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Als Kontrollgruppe diente endomyokardiales Gewebe von sieben Individuen bei denen letztlich eine Herzerkrankung ausgeschlossen werden konnte.¶   Ergebnisse: Die Menge an EXCH mRNA betrug im Myokard der Kontrollgruppe 2,6±1,2amol\u002Fng totaler RNA und unterschied sich nicht von der Menge an EXCH mRNA bei AS (1,8±1,4amol\u002Fng totaler RNA), AI (1,9±0,8amol\u002Fng) oder MI (2,2±2,1amol\u002Fng). Im Myokard explantierter terminal insuffizienter Herzen fand sich dagegen eine deutlich erhöhte Menge an EXCH mRNA (8,9±1,9amol\u002Fng).¶Zwischen der EXCH-Transkription und dem Schweregrad des Vitiums oder der konsekutiven Einschränkung der linksventrikulären Pumpfunktion bestand kein Zusammenhang: Cardiac Index (CI) >3,5l\u002Fmin\u002Fm2 (EXCH 1,4±1,1amol\u002Fng totaler RNA); CI 3,5–2,4 (EXCH 2,5±1,8); CI \u003C2,4 (EXCH 1,8±1,0); EF-angio >50% (EXCH 1,9±1,8), EF-angio ≤50% (EXCH 1,9±0,9); EF-RNV >50% (EXCH 2,4±1,8), EF-RNV ≤50% (EXCH 1,7±1,0).¶   Schlussfolgerung: Die myokardialen EXCH-Transkriptionsmengen ändern sich bei HKF nicht parallel zur Ausbildung einer myokardialen Pumpfunktionsstörung. 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