Enhanced Electron Flux and Reduced Calmodulin Dissociation May Explain “Calcium-independent” eNOS Activation by Phosphorylation

Journal of Biological Chemistry - Tập 275 - Trang 6123-6128 - 2000
Timothy J. McCabe1, David Fulton1, Linda J. Roman2, William C. Sessa1
1Department of Pharmacology and Molecular Cardiobiology Program, Boyer Center for Molecular Medicine, Yale University School of Medicine, New Haven, Connecticut 06536
2Department of Biochemistry, The University of Texas Health Science Center at San Antonio, Texas 78284-3900