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Journal of Medicine and Pharmacy","Tạp chí Y Dược học Cần Thơ",{"EN":487,"VI":488},"\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">04\u002F10\u002F2015 Ministry of Information and Communications allowed Can Tho journal of medicine and pharmacy to operate (102 \u002FGP-BTTTT)\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">07\u002F16\u002F2015 Can Tho journal of medicine and pharmacy is internationally recognized: ISSN 2354-1210\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">In 2016, The journal has been included in the list of medical science journals by The State Council for professorship which is awarded a work score of 0-0.5 points for a published article.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Can Tho Journal of Medicine and Pharmacy welcome original works that haven’t been submitted or published in other medical journals. Posts must contain content related to one of the journal’s categories.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The content published\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The journal is divided into 3 categories:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Scientific research article: are valuable scientific works, which have been researched and accepted.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Overview of medicine, biology and pharmacy: serving the objective of continuing training in the fields of medicine, biology and pharmacy; to systematize classical and modern knowledge.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Update information on new knowledge about medicine, biology, pharmacy in the country and in the world.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Scope\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Publication and introduction of scientific research in the fields:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Medicine (internal medicine, surgery, pediatrics, obstetrics and gynecology, odonto-stomatology, laboratory, oncology, traditional medicine, nursing).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Biology (genetics, biotechnology).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Pharmacology (pharmaceutics, drug quality analysis-control, synthetic pharmaceutical chemistry, biochemistry, pharmacognosy, botany, clinical pharmacy).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- To enhance the quality of undergraduate, postgraduate education, scientifically researching and meet the necessary treatment in hospital.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Introducing the updated domestic and oversea information about science technology to promote scientific research and exchanging technology in local, other universities.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Exchanging pharmaceutical and medical information for social health developing in the Mekong Delta and Vietnam.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The object\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Postgraduate students, student of Can Tho University of Medicine and Pharmacy, scientists from schools, research institutes, hospitals, health centers, pharmaceutical companies of the Mekong Delta; other provinces and regions in Vietnam and other country.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Address\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Headquarters of Can Tho Journal of Medicine and Pharmacy, located Scientific Research and International Cooperation Office: 179 Nguyen Van Cu Street, An Khanh Ward, Ninh Kieu District, Can Tho City, Vietnam.\u003C\u002Fspan>\u003C\u002Fp>","\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Ngày 16\u002F7\u002F2015, Tạp chí Y Dược học Cần Thơ được cấp chỉ số quốc tế: ISSN 2354-1210.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 4\u002F2016, Tạp chí đã được Hội đồng Giáo sư ngành Y đưa vào danh sách các tạp chí khoa học Y học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Năm 2020 Tạp chí Y Dược học Cần Thơ đã được phê duyệt vào danh mục của các Hội đồng Giáo sư ngành Dược học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ ra 12 số\u002Fnăm, 180-200 trang\u002Fsố.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 12\u002F2022 Tạp chí Y Dược học Cần Thơ là thành viên của hệ thống Crossref và từ tháng 01\u002F2023 tạp chí thực hiện bình duyệt online kín 2 chiều nhằm tăng tính minh bạch, tin cậy của các công trình nghiên cứu khoa học và đảm bảo tốt nhất chất lượng khoa học của bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ, mục đích và phạm vi của tạp chí\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ và mục đích hoạt động của tạp chí: xuất bản nhằm mục đích phổ biến kết quả từ các đề tài nghiên cứu khoa học; giao lưu trao đổi khoa học, chia sẻ kinh nghiệm, học tập, đồng thời cập nhật thông tin khoa học mới trong các lĩnh vực y, sinh, dược học trong và ngoài nước.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phạm vi của tạp chí: Tạp chí xuất bản được chia thành 3 chuyên mục: (i) Bài báo nghiên cứu khoa học là kết quả công trình nghiên cứu khoa học có giá trị đã được triển khai nghiên cứu, (ii) Bài tổng quan y, sinh, dược học: phục vụ mục tiêu đào tạo liên tục trong lĩnh vực y, sinh, dược học; nhằm hệ thống hóa những kiến thức kinh điển và hiện đại; (iii) Thông tin cập nhật kiến thức mới về y, sinh, dược học trong nước và trên thế giới.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Chính sách truy cập mở\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ áp dụng chính sách truy cập mở đối với các bài báo đã xuất bản đến với độc giả, nhằm mở rộng cơ hội tiếp cận các kết quả nghiên cứu chất lượng cao và tăng cường trao đổi kiến thức. Tạp chí đăng tải trực tuyến (miễn phí) toàn văn các bài báo được công bố trên website của Tạp chí (https:\u002F\u002Ftapchi.ctump.edu.vn).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đạo đức xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ cam kết tuân thủ đạo đức xuất bản phù hợp với các hướng dẫn và tiêu chuẩn của the Committee on Publication Ethics (COPE), tuân thủ các nguyên tắc của COPE’s Core Practices, Best Practices Guidelines for Journal Editors và Guidelines on Good Publication Practices.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Bản thảo bài báo chỉ được chấp nhận khi được tác giả chịu trách nhiệm chính cam kết các nội dung sau: Các nội dung của bản thảo chưa được đăng tải toàn bộ hoặc một phần ở các tạp chí khác; Tất cả các tác giả đều có đóng góp một cách đáng kể vào quá trình nghiên cứu hoặc chuẩn bị bản thảo và cùng chịu trách nhiệm về các nội dung của bản thảo; Tuân thủ các biện pháp đảm bảo đạo đức nghiên cứu (ví dụ thỏa thuận đồng ý tham gia nghiên cứu).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Cam kết bảo mật\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí cam kết thực hiện và tuân thủ các quy định của luật và các văn bản hướng dẫn liên quan đến bảo mật thông tin cá nhân trên không gian mạng. Các thông tin mà người dùng (tác giả, độc giả, biên tập viên, người phản biện) nhập vào các biểu mẫu trên Hệ thống Quản lý xuất bản trực tuyến của tạp chí chỉ được sử dụng vào các mục đích đã được tuyên bố rõ ràng và sẽ không được cung cấp cho bất kỳ bên thứ ba nào khác, hay dùng vào bất kỳ mục đích nào khác.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phí gửi bài\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng bài: 1.000.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng nhanh: 1.500.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với tác giả là cán bộ viên chức thuộc Trường Đại học Y Dược Cần Thơ thì được hỗ trợ 50% lệ phí gửi đăng bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với sinh viên thực hiện đề tài nghiên cứu khoa học cấp trường được hỗ trợ 100% lệ phí đăng bài ( Tác giả gửi đính kèm “ Quyết định về việc giao tổ chức thực hiện đề tài nghiên cứu khoa học cấp Trường của sinh viên”).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Hình thức nộp lệ phí:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Tiền mặt:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Nộp trực tiếp tại Phòng Tài chính - Kế toán, Trường Đại học Y Dược Cần Thơ, số 179 Nguyễn Văn Cừ, P. An Khánh, Q. Ninh Kiều, thành phố Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Chuyển khoản:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tên Tài khoản: Trường ĐHYD Cần Thơ, Số TK: 0111000115668, tại ngân hàng Vietcombank chi nhánh Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Thời gian: Áp dụng từ ngày 01\u002F02\u002F2023.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">* Phí gửi bài không được hoàn trả khi bài viết bị từ chối hoặc tác giả xin rút bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Quy trình phản biện bài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ thực hiện quy trình phản biện kín hai chiều nghiêm ngặt. Danh tính của những người phản biện không được tiết lộ cho các tác giả và ngược lại. Quy trình thẩm định bài báo đăng gồm các bước sau:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tiếp nhận bản thảo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tác giả liên hệ gửi bản thảo đến Tạp chí qua hệ thống trực tuyến tại website: https:\u002F\u002Ftapchi.ctump.edu.vn. Hướng dẫn về cách đăng ký, gửi bài và chuẩn bị bản thảo được cung cấp trên website của Tạp chí.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sàng lọc sơ bộ\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sau khi Tòa soạn nhận được bài báo của tác giả, Ban Thư ký sẽ tiến hành kiểm tra sơ bộ bài báo (các yêu cầu về nội dung và hình thức). Những bài báo không đúng quy cách hoặc có nội dung không phù hợp hoặc vi phạm bản quyền sẽ bị từ chối (Ban Thư ký thông báo phản hồi đến tác giả trong vòng 1 tuần). Những bài báo đủ điều kiện, được Ban Thư ký tòa soạn chuyển đến Ban Biên tập có cùng chuyên môn với nội dung bài báo để đề xuất người phản biện. Thời gian kể từ khi Ban Biên tập nhận bài báo đến khi đề xuất người phản biện bài báo chậm nhất là 5 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Vòng phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký gửi bài và yêu cầu phản biện đến 02 phản biện độc lập.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Các phản biện gởi nhận xét cho Ban Thư ký. Thời gian từ khi gửi bài cho phản biện đến khi nhận ý kiến của phản biện tối đa là 20 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xử ký kết quả phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Nếu ý kiến đồng ý cho đăng và không cần chỉnh sửa, Ban Thư ký tiếp tục đăng bài theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Nếu ý kiến đồng ý đăng và cần chỉnh sửa, Ban Thư ký sẽ thông tin đến tác giả chỉnh sửa theo yêu cầu của người phản biện. Thời gian chỉnh sửa và gửi lại kéo dài không quá 2 tuần, từ khi tác giả bài báo nhận được thông tin (Quá trình này có thể lặp lại tối đa 2 lần\u002F1 bài báo). Khi có sự thống nhất, đồng ý của người phản biện; bài báo được tiếp tục đăng theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Những bài báo có chất lượng không đạt yêu cầu, cả 2 phản biện không đồng ý cho đăng sẽ bị Tòa soạn từ chối đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký tổng hợp các bản thảo đã được tác giả hoàn thiện sau thẩm định trình Ban Biên tập xem xét, Tổng Biên tập phê duyệt, quyết định bài đăng theo các tiêu chí: sự phù hợp nội dung với tôn chỉ và mục đích, thể loại bài viết (ưu tiên các bài có bài có nghiên cứu chuyên sâu, hàm lượng khoa học cao), đóng góp mới bài báo, bài báo được ưu tiên đăng trong số gần nhất của Tạp chí theo thứ tự: tính thời sự, chất lượng bài báo và thời gian gửi bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Ban Biên tập và Ban Thư ký biên tập bản thảo, chế bản, đọc rà soát lỗi. Thời gian hoàn thành từ 10-15 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Ban Thư ký có trách nhiệm thông báo cho tác giả bài báo (bằng e-mail) về tình hình phê duyệt bài báo, thời gian, số kỳ, tập xuất bản bài báo theo qui định.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">4. Danh sách bài báo theo số Tạp chí được in ấn và phát hành trong năm định kỳ được công bố chính thức trên website: https:\u002F\u002Ftapchi.ctump.edu.vn\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>",{"VOID":490},"wcQ1uqwAAAAJ","2023-05-30T08:17:21.868+00:00",[],[494],{"id":495,"createTime":28,"updateTime":28,"relativeEntities":496,"slug":28,"properties":497,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":507,"parentIds":508,"statistic":28},"6413896b-eca9-442b-a73f-182a58a0ce40",[],{"title":498,"address":501,"country":504,"abbreviation":505},{"EN":499,"VI":500},"Can Tho University of Medicine and Pharmacy","Trường Đại học Y Dược Cần Thơ",{"EN":502,"VI":503},"No 179, Nguyen Van Cu street, An Khanh ward, Ninh Kieu district, Can Tho city, Vietnam","Số 179, đường Nguyễn Văn Cừ, phường An Khánh, quận Ninh Kiều, thành phố Cần Thơ, Việt Nam",{"VOID":15},{"VOID":506},"ctump","http:\u002F\u002Fwww.ctump.edu.vn\u002F",[],[],"https:\u002F\u002Ftapchi.ctump.edu.vn\u002Findex.php\u002Fctump",{"impactFactor":32,"impactFactorByYear":512,"i10Index":32,"i10IndexLast5Year":32,"totalPublication":514,"totalPublicationByYear":515,"totalCitation":520,"totalCitationByYear":521,"totalCitationPerPublication":108,"totalCitationPerPublicationByYear":523,"hindexLast5Year":45,"hindex":45},{"2022":513,"2023":111,"2024":106},0.01,1556,{"2020":47,"2021":516,"2022":517,"2023":518,"2024":519,"2025":122},57,306,801,358,161,{"2021":146,"2022":280,"2023":522},99,{"2021":524,"2022":318,"2023":104},0.23,{"impactFactor":28,"impactFactorByYear":28,"i10Index":123,"i10IndexLast5Year":123,"totalPublication":526,"totalPublicationByYear":527,"totalCitation":526,"totalCitationByYear":528,"totalCitationPerPublication":40,"totalCitationPerPublicationByYear":531,"hindexLast5Year":49,"hindex":49},476,{"0":205,"2019":123,"2021":139,"2022":459,"2023":451,"2024":357,"2025":49,"2026":48},{"2021":42,"2022":123,"2023":161,"2024":529,"2025":360,"2026":530},136,83,{"2021":105,"2022":513,"2023":532,"2024":127,"2025":533,"2026":534},0.62,25.43,13.83,{"id":536,"createTime":537,"updateTime":382,"relativeEntities":538,"slug":539,"properties":540,"entityType":25,"verifyStatus":26,"verifyTime":28,"verifyNote":28,"languages":552,"translateLanguages":28,"viewCount":133,"subjectFields":553,"manageAffiliations":554,"indexDatabases":555,"url":556,"thumbnailPath":557,"statistic":558,"gsStatistic":594,"type":55,"analyzePriority":28},"6984a56a-db70-403b-9cc4-4013e1ceaffa","2023-05-09T06:47:40.346+00:00",[],"T%E1%BA%A1p%20ch%C3%AD%20Nghi%C3%AAn%20c%E1%BB%A9u%20n%C6%B0%E1%BB%9Bc%20ngo%C3%A0i",{"country":541,"issn":542,"title":544,"introduce":547,"gsId":550},{"VOID":15},{"VOID":543},"25252445",{"EN":545,"VI":546},"VNU Journal of Foreign Studies","Tạp chí Nghiên cứu nước ngoài",{"EN":548,"VI":549},"{\"ops\":[{\"insert\":\"\\n\\nThe \\n\"},{\"attributes\":{\"italic\":true},\"insert\":\"VNU Journal of Science\"},{\"insert\":\"\\n was established in 1985 for the publication of national and international research papers in all fields of natural sciences and technology, social sciences and humanities. 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Transjugular intrahepatic portosystemic shunt (TIPS) is a well-established procedure for treating portal hypertension, but the safety of TIPS during pregnancy is largely unknown. In this series, we review five patients who underwent TIPS placement while pregnant and describe their clinical outcomes. Five pregnant patients with cirrhosis and portal hypertension underwent elective TIPS for complications of portal hypertension (four for secondary prevention of variceal bleeding and one for refractory ascites). Outcomes measured were recurrent bleeding episodes or need for further paracenteses during pregnancy, estimated radiation dose to the fetus and gestational age at delivery. All patients were followed after delivery to evaluate technical and clinical success of the procedure. All five patients survived pregnancy and went on to deliver successfully. When TIPS was performed for secondary prevention of variceal bleeding (n = 4), no patients demonstrated variceal bleeding after TIPS placement. When TIPS was performed for refractory ascites (n = 1), no further paracenteses were required. All patients delivered successfully, albeit prematurely. Average radiation dose estimated to the fetus was 16.3 mGy. This series suggests that TIPS can be performed in selective pregnant patients with portal hypertension, with little added risk to the mother or fetus.",{"EN":1056},"Transjugular Intrahepatic Portosystemic Shunt Placement During Pregnancy: A Case Series of Five Patients",{"VOID":1058},"Russell MA, Craigo SD (1998) Cirrhosis and portal hypertension in pregnancy. Semin Perinatol 22:156–165\nLeung W, Wong F (2011) Medical management of ascites. Expert Opin Pharmacother 12(8):1269–1283\nBiecker E (2013) Portal hypertension and gastrointestinal bleeding: diagnosis, prevention and management. World J Gastroenterol 19(31):5035–5050\nFidelman N, Kwan SW, LaBerge JM et al (2012) The transjugular intrahepatic portosystemic shunt: an update. Am J Roentgenol 199(4):746–755\nWildberger JE, Vorwerk D, Winograd R et al (1998) New TIPS placement in pregnancy in recurrent esophageal varices hemorrhage—assessment of fetal radiation exposure. Rofo 169(4):429–431\nSavage C, Patel J, Lepe MR et al (2007) Transjugular intrahepatic portosystemic shunt creation for recurrent gastrointestinal bleeding during pregnancy. J Vasc Interv Radiol 18(7):902–904\nLodato F, Cappelli A, Montagnani M et al (2008) Transjugular intrahepatic portosystemic shunt: a case report of rescue management of unrestrainable variceal bleeding in a pregnant woman. Dig Liver Dis 40(5):387–390\nWieseler KM, Bhargava P, Kanal KM et al (2010) Imaging in pregnant patients: examination appropriateness. Radiographics 30(5):1215–1229 (discussion 1230–1233)\nHurwitz LM, Yoshizumi T, Reiman RE et al (2006) Radiation dose to the fetus from body MDCT during early gestation. Am J Roentgenol 186(3):871–876\nDauer LT, Thornton RH, Miller DL et al (2012) Radiation management for interventions using fluoroscopic or computed tomographic guidance during pregnancy: a joint guideline of the Society of Interventional Radiology and the Cardiovascular and Interventional Radiological Society of Europe with Endorsement by the Canadian Interventional Radiology Association. J Vasc Interv Radiol 23(1):19–32\nHuda W (2009) Review of radiologic physics, 3rd edn. Lippincott Williams & Wilkins, Philadelphia\nHomburg R, Bayer I, Lurie B (1988) Bleeding esophageal varices in pregnancy. A report of two cases. J Reprod Med 33:784–786\nBritton RC (1982) Pregnancy and esophageal varices. Am J Surg 143:421–425\nZeeman GG, Moise JK (1999) Prophylactic banding of severe esophageal varices associated with liver cirrhosis in pregnancy. Obstet Gynecol 94:842\nTan J, Surti B, Saab S (2008) Pregnancy and cirrhosis. Liver Transpl 14:1081–1091\nHay JE (2008) Liver disease in pregnancy. Hepatology 47:1067–1076\nHebert MF, Carr DB, Anderson GD et al (2005) Pharmacokinetics and pharmacodynamics of atenolol during pregnancy and postpartum. J Clin Pharmacol 45(1):25–33\nAnderson GD, Carr DB (2009) Effect of pregnancy on the pharmacokinetics of antihypertensive drugs. Clin Pharmacokinet 48(3):159–168\nWagner LK, Lester RG, Saldana LR (1997) Exposure of the pregnant patient to diagnostic radiations: a guide to medical management, 2nd edn. Medical Physics, Madison\nAmerican College of Radiology (2008) ACR practice guideline for imaging pregnant or potentially pregnant adolescents and women with ionizing radiation. American College of Radiology, Reston\nMartin JN Jr, Ridgway LE 3rd, Connors JJ et al (1990) Angiographic arterial embolization and computed tomography-directed drainage for the management of hemorrhage and infection with abdominal pregnancy. Obstet Gynecol 76(5 pt 2):941–945\nBrent RL (2009) Saving lives and changing family histories: appropriate counseling of pregnant women and men and women of reproductive age, concerning the risk of diagnostic radiation exposures during and before pregnancy. Am J Obstet Gynecol 200(1):4–24\nBushberg JT (2002) The essential physics of medical imaging, 2nd edn, Lippincott Williams & Wilkins, Philadelphia\nFarsad K, Fuss C, Kolbeck KJ et al (2012) Transjugular intrahepatic portosystemic shunt creation using intravascular ultrasound guidance. J Vasc Interv Radiol 23(12):1594–1602\nRaza SA, Walser E, Hernandez A et al (2006) Transhepatic puncture of portal and hepatic veins for TIPS using a single-needle pass under sonographic guidance. 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objective of this study was to investigate the effect of transcatheter arterial embolization (TAE) with N-butyl-2-cyanoacrylate (NBCA)–Lipiodol mixture in patients with bronchial artery aneurysm (BAA). From January 2005 to January 2010, five patients presenting hemoptysis with six BAAs were treated with NBCA–Lipiodol mixture, including intra-aneurysm embolization (IAE) in one patient. Adjuvant embolization with spherical polyvinyl alcohol (PVA) embolic microparticles or NBCA was first performed to embolize the distal engorged bronchiectatic arteries. Bronchial arterial angiography showed six BAAs (four in the right lobe and two in the left lobe) and some engorged, tortuous bronchial arteries. TAE through microcatheter was successful in all cases. Postembolization angiogram demonstrated the NBCA cast and total occlusion of BAAs and bronchiectatic engorged vessels. After these procedures, hemoptysis completely disappeared in all patients. Follow-up computed tomography (CT) scan was performed at an average of 3 months (range 2 to 6), which showed no enhancement of BAAs and accumulation of NBCA. 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Ann Thorac Surg 73:632–633\nHaddad R (2004) Bronchial artery aneurysm. Eur J Cardiothorac Surg 26:213\nKalva SP, Wicky S (2006) Mediastinal bronchial artery aneurysms: endovascular therapy in two patients. Catheter Cardiovasc Interv 68:858–861\nTsolaki E, Salviato E, Coen M, Galeotti R, Mascoli F (2007) Double right bronchial artery aneurysm treated with combined procedures. Eur J Vasc Endovasc Surg 34:537–539\nMisselt AJ, Krowka MJ, Misra S (2010) Successful coil embolization of mediastinal bronchial artery aneurysm. J Vasc Interv Radiol 21:295–296\nPugnale M, Portier F, Lamarre A, Halkic N, Riis HB, Wicky S et al (2001) Hemomediastinum caused by rupture of a bronchial artery aneurysm: successful treatment by embolization with N-butyl-2-cyanoacrylate. J Vasc Interv Radiol 12:1351–1352\nAburano H, Kawamori Y, Horiti Y, Kitagawa K, Sanada J, Matsui O (2006) Bronchial artery aneurysm embolization with NBCA. Cardiovasc Interv Radiol 29:1141–1143\nMizuguchi S, Inoue K, Kida A, Isota M, Hige K, Aoyama T et al (2009) Ruptured bronchial artery aneurysm associated with bronchiectasis: a case report. Ann Thorac Cardiovasc Surg 15:115–118\nShaer AH, Bashist B (1989) Computed tomography of bronchial artery aneurysm with erosion into the esophagus. J Comput Assist Tomogr 13:1069–1071\nHoffmann V, Ysebaert D, De Schepper A, Colpaert C, Jorens P (1996) Acute superior vena cava obstruction after rupture of a bronchial artery aneurysm. Chest 110:1356–1358\nOka M, Fukuda M, Terashi K, Takatani H, Narasaki F, Nakano R et al (1997) Bronchial artery aneurysm as a cause of atelectasis. Intern Med 36:917–919\nKalangos A, Khatchatourian G, Panos A, Faidutti B (1997) Ruptured mediastinal bronchial artery aneurysm: a dilemma of diagnosis and therapeutic approach. J Thorac Cardio Vasc Surg 114:853–856\nCearlock JR, Fontaine AB, Urbaneja A, Spigos DG (1995) Endovascular treatment of a posttraumatic bronchial artery pseudoaneurysm. J Vasc Intev Radiol 6:495–496\nSanchez E, Alados P, Zurera L, Canis M, Muñoz I, Casares J et al (2007) Bronchial artery aneurysm treated with aortic stent graft and fibrin sealant. Ann Thorac Surg 83:693–695\nChun JY, Morgan R, Belli AM (2010) Radiological management of hemoptysis: a comprehensive review of diagnostic imaging and bronchial arterial embolization. Cardiovasc Interv Radiol 33:240–250\nKervancioglu S, Andic C, Bayram N, Telli C, Sarica A, Sirikci A (2008) Bronchial artery embolization in the management of pulmonary parenchymal endometriosis with hemoptysis. Cardiovasc Interv Radiol 31:824–827\nYonemitsu T, Kawai N, Sato M, Tanihata H, Takasaka I, Nakai M et al (2009) Evaluation of transcatheter arterial embolization with gelatin sponge particles, microcoils, and N-butyl cyanoacrylate for acute arterial bleeding in a coagulopathic condition. J Vasc Interv Radiol 20:1176–1187\nSakai T, Razavi MK, Semba CP, Kee ST, Sze DY, Dake MD (1998) Percutaneous treatment of bronchial artery aneurysm with use of transcatheter coil embolization and thoracic aortic stent-graft placement. J Vasc Interv Radiol 9:1025–1028\nKasashima F, Endo M, Kosugi I, Matsumoto Y, Abe Y, Sasaki H et al (2003) Mediastinal bronchial artery aneurysm treated with a stent-graft. J Endovasc Ther 10:381–385\nBaltacioğlu F, Cimşit NC, Bostanci K, Yüksel M, Kodalli N (2010) Transarterial microcatheter glue embolization of the bronchial artery for life-threatening hemoptysis: technical and clinical results. 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Ablation of Liver Tumours: The Crucial Role of 3D Imaging",{"VOID":1414},"Crocetti L, de Baere T, Pereira PL, Tarantino FP. CIRSE standards of practice on thermal ablation of liver tumours. Cardiovasc Interv Radiol. 2020. https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs00270-020-02471-z.\nCalandri M, Mauri G, Yevich S, Gazzera C, Basile D, Gatti M, Veltri A, Fonio P. Fusion imaging and virtual navigation to guide percutaneous thermal ablation of hepatocellular carcinoma: a review of the literature. Cardiovasc Interv Radiol. 2019;42(5):639–47. https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs00270-019-02167-z.\nMonfardini L, Orsi F, Caserta R, Sallemi C, Della Vigna P, Bonomo G, Varano G, Solbiati L, Mauri G. Ultrasound and cone beam CT fusion for liver ablation: technical note. Int J Hyperthermia. 2018;35(1):500–4. https:\u002F\u002Fdoi.org\u002F10.1080\u002F02656736.2018.1509237.\nCrocetti L, de Baere T, Lencioni R. Quality improvement guidelines for radiofrequency ablation of liver tumours. Cardiovasc Interv Radiol. 2010;33:11–7. https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs00270-009-9736-y.\nSolbiati M, Muglia R, Goldberg SN, Ierace T, Rotilio A, Passera KM, et al. A novel software platform for volumetric assessment of ablation completeness. Int J Hyperthermia. 2019;36(1):337–43.",{"VOID":1416},"10.1007\u002Fs00270-020-02560-z","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs00270-020-02560-z",[1419,1443,1467,1480],{"id":1420,"sortIndex":32,"researcher":28,"roles":1421,"affiliations":1422,"properties":1440},"e62a4e2c-3ffe-4e4e-bbf7-472e2eb6e61a",[1068],[1423,1431],{"id":1424,"sortIndex":32,"affiliation":1425,"properties":28},"0fe7acf9-a3e5-44d3-ab40-f3fd06f99b77",{"id":1424,"createTime":28,"updateTime":28,"relativeEntities":1426,"slug":28,"properties":1427,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1430,"statistic":28},[],{"title":1428},{"VI":1429},"Division of Interventional Radiology, IEO, European Institute of Oncology IRCCS, Milan, Italy",[],{"id":1432,"sortIndex":40,"affiliation":1433,"properties":1439},"a01cd6f0-e6c4-4eec-b470-9ee18f469eeb",{"id":1432,"createTime":28,"updateTime":28,"relativeEntities":1434,"slug":28,"properties":1435,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1438,"statistic":28},[],{"title":1436},{"VI":1437},"Department of Oncology and Hematolgy-Oncology, Università degli studi di Milano, Milan, Italy",[],{},{"title":1441},{"VI":1442},"Giovanni Mauri",{"id":1444,"sortIndex":40,"researcher":28,"roles":1445,"affiliations":1446,"properties":1464},"da20236d-7276-4748-88be-0a2efec04d04",[1068],[1447,1455],{"id":1448,"sortIndex":32,"affiliation":1449,"properties":28},"b2c9560f-e80e-460e-9195-5ca1071bb397",{"id":1448,"createTime":28,"updateTime":28,"relativeEntities":1450,"slug":28,"properties":1451,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1454,"statistic":28},[],{"title":1452},{"VI":1453},"Department of Biomedical Sciences, Humanitas University, Pieve Emanuele, Milan, Italy",[],{"id":1456,"sortIndex":40,"affiliation":1457,"properties":1463},"bd4f66b6-6495-4891-9972-b6f70f891e86",{"id":1456,"createTime":28,"updateTime":28,"relativeEntities":1458,"slug":28,"properties":1459,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1462,"statistic":28},[],{"title":1460},{"VI":1461},"Department of Radiology, Humanitas Clinical and Research Hospital, Rozzano, Milan, Italy",[],{},{"title":1465},{"VI":1466},"Luigi Solbiati",{"id":1468,"sortIndex":123,"researcher":28,"roles":1469,"affiliations":1470,"properties":1477},"3bfccbf0-a68c-44cf-b93c-41dfe4daba3f",[1068],[1471],{"id":1424,"sortIndex":32,"affiliation":1472,"properties":28},{"id":1424,"createTime":28,"updateTime":28,"relativeEntities":1473,"slug":28,"properties":1474,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1476,"statistic":28},[],{"title":1475},{"VI":1429},[],{"title":1478},{"VI":1479},"Franco Orsi",{"id":1481,"sortIndex":42,"researcher":28,"roles":1482,"affiliations":1483,"properties":1492},"58bb8852-efc7-40d5-8f11-98dc67130fd3",[1068],[1484],{"id":1485,"sortIndex":32,"affiliation":1486,"properties":28},"931dbbe2-ecdf-4c4a-b3f0-46380aefeac8",{"id":1485,"createTime":28,"updateTime":28,"relativeEntities":1487,"slug":28,"properties":1488,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1491,"statistic":28},[],{"title":1489},{"VI":1490},"Division of Interventional Radiology, Fondazione Poliambulanza, Brescia, Italy",[],{"title":1493},{"VI":1494},"Lorenzo Monfardini",{"url":1417,"publisher":1496,"properties":1542},{"id":868,"createTime":869,"updateTime":870,"relativeEntities":1497,"slug":872,"properties":1498,"entityType":25,"verifyStatus":880,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":32,"subjectFields":1502,"manageAffiliations":1511,"indexDatabases":1522,"url":28,"thumbnailPath":28,"statistic":1537,"gsStatistic":28,"type":28,"analyzePriority":28},[],{"issn":1499,"title":1500,"eissn":1501},{"VOID":875},{"EN":877},{"VOID":879},[1503,1507],{"id":883,"createTime":28,"updateTime":28,"relativeEntities":1504,"label":1505,"description":1506,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":886},{},{"id":889,"createTime":28,"updateTime":28,"relativeEntities":1508,"label":1509,"description":1510,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":892},{},[1512,1517],{"id":896,"createTime":28,"updateTime":28,"relativeEntities":1513,"slug":28,"properties":1514,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1516,"statistic":28},[],{"title":1515},{"EN":900},[],{"id":903,"createTime":28,"updateTime":28,"relativeEntities":1518,"slug":28,"properties":1519,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1521,"statistic":28},[],{"title":1520},{"EN":907},[909],[1523,1530],{"id":929,"indexDatabase":1524,"url":935,"indexYears":936,"academicFieldIds":1529,"indexDatabaseRanking":940},{"id":775,"createTime":28,"updateTime":28,"relativeEntities":1525,"label":1526,"description":1527,"key":781,"publicationTags":1528,"standard":28},[],{"EN":778,"VI":778},{"EN":778,"VI":780},[783],[938,939],{"id":912,"indexDatabase":1531,"url":924,"indexYears":28,"academicFieldIds":1536,"indexDatabaseRanking":28},{"id":914,"createTime":28,"updateTime":28,"relativeEntities":1532,"label":1533,"description":1534,"key":921,"publicationTags":1535,"standard":28},[],{"EN":917,"VI":917},{"EN":919,"VI":920},[923,813],[926,927],{"impactFactor":32,"impactFactorByYear":1538,"i10Index":944,"i10IndexLast5Year":281,"totalPublication":945,"totalPublicationByYear":1539,"totalCitation":965,"totalCitationByYear":1540,"totalCitationPerPublication":1005,"totalCitationPerPublicationByYear":1541,"hindexLast5Year":208,"hindex":208},{"2012":225,"2013":943,"2014":840,"2015":319,"2016":422,"2017":440,"2018":840,"2019":696,"2020":221,"2021":840,"2022":170,"2023":284},{"1978":202,"1979":142,"1980":516,"1981":206,"1982":516,"1983":200,"1984":160,"1985":152,"1986":358,"1987":428,"1988":564,"1989":947,"1990":161,"1991":599,"1992":157,"1993":428,"1994":333,"1995":155,"1996":530,"1997":567,"1998":161,"1999":209,"2000":334,"2001":567,"2002":948,"2003":154,"2004":949,"2005":684,"2006":950,"2007":951,"2008":952,"2009":953,"2010":954,"2011":836,"2012":955,"2013":956,"2014":957,"2015":570,"2016":958,"2017":959,"2018":614,"2019":960,"2020":961,"2021":962,"2022":963,"2023":964,"2024":600},{"1978":122,"1979":967,"1980":325,"1981":968,"1982":969,"1983":970,"1984":971,"1985":972,"1986":522,"1987":156,"1988":831,"1989":973,"1990":210,"1991":974,"1992":605,"1993":975,"1994":976,"1995":977,"1996":978,"1997":979,"1998":980,"1999":981,"2000":982,"2001":983,"2002":984,"2003":985,"2004":986,"2005":987,"2006":988,"2007":989,"2008":990,"2009":991,"2010":992,"2011":993,"2012":994,"2013":995,"2014":996,"2015":997,"2016":998,"2017":999,"2018":1000,"2019":1001,"2020":1002,"2021":1003,"2022":1004,"2023":137,"2024":145},{"1978":286,"1979":1007,"1980":1008,"1981":694,"1982":1005,"1983":1009,"1984":1010,"1985":1011,"1986":1012,"1987":1013,"1988":1014,"1989":1015,"1990":1016,"1991":1017,"1992":1018,"1993":1019,"1994":1020,"1995":1021,"1996":1005,"1997":1022,"1998":1023,"1999":1023,"2000":1024,"2001":1025,"2002":1026,"2003":1027,"2004":593,"2005":1028,"2006":1029,"2007":1030,"2008":1031,"2009":1032,"2010":1033,"2011":1034,"2012":1035,"2013":1036,"2014":1037,"2015":593,"2016":1038,"2017":1039,"2018":1040,"2019":177,"2020":1041,"2021":741,"2022":338,"2023":365,"2024":104},{"pages":1543,"volume":1545},{"VOID":1544},"1416-1417",{"VOID":1546},"43","2020-06-17",2020,[940,923],{"id":1551,"createTime":1552,"updateTime":1553,"relativeEntities":1554,"slug":1555,"properties":1556,"entityType":1061,"verifyStatus":26,"verifyTime":1565,"verifyNote":1062,"languages":28,"translateLanguages":28,"viewCount":32,"primaryUrl":1566,"fullTextUrl":28,"authors":1567,"publicationType":1165,"publisherRelationship":1613,"citationCount":28,"citationInfo":28,"publishDate":1665,"publishYear":1666,"citationAnalyzeStatus":880,"lastCitationAnalyze":28,"indexDatabases":1667,"openAccess":28,"references":28,"isForceReanalyzing":1221},"00147083-b43c-47be-96e1-f6db6cdad091","2023-12-14T16:09:55.376+00:00","2025-01-07T02:10:14.882+00:00",[],"Selective-thrombolysis-in-acute-deep-vein-thrombosis-Evaluation-of-adjuvant-therapy-in-vivo",{"abstract":1557,"title":1559,"references":1561,"doi":1563},{"EN":1558},"Purpose: To evaluate in a porcine model of acute deep vein thrombosis (DVT) the efficacy of dalteparin and antithrombin with respect to heparin for local adjuvant therapy during selective thrombolysis, and the utility of nitroglycerin and iloprost as heparin supplements. Methods: DVT was induced in both hind limbs using a previously described technique (n=20). Thirty minutes later, the animal was heparinized (2500 IU IV), and bilateral sequestrated thrombolysis was performed using 8 mg alteplase: both external iliac veins were endoluminally occluded with Swan-Ganz catheters, and a multi-sideport infusion wire coaxially introduced through each catheter and advanced into the ipsilateral popliteal vein. In the control limbs, tissue plasminogen activator (tPA) 8 mg was injected as 0.8-ml boluses at 3-min intervals for 2 hr as a 0.25-mg\u002Fml solution containing heparin 50 IU\u002Fml (n=20). On the contralateral side, heparin was substituted with either dalteparin 50 IU\u002Fml (n=5) or antithrombin 12.5 IU\u002Fml (n=5), or supplemented with either nitroglycerin 0.075 mg\u002Fml (n=5) or iloprost (150 ng\u002Fml) (n=5). Blood samples were taken at predetermined intervals to measure the activated partial thromboplastin time (aPTT), prothrombin time (PT), and fibrinogen concentration. At autopsy, the thrombus mass in the iliofemoral veins was measured, and the extent of residual thrombosis in the venous tributaries graded at four sites. Results: Bilateral thrombolysis was successfully completed in all animals. The median thrombus mass in the iliofemoral veins after thrombolysis was 0.48 g (range 0.06–1.58 g), 0.95 g (0.59–1.29 g), 0.74 g (0.52–0.96 g), and 0.29 g (0.0–0.77 g) for dalteparin, antithrombin, iloprost, and nitroglycerin respectively, as compared with 0.53 g (0.18–0.88 g) (p=0.69), 0.97 g (0.46–1.15 g) (p=0.69), 0.53 g (0.48–1.10 g) (p=0.69), and 0.18 g (0.13–1.04 g) (p=0.5) for the respective controls. Likewise, the severity of residual thrombosis in the venous tributaries was not affected by the constituents of adjuvant therapy. Nitroglycerin induced a small drop in blood pressure, which was transient. The temporal change in aPTT was similar in all four groups. Invariably PT progressively shortened during thrombolysis (p=0.0001); this effect was somewhat blunted with antithrombin. Fibrinogen levels demonstrated a time-dependent increase (p=0.004) that was not influenced by the adjuvant therapy used. Conclusions: Dalteparin or antithrombin demonstrated no appreciable advantage over heparin as local adjuvant therapy for selective venous thrombolysis. Supplementation of heparin with iloprost or nitroglycerin also had virtually no effect on thrombolytic efficacy.",{"EN":1560},"Selective thrombolysis in acute deep vein thrombosis: Evaluation of adjuvant therapy in vivo",{"VOID":1562},"Semba CP, Dake M (1994) Iliofemoral deep venous thrombosis: Aggressive therapy with catheter-directed thrombolysis. Radiology 191:487–494\nBjarnason H, Kruse JR, Asinger DA, Nazarian GK, Dietz CA Jr, Caldwell MD, Key NS, Hirsch AT, Hunter DW (1997) Iliofemoral deep vein thrombosis: Safety and efficacy outcome during 5 years of catheter-directed thrombolytic therapy. J Vasc Interv Radiol 8:405–418\nThéry C, Bauchart JJ, Lesenne M, Asseman P, Flajollet JG, Legghe R, Marache P (1992) Predictive factors of effectiveness of streptokinase in deep venous thrombosis. Am J Cardiol 69:117–122\nMewissen MW (1997) Catheter-directed thrombolytic therapy of the lower extremity: Interim data report from the data collection center. J Vasc Interv Radiol 8[Suppl]:50–52\nDiamond SL, Anand A (1993) Inner clot diffusion and permeation during fibrinolysis. Biophys J 65:2622–2643\nRoy S, Lærum F, Brosstad F (1998) Quantitative evaluation of thrombolysis techniques: Influence of catheter characteristics and delivery parameters. 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Thromb Haemost 71:300–304\nHoppensteadt DA, Jeske W, Fareed J, Bermes EW JR (1995) The role of tissue factor pathway inhibitor in the mediation of antithrombotic actions of heparin and low molecular weight heparins. Blood Coagul Fibrinolysis 6[Suppl]:S57-S64\nFalkon L, Garí M, Barbanoj M, Amiral J, Fontcuberta J (1998) Tissue factor pathway inhibitor and anti-FXa kinetic profiles of a new low-molecular-mass heparin, Bemiparin, at therapeutic subcutaneous doses. Blood Coag Fibrinolysis 9:137–141\nMontalescot G, Philippe F, Ankri A, Vicaut E, Bearez F, Poulard JE, Carrie D, Flammang D, Dutoit A, Carayon A, Jardel C, Chevot M, Bastard JP, Bigonzi F, Thomas D (1998) Early increase of von Willebrand factor predicts adverse outcome in unstable coronary artery disease: Beneficial effects of enoxaparin. Circulation 98:294–299\nAntman EM, Handin R (1998) Low-molecular-weight heparins: An intriguing new twist with profound implications. Circulation 98:287–289\nBacher P, Welzel D, Iqbal O, Hoppensteadt D, Callas D, Walenga JM, Fareed J (1992) The thrombolytic potency of LMW-heparin compared to urokinase in rabbit jugular vein clot lysis model. Thromb Res 66:151–158\nNilsen DWT, Gøransson L, Larsen A-I, Hetland Ø, Kierulf P (1997) Systemic thrombin generation and activity resistant to low molecular weight heparin administered prior to streptokinase in patients with acute myocardial infarction. Thromb Haemost 77:57–61\nGrosset AB, Spiro TE, Beynon J, Rodgers GM (1997) Enoxaparin, a low-molecular weight heparin suppresses prothrombin activation more effectively than unfractionated heparin in patients treated for venous thromboembolism. Thromb Res 86:349–354\nWolzt M, Eder M, Weltermann A, Entlicher J, Eichler H-G, Kyrle PA (1997) Comparison of the effects of different low molecuar weight heparins on the haemostatic system activation in man. Thromb Haemost 78:876–879\nStassen J-M, Rappold MJ, Valinthout I, Collen D (1993) Comparative effects of enoxaparin and heparin on arterial and venous clot lysis with alteplase in dogs. Thromb Haemost 69:454–459\nMelissari E, Monte G, Lindo VS, Pemberton KD, Wilson NV, Edmondson R, Das S, Kakker VV (1992) Congenital thrombophilia among patients with venous thromboembolism. Blood Coagul Fibrinolysis 3:749–758\nFinazzi G, Barbui T (1994) Different incidence of venous thrombosis in patients with inherited deficiencies of antithrombin III, protein C and protein S. Thromb Haemost 71:15–18\nHumphries JE (1993) Acquired antithrombin III deficiency: Replacement with antithrombin III concentrates in a patient with protein S deficiency accelerates response to therapy. Acta Haematol 90:151–154\nPatton DF, Harper JL, Wooldridge TN, Gordon BG, Coccia P, Haire WD (1996) Treatment of veno-occlusive disease of the liver with bolus tissue plasminogen activator and continuous infusion of antithrombin III concentrate. Bone Marrow Transplant 17:443–447\nGrip L, Blömback M, Egberg N, Olsson Å, Svane B, Velander M (1997) Antithrombin III supplementation for patients undergoing PTCA for unstable angina pectoris: A controlled randomized double-blind pilot study. Eur Heart J 18:443–449\nReiter W, Ehrensberger H, Steinbrückner B, Keller F (1995) Parameters of haemostasis during acute venous thrombosis. Thromb Haemost 74:596–601\nBalk R, Emerson T, Fourrier F, Kruse JA, Mammen EF, Schuster H-P, Vinazzer H (1998) Therapeutic use of antithrombin concentrate in sepsis. Semin Thromb Hemost 24:183–194\nSchächinger V, Allert M, Kasper W, Just H, Vach W, Zeiher AM (1994) Adjunctive intracoronary infusion of antithrombin III during percutaneous transluminal coronary angioplasty: Results of a prospective, randomized trial. Circulation 90:2258–2266\nRao GHR, Reddy KR, White JG (1980) The influence of epinephrine on prostacyclin (PGI2) induced disaggregation of ADP aggregated platelets. Prostaglandins Leukotrienes Med 4:385–397\nSchafer AI, Zavoico GB, Loscalzo J, Maas AK (1987) Synergistic inhibition of platelet activation by plasmin and prostaglandin. Blood 69:1504–1507\nIocaviello L, de Curtis A, d’Amato MC, Amore C, Buczko W, de Gaetano G, Donati MB (1994) Prostacyclin is required for t-PA release after venous occlusion. Am J Physiol 266:H429-H434\nDarius H, Binz C, Veit K, Fisch A, Meyer J (1995) Platelet receptor desensitization induced by elevated prostacyclin levels causes platelet endothelial cell adhesion. J Am Coll Cardiol 26:800–806\nMacDermot J (1986) Desensitization of prostacyclin responsiveness in platelets: Apparent differences in the mechanism in vitro and in vivo. Biochem Pharmacol 35:2645–2649\nBecker RC, Corrao JM, Bovill EG, Gore JM, Baker SP, Miller ML, Lucas FV, Alpert JA (1990) Intravenous nitroglycerin-induced heparin resistance: A qualitative antithrombin III abnormality. Am Heart J 119:1254–1261\nBrack MJ, More RS, Hubner PJB, Gershlick AH (1993) The effect of low dose nitroglycerine on plasma heparin concentrations and activated plasma thromboplastin times. Blood Coag Fibrinolysis 4:183–186\nVillanueva GB, Parret Y, Danishefsky I (1980) Tryptophan residue at the tryptophan binding site of antithrombin III. Arch Biochem Biophys 203:453–457\nThomas GR, Thibodeaux H, Errett CJ, Badillo JM, Wu DT, Renfino CJ, Key BA, Bennett WF (1996) Limiting systemic plasminogenolysis reduces the bleeding potential for tissue-type plasminogen activators but not for streptokinase. Thromb Haemost 75:915–920\nBounameaux H, Banga JD, Bluhmki E, Coccheri S, Fiessinger J-N, Haarmann W, Lockner D, Mahler F, Ninet J, Schneider P-A, de Torrente A, van der Meer J, Verhaeghe R (1992) Double-blind, randomized comparison of systemic continuous infusion of 0.25 versus 0.50 mg\u002Fkg\u002F24 h of alteplase over 3–7 days for treatment of deep venous thrombosis: Results of the European Thrombolysis with rt-PA in Venous Thrombosis (ETTT) Trial. Thromb Haemost 67:306–309\nSchrör K, Darius H, Matzky R, Ohlendorf R (1981) The antiplatelet and cardiovascular effects of a new carbacyclin derivative (ZK36 374)-equipotent to PGI2 in vitro. Naunyn Schiemedebergs Arch Pharmacol 316:252–255\nKovacs IB, Mayou SC, Kirby JD (1991) Infusion of a stable prostacyclin analogue, iloprost, to patients with peripheral vascular disease: Lack of antiplatelet effect but risk of thrombosis. Am J Med 90:41–45\nKamikubo Y, Hamuro T, Takemoto S, Nakahara Y, Kamei S, Nakagaki T, Miyamoto S, Funatsu A, Kato H (1998) A kinetic analysis of the interaction of human recombinant tissue factor pathway inhibitor with factor Xa utilizing an immunoassay and the effect of antithrombin III\u002Fheparin on the complex formation. Thromb Res 89:179–186\nLaake K (1974) Cold-promoted activation of factor VII in human plasma: Studies on associated acyl arginine esterase activity. Thromb Res 4:769–774\nCollen D (1988) Synergism of thrombolytic agents: Investigational procedures and clinical potential. Circulation 77:731–735\nBagge L, Wahlberg T, Holmer E, Tydén H, Nyström SO (1994) Low-molecular-weight-heparin (Fragmin) versus heparin for anticoagulation during cardiopulmonary bypass in open heart surgery using a pig model. Blood Coag Fibrinolysis 5:265–272",{"VOID":1564},"10.1007\u002Fs002709900415","2025-01-07T02:10:14.881+00:00","http:\u002F\u002Flink.springer.com\u002F10.1007\u002Fs002709900415",[1568,1583,1598],{"id":1569,"sortIndex":32,"researcher":28,"roles":1570,"affiliations":1571,"properties":1580},"8b1a6c37-0dcc-4ad5-b641-fec3556ea75f",[1068],[1572],{"id":1573,"sortIndex":32,"affiliation":1574,"properties":28},"2c46b904-a731-4916-b688-bef8d6d23b06",{"id":1573,"createTime":28,"updateTime":28,"relativeEntities":1575,"slug":28,"properties":1576,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1579,"statistic":28},[],{"title":1577},{"VI":1578},"Institute for Surgical Research, National Hospital, Oslo, Norway",[],{"title":1581},{"VI":1582},"Sumit Roy",{"id":1584,"sortIndex":40,"researcher":28,"roles":1585,"affiliations":1586,"properties":1595},"4a8a9265-1303-4b39-afee-425305cc6a79",[1068],[1587],{"id":1588,"sortIndex":32,"affiliation":1589,"properties":28},"fadf2856-a81c-488f-b1c0-5245737f46a7",{"id":1588,"createTime":28,"updateTime":28,"relativeEntities":1590,"slug":28,"properties":1591,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1594,"statistic":28},[],{"title":1592},{"VI":1593},"Research Institute for Internal Medicine, National Hospital, Oslo, Norway",[],{"title":1596},{"VI":1597},"Frank Brosstad",{"id":1599,"sortIndex":123,"researcher":28,"roles":1600,"affiliations":1601,"properties":1610},"f1a45f20-9641-4c40-b6ea-1df89eda126c",[1068],[1602],{"id":1603,"sortIndex":32,"affiliation":1604,"properties":28},"3235e394-810c-411f-855e-15506c1af4a4",{"id":1603,"createTime":28,"updateTime":28,"relativeEntities":1605,"slug":28,"properties":1606,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1609,"statistic":28},[],{"title":1607},{"VI":1608},"Department of Physiology, Institute for Biology, University of Oslo, Oslo, Norway",[],{"title":1611},{"VI":1612},"Kjell S. 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results of 125 urokinase (UK) infusions in 108 patients were analyzed. Results showed total clot lysis in 61% of patients, partial lysis in 24%, and no lysis in 15%. Predictably, patients with acute occlusions had the highest incidence of complete clot lysis. However, clots older than 3 weeks had a surprisingly high lysis rate of 57%. None of the patients had a fibrinogen level below 100 mg\u002FdL. Overall incidence of bleeding (major and minor) was 15%; patients having the most severe bleeding were on concomitant anticoagulation. We conclude that UK is a safe and effective therapeutic option in patients with acute and chronic obstruction. Significant bleeding episodes are uncommon during UK therapy and are related primarily to ongoing anticoagulation.",{"EN":1678},"Technique and results of “low-dose” infusion",{"VOID":1680},"Dotter CT, Rosch J, Seamen AJ (1974) Selective clot lysis with low dose streptokinase. Radiology 111:31–37\nKatzen BT, van Breda A (1981) Low dose streptokinase in the treatment of arterial occlusions. AJR 136:1171–1178\nKatzen BT (1984) Percutaneous transluminal angioplasty for arterial disease of the lower extremities. AJR 142:23–25\nvan Breda A, Waltman AC (1983) Low dose thrombolysis in the treatment of arterial occlusive disease. Int Angiol 2:75–83\nTotty WG, Gilula LA, McClennan BL, Ahmed P, Sherman L (1982) Low dose intravascular fibrinolytic therapy. Radiology 143:59–69\nvan Breda A, Robison JC, Feldman L (1984) Local thrombolysis in the treatment of arterial graft occlusions. J Vasc Surg 1:103–112\nHargrove WC, Berkowitz HD, Freiman DB, McClean G, Ring EJ, Roberts B (1982) Recanalization of totally occluded femoropopliteal vein grafts with low-dose streptokinase infusion. Surgery 92:890–895\nBecker GJ, Rabe FE, Richmond BD, Holden RW, Yune HY, Dilley RS, Bang NU, Glover JL, Klatte EC (1983) Low dose fibrinolytic therapy. Radiology 148:663–670\nvan Breda A, Katzen BT, Deutsch AS (1987) Urokinase vs streptokinase in local thrombolysis. Presented at 70th Scientific Assembly and Ann Meet. Radiol Soc North Am, Washington, DC. Radiology 165:109–111\nMcNamara TO, Fischer JR (1985) Thrombolysis of peripheral arterial and graft occlusions: Improved results using high dose urokinase. AJR 144:769–775\nKatzen BT, Edwards KC, Albert AS, van Breda A (1984) Low dose fibrinolysis in peripheral vascular disease. J Vasc Dis 1:718–722",{"VOID":1682},"10.1007\u002FBF02577096","http:\u002F\u002Flink.springer.com\u002F10.1007\u002FBF02577096",[1685],{"id":1686,"sortIndex":32,"researcher":28,"roles":1687,"affiliations":1688,"properties":1697},"1eb4e8ec-1b33-4d3c-aba1-80fdaf480a4f",[1068],[1689],{"id":1690,"sortIndex":32,"affiliation":1691,"properties":28},"53b93a24-59b9-4a02-ad71-3f5e437beaca",{"id":1690,"createTime":28,"updateTime":28,"relativeEntities":1692,"slug":28,"properties":1693,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1696,"statistic":28},[],{"title":1694},{"VI":1695},"Department of Radiology, Alexandria Hospital, Alexandria, USA",[],{"title":1698},{"VI":1699},"Barry T. Katzen",{"url":1683,"publisher":1701,"properties":1747},{"id":868,"createTime":869,"updateTime":870,"relativeEntities":1702,"slug":872,"properties":1703,"entityType":25,"verifyStatus":880,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":32,"subjectFields":1707,"manageAffiliations":1716,"indexDatabases":1727,"url":28,"thumbnailPath":28,"statistic":1742,"gsStatistic":28,"type":28,"analyzePriority":28},[],{"issn":1704,"title":1705,"eissn":1706},{"VOID":875},{"EN":877},{"VOID":879},[1708,1712],{"id":883,"createTime":28,"updateTime":28,"relativeEntities":1709,"label":1710,"description":1711,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":886},{},{"id":889,"createTime":28,"updateTime":28,"relativeEntities":1713,"label":1714,"description":1715,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":892},{},[1717,1722],{"id":896,"createTime":28,"updateTime":28,"relativeEntities":1718,"slug":28,"properties":1719,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1721,"statistic":28},[],{"title":1720},{"EN":900},[],{"id":903,"createTime":28,"updateTime":28,"relativeEntities":1723,"slug":28,"properties":1724,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1726,"statistic":28},[],{"title":1725},{"EN":907},[909],[1728,1735],{"id":929,"indexDatabase":1729,"url":935,"indexYears":936,"academicFieldIds":1734,"indexDatabaseRanking":940},{"id":775,"createTime":28,"updateTime":28,"relativeEntities":1730,"label":1731,"description":1732,"key":781,"publicationTags":1733,"standard":28},[],{"EN":778,"VI":778},{"EN":778,"VI":780},[783],[938,939],{"id":912,"indexDatabase":1736,"url":924,"indexYears":28,"academicFieldIds":1741,"indexDatabaseRanking":28},{"id":914,"createTime":28,"updateTime":28,"relativeEntities":1737,"label":1738,"description":1739,"key":921,"publicationTags":1740,"standard":28},[],{"EN":917,"VI":917},{"EN":919,"VI":920},[923,813],[926,927],{"impactFactor":32,"impactFactorByYear":1743,"i10Index":944,"i10IndexLast5Year":281,"totalPublication":945,"totalPublicationByYear":1744,"totalCitation":965,"totalCitationByYear":1745,"totalCitationPerPublication":1005,"totalCitationPerPublicationByYear":1746,"hindexLast5Year":208,"hindex":208},{"2012":225,"2013":943,"2014":840,"2015":319,"2016":422,"2017":440,"2018":840,"2019":696,"2020":221,"2021":840,"2022":170,"2023":284},{"1978":202,"1979":142,"1980":516,"1981":206,"1982":516,"1983":200,"1984":160,"1985":152,"1986":358,"1987":428,"1988":564,"1989":947,"1990":161,"1991":599,"1992":157,"1993":428,"1994":333,"1995":155,"1996":530,"1997":567,"1998":161,"1999":209,"2000":334,"2001":567,"2002":948,"2003":154,"2004":949,"2005":684,"2006":950,"2007":951,"2008":952,"2009":953,"2010":954,"2011":836,"2012":955,"2013":956,"2014":957,"2015":570,"2016":958,"2017":959,"2018":614,"2019":960,"2020":961,"2021":962,"2022":963,"2023":964,"2024":600},{"1978":122,"1979":967,"1980":325,"1981":968,"1982":969,"1983":970,"1984":971,"1985":972,"1986":522,"1987":156,"1988":831,"1989":973,"1990":210,"1991":974,"1992":605,"1993":975,"1994":976,"1995":977,"1996":978,"1997":979,"1998":980,"1999":981,"2000":982,"2001":983,"2002":984,"2003":985,"2004":986,"2005":987,"2006":988,"2007":989,"2008":990,"2009":991,"2010":992,"2011":993,"2012":994,"2013":995,"2014":996,"2015":997,"2016":998,"2017":999,"2018":1000,"2019":1001,"2020":1002,"2021":1003,"2022":1004,"2023":137,"2024":145},{"1978":286,"1979":1007,"1980":1008,"1981":694,"1982":1005,"1983":1009,"1984":1010,"1985":1011,"1986":1012,"1987":1013,"1988":1014,"1989":1015,"1990":1016,"1991":1017,"1992":1018,"1993":1019,"1994":1020,"1995":1021,"1996":1005,"1997":1022,"1998":1023,"1999":1023,"2000":1024,"2001":1025,"2002":1026,"2003":1027,"2004":593,"2005":1028,"2006":1029,"2007":1030,"2008":1031,"2009":1032,"2010":1033,"2011":1034,"2012":1035,"2013":1036,"2014":1037,"2015":593,"2016":1038,"2017":1039,"2018":1040,"2019":177,"2020":1041,"2021":741,"2022":338,"2023":365,"2024":104},{"pages":1748,"volume":1750},{"VOID":1749},"S39-S47",{"VOID":1751},"11","1988-01-01",1988,[940,923],{"id":1756,"createTime":1757,"updateTime":1758,"relativeEntities":1759,"slug":1760,"properties":1761,"entityType":1061,"verifyStatus":26,"verifyTime":1758,"verifyNote":1062,"languages":28,"translateLanguages":28,"viewCount":32,"primaryUrl":1770,"fullTextUrl":28,"authors":1771,"publicationType":1165,"publisherRelationship":1828,"citationCount":28,"citationInfo":28,"publishDate":1880,"publishYear":1881,"citationAnalyzeStatus":880,"lastCitationAnalyze":28,"indexDatabases":1882,"openAccess":28,"references":28,"isForceReanalyzing":1221},"004f5d1b-f706-4711-a91e-8405f8712540","2023-12-06T22:22:03.587+00:00","2025-02-06T19:34:26.147+00:00",[],"Direct-Oral-Anticoagulants-An-Overview-for-the-Interventional-Radiologist",{"abstract":1762,"title":1764,"references":1766,"doi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direct oral anticoagulants (DOACs) have emerged as a good alternative for the treatment of thromboembolic diseases, and their use in clinical practice is increasing rapidly. The DOACs act by blocking the activity of one single step in the coagulation cascade. These drugs act downstream in the common pathway of the coagulation cascade by directly antagonising the action of thrombin or factor Xa. The development of DOACs represents a paradigm shift from the oral vitamin K antagonists such as warfarin. This article aims to describe the properties of the currently available DOACs including pharmacology and dosing. We also address the strategies for periprocedural management and reversal of anticoagulation of patients treated with these agents.",{"EN":1765},"Direct Oral Anticoagulants: An Overview for the Interventional Radiologist",{"VOID":1767},"Mackman N. Triggers, targets and treatments for thrombosis. Nature. 2008;451:914–8.\nRosamond W, Flegal K, Friday G, Furie K, Go A, Greenlund K, et al. Heart disease and stroke statistics—2007 update: a report from the American Heart Association Statistics Committee and Stroke Statistics Subcommittee. Circulation. 2007;115:e69–171.\nDouketis JD, Berger PB, Dunn AS, Jaffer AK, Spyropoulos AC, Becker RC, et al. The perioperative management of antithrombotic therapy *. CHEST J. 2008;133:299S.\nPatel IJ, Davidson JC, Nikolic B, Salazar GM, Schwartzberg MS, Walker TG, et al. Consensus guidelines for periprocedural management of coagulation status and hemostasis risk in percutaneous image-guided interventions. J Vasc Interv Radiol. 2012;23:727–36.\nBarnes GD, Ageno W, Ansell J, Kaatz S, Husted S, Lip GYH, et al. Recommendation on the nomenclature for oral anticoagulants: communication from the SSC of the ISTH: comment. J Thromb Haemost. 2015;13:2130–2.\nWeitz JI. Anticoagulation therapy in 2015: where we are and where we are going. J Thromb Thrombolysis. 2015;39:264–72.\nCamm AJ, Lip GYH, De Caterina R, Savelieva I, Atar D, Hohnloser SH, et al. 2012 focused update of the ESC Guidelines for the management of atrial fibrillation: an update of the 2010 ESC Guidelines for the management of atrial fibrillation. Developed with the special contribution of the European Heart Rhythm Association. Eur Heart J. 2012;33:2719–47.\nChan NC, Eikelboom JW, Weitz JI. Evolving treatments for arterial and venous thrombosis. Circ Res. 2016;118:1409–24.\nWeinz C, Schwarz T, Kubitza D, Mueck W, Lang D. Metabolism and excretion of rivaroxaban, an oral, direct factor Xa inhibitor, in rats, dogs, and humans. Drug Metab Dispos. 2009;37:1056–64.\nFrost C, Wang J, Nepal S, Schuster A, Barrett YC, Mosqueda-Garcia R, et al. Apixaban, an oral, direct factor Xa inhibitor: single dose safety, pharmacokinetics, pharmacodynamics and food effect in healthy subjects. Br J Clin Pharmacol. 2013;75:476–87.\nOgata K, Mendell-Harary J, Tachibana M, Masumoto H, Oguma T, Kojima M, et al. Clinical safety, tolerability, pharmacokinetics, and pharmacodynamics of the novel factor Xa inhibitor edoxaban in healthy volunteers. J Clin Pharmacol. 2010;50:743–53.\nStangier J. Clinical pharmacokinetics and pharmacodynamics of the oral direct thrombin inhibitor dabigatran etexilate. Clin Pharmacokinet. 2008;47:285–95.\nChai-Adisaksopha C, Crowther M, Isayama T, Lim W, Kearon C, Gent M, et al. The impact of bleeding complications in patients receiving target-specific oral anticoagulants: a systematic review and meta-analysis. Blood. 2014;124:2450–8.\nHart RG, Diener H-C, Yang S, Connolly SJ, Wallentin L, Reilly PA, et al. Intracranial hemorrhage in atrial fibrillation patients during anticoagulation with warfarin or dabigatran: the RE-LY trial. Stroke. 2012;43:1511–7.\nHylek EM, Held C, Alexander JH, Lopes RD, De Caterina R, Wojdyla DM, et al. Major bleeding in patients with atrial fibrillation receiving apixaban or warfarin. J Am Coll Cardiol. 2014;63:2141–7.\nStangier J, Rathgen K, Stähle H, Gansser D, Roth W. The pharmacokinetics, pharmacodynamics and tolerability of dabigatran etexilate, a new oral direct thrombin inhibitor, in healthy male subjects. Br J Clin Pharmacol. 2007;64:292–303.\nBlech S, Ebner T, Ludwig-Schwellinger E, Stangier J, Roth W, American Society for Pharmacology and Experimental Therapeutics. The metabolism and disposition of the oral direct thrombin inhibitor, dabigatran, in humans. Drug Metab Dispos. 2008;36:386–99.\nHariharan S, Madabushi R. Clinical pharmacology basis of deriving dosing recommendations for dabigatran in patients with severe renal impairment. J Clin Pharmacol. 2012;52:119S–25S.\nWittkowsky AK. Novel oral anticoagulants and their role in clinical practice. Pharmacotherapy. 2011;31:1175–91.\nStangier J, Rathgen K, Stähle H, Mazur D. Influence of renal impairment on the pharmacokinetics and pharmacodynamics of oral dabigatran etexilate: an open-label, parallel-group, single-centre study. Clin Pharmacokinet. 2010;49:259–68.\nStampfuss J, Kubitza D, Becka M, Mueck W. The effect of food on the absorption and pharmacokinetics of rivaroxaban. Int J Clin Pharmacol Ther. 2013;51:549–61.\nMueck W, Schwers S, Stampfuss J. Rivaroxaban and other novel oral anticoagulants: pharmacokinetics in healthy subjects, specific patient populations and relevance of coagulation monitoring. Thromb J. 2013;11:1.\nKubitza D, Becka M, Wensing G, Voith B, Zuehlsdorf M. Safety, pharmacodynamics, and pharmacokinetics of BAY 59-7939–an oral, direct factor Xa inhibitor–after multiple dosing in healthy male subjects. Eur J Clin Pharmacol. 2005;61:873–80.\nFox KAA, Piccini JP, Wojdyla D, Becker RC, Halperin JL, Nessel CC, et al. Prevention of stroke and systemic embolism with rivaroxaban compared with warfarin in patients with non-valvular atrial fibrillation and moderate renal impairment. Eur Heart J. 2011;32:2387–94.\nMartinelli I, Lensing AWA, Middeldorp S, Levi M, Beyer-Westendorf J, van Bellen B, et al. Recurrent venous thromboembolism and abnormal uterine bleeding with anticoagulant and hormone therapy use. Blood. 2016;127:1417–25.\nWang L, Zhang D, Raghavan N, Yao M, Ma L, Frost CE, et al. In vitro assessment of metabolic drug-drug interaction potential of apixaban through cytochrome P450 phenotyping, inhibition, and induction studies. Drug Metab Dispos. 2010;38:448–58.\nAcharya T, Deedwania P. An evidence-based review of edoxaban and its role in stroke prevention in patients with nonvalvular atrial fibrillation. Core Evid. 2015;10:63–73.\nMatsushima N, Lee F, Sato T, Weiss D, Mendell J. Bioavailability and safety of the factor Xa inhibitor edoxaban and the effects of quinidine in healthy subjects. Clin Pharmacol Drug Dev. 2013;2:358–66.\nHealey JS, Eikelboom J, Douketis J, Wallentin L, Oldgren J, Yang S, et al. Periprocedural bleeding and thromboembolic events with dabigatran compared with warfarin: results from the randomized evaluation of long-term anticoagulation therapy (RE-LY) randomized trial. Circulation. 2012;126:343–8.\nSherwood MW, Douketis JD, Patel MR, Piccini JP, Hellkamp AS, Lokhnygina Y, et al. Outcomes of temporary interruption of rivaroxaban compared with warfarin in patients with nonvalvular atrial fibrillation: results from ROCKET AF. Circulation. 2014;129:1850–9. doi:10.1161\u002FCIRCULATIONAHA.113.005754.\nHeidbuchel H, Verhamme P, Alings M, Antz M, Hacke W, Oldgren J. EHRA practical guide on the use of new oral anticoagulants in patients with non-valvular atrial fibrillation: executive summary. Eur Heart J. 2013;34(27):2094–106.\nBaron TH, Kamath PS, McBane RD. Management of antithrombotic therapy in patients undergoing invasive procedures. N Engl J Med. 2013;368:2113–24.\nMakris M, Van Veen JJ, Tait CR, Mumford AD, Laffan M. Guideline on the management of bleeding in patients on antithrombotic agents. Br J Haematol. 2013;160:35–46.\nSchulman S. Advantages and limitations of the new anticoagulants. J Intern Med. 2014;275:1–11.\nHawes EM, Deal AM, Funk-Adcock D, Gosselin R, Jeanneret C, Cook AM, et al. Performance of coagulation tests in patients on therapeutic doses of dabigatran: a cross-sectional pharmacodynamic study based on peak and trough plasma levels. J Thromb Haemost. 2013;11:1493–502.\nBecker RC, Yang H, Barrett Y, Mohan P, Wang J, Wallentin L, et al. Chromogenic laboratory assays to measure the factor Xa-inhibiting properties of apixaban—an oral, direct and selective factor Xa inhibitor. J Thromb Thrombolysis. 2011;32:183–7.\nBeyer-Westendorf J, Gelbricht V, Förster K, Ebertz F, Köhler C, Werth S, et al. Peri-interventional management of novel oral anticoagulants in daily care: results from the prospective Dresden NOAC registry. Eur Heart J. 2014;35:1888–96.\nZhou W, Schwarting S, Illanes S, Liesz A, Middelhoff M, Zorn M, et al. Hemostatic therapy in experimental intracerebral hemorrhage associated with the direct thrombin inhibitor dabigatran. Stroke. 2011;42:3594–9.\nPragst I, Zeitler SH, Doerr B, Kaspereit FJ, Herzog E, Dickneite G, et al. Reversal of dabigatran anticoagulation by prothrombin complex concentrate (Beriplex P\u002FN) in a rabbit model. J Thromb Haemost. 2016;10:1841–8.\nZahir H, Brown KS, Vandell AG, Desai M, Maa J-F, Dishy V, et al. Edoxaban effects on bleeding following punch biopsy and reversal by a 4-factor prothrombin complex concentrate. Circulation. 2015;131:82–90.\nPerlstein I, Wang Z, Song Y, Wang J, Bedford B, Chang M, American Society of Hematology, et al. Reversal of apixaban anticoagulation by 4-factor prothrombin complex concentrates in healthy subjects. Blood. 2014;124:345.\nEscolar G, Fernandez-Gallego V, Arellano-Rodrigo E, Roquer J, Reverter JC, Sanz VV, et al. Reversal of apixaban induced alterations in hemostasis by different coagulation factor concentrates: significance of studies in vitro with circulating human blood. PLoS ONE. 2013;8:e78696.\nLevi M, Levy JH, Andersen HF, Truloff D. Safety of recombinant activated factor VII in randomized clinical trials. N Engl J Med. 2010;363:1791–800.\nSchulman S, Crowther MA, Administration USF and D, Connolly S, Ezekowitz M, Yusuf S, et al. How I treat with anticoagulants in 2012: new and old anticoagulants, and when and how to switch. Blood. 2012;119:3016–23.\nDouketis JD, Spyropoulos AC, Spencer FA, Mayr M, Jaffer AK, Eckman MH, et al. Perioperative management of antithrombotic therapy. Antithrombotic therapy and prevention of thrombosis, 9th ed: American College of Chest Physicians evidence-based clinical practice guidelines. Chest. 2012;141:e326S–50S.\nFaraoni D, Levy JH, Albaladejo P, Samama C-M, Groupe d’Intérêt en Hémostase Périopératoire. Updates in the perioperative and emergency management of non-vitamin K antagonist oral anticoagulants. Crit Care. 2015;19:203.\nGould MK, Garcia DA, Wren SM, Karanicolas PJ, Arcelus JI, Heit JA, et al. Prevention of VTE in nonorthopedic surgical patients. Chest. 2012;141:e227S–77S.\nSpyropoulos AC, Douketis JD. How I treat anticoagulated patients undergoing an elective procedure or surgery. Blood. 2012;120:2954–62.\nMajeed A, Hwang HG, Connolly SJ, Eikelboom JW, Ezekowitz MD, Wallentin L, et al. Management and outcomes of major bleeding during treatment with dabigatran or warfarin. Circulation. 2013;128(21):2325–32.\nSiegal DM, Curnutte JT, Connolly SJ, Lu G, Conley PB, Wiens BL, et al. Andexanet alfa for the reversal of factor Xa inhibitor activity. N Engl J Med. 2015;373:2413–24.\nAnsell JE, Bakhru SH, Laulicht BE, Steiner SS, Grosso M, Brown K, et al. Use of PER977 to reverse the anticoagulant effect of edoxaban. N Engl J Med. 2014;371:2141–2.\nMartin K, Beyer-Westendorf J, Davidson BL, Huisman MV, Sandset PM, Moll S. Use of the direct oral anticoagulants in obese patients: guidance from the SSC of the ISTH. J Thromb Haemost. 2016;14:1308–13.\nVanassche T, Hirsh J, Eikelboom JW, Ginsberg JS. Organ-specific bleeding patterns of anticoagulant therapy: lessons from clinical trials. Thromb Haemost. 2014;112:918–23.\nRaghavan N, Frost CE, Yu Z, He K, Zhang H, Humphreys WG, et al. Apixaban metabolism and pharmacokinetics after oral administration to humans. Drug Metab Dispos. 2009;37:74–81.\nBathala MS, Masumoto H, Oguma T, He L, Lowrie C, Mendell J. Pharmacokinetics, biotransformation, and mass balance of edoxaban, a selective, direct factor Xa inhibitor, in humans. Drug Metab Dispos. 2012;40:2250–5.\nKamath SD, McMahon BJ. Update on anticoagulation: what the interventional radiologist needs to know. 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a series of 18 patients with atrial myxoma (16 left, 2 right) the tumors were consistently identified by right ventriculography and generally visualized by left ventriculography in patients with left atrial myxoma examined owing to concomitant mitral insufficiency caused by tumor invasion at the site of mitral valve closure. Echocardiography was suited to early diagnosis of suggested tumor. Pseudoaneurysms in the cerebral and coronary arteries, apparently caused by myxomatous emboli with weakening of vessel lumen, were noted in several patients; therefore, selective coronary and cerebral arteriography should be performed preoperatively in patients with symptoms of thromboembolism. We stress that echocardiography should be performed routinely after surgery to disclose recurrences, while arteriographic control studies should be added in cases with vascular anomalies seen peeoperatively.",{"EN":1893},"Angiographic and echocardiographic observations in surgical patients with atrial myxoma",{"VOID":1895},"Prichard RW (1951) Tumors of the heart. Review of the subject and report of 150 cases. Arch Pathol 51:98–128\nGoldberg HP, Glenn F, Dotter CT, Steinberg I (1952) Myxoma of the left atrium: Diagnosis made during life with operative and postmortem findings. Circulation 6:762–767\nCrafoord C (1955) Panel discussion on late results of mitral commissurotomy. In: Lamb CR (ed) Henry Ford Hospital, Detroit, International Symposium on Cardiovascular Surgery: Studies in Physiology, Diagnosis and Techniques. p. 202. Saunders, Philadelphia\nAbrams HL, Adams DF, Grant HA (1971) The radiology of tumors of the heart Radiol Clin N Am 9:299–326\nPopp RL, Harrison DC (1969) Ultrasound for the diagnosis of atrial tumor. Ann Intern Med 71:785–787\nDonahoo JS, Weiss JL, Gardner J, Fortuin NJ, Brawley RK (1979) Current management of atrial myxoma with emphasis on a new diagnostic technique. Ann Surg 189:763–768\nNasser WK, Davis RH, Dillon JC, Tavel ME, Helmen CH, Feigenbaum H, Fisch C (1972) Atrial myxoma. 2. Phonocardiographic, echocardiographic, hemodynamic, and angiographic features in nine cases. Am Heart J, 83:810–824\nBulkley BH, Hutchins GM (1979) Atrial myxomas: A 50-year review. Am Heart J 97:639–643\nStake G, Jervell J, Frøysaker T (1978) Cardiac myxoma. Clinical and roentgenological aspects in 7 cases. J Oslo City Hosp 28:139–143\nGreenwood WF (1968) Profile of atrial myxoma. Am J Cardiol 21:367–375\nMarshall WH, Steiner RM, Wexler L (1969) Tumor vascularity in left atrial myxoma demonstrated by selective coronary arteriography. Radiology 93:815–816\nStoane L, Allen JH, Collins HA (1966) Radiological observations in cerebral embolization from left heart myxomas. Radiology 87:262–266\nNew PFJ, Price DL, Carter B (1970) Cerebral angiography in cardiac myxoma. Correlation of angiographic and histopathological findings. Radiology 96:335–345\nDesousa AL, Muller J, Campbell RL, Batnitzky S, Rankin L (1978) Atrial myxoma: A review of the neurological complications, cations, metases, and recurrences. J Neurol Neurosurg Psych 41:1119–1124\nBurton C, Johnston J (1970) Multiple cerebral aneurysms and cardiac myxoma. N Engl J Med, 282:35–36\nSemb BKH, Enge IP (1975) The effect of hypoxia on coronary circulation time. Scand J Thor Cardiovasc Surg 9:34–38\nMartinez EC, Giles TD, Burch GE (1974) Echocardiographic diagnosis of left atrial myxoma. Am J Cardiol 33:281–285\nAttar S, Lee YS, Singleton R, Scherlis L, David R, McLaughlin JS (1980) Cardiac myoma. Ann Thorac Surg 29:397–405\nFarah MG (1975) Familial atrial myxoma. Ann Intern Med 83:358–360\nBalk AHM, Wagenaar SS, Bruschke AVG (1979) Bilateral cardiac myxomas and peripheral myxomas in a patient with recent myocardial infarction. Am J Cardiol 44:767–770",{"VOID":1897},"10.1007\u002FBF02552875","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002FBF02552875",[1900,1924,1944,1964],{"id":1901,"sortIndex":32,"researcher":28,"roles":1902,"affiliations":1903,"properties":1921},"86e642c6-235b-4f78-95e9-8d46ad46d2fe",[1068],[1904,1912],{"id":1905,"sortIndex":32,"affiliation":1906,"properties":28},"faec27a0-f22e-4628-a5dc-99c9084b3d56",{"id":1905,"createTime":28,"updateTime":28,"relativeEntities":1907,"slug":28,"properties":1908,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1911,"statistic":28},[],{"title":1909},{"VI":1910},"Department of Cardiovascular Surgery and Radiology, The National Hospital of Norway, Oslo, Norway",[],{"id":1913,"sortIndex":40,"affiliation":1914,"properties":1920},"5f6ea331-3ba1-4a34-859a-46ccc1e7b7bb",{"id":1913,"createTime":28,"updateTime":28,"relativeEntities":1915,"slug":28,"properties":1916,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1919,"statistic":28},[],{"title":1917},{"VI":1918},"Thoracic and Cardiovascular Surgical Clinic, The Karolinska Hospital, Stockholm, Sweden",[],{},{"title":1922},{"VI":1923},"Bjarne K. 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cứu này được thiết kế để đánh giá hiệu quả lâm sàng và độ an toàn của thuyên tắc động mạch qua ống thông (TAE) với N-Butyl Cyanoacrylate (NBCA) cho cơn chảy máu hoạt động trong mô cơ do nhiều nguyên nhân và vị trí giải phẫu khác nhau. Mười tám bệnh nhân có hematoma với giả phình mạch và\u002Fhoặc sự thoát dịch hoạt động của chất tương phản đã trải qua TAE với NBCA. Các nguyên nhân của hematoma bao gồm chấn thương, biến chứng sau phẫu thuật và rối loạn đông máu (do bệnh nền hoặc điều trị thuốc chống đông). Các vị trí thuyên tắc bao gồm thành ngực, thành bụng, sau phúc mạc và chi. TAE được thực hiện bằng cách sử dụng hỗn hợp NBCA và dầu iod từ 1:3 đến 1:5, hoặc chỉ đơn thuần (n = 15) hoặc kết hợp với microcoil (n = 3). Tỷ lệ thành công kỹ thuật và lâm sàng, các biến chứng liên quan đến quy trình và kết quả lâm sàng đã được đánh giá. Tỷ lệ thành công kỹ thuật và lâm sàng lần lượt là 100% và 83% (15\u002F18). Hai bệnh nhân đã qua đời trong thời gian nhập viện do các bệnh lý kèm theo khác. Một bệnh nhân đã qua đời do chảy máu tái phát ở vị trí khác. Không có biến chứng nghiêm trọng nào liên quan đến quy trình thuyên tắc. TAE với NBCA là phương pháp điều trị hiệu quả và an toàn cho cơn chảy máu hoạt động trong cơ.","This study was designed to evaluate the clinical efficacy and safety of transcatheter arterial embolization (TAE) with n-butyl cyanoacrylate (NBCA) for intramuscular active hemorrhage of varied etiologies and anatomic sites. Eighteen patients who demonstrated hematoma with pseudoaneurysm and\u002For active extravasation of contrast media underwent TAE with NBCA. Etiologies of hematoma included trauma, postoperative complication, and coagulopathy (due to underlying disease or anticoagulation therapy). Sites of embolization included chest wall, abdomen wall, retroperitoneum, and extremity. TAE was performed by using 1:3 to 1:5 mixtures of NBCA and iodized oil, either solely (n = 15) or in combination with microcoil (n = 3). The technical and clinical success rate, procedure-related complications, and clinical outcomes were evaluated. The technical and clinical success rates were 100% and 83% (15\u002F18), respectively. Two patients expired while admitted due to other comorbidities. One patient expired due to recurrent bleeding at another site. There were no serious complications relating to the embolization procedure. TAE with NBCA is effective and safe treatment modality for intramuscular active hemorrhage.",{"EN":2050,"VI":2051},"Transcatheter Arterial Embolization of Intramuscular Active Hemorrhage with N-Butyl Cyanoacrylate","Thuyên tắc động mạch qua ống thông với N-Butyl Cyanoacrylate trong cơn chảy máu hoạt động tại cơ",{"VI":2053},"thuyên tắc động mạch, N-Butyl Cyanoacrylate, chảy máu hoạt động trong cơ, độ an toàn, hiệu quả lâm sàng",{"VOID":2055},"Sclafani SJ, Cooper R, Shaftan GW et al (1986) Arterial trauma: diagnostic and therapeutic angiography. Radiology 161(1):165–172\nSharafuddin MJ, Andresen KJ, Sun S et al (2001) Spontaneous extraperitoneal hemorrhage with hemodynamic collapse in patients undergoing anticoagulation: management with selective arterial embolization. J Vasc Interv Radiol 12(10):1231–1234\nRimola J, Perendreu J, Falcó J et al (2007) Percutaneous arterial embolization in the management of rectus sheath hematoma. Am J Roentgenol 188(6):W497–W502\nn-BCA Trial Investigators (2002) N-butyl cyanoacrylate embolization of cerebral arteriovenous malformations: results of a prospective, randomized, multi-center trial. Am J Neuroradiol 23(5):748–755\nYamakado K, Nakatsuka A, Tanaka N et al (2000) Transcatheter arterial embolization of ruptured pseudoaneurysms with coils and n-butyl cyanoacrylate. J Vasc Interv Radiol 11(1):66–72\nKish JW, Katz MD, Marx MV et al (2004) N-butyl cyanoacrylate embolization for control of acute arterial hemorrhage. J Vasc Interv Radiol 15(7):689–695\nToyoda H, Nakano S, Kumada T et al (1996) Estimation of usefulness of N-butyl-2-cyanoacrylate-lipiodol mixture in transcatheter arterial embolization for urgent control of life-threatening massive bleeding from gastric or duodenal ulcer. J Gastroenterol Hepatol 11(3):252–258\nJae HJ, Chung JW, Jung AY et al (2007) Transcatheter arterial embolization of nonvariceal upper gastrointestinal bleeding with N-butyl cyanoacrylate. Korean J Radiol 8(1):48–56\nLee CW, Liu KL, Wang HP et al (2007) Transcatheter arterial embolization of acute upper gastrointestinal tract bleeding with N-butyl-2-cyanoacrylate. J Vasc Interv Radiol 18(2):209–216\nCromwell LD, Kerber CW (1979) Modification of cyanoacrylate for therapeutic embolization: preliminary experience. AJR Am J Roentgenol 132(5):799–801\nPollak JS, White RI Jr (2001) The use of cyanoacrylate adhesives in peripheral embolization. J Vasc Interv Radiol 12(8):907–913\nSchenker MP, Duszak R Jr, Soulen MC et al (2001) Upper gastrointestinal hemorrhage and transcatheter embolotherapy: clinical and technical factors impacting success and survival. J Vasc Interv Radiol 12(11):1263–1271\nSavader SJ, Trerotola SO, Merine DS et al (1992) Hemobilia after percutaneous transhepatic biliary drainage: treatment with transcatheter embolotherapy. J Vasc Interv Radiol 3(2):345–352\nde Baere T, Ousehal A, Kuoch V et al (1998) Endovascular management of bleeding iliac artery pseudoaneurysms complicating radiation therapy for pelvic malignancies. AJR Am J Roentgenol 170(2):349–353\nKim BS, Do HM, Razavi M (2004) N-butyl cyanoacrylate glue embolization of splenic artery aneurysms. J Vasc Interv Radiol 15(1 Pt 1):91–94\nDrooz AT, Lewis CA, Allen TE et al (2003) Quality improvement guidelines for percutaneous transcatheter embolization. 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compare procedure-related variables, safety, renal function, and oncologic outcomes in patients undergoing percutaneous cryoablation (CA) of renal tumors with MRI- or CT-guidance.\n Patient, tumour, procedure, and follow-up data were collected and analysed. MRI and CT groups were matched using a coarsened exact approach according to patient's gender and age, tumour grade, size and location. P \u003C 0.05 was considered statistically significant. Two-hundred fifty-three patients (266 tumors) were retrospectively selected. Following the coarsened exact matching 46 patients (46 tumors) in the MRI group and 42 patients (42 tumors) in the CT group were matched. There were no significant baseline differences between the two populations except for the duration of follow-up (P = 0.002) and renal function (P = 0.002). On average MRI-guided CA lasted 21 min longer than CT-guided ones (P = 0.005). Following CA, complication rates (6.5% for MRI vs 14.3% for CT; P = 0.30) and GFR decline (mean − 13.1 ± 15.8%; range − 64.5–15.0 for MRI; mean − 8.1 ± 14.8%; range − 52.5–20.4; for CT; P = 0.13) were similar in both groups. The 5-year local progression-free, cancer-specific and overall survivals in the MRI and CT groups were 94.0% (95% CI 86.3%–100.0%) and 90.8% (95% CI 81.3%–100.0%; P = 0.55), 100.0% (95% CI 100.0%–100.0%) and 100.0% (95% CI 100.0%–100.0%; P = 1), and 83.7% (95% CI 64.0%–100.0%) and 76.2% (95% CI 62.0%–93.6%; P = 0.41), respectively. Apart from increased procedural times associated with MRI-guided CA of renal tumors compared to CT-guidance, both modalities demonstrate similar safety, GFR decline and oncologic outcomes.",{"EN":2194},"MRI- Versus CT-Guided Renal Tumor Cryoablation: Is There a Difference?",{"VOID":2196},"Ljungberg B, Albiges L, Abu-Ghanem Y, et al. European Association of Urology guidelines on renal cell carcinoma: the 2022 update. Eur Urol. 2022. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.eururo.2022.03.006.\nCazzato R, Garnon J, Ramamurthy N, et al. Percutaneous image-guided cryoablation: current applications and results in the oncologic field. Med Oncol. 2016;33:1–6.\nBorgbjerg J, Bylling T, Andersen G, Thygesen J, Mikkelsen A, Nielsen TK. CT-guided cryoablation of renal cancer: radiation burden and the associated risk of secondary cancer from procedural- and follow-up imaging. Abdom Radiol. 2020;45(11):3581–8. https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs00261-020-02527-1.\nZhong J, Gallagher M, et al. Radiation dose reduction in CT-guided cryoablation of renal tumors. Diagn Interv Radiol. 2021;27(2):244–8. https:\u002F\u002Fdoi.org\u002F10.5152\u002Fdir.2021.19548.\nCazzato RL, De Marini P, Leonard-Lorant I, et al. Safety and oncologic outcomes of magnetic resonance imaging-guided cryoablation of renal cell carcinoma: a 10-year single-center experience. Invest Radiol. 2021;56(3):153–62. https:\u002F\u002Fdoi.org\u002F10.1097\u002FRLI.0000000000000719.\nDe Marini P, Cazzato RL, Garnon J, et al. Safety and oncologic efficacy of percutaneous MRI-guided cryoablation of intraparenchymal renal cancers. Diagn Interv Imaging. 2021;102(9):531–8. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.diii.2021.04.002.\nSilverman SG, Tuncali K, vanSonnenberg E, et al. Renal tumors: MR imaging–guided percutaneous cryotherapy—initial experience in 23 patients. Radiology. 2005;236(2):716–24. https:\u002F\u002Fdoi.org\u002F10.1148\u002Fradiol.2362041107.\nCazzato R, Garnon J, Shaygi B, et al. How to perform a routine cryoablation under MRI guidance. Top Magn Reson Imaging. 2018. https:\u002F\u002Fdoi.org\u002F10.1097\u002FRMR.0000000000000158.\nBhagavatula SK, Tuncali K, Shyn PB, Levesque VM, Chang SL, Silverman SG. Percutaneous CT- and MRI-guided cryoablation of cT1 renal cell carcinoma: intermediate- to long-term outcomes in 307 patients. Radiology. 2020;296(3):687–95. https:\u002F\u002Fdoi.org\u002F10.1148\u002Fradiol.2020200149.\nHadi M, Walker C, Desborough M, et al. CIRSE standards of practice on peri-operative anticoagulation management during interventional radiology procedures. Cardiovasc Intervent Radiol. 2021;44(4):523–36. https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs00270-020-02763-4.\nKutikov A, Uzzo RG. The RENAL nephrometry score: a comprehensive standardized system for quantitating renal tumor size, location and depth. J Urol. 2009;182(3):844–53. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.juro.2009.05.035.\nClavien PA, Barkun J, de Oliveira ML, et al. The Clavien-Dindo classification of surgical complications: five-year experience. Ann Surg. 2009;250(2):187–96. https:\u002F\u002Fdoi.org\u002F10.1097\u002FSLA.0b013e3181b13ca2.\nGarnon J, Van Strijen MJ, Nielsen TK, et al. Safety of percutaneous renal cryoablation: an international multicentre experience from the EuRECA retrospective percutaneous database. Eur Radiol. 2019;29(11):6293–9. https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs00330-019-06191-y.\nKakarala B, Frangakis CE, Rodriguez R, Georgiades CS. Hemorrhagic complications of percutaneous cryoablation for renal tumors: results from a 7-year prospective study. Cardiovasc Intervent Radiol. 2016;39(11):1604–10. https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs00270-016-1419-x.\nBlute ML Jr, Okhunov Z, Moreira DM, et al. Image-guided percutaneous renal cryoablation: preoperative risk factors for recurrence and complications: RISK FACTORS FOR RECURRENCE AFTER PERCUTANEOUS CRYOABLATION. BJU Int. 2013;111(4b):E181–5. https:\u002F\u002Fdoi.org\u002F10.1111\u002Fj.1464-410X.2012.11538.x.\nBreen DJ, King AJ, Patel N, Lockyer R, Hayes M. Image-guided cryoablation for sporadic renal cell carcinoma: three- and 5-year outcomes in 220 patients with biopsy-proven renal cell carcinoma. Radiology. 2018;289(2):554–61. https:\u002F\u002Fdoi.org\u002F10.1148\u002Fradiol.2018180249.\nOkhunov Z, Moreira DM, del Junco M, et al. predictors of complications after percutaneous image-guided renal cryoablation for T1a renal cortical neoplasms. J Endourol. 2017;31(1):7–13. https:\u002F\u002Fdoi.org\u002F10.1089\u002Fend.2016.0684.\nKim EH, Tanagho YS, Bhayani SB, Saad NE, Benway BM, Figenshau RS. Percutaneous cryoablation of renal masses: Washington University experience of treating 129 tumours: Percutaneous cryoablation of renal masses. BJU Int. 2013;111(6):872–9. https:\u002F\u002Fdoi.org\u002F10.1111\u002Fj.1464-410X.2012.11432.x.\nKim EH, Tanagho YS, Saad NE, Bhayani SB, Figenshau RS. Comparison of laparoscopic and percutaneous cryoablation for treatment of renal masses. Urology. 2014;83(5):1081–7. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.urology.2013.10.081.\nPickersgill NA, Vetter JM, Kim EH, et al. Ten-year experience with percutaneous cryoablation of renal tumors: tumor size predicts disease progression. 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(TACE) improves survival in cirrhotic patients with hepatocellular carcinoma (HCC). The optimal schedule, or whether embolization (TAE) alone gives the same survival advantage, is not known. To evaluate whether specific patient characteristics and\u002For radiological transarterial techniques result in better outcomes. A PubMed search was carried out for cohort and randomized trials (n = 175) testing transarterial therapies; meta-analysis was performed where appropriate. Anticancer drugs were used as sole agent in 75% of cases (double 15% and triple 6%): doxorubicin (36%), cisplatin (31%), epirubicin (12%), mitoxantrone (8%), mitomycin (8%), and SMANCS (5%). Embolizing agents used were: gelatin sponge particles (71%), polyvinyl alcohol (PVA) particles (8%), degradable starch microspheres (DSM) (4%), and embospheres (4%). Sessions per patient were 2.5 ± 1.5 (interval: 2 months). Objective response was 40 ± 20%; survival rates at 1, 2, 3, and 5 years were: 62 ± 20%, 42 ± 17%, 30 ± 15%, and 19 ± 16%, respectively, and survival time was 18 ± 9.5 months. The post-TACE complications were: acute liver failure, 7.5% (range 0–49%); acute renal failure, 1.8% (0–13%); encephalopathy, 1.8% (0–16%); ascites, 8.3% (0–52%); upper gastrointestinal bleeding; 3% (0–22%); and hepatic or splenic abscess, 1.3% (0–2.5%). Treatment-related mortality was 2.4% (0–9.5%), mainly due to acute liver failure. Our meta-analysis of nine randomized controlled trials (RCTs) confirmed that TACE improves survival; but a meta-analysis of TACE versus TAE alone (3 RCTs, 412 patients) demonstrated no survival difference. No chemotherapeutic agent appears better than any other. There is no evidence for benefit with lipiodol. Gelatin sponge is the most used embolic agent, but PVA particles may be better. TAE appears as effective as TACE. New strategies to reduce the risk of post-TACE complications are required.",{"EN":2446},"Transarterial Therapy for Hepatocellular Carcinoma: Which Technique Is More Effective? A Systematic Review of Cohort and Randomized Studies",{"VOID":2448},"Llovet JM, Burroughs A, Bruix J (2003) Hepatocellular carcinoma. Lancet 362:1907–1917\nCamma C, Schepis F, Orlando A, Albanese M, Shahied L, Trevisani F, Andreone P, Craxi A, Cottone M (2002) Transarterial chemoembolization for unresectable hepatocellular carcinoma: Meta-analysis of randomized controlled trials. Radiology 224:47–54\nLlovet JM, Bruix J (2003) Systematic review of randomized trials for unresectable hepatocellular carcinoma: Chemoembolization improves survival. Hepatology 37:429–442\nLlovet JM, Real MI, Montana X, Planas R, Coll S, Aponte J, Ayuso C, Sala M, Muchart J, Sola R, Rodes J, Bruix J (2002) Arterial embolisation or chemoembolisation versus symptomatic treatment in patients with unresectable hepatocellular carcinoma: a randomised controlled trial. 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