No evidence for an association between the -871 T/C promoter polymorphism in the B-cell-activating factor gene and primary Sjögren's syndrome

Arthritis Research & Therapy - Tập 8 - Trang 1-5 - 2006
Jacques-Eric Gottenberg1, Jérémie Sellam1, Marc Ittah1, Frédéric Lavie1, Alexis Proust2, Habib Zouali3, Christelle Sordet4, Jean Sibilia4, Robert P Kimberly5, Xavier Mariette1, Corinne Miceli-Richard1
1Rhumatologie, INSERM E 109, Hôpital Bicêtre, Assistance Publique-Hôpitaux de Paris, Université Paris-Sud 11, Le Kremlin Bicêtre, France
2Hématologie, INSERM E 109, Hôpital de Bicêtre, Assistance Publique-Hôpitaux de Paris (AP-HP), Le Kremlin Bicêtre, France
3Fondation Jean Dausset, Centre d'Etude du Polymorphisme Humain (CEPH), Paris, France
4Rhumatologie, Centre Hospitalier Universitaire de Strasbourg, Strasbourg, France
5Division of Clinical Immunology and Rheumatology, University of Alabama, Birmingham, USA

Tóm tắt

Polyclonal B cell activation might be related to pathogenic over-expression of B-cell-activating factor (BAFF) in primary Sjögren's syndrome (pSS) and other autoimmune diseases. We therefore investigated whether BAFF over-expression in pSS could be a primary, genetically determined event that leads to the disease. The complete BAFF gene was sequenced in Caucasian pSS patients and control individuals. The only single nucleotide polymorphism frequently observed, namely -871 T/C in the promoter region, was then genotyped in 162 French patients with pSS and 90 French control individuals. No significant differences in allele (T allele frequency: 49.7% in patients with pSS versus 50% in controls; P = 0.94) and genotype frequencies of BAFF polymorphism were detected between pSS patients and control individuals. BAFF gene polymorphism was not associated with a specific pattern of antibody secretion either. T allele carriers had significantly increased BAFF protein serum levels (mean values of 8.6 and 5.7 ng/ml in patients with TT and TC genotypes, respectively, versus 3.3 ng/ml in patients with CC genotype; P = 0.01), although no correlation was observed between BAFF polymorphism and mRNA level. In conclusion, BAFF gene polymorphism is neither involved in genetic predisposition to pSS nor associated with a specific pattern of antibody production.

Tài liệu tham khảo

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