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Journal of Medicine and Pharmacy","Tạp chí Y Dược học Cần Thơ",{"EN":487,"VI":488},"\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">04\u002F10\u002F2015 Ministry of Information and Communications allowed Can Tho journal of medicine and pharmacy to operate (102 \u002FGP-BTTTT)\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">07\u002F16\u002F2015 Can Tho journal of medicine and pharmacy is internationally recognized: ISSN 2354-1210\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">In 2016, The journal has been included in the list of medical science journals by The State Council for professorship which is awarded a work score of 0-0.5 points for a published article.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Can Tho Journal of Medicine and Pharmacy welcome original works that haven’t been submitted or published in other medical journals. Posts must contain content related to one of the journal’s categories.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The content published\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The journal is divided into 3 categories:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Scientific research article: are valuable scientific works, which have been researched and accepted.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Overview of medicine, biology and pharmacy: serving the objective of continuing training in the fields of medicine, biology and pharmacy; to systematize classical and modern knowledge.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Update information on new knowledge about medicine, biology, pharmacy in the country and in the world.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Scope\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Publication and introduction of scientific research in the fields:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Medicine (internal medicine, surgery, pediatrics, obstetrics and gynecology, odonto-stomatology, laboratory, oncology, traditional medicine, nursing).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Biology (genetics, biotechnology).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Pharmacology (pharmaceutics, drug quality analysis-control, synthetic pharmaceutical chemistry, biochemistry, pharmacognosy, botany, clinical pharmacy).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- To enhance the quality of undergraduate, postgraduate education, scientifically researching and meet the necessary treatment in hospital.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Introducing the updated domestic and oversea information about science technology to promote scientific research and exchanging technology in local, other universities.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Exchanging pharmaceutical and medical information for social health developing in the Mekong Delta and Vietnam.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The object\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Postgraduate students, student of Can Tho University of Medicine and Pharmacy, scientists from schools, research institutes, hospitals, health centers, pharmaceutical companies of the Mekong Delta; other provinces and regions in Vietnam and other country.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Address\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Headquarters of Can Tho Journal of Medicine and Pharmacy, located Scientific Research and International Cooperation Office: 179 Nguyen Van Cu Street, An Khanh Ward, Ninh Kieu District, Can Tho City, Vietnam.\u003C\u002Fspan>\u003C\u002Fp>","\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Ngày 16\u002F7\u002F2015, Tạp chí Y Dược học Cần Thơ được cấp chỉ số quốc tế: ISSN 2354-1210.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 4\u002F2016, Tạp chí đã được Hội đồng Giáo sư ngành Y đưa vào danh sách các tạp chí khoa học Y học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Năm 2020 Tạp chí Y Dược học Cần Thơ đã được phê duyệt vào danh mục của các Hội đồng Giáo sư ngành Dược học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ ra 12 số\u002Fnăm, 180-200 trang\u002Fsố.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 12\u002F2022 Tạp chí Y Dược học Cần Thơ là thành viên của hệ thống Crossref và từ tháng 01\u002F2023 tạp chí thực hiện bình duyệt online kín 2 chiều nhằm tăng tính minh bạch, tin cậy của các công trình nghiên cứu khoa học và đảm bảo tốt nhất chất lượng khoa học của bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ, mục đích và phạm vi của tạp chí\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ và mục đích hoạt động của tạp chí: xuất bản nhằm mục đích phổ biến kết quả từ các đề tài nghiên cứu khoa học; giao lưu trao đổi khoa học, chia sẻ kinh nghiệm, học tập, đồng thời cập nhật thông tin khoa học mới trong các lĩnh vực y, sinh, dược học trong và ngoài nước.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phạm vi của tạp chí: Tạp chí xuất bản được chia thành 3 chuyên mục: (i) Bài báo nghiên cứu khoa học là kết quả công trình nghiên cứu khoa học có giá trị đã được triển khai nghiên cứu, (ii) Bài tổng quan y, sinh, dược học: phục vụ mục tiêu đào tạo liên tục trong lĩnh vực y, sinh, dược học; nhằm hệ thống hóa những kiến thức kinh điển và hiện đại; (iii) Thông tin cập nhật kiến thức mới về y, sinh, dược học trong nước và trên thế giới.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Chính sách truy cập mở\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ áp dụng chính sách truy cập mở đối với các bài báo đã xuất bản đến với độc giả, nhằm mở rộng cơ hội tiếp cận các kết quả nghiên cứu chất lượng cao và tăng cường trao đổi kiến thức. Tạp chí đăng tải trực tuyến (miễn phí) toàn văn các bài báo được công bố trên website của Tạp chí (https:\u002F\u002Ftapchi.ctump.edu.vn).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đạo đức xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ cam kết tuân thủ đạo đức xuất bản phù hợp với các hướng dẫn và tiêu chuẩn của the Committee on Publication Ethics (COPE), tuân thủ các nguyên tắc của COPE’s Core Practices, Best Practices Guidelines for Journal Editors và Guidelines on Good Publication Practices.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Bản thảo bài báo chỉ được chấp nhận khi được tác giả chịu trách nhiệm chính cam kết các nội dung sau: Các nội dung của bản thảo chưa được đăng tải toàn bộ hoặc một phần ở các tạp chí khác; Tất cả các tác giả đều có đóng góp một cách đáng kể vào quá trình nghiên cứu hoặc chuẩn bị bản thảo và cùng chịu trách nhiệm về các nội dung của bản thảo; Tuân thủ các biện pháp đảm bảo đạo đức nghiên cứu (ví dụ thỏa thuận đồng ý tham gia nghiên cứu).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Cam kết bảo mật\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí cam kết thực hiện và tuân thủ các quy định của luật và các văn bản hướng dẫn liên quan đến bảo mật thông tin cá nhân trên không gian mạng. Các thông tin mà người dùng (tác giả, độc giả, biên tập viên, người phản biện) nhập vào các biểu mẫu trên Hệ thống Quản lý xuất bản trực tuyến của tạp chí chỉ được sử dụng vào các mục đích đã được tuyên bố rõ ràng và sẽ không được cung cấp cho bất kỳ bên thứ ba nào khác, hay dùng vào bất kỳ mục đích nào khác.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phí gửi bài\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng bài: 1.000.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng nhanh: 1.500.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với tác giả là cán bộ viên chức thuộc Trường Đại học Y Dược Cần Thơ thì được hỗ trợ 50% lệ phí gửi đăng bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với sinh viên thực hiện đề tài nghiên cứu khoa học cấp trường được hỗ trợ 100% lệ phí đăng bài ( Tác giả gửi đính kèm “ Quyết định về việc giao tổ chức thực hiện đề tài nghiên cứu khoa học cấp Trường của sinh viên”).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Hình thức nộp lệ phí:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Tiền mặt:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Nộp trực tiếp tại Phòng Tài chính - Kế toán, Trường Đại học Y Dược Cần Thơ, số 179 Nguyễn Văn Cừ, P. An Khánh, Q. Ninh Kiều, thành phố Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Chuyển khoản:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tên Tài khoản: Trường ĐHYD Cần Thơ, Số TK: 0111000115668, tại ngân hàng Vietcombank chi nhánh Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Thời gian: Áp dụng từ ngày 01\u002F02\u002F2023.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">* Phí gửi bài không được hoàn trả khi bài viết bị từ chối hoặc tác giả xin rút bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Quy trình phản biện bài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ thực hiện quy trình phản biện kín hai chiều nghiêm ngặt. Danh tính của những người phản biện không được tiết lộ cho các tác giả và ngược lại. Quy trình thẩm định bài báo đăng gồm các bước sau:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tiếp nhận bản thảo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tác giả liên hệ gửi bản thảo đến Tạp chí qua hệ thống trực tuyến tại website: https:\u002F\u002Ftapchi.ctump.edu.vn. Hướng dẫn về cách đăng ký, gửi bài và chuẩn bị bản thảo được cung cấp trên website của Tạp chí.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sàng lọc sơ bộ\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sau khi Tòa soạn nhận được bài báo của tác giả, Ban Thư ký sẽ tiến hành kiểm tra sơ bộ bài báo (các yêu cầu về nội dung và hình thức). Những bài báo không đúng quy cách hoặc có nội dung không phù hợp hoặc vi phạm bản quyền sẽ bị từ chối (Ban Thư ký thông báo phản hồi đến tác giả trong vòng 1 tuần). Những bài báo đủ điều kiện, được Ban Thư ký tòa soạn chuyển đến Ban Biên tập có cùng chuyên môn với nội dung bài báo để đề xuất người phản biện. Thời gian kể từ khi Ban Biên tập nhận bài báo đến khi đề xuất người phản biện bài báo chậm nhất là 5 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Vòng phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký gửi bài và yêu cầu phản biện đến 02 phản biện độc lập.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Các phản biện gởi nhận xét cho Ban Thư ký. Thời gian từ khi gửi bài cho phản biện đến khi nhận ý kiến của phản biện tối đa là 20 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xử ký kết quả phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Nếu ý kiến đồng ý cho đăng và không cần chỉnh sửa, Ban Thư ký tiếp tục đăng bài theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Nếu ý kiến đồng ý đăng và cần chỉnh sửa, Ban Thư ký sẽ thông tin đến tác giả chỉnh sửa theo yêu cầu của người phản biện. Thời gian chỉnh sửa và gửi lại kéo dài không quá 2 tuần, từ khi tác giả bài báo nhận được thông tin (Quá trình này có thể lặp lại tối đa 2 lần\u002F1 bài báo). Khi có sự thống nhất, đồng ý của người phản biện; bài báo được tiếp tục đăng theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Những bài báo có chất lượng không đạt yêu cầu, cả 2 phản biện không đồng ý cho đăng sẽ bị Tòa soạn từ chối đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký tổng hợp các bản thảo đã được tác giả hoàn thiện sau thẩm định trình Ban Biên tập xem xét, Tổng Biên tập phê duyệt, quyết định bài đăng theo các tiêu chí: sự phù hợp nội dung với tôn chỉ và mục đích, thể loại bài viết (ưu tiên các bài có bài có nghiên cứu chuyên sâu, hàm lượng khoa học cao), đóng góp mới bài báo, bài báo được ưu tiên đăng trong số gần nhất của Tạp chí theo thứ tự: tính thời sự, chất lượng bài báo và thời gian gửi bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Ban Biên tập và Ban Thư ký biên tập bản thảo, chế bản, đọc rà soát lỗi. Thời gian hoàn thành từ 10-15 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Ban Thư ký có trách nhiệm thông báo cho tác giả bài báo (bằng e-mail) về tình hình phê duyệt bài báo, thời gian, số kỳ, tập xuất bản bài báo theo qui định.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">4. Danh sách bài báo theo số Tạp chí được in ấn và phát hành trong năm định kỳ được công bố chính thức trên website: https:\u002F\u002Ftapchi.ctump.edu.vn\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>",{"VOID":490},"wcQ1uqwAAAAJ","2023-05-30T08:17:21.868+00:00",[],[494],{"id":495,"createTime":28,"updateTime":28,"relativeEntities":496,"slug":28,"properties":497,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":507,"parentIds":508,"statistic":28},"6413896b-eca9-442b-a73f-182a58a0ce40",[],{"title":498,"address":501,"country":504,"abbreviation":505},{"EN":499,"VI":500},"Can Tho University of Medicine and Pharmacy","Trường Đại học Y Dược Cần Thơ",{"EN":502,"VI":503},"No 179, Nguyen Van Cu street, An Khanh ward, Ninh Kieu district, Can Tho city, Vietnam","Số 179, đường Nguyễn Văn Cừ, phường An Khánh, quận Ninh Kiều, thành phố Cần Thơ, Việt Nam",{"VOID":15},{"VOID":506},"ctump","http:\u002F\u002Fwww.ctump.edu.vn\u002F",[],[],"https:\u002F\u002Ftapchi.ctump.edu.vn\u002Findex.php\u002Fctump",{"impactFactor":32,"impactFactorByYear":512,"i10Index":32,"i10IndexLast5Year":32,"totalPublication":514,"totalPublicationByYear":515,"totalCitation":520,"totalCitationByYear":521,"totalCitationPerPublication":108,"totalCitationPerPublicationByYear":523,"hindexLast5Year":45,"hindex":45},{"2022":513,"2023":111,"2024":106},0.01,1556,{"2020":47,"2021":516,"2022":517,"2023":518,"2024":519,"2025":122},57,306,801,358,161,{"2021":146,"2022":280,"2023":522},99,{"2021":524,"2022":318,"2023":104},0.23,{"impactFactor":28,"impactFactorByYear":28,"i10Index":123,"i10IndexLast5Year":123,"totalPublication":526,"totalPublicationByYear":527,"totalCitation":526,"totalCitationByYear":528,"totalCitationPerPublication":40,"totalCitationPerPublicationByYear":531,"hindexLast5Year":49,"hindex":49},476,{"0":205,"2019":123,"2021":139,"2022":459,"2023":451,"2024":357,"2025":49,"2026":48},{"2021":42,"2022":123,"2023":161,"2024":529,"2025":360,"2026":530},136,83,{"2021":105,"2022":513,"2023":532,"2024":127,"2025":533,"2026":534},0.62,25.43,13.83,{"id":536,"createTime":537,"updateTime":382,"relativeEntities":538,"slug":539,"properties":540,"entityType":25,"verifyStatus":26,"verifyTime":28,"verifyNote":28,"languages":552,"translateLanguages":28,"viewCount":133,"subjectFields":553,"manageAffiliations":554,"indexDatabases":555,"url":556,"thumbnailPath":557,"statistic":558,"gsStatistic":594,"type":55,"analyzePriority":28},"6984a56a-db70-403b-9cc4-4013e1ceaffa","2023-05-09T06:47:40.346+00:00",[],"T%E1%BA%A1p%20ch%C3%AD%20Nghi%C3%AAn%20c%E1%BB%A9u%20n%C6%B0%E1%BB%9Bc%20ngo%C3%A0i",{"country":541,"issn":542,"title":544,"introduce":547,"gsId":550},{"VOID":15},{"VOID":543},"25252445",{"EN":545,"VI":546},"VNU Journal of Foreign Studies","Tạp chí Nghiên cứu nước ngoài",{"EN":548,"VI":549},"{\"ops\":[{\"insert\":\"\\n\\nThe \\n\"},{\"attributes\":{\"italic\":true},\"insert\":\"VNU Journal of Science\"},{\"insert\":\"\\n was established in 1985 for the publication of national and international research papers in all fields of natural sciences and technology, social sciences and humanities. 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K.: Der Blutzucker. InOppenheimers Handbuch der Biochemie, 2. Aufl., Erg. werk Bd. 2, S. 86, Jena: Gustav Fischer 1934.\nBietti, G.: Histologische Untersuchungen über Veränderungen am Auge nach parenteraler Einführung von Organextrakten. Weiterer Beitrag zur Kenntnis der Wirkungsweise kapillargiftiger Stoffe aufs Auge. Arch. Augenheilk.105, 603 (1932).\nFilatow, V. P., I. G. Jerschkowitsch u.V. E. Chewalow: Experimentelle Studien über die Wirkung der Muskelarbeit auf den Augendruck. Vestn. Oftalm.11, 161 (1937). Ref. Zbl. Ophthalm.40, 574 (1938).\nFreund, H.: Über die pharmakologischen Wirkungen des defibrinierten Blutes. Arch. exper. Path. u. Pharmakol.86, 266 (1920);88, 39 (1920).\nGreen: Clin. Sci.8, 65 (1949).Hallmann L.: Klinische Chemie und Mikroskopie. Stuttgart: Georg Thieme 1955.Hertel, E.: Experimentelle Untersuchungen über die Abhängigkeit des Augendruckes von der Blutbeschaffenheit. Graefes Arch.88, 197 (1914).\nHinsberg, K.: Beitrag inB. Flaschenträger u.E. Lehnartz, Physiologische Chemie. Bd. 2. Der Stoffwechsel. Erster Teil, Bandteil a, S. 324. Berlin: Springer 1954.\nIsaac, S., u.R. Siegel: Physiologie und Pathologie des intermediären Kohlehydratstoffwechsels. InBethe-Bergmanns Handbuch der pathologischen Physiologie, Bd. V, S. 469. Berlin: Springer 1928.\nJohnson, R. E., u.H. T. Edwards: Lactate and pyruvate in blood and urine after exercise. J. of Biol. Chem.118, 427 (1937).\nKlein, J. R. D.: The effects of administration of glucose and insulin on blood pyruvate and lactate in diabetes mellitus. J. of Biol. Chem..145, 35 (1942).\nLehmann, G.: Der Stoffwechsel bei schwerer Körperarbeit. In Handbuch der Biochemie, 2. Aufl., Erg.-werk Bd. III, S. 113–131 (1936).\nMargaria, R., eC. Talenti: Veranderungen desPh des Blutes nach Muskelarbeit verschiedener Art. Arch. di Fisiol.32, 165 (1933).\nMatakas, F.: Zur Methodik des Milchsäurenachweises im Gewebe und Blut. Biochem. Z.239, 417 (1931).\nMather, R. W., Ch. R. Shuman andS. I. Harrison: Influences of curare and eserine on ocular tension following electroshock. Amer. J. Ophthalm.37, 859 (1954).\nMohos, E., u.A. Scholtz: Blutmilchsäureuntersuchungen bei Basedow. Dtsch. Arch. klin. Med.171, 397 (1931).\nPassow, A.: Über die augendrucksenkende Wirkung körpereigener Substanzen. Arch. Augenheilk.104, 490 (1931).\nPerger, H.: Über die Resynthese der Milchsäure bei Kreislaufkranken. Klin. Wschr.1927 II, S. 1324.\nPeters, J. P., andD. van Slyke: Quant. Clin. Chemistry1, 208 (1946).\nPoos, F.: Klinische Untersuchungen über die Beziehungen zwischen Osmose, Blutdruck und Augendruck. I. Hypertonie durch Senkung des Blutzuckerspiegels bei jugendlichem Diabetes und Glaukom mit Diabetes. Klin. Mbl. Augenheilk.84, 340 (1930.\nRömer, P.: Experimentelles über Hypotonie. Klin. Mbl. Augenheilk.51, II, 592 (1913).\nSchulz, Fr. N.: inWintersteins Handbuch der vergleichenden Physiologie, Bd. I, 1. Hälfte, S. 1111. Jena: Gustav Fischer 1925.\nSzakall, A.: Über den Phosphatstoffwechsel bei Muskelarbeit. Arb. physiol.8, 316 (1934).\nSzily, A. v.: Arch. exper. Path. u. Pharmakol.157, 53 (1930).\nWelt, L. G., J. Orloff, D. M. Kydd u.J. E. Oltmann: An example of cellular hyperosmolarity. J. Clin. Invest.29, 935 (1950).\nWolff, L. K., u.S. E. de Jongh: (1) Über eine augendruckerniedrigende Substanz. Klin. Wschr.1925, 344.\n: (2) Über eine Substanz, die imstande ist, den Augendruck herabzusetzen. Biochem. Z.163, 428 (1925).\nWolff, L. K., u.S. E. de Jongh: (3) Über augendruckerniedrigende Mittel beim Glaukom. Klin. Wschr.1925, 2489.\nZipf, K.: (1) Die chemische Natur des Frühgiftes. Arch. exper. Path. u. Pharmakol.157, 97 (1930).\n: (2) Die chemische Natur der “depressorischen Substanz” des Blutes. Arch. exper. Path. u. 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The study examines the association between retinochoroidal parameters and renal impairment in hypertensive, non-diabetic patients. This is a cross-sectional study on Caucasian patients with systemic arterial hypertension with different levels of renal function. All subjects were studied by blood chemistry, urine examination, microalbuminuria and blood pressure. Complete eye examination was completed with swept source optical coherence tomography (SS-OCT) and optical coherence tomography angiography (OCTA) scans of macular region. Patients were divided in groups: LowGFR and HighGFR and CKD− and CKD+, according to the value of glomerular filtrate (GFR) and albuminuria. LowGFR and CKD+ groups included patients with clinical kidney impairment. One hundred and twenty eyes of 120 hypertensive patients were evaluated. The mean retinal thickness was thinner in CKD+ versus CKD− group (p \u003C 0.009). LowGFR and CKD+ groups showed thinner choroidal values than HighGFR (p \u003C 0.02) and CKD− (p \u003C 0.001) groups. OCTA showed lower density in LowGFR than in HighGFR group (p \u003C 0.001) and in CKD+ versus CKD− group (p \u003C 0.001). Albuminuria was inversely related to choroidal and retinal thickness measures (p \u003C 0.001) and to the indices of superficial parafoveal (p \u003C 0.05) and foveal (p \u003C 0.05) vascular densities. CKD is associated with retinal thinning, eGFR and decreasing renal function with progressive reduction of choroidal and retinal vascular density. SS-OCT and OCTA documented close association between CKD and reduction of both choroidal thickness and paracentral retinal vascular density in hypertensive patients.",{"EN":1078},"Retinal and choroidal vasculature changes associated with chronic kidney disease",{"VOID":1080},"Jorgensen T, Capewell S, Prescott E et al (2013) Population-level changes to promote cardiovascular health. Eur J Prev Cardiol 20(3):409–421\nMendis S, Davis S, Norrving B (2015) Organizational update: the world health organization global status report on noncommunicable diseases 2014; one more landmark step in the combat against stroke and vascular disease. Stroke 46(5):e121–e122\nLewington S, Clarke R, Qizilbash N, Prospective Studies Collaboration et al (2002) Age-specific relevance of usual blood pressure to vascular mortality: a meta-analysis of individual data for one million adults in 61 prospective studies. Lancet 360(9349):1903–1913\nMulè G, Castiglia A, Cusumano C et al (2017) Subclinical kidney damage in hypertensive patients: a renal window opened on the cardiovascular system. Focus on microalbuminuria. Adv Exp Med Biol 956:279–306\nWong CW, Wong TY, Cheng CY et al (2014) Kidney and eye diseases: common risk factors, etiological mechanisms, and pathways. Kidney Int 85(6):1290–1302\nWilkinson-Berka JL, Agrotis A et al (2012) The retinal renin-angiotensin system: roles of angiotensin II and aldosterone. Peptides 36:142–150\nBenigni A, Cassis P, Remuzzi G (2010) Angiotensin II revisited: new roles in inflammation, immunology and aging. EMBO Mol Med 2:247–257\nWong TY, Coresh J, Klein R et al (2004) Retinal microvascular abnormalities and renal dysfunction: the atherosclerosis risk in communities study. J Am Soc Nephrol 15(9):2469–2476\nGrunwald JE, Alexander J, Maguire M, CRIC Study Group et al (2010) Prevalence of ocular fundus pathology in patients with chronic kidney disease. Clin J Am Soc Nephrol 5(5):867–873\nFerrara D, Waheed NK, Duker JS (2016) Investigating the choriocapillaris and choroidal vasculature with new optical coherence tomography technologies. Prog Retin Eye Res 52:130–155\nESH\u002FESC task Force for the Management of Arterial Hypertension (2013) 2013 Practice guidelines for the management of arterial hypertension of the European Society of Hypertension (ESH) and the European Society of Cardiology (ESC): ESH\u002FESC task force for the management of arterial hypertension. J Hypertens 31(10):1925–1938\nDownie LE, Hodgson LA, Dsylva C et al (2013) Hypertensive retinopathy: comparing the Keith-Wagener-Barker to a simplified classification. J Hypertens 31(5):960–965\nBalmforth C, van Bragt JJ, Ruijs T et al (2016) Chorioretinal thinning in chronic kidney disease links to inflammation and endothelial dysfunction. JCI Insight 8(1):20 e89173\nMulè G, Vadalà M, Geraci G, Cottone S (2018) Retinal vascular imaging in cardiovascular medicine: new tools for an old examination. Atherosclerosis. 268:188–190. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.atherosclerosis.2017.11.001\nNickla DL, Wallman J (2010) The multifunctional choroid. Prog Retin Eye Res 29:144–168\nLinsenmeier RA, Padnick-Silver L (2000) Metabolic dependence of photoreceptors on the choroid in the normal and detached retina. Invest Ophthalmol Vis Sci 41:3117–3123\nSpraul CW, Lang GE, Lang GK et al (2002) Morphometric changes of the choriocapillaris and the choroidal vasculature in eyes with advanced glaucomatous changes. Vis Res 42:923–932\nBrown JS, Flitcroft DI, Ying G et al (2009) In vivo human choroidal thickness measurements: evidence for diurnal fluctuations. Invest Ophthalmol Vis Sci 50:5–12\nGao SS, Jia Y, Zhang M et al (2016) Optical coherence tomography angiography. Invest Ophthalmol Vis Sci 57(9):OCT27–OCT36\nCerasola G, Cottone S, Mule G (2010) The progressive pathway of microalbuminuria: from early marker of renal damage to strong cardiovascular risk predictor. J Hypertens 28(12):2357–2369\nChen YH, Chen HS, Tarng DC (2012) More impact of microalbuminuria on retinopathy than moderately reduced GFR among type 2 diabetic patients. Diabetes Care 35(4):803–808\nSchlaich MP, Socratous F, Hennebry S et al (2009) Sympathetic activation in chronic renal failure. J Am Soc Nephrol 20(5):933–939\nWallman J, Wildsoet C, Xu A et al (1995) Moving the retina: choroidal modulation of refractive state. Vis Res 35(1):37–50\nCopete S, Flores-Moreno I, Montero JA et al (2014) Direct comparison of spectral-domain and swept-source OCT in the measurement of choroidal thickness in normal eyes. Br J Ophthalmol 98(3):334–338\nBahrami B, Ewe SYP, Hong T et al (2017) Influence of retinal pathology on the reliability of macular thickness measurement: a comparison between optical coherence tomography devices. Ophthalmic Surg Lasers Imaging Retina 48(4):319–325\nUlaş F, Doğan Ü, Keleş A et al (2013) Evaluation of choroidal and retinal thickness measurements using optical coherence tomography in non-diabetic haemodialysis patients. Int Ophthalmol 33(5):533–539\nJung JW, Chin HS, Lee DH et al (2014) Changes in subfoveal choroidal thickness and choroidal extravascular density by spectral domain optical coherence tomography after haemodialysis: a pilot study. Br J Ophthalmol 98(2):207–212\nRuiz-Medrano J, Flores-Moreno I, Peña-García P et al (2014) Macular choroidal thickness profile in a healthy population measured by swept-source optical coherence tomography. Invest Ophthalmol Vis Sci 55(6):3532–3542\nMasis M, Hernandez E, Wu L (2011) Choroidal thickness in patients with systemic hypertension. ARVO Meeting Abstract Invest Ophthalmol Vis Sci (52):5296\nAkay F, Gundogan FC, Yolcu U et al (2016) Choroidal thickness in systemic arterial hypertension. Eur J Ophthalmol 26(2):152–157\nLiao MT, Sung CC, Hung KC et al (2012) Insulin resistance in patients with chronic kidney disease. J Biomed Biotechnol 2012:691369\nChen J, Muntner P, Hamm LL et al (2003) Insulin resistance and risk of chronic kidney disease in nondiabetic US adults. J Am Soc Nephrol 14:469–477",{"VOID":1082},"10.1007\u002Fs00417-019-04358-3","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs00417-019-04358-3",[1085,1100,1113,1126,1139,1154],{"id":1086,"sortIndex":32,"researcher":28,"roles":1087,"affiliations":1088,"properties":1097,"displayName":1099,"givenName":28,"familyName":28},"254f9257-7d80-4ac5-80e7-a3996b761453",[984],[1089],{"id":1090,"sortIndex":32,"affiliation":1091,"properties":28},"73c24a42-e36a-4ff4-b6c2-eabf29e95467",{"id":1090,"createTime":28,"updateTime":28,"relativeEntities":1092,"slug":28,"properties":1093,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1096,"statistic":28},[],{"title":1094},{"VI":1095},"Dipartimento di Biomedicina Sperimentale e Neuroscienze cliniche, Sezione di Oftalmologia, University of Palermo, Palermo, Italy",[],{"title":1098},{"VI":1099},"Maria 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compare corneal tomographic parameters between Hispanic White and non-Hispanic White patients using Pentacam data. This retrospective study evaluated preoperative Pentacam data from 641 patients 50 years or older who underwent surgery for senile cataract and self-identified as Hispanic or non-Hispanic White. Patients of non-White race or multiethnic groups, or a history of surgery, trauma, or any abnormality of the cornea or anterior segment were excluded. Cornea and anterior segment parameters, as measured with Pentacam, were then compared between Hispanics and non-Hispanics. There were 352 Hispanic White and 289 non-Hispanic White patients. These included 231 men and 410 women, with a mean age of 69.5 ± 8.2 years. There were no significant differences between Hispanics and non-Hispanics in front or back keratometry or amount of front astigmatism. However, Hispanics had a greater amount of back astigmatism (0.36 ± 0.19 vs 0.32 ± 0.17 diopter, P = 0.04). Moreover, there was a statistically significant difference in front steep axis of the left eyes between Hispanics and non-Hispanics (97.8 ± 47.9 vs 108.2 ± 48.9 deg, P = 0.01), and a marginally significant difference in front steep axis of the right eyes (81.0 ± 48.2 vs 73.5 ± 49.9 deg, P = 0.06). Hispanics also had a lower vertex pachymetry (548.1 ± 34.5 vs 553.4 ± 37.4 μm, P = 0.04) and a smaller anterior chamber volume (134.7 ± 39.0 vs 146.1 ± 39.9 mm3, P \u003C 0.001). There are some differences in cornea and anterior segment parameters between Hispanics and non-Hispanics 50 years or older who underwent surgery for senile cataract. However, such differences may not be clinically significant.",{"EN":1233},"Comparison of corneal tomographic parameters between Hispanic and non-Hispanic patients",{"VOID":1235},"Broman AT, Congdon NG, Bandeen-Roche K, Quigley HA (2007) Influence of corneal structure, corneal responsiveness, and other ocular parameters on tonometric measurement of intraocular pressure. J Glaucoma 16:581–588\nHerndon LW (2006) Measuring intraocular pressure-adjustments for corneal thickness and new technologies. Curr Opin Ophthalmol 17:115–119\nPiñero DP, Alió JL, Alesón A, Vergara ME, Miranda M (2010) Corneal volume, pachymetry, and correlation of anterior and posterior corneal shape in subclinical and different stages of clinical keratoconus. J Cataract Refract Surg 36:814–825\nFriedman MW, Wright ES (1952) Hereditary microcornea and cataract in five generations. Am J Ophthalmol 35:1017–1021\nCarney LG, Mainstone JC, Henderson BA (1997) Corneal topography and myopia. A cross-sectional study. Invest Ophthalmol Vis Sci 38:311–320\nHashemi H, Asgari S, Emamian MH, Mehravaran S, Fotouhi A (2015) Age-related changes in corneal curvature and shape. Cornea 34:1456–1458\nAlsbirk PH (2009) Corneal Thickness: I. Age variation, sex difference and oculometric correlations. Acta Ophthalmol 56:95–104\nTong J, Phu J, Kalloniatis M, Zangerl B (2020) Modeling changes in corneal parameters with age: implications for corneal disease detection. Am J Ophthalmol 209:117–131\nOrucoglu F, Akman M, Onal S (2015) Analysis of age, refractive error and gender related changes of the cornea and the anterior segment of the eye with Scheimpflug imaging. Contact Lens Anterior Eye 38:345–350\nGoto T, Klyce SD, Zheng X, Maeda N, Kuroda T, Ide C (2001) Gender- and age-related differences in corneal topography. Cornea 20:270–276\nDimasi DP, Burdon KP, Craig JE (2010) The genetics of central corneal thickness. Br J Ophthalmol 94:971–976\nShimmyo M, Ross AJ, Moy A, Mostafavi R (2003) Intraocular pressure, Goldmann applanation tension, corneal thickness, and corneal curvature in Caucasians, Asians, Hispanics, and African Americans. Am J Ophthalmol 136:603–613\nAng M, Chong W, Huang H, Wong TY, He MG, Aung T, Mehta JS (2014) Determinants of posterior corneal biometric measurements in a multi-ethnic Asian population. PLoS One 9(7):e101483\nPan CW, Cheng CY, Sabanayagam C, Chew M, Lam J, Ang M, Wong TY (2014) Ethnic variation in central corneal refractive power and steep cornea in Asians. Ophthalmic Epidemiol 21:99–105\nLam BC, Weiss M, Jing F, Zhu C, Johnson DA, Kheirkhah A (2022) Comparison of ocular biometric parameters between Hispanic and non-Hispanic ethnicities in White adults undergoing cataract surgery. Eye Contact Lens 48:391–395\nLee RY, Chon BH, Lin SC, He M, Lin SC (2015) Association of ocular conditions with narrow angles in different ethnicities. Am J Ophthalmol 160:506-515.e1\nHalawa OA, Jin Q, Pasquale LR, Kang JH, Lorch AC, Sobrin L, Miller JW, Li Y, Eslami M, Wang M, Zebardast N, Elze T (2022) Race and ethnicity differences in disease severity and visual field progression among glaucoma patients. Am J Ophthalmol 242:69–76\nStein JD, Kim DS, Niziol LM, Talwar N, Nan B, Musch DC, Richards JE (2011) Differences in rates of glaucoma among Asian Americans and other racial groups, and among various Asian ethnic groups. Ophthalmology 118:1031–1037\nEmanuele N, Sacks J, Klein R, Reda D, Anderson R, Duckworth W, Abraira C, Veterans Affairs Diabetes Trial Group (2005) Ethnicity, race, and baseline retinopathy correlates in the veterans affairs diabetes trial. Diabetes Care 28:1954–1958\nRabsilber TM, Khoramnia R, Auffarth GU (2006) Anterior chamber measurements using Pentacam rotating Scheimpflug camera. J Cataract Refract Surg 32:456–459\nHo JD, Tsai CY, Tsai RJF, Kuo LL, Tsai IL, Liou SW (2008) Validity of the keratometric index: Evaluation by the Pentacam rotating Scheimpflug camera. J Cataract Refract Surg 34:137–145\nShankar H, Taranath D, Santhirathelagan CT, Pesudovs K (2008) Anterior segment biometry with the Pentacam: Comprehensive assessment of repeatability of automated measurements. J Cataract Refract Surg 34:103–113\nMcAlinden C, Khadka J, Pesudovs K (2011) A Comprehensive evaluation of the precision (repeatability and reproducibility) of the Oculus Pentacam HR. Invest Opthalmol Vis Sci 52:7731–7737\n2020 Census State Redistricting Data (Public Law 94-171) Summary file prepared by the U.S. Census Bureau, 2021. https:\u002F\u002Fwww2.census.gov\u002Fprograms-surveys\u002Fdecennial\u002F2020\u002Fdata\u002Fredistricting-supplementary-tables\u002Fredistricting-supplementary-table-04.pdf\nAghaian E, Choe JE, Lin S, Stamper RL (2004) Central corneal thickness of Caucasians, Chinese, Hispanics, Filipinos, African Americans, and Japanese in a glaucoma clinic. Ophthalmology 111:2211–2219\nWang D, Amoozgar B, Porco T, Wang Z, Lin SC (2017) Ethnic differences in lens parameters measured by ocular biometry in a cataract surgery population. PLoS One 12(6):e0179836\nLeung CK, Palmiero PM, Weinreb RN, Li H, Sbeity Z, Dorairaj S, Leung D, Liu S, Liebmann JM, Congdon N, Lam DS, Ritch R (2010) Comparisons of anterior segment biometry between Chinese and Caucasians using anterior segment optical coherence tomography. Br J Ophthalmol 94:1184–1189\nFoster PJ (2002) The epidemiology of primary angle closure and associated glaucomatous optic neuropathy. Semin Ophthalmol 17:50–58\nWang YE, Li Y, Wang D, He M, Lin S (2013) Comparison of factors associated with occludable angle between American Caucasians and ethnic Chinese. Invest Opthalmol Vis Sci 54:7717–7723\nTwelker JD, Mitchell GL, Messer DH, Bhakta R, Jones LA, Mutti DO, Cotter SA, Klenstein RN, Manny RE, Zadnik K, CLEERE Study Group (2009) Children’s ocular components and age, gender, and ethnicity. Optom Vis Sci 86:918–935\nFozailoff A, Tarczy-Hornoch K, Cotter S, Wen G, Lin J, Borchert M, Azen S, Varma R, Writing Committee for the MEPEDS Study Group (2011) Prevalence of astigmatism in 6- to 72-month-old African American and Hispanic children: The Multi-Ethnic Pediatric Eye Disease Study. Ophthalmology 118:284–293\nHuynh SC, Kifley A, Rose KA, Morgan I, Heller GZ, Mitchell P (2006) Astigmatism and its components in 6-year-old children. Invest Opthalmol Vis Sci 47:55–64\nWen G, Tarczy-Hornoch K, McKean-Cowdin R, Cotter SA, Borchert M, Lin J, Kim J, Varma R (2013) Prevalence of myopia, hyperopia, and astigmatism in non-Hispanic White and Asian children. Ophthalmology 120:2109–2116\nO’Donnell C, Maldonado-Codina C (2005) Agreement and repeatability of central thickness measurement in normal corneas using ultrasound pachymetry and the Oculus Pentacam. Cornea 24:920–924\nAl-Mezaine HS, Al-Amro SA, Kangave D, Sadaawy A, Wehaib TA, Al-Obeidan S (2008) Comparison between central corneal thickness measurements by oculus pentacam and ultrasonic pachymetry. Int Ophthalmol 28:333–338\nHo T, Cheng ACK, Rao SK, Lau S, Leung CKS, Lam DSC (2007) Central corneal thickness measurements using Orbscan II, Visante, ultrasound, and Pentacam pachymetry after laser in situ keratomileusis for myopia. J Cataract Refract Surg 33:1177–1182\nUçakhan ÖÖ, Özkan M, Kanpolat A (2006) Corneal thickness measurements in normal and keratoconic eyes: Pentacam comprehensive eye scanner versus noncontact specular microscopy and ultrasound pachymetry. J Cataract Refract Surg 32:970–977\nStodtmeister R (1998) Applanation tonometry and correction according to corneal thickness. Acta Ophthalmol Scand 76:319–324\nDoughty MJ, Zaman ML (2000) Human corneal thickness and its impact on intraocular pressure measures: a review and meta-analysis approach. Surv Ophthalmol 44:367–408\nQuigley HA, West SK, Rodriguez J, Munoz B, Klein R, Snyder R (2001) The prevalence of glaucoma in a population-based study of Hispanic Subjects. Arch Ophthalmol 119:1819–1826\nWang D, Qi M, He M, Wu L, Lin S (2012) Ethnic difference of the anterior chamber area and volume and its association with angle width. Invest Opthalmol Vis Sci 53:3139–3144",{"VOID":1237},"10.1007\u002Fs00417-023-06167-1","https:\u002F\u002Flink.springer.com\u002F10.1007\u002Fs00417-023-06167-1",[1240,1255,1268,1281,1294],{"id":1241,"sortIndex":32,"researcher":28,"roles":1242,"affiliations":1243,"properties":1252,"displayName":1254,"givenName":28,"familyName":28},"28c32fec-da7c-4ce7-8596-3ed2159a50fe",[984],[1244],{"id":1245,"sortIndex":32,"affiliation":1246,"properties":28},"0ff8a370-2dc9-475d-8f0d-c2b502e73d8d",{"id":1245,"createTime":28,"updateTime":28,"relativeEntities":1247,"slug":28,"properties":1248,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1251,"statistic":28},[],{"title":1249},{"VI":1250},"Department of Ophthalmology, UT Health San Antonio, San Antonio, USA",[],{"title":1253},{"VI":1254},"So Yeon 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assessed the ability of deep learning (DL) models to distinguish between tear meniscus of lacrimal duct obstruction (LDO) patients and normal subjects using anterior segment optical coherence tomography (ASOCT) images. The study included 117 ASOCT images (19 men and 98 women; mean age, 66.6 ± 13.6 years) from 101 LDO patients and 113 ASOCT images (29 men and 84 women; mean age, 38.3 ± 19.9 years) from 71 normal subjects. We trained to construct 9 single and 502 ensemble DL models with 9 different network structures, and calculated the area under the curve (AUC), sensitivity, and specificity to compare the distinguishing abilities of these single and ensemble DL models. For the highest single DL model (DenseNet169), the AUC, sensitivity, and specificity for distinguishing LDO were 0.778, 64.6%, and 72.1%, respectively. For the highest ensemble DL model (VGG16, ResNet50, DenseNet121, DenseNet169, InceptionResNetV2, InceptionV3, and Xception), the AUC, sensitivity, and specificity for distinguishing LDO were 0.824, 84.8%, and 58.8%, respectively. The heat maps indicated that these DL models placed their focus on the tear meniscus region of the ASOCT images. The combination of DL and ASOCT images could distinguish between tear meniscus of LDO patients and normal subjects with a high level of accuracy. These results suggest that DL might be useful for automatic screening of patients for LDO.",{"EN":1382},"Automatic screening of tear meniscus from lacrimal duct obstructions using anterior segment optical coherence tomography images by deep learning",{"VOID":1384},"Mishima S, Gasset A, Klyce SD Jr et al (1966) Determination of tear volume and tear flow. Investig Ophthalmol 5:264–276\nMaurice DM (1973) The dynamics and drainage of tears. Int Ophthalmol Clin 13:103–116\nShen M, Li J, Wang J et al (2009) Upper and lower tear menisci in the diagnosis of dry eye. Invest Ophthalmol Vis Sci 50:2722–2726\nRoh JH, Chi MJ (2010) Efficacy of dye disappearance test and tear meniscus height in diagnosis and postoperative assessment of nasolacrimal duct obstruction. 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Cont Lens Anterior Eye 25:57–65",{"VOID":1386},"10.1007\u002Fs00417-021-05078-3","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs00417-021-05078-3",[1389,1404,1426,1446,1466,1479,1492],{"id":1390,"sortIndex":32,"researcher":28,"roles":1391,"affiliations":1392,"properties":1401,"displayName":1403,"givenName":28,"familyName":28},"22611f88-e191-44dc-92e7-6e2944042e47",[984],[1393],{"id":1394,"sortIndex":32,"affiliation":1395,"properties":28},"2fdb2f54-b824-4062-b9ee-c6841d294c46",{"id":1394,"createTime":28,"updateTime":28,"relativeEntities":1396,"slug":28,"properties":1397,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1400,"statistic":28},[],{"title":1398},{"VI":1399},"Department of Ophthalmology, Tsukazaki Hospital, Himeji City, Japan",[],{"title":1402},{"VI":1403},"Hitoshi 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dem Elektroretinogramm (ERG) in situ, dem ERG der isolierten Netzhaut, der Einzelfaserableitung vom N. opticus und dem visuell evozierten Potential der Sehrinde wurde der Einfluß von NP 207 (Piperidylchloro-phenothiazin) sowohl bei „akuter“ als auch bei „chronischer“ Verabreichung an Katzen studiert. Im ERG zeigte sich eine dosisabhängige starke Abnahme der c-Wellenamplitude, a- und b-Wellen werden weniger beeinflußt. Die Spontanaktivität im N. opticus war stark vermindert, die phasische Reaktion wurde nicht einheitlich beeinflußt. Das evozierte Potential wurde kaum verändert. Im „akuten“ Experiment an der isolierten Netzhaut konnte gezeigt werden, daß die Effekte teilweise reversibel sind. Im „chronischen“ Versuch kehrte sogar nach 31wöchiger Applikation nach einem medikationsfreien Intervall von 3 Tagen normale Einzelfaseraktivität des N. opticus wieder. Die starke Abnahme der Amplitude der c-Welle und der Frequenz der Spontanaktivität lassen gemeinsam mit den primären histologischen Schäden an den Receptoraußengliedern die Receptoren als wichtigsten Angriffspunkt für die schädigende Wirkung des NP 207 vermuten.",{"EN":1573},"Tierexperimentelle Untersuchungen mit einer das visuelle System schädigenden Substanz (NP 207)",{"VOID":1575},"Anden, N.-E., Butcher, S. G., Corrodi, H., Fuxe, K., Ungerstedt, U.: Receptor activity and turnover of dopamine and nor-adrenaline after neuroleptics. Europ. J. Pharmacol. 11, 303–314 (1970)\nAnden, N.-E., Dahlström, A., Fuxe, K., Hökfelt, T.: The effect of haloperidol and chlorpromazine on the amine levels of central monoamine neurons. Acta physiol. scand. 68, 419–420 (1966)\nBrown, K. T., Wiesel, T. N.: Intraretinal recording in the unopened cat eye. Amer. J. Ophthal. 46, 91–96 (1958)\nBrown, K. T., Wiesel, T. N.: Analysis of the intraretinal electroretinogram in the intact cat eye. J. Physiol. (Lond.) 158, 229–256 (1961)\nBurian, H. M., Fletcher, M. C.: Visual functions in patients with retinal pigmentary degeneration following the use of NP 207. Arch. Ophthal. 60, 612–629 (1958)\nCervetto, L., McNichol, E. F.: Inactivation of horizontal cells in turtle retina by glutamate and aspartate. Science 178, 767–768 (1972)\nGoar, E. L., Fletcher, M. C.: Toxic chorioretinopathy following the use of NP 207. Amer. J. Ophthal. 44, 603 (1957)\nHäggendal, J., Malmfors, T.: Identification and cellular localisation of the catecholamines in the retina and the choroid of the rabbit. Acta physiol. scand. 64, 58–66 (1965)\nHanitzsch, R.: Intraretinal isolation of P III subcomponents in the isolated rabbit retina after treatment with sodium aspartate. Vision Res. 13, 2093–2102 (1973)\nHanitzsch, R., Bornschein, H.: Spezielle Überlebensbedingungen für isolierte Netzhäute verschiedener Warmblüter. Experentia (Basel) 21, 484 (1965)\nHeiss, W.-D., Heilig, P., Hoyer, J.: Die Wirkung von Psychopharmaka auf die Aktivität retinaler Neurone. Vision Res. 9, 493–506 (1969)\nHeiss, W.-D., Hoyer, J., Heilig, P.: Die Wirkung von Psychopharmaka auf visuell evozierte Potentiale der Katze. Vision Res. 9, 507–513 (1969)\nKishida, K., Naka, K. J.: Amino acids and the spikes from the retinal ganglion cells. Science 156, 648–649 (1967)\nKramer, G. S.: Dopamine: A retinal neurotransmitter I. Retinal uptake, storage and light stimulated release of H3-dopamine in vivo. Invest. Ophthal. 10, 438–452 (1971)\nLützow, A. v., Hommer, K.: Influences of some drugs on the ERG of the isolated rabbit retina, 3rd Congr. Europ. Soc. Ophthal., Amsterdam 1968. Ophthalmologica (Basel) Additamentum ad vol. 158, 647–652 (1969)\nLützow, A. v., Wündsch, L.: Grenzen der zeitlichen Summation im Doppelreiz-ERG des Kaninchens. Vision Res. 7, 565–571 (1967)\nMacaione, S.: Localization of GABA system in rat retina. J. Neurochem. 19, 1397–1400 (1972)\nMay, R. H., Selymes, P., Weekly, R. O., Potts, A. M.: Thioridazine therapy: Results and complications. J. Nerv. ment. Dis. 130, 230 (1960)\nMeier-Ruge, W.: Medikamentöse Retinopathie. Stuttgart: Thieme 1967\nMiller, R. F., Dowling, J. E.: Intracellular responses of the Müller (glial) cells of the mudpuppy retina: Their relation to b-wave of the electroretinogram. J. Neurophysiol. 33, 323–341 (1970)\nNoell, W. K.: The effect of iodoacetate on the vertebrate retina. J. cell. comp. Physiol. 37, 283–307 (1951)\nNoell, W. K.: The origin of the electroretinogram. Amer. J. Ophthal. 38, 78–93 (1954)\nSchubert, G., Bornschein, H.: Spezifische Schädigung von Netzhautelementen durch Jodazetat. Experientia (Basel) 7, 461 (1951)\nSillman, A. J., Ito, H., Tomita, T.: Studies on the mass receptor potential of the isolated frog retina I. General properties of the response. Vision Res. 9, 1435–1442 (1969)\nSteinberg, R. H., Schmidt, R., Brown, K. T.: Intracellular responses to light from the cat's pigment epithelium: origin of the c-wave of the electroretinogram. Nature (Lond.) 227, 728–730 (1970)\nStraschill, M., Perwein, J.: The inhibition of retinal ganglion cells by catecholamines and γ-aminobutyric acid. Pflügers Arch. 312, 45–54 (1969)\nTomita, T.: Electrical activity in the vertebrate retina. J. opt. Soc. 53, 49–57 (1963)\nVerrey, F.: Dégénérescence pigmentaire de la rétine d'origine médicamenteuse. Ophthalmologica (Basel) 131, 296 (1956)\nWerblin, F. S., Dowling, J. E.: Organization of the retina of the mudpuppy, Necturus maculosus II. Intracellular recording. J. Neurophysiol. 32, 339–355 (1969)",{"VOID":1577},"10.1007\u002FBF00414332","2024-12-14T11:50:41.076+00:00","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002FBF00414332",[1581,1596,1609,1622,1635,1648,1661,1674],{"id":1582,"sortIndex":32,"researcher":28,"roles":1583,"affiliations":1584,"properties":1593,"displayName":1595,"givenName":28,"familyName":28},"5120ae72-cb44-4c4a-89fb-db51b3060d94",[984],[1585],{"id":1586,"sortIndex":32,"affiliation":1587,"properties":28},"447e19d8-d6ab-4926-b22a-b53236bcb8fe",{"id":1586,"createTime":28,"updateTime":28,"relativeEntities":1588,"slug":28,"properties":1589,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1592,"statistic":28},[],{"title":1590},{"VI":1591},"Institut für allgemeine und vergleichende Physiologie, II. Augenklinik und Neurologische Klinik der Universität Wien, Österreich",[],{"title":1594},{"VI":1595},"H. 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It is characterized by three pathophysiological components, namely microangiopathy, neurodegeneration, and inflammation. We recently reported that intraperitoneal administration of BNN27, a novel neurosteroidal microneurotrophin, reversed the diabetes-induced neurodegeneration and inflammation in rats treated with streptozotocin (STZ), by activating the NGF TrkA and p75 receptors. The aim of the present study was to investigate the efficacy of BNN27 to protect retinal neurons when applied topically as eye drops in the same model. The STZ rat model of DR was employed. BNN27 was administered as eye drops to diabetic Sprague-Dawley rats for 7 days, 4 weeks post-STZ (70 mg\u002Fkg) injection. Immunohistochemistry and western blot analyses were employed to examine the viability of retinal neurons in control, diabetic, and diabetic-treated animals and the involvement of the TrkA receptor and its downstream signaling ERK1\u002F2 kinases, respectively. BNN27 reversed the STZ-induced attenuation of the immunoreactive brain nitric oxide synthetase (bNOS)- and tyrosine hydroxylase (TH)-expressing amacrine cells and neurofilament (NFL)-expressing ganglion cell axons in a dose-dependent manner. In addition, BNN27 activated\u002Fphosphorylated the TrkA receptor and its downstream prosurvival signaling pathway, ERK1\u002F2 kinases. The results of this study provide solid evidence regarding the efficacy of BNN27 as a neuroprotectant to the diabetic retina when administered topically, and suggest that its pharmacodynamic and pharmacokinetic profiles render it a putative therapeutic for diabetic retinopathy.",{"EN":1753},"Effect of topical administration of the microneurotrophin BNN27 in the diabetic rat retina",{"VOID":1755},"Yau JW, Rogers SL, Kawasaki R et al (2012) META-analysis for eye disease (META-EYE) study group. Diabetes Care 35:556–564\nAntonetti DA, Barber AJ, Bronson SK et al (2006) Diabetic retinopathy seeing beyond glucose-induced microvascular disease. Diabetes. 55:2041–2411\nAntonetti DA, Klein R, Gardner TW (2012) Mechanisms of disease diabetic retinopathy. N Engl J Med 366:1227–1239\nBarber AJ, Lieth E, Khin SA, Antonetti DA, Buchanan AG, Gardner TW, The Penn State Retina Research Group (1998) Neural apoptosis in the retina during experimental and human diabetes. Early onset and effect of insulin. J Clin Invest 102:783–791\nSimó R, Hernández C (2014) Neurodegeneration in the diabetic eye: new insights and therapeutic perspectives. Trends Endocrinol Metab 25:23–33\nTang J, Kern TS (2011) Inflammation in diabetic retinopathy. Prog Retin Eye Res 30:343–358\nSolomon SD, Chew E, Duh EJ et al (2017) Diabetic retinopathy: a position statement by the American Diabetes Association. Diabetes Care 40:412–418\nStitt AW, Curtis TM et al (2016) The progress in understanding and treatment of diabetic retinopathy. Prog Retinal Eye Res 51:156–186\nPark SH, Park JW, Park SJ et al (2003) Apoptotic death of photoreceptors in the streptozotocin-induced diabetic rat retina. Diabetologia 46:1260–1268\nKizawa J, Machida S, Kobayashi T et al (2006) Changes of oscillatory potentials and photopic negative response in patients with early diabetic retinopathy. Jpn J 50(4):367–373\nCarrasco E, Hernández C, Miralles A et al (2007) Lower somatostatin expression is an early event in diabetic retinopathy and is associated with retinal neurodegeneration. Diabetes Care 30:2902–2908\nCarrasco E, Hernández C, de Torres I et al (2008) Lowered cortistatin expression is an early event in the human diabetic retina and is associated with apoptosis and glial activation. Mol Vis 14:1496–1502\nSimó R, Stitt AW, Gardner TW (2018) Neurodegeneration in diabetic retinopathy: does it really matter? Diabetologia 61:1902–1912\nIbán-Arias R, Lisa S, Mastrodimou N et al. (2018) The synthetic microneurotrophin BNN27 affects retinal function in rats with streptozotocin-induced diabetes. Diabetes 67(2):321–333\nCalogeropoulou T, Avlonitis N, Minas V et al (2009) Novel dehydroepiandrosterone derivatives with antiapoptotic, neuroprotective activity. J Med Chem 52:6569–6587\nPediaditakis I, Efstathopoulos P, Prousis KC et al (2016a) Selective and differential interactions of BNN27, a novel C17-spiroepoxy steroid derivative, with TrkA receptors, regulating neuronal survival and differentiation. Neuropharmacol 111:266–282\nPediaditakis I, Kourgiantaki A, Prousis KC et al (2016b) BNN27, a 17-spiroepoxy steroid derivative, interacts with and activates p75neurotrophin receptor, rescuing cerebellar granule neurons from apoptosis. Front Pharmacol. https:\u002F\u002Fdoi.org\u002F10.3389\u002Ffphar.2016.00512\nBennett J, O’Brien L, Brohawn D (2016) Pharmacological properties of microneurotrophin drugs developed for treatment of amyotrophic lateral sclerosis. Biochem Pharmacol 117:68–77\nKokona D, Charalampopoulos I, Pediaditakis I et al (2012) The neurosteroid dehydroepiandrosterone (DHEA) protects the retina from AMPA-induced excitotoxicity: NGF TrkA receptor involvement. Neuropharmacol 62:2106–2117\nMysona BA, Shanab A, Elshaer S, El-Remessy AB (2014) Nerve growth factor in diabetic retinopathy: beyond neurons. Expert Rev Ophthalmol 9:99–107\nBonetto G, Charalampopoulos I, Gravanis A, Karagogeos D (2017) The novel synthetic microneurotrophin BNN27 protects mature oligodendrocytes against cuprizone-induced death, through the NGF receptor TrkA. Glia 65:1376–1394.24\nTsoka P, Matsumoto H, Maidana DE et al (2018) Effects of BNN27, a novel C17-spiroepoxy steroid derivative, on experimental retinal detachment-induced photoreceptor cell death. Sci Rep 8:10661.25\nAkiyama H, Nakazawa T, Shimura M et al (2002) Presence of mitogen-activated protein kinase in retinal Müller cells and its neuroprotective effect ischemia-reperfusion injury. Neuroreport 13:2103–2107\nHernández C, García-Ramírez M, Corraliza L et al (2013) Topical administration of somatostatin prevents retinal neurodegeneration in experimental diabetes. Diabetes. 62:2569–2578\nKiagiadaki F, Thermos K (2008) Effect of intravitreal administration of somatostatin and sst2 analogs on AMPA-induced neurotoxicity in rat retina. Invest Ophthalmol Vis Sci 49:3080–3089\nBogdanov P, Simó-Servat O, Sampedro J et al (2018) Topical administration of bosentan prevents retinal neurodegeneration in experimental diabetes. Int J Mol Sci 19:3578. https:\u002F\u002Fdoi.org\u002F10.3390\u002Fijms19113578\nLambiase A, Aloe L, Centofanti M et al (2009) Experimental and clinical evidence of neuroprotection by nerve growth factor eye drops: implications for glaucoma. Proc Natl Acad Sci U S A 106:13469–13474\nColafrancesco V, Parisi V, Sposato V et al (2011) Ocular application of nerve growth factor protects degenerating retinal ganglion cells in a rat model of glaucoma. J Glaucoma 20:100–108\nMesentier-Louro LA, Rosso P, Carito V et al. (2019) Nerve growth factor role on retinal ganglion cell survival and axon regrowth: effects of ocular administration in experimental model of optic nerve injury. Mol Neurobiol 56: 1056. https:\u002F\u002Fdoi.org\u002Fhttps:\u002F\u002Fdoi.org\u002F10.1007\u002Fs12035-018-1154-1\nTirassa P, Rosso P, Iannitelli A (2018) Ocular nerve growth factor (NGF) and NGF eye drop application as paradigms to investigate NGF neuroprotective and reparative actions. 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Four weeks later, hemoglobin released from lysis of the original intravitreal clot had formed a thick layer on the retina. At this stage, many macrophages were conspicuous on the retinal surface and, in addition, in seven of the eight eyes, small cellular membranes were found by scanning electron microscopy. Light and transmission electron microscopy showed these membranes to be derived from accessory glial cells and their progeny. The membranes resembled the simple epiretinal membranes that occur in human eyes.",{"EN":2059},"Origin of simple glial epiretinal membranes in an animal model",{"VOID":2061},"Algvere P, Kock E (1983) Experimental epiretinal membranes induced by intravitreal carbon particles. Am J Ophthalmol 96:345–353\nBellhorn MB, Friedman AH, Wise GN, Henkind P (1975) Ultrastructure and clinicopathologic correlation of idiopathic preretinal macular fibrosis. Am J Ophthalmol 79:366–373\nBennett TO, Peyman GA, Vlchek JK (1975) Intravitreal injection of autologous blood in primates. Can J Ophthalmol 10:248–254\nBurke JM (1980) Phagocytes that invade the vitreous after injury stimulate DNA synthesis in neural retina in vitro. Graefe's Arch Clin Exp Ophthalmol 214:223–227\nBurke JM (1981) Vitreal superoxide and superoxide dismutase after hemorrhagic injury: the role of invasive cells. Invest Ophthalmol Vis Sci 20:435–441\nCampochiaro PA, Glaser BM (1985) Platelet-derived growth factor is chemotactic for human retinal pigment epithelial cells. Arch Ophthalmol 103:576–579\nCampochiaro PA, Jerdan JA, Glaser BM (1984) Serum contains chemoattractants for human retinal pigment epithelial cells. Arch Ophthalmol 102:1830–1833\nCampochiaro PA, Jerdan JA, Glaser BM, Cardin A, Michels RG (1985a) Vitreous aspirates from patients with proliferative vitreoretinopathy stimulate retinal pigment epithelial cell migration. Arch Ophthalmol 103:1403–1405\nCampochiaro PA, Kaden IH, Vidaurri-Leal J, Glaser BM (1985b) Cryotherapy enhances intravitreal dispersion of viable retinal pigment epithelial cells. Arch Ophthalmol 103:434–436\nCleary PE, Ryan SJ (1979) Method of production and natural history of experimental posterior penetrating eye injury in the rhesus monkey. Am J Ophthalmol 88:212–220\nConstable IJ, Horne R, Slatter DH, Chester GH, Cooper RL (1981) Regeneration of retinal limiting membranes after chorioretinal biopsy in dogs. Invest Ophthalmol Vis Sci 20:246–251\nEhrenberg M, Thresher RJ, Machemer R (1984) Vitreous hemorrhage nontoxic to retina as a stimulator of glial and fibrous proliferation. Am J Ophthalmol 97:611–626\nFoos RY (1974) Vitreoretinal juncture — simple epiretinal membranes. Graefe's Arch Clin Exp Ophthalmol 189:231–250\nFoos RY (1977) Surface wrinkling retinopathy. In: Freeman HM, Hirose T, Schepens CL (eds) Vitreous surgery and advances in fundus diagnosis and treatment. Appleton-Century-Crofts, New York, pp 23–38\nFoos RY, Gloor BP (1975) Vitreoretinal juncture; healing of experimental wounds. Graefe's Arch Clin Exp Ophthalmol 196:213–230\nForrester JV, Grierson I, Lee WR (1978) Comparative studies of erythrophagocytosis in the rabbit and human vitreous. Graefe's Arch Clin Exp Ophthalmol 208:143–158\nHiscott PS, Grierson I, Trombetta CJ, Rahi AHS, Marshall J, McLeod D (1984) Retinal and epiretinal glia — an immunohistochemical study. Br J Ophthalmol 68:698–707\nLaqua H, Machemer R (1975) Glial cell proliferation in retinal detachment (massive periretinal proliferation). Am J Ophthalmol 80:602–618\nMachemer R (1978) Pathogenesis and classification of massive periretinal proliferation. Br J Ophthalmol 62:737–747\nMachemer R, van Horn D, Aaberg TM (1978) Pigmented epithelial proliferation in human retinal detachment with massive periretinal proliferation. Am J Ophthalmol 85:181–191\nMcCord J, Stokes S, Wong K (1979) Superoxide radical as a phagocyte-produced chemical mediator of inflammation. In: Weissman G (ed) Advances in inflammation research, vol 1. Raven Press, New York, pp 273–280\nMcLeod D, Marshall J, Grierson I (1981) Epimacular membrane peeling. Trans Ophthalmol Soc UK 101:170–180\nMiller B, Miller H, Patterson R, Ryan SJ (1986) Retinal wound healing. Cellular activity at the vitreoretinal interface. Arch Ophthalmol 104:281–285\nNewsome DA, Rodrigues MM, Machemer R (1981) Human massive periretinal proliferation. In vitro characteristics of cellular components. Arch Ophthalmol 99:873–880\nRoth AM, Foos RY (1971) Surface wrinkling retinopathy in eyes enucleated at autopsy. Trans Am Acad Ophthalmol Otolaryngol 75:1047–1058\nSmelser GK, Ishikawa T, Pei YF (1965) Electron microscopic studies of intraretinal spaces; diffusion of particulate materials. In: Rohen JW (ed) The structure of the eye. Schattauer, Stuttgart, pp 109–115\nTopping TM, Abrams GW, Machemer R (1979) Experimental double-perforating injury of the posterior segment in rabbit eyes. The natural history of intraocular proliferation. Arch Ophthalmol 97:735–742\nWise GN (1975) Clinical features of idiopathic preretinal macular fibrosis. Am J Ophthalmol 79:349–357\nYeo JH, Sadeghi J, Campochiaro PA, Green WR, Glaser BM (1986) Intravitreous fibronectin and platelet-derived growth factor. New model for traction retinal detachment. Arch Ophthalmol 104:417–421",{"VOID":2063},"10.1007\u002FBF02334169","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002FBF02334169",[2066],{"id":2067,"sortIndex":32,"researcher":28,"roles":2068,"affiliations":2069,"properties":2087,"displayName":2089,"givenName":28,"familyName":28},"d662a109-bdd1-40b2-9ae9-290d319c3629",[984],[2070,2078],{"id":2071,"sortIndex":32,"affiliation":2072,"properties":28},"a12873f0-f325-4e29-936e-ef64248a1fb4",{"id":2071,"createTime":28,"updateTime":28,"relativeEntities":2073,"slug":28,"properties":2074,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2077,"statistic":28},[],{"title":2075},{"EN":2076},"Department of Ophthalmology, University of Southern California School of Medicine, Los Angeles, USA.",[],{"id":2079,"sortIndex":40,"affiliation":2080,"properties":2086},"9b9f7dde-a068-4d9b-b0b8-9d4fd873cf61",{"id":2079,"createTime":28,"updateTime":28,"relativeEntities":2081,"slug":28,"properties":2082,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2085,"statistic":28},[],{"title":2083},{"VI":2084},"Estelle Doheny Eye Foundation, Los Angeles, USA",[],{},{"title":2088},{"VI":2089},"J. S. Lean",{"url":2064,"publisher":2091,"properties":2140},{"id":868,"createTime":869,"updateTime":870,"relativeEntities":2092,"slug":872,"properties":2093,"entityType":25,"verifyStatus":878,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":32,"subjectFields":2096,"manageAffiliations":2109,"indexDatabases":2120,"url":28,"thumbnailPath":28,"statistic":2135,"gsStatistic":28,"type":28,"analyzePriority":28},[],{"issn":2094,"title":2095},{"VOID":875},{"EN":877},[2097,2101,2105],{"id":881,"createTime":28,"updateTime":28,"relativeEntities":2098,"label":2099,"description":2100,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":884},{},{"id":887,"createTime":28,"updateTime":28,"relativeEntities":2102,"label":2103,"description":2104,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":890},{},{"id":893,"createTime":28,"updateTime":28,"relativeEntities":2106,"label":2107,"description":2108,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":896},{},[2110,2115],{"id":900,"createTime":28,"updateTime":28,"relativeEntities":2111,"slug":28,"properties":2112,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2114,"statistic":28},[],{"title":2113},{"EN":904},[906],{"id":908,"createTime":28,"updateTime":28,"relativeEntities":2116,"slug":28,"properties":2117,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2119,"statistic":28},[],{"title":2118},{"EN":912},[],[2121,2128],{"id":916,"indexDatabase":2122,"url":928,"indexYears":28,"academicFieldIds":2127,"indexDatabaseRanking":28},{"id":918,"createTime":28,"updateTime":28,"relativeEntities":2123,"label":2124,"description":2125,"key":925,"publicationTags":2126,"standard":28},[],{"EN":921,"VI":921},{"EN":923,"VI":924},[927,813],[930],{"id":932,"indexDatabase":2129,"url":938,"indexYears":939,"academicFieldIds":2134,"indexDatabaseRanking":944},{"id":775,"createTime":28,"updateTime":28,"relativeEntities":2130,"label":2131,"description":2132,"key":781,"publicationTags":2133,"standard":28},[],{"EN":778,"VI":778},{"EN":778,"VI":780},[783],[941,942,943],{"impactFactor":32,"impactFactorByYear":2136,"i10Index":142,"i10IndexLast5Year":32,"totalPublication":280,"totalPublicationByYear":2137,"totalCitation":948,"totalCitationByYear":2138,"totalCitationPerPublication":954,"totalCitationPerPublicationByYear":2139,"hindexLast5Year":129,"hindex":129},{},{"1998":42,"2000":46,"2001":135,"2002":126,"2003":357},{"1998":599,"2000":950,"2001":951,"2002":952,"2003":953},{"1998":956,"2000":957,"2001":958,"2002":140,"2003":959},{"pages":2141,"volume":2143},{"VOID":2142},"421-425",{"VOID":2144},"225","1987-11-01",1987,[927,944],{"id":2149,"createTime":2150,"updateTime":2151,"relativeEntities":2152,"slug":2153,"properties":2154,"entityType":977,"verifyStatus":26,"verifyTime":2151,"verifyNote":978,"languages":28,"translateLanguages":28,"viewCount":32,"primaryUrl":2163,"fullTextUrl":28,"authors":2164,"publicationType":1010,"publisherRelationship":2180,"citationCount":28,"citationInfo":28,"publishDate":2235,"publishYear":2236,"citationAnalyzeStatus":878,"lastCitationAnalyze":28,"indexDatabases":2237,"openAccess":28,"references":28,"isForceReanalyzing":1069},"003ff1f2-fc8b-499b-940e-b300e583c7e9","2024-02-10T07:20:29.552+00:00","2025-01-02T08:39:24.885+00:00",[],"R%C3%B6ntgenphotographie-des-Foramen-opticum-bei-Geschw%C3%BClsten-und-Erkrankungen-des-Sehnerven",{"abstract":2155,"title":2157,"references":2159,"doi":2161},{"EN":2156},"Röntgenographie des Foramen opticum kann abgesehen von Untersuchung auf Fissuren, Frakturen und Callus auch benutzt werden zur Beurteilung von Größe und Form des Sehnervenloches. — Man muß bei der Beurteilung immer sehr vorsichtig sein. Die Operation, entlang dem lateralen Weg, gibt eine schöne Einsicht in die Verhältnisse am intrakraniellen Sehnervenloch und gestattet in ziemlich leichter Weise den Sehnervenkanal zu erweitern, Geschwülste zu entfernen oder zu verkleinern. Diese Operation kann benutzt werden, wenn der Kanal stark deformiert oder absolut zu klein ist, z. B. bei Veränderungen des Sehnerven, bei Turmschädel, Frakturen usw.; und wenn er durch Geschwülste vergrößert ist, weil er relativ zu eng war. Ob man zu dieser immerhin beträchtlichen Operation schreiten wird, muß abhängen von der Weise, in welcher das Leben oder das Sehvermögen bedroht sind. M. E. soll man die Operation der Sehschärfe wegen nur vornehmen, wenn beide Augen derartig bedroht sind, daß Erblindung zu erwarten ist.",{"EN":2158},"Röntgenphotographie des Foramen opticum bei Geschwülsten und Erkrankungen des Sehnerven",{"VOID":2160},"Klin. Monatsbl. f. Augenheilk.51, 2, S. 1. 1913.\nArch. f. klin. Chirurg.124, Heft 2, S. 199.\nAmerican journ. of ophthalmology 1922, S. 169.\nByers Studies from the royal Victoria hospital Montreal6, Nr. 1, S. 3.\nHudson, Royal London Ophthalmic Hospital Reports XVIII.3, 317.\nVerhoeff, Arch. of ophth.51, 120–140, 239–254. 1922.\nBenedict, Americ. journ. of ophth. 1923, S. 183.\nvon Hippel, Die Krankheiten des Sehnerven. Handbuch der gesamten Augenheilkunde S. 535.\nvan Duyse, Arch. d'opht.40, 385.\nMartin undCushing, Arch. of ophth.52, Nr. 3, S. 209.\nv. Graefes Arch. f. Ophth.93, 216. 1917.\nRhese, Arch. f. Laryngol.24, Heft 3.\nv. Graefes Arch. f. Ophth.105, 880. 1921 und111, 1. 1923.\nMitgeteilt in der „Amsterdamsche neurologenvereeniging,1. III. 1923; Nederlandsch Tijdschr. v. geneesk 1923 und in The Oxford Ophthalmological congres, July 1923. Transact. of the ophth. soc. of Kingdom43, 534. 1923.\nO. Steuer, Zeitschr. f. Hals-, Nasen- u. Ohrenheilk.4, Heft 1, S. 130. 1922.\nTendeloo, Allgemeine Pathologie 1919, S. 378.\nZentrlbl. für die ges. Ophthalmologie12, H. 11, S. 493.\nLeon E. White, Transactions of the 28 annual meeting of the Academy of Ophthalmology and Oto-laryngology. Oktober 1923, S. 20.\nH. A. Goalwin, Ophthalmol. Gesellsch. in Wien, März 1924; Klin. Monatsbl. f. Augenheilk.72, S. 543.",{"VOID":2162},"10.1007\u002FBF02283712","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002FBF02283712",[2165],{"id":2166,"sortIndex":32,"researcher":28,"roles":2167,"affiliations":2168,"properties":2177,"displayName":2179,"givenName":28,"familyName":28},"afebc2bf-b6e0-4df7-b7ef-56890ca32b57",[984],[2169],{"id":2170,"sortIndex":32,"affiliation":2171,"properties":28},"aa824a51-02a3-44b2-84a6-5fba987d4293",{"id":2170,"createTime":28,"updateTime":28,"relativeEntities":2172,"slug":28,"properties":2173,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2176,"statistic":28},[],{"title":2174},{"VI":2175},"Leiden, Holland",[],{"title":2178},{"VI":2179},"J. van der Hoeve",{"url":2163,"publisher":2181,"properties":2230},{"id":868,"createTime":869,"updateTime":870,"relativeEntities":2182,"slug":872,"properties":2183,"entityType":25,"verifyStatus":878,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":32,"subjectFields":2186,"manageAffiliations":2199,"indexDatabases":2210,"url":28,"thumbnailPath":28,"statistic":2225,"gsStatistic":28,"type":28,"analyzePriority":28},[],{"issn":2184,"title":2185},{"VOID":875},{"EN":877},[2187,2191,2195],{"id":881,"createTime":28,"updateTime":28,"relativeEntities":2188,"label":2189,"description":2190,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":884},{},{"id":887,"createTime":28,"updateTime":28,"relativeEntities":2192,"label":2193,"description":2194,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":890},{},{"id":893,"createTime":28,"updateTime":28,"relativeEntities":2196,"label":2197,"description":2198,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":896},{},[2200,2205],{"id":900,"createTime":28,"updateTime":28,"relativeEntities":2201,"slug":28,"properties":2202,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2204,"statistic":28},[],{"title":2203},{"EN":904},[906],{"id":908,"createTime":28,"updateTime":28,"relativeEntities":2206,"slug":28,"properties":2207,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2209,"statistic":28},[],{"title":2208},{"EN":912},[],[2211,2218],{"id":916,"indexDatabase":2212,"url":928,"indexYears":28,"academicFieldIds":2217,"indexDatabaseRanking":28},{"id":918,"createTime":28,"updateTime":28,"relativeEntities":2213,"label":2214,"description":2215,"key":925,"publicationTags":2216,"standard":28},[],{"EN":921,"VI":921},{"EN":923,"VI":924},[927,813],[930],{"id":932,"indexDatabase":2219,"url":938,"indexYears":939,"academicFieldIds":2224,"indexDatabaseRanking":944},{"id":775,"createTime":28,"updateTime":28,"relativeEntities":2220,"label":2221,"description":2222,"key":781,"publicationTags":2223,"standard":28},[],{"EN":778,"VI":778},{"EN":778,"VI":780},[783],[941,942,943],{"impactFactor":32,"impactFactorByYear":2226,"i10Index":142,"i10IndexLast5Year":32,"totalPublication":280,"totalPublicationByYear":2227,"totalCitation":948,"totalCitationByYear":2228,"totalCitationPerPublication":954,"totalCitationPerPublicationByYear":2229,"hindexLast5Year":129,"hindex":129},{},{"1998":42,"2000":46,"2001":135,"2002":126,"2003":357},{"1998":599,"2000":950,"2001":951,"2002":952,"2003":953},{"1998":956,"2000":957,"2001":958,"2002":140,"2003":959},{"pages":2231,"volume":2233},{"VOID":2232},"355-369",{"VOID":2234},"115","1925-01-01",1925,[927,944],{"id":2239,"createTime":2240,"updateTime":2241,"relativeEntities":2242,"slug":2243,"properties":2244,"entityType":977,"verifyStatus":26,"verifyTime":2241,"verifyNote":978,"languages":28,"translateLanguages":28,"viewCount":32,"primaryUrl":2253,"fullTextUrl":28,"authors":2254,"publicationType":1010,"publisherRelationship":2354,"citationCount":28,"citationInfo":28,"publishDate":2409,"publishYear":2410,"citationAnalyzeStatus":878,"lastCitationAnalyze":28,"indexDatabases":2411,"openAccess":28,"references":28,"isForceReanalyzing":1069},"004c592c-c28e-492c-83d3-f2935a46c436","2024-01-16T08:10:06.574+00:00","2024-12-06T17:41:51.382+00:00",[],"Matrix-metalloproteinase-MMP-2-9-and-tissue-inhibitor-TIMP-1-2-activity-in-tear-samples-of-pediatric-type-1-diabetic-patients",{"abstract":2245,"title":2247,"references":2249,"doi":2251},{"EN":2246},"The presence of matrix metalloproteinase (MMP-2, -9) and tissue inhibitor (TIMP-1, -2) activity in tear samples of pediatric type 1 diabetes mellitus (DM) patients and potential correlations with clinical parameters (Schirmer testing, glycosylated hemoglobin-HBA1C) were investigated. Tear samples from the right eyes of 27 type 1 DM patients and 17 healthy control subjects were included in this study. The MMP gelatinolytic activity was determined by gelatin zymography analysis using sodium dodecyl sulphate–polyacrylamide gel electrophoresis (SDS-PAGE), while MMP and TIMP concentrations (in ng\u002Fml) were quantified in tears of type 1 diabetic patients and healthy controls, with the use of enzyme-linked immunosorbent assay (ELISA). MMP-9, TIMP-1, -2 levels, MMP-9\u002FTIMP-1, and MMP-9\u002FTIMP-2 ratios in the patient group were significantly elevated. There was a significant correlation between TIMP-2 and HBA1C values, as well as between MMP-2 and MMP-9. Significant correlations between TIMP-2 and HBA1C and between Schirmer test results and HBA1C were revealed. Significant increase in tear MMP and TIMP levels in pediatric type 1 diabetic patients may be suggestive of disease progression and localized pathologic remodelling. Further studies are required in order to ascertain whether MMPs and TIMPs could be employed as indicators of early disease progression.",{"EN":2248},"Matrix metalloproteinase (MMP-2, -9) and tissue inhibitor (TIMP-1, -2) activity in tear samples of pediatric type 1 diabetic patients",{"VOID":2250},"Schultz RO, Van Horn DL, Peters MA, Klewin KM, Schutten WH (1981) Diabetic keratopathy. Trans Am Ophthalmol Soc 79:180–199\nSchultz RO, Matsuda M, Yee RW, Edelhauser HF, Schultz KJ (1984) Corneal endothelial changes in type I and type II diabetes mellitus. Am J Ophthalmol 98:401–410\nFriend J, Thoft RA (1984) The diabetic cornea. Int Ophthalmol Clin 24:111–123\nHerse PR (1988) A review of manifestations of diabetes mellitus in the anterior eye and cornea. Am J Optom Physiol Opt 65:224–230\nLiu J, Shi B, He S, Yao X, Willcox MD, Zhao Z (2010) Changes to tear cytokines of type 2 diabetic patients with or without retinopathy. Mol Vis 16:2931–2938\nNepp J, Abela C, Polzer I, Derbolav A, Wedrich A (2000) Is there a correlation between the severity of diabetic retinopathy and keratoconjunctivitis sicca? Cornea 19:487–491\nSethi CS, Bailey TA, Luthert PJ, Chong NH (2000) Matrix metalloproteinase biology applied to vitreoretinal disorders. Br J Ophthalmol 84:654–666\nSivak JM, Fini E (2002) MMPs in the eye: emerging roles for matrix metallo-proteinases in ocular pathology. Prog Retin Eye Res 21:1–14\nFowlkes JL, Serra DM, Bunn RC, Thrailkill KM, Enghild JJ, Nagase H (2004) Regulation of insulin-like growth factor (IGF)-I action by matrix metalloproteinase-3 involves selective disruption of IGF-I\u002FIGF-binding protein-3 complexes. Endocrinology 145:620–626\nBorden P, Heller RA (1997) Transcriptional control of matrix metalloproteinases and the tissue inhibitors of matrix metalloproteinases. Crit Rev Euk Gene Exp 7:159–178\nClark IM, Swingler TE, Sampieri CL, Edwards DR (2008) The regulation of matrix metalloproteinases and their inhibitors. Int J Biochem Cell Biol 40:1362–1378\nMäkitalo L, Kolho KL, Karikoski R, Anthoni H, Saarialho-Kere U (2010) Expression profiles of matrix metalloproteinases and their inhibitors in colonic inflammation related to pediatric inflammatory bowel disease. Scand J Gastroenterol 45:862–871\nMusiał K, Zwolińska D (2011) Matrix metalloproteinases (MMP-2, -9) and their tissue inhibitors (TIMP-1, 2) as novel markers of stress response and atherogenesis in children with chronic kidney disease (CKD) on conservative treatment. Cell Stress Chaperones 16:97–103\nPapakonstantinou E, Dionyssopoulos A, Aletras AJ, Pesintzaki C, Minas A, Karakiulakis G (2004) Expression of matrix metalloproteinases and their endogenous tissue inhibitors in skin lesions from patients with tuberous sclerosis. J Am Acad Dermatol 51:526–533\nSenzaki H (2006) The pathophysiology of coronary artery aneurysms in Kawasaki disease: role of matrix metalloproteinases. Arch Dis Child 91:847–851\nBoden G, Song W, Pashko L, Kresge K (2008) In vivo effects of insulin and free fatty acids on matrix metalloproteinases in rat aorta. Diabetes 57:476–483\nDerosa G, D’Angelo A, Tinelli C, Devangelio E, Consoli A, Miccoli R, Penno G, Del Prato S, Paniga S, Cicero AF (2007) Evaluation of metalloproteinase 2 and 9 levels and their inhibitors in diabetic and healthy subjects. Diabetes Metab 33:129–134\nYang C, Zhu P, Yan L, Chen L, Meng R, Lao G (2009) Dynamic changes in matrix metalloproteinase 9 and tissue inhibitor of metalloproteinase 1 levels during wound healing in diabetic rats. J Am Pediatr Med Assoc 99:489–496\nAcera A, Rocha G, Vecino E, Durán JA (2008) Inflammatory markers in the tears of patients with ocular surface disease. Ophthalmic Res 40:315–321\nLema I, Sobrino T, Durán JA, Díez-Feijoo E (2009) Subclinical keratoconus and inflammatory molecules from tears. Br J Ophthalmol 93:820–824\nRohini G, Murugeswari P, Prajna NV, Lalitha P, Muthukkaruppan V (2007) Matrix metalloproteinases (MMP-8, MMP-9) and the tissue inhibitors of metalloproteinases (TIMP-1, TIMP-2) in patients with fungal keratitis. Cornea 26:207–211\nGunczler P, Lanes R, Soros A, Verdu L, Ramon Y, Guevara B, Beer N (2006) Coronary artery calcification, serum lipids, lipoproteins, and peripheral inflammatory markers in adolescents and young adults with type 1 diabetes. J Pediatr 149:320–323\nRysz J, Banach M, Stolarek RA, Pasnik J, Cialkowska-Rysz A, Koktysz R, Piechota M, Baj Z (2007) Serum matrix metalloproteinases MMP-2 and MMP-9 and metalloproteinase tissue inhibitors TIMP-1 and TIMP-2 in diabetic nephropathy. J Nephrol 20:444–452\nThrailkill KM, Bunn RC, Moreau CS, Cockrell GE, Simpson PM, Coleman HN, Frindik JP, Kemp SF, Fowlkes JL (2007) Matrix metalloproteinase-2 dysregulation in type 1 diabetes. Diabetes Care 30:2321–2326\nThrailkill KM, Bunn RC, Fowlkes JL (2009) Matrix metalloproteinases: their potential role in the pathogenesis of diabetic nephropathy. Endocrine 35:1–10\nAmerican Diabetes Association(2009) Executive summary: standards of medical care in diabetes—2009. Diabetes Care 32:S6–S12\nSymeonidis C, Papakonstantinou E, Souliou E, Karakiulakis G, Dimitrakos SA, Diza E (2011) Correlation of matrix metalloproteinase levels with the grade of proliferative vitreoretinopathy in the subretinal fluid and vitreous during rhegmatogenous retinal detachment. Acta Ophthalmol 89:339–345\nDeath AK, Fisher EJ, McGrath KC, Yue DK (2003) High glucose alters matrix metalloproteinase expression in two key vascular cells: potential impact on atherosclerosis in diabetes. Atherosclerosis 168:263–269\nPugliese G, Pricci F, Pugliese F, Mene P, Lenti L, Andreani D, Galli G, Casini A, Bianchi S, Rotella CM (1994) Mechanisms of glucose-enhanced extracellular matrix accumulation in rat glomerular mesangial cells. Diabetes 43:478–490\nGoebbels M (2000) Tear secretion and tear film function in insulin dependent diabetics. Br J Ophthalmol 84:19–21\nGrus FH, Sabuncuo P, Dick HB, Augustin AJ, Pfeiffer N (2002) Changes in the tear proteins of diabetic patients. BMC Ophthalmol 2:4\nDerosa G, Avanzini MA, Geroldi D, Fogari R, Lorini R, De Silvestri A, Tinelli C, Rondini G, d’Annunzio G (2005) Matrix metalloproteinase 2 may be a marker of microangiopathy in children and adolescents with type 1 diabetes mellitus. Diabetes Res Clin Pract 70:119–125\nParshley DE, Bradley JMB, Samples JR, Van Buskirk EM, Acott TS (1995) Early changes in matrix metalloproteinases and inhibitors after in vitro laser treatment to the trabecular meshwork. 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