Post-translational glycosylation of target proteins implicate molecular mimicry in the pathogenesis of HTLV-1 associated neurological disease

Journal of Neuroimmunology - Tập 204 - Trang 140-148 - 2008
Sangmin Lee1,2,3, Yoojin Shin1,2,3, Jacob Marler1,2, Michael C. Levin1,2,4,3,5
1Research Service, Veterans Affairs Medical Center, Memphis, TN, USA
2Department of Neurology, University of Tennessee Health Sciences Center, Memphis, TN, USA
3Center for the Neurobiology of Brain Diseases, University of Tennessee Health, Sciences Center, Memphis, TN, USA
4Department of Anatomy and Neurobiology, University of Tennessee Health Sciences Center, Memphis, TN, USA
5Center of Excellence in Connective Tissue Diseases, University of Tennessee Health, Sciences Center, Memphis, TN, USA

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Thông tin xuất bản

Journal of Neuroimmunology

Tập 204

140-148

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