[{"data":1,"prerenderedAt":-1},["ShallowReactive",2],{"_public_publisher_byId_8162add8-64dc-443b-84ef-acb717121ca9":3,"_public_publication_all{\"sortAscending\":false,\"sortField\":\"updateTime\",\"page\":0,\"size\":10,\"facet\":true,\"searchKey\":\"publisherId:8162add8-64dc-443b-84ef-acb717121ca9,\"}":36},{"code":4,"data":5,"meta":18},"SUCCESS",{"id":6,"createTime":7,"updateTime":8,"relativeEntities":9,"slug":10,"properties":11,"entityType":16,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":19,"subjectFields":20,"manageAffiliations":21,"indexDatabases":22,"url":23,"thumbnailPath":18,"statistic":24,"gsStatistic":18,"type":35,"analyzePriority":18},"8162add8-64dc-443b-84ef-acb717121ca9","2024-04-20T10:22:10.695+00:00","2026-07-21T23:12:32.041+00:00",[],"T%E1%BA%A1p-ch%C3%AD-Tim-m%E1%BA%A1ch-h%E1%BB%8Dc-Vi%E1%BB%87t-Nam",{"issn":12,"title":14},{"VOID":13},"18592848",{"VOID":15},"Tạp chí Tim mạch học Việt Nam","PUBLISHER","PENDING",null,0,[],[],[],"https:\u002F\u002Fjvc.vnha.org.vn\u002Ftmh",{"impactFactor":19,"impactFactorByYear":25,"i10Index":19,"i10IndexLast5Year":19,"totalPublication":26,"totalPublicationByYear":27,"totalCitation":30,"totalCitationByYear":31,"totalCitationPerPublication":32,"totalCitationPerPublicationByYear":33,"hindexLast5Year":28,"hindex":28},{},12,{"2015":28,"2016":28,"2018":29,"2019":28,"2021":29,"2022":28,"2023":30},1,3,2,{"2015":28,"2018":28},0.17,{"2015":28,"2018":34},0.33,"JOURNAL",{"meta":37,"data":39},{"total":38},"27",[40,214,381,564,739,826,917,1082,1179,1285],{"id":41,"createTime":42,"updateTime":43,"relativeEntities":44,"slug":45,"properties":46,"entityType":59,"verifyStatus":60,"verifyTime":42,"verifyNote":61,"languages":62,"translateLanguages":18,"viewCount":19,"primaryUrl":64,"fullTextUrl":65,"authors":66,"publicationType":151,"publisherRelationship":152,"citationCount":19,"citationInfo":171,"publishDate":174,"publishYear":172,"citationAnalyzeStatus":175,"lastCitationAnalyze":176,"indexDatabases":177,"openAccess":18,"references":178,"isForceReanalyzing":213},"31ccfd87-4be1-4316-97a6-18d9903f5ddb","2024-06-06T01:17:05.189+00:00","2026-07-29T19:55:09.147+00:00",[],"%C4%90%E1%BA%B7c-%C4%91i%E1%BB%83m-r%E1%BB%91i-lo%E1%BA%A1n-lipid-m%C3%A1u-%E1%BB%9F-ng%C6%B0%E1%BB%9Di-m%E1%BA%AFc-b%E1%BB%87nh-%C4%91%E1%BB%99ng-m%E1%BA%A1ch-chi-d%C6%B0%E1%BB%9Bi",{"abstract":47,"title":50,"gsPaper":53,"keywords":55,"doi":57},{"VI":48,"EN":49},"Mục tiêu nghiên cứu là mô tả đặc điểm lipid máu ở người mắc bệnh động mạch chi dưới (BĐMCD). Nghiên cứu mô tả cắt ngang bao gồm những người mắc BĐMCD điều trị tại Viện Tim mạch, Bệnh viện Bạch Mai. Kết quả nghiên cứu ghi nhận trong 229 người mắc BĐMCD, có 78,6% là nam giới, 63,3% có tổn thương Rutherford mức độ IV-VI. Tỉ lệ bệnh nhân có mức cholesterol toàn phần >5,2mmol\u002FL, LDL-C >3,6 mmol\u002FL, HDL-C <1,0 mmol\u002FL và triglycerid >1,7 mmol\u002FL lương ứng là  25,8%, 18,8%, 50,2% và 42,8%. Tuy nhiên, chỉ 5,2% bệnh nhân sử dụng statin cường độ mạnh là rosuvastatin 20 mg. Tóm lại, rối loạn lipid máu là phổ biến ở người mắc BĐMCD.","The objective of this study was to characterize blood lipids in individuals with lower extremity arterial disease (LEAD). The cross-sectional descriptive study included individuals with LEAD who were treated at the Vietnam National Heart Institute, Bach Mai Hospital. The results of the study showed that among 229 individuals with LEAD, 78.6% were men, and 63.3% had a Rutherford classification of grade IV-VI. The rate of patients with plasma levels of total cholesterol >5.2mmol\u002FL, LDL-C >3.6 mmol\u002FL, HDL-C <1.0 mmol\u002FL, and triglyceride >1.7 mmol\u002FL was 25.8%, 18.8%, 50.2%, and 42.8%, respectively. However, only 5.2% of patients were used high-intensity statins such as rosuvastatin 20 mg. In summary, the study found that dyslipidemia was common in patients with LEAD.",{"EN":51,"VI":52},"Dyslipidemia patterns in patients with peripheral artery disease","Đặc điểm rối loạn lipid máu ở người mắc bệnh động mạch chi dưới",{"VOID":54},"4857528313911206520",{"VI":56},"Bệnh động mạch chi dưới,Rối loạn lipid máu,Cholesterol,Statin",{"VOID":58},"10.58354\u002Fjvc.106.2023.479","PUBLICATION","VERIFIED","Auto Verify",[63],"VI","https:\u002F\u002Fjvc.vnha.org.vn\u002Ftmh\u002Farticle\u002Fview\u002F479","https:\u002F\u002Fjvc.vnha.org.vn\u002Ftmh\u002Farticle\u002Fdownload\u002F479\u002F650",[67,95,113,131],{"id":68,"sortIndex":19,"researcher":18,"roles":69,"affiliations":70,"properties":90},"c164c141-d56f-4508-879d-99990f3c40f5",[],[71],{"id":72,"sortIndex":19,"affiliation":73,"properties":88},"fbda2311-d0c4-4fac-aba0-6b61a38a8301",{"id":72,"createTime":18,"updateTime":18,"relativeEntities":74,"slug":18,"properties":75,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":86,"parentIds":87,"statistic":18},[],{"title":76,"address":79,"country":82,"abbreviation":84},{"EN":77,"VI":78},"Hanoi Medical University","Trường Đại học Y Hà Nội",{"EN":80,"VI":81},"No. 01 Ton That Tung street, Trung Tu ward, Dong Da district, Hanoi city, Vietnam","Số 1 đường Tôn Thất Tùng, phường Trung Tự, quận Đống Đa, thành phố Hà Nội, Việt Nam",{"VOID":83},"VN",{"VOID":85},"HMU","https:\u002F\u002Fhmu.edu.vn\u002F",[],{"title":89},{"EN":78},{"title":91,"gsAuthor":93},{"EN":92},"Ngọc Thanh Kim",{"VOID":94},"chicgEwAAAAJ",{"id":96,"sortIndex":28,"researcher":18,"roles":97,"affiliations":98,"properties":110},"5f1bd7b2-b205-4da3-b716-3824e3e0ebc0",[],[99],{"id":72,"sortIndex":19,"affiliation":100,"properties":108},{"id":72,"createTime":18,"updateTime":18,"relativeEntities":101,"slug":18,"properties":102,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":86,"parentIds":107,"statistic":18},[],{"title":103,"address":104,"country":105,"abbreviation":106},{"EN":77,"VI":78},{"EN":80,"VI":81},{"VOID":83},{"VOID":85},[],{"title":109},{"EN":78},{"title":111},{"EN":112},"Hùng Tiến Lê",{"id":114,"sortIndex":30,"researcher":18,"roles":115,"affiliations":116,"properties":128},"4a9b4297-e964-498e-bb2b-a019dca24a3e",[],[117],{"id":72,"sortIndex":19,"affiliation":118,"properties":126},{"id":72,"createTime":18,"updateTime":18,"relativeEntities":119,"slug":18,"properties":120,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":86,"parentIds":125,"statistic":18},[],{"title":121,"address":122,"country":123,"abbreviation":124},{"EN":77,"VI":78},{"EN":80,"VI":81},{"VOID":83},{"VOID":85},[],{"title":127},{"EN":78},{"title":129},{"EN":130},"Tuấn Hải Nguyễn",{"id":132,"sortIndex":29,"researcher":18,"roles":133,"affiliations":134,"properties":146},"6383c035-ff9e-4dba-a0fc-fe7ac4de7777",[],[135],{"id":72,"sortIndex":19,"affiliation":136,"properties":144},{"id":72,"createTime":18,"updateTime":18,"relativeEntities":137,"slug":18,"properties":138,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":86,"parentIds":143,"statistic":18},[],{"title":139,"address":140,"country":141,"abbreviation":142},{"EN":77,"VI":78},{"EN":80,"VI":81},{"VOID":83},{"VOID":85},[],{"title":145},{"EN":78},{"title":147,"gsAuthor":149},{"EN":148},"Thanh Hương Trương",{"VOID":150},"gvDwpdYAAAAJ","ARTICLE",{"url":18,"publisher":153,"properties":166},{"id":6,"createTime":7,"updateTime":8,"relativeEntities":154,"slug":10,"properties":155,"entityType":16,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":19,"subjectFields":158,"manageAffiliations":159,"indexDatabases":160,"url":23,"thumbnailPath":18,"statistic":161,"gsStatistic":18,"type":35,"analyzePriority":18},[],{"issn":156,"title":157},{"VOID":13},{"VOID":15},[],[],[],{"impactFactor":19,"impactFactorByYear":162,"i10Index":19,"i10IndexLast5Year":19,"totalPublication":26,"totalPublicationByYear":163,"totalCitation":30,"totalCitationByYear":164,"totalCitationPerPublication":32,"totalCitationPerPublicationByYear":165,"hindexLast5Year":28,"hindex":28},{},{"2015":28,"2016":28,"2018":29,"2019":28,"2021":29,"2022":28,"2023":30},{"2015":28,"2018":28},{"2015":28,"2018":34},{"issue":167,"pages":169},{"VOID":168},"106",{"VOID":170},"51-55",{"total":19,"publishYear":172,"statisticByYear":173},2023,{},"2023-08-01","DONE_ANALYZE_CITATION","2026-07-29T19:55:09.146+00:00",[],[179,181,183,185,187,189,191,193,195,197,199,201,203,205,207,209,211],{"id":18,"text":180,"url":18,"identifiers":18},"Song, P., et al., Global, regional, and national prevalence and risk factors for peripheral artery disease in 2015: an updated systematic review and analysis. Lancet Glob Health, 2019. 7(8): p. e1020-e1030.",{"id":18,"text":182,"url":18,"identifiers":18},"Aboyans, V., et al., 2017 ESC Guidelines on the Diagnosis and Treatment of Peripheral Arterial Diseases, in collaboration with the European Society for Vascular Surgery (ESVS): Document covering atherosclerotic disease of extracranial carotid and vertebral, mesenteric, renal, upper and lower extremity arteriesEndorsed by: the European Stroke Organization (ESO)The Task Force for the Diagnosis and Treatment of Peripheral Arterial Diseases of the European Society of Cardiology (ESC) and of the European Society for Vascular Surgery (ESVS). Eur Heart J, 2018. 39(9): p. 763-816.",{"id":18,"text":184,"url":18,"identifiers":18},"Nguyễn Trường Sơn, et al., Hướng dẫn chẩn đoán và điều trị bệnh động mạch chi dưới. Tạp chí Tim mạch học Việt Nam, 2022(102): p. 5-19.",{"id":18,"text":186,"url":18,"identifiers":18},"Trần Xuân Thủy và Đinh Thị Thu Hương, Một số yếu tố liên quan tới chỉ số huyết áp tâm thu cổ chân- cánh tay (ABI) ở bệnh nhân bị bệnh động mạch chi dưới. Tạp chí y học Việt Nam, 2021. 500: p. 116-119.",{"id":18,"text":188,"url":18,"identifiers":18},"Yi, C., et al., Association between lipoprotein(a) and peripheral arterial disease in coronary artery bypass grafting patients. Clin Cardiol, 2023. 46(5): p. 512-520.",{"id":18,"text":190,"url":18,"identifiers":18},"Verwer, M.C., et al., High lipoprotein(a) is associated with major adverse limb events after femoral artery endarterectomy. Atherosclerosis, 2022. 349: p. 196-203.",{"id":18,"text":192,"url":18,"identifiers":18},"West, R., Tobacco smoking: Health impact, prevalence, correlates and interventions. Psychol Health, 2017. 32(8): p. 1018-1036.",{"id":18,"text":194,"url":18,"identifiers":18},"Aday, A.W. and B.M. Everett, Dyslipidemia Profiles in Patients with Peripheral Artery Disease. Curr Cardiol Rep, 2019. 21(6): p. 42.",{"id":18,"text":196,"url":18,"identifiers":18},"Murabito, J.M., et al., Intermittent claudication. A risk profile from The Framingham Heart Study. Circulation, 1997. 96(1): p. 44-9.",{"id":18,"text":198,"url":18,"identifiers":18},"Ness, J., W.S. Aronow, and C. Ahn, Risk factors for symptomatic peripheral arterial disease in older persons in an academic hospital-based geriatrics practice. J Am Geriatr Soc, 2000. 48(3): p. 312-4.",{"id":18,"text":200,"url":18,"identifiers":18},"Murabito, J.M., et al., Prevalence and clinical correlates of peripheral arterial disease in the Framingham Offspring Study. Am Heart J, 2002. 143(6): p. 961-5.",{"id":18,"text":202,"url":18,"identifiers":18},"Mach, F., et al., 2019 ESC\u002FEAS Guidelines for the management of dyslipidaemias: lipid modification to reduce cardiovascular risk: The Task Force for the management of dyslipidaemias of the European Society of Cardiology (ESC) and European Atherosclerosis Society (EAS). European Heart Journal, 2019. 41(1): p. 111-188.",{"id":18,"text":204,"url":18,"identifiers":18},"Visseren, F.L.J., et al., 2021 ESC Guidelines on cardiovascular disease prevention in clinical practice: Developed by the Task Force for cardiovascular disease prevention in clinical practice with representatives of the European Society of Cardiology and 12 medical societies With the special contribution of the European Association of Preventive Cardiology (EAPC). European Heart Journal, 2021. 42(34): p. 3227-3337.",{"id":18,"text":206,"url":18,"identifiers":18},"Olov Wikluynd, et al., The Clinician’s Handbook: Dyslipidaemia and Atherosclerosis Prevention, Diagnosis and Treatment. 2023: European Atherosclerosis Society.",{"id":18,"text":208,"url":18,"identifiers":18},"Hirsch, A.T. and A.M. Gotto, Jr., Undertreatment of dyslipidemia in peripheral arterial disease and other high-risk populations: an opportunity for cardiovascular disease reduction. Vasc Med, 2002. 7(4): p. 323-31.",{"id":18,"text":210,"url":18,"identifiers":18},"Thalmann, I., et al., Population-wide cohort study of statin use for the secondary cardiovascular disease prevention in Scotland in 2009-2017. Heart, 2023. 109(5): p. 388-395.",{"id":18,"text":212,"url":18,"identifiers":18},"Aboyans, V. and L. Chastaingt, What LEADs to the under-treatment of patients with lower-extremity artery disease? European Journal of Preventive Cardiology, 2023.",false,{"id":215,"createTime":216,"updateTime":217,"relativeEntities":218,"slug":219,"properties":220,"entityType":59,"verifyStatus":60,"verifyTime":227,"verifyNote":61,"languages":228,"translateLanguages":18,"viewCount":19,"primaryUrl":229,"fullTextUrl":230,"authors":231,"publicationType":151,"publisherRelationship":284,"citationCount":19,"citationInfo":298,"publishDate":301,"publishYear":299,"citationAnalyzeStatus":17,"lastCitationAnalyze":302,"indexDatabases":303,"openAccess":18,"references":304,"isForceReanalyzing":213},"da295352-a074-4cf2-988f-14d3cb1616d4","2024-04-11T18:09:23.516+00:00","2026-07-23T13:46:53.368+00:00",[],"Y%E1%BA%BFu-t%E1%BB%91-di-truy%E1%BB%81n-trong-b%E1%BB%87nh-l%C3%BD-%C4%91%E1%BB%99ng-m%E1%BA%A1ch-v%C3%A0nh-vai-tr%C3%B2-c%E1%BB%A7a-c%C3%A1c-nghi%C3%AAn-c%E1%BB%A9u-li%C3%AAn-quan-%C4%91%E1%BA%BFn-sinh-h%E1%BB%8Dc-ph%C3%A2n-t%E1%BB%AD",{"title":221,"gsPaper":223,"abstract":225},{"VI":222},"Yếu tố di truyền trong bệnh lý động mạch vành - vai trò của các nghiên cứu liên quan đến sinh học phân tử",{"VOID":224},"14093785667933274826",{"VI":226},"Bệnh tim mạch là một trong những nguyên nhân gây tử vong hàng đầu thế giới. Ở Việt Nam, 1\u002F4 số người tử vong mỗi năm do mắc các vấn đề về tim mạch, trong đó bệnh lý động mạch vành (ĐMV) là một trong những nguyên nhân thường gặp nhất.\r\nVề mặt lâm sàng, xơ vữa động mạch do hậu quả của sự lắng đọng cholesterol và quá trình viêm trong thành động mạch, cả hai đều được kích hoạt bởi các yếu tố nguy cơ truyền thống như tăng huyết áp, tăng lipid máu hoặc đái tháo đường. Một yếu tố nguy cơ khác là đột biến gen được xác định ở cá thể có tiền sử gia đình dương tính. Trong những năm gần đây, nhiều nghiên cứu sinh học phân tử đã xác định vai trò tác động mạch mẽ của các biến thể di truyền đến biểu hiện của xơ vữa ĐMV. Kết quả là đã có164 locus (vị trí) trên nhiễm sắc thể (NST) được xác định bởi các nghiên cứu liên kết trên toàn bộ hệ gen (GWAS) tác động tới nguy cơ mắc bệnh ĐMV. Trên thực tế nghiên cứu, tất cả các đột biến gen nguy cơ được phát hiện bởi GWAS thường được tìm thấy trong quần thể dân số chung, như mỗi cá thể khu vực Tây Âu mang từ 130 đến 190 alen nguy cơ tại các locus đã được biết đến trong toàn bộ hệ gen (có 0, 1 hoặc 2 alen nguy cơ trên mỗi locus). Có thể giả định rằng, cách phân bố đa dạng này khiến cho con người dễ bị ảnh hưởng bởi các yếu tố lối sống, làm tăng nguy cơ xơ vữa ĐMV. Cho đến nay, đã có nhiều đột biến gen liên quan đến nguy cơ bệnh ĐMV đã được phát hiện và các nhà khoa học đã cố gắng nhóm các đột biến gen này vào trong các nhóm chức năng để từ đó đưa ra các quan điểm phòng ngừa hoặc điều trị. Tại Việt Nam, từ năm 2013, sau khi phát hiện đột biến gen Fibrinogen beta (FGB) ở một bệnh nhân nhồi máu cơ tim (NMCT) có cơ địa dễ hình thành huyết khối, đã có nhiều nghiên cứu khác trong lĩnh vực này được tiến hành nhằm xác định các đột biến gen mới trong các bệnh lý tim mạch nói chung và bệnh lý ĐMV nói riêng. Điều này có ý nghĩa rất lớn trong việc ghi nhận đặc điểm đột biến gen tác động đến tỉ lệ mắc bệnh lý ĐMV nói riêng ở Việt Nam, góp phần thay đổi nhận thức của nhân viên y tế trong sàng lọc, chẩn đoán cũng như điều trị các yếu tố nguy cơ liên quan đến đột biến gen cũng như các tổn thương tim mạch liên quan.","2025-01-10T03:32:12.164+00:00",[63],"https:\u002F\u002Fjvc.vnha.org.vn\u002Ftmh\u002Farticle\u002Fview\u002F417","https:\u002F\u002Fjvc.vnha.org.vn\u002Ftmh\u002Farticle\u002Fdownload\u002F417\u002F401",[232,249,269],{"id":233,"sortIndex":19,"researcher":18,"roles":234,"affiliations":235,"properties":245},"62ff1793-956d-4c68-af45-b26fde14114c",[],[236],{"id":237,"sortIndex":19,"affiliation":238,"properties":18},"fc692af9-e7c6-407c-a29f-ad535920d306",{"id":237,"createTime":18,"updateTime":18,"relativeEntities":239,"slug":18,"properties":240,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":243,"statistic":18},[],{"title":241},{"VI":242}," Viện Tim mạch Việt Nam - Bệnh viện Bạch Mai",[244],"d18f005f-5831-42cd-8bd3-630a3a5767fb",{"title":246,"gsAuthor":248},{"EN":247},"Trương Thanh Hương",{"VOID":150},{"id":250,"sortIndex":28,"researcher":18,"roles":251,"affiliations":252,"properties":266},"82eb6d17-4c41-4638-967f-9c1245d2c872",[],[253],{"id":254,"sortIndex":19,"affiliation":255,"properties":264},"053efbfe-ef28-4a48-8865-fe5d3062fa75",{"id":254,"createTime":18,"updateTime":18,"relativeEntities":256,"slug":18,"properties":257,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":262,"parentIds":263,"statistic":18},[],{"title":258,"country":261},{"EN":259,"VI":260},"Vietnam National Heart Institute, Bach Mai Hospital","Viện Tim Mạch Việt Nam, Bệnh Viện Bạch Mai",{"VOID":83},"http:\u002F\u002Fvientimmach.vn\u002F",[244],{"title":265},{"EN":242},{"title":267},{"EN":268},"Nguyễn Thị Mai Ngọc",{"id":270,"sortIndex":30,"researcher":18,"roles":271,"affiliations":272,"properties":279},"2557dbd8-3ade-4d8a-998a-02953b5fe50b",[],[273],{"id":237,"sortIndex":19,"affiliation":274,"properties":18},{"id":237,"createTime":18,"updateTime":18,"relativeEntities":275,"slug":18,"properties":276,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":278,"statistic":18},[],{"title":277},{"VI":242},[244],{"title":280,"gsAuthor":282},{"EN":281},"Nguyễn Thị Duyên",{"VOID":283},"u7Y3lwwAAAAJ",{"url":18,"publisher":285,"properties":18},{"id":6,"createTime":7,"updateTime":8,"relativeEntities":286,"slug":10,"properties":287,"entityType":16,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":19,"subjectFields":290,"manageAffiliations":291,"indexDatabases":292,"url":23,"thumbnailPath":18,"statistic":293,"gsStatistic":18,"type":35,"analyzePriority":18},[],{"issn":288,"title":289},{"VOID":13},{"VOID":15},[],[],[],{"impactFactor":19,"impactFactorByYear":294,"i10Index":19,"i10IndexLast5Year":19,"totalPublication":26,"totalPublicationByYear":295,"totalCitation":30,"totalCitationByYear":296,"totalCitationPerPublication":32,"totalCitationPerPublicationByYear":297,"hindexLast5Year":28,"hindex":28},{},{"2015":28,"2016":28,"2018":29,"2019":28,"2021":29,"2022":28,"2023":30},{"2015":28,"2018":28},{"2015":28,"2018":34},{"total":19,"publishYear":299,"statisticByYear":300},2018,{},"2018-10-01","2026-07-23T13:46:53.367+00:00",[],[305,307,309,311,313,315,317,319,321,323,325,327,329,331,333,335,337,339,341,343,345,347,349,351,353,355,357,359,361,363,365,367,369,371,373,375,377,379],{"id":18,"text":306,"url":18,"identifiers":18},"1. Myers RH, Kiely DK, Cupples LA, Kannel WB (1990) Parental history is an independent risk factor for coronary artery disease: the Framingham Study. Am Heart J 120:963–969.",{"id":18,"text":308,"url":18,"identifiers":18},"2. Marenberg ME, Risch N, Berkman LF, Floderus B, de Faire U (1994) Genetic susceptibility to death from coronary heart disease in a study of twins. N Engl J Med 330:1041–1046.",{"id":18,"text":310,"url":18,"identifiers":18},"3. Erdmann J, Stark K, Esslinger UB et al (2013) Dysfunctional nitric oxide signalling increases risk of myocardial infarction. Nature 504:432–436.",{"id":18,"text":312,"url":18,"identifiers":18},"4. Goldstein JL, Brown MS (2009) “History of discovery: the LDL receptor”. Arterioscler Thromb Vasc Biol 29:431–438",{"id":18,"text":314,"url":18,"identifiers":18},"5. Kessler T, Vilne B, Schunkert H (2016) The impact of genome- wide association studies on the pathophysiology and therapy of cardiovascular disease. EMBO Mol Med 8:688–701",{"id":18,"text":316,"url":18,"identifiers":18},"6. Samani NJ, Erdmann J, Hall AS, Hengstenberg C et al (2007) WTCCC, Cardiogenics. Genomewide association analysis of coronary artery disease. N Engl J Med 357:443–453",{"id":18,"text":318,"url":18,"identifiers":18},"7. Erdmann J, Grosshennig A, Braund PS et al (2009) New suscep- tibility locus for coronary artery disease on chromosome 3q22.3. Nat Genet 41:280–282.",{"id":18,"text":320,"url":18,"identifiers":18},"8. Schunkert H, Konig IR, Kathiresan S et al (2011). Large-scale association analysis identi es 13 new susceptibility loci for coronary artery disease. Nat Genet 43:333–338.",{"id":18,"text":322,"url":18,"identifiers":18},"9. Nelson CP, Goel A, Butterworth AS et al (2017) Association analyses based on false discovery rate implicate new loci for coronary artery disease. Nat Genet 49:1385–1391.",{"id":18,"text":324,"url":18,"identifiers":18},"10. Van der Harst P, Verweij N (2018) The identication of 64 novel genetic loci provides an expanded view on the genetic architecture of coronary artery disease. Circ Res 122:433–443.",{"id":18,"text":326,"url":18,"identifiers":18},"11. Erdmann J, Kessler T, Munoz Venegas L, Schunkert H (2018). A decade of genome-wide association studies for coronary artery disease: the challenges ahead. Cardiovasc Res. https:\u002F\u002Fdoi. org\u002F10.1093\u002Fcvr\u002F cvy084.",{"id":18,"text":328,"url":18,"identifiers":18},"12. Visscher PM, Wray NR, Zhang Q, Sklar P, McCarthy MI, Brown MA, Yang J (2017) 10 years of GWAS discovery: biology, func- tion, and translation. Am J Hum Genet 101(1):5–22.",{"id":18,"text":330,"url":18,"identifiers":18},"13. Stitziel NO, Stirrups KE, Masca NG et al (2016) Coding variation in ANGPTL4, LPL, and SVEP1 and the risk of coronary disease. N Engl J Med 374:1134–1144.",{"id":18,"text":332,"url":18,"identifiers":18},"14. Webb TR, Erdmann J, Stirrups KE et al (2017) Systematic evaluation of pleiotropyidenties 6 further loci associated with coronary artery disease. J Am Coll Cardiol 69:823–836",{"id":18,"text":334,"url":18,"identifiers":18},"15. Schunkert H (2016) Family or SNPs: what counts for hereditary risk of coronary artery disease? Eur Heart J 37:568–571",{"id":18,"text":336,"url":18,"identifiers":18},"16. Braenne I, Kleinecke M, Reiz B et al (2016) Systematic analysis of variants related to familial hypercholesterolemia in families with premature myocardial infarction. Eur J Hum Genet 24:191–197",{"id":18,"text":338,"url":18,"identifiers":18},"17. Stranneheim H, Wedell A (2016) Exome and genome sequenc- ing: a revolution for the discovery and diagnosis of monogenic disorders. J Intern Med 279(1):3–15",{"id":18,"text":340,"url":18,"identifiers":18},"18. Trương Thanh Hương và cs. Phát hiện đột biến gen Fibrinogen beta (FGB) ở một bệnh nhân nhồi máu cơ tim có cơ địa dễ hình thành huyết khối, Y học Việt Nam, 1\u002F2013, tr. 58.",{"id":18,"text":342,"url":18,"identifiers":18},"19. Trương Thanh Hương, Dương Đức Hùng, Nguyễn Thị Mai Ngọc, Lương Thị Lan Anh, Nguyễn Thị Trang. Phát hiện đột biến gen tổng hợp chuỗi nặng beta-myosin (YH7) trong bệnh cơ tim phì đại. Tạp chí Tim mạch học Việt Nam, số 65\u002F2014",{"id":18,"text":344,"url":18,"identifiers":18},"20. Gerald F Watts, Phillip YA Ding, Thanh Huong Truong et al. Translational Research for Improving the Care of Familial Hypercholesterolemia: The “Ten Countries Study” and Beyond. J Atheroscler Thromb, 2016; 23: 891-900. doi: 10.5551\u002Fjat.35949.",{"id":18,"text":346,"url":18,"identifiers":18},"21. Kim Ngọc Thanh, Trần Trung Thành, Trần Đức Huy, Lê Hồng An, Trương Thanh Hương. Nghiên cứu bước đầu về tình trạng bệnh tăng Cholesterol máu có tính chất gia đình ở các trường hợp mắc bệnh ĐMV. Tạp chí tim mạch học Việt Nam – số 81.2018",{"id":18,"text":348,"url":18,"identifiers":18},"22. Nguyễn Thị Thúy Mậu, Vũ Ngọc Trung, Nguyễn Hữu Hiếu, Nguyễn Thanh Thúy, Vũ Thị Thơm, Đinh Đoàn Long, Trịnh Hoàng Hà. Bước đầu đánh giá mối liên quan giữa đa hình gen CYP2C19 và độ ngưng tập tiểu cầu trên bệnh nhân nhồi máu cơ tim cấp tại một số bệnh viện ở Hà Nội. Tạp chí Khoa học ĐHQGHN: Khoa học Y Dược, Tập 33, Số 2 (2017) 68-74",{"id":18,"text":350,"url":18,"identifiers":18},"23. Khera AV, Won HH, Peloso GM, et al (2017) Association of rare and common variation in the",{"id":18,"text":352,"url":18,"identifiers":18},"lipoprotein lipase gene with coronary artery disease. JAMA 7(9):937–946 317.",{"id":18,"text":354,"url":18,"identifiers":18},"24. Ukraintseva S, Yashin A, Arbeev K, Kulminski A, Akushevich I, Wu D, Joshi G, Land KC, Stallard E (2016) Puzzling role of genetic risk factors in human longevity: “risk alleles” as pro-lon- gevity variants. Biogerontology 17(1):109–127",{"id":18,"text":356,"url":18,"identifiers":18},"25. Jansen H, Samani NJ, Schunkert H (2014) Mendelian randomiza- tion studies in coronary artery disease. Eur Heart J 35:1917–1924",{"id":18,"text":358,"url":18,"identifiers":18},"26. Voight BF, Peloso GM, Orho-Melander M, et al (2008) Schunkert HLifelong reduction of LDL- cholesterol related to a common variant in the LDL-receptor gene decreases the risk of coronary artery disease a Mendelian Randomisation study. PLoS One 3:e2986",{"id":18,"text":360,"url":18,"identifiers":18},"27. Linsel-Nitschke P, Götz A, Erdmann J, et al (2008) Schunkert HLifelong reduction of LDL",{"id":18,"text":362,"url":18,"identifiers":18},"-cholesterol related to a common variant in the LDL-receptor gene decreases the risk of coronary artery disease a Mendelian Randomisation study. PLoS One 3:e2986",{"id":18,"text":364,"url":18,"identifiers":18},"28. Nelson MR, Tipney H, Painter JL, Shen J, Nicoletti P, Shen Y, Floratos A, Sham PC, Li MJ, Wang J, Cardon LR, Whittaker JC, Sanseau P (2015) The support of human genetic evidence for approved drug indications. Nat Genet 47:856–860",{"id":18,"text":366,"url":18,"identifiers":18},"29. Abraham G, Havulinna AS, Bhalala OG, Byars SG, De Livera AM, Yetukuri L, Tikkanen E, Perola M, Schunkert H, Sijbrands EJ, Palotie A, Samani NJ, Salomaa V, Ripatti S, Inouye M (2016) Genomic prediction of coronary heart disease. Eur Heart J 37:3267–3278",{"id":18,"text":368,"url":18,"identifiers":18},"30. International Consortium for Blood Pressure Genome-Wide Association Studies, Ehret GB, Munroe PB, Rice KM, Bochud M et al. Genetic variants in novel pathways in uence blood pressure and cardiovascular disease risk. Nature 2011; 478(7367):103–109",{"id":18,"text":370,"url":18,"identifiers":18},"31. Morris AP, Voight BF, Teslovich TM, et al. Diabetes Genetics Replication And Meta- analysis (DIAGRAM) Consortium. Large-scale association analysis provides insights into the genetic architecture and pathophysiology of type 2 diabetes. Nat Genet. 2012;44(9):981–990",{"id":18,"text":372,"url":18,"identifiers":18},"32. Willer CJ, Schmidt EM, Sengupta S, et al (2018) Network analysis of coronary artery disease risk genes elucidates disease mechanisms and druggable targets. Sci Rep 8:3434",{"id":18,"text":374,"url":18,"identifiers":18},"33. Lempiäinen H, Brænne I, Michoel T, et al (2018) Network analysis of coronary artery disease risk genes elucidates disease mechanisms and druggable targets. Sci Rep 8:3434",{"id":18,"text":376,"url":18,"identifiers":18},"34. Braenne I, Civelek M, Vilne B, et al (2015). Prediction of causal candidate genes in coronary artery disease loci. Arterioscler Thromb Vasc Biol 35:2207–2217",{"id":18,"text":378,"url":18,"identifiers":18},"35. Kessler T, Wobst J, Wolf B, et al (2017). Functional characterization of the GUCY1A3 coronary artery disease risk locus. Circulation 136:476–489",{"id":18,"text":380,"url":18,"identifiers":18},"36. Chen CC1, Chen LL, Hsu YT, Liu KJ, Fan CS, Huang TS (2014) The endothelin-integrin axis is involved in macrophage-induced breast cancer cell chemotactic interactions with endothelial cells. J Biol Chem 289:10029–10044.",{"id":382,"createTime":383,"updateTime":383,"relativeEntities":384,"slug":385,"properties":386,"entityType":59,"verifyStatus":60,"verifyTime":393,"verifyNote":61,"languages":394,"translateLanguages":18,"viewCount":19,"primaryUrl":395,"fullTextUrl":18,"authors":396,"publicationType":151,"publisherRelationship":512,"citationCount":18,"citationInfo":18,"publishDate":533,"publishYear":172,"citationAnalyzeStatus":17,"lastCitationAnalyze":18,"indexDatabases":534,"openAccess":18,"references":535,"isForceReanalyzing":213},"c5357d3b-af9a-4a9a-9c2d-6198bf049608","2026-07-21T23:12:32.038+00:00",[],"Assessment-of-left-ventricular-systolic-function-in-patients-with-chronic-coronary-syndrome-and-heart-failure-with-reduced-ejection-fraction-after-percutaneous-coronary-intervention",{"title":387,"doi":389,"abstract":391},{"EN":388,"VI":388},"Assessment of left ventricular systolic function in patients with chronic coronary syndrome and heart failure with reduced ejection fraction after percutaneous coronary intervention",{"VOID":390},"10.58354\u002Fjvc.107e.2023.705",{"VI":392,"EN":392},"Objective: Describe the characteristic and change of left ventricular systolic function after percutaneous coronary intervention in patients with chronic coronary syndrome and heart failure with reduced ejection fraction after percutaneous coronary intervention. Methods: This prospective study involved the monitoring of 40 patients who had an ejection fraction of 40% or less and were diagnosed with chronic coronary artery disease. These patients underwent a successful percutaneous coronary intervention at the Vietnam National Heart Institute and Hanoi Medical University Hospital between September 2022 and August 2023. The main objective is to evaluate the left ventricular systolic function following the intervention using 2D echocardiography. Results: A total of 40 patients (34 male and 6 females) with a mean age of 68.3 ± 10.2, were assessed for left ventricular systolic function using 2D echocardiography before and after the intervention. In paired assessment at 90-day follow-up, baseline LVEF improved significantly (before intervention: 32.8 ± 7.2% and after: 38,7 ± 7,3%, p \u003C 0.01). Left ventricular longitudinal strain improved significantly on all cross-sections such as: 4-chamber GLS (before: -10.4 ± 3.97%, after: -13.6 ± 4.3%, p \u003C 0.01), 2-chamber GLS (before: -10.6 ± 3.8%, after: -13.1 ± 4.2%, p \u003C 0.01), 3-chamber GLS (before: -9.8 ± 3.7%, after: -12.3 ± 4.6%, p \u003C 0.01), GLS Avg (before: -10.3 ± 3.6%, after: -13 ± 4.1%, p\u003C 0.01), basal GLS (before: -11.2 ± 3.8%, after: -14 ± 4.5%, p \u003C 0.01), middle GLS (before: -9.3 ± 4.4%, after: -11.8 ± 3.9%, p \u003C 0.01), apical GLS (before: -11.5 ± 5.1%, after: -14.3 ± 5.9%, p \u003C 0.01). In comparison with the incomplete-revascularization group, there was a significant improvement in left ventricular systolic function in the complete-revascularization group (OR= 22.17, p \u003C 0.01). Conclusions: In patients with chronic coronary syndrome and a reduced left ventricular ejection fraction, the systolic function of the left ventricle was enhanced following percutaneous coronary intervention, particularly in the group that underwent complete revascularization.","2026-07-21T23:12:32.037+00:00",[63],"http:\u002F\u002Fjvc.vnha.org.vn\u002Ftmh\u002Farticle\u002Fview\u002F705",[397,411,424,439,454,468,484,498],{"id":398,"sortIndex":19,"researcher":18,"roles":399,"affiliations":400,"properties":408},"8893dab4-83d2-42ae-91de-3e1ea95b52e6",[],[401],{"id":402,"sortIndex":19,"affiliation":403,"properties":18},"90f08c87-4a3d-4ba4-bee8-9f2130d79fa8",{"id":402,"createTime":18,"updateTime":18,"relativeEntities":404,"slug":18,"properties":405,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":407,"statistic":18},[],{"title":406},{"VI":77},[],{"title":409},{"VI":410},"Dao Anh 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Hoa",{"url":18,"publisher":513,"properties":526},{"id":6,"createTime":7,"updateTime":8,"relativeEntities":514,"slug":10,"properties":515,"entityType":16,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":19,"subjectFields":518,"manageAffiliations":519,"indexDatabases":520,"url":23,"thumbnailPath":18,"statistic":521,"gsStatistic":18,"type":35,"analyzePriority":18},[],{"issn":516,"title":517},{"VOID":13},{"VOID":15},[],[],[],{"impactFactor":19,"impactFactorByYear":522,"i10Index":19,"i10IndexLast5Year":19,"totalPublication":26,"totalPublicationByYear":523,"totalCitation":30,"totalCitationByYear":524,"totalCitationPerPublication":32,"totalCitationPerPublicationByYear":525,"hindexLast5Year":28,"hindex":28},{},{"2015":28,"2016":28,"2018":29,"2019":28,"2021":29,"2022":28,"2023":30},{"2015":28,"2018":28},{"2015":28,"2018":34},{"issue":527,"pages":529,"volume":531},{"VOID":528},"107",{"VOID":530},"",{"VOID":532},"1","2023-11-30",[],[536,538,540,542,544,546,548,550,552,554,556,558,560,562],{"id":18,"text":537,"url":18,"identifiers":18},"Vedin O, Lam CSP, Koh AS, et al. Significance of Ischemic Heart Disease in Patients With Heart Failure and Preserved, Midrange, and Reduced Ejection Fraction: A Nationwide Cohort Study. Circ: Heart Failure. 2017;10(6):e003875. doi: 10.1161\u002FCIRCHEARTFAILURE.117.003875",{"id":18,"text":539,"url":18,"identifiers":18},"Perera D, Clayton T, O’Kane PD, et al. Percutaneous Revascularization for Ischemic Left Ventricular Dysfunction. N Engl J Med. 2022;387(15):1351-1360. doi: 10.1056\u002FNEJMoa2206606",{"id":18,"text":541,"url":18,"identifiers":18},"Velazquez EJ, Lee KL, Jones RH, et al. Coronary-Artery Bypass Surgery in Patients with Ischemic Cardiomyopathy. N Engl J Med. 2016;374(16):1511-1520. doi: 10.1056\u002FNEJMoa1602001",{"id":18,"text":543,"url":18,"identifiers":18},"DeVore AD, Yow E, Krucoff MW, et al. Percutaneous coronary intervention outcomes in patients with stable coronary disease and left ventricular systolic dysfunction. ESC Heart Failure. 2019;6(6):1233-1242. doi: 10.1002\u002Fehf2.12510",{"id":18,"text":545,"url":18,"identifiers":18},"Wolff G, Dimitroulis D, Andreotti F, et al. Survival Benefits of Invasive Versus Conservative Strategies in Heart Failure in Patients With Reduced Ejection Fraction and Coronary Artery Disease: A Meta-Analysis. Circ: Heart Failure. 2017;10(1):e003255. doi: 10.1161\u002FCIRCHEARTFAILURE.116.003255",{"id":18,"text":547,"url":18,"identifiers":18},"Pathak S, Lai FY, Miksza J, et al. Surgical or percutaneous coronary revascularization for heart failure: an in silico model using routinely collected health data to emulate a clinical trial. European Heart Journal. 2023;44(5):351-364. doi: 10.1093\u002Feurheartj\u002Fehac670",{"id":18,"text":549,"url":18,"identifiers":18},"Velazquez EJ, Lee KL, Deja MA, et al. Coronary-Artery Bypass Surgery in Patients with Left Ventricular Dysfunction. N Engl J Med. 2011;364(17):1607-1616. doi: 10.1056\u002FNEJMoa1100356",{"id":18,"text":551,"url":18,"identifiers":18},"Smiseth OA, Torp H, Opdahl A, et al. Myocardial strain imaging: how useful is it in clinical decision making? Eur Heart J. 2016;37(15):1196-1207. doi: 10.1093\u002Feurheartj\u002Fehv529",{"id":18,"text":553,"url":18,"identifiers":18},"Blessberger H, Binder T. Two dimensional speckle tracking echocardiography: clinical applications. Heart. 2010;96(24):2032-2040. doi: 10.1136\u002Fhrt.2010.199885",{"id":18,"text":555,"url":18,"identifiers":18},"Lobo AS, Cantu SM, Sharkey SW, et al. Revascularization in Patients With Spontaneous Coronary Artery Dissection and ST-Segment Elevation Myocardial Infarction. Journal of the American College of Cardiology. 2019;74(10):1290-1300. doi: 10.1016\u002Fj.jacc.2019.06.065",{"id":18,"text":557,"url":18,"identifiers":18},"Bangalore S, Guo Y, Samadashvili Z, et al. Outcomes With Complete Versus Incomplete Revascularization in Patients With Multivessel Coronary Disease Undergoing Percutaneous Coronary Intervention With Everolimus Eluting Stents. The American Journal of Cardiology. 2020;125(3):362-369. doi: 10.1016\u002Fj.amjcard.2019.10.022",{"id":18,"text":559,"url":18,"identifiers":18},"Kramer DG, Trikalinos TA, Kent DM, et al. Quantitative Evaluation of Drug or Device Effects on Ventricular Remodeling as Predictors of Therapeutic Effects on Mortality in Patients With Heart Failure and Reduced Ejection Fraction. Journal of the American College of Cardiology. 2010;56(5):392-406. doi: 10.1016\u002Fj.jacc.2010.05.011",{"id":18,"text":561,"url":18,"identifiers":18},"Adachi Y, Sakakura K, Wada H, et al. Determinants of Left Ventricular Systolic Function Improvement Following Coronary Artery Revascularization in Heart Failure Patients With Reduced Ejection Fraction (HFrEF). Int Heart J. 2016;57(5):565-572. doi: 10.1536\u002Fihj.16-087",{"id":18,"text":563,"url":18,"identifiers":18},"Russo JJ, Prasad M, Doshi D, et al. Improvement in left ventricular function following higher‐risk percutaneous coronary intervention in patients with ischemic cardiomyopathy. Cathet Cardio Intervent. 2020;96(4):764-770. doi: 10.1002\u002Fccd.28557",{"id":565,"createTime":566,"updateTime":567,"relativeEntities":568,"slug":569,"properties":570,"entityType":59,"verifyStatus":60,"verifyTime":577,"verifyNote":61,"languages":578,"translateLanguages":18,"viewCount":19,"primaryUrl":579,"fullTextUrl":580,"authors":581,"publicationType":151,"publisherRelationship":671,"citationCount":19,"citationInfo":690,"publishDate":693,"publishYear":691,"citationAnalyzeStatus":17,"lastCitationAnalyze":694,"indexDatabases":695,"openAccess":18,"references":696,"isForceReanalyzing":213},"0ffe2952-d669-4c15-98e4-7d4d93962658","2024-11-25T18:34:42.604+00:00","2026-07-20T15:05:25.775+00:00",[],"R%E1%BB%91i-lo%E1%BA%A1n-ch%E1%BB%A9c-n%C4%83ng-th%E1%BA%ADn-%E1%BB%9F-b%E1%BB%87nh-nh%C3%A2n-tim-b%E1%BA%A9m-sinh-ng%C6%B0%E1%BB%9Di-l%E1%BB%9Bn-v%C3%A0-c%C3%A1c-y%E1%BA%BFu-t%E1%BB%91-li%C3%AAn-quan",{"title":571,"gsPaper":573,"abstract":575},{"VI":572},"Rối loạn chức năng thận ở bệnh nhân tim bẩm sinh người lớn và các yếu tố liên quan",{"VOID":574},"930691213892245242",{"VI":576},"Mục tiêu nghiên cứu: Tại Việt Nam, các hiểu biết về chức năng thận ở người lớn mắc tim bẩm sinh (TBS) còn hạn chế. Nghiên cứu này nhằm xác định tỉ lệ rối loạn chức năng thận, và các yếu tố liên quan đến rối loạn chức năng thận ở người lớn mắc TBS.\r\nPhương pháp nghiên cứu: Đây là nghiên cứu mô tả cắt ngang, gồm 230 bệnh nhân TBS >16 tuổi. Các thông tin được thu thập bao gồm ure, creatinine huyết thanh, mức lọc cầu thận (MLCT), tuổi, giới, phân suất tống máu thất trái, NT-proBNP huyết thanh, tăng áp động mạch phổi, rung nhĩ, thiếu máu, đa hồng cầu. Tỉ suất chênh (OR) cho MLCT <90ml\u002Fphút\u002F1.73 m2 da được xác định.Kết quả: 73.1% bệnh nhân có rối loạn chức năng thận, bao gồm 9.6% có MLCT >120 ml\u002Fphút\u002F1.73 m2 da và 63.5% có MLCT <90 ml\u002Fphút\u002F1.73 m2 da. Ở bệnh nhân TBS shunt trái-phải, tỉ suất chênh OR bằng 6.58 (CI95%: 1.50 – 28.96) lần so sánh giữa nhóm tuổi >60 và ≤60, và tỉ suất chênh OR bằng 2.65 (CI95%: 1.24 – 5.67) lần khi so sánh giữa nhóm SpO2 < 95% và SpO2 ≥95%.\r\nKết luận: Rối loạn chức năng thận là thường gặp ở TBS người lớn. Các yếu tố liên quan đến rối loạn chức năng thận ở là tuổi >60 và SpO2 <95%.\r\nTừ khóa: Rối loạn chức năng thận, tim bẩm sinh, người lớn","2024-11-25T18:34:42.603+00:00",[63],"https:\u002F\u002Fjvc.vnha.org.vn\u002Ftmh\u002Farticle\u002Fview\u002F372","https:\u002F\u002Fjvc.vnha.org.vn\u002Ftmh\u002Farticle\u002Fdownload\u002F372\u002F356",[582,596,615,634,647,659],{"id":583,"sortIndex":19,"researcher":18,"roles":584,"affiliations":585,"properties":592},"cc7fae4c-5ef5-4b4f-9e56-8f97d5c77363",[],[586],{"id":237,"sortIndex":19,"affiliation":587,"properties":18},{"id":237,"createTime":18,"updateTime":18,"relativeEntities":588,"slug":18,"properties":589,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":591,"statistic":18},[],{"title":590},{"VI":242},[244],{"title":593,"gsAuthor":595},{"EN":594},"Kim Ngọc Thanh",{"VOID":94},{"id":597,"sortIndex":28,"researcher":18,"roles":598,"affiliations":599,"properties":612},"126a8e33-f80f-48e3-b124-85371e452d28",[],[600],{"id":72,"sortIndex":19,"affiliation":601,"properties":609},{"id":72,"createTime":18,"updateTime":18,"relativeEntities":602,"slug":18,"properties":603,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":86,"parentIds":608,"statistic":18},[],{"title":604,"address":605,"country":606,"abbreviation":607},{"EN":77,"VI":78},{"EN":80,"VI":81},{"VOID":83},{"VOID":85},[],{"title":610},{"EN":611}," Đại học Y Hà Nội",{"title":613},{"EN":614},"Phan Nhật Quang",{"id":616,"sortIndex":30,"researcher":18,"roles":617,"affiliations":618,"properties":631},"58398114-f9f0-4b47-86c1-350c9e05534f",[],[619],{"id":72,"sortIndex":19,"affiliation":620,"properties":628},{"id":72,"createTime":18,"updateTime":18,"relativeEntities":621,"slug":18,"properties":622,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":86,"parentIds":627,"statistic":18},[],{"title":623,"address":624,"country":625,"abbreviation":626},{"EN":77,"VI":78},{"EN":80,"VI":81},{"VOID":83},{"VOID":85},[],{"title":629},{"EN":630},"Đại học Y Hà Nội",{"title":632},{"EN":633},"Đinh Thị Kim Dung",{"id":635,"sortIndex":29,"researcher":18,"roles":636,"affiliations":637,"properties":644},"7ac6b27f-1b15-4ecc-9783-77e47020d5fb",[],[638],{"id":237,"sortIndex":19,"affiliation":639,"properties":18},{"id":237,"createTime":18,"updateTime":18,"relativeEntities":640,"slug":18,"properties":641,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":643,"statistic":18},[],{"title":642},{"VI":242},[244],{"title":645},{"EN":646},"Lê Thanh Tùng",{"id":648,"sortIndex":456,"researcher":18,"roles":649,"affiliations":650,"properties":657},"74fa8956-cbff-474f-bb0c-6fdf25ddb7b9",[],[651],{"id":237,"sortIndex":19,"affiliation":652,"properties":18},{"id":237,"createTime":18,"updateTime":18,"relativeEntities":653,"slug":18,"properties":654,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":656,"statistic":18},[],{"title":655},{"VI":242},[244],{"title":658},{"EN":268},{"id":660,"sortIndex":470,"researcher":18,"roles":661,"affiliations":662,"properties":669},"5f5f2037-78e8-40fc-bebe-71fff516cd02",[],[663],{"id":237,"sortIndex":19,"affiliation":664,"properties":18},{"id":237,"createTime":18,"updateTime":18,"relativeEntities":665,"slug":18,"properties":666,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":668,"statistic":18},[],{"title":667},{"VI":242},[244],{"title":670},{"EN":247},{"url":18,"publisher":672,"properties":685},{"id":6,"createTime":7,"updateTime":8,"relativeEntities":673,"slug":10,"properties":674,"entityType":16,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":19,"subjectFields":677,"manageAffiliations":678,"indexDatabases":679,"url":23,"thumbnailPath":18,"statistic":680,"gsStatistic":18,"type":35,"analyzePriority":18},[],{"issn":675,"title":676},{"VOID":13},{"VOID":15},[],[],[],{"impactFactor":19,"impactFactorByYear":681,"i10Index":19,"i10IndexLast5Year":19,"totalPublication":26,"totalPublicationByYear":682,"totalCitation":30,"totalCitationByYear":683,"totalCitationPerPublication":32,"totalCitationPerPublicationByYear":684,"hindexLast5Year":28,"hindex":28},{},{"2015":28,"2016":28,"2018":29,"2019":28,"2021":29,"2022":28,"2023":30},{"2015":28,"2018":28},{"2015":28,"2018":34},{"issue":686,"pages":688},{"VOID":687},"87",{"VOID":689},"12-20",{"total":19,"publishYear":691,"statisticByYear":692},2019,{},"2019-04-01","2026-07-20T15:05:25.774+00:00",[],[697,699,701,703,705,707,709,711,713,715,717,719,721,723,725,727,729,731,733,735,737],{"id":18,"text":698,"url":18,"identifiers":18},"1. Baumgartner H, Bonhoeffer P, De Groot NM, et al. ESC Guidelines for the management of grown-up congenital heart disease version 2010. Eur Heart J. 2010 Dec;31(23):2915-57.",{"id":18,"text":700,"url":18,"identifiers":18},"2. Torres-Cosme, Rolón, Aguinaga M, et al. Mortality from Congenital Heart Disease in Mexico: A Problem on the Rise. PLoS One. 2016 Mar 3;11(3):e0150422.",{"id":18,"text":702,"url":18,"identifiers":18},"3. Naidu P, Grigg L, Zentner D. Mortality in adults with congenital heart disease. Int J Cardiol. 2017 Oct 15;245:125-130.",{"id":18,"text":704,"url":18,"identifiers":18},"4. Amanda L.H, Lorna Swan. Quality of life in adults living with congenital heart disease: beyond morbidity and mortality. J Thorac Dis. 2016 Dec;8(12):E1632-E1636.",{"id":18,"text":706,"url":18,"identifiers":18},"5. Catherine M, Mohammed A.A, Gonzalo G.G. Chronic kidney disease in congenital heart disease patients: a narrative review of evidence. Can J Kidney Health Dis. 2015 Aug 11;2:27.",{"id":18,"text":708,"url":18,"identifiers":18},"6. Agras P.I, Derbent M, Ozcay F, et al. Effect of congenital heart disease on renal function in childhood. Nephron Physiol. 2005;99(1):p10-5.",{"id":18,"text":710,"url":18,"identifiers":18},"7. Krull F, Ehrich J.H, Wurster U, et al. Renal involvement in patients with congenital cyanotic heart disease. Acta Paediatr Scand. 1991 Dec;80(12):1214-9.",{"id":18,"text":712,"url":18,"identifiers":18},"8. Ariane J.M, Andrew S. M, Raluca I, et al. Congenital Heart Disease in the General Population: Changing Prevalence and Age Distribution. Circulation. 2007 Jan 16;115(2):163-72. Epub 2007 Jan 8.",{"id":18,"text":714,"url":18,"identifiers":18},"9. P. Engelfriet, B.J.M. Mulder. Gender differences in adult congenital heart disease. Neth Heart J. 2009 Nov;17(11):414-7.",{"id":18,"text":716,"url":18,"identifiers":18},"10. Graham J.R, Gary D.W, Mor Barzel M.A. Estimates of Life Expectancy by Adolescents and Young Adults With Congenital Heart Disease. J Am Coll Cardiol. 2006 Jul 18;48(2):349-55. Epub 2006 Jun 22.",{"id":18,"text":718,"url":18,"identifiers":18},"11. Paniagua R, Ramos A, Fabian R, et al. Chronic kidney disease and dialysis in Mexico. Perit Dial Int. 2007 Jul-Aug;27(4):405-9.",{"id":18,"text":720,"url":18,"identifiers":18},"12. Cobo G, Hecking M, Port FK, et al. Sex and gender differences in chronic kidney disease: progression to end-stage renal disease and haemodialysis. Clin Sci (Lond). 2016 Jul 1;130(14):1147-63.",{"id":18,"text":722,"url":18,"identifiers":18},"13. Davies D.F, Shock NW. Age changes in glomerular filtration rate, effective renal plasma flow and tubular excretory capacity in adult males. J Clin Invest. 1950 May;29(5):496-507.",{"id":18,"text":724,"url":18,"identifiers":18},"14. Collins A.J, Foley R.N, Chavers B, et al. United States Renal Data System 2011 Annual Data Report: Atlas of chronic kidney disease & endstage renal disease in the United States. Am J Kidney Dis. 2012 Jan;59(1 Suppl 1):A7, e1-420.",{"id":18,"text":726,"url":18,"identifiers":18},"15. N.P. Nickel, O’Leary, E.L Brittain, et al. Kidney dysfunction in patients with pulmonary arterial hypertension. Pulm Circ. 2017 Mar 13;7(1):38-54.",{"id":18,"text":728,"url":18,"identifiers":18},"16. Chan Soon Park, Jin Joo Park, Young Oh, et al. Relation of Renal Function with Left Ventricular Systolic Function and NT-proBNP Level and Its Prognostic Implication in Heart Failure with Preserved versus Reduced Ejection Fraction: an analysis from the Korean Heart Failure (KorHF) Registry. Korean Circ J. 2017 Sep;47(5):727-741.",{"id":18,"text":730,"url":18,"identifiers":18},"17. Franczyk B, Gluba B.A, Ciałkowska-Rysz, et al. The Problem of Atrial Fibrillation in Patients with Chronic Kidney Disease. Curr Vasc Pharmacol. 2016;14(3):260-5.",{"id":18,"text":732,"url":18,"identifiers":18},"18. Donald S.S, Dov Wexler, Adrian Iaina, et al. The role of anemia in the progression of congestive heart failure. Is there a place for erythropoietin and intravenous iron? J Nephrol. 2004 Nov-Dec;17(6):749-61.",{"id":18,"text":734,"url":18,"identifiers":18},"19. Yosuke Hirakawa, Tetsuhiro Tanaka, Masaomi Nangaku. Renal Hypoxia in CKD; Pathophysiology and Detecting Methods. Front Physiol. 2017 Feb 21;8:99.",{"id":18,"text":736,"url":18,"identifiers":18},"20. Truong L.D, Farhood A, Tasby J, et al. Experimental chronic renal ischemia: morphologic and immunologic studies. Kidney Int. 1992 Jun;41(6):1676-89.",{"id":18,"text":738,"url":18,"identifiers":18},"21. Perloff J.K, Latta H, Barsotti P. Pathogenesis of the glomerular abnormality in cyanotic congenital heart disease. Am J Cardiol. 2000 Dec 1;86(11):1198-204.",{"id":740,"createTime":741,"updateTime":742,"relativeEntities":743,"slug":744,"properties":745,"entityType":59,"verifyStatus":60,"verifyTime":756,"verifyNote":61,"languages":18,"translateLanguages":18,"viewCount":19,"primaryUrl":757,"fullTextUrl":758,"authors":759,"publicationType":151,"publisherRelationship":802,"citationCount":19,"citationInfo":821,"publishDate":824,"publishYear":822,"citationAnalyzeStatus":17,"lastCitationAnalyze":742,"indexDatabases":825,"openAccess":18,"references":18,"isForceReanalyzing":213},"f6808488-a97a-43c5-981a-13ba7811f661","2024-01-26T02:35:14.327+00:00","2026-07-15T12:48:59.221+00:00",[],"%C4%90%C3%A1nh-gi%C3%A1-t%C3%ACnh-tr%E1%BA%A1ng-suy-tim-%E1%BB%9F-b%E1%BB%87nh-nh%C3%A2n-nh%E1%BB%93i-m%C3%A1u-c%C6%A1-tim-c%E1%BA%A5p-ST-ch%C3%AAnh-l%C3%AAn-tr%C6%B0%C6%A1-c-v%C3%A0-sau-can-thi%E1%BB%87p-%C4%91%E1%BB%99ng-m%E1%BA%A1ch-v%C3%A0nh",{"abstract":746,"title":748,"gsPaper":750,"references":752,"doi":754},{"VI":747},"Mục tiêu: Khảo sát sự biến đổi hình thái và chức năng thất trái của bệnh nhân nhồi máu cơ tim cấp ST chênh lên trước và sau can thiệp tại thời điểm 48 giờ và 3 tháng bằng siêu âm tim.\r\nĐối tượng nghiên cứu: Trong thời gian từ tháng 02\u002F2020 đến 09\u002F2020 chúng tôi đã tiến hành nghiên cứu trên 97 bệnh nhân bệnh nhồi máu cơ tim cấp ST chênh lên được can thiệp động mạch vành qua da.\r\nPhương pháp nghiên cứu: Nghiên cứu tiến cứu quan sát.\r\nKết quả: Khối lượng cơ thất trái giảm từ 195,2 ± 65,8 gr xuống 170,2 ± 51,1 gr, thể tích thất trái cuối tâm trương giảm từ 105,2 ± 37,4 mm xuống 95,5 ± 41,3 mm, thể tích thất trái cuối tâm thu giảm từ 57,3± 45,2 mm xuống 49,8 ± 50,3 mm. Chức năng tâm thu thất trái (EF) sau 3 tháng can thiệp động mạch vành qua da của nhóm EF ≤ 45 % tăng lên đáng kể từ 39,3 ± 11,2 % lên 45,85 ± 7,56 %, (p<0,05),ngược lại nhóm EF > 45 % cũng có sự biến đổi từ 57,7 ± 14,4% lên 60,1 ± 13,3 %, (p>0,05).\r\nKết luận: Sau can thiệp động mạch vành qua da ở thời điểm 3 tháng, khối lượng cơ thất trái, thể tích thất trái cuối tâm thu và cuối tâm trương có sự thay đổi đáng kể. Chức năng tâm thu thất trái (EF) sau 3 tháng can thiệp động mạch vành qua da nhóm EF ≤ 45 % tăng lên có ý nghĩa thống kê.\r\nTừ khóa: Can thiệp động mạch vành qua da, siêu âm tim, chức năng thất trái, khối lượng cơ thất trái, thể tích cuối tâm trương.",{"VI":749},"Đánh giá tình trạng suy tim ở bệnh nhân nhồi máu cơ tim cấp ST chênh lên trước và sau can thiệp động mạch vành",{"VOID":751},"1763498364152536567",{"VOID":753},"TIẾNG VIỆT\n1. TrươngQuangBình,(2011), “Vai trò của can thiệp động mạch vành qua da trong điều trị bệnh động mạch vành”, Canthiệpđộngmạchvànhtrongthựchànhlâmsàng,NxbYhọc,TPHồChíMinh, pp. 11-31.\n2. TrươngQuangBình,(2011), \"Lịch sử về bệnh động mạch vành và về điều trị can thiệp động mạch vành\", Canthiệpđộngmạchvànhtrongthựchànhlâmsàng,NxbYhọc,TPHồChíMinh, tr. 1-10.\n3. NguyễnThịKimChung,(2004), “Tình hình nhồi máu cơ tim tại bệnh viện Đà Nẵng”, Tạp chí Tim mạch học Việt Nam, 37, tr. 38-39.\n4. Trương Đức Công, Vũ Đình Hùng, (2011), “Nghiên cứu biến đổi hình thái thất trái trên siêu âm ở bệnh nhân đau thắt ngực”, Tạp chí y học TP. Hồ Chí Minh, tập 15, phụ bản số 1, tr. 130-134.\n5. TrầnVănDương,PhạmGiaKhải,,(2000), “Kỹ thuật chụp động mạch vành chọn lọc: Một số kinh nghiệm qua 152 bệnh nhân tim mạch được chụp động mạch vành tại Viện Tim mạch Việt Nam”, Tạp chí tim mạch học, 21, tr. 19-72.\n6. Huỳnh Văn Minh, (2008), “Chụp động mạch vành”, Giáo trình sau đại học tim mạch học, Nxb Đại học Huế, Huế, pp. 320-331.\n7. HuỳnhVănMinh,(2008), “Suy mạch vành”, Giáotrìnhsauđạihọc Tim mạch học, Nxb Đại học Huế, Huế,, tr. 118-128.\n8. ThânHàNgọcThêvàcs,(2012), “Đánh giá hiệu quả của can thiệp động mạch vành qua da dựa trên cộng hưởng từ tim mạch”, Y học TP Hồ Chí Minh, tập 16, phụ bản của số 1, tr. 125-134.\n9. NguyễnQuangTuấn,(2005), “Nghiên cứu hiệu quả của phương pháp can thiệp động mạch vành qua da trong điều trị nhồi máu cơ tim cấp”, Luận án Tiến sỹ Y họcTrường Đại học Y Hà Nội, tr. 80 - 90.\nTIẾNG ANH\n10. Connolly HM, (2007), Braunwald’s Heart Disease, A Textbook of Cardiovascular Medicine. 8th ed, pp. 227-326.\n11. Hammermeister K E, DeRouen T A, Dodge H T, (1979), “Variables predictive of survival in patients with coronary disease. Selection by univariate and multivariate analyses from the clinical, electrocardiographic, exercise, arteriographic, and quantitative angiographic evaluations”, Circulation, 59 (3), pp. 421-430.\n12. LaneGEHDJ,(2007), Primarypercutaneouscoronaryinterventioninthemanagementofacutemyocardial infarction,Braunwald’s Heart Disease: A Textbook of Cardiovascular Medicine. 8th ed, pp. 1301-11.\n13. LaroseE,Rodés-CabauJ,PibarotP,RinfretS,etal,(2010), “Predicting late myocardial recovery and outcomes in the early hours of ST-segment elevation myocardial infarction traditional measures compared with microvascular obstruction, salvaged myocardium, and necrosis characteristics by cardiovascular magnetic resonance”, J Am Coll Cardiol, 55 (22), pp. 2459-2469.\n14. Talantbek Batyraliev, (2012), “Biodegradable Stents: Expected Revolution Ininterventional Cardiology”, Türk Girişimsel Kard Der 16, pp. 103-107.",{"VOID":755},"10.58354\u002Fjvc.97.2021.126","2025-01-09T03:27:02.582+00:00","https:\u002F\u002Fjvc.vnha.org.vn\u002Ftmh\u002Farticle\u002Fview\u002F126","https:\u002F\u002Fjvc.vnha.org.vn\u002Ftmh\u002Farticle\u002Fdownload\u002F126\u002F148",[760,776,789],{"id":761,"sortIndex":19,"researcher":18,"roles":762,"affiliations":764,"properties":773},"fe18aac3-4881-4411-8650-ba76d90dbf20",[763],"AUTHOR",[765],{"id":766,"sortIndex":19,"affiliation":767,"properties":18},"61ca5669-9bb8-4d49-884b-d7322e5b1156",{"id":766,"createTime":18,"updateTime":18,"relativeEntities":768,"slug":18,"properties":769,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":772,"statistic":18},[],{"title":770},{"VI":771},"Bệnh viện Trung Ương Huế",[],{"title":774},{"VI":775},"Hồ Anh Bình",{"id":777,"sortIndex":28,"researcher":18,"roles":778,"affiliations":779,"properties":786},"2134b75f-50b2-4fcc-a904-64edb3036e39",[763],[780],{"id":766,"sortIndex":19,"affiliation":781,"properties":18},{"id":766,"createTime":18,"updateTime":18,"relativeEntities":782,"slug":18,"properties":783,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":785,"statistic":18},[],{"title":784},{"VI":771},[],{"title":787},{"VI":788},"Nguyễn Thị Bích Ngọc",{"id":790,"sortIndex":30,"researcher":18,"roles":791,"affiliations":792,"properties":799},"b0542b5b-3318-457c-8a31-89cb508ccd4d",[763],[793],{"id":766,"sortIndex":19,"affiliation":794,"properties":18},{"id":766,"createTime":18,"updateTime":18,"relativeEntities":795,"slug":18,"properties":796,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":798,"statistic":18},[],{"title":797},{"VI":771},[],{"title":800},{"VI":801},"Lê Văn Duy",{"url":757,"publisher":803,"properties":816},{"id":6,"createTime":7,"updateTime":8,"relativeEntities":804,"slug":10,"properties":805,"entityType":16,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":19,"subjectFields":808,"manageAffiliations":809,"indexDatabases":810,"url":23,"thumbnailPath":18,"statistic":811,"gsStatistic":18,"type":35,"analyzePriority":18},[],{"issn":806,"title":807},{"VOID":13},{"VOID":15},[],[],[],{"impactFactor":19,"impactFactorByYear":812,"i10Index":19,"i10IndexLast5Year":19,"totalPublication":26,"totalPublicationByYear":813,"totalCitation":30,"totalCitationByYear":814,"totalCitationPerPublication":32,"totalCitationPerPublicationByYear":815,"hindexLast5Year":28,"hindex":28},{},{"2015":28,"2016":28,"2018":29,"2019":28,"2021":29,"2022":28,"2023":30},{"2015":28,"2018":28},{"2015":28,"2018":34},{"issue":817,"pages":819},{"VOID":818},"97",{"VOID":820},"88-95",{"total":19,"publishYear":822,"statisticByYear":823},2021,{},"2021-01-01",[],{"id":827,"createTime":828,"updateTime":829,"relativeEntities":830,"slug":831,"properties":832,"entityType":59,"verifyStatus":60,"verifyTime":841,"verifyNote":61,"languages":18,"translateLanguages":18,"viewCount":19,"primaryUrl":842,"fullTextUrl":843,"authors":844,"publicationType":151,"publisherRelationship":893,"citationCount":19,"citationInfo":912,"publishDate":915,"publishYear":913,"citationAnalyzeStatus":17,"lastCitationAnalyze":829,"indexDatabases":916,"openAccess":18,"references":18,"isForceReanalyzing":213},"08216f12-3795-4f1a-99f3-609a5bae95e2","2024-02-20T18:46:09.177+00:00","2026-07-12T15:00:53.492+00:00",[],"K%E1%BA%BFt-qu%E1%BA%A3-t%E1%BB%A9c-th%E1%BB%9Di-v%C3%A0-trung-h%E1%BA%A1n-c%E1%BB%A7a-ph%C6%B0%C6%A1ng-ph%C3%A1p-b%C3%ADt-th%C3%B4ng-li%C3%AAn-th%E1%BA%A5t-kh%C3%B4ng-c%C3%B3-g%E1%BB%9D-van-%C4%91%E1%BB%99ng-m%E1%BA%A1ch-ch%E1%BB%A7-b%E1%BA%B1ng-d%E1%BB%A5ng-c%E1%BB%A5-ADOII-qua-%C4%91%C6%B0%E1%BB%9Dng-%E1%BB%91ng-th%C3%B4ng",{"abstract":833,"title":835,"gsPaper":837,"references":839},{"VI":834},"Mục tiêu: Mô tả kết quả trung hạn của phương pháp bít thông liên thất không có gờ van động mạch chủ bằng dụng cụ ADOII qua đường ống thông.\r\nĐối tượng và phương pháp nghiên cứu: nghiên cứu quan sát trên 31 bệnh nhân được chẩn đoán thông liên thất không có gờ van động mạch chủ (TLT phần quanh màng lan đến sát van động mạch chủ và thông liên thất dưới đại động mạch) đã được can thiệp bằng dụng cụ ADO II qua đường ống thông tại Viện Tim mạch Việt Nam từ tháng 4\u002F2015 đến tháng 4\u002F2021\r\nKết quả: có tổng số 31 bệnh nhân thông liên thất không có gờ van động mạch chủ (24 BN TLT phần quanh màng không có gờ van ĐMC; 27 BN TLT dưới hai đại động mạch), gồm 20 BN nam và 11 BN nữ. Tuổi trung bình 17,84 (2-46 tuổi), cân nặng trung bình 34,81 kg (10 – 60 kg) đường kính trung bình thông liên thất phía thất phải: 3,65\r\n± 1,15 mm; đường kính trung bình thông liên thất phía thất trái: 5,00 ± 1,77 mm được can thiệp bít lỗ thông bằng dụng cụ ADOII. Tỷ lệ thành công của thủ thuật là 96,77%. Sau can thiệp, áp lực động mạch phổi giảm có ý nghĩa thống kê so với trước can thiệp với p = 0,03. Thời gian theo dõi sau can thiệp trong nghiên cứu là 45,39 ± 31,81 tháng (6- 72 tháng). Tại thời điểm 6 tháng sau can thiệp, gần\r\n \r\ncan thiệp, sau 60 tháng, tỷ lệ bệnh nhân còn triệu chứng khó thở tức ngực chỉ chiếm gần 5% so với trước can thiệp. Tại thời điểm 6 tháng sau can thiệp, có 82,75% bệnh nhân hết shunt tồn lưu, tại thời điểm 60 tháng sau can thiệp, chỉ còn 11,11% bệnh nhân còn shunt tồn lưu nhỏ và rất nhỏ, không ảnh hưởng tới huyết động. Tỷ lệ hở van động mạch chủ, van nhĩ thất trước và sau can thiệp khác biệt không có ý nghĩa thống kê. Không có trường hợp nào ghi nhận bloc nhĩ thất, không có trường hợp nào ghi nhận các biến chứng trầm trọng: tràn dịch màng tim, viêm nội tâm mạc nhiễm khuẩn, hở van tim tiến triển. Không có trường hợp nào di lệch dụng cụ phải xử trí, không có trường hợp nào tử vong trong quá trình theo dõi.\r\nKết luận: Tỷ lệ thành công của phương pháp bít thông liên thất không có gờ van động mạch chủ bằng dụng cụ ADOII qua đường ống thông trong nghiên cứu đạt 96,77%. Tỷ lệ bít kín hoàn toàn đạt 82,75% sau can thiệp 6 tháng và 99,89% sau can thiệp 60 tháng. Không có biến chứng trầm trọng: hở van động mạch chủ, hở van nhĩ thất tiến triển, rối loạn nhịp tim trong thời gian theo dõi 45,39 ± 31,81 tháng (6-72 tháng).\r\nTừ khóa: thông liên thất, gờ van động mạch chủ, can thiệp qua đường ống thông.",{"VI":836},"Kết quả tức thời và trung hạn của phương pháp bít thông liên thất không có gờ van động mạch chủ bằng dụng cụ ADOII qua đường ống thông",{"VOID":838},"18034249183042264254",{"VOID":840},"1. T. Carol. Diseases of the Human Body. Philadelphia. Fifth. PA: F.A Davis Company; 2011.\n2. Van der Linde D, Konings EEM, Slager MA, et al. Birth prevalence of congenital heart disease worldwide: a systematic review and meta-analysis. J Am Coll Cardiol. 2011;58(21):2241-2247. doi:10.1016\u002Fj.jacc.2011.08.025.\n3. Phạm Nguyễn Vinh. Bệnh Học Tim Mạch Tập II. Nhà xuất bản Y học.; 2008.\n4. Zhou T, Luo Y, Zhou X. Transcatheter Closure vs Surgical Closure of Ventricular Septal Defect in China: A Meta-Analysis. Cross-Cult Commun. 2015;11(9):57-65. doi:10.3968\u002F%x\n5. Phạm Tuấn Việt. Đánh giá kết quả ngắn hạn và trung hạn can thiệp bít thông liên thất bằng dụng cụ qua đường ống thông ở người trưởng thành. Published online 2015.\n6. Nguyễn Lân Hiếu. Lâm Sàng Tim Bẩm Sinh. Nhà xuất bản Đại học Quốc Gia Hà Nội; 2021.\n7. Kanaan M, Ewert P, Berger F, Assa S, Schubert S. Follow-Up of Patients with Interventional Closure of Ventricular Septal Defects with Amplatzer Duct Occluder II. Pediatr Cardiol. 2015;36(2):379-385. doi:10.1007\u002F s00246-014-1017-0.\n8. Kuswiyanto RB, Rahayuningsih SE, Apandi PR, Hilmanto D, Bashari MH. Transcatheter closure of doubly committed subarterial ventricular septal defect: Early to one-year outcome. Int J Cardiol Congenit Heart Dis. 2021;2:100081. doi:10.1016\u002Fj.ijcchd.2021.100081.\n9. Masura J, Gao W, Gavora P, et al. Percutaneous Closure of Perimembranous Ventricular Septal Defects with the Eccentric Amplatzer Device: Multicenter Follow-up Study. Pediatr Cardiol. 2005;26(3):216-219. doi:10.1007\u002F s00246-005-1003-7.\n10. Koneti NR, Penumatsa RR, Kanchi V, Arramraj SK, S. J, Bhupathiraju S. Retrograde transcatheter closure of ventricular septal defects in children using the Amplatzer Duct Occluder II. Catheter Cardiovasc Interv. 2011;77(2):252-259. doi:10.1002\u002Fccd.22675.","2025-01-12T05:47:32.249+00:00","https:\u002F\u002Fjvc.vnha.org.vn\u002Ftmh\u002Farticle\u002Fview\u002F338","https:\u002F\u002Fjvc.vnha.org.vn\u002Ftmh\u002Farticle\u002Fdownload\u002F338\u002F332",[845,860,877],{"id":846,"sortIndex":19,"researcher":18,"roles":847,"affiliations":848,"properties":857},"0931339a-994d-44cd-9844-8032ec5a9920",[763],[849],{"id":850,"sortIndex":19,"affiliation":851,"properties":18},"f4021d3f-ef0f-432c-b4d7-5b6b5a229233",{"id":850,"createTime":18,"updateTime":18,"relativeEntities":852,"slug":18,"properties":853,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":856,"statistic":18},[],{"title":854},{"VI":855},"Bộ môn Tim mạch - Trường Đại học Y Hà Nội",[],{"title":858},{"VI":859},"Hoàng Kim Quân",{"id":861,"sortIndex":28,"researcher":18,"roles":862,"affiliations":863,"properties":874},"fa6af91a-a113-40d6-a2b4-862f56e6b042",[763],[864],{"id":254,"sortIndex":19,"affiliation":865,"properties":871},{"id":254,"createTime":18,"updateTime":18,"relativeEntities":866,"slug":18,"properties":867,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":262,"parentIds":870,"statistic":18},[],{"title":868,"country":869},{"EN":259,"VI":260},{"VOID":83},[244],{"title":872},{"VI":873},"Viện Tim mạch Việt Nam - Bệnh viện Bạch Mai",{"title":875},{"VI":876},"Nguyễn Văn Hiếu",{"id":878,"sortIndex":30,"researcher":18,"roles":879,"affiliations":880,"properties":890},"4a655cfe-f3df-4368-9133-67d24e570ddd",[763],[881],{"id":882,"sortIndex":19,"affiliation":883,"properties":18},"e79a8923-ee23-4b7e-b2ad-85e9030ed8f7",{"id":882,"createTime":18,"updateTime":18,"relativeEntities":884,"slug":18,"properties":885,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":888,"statistic":18},[],{"title":886},{"VI":887},"Bộ môn Tim mạch - Trường Đại học Y Hà Nội, Bệnh viện Đại học Y Hà Nội ",[72,889],"465dd74f-be4f-41fc-bbfb-79b8d504dbe7",{"title":891},{"VI":892},"Nguyễn Lân Hiếu",{"url":842,"publisher":894,"properties":907},{"id":6,"createTime":7,"updateTime":8,"relativeEntities":895,"slug":10,"properties":896,"entityType":16,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":19,"subjectFields":899,"manageAffiliations":900,"indexDatabases":901,"url":23,"thumbnailPath":18,"statistic":902,"gsStatistic":18,"type":35,"analyzePriority":18},[],{"issn":897,"title":898},{"VOID":13},{"VOID":15},[],[],[],{"impactFactor":19,"impactFactorByYear":903,"i10Index":19,"i10IndexLast5Year":19,"totalPublication":26,"totalPublicationByYear":904,"totalCitation":30,"totalCitationByYear":905,"totalCitationPerPublication":32,"totalCitationPerPublicationByYear":906,"hindexLast5Year":28,"hindex":28},{},{"2015":28,"2016":28,"2018":29,"2019":28,"2021":29,"2022":28,"2023":30},{"2015":28,"2018":28},{"2015":28,"2018":34},{"issue":908,"pages":910},{"VOID":909},"101",{"VOID":911},"62-68",{"total":19,"publishYear":913,"statisticByYear":914},2022,{},"2022-09-01",[],{"id":918,"createTime":919,"updateTime":920,"relativeEntities":921,"slug":922,"properties":923,"entityType":59,"verifyStatus":60,"verifyTime":919,"verifyNote":61,"languages":932,"translateLanguages":18,"viewCount":19,"primaryUrl":933,"fullTextUrl":934,"authors":935,"publicationType":151,"publisherRelationship":1017,"citationCount":19,"citationInfo":1036,"publishDate":824,"publishYear":822,"citationAnalyzeStatus":17,"lastCitationAnalyze":920,"indexDatabases":1038,"openAccess":18,"references":1039,"isForceReanalyzing":213},"aa60ea34-a638-4f44-8324-1347d0680878","2025-02-24T07:45:39.432+00:00","2026-06-22T09:33:33.154+00:00",[],"%C4%90a-h%C3%ACnh-gen-COX-1-kh%C3%A1ng-Aspirin-tr%C3%AAn-b%E1%BB%87nh-nh%C3%A2n-c%C3%B3-b%E1%BB%87nh-%C4%91%E1%BB%99ng-m%E1%BA%A1ch-v%C3%A0nh",{"abstract":924,"title":926,"gsPaper":928,"doi":930},{"VI":925},"Tổng quan: Bệnh động mạch vành là nguyên nhân gây tử vong hàng đầu trên thế giới. Kháng aspirin đã được báo cáo trong bệnh lý động mạch vành do ảnh hưởng bởi các đa hình gen Cyclooxygenase-1 (COX-1).\r\nMục tiêu: Xác định tỷ lệ một số đa hình gen COX-1 thường gặp ảnh hưởng đến chuyển hóa aspirin (C-1676T, C644A, G128A, C50T, C22T) và mối liên quan với độ ngưng tập tiểu cầu trên bệnh nhân có bệnh động mạch vành.\r\nPhương pháp: 54 bệnh nhân mắc bệnh động mạch vành điều trị bằng aspirin hàng ngày với liều từ 75-100mg tối thiểu trên 2 tuần được đưa vào nghiên cứu. Hoạt động của aspirin được đánh giá bằng xét nghiệm đo độ ngưng tập tiểu cầu (NTTC) qua chất kết tập ADP.\r\nKết quả: Tần suất của các đa hình C-1676T, C644A, G128A, C22T lần lượt là 81,48%, 14,81%, 3,7% và 3,7%. Đa hình C50T không được tìm thấy. Bệnh nhân mang đa hình C-1676T cho thấy độ ngưng tập tiểu cầu cao hơn với bệnh nhân không mang đi hình gen COX-1 với p= 0,02.\r\nKết luận: Đa hình C-1676T xuất hiện nhiều nhất chiếm 81,48% trong nhóm bệnh nhân có bệnh động mạch vành. Sự xuất hiện của đa hình C-1676T chứng minh làm giảm đáp ứng của aspirin bằng xét nghiệm đo độ ngưng tập tiểu cầu.\r\nTừkhóa: Aspirin, độ NTTC,đa hình gen COX-1.",{"VI":927},"Đa hình gen COX-1 kháng Aspirin trên bệnh nhân có bệnh động mạch 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Geneva,Switzerlan(2012), CardiovascularDisease:GlobalAtlason Cardiovascular Disease Prevention and Control, World Health Organization.",{"id":18,"text":1043,"url":18,"identifiers":18},"2. BhattD.L.,FoxK.A.A.,HackeW.,etal.(2006). Clopidogrel and Aspirin versus Aspirin Alone for the Prevention of Atherothrombotic Events. N Engl J Med, 354(16), 1706–1717.",{"id":18,"text":1045,"url":18,"identifiers":18},"3. KrasopoulosG.,BristerS.J.,BeattieW.S.,etal.(2008). Aspirin “resistance” and risk of cardiovascular morbidity: systematic review and meta-analysis. BMJ, 336(7637), 195–198.",{"id":18,"text":1047,"url":18,"identifiers":18},"4. Lý Tuấn Khải, Quách Hữu Trung và Vũ Điện Biên (2011). Nghiên cứu kháng aspirin ở bệnh nhân bệnh mạch vành dùng aspirin dài ngày. Nghiên cứu Y Học, 15, 358–364.",{"id":18,"text":1049,"url":18,"identifiers":18},"5. YokoyamaC.andTanabe T. (1989). Cloning of human gene encoding prostaglandin endoperoxide synthase and primary structure of the enzyme. Biochem Biophys Res Commun, 165(2), 888–894.",{"id":18,"text":1051,"url":18,"identifiers":18},"6. EikelboomJ.W.,HirshJ.,SpencerF.A.,etal.(2012). Antiplatelet drugs: Antithrombotic Therapy and Prevention of Thrombosis, 9th ed: American College of Chest Physicians Evidence-Based Clinical Practice Guidelines. Chest, 141(2 Suppl), e89S-e119S.",{"id":18,"text":1053,"url":18,"identifiers":18},"7. Arisawa T., Tahara T., Shibata T., et al. (2007). Association between genetic polymorphisms in thecyclooxygenase-1 gene promoter and peptic ulcers in Japan. Int J Mol Med.",{"id":18,"text":1055,"url":18,"identifiers":18},"8. Fan L., Cao J., Liu L., et al. (2013). Frequency, Risk Factors, Prognosis, and Genetic Polymorphism of the Cyclooxygenase-1 Gene for Aspirin Resistance in Elderly Chinese Patients with Cardiovascular Disease. Gerontology, 59(2), 122–131.",{"id":18,"text":1057,"url":18,"identifiers":18},"9. ZhaoL.,FangJ.,ZhouM.,etal.(2019). Interaction between COX-1 and COX-2 increases susceptibility to ischemic stroke in a Chinese population. BMC Neurol, 19.",{"id":18,"text":1059,"url":18,"identifiers":18},"10. XueM.,YangX.,YangL.,etal.(2017). rs5911 and rs3842788 Genetic Polymorphism, Blood Stasis Syndrome, and Plasma TXB2 and hs-CRP Levels Are Associated with Aspirin Resistance in Chinese Chronic Stable Angina Patients. Evid-Based Complement Altern Med ECAM, 2017, 9037094.",{"id":18,"text":1061,"url":18,"identifiers":18},"11. Yi X., Cheng W., Lin J., et al. (2016). Interaction between COX-1 and COX-2 Variants Associated with Aspirin Resistance in Chinese Stroke Patients. J Stroke Cerebrovasc Dis Off J Natl Stroke Assoc, 25(9), 2136–2144.",{"id":18,"text":1063,"url":18,"identifiers":18},"12. Wong K.-C., Zhang J., Yan S., et al. (2013). DNA Sequencing Technologies: Sequencing Data Protocols and Bioinformatics Tools. ACM Comput Surv, 52, 1–30.",{"id":18,"text":1065,"url":18,"identifiers":18},"13. NguyễnNgọcMinhvàNguyễnĐìnhÁi. Cầmmáu-Đôngmáuứngdụngvàkỹthuậtvàứngdụngtrong chẩn đoán lâm sàng, Nhà xuất bản Y Học.",{"id":18,"text":1067,"url":18,"identifiers":18},"14. NguyễnThịNữ,CungThịTývàĐỗTrungPhấn(1997). Chỉ số ngưng tập tiểu cầu ở người trưởng thành Việt Nam bình thường. Y học Việt Nam, 66–68.",{"id":18,"text":1069,"url":18,"identifiers":18},"15. Rocca B. and Petrucci G. (2012). Variability in the Responsiveness to Low-Dose Aspirin: Pharmacological and Disease-Related Mechanisms. Thrombosis, 2012.",{"id":18,"text":1071,"url":18,"identifiers":18},"16. KuliczkowskiW.,WitkowskiA.,PolonskiL.,etal.(2009). Interindividual variability in the response to oral antiplatelet drugs: a position paper of the Working Group on antiplatelet drugs resistance appointed by the Section of Cardiovascular Interventions of the Polish Cardiac Society, endorsed by the Working Group on Thrombosis of the European Society of Cardiology. Eur Heart J, 30(4), 426–435.",{"id":18,"text":1073,"url":18,"identifiers":18},"17. CYCLOOXYGENASE-1 GENE - SNP - NCBI.",{"id":18,"text":1075,"url":18,"identifiers":18},"18. LeeC.R.,BottoneF.G.,KrahnJ.M.,etal. (2007). Identification and functional characterization of polymorphisms in human cyclooxygenase-1 (PTGS1). Pharmacogenet Genomics, 17(2), 145–160.",{"id":18,"text":1077,"url":18,"identifiers":18},"19. Maree A.O., Curtin R.J., Chubb A., et al. (2005). Cyclooxygenase-1 haplotype modulates platelet response to aspirin. J Thromb Haemost, 3(10), 2340–2345.",{"id":18,"text":1079,"url":18,"identifiers":18},"v20. WangZ.,ChenY.,HuS.,etal.(2017). A Meta-analysis of the Association of COX-1 Gene rs3842788 and rs1330344 Polymorphism with Aspirin Resistance in Chinese. J Med Diagn Methods, 06(04).",{"id":18,"text":1081,"url":18,"identifiers":18},"21. Yi X., Wang C., Zhou Q., et al. (2017). Interaction among COX-2, P2Y1 and GPIIIa gene variants is associated with aspirin resistance and early neurological deterioration in Chinese stroke patients. 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Wilkerson Group Survey, 1998. Congestive heart failure worldwilde market, clinical status and product development. New medcine, Inc. 1997: 1-40.",{"id":18,"text":1276,"url":18,"identifiers":18},"2. Phạm Việt Tuân và cộng sự. Luận văn tốt nghiệp cao học 2009.",{"id":18,"text":1278,"url":18,"identifiers":18},"3. Boriani G, Muller CP, Seidl KH et al (2006); Resynchronization for the HemodYnamic Treatment for Heart Failure Management II Investigators. Randomized comparison of simultaneous biventricular stimulation versus optimized interventricular delay in cardiac resynchronization therapy. The Resynchronization for the HemodYnamic Treatment for Hear t Fai lure Management I I implantable cardioverter defi brillator (RHYTHM II ICD) study. Am Heart J 2006; 151: 1050–1058.",{"id":18,"text":1280,"url":18,"identifiers":18},"4. Baker JH, McKenzie J, Beau S et al (2007). Acute evaluation of programmer-guided AV\u002FPV and VV delay optimization comparing an IEGM method and echocardiogram for cardiac resynchronization therapy in heart failure patients and dual-chamber ICD implants. J Cardiovasc Electrophysiol 2007; 18: 185–191.",{"id":18,"text":1282,"url":18,"identifiers":18},"5. Stanton T, Hawkins NM, Hogg KJ, Goodfield NE, Petrie MC, McMurray JJ. How should we optimize cardiac resynchronization therapy? Eur Heart J. 2008 Oct;29(20):2458-72.",{"id":18,"text":1284,"url":18,"identifiers":18},"6. Ypenburg C, Van De Veire N, Westenberg JJ, Bleeker GB, Marsan NA, Henneman MM, Van Der Wall EE, Schalij MJ, Abraham TP, Barold SS, Bax JJ.Noninvasive imaging in cardiac resynchronization therapy--Part 2: Follow-up and optimization of settings. Pacing Clin Electrophysiol. 2008 Dec;31(12):1628-39.",{"id":1286,"createTime":1287,"updateTime":1288,"relativeEntities":1289,"slug":1290,"properties":1291,"entityType":59,"verifyStatus":60,"verifyTime":1300,"verifyNote":61,"languages":18,"translateLanguages":18,"viewCount":19,"primaryUrl":1301,"fullTextUrl":18,"authors":1302,"publicationType":151,"publisherRelationship":1437,"citationCount":19,"citationInfo":1456,"publishDate":301,"publishYear":299,"citationAnalyzeStatus":17,"lastCitationAnalyze":1458,"indexDatabases":1459,"openAccess":18,"references":18,"isForceReanalyzing":213},"a7073a64-1272-4cbd-a5ff-34f5ba90356b","2024-01-22T18:38:33.333+00:00","2026-05-12T16:44:08.269+00:00",[],"Can-thi%E1%BB%87p-Tim-b%E1%BA%A9m-sinh-t%E1%BA%A1i-Vi%E1%BB%87n-Tim-m%E1%BA%A1ch-Vi%E1%BB%87t-Nam-B%E1%BB%87nh-vi%E1%BB%87n-B%E1%BA%A1ch-Mai-k%E1%BA%BFt-qu%E1%BA%A3-sau-2-n%C4%83m-nh%C3%ACn-l%E1%BA%A1i-2015-2016",{"abstract":1292,"title":1294,"gsPaper":1296,"references":1298},{"VI":1293},"Giới thiệu: Số lượng bệnh nhân mắc tim bẩm sinh được can thiệp bằng đường ống thông ngày càng tăng ở Việt Nam.\r\nMục tiêu: Nghiên cứu chúng tôi nhằm báo cáo kết quả, hiệu quả và biến chứng can thiệp bằng ống thông cho bệnh nhân tim bẩm sinh tại Viện Tim mạch Việt Nam 2015-2016.\r\nPhương pháp: Chúng tôi tuyển chọn 1265 bệnh nhân (tuổi trung bình: 25,8 ± 17.0; nhỏ nhất 6 tháng tuổi, cao nhất 76 tuổi) có bệnh tim bẩm sinh mà được thực hiện can thiệp qua da từ tháng 1\u002F2015 đến tháng 12\u002F2016. Toàn bộ dữ liệu lâm sàng được thu thập hồi cứu dựa trên dữ liệu bệnh viện và bệnh án bệnh nhân.\r\nKết quả: Nghiên cứu của chúng tôi ghi nhận gồm 1265 ca can thiệp TBS\u002F22.416 ca nhập viện nội trú (5,64%), trong đó chủ yếu là thông liên nhĩ (TLN), thông liên thất (TLT) và ống động mạch (ÔĐM), cụ thể như sau: 499 ca TLN (39,6%), 158 ca TLT (12,5%), 251 ca CODM (19.8%), 52 ca hẹp van động mạch phổi và 7 ca nong van ĐMC.\r\nThủ thuật can thiệp thành công trên 1194\u002F1265 ca (94,4%). Biến chứng sớm chính xảy ra ở 71 ca (5,6%), trong đó bao gồm 52 ca rối loạn nhịp, 10 ca đái máu, 1 ca ép tim do tràn máu màng ngoài tim cấp và 2 ca di lệch dụng cụ (1 TLT, 1 TLN). Biến chứng khác là shunt tồn lưu nhỏ ở 6 ca TLT, TLN. Thời gian theo dõi sau can thiệp là 3 ngày. Chúng tôi không ghi nhận tắc mạch não, nhồi máu cơ tim, và tử vong trong quá trình can thiệp cũng như thời gian theo dõi.\r\nKết luận: Phương pháp can thiệp bằng ống thông theo đường dưới da cho bệnh nhân tim bẩm sinh là hiệu quả và an toàn trong đại đa số các ca. Tỷ lệ biến chứng sớm sau can thiệp là 5,6%.\r\nTừ khóa: Bệnh tim bẩm sinh, biến chứng sớm sau can thiệp, can thiệp tim bẩm sinh.",{"VI":1295},"Can thiệp Tim bẩm sinh tại Viện Tim mạch Việt Nam - Bệnh viện Bạch Mai: kết quả sau 2 năm nhìn lại 2015-2016",{"VOID":1297},"3797842079972816911",{"VOID":1299},"1. Theo Vos, Ryan M. Barber, Brad Bell và các cộng sự., \"Global, regional, and national incidence, prevalence, and years lived with disability for 301 acute and chronic diseases and injuries in 188 countries, 1990–2013: a systematic analysis for the Global Burden of Disease Study 2013\", The Lancet, 386(9995), tr. 743-800.\n2. Ariane J. Marelli, Raluca Ionescu-Ittu, Andrew S. Mackie và các cộng sự. (2014), \"Lifetime Prevalence of Congenital Heart Disease in the General Population From 2000 to 2010\", Circulation, 130(9), tr. 749-756.\n3. Yumi Shiina, Tomohiko Toyoda, Yasutaka Kawasoe và các cộng sự., \"Prevalence of adult patients with congenital heart disease in Japan\", International Journal of Cardiology, 146(1), tr. 13-16.\n4. Nguyễn Lân Việt (2010 ), \"nghiên cứu mô hình bệnh tạt ở bệnh nhân điều trị nội trú tại Viện Tim Mạch Việt Nam trong thời gian 2003-2007\", Tạp chí Tim mạch học số 52.\n5. Phạm Mạnh Hùng: can thiệp tim bẩm sinh: làm gì? bao giờ? khi nào.\n6. Carianne L. Verheugt, Cuno S.P.M. Uiterwaal, Enno T. van der Velde và các cộng sự. (2008), \"Gender and Outcome in Adult Congenital Heart Disease\", Circulation, 118(1), tr. 26-32.\n7. Lê Thành Khánh Vân (2015), \"Điều trị bệnh tim bẩm sinh tại bệnh viện Chợ Rẫy \", Tạp chí y học thực hành.\n8. P. Engelfriet và B. Mulder (2009), \"Gender differences in adult congenital heart disease\", Neth Heart J, 17(11), tr. 414-7.\n9. Nguyễn Thị Nhung (2016): Nhận xét đặc điểm tim bẩm sinh người lớn tại Viện Tim mạch quốc gia- Luận văn tốt nghiệp BS đa khoa 2016.\n10. Nelson textbook of pediatrics 20th (Daniel Bernstein,), Elsevier.\n11. Trần Thị Mai Hồng (2012), \"Nghiên cứu sự thay đổi các chỉ số huyết học ở bệnh nhân tim bẩm sinh.\",\nLuận văn tốt nghiệp bác sĩ đa khoa.\n12. Lê Thị Kim Dung, \"Phân loại tim bẩm sinh điều trị tại khoa Nhi bệnh viện Đa khoa Thái Nguyên. \".\n13. Nguyễn Thị Thanh Hương (2010), \"Đặc điểm bệnh lý tim bẩm sinh ở trẻ sơ sinh tại Bệnh viện Nhi đồng 1. \", Tạp chí y học thành phố Hồ Chí Minh.\n14. Ths.Bs. Hoàng Thị Phú Bằng và cộng sự (2017): Can thiệp bằng ống thông ở bệnh nhân Tim bẩm sinh: nghiên cứu hồi cứu tại Việt Nam, báo cáo hội nghị can thiệp tim mạch toàn quốc 11\u002F2017 tại Cần 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