Genomic mutations in the katG, inhA and aphC genes are useful for the prediction of isoniazid resistance in Mycobacterium tuberculosis isolates from Kwazulu Natal, South Africa
Tài liệu tham khảo
Telenti, 1993, Detection of rifampicin-resistance mutations in Mycobacterium tuberculosis, Lancet, 341, 647, 10.1016/0140-6736(93)90417-F
Zhang, 1992, The catalase peroxidase gene and isoniazid resistance of Mycobacterium tuberculosis, Nature, 358, 501, 10.1038/358591a0
Banerjee, 1994, inhA a gene encoding a target for isoniazid and ethionomide in Mycobacterium tuberculosis, Science, 263, 227, 10.1126/science.8284673
Rozwarski, 1998, Modification of the NADH of the isoniazid target (inhA) from Mycobacterium tuberculosis, Science, 279, 98, 10.1126/science.279.5347.98
Musser, 1996, Characterization of the catalase-peroxidase gene (katG) and inhA locus in isoniazid-resistant and -susceptible strains of Mycobacterium tuberculosis by automated DNA sequencing: restricted array of mutations associated with drug resistance, J Inf Dis, 173, 196, 10.1093/infdis/173.1.196
Deretic, 1995, Mycobacterium tuberculosis is a natural mutant with an inactivated oxidative- strees regulatory gene: implications for sensitivity to isoniazid, Mol Microbiol, 17, 889, 10.1111/j.1365-2958.1995.mmi_17050889.x
Sherman, 1995, Disparate responses to oxidative stress in saprophytic and pathogenic mycobacteria, Proc Natl Acad Sci USA, 92, 6625, 10.1073/pnas.92.14.6625
Wilson, 1996, ahpC, a gene involved in isoniazid resistance of the Mycobacterium tuberculosis complex, Mol Microbiol, 19, 1025, 10.1046/j.1365-2958.1996.449980.x
Mduli, 1998, Inhibition of a Mycobacterium tuberculosis beta Ketoacyl ACP synthase by isoniazid, Science, 28, 1607, 10.1126/science.280.5369.1607
Smith, A, N, Personal communication, Department of Virology, University of Natal, Kwazulu Natal, Durban, South Africa
Kiepiela, 1998, Comparison of PCR-heteroduplex, Line probe assay and characterization by automated sequencing of rifampicin resistance in Mycobacterium tuberculosis strains in Kwazulu Natal, South Africa. Microbiol, Drug Resistance, 4, 260
Kent, P, T, Kubica, G, P, Public health mycobacteriology – guide for the level III laboratory. Centers for Disease Control, U.S, 1985
Wellstood, 1993, Diagnostic mycobacteriology: current challenges and technologies, Lab Med, 24, 357, 10.1093/labmed/24.6.357
Heifets, 1991
Griffith, 1967, Drug susceptibility tests for tuberculosis using drug impregnated discs, Am Clin Pathol, 47, 812, 10.1093/ajcp/47.6_ts.812
Boom, 1990, Rapid and simple method for purification of nucleic acids, J Clin Microbiol, 28, 495, 10.1128/JCM.28.3.495-503.1990
Heym, 1995, Missense mutations in the catalase-peroxidase gene. KatG, are associated with isoniazid resistance inMycobacterium tuberculosis, Mol Microbiol, 15, 235, 10.1111/j.1365-2958.1995.tb02238.x
Telenti, 1997, Genotypic assessment of isoniazid and rifampicin resistance in Mycobacterium tuberculosis: a blind study at reference laboratory level, J Clin Microbiol, 35, 719, 10.1128/JCM.35.3.719-723.1997
Warren, 1996, Genotyoping of Mycobacterium tuberculosis with additional markers enhances accuracy in epidemiological studies, J Clin Microbiol, 34, 2219, 10.1128/JCM.34.9.2219-2224.1996
Van Emden, 1993, Strain identification of Mycobacterium tuberculosis by DNA fingerprinting: recommendation for a standardised methodology, J Clin Microbiol, 31, 406, 10.1128/JCM.31.2.406-409.1993
Cave, 1994, Stability of DNA fingerprinting pattern produced with IS6110 in strains of Mycobacterium tuberculosis, J Clin Mircobiol, 32, 262, 10.1128/JCM.32.1.262-266.1994
Yang, 1994, Restriction fragment length polymorphism of Mycobacterium tuberculosis strains isolated from Greenland during 1992: evidence of tuberculosis transmission between Greenland and Denmark, J Clin Microbiol, 32, 3018, 10.1128/JCM.32.12.3018-3025.1994
Van Soolingen, 1994, DNA fingerprinting of Mycobacterium tuberculosis, Meth Enzymol, 235, 196, 10.1016/0076-6879(94)35141-4
Zhang, 1996, Molecular basis for the exquisite sensitivity of Mycobacterium tuberculosis to isoniazid, Proc Natl Acad Sci USA, 93, 1312, 10.1073/pnas.93.23.13212
Sreevatsan, 1997, Analysis of the oxyR-ahpc region in isoniazid resistant and – susceptible Mycobacterium tuberculosis– complex organisms recovered from diseased humans and animals in diverse localities, Antimicrob Agents Chemother, 41, 600, 10.1128/AAC.41.3.600
Uhi, J, R, Cockerill, III, F R, Kline, B, C, Rapid simultaneous detection of Mycobacterium tuberculosis (MTB) and isoniazid resistance directly from sputum, 1997
Cockerill, 1995, Rapid identification of a point mutation of the Mycobacterium tuberculosis catalase-peroxidase (katG) gene associated with isoniazid resistance, J Inf Dis, 175, 240, 10.1093/infdis/171.1.240
Rouse, 1995, Characterization of the katG and inhA genes of isoniazid resistant clinical isolates ofMycobacterium tuberculosis, Antimicrob Agents Chemother, 39, 2472, 10.1128/AAC.39.11.2472
Pretorius, 1995, Mutations in katG gene sequences in isoniazid-resistant clinical isolates of Mycobacterium tuberculosis are rare, Antimicrob Agents Chemother, 39, 2276, 10.1128/AAC.39.10.2276
Morris, 1995, Molecular mechanisms of multiple drug resistance in clinical isolates of Mycobacterium tuberculosis, J Infect Dis, 171, 954, 10.1093/infdis/171.4.954
Martilla, 1996, katG mrtations in isoniazid-resistant Mycobacterium tuberculosis isolates recovered from finnish patients, Antimicrob Agents Chemother, 40, 2187, 10.1128/AAC.40.9.2187
Donber, 1997, The usefulness of Mycobacterium tuberculosis genomic mutations in the genes katG and inhA for the prediction of isoniazid resistance, Int J Tuberc Lung Dis, 1, 365
Namchankin, 1997, Detection of resistance to isoniazid, rifampicin and streptomycin in clinical isolates of Mycobacterium tuberculosis by molecular methods, Clin Inf Dis, 24, 894, 10.1093/clinids/24.5.894
Haas, 1997, Molecular analysis of katG gene mutations in strains of Mycobacterium tuberculosis complex from Africa, Antimicrob Agents Chemother, 41, 1601, 10.1128/AAC.41.7.1601
Wengenack, 1997, Recombinant Mycobacterium tuberculosis KatG (S315T) is a competent catalase-peroxidase with reduced activity toward isoniazid, J Infect Dis, 176, 722, 10.1086/514096
Altamirano, 1994, Mutations in the catalase–peroxidase gene from isoniazid-resistant Mycobacterium tuberculosis isolates, J Infect Dis, 169, 1162, 10.1093/infdis/169.5.1162
Heym, 1994, Implications of multidrug resistance for the future short-course chemotherapy of tuberculosis: a molecular study, Lancet, 344, 293, 10.1016/S0140-6736(94)91338-2
Stoeckle, 1993, Catalase-peroxidase gene sequences in isoniazid-sensitive and resistant strains of Mycobacterium tuberculosis from New York City, J Infect Dis, 168, 1063, 10.1093/infdis/168.4.1063
Kapur, 1995, Rapid Mycobacterium species assignment and unambiguous identification of mutations associated with antimicrobial resistance in Mycobacterium tuberculosis by automated DNA sequencing, Arch Pathol Lab Med, 119, 131
Ristow, 1995, New isoniazid/ethionamide resistance gene mutation and screening for multi-drug resistant Mycobacterium tuberculosis strains, Lancet, 346, 502, 10.1016/S0140-6736(95)91351-3
Glavac, 1993, Optimization of the single-strand conformation polymorphism (SSCP) technique for detection of point mutations, Hum Mutat, 2, 404, 10.1002/humu.1380020513
Sheffield, 1993, The sensitivity of single-strand conformation polymorphism analysis for the detection of single base substitutions, Genomics, 16, 325, 10.1006/geno.1993.1193
Victor, 1996, katG mutations in isoniazid-resistant strains of Mycobacterium tuberculosis are not infrequent (Letter to the editor), Antimicrob Agents Chemother, 40, 1572, 10.1128/AAC.40.6.1572