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Journal of Medicine and Pharmacy","Tạp chí Y Dược học Cần Thơ",{"EN":487,"VI":488},"\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">04\u002F10\u002F2015 Ministry of Information and Communications allowed Can Tho journal of medicine and pharmacy to operate (102 \u002FGP-BTTTT)\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">07\u002F16\u002F2015 Can Tho journal of medicine and pharmacy is internationally recognized: ISSN 2354-1210\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">In 2016, The journal has been included in the list of medical science journals by The State Council for professorship which is awarded a work score of 0-0.5 points for a published article.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Can Tho Journal of Medicine and Pharmacy welcome original works that haven’t been submitted or published in other medical journals. Posts must contain content related to one of the journal’s categories.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The content published\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The journal is divided into 3 categories:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Scientific research article: are valuable scientific works, which have been researched and accepted.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Overview of medicine, biology and pharmacy: serving the objective of continuing training in the fields of medicine, biology and pharmacy; to systematize classical and modern knowledge.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Update information on new knowledge about medicine, biology, pharmacy in the country and in the world.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Scope\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Publication and introduction of scientific research in the fields:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Medicine (internal medicine, surgery, pediatrics, obstetrics and gynecology, odonto-stomatology, laboratory, oncology, traditional medicine, nursing).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Biology (genetics, biotechnology).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Pharmacology (pharmaceutics, drug quality analysis-control, synthetic pharmaceutical chemistry, biochemistry, pharmacognosy, botany, clinical pharmacy).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- To enhance the quality of undergraduate, postgraduate education, scientifically researching and meet the necessary treatment in hospital.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Introducing the updated domestic and oversea information about science technology to promote scientific research and exchanging technology in local, other universities.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Exchanging pharmaceutical and medical information for social health developing in the Mekong Delta and Vietnam.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The object\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Postgraduate students, student of Can Tho University of Medicine and Pharmacy, scientists from schools, research institutes, hospitals, health centers, pharmaceutical companies of the Mekong Delta; other provinces and regions in Vietnam and other country.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Address\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Headquarters of Can Tho Journal of Medicine and Pharmacy, located Scientific Research and International Cooperation Office: 179 Nguyen Van Cu Street, An Khanh Ward, Ninh Kieu District, Can Tho City, Vietnam.\u003C\u002Fspan>\u003C\u002Fp>","\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Ngày 16\u002F7\u002F2015, Tạp chí Y Dược học Cần Thơ được cấp chỉ số quốc tế: ISSN 2354-1210.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 4\u002F2016, Tạp chí đã được Hội đồng Giáo sư ngành Y đưa vào danh sách các tạp chí khoa học Y học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Năm 2020 Tạp chí Y Dược học Cần Thơ đã được phê duyệt vào danh mục của các Hội đồng Giáo sư ngành Dược học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ ra 12 số\u002Fnăm, 180-200 trang\u002Fsố.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 12\u002F2022 Tạp chí Y Dược học Cần Thơ là thành viên của hệ thống Crossref và từ tháng 01\u002F2023 tạp chí thực hiện bình duyệt online kín 2 chiều nhằm tăng tính minh bạch, tin cậy của các công trình nghiên cứu khoa học và đảm bảo tốt nhất chất lượng khoa học của bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ, mục đích và phạm vi của tạp chí\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ và mục đích hoạt động của tạp chí: xuất bản nhằm mục đích phổ biến kết quả từ các đề tài nghiên cứu khoa học; giao lưu trao đổi khoa học, chia sẻ kinh nghiệm, học tập, đồng thời cập nhật thông tin khoa học mới trong các lĩnh vực y, sinh, dược học trong và ngoài nước.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phạm vi của tạp chí: Tạp chí xuất bản được chia thành 3 chuyên mục: (i) Bài báo nghiên cứu khoa học là kết quả công trình nghiên cứu khoa học có giá trị đã được triển khai nghiên cứu, (ii) Bài tổng quan y, sinh, dược học: phục vụ mục tiêu đào tạo liên tục trong lĩnh vực y, sinh, dược học; nhằm hệ thống hóa những kiến thức kinh điển và hiện đại; (iii) Thông tin cập nhật kiến thức mới về y, sinh, dược học trong nước và trên thế giới.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Chính sách truy cập mở\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ áp dụng chính sách truy cập mở đối với các bài báo đã xuất bản đến với độc giả, nhằm mở rộng cơ hội tiếp cận các kết quả nghiên cứu chất lượng cao và tăng cường trao đổi kiến thức. Tạp chí đăng tải trực tuyến (miễn phí) toàn văn các bài báo được công bố trên website của Tạp chí (https:\u002F\u002Ftapchi.ctump.edu.vn).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đạo đức xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ cam kết tuân thủ đạo đức xuất bản phù hợp với các hướng dẫn và tiêu chuẩn của the Committee on Publication Ethics (COPE), tuân thủ các nguyên tắc của COPE’s Core Practices, Best Practices Guidelines for Journal Editors và Guidelines on Good Publication Practices.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Bản thảo bài báo chỉ được chấp nhận khi được tác giả chịu trách nhiệm chính cam kết các nội dung sau: Các nội dung của bản thảo chưa được đăng tải toàn bộ hoặc một phần ở các tạp chí khác; Tất cả các tác giả đều có đóng góp một cách đáng kể vào quá trình nghiên cứu hoặc chuẩn bị bản thảo và cùng chịu trách nhiệm về các nội dung của bản thảo; Tuân thủ các biện pháp đảm bảo đạo đức nghiên cứu (ví dụ thỏa thuận đồng ý tham gia nghiên cứu).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Cam kết bảo mật\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí cam kết thực hiện và tuân thủ các quy định của luật và các văn bản hướng dẫn liên quan đến bảo mật thông tin cá nhân trên không gian mạng. Các thông tin mà người dùng (tác giả, độc giả, biên tập viên, người phản biện) nhập vào các biểu mẫu trên Hệ thống Quản lý xuất bản trực tuyến của tạp chí chỉ được sử dụng vào các mục đích đã được tuyên bố rõ ràng và sẽ không được cung cấp cho bất kỳ bên thứ ba nào khác, hay dùng vào bất kỳ mục đích nào khác.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phí gửi bài\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng bài: 1.000.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng nhanh: 1.500.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với tác giả là cán bộ viên chức thuộc Trường Đại học Y Dược Cần Thơ thì được hỗ trợ 50% lệ phí gửi đăng bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với sinh viên thực hiện đề tài nghiên cứu khoa học cấp trường được hỗ trợ 100% lệ phí đăng bài ( Tác giả gửi đính kèm “ Quyết định về việc giao tổ chức thực hiện đề tài nghiên cứu khoa học cấp Trường của sinh viên”).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Hình thức nộp lệ phí:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Tiền mặt:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Nộp trực tiếp tại Phòng Tài chính - Kế toán, Trường Đại học Y Dược Cần Thơ, số 179 Nguyễn Văn Cừ, P. An Khánh, Q. Ninh Kiều, thành phố Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Chuyển khoản:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tên Tài khoản: Trường ĐHYD Cần Thơ, Số TK: 0111000115668, tại ngân hàng Vietcombank chi nhánh Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Thời gian: Áp dụng từ ngày 01\u002F02\u002F2023.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">* Phí gửi bài không được hoàn trả khi bài viết bị từ chối hoặc tác giả xin rút bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Quy trình phản biện bài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ thực hiện quy trình phản biện kín hai chiều nghiêm ngặt. Danh tính của những người phản biện không được tiết lộ cho các tác giả và ngược lại. Quy trình thẩm định bài báo đăng gồm các bước sau:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tiếp nhận bản thảo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tác giả liên hệ gửi bản thảo đến Tạp chí qua hệ thống trực tuyến tại website: https:\u002F\u002Ftapchi.ctump.edu.vn. Hướng dẫn về cách đăng ký, gửi bài và chuẩn bị bản thảo được cung cấp trên website của Tạp chí.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sàng lọc sơ bộ\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sau khi Tòa soạn nhận được bài báo của tác giả, Ban Thư ký sẽ tiến hành kiểm tra sơ bộ bài báo (các yêu cầu về nội dung và hình thức). Những bài báo không đúng quy cách hoặc có nội dung không phù hợp hoặc vi phạm bản quyền sẽ bị từ chối (Ban Thư ký thông báo phản hồi đến tác giả trong vòng 1 tuần). Những bài báo đủ điều kiện, được Ban Thư ký tòa soạn chuyển đến Ban Biên tập có cùng chuyên môn với nội dung bài báo để đề xuất người phản biện. Thời gian kể từ khi Ban Biên tập nhận bài báo đến khi đề xuất người phản biện bài báo chậm nhất là 5 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Vòng phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký gửi bài và yêu cầu phản biện đến 02 phản biện độc lập.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Các phản biện gởi nhận xét cho Ban Thư ký. Thời gian từ khi gửi bài cho phản biện đến khi nhận ý kiến của phản biện tối đa là 20 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xử ký kết quả phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Nếu ý kiến đồng ý cho đăng và không cần chỉnh sửa, Ban Thư ký tiếp tục đăng bài theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Nếu ý kiến đồng ý đăng và cần chỉnh sửa, Ban Thư ký sẽ thông tin đến tác giả chỉnh sửa theo yêu cầu của người phản biện. Thời gian chỉnh sửa và gửi lại kéo dài không quá 2 tuần, từ khi tác giả bài báo nhận được thông tin (Quá trình này có thể lặp lại tối đa 2 lần\u002F1 bài báo). Khi có sự thống nhất, đồng ý của người phản biện; bài báo được tiếp tục đăng theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Những bài báo có chất lượng không đạt yêu cầu, cả 2 phản biện không đồng ý cho đăng sẽ bị Tòa soạn từ chối đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký tổng hợp các bản thảo đã được tác giả hoàn thiện sau thẩm định trình Ban Biên tập xem xét, Tổng Biên tập phê duyệt, quyết định bài đăng theo các tiêu chí: sự phù hợp nội dung với tôn chỉ và mục đích, thể loại bài viết (ưu tiên các bài có bài có nghiên cứu chuyên sâu, hàm lượng khoa học cao), đóng góp mới bài báo, bài báo được ưu tiên đăng trong số gần nhất của Tạp chí theo thứ tự: tính thời sự, chất lượng bài báo và thời gian gửi bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Ban Biên tập và Ban Thư ký biên tập bản thảo, chế bản, đọc rà soát lỗi. Thời gian hoàn thành từ 10-15 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Ban Thư ký có trách nhiệm thông báo cho tác giả bài báo (bằng e-mail) về tình hình phê duyệt bài báo, thời gian, số kỳ, tập xuất bản bài báo theo qui định.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">4. Danh sách bài báo theo số Tạp chí được in ấn và phát hành trong năm định kỳ được công bố chính thức trên website: https:\u002F\u002Ftapchi.ctump.edu.vn\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>",{"VOID":490},"wcQ1uqwAAAAJ","2023-05-30T08:17:21.868+00:00",[],[494],{"id":495,"createTime":28,"updateTime":28,"relativeEntities":496,"slug":28,"properties":497,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":507,"parentIds":508,"statistic":28},"6413896b-eca9-442b-a73f-182a58a0ce40",[],{"title":498,"address":501,"country":504,"abbreviation":505},{"EN":499,"VI":500},"Can Tho University of Medicine and Pharmacy","Trường Đại học Y Dược Cần Thơ",{"EN":502,"VI":503},"No 179, Nguyen Van Cu street, An Khanh ward, Ninh Kieu district, Can Tho city, Vietnam","Số 179, đường Nguyễn Văn Cừ, phường An Khánh, quận Ninh Kiều, thành phố Cần Thơ, Việt Nam",{"VOID":15},{"VOID":506},"ctump","http:\u002F\u002Fwww.ctump.edu.vn\u002F",[],[],"https:\u002F\u002Ftapchi.ctump.edu.vn\u002Findex.php\u002Fctump",{"impactFactor":32,"impactFactorByYear":512,"i10Index":32,"i10IndexLast5Year":32,"totalPublication":514,"totalPublicationByYear":515,"totalCitation":520,"totalCitationByYear":521,"totalCitationPerPublication":108,"totalCitationPerPublicationByYear":523,"hindexLast5Year":45,"hindex":45},{"2022":513,"2023":111,"2024":106},0.01,1556,{"2020":47,"2021":516,"2022":517,"2023":518,"2024":519,"2025":122},57,306,801,358,161,{"2021":146,"2022":280,"2023":522},99,{"2021":524,"2022":318,"2023":104},0.23,{"impactFactor":28,"impactFactorByYear":28,"i10Index":123,"i10IndexLast5Year":123,"totalPublication":526,"totalPublicationByYear":527,"totalCitation":526,"totalCitationByYear":528,"totalCitationPerPublication":40,"totalCitationPerPublicationByYear":531,"hindexLast5Year":49,"hindex":49},476,{"0":205,"2019":123,"2021":139,"2022":459,"2023":451,"2024":357,"2025":49,"2026":48},{"2021":42,"2022":123,"2023":161,"2024":529,"2025":360,"2026":530},136,83,{"2021":105,"2022":513,"2023":532,"2024":127,"2025":533,"2026":534},0.62,25.43,13.83,{"id":536,"createTime":537,"updateTime":382,"relativeEntities":538,"slug":539,"properties":540,"entityType":25,"verifyStatus":26,"verifyTime":28,"verifyNote":28,"languages":552,"translateLanguages":28,"viewCount":133,"subjectFields":553,"manageAffiliations":554,"indexDatabases":555,"url":556,"thumbnailPath":557,"statistic":558,"gsStatistic":594,"type":55,"analyzePriority":28},"6984a56a-db70-403b-9cc4-4013e1ceaffa","2023-05-09T06:47:40.346+00:00",[],"T%E1%BA%A1p%20ch%C3%AD%20Nghi%C3%AAn%20c%E1%BB%A9u%20n%C6%B0%E1%BB%9Bc%20ngo%C3%A0i",{"country":541,"issn":542,"title":544,"introduce":547,"gsId":550},{"VOID":15},{"VOID":543},"25252445",{"EN":545,"VI":546},"VNU Journal of Foreign Studies","Tạp chí Nghiên cứu nước ngoài",{"EN":548,"VI":549},"{\"ops\":[{\"insert\":\"\\n\\nThe \\n\"},{\"attributes\":{\"italic\":true},\"insert\":\"VNU Journal of Science\"},{\"insert\":\"\\n was established in 1985 for the publication of national and international research papers in all fields of natural sciences and technology, social sciences and humanities. 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Subjects older than 12 years with massive PE requiring pleural drainage were included in this study. Hemodynamic parameters were collected using transthoracic echocardiography at pre-drainage, immediately post-drainage, and 24 h after drainage. We enrolled 47subjects in this prospective study from June 9, 2015 to September 18, 2016 in Beijing Chaoyang Hospital and 28 subjects were analyzed finally. Draining large-volume PE led to a progressive increase in left ventricular end-diastolic volume index, left atrial volume index, right ventricular area, right atrial area, left ventricular ejection fraction, stroke volume, and tricuspid annular plane systolic excursion, both immediately (P \u003C 0.05) and 24 h after drainage (P \u003C 0.05). The cardiac diastolic measurement ratios of early-transmitral flow velocity to diastolic mitral annular velocity and myocardial performance index decreased significantly following drainage (P \u003C 0.05). More parameters were influenced by left-sided PE drainage. The correlation between effusion volume and changes in echocardiographic measurements was not statistically significant. Improved preload, and systolic and diastolic function is pivotal for hemodynamic change after draining large PE volumes. Subjects experienced improved cardiac hemodynamics following PE drainage, underlining the beneficial therapeutic and subjective effects.",{"EN":987},"Improved heart hemodynamics after draining large-volume pleural effusion: a prospective cohort study",{"VOID":989},"Mitrouska I, Klimathianaki M, Siafakas NM. Effects of pleural effusion on respiratory function. Can Respir J. 2004;11(7):499–503.\nEstenne M, Yernault JC, De Troyer A. Mechanism of relief of dyspnea after thoracocentesis in patients with large pleural effusions. Am J Med. 1983;74(5):813–9.\nLight RW, Stansbury DW, Brown SE. The relationship between pleural pressures and changes in pulmonary function after therapeutic thoracentesis. Am Rev Respir Dis. 1986;133(4):658–61.\nKaplan LM, Epstein SK, Schwartz SL, Cao QL, Pandian NG. Clinical, echocardiographic, and hemodynamic evidence of cardiac tamponade caused by large pleural effusions. Am J Respir Crit Care Med. 1995;151(3 Pt 1):904–8.\nKopterides P, Lignos M, Papanikolaou S, Papadomichelakis E, Mentzelopoulos S, Armaganidis A, et al. Pleural effusion causing cardiac tamponade: report of two cases and review of the literature. Heart Lung. 2006;35(1):66–7.\nBroch O, Gruenewald M, Renner J, Meybohm P, Schöttler J, Heß K, et al. Dynamic and volumetric variables reliably predict fluid responsiveness in a porcine model with pleural effusion. PLoS One. 2013;8(2):e56267.\nHermansen JF, Juhl-Olsen P, Frederiksen CA, Christiansen LK, Hørlyck A, Sloth E. Drainage of large pleural effusions increases left ventricular preload. J Cardiothorac Vasc Anesth. 2014;28(4):885–9.\nSadaniantz A, Anastacio R, Verma V, Aprahamian N. The incidence of diastolic right atrial collapse in patients with pleural effusion in the absence of pericardial effusion. Echocardiography. 2003;20(3):211–5.\nWang F, Wang Z, Tong Z, Xu L, Wang X, Wu Y. A pilot study of autofluorescence in the diagnosis of pleural disease. Chest. 2015;147(5):1395–400.\nWang XJ, Yang Y, Wang Z, Xu LL, Wu YB, Zhang J, et al. Efficacy and safety of diagnostic thoracoscopy in undiagnosed pleural effusions. Respiration. 2015;90(3):251–5.\nLang RM, Badano LP, Mor-Avi V, Afilalo J, Armstrong A, Ernande L, et al. Recommendations for cardiac chamber quantification by echocardiography in adults: an update from the American Society of Echocardiography and the European Association of Cardiovascular Imaging. J Am Soc Echocardiogr. 2015;28(1):1–39.\nLight RW, Jenkinson SG, Minh VD, George RB. Observations on pleural fluid pressures as fluid is withdrawn during thoracentesis. Am Rev Respir Dis. 1980;121(5):799–804.\nVaska K, Wann LS, Sagar K, Klopfenstein HS. Pleural effusion as a cause of right ventricular diastolic collapse. Circulation. 1992;86(2):609–17.\nChidambaram S, Sangareddi V, Ganesan G, Dhandapani VE, Ravi MS, Meenakshi K, et al. An echocardiographic assessment of cardiovascular hemodynamics in patients with large pleural effusion. Indian Heart J. 2013;65(6):666–70.\nGraf J, Formenti P, Santos A, Gard K, Adams A, Tashjian J, et al. Pleural effusion complicates monitoring of respiratory mechanics. Crit Care Med. 2011;39(10):2294–9.\nAhmed SH, Ouzounian SP, Dirusso S, Sullivan T, Savino J, Del Guercio L. Hemodynamic and pulmonary changes after drainage of significant pleural effusions in critically ill, mechanically ventilated surgical patients. J Trauma. 2004;57(6):1184–8.\nLan CC, Hsu HH, Wu CP, Lee SC, Peng CK, Chang H. Influences of pleural effusion on respiratory mechanics, gas exchange, hemodynamics, and recruitment effects in acute respiratory distress syndrome. J Surg Res. 2014;186(1):346–53.\nNishida O, Arellano R, Cheng DC, DeMajo W, Kavanagh BP. Gas exchange and hemodynamics in experimental pleural effusion. Crit Care Med. 1999;27(3):583–7.\nChang SC, Shiao GM, Perng RP. Postural effect on gas exchange in patients with unilateral pleural effusions. Chest. 1989;96(1):60–3.\nRomero S, Martin C, Hernandez L, Arriero JM, Benito N, Gil J. Effect of body position on gas exchange in patients with unilateral pleural effusion: influence of effusion volume. 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questionnaires are commonly used in epidemiology, but may be susceptible to misclassification, especially if answers are given on behalf of others, e.g. children or parents. The aim was to determine agreement and analyse predictors of disagreement in parents’ reports of offspring asthma, and in offspring reports of parents’ asthma. In the Respiratory Health in Northern Europe, Spain and Australia (RHINESSA) generation study, 6752 offspring (age range 18–51 years) and their parents (age range 39–66 years) reported their own and each other’s asthma status. Agreement between asthma reports from offspring and parents was determined by calculating sensitivity, specificity, positive and negative predictive value and Cohen’s kappa. The participants’ own answers regarding themselves were defined as the gold standard. To investigate predictors for disagreement logistic regression analyses were performed to obtain odds ratios (OR) with 95% confidence intervals (CI) for sex, smoking status, education, comorbidity and severity of asthma. Agreement was good for parental report of offspring early onset asthma (\u003C 10 years, Cohen’s kappa 0.72) and moderate for offspring later onset asthma (Cohen’s kappa 0.46). Specificity was 0.99 for both, and sensitivity was 0.68 and 0.36, respectively. For offspring report of maternal and paternal asthma the agreement was good (Cohen’s kappa 0.69 and 0.68), specificity was 0.96 and 0.97, and sensitivity was 0.72 and 0.68, respectively. The positive predictive value (PPV) was lowest for offspring report of maternal asthma (0.75), and highest for parents’ report of early onset asthma in the offspring (0.83). The negative predictive value (NPV) was high for all four groups (0.94–0.97). In multivariate analyses current smokers (OR = 1.46 [95% CI 1.05, 2.02]) and fathers (OR = 1.31 [95% CI 1.08, 1.59]) were more likely to report offspring asthma incorrectly. Offspring wheeze was associated with reporting parental asthma incorrectly (OR = 1.60 [95% CI 1.21, 2.11]), both under- and over reporting. Asthma reports across generations show moderate to good agreement, making information from other generations a useful tool in the absence of direct reports.",{"EN":1192},"Agreement in reporting of asthma by parents or offspring – the RHINESSA generation study",{"VOID":1194},"Strippoli MP, Silverman M, Michel G, Kuehni CE. A parent-completed respiratory questionnaire for 1-year-old children: repeatability. Arch Dis Child. 2007;92(10):861–5.\nBeasley R, Semprini A, Mitchell EA. Risk factors for asthma: is prevention possible? Lancet. 2015;386(9998):1075–85.\nSoubry A, Hoyo C, Jirtle RL, Murphy SK. A paternal environmental legacy: evidence for epigenetic inheritance through the male germ line. BioEssays. 2014;36(4):359–71.\nHansen S, Strom M, Maslova E, Mortensen EL, Granstrom C, Olsen SF. A comparison of three methods to measure asthma in epidemiologic studies: results from the Danish national birth cohort. PLoS One. 2012;7(5):e36328.\nAsher MI, Keil U, Anderson HR, Beasley R, Crane J, Martinez F, et al. International study of asthma and allergies in childhood (ISAAC): rationale and methods. Eur Respir J. 1995;8(3):483–91.\nMinasyan A, Babajanyan A, Campbell DE, Nanan R. Validation of a comprehensive early childhood allergy questionnaire. Pediatr Allergy Immunol. 2015;26(6):522–9.\nKim MH, Kwon JW, Kim HB, Song Y, Yu J, Kim WK, et al. Parent-reported ISAAC written questionnaire may underestimate the prevalence of asthma in children aged 10-12 years. Pediatr Pulmonol. 2012;47(1):36–43.\nHansen TE, Evjenth B, Holt J. Validation of a questionnaire against clinical assessment in the diagnosis of asthma in school children. J Asthma. 2015;52(3):262–7.\nPekkanen J, Pearce N. Defining asthma in epidemiological studies. Eur Respir J. 1999;14(4):951–7.\nBrozek G, Zejda J, Farnik M, Wypych-Slusarska A, Skoczynski S. Validation of simple and composed screening test of childhood asthma. Eur Respir J. 2016;48(suppl 60). https:\u002F\u002Fdoi.org\u002F10.1183\u002F13993003.congress-2016.PA4231.\nSundblad GM, Saartok T, Engstrom LM. Child-parent agreement on reports of disease, injury and pain. BMC Public Health. 2006;6:276.\nMiller JE. Predictors of asthma in young children: does reporting source affect our conclusions? Am J Epidemiol. 2001;154(3):245–50.\nSole D, Vanna AT, Yamada E, Rizzo MC, Naspitz CK. International study of asthma and allergies in childhood (ISAAC) written questionnaire: validation of the asthma component among Brazilian children. J Investig Allergol Clin Immunol. 1998;8(6):376–82.\nBraun-Fahrlander C, Gassner M, Grize L, Minder CE, Varonier HS, Vuille JC, et al. Comparison of responses to an asthma symptom questionnaire (ISAAC core questions) completed by adolescents and their parents. SCARPOL-Team. Swiss Study on Childhood Allergy and Respiratory Symptoms with respect to Air Pollution. Pediatr Pulmonol. 1998;25(3):159–66.\nLi YF, Langholz B, Salam MT, Gilliland FD. Maternal and grandmaternal smoking patterns are associated with early childhood asthma. Chest. 2005;127(4):1232–41.\nMiller LL, Henderson J, Northstone K, Pembrey M, Golding J. Do grandmaternal smoking patterns influence the etiology of childhood asthma? Chest. 2014;145(6):1213–8.\nSvanes C, Koplin J, Skulstad SM, Johannessen A, Bertelsen RJ, Benediktsdottir B, et al. Father's environment before conception and asthma risk in his children: a multi-generation analysis of the respiratory health in northern Europe study. Int J Epidemiol. 2017;46(1):235–45.\nArshad SH, Karmaus W, Zhang H, Holloway JW. Multigenerational cohorts in patients with asthma and allergy. J Allergy Clin Immunol. 2017;139(2):415–21.\nLodge CJ, Braback L, Lowe AJ, Dharmage SC, Olsson D, Forsberg B. Grandmaternal smoking increases asthma risk in grandchildren: a nationwide Swedish cohort. Clin Exp Allergy. 2018;48(2):167-74.\nJohannessen A, Verlato G, Benediktsdottir B, Forsberg B, Franklin K, Gislason T, et al. Longterm follow-up in European respiratory health studies - patterns and implications. BMC Pulm Med. 2014;14:63.\nToren K, Gislason T, Omenaas E, Jogi R, Forsberg B, Nystrom L, et al. A prospective study of asthma incidence and its predictors: the RHINE study. Eur Respir J. 2004;24(6):942–6.\nD A. Practical statistics for medical research. London: Chapman and Hall; 1991.\nBrunekreef B, Groot B, Rijcken B, Hoek G, Steenbekkers A, de Boer A. Reproducibility of childhood respiratory symptom questions. Eur Respir J. 1992;5(8):930–5.\nYang CL, To T, Foty RG, Stieb DM, Dell SD. Verifying a questionnaire diagnosis of asthma in children using health claims data. BMC Pulm Med. 2011;11:52.\nHederos CA, Hasselgren M, Hedlin G, Bornehag CG. Comparison of clinically diagnosed asthma with parental assessment of children's asthma in a questionnaire. Pediatr Allergy Immunol. 2007;18(2):135–41.\nCornish RP, Henderson J, Boyd AW, Granell R, Van Staa T, Macleod J. Validating childhood asthma in an epidemiological study using linked electronic patient records. BMJ Open. 2014;4(4):e005345.\nVissing NH, Jensen SM, Bisgaard H. Validity of information on atopic disease and other illness in young children reported by parents in a prospective birth cohort study. BMC Med Res Methodol. 2012;12:160.\nLund KE, Helgason AR. Environmental tobacco smoke in Norwegian homes, 1995 and 2001: changes in children’s exposure and parents attitudes and health risk awareness. Eur J Pub Health. 2005;15(2):123–7.\nKulig M, Bergmann R, Edenharter G, Wahn U, Multicenter Allergy Study Group. Does allergy in parents depend on allergy in their children? Recall bias in parental questioning of atopic diseases. J Allergy Clin Immunol. 2000;105(2 Pt 1):274–8.\nDanell CS, Bergstrom A, Wahlgren CF, Hallner E, Bohme M, Kull I. Parents and school children reported symptoms and treatment of allergic disease differently. J Clin Epidemiol. 2013;66(7):783–9.\nNilsen RM, Suren P, Gunnes N, Alsaker ER, Bresnahan M, Hirtz D, et al. Analysis of self-selection bias in a population-based cohort study of autism spectrum disorders. Paediatr Perinat Epidemiol. 2013;27(6):553–63.\nFuru K, Karlstad O, Skurtveit S, Haberg SE, Nafstad P, London SJ, et al. High validity of mother-reported use of antiasthmatics among children: a comparison with a population-based prescription database. J Clin Epidemiol. 2011;64(8):878–84.\nde Marco R, Cerveri I, Bugiani M, Ferrari M, Verlato G. An undetected burden of asthma in Italy: the relationship between clinical and epidemiological diagnosis of asthma. Eur Respir J. 1998;11(3):599–605.",{"VOID":1196},"10.1186\u002Fs12890-018-0687-4","https:\u002F\u002Fbmcpulmmed.biomedcentral.com\u002Farticles\u002F10.1186\u002Fs12890-018-0687-4",[1199,1214,1236,1251,1264,1279,1294,1309,1324,1339,1354,1367,1382,1406,1421,1445,1458],{"id":1200,"sortIndex":32,"researcher":28,"roles":1201,"affiliations":1202,"properties":1211,"displayName":1213,"givenName":28,"familyName":28},"1f5c4833-dd39-4694-a8a4-9a6c54f693af",[1000],[1203],{"id":1204,"sortIndex":32,"affiliation":1205,"properties":28},"d1bac50c-c652-4e2e-8dc6-2b9ac527d2e3",{"id":1204,"createTime":28,"updateTime":28,"relativeEntities":1206,"slug":28,"properties":1207,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1210,"statistic":28},[],{"title":1208},{"VI":1209},"Department of Occupational Medicine, Haukeland University Hospital, Bergen, Norway",[],{"title":1212},{"VI":1213},"Ingrid N. 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most commonly isolated organisms in a parapneumonic effusion include S. pneumoniae, H. influenzae, and S. aureus. If unusual organisms are isolated from the pleural space, further investigation is warranted to locate the primary source. We present a patient with an infected chronic renal cyst found to have an empyema secondary to Proteus mirabilis to highlight the importance of further diagnostic workup when encountering unusual organisms in the pleural space. A 40-year-old African-American female, with a past medical history of asthma and sickle cell trait, presented with 5 weeks of upper respiratory tract symptoms and chest pain. A computed tomography angiogram (CTA) of the chest was negative for a pulmonary embolism but revealed a loculated left sided pleural effusion with associated left-lower lobe consolidation. She was started on empiric antibiotics, and a chest tube was inserted with drainage of frank pus. Fluid gram stain was positive for gram negative rods. Intrapleural fibrinolytics were administered for 72 h given the presence of loculations. With no improvement following fibrinolytics, she was taken to the operating room for large bore chest tube placement and left visceral pleura decortication. Pleural fluid cultures speciated to Proteus mirabilis, so further cross-sectional imaging of her abdomen\u002Fpelvis was pursued to evaluate for a primary source. A complex cystic lesion in the upper pole of the left kidney that communicated with the ipsilateral diaphragm was identified. Subsequent drainage and culture of the renal cyst was positive for Proteus mirabilis. Given clinical improvement following these interventions she was discharged with an extended course of antibiotics with plans for repeat imaging following completion of treatment. While cases of Proteus mirabilis empyema have previously been reported as a consequence of conditions such as pyelonephritis, we present, to our knowledge, the first case of a Proteus mirabilis empyema as a consequence of an infected renal cyst communicating with the pleural space. This study highlights that further evaluation with cross-sectional imaging is warranted when unusual organisms are found in the pleural space. Anatomic abnormalities that become apparent on imaging may help elucidate the source of infection.",{"EN":1536},"Proteus empyema as a rare complication from an infected renal cyst, a case report",{"VOID":1538},"McCauley L, Dean N. Pneumonia and empyema: causal, casual or unknown. J Thorac Dis. 2015;7(6):992–8.\nChapman S, Robert J. The management of pleural space infections. Respirology. 2004;9(1):4–11.\nRosenstengel A. Pleural infection-current diagnosis and management. J Thorac Dis. 2012;4(2):186.\nPatel S, Holland S, Fullerton D. P. mirabilis Pleural Empyema Secondary to a Staghorn Calculus in the Left Kidney. Am Thorac Soc. 2018;A3597.\nO'Hara C, Brenner F, Miller J. Classification, identification, and clinical significance of Proteus, Providencia, and Morganella. Clin Microbiol Rev. 2000;13(4):534–46.\nHernández P, Suárez R, Gilart F. Pleural empyema secondary to pyonephrosis. Arch Bronconeumol. 2008;44(5):285.\nLacort F, Gutierrez F, Gonzalez R, Antuna B, Herrero F, Espina A. Massive pleural empyema caused by pyonephrosis. An Med Interna. 2001;18(3):147–8.\nPine J, Hollman J. Elevated pleural fluid pH in Proteus mirabilis empyema. Chest. 1983;84(1):109–11.\nRahman N, Mishra E, Davies H, Davies R, Lee Y. Clinically important factors influencing the diagnostic measurement of pleural fluid pH and glucose. Am J Respir Crit Care Med. 2008;178(5):483–90.\nTillotson J, Lerner A. Characteristics of pneumonias caused by Bacillus proteus. Ann Intern Med. 1968;68(2):287–94.",{"VOID":1540},"10.1186\u002Fs12890-020-01346-w","https:\u002F\u002Fbmcpulmmed.biomedcentral.com\u002Farticles\u002F10.1186\u002Fs12890-020-01346-w",[1543,1558,1573,1588,1606,1619,1634],{"id":1544,"sortIndex":32,"researcher":28,"roles":1545,"affiliations":1546,"properties":1555,"displayName":1557,"givenName":28,"familyName":28},"93ccfd80-e0eb-47d7-a179-1e30040af9df",[1000],[1547],{"id":1548,"sortIndex":32,"affiliation":1549,"properties":28},"1061de9f-5f1f-45f1-9a3d-8891d96e66ca",{"id":1548,"createTime":28,"updateTime":28,"relativeEntities":1550,"slug":28,"properties":1551,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1554,"statistic":28},[],{"title":1552},{"VI":1553},"Department of Medicine, University of Virginia, Charlottesville, USA",[],{"title":1556},{"VI":1557},"Kranthikiran 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Similar findings may result from a congenital portosystemic shunt without liver disease. An adverse consequence of such shunts is intrapulmonary vascular dilatation, which affects the microvascular gas exchange units for oxygen. Here, we describe a toddler with chronic cyanosis, exercise intolerance, and finger clubbing due to a malformation shunt between the portal vein and the inferior vena cava. A transcatheter embolization of the shunt resulted in resolution of his findings. Congenital portosystemic shunts need to be considered in the differential diagnosis of cyanosis.",{"EN":1707},"Hypoxia due to intrapulmonary vascular dilatation in a toddler with a congenital portacaval shunt: case report",{"VOID":1709},"Rodríguez-Roisin R, Krowka MJ, Hervé P, Fallon MB, et al. Pulmonary-hepatic vascular disorders (PHD). Eur Respir J. 2004;24(5):861–80.\nBernard O, Franchi-Abella S, Branchereau S, Pariente D, Gauthier F, Jacquemin E. Congenital portosystemic shunts in children: recognition, evaluation, and management. Semin Liver Dis. 2012;32(04):273–87.\nKrowka MJ, Fallon MB, Kawut SM, Fuhrmann V, Hei mbach JK, Ramsay MA, et al. International liver transplant society practice guidelines: diagnosis and management of hepatopulmonary syndrome and portopulmonary hypertension. Transplantation. 2016;100(7):1440–52.\nSoulaidopoulos S, Cholongitas E, Giannakoulas G, Vlachou M, Goulis I. Review article: update on current and emergent data on hepatopulmonary syndrome. World J Gastroenterol. 2018;24(12):1285–98.\nWoods TD, Patel A. A critical review of patent foramen ovale detection using saline contrast echocardiography: when bubbles lie. J Am Soc Echocardiogr. 2006;19(2):215–22.\nSchraufnagel DE, Malik R, Goel V, Ohara N, Chang SW. Lung capillary changes in hepatic cirrhosis in rats. Am J Phys. 1997;272(1 Pt 1):L139–47.\nSchraufnagel DE, Kay JM. Structural and pathologic changes in the lung vasculature in chronic liver disease. Clin Chest Med. 1996;17(1):1–15.\nDuke JW, Elliott JE, Lovering AT. Clinical consideration for techniques to detect and quantify blood flow through intrapulmonary arteriovenous anastomoses: lessons from physiological studies. Echocardiography. 2015;32(Suppl 3):S195–204.\nDavis HH 2nd, Schwartz DJ, Lefrak SS, Susman N, Schainker BA. Alveolar-capillary oxygen disequilibrium in hepatic cirrhosis. Chest. 1978;73(4):507–11.\nGomez FP, Martinez-Palli G, Barbera JA, Roca J, Navasa M, Rodriguez-Roisin R. Gas exchange mechanism of orthodeoxia in hepatopulmonary syndrome. Hepatology. 2004;40(3):660–6.\nAlonso-Gamarra E, Parron M, Perez A, Prieto C, Hierro L, Lopez-Santamaria M. Clinical and radiologic manifestations of congenital extrahepatic portosystemic shunts: a comprehensive review. Radiographics. 2011;31(3):707–22.\nUchino T, Matsuda I, Endo F. The long-term prognosis of congenital portosystemic venous shunt. J Pediatr. 1999;135(2 Pt 1):254–6.",{"VOID":1711},"10.1186\u002Fs12890-019-0788-8","https:\u002F\u002Fbmcpulmmed.biomedcentral.com\u002Farticles\u002F10.1186\u002Fs12890-019-0788-8",[1714,1729,1744,1757,1772],{"id":1715,"sortIndex":32,"researcher":28,"roles":1716,"affiliations":1717,"properties":1726,"displayName":1728,"givenName":28,"familyName":28},"e7aa4e4c-0662-4986-b1de-4ee1ec2d3c8a",[1000],[1718],{"id":1719,"sortIndex":32,"affiliation":1720,"properties":28},"0a2fa860-b900-4c10-a0ce-cbdf235315c4",{"id":1719,"createTime":28,"updateTime":28,"relativeEntities":1721,"slug":28,"properties":1722,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1725,"statistic":28},[],{"title":1723},{"VI":1724},"Departments of Pediatrics at Tawam Hospital, Alain City, United Arab Emirates",[],{"title":1727},{"VI":1728},"Mohammed T. Alsamri",{"id":1730,"sortIndex":40,"researcher":28,"roles":1731,"affiliations":1732,"properties":1741,"displayName":1743,"givenName":28,"familyName":28},"0ef897c0-cd04-4909-900e-239bf0221c7b",[1000],[1733],{"id":1734,"sortIndex":32,"affiliation":1735,"properties":28},"99fc9061-73d0-4b2c-a892-276846191a15",{"id":1734,"createTime":28,"updateTime":28,"relativeEntities":1736,"slug":28,"properties":1737,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1740,"statistic":28},[],{"title":1738},{"VI":1739},"KidsHeart Medical Center, Alain City, United Arab Emirates",[],{"title":1742},{"VI":1743},"Mohamed A. Hamdan",{"id":1745,"sortIndex":123,"researcher":28,"roles":1746,"affiliations":1747,"properties":1754,"displayName":1756,"givenName":28,"familyName":28},"c58b0605-94de-4d41-940e-1cf292077992",[1000],[1748],{"id":1734,"sortIndex":32,"affiliation":1749,"properties":28},{"id":1734,"createTime":28,"updateTime":28,"relativeEntities":1750,"slug":28,"properties":1751,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1753,"statistic":28},[],{"title":1752},{"VI":1739},[],{"title":1755},{"VI":1756},"Mohamed Sulaiman",{"id":1758,"sortIndex":42,"researcher":28,"roles":1759,"affiliations":1760,"properties":1769,"displayName":1771,"givenName":28,"familyName":28},"81b1a6c9-c4ed-4351-9ace-361b810b88d5",[1000],[1761],{"id":1762,"sortIndex":32,"affiliation":1763,"properties":28},"1249d41a-a91f-45b7-8f98-89a99b1cbc32",{"id":1762,"createTime":28,"updateTime":28,"relativeEntities":1764,"slug":28,"properties":1765,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1768,"statistic":28},[],{"title":1766},{"VI":1767},"College of Medicine and Health Sciences - UAE University, Alain City, United Arab Emirates",[],{"title":1770},{"VI":1771},"Hassib Narchi",{"id":1773,"sortIndex":45,"researcher":28,"roles":1774,"affiliations":1775,"properties":1782,"displayName":1784,"givenName":28,"familyName":28},"cf07f584-ea92-4294-8d49-3e114c674716",[1000],[1776],{"id":1762,"sortIndex":32,"affiliation":1777,"properties":28},{"id":1762,"createTime":28,"updateTime":28,"relativeEntities":1778,"slug":28,"properties":1779,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1781,"statistic":28},[],{"title":1780},{"VI":1767},[],{"title":1783},{"VI":1784},"Abdul-Kader Souid",{"url":1712,"publisher":1786,"properties":1827},{"id":868,"createTime":869,"updateTime":870,"relativeEntities":1787,"slug":872,"properties":1788,"entityType":25,"verifyStatus":878,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":32,"subjectFields":1791,"manageAffiliations":1796,"indexDatabases":1807,"url":28,"thumbnailPath":28,"statistic":1822,"gsStatistic":28,"type":28,"analyzePriority":28},[],{"title":1789,"eissn":1790},{"EN":875},{"VOID":877},[1792],{"id":881,"createTime":28,"updateTime":28,"relativeEntities":1793,"label":1794,"description":1795,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":884},{},[1797,1802],{"id":888,"createTime":28,"updateTime":28,"relativeEntities":1798,"slug":28,"properties":1799,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1801,"statistic":28},[],{"title":1800},{"EN":892},[],{"id":895,"createTime":28,"updateTime":28,"relativeEntities":1803,"slug":28,"properties":1804,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1806,"statistic":28},[],{"title":1805},{"EN":899},[],[1808,1815],{"id":919,"indexDatabase":1809,"url":925,"indexYears":926,"academicFieldIds":1814,"indexDatabaseRanking":929},{"id":775,"createTime":28,"updateTime":28,"relativeEntities":1810,"label":1811,"description":1812,"key":781,"publicationTags":1813,"standard":28},[],{"EN":778,"VI":778},{"EN":778,"VI":780},[783],[928],{"id":903,"indexDatabase":1816,"url":915,"indexYears":28,"academicFieldIds":1821,"indexDatabaseRanking":28},{"id":905,"createTime":28,"updateTime":28,"relativeEntities":1817,"label":1818,"description":1819,"key":912,"publicationTags":1820,"standard":28},[],{"EN":908,"VI":908},{"EN":910,"VI":911},[914,813],[917],{"impactFactor":32,"impactFactorByYear":1823,"i10Index":934,"i10IndexLast5Year":516,"totalPublication":935,"totalPublicationByYear":1824,"totalCitation":942,"totalCitationByYear":1825,"totalCitationPerPublication":372,"totalCitationPerPublicationByYear":1826,"hindexLast5Year":196,"hindex":196},{"2012":319,"2013":932,"2014":229,"2015":346,"2016":227,"2017":228,"2018":709,"2019":226,"2020":114,"2021":368,"2022":119,"2023":933},{"2001":45,"2002":42,"2003":42,"2004":205,"2005":205,"2006":135,"2007":146,"2008":129,"2009":278,"2010":279,"2011":278,"2012":689,"2013":436,"2014":529,"2015":937,"2016":938,"2017":359,"2018":331,"2019":836,"2020":606,"2021":939,"2022":940,"2023":941,"2024":687},{"2001":45,"2004":944,"2006":361,"2007":135,"2008":329,"2009":945,"2010":612,"2011":946,"2012":947,"2013":948,"2014":949,"2015":950,"2016":951,"2017":952,"2018":953,"2019":954,"2020":955,"2021":956,"2022":957},{"2001":40,"2004":959,"2006":960,"2007":961,"2008":962,"2009":963,"2010":964,"2011":965,"2012":966,"2013":45,"2014":967,"2015":968,"2016":969,"2017":970,"2018":971,"2019":972,"2020":371,"2021":363,"2022":462},{"pages":1828,"volume":1830},{"VOID":1829},"1-6",{"VOID":1831},"19","2019-02-22",2019,[914,929],{"id":1836,"createTime":1837,"updateTime":1838,"relativeEntities":1839,"slug":1840,"properties":1841,"entityType":992,"verifyStatus":26,"verifyTime":1838,"verifyNote":994,"languages":28,"translateLanguages":28,"viewCount":32,"primaryUrl":1850,"fullTextUrl":28,"authors":1851,"publicationType":1132,"publisherRelationship":2016,"citationCount":28,"citationInfo":28,"publishDate":2063,"publishYear":2064,"citationAnalyzeStatus":878,"lastCitationAnalyze":28,"indexDatabases":2065,"openAccess":28,"references":28,"isForceReanalyzing":1183},"0061091d-2083-456c-81ca-f814dcc0fb02","2023-12-26T01:55:49.433+00:00","2024-09-20T19:18:56.063+00:00",[],"Prevalence-of-asthma-and-other-allergic-conditions-in-Colombia-2009-2010-a-cross-sectional-study",{"abstract":1842,"title":1844,"references":1846,"doi":1848},{"EN":1843},"While it is suggested that the prevalence of asthma in developed countries may have stabilized, this is not clear in currently developing countries. Current available information for both adults and children simultaneously on the burden and impact of allergic conditions in Colombia and in many Latin American countries is limited. The objectives of this study were to estimate the prevalence for asthma, allergic rhinitis (AR), atopic eczema (AE), and atopy in six colombian cities; to quantify costs to the patient and her\u002Fhis family; and to determine levels of Immunoglobulin E (IgE) in asthmatic and healthy subjects. We conducted a cross-sectional, population-based study in six cities during the academic year 2009–2010. We used a school-based design for subjects between 5–17 years old. We carried out a community-based strategy for subjects between 1–4 years old and adults between 18–59 years old. Serum samples for total and antigen-specific (IgE) levels were collected using a population-based, nested, case–control design. We obtained information on 5978 subjects. The largest sample of subjects was collected in Bogotá (2392). The current prevalence of asthma symptoms was 12% (95% CI, 10.5-13.7), with 43% (95% CI, 36.3-49.2) reporting having required an emergency department visit or hospitalization in the past 12 months. Physician diagnosed asthma was 7% (95% CI, 6.1-8.0). The current prevalence of AR symptoms was 32% (95% CI, 29.5-33.9), and of AE symptoms was 14% (95% CI, 12.5-15.3). We collected blood samples from 855 subjects; 60.2% of asthmatics and 40.6% of controls could be classified as atopic. In Colombia, symptom prevalence for asthma, AR and AE, as well as levels of atopy, are substantial. Specifically for asthma, symptom severity and absence from work or study due to symptoms are important. These primary care sensitive conditions remain an unmet public health burden in developing countries such as Colombia.",{"EN":1845},"Prevalence of asthma and other allergic conditions in Colombia 2009–2010: a cross-sectional study",{"VOID":1847},"Eder W, Ege MJ, von Mutius E: The asthma epidemic. N Engl J Med. 2006, 355: 2226-2235. 10.1056\u002FNEJMra054308.\nLawson JA, Senthilselvan A: Asthma epidemiology: has the crisis passed?. Curr Opin Pulm Med. 2004, 11: 79-84.\nVon Hertzen L, Haahtela T: Signs of reversing trends in the prevalence of asthma. Allergy. 2005, 60: 283-292. 10.1111\u002Fj.1398-9995.2005.00769.x.\nAsher MI, Montefort S, Bjorksten B, Lai CK, Strachan DP, Weiland SK, Williams H, ISAAC Phase Three Study Group: Worldwide time trends in the prevalence of symptoms of Asthma, Rhinoconjuctivitis, and eczema in childhood. Lancet. 2006, 368: 733-743. 10.1016\u002FS0140-6736(06)69283-0.\nOdhiambo JA, Williams HC, Clayton TO, Robertson CF, Asher MI: Global variations in prevalence of eczema symptoms in children from ISAAC phase three. J Allergy Clin Inmunol. 2009, 124: 1251-1258. 10.1016\u002Fj.jaci.2009.10.009.\nAit-Khaled N, Pearce N, Anderson HR, Ellwood P, Montefort S, Shah J: Global map of the prevalence of symptoms of rhinoconjunctivitis in children: the international study of asthma and allergies in childhood (ISAAC) phase three. Allergy. 2009, 64: 123-148. 10.1111\u002Fj.1398-9995.2008.01884.x.\nFord ES: The epidemiology of obesity and asthma. J Allergy Clin Inmunol. 2005, 115: 897-909. 10.1016\u002Fj.jaci.2004.11.050.\nThompson CC, Clark S, Camargo CA: BMI and asthma severity among adults presenting to the emergency department. Chest. 2003, 124: 795-802. 10.1378\u002Fchest.124.3.795.\nEllwood P, Asher MI, Bjorksten B, Burr M, Pearce N, Robertson CF: Diet and asthma, allergic rhinoconjunctivitis and atopic eczema symptom prevalence: an ecological analysis of the International Study of Asthma and Allergies in Childhood (ISAAC) data. ISAAC Phase One Study Group. Eur Respir J. 2001, 17: 436-443. 10.1183\u002F09031936.01.17304360.\nDevereux G, Seaton A: Diet as a risk factor for atopy and asthma. J Allergy Clin Immunol. 2005, 115: 1109-1117. 10.1016\u002Fj.jaci.2004.12.1139.\nStewart AW, Mitchell EA, Pearce N, Strachan DP, Weilandon SK: The relationship of per capita gross national product to the prevalence of symptoms of asthma and other atopic diseases in children (ISAAC). Int J Epidemiol. 2001, 30: 173-179. 10.1093\u002Fije\u002F30.1.173.\nWickens K, Pearce N, Crane J, Beasley R: Antibiotic use in early childhood and the development of asthma. Clin Exp Allergy. 1999, 29: 766-771. 10.1046\u002Fj.1365-2222.1999.00536.x.\nNewson RB, Shaheen SO, Chinn S, Burney PG: Paracetamol sales and atopic disease in children and adults: an ecological analysis. Eur Respir J. 2000, 16: 817-823. 10.1183\u002F09031936.00.16581700.\nSolé D, Wandalsen G, Camelo-Nunes IC, Naspitz CK: Prevalence of symptoms of asthma, rhinitis and atopic eczema among Brazilian children and adolescents identified by the international study of asthma and allergies in childhood (ISAAC)-phase III. J Pediatr (Rio J). 2006, 82: 341-346.\nMcCarthy M: Researchers call for more research into Asthma in Latinamerica. Lancet. 2003, 361: 1797-10.1016\u002FS0140-6736(03)13452-6.\nWHO\u002FPAHO: Health indicators, Andean Region., http:\u002F\u002Fais.paho.org\u002Fphip\u002Fviz\u002FIndicadoresdesalud_regionandina_esp.aspAccessed March 11, 2012\nDennis R, Caraballo L, García E, Aristizabal G, Córdoba H, Rodriguez MN, Rojas MX, Orduz C, Cardona R, Blanco A, Egea E, Verbel C, Cala LL: Asthma and other allergic conditions in Colombia: a study in 6 cities. Ann Allergy Asthma Immunol. 2004, 93: 568-574. 10.1016\u002FS1081-1206(10)61265-3.\nGarcia E, Aristizabal G, Vasquez C, Rodriguez-Martinez CE, Sarmiento OL, Satizabal Cl: Prevalence of and Factors Associated with Current Asthma Symptoms in School Children aged 6–7 and 13–14 years old in Bogota, Colombia. Pediatr Allergy Immunol. 2008, 19: 307-314. 10.1111\u002Fj.1399-3038.2007.00650.x.\nRodriguez C, Sossa M, Goss CH: Factors associated with severe disease in a population of asthmatic children in Bogotá, Colombia. J Asthma. 2008, 45: 141-147. 10.1080\u002F02770900701840253.\nInvestigación de educación formal: DANE. Departamento administrativo nacional de estadística. , , , http:\u002F\u002Fwww.dane.gov.co\u002Findex.php?option=com_content&task=category&sectionid=47&id=79&Itemid=261] Accessed March 26, 2009\nSärndal CE, Swensson B, Wretman J: Introduction to more complex estimation problems. Model Assisted Survey Sampling. 1992, Springer-Verlag, New York, PA, 172-190.\nAsher MI, Keil U, Anderson HR, Beasley R, Crane J, Martinez F, Mitchell EA, Pearce N, Sibbald B, Stewart AW, Strachan D, Weiland SK, Williams HC: International study of asthma and allergies in childhood (ISAAC): rationale and methods. Eur Resp J. 1995, 8: 483-491. 10.1183\u002F09031936.95.08030483.\nWoodruff RSA: simple method for approximating the variance of a complicated estimate. J Am Stat Assoc. 1971, 66: 411-414. 10.1080\u002F01621459.1971.10482279.\nSubbarao P, Mandhane PJ, Sears MR: Asthma: epidemiology, etiology and risk factors. CMAJ. 2009, 181: 181-190.\nMallol J, Solé D, Baeza-Bacab M, Aguirre-Camposano V, Soto-Quiros M, Baena-Cagnani C: Regional variation in asthma symptom prevalence in Latinamerican children. J Asthma. 2010, 47: 644-650. 10.3109\u002F02770901003686480.\nPeñaranda A, Aristizabal G, García E, Vásquez C, Rodríguez-Martinez CE: Rhinoconjunctivitis prevalence and associated factors in school children aged 6–7 and 13–14 years old in Bogota, Colombia. Int J Pediatr Otorhinolaryngol. 2012, : -Jan 31.[E-publication ahead of print]\nCooper PJ, Chico ME, Rodrigues LC, Strachan DP, Anderson HR, Rodrigez EA, Gaus DP: Risk factors for atopy among school children in a rural area of Latin America. Clin Exp Allergy. 2004, 34: 845-852. 10.1111\u002Fj.1365-2222.2004.01958.x.\nCooper PJ, Rodrigues LC, Cruz AA, Barreto ML: Asthma in Latin America: a public health challenge and research opportunity. Allergy. 2009, 64: 5-17. 10.1111\u002Fj.1398-9995.2008.01902.x.\nCaudri G, Wijga A, Schipper MA, Hoekstra M, Postma D, Koppelman G: Predicting the long term prognosis of children with symptoms suggestive of asthma at pre-school age. J Allergy Clin Inmunol. 2009, 124: 903-910. 10.1016\u002Fj.jaci.2009.06.045.\nMartinez FD: The connection between early life wheezing and subsequent asthma: The viral march. Allergol Immunopathol (Madr). 2009, 37: 249-251. 10.1016\u002Fj.aller.2009.06.008.\nNeffens H, Fritscher C, Schacht FC, Chiarella P, Soriano JB, Mechali D: Asthma control in Latin America: the asthma insights and reality in Latin America (AIRLA) survey. Rev Panam Salud Publica. 2005, 17: 191-197.\nRabe KF, Adachi M, Lai CKW, Soriano JB, Vermeire PA, Weiss KB, Weiss ST: Worldwide severity and control of asthma in children and adults. J Allergy Clin Inmunol. 2004, 114: 40-47. 10.1016\u002Fj.jaci.2004.04.042.\nCaminal J, Starfield B, Sanchez E, Casanova C, Morales M: The role of primary care in preventing ambulatory care sensitive conditions. Eur J Public Health. 2004, 14: 246-251. 10.1093\u002Feurpub\u002F14.3.246.\nGodard P, Chanez P, Siraudin P, Nicoloyannis N, Guru G: Costs of asthma are correlated with severity: a 1 year prospective study. Eur Respir J. 2002, 19: 61-67. 10.1183\u002F09031936.02.00232001.\nFuhlbrigge AL, Adams RJ, Guibert TW, Grant E: The burden of asthma in the United States. Am J Respir Crit Care Med. 2002, 166: 1044-1049. 10.1164\u002Frccm.2107057.\nGergen PJ, Arbes SJ, Calatroni A, Mitchell HE, Zeldin DC: Total IgE levels and asthma prevalence in the US population. J Allergy Clin Inmunol. 2009, 124: 447-453. 10.1016\u002Fj.jaci.2009.06.011.\nWeynmayr G, Weiland SK, Bjorksten B, Brunekreef B, Buchele G, Cookson WO, García-Marcos L, Gotua M, Gratziou C, van Hage M, von Mutius E, Riikjarv MA, Rzehak P, Stein RT, Strachan DP, Tsanakas J, Wickens K, Won GW, ISAAC Phase Two Study Group: Atopic sensitization and the international variation of asthma symptom prevalence in children. Am J Respir Crit Care Med. 2007, 176: 565-574. 10.1164\u002Frccm.200607-994OC.\nAcevedo N, Erler A, Briza P, Puccio F, Ferreira F, Caraballo L: Allergenicity ofAscaris lumbricoidestropomyosin and IgE sensitization among asthmatic patients in a tropical environment. Int Arch Allergy Immunol. 2011, 154: 195-206. 10.1159\u002F000321106.\nAcevedo N, Sánchez J, Erler A, Mercado D, Briza P, Kennedy M, Fernandez A, Gutierrez M, Chua KY, Cheong N, Jiménez S, Puerta L, Caraballo L: IgE cross-reactivity betweenAscarisand domestic mite allergens: the role of tropomyosin and the nematode polyprotein ABA-1. Allergy. 2009, 64: 1635-1643. 10.1111\u002Fj.1398-9995.2009.02084.x.\nCaraballo L, Acevedo N: Allergy in the tropics: the impact of cross-reactivity between mites and ascaris. Front Biosci. 2011, E3: 51-64.\nSolé D, Mallol J, Camelo-Nunes IC, Wandalsen GF: Prevalence of rhinitis related symptoms in Latin American children – results of the International Study of Asthma and Allergies in Childhood (ISAAC) phase three. Pediatr Allergy Immunol. 2010, 21: l27-l36.\nGarcia-Marcos L, Quirós AB, Hernández GG, Guillén-Grima F, Díaz CG, Ureña IC, Pena AA, Monge RB, Suárez-Varela MM, Varela AL, Cabanillas PG, Garrido JB: Stabilization of Asthma prevalence among adolescents and increase among schoolchildren in Spain. Allergy. 2004, 59: 1301-1307. 10.1111\u002Fj.1398-9995.2004.00562.x.\nShayck V, Smit HA: The prevalence of asthma in children: a reversing trend. Eur Respir J. 2005, 26: 647-650.\nWeiland SK, Pearce N: Asthma prevalence in adults: good news?. Thorax. 2004, 59: 637-638. 10.1136\u002Fthx.2004.026302.\nThe pre-publication history for this paper can be accessed here:http:\u002F\u002Fwww.biomedcentral.com\u002F1471-2466\u002F12\u002F17\u002Fprepub",{"VOID":1849},"10.1186\u002F1471-2466-12-17","https:\u002F\u002Fbmcpulmmed.biomedcentral.com\u002Farticles\u002F10.1186\u002F1471-2466-12-17",[1852,1867,1882,1897,1912,1925,1949,1964,1977,1990,2003],{"id":1853,"sortIndex":32,"researcher":28,"roles":1854,"affiliations":1855,"properties":1864,"displayName":1866,"givenName":28,"familyName":28},"bce226e9-2af2-418c-9312-1ba6b9d7e578",[1000],[1856],{"id":1857,"sortIndex":32,"affiliation":1858,"properties":28},"47b5f5c7-77a5-422d-8c0e-155ca0e443d6",{"id":1857,"createTime":28,"updateTime":28,"relativeEntities":1859,"slug":28,"properties":1860,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1863,"statistic":28},[],{"title":1861},{"VI":1862},"Research 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improve the understanding of acute fibrinous and organizing pneumonia (AFOP), we present one case of AFOP proven by percutaneous lung biopsy along with clinical features, chest imaging and pathology. A 50-year-old man was admitted to our department after he was given empiric therapy for community-acquired pneumonia (CAP). The clinical symptoms of the patient were dry cough, chills, night sweats and high fevers. Chest computed tomography (CT) scan showed a high-density shadow in the right lung lobe, similar to lobular pneumonia. The patient was preliminarily diagnosed with community-acquired pneumonia; however, antibacterial treatment was ineffective. To confirm the diagnosis, we performed bronchoscopy and percutaneous lung biopsy; pathology was consistent with AFOP. After he was treated with glucocorticoids, the patient’s symptoms were relieved, and the shadow seen on imaging dissipated during the follow-up period. AFOP is a rare histopathological diagnosis that can be easily misdiagnosed. Clinicians need to consider the possibility of AFOP in the case of invalid antibacterial therapy.",{"EN":2076},"A case of herbicide-induced acute fibrinous and organizing pneumonia?",{"VOID":2078},"Beasley MB, Franks TJ, Galvin JR, Gochuico B, Travis WD. Acute fibrinous and organizing pneumonia: a histological pattern of lung injury and possible variant of diffuse alveolar damage. Arch Pathol Lab Med. 2002;126(9):1064–70.\nTravis WD, Costabel U, Hansell DM, King TJ, Lynch DA, Nicholson AG, Ryerson CJ, Ryu JH, Selman M, Wells AU, et al. An official American Thoracic Society\u002FEuropean Respiratory Society statement: update of the international multidisciplinary classification of the idiopathic interstitial pneumonias. Am J Respir Crit Care Med. 2013;188(6):733–48.\nDai JH, Li H, Shen W, Miao LY, Xiao YL, Huang M, Cao MS, Wang Y, Zhu B, Meng FQ, et al. Clinical and radiological profile of acute Fibrinous and organizing pneumonia: a retrospective study. Chin Med J. 2015;128(20):2701–6.\nGomes R, Padrao E, Dabo H, Soares PF, Mota P, Melo N, Jesus JM, Cunha R, Guimaraes S, Souto MC, et al. Acute fibrinous and organizing pneumonia: a report of 13 cases in a tertiary university hospital. Medicine (Baltimore). 2016;95(27):e4073.\nKligerman SJ, Franks TJ, Galvin JR. From the radiologic pathology archives: organization and fibrosis as a response to lung injury in diffuse alveolar damage, organizing pneumonia, and acute fibrinous and organizing pneumonia. Radiographics. 2013;33(7):1951–75.\nHashisako M, Fukuoka J. Pathology of idiopathic interstitial pneumonias. Clin Med Insights Circ Respir Pulm Med. 2015;9(Suppl 1):123–33.\nCincotta DR, Sebire NJ, Lim E, Peters MJ. Fatal acute fibrinous and organizing pneumonia in an infant: the histopathologic variability of acute respiratory distress syndrome. Pediatr Crit Care Med. 2007;8(4):378–82.\nParaskeva M, McLean C, Ellis S, Bailey M, Williams T, Levvey B, Snell GI, Westall GP. Acute fibrinoid organizing pneumonia after lung transplantation. Am J Respir Crit Care Med. 2013;187(12):1360–8.\nValim V, Rocha RH, Couto RB, Paixao TS, Serrano EV. Acute fibrinous and organizing pneumonia and undifferentiated connective tissue disease: a case report. Case Rep Rheumatol. 2012;2012:549298.\nFasanya A, Gandhi V, DiCarlo C, Thirumala R. Acute fibrinous and organizing pneumonia in a patient with Sjogren's syndrome. Respir Med Case Rep. 2017;20:28–30.\nChiu KY, Li JG, Gu YY. A case report of acute fibrinous and organizing pneumonia with pneumothorax and avian exposure history. Clin Respir J. 2016; doi:10.1111\u002Fcrj.12553.\nOtto C, Huzly D, Kemna L, Huttel A, Benk C, Rieg S, Ploenes T, Werner M, Kayser G. Acute fibrinous and organizing pneumonia associated with influenza a\u002FH1N1 pneumonia after lung transplantation. BMC Pulm Med. 2013;13:30.\nPiciucchi S, Dubini A, Tomassetti S, Casoni G, Ravaglia C, Poletti V. A case of amiodarone-induced acute fibrinous and organizing pneumonia mimicking mesothelioma. Am J Respir Crit Care Med. 2015;191(1):104–6.\nKamijo Y, Takai M, Sakamoto T. A multicenter retrospective survey of poisoning after ingestion of herbicides containing glyphosate potassium salt or other glyphosate salts in Japan. 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fluid nebulization is highly recommend to combat the complications following tracheostomy, yet the understandings on the role of osmolality in saline solution for nebulization remain unclear. To investigate the biological changes in the early stage after tracheostomy, to verify the efficacy of saline fluid nebulization and explore the potential role of osmolality of saline nebulization after tracheostomy. Sprague-Dawley rats undergone tracheostomy were taken for study model, the sputum viscosity was detected by rotational viscometer, the expressions of TNF-α, AQP4 in bronchoalveolar lavage fluid were assessed by western blot analysis, and the histological changes in endothelium were evaluated by HE staining and scanning electron microscopy (SEM). Study results revealed that tracheostomy gave rise to the increase of sputum viscosity, TNF-α and AQP4 expression, mucosa and cilia damage, yet the saline fluid nebulization could significantly decrease the changes of those indicators, besides, the hypertonic, isotonic and hypertonic saline nebulization produced different efficacy. Osmolality plays an important role in the saline fluid nebulization after tracheostomy, and 3% saline fluid nebulization seems to be more beneficial, further studies on the role of osmolality in saline fluid nebulization are warranted.",{"EN":2227},"The role of osmolality in saline fluid nebulization after tracheostomy: time for changing?",{"VOID":2229},"Mehta AB, Cooke CR, Wiener RS, Walkey AJ. Hospital Variation in Early Tracheostomy in the United States: A Population-Based Study. Crit Care Med. 2016;44(8):1506–14.\nHyde GA, Savage SA, Zarzaur BL, Hart-Hyde JE, Schaefer CB, Croce MA, Fabian TC. Early tracheostomy in trauma patients saves time and money. Injury. 2015;46(1):110–4.\nSofi K, Wani T. Effect of tracheostomy on pulmonary mechanics: An observational study. Saudi J Anaesth. 2010;4(1):2–5.\nCampisi P, Forte V. Pediatric tracheostomy. Semin Pediatr Surg. 2016;25(3):191–5.\nMarshall RV, Haas PJ, Schweinfurth JM, Replogle WH. Tracheotomy Outcomes in Super Obese Patients. JAMA Otolaryngol Head Neck Surg. 2016;142(8):772–6.\nEdwards JD, Houtrow AJ, Lucas AR, Miller RL, Keens TG, Panitch HB, Dudley RA. Children and Young Adults Who Received Tracheostomies or Were Initiated on Long-Term Ventilation in PICUs. Pediatr Crit Care Med. 2016;17(8):e324–34.\nColandrea M, Eckardt P. Improving Tracheostomy Care Delivery: Instituting Clinical Care Pathways and Nursing Education to Improve Patient Outcomes. ORL Head Neck Nurs. 2016;34(1):7–16.\nDawson D. Essential principles: tracheostomy care in the adult patient. Nurs Crit Care. 2014;19(2):63–72.\nPaul F. Tracheostomy care and management in general wards and community settings: literature review. Nurs Crit Care. 2010;15(2):76–85.\nDres M, Ferre A, Becquemin MH, Dessanges JF, Reychler G, Durand M, Escabasse V, Sauvaget E, Dubus JC. [Inhalation therapy: provocation tests, infectious risks, acute bronchiolitis and ENT diseases. GAT aerosolstorming, Paris. Rev Mal Respir. 2012;29(10):1186–97.\nGupta HV, Gupta VV, Kaur G, Baidwan AS, George PP, Shah JC, Shinde K, Malik R, Chitkara N, Bajaj KV. Effectiveness of 3% hypertonic saline nebulization in acute bronchiolitis among Indian children: A quasi-experimental study. Persp Clin Res. 2016;7(2):88–93.\nGupta S, Ahmed F, Lodha R, Gupta YK, Kabra SK. Comparison of effects of 3 and 7% hypertonic saline nebulization on lung function in children with cystic fibrosis: a double-blind randomized, controlled trial. J Trop Pediatr. 2012;58(5):375–81.\nHuang H, Li C, Wu Y. Application of oxygen jet nebulization with 0.45% saline in tracheotomized patients. J Nurs Sci. 2007;22(10):2.\nLi L, Zhou F, Sang X, Huang Y. Comparative study of effect of different airway humidification fluid in tracheotomy patients. Med Innovation China. 2014;14:2.\nLi S, Lu Q, Deng H, Pang S. Effect of different saline solutions on sputum inducing in patients with tracheostomy. J Nurs Training. 2016;31(6):3.\nEigl S, Hoenigl M, Spiess B, Heldt S, Prattes J, Neumeister P, Wolfler A, Rabensteiner J, Prueller F, Krause R, et al. Galactomannan testing and Aspergillus PCR in same-day bronchoalveolar lavage and blood samples for diagnosis of invasive aspergillosis. Med Mycol. 2016;23(10):641–9.\nYang SH, Lin JC, Wu SY, Huang KL, Jung F, Ma MC, Wang Hsu GS, Jow GM. Membrane translocation of IL-33 receptor in ventilator induced lung injury. PLoS One. 2015;10(3):e0121391.\nWang C, Yan M, Jiang H, Wang Q, Guan X, Chen J, Wang C. Protective effects of puerarin on acute lung and cerebrum injury induced by hypobaric hypoxia via the regulation of aquaporin (AQP) via NF-kappaB signaling pathway. Int Immunopharmacol. 2016;40:300–9.\nZhang WG, He L, Shi XM, Wu SS, Zhang B, Mei L, Xu YJ, Zhang ZX, Zhao JP, Zhang HL. Regulation of transplanted mesenchymal stem cells by the lung progenitor niche in rats with chronic obstructive pulmonary disease. Respir Res. 2014;15:33.\nNakajima H, Nakajima HO, Dembowsky K, Pasumarthi KB, Field LJ. Cardiomyocyte cell cycle activation ameliorates fibrosis in the atrium. Circ Res. 2006;98(1):141–8.\nCandau-Alvarez A, Gil-Campos M, De la Torre-Aguilar MJ, Llorente-Cantarero F, Lopez-Miranda J, Perez-Navero JL. Early modification in drainage of interleukin-1beta and tumor necrosis factor-alpha best predicts surgical-site infection after cervical neck dissection for oral cancer. J Oral Maxillofac Surg. 2015;73(6):1189–98.\nOkuyucu S, Akoglu E, Babayigit C, Akoglu S, Dagli S. Ex vivo tracheal mucociliary clearance in rats: comparisons of nebulization versus irrigation with lactated ringer and saline solutions. J Otolaryngol Head Neck Surg. 2009;38(4):422–6.\nSong Y, Jayaraman S, Yang B, Matthay MA, Verkman AS. Role of aquaporin water channels in airway fluid transport, humidification, and surface liquid hydration. J Gen Physiol. 2001;117(6):573–82.\nRuddy MK, Drazen JM, Pitkanen OM, Rafii B, O’Brodovich HM, Harris HW. Modulation of aquaporin 4 and the amiloride-inhibitable sodium channel in perinatal rat lung epithelial cells. Am J Physiol. 1998;274(6 Pt 1):L1066–1072.\nCampos KK, Leal SF, Costa DC, de Lima WG, Bezerra FS. Long-term exposure to ultrasonically nebulized distilled water and saline causes cellular influx and oxidative stress in lung tissue of rats. Exp Lung Res. 2015;41(10):546–53.\nBilton D, Stanford G. The expanding armamentarium of drugs to aid sputum clearance: how should they be used to optimize care? Curr Opin Pulm Med. 2014;20(6):601–6.\nGoldman G, Welbourn R, Kobzik L, Valeri CR, Shepro D, Hechtman HB. Tumor necrosis factor-alpha mediates acid aspiration-induced systemic organ injury. Ann Surg. 1990;212(4):513–9. discussion 519–520.\nLewis CA, Martin GS. Understanding and managing fluid balance in patients with acute lung injury. Curr Opin Crit Care. 2004;10(1):13–7.\nMatsui H, Davis CW, Tarran R, Boucher RC. Osmotic water permeabilities of cultured, well-differentiated normal and cystic fibrosis airway epithelia. J Clin Invest. 2000;105(10):1419–27.\nFarinas J, Kneen M, Moore M, Verkman AS. Plasma membrane water permeability of cultured cells and epithelia measured by light microscopy with spatial filtering. J Gen Physiol. 1997;110(3):283–96.\nVerkman AS, Matthay MA, Song Y. Aquaporin water channels and lung physiology. Am J Physiol Lung Cell Mol Physiol. 2000;278(5):L867–879.\nWabnitz DA, Wormald PJ. A blinded, randomized, controlled study on the effect of buffered 0.9% and 3% sodium chloride intranasal sprays on ciliary beat frequency. Laryngoscope. 2005;115(5):803–5.\nMa W, Korngreen A, Uzlaner N, Priel Z, Silberberg SD. Extracellular sodium regulates airway ciliary motility by inhibiting a P2X receptor. Nature. 1999;400(6747):894–7.\nBoek WM, Keles N, Graamans K, Huizing EH. Physiologic and hypertonic saline solutions impair ciliary activity in vitro. Laryngoscope. 1999;109(3):396–9.\nMin YG, Lee KS, Yun JB, Rhee CS, Rhyoo C, Koh YY, Yi WJ, Park KS. Hypertonic saline decreases ciliary movement in human nasal epithelium in vitro. J Otolaryngol Head Neck Surg. 2001;124(3):313–6.",{"VOID":2231},"10.1186\u002Fs12890-016-0342-x","https:\u002F\u002Fbmcpulmmed.biomedcentral.com\u002Farticles\u002F10.1186\u002Fs12890-016-0342-x",[2234,2249,2262,2277,2290,2303],{"id":2235,"sortIndex":32,"researcher":28,"roles":2236,"affiliations":2237,"properties":2246,"displayName":2248,"givenName":28,"familyName":28},"9fc566f3-0bce-44c0-b243-5d3d3597a9ab",[1000],[2238],{"id":2239,"sortIndex":32,"affiliation":2240,"properties":28},"cb13485c-4c22-453c-8770-397ccc45c9b2",{"id":2239,"createTime":28,"updateTime":28,"relativeEntities":2241,"slug":28,"properties":2242,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2245,"statistic":28},[],{"title":2243},{"VI":2244},"Nursing School of Soochow University, Su Zhou, People’s Republic of China",[],{"title":2247},{"VI":2248},"Zunjia 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ratio (NLR) is a biomarker of inflammation in chronic obstructive pulmonary disease (COPD) patients. But, a meaningful threshold and the longitudinal changes are unknown. We aimed to investigate the association between NLR and the clinical characteristics of COPD patients and to determine a meaningful threshold and the longitudinal changes for NLR. Keio University and its affiliate hospitals conducted an observational COPD cohort study over 3 years. We performed a blood examination and a pulmonary function test. Blood examination was completed at baseline and annually thereafter, at a time when the disease was stable. Two hundred seventy-four patients who had at least 3 blood examinations over 3 years were included. Baseline NLR was correlated with baseline C-reactive protein (CRP) (r = 0.18, p = 0.003) and SAA (r = 0.34, p \u003C  0.001). We defined an NLR score of 2.7 as the arbitrary cut-off value based on upper quartile points. COPD patients with NLR ≥ 2.7 were older (p = 0.037), had a lower BMI (p = 0.005) and a lower %FEV1 (p = 0.0003) compared to patients with NLR \u003C 2.7. Receiver-operating-characteristic (ROC) curves showed the optimal cutoff for the baseline NLR in the predicting moderate\u002Fsevere exacerbation to be 2.7, which was same as the upper quartile points. Follow-up analysis over 3 years revealed that the differences in the trends of NLR among the three groups based on the categories of exacerbations (moderate or severe, mild, no exacerbation) were significant (p = 0.006). NLR is associated with COPD severity and exacerbations. For predicting exacerbations, we estimated the threshold of NLR to be 2.7 at baseline. Clinical trial registered with the University Hospital Medication Information Network (\n                    UMIN000003470\n                    \n                  , April 10, 2010).",{"EN":2375},"Clinical utility of blood neutrophil-lymphocyte ratio in Japanese COPD patients",{"VOID":2377},"Rabe KF, Watz H. Chronic obstructive pulmonary disease. Lancet. 2017;389(10082):1931–40.\nBarnes PJ. Inflammatory mechanisms in patients with chronic obstructive pulmonary disease. J Allergy Clin Immunol. 2016;138(1):16–27.\nDahl M, Vestbo J, Lange P, Bojesen SE, Tybjaerg-Hansen A, Nordestgaard BG. C-reactive protein as a predictor of prognosis in chronic obstructive pulmonary disease. Am J Respir Crit Care Med. 2007;175(3):250–5.\nCelli BR, Locantore N, Yates J, Tal-Singer R, Miller BE, Bakke P, Calverley P, Coxson H, Crim C, Edwards LD, et al. Inflammatory biomarkers improve clinical prediction of mortality in chronic obstructive pulmonary disease. Am J Respir Crit Care Med. 2012;185(10):1065–72.\nMehrotra N, Freire AX, Bauer DC, Harris TB, Newman AB, Kritchevsky SB, Meibohm B, Health ABCS. Predictors of mortality in elderly subjects with obstructive airway disease: the PILE score. Ann Epidemiol. 2010;20(3):223–32.\nCalfee CS, Ware LB, Glidden DV, Eisner MD, Parsons PE, Thompson BT, Matthay MA, National Heart B. Lung institute acute respiratory distress syndrome N: use of risk reclassification with multiple biomarkers improves mortality prediction in acute lung injury. Crit Care Med. 2011;39(4):711–7.\nBozinovski S, Hutchinson A, Thompson M, Macgregor L, Black J, Giannakis E, Karlsson AS, Silvestrini R, Smallwood D, Vlahos R, et al. Serum amyloid a is a biomarker of acute exacerbations of chronic obstructive pulmonary disease. Am J Respir Crit Care Med. 2008;177(3):269–78.\nYonchuk JG, Silverman EK, Bowler RP, Agusti A, Lomas DA, Miller BE, Tal-Singer R, Mayer RJ. Circulating soluble receptor for advanced glycation end products (sRAGE) as a biomarker of emphysema and the RAGE axis in the lung. Am J Respir Crit Care Med. 2015;192(7):785–92.\nTamhane UU, Aneja S, Montgomery D, Rogers EK, Eagle KA, Gurm HS. Association between admission neutrophil to lymphocyte ratio and outcomes in patients with acute coronary syndrome. Am J Cardiol. 2008;102(6):653–7.\nAzab B, Jaglall N, Atallah JP, Lamet A, Raja-Surya V, Farah B, Lesser M, Widmann WD. Neutrophil-lymphocyte ratio as a predictor of adverse outcomes of acute pancreatitis. Pancreatology. 2011;11(4):445–52.\nYoon NB, Son C, Um SJ. Role of the neutrophil-lymphocyte count ratio in the differential diagnosis between pulmonary tuberculosis and bacterial community-acquired pneumonia. Ann Lab Med. 2013;33(2):105–10.\nTaylan M, Demir M, Kaya H, Selimoglu Sen H, Abakay O, Carkanat AI, Abakay A, Tanrikulu AC, Sezgi C. Alterations of the neutrophil-lymphocyte ratio during the period of stable and acute exacerbation of chronic obstructive pulmonary disease patients. Clin Respir J. 2017;11(3):311–7.\nGunay E, Sarinc Ulasli S, Akar O, Ahsen A, Gunay S, Koyuncu T, Unlu M. Neutrophil-to-lymphocyte ratio in chronic obstructive pulmonary disease: a retrospective study. Inflammation. 2014;37(2):374–80.\nLee H, Um SJ, Kim YS, Kim DK, Jang AS, Choi HS, Kim YH, Kim TE, Yoo KH, Jung KS. Association of the neutrophil-to-lymphocyte ratio with lung function and exacerbations in patients with chronic obstructive pulmonary disease. PLoS One. 2016;11(6):e0156511.\nFurutate R, Ishii T, Motegi T, Hattori K, Kusunoki Y, Gemma A, Kida K. The neutrophil to lymphocyte ratio is related to disease severity and exacerbation in patients with chronic obstructive pulmonary disease. Intern Med. 2016;55(3):223–9.\nCho KM, Park H, Oh DY, Kim TY, Lee KH, Han SW, Im SA, Kim TY, Bang YJ. Neutrophil-to-lymphocyte ratio, platelet-to-lymphocyte ratio, and their dynamic changes during chemotherapy is useful to predict a more accurate prognosis of advanced biliary tract cancer. Oncotarget. 2017;8(2):2329–41.\nJin F, Han A, Shi F, Kong L, Yu J. The postoperative neutrophil-to-lymphocyte ratio and changes in this ratio predict survival after the complete resection of stage I non-small cell lung cancer. Onco Targets Ther. 2016;9:6529–37.\nVanfleteren L, Spruit MA, Wouters EFM, Franssen FME. Management of chronic obstructive pulmonary disease beyond the lungs. Lancet Respir Med. 2016;4(11):911–24.\nMiller J, Edwards LD, Agusti A, Bakke P, Calverley PM, Celli B, Coxson HO, Crim C, Lomas DA, Miller BE, et al. Comorbidity, systemic inflammation and outcomes in the ECLIPSE cohort. Respir Med. 2013;107(9):1376–84.\nMiyazaki M, Nakamura H, Chubachi S, Sasaki M, Haraguchi M, Yoshida S, Tsuduki K, Shirahata T, Takahashi S, Minematsu N, et al. Analysis of comorbid factors that increase the COPD assessment test scores. Respir Res. 2014;15:13.\nChubachi S, Sato M, Kameyama N, Tsutsumi A, Sasaki M, Tateno H, Nakamura H, Asano K, Betsuyaku T, Keio CCRG. Identification of five clusters of comorbidities in a longitudinal Japanese chronic obstructive pulmonary disease cohort. Respir Med. 2016;117:272–9.\nIrie H, Chubachi S, Sato M, Tsutsumi A, Nakachi I, Miyao N, Nishio K, Nakamura H, Asano K, Betsuyaku T. Characteristics of patients with increasing COPD assessment test scores within 3 years. Respir Med. 2017;131:101–8.\nChubachi S, Nakamura H, Sasaki M, Haraguchi M, Miyazaki M, Takahashi S, Tanaka K, Funatsu Y, Asano K, Betsuyaku T, et al. Polymorphism of LRP5 gene and emphysema severity are associated with osteoporosis in Japanese patients with or at risk for COPD. Respirology. 2015;20(2):286–95.\nSato M, Chubachi S, Sasaki M, Haraguchi M, Kameyama N, Tsutsumi A, Takahashi S, Nakamura H, Asano K, Betsuyaku T. Impact of mild exacerbation on COPD symptoms in a Japanese cohort. Int J Chron Obstruct Pulmon Dis. 2016;11:1269–78.\nParr DG, Stoel BC, Stolk J, Stockley RA. Validation of computed tomographic lung densitometry for monitoring emphysema in alpha1-antitrypsin deficiency. Thorax. 2006;61(6):485–90.\nJones PW, Harding G, Berry P, Wiklund I, Chen WH, Kline Leidy N. Development and first validation of the COPD Assessment Test. Eur Respir J. 2009;34(3):648–54.\nKwon N, Amin M, Hui DS, Jung KS, Lim SY, Ta HD, Thai TTL, Yunus F, Jones PW. Validity of the COPD assessment test translated into local languages for Asian patients. Chest. 2013;143(3):703–10.\nHajiro T, Nishimura K, Tsukino M, Ikeda A, Koyama H, Izumi T. Analysis of clinical methods used to evaluate dyspnea in patients with chronic obstructive pulmonary disease. Am J Respir Crit Care Med. 1998;158(4):1185–9.\nHajiro T, Nishimura K, Tsukino M, Ikeda A, Koyama H, Izumi T. Comparison of discriminative properties among disease-specific questionnaires for measuring health-related quality of life in patients with chronic obstructive pulmonary disease. Am J Respir Crit Care Med. 1998;157(3 Pt 1):785–90.\nJones PW, Quirk FH, Baveystock CM, Littlejohns P. A self-complete measure of health status for chronic airflow limitation. The St. George’s Respiratory Questionnaire. Am Rev Respir Dis. 1992;145(6):1321–7.\nGlobal strategy for the diagnosis, management, and prevention of chronic obstructive pulmonary disease. http:\u002F\u002Fgoldcopd.org\u002Fgold-2017-global-strategy-diagnosis-management-prevention-copd\u002F. Accessed 28 Feb 2018.\nPark CH, Han DS, Jeong JY, Eun CS, Yoo KS, Jeon YC, Sohn JH. The optimal cut-off value of neutrophil-to-lymphocyte ratio for predicting prognosis in adult patients with Henoch-Schonlein Purpura. PLoS One. 2016;11(4):e0153238.\nLuo G, Liu C, Cheng H, Jin K, Guo M, Lu Y, Long J, Xu J, Ni Q, Chen J, et al. Neutrophil-lymphocyte ratio predicts survival in pancreatic neuroendocrine tumors. Oncol Lett. 2017;13(4):2454–8.\nRutgers SR, Postma DS, ten Hacken NH, Kauffman HF, van Der Mark TW, Koeter GH, Timens W. Ongoing airway inflammation in patients with COPD who do not currently smoke. Thorax. 2000;55(1):12–8.\nKido T, Tamagawa E, Bai N, Suda K, Yang HH, Li Y, Chiang G, Yatera K, Mukae H, Sin DD, et al. Particulate matter induces translocation of IL-6 from the lung to the systemic circulation. Am J Respir Cell Mol Biol. 2011;44(2):197–204.\nDemedts IK, Morel-Montero A, Lebecque S, Pacheco Y, Cataldo D, Joos GF, Pauwels RA, Brusselle GG. Elevated MMP-12 protein levels in induced sputum from patients with COPD. Thorax. 2006;61(3):196–201.\nO'Donnell RA, Peebles C, Ward JA, Daraker A, Angco G, Broberg P, Pierrou S, Lund J, Holgate ST, Davies DE, et al. Relationship between peripheral airway dysfunction, airway obstruction, and neutrophilic inflammation in COPD. Thorax. 2004;59(10):837–42.\nSchwartz J, Weiss ST. Cigarette smoking and peripheral blood leukocyte differentials. Ann Epidemiol. 1994;4(3):236–42.\nPatel IS, Seemungal TA, Wilks M, Lloyd-Owen SJ, Donaldson GC, Wedzicha JA. Relationship between bacterial colonisation and the frequency, character, and severity of COPD exacerbations. Thorax. 2002;57(9):759–64.\nMcNerlan SE, Alexander HD, Rea IM. Age-related reference intervals for lymphocyte subsets in whole blood of healthy individuals. Scand J Clin Lab Invest. 1999;59(2):89–92.\nFraker PJ, Lill-Elghanian DA. The many roles of apoptosis in immunity as modified by aging and nutritional status. J Nutr Health Aging. 2004;8(1):56–63.\nFabbri LM, Rabe KF. From COPD to chronic systemic inflammatory syndrome? Lancet. 2007;370(9589):797–9.\nBhat T, Teli S, Rijal J, Bhat H, Raza M, Khoueiry G, Meghani M, Akhtar M, Costantino T. Neutrophil to lymphocyte ratio and cardiovascular diseases: a review. Expert Rev Cardiovasc Ther. 2013;11(1):55–9.\nVestbo J, Hurd SS, Agusti AG, Jones PW, Vogelmeier C, Anzueto A, Barnes PJ, Fabbri LM, Martinez FJ, Nishimura M, et al. Global strategy for the diagnosis, management, and prevention of chronic obstructive pulmonary disease: GOLD executive summary. Am J Respir Crit Care Med. 2013;187(4):347–65.\nPavord ID, Jones PW, Burgel PR, Rabe KF. Exacerbations of COPD. Int J Chron Obstruct Pulmon Dis. 2016;11 Spec Iss:21–30.\nVedel-Krogh S, Nielsen SF, Lange P, Vestbo J, Nordestgaard BG. Blood eosinophils and exacerbations in chronic obstructive pulmonary disease. The Copenhagen general population study. Am J Respir Crit Care Med. 2016;193(9):965–74.\nPavord ID, Lettis S, Locantore N, Pascoe S, Jones PW, Wedzicha JA, Barnes NC. Blood eosinophils and inhaled corticosteroid\u002Flong-acting beta-2 agonist efficacy in COPD. Thorax. 2016;71(2):118–25.\nPascoe S, Locantore N, Dransfield MT, Barnes NC, Pavord ID. Blood eosinophil counts, exacerbations, and response to the addition of inhaled fluticasone furoate to vilanterol in patients with chronic obstructive pulmonary disease: a secondary analysis of data from two parallel randomised controlled trials. Lancet Respir Med. 2015;3(6):435–42.\nSong JH, Lee CH, Kim JW, Lee WY, Jung JY, Park JH, Jung KS, Yoo KH, Park YB, Kim DK. Clinical implications of blood eosinophil count in patients with non-asthma-COPD overlap syndrome COPD. Int J Chron Obstruct Pulmon Dis. 2017;12:2455–64.\nO'Reilly PJ, Jackson PL, Wells JM, Dransfield MT, Scanlon PD, Blalock JE. Sputum PGP is reduced by azithromycin treatment in patients with COPD and correlates with exacerbations. BMJ Open. 2013;3(12):e004140.\nWells JM, Jackson PL, Viera L, Bhatt SP, Gautney J, Handley G, King RW, Xu X, Gaggar A, Bailey WC, et al. A randomized, placebo-controlled trial of roflumilast. Effect on proline-glycine-proline and neutrophilic inflammation in chronic obstructive pulmonary disease. Am J Respir Crit Care Med. 2015;192(8):934–42.\nSorensen AK, Holmgaard DB, Mygind LH, Johansen J, Pedersen C. Neutrophil-to-lymphocyte ratio, calprotectin and YKL-40 in patients with chronic obstructive pulmonary disease: correlations and 5-year mortality - a cohort study. J Inflamm (Lond). 2015;12:20.",{"VOID":2379},"10.1186\u002Fs12890-018-0639-z","https:\u002F\u002Fbmcpulmmed.biomedcentral.com\u002Farticles\u002F10.1186\u002Fs12890-018-0639-z",[2382,2397,2410,2423,2436,2449,2462,2477,2492,2507,2522],{"id":2383,"sortIndex":32,"researcher":28,"roles":2384,"affiliations":2385,"properties":2394,"displayName":2396,"givenName":28,"familyName":28},"088f9627-de7f-41c7-a642-de35a16f62ef",[1000],[2386],{"id":2387,"sortIndex":32,"affiliation":2388,"properties":28},"19bcd76a-7f51-43b8-b8e8-8ee7f2c9f47f",{"id":2387,"createTime":28,"updateTime":28,"relativeEntities":2389,"slug":28,"properties":2390,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2393,"statistic":28},[],{"title":2391},{"VI":2392},"Division of Pulmonary Medicine, Department of Medicine, Keio University, School of Medicine, Tokyo, Japan",[],{"title":2395},{"VI":2396},"Kaori 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remodeling due to increased airway smooth muscle cell (ASMC) mass, likely due to enhanced proliferation, hypertrophy, and migration, has been proven to be highly correlated with decreased lung function in asthma patients. Vascular endothelial growth factor (VEGF) mediates vascular and extravascular remodeling and inflammation and has been proven to be involved in the progression of asthma. Previous studies have focused on the effects of VEGF on ASMC proliferation, but few researchers have focused on the effects of VEGF on human ASMC migration. The purpose of this study was to explore the effect of VEGF on the migration of ASMCs and its related signaling pathway mechanism to provide evidence for the treatment of airway remodeling. We examined the effects of VEGF induction on ASMC migration and explored the mechanisms involved in ASMC migration. We found by wound healing and Transwell assays that VEGF promoted ASMC migration. Through the Cell Counting Kit-8 (CCK-8) experiment, we found that VEGF had no significant effect on the proliferation of ASMCs, which excluded the involvement of cell proliferation in the process of wound healing. Moreover, a cellular immunofluorescence assay showed that VEGF promoted F-actin reorganization, and Western blotting showed that VEGF improved RhoA activation and myosin phosphatase targeting subunit-1 (MYPT1) and myosin light chain (MLC) phosphorylation in ASMCs. Treatment with the ROCK inhibitor Y27632 significantly attenuated the effects of VEGF on MYPT1\u002FMLC activation and cell migration. In conclusion, the results suggest that the promigratory function of VEGF activates the RhoA\u002FROCK pathway, induces F-actin reorganization, improves the migration of ASMCs, and provides a better rationale for targeting the RhoA\u002FROCK pathway for therapeutic approaches in airway remodeling.",{"EN":2591},"Vascular endothelial growth factor induces the migration of human airway smooth muscle cells by activating the RhoA\u002FROCK pathway",{"VOID":2593},"Papi A, Brightling C, Pedersen SE, Reddel HK. Asthma. Lancet. 2018;391(10122):783–800.\nPayne DN, Rogers AV, Adelroth E, Bandi V, Guntupalli KK, Bush A, Jeffery PK. Early thickening of the reticular basement membrane in children with difficult asthma. Am J Respir Crit Care Med. 2003;167(1):78–82.\nNj V, Els P, Jc K. Pauwels Ra: Fluticasone inhibits but does not reverse allergen-induced structural airway changes. Am J Resp Crit Care. 2001;163(3):674–9.\nZhang Y, Saradna A, Ratan R, Ke X, Tu W, Do DC, Hu C, Gao P. RhoA\u002FRho-kinases in asthma: from pathogenesis to therapeutic targets. Clin Transl Immunology. 2020;9(5):e01134.\nPepe C, Foley S, Shannon J, Lemiere C, Olivenstein R, Ernst P, Ludwig MS, Martin JG, Hamid Q. Differences in airway remodeling between subjects with severe and moderate asthma. J Allergy Clin Immunol. 2005;116(3):544–9.\nKaminska M, Foley S, Maghni K, Storness-Bliss C, Coxson H, Ghezzo H, Lemiere C, Olivenstein R, Ernst P, Hamid Q, et al. Airway remodeling in subjects with severe asthma with or without chronic persistent airflow obstruction. J Allergy Clin Immunol. 2009;124(1):45–51.\nJames AL, Elliot JG, Jones RL, Carroll ML, Mauad T, Bai TR, Abramson MJ, McKay KO, Green FH. Airway smooth muscle hypertrophy and hyperplasia in asthma. Am J Respir Crit Care Med. 2012;185(10):1058–64.\nGizycki MJ, Adelroth E, Rogers AV, O'Byrne PM, Jeffery PK. Myofibroblast involvement in the allergen-induced late response in mild atopic asthma. Am J Respir Cell Mol Biol. 1997;16(6):664–73.\nAsai K, Kanazawa H, Kamoi H, Shiraishi S, Hirata K, Yoshikawa J. Increased levels of vascular endothelial growth factor in induced sputum in asthmatic patients. Clin Exp Allergy. 2003;33(5):595–9.\nKanazawa H, Hirata K, Yoshikawa J. Involvement of vascular endothelial growth factor in exercise induced bronchoconstriction in asthmatic patients. Thorax. 2002;57(10):885–8.\nMeyer N, Akdis CA. Vascular endothelial growth factor as a key inducer of angiogenesis in the asthmatic airways. Curr Allergy Asthm R. 2013;13(1):1–9.\nPei QM, Jiang P, Yang M, Qian XJ, Liu JB, Zheng H, Zhao LH, Kim SH. Upregulation of a disintegrin and metalloproteinase-33 by VEGF in human airway smooth muscle cells: implications for asthma. Cell Cycle. 2016;15(20):2819–26.\nBanerjee S, Mehta S, Haque I, Sengupta K, Dhar K, Kambhampati S, Van Veldhuizen PJ, Banerjee SK. VEGF-A165 induces human aortic smooth muscle cell migration by activating neuropilin-1-VEGFR1-PI3K axis. Biochem –Us. 2008;47(11):3345–51.\nStone OA, Carter JG, Lin PC, Paleolog E, Machado MJ, Bates DO. Differential regulation of blood flow-induced neovascularization and mural cell recruitment by vascular endothelial growth factor and angiopoietin signalling. J Physiol. 2017;595(5):1575–91.\nYamada KM, Sixt M. Mechanisms of 3D cell migration. Nat Rev Mol Cell Biol. 2019;20(12):738–52.\nWarner H, Wilson BJ, Caswell PT. Control of adhesion and protrusion in cell migration by rho GTPases. Curr Opin Cell Biol. 2019;56:64–70.\nHall A. Rho GTPases and the control of cell behaviour. Biochem Soc Trans. 2005;33(Pt 5):891–5.\nTian W, Chen W, Chen Y, Lin C, Huang J, Song Y, Yang Y. The role and mechanism of 1,25-dihydroxyvitamin D3 in regulating the rho-kinase signaling pathway in asthmatic rats. Translat Pediatr. 2021;10(4):773–82.\nXu C, Wu X, Lu M, Tang L, Yao H, Wang J, Ji X, Hussain M, Wu J, Wu X. Protein tyrosine phosphatase 11 acts through RhoA\u002FROCK to regulate eosinophil accumulation in the allergic airway. Faseb J. 2019;33(11):11706–20.\nLauffenburger DA, Horwitz AF. Cell migration: a physically integrated molecular process. Cell. 1996;84(3):359–69.\nQu C, Yang W, Kan Y, Zuo H, Wu M, Zhang Q, Wang H, Wang D, Chen J. RhoA\u002FROCK signaling regulates Drp1-mediated mitochondrial fission during collective cell migration. Front Cell Dev Biol. 2022;10:882581.\nLv C, Liao G, Wu L, Li J, Gao Y. Effects of different intervention factors on vascular endothelial growth factor-induced human airway smooth muscle cell migration. Can Respir J. 2022;2022(1):6879539.\nSenger DR, Galli SJ, Dvorak AM, Perruzzi CA, Harvey VS, Dvorak HF. Tumor cells secrete a vascular permeability factor that promotes accumulation of ascites fluid. Science. 1983;219(4587):983–5.\nSalter B, Pray C, Radford K, Martin JG, Nair P. Regulation of human airway smooth muscle cell migration and relevance to asthma. Respir Res. 2017;18(1):156.\nPei QM, Jiang P, Yang M, Qian XJ, Liu JB, Kim SH. Roxithromycin inhibits VEGF-induced human airway smooth muscle cell proliferation: opportunities for the treatment of asthma. Exp Cell Res. 2016;347(2):378–84.\nKim SH, Pei QM, Jiang P, Yang M, Qian XJ, Liu JB. Role of licochalcone a in VEGF-induced proliferation of human airway smooth muscle cells: implications for asthma. Growth Factors. 2017;35(1):39–47.\nKim SH, Pei QM, Jiang P, Yang M, Qian XJ, Liu JB. Effect of active vitamin D3 on VEGF-induced ADAM33 expression and proliferation in human airway smooth muscle cells: implications for asthma treatment. Respir Res. 2017;18(1):7.\nStamatiou R, Paraskeva E, Gourgoulianis K, Molyvdas PA, Hatziefthimiou A. Cytokines and growth factors promote airway smooth muscle cell proliferation. ISRN Inflamm. 2012;2012:731472.\nOda T, Iwasa M, Aihara T, Maéda Y, Narita A. The nature of the globular- to fibrous-actin transition. Nature. 2009;457(7228):441–5.\nHall A, Nobes CD. Rho GTPases: molecular switches that control the organization and dynamics of the actin cytoskeleton. Philosophical transactions of the Royal Society of London. Series B: Biolog Sci. 2000;355(1399):965–70.\nCiocanel MV, Chandrasekaran A, Mager C, Ni Q, Papoian GA, Dawes A. Simulated actin reorganization mediated by motor proteins. PLoS Comput Biol. 2022;18(4):e1010026.\nTang L, Dai F, Liu Y, Yu X, Huang C, Wang Y, Yao W. RhoA\u002FROCK signaling regulates smooth muscle phenotypic modulation and vascular remodeling via the JNK pathway and vimentin cytoskeleton. Pharmacol Res. 2018;133:201–12.\nRaftopoulou M, Hall A. Cell migration: rho GTPases lead the way. Dev Biol. 2004;265(1):23–32.\nBrito C, Sousa S. Non-muscle myosin 2A (NM2A): structure, regulation and function. Cells-Basel. 2020;9(7):1590.\nAi S, Kuzuya M, Koike T, Asai T, Kanda S, Maeda K, Shibata T, Iguchi A. Rho-rho kinase is involved in smooth muscle cell migration through myosin light chain phosphorylation-dependent and independent pathways. Atherosclerosis. 2001;155(2):321–7.\nPette D, Staron RS. Myosin isoforms, muscle fiber types, and transitions. Microsc Res Tech. 2000;50(6):500–9.\nYang T, Fang F, Chen Y, Ma J, Xiao Z, Zou S, Zheng N, Yan D, Liao S, Chen S, et al. Elevated plasma interleukin-37 playing an important role in acute coronary syndrome through suppression of ROCK activation. Oncotarget. 2017;8(6):9686–95.\nGao N, Tsai MH, Chang AN, He W, Chen CP, Zhu M, Kamm KE, Stull JT. Physiological vs. pharmacological signalling to myosin phosphorylation in airway smooth muscle. J Physiol. 2017;595(19):6231–47.\nRodríguez-Trillo A, Pena C, García S, Pérez-Pampín E, Rodríguez-López M, Mera-Varela A, González A, Conde C. ROCK inhibition with Y-27632 reduces joint inflammation and damage in serum-induced arthritis model and decreases in vitro osteoclastogenesis in patients with early arthritis. Front Immunol. 2022:13.",{"VOID":2595},"10.1186\u002Fs12890-023-02803-y","https:\u002F\u002Fbmcpulmmed.biomedcentral.com\u002Farticles\u002F10.1186\u002Fs12890-023-02803-y",[2598,2613,2626,2639],{"id":2599,"sortIndex":32,"researcher":28,"roles":2600,"affiliations":2601,"properties":2610,"displayName":2612,"givenName":28,"familyName":28},"7da37945-a0d7-49f8-ad2e-574e71c7b06c",[1000],[2602],{"id":2603,"sortIndex":32,"affiliation":2604,"properties":28},"da507db1-cedf-40cc-a081-133c1951a248",{"id":2603,"createTime":28,"updateTime":28,"relativeEntities":2605,"slug":28,"properties":2606,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2609,"statistic":28},[],{"title":2607},{"VI":2608},"Department of Pulmonary and Critical Care Medicine; Guangdong Provincial Key Laboratory of Major Obstetric Diseases, Guangdong Provincial Clinical Research Center for Obstetrics and Gynecology, The Third Affiliated Hospital of Guangzhou Medical University, Guangzhou, China",[],{"title":2611},{"VI":2612},"Chengtian Lv",{"id":2614,"sortIndex":40,"researcher":28,"roles":2615,"affiliations":2616,"properties":2623,"displayName":2625,"givenName":28,"familyName":28},"2d8ffc52-d914-412b-93c2-6621123de418",[1000],[2617],{"id":2603,"sortIndex":32,"affiliation":2618,"properties":28},{"id":2603,"createTime":28,"updateTime":28,"relativeEntities":2619,"slug":28,"properties":2620,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2622,"statistic":28},[],{"title":2621},{"VI":2608},[],{"title":2624},{"VI":2625},"Yuwen Huang",{"id":2627,"sortIndex":123,"researcher":28,"roles":2628,"affiliations":2629,"properties":2636,"displayName":2638,"givenName":28,"familyName":28},"37738782-bd6c-4b41-b901-0f71b8dffb39",[1000],[2630],{"id":2603,"sortIndex":32,"affiliation":2631,"properties":28},{"id":2603,"createTime":28,"updateTime":28,"relativeEntities":2632,"slug":28,"properties":2633,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2635,"statistic":28},[],{"title":2634},{"VI":2608},[],{"title":2637},{"VI":2638},"Ruirong Yan",{"id":2640,"sortIndex":42,"researcher":28,"roles":2641,"affiliations":2642,"properties":2649,"displayName":2651,"givenName":28,"familyName":28},"76682d69-b0fa-4fd1-91fc-1736ed12474b",[1000],[2643],{"id":2603,"sortIndex":32,"affiliation":2644,"properties":28},{"id":2603,"createTime":28,"updateTime":28,"relativeEntities":2645,"slug":28,"properties":2646,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2648,"statistic":28},[],{"title":2647},{"VI":2608},[],{"title":2650},{"VI":2651},"Yuanmei Gao",{"url":2596,"publisher":2653,"properties":2694},{"id":868,"createTime":869,"updateTime":870,"relativeEntities":2654,"slug":872,"properties":2655,"entityType":25,"verifyStatus":878,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":32,"subjectFields":2658,"manageAffiliations":2663,"indexDatabases":2674,"url":28,"thumbnailPath":28,"statistic":2689,"gsStatistic":28,"type":28,"analyzePriority":28},[],{"title":2656,"eissn":2657},{"EN":875},{"VOID":877},[2659],{"id":881,"createTime":28,"updateTime":28,"relativeEntities":2660,"label":2661,"description":2662,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":884},{},[2664,2669],{"id":888,"createTime":28,"updateTime":28,"relativeEntities":2665,"slug":28,"properties":2666,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2668,"statistic":28},[],{"title":2667},{"EN":892},[],{"id":895,"createTime":28,"updateTime":28,"relativeEntities":2670,"slug":28,"properties":2671,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2673,"statistic":28},[],{"title":2672},{"EN":899},[],[2675,2682],{"id":919,"indexDatabase":2676,"url":925,"indexYears":926,"academicFieldIds":2681,"indexDatabaseRanking":929},{"id":775,"createTime":28,"updateTime":28,"relativeEntities":2677,"label":2678,"description":2679,"key":781,"publicationTags":2680,"standard":28},[],{"EN":778,"VI":778},{"EN":778,"VI":780},[783],[928],{"id":903,"indexDatabase":2683,"url":915,"indexYears":28,"academicFieldIds":2688,"indexDatabaseRanking":28},{"id":905,"createTime":28,"updateTime":28,"relativeEntities":2684,"label":2685,"description":2686,"key":912,"publicationTags":2687,"standard":28},[],{"EN":908,"VI":908},{"EN":910,"VI":911},[914,813],[917],{"impactFactor":32,"impactFactorByYear":2690,"i10Index":934,"i10IndexLast5Year":516,"totalPublication":935,"totalPublicationByYear":2691,"totalCitation":942,"totalCitationByYear":2692,"totalCitationPerPublication":372,"totalCitationPerPublicationByYear":2693,"hindexLast5Year":196,"hindex":196},{"2012":319,"2013":932,"2014":229,"2015":346,"2016":227,"2017":228,"2018":709,"2019":226,"2020":114,"2021":368,"2022":119,"2023":933},{"2001":45,"2002":42,"2003":42,"2004":205,"2005":205,"2006":135,"2007":146,"2008":129,"2009":278,"2010":279,"2011":278,"2012":689,"2013":436,"2014":529,"2015":937,"2016":938,"2017":359,"2018":331,"2019":836,"2020":606,"2021":939,"2022":940,"2023":941,"2024":687},{"2001":45,"2004":944,"2006":361,"2007":135,"2008":329,"2009":945,"2010":612,"2011":946,"2012":947,"2013":948,"2014":949,"2015":950,"2016":951,"2017":952,"2018":953,"2019":954,"2020":955,"2021":956,"2022":957},{"2001":40,"2004":959,"2006":960,"2007":961,"2008":962,"2009":963,"2010":964,"2011":965,"2012":966,"2013":45,"2014":967,"2015":968,"2016":969,"2017":970,"2018":971,"2019":972,"2020":371,"2021":363,"2022":462},{"pages":2695,"volume":2696},{"VOID":2579},{"VOID":2697},"23","2023-12-13",2023,[914,929],{"id":2702,"createTime":2703,"updateTime":2704,"relativeEntities":2705,"slug":2706,"properties":2707,"entityType":992,"verifyStatus":26,"verifyTime":2704,"verifyNote":994,"languages":28,"translateLanguages":28,"viewCount":32,"primaryUrl":2716,"fullTextUrl":28,"authors":2717,"publicationType":1132,"publisherRelationship":2776,"citationCount":28,"citationInfo":28,"publishDate":2822,"publishYear":2823,"citationAnalyzeStatus":878,"lastCitationAnalyze":28,"indexDatabases":2824,"openAccess":28,"references":28,"isForceReanalyzing":1183},"00fc9ee1-47ff-43c5-81d3-8bc8bbe25bc0","2024-01-08T14:18:33.869+00:00","2024-09-19T06:00:20.665+00:00",[],"Comparison-of-postoperative-cough-related-quality-of-life-and-recovery-between-sublobectomy-and-lobectomy-for-early-stage-non-small-cell-lung-cancer-patients-a-longitudinal-study",{"abstract":2708,"title":2710,"references":2712,"doi":2714},{"EN":2709},"Cough is a common complication after pulmonary surgery. Previous studies lacked a standard measure to assess postoperative cough-related quality of life and recovery. The purpose of this study is to compare postoperative cough regarding changes in health-related quality of life (HRQOL) and recovery trajectory between video-assisted thoracic surgery (VATS) lobectomy and sublobectomy (segmentectomy or wedge resection) for early-stage non-small cell lung cancer (NSCLC) patients via the Leicester Cough Questionnaire in Mandarin Chinese (LCQ-MC). Overall, 156 patients with NSCLC underwent either VATS lobectomy or VATS sublobectomy; LCQ-MC was used to report the impact of postoperative cough on HRQOL for 6 months after surgery. The total scores of LCQ-MC range from 3 to 21, with a higher score indicating better health. Recovery from postoperative cough was defined as LCQ-MC scores returning to preoperative levels. The sensitivity of LCQ-MC to changes in postoperative cough recovery over time was evaluated via its ability to distinguish between surgery types. The VATS sublobectomy group reported significantly higher mean LCQ-MC scores at 1 month after surgery, but no significant difference postoperatively at 3 and 6 months after surgery, and returned to preoperative physical (69 vs. 99 days), psychological (67 vs. 99 days), social (50 vs. 98 days) and total (69 vs. 99 days) scores faster than the VATS lobectomy group (all p \u003C 0.05). VATS sublobectomy had generally better HRQOL and faster recovery of postoperative cough than VATS lobectomy. In addition, the LCQ-MC performed satisfactorily in describing the longitudinal changes in postoperative cough.",{"EN":2711},"Comparison of postoperative cough-related quality of life and recovery between sublobectomy and lobectomy for early-stage non-small cell lung cancer patients: a longitudinal study",{"VOID":2713},"Sarna L, Evangelista L, Tashkin D, Padilla G, Holmes C, Brecht ML, Grannis F. Impact of respiratory symptoms and pulmonary function on quality of life of long-term survivors of non-small cell lung cancer. Chest. 2004;125(2):439–45.\nYang P, Cheville AL, Wampfler JA, Garces YI, Jatoi A, Clark MM, Cassivi SD, Midthun DE, Marks RS, Aubry MC, et al. Quality of life and symptom burden among long-term lung cancer survivors. J Thorac Oncol Off Publ Int Assoc Study Lung Cancer. 2012;7(1):64–70.\nLin R, Che G. Risk factors of cough in non-small cell lung cancer patients after video-assisted thoracoscopic surgery. J Thorac Dis. 2018;10(9):5368–75.\nXie M-r, Zhu Y-f, Zhou M-q, Wu S-b, Xu G-w, Xu S-b, Xu M-q. Analysis of factors related to chronic cough after lung cancer surgery. Thoracic Cancer. 2019;10(4):898–903.\nKvale PA. Chronic cough due to lung tumors: ACCP evidence-based clinical practice guidelines. Chest. 2006;129(1 Suppl):147s–53s.\nAgha R, Abdall-Razak A, Crossley E, Dowlut N, Iosifidis C, Mathew G, Beamishaj BM, Millham FH, et al. Guideline: strengthening the reporting of cohort studies in surgery. Int J Surg. 2019;2019(72):156–65.\nChu XY, Xue ZQ, Liu Y, Zhang LB, Hou XB, Hua YU. Single utility port complete video-assisted thoracoscopic surgery lobectomy for patients with early-stage lung cancer. Chin J Clin Thorac Cardiovasc Surg. 2012;2012:3165.\nZhi XY, Yu JM, Shi YK. Chinese guidelines on the diagnosis and treatment of primary lung cancer (2015 version). Cancer. 2015;121(Suppl 17):3165–81.\nDarling GE, Allen MS, Decker PA, Ballman K, Malthaner RA, Inculet RI, Jones DR, McKenna RJ, Landreneau RJ, Rusch VW, et al. Randomized trial of mediastinal lymph node sampling versus complete lymphadenectomy during pulmonary resection in the patient with N0 or N1 (less than hilar) non-small cell carcinoma: results of the American College of Surgery Oncology Group Z0030 Trial. J Thorac Cardiovasc Surg. 2011;141(3):662–70.\nAsamura H, Chansky K, Crowley J, Goldstraw P, Rusch VW, Vansteenkiste JF, Watanabe H, Wu YL, Zielinski M, Ball D, et al. The International Association for the study of lung cancer lung cancer staging project: proposals for the revision of the N descriptors in the forthcoming 8th edition of the TNM Classification for Lung Cancer. J Thoracic Oncol Off Publ Int Assoc Study Lung Cancer. 2015;10(12):1675–84.\nBirring SS, Prudon B, Carr AJ, Singh SJ, Morgan MD, Pavord ID. Development of a symptom specific health status measure for patients with chronic cough: Leicester Cough Questionnaire (LCQ). Thorax. 2003;58(4):339–43.\nGao YH, Guan WJ, Xu G, Gao Y, Lin ZY, Tang Y, Lin ZM, Li HM, Luo Q, Zhong NS, et al. Validation of the Mandarin Chinese version of the Leicester Cough Questionnaire in bronchiectasis. Int J Tuberc Lung Dis. 2014;18(12):1431–7.\nLin R, Che G. Validation of the Mandarin Chinese version of the Leicester Cough Questionnaire in non-small cell lung cancer patients after surgery. Thorac Cancer. 2018;9(4):486–90.\nTravis WD, Brambilla E, Burke AP, Marx A, Nicholson AG. Introduction to The 2015 World Health Organization Classification of Tumors of the Lung, Pleura, Thymus, and Heart. J Thorac Oncol Off Publ Int Assoc Study Lung Cancer. 2015;10(9):1240–2.\nDetterbeck FC, Boffa DJ, Kim AW, Tanoue LT. The eighth edition lung cancer stage classification. Chest. 2017;151(1):193–203.\nRusch VW, Asamura H, Watanabe H, Giroux DJ, Rami-Porta R, Goldstraw P. The IASLC lung cancer staging project: a proposal for a new international lymph node map in the forthcoming seventh edition of the TNM classification for lung cancer. J Thorac Oncol Off Publ Int Assoc Study Lung Cancer. 2009;4(5):568–77.\nBirring SS, Spinou A. How best to measure cough clinically. Curr Opin Pharmacol. 2015;22:37–40.\nGinsberg RJ, Rubinstein LV. Randomized trial of lobectomy versus limited resection for T1 N0 non-small cell lung cancer: Lung Cancer Study Group. Ann Thorac Surg. 1995;60(3):615–22.\nZhang Z, Feng H, Zhao H, Hu J, Liu L, Liu Y, Li X, Xu L, Li Y, Lu X, et al. Sublobar resection is associated with better perioperative outcomes in elderly patients with clinical stage I non-small cell lung cancer: a multicenter retrospective cohort study. J Thorac Dis. 2019;11(5):1838–48.\nDivisi D, De Vico A, Zaccagna G, Crisci R. Lobectomy versus sublobar resection in patients with non-small cell lung cancer: a systematic review. J Thorac Dis. 2020;12(6):3357–62.\nMageed NA, El-Ghonaimy YA, Elgamal MA, Hamza U. Acute effects of lobectomy on right ventricular ejection fraction and mixed venous oxygen saturation. Ann Saudi Med. 2005;25(6):481–5.\nSeok Y, Cho S, Lee JY, Yang HC, Kim K, Jheon S. The effect of postoperative change in bronchial angle on postoperative pulmonary function after upper lobectomy in lung cancer patients. Interact Cardiovasc Thorac Surg. 2014;18(2):183.\nCanning BJ, Chang AB, Bolser DC, Smith JA, Mazzone SB, McGarvey L. Anatomy and neurophysiology of cough: CHEST guideline and expert panel report. Chest. 2014;146(6):1633–48.\nIyer S, Roughley A, Rider A, Taylor-Stokes G. The symptom burden of non-small cell lung cancer in the USA: a real-world cross-sectional study. Supportive Care Cancer Off J Multinatl Assoc Supportive Care Cancer. 2014;22(1):181–7.\nLi WW, Lee TW, Yim AP. Quality of life after lung cancer resection. Thorac Cardiovasc Surg. 2004;14(3):353–65.\nPoghosyan H, Sheldon LK, Leveille SG, Cooley ME. Health-related quality of life after surgical treatment in patients with non-small cell lung cancer: a systematic review. Lung cancer (Amsterdam, Netherlands). 2013;81(1):11–26.\nCheng X, Yang Y, Shentu Y, Ding Z, Zhou Q, Tan Q, Luo Q. Remote monitoring of patient recovery following lung cancer surgery: a messenger application approach. J Thorac Dis. 2021;13(2):1162–71.\nBilimoria KY, Cella D, Butt Z. Current challenges in using patient-reported outcomes for surgical care and performance measurement: everybody wants to hear from the patient, but are we ready to listen? JAMA Surg. 2014;149:505.\nMorice AH, Fontana GA, Belvisi MG, Birring SS, Chung KF, Dicpinigaitis PV, Kastelik JA, McGarvey LP, Smith JA, Tatar M, et al. ERS guidelines on the assessment of cough. Eur Respir J. 2007;29(6):1256–76.\nAltorki NK, Wang X, Wigle D, Gu L, Darling G, Ashrafi AS, Landrenau R, Miller D, Liberman M, Jones DR, et al. Perioperative mortality and morbidity after sublobar versus lobar resection for early-stage non-small-cell lung cancer: post-hoc analysis of an international, randomised, phase 3 trial (CALGB\u002FAlliance 140503). Lancet Respir Med. 2018;6(12):915–24.\nSuzuki K, Saji H, Aokage K, Watanabe SI, Okada M, Mizusawa J, Nakajima R, Tsuboi M, Nakamura S, Nakamura K, et al. Comparison of pulmonary segmentectomy and lobectomy: safety results of a randomized trial. J Thorac Cardiovasc Surg. 2019;158(3):895–907.\nCrosby RD, Kolotkin RL, Williams GR. Defining clinically meaningful change in health-related quality of life. Value Health. 2003;56(5):395–407.\nRaj AA, Pavord DI, Birring SS. Clinical cough IV: What is the minimal important difference for the Leicester Cough Questionnaire? 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