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Journal of Medicine and Pharmacy","Tạp chí Y Dược học Cần Thơ",{"EN":487,"VI":488},"\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">04\u002F10\u002F2015 Ministry of Information and Communications allowed Can Tho journal of medicine and pharmacy to operate (102 \u002FGP-BTTTT)\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">07\u002F16\u002F2015 Can Tho journal of medicine and pharmacy is internationally recognized: ISSN 2354-1210\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">In 2016, The journal has been included in the list of medical science journals by The State Council for professorship which is awarded a work score of 0-0.5 points for a published article.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Can Tho Journal of Medicine and Pharmacy welcome original works that haven’t been submitted or published in other medical journals. Posts must contain content related to one of the journal’s categories.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The content published\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The journal is divided into 3 categories:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Scientific research article: are valuable scientific works, which have been researched and accepted.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Overview of medicine, biology and pharmacy: serving the objective of continuing training in the fields of medicine, biology and pharmacy; to systematize classical and modern knowledge.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Update information on new knowledge about medicine, biology, pharmacy in the country and in the world.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Scope\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Publication and introduction of scientific research in the fields:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Medicine (internal medicine, surgery, pediatrics, obstetrics and gynecology, odonto-stomatology, laboratory, oncology, traditional medicine, nursing).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Biology (genetics, biotechnology).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Pharmacology (pharmaceutics, drug quality analysis-control, synthetic pharmaceutical chemistry, biochemistry, pharmacognosy, botany, clinical pharmacy).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- To enhance the quality of undergraduate, postgraduate education, scientifically researching and meet the necessary treatment in hospital.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Introducing the updated domestic and oversea information about science technology to promote scientific research and exchanging technology in local, other universities.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Exchanging pharmaceutical and medical information for social health developing in the Mekong Delta and Vietnam.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The object\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Postgraduate students, student of Can Tho University of Medicine and Pharmacy, scientists from schools, research institutes, hospitals, health centers, pharmaceutical companies of the Mekong Delta; other provinces and regions in Vietnam and other country.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Address\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Headquarters of Can Tho Journal of Medicine and Pharmacy, located Scientific Research and International Cooperation Office: 179 Nguyen Van Cu Street, An Khanh Ward, Ninh Kieu District, Can Tho City, Vietnam.\u003C\u002Fspan>\u003C\u002Fp>","\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Ngày 16\u002F7\u002F2015, Tạp chí Y Dược học Cần Thơ được cấp chỉ số quốc tế: ISSN 2354-1210.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 4\u002F2016, Tạp chí đã được Hội đồng Giáo sư ngành Y đưa vào danh sách các tạp chí khoa học Y học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Năm 2020 Tạp chí Y Dược học Cần Thơ đã được phê duyệt vào danh mục của các Hội đồng Giáo sư ngành Dược học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ ra 12 số\u002Fnăm, 180-200 trang\u002Fsố.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 12\u002F2022 Tạp chí Y Dược học Cần Thơ là thành viên của hệ thống Crossref và từ tháng 01\u002F2023 tạp chí thực hiện bình duyệt online kín 2 chiều nhằm tăng tính minh bạch, tin cậy của các công trình nghiên cứu khoa học và đảm bảo tốt nhất chất lượng khoa học của bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ, mục đích và phạm vi của tạp chí\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ và mục đích hoạt động của tạp chí: xuất bản nhằm mục đích phổ biến kết quả từ các đề tài nghiên cứu khoa học; giao lưu trao đổi khoa học, chia sẻ kinh nghiệm, học tập, đồng thời cập nhật thông tin khoa học mới trong các lĩnh vực y, sinh, dược học trong và ngoài nước.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phạm vi của tạp chí: Tạp chí xuất bản được chia thành 3 chuyên mục: (i) Bài báo nghiên cứu khoa học là kết quả công trình nghiên cứu khoa học có giá trị đã được triển khai nghiên cứu, (ii) Bài tổng quan y, sinh, dược học: phục vụ mục tiêu đào tạo liên tục trong lĩnh vực y, sinh, dược học; nhằm hệ thống hóa những kiến thức kinh điển và hiện đại; (iii) Thông tin cập nhật kiến thức mới về y, sinh, dược học trong nước và trên thế giới.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Chính sách truy cập mở\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ áp dụng chính sách truy cập mở đối với các bài báo đã xuất bản đến với độc giả, nhằm mở rộng cơ hội tiếp cận các kết quả nghiên cứu chất lượng cao và tăng cường trao đổi kiến thức. Tạp chí đăng tải trực tuyến (miễn phí) toàn văn các bài báo được công bố trên website của Tạp chí (https:\u002F\u002Ftapchi.ctump.edu.vn).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đạo đức xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ cam kết tuân thủ đạo đức xuất bản phù hợp với các hướng dẫn và tiêu chuẩn của the Committee on Publication Ethics (COPE), tuân thủ các nguyên tắc của COPE’s Core Practices, Best Practices Guidelines for Journal Editors và Guidelines on Good Publication Practices.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Bản thảo bài báo chỉ được chấp nhận khi được tác giả chịu trách nhiệm chính cam kết các nội dung sau: Các nội dung của bản thảo chưa được đăng tải toàn bộ hoặc một phần ở các tạp chí khác; Tất cả các tác giả đều có đóng góp một cách đáng kể vào quá trình nghiên cứu hoặc chuẩn bị bản thảo và cùng chịu trách nhiệm về các nội dung của bản thảo; Tuân thủ các biện pháp đảm bảo đạo đức nghiên cứu (ví dụ thỏa thuận đồng ý tham gia nghiên cứu).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Cam kết bảo mật\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí cam kết thực hiện và tuân thủ các quy định của luật và các văn bản hướng dẫn liên quan đến bảo mật thông tin cá nhân trên không gian mạng. Các thông tin mà người dùng (tác giả, độc giả, biên tập viên, người phản biện) nhập vào các biểu mẫu trên Hệ thống Quản lý xuất bản trực tuyến của tạp chí chỉ được sử dụng vào các mục đích đã được tuyên bố rõ ràng và sẽ không được cung cấp cho bất kỳ bên thứ ba nào khác, hay dùng vào bất kỳ mục đích nào khác.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phí gửi bài\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng bài: 1.000.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng nhanh: 1.500.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với tác giả là cán bộ viên chức thuộc Trường Đại học Y Dược Cần Thơ thì được hỗ trợ 50% lệ phí gửi đăng bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với sinh viên thực hiện đề tài nghiên cứu khoa học cấp trường được hỗ trợ 100% lệ phí đăng bài ( Tác giả gửi đính kèm “ Quyết định về việc giao tổ chức thực hiện đề tài nghiên cứu khoa học cấp Trường của sinh viên”).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Hình thức nộp lệ phí:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Tiền mặt:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Nộp trực tiếp tại Phòng Tài chính - Kế toán, Trường Đại học Y Dược Cần Thơ, số 179 Nguyễn Văn Cừ, P. An Khánh, Q. Ninh Kiều, thành phố Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Chuyển khoản:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tên Tài khoản: Trường ĐHYD Cần Thơ, Số TK: 0111000115668, tại ngân hàng Vietcombank chi nhánh Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Thời gian: Áp dụng từ ngày 01\u002F02\u002F2023.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">* Phí gửi bài không được hoàn trả khi bài viết bị từ chối hoặc tác giả xin rút bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Quy trình phản biện bài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ thực hiện quy trình phản biện kín hai chiều nghiêm ngặt. Danh tính của những người phản biện không được tiết lộ cho các tác giả và ngược lại. Quy trình thẩm định bài báo đăng gồm các bước sau:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tiếp nhận bản thảo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tác giả liên hệ gửi bản thảo đến Tạp chí qua hệ thống trực tuyến tại website: https:\u002F\u002Ftapchi.ctump.edu.vn. Hướng dẫn về cách đăng ký, gửi bài và chuẩn bị bản thảo được cung cấp trên website của Tạp chí.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sàng lọc sơ bộ\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sau khi Tòa soạn nhận được bài báo của tác giả, Ban Thư ký sẽ tiến hành kiểm tra sơ bộ bài báo (các yêu cầu về nội dung và hình thức). Những bài báo không đúng quy cách hoặc có nội dung không phù hợp hoặc vi phạm bản quyền sẽ bị từ chối (Ban Thư ký thông báo phản hồi đến tác giả trong vòng 1 tuần). Những bài báo đủ điều kiện, được Ban Thư ký tòa soạn chuyển đến Ban Biên tập có cùng chuyên môn với nội dung bài báo để đề xuất người phản biện. Thời gian kể từ khi Ban Biên tập nhận bài báo đến khi đề xuất người phản biện bài báo chậm nhất là 5 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Vòng phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký gửi bài và yêu cầu phản biện đến 02 phản biện độc lập.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Các phản biện gởi nhận xét cho Ban Thư ký. Thời gian từ khi gửi bài cho phản biện đến khi nhận ý kiến của phản biện tối đa là 20 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xử ký kết quả phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Nếu ý kiến đồng ý cho đăng và không cần chỉnh sửa, Ban Thư ký tiếp tục đăng bài theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Nếu ý kiến đồng ý đăng và cần chỉnh sửa, Ban Thư ký sẽ thông tin đến tác giả chỉnh sửa theo yêu cầu của người phản biện. Thời gian chỉnh sửa và gửi lại kéo dài không quá 2 tuần, từ khi tác giả bài báo nhận được thông tin (Quá trình này có thể lặp lại tối đa 2 lần\u002F1 bài báo). Khi có sự thống nhất, đồng ý của người phản biện; bài báo được tiếp tục đăng theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Những bài báo có chất lượng không đạt yêu cầu, cả 2 phản biện không đồng ý cho đăng sẽ bị Tòa soạn từ chối đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký tổng hợp các bản thảo đã được tác giả hoàn thiện sau thẩm định trình Ban Biên tập xem xét, Tổng Biên tập phê duyệt, quyết định bài đăng theo các tiêu chí: sự phù hợp nội dung với tôn chỉ và mục đích, thể loại bài viết (ưu tiên các bài có bài có nghiên cứu chuyên sâu, hàm lượng khoa học cao), đóng góp mới bài báo, bài báo được ưu tiên đăng trong số gần nhất của Tạp chí theo thứ tự: tính thời sự, chất lượng bài báo và thời gian gửi bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Ban Biên tập và Ban Thư ký biên tập bản thảo, chế bản, đọc rà soát lỗi. Thời gian hoàn thành từ 10-15 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Ban Thư ký có trách nhiệm thông báo cho tác giả bài báo (bằng e-mail) về tình hình phê duyệt bài báo, thời gian, số kỳ, tập xuất bản bài báo theo qui định.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">4. Danh sách bài báo theo số Tạp chí được in ấn và phát hành trong năm định kỳ được công bố chính thức trên website: https:\u002F\u002Ftapchi.ctump.edu.vn\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>",{"VOID":490},"wcQ1uqwAAAAJ","2023-05-30T08:17:21.868+00:00",[],[494],{"id":495,"createTime":28,"updateTime":28,"relativeEntities":496,"slug":28,"properties":497,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":507,"parentIds":508,"statistic":28},"6413896b-eca9-442b-a73f-182a58a0ce40",[],{"title":498,"address":501,"country":504,"abbreviation":505},{"EN":499,"VI":500},"Can Tho University of Medicine and Pharmacy","Trường Đại học Y Dược Cần Thơ",{"EN":502,"VI":503},"No 179, Nguyen Van Cu street, An Khanh ward, Ninh Kieu district, Can Tho city, Vietnam","Số 179, đường Nguyễn Văn Cừ, phường An Khánh, quận Ninh Kiều, thành phố Cần Thơ, Việt Nam",{"VOID":15},{"VOID":506},"ctump","http:\u002F\u002Fwww.ctump.edu.vn\u002F",[],[],"https:\u002F\u002Ftapchi.ctump.edu.vn\u002Findex.php\u002Fctump",{"impactFactor":32,"impactFactorByYear":512,"i10Index":32,"i10IndexLast5Year":32,"totalPublication":514,"totalPublicationByYear":515,"totalCitation":520,"totalCitationByYear":521,"totalCitationPerPublication":108,"totalCitationPerPublicationByYear":523,"hindexLast5Year":45,"hindex":45},{"2022":513,"2023":111,"2024":106},0.01,1556,{"2020":47,"2021":516,"2022":517,"2023":518,"2024":519,"2025":122},57,306,801,358,161,{"2021":146,"2022":280,"2023":522},99,{"2021":524,"2022":318,"2023":104},0.23,{"impactFactor":28,"impactFactorByYear":28,"i10Index":123,"i10IndexLast5Year":123,"totalPublication":526,"totalPublicationByYear":527,"totalCitation":526,"totalCitationByYear":528,"totalCitationPerPublication":40,"totalCitationPerPublicationByYear":531,"hindexLast5Year":49,"hindex":49},476,{"0":205,"2019":123,"2021":139,"2022":459,"2023":451,"2024":357,"2025":49,"2026":48},{"2021":42,"2022":123,"2023":161,"2024":529,"2025":360,"2026":530},136,83,{"2021":105,"2022":513,"2023":532,"2024":127,"2025":533,"2026":534},0.62,25.43,13.83,{"id":536,"createTime":537,"updateTime":382,"relativeEntities":538,"slug":539,"properties":540,"entityType":25,"verifyStatus":26,"verifyTime":28,"verifyNote":28,"languages":552,"translateLanguages":28,"viewCount":133,"subjectFields":553,"manageAffiliations":554,"indexDatabases":555,"url":556,"thumbnailPath":557,"statistic":558,"gsStatistic":594,"type":55,"analyzePriority":28},"6984a56a-db70-403b-9cc4-4013e1ceaffa","2023-05-09T06:47:40.346+00:00",[],"T%E1%BA%A1p%20ch%C3%AD%20Nghi%C3%AAn%20c%E1%BB%A9u%20n%C6%B0%E1%BB%9Bc%20ngo%C3%A0i",{"country":541,"issn":542,"title":544,"introduce":547,"gsId":550},{"VOID":15},{"VOID":543},"25252445",{"EN":545,"VI":546},"VNU Journal of Foreign Studies","Tạp chí Nghiên cứu nước ngoài",{"EN":548,"VI":549},"{\"ops\":[{\"insert\":\"\\n\\nThe \\n\"},{\"attributes\":{\"italic\":true},\"insert\":\"VNU Journal of Science\"},{\"insert\":\"\\n was established in 1985 for the publication of national and international research papers in all fields of natural sciences and technology, social sciences and humanities. 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In this study we investigated the expressions of ET-1, ET-3, and their receptors, ETA and ETB, in a rat chronic renal transplant rejection model. Renal allografts were performed (F344 → Lewis) with bilateral nephrectomy in recipients. For isograft control, lewis → lewis transplantations were performed. All recipients were sacrificed 140 days after transplantation and the grafts were analyzed histologically. ET-1 and ET-3 protein expression in grafts was measured by immunohistochemistry and ELISA. Semiquantitative RT-PCR methods were used for mRNA levels of ET-1, ET-3, ETA and ETB. No evidence of chronical rejection was manifested in isografts. The allografted rats showed proteinuria and increased serum creatinine levels. Histologically, renal allografts showed atrophy and sclerosis of the glomeruli, cortical scarring and vascular intimal thickening. Immunohistochemically, ET-1 and ET-3 were localized in the convoluted tubules, collecting ducts, endothelium and smooth muscle cells of the large blood vessels. Significantly increased staining for ET-1 and ET-3 were found in allografts compared to isografts. Simultaneously, ELISA for ET-1 and ET-3 showed elevated protein concentrations in allografts compared to isografts. Allografts showed significantly increased ET-1- and ET-3 mRNA compared to isografts. On the other hand, a significant down regulation of the ETA mRNA was noted, and the ETB mRNA remained unchanged. The data from the present study suggest that alteration of ET system may be of importance in the pathogenesis of chronic renal transplant rejection.","PUBLICATION","2025-02-08T07:06:08.480+00:00","Auto Verify","https:\u002F\u002Fonlinelibrary.wiley.com\u002Fresolve\u002Fdoi?DOI=10.1007\u002Fs001470050683",[963,979,994,1007,1020],{"id":964,"sortIndex":32,"researcher":28,"roles":965,"affiliations":967,"properties":976,"displayName":978,"givenName":28,"familyName":28},"8689099e-b8a6-45a8-a856-5a2af66d4795",[966],"AUTHOR",[968],{"id":969,"sortIndex":32,"affiliation":970,"properties":28},"6d76f874-f2b2-486a-bedf-42163fbfd58f",{"id":969,"createTime":28,"updateTime":28,"relativeEntities":971,"slug":28,"properties":972,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":975,"statistic":28},[],{"title":973},{"VI":974},"Department of Pathology,  University of Western Ontario, London Health Sciences Center, London, Ontario, N6A 5C1, Canada, , CA",[],{"title":977},{"VI":978},"Diana Xi Deng",{"id":980,"sortIndex":40,"researcher":28,"roles":981,"affiliations":982,"properties":991,"displayName":993,"givenName":28,"familyName":28},"789f9fa8-47d7-4a73-99db-010a853ac1c2",[966],[983],{"id":984,"sortIndex":32,"affiliation":985,"properties":28},"d641e9be-fbc5-46cc-981f-66ab9c742589",{"id":984,"createTime":28,"updateTime":28,"relativeEntities":986,"slug":28,"properties":987,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":990,"statistic":28},[],{"title":988},{"VI":989},"Department of Surgery,  University of Western Ontario, London Health Sciences Center, London, Ontario, N6A 5C1, Canada, , CA",[],{"title":992},{"VI":993},"Jifu Jiang",{"id":995,"sortIndex":123,"researcher":28,"roles":996,"affiliations":997,"properties":1004,"displayName":1006,"givenName":28,"familyName":28},"98c4b7fe-7a56-4a4f-bfbc-1e82647ee181",[966],[998],{"id":969,"sortIndex":32,"affiliation":999,"properties":28},{"id":969,"createTime":28,"updateTime":28,"relativeEntities":1000,"slug":28,"properties":1001,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1003,"statistic":28},[],{"title":1002},{"VI":974},[],{"title":1005},{"VI":1006},"Bertha Garcia",{"id":1008,"sortIndex":42,"researcher":28,"roles":1009,"affiliations":1010,"properties":1017,"displayName":1019,"givenName":28,"familyName":28},"bdb82e43-976c-453f-a8b7-dc142310b248",[966],[1011],{"id":969,"sortIndex":32,"affiliation":1012,"properties":28},{"id":969,"createTime":28,"updateTime":28,"relativeEntities":1013,"slug":28,"properties":1014,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1016,"statistic":28},[],{"title":1015},{"VI":974},[],{"title":1018},{"VI":1019},"Robert Zhong",{"id":1021,"sortIndex":45,"researcher":28,"roles":1022,"affiliations":1023,"properties":1032,"displayName":1034,"givenName":28,"familyName":28},"e9a05fc5-f8ed-4a5f-b527-48bfbc110e53",[966],[1024],{"id":1025,"sortIndex":32,"affiliation":1026,"properties":28},"d72372aa-89c1-4a54-81ca-d36bbab0e6b6",{"id":1025,"createTime":28,"updateTime":28,"relativeEntities":1027,"slug":28,"properties":1028,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1031,"statistic":28},[],{"title":1029},{"VI":1030},"Department of Pathology, The University of Western Ontario, London Health Sciences Center, London, Ontario, N6A 5C1, Canada e-mail:schakrab@julian.uwo.ca Tel.: + 1-5 19-6 63-33 81 Fax: + 1-5 19-6 61-33 70, , CA",[],{"title":1033},{"VI":1034},"S. 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alterations are a well-known phenomenon that influence the outcome of orthotopic liver transplantation (OLT). Whether or not myocardial dysfunction, which has various causes, contributes to this instability is still debated. Previous transesophageal echocardiography (TEE) studies have presented controversial data, not leading to final clarification. This is mainly because the impact of other contributing factors (inotropic support, alternating preload conditions and temperature) remained unaccounted for. We therefore measured the left ventricular shortening fraction (LVSF), a parameter reflecting myocardial contractility, in 10 consecutive patients undergoing OLT without veno-venous bypass. We measured during preparation (PP), during the anhepatic (AP) phase and the immediate reperfusion phase (RP). During the AP we observed a significant decrease of LVSF which never fell to subnormal levels in the majority of our patients, whereas during the RP, LVSF returned to PP values. These findings support the assumption that myocardial function is influenced by OLT, but that it plays only a minor role in the occurrence of hemodynamic instability, which could mainly be attributed to volume fluctuations.",{"EN":1163},"Assessment of ventricular contractile function during orthotopic liver transplantation",{"EN":1165},"",{"VOID":1167},"De Wolf AM, Begliomini B, Gasoir TA, Kang Y, Pinsky MR. Right ventricular function during orthotopic liver transplantation. Anesth Analg 1993; 76:562.\nDe la Morena G, Acosta F, Villegas M et al. Ventricular function during liver reperfusion in hepatic transplantation. A transesophageal echocardiographic study. Transplantation 1994; 58:306.\nSteininger R, Roth E, Fugger R et al. Transhepatic metabolism of TNF-alpha, IL-6, and endotoxin in the early hepatic reperfusion period after human liver transplantation. Transplantation 1994; 58:179.\nBlanot S, Gillon MC, Ecoffey C. Circulating endotoxins during orthotopic liver transplantation and post-reperfusion syndrome. Lancet 1993; 342:860.\nSteltzer H, Blazek ZG, Gabriel A et al. Two-dimensional transesophageal echocardiography in early diagnosis and treatment of hemodynamic disturbances during liver transplantation. Transpl Proc 1991; 23:1957.\nDe Wolf A. Hemodynamic monitoring during orthotopic liver transplantation. Transpl Proc 1993; 25:1993.\nNasraway SA, Klein RD, Spanier TB, Rohrer RJ, Freeman RB, Rand WM. Hemodynamic correlates of outcome in patients undergoing orthotopic liver transplantation. Evidence for early postoperative myocardial depression. Chest 1995; 107:218.\nEstrin JA, Belani KG, Ascher NL, Lura D, Payne W, Najarian JS. Hemodynamic changes on clamping and unclamping of major vessels during liver transplantation. Transpl Proc 1989; 21:3500.\nDe La Morena G, Acosta F, Villegas M, Olivares A, Contereras RF, Bueno FS, Sanchez-Munoz J et al. Transesophageal echocardiographic evaluation of left ventricular function during orthotopic liver transplantation. Transpl Proc 1993; 25:1832.\nKrenn GC, Plöchl W, Nikolic A, et al. Intrathoracic fluid volumes and pulmonary function during orthotopic liver transplantation. Transplantation 2000; 15:2394.\nGlauser FL. Systemic Hemodynamic and cardiac function changes in patients undergoing orthotopic liver transplantation. 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Factors potentially affecting graft outcome, such as sex, donor and recipient's age, HLA A-B match, cold ischaemia time (CIT), delayed graft function (DGF), serum creatinine at 1 month (Cr1), rejection episodes within 3 months (R3), and type of brain death (BD), were considered. The slopes of the plots by donor age, DGF, HLA A-B match, R3, Cr1 and BD appear to have a significant influence throughout the observation period, with different time-dependent effects on graft survival. Slopes for DGF, Cr1, and age of donor are positive (increased hazard), while slopes for HLA match and BD are negative (decreased hazard). Estimated regression functions for DGF, donor age and Cr1 show a prompt slope (within 3 months); the covariate R3 has a clear influence for about 5 years, and then seems to disappear; and BD appears to have a consistent effect over the entire period. 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All anti-pig immunoglobulins (i.e., IgG, IgA, and IgM) were localized on the cell surface by the use of an indirect immunofluorescence technique. Anti-pig immunoglobulins also competed for the pig cell surface epitopes with Griffonia simplicifolia lectin (GS-I-B4), which is specific for α-galactosyl residues. This study provides further evidence that preformed human antibodies recognizing α-glactosyl-containing epitopes (anti-gal antibodies) could be an important factor in hyperacute rejection of pig organs.",{"EN":1439},"Binding and specificity of major immunoglobulin classes of preformed human anti-pig heart antibodies",{"VOID":1441},"Auchincloss H Jr (1980) Xenogeneic transplantation. Transplantation 46:1–20\nFischel RJ, Bolman RM, Platt JL, Najarian JS, Bach FH, Matas AJ (1990) Removal of IgM anti-endothelial antibodies results in prolonged cardiac xenograft survival. Transplant Proc 22:1077–1078\nGalili U, Rachmilewitz EA, Peleg A, Flechner I (1984) A unique natural human IgG antibody with anti-α-galacosyl specificity. J Exp Med 160:1519–1531\nGalili U, Macher BA, Buehler J, Shohet SB (1985) Human natural anti-α-galactosyl IgG. II. The specific recognition of α(1–3)-linked galactose residues. J Exp Med 162:573–582\nGood AH, Cooper DKC, Malcolm AJ, Ippolito RM, Koren E, Neethling FA, Ye Y, Zuhdi N, Lamontagne LR (1992) Identification of carbohydrate structures which bind human anti-porcine antibodies: implications for discordant xenografting in humans. Transplant Proc 24:559–562\nHammer C (1987) Isohemagglutinins and preformed natural antibodies in xenogeneic organ transplantation. Transplant Proc 19:4443–4447\nKoren E, Neethling FA, Ye Y, Niekrasz M, Baker J, Martin M, Zuhdi N, Cooper DKC (1992) Heterogeneity of preformed human anti-pig xenogeneic antibodies. Transplant Proc 24:598–601\nMurphy LA, Goldstein IJ (1977) Five α-D-galactopyranosyl-binding isolectins from Bandeiraea simplicifolia seeds. J Biol Chem 252:4739–4742\nOtte KE, Andersen N, Jorgensen KA, Kristensen T, Barfort P, Starklint H, Larsen S, Kemp E (1990) Xenoperfusion of pig kidney with human AB or 0 whole blood. Transplant Proc 22:1090\nPaul LC (1991) Mechanism of humoral rejection. In: Cooper DKC, Kemp E, Reemtsma K, White DJG (eds) Xenotransplantation. Springer, Heidelberg, pp 47–67\nPlatt JL, Lindman BJ, Chen H, Spitalnik SL, Bach FH (1990) Endothelial cell antigens recognized by xenoreactive human natural antibodies. Transplantation 50:817–822\nPlatt JL, Vercellotti GM, Dalmasso AP, Matas AJ, Bolman RM, Najarian JS, Bach FH (1990) Transplantation of discordant xenografts: a review of progress. 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There has only been one report on the development of a brain tumor metastasis in liver transplantation. From September 1988 to January 1993, 342 donor hepatectomies with subsequent transplantation were performed at our center. The main donor diagnoses included subarachnoidal bleeding (n=128; 37.4%), isolated head injury (n=114; 33.3%), multiple injuries (n=55; 16.1%), primary cerebral neoplasia (n=13; 3.8%), and other (n=32; 9.4%). Primary cerebral neoplasia included glioblastoma (n=4), meningioma (n=3), astrocytoma (n=2), angioma (n=2), neurocytoma (n=1), and ependymoma (n=1). In the group of donors suffering from primary cerebral neoplasia, procured organs other than the liver included kidneys (n=20), combined kidneys and pancreata (n=1), pancreata (n=2) hearts (n=8), combined hearts and lungs (n=1), and single lungs (n=1). Follow-up of the respective graft recipients ranged from 28 to 68 months (median 43 months). Recurrent malignancy was observed once, in a liver graft recipient. The donor, a 48-year-old female, had undergone surgical resection of an intracerebral multiform glioblastoma and died 4 months later of a relapse in the brain stem. The 28-year-old female recipient had undergone transplantation for an autoimmune-hepatitic cirrhosis. Four months later, histopathological examination of an intraperitoneal and intrahepatic mass revealed a poorly differentiated, small-cell pleomorphic cancer, identified as a glioma metastasis by S100-and glial fibrillary acidic protein immunohistochemical staining. The patient died 6 months post-transplantation. On autopsy, no further neoplastic lesions were detected. Our review adds a second reported case of a liver graft-transmitted brain tumor to the literature and the fourth donor-related malignancy after hepatic transplantation in general.",{"EN":1602},"Liver graft-transmitted glioblastoma multiforme. A case report and experience with 13 multiorgan donors suffering from primary cerebral neoplasia",{"VOID":1604},"Baquero A, Foote J, Kottle S, Raja R, Mendez M, Noumoff J, Cavarocchi N, Cohn JB, Bannett AD (1988) Inadvertent transplantation of choriocarcinoma into four recipients Transplant Proc 20: 98–100\nBaquero A, Penn I, Bannett A, Werner DJ, Kim P (1988) Misdiagnosis of metastatic cerebral choriocarcinoma in female cadaver donors. Transplant Proc 20: 776–777\nHoffmann HJ, Duffner PK (1985) Extraneural metastases of central nervous system tumors. Cancer 56: 1778–1782\nJackson AM, Graham DI (1978) Remote metastases from intracranial tumours. J Clin Pathol 31: 794–802\nKimura T, Budka H, Soler-Federspiel S (1986) An immunohistochemical comparison of the glia-associated proteins, glial fibrillary acidic protein (GFAP), and S-100 protein (S-100P) in human brain tumors. Clin Neuropathol 5: 21–27\nLefrancois N, Touraine JL, Cantarovich D, Cantarovich F, Faure JL, Dubernard JM, Dureau G, Colpart JJ, Bouvier R, Traeger J (1987) Transmission of medulloblastoma from cadaver donor to three organ transplant recipients. Transplant Proc 19: 2242\nLeifer D, Moore T, Ukena T, Wilner D, Thor A, Hedley-White ET (1989) Multifocal glioblastoma with liver metastases in the absence of surgery: case report. J Neurosurg 71: 772–776\nMorse JH, Turcotte JG, Merion R, Campbell DA Jr, Burtch GD, Lucey MR (1990) Development of a malignant tumor in a liver transplant graft procured from a donor with a cerebral neoplasm. Transplantation 50: 875–877\nNeuhaus P, Bechstein WO, Blumhardt G, Wiens M, Lemmens P, Langrehr JM, Lohmann R, Steffen R, Schlag H, Slama KJ, Lobeck H (1993) Comparison of quadruple immunosuppression after liver transplantation with ATG or IL-2 receptor antibody. Transplantation 55: 1320–1327\nNeuhaus P, Blumhardt G, Bechstein WO, Steften R, Platz KP, Keck H (1994) Technique and results of biliary reconstruction using side-to-side choledoch-ocholedochostomy in 300 orthotopic liver transplants. Ann Surg 219: 426–434\nPasquier B, Pasquier D, N'Golet A, Panh MH, Coudere P (1980) Extraneural metastases of astrocytomas and glioblastomas: clinicopathological study of two cases and review of the literature. Cancer 45: 112–125\nPenn I (1991) Donor transmitted disease: cancer. Transplant Proc 23: 2629–2631\nPenn I (1993) Malignancy in transplanted organs. Transpl Int 6: 1–3\nPerentes E, Rubinstein LJ (1987) Recent application of immunoperoxidase histochemistry in human neuro-oncology. Arch Pathol Lab Med 111: 796–812\nSpiro IJ, Yandell DW, Li C, Saini S, Ferry J, Powelson J, Katkov WN, Cosimi AB (1993) Brief report: lymphoma of donor origin occurring in the porta hepatis of a transplanted liver. N Engl J Med 329: 27–29\nZimmermann HM (1957) The natural history of intracranial neoplasms with special reference to the gliomas. Am J Surg 93: 913–924",{"VOID":1606},"10.1007\u002FBF00335707","https:\u002F\u002Fonlinelibrary.wiley.com\u002Fdoi\u002F10.1111\u002Fj.1432-2277.1996.tb00903.x",[1609,1624,1637,1650,1663,1678],{"id":1610,"sortIndex":32,"researcher":28,"roles":1611,"affiliations":1612,"properties":1621,"displayName":1623,"givenName":28,"familyName":28},"51ff505a-63a2-4d6c-aa15-146f37bd2bdb",[966],[1613],{"id":1614,"sortIndex":32,"affiliation":1615,"properties":28},"2c869421-3413-4929-a8f2-599fdfab0faa",{"id":1614,"createTime":28,"updateTime":28,"relativeEntities":1616,"slug":28,"properties":1617,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1620,"statistic":28},[],{"title":1618},{"VI":1619},"Department of Surgery, Virchow Clinic, Humboldt University, Berlin, Germany",[],{"title":1622},{"VI":1623},"Sven Jonas",{"id":1625,"sortIndex":40,"researcher":28,"roles":1626,"affiliations":1627,"properties":1634,"displayName":1636,"givenName":28,"familyName":28},"e1471f22-5690-4c61-b606-3d2f4610225f",[966],[1628],{"id":1614,"sortIndex":32,"affiliation":1629,"properties":28},{"id":1614,"createTime":28,"updateTime":28,"relativeEntities":1630,"slug":28,"properties":1631,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1633,"statistic":28},[],{"title":1632},{"VI":1619},[],{"title":1635},{"VI":1636},"Wolf O. 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In this study, costs of acute and of chronic LTx patients were determined in a retrospective study. Files of 100 consecutive patients who underwent LTx in 1993–1997 were studied. Costs up to 1 year after LTx were Euro 107,675 (chronic liver failure) and Euro 90,792 (acute liver failure). The difference was mainly caused by higher hospitalisation costs and higher personnel costs for chronic liver failure. Medication costs for acute liver failure were higher, due to a high administration rate of expensive anti-HBs immunoglobulin therapy in patients with viral hepatitis B. LTx for chronic liver failure is more costly and seems to be more cost-effective than LTx for acute liver failure, since 1-year survival is higher in patients who underwent transplantation for chronic liver failure.","https:\u002F\u002Fonlinelibrary.wiley.com\u002Fresolve\u002Fdoi?DOI=10.1007\u002Fs001470050852",[1757,1772,1787,1802,1815,1828],{"id":1758,"sortIndex":32,"researcher":28,"roles":1759,"affiliations":1760,"properties":1769,"displayName":1771,"givenName":28,"familyName":28},"e52ed6d0-1330-4de5-a6c3-d429173409e5",[966],[1761],{"id":1762,"sortIndex":32,"affiliation":1763,"properties":28},"10b3a0a4-eab2-4c1c-a550-3e6d5b02111c",{"id":1762,"createTime":28,"updateTime":28,"relativeEntities":1764,"slug":28,"properties":1765,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1768,"statistic":28},[],{"title":1766},{"VI":1767},"Department of Medical Technology Assessment, Erasmus University Medical Centre, P. O. Box 1738,  3000 DR Rotterdam, The Netherlands e-mail: vanagthoven@bmg.eur.nl Tel.: + 31-1 04 08-85 74 Fax: + 31-1 04 08-90 94, , NL",[],{"title":1770},{"VI":1771},"Michel van Agthoven",{"id":1773,"sortIndex":40,"researcher":28,"roles":1774,"affiliations":1775,"properties":1784,"displayName":1786,"givenName":28,"familyName":28},"04467777-2eb9-4ca9-9d13-0898f6bf55f4",[966],[1776],{"id":1777,"sortIndex":32,"affiliation":1778,"properties":28},"dfd1177f-5594-4d4f-8063-678cdf65b5f3",{"id":1777,"createTime":28,"updateTime":28,"relativeEntities":1779,"slug":28,"properties":1780,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1783,"statistic":28},[],{"title":1781},{"VI":1782},"Department of Hepatogastroenterology, Erasmus University Medical Centre,  P. O. Box 1738, 3000 DR Rotterdam, The Netherlands, , NL",[],{"title":1785},{"VI":1786},"Herold J. 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OLTx was performed in 11 pairs of pigs via a venovenous bypass. Six of these grafts were transplanted immediately (group A), while the other five were preserved in University of Wisconsin (UW) solution for 24 h and then transplanted (group B). All grafts were flushed with 300 ml of chilled (4°C) Ringer's lactate solution before reperfusion of the graft, when 20 ml of effluent from the graft was collected and the concentrations of ammonia, lactic acid, GOT, and LDH were measured. Four of the six pigs in group A survived longer than 3 days, while the other two pigs died of causes other than graft dysfunction. All five pigs in group B died either of hemoperitoneum or hemodynamic instability due to liver failure. The histology of postperfusion biopsies in group A showed minimal pathological changes, while the grafts in group B revealed moderate to severe ischemic injuries. Ammonia and lactic acid in the effluent of group B were significantly higher than those of group A (1511±216 vs 417±333 μg\u002Fdl and 114.1±12.2 vs 91.4±12.2 mg\u002Fdl, respectively; P\u003C0.05 in both cases). Before reperfusion, the rate of total adenine nucleotides in all of the substances in the graft, which were measured using high performance liquid chromatography (HPLC), inversely correlated with the ammonia levels in the effluent. We conclude that an analysis of the effluent, (i.e. the levels of ammonia and lactic acid), flushed from a hepatic graft before reperfusion could serve as a predictor of hepatic graft viability.",{"EN":1898},"Prediction of hepatic graft viability before reperfusion: an analysis of effluent from porcine allografts",{"VOID":1900},"Burdelski M, Oellerich M, Raude E, Lamesch P, Ringe B, Raith H, Scheruhn M, Westphal C, Worm M, Bortfeld S, Schulz M, Wittekind C, Hoyer PF, Pichlmayr R (1988) A novel approach to assessment of liver function in donors. Transplant Proc 20: 591–593\nBusuttil RW, Colonna JO II, Hiatt JR, Brems JJ, Khoury GI, Goldstein LI, Quinones-Baldrich WJ, Abdul-Rasool IH, Ramming KP (1987) The first 100 liver transplants at UCLA. Ann Surg 206: 387–402\nDemetris AJ, Jaffe R, Tzakis A, Ramsey G, Todo S, Belle S, Esquivel CO, Shapiro R, Markus B, Mroczek E, Thiel DH van, Sysyn G, Gordon R, Makowka L, Starzl TE (1988) Antibody mediated rejection in human orthotopic liver allografts: a study of liver transplantation across ABO blood group barriers. Am J Pathol 132: 489–509\nFath J, Ascher NL, Konstantinideds FN, Bloomer J, Sharp H, Najarian JS, Cerra FB (1984) Metabolism during hepatic transplantation: indicator of allograft function. Surgery 96: 664–674\nGugenheim J, Samuel D, Reynes M, Bismuth H (1990) Liver transplantation across ABO blood group barriers. Lancet 336: 519–523\nHigashi H, Yanaga K, Shimada M, Makowka L, Thiel DH van, Starzl TE (1990) Lecithin: cholesterol acyltransferase activity in brain-dead donors: a predictor of allograft viability in clinical liver transplantation. Transplant Proc 22: 433–434\nIsai H, Sheil AGR, Bell R, Woodman K, Painteer DM, Earl J, Kirwan PD, Lin WG (1990) A comparison of UW solution with and without hydroxyethyl starch for liver preservation using the isolated porcine liver perfusion model. Transplant Proc 22: 2152–2153\nJenkins R, Bosari S, Khettry U, Clowes GHA Jr, Pearl RH, Trey C (1986) Survival from hepatic transplantation: relationship of protein synthesis to histological abnormality in patient selection and postoperative management. Ann Surg 204: 364–374\nJennings RB, Reimer KA (1981) Lethal myocardial ischemic injury. Am J Pathol 102: 241–255\nKalayoglu M, Sollinger HW, Stratta RJ, D'Alessandro AM, Hoffmann RM, Pirsch JD, Belzer FO (1988) Extended preservation of the liver for clinical transplantation. Lancet I: 617–619\nKamiike W, Burdelski M, Steinhoff G, Ringe B, Lauchart W, Pichlmayr R (1988) Adenine nucleotide metabolism and its relation to organ viability in human liver transplantation. Transplantation 45: 138–143\nLanir A, Jenkins RL, Caldwell L, Lee RGL, Khettry U, Clouse ME (1988) Hepatic transplantation survival: correlation with adenine nucleotide level in donor liver. Hepatology. 8: 471–475\nOellerich M, Burdelski M, Ringe B, Lamesch P, Gubernatis G, Bunzendahl H, Pichlmayr R, Herrmann H (1990) Lignocaine metabolism formation as a measure of pre-transplant liver function. Lancet I: 640–642\nShaw BW, Gordon RD, Iwatsuki S, Starzl TE (1985) Hepatic retransplantation. Transplant Proc 17: 264–271\nShimada M, Yanaga K, Makowka L, Kakizoe S, Thiel DH van, Starzl TE (1989) Significance of lecithin: cholesterol acyltransferase (LCAT) activity as a prognotic indicator of early allograft function in clinical liver transplantation. Transplantation 48: 600–603\nShimada M, Fukuzawa K, Itasaka H, Takenaka K, Sugimachi K (1991) A simple and reliable assessment of liver allograft quality by analysis of effluent flushed from the graft before liver transplantation in dogs. Res Surg 3: 49–52\nStarzl TE, Demetris AJ, Thiel DH van (1989) Liver transplantation (second of two parts). N Engl J Med 321: 1092–1099\nSvensson KL, Persson H, Henriksson BA, Karlberg I, Sonander H, Lundholm K, Stenquist O, Schersten T (1989) Whole body gas exchange: amino acid and lactate clearance as indicators of initial and early allograft viability in clinical liver transplantation. Surgery 105: 472–480\nTaki Y, Gubernatis G, Yamaoka Y, Oellerich M, Yamamoto Y, Ringe B, Okamoto R, Bunzendahl H, Beneking M, Burdelski M, Bornscheuer A, Ozawa K, Pichlmayr R (1990) Significance of arterial ketone body ratio measurement in human liver transplantation. Transplantation 49: 535–539\nTodo S, Nery J, Yanaga K, Podesta L, Gordon RD, Starzl TE (1989) Extended preservation of human liver grafts with U. W. solution. JAMA 261: 711–714\nYanaga K, Makowka L, Lebeau G, Hwang RR, Shimada M, Kakizoe S, Demetris AL, Starzl TE (1990) A new liver perfusion and preservation system for transplantation research in large animals. 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plus CMV immunoglobulin prophylaxis and early therapy with ganciclovir are effective and safe in CMV high-risk renal transplant pediatric recipients",{"VOID":2070},"10.1007\u002Fs001470050444",{"EN":2072}," Cytomegalovirus (CMV) infection is still a major cause of morbidity in high-risk renal transplant recipients. In the present report, we have reviewed our records of renal transplant pediatric recipients (RTPR; mean age 14.1 ± 4.9 years) since 1991, when we started a policy of CMV prophylaxis constituting high-dose oral acyclovir plus CMV hyperimmune immunoglobulins (Hlg) followed by early i. v. ganciclovir therapy in high-risk patients (i. e., CMV donor + \u002F recipient −). Four patients received a kidney from a living relative (LR), 2 patients had one previous transplant, and 1 had a combined liver – kidney transplant. Thirty-three patients who were negative for CMV antibodies (ab) before transplantation received a kidney from CMV ab positive donors. The immunosuppressive regimen included cyclosporine A and steroids, with the addition of azathioprine in the 4 patients who received an LR kidney. Serial assessments for CMV antigenemia (pp 65) were routinely performed for 6 months after transplantation to define CMV infection. Among the 33 CMV seronegative recipients (R −) who received the graft from a CMV seropositive donor (D +), 18 (54.5 %) experienced CMV infection, whereas among the 28 CMV R +, who received a graft from a CMV D +, 11 (39.3 %) experienced CMV infection. With regard to CMV − related symptoms, only 2 patients suffered from a CMV syndrome (fever and leukopenia in 1 patient, fever and arthralgia in the other). In no case did the spectrum of CMV disease occur; only minor symptoms were present in 7 of the remaining CMV-infected patients (fever in 6 and leukopenia in 1). Rejection episodes and renal function did not differ between CMV-infected and non-CMV-infected patients. Our experiences support the use of prophylactic acyclovir plus CMV HIg followed by early therapy with i. v. ganciclovir to combat the risk of increased morbidity in high risk RTPR.","https:\u002F\u002Fonlinelibrary.wiley.com\u002Fresolve\u002Fdoi?DOI=10.1007\u002Fs001470050444",[2075,2090,2105,2120,2135,2148,2161,2174,2189,2202,2215],{"id":2076,"sortIndex":32,"researcher":28,"roles":2077,"affiliations":2078,"properties":2087,"displayName":2089,"givenName":28,"familyName":28},"96b1b525-7e0e-4c93-a8f3-fd03965cfef5",[966],[2079],{"id":2080,"sortIndex":32,"affiliation":2081,"properties":28},"98382f8b-19fc-4e60-ae01-27cf040ea750",{"id":2080,"createTime":28,"updateTime":28,"relativeEntities":2082,"slug":28,"properties":2083,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2086,"statistic":28},[],{"title":2084},{"VI":2085},"Department of Nephrology,  G. Gaslini Institute, Largo G. 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Some studies suggest that livers from fasted animals (liver glycogen-depleted) are more readily injured than livers from fed animals. Our previous study suggested the opposite, i.e., livers from donors fasted for 4 days were significantly more viable on orthotopic liver transplantation. Fasting may decrease the sensitivity of the liver to an inflammatory response or block Kupffer cell activation following transplantation. Thus, long-term fasting may be beneficial for reasons unrelated to liver glycogen content. In this study we attempted to separate out the roles of fasting and liver glycogen in liver transplant outcome by fasting donors for 2 days and then feeding them only glucose to elevate liver glycogen. Rats (Brown Norway) were fed (standard diet), fasted (4 days), or fasted 2 days and then fed glucose (in water) for 2 days. Livers were preserved for either 30 or 44 h in UW solution and transplanted. Four-day fasting of the donor improved the survival rate in liver transplantation (50%–100% in 30-h cold storage, 29%–83% in 44-h cold storage). However, feeding glucose for 2 days to fasted animals caused a decrease in survival in this series of transplants (40% in 30-h cold storage, 0% in 44-h cold storage). In the glucose-fed group, liver glycogen was 240% of that in the control group. This suggests that the presence of a high concentration of liver glycogen is not beneficial to the preserved and transplanted rat liver.",{"EN":2289},"Liver glycogen in fasted rat livers does not improve outcome of liver transplantation",{"VOID":2291},"Boudjema K, Lindell SL, Southard JH, Belzer FO (1990) The effect of fasting on the quality of liver preservation by simple cold storage. Transplantation 50:943–948\nCaldwell-Kenkel JC, Currin RT, Tanaka Y, Thurman RG, Lemasters JJ (1989) Reperfusion injury to endothelial cells following cold ischemic storage of rat livers. Hepatology 10:292–299\nCreig PD, Foster J, Superina RA, Strasberg SM, Mohamed M, Blendis LM, Taylor BR, Levy GA, Langer B (1990) Donor-specific factors predict graft function following liver transplantation. Transplant Proc 22:2072–2073\nCywes R, Creig PD, Sanabria JR, Clavien PA, Levy GA, Harvey PRC, Strasberg SM (1992) Effect of intraportal glucose infusion on hepatic glycogen content and degradation and outcome of liver transplantation. Ann Surg 216:235–246\nKamada N, Calne RY (1979) Orthotopic liver transplantation in the rat. Technique using cuff for portal vein anastomosis and biliary drainage. Transplantation 28:47–50\nKrahenbuhl S, Weber FL, Brass EP (1991) Decreased hepatic glycogen content and accelarated response to starvation in rats with carbon tetrachloride-induced cirrhosis. Hepatology 14:1189–1193\nLemasters JL, Caldwell-Kenkel JC, Currin RT, Tanaka Y, Marzi I, Thurman RG (1989) Endothelial cell killing and activation of Kupffer cells following reperfusion of rat liver stored in Eurocollins solution. In: Wisse E, Knook DL, Decker K (eds) Kupffer Cell Foundation, Rijswijk, pp 277–282\nLindell SL, Southard JH, Hansen T, Rankins M, Danielewitz R, Belzer FO (1996) Donor nutritional status: a determinant of liver preservation injury. Transplantation (in press)\nMor E, Klintmalm GB, Gonwa TA (1992) The use of marginal donors for liver transplantation: retrospective study of 365 liver donors. Transplantation 53:383–386\nMorgan GR, Sanabria JR, Clavien PA (1991) Correlation of donor nutritional status with sinusoidal lining cell viability and liver function in the rat. Transplantation 51:1176–1183\nNishihara M, Sumimoto R, Fukuda Y, Southard JH, Dohi K (in press) Inhibition of warm ischemic injury to rat liver, pancreas and heart grafts by controlling the nutritional status of both donor and recipient. Surg Today\nPurim J, Woerden WF van, Knol E (1989) Donor data in liver grafts with primary non-function — a preliminary analysis by the European liver registry. Transplant Proc 21:2383–2384\nSouthard JH, Lutz MF, Ametani MS, Belzer FO (1984) Stimulation of ATP synthesis in hypothermically perfused dog kidneys by adenosine and phosphate. Cryobiology 21:13–18\nSumimoto R, Southard JH, Belzer FO (1993) Livers from fasted rats acquire resistance to warm and cold ischemic injury. 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