Identification of an Autophagy Defect in Smokers’ Alveolar Macrophages

Journal of Immunology - Tập 185 Số 9 - Trang 5425-5435 - 2010
Martha M. Monick1, Linda S. Powers2, Katherine S. Walters3, Nina Lovan2, Michael S. Zhang2, Alicia K. Gerke2, Sif Hansdóttir2, Gary W. Hunninghake2,4
1Department of Medicine, Carver College of Medicine, University of Iowa, Iowa City, IA 52242, USA.
2*Department of Medicine, Carver College of Medicine,
3†Office of the Vice President for Research, and
4‡Institute for Clinical and Translational Science, University of Iowa, Iowa City, IA 52242

Tóm tắt

Abstract

Alveolar macrophages are essential for clearing bacteria from the alveolar surface and preventing microbe-induced infections. It is well documented that smokers have an increased incidence of infections, in particular lung infections. Alveolar macrophages accumulate in smokers’ lungs, but they have a functional immune deficit. In this study, we identify an autophagy defect in smokers’ alveolar macrophages. Smokers’ alveolar macrophages accumulate both autophagosomes and p62, a marker of autophagic flux. The decrease in the process of autophagy leads to impaired protein aggregate clearance, dysfunctional mitochondria, and defective delivery of bacteria to lysosomes. This study identifies the autophagy pathway as a potential target for interventions designed to decrease infection rates in smokers and possibly in individuals with high environmental particulate exposure.

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