Prelamin A is involved in early steps of muscle differentiation

Gruenbaum, 2005, The nuclear lamina comes of age, Nat. Rev. Mol. Cell Biol., 6, 21, 10.1038/nrm1550

Rusinol, 2006, Farnesylated lamins, progeroid syndromes and farnesyl transferase inhibitors, J. Cell. Sci., 119, 3265, 10.1242/jcs.03156

Chaly, 1996, Remodelling of the nuclear periphery during muscle cell differentiation in vitro, J. Cell. Biochem., 62, 76, 10.1002/(SICI)1097-4644(199607)62:1<76::AID-JCB9>3.0.CO;2-Z

Muralikrishna, 2001, Distinct changes in intranuclear lamin A/C organization during myoblast differentiation, J. Cell. Sci., 114, 4001, 10.1242/jcs.114.22.4001

Lattanzi, 2003, Association of emerin with nuclear and cytoplasmic actin is regulated in differentiating myoblasts, Biochem. Biophys. Res. Commun., 303, 764, 10.1016/S0006-291X(03)00415-7

Cenni, 2005, Lamin A N-terminal phosphorylation is associated with myoblast activation: impairment in Emery–Dreifuss muscular dystrophy, J. Med. Genet., 42, 214, 10.1136/jmg.2004.026112

Lourim, 1992, Expression of wild-type and nuclear localization-deficient human lamin A in chick myogenic cells, J. Cell. Sci., 103, 863, 10.1242/jcs.103.3.863

Frock, 2006, Lamin A/C and emerin are critical for skeletal muscle satellite cell differentiation, Genes Dev., 20, 486, 10.1101/gad.1364906

Vlcek, 2007, Lamins and lamin-associated proteins in aging and disease, Curr. Opin. Cell Biol., 19, 298, 10.1016/j.ceb.2007.04.001

Favreau, 2004, Expression of a mutant lamin A that causes Emery–Dreifuss muscular dystrophy inhibits in vitro differentiation of C2C12 myoblasts, Mol. Cell. Biol., 24, 1481, 10.1128/MCB.24.4.1481-1492.2004

Markiewicz, 2005, Remodelling of the nuclear lamina and nucleoskeleton is required for skeletal muscle differentiation in vitro, J. Cell. Sci., 118, 409, 10.1242/jcs.01630

Kirschner, 2005, p.S143F mutation in lamin A/C: a new phenotype combining myopathy and progeria, Ann. Neurol., 57, 148, 10.1002/ana.20359

Spuler, 2007, Muscle and nerve pathology in Dunnigan familial partial lipodystrophy, Neurology, 68, 677, 10.1212/01.wnl.0000255939.73424.f8

Broers, 2006, Nuclear lamins: laminopathies and their role in premature ageing, Physiol. Rev., 86, 967, 10.1152/physrev.00047.2005

Hennekam, 2006, Hutchinson–Gilford progeria syndrome: review of the phenotype, Am. J. Med. Genet. A, 140, 2603, 10.1002/ajmg.a.31346

Maraldi, 2007, Involvement of prelamin A in laminopathies, Crit. Rev. Eukaryot. Gene Expr., 17, 317, 10.1615/CritRevEukarGeneExpr.v17.i4.50

Capanni, 2005, Altered pre-lamin A processing is a common mechanism leading to lipodystrophy, Hum. Mol. Genet., 14, 1489, 10.1093/hmg/ddi158

Maraldi, 2006, Nuclear envelope proteins and chromatin arrangement: a pathogenic mechanism for laminopathies, Eur. J. Histochem., 50, 1

Lattanzi, 2007, Pre-Lamin A processing is linked to heterochromatin organization, J. Cell. Biochem., 102, 1149, 10.1002/jcb.21467

Mattioli, 2008, Drugs affecting prelamin A processing: effects on heterochromatin organization, Exp. Cell Res., 314, 453, 10.1016/j.yexcr.2007.11.012

Dorner, 2006, Lamina-associated polypeptide 2alpha regulates cell cycle progression and differentiation via the retinoblastoma-E2F pathway, J. Cell Biol., 173, 83, 10.1083/jcb.200511149

Capozza, 2005, Caveolin-3 knockout mice show increased adiposity and whole body insulin resistance, with ligand-induced insulin receptor instability in skeletal muscle, Am. J. Physiol. Cell Physiol., 288, C1317, 10.1152/ajpcell.00489.2004

Johnson, 2004, Statins induce apoptosis in rat and human myotube cultures by inhibiting protein geranylgeranylation but not ubiquinone, Toxicol. Appl. Pharmacol., 200, 237, 10.1016/j.taap.2004.04.010

Dagenais, 1985, Detection in BHK cells of a precursor form for lamin A, Exp. Cell Res., 161, 269, 10.1016/0014-4827(85)90084-9

Shackleton, 2005, Compound heterozygous ZMPSTE24 mutations reduce prelamin A processing and result in a severe progeroid phenotype, J. Med. Genet., 42, e36, 10.1136/jmg.2004.029751

Melcon, 2006, Loss of emerin at the nuclear envelope disrupts the Rb1/E2F and MyoD pathways during muscle regeneration, Hum. Mol. Genet., 15, 637, 10.1093/hmg/ddi479

Taylor, 2005, Thymopoietin (lamina-associated polypeptide 2) gene mutation associated with dilated cardiomyopathy, Hum. Mutat., 26, 566, 10.1002/humu.20250

Pekovic, 2007, Nucleoplasmic LAP2alpha-lamin A complexes are required to maintain a proliferative state in human fibroblasts, J. Cell Biol., 176, 163, 10.1083/jcb.200606139

Scaffidi, 2005, Reversal of the cellular phenotype in the premature aging disease Hutchinson–Gilford progeria syndrome, Nat. Med., 11, 440, 10.1038/nm1204

Caron, 2007, Human lipodystrophies linked to mutations in A-type lamins and to HIV protease inhibitor therapy are both associated with prelamin A accumulation, oxidative stress and premature cellular senescence, Cell Death Differ., 14, 1759, 10.1038/sj.cdd.4402197

Columbaro, 2005, Rescue of heterochromatin organization in Hutchinson–Gilford progeria by drug treatment, Cell. Mol. Life Sci., 62, 2669, 10.1007/s00018-005-5318-6

Filesi, 2005, Alterations of nuclear envelope and chromatin organization in mandibuloacral dysplasia, a rare form of laminopathy, Physiol. Genomics, 23, 150, 10.1152/physiolgenomics.00060.2005

Shumaker, 2006, Mutant nuclear lamin A leads to progressive alterations of epigenetic control in premature aging, Proc. Natl. Acad. Sci. U. S. A., 103, 8703, 10.1073/pnas.0602569103

Volonte, 2003, Modulation of myoblast fusion by caveolin-3 in dystrophic skeletal muscle cells: implications for Duchenne muscular dystrophy and limb-girdle muscular dystrophy-1C, Mol. Biol. Cell, 14, 4075, 10.1091/mbc.e03-03-0161

Shimizu, 2005, Gene mutations in adult Japanese patients with dilated cardiomyopathy, Circ. J., 69, 150, 10.1253/circj.69.150