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dispersed human T and TC mast cells, J Immunol, 138, 2611, 10.4049\u002Fjimmunol.138.8.2611\nRicciotti, 2011, Prostaglandins and inflammation, Arterioscler Thromb Vasc Biol, 31, 986, 10.1161\u002FATVBAHA.110.207449\nYamaga, 2020, Decreased intracellular histamine concentration and basophil activation in anaphylaxis, Allergol Int, 69, 78, 10.1016\u002Fj.alit.2019.05.009\nGiavina-Bianchi, 2017, Basophil activation test is a relevant biomarker of the outcome of rapid desensitization in platinum compounds-allergy, J Allergy Clin Immunol Pract, 5, 728, 10.1016\u002Fj.jaip.2016.11.006\nGotlib, 2013, International Working Group-Myeloproliferative Neoplasms Research and Treatment (IWG-MRT) & European Competence Network on Mastocytosis (ECNM) consensus response criteria in advanced systemic mastocytosis, Blood, 121, 2393, 10.1182\u002Fblood-2012-09-458521\nAkin, 2010, Mast cell activation syndrome: proposed diagnostic criteria, J Allergy Clin Immunol, 126, 1099, 10.1016\u002Fj.jaci.2010.08.035\nValent, 2012, Definitions, criteria and global classification of mast cell disorders with special reference to mast cell activation syndromes: a consensus proposal, Int Arch Allergy Immunol, 157, 215, 10.1159\u002F000328760\nValent, 2019, Proposed diagnostic algorithm for patients with suspected mast cell activation syndrome, J Allergy Clin Immunol Pract, 7, 1125, 10.1016\u002Fj.jaip.2019.01.006\nHamilton, 2018, Nonclonal mast cell activation syndrome: a growing body of evidence, Immunol Allergy Clin North Am, 38, 469, 10.1016\u002Fj.iac.2018.04.002\nHartmann, 2016, Cutaneous manifestations in patients with mastocytosis: consensus report of the European Competence Network on Mastocytosis; the American Academy of Allergy, Asthma & Immunology; and the European Academy of Allergology and Clinical Immunology, J Allergy Clin Immunol, 137, 35, 10.1016\u002Fj.jaci.2015.08.034\nHamilton, 2011, Mast cell activation syndrome: a newly recognized disorder with systemic clinical manifestations, J Allergy Clin Immunol, 128, 147, 10.1016\u002Fj.jaci.2011.04.037\nPatterson, 1995, Idiopathic anaphylaxis: an attempt to estimate the incidence in the United States, Arch Intern Med, 155, 869, 10.1001\u002Farchinte.1995.00430080119014\nKlein, 1995, Underreporting of anaphylaxis in a community emergency room, J Allergy Clin Immunol, 95, 637, 10.1016\u002FS0091-6749(95)70329-2\nTejedor Alonso, 2015, Epidemiology of anaphylaxis, Clin Exp Allergy, 45, 1027, 10.1111\u002Fcea.12418\nYocum, 1999, Epidemiology of anaphylaxis in Olmsted county: a population-based study, J Allergy Clin Immunol, 104, 452, 10.1016\u002FS0091-6749(99)70392-1\nWood, 2014, Anaphylaxis in America: the prevalence and characteristics of anaphylaxis in the United States, J Allergy Clin Immunol, 133, 461, 10.1016\u002Fj.jaci.2013.08.016\nLieberman, 2015, Anaphylaxis—a practice parameter update 2015, Ann Allergy Asthma Immunol, 115, 341, 10.1016\u002Fj.anai.2015.07.019\nPattanaik, 2018, The changing face of anaphylaxis in adults and adolescents, Ann Allergy Asthma Immunol, 121, 594, 10.1016\u002Fj.anai.2018.07.017\nWebb, 2006, Anaphylaxis: a review of 601 cases, Ann Allergy Asthma Immunol, 97, 39, 10.1016\u002FS1081-1206(10)61367-1\nDitto, 1996, Idiopathic anaphylaxis: a series of 335 cases, Ann Allergy Asthma Immunol, 77, 285, 10.1016\u002FS1081-1206(10)63322-4\nAlonso, 2002, Idiopathic anaphylaxis: a descriptive study of 81 patients in Spain, Ann Allergy Asthma Immunol, 88, 313, 10.1016\u002FS1081-1206(10)62014-5\nKaliner, 1981, Effects of infused histamine: analysis of the effects of H-1 and H-2 histamine receptor antagonists on cardiovascular and pulmonary responses, J Allergy Clin Immunol, 68, 365, 10.1016\u002F0091-6749(81)90134-2\nIff, 1966, Mechanisms of anaphylaxis in the mouse: similarity of shock induced by anaphylaxis and by mixtures of histamine and serotonin, Int Arch Allergy Appl Immunol, 40, 313, 10.1159\u002F000229815\nSimons, 2010, Anaphylaxis, 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Food Allergy Biomarkers",{"VOID":1210},"[\"13709343017518301559\"]",{"VOID":1212},"Sampson, 2012, Standardizing double-blind, placebo-controlled oral food challenges: American Academy of Allergy, Asthma & Immunology-European Academy of Allergy and Clinical Immunology PRACTALL consensus report, J Allergy Clin Immunol, 130, 1260, 10.1016\u002Fj.jaci.2012.10.017\nGrabenhenrich, 2017, A new framework for the documentation and interpretation of oral food challenges in population-based and clinical research, Allergy, 72, 453, 10.1111\u002Fall.13049\nSampson, 1997, Relationship between food-specific IgE concentrations and the risk of positive food challenges in children and adolescents, J Allergy Clin Immunol, 100, 444, 10.1016\u002FS0091-6749(97)70133-7\nSampson, 2001, Utility of food-specific IgE concentrations in predicting symptomatic food allergy, J Allergy Clin Immunol, 107, 891, 10.1067\u002Fmai.2001.114708\nPerry, 2004, The relationship of allergen-specific IgE levels and oral food challenge outcome, J Allergy Clin Immunol, 114, 144, 10.1016\u002Fj.jaci.2004.04.009\nCelik-Bilgili, 2005, The predictive value of specific immunoglobulin E levels in serum for the outcome of oral food challenges, Clin Exp Allergy, 35, 268, 10.1111\u002Fj.1365-2222.2005.02150.x\nKomata, 2007, The predictive relationship of food-specific serum IgE concentrations to challenge outcomes for egg and milk varies by patient age, J Allergy Clin Immunol, 119, 1272, 10.1016\u002Fj.jaci.2007.01.038\nNicolaou, 2010, Allergy or tolerance in children sensitized to peanut: prevalence and differentiation using component-resolved diagnostics, J Allergy Clin Immunol, 125, 191, 10.1016\u002Fj.jaci.2009.10.008\nDang, 2012, Increasing the accuracy of peanut allergy diagnosis by using Ara h 2, J Allergy Clin Immunol, 129, 1056, 10.1016\u002Fj.jaci.2012.01.056\nKlemans, 2013, Ara h 2 is the best predictor for peanut allergy in adults, J Allergy Clin Immunol Pract, 1, 632, 10.1016\u002Fj.jaip.2013.07.014\nMasthoff, 2013, Sensitization to Cor a 9 and Cor a 14 is highly specific for a hazelnut allergy with objective symptoms in Dutch children and adults, J Allergy Clin Immunol, 132, 393, 10.1016\u002Fj.jaci.2013.02.024\nShreffler, 2004, Microarray immunoassay: association of clinical history, in vitro IgE function, and heterogeneity of allergenic peanut epitopes, J Allergy Clin Immunol, 113, 776, 10.1016\u002Fj.jaci.2003.12.588\nLin, 2012, A bioinformatics approach to identify patients with symptomatic peanut allergy using peptide microarray immunoassay, J Allergy Clin Immunol, 129, 1321, 10.1016\u002Fj.jaci.2012.02.012\nSackesen, 2019, A new Luminex-based peptide assay to identify reactivity to baked, fermented and whole milk, Allergy, 74, 327, 10.1111\u002Fall.13581\nSuprun, 2018, Peanut epitope-specific IgE binding can predict clinical peanut allergy, Allergy, 73, 116\nNugraha, 2019, Conservation analysis of B-cell allergen epitopes to predict clinical cross-reactivity between shellfish and inhalant invertebrate allergens, Front Immunol, 10, 2676, 10.3389\u002Ffimmu.2019.02676\nBoyce, 2010, Guidelines for the diagnosis and management of food allergy in the United States: report of the NIAID-sponsored expert panel, J Allergy Clin Immunol, 126, S1, 10.1016\u002Fj.jaci.2010.10.008\nStapel, 2008, Testing for IgG4 against foods is not recommended as a diagnostic tool: EAACI Task Force Report, Allergy, 63, 793, 10.1111\u002Fj.1398-9995.2008.01705.x\nHusby, 1985, Humoral immunity to dietary antigens in healthy adults. 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133, 492, 10.1016\u002Fj.jaci.2013.12.1041\nPeters, 2015, Natural history of peanut allergy and predictors of resolution in the first 4 years of life: a population-based assessment, J Allergy Clin Immunol, 135, 1257, 10.1016\u002Fj.jaci.2015.01.002\nVickery, 2014, Sustained unresponsiveness to peanut in subjects who have completed peanut oral immunotherapy, J Allergy Clin Immunol, 133, 468, 10.1016\u002Fj.jaci.2013.11.007\nChinthrajah, 2019, Sustained outcomes in oral immunotherapy for peanut allergy (POISED study): a large, randomised, double-blind, placebo-controlled, phase 2 study, Lancet, 394, 1437, 10.1016\u002FS0140-6736(19)31793-3\nDang, 2019, Egg allergen specific IgE diversity predicts resolution of egg allergy in the population cohort HealthNuts, Allergy, 74, 318, 10.1111\u002Fall.13572\nCaubet, 2017, Natural tolerance development in cow's milk allergic children: IgE and IgG4 epitope binding, Allergy, 72, 1677, 10.1111\u002Fall.13167\nWood, 2016, A randomized, double-blind, placebo-controlled study of omalizumab combined with oral immunotherapy for the treatment of cow's milk allergy, J Allergy Clin Immunol, 137, 1103, 10.1016\u002Fj.jaci.2015.10.005\nKulis, 2012, Increased peanut-specific IgA levels in saliva correlate with food challenge outcomes after peanut sublingual immunotherapy, J Allergy Clin Immunol, 129, 1159, 10.1016\u002Fj.jaci.2011.11.045\nWright, 2016, Component-resolved analysis of IgA, IgE, and IgG4 during egg OIT identifies markers associated with sustained unresponsiveness, Allergy, 71, 1552, 10.1111\u002Fall.12895\nBurton, 2018, Allergen-specific IgG antibody signaling through FcgammaRIIb promotes food tolerance, J Allergy Clin Immunol, 141, 189, 10.1016\u002Fj.jaci.2017.03.045\nBurks, 2012, Oral immunotherapy for treatment of egg allergy in children, N Engl J Med, 367, 233, 10.1056\u002FNEJMoa1200435\nHoffmann, 2015, The clinical utility of basophil activation testing in diagnosis and monitoring of allergic disease, Allergy, 70, 1393, 10.1111\u002Fall.12698\nKnol, 1991, Monitoring human basophil activation via CD63 monoclonal antibody 435, J Allergy Clin Immunol, 88, 328, 10.1016\u002F0091-6749(91)90094-5\nBuhring, 1999, The monoclonal antibody 97A6 defines a novel surface antigen expressed on human basophils and their multipotent and unipotent progenitors, Blood, 94, 2343\nPatil, 2012, Immunology in the Clinic Review Series; focus on allergies: basophils as biomarkers for assessing immune modulation, Clin Exp Immunol, 167, 59, 10.1111\u002Fj.1365-2249.2011.04503.x\nSantos, 2014, Basophil activation test discriminates between allergy and tolerance in peanut-sensitized children, J Allergy Clin Immunol, 134, 645, 10.1016\u002Fj.jaci.2014.04.039\nSantos, 2017, Road map for the clinical application of the basophil activation test in food allergy, Clin Exp Allergy, 47, 1115, 10.1111\u002Fcea.12964\nSantos, 2015, Distinct parameters of the basophil activation test reflect the severity and threshold of allergic reactions to peanut, J Allergy Clin Immunol, 135, 179, 10.1016\u002Fj.jaci.2014.09.001\nvan Erp, 2017, The IgE and basophil responses to Ara h 2 and Ara h 6 are good predictors of peanut allergy, J Allergy Clin Immunol, 139, 358, 10.1016\u002Fj.jaci.2016.06.041\nRuinemans-Koerts, 2019, The basophil activation test reduces the need for a food challenge test in children suspected of IgE-mediated cow's milk allergy, Clin Exp Allergy, 49, 350, 10.1111\u002Fcea.13307\nFord, 2013, Basophil reactivity, wheal size, and immunoglobulin levels distinguish degrees of cow's milk tolerance, J Allergy Clin Immunol, 131, 180, 10.1016\u002Fj.jaci.2012.06.003\nRubio, 2011, Benefit of the basophil activation test in deciding when to reintroduce cow's milk in allergic children, Allergy, 66, 92, 10.1111\u002Fj.1398-9995.2010.02432.x\nVila, 2013, Decrease in antigen-specific CD63 basophil expression is associated with the development of tolerance to egg by SOTI in children, Pediatr Allergy Immunol, 24, 463, 10.1111\u002Fpai.12070\nJones, 2009, Clinical efficacy and immune regulation with peanut oral immunotherapy, J Allergy Clin Immunol, 124, 292, 10.1016\u002Fj.jaci.2009.05.022\nPatil, 2019, Early decrease in basophil sensitivity to Ara h 2 precedes sustained unresponsiveness after peanut oral immunotherapy, J Allergy Clin Immunol, 144, 1310, 10.1016\u002Fj.jaci.2019.07.028\nThyagarajan, 2012, Evidence of pathway-specific basophil anergy induced by peanut oral immunotherapy in peanut-allergic children, Clin Exp Allergy, 42, 1197, 10.1111\u002Fj.1365-2222.2012.04028.x\nVarshney, 2011, A randomized controlled study of peanut oral immunotherapy: clinical desensitization and modulation of the allergic response, J Allergy Clin Immunol, 127, 654, 10.1016\u002Fj.jaci.2010.12.1111\nTsai, 2020, Sustained successful peanut oral immunotherapy associated with low basophil activation and peanut-specific IgE, J Allergy Clin Immunol, 145, 885, 10.1016\u002Fj.jaci.2019.10.038\nKim, 2011, Sublingual immunotherapy for peanut allergy: clinical and immunologic evidence of desensitization, J Allergy Clin Immunol, 127, 640, 10.1016\u002Fj.jaci.2010.12.1083\nKim, 2019, Long-term sublingual immunotherapy for peanut allergy in children: clinical and immunologic evidence of desensitization, J Allergy Clin Immunol, 144, 1320, 10.1016\u002Fj.jaci.2019.07.030\nJones, 2017, Epicutaneous immunotherapy for the treatment of peanut allergy in children and young adults, J Allergy Clin Immunol, 139, 1242, 10.1016\u002Fj.jaci.2016.08.017\nKulis, 2019, High- and low-dose oral immunotherapy similarly suppress pro-allergic cytokines and basophil activation in young children, Clin Exp Allergy, 49, 180, 10.1111\u002Fcea.13256\nGorelik, 2015, Suppression of the immunologic response to peanut during immunotherapy is often transient, J Allergy Clin Immunol, 135, 1283, 10.1016\u002Fj.jaci.2014.11.010\nTurcanu, 2003, Characterization of lymphocyte responses to peanuts in normal children, peanut-allergic children, and allergic children who acquired tolerance to peanuts, J Clin Invest, 111, 1065, 10.1172\u002FJCI200316142\nDeLong, 2011, Ara h 1-reactive T cells in individuals with peanut allergy, J Allergy Clin Immunol, 127, 1211, 10.1016\u002Fj.jaci.2011.02.028\nWisniewski, 2015, Analysis of cytokine production by peanut-reactive T cells identifies residual Th2 effectors in highly allergic children who received peanut oral immunotherapy, Clin Exp Allergy, 45, 1201, 10.1111\u002Fcea.12537\nChiang, 2018, Single-cell profiling of peanut-responsive T cells in patients with peanut allergy reveals heterogeneous effector TH2 subsets, J Allergy Clin Immunol, 141, 2107, 10.1016\u002Fj.jaci.2017.11.060\nWeissler, 2018, Identification and analysis of peanut-specific effector T and regulatory T cells in children allergic and tolerant to peanut, J Allergy Clin Immunol, 141, 1699, 10.1016\u002Fj.jaci.2018.01.035\nBrough, 2014, IL-9 is a key component of memory TH cell peanut-specific responses from children with peanut allergy, J Allergy Clin Immunol, 134, 1329, 10.1016\u002Fj.jaci.2014.06.032\nWambre, 2017, A phenotypically and functionally distinct human TH2 cell subpopulation is associated with allergic disorders, Sci Transl Med, 9, eaam9171, 10.1126\u002Fscitranslmed.aam9171\nRuiter, 2020, Expansion of the CD4(+) effector T-cell repertoire characterizes peanut-allergic patients with heightened clinical sensitivity, J Allergy Clin Immunol, 145, 270, 10.1016\u002Fj.jaci.2019.09.033\nBerin, 2018, Egg-specific IgE and basophil activation but not egg-specific T-cell counts correlate with phenotypes of clinical egg allergy, J Allergy Clin Immunol, 142, 149, 10.1016\u002Fj.jaci.2018.01.044\nKosoy, 2016, Transcriptional profiling of egg allergy and relationship to disease phenotype, PLoS One, 11, e0163831, 10.1371\u002Fjournal.pone.0163831\nQamar, 2015, Naturally occurring tolerance acquisition to foods in previously allergic children is characterized by antigen specificity and associated with increased subsets of regulatory T cells, Clin Exp Allergy, 45, 1663, 10.1111\u002Fcea.12570\nRyan, 2016, Successful immunotherapy induces previously unidentified allergen-specific CD4+ T-cell subsets, Proc Natl Acad Sci U S A, 113, E1286, 10.1073\u002Fpnas.1520180113\nWatson, 2017, Integrative transcriptomic analysis reveals key drivers of acute peanut allergic reactions, Nat Commun, 8, 1943, 10.1038\u002Fs41467-017-02188-7\nDo, 2020, Dual transcriptomic and epigenomic study of reaction severity in peanut allergic children, J Allergy Clin Immunol, 145, 1219, 10.1016\u002Fj.jaci.2019.10.040\nMartino, 2012, T-cell activation genes differentially expressed at birth in CD4+ T-cells from children who develop IgE food allergy, Allergy, 67, 191, 10.1111\u002Fj.1398-9995.2011.02737.x\nMartino, 2015, Blood DNA methylation biomarkers predict clinical reactivity in food-sensitized infants, J Allergy Clin Immunol, 135, 1319, 10.1016\u002Fj.jaci.2014.12.1933\nMartino, 2018, Epigenetic dysregulation of naive CD4+ T-cell activation genes in childhood food allergy, Nat Commun, 9, 3308, 10.1038\u002Fs41467-018-05608-4\nBunyavanich, 2019, Food allergy: could the gut microbiota hold the key?, Nat Rev Gastroenterol Hepatol, 16, 201, 10.1038\u002Fs41575-019-0123-0\nZhao, 2019, The gut microbiome in food allergy, Ann Allergy Asthma Immunol, 122, 276, 10.1016\u002Fj.anai.2018.12.012\nHo, 2018, Role of the microbiome in food allergy, Curr Allergy Asthma Rep, 18, 27, 10.1007\u002Fs11882-018-0780-z\nBunyavanich, 2019, Food allergy and the microbiome: current understandings and future directions, J Allergy Clin Immunol, 144, 1468, 10.1016\u002Fj.jaci.2019.10.019\nFazlollahi, 2018, Early-life gut microbiome and egg allergy, Allergy, 73, 1515, 10.1111\u002Fall.13389\nBerni Canani, 2016, Lactobacillus rhamnosus GG-supplemented formula expands butyrate-producing bacterial strains in food allergic infants, ISME J, 10, 742, 10.1038\u002Fismej.2015.151\nBunyavanich, 2016, 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inhibitor deficiency: patient registry and approach to the prevalence in Spain, Ann Allergy Asthma Immunol, 94, 498, 10.1016\u002FS1081-1206(10)61121-0\nFarkas, 2010, Pediatric hereditary angioedema due to C1-inhibitor deficiency, Allergy Asthma Clin Immunol, 6, 18, 10.1186\u002F1710-1492-6-18\nPappalardo, 2000, Frequent de novo mutations and exon deletions in the C1 inhibitor gene of patients with angioedema, J Allergy Clin Immunol, 106, 1147, 10.1067\u002Fmai.2000.110471\nBoyle, 2005, Hereditary angio-oedema in children: a management guideline, Pediatr Allergy Immunol, 16, 288, 10.1111\u002Fj.1399-3038.2005.00275.x\nSchneider, 2013, C1-INH concentrate for treatment of acute hereditary angioedema: a pediatric cohort from the I.M.P.A.C.T. studies, Pediatr Allergy Immunol, 24, 54, 10.1111\u002Fpai.12024\nAbinun, 1999, Hereditary angio-oedema in children, Lancet, 353, 2242, 10.1016\u002FS0140-6736(05)76288-7\nAgostoni, 1992, Hereditary and acquired C1-inhibitor deficiency: biological and 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