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In the active LN population, serum and urine PGRN levels were not only significantly correlated with SLEDAI score, rSLEDAI score, and activity index, but also had a considerable association with several key markers reflecting the disease activity of LN, including serum levels of complement component 3 and ds-DNA. Nevertheless, neither of the two indicators were correlated with the pathological classification of LN, chronicity index, serum creatinine level, and 24-h urine protein levels. 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Arthritis Rheum 35(6):630–640",{},{"id":403,"text":457,"url":405,"identifiers":458},"Weening JJ, D’Agati VD, Schwartz MM et al (2004) The classification of glomerulonephritis in systemic lupus erythematosus revisited. J Am Soc Nephrol 15(2):241–250",{"doi":407},{"id":460,"text":461,"url":462,"identifiers":463},"f007c493-4136-4e2a-8bb4-87566388b0af","Austin HA 3rd, Muenz LR, Joyce KM, Antonovych TT, Balow JE (1984) Diffuse proliferative lupus nephritis: identification of specific pathologic features affecting renal outcome. Kidney Int 25(4):689–695","https:\u002F\u002Fwww.sciencedirect.com\u002Fscience\u002Farticle\u002Fpii\u002FS0085253815331847",{"doi":464},"10.1038\u002Fki.1984.75",{"id":403,"text":466,"url":405,"identifiers":467},"Bradford MM (1976) A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding. Anal Biochem 72:248–254",{"doi":407},{"id":403,"text":469,"url":405,"identifiers":470},"DeLong ER, DeLong DM, Clarke-Pearson DL (1988) Comparing the areas under two or more correlated receiver operating characteristic curves: a nonparametric approach. Biometrics 44(3):837–845",{"doi":407},{"id":403,"text":472,"url":405,"identifiers":473},"Lech M, Anders HJ (2013) The pathogenesis of lupus nephritis. J Am Soc Nephrol 24(9):1357–1366",{"doi":407},{"id":403,"text":475,"url":405,"identifiers":476},"Borchers AT, Leibushor N, Naguwa SM, Cheema GS, Shoenfeld Y, Gershwin ME (2012) Lupus nephritis: a critical review. Autoimmun Rev 12(2):174–194",{"doi":407},{"id":18,"text":478,"url":18,"identifiers":479},"Lorenz G, Desai J, Anders HJ (2014) Lupus nephritis: update on mechanisms of systemic autoimmunity and kidney immunopathology. Curr Opin Nephrol Hypertens 23(3):211–217",{},{"id":403,"text":481,"url":405,"identifiers":482},"van der Vlag J, Berden JH (2011) Lupus nephritis: role of antinucleosome autoantibodies. Semin Nephrol 31(4):376–389",{"doi":407},{"id":403,"text":484,"url":405,"identifiers":485},"Sprangers B, Monahan M, Appel GB (2012) Diagnosis and treatment of lupus nephritis flares—an update. Nat Rev Nephrol 8(12):709–717",{"doi":407},{"id":403,"text":487,"url":405,"identifiers":488},"Cash H, Relle M, Menke J et al (2010) Interleukin 6 (IL-6) deficiency delays lupus nephritis in MRL-Faslpr mice: the IL-6 pathway as a new therapeutic target in treatment of autoimmune kidney disease in systemic lupus erythematosus. J Rheumatol 37(1):60–70",{"doi":407},{"id":490,"text":491,"url":492,"identifiers":493},"a4c8c9c1-4c5f-4a32-83fe-2c59f01e53d2","Anders HJ, Fogo AB (2014) Immunopathology of lupus nephritis. Semin Immunopathol 36(4):443–459","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs00281-013-0413-5",{"doi":494},"10.1007\u002Fs00281-013-0413-5",{"id":403,"text":496,"url":405,"identifiers":497},"Frieri M, Samih MA, Dzhindzhikhashvili M, Liu H, Balsam L, Rubinstein S (2012) Toll-like receptor 9 and vascular endothelial growth factor levels in human kidneys from lupus nephritis patients. J Nephrol 25(6):1041–1046",{"doi":407},{"id":403,"text":499,"url":405,"identifiers":500},"Machida H, Ito S, Hirose T et al (2010) Expression of Toll-like receptor 9 in renal podocytes in childhood-onset active and inactive lupus nephritis. Nephrol Dial Transplant 25(8):2530–2537",{"doi":407},{"id":403,"text":502,"url":405,"identifiers":503},"Tang W, Lu Y, Tian QY et al (2011) The growth factor progranulin binds to TNF receptors and is therapeutic against inflammatory arthritis in mice. Science 332(6028):478–484",{"doi":407},{"id":403,"text":505,"url":405,"identifiers":506},"Postal M, Appenzeller S (2011) The role of tumor necrosis factor-alpha (TNF-alpha) in the pathogenesis of systemic lupus erythematosus. Cytokine 56(3):537–543",{"doi":407},false,{"id":509,"createTime":510,"updateTime":511,"relativeEntities":512,"slug":513,"properties":514,"entityType":246,"verifyStatus":247,"verifyTime":525,"verifyNote":249,"languages":18,"translateLanguages":18,"viewCount":19,"primaryUrl":526,"fullTextUrl":18,"authors":527,"publicationType":339,"publisherRelationship":593,"citationCount":18,"citationInfo":18,"publishDate":648,"publishYear":649,"citationAnalyzeStatus":650,"lastCitationAnalyze":651,"indexDatabases":652,"openAccess":18,"references":18,"isForceReanalyzing":507},"447fe7ff-5021-4fe0-8183-adfa70ef21d8","2024-01-04T06:51:48.302+00:00","2026-08-24T20:06:36.224+00:00",[],"Prevalence-and-annual-incidence-of-vertebral-fractures-in-patients-with-ankylosing-spondylitis",{"abstract":515,"title":517,"gsPaper":519,"references":521,"doi":523},{"EN":516},"Objectives To evaluate the prevalence and annual incidence of clinically-manifest vertebral fractures among patients with ankylosing spondylitis (AS). Method: Coordinated by the Ankylosing Spondylitis International Federation, a self-administered general questionnaire which included some questions on gender, age, age at onset of disease, and a history of vertebral fracture was inserted in one issue of the membership journals of the AS patient organizations in Germany and Austria. Results: Among the 1,071 patients responding who all had indicated that the diagnosis of AS had been established or confirmed by a physician, 61 (5.7%) indicated a history of vertebral fracture, 15 of them (1.4%) without an accident. The prevalence of vertebral fractures was 6.2% among male AS patients and 4.6% among females (NS), and 4.8% among HLA-B27+ patients and 9.9% among HLA-B27− patients (p\u003C0.05). Spinal fractures occurred more often among AS patients with peripheral arthritis (7.1%) than among patients with axial involvement only (3.1%, p\u003C0.01). The average delay between disease onset and diagnosis of AS was 10.5 years for patients with a vertebral fracture, compared to 8.7 years for patients without any such event (p\u003C0.05). Among patients with a disease duration ≥42 years, the prevalence of vertebral fractures was 14%. The annual incidence of vertebral fractures which occurred without an accident had a maximum of 0.1% per annum at a disease duration of 20–35 years, whereas the incidence of vertebral fractures caused by an accident increased continuously with increasing disease duration, amounting to 1.3% per annum after a disease duration of 45 years. Conclusion: A considerable proportion of AS patients will experience a vertebral fracture during the course of the disease, in particular if peripheral joints are also involved.",{"EN":518},"Prevalence and annual incidence of vertebral fractures in patients with ankylosing spondylitis",{"VOID":520},"[]",{"VOID":522},"van der Linden SM, Valkenburg HA, de Jongh BM, Cats A (1984) The risk of developing ankylosing spondylitis in HLA-B27 positive individuals: a comparison of relatives of spondylitis patients with the general population. Arthritis Rheum 27:241–249\nvan der Linden SM (1991) Newer clinical, epidemiology and genetical findings in ankylosing spondylitis and related spondylarthropathies. In: Lipsky PE, Taurog JD (eds) HLA-B27+ Spondylarthropathies. Elsevier Science Publishing, New York, pp 145–151\nBraun J, Bollow M, Remlinger G, Eggens U, Rudwaleit M, Distler A, Sieper J (1998) Prevalence of spondylarthropathies in HLA-B27 positive and negative blood donors. Arthritis Rheum 41:58–67\nBruges-Armas J, Lima C, Peixoto MJ, Santos P, Mendonca D, da Silva BM, Herrero-Beaumont G, Calin A (2002) Prevalence of spondyloarthritis in Terceira, Azores: a population based study. Ann Rheum Dis 61:551–553\nFeldtkeller E (1999) Erkrankungsalter und Diagnoseverzögerung bei Spondylarthropathien. Z Rheumatol 58:21–30\nFeldtkeller E, Lemmel EM (1999) Zur Situation von Spondyloarthritis-Patienten. Ergebnisse einer Repräsentativbefragung der Deutschen Vereinigung Morbus Bechterew. Novartis Pharma Verlag, Nürnberg\nFeldtkeller E, Bruckel J, Khan MA (2000) Scientific contributions of ankylosing spondylitis patient advocacy groups. Curr Opin Rheumatol 12:239–247\nFeldtkeller E (1998) Unterschiede im Krankheitsverlauf männlicher und weiblicher Spondylarthritis-Patienten. Akt Rheumatol 23:145–153\nEl Maghraoui A, Borderie D, Cherruau B, Edouard R, Dougados M, Roux C (1999) Osteoporosis, body composition, and bone turnover in ankylosing spondylitis. J Rheumatol 26:2205–2209\nLange U (2002) Ankylosierende Spondylitis und Osteoporose—eine aktuelle Übersicht. Akt Rheumatol 27:147–151\nBölzner C, Müller A, Bräunig E, Hein G (2003) Kollagenabbauaktivität bei Spondylitis ankylosans in Beziehung zu krankheitsrelevanten Daten. Z Rheumatol 62:459–467\nSchilling F (2003) Osteoporose der ankylosierenden Spondylitis in Abhängigkeit von deren Verlaufstypen. Z Rheumatol 62:492–495\nBessant R, Keat A (2002) How should clinicians manage osteoporosis in ankylosing spondylitis?. J Rheumatol 29:1511–1519\nGran JT, Husby G (1998) Clinical, epidemiological, and therapeutic aspects of ankylosing spondylitis. Curr Opin Rheumatol 10:292–298\nDevogelaer JP, Maldague B, Malghem J, Nagant de Deuxchaisnes C (1992) Appendicular and vertebral bone mass in ankylosing spondylitis. A comparison of plain radiographs with single- and dual-photon absorptiometry and with quantitative computed tomography. Arthritis Rheum 35:1062–1067\nDonnelly S, Jawed S, Meija A, Doyle DV (1995) Effect of syndesmophyte formation on lumbar spine bone mineral density in patients with ankylosing spondylitis. Br J Rheumatol 34(Suppl):117\nLee YS, Schlotzhauer T, Ott SM, van Vollenhoven RF, Hunter J, Marcus R, McGuire JL (1997) Skeletal status of men with early and late ankylosing spondylitis. Am J Med 103:233–241\nBronson WD, Walker SE, Hillman LS, Keisler D, Hoyt T, Allen SH (1998) Bone mineral density and biochemical markers of bone metabolism in ankylosing spondylitis. J Rheumatol 25:929–935\nReid DM, Nicoll JJ, Kennedy NJS, Smith MA, Tothill P, Nuki G (1986) Bone mass in ankylosing spondylitis. J Rheumatol 13:932–935\nDonnelly S, Doyle DV, Denton A, Rolfe I, McCloskey EV, Spector TD (1994) Bone mineral density and vertebral compression fracture rates in ankylosing spondylitis. Ann Rheum Dis 53:117–121\nWill R, Palmer R, Bhalla AK, Ring EFJ, Calin A (1989) Osteoporosis in early ankylosing spondylitis: a primary pathologic event?. Lancet 2:1483–1485\nHanson CA, Shagrin JW, Duncan H (1971) Vertebral osteoporosis in ankylosing spondylitis. Clin Orthop 74:59–64\nMartel W (1978) Spinal pseudoarthrosis: a complication of ankylosing spondylitis. Arthritis Rheum 21:485–490\nHunter T, Dubo HIC (1983) Spinal fractures complicating ankylosing spondylitis. A long-term follow-up study. Arthritis Rheum 26:751–759\nRalston SH, Urquhart GDK, Brzeski M, Sturrock RD (1990) Prevalence of vertebral compression fractures due to osteoporosis in ankylosing spondylitis. BMJ 300:563–565\nCooper C, Carbone L, Michet CJ, Atkinson EJ, O’Fallon WM, Melton LJ 3rd (1994) Fracture risk in patients with ankylosing spondylitis: a population based study. J Rheumatol 21:1877–1882\nMitra D, Elvins DM, Speden DJ, Collins AJ (2000) The prevalence of vertebral fractures in mild ankylosing spondylitis and their relationship to bone mineral density. Rheumatology 39:85–89\nHitchon PW, From AM, Brenton BS, Glaser JA, Torner JC (2002) Fractures of the thoracolumbar spine complicating ankylosing spondylitis. J Neurosurg 97(Suppl 2):218–222\nFeldtkeller E, Khan MA, van der Heijde D, van der Linden S, Braun J (2003) Age at disease onset and diagnosis delay in HLA-B27 negative vs. positive patients with ankylosing spondylitis. Rheumatol Int 23:61–66\nVosse D, Feldtkeller E, Erlendssen J, Geusens P, van der Linden S (2004) Clinically manifest vertebral fractures in patients with ankylosing spondylitis (to be published in J Rheumatol)\nBrophy S, Mackay K, Al-Saidi A, Taylor G, Calin A (2002) The natural history of ankylosing spondylitis as defined by radiological progression. J Rheumatol 29:1236–1243",{"VOID":524},"10.1007\u002Fs00296-004-0556-8","2024-08-31T15:13:28.233+00:00","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs00296-004-0556-8",[528,543,558,580],{"id":529,"sortIndex":19,"researcher":18,"roles":530,"affiliations":531,"properties":540,"displayName":542,"givenName":18,"familyName":18},"d02fea52-f82e-4620-b24b-bbb766035803",[255],[532],{"id":533,"sortIndex":19,"affiliation":534,"properties":18},"cb54577e-a9c0-47fe-b25b-ac3c813a7f4b",{"id":533,"createTime":18,"updateTime":18,"relativeEntities":535,"slug":18,"properties":536,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":539,"statistic":18},[],{"title":537},{"VI":538},"Ankylosing Spondylitis International Federation, München, Germany",[],{"title":541},{"VI":542},"Ernst Feldtkeller",{"id":544,"sortIndex":190,"researcher":18,"roles":545,"affiliations":546,"properties":555,"displayName":557,"givenName":18,"familyName":18},"a12e9f9b-f1b2-4c8d-a4dc-40a3de711b63",[255],[547],{"id":548,"sortIndex":19,"affiliation":549,"properties":18},"68a209f4-ed87-43f1-914a-3d62c8b4a9a7",{"id":548,"createTime":18,"updateTime":18,"relativeEntities":550,"slug":18,"properties":551,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":554,"statistic":18},[],{"title":552},{"VI":553},"Department of Medicine, University Hospital Maastricht, Maastricht, The Netherlands",[],{"title":556},{"VI":557},"Debby Vosse",{"id":559,"sortIndex":283,"researcher":18,"roles":560,"affiliations":561,"properties":577,"displayName":579,"givenName":18,"familyName":18},"288a583e-1c49-4fac-9176-0333bab9ca6a",[255],[562,568],{"id":548,"sortIndex":19,"affiliation":563,"properties":18},{"id":548,"createTime":18,"updateTime":18,"relativeEntities":564,"slug":18,"properties":565,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":567,"statistic":18},[],{"title":566},{"VI":553},[],{"id":569,"sortIndex":190,"affiliation":570,"properties":576},"92c230c7-a2e0-4a53-96a4-31c4269f683b",{"id":569,"createTime":18,"updateTime":18,"relativeEntities":571,"slug":18,"properties":572,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":575,"statistic":18},[],{"title":573},{"VI":574},"Department of Rheumatology, Limburg University Center, Diepenbeek, Belgium",[],{},{"title":578},{"VI":579},"Piet Geusens",{"id":581,"sortIndex":297,"researcher":18,"roles":582,"affiliations":583,"properties":590,"displayName":592,"givenName":18,"familyName":18},"cc3d19b9-5519-4a46-b8fb-e11b108b56f5",[255],[584],{"id":548,"sortIndex":19,"affiliation":585,"properties":18},{"id":548,"createTime":18,"updateTime":18,"relativeEntities":586,"slug":18,"properties":587,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":589,"statistic":18},[],{"title":588},{"VI":553},[],{"title":591},{"VI":592},"Sjef van der 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study oral health in patients with rheumatoid arthritis (RA) with emphasis on disease activity and treatment of RA. In this prospective cohort study 81 RA patients [53 early untreated RA (EURA) and 28 chronic RA (CRA) patients with inadequate response to synthetic disease modifying antirheumatic drugs (DMARDs)], underwent rheumatological [Disease Activity Score (28-joint) DAS28] and dental examinations [Total Dental Index (TDI), Decayed Missing Filled Teeth (DMFT) and Decayed Missing Filled Surfaces (DMFS)]. For controls, 43 volunteers were examined. After the examinations, EURA patients started treatment with synthetic DMARDs, oral and intra-articular glucocorticoids. CRA patients were candidates for biological DMARDs. The patients were re-examined mean 16 months later. Results were analyzed with descriptive statistics and logistic regression. TDI was higher in both RA groups at baseline compared to controls [EURA: 2 (2–3); CRA: 2 (1–3); controls 1 (1–3), p = 0.045]. DMFT [rs 0.561 (p = 0.002)] and DMFS [rs 0.581 (p = 0.001)] associated with DAS28 at baseline in CRA patients. After follow-up, DAS28 associated positively with DMFT [rs 0.384 (p = 0.016)] and DMFS [rs 0.334 (p = 0.038)] in EURA patients; as well as in CRA patients DMFT [rs 0.672 (p = 0.001)], DMFS [rs 0.650 (p = 0.001)]. RA patients already in the early phase of the disease had poorer oral health compared to controls. The caries indices associated with the activity of RA in both patient groups. Oral status may thus contribute to the development and further relate to the activity of RA.",{"EN":663},"Activity of rheumatoid arthritis correlates with oral inflammatory burden",{"VOID":665},"[\"5530265903484232222\"]",{"VOID":667},"10.1007\u002Fs00296-018-4108-z","2024-05-03T11:50:55.559+00:00","http:\u002F\u002Flink.springer.com\u002F10.1007\u002Fs00296-018-4108-z",[671,686,699,716,729,744,759,773,789,803,816,830],{"id":672,"sortIndex":19,"researcher":18,"roles":673,"affiliations":674,"properties":683,"displayName":685,"givenName":18,"familyName":18},"860b5bb7-90b5-435c-bc5a-f7947f5bf144",[255],[675],{"id":676,"sortIndex":19,"affiliation":677,"properties":18},"711207df-c423-4149-b6e6-270d97ee98cb",{"id":676,"createTime":18,"updateTime":18,"relativeEntities":678,"slug":18,"properties":679,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":682,"statistic":18},[],{"title":680},{"VI":681},"Department of Oral and Maxillofacial Diseases, Helsinki University Hospital, University of Helsinki, Helsinki, Finland",[],{"title":684},{"VI":685},"Leena 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Ann Rheum Dis 61:554–558. https:\u002F\u002Fdoi.org\u002F10.1136\u002Fard.61.6.554","https:\u002F\u002Fard.bmj.com\u002Flookup\u002Fdoi\u002F10.1136\u002Fard.61.6.554",{"doi":1041},"10.1136\u002Fard.61.6.554",{"id":1043,"text":1044,"url":1045,"identifiers":1046},"e0955816-41c9-4640-a424-3214bd8ad0d3","Akpek EK, Lindsley KB, Adyanthaya RS, Swamy R, Baer AN, McDonnell PJ (2011) Treatment of Sjögren’s syndrome-associated dry eye an evidence-based review. Ophthalmology 118:1242–1252. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.ophtha.2010.12.016","https:\u002F\u002Flinkinghub.elsevier.com\u002Fretrieve\u002Fpii\u002FS0161642010013308",{"doi":1047},"10.1016\u002Fj.ophtha.2010.12.016",{"id":1049,"text":1050,"url":1051,"identifiers":1052},"8e0f6c3a-3481-498b-beff-d5bef5b3a5d2","Shahin AA, El-Agha S, El-Azkalany GS (2014) The effect of leflunomide on the eye dryness in secondary Sjögren’s syndrome associated with rheumatoid arthritis and in rheumatoid arthritis patients. Clin Rheumatol 33:925–930. https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs10067-014-2548-8","http:\u002F\u002Flink.springer.com\u002F10.1007\u002Fs10067-014-2548-8",{"doi":1053},"10.1007\u002Fs10067-014-2548-8",{"id":1055,"text":1056,"url":1057,"identifiers":1058},"f4e5770b-7264-430b-a563-84ae2d5122e5","Tsuboi H, Matsumoto I, Hagiwara S, Hirota T, Takahashi H, Ebe H, Yokosawa M, Yagishita M, Takahashi H, Kurata I, Ohyama A, Honda F, Asashima H, Miki H, Umeda N, Kondo Y, Hirata S, Saito K, Tanaka Y, Horai Y, Nakamura H, Kawakami A, Sumida T (2016) Effectiveness of abatacept for patients with Sjögren’s syndrome associated with rheumatoid arthritis. An open label, multicenter, one-year, prospective study: ROSE (Rheumatoid Arthritis with Orencia Trial toward Sjögren’s syndrome Endocrinopathy) trial. Mod Rheumatol 25:187–193. https:\u002F\u002Fdoi.org\u002F10.3109\u002F14397595.2016.1158773","https:\u002F\u002Facademic.oup.com\u002Fmr\u002Farticle\u002F26\u002F6\u002F891-899\u002F6301263",{"doi":1059},"10.3109\u002F14397595.2016.1158773",{"id":403,"text":1061,"url":405,"identifiers":1062},"Hazes JM, Dijkmans BA, Vandenbroucke JP, de Vries RR, Cats A (1990) Lifestyle and the risk of rheumatoid arthritis: cigarette smoking and alcohol consumption. Ann Rheum Dis 49:980–982",{"doi":407},{"id":1064,"text":1065,"url":1066,"identifiers":1067},"87626f7c-acaa-4899-b81e-7ab57e67ccc1","Krishnan E, Sokka T, Hannonen P (2003) Smoking-gender interaction and risk for rheumatoid arthritis. 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Eur J Public Health 25:539–546. https:\u002F\u002Fdoi.org\u002F10.1093\u002Feurpub\u002Fcku174","https:\u002F\u002Facademic.oup.com\u002Feurpub\u002Farticle-lookup\u002Fdoi\u002F10.1093\u002Feurpub\u002Fcku174",{"doi":1080},"10.1093\u002Feurpub\u002Fcku174",{"id":1082,"createTime":1083,"updateTime":1084,"relativeEntities":1085,"slug":1086,"properties":1087,"entityType":246,"verifyStatus":247,"verifyTime":1096,"verifyNote":249,"languages":18,"translateLanguages":18,"viewCount":19,"primaryUrl":1097,"fullTextUrl":18,"authors":1098,"publicationType":339,"publisherRelationship":1209,"citationCount":19,"citationInfo":1264,"publishDate":1267,"publishYear":1265,"citationAnalyzeStatus":399,"lastCitationAnalyze":1084,"indexDatabases":1268,"openAccess":18,"references":1269,"isForceReanalyzing":507},"13e59dd3-5d39-4451-806f-b7ba327c690a","2024-01-04T21:57:59.824+00:00","2026-08-15T17:05:17.846+00:00",[],"A-case-of-ANCA-associated-vasculitis-presenting-with-calf-claudication",{"abstract":1088,"title":1090,"gsPaper":1092,"doi":1094},{"EN":1089},"Wegener’s granulomatosis (WG), Churg–Strauss syndrome, and microscopic polyangiitis (MPA) are closely related small vessel vasculitides characterized by anti-neutrophil cytoplasmic antibodies (ANCA). Although there were some reports of MPA presenting with claudication, there are very few reports on WG presenting with claudication of calf muscles. We report an unusual case of ANCA-associated vasculitis in a 75-year-old man who presented with bilateral calf claudication. Comprehensive evaluations, including electromyography, nerve conduction study, lower extremity magnetic resonance imaging, and Doppler scan, did not reveal any other cause of bilateral calf claudication. P-ANCA and anti-myeloperoxidase antibody was positive, but the anti-proteinase 3 antibody was negative. Chest computed tomography scan showed subpleural honeycombing and reticulation, predominantly in both basal lung areas. Biopsy of the calf muscle showed granulomatous vasculitis. Kidney biopsy was also performed which revealed focal necrotizing glomerulonephritis. Our patient does not exhibit typical clinical features for WG, but histopathologic findings of necrotizing granulomatous vasculitis in calf muscle biopsy is highly suggestive of WG.",{"EN":1091},"A case of ANCA-associated vasculitis presenting with calf 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Chest 97:906–912",{},{"id":403,"text":1298,"url":405,"identifiers":1299},"Jindal SK, Agarwal R (2005) Autoimmunity and interstitial lung disease. Curr Opin Pulm Med 11:438–446",{"doi":407},{"id":403,"text":1301,"url":405,"identifiers":1302},"Mukhtyar C, Guillevin L, Cid MC, Dasgupta B, de Groot K, Gross W, Hauser T, Hellmich B, Jayne D, Kallenberg CG, Merkel PA, Raspe H, Salvarani C, Scott DG, Stegeman C, Watts R, Westman K, Witter J, Yazici H, Luqmani R (2009) EULAR recommendations for the management of primary small and medium vessel vasculitis. Ann Rheum Dis 68:310–317",{"doi":407},{"id":403,"text":1304,"url":405,"identifiers":1305},"Seo P, Stone JH (2004) The antineutrophil cytoplasmic antibody-associated vasculitides. 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Arthritis Rheum 43:1021–1032",{"doi":407},{"id":403,"text":1313,"url":405,"identifiers":1314},"Jayne D, Rasmussen N, Andrassy K, Bacon P, Tervaert JW, Dadoniene J, Ekstrand A, Gaskin G, Gregorini G, de Groot K, Gross W, Hagen EC, Mirapeix E, Pettersson E, Siegert C, Sinico A, Tesar V, Westman K, Pusey C (2003) A randomized trial of maintenance therapy for vasculitis associated with antineutrophil cytoplasmic autoantibodies. N Engl J Med 349:36–44",{"doi":407},{"id":403,"text":1316,"url":405,"identifiers":1317},"Sanchez-Cano D, Callejas-Rubio JL, Ortego-Centeno N (2008) Effect of rituximab on refractory Wegener granulomatosis with predominant granulomatous disease. J Clin Rheumatol 14:92–93",{"doi":407},{"id":403,"text":1319,"url":405,"identifiers":1320},"Patel AM, Lehman TJ (2008) Rituximab for severe refractory pediatric Wegener granulomatosis. J Clin Rheumatol 14:278–280",{"doi":407},{"id":403,"text":1322,"url":405,"identifiers":1323},"Taylor SR, Salama AD, Joshi L, Pusey CD, Lightman SL (2009) Rituximab is effective in the treatment of refractory ophthalmic Wegener’s granulomatosis. Arthritis Rheum 60:1540–1547",{"doi":407},{"id":1325,"createTime":1326,"updateTime":1327,"relativeEntities":1328,"slug":1329,"properties":1330,"entityType":246,"verifyStatus":247,"verifyTime":1339,"verifyNote":249,"languages":18,"translateLanguages":18,"viewCount":19,"primaryUrl":1340,"fullTextUrl":18,"authors":1341,"publicationType":339,"publisherRelationship":1456,"citationCount":311,"citationInfo":1511,"publishDate":1514,"publishYear":1512,"citationAnalyzeStatus":399,"lastCitationAnalyze":1327,"indexDatabases":1515,"openAccess":18,"references":18,"isForceReanalyzing":507},"3038dedf-8c26-416f-810d-d7d12d27d9c3","2023-12-12T19:42:39.800+00:00","2026-08-15T07:51:10.774+00:00",[],"Antiperinuclear-factor-clinical-serological-and-genetic-correlates-in-Israeli-patients-with-rheumatoid-arthritis",{"abstract":1331,"title":1333,"gsPaper":1335,"doi":1337},{"EN":1332}," The possible association between the presence of antiperinuclear factor (APF) and clinical and genetic parameters was investigated in 54 Israeli patients with rheumatoid arthritis (RA). Rheumatoid factor (RF) was detected in the sera of 43 patients (80%) and APF was positive in 33 (61%). No significant statistical differences were found in the presence of HLA-DR4 and\u002For DR1 between APF-positive and -negative patients. Furthermore, neither the Ritchie articular index nor the patient's functional class correlated with the presence of APF. The results of our study suggested that although Israeli patients have a different genetic background, the presence and behaviour of APF is similar to that of other Caucasian populations.",{"EN":1334},"Antiperinuclear factor – clinical, serological and genetic correlates in Israeli patients with rheumatoid arthritis",{"VOID":1336},"[\"17061724699237914648\"]",{"VOID":1338},"10.1007\u002Fs002960050025","2024-06-24T17:38:11.045+00:00","http:\u002F\u002Flink.springer.com\u002F10.1007\u002Fs002960050025",[1342,1357,1372,1387,1400,1415,1428,1443],{"id":1343,"sortIndex":19,"researcher":18,"roles":1344,"affiliations":1345,"properties":1354,"displayName":1356,"givenName":18,"familyName":18},"00351814-7741-4dc7-9c6b-f430ace6876c",[255],[1346],{"id":1347,"sortIndex":19,"affiliation":1348,"properties":18},"12d58a65-8cc9-484b-a856-b8525f71a1a0",{"id":1347,"createTime":18,"updateTime":18,"relativeEntities":1349,"slug":18,"properties":1350,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":1353,"statistic":18},[],{"title":1351},{"VI":1352},"Department of Rheumatology, Tel Aviv Souraski Medical Centre, 6 Weizmann Street, Tel Aviv, Israel Fax: 972-3-6 97 45 77, , IL",[],{"title":1355},{"VI":1356},"M. 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dục là một khía cạnh phức tạp trong cuộc sống của con người và không chỉ đơn thuần là hành động tình dục. Chức năng tình dục bình thường bao gồm hoạt động tình dục với sự chuyển tiếp qua các giai đoạn từ hưng phấn đến thư giãn mà không gặp phải vấn đề gì, và với cảm giác thỏa mãn, hoàn thiện và vui vẻ. Các bệnh thấp khớp có thể ảnh hưởng đến tất cả các khía cạnh của cuộc sống, bao gồm cả chức năng tình dục. Các nguyên nhân gây rối loạn chức năng tình dục là đa yếu tố và bao gồm các yếu tố liên quan đến bệnh tật cũng như liệu pháp điều trị. Ở những bệnh nhân viêm khớp dạng thấp và viêm cột sống dính khớp, đau đớn và trầm cảm có thể là những yếu tố chính góp phần vào rối loạn chức năng tình dục. Ngược lại, ở phụ nữ bị hội chứng Sjögren, lupus ban đỏ hệ thống và xơ cứng hệ thống, rối loạn chức năng tình dục dường như có mối liên hệ chặt chẽ nhất với sự khó chịu hoặc đau đớn ở âm đạo trong quá trình giao hợp. Cuối cùng, rối loạn chức năng tình dục ở bệnh nhân fibromyalgia có thể chủ yếu liên quan đến trầm cảm, nhưng các triệu chứng đặc trưng của fibromyalgia (đau tổng quát, cứng khớp, mệt mỏi và mất ngủ) có thể góp phần vào sự xuất hiện của rối loạn chức năng tình dục. Việc điều trị rối loạn chức năng tình dục sẽ tùy thuộc vào các triệu chứng cụ thể của bệnh nhân, tuy nhiên, có một số khuyến nghị chung bao gồm: khám phá các tư thế khác nhau, sử dụng thuốc giảm đau, áp dụng nhiệt và thuốc giãn cơ trước khi hoạt động tình dục và khám phá các phương pháp biểu đạt tình dục thay thế. Đây là một bài tổng quan hệ thống về tác động của một số bệnh thấp khớp đối với chức năng tình dục. Đến nay, chưa có tổng quan nào trước đây về chủ đề này.","Sexuality is a complex aspect of the human being’s life and is more than of only the sexual act. Normal sexual functioning consists of sexual activity with transition through the phases from arousal to relaxation with no problems, and with a feeling of pleasure, fulfillment and satisfaction. Rheumatic diseases may affect all aspects of life including sexual functioning. The reasons for disturbing sexual functioning are multifactorial and comprise disease-related factors as well as therapy. In rheumatoid arthritis and ankylosing spondylitis patients, pain and depression could be the principal factors contributing to sexual dysfunction. On the other hand, in women with Sjögren’s syndrome, systemic lupus erythematosus and systemic sclerosis sexual dysfunction is apparently most associated to vaginal discomfort or pain during intercourse. Finally, sexual dysfunction in patients with fibromyalgia could be principally associated with depression, but the characteristic symptoms of fibromyalgia (generalized pain, stiffness, fatigue and poor sleep) may contribute to the occurrence of sexual dysfunction. The treatment of sexual dysfunction will depend on the specific patient’s symptoms, however, there are some general recommendations including: exploring different positions, using analgesics drug, heat and muscle relaxants before sexual activity and exploring alternative methods of sexual expression. This is a systemic review about the impact of several rheumatic diseases on sexual functioning. There are no previous overviews about this topic so far.",{"EN":1527,"VI":1528},"The impact of rheumatic diseases on sexual function","Tác động của các bệnh thấp khớp lên chức năng tình dục",{"VOID":1530},"[\"9704588257962456964\"]",{"EN":1532,"VI":1533},"","bệnh thấp khớp, chức năng tình dục, viêm khớp dạng thấp, viêm cột sống dính khớp, hội chứng Sjögren, lupus ban đỏ hệ thống, xơ cứng hệ thống, fibromyalgia, rối loạn chức năng tình dục",{"VOID":1535},"Prady J, Vale A, Hill J (1998) Body image and sexuality. In: Hill J (ed) Rheumatology nursing: a creative approach. Churchill Livingstone, Edinburgh, pp 109–124\nWells D (2000) Caring for sexuality in health and illness. Churchill Livingstone, Edinburgh\nPitts M (1996) Sexual health: the psychology of preventative health. Routledge, London\nØstensen M (2004) New insights into sexual functioning and fertility in rheumatic diseases. Best Pract Res Clin Rheumatol 18:219–232. doi:10.1016\u002Fj.berh.2004.01.002\nStrax TE (1991) Psychosocial issues faced by adolescents and young adults with disabilities. Pediatr Ann 20:501–506\nSelekman J, McIlvain-Simpson G (1991) Sex and sexuality for the adolescent with a chronic condition. Pediatr Nurs 17:535–538\nHamilton A (1980) Sexual problems in arthritis and allied conditions. Int Rehabil Med 3:38–42\nBritto MT, Rosenthal SL, Taylor J et al (2000) Improving rheumatologists’ screening for alcohol use and sexual activity. Arch Pediatr Adolesc Med 154(5):478–483\nSerrant-Green L (2001) Inequality in provision of sexual health information. Prof Nurse 16:1038–1042\nRoyal College of Nursing (2001) RCN sexual health strategy. Guidance for nursing staff (publication code 001 525). Royal College of Nursing, London\nElst R, Sybesma T, van der Stadt RJ et al (1984) Sexual problems in rheumatoid arthritis and ankylosing spondylitis. Arthr Rheum 27:217–220. doi:10.1002\u002Fart.1780270214\nEhrlich GE (1983) Social, economic, psychologic, and sexual outcomes in rheumatoid arthritis. Am J Med 75:27–34. doi:10.1016\u002F0002-9343(83)90472-2\nBaldursson H, Brattstrom H (1979) Sexual difficulties and total hip replacement in rheumatoid arthritis. Scand J Rheumatol 8:214–216. doi:10.3109\u002F03009747909114625\nBlake DJ, Maisiak R, Koplan A et al (1988) Sexual dysfunction among patients with arthritis. Clin Rheumatol 7:50–60. doi:10.1007\u002FBF02284057\nBrown GMM, Dare CM, Smith PR et al (1987) Important problems identified by patients with chronic arthritis. S Afr Med J 72:126–128\nFerguson K, Figley B (1979) Sexuality and rheumatic disease: a prospective study. Sex Disabil 2:130–138. doi:10.1007\u002FBF01102472\nGordon D, Beastall GH, Thomson JA et al (1986) Androgenic status and sexual function in males with rheumatoid arthritis and ankylosing spondylitis. QJM 60:671–679\nHill RH, Herstein A, Walters K (1977) Juvenile rheumatoid arthritis: follow-up into adulthood-medical, sexual and social status. Can Med Assoc J 114:790–794\nKraaimaat FW, Bakker AH, Janssen E et al (1996) Intrusiveness of rheumatoid arthritis on sexuality in male and female patients living with a spouse. Arthr Care Res 9:120–125. doi:10.1002\u002F1529-0131(199604)9:2\u003C120::AID-ANR1790090208>3.0.CO;2-T\nYoshino S, Uchida S (1981) Sexual problems of women with rheumatoid arthritis. Arch Phys Med Rehabil 62(3):122–123\nBlake DJ, Maisiak R, Alarcon GS et al (1987) Sexual quality-of-life of patients with arthritis compared to arthritis-free controls. J Rheumatol 14(3):570–576\nBhadauria S, Moser DK, Clements PJ et al (1995) Genital tract abnormalities and female sexual function impairment in systemic sclerosis. Am J Obstet Gynecol 172:580–587. doi:10.1016\u002F0002-9378(95)90576-6\nHill J, Bird H, Thorpe R (2003) Effects of rheumatoid arthritis on sexual activity and relationships. Rheumatology (Oxford) 42(2):280–286. doi:10.1093\u002Frheumatology\u002Fkeg079\nYoshino S, Fujimori J, Morishige T et al (1984) Bilateral joint replacement of hip and knee joints in patients with rheumatoid arthritis. Arch Orthop Trauma Surg 103(1):1–4. doi:10.1007\u002FBF00451311\nØstensen M, Almberg K, Koksvik H (2000) Sex, reproduction, and gynecological disease in young adults with a history of juvenile chronic arthritis. J Rheumatol 27:1783–1787\nPackham JC, Hall MA, Pimm TJ (2002) Long-term follow-up of 246 adults with juvenile idiopathic arthritis: predictive factors for mood and pain. Rheumatology (Oxford) 41(12):1444–1449. doi:10.1093\u002Frheumatology\u002F41.12.1444\nvan Lankveld W, Ruiterkamp G, Naring G et al (2004) Marital and sexual satisfaction in patients with RA and their spouses. Scand J Rheumatol 33(6):405–408. doi:10.1080\u002F03009740410010317\nChannon LD, Ballinger SE (1986) Some aspects of sexuality and vaginal symptoms during menopause and their relation to anxiety and depression. Br J Med Psychol 59:173–180\nFrohlich P, Meston C (2002) Sexual functioning and self-reported depressive symptoms among college women. J Sex Res 39:321–325\nGutweniger S, Kopp M, Mur E et al (1999) Body image of women with rheumatoid arthritis. Clin Exp Rheumatol 17(4):413–417\nAbdel-Nasser AM, Ali EI (2006) Determinants of sexual disability and dissatisfaction in female patients with rheumatoid arthritis. Clin Rheumatol 25(6):822–830. doi:10.1007\u002Fs10067-005-0175-0\nvan Berlo WT, van de Wiel HB, Taal E et al (2007) Sexual functioning of people with rheumatoid arthritis: a multicenter study. Clin Rheumatol 26(1):30–38. doi:10.1007\u002Fs10067-006-0216-3\nKarlsson B, Berglin E, Wållberg-Jonsson S (2006) Life satisfaction in early rheumatoid arthritis: a prospective study. Scand J Occup Ther 13(3):193–199. doi:10.1080\u002F11038120500462337\nMajerovitz SD, Revenson TA (1994) Sexuality and rheumatic disease: the significance of gender. Arthr Care Res 7(1):29–34. doi:10.1002\u002Fart.1790070107\nDalyan M, Güner A, Tuncer S et al (1999) Disability in ankylosing spondylitis. Disabil Rehabil 21:74–79. doi:10.1080\u002F096382899298007\nBraun J, Sieper J (2000) Inception cohorts for spondyloarthropathies. Z Rheumatol 59:117–121. doi:10.1007\u002Fs003930050215\nZink A, Braun J, Listing J et al (2000) Disability and handicap in rheumatoid arthritis and ankylosing-spondylitis results from the German rheumatological database. J Rheumatol 27:613–622\nFeldtkeller E, Bruckel J, Khan MA (2000) Scientific contribution of ankylosing spondylitis patient advocacy groups. Curr Opin Rheumatol 12(4):239–247. doi:10.1097\u002F00002281-200007000-00002\nBraunstein EM, Martel W, Moidel R (1982) Ankylosing spondylitis in men and women: a clinical and radiographic comparison. Radiology 144:91–94\nJimenez-Balderas FJ, Mintz G (1993) Ankylosing spondylitis: clinical course in women and men. J Rheumatol 20:2069–2072\nResnick D, Dwosh IL, Goergen TG et al (1976) Clinical and radiographic abnormalities in ankylosing spondylitis: a comparison of men and women. Radiology 119:293–297\nPirildar T, Müezzinoğlu T, Pirildar S (2004) Sexual function in ankylosing spondylitis: a study of 65 men. J Urol 171(4):1598–1600. doi:10.1097\u002F01.ju.0000117867.44858.ba\nDincer U, Cakar E, Kiralp MZ et al (2007) Assessment of sexual dysfunction in male patients with ankylosing spondylitis. Rheumatol Int 27(6):561–566. doi:10.1007\u002Fs00296-006-0248-7\nOzgül A, Peker F, Taskaynatan MA et al (2006) Effect of ankylosing spondylitis on health-related quality of life and different aspects of social life in young patients. Clin Rheumatol 25(2):168–174. doi:10.1007\u002Fs10067-005-1150-5\nMoutsopoulus H, Talal N (1987) Immunological abnormalities in Sjögren’s syndrome. In: Talal N, Moutsopoulus HM, Kassan SS (eds) Sjögren’s syndrome. Clinical and immunological aspects. Springer, Berlin, pp 258–265\nSato E, Ariga H, Sullivan D (1992) Impact of androgen therapy in Sjögren’s syndrome: hormonal influence on lymphocyte populations and la expression in lacrimal glands of MRL\u002FMp-lpr\u002Flpr mice. Invest Ophthalmol Vis Sci 33:2537–2545\nSato E, Sullivan D (1994) Comparative influence of steroid hormones and immunosuppressive agents on autoimmune expression in lacrimal glands of a female mouse model of Sjögren’s syndrome. Invest Ophthalmol Vis Sci 35:2632–2642\nSullivan D, Edwards J (1997) Androgen stimulation of lacrimal gland function in mouse models of Sjögren’s syndrome. J Steroid Biochem Mol Biol 60:237–245. doi:10.1016\u002FS0960-0760(96)00190-2\nValtysdottir ST, Wide L, Hallgren R (2003) Mental wellbeing and quality of sexual life in women with primary Sjögren’s syndrome are related to circulating dehydroepiandrosterone sulphate. Ann Rheum Dis 62(9):875–879. doi:10.1136\u002Fard.62.9.875\nMcCoy N, Davidson J (1985) A longitudinal study of the effects of menopause on sexuality. Maturitas 7:203–210. doi:10.1016\u002F0378-5122(85)90041-6\nDupuy HJ (1984) The psychological general well-being (PGWB) index. In: Wenger NK, Mattson ME, Furberg CF, Elinson J (eds) Assessment of quality of life in clinical trials of cardiovascular therapies. Le Jacq Publishing, Hoboken, pp 170–183\nHartkamp A, Geenen R, Godaert GL et al (2008) Effect of dehydroepiandrosterone administration on fatigue, well-being, and functioning in women with primary Sjögren syndrome: a randomised controlled trial. Ann Rheum Dis 67(1):91–97. doi:10.1136\u002Fard.2007.071563\nMulherin MD, Sheeran TP, Kum Araratne DS et al (1997) Sjögren’s syndrome in women presenting with chronic dyspareunia. Br J Obstet Gynaecol 104:1019–1023\nMarchesoni D, Mozzanega B, De Sandre P et al (1995) Gynaecological aspects of primary Sjogren’s syndrome. Eur J Obstet Gynecol Reprod Biol 63:49–53. doi:10.1016\u002F0301-2115(95)02224-U\nSkopouli FN, Papanikolaou S, Malamou-Mitsi V et al (1994) Obstetric and gynecologic profile in patients with primary Sjögren’s syndrome. Ann Rheum Dis 53:569–573. doi:10.1136\u002Fard.53.9.569\nD’Cruz DP, Khamashta MA, Hughes GR (2007) Systemic lupus erythematosus. Lancet 369(9561):587–596. doi:10.1016\u002FS0140-6736(07)60279-7\nErmann J, Bermas BL (2007) The biology behind the new therapies for SLE. Int J Clin Pract 61(12):2113–2119. doi:10.1111\u002Fj.1742-1241.2007.01528.x\nStein H, Walters K, Dillon A et al (1986) Systemic lupus erythematosus—a medical and social profile. J Rheumatol 13(3):570–576\nCurry SL, Levine SB, Corty E et al (1994) The impact of systemic lupus erythematosus on women’s sexual functioning. J Rheumatol 21:2254–2260\nFolomeev M, Alekberova Z (1990) Impotence in systemic lupus erythematosus. J Rheumatol 17:117–118\nBoomsma MM, Bijl M, Stegeman CA et al (2002) Patients’ perceptions of the effects of systemic lupus erythematosus on health, function, income, and interpersonal relationships: a comparison with Wegener’s granulomatosis. Arthr Rheum 47(2):196–201. doi:10.1002\u002Fart.10341\nSutcliffe N, Clarke AE, Levinton C et al (1999) Associates of health status in patients with systemic lupus erythematosus. J Rheumatol 26(11):2352–2356\nMedsger TA Jr (2001) Systemic sclerosis (scleroderma): clinical aspects. In: Koopman WJ (ed) Arthritis and allied conditions, 14th edn. Lippincott, Philadelphia, pp 1590–1624\nLeRoy EC, Black C, Fleischmajer R et al (1988) Scleroderma (systemic sclerosis): classification, subsets, and pathogenesis. J Rheumatol 15:202–205\nLally EV, Jimenez SA, Kaplan SR (1988) Progressive systemic sclerosis: mode of presentation, rapidly progressive disease course, and mortality based on analysis of 91 patients. Semin Arthr Rheum 18:1–13. doi:10.1016\u002F0049-0172(88)90030-3\nSimeon CP, Fonollosa V, Vilardell M et al (1994) Impotence and Peyronie’s disease in systemic sclerosis. Clin Exp Rheumatol 12:464\nHong P, Pope J, Ouimet JM et al (2004) Erectile dysfunction associated with scleroderma: a case-control study of men with scleroderma and rheumatoid arthritis. J Rheumatol 31:508–513\nLally EV, Jimenez SA (1981) Impotence in progressively systemic sclerosis. Ann Intern Med 95:150–153\nNehra A, Hall SJ, Basile G et al (1995) Systemic sclerosis and impotence: a clinicopathological correlation. J Urol 153:1140–1146. doi:10.1016\u002FS0022-5347(01)67533-5\nSukenik S, Horowitz J, Buskila D et al (1987) Impotence in systemic sclerosis. Ann Intern Med 106:910–911\nLofti MA, Varga J, Hirsch IH (1995) Erectile dysfunction in systemic sclerosis. Urology 45:879–881. doi:10.1016\u002FS0090-4295(99)80100-9\nNowlin NS, Brick JE, Weaver DJ et al (1986) Impotence in scleroderma. Ann Intern Med 104:794–798\nMarumo K, Nakashima J, Murai M (2001) Age-related prevalence of erectile dysfunction in Japan: assessment by the International Index of Erectile Dysfunction. Int J Urol 8:53–59. doi:10.1046\u002Fj.1442-2042.2001.00258.x\nSukenik S, Abarbanel J, Buskila D et al (1987) Impotence, carpal tunnel syndrome and peripheral neuropathy as presenting symptoms in progressive systemic sclerosis. J Rheumatol 14:641–643\nOstojic P, Damjanov N (2007) The impact of depression, microvasculopathy, and fibrosis on development of erectile dysfunction in men with systemic sclerosis. Clin Rheumatol 26(10):1671–1674. doi:10.1007\u002Fs10067-007-0567-4\nProietti M, Aversa A, Letizia C et al (2007) Erectile dysfunction in systemic sclerosis: effects of longterm inhibition of phosphodiesterase type-5 on erectile function and plasma endothelin-1 levels. J Rheumatol 34(8):1712–1717\nYunus MB, Inanıcı F (2001) Clinical characteristics and biopathophysiological mechanisms of fibromyalgia syndrome. In: Baldry P (ed) Myofascial pain and fibromyalgia syndromes: a clinical guide to diagnosis and management. Churchill Livingstone, Edinburgh, pp 351–377\nYunus MB, Inanıcı F (2002) Fibromyalgia syndrome: clinical features, diagnosis, and biopathophysiologic mechanisms. In: Rachlin ES, Rachlin I (eds) Myofascial pain and fibromyalgia: trigger point management. Mosby, Philadelphia, pp 3–31\nAydin G, Başar MM, Keleş I et al (2006) Relationship between sexual dysfunction and psychiatric status in premenopausal women with fibromyalgia. Urology 67(1):156–161. doi:10.1016\u002Fj.urology.2005.08.007\nWallace DJ, Brock J (1999) Making sense of fibromyalgia. Oxford University Press, New York\nPanush RS, Mihailescu GD, Gornisiewicz MT et al (2000) Sex and arthritis. Bull Rheum Dis 49(7):1–4\nAbdel-Nasser AM, Abd El-Azim S, Taal E et al (1998) Depression and depressive symptoms in rheumatoid arthritis patients: an analysis of their occurrence and determinants. 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idiopathic skeletal hyperostosis (DISH) is characterized by ossification of different entheses. Psoriatic arthritis (PsA) is a seronegative spondyloarthritis associated with psoriasis. Given the possible overlap of the two diseases, we assessed whether DISH presence may affect PsA clinical outcomes. Also, predictors of DISH presence in the cohort were investigated. Consecutive PsA patients from two Italian Rheumatology Research Units were enrolled. Subjects were splitted into two groups, according to the current treatment (TNF-α blockers or traditional DMARDs). All patients underwent a rheumatologic examination, blood sample collections and spine radiographs. Information about traditional vascular risk factors was recorded. In each patient, the presence of minimal disease activity was evaluated and the presence of DISH was established according to the Resnick and Niwayama criteria. Among the 80 enrolled subjects (57.5 % men, mean age 56.5 ± 11.1 years), the overall prevalence of DISH was 30.0 %. Patients with DISH were older, with higher BMI and waist circumference. DISH subjects showed worsen BASMI, HAQ and ESR. In a multivariate regression model, BASMI was a significant predictor of DISH presence (OR 3.027, 95 % CI 1.449–6.325, p = 0.003). The prevalence of MDA was lower in DISH patients than in no-DISH (16.7 vs 41.1 %, p = 0.041), and the presence of DISH was a predictor of not achieving MDA (OR 3.485, 95 % CI 1.051–11.550, p = 0.041). PsA subjects with DISH showed worsen indices of spine mobility and articular function and lower prevalence of minimal disease activity than no-DISH patients.",{"EN":1632},"The impact of concomitant diffuse idiopathic skeletal hyperostosis on the achievement of minimal disease activity in subjects with psoriatic arthritis",{"VOID":1634},"[\"13604345489676368447\"]",{"VOID":1636},"10.1007\u002Fs00296-015-3303-4","2024-04-30T09:51:44.266+00:00","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs00296-015-3303-4",[1640,1655,1672,1685,1698,1713,1726],{"id":1641,"sortIndex":19,"researcher":18,"roles":1642,"affiliations":1643,"properties":1652,"displayName":1654,"givenName":18,"familyName":18},"9a72d8df-925a-4e8b-9ad4-668ee0e6a969",[255],[1644],{"id":1645,"sortIndex":19,"affiliation":1646,"properties":18},"6d9ee62c-f66c-4295-aa9c-ae75f66b90d2",{"id":1645,"createTime":18,"updateTime":18,"relativeEntities":1647,"slug":18,"properties":1648,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":1651,"statistic":18},[],{"title":1649},{"VI":1650},"Rheumatology and Rehabilitation Research Unit, “Salvatore Maugeri” Foundation I.R.C.C.S. Scientific Institute of Telese Terme, Telese Terme, Italy",[],{"title":1653},{"VI":1654},"Nicola Pappone",{"id":1656,"sortIndex":190,"researcher":18,"roles":1657,"affiliations":1658,"properties":1667,"displayName":1669,"givenName":18,"familyName":18},"6bad6e92-e3d1-41a1-bac3-b2472582fafb",[255],[1659],{"id":1660,"sortIndex":19,"affiliation":1661,"properties":18},"c9406402-cf7f-40a3-86e6-e722093fd62c",{"id":1660,"createTime":18,"updateTime":18,"relativeEntities":1662,"slug":18,"properties":1663,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":1666,"statistic":18},[],{"title":1664},{"VI":1665},"Department of Clinical Medicine and Surgery, Federico II University, Naples, Italy",[],{"title":1668,"gsAuthor":1670},{"VI":1669},"Matteo Nicola Dario Di 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objective of this study was to analyze the un-stimulated and stimulated release of superoxide anion (O2\n                −) by granulocytes and monocytes in juvenile systemic lupus erythematosus (jSLE). The un-stimulated and phorbol myristate acetate (PMA, 30 nM)-induced O2\n                −by granulocytes and monocytes were determined in six different times of incubation in patients with 23 jSLE and 28 controls. The analysis compared the jSLE group, which was classified into two subgroups by SLEDAI in one inactive subgroup (score \u003C3) (n = 13 patients) and one active subgroup (score ≥3) (n = 10 patients) to the same control group. At time of blood withdrawal, 13 (56.52%) had inactive and 10 (43.47%) patients had active SLE. jSLE patients’ granulocytes and monocytes had always a lower un-stimulated O2\n                − production when compared to controls. Stimulated granulocytes had an increased O2\n                − production at baseline followed by a significant lower production at 60 min in jSLE when compared to controls. Stimulated monocytes had a similar O2\n                − production among patients with jSLE and controls. The results suggest a defect in phagocytic function in jSLE. The significant higher release of O2\n                − in the assays of the stimulated granulocytes, in the initial instances, the so-called respiratory burst, could be attributed to the inflammatory state of phagocytes.",{"EN":2020},"Superoxide release in juvenile systemic lupus erythematosus",{"VOID":2022},"[\"1813082005907647725\"]",{"VOID":2024},"Waszczykowska E, Robak E, Wozniacka A, Narbutt J, Torzecka JD, Sysa-Jedrzejowska A (1999) Estimation of SLE activity based on the serum level of chosen cytokines and superoxide radical generation. Mediators Inflamm 8:93–100\nAntonaccio F, Bassissi P (1994) Free radicals. Chron Dermatol 4:1017–1033\nButtke TM, Sandstrom PA (1994) Oxidative stress as a mediator of apoptosis. Immunol Today 25:111–121\nKnight JA (1995) Diseases related to oxygen derived free radicals. Ann Clin Lab Sci 25:11–121\nAruoma OI (1994) Nutrition and health aspects of free radicals and antioxidants. Food Chem Toxicol 32:671–683\nKomatsu H, Tsukimori K, Hata K, Satoh S, Nakano H (2001) The characterization of superoxide production of human neonatal neutrophil. Early Hum Dev 65:11–19\nPerskin MH, Cronstein BN (1992) Age-related changes in neutrophil structure and function. Mech Ageing Dev 64:303–313\nPoligano A, Tortorella C, Venezia A, Jirillo E, Antonaci S (1994) Age-associated changes of neutrophils responsiveness in a human healthy elderly population. Cytobios 80:145–153\nTan EM, Cohen AS, Fries JF et al (1982) The 1982 revised criteria for the classification of systemic lupus erythematosus. Arthritis Rheum 25:1271–1277\nLiang MH, Corzillius M, Bae SC et al (1999) The American College Nomenclature and case definitions for neuropsychiatric lupus syndrome. Arthritis Rheum 42:599–608\nThe Committee on prognosis studies in SLE, Bombardier C, Gladman DD, Urowitz M, Caron D, Chang C (1992) Derivation of the SLEDAI: a disease activity index for lupus patients. Arthritis Rheum 35:630–640\nHarris EN, Gharavi AE, Patel SP, Hughes GR (1987) Evaluation of the anticardiolipin antibody test: report of an international workshop held 4 April 1986. Clin Exp Immunol 68:215–222\nBrandt JT, Triplett DA, Scharer I (1995) Criteria for the diagnosis of lupus anticoagulant: an update. Thromb Haemost 74:1185–1190\nBoyum A (1968) Isolation of leucocytes from human blood. A two-phase system for removal of red cells with methylcellulose as erythrocyte-aggregating agent. Scand J Clin Lab Invest Suppl 97:9–29\nMcCord J, Fridovich I (1969) Superoxide dismutase: an enzymatic function for erythrocuprein (hemocuprein). J Biol Chem 244:6049–6055\nCondino-Neto A, Newburger PE (1988) NADPH oxidase activity and cytochrome b558 content of human Epstein-Barr-virus-transformed B lymphocytes correlate with expression of genes encoding components of the oxidase system. Arch Biochem Biophys 360:158–164\nAlves CM, Marzocchi-Machado CM, Louzada-Junior P, Azzolini AE, Polizello AC, de Carvalho IF, Lucisano-Valim YM (2007) Superoxide anion production by neutrophils is associated with prevalent clinical manifestations in systemic lupus erythematosus. Clin Rheumatol 27:701–708\nValko M, Leibfritz D, Moncol J et al (2007) Free radicals and antioxidants in normal physiological functions and human disease. Int J Biochem Cell Biol 39:44–84\nKurien BT, Hensley K, Bachmann M et al (2006) Oxidatively modified autoantigens in autoimmune diseases. Free Radic Biol Med 41:549–556\nBarnes PJ (1990) Reactive oxygen species and airway inflammation. Free Radic Biol Med 9:235–243\nAra J, Ali R (1992) Reactive oxygen species modified DNA fragments of varying size are the preferred antigen for human anti-DNA autoantibodies. Immunol Lett 34:195–200\nWitt C, Neuhans K, Winsel K, Brenke A, Hiepe F, Volk HD (1991) Bronchoalveolar lavage in patients with systemic lupus erythematosus: characterisation of cell activity by cytofluorometry, chemiluminescence and differential cell count. Z Erkr Atmungsorgane 176:21–29\nBenke PJ, Levcowitz H, Pampe J, Tozman E (1990) Scavengers of free radical oxygen effect in the generation of low molecular weight DNA in stimulated lymphocytes from patients with systemic lupus erythematosus. Metabolism 39:1278–1284\nMiesel R, Hartung R, Kroeger H (1996) Priming of NADPH oxidase by tumor necrosis factor alpha in patients with inflammatory and autoimmune rheumatic diseases. Inflammation 20:427–438\nGyimesi E, Kavai M, Kiss E, Csipö I, Szücs G, Szegedi G (1993) Triggering of respiratory burst by phagocytosis in monocytes of patients with systemic lupus erythematosus. Clin Exp Immunol 94:140–144\nSambo P, Jannino L, Candela M, Salvi A, Donini M, Dusi S, Luchetti MM, Gabrielli A (1999) Monocytes of patients with systemic sclerosis (scleroderma un-stimulated release in vitro increased amounts of superoxide anion. J Invest Dermatol 112:78–84\nCasellas AM, Prat A, Llera A, Manni J, Boveris A, Sarano JF (1991) Increased superoxide production by polymorphonuclear leukocytes in systemic lupus erythematosus. Clin Exp Rheumatol 9:511–514\nStewart AG, Dubbin PN, Harris T, Dusting GJ (1990) Platelet-activating factor may act as a second messenger in the release of icosanoids and superoxide anions from leukocytes and endothelial cells. Proc Natl Acad Sci USA 87:3215–3219\nMoon DG, van der Zee H, Weston LK, Gudewicz PW, Fenton JW 2nd, Kaplan JE (1990) Platelet modulation of neutrophil superoxide anion production. Thromb Haemost 63:91–96\nDobrinich R, Spagnuolo PJ (1991) Binding of C-reactive protein to human neutrophils. Inhibition of respiratory burst activity. Arthritis Rheum 34:1031–1038\nMarzocchi-Machado CM, Alves CM, Azzolini AE, Polizello AC, Carvalho IF, Lucisano-Valim YM (2002) Fcgamma and complement receptors: expression, role and co-operation in mediating the oxidative burst and degranulation of neutrophils of Brazilian systemic lupus erythematosus patients. Lupus 11:240–248",{"VOID":2026},"10.1007\u002Fs00296-011-1918-7","2024-05-16T13:24:35.953+00:00","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs00296-011-1918-7",[2030,2045,2062,2079,2092],{"id":2031,"sortIndex":19,"researcher":18,"roles":2032,"affiliations":2033,"properties":2042,"displayName":2044,"givenName":18,"familyName":18},"900402f7-44ca-45f6-b38d-1d390a1ddf92",[255],[2034],{"id":2035,"sortIndex":19,"affiliation":2036,"properties":18},"f72894af-7082-4936-9060-fb5033660c83",{"id":2035,"createTime":18,"updateTime":18,"relativeEntities":2037,"slug":18,"properties":2038,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":2041,"statistic":18},[],{"title":2039},{"VI":2040},"Department of Pediatrics and Medicine-Rheumatology Unit, Faculty of Medical Science, State University of Campinas, Campinas, Brazil",[],{"title":2043},{"VI":2044},"Roberto Marini",{"id":2046,"sortIndex":190,"researcher":18,"roles":2047,"affiliations":2048,"properties":2057,"displayName":2059,"givenName":18,"familyName":18},"5aab9c7f-e703-454e-a8e8-37a42e989962",[255],[2049],{"id":2050,"sortIndex":19,"affiliation":2051,"properties":18},"9ba9d31a-22bb-458c-a666-2779dbca49d9",{"id":2050,"createTime":18,"updateTime":18,"relativeEntities":2052,"slug":18,"properties":2053,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":2056,"statistic":18},[],{"title":2054},{"VI":2055},"Center for Investigation in Pediatrics, Department of Pediatics and Medicine-Rheumatology Unit, Faculty of Medical Science, State University of Campinas, Campinas, Brazil",[],{"title":2058,"gsAuthor":2060},{"VI":2059},"Antonio Condino-Neto",{"VOID":2061},"[\"9iqiCJIAAAAJ\"]",{"id":2063,"sortIndex":283,"researcher":18,"roles":2064,"affiliations":2065,"properties":2074,"displayName":2076,"givenName":18,"familyName":18},"ecae9bc3-b4f6-48e6-aef6-2bc020b11efc",[255],[2066],{"id":2067,"sortIndex":19,"affiliation":2068,"properties":18},"c1ef6983-aeb7-4454-9293-d4f8f9f4ba8c",{"id":2067,"createTime":18,"updateTime":18,"relativeEntities":2069,"slug":18,"properties":2070,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":2073,"statistic":18},[],{"title":2071},{"VI":2072},"Rheumatology Division, Department of Medicine, Faculty of Medical Science, State University of Campinas, Campinas, Brazil",[],{"title":2075,"gsAuthor":2077},{"VI":2076},"Simone Appenzeller",{"VOID":2078},"[\"_q9rYY8AAAAJ\"]",{"id":2080,"sortIndex":297,"researcher":18,"roles":2081,"affiliations":2082,"properties":2089,"displayName":2091,"givenName":18,"familyName":18},"070d245e-2923-471a-a809-93b247e14e47",[255],[2083],{"id":2050,"sortIndex":19,"affiliation":2084,"properties":18},{"id":2050,"createTime":18,"updateTime":18,"relativeEntities":2085,"slug":18,"properties":2086,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":2088,"statistic":18},[],{"title":2087},{"VI":2055},[],{"title":2090},{"VI":2091},"André M. 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examined the neurological involvement of ankylosing spondylitis (AS) are limited. This study aimed to assess the frequency of myelopathy, radiculopathy and myopathy in AS correlating them to the clinical, radiological and laboratory parameters. Included were 24 patients with AS. Axial status was assessed using bath ankylosing spondylitis metrology index (BASMI). Patients underwent (a) standard cervical and lumbar spine and sacroiliac joint radiography, (b) somatosensory (SSEP) and magnetic motor (MEP) evoked potentials of upper and lower limbs, (c) electromyography (EMG) of trapezius and supraspinatus muscles. Patients’ mean age and duration of illness were 36 and 5.99 years. Bath ankylosing spondylitis metrology index mean score was 4.6. Twenty-five percent (n = 6) of patients had neurological manifestations, 8.3% of them had myelopathy and 16.7% had radiculopathy. Ossification of the posterior (OPLL) and anterior (OALL) longitudinal ligaments were found in 8.3% (n = 2) and 4.2% (n = 1). About 70.8% (n = 17) had ≥1 neurophysiological test abnormalities. Twelve patients (50%) had SSEP abnormalities, seven had prolonged central conduction time (CCT) of median and\u002For ulnar nerves suggesting cervical myelopathy. Six had delayed peripheral or root latencies at Erb’s or interpeak latency (Erb’s-C5) suggesting radiculopathy. Motor evoked potentials was abnormal in 54% (n = 13). Twelve (50%) and five (20.8%) patients had abnormal MEP of upper limbs and lower limbs, respectively. About 50% (n = 12) had myopathic features of trapezius and supraspinatus muscles. Only 8.3% (n = 2) had neuropathic features. We concluded that subclinical neurological complications are frequent in AS compared to clinically manifest complications. Somatosensory evoked potential and MEP are useful to identify AS patients prone to develop neurological complications.",{"EN":2174},"Neurological complications of ankylosing spondylitis: neurophysiological assessment",{"VOID":2176},"[\"13425922913217747137\"]",{"VOID":2178},"10.1007\u002Fs00296-009-0841-7","2024-05-01T13:28:47.131+00:00","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs00296-009-0841-7",[2182,2199,2214,2237,2250],{"id":2183,"sortIndex":19,"researcher":18,"roles":2184,"affiliations":2185,"properties":2194,"displayName":2196,"givenName":18,"familyName":18},"1a85be0a-5f38-4908-9f47-b4efc384cf3c",[255],[2186],{"id":2187,"sortIndex":19,"affiliation":2188,"properties":18},"6a32cf89-7747-4266-84fc-561f5fdc17f5",{"id":2187,"createTime":18,"updateTime":18,"relativeEntities":2189,"slug":18,"properties":2190,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":2193,"statistic":18},[],{"title":2191},{"VI":2192},"Department of Neurology and Psychiatry, Assiut University Hospital, Assiut, Egypt",[],{"title":2195,"gsAuthor":2197},{"VI":2196},"Eman M. Khedr",{"VOID":2198},"[\"QDjHpNEAAAAJ\"]",{"id":2200,"sortIndex":190,"researcher":18,"roles":2201,"affiliations":2202,"properties":2211,"displayName":2213,"givenName":18,"familyName":18},"31c8214e-0eae-4807-afb1-90152ac68410",[255],[2203],{"id":2204,"sortIndex":19,"affiliation":2205,"properties":18},"10a6d9a6-35db-4341-8088-6bb61430ac17",{"id":2204,"createTime":18,"updateTime":18,"relativeEntities":2206,"slug":18,"properties":2207,"entityType":18,"verifyStatus":18,"verifyTime":18,"verifyNote":18,"languages":18,"translateLanguages":18,"viewCount":18,"url":18,"parentIds":2210,"statistic":18},[],{"title":2208},{"VI":2209},"Department of Rheumatology and Rehabilitation, Assiut University Hospital, Assiut, Egypt",[],{"title":2212},{"VI":2213},"Sonia M. 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