Rat two-generation reproductive toxicity study of bisphenol A

Krishnan, 1993, Bisphenol-A, Endocrinology, 132, 2279, 10.1210/en.132.6.2279 Olea, 1996, Estrogenicity of resin-based composites and sealants used in dentistry, Environ Health Perspect, 104, 298, 10.1289/ehp.96104298 Nagel, 1977, Relative binding affinity-serum modified access (RBA-SMA) assay predicts the relative in vivo bioactivity of the xenoestrogens bisphenol A and octylphenol, Environ Health Perspect, 105, 70, 10.1289/ehp.9710570 Gaido, 1997, Evaluation of chemicals with endocrine modulating activity in a yeast-based steroid hormone receptor gene transcription assay, Toxicol Appl Pharmacol, 143, 205, 10.1006/taap.1996.8069 Sohoni, 1998, Several environmental oestrogens are also anti-androgens, J Endocrinol, 158, 327, 10.1677/joe.0.1580327 Milligan, 1998, Relative potency of xenobiotic estrogens in an acute in vivo mammalian assay, Environ Health Perspect, 106, 23, 10.1289/ehp.9810623 Law, 2000, Estrogenic activity of octylphenol, nonylphenol, bisphenol A and methoxychlor in rats, Toxicol Sci, 54, 154, 10.1093/toxsci/54.1.154 Goloubkova, 2000, Effects of xenoestrogen bisphenol A on uterine and pituitary weight, serum prolactin levels and immunoreactive prolactin cells in ovariectomized Wistar rats, Arch Toxicol, 74, 92, 10.1007/s002040050658 Morrisey, 1987, The developmetal toxicity of bisphenol A in rats and mice, Fundam Appl Toxicol, 8, 571, 10.1016/0272-0590(87)90142-4 Morrissey, 1989, Results and evaluations of 48 continuous breeding reproduction studies conducted in mice, Fundam Appl Toxicol, 13, 747, 10.1016/0272-0590(89)90332-1 Kwon, 2000, Pubertal development and reproductive functions of crl, Toxicol Sci., 55, 399, 10.1093/toxsci/55.2.399 vom Saal, 1998, A physiologically based approach to the study of bisphenol A, and other estrogenic chemicals on the size of reproductive organs, daily sperm production, and bihavior, Toxicol Industrial Health, 14, 239, 10.1177/074823379801400115 Gupta, 2000, Reproductive malformation of the male offspring following maternal exposure to estrogenic chemicals, Proc Soc Exp Biol Med, 224, 61, 10.1046/j.1525-1373.2000.22402.x Cagen, 1999, Normal reproductive organ development in CF-1 mice following prenatal exposure to bisphenol A, Toxicol Sci, 50, 36, 10.1093/toxsci/50.1.36 Ashby, 1999, Lack of effects for low dose levels of bisphenol A and diethylstilbestrol on the prostate gland of CF1 mice exposed in utero, Regulatory Toxicol Pharmacol, 30, 156, 10.1006/rtph.1999.1317 Cagen, 1999, Normal reproductive organ development in Wistar rats exposed to bisphenol A in the drinking water, Regulatory Toxicol Pharmacol, 30, 130, 10.1006/rtph.1999.1340 Elswick, 2000, Effects of perinatal exposure to low doses of bisphenol A in male offspring of Sprague-Dawley rats, Toxicol Sci, 54, 256 Welsch, 2000, Effects of perinatal expsure to low doses of bisphenol A on female offspring of Spragure-Dawley rats, Toxicol Sci, 54, 256 MAFF (Ministry of Agriculture, Forestry, and Fisheries) of Japan. Guidance on Toxicological Study Data for Application of Agriculture Chemical Registration, 59 NohSan No. 4200, January 1985. OECD (Organization for Economic Cooperation, and Development). OECD Guideline for Testing of Chemicals. 416 Two-Generation Reproduction Toxicity Study, May. 1983. ICH (International Conference on Harmonisation of Technical Requirements for Registration of Pharmaceuticals for Human Use). ICH Harmonized Tripartite Guideline: Detection of Toxicity to Reproduction for Medicinal Products, June. 1993. EA (Environment Agency), MHW (Ministry of Health, and Welfare), and MITI (Ministry of International Trade, and Industry) of Japan. Chemical Substances Control Law, November 1988. Biel, 1940, Early age differences in maze performance in the albino rats, J Genet Psychol, 56, 439 Vandenbergh, 1995, The anogenital distance index, a predictor of the intrauterine position effects on reproduction in female house mice, Laboratory Anim Sci, 45, 567 Gallavan, 1999, Interpreting the toxicologic significance of alterations in anogenital distance, Reprod Toxicol, 13, 383, 10.1016/S0890-6238(99)00036-2 Farabollini, 1999, Perinatal exposure to the estrogenic pollutant bisphenol A affects behavior in male and female rats, Pharmacol Biochem Behavior, 64, 687, 10.1016/S0091-3057(99)00136-7 Steinmetz, 1997, The environmental estrogen bisphenol A stimulates prolactin release in vitro and in vivo, Endocrinology, 138, 1780, 10.1210/en.138.5.1780 Steinmets, 1998, The xenoestrogen bisphenol A induces growth, differentiation, and c-fos gene expression in the female reproductive tract, Endocrinology, 139, 2741, 10.1210/en.139.6.2741 Reel, 1996, Survey and assessment of mammalian estrogen biological assays for hazard characterization, Fundam Appl Toxicol, 34, 288, 10.1006/faat.1996.0198 Sheehan, 2000, Activity of environmentally relevant low doses of endocrine disruptors and the bisphenol A controversy, Proc Soc Exp Biol Med, 224, 57, 10.1046/j.1525-1373.2000.22401.x Ashby, 1998, Uterotrophic activity of bisphenol A in the immature rat, Environ Health Perspect, 106, 719, 10.1289/ehp.98106719 Papaconstantinou, 2000, Bisphenol A-induced increase in uterine weight, and alterations in uterine morphology in ovariectomized B6C3F1 mice, Toxicol Sci, 56, 332, 10.1093/toxsci/56.2.332 Nagao, 1999, Reproductive function in rats exposed neonatally to bisphenol A and estradiol benzoate, Reprod toxicol, 13, 303, 10.1016/S0890-6238(99)00017-9 Colerangle, 1997, Profound effects of the weak environmental estrogen-like chemical bisphenol A on the growth of the mammary gland of Noble rats, J Steroid Biochem MolBiol, 60, 153, 10.1016/S0960-0760(96)00130-6 Howdeshell, 1999, Exposure to bisphenol A advances puberty, Nature, 401, 763, 10.1038/44517 Miyakoda, 1999, Passage of bisphenol A into fetus of the pregnant rat, J Health Sci, 45, 318, 10.1248/jhs.45.318 Takahashi, 2000, Disposion of orally administered 2,2-bis(4-hydroxyphenol)propane (bisphenol A) in pregnant rats and the placental transfer to fetuses, Environ Health Perspect, 108, 931, 10.1289/ehp.00108931 Pottenger, 2000, The relative bioavailability and metabolism of bisphenol A in rats is dependent upon the route of administration, Toxicol Sci, 54, 3, 10.1093/toxsci/54.1.3