Multiple, brief coronary occlusions during early reperfusion protect rabbit hearts by targeting cell signaling pathways

Journal of the American College of Cardiology - Tập 44 - Trang 1103-1110 - 2004
Xi-Ming Yang1, J. Bradley Proctor1, Lin Cui1, Thomas Krieg1, James M. Downey1, Michael V. Cohen1,2
1Physiology
2Medicine, University of South Alabama, College of Medicine, Mobile, Alabama, USA

Tài liệu tham khảo

Cohen, 2001, Ischemic preconditioning: description, mechanism, and significance, 867 Becker, 1987, Myocardial consequences of reperfusion, Prog Cardiovasc Dis, 30, 23, 10.1016/0033-0620(87)90009-0 Farb, 1993, Myocardial infarct extension during reperfusion after coronary artery occlusion: pathologic evidence, J Am Coll Cardiol, 21, 1245, 10.1016/0735-1097(93)90253-W Vanden Hoek, 1996, Reperfusion injury in cardiac myocytes after simulated ischemia, Am J Physiol, 270, H1334 Matsumura, 1998, Progression of myocardial necrosis during reperfusion of ischemic myocardium, Circulation, 97, 795, 10.1161/01.CIR.97.8.795 Piper, 1998, A fresh look at reperfusion injury, Cardiovasc Res, 38, 291, 10.1016/S0008-6363(98)00033-9 Olafsson, 1987, Reduction of reperfusion injury in the canine preparation by intracoronary adenosine, Circulation, 76, 1135, 10.1161/01.CIR.76.5.1135 Goto, 1991, Adenosine infusion during early reperfusion failed to limit myocardial infarct size in a collateral deficient species, Cardiovasc Res, 25, 943, 10.1093/cvr/25.11.943 Todd, 1996, Intravascular adenosine at reperfusion reduces infarct size and neutrophil adherence, Ann Thorac Surg, 62, 1364, 10.1016/0003-4975(96)00495-X Vander Heide, 1996, Effect of adenosine therapy at reperfusion on myocardial infarct size in dogs, Cardiovasc Res, 31, 711, 10.1016/S0008-6363(95)00235-9 Xu, 2001, AMP 579 reduces contracture and limits infarction in rabbit heart by activating adenosine A2receptors, J Cardiovasc Pharmacol, 38, 474, 10.1097/00005344-200109000-00016 Smits, 1998, Cardioprotective effects of the novel adenosine A1/A2receptor agonist AMP 579 in a porcine model of myocardial infarction, J Pharmacol Exp Ther, 286, 611 Budde, 2000, Comparative study of AMP579 and adenosine in inhibition of neutrophil-mediated vascular and myocardial injury during 24 h of reperfusion, Cardiovasc Res, 47, 294, 10.1016/S0008-6363(00)00115-2 Baxter, 2000, Amp579, an A1/A2A agonist, limits infarct size at reperfusion via a p42/p44 MAPK-dependent pathway, Circulation, 102, II212 Xu, 2000, Limitation of infarct size in rabbit hearts by the novel adenosine receptor agonist AMP 579 administered at reperfusion, J Mol Cell Cardiol, 32, 2339, 10.1006/jmcc.2000.1264 Yang, 2004, NECA and bradykinin at reperfusion reduce infarction in rabbit hearts by signaling through PI3K, ERK, and NO, J Mol Cell Cardiol, 36, 411, 10.1016/j.yjmcc.2003.12.008 Zhang, 2003, CGX-1051, a peptide from Conus snail venom, attenuates infarction in rabbit hearts when administered at reperfusion, J Cardiovasc Pharmacol, 42, 764, 10.1097/00005344-200312000-00011 Bell, 2003, Bradykinin limits infarction when administered as an adjunct to reperfusion in mouse heart: the role of PI3K, Akt and eNOS, J Mol Cell Cardiol, 35, 185, 10.1016/S0022-2828(02)00310-3 Liao, 2002, Cardiotrophin-1 (CT-1) can protect the adult heart from injury when added both prior to ischaemia and at reperfusion, Cardiovasc Res, 53, 902, 10.1016/S0008-6363(01)00531-4 Jonassen, 2001, Myocardial protection by insulin at reperfusion requires early administration and is mediated via Akt and p70s6 kinase cell-survival signaling, Circ Res, 89, 1191, 10.1161/hh2401.101385 Baxter, 2001, Cardioprotective effects of transforming growth factor-β1 during early reoxygenation or reperfusion are mediated by p42/p44 MAPK, J Cardiovasc Pharmacol, 38, 930, 10.1097/00005344-200112000-00015 Zhao, 2003, Inhibition of myocardial injury by ischemic postconditioning during reperfusion: comparison with ischemic preconditioning, Am J Physiol, 285, H579 Yao, 1994, A comparison of adenosine-induced cardioprotection and ischemic preconditioning in dogs: efficacy, time course, and role of KATPchannels, Circulation, 89, 1229, 10.1161/01.CIR.89.3.1229 Takano, 2000, Differential role of KATPchannels in late preconditioning against myocardial stunning and infarction in rabbits, Am J Physiol, 279, H2350 Fryer, 2001, ERK and p38 MAP kinase activation are components of opioid-induced delayed cardioprotection, Basic Res Cardiol, 96, 136, 10.1007/s003950170063 Patel, 1993, Inhibition of nitric oxide limits infarct size in the in siturabbit heart, Biochem Biophys Res Commun, 194, 234, 10.1006/bbrc.1993.1809 Murry, 1986, Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium, Circulation, 74, 1124, 10.1161/01.CIR.74.5.1124 Oldenburg, 2003, Mitochondrial KATPchannels in preconditioning, J Mol Cell Cardiol, 35, 569, 10.1016/S0022-2828(03)00115-9 Oldenburg, 2003, Bradykinin induces mitochondrial ROS generation via NO, cGMP, PKG, and mitoKATPchannel opening and leads to cardioprotection, Am J Physiol, 286, H468 Xu, 2003, Timing and duration of administration are crucial for antiinfarct effect of AMP 579 infused at reperfusion in rabbit heart, Heart Dis, 5, 368, 10.1097/01.hdx.0000098614.29006.a7 Komalavilas, 1999, Activation of mitogen-activated protein kinase pathways by cyclic GMP and cyclic GMP-dependent protein kinase in contractile vascular smooth muscle cells, J Biol Chem, 274, 34301, 10.1074/jbc.274.48.34301 Kim, 1999, Ischemia induced activation of heat shock protein 27 kinases and casein kinase 2 in the preconditioned rabbit heart, Biochem Cell Biol, 77, 559, 10.1139/o99-065