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Journal of Medicine and Pharmacy","Tạp chí Y Dược học Cần Thơ",{"EN":487,"VI":488},"\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">04\u002F10\u002F2015 Ministry of Information and Communications allowed Can Tho journal of medicine and pharmacy to operate (102 \u002FGP-BTTTT)\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">07\u002F16\u002F2015 Can Tho journal of medicine and pharmacy is internationally recognized: ISSN 2354-1210\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">In 2016, The journal has been included in the list of medical science journals by The State Council for professorship which is awarded a work score of 0-0.5 points for a published article.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Can Tho Journal of Medicine and Pharmacy welcome original works that haven’t been submitted or published in other medical journals. Posts must contain content related to one of the journal’s categories.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The content published\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The journal is divided into 3 categories:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Scientific research article: are valuable scientific works, which have been researched and accepted.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Overview of medicine, biology and pharmacy: serving the objective of continuing training in the fields of medicine, biology and pharmacy; to systematize classical and modern knowledge.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Update information on new knowledge about medicine, biology, pharmacy in the country and in the world.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Scope\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Publication and introduction of scientific research in the fields:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Medicine (internal medicine, surgery, pediatrics, obstetrics and gynecology, odonto-stomatology, laboratory, oncology, traditional medicine, nursing).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Biology (genetics, biotechnology).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Pharmacology (pharmaceutics, drug quality analysis-control, synthetic pharmaceutical chemistry, biochemistry, pharmacognosy, botany, clinical pharmacy).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- To enhance the quality of undergraduate, postgraduate education, scientifically researching and meet the necessary treatment in hospital.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Introducing the updated domestic and oversea information about science technology to promote scientific research and exchanging technology in local, other universities.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Exchanging pharmaceutical and medical information for social health developing in the Mekong Delta and Vietnam.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The object\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Postgraduate students, student of Can Tho University of Medicine and Pharmacy, scientists from schools, research institutes, hospitals, health centers, pharmaceutical companies of the Mekong Delta; other provinces and regions in Vietnam and other country.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Address\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Headquarters of Can Tho Journal of Medicine and Pharmacy, located Scientific Research and International Cooperation Office: 179 Nguyen Van Cu Street, An Khanh Ward, Ninh Kieu District, Can Tho City, Vietnam.\u003C\u002Fspan>\u003C\u002Fp>","\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Ngày 16\u002F7\u002F2015, Tạp chí Y Dược học Cần Thơ được cấp chỉ số quốc tế: ISSN 2354-1210.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 4\u002F2016, Tạp chí đã được Hội đồng Giáo sư ngành Y đưa vào danh sách các tạp chí khoa học Y học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Năm 2020 Tạp chí Y Dược học Cần Thơ đã được phê duyệt vào danh mục của các Hội đồng Giáo sư ngành Dược học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ ra 12 số\u002Fnăm, 180-200 trang\u002Fsố.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 12\u002F2022 Tạp chí Y Dược học Cần Thơ là thành viên của hệ thống Crossref và từ tháng 01\u002F2023 tạp chí thực hiện bình duyệt online kín 2 chiều nhằm tăng tính minh bạch, tin cậy của các công trình nghiên cứu khoa học và đảm bảo tốt nhất chất lượng khoa học của bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ, mục đích và phạm vi của tạp chí\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ và mục đích hoạt động của tạp chí: xuất bản nhằm mục đích phổ biến kết quả từ các đề tài nghiên cứu khoa học; giao lưu trao đổi khoa học, chia sẻ kinh nghiệm, học tập, đồng thời cập nhật thông tin khoa học mới trong các lĩnh vực y, sinh, dược học trong và ngoài nước.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phạm vi của tạp chí: Tạp chí xuất bản được chia thành 3 chuyên mục: (i) Bài báo nghiên cứu khoa học là kết quả công trình nghiên cứu khoa học có giá trị đã được triển khai nghiên cứu, (ii) Bài tổng quan y, sinh, dược học: phục vụ mục tiêu đào tạo liên tục trong lĩnh vực y, sinh, dược học; nhằm hệ thống hóa những kiến thức kinh điển và hiện đại; (iii) Thông tin cập nhật kiến thức mới về y, sinh, dược học trong nước và trên thế giới.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Chính sách truy cập mở\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ áp dụng chính sách truy cập mở đối với các bài báo đã xuất bản đến với độc giả, nhằm mở rộng cơ hội tiếp cận các kết quả nghiên cứu chất lượng cao và tăng cường trao đổi kiến thức. Tạp chí đăng tải trực tuyến (miễn phí) toàn văn các bài báo được công bố trên website của Tạp chí (https:\u002F\u002Ftapchi.ctump.edu.vn).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đạo đức xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ cam kết tuân thủ đạo đức xuất bản phù hợp với các hướng dẫn và tiêu chuẩn của the Committee on Publication Ethics (COPE), tuân thủ các nguyên tắc của COPE’s Core Practices, Best Practices Guidelines for Journal Editors và Guidelines on Good Publication Practices.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Bản thảo bài báo chỉ được chấp nhận khi được tác giả chịu trách nhiệm chính cam kết các nội dung sau: Các nội dung của bản thảo chưa được đăng tải toàn bộ hoặc một phần ở các tạp chí khác; Tất cả các tác giả đều có đóng góp một cách đáng kể vào quá trình nghiên cứu hoặc chuẩn bị bản thảo và cùng chịu trách nhiệm về các nội dung của bản thảo; Tuân thủ các biện pháp đảm bảo đạo đức nghiên cứu (ví dụ thỏa thuận đồng ý tham gia nghiên cứu).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Cam kết bảo mật\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí cam kết thực hiện và tuân thủ các quy định của luật và các văn bản hướng dẫn liên quan đến bảo mật thông tin cá nhân trên không gian mạng. Các thông tin mà người dùng (tác giả, độc giả, biên tập viên, người phản biện) nhập vào các biểu mẫu trên Hệ thống Quản lý xuất bản trực tuyến của tạp chí chỉ được sử dụng vào các mục đích đã được tuyên bố rõ ràng và sẽ không được cung cấp cho bất kỳ bên thứ ba nào khác, hay dùng vào bất kỳ mục đích nào khác.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phí gửi bài\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng bài: 1.000.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng nhanh: 1.500.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với tác giả là cán bộ viên chức thuộc Trường Đại học Y Dược Cần Thơ thì được hỗ trợ 50% lệ phí gửi đăng bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với sinh viên thực hiện đề tài nghiên cứu khoa học cấp trường được hỗ trợ 100% lệ phí đăng bài ( Tác giả gửi đính kèm “ Quyết định về việc giao tổ chức thực hiện đề tài nghiên cứu khoa học cấp Trường của sinh viên”).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Hình thức nộp lệ phí:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Tiền mặt:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Nộp trực tiếp tại Phòng Tài chính - Kế toán, Trường Đại học Y Dược Cần Thơ, số 179 Nguyễn Văn Cừ, P. An Khánh, Q. Ninh Kiều, thành phố Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Chuyển khoản:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tên Tài khoản: Trường ĐHYD Cần Thơ, Số TK: 0111000115668, tại ngân hàng Vietcombank chi nhánh Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Thời gian: Áp dụng từ ngày 01\u002F02\u002F2023.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">* Phí gửi bài không được hoàn trả khi bài viết bị từ chối hoặc tác giả xin rút bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Quy trình phản biện bài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ thực hiện quy trình phản biện kín hai chiều nghiêm ngặt. Danh tính của những người phản biện không được tiết lộ cho các tác giả và ngược lại. Quy trình thẩm định bài báo đăng gồm các bước sau:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tiếp nhận bản thảo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tác giả liên hệ gửi bản thảo đến Tạp chí qua hệ thống trực tuyến tại website: https:\u002F\u002Ftapchi.ctump.edu.vn. Hướng dẫn về cách đăng ký, gửi bài và chuẩn bị bản thảo được cung cấp trên website của Tạp chí.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sàng lọc sơ bộ\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sau khi Tòa soạn nhận được bài báo của tác giả, Ban Thư ký sẽ tiến hành kiểm tra sơ bộ bài báo (các yêu cầu về nội dung và hình thức). Những bài báo không đúng quy cách hoặc có nội dung không phù hợp hoặc vi phạm bản quyền sẽ bị từ chối (Ban Thư ký thông báo phản hồi đến tác giả trong vòng 1 tuần). Những bài báo đủ điều kiện, được Ban Thư ký tòa soạn chuyển đến Ban Biên tập có cùng chuyên môn với nội dung bài báo để đề xuất người phản biện. Thời gian kể từ khi Ban Biên tập nhận bài báo đến khi đề xuất người phản biện bài báo chậm nhất là 5 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Vòng phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký gửi bài và yêu cầu phản biện đến 02 phản biện độc lập.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Các phản biện gởi nhận xét cho Ban Thư ký. Thời gian từ khi gửi bài cho phản biện đến khi nhận ý kiến của phản biện tối đa là 20 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xử ký kết quả phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Nếu ý kiến đồng ý cho đăng và không cần chỉnh sửa, Ban Thư ký tiếp tục đăng bài theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Nếu ý kiến đồng ý đăng và cần chỉnh sửa, Ban Thư ký sẽ thông tin đến tác giả chỉnh sửa theo yêu cầu của người phản biện. Thời gian chỉnh sửa và gửi lại kéo dài không quá 2 tuần, từ khi tác giả bài báo nhận được thông tin (Quá trình này có thể lặp lại tối đa 2 lần\u002F1 bài báo). Khi có sự thống nhất, đồng ý của người phản biện; bài báo được tiếp tục đăng theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Những bài báo có chất lượng không đạt yêu cầu, cả 2 phản biện không đồng ý cho đăng sẽ bị Tòa soạn từ chối đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký tổng hợp các bản thảo đã được tác giả hoàn thiện sau thẩm định trình Ban Biên tập xem xét, Tổng Biên tập phê duyệt, quyết định bài đăng theo các tiêu chí: sự phù hợp nội dung với tôn chỉ và mục đích, thể loại bài viết (ưu tiên các bài có bài có nghiên cứu chuyên sâu, hàm lượng khoa học cao), đóng góp mới bài báo, bài báo được ưu tiên đăng trong số gần nhất của Tạp chí theo thứ tự: tính thời sự, chất lượng bài báo và thời gian gửi bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Ban Biên tập và Ban Thư ký biên tập bản thảo, chế bản, đọc rà soát lỗi. Thời gian hoàn thành từ 10-15 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Ban Thư ký có trách nhiệm thông báo cho tác giả bài báo (bằng e-mail) về tình hình phê duyệt bài báo, thời gian, số kỳ, tập xuất bản bài báo theo qui định.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">4. Danh sách bài báo theo số Tạp chí được in ấn và phát hành trong năm định kỳ được công bố chính thức trên website: https:\u002F\u002Ftapchi.ctump.edu.vn\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>",{"VOID":490},"wcQ1uqwAAAAJ","2023-05-30T08:17:21.868+00:00",[],[494],{"id":495,"createTime":28,"updateTime":28,"relativeEntities":496,"slug":28,"properties":497,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":507,"parentIds":508,"statistic":28},"6413896b-eca9-442b-a73f-182a58a0ce40",[],{"title":498,"address":501,"country":504,"abbreviation":505},{"EN":499,"VI":500},"Can Tho University of Medicine and Pharmacy","Trường Đại học Y Dược Cần Thơ",{"EN":502,"VI":503},"No 179, Nguyen Van Cu street, An Khanh ward, Ninh Kieu district, Can Tho city, Vietnam","Số 179, đường Nguyễn Văn Cừ, phường An Khánh, quận Ninh Kiều, thành phố Cần Thơ, Việt Nam",{"VOID":15},{"VOID":506},"ctump","http:\u002F\u002Fwww.ctump.edu.vn\u002F",[],[],"https:\u002F\u002Ftapchi.ctump.edu.vn\u002Findex.php\u002Fctump",{"impactFactor":32,"impactFactorByYear":512,"i10Index":32,"i10IndexLast5Year":32,"totalPublication":514,"totalPublicationByYear":515,"totalCitation":520,"totalCitationByYear":521,"totalCitationPerPublication":108,"totalCitationPerPublicationByYear":523,"hindexLast5Year":45,"hindex":45},{"2022":513,"2023":111,"2024":106},0.01,1556,{"2020":47,"2021":516,"2022":517,"2023":518,"2024":519,"2025":122},57,306,801,358,161,{"2021":146,"2022":280,"2023":522},99,{"2021":524,"2022":318,"2023":104},0.23,{"impactFactor":28,"impactFactorByYear":28,"i10Index":123,"i10IndexLast5Year":123,"totalPublication":526,"totalPublicationByYear":527,"totalCitation":526,"totalCitationByYear":528,"totalCitationPerPublication":40,"totalCitationPerPublicationByYear":531,"hindexLast5Year":49,"hindex":49},476,{"0":205,"2019":123,"2021":139,"2022":459,"2023":451,"2024":357,"2025":49,"2026":48},{"2021":42,"2022":123,"2023":161,"2024":529,"2025":360,"2026":530},136,83,{"2021":105,"2022":513,"2023":532,"2024":127,"2025":533,"2026":534},0.62,25.43,13.83,{"id":536,"createTime":537,"updateTime":382,"relativeEntities":538,"slug":539,"properties":540,"entityType":25,"verifyStatus":26,"verifyTime":28,"verifyNote":28,"languages":552,"translateLanguages":28,"viewCount":133,"subjectFields":553,"manageAffiliations":554,"indexDatabases":555,"url":556,"thumbnailPath":557,"statistic":558,"gsStatistic":594,"type":55,"analyzePriority":28},"6984a56a-db70-403b-9cc4-4013e1ceaffa","2023-05-09T06:47:40.346+00:00",[],"T%E1%BA%A1p%20ch%C3%AD%20Nghi%C3%AAn%20c%E1%BB%A9u%20n%C6%B0%E1%BB%9Bc%20ngo%C3%A0i",{"country":541,"issn":542,"title":544,"introduce":547,"gsId":550},{"VOID":15},{"VOID":543},"25252445",{"EN":545,"VI":546},"VNU Journal of Foreign Studies","Tạp chí Nghiên cứu nước ngoài",{"EN":548,"VI":549},"{\"ops\":[{\"insert\":\"\\n\\nThe \\n\"},{\"attributes\":{\"italic\":true},\"insert\":\"VNU Journal of Science\"},{\"insert\":\"\\n was established in 1985 for the publication of national and international research papers in all fields of natural sciences and technology, social sciences and humanities. 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research has indicated greater muscle activation is needed for children (CH) to match relative intensity submaximal contractions in comparison with adults (AD). However, no study has compared motor unit (MU) firing and recruitment patterns between children and adults. Therefore, MU action potential amplitudes (MUAPAMP) and firing rates were examined during two repetitive submaximal contractions of the first dorsal interosseous in children and adults. Twenty-two children (age 9.0 ± 0.8 years) and 13 adults (age 22.9 ± 4.8 years) completed three maximum voluntary contractions (MVC) and two repetitive isometric contractions at 30% MVC for 40 s. Surface electromyography (EMG) was recorded and decomposed into action potential trains. MUAPAMPS, recruitment thresholds (RTs), and mean firing rates (MFRs) were calculated, and EMG amplitude was normalized (N-EMG) to MVC. For each subject and repetition, linear MFR vs. RT and exponential MUAPAMP vs. RT and MFR vs. MUAPAMP relationships were calculated. N-EMG (P = 0.001, CH = 56.5 ± 31.7%, AD = 30.3 ± 9.1%), MFRs regardless of RT, according to greater y-intercepts of the MFR vs. RT relationships [P = 0.013, CH = 31.1 ± 5.1 pulses per second (pps), AD = 25.9 ± 4.3 pps] and MFRs of MUs with smaller action potential amplitudes (P = 0.017, CH = 29.4 ± 6.8 pps, AD = 23.5 ± 3.5 pps), were greater for children. MUAPAMPS in relation with RT were similar between groups except the highest threshold MUs (RT = 28% MVC) were greater for the adults (1.02 ± 0.43 mV) than children (0.67 ± 0.24 mV) (P = 0.010). Muscle activation and MU firing rates were greater for children, which likely indicated a greater operating point of MU control in comparison with adults during an isometric contraction performed at a relative submaximal intensity.",{"EN":1099},"Motor unit action potential amplitudes and firing rates during repetitive muscle actions of the first dorsal interosseous in children and adults",{"VOID":1101},"Adam A, De Luca CJ (2005) Firing rates of motor units in human vastus lateralis muscle during fatiguing isometric contractions. J Appl Physiol (1985) 99:268–280. https:\u002F\u002Fdoi.org\u002F10.1152\u002Fjapplphysiol.01344.2004\nBenjamini Y, Hochberg Y (1995) Controlling the false discovery rate—a practical and powerful approach to multiple testing. J R Stat Soc B 57:289–300\nBurnett RA, Laidlaw DH, Enoka RM (2000) Coactivation of the antagonist muscle does not covary with steadiness in old adults. 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Appl Physiol Nutr Metab. https:\u002F\u002Fdoi.org\u002F10.1139\u002Fapnm-2017-0646\nStock MS, Beck TW, Defreitas JM (2012) Effects of fatigue on motor unit firing rate versus recruitment threshold relationships. Muscle Nerve 45:100–109. https:\u002F\u002Fdoi.org\u002F10.1002\u002Fmus.22266\nTanji J, Kato M (1973) Firing rate of individual motor units in voluntary contraction of abductor digiti minimi muscle in man. Exp Neurol 40:771–783\nTrevino MA et al (2018) Sex-related differences in muscle size explained by amplitudes of higher-threshold motor unit action potentials and muscle fiber typing. Acta Physiol. https:\u002F\u002Fdoi.org\u002F10.1111\u002Fapha.13151\nWatanabe K, Holobar A, Kouzaki M, Ogawa M, Akima H, Moritani T (2016) Age-related changes in motor unit firing pattern of vastus lateralis muscle during low-moderate contraction. 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elucidate the characteristics of vasomotor control in glabrous and nonglabrous skin during dynamic exercise, we compared the vascular responses in both areas to increasing core temperature during the cycle exercise for 30 min at different intensities in the range 20–60% of peak oxygen consumption (VO2peak) in a total of 13 male and four female subjects in two experimental protocols. Skin blood flow was monitored using laser Doppler flowmetry. In protocol 1, the slope of the relationship between esophageal temperature (T\n                        es) and cutaneous vascular conductance (CVC) in the early phase of the exercise decreased (P \u003C 0.05) with increasing exercise intensity at glabrous sites (palm) but not nonglabrous sites (dorsal hand). In protocol 2, to examine whether a difference in vascular responses in the two areas is due to the adrenergic vasoconstrictor system, the release of norepinephrine from adrenergic nerves in forearm and palmar skin was blocked locally by iontophoresis of bretylium tosylate (BT). The administration of BT diminished completely the change of CVC in the palm during the exercise but did not alter the response in the forearm compared with the untreated site. In the two areas, neither the T\n                        es threshold for vasodilation nor the change in CVC above the threshold in the middle and late phase of the exercise was influenced by the intensity of the exercise. These results suggest that, in the early phase of the exercise, light-to-moderate exercise reduces in an intensity-dependent manner the thermal sensitivity for vasodilation in glabrous skin but not nonglabrous skin via an adrenergic vasoconstrictor pathway.",{"EN":1270},"Different vascular responses in glabrous and nonglabrous skin with increasing core temperature during exercise",{"VOID":1272},"Abramson DI (ed) (1967) Vascular responses in skin: circulation in the extremities. Academic, pp 114–138\nChristensen EH, Nielsen M, Hannisdahl B (1942) Investigations of the circulation in the skin at the beginning of muscular work. Acta Physiol Scand 4:162–170\nGreenfield ADM (ed) (1963) The circulation through the skin: handbook of physiology. Circulation, Sect 2, vol 2, Chapt 39. American Physiological Society, Washington, pp 1325–1351\nHirata K, Nagasaka T, Hirai A, Hirashita M, Takahara T (1984) Cutaneous vascular tone during heat load modified by exercise intensity. J Therm Biol 9:117–120\nHirata K, Nagasaka T, Hirai A, Hirashita M, Takahara T, Nunomura T (1986) Effects of human menstrual cycle on thermoregulatory vasodilation during exercise. Eur J Appl Physiol 54:559–565\nJohnson JM (1979) Responses of forearm blood flow to graded leg exercise in man. J Appl Physiol 46:457–462\nJohnson JM (1986) Nonthermoregulatory control of human skin blood flow. J Appl Physiol 61:1613–1622\nJohnson JM (1992) Exercise and cutaneous circulation. Exerc Sport Sci Rev 20:59–97\nJohnson JM, Park MK (1981) Effect of upright exercise and on threshold for cutaneous vasodilation and sweating. J Appl Physiol 35:814–818\nJohnson JM, Park MK (1982) Effect of heat stress on cutaneous vascular responses to the initiation of exercise. J Appl Physiol 53:744–749\nJohnson JM, Proppe DW (eds) (1996) Cardiovascular adjustments to heat stress. In: Handbook of physiology: environmental physiology. Oxford University Press, New York, pp 215–243\nJohnson JM, Taylor WF, Shepherd AP, Park MK (1984) Laser-Doppler measurement of skin blood flow: comparison with plethysmography. J Appl Physiol 56:798–803\nJohnson JM, Pérgola PE, Liao FK, Kellogg DL Jr, Crandall CG (1995) Skin of the dorsal aspect of human hands and fingers possesses an active vasodilator system. J Appl Physiol 78:948–954\nKellogg DL Jr, Johnson JM, Kosiba W (1989) Selective abolition of adrenergic vasoconstrictor responses in skin by local iontophoresis of bretylium. Am J Physiol 257:H1599–H1606\nKellogg DL Jr, Johnson JM, Kosiba WA (1991) Competition between cutaneous active vasoconstriction and active vasodilation during exercise in humans. Am J Physiol 261:H1184–H1189\nKellogg DL, Pérgola PE, Piest KL, Kosiba WA, Crandall CG, Johnson JM (1995) Cutaneous active vasodilation in humans is mediated by cholinergic nerve cotransmission. Circ Res 77:1222–1228\nKondo N (1999) The control of sweating rate and skin blood flow during exercise. In: Nose H, Gisolfi CV, Imaizumi K (eds) Exercise, nutrition, and environmental stress, vol 1. Cooper, Traverse City, pp 153–178\nKondo N, Takano S, Aoki K, Shibasaki M, Tominaga H, Inoue Y (1998) Regional differences in the effect of exercise intensity on thermoregulatory sweating and cutaneous vasodilation. Acta Physiol Scand 164:71–78\nMack GW, Nose H, Takamata A, Okuno T, Morimoto T (1994) Influence of exercise intensity and plasma volume on active cutaneous vasodilation in humans. Med Sci Sports Exerc 26:209–216\nMitono H, Endoh H, Okazaki K, Ichinose T, Masuki S, Takamata A, Nose H (2005) Acute hypoosmolality attenuates the suppression of cutaneous vasodilation with increased exercise intensity. J Appl Physiol 99:902–908\nRowell LB (1983) Cardiovascular aspects of human thermoregulation. Circ Res 52:367–379\nSaad AR, Stephens DP, Bennett LAT, Charkoudian N, Kosiba WA, Johnson JM (2001) Influence of isometric exercise on blood flow and sweating in glabrous and nonglabrous human skin. J Appl Physiol 91:2487–2492\nSmolander J, Saalo J, Korhonen O (1991) Effect of work load on cutaneous vascular response to exercise. J Appl Physiol 71:1614–1619\nTakeno Y, Kamijo Y, Nose H (2001) Thermoregulatory and aerobic changes after endurance training in a hypobaric hypoxic and warm environment. J Appl Physiol 91:1520–1528\nTaylor WF, Johnson JM, O’Leary D, Park MK (1984a) Effect of high local temperature on reflex cutaneous vasodilation. J Appl Physiol 57:191–196\nTaylor WF, Johnson JM, O’Leary D, Park MK (1984b) Modification of the cutaneous vascular response to exercise by local skin temperature. J Appl Physiol 57:1878–1884\nTaylor WF, Johnson JM, Kosiba WA, Kwan CM (1988) Graded cutaneous vascular responses to dynamic leg exercise. J Appl Physiol 64:1803–1809\nWenger CB, Roberts MF, Stolwijk JAJ, Nadel ER (1975) Forearm blood flow during body temperature transients produced by leg exercise. J Appl Physiol 38:58–63\nYamazaki F (2002) Vasomotor responses in glabrous and nonglabrous skin during sinusoidal exercise. Med Sci Sports Exerc 34:767–772\nYamazaki F, Sone R (2003a) Evaluation of thermoregulatory responses during exercise and exercise prescription. In: Nose H, Mack G, Imaizumi K (eds) Exercise, nutrition, and environmental stress, vol 3. Cooper, Traverse City, pp 155–177\nYamazaki F, Sone R (2003b) Skin vascular response in the hand during sinusoidal exercise in physically trained subjects. Eur J Appl Physiol 90:159–164\nYanagimoto S, Kuwahara T, Zhang Y, Koga S, Inoue Y, Kondo N (2003) Intensity-dependent thermoregulatory responses at the onset of dynamic exercise in mildly heated humans. 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Subjects were allocated to two training groups, high (HI, n=10) and low (LO, n=10) intensity. The HI group performed eight sets of 8 revolutions at 80% of the maximum resistance to complete 2 pedal revolutions (2RM); the LO group performed eight sets of 16 pedal revolutions at 40% of 2RM. Subjects were tested twice before, as control period (−4 weeks and 0 weeks) and once after training (16 weeks) for HRV, maximum voluntary contraction (MVC) of knee extensors and peak power (P\n                        \n                        p) of lower limbs by jumping on a force platform. HRV was measured using time and frequency domain parameters. Two-way ANOVA for repeated measures was performed on all variables (P\u003C0.05). Results showed no differences between training groups. Following training HRV was not modified, while MVC and P\n                        \n                        p significantly increased. 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Am J Physiol 280:H1145–H1150\nBoutcher SH, Stein P (1995) Association between heart rate variability and training response in sedentary middle-aged men. Eur J Appl Physiol 70:75–80\nDavy KP, Miniclier NL, Taylor A, Stevenson ET, Seals DR (1996) Elevated heart rate variability in physically active postmenopausal women: a cardioprotective effect? Am J Physiol 271:–H455–H460\nDekker JM, Schouten EG, Klootwijk P, Pool J, Swenne CA, Kromhout D (1997) Heart rate variability from short electrocardiographic recording predicts mortality from all causes in middle-aged and elderly men. The Zutphen study. Am J Epidemiol 145:899–908\nDe Meersman RE (1992) Respiratory sinus arrhythmia alteration following training in endurance athletes. Eur J Appl Physiol 64:434–436\nDe Vito G, Bernardi M, Forte R, Pulejo C, Macaluso A, Figura F (1998) Determinants of maximal instantaneous muscle power in women aged 50–75 years. Eur J Appl Physiol 78:59–64\nDreifus LS, Agarwal JB, Botvinick EH, Ferdinand KC, Fisch C, Fisher JD, Kennedy JW, Kerber RE, Lambert CR, Okike ON, Prystowsky EN, Saksen SV, Schroeder JS, Williams DO (1993) Heart rate variability for risk stratification of life-threatening arrhythmias. J Am Coll Cardiol 22:948–950\nFiatarone MA, O'Neill EF, Ryan ND (1994) Exercise training and nutritional supplementation for physical frailty in very elderly people. N Engl J Med 330:1769–1775\nFrontera WR, Meredith CN, O'Reilly KP, Knuttgen HG, Evans WJ (1988) Strength conditioning in older men: skeletal muscle hypertrophy and improved function. J Appl Physiol 64:1038–1044\nGregoire J, Tuck S, Yamamoto Y, Hughson RL (1996) Heart rate variability at rest and exercise: influence of age, gender and physical training. Can J Appl Physiol 21:455–470\nGreig CA, Young A, Skelton DA, Pippet E, Butler FMM, Mahmud SM (1994) Exercise studies with elderly volunteers. Age Ageing 23:185–189\nHaennel R, Teo KK, Quinney A, Kappagoda T (1989) Effects of hydraulic circuit training on cardiovascular function. Med Sci Sports Exerc 21:605–612\nHuikuri HV, Pikkujamsa SM, Airaksinen KEJ, Ikaheimo MJ, Rantala AO, Kauma H, Lilja M, Kesaniemi A (1996) Sex-related differences in autonomic modulation of heart rate in middle aged subjects. Circulation 94:122–125\nJensen-Urstad K, Saltin B, Ericson M, Storck N, Jensen-Urstad M (1997a) Pronounced resting bradycardia in male elite runners is associated with high heart rate variability. Scand J Med Sci Sports 7:274–278\nJensen-Urstad K, Storck N, Bouvier F, Ericson M, Lindblad IE, Jensen-Urstad M (1997b) Heart rate variability is related to age and gender Acta Physiol Scand 160:235–241\nLoimaala A, Sievanen H, Laukkanen R, Parkka J, Vuori I, Huikuri H (1999) Accuracy of a novel real-time microprocessor QRS detector for heart rate variability assessment. Clin Physiol 19:84–88\nLoimaala A, Huikuri H, Oja P, Pasanen M, Vuori I (2000) Controlled 5-mo aerobic training improves heart rate but not heart rate variability or baroreflex sensitivity. J Appl Physiol 89:1825–1829\nMelanson ED (2000) Resting heart rate varibility in men varying in habitual physical activity. Med Sci Sports Exerc 32:1894–1901\nMelanson EL, Freedson PS (2001) The effect of endurance training on resting heart rate variability in sedentary adult males. Eur J Appl Physiol 85:442–449\nPagani M, Lombardi F, Guzzetti S, Rimoldi O, Furlan R, Pizzinelli P, Sandrone G, Malfatto G, Dell'Orto S, Piccaluga E, Turiel M, Baselli G, Cerutti S, Malliani A (1986) Power spectral analysis of heart rate and arterial pressure variabilities as a marker of sympatho–vagal interaction in man and conscious dog. Circ Res 59:178–193\nPerini R, Milesi S, Fisher NM, Pendergast DR, Veicsteinas A (2000) Heart rate variability during dynamic exercise in elderly males and females. Eur J Appl Physiol 82:8–15\nPerini R, Fisher NM, Veicsteinas A, Pendergast DR (2002) Aerobic training and cardiovascular responses at rest and during exercise in older men and women. Med Sci Sports Exerc 34:700–708\nRay CA, Carrasco DI (2000) Isometric handgrip training reduces arterial pressure at rest without changes in sympathetic nerve activity. Am J Heart Circ Physiol 279:H245–H249\nRay CA, Hume KM (1997) Sympathetic neural adaptations to exercise training in humans: insights from microneurography. Med Sci Sports Exerc 30:387–391\nRuha A, Sallinen S, Nissila S (1997) A real-time microprocessor QRS detector system with a 1-ms timing accuracy for the measurements of ambulatory HRV. Trans Biomed Eng 44:159–167\nSchuit AJ, Van Amelsvoort LGPM, Verheij TC, Rijneke RD, Maan AC, Swenne CA, Schouten EG (1998) Exercise training and heart rate variability in older people. Med Sci Sports Exerc 31:816–821\nSeals D, Chase PB (1989) Influence of physical training on heart rate variability and baroreflex circulatory control. J Appl Physiol 66:1886–1895\nShephard RJ (1997) Aging, physical activity and health. Human Kinetics, Champaign, Ill.\nSinoway LI, Rea RF, Mosher TJ, Smith MB, Mark AL (1992) Hydrogen ion concentration is not the sole determinant of muscle metaboreceptor responses in humans. J Clin Invest 89:1875–1884\nSipila S, Suominen H (1995) Effects of strength and endurance training on thigh and leg muscle mass and composition in elderly women. J Appl Physiol 78:334–340\nTask Force of the European Society of Cardiology the North American Society of Pacing Electrophysiology (1996) Heart rate variability. Standards of measurements, physiological interpretation and clinical use. Circulation 93:1043–1065\nUmetani K, Singer DH, McCraty R, Atkinson M (1998) Twenty-four hour time domain heart rate variability and heart rate: relations to age and gender over nine decades. J Am Coll Cardiol 31:593–601\nVan Hoof R, Macor F, Lijnen P, Staessen J, Thijs L, Vanhees L, Fagard R (1996) Effect of strength training on blood pressure measured in various training conditions in sedentary men. 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study aimed to reveal the neural and muscular adjustments following a repeated-sprint (RS) running exercise. Sixteen subjects performed a series of neuromuscular tests before, immediately after and 30 min (passive recovery) post-RS exercise (12 × 40 m sprints interspaced by 30 s of passive recovery). Sprint times significantly lengthened over repetitions (+17% from the first to the last sprint; P \u003C 0.05). After RS running exercise, maximal voluntary contraction torque of the plantar flexors (−11 ± 7.3%), muscle activation (twitch interpolation) (−2.7 ± 3.4%) and soleus maximal M-wave amplitude (−20 ± 17%) were significantly (P \u003C 0.05) reduced but returned close to baseline after 30 min. Both soleus EMG activity and maximal Hoffmann reflex normalized with respect to M-wave amplitude did not change during the whole experiment. From pre- to post-RS exercise, evoked twitch response was characterized by lower peak torque and maximal rate of torque development (−13 and −11%, respectively, P \u003C 0.05), but was not different from baseline after recovery. Peak tetanus at 20 and 80 Hz were 17 and 8% lower (P \u003C 0.05) in the fatigued state, respectively. Acute muscle fatigue induced by RS running exercise is mainly peripheral as the short-term (30 min) recovery pattern of plantar flexors contractile properties follows that of the voluntary force-generating capacity.",{"EN":1615},"Neural and muscular adjustments following repeated running sprints",{"VOID":1617},"Andersen B, Westlund B, Krarup C (2003) Failure of activation of spinal motoneurones after muscle fatigue in healthy subjects studied by transcranial magnetic stimulation. J Physiol 551(Pt1):345–356\nAvela J, Finni J, Komi PV (2006) Excitability of the soleus reflex arc during intensive stretch-shortening cycle exercise in two power-trained athlete groups. Eur J Appl Physiol 97:449–486\nBalsom PD, Seger JY, Sjödin B, Ekblom B (1992) Maximal-intensity intermittent exercise: effect of recovery duration. Int J Sports Med 13(7):528–533\nBillaut F, Basset FA (2007) Effect of different recovery patterns on repeated-sprint ability and neuromuscular responses. J Sports Sci 28(8):905–913\nBillaut F, Basset FA, Falgairette G (2005) Muscle coordination changes during intermittent cycling sprints. Neurosci Lett 380(3):265–269\nBishop D, Edge J, Goodman C (2004) Muscle buffer capacity and anaerobic fitness are associated with repeated-sprint ability in women. Eur J Appl Physiol 92:540–547\nBringard A, Denis R, Belluye N, Perrey S (2007) External elastic compression and muscle function in humans. Sci Sports 22:3–13\nClausen T, Nielsen OB, Harrison AP, Flatman JA, Overgaard K (1998) The Na+, K+ pump and muscle excitability. Acta Physiol Scand 162:183–190\nDavies CT, Young K (1983) Effect of temperature on the contractile properties and muscle power of triceps surae in humans. J Appl Physiol 55(1):191–195\nDrust B, Rasmussen P, Mohr M, Nielsen B, Nybo L (2005) Elevations in core and muscle temperature impairs repeated sprint performance. Acta Physiol Scand 183(2):181–190\nDupont G, Millet GP, Guinhouya C (2005) Berthoin relationship between oxygen uptake kinetics and performance in repeated running sprints. Eur J Appl Physiol 95(1):27–34\nEnoka RM, Stuart DG (1992) Neurobiology of muscle fatigue. J Appl Physiol 72:1631–1648\nFarina D, Merletti R, Enoka RM (2004) The extraction of neural strategies from the surface EMG. J Appl Physiol 96:1486–1495\nFitts RH, Metzger JM (1991) Mechanisms of muscular fatigue. In: Poortmans JR (ed) Principles of exercise biochemistry. Karger, Basel, pp 212–229\nGibson H, Edwards RH (1985) Muscular exercise and fatigue. Sports Med 2(2):120–132\nGill ND, Beaven CM, Cook C (2006) Effectiveness of post-match recovery strategies in rugby players. Br J Sports Med 40:260–263\nGirard O, Lattier G, Maffiuletti NA, Micallef JP, Millet GP (2008) Neuromuscular fatigue during a prolonged intermittent exercise: application to tennis. J Electromyogr Kinesiol 18(6):1038–1046\nGirard O, Racinais S, Micallef J-P, Millet GP (2009) Spinal modulations accompany peripheral fatigue during prolonged tennis playing. Scand J Med Sci Sport (in press)\nGodt RE, Nosek TM (1989) Changes of intracellular milieu with fatigue or hypoxia depress contraction of skinned rabbit skeletal and cardiac muscle. J Physiol 412:155–180\nHautier CA, Arsac LM, Deghdegh K, Souquet J, Belli A, Lacour JR (2000) Influence of fatigue on EMG\u002Fforce ratio and cocontraction in cycling. Med Sci Sports Exerc 32(4):839–843\nJones DA (1996) High- and low-frequency fatigue revisited. Acta Physiol Scand 156:265–270\nKalmar JM, Cafarelli E (1999) Effects of caffeine on neuromuscular function. J Appl Physiol 87(2):801–808\nKinugasa R, Akima H, Ota A, Ohta A, Sugiura K, Kuno SY (2004) Short-term creatine supplementation does not improve muscle activation or sprint performance in humans. Eur J Appl Physiol 91(2–3):230–237\nLattier G, Millet GY, Martin A, Martin V (2004) Fatigue and recovery after high-intensity exercise Part II: recovery interventions. Int J Sports Med 25(7):509–515\nLavender G, Bird SR (1989) Effect of sodium bicarbonate ingestion upon repeated sprints. Br J Sports Med 23:41–45\nMartin V, Millet GY, Lattier G, Perrod L (2005) Why does knee extensor muscles torque decrease after eccentric-type exercise? J Sports Med Phys Fitness 45(2):143–151\nMendez-Villanueva A, Hamer P, Bishop D (2007) Physical fitness and performance fatigue responses during repeated sprints matched for initial mechanical output. Med Sci Sports Exerc 39(12):2219–2225\nMendez-Villanueva A, Hamer P, Bishop D (2008) Fatigue in repeated-sprint exercise is related to muscle power factors and reduced neuromuscular activity. Eur J Appl Physiol 103(4):411–419\nMillet GY, Lepers R (2004) Alterations of neuromuscular function after prolonged running, cycling and skiing exercises. Sports Med 34:105–116\nMorana C, Perrey S (2009) Time course of postactivation potentiation during intermittent submaximal fatiguing contractions in endurance- and power-trained athletes. J Strength Cond Res 23(5):1456–1464\nRacinais S, Girard O (2009) ITT: a tool to demonstrate muscle inactivation rather than to calculate a percentage of activation. In: Comments on point:counterpoint: the interpolated twitch does\u002Fdoes not provide a valid measure of the voluntary activation of muscle. J Appl Physiol 107:361\nRacinais S, Bishop D, Denis R, Lattier G, Mendez-Villaneuva A, Perrey S (2007a) Muscle de-oxygenation and neural drive to the muscle during repeated sprint cycling. Med Sci Sports Exerc 39:268–274\nRacinais S, Girard O, Micallef JP, Perrey S (2007b) Failed excitability of spinal motoneurons induced by prolonged running exercise. J Neurophysiol 97:596–603\nRupp T, Girard O, Perrey S (2009) Redetermination of the optimal intensity modifies resting H-reflex recovery after a sustained moderate-intensity muscle contraction. Muscle Nerve (in press)\nSale D, Quinlan J, Marsh E, McComas AJ, Belanger AY (1982) Influence of joint position on ankle plantar flexion in humans. J Appl Physiol 52:1636–1642\nSchieppati M (1987) The Hoffman reflex: a means for assessing spinal reflex excitability and its descending control in man. Prog Neurobiol 28:345–376\nSinoway LI, Hill JM, Pickar JG, Kaufman MP (1993) Effects of contraction and lactic acid on the discharge of group III muscle afferents in cats. J Neurophysiol 69(4):1053–1059\nSkof B, Strojnik V (2006) Neuro-muscular fatigue and recovery dynamics following anaerobic interval workload. Int J Sports Med 27(3):220–225\nStrojnik V, Komi PV (1998) Neuromuscular fatigue after maximal stretch-shortening cycle exercise. J Appl Physiol 84(1):344–350\nTaylor JL, Todd G, Gandevia SC (2006) Evidence for a supraspinal contribution to human muscle fatigue. 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present study was performed to investigate the effect of food intake on thermoregulatory vasodilatation in seven healthy male volunteers. The changes in oesophageal (T\noes) and mean skin temperatures, finger and forearm blood flows (BF), oxygen consumption (VO2) and heart rate (f\nc) with and without food intake were measured before and during a 40-min exercise at an intensity of 35% maximal O2 consumption at an ambient temperature of 25°C. Exercise commenced 60 min after food intake. Ingestion of food equivalent to 50.2 kJ · kg body mass−1 elevated mean body temperature, BF,VO2 andf\nc in 60 min. Four subjects responded to exercise with a marked increase in finger BF and with no sweating (non-sweating group), while the other three responded with perspiration over almost the whole skin area and with little change in finger BF. Further analyses were made mainly in the non-sweating group. The postprandial increases inT\noes, BF,VO2 andf\nc were persistent during exercise. The rate of increase in finger BF with the increase inT\noes and mean body temperature was significantly greater with food intake than without. However, there was no difference in the response of forearm BF to exercise between the two conditions. These results suggested that food intake enhanced finger BF response to the increase in deep body temperature during exercise. It was also concluded that there was a regional difference in cutaneous vasomotor response to thermal load in the post-prandial subjects.",{"EN":1769},"Enhancement of finger blood flow response of postprandial human subjects to the increase in body temperature during exercise",{"VOID":1771},"Fronek K, Stahlgren LH (1968) Systemic and regional hemodynamic changes during food intake and digestion in nonanesthetized dogs. Circ Res 23:687–692\nHales JRS, Fawcett AA, Bennett JW, Needham AD (1978) Thermal control of blood flow through capillaries and arteriovenous anastomoses in skin of sheep. Pflügers Arch 378:55–63\nHirai A, Hirata K, Hirashita M, Takahata T (1989) Effect of food intake on skin temperatures and thermal sensation (in Japanese). Hokuriku Taiikugakkai Kiyo 25:25–29\nHirata K, Nagasaka T, Hirai A, Hirashita M, Takahata T (1983) Peripheral vascular tone during heat load is modified by exercise intensity. Eur J Appl Physiol 51:7–15\nHirata K, Nagasaka T, Hirai A, Hirashita M, Takahata T (1986) Effects of human menstrual cycle on thermoregulatory vasodilation during exercise. Eur J Appl Physiol 54:559–565\nHirata K, Nagasaka T, Noda Y (1989a) Venous return from distal regions affects heat loss from the arms and legs during exercise-induced thermal loads. Eur J Appl Physiol 58:865–872\nHirata K, Nagasaka T, Hirashita M, Takahata T (1989b) Increase in evaporative and non-evaporative heat loss from the forearm depends on venous return from the hand during exercise. In: Mercer JB (ed) Thermal Physiology 1989, International Congress series 871 Excerpta Medica, Elsevier, Amsterdam, pp 155–159\nHonda N (1962) Temperature compensation for mercury strain gauge used in plethysmography. J Appl Physiol 17:572–574\nJohnson JM, Rowell LB, Brengelmann GL (1974) Modification of the skin blood flow-body temperature relationship by upright exercise. J Appl Physiol 37:880–886\nJones WB, Thomas HD, Reeves TJ (1965) Circulatory and ventilatory responses to postprandial exercise. Am Heart J 69:668–676\nKelbæk H, Munck O, Christensen NJ, Godtfredsen J (1987) Autonomic nervous control of postprandial hemodynamic changes at rest and upright exercise. J Appl Physiol 63:1862–1865\nLeBlanc J, Brondel L (1985) Role of palatability on meal-induced thermogenesis in human subjects. Am J Physiol 246:E333-E336\nLeBlanc J, Cabanac M, Samson P (1984) Reduced postprandial heat production with gavage as compared with meal feeding in human subjects. Am J Physiol 246:E95-E101\nMashford ML, Nilsson G, Rokaeus A, Rosell S (1978) The effect of food ingestion on circulating neurotensin-like immunoreactivity (NTLI) in the human. Acta Physiol Scand 104:244–246\nMorley JE, Levine AS, Gosnell BA, Mitchell JE, Krahn DD, Nizielski SE (1985) Peptides and feeding. Peptides 6:181–192\nNadel ER (1980) Circulatory and thermal regulations during exercise. Fed Proc 39:1491–1497\nNadel ER, Pandolf KB, Roberts MF, Stolwijk JAJ (1974) Mechanisms of thermal acclimation to exercise and heat. J App] Physiol 37:515–520\nNagasaka T, Shido O (1989) Locomotor activity and heat production of rats on restricted two-hour feeding regimes. Jpn J Biometeorol 26:85–90\nNelms JD (1963) Functional anatomy of skin related to temperature regulation. Fed Proc 22:933–936\nNichols J, Ross S, Patterson P (1988) Thermic effect of food at rest and following swim exercise in trained college men and women. Ann Nutr Metab 32:215–219\nNielsen B (1987) Does diet-induced thermogenesis change the preferred ambient temperature of humans? Eur J Appl Physiol 56:474–478\nSegal KR, Gutin B, Albu J, Pi-Sunyer FX (1987) Thermic effects of food and exercise in lean and obese men of similar lean body mass. Am J Physiol 252:E110-E117\nSherman JL (1963) Normal arteriovenous anastomoses. Medicine (Baltimore) 42:247–267\nShido O, Nagasaka T (1985) Effects of intraventricular neurotensin on blood pressure and heat balance in rats. Jpn J Physiol 35:311–320\nShido O, Yoneda Y, Nagasaka T (1989) Changes in brown adipose tissue metabolism following intraventricular vasoactive intestinal peptide and other gastrointestinal peptides in rats. Jpn J Physiol 39:359–369\nSteffens AB, Gugten JVD, Godeke J, Luiten PGM, Strubbe JH (1986) Meal-induced increases in parasympathetic and sympathetic activity elicit simultaneous rises in plasma insulin and free fatty acids. Physiol Behav 37:119–122\nStephenson LA, Wenger CB, O'Donovan BH, Nadel ER (1984) Circadian rhythm in sweating and cutaneous blood flow. Am J Physiol 246:R321-R324\nTakano N, Kotani M (1989) Influence of food intake on cold-induced vasodilation of finger. Jpn J Physiol 39:755–765\nWelle SL (1984) Metabolic responses to a meal during rest and low-intensity exercise. Am J Clin Nutr 40:990–994\nWelle SL, Lilavivathana U, Campbell RG (1980) Increased plasma norepinephrine concentrations and metabolic rates following glucose ingestion in man. Metabolism 29:806–809\nWelle SL, Lilavivathana U, Campbell RG (1981) Thermic effect of feeding in man. Metabolism 30:953–960\nWhitney RJ (1953) The measurement of volume changes in human limbs. J Physiol 121:1–27\nWoods SC, West DB, Stein LJ, Mckay LD, Lotter EC, Porte SG, Kenney NJ, Porte D Jr (1981) Peptides and the control of meal size. 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the last few years, epidemiological studies have shown correlations between the low endurance capacity of the erector spinae muscle and low back pain (LBP). In this study, the function of the erector spinae muscle and localized muscle fatigue in LBP were investigated in both LBP patients and control subjects without LBP, using surface multi-channel electromyograms (EMG). Four positions (L1, L2, L4, L5) were chosen as representative locations for the different functions of the erector spinae muscle. Median frequency (f\nm) parameters of an EMG power density spectrum were monitored to quantify localized muscle fatigue. This research indicated a statistically significant difference (P \u003C 0.05) in f\nm, parameters between the LBP patients and the control subjects. The integrated electromyogram (iEMG) signals of LBP patients were considerably higher than for the control subjects during endurance isometric contraction. Our research suggested that human fatiguing erector spinae muscle, in submaximal voluntary contractions tests, induced a different firing order of motor unit spikes between two groups.",{"EN":1896},"Spectral analysis of electromyogram in lumbar muscles: fatigue induced endurance contraction",{"VOID":1898},"Bagnall KM, Ford DM, McFadden KD, Greenhill BJ, Raso VJ (1984) The histochemical composition of human vertebral muscle. Spine 9:470–473\nBerkson M, Schultz A, Nachemson A, Andersson G (1977) Voluntary strengths of male adults with acute low back syndrome. Clin Orthop 129:84–95\nBiering-Sorensen F (1983) A prospective study of low back pain in a general population, I. Occurrence, recurrence and aetiology. Scand J Rehabil Med 15:71–79\nBiering-Sorensen F (1984) Physical measurements as risk indicators for low-back trouble over a one-year period. Spine 9:106–119\nBigland-Ritchie B, Jones DA, Wood JJ (1979) Excitation frequency and muscle fatigue: electrical responses during human voluntary and stimulated contractions. Exp Neurol 64:414–427\nBigland-Ritchie B, Donnovan EF, Roussos CS (1981) Conduction velocity and EMG power spectrum changes in fatigue of sustained maximal efforts. J Appl Physiol 51:1300–1305\nBroman H, Bilotto G, De Luca CJ (1985) Myoelectric signal conduction velocity and spectral parameters: influence of force and time. J Appl Physiol 58:1428–1437\nCollins GA, Cohen MJ, Naliboff BD, Schandler SL (1982) Comparative analysis of paraspinal and frontals EMG, heart rate and skin conductance in chronic low back pain patient and normals to various postures and stresses. Scand J Rehabil Med 14:39–46\nCram JR, Steger JC (1983) EMG scanning in the diagnosis of chronic pain. Biofeedback Self Regul 8:229–241\nDe Luca CJ (1993) Use of the surface EMG signal for performance evaluation of back muscles. Muscle Nerve 16:210–216\nDe Luca CJ, Creigh JL (1985) Do the firing statistics of motor units modify the frequency content of the EMG signal during sustained contractions? In: Winter DA (ed) Biomechanics IXA. Human Kinetics, Champaign, Ill., pp 287–292\nFidler MW, Jowett RL, Troup JDG (1974) Histochemical study of the function of multifidus in scoliosis. In: Zorab PA (ed) Scoliosis and muscle. Heinemann Medical, pp 184–192\nFrymoyer JW, Pope MH, Constanza MC (1980) Epidemiologic studies of low back pain. Spine 5:419–423\nFurness R, Jessop J, Lippold OJC (1977) Long lasting increases in the tremor of human hand muscles following brief, strong efforts. J Physiol (Lond) 265:821–831\nHagberg M (1981) Muscular endurance and surface electromyogram in isometric and dynamic exercise. J Appl Physiol Respir Environ Exerc Physiol 18:44–49\nHansen JW (1964) Postoperative management in lumbar disc protrusions. Acta Orthop Scand [Supp] 71:1–47\nHoyt WH, Hunt HH, De Pouw MA (1981) Electromyographic assessment of chronic low back pain syndrome. J Am Osteopath Assoc 80:57–59\nJayasinghe WJ, Harding RH, Anderson JAD, Sweetman BJ (1978) An electromyographic investigation of postural fatigue in low back pain: a preliminary study. Electromyogr Clin Neurophysiol 18:191–198\nJorgensen K, Nicolaisen T (1986) Two methods for determining trunk extensor endurance. A comparative study. Eur J Appl Physiol 55:639–644\nJorgensen K, Nicolaisen T (1987) Trunk extensor endurance: determination and relation to low-back trouble. Ergonomics 30:259–267\nJorgensen K, Fallentin N, Krogh-Lund C, Jensen B (1988) Electromyography and fatigue during prolonged, low-level static contractions. Eur J Appl Physiol 57:316–321\nKadefors R, Kaiser E, Petersen I (1968) Dynamic spectrum analysis of myopotentials with special reference to muscle fatigue. Electromyography 8:39–74\nKomi PV, Tesch P (1979) EMG frequency power spectrum, muscle structure, and fatigue during dynamic contractions in man. Eur J Appl Physiol 42:41–50\nLindstrom L, Magnusson R, Petersen I (1970) Muscle fatigue and action potential conduction velocity changes studies with frequency analysis of EMG signals. Electromyogr Clin Neurophysiol 43:750–754\nLindstrom L, Kadefors R, Petersen I (1977) An electromyographic index for localized muscle fatigue. J Appl Physiol 43:750–754\nLinssen WHJP, Stegeman DF, Joosten EMG, Binkhorst RA, Merks MJH, Laak HJ ter, Notermans SLH (1991) Fatigue in type I fiber predominance: a muscle force and surface EMG study on the relative role of type I and type II muscle fiber. Muscle Nerve 14:829–837\nMills K, Edwards RHT (1984) Muscle fatigue in myophosphorylase deficiency: power spectral analysis of electromyogram. Electroencephalogr Clin Neurophysiol 57:330–335\nMoritani T, Muro M (1987) Motor unit activity and surface electromyogram power spectrum during increasing force of contraction. Eur J Appl Physiol 56:206–265\nMoritani T, Nagata A, Muro M (1982) Electromyographic manifestations of muscle fatigue. Med Sci Sport Exerc 14:198–202\nMoritani T, Tanaka H, Yoshida T, Ishii C, Shindo C (1984) Relationship between myoelectric signals and blood lactate during forearm incremental exercise. Am J Phys Med 63:122–132\nMoritani T, Gaffney FD, Carmichael T, Hargis J (1985a) Interrelationships among muscle fiber types, electromyogram, and blood pressure during fatiguing isometric contraction. In: Winter DA (ed) Biomechanics IX-A, Human Kinetics, Champaign, Ill., pp 287–292\nMoritani T, Muro M, Kijima A, Gaffney FD, Petersen D (1985b) Electromechanical changes during electrically induced and maximal voluntary contractions: surface and intramuscular EMG responses during sustained maximal voluntary contraction. Exp Neurol 88:484–499\nMoritani T, Muro M, Nagata A (1986) Intramuscular and surface electromyogram changes during muscle fatigue. J Appl Physiol 60:1179–1185\nNachemson A, Lindh M (1969) Measurement of abdominal and back muscle strength with and without low back pain. Scand J Rehabil Med 1:60–65\nNicolaisen T, Jorgensen K (1985) Trunk strength, back muscle endurance, and low-back trouble. Scand J Rehabil Med 17:121–127\nPedersen OF, Petersen R, Schack Staffeldt E (1975) Back pain and isometric back muscle strength of workers in a Danish fact Scand J Rehabil Med 7:125–128\nRoy SH, De Luca CJ, Casavant DA (1989) Lumbar muscle fatigue and chronic lower back pain. Spine 14:992–1001\nRoy SH, De Luca CJ, Snyder-Mackler L, Emley MS, Crenshaw RL, Lyons JP (1990) Fatigue, recovery, and low back pain in varsity rowers. Med Sci Sports Exerc 22:463–469\nSeidel H, Beyer H, Brauer D (1987) Electromyographic evaluation of back muscle fatigue with repeated sustained contractions of different strengths. Eur J Appl Physiol 56:592–602\nSirca A, Kostevc V (1985) The fiber type composition of thoracic and lumbar paravertebral muscle in man. J Anat 141:131–137\nThorstensson A, Carlson H (1987) Fiber types in human lumbar back muscles. Acta Physiol Scand 131:195–202\nVan Dieen JH, Toussaint HM, Thissen C, Van de Ven A (1993) Spectral analysis of EMG during intermittent isometric fatiguing exercise. Ergonomics 36:404–414\nViitasalo JT, Komi PV (1977) Signal characteristics of EMG during fatigue. Eur J Appl Physiol 37:111–121\nZwarts MJ, Van Weerden TW, Haenen HTM (1987) Relationship between average muscle fiber conduction velocity and EMG power spectra during isometric contraction, recovery and applied ishemia. 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upper limit of brachial artery (BA) flow-mediated dilation (FMD) has not been thoroughly interrogated, and long duration occlusion + handgrip exercise may create larger shear stress stimuli than previous manipulations. To determine whether novel combinations of occlusion + handgrip exercise can extend the range of FMD stimulus–response relationship characterization and permit identification of a BA-FMD response ceiling. Ten healthy subjects performed eight reactive hyperemia (RH) FMD trials: 5, 10, and 15 min of occlusion (5RH, 10RH, 15RH); 5, 10 and 15 min of occlusion + 3-min ischemic exercise (IE) (5IE, 10IE, 15IE); 10 and 15 min of occlusion + 3-min IE + 4-min post-occlusion exercise (PE) (10IEPE, 15IEPE). Shear stress was estimated as shear rate (SR = blood velocity\u002FBA diameter; (ultrasound assessment)) (SR stimulus = area under the curve (AUC) until peak diameter). Data are mean ± SE. There were no differences in SR-AUC among IE and IEPE trials (p > 0.70), however, IE consistently increased the SR-AUC (IE + IEPE trial average 17,845.1 ± 2,023.3 a.u.) vs. the 5RH and 10RH trials (4,943.0 ± 428.4 a.u., 6,800.6 ± 805.9 a.u.) (p \u003C 0.05). The %FMD ranged from 7.3 ± 0.8 % (5RH) to 19.1 ± 2.0 % (15IEPE) (p \u003C 0.001) with no differences among IE and IEPE trials (p > 0.16). FMD increased with increasing SR-AUC (all subjects, all trials: r\n                  2 0.36, p \u003C 0.001) The stimulus created by brief (5 min) occlusion + ischemic exercise was not significantly enhanced by prolonging occlusion or continuing to exercise post-occlusion. The FMD response did not clearly plateau with increasing stimulus magnitude; however, the FMD capacity was shown to be more than double the FMD magnitude that was elicited with a standard 5-min occlusion test.",{"EN":2047},"Can a combination of handgrip exercise and prolonged forearm occlusion elicit a maximal brachial artery FMD response?",{"VOID":2049},"Agewall S, Hulthe J, Fagerberg B, Gottfridsson B, Wikstrand J (2002) Post-occlusion brachial artery vasodilatation after ischaemic handgrip exercise is nitric oxide mediated. Clin Physiol Funct Imaging 22:18–23\nBellien J, Iacob M, Gutierrez L, Isabelle M, Lahary A, Thuillez C, Joannides R (2006) Crucial role of NO and endothelium-derived hyperpolarizing factor in human sustained conduit artery flow-mediated dilatation. Hypertension 48:1088–1094\nBetik AC, Luckham VB, Hughson RL (2004) Flow-mediated dilation in human brachial artery after different circulatory occlusion conditions. Am J Physiol Heart Circ Physiol 286:H442–H448\nBirk GK, Dawson EA, Atkinson C, Haynes A, Cable NT, Thijssen DH, Green DJ (2012) Brachial artery adaptation to lower limb exercise training: role of shear stress. J Appl Physiol 112(1985):1653–1658\nCelermajer DS, Sorensen KE, Gooch VM, Spiegelhalter DJ, Miller OI, Sullivan ID, Lloyd JK, Deanfield JE (1992) Non-invasive detection of endothelial dysfunction in children and adults at risk of atherosclerosis. Lancet 340:1111–1115\nCorretti MC, Plotnick GD, Vogel RA (1995) Technical aspects of evaluating brachial artery vasodilatation using high-frequency ultrasound. Am J Physiol 268:H1397–H1404\nDoshi SN, Naka KK, Payne N, Jones CJ, Ashton M, Lewis MJ, Goodfellow J (2001) Flow-mediated dilatation following wrist and upper arm occlusion in humans: the contribution of nitric oxide. Clin Sci (Lond) 101:629–635\nDyson KS, Shoemaker JK, Hughson RL (2006) Effect of acute sympathetic nervous system activation on flow-mediated dilation of brachial artery. Am J Physiol Heart Circ Physiol 290:H1446–H1453\nGreen DJ, Jones H, Thijssen D, Cable NT, Atkinson G (2011) Flow-mediated dilation and cardiovascular event prediction: does nitric oxide matter? Hypertension 57:363–369\nGreen DJ, Dawson EA, Groenewoud HM, Jones H, Thijssen DH (2014) Is flow-mediated dilation nitric oxide mediated?: a meta-analysis. Hypertension 63:376–382\nHarris RA, Nishiyama SK, Wray DW, Tedjasaputra V, Bailey DM, Richardson RS (2009) The effect of oral antioxidants on brachial artery flow-mediated dilation following 5 and 10 min of ischemia. Eur J Appl Physiol 107:445–453\nHijmering ML, Stroes ES, Olijhoek J, Hutten BA, Blankestijn PJ, Rabelink TJ (2002) Sympathetic activation markedly reduces endothelium-dependent, flow-mediated vasodilation. J Am Coll Cardiol 39:683–688\nIshibashi Y, Takahashi N, Shimada T, Sugamori T, Sakane T, Umeno T, Hirano Y, Oyake N, Murakami Y (2006) Short duration of reactive hyperemia in the forearm of subjects with multiple cardiovascular risk factors. Circ J 70:115–123\nJazuli F, Pyke KE (2011) The impact of baseline artery diameter on flow-mediated vasodilation: a comparison of brachial and radial artery responses to matched levels of shear stress. Am J Physiol Heart Circ Physiol 301:H1667–H1677\nJoannides R, Bakkali E, Richard V, Benoist A, Moore N, Thuillez C (1997) Evaluation of the determinants of flow-mediated radial artery vasodilatation in humans. Clin Exp Hypertens 19:813–826\nKooijman M, Thijssen DH, de Groot PC, Bleeker MW, van Kuppevelt HJ, Green DJ, Rongen GA, Smits P, Hopman MT (2008) Flow-mediated dilatation in the superficial femoral artery is nitric oxide mediated in humans. J Physiol 586:1137–1145\nLeeson P, Thorne S, Donald A, Mullen M, Clarkson P, Deanfield J (1997) Non-invasive measurement of endothelial function: effect on brachial artery dilatation of graded endothelial dependent and independent stimuli. Heart 78:22–27\nLieberman EH, Gerhard MD, Uehata A, Selwyn AP, Ganz P, Yeung AC, Creager MA (1996) Flow-induced vasodilation of the human brachial artery is impaired in patients \u003C40 years of age with coronary artery disease. Am J Cardiol 78:1210–1214\nMark AL, Victor RG, Nerhed C, Wallin BG (1985) Microneurographic studies of the mechanisms of sympathetic nerve responses to static exercise in humans. Circ Res 57:461–469\nMitchell GF, Parise H, Vita JA, Larson MG, Warner E, Keaney JF Jr, Keyes MJ, Levy D, Vasan RS, Benjamin EJ (2004) Local shear stress and brachial artery flow-mediated dilation: the Framingham Heart Study. Hypertension 44:134–139\nMullen MJ, Kharbanda RK, Cross J, Donald AE, Taylor M, Vallance P, Deanfield JE, MacAllister RJ (2001) Heterogenous nature of flow-mediated dilatation in human conduit arteries in vivo: relevance to endothelial dysfunction in hypercholesterolemia. Circ Res 88:145–151\nNaylor LH, Weisbrod CJ, O’Driscoll G, Green DJ (2005) Measuring peripheral resistance and conduit arterial structure in humans using doppler ultrasound. J Appl Physiol 98:2311–2315\nNeunteufl T, Katzenschlager R, Hassan A, Klaar U, Schwarzacher S, Glogar D, Bauer P, Weidinger F (1997) Systemic endothelial dysfunction is related to the extent and severity of coronary artery disease. Atherosclerosis 129:111–118\nPadilla J, Johnson BD, Newcomer SC, Wilhite DP, Mickleborough TD, Fly AD, Mather KJ, Wallace JP (2009) Adjusting flow-mediated dilation for shear stress stimulus allows demonstration of endothelial dysfunction in a population with moderate cardiovascular risk. J Vasc Res 46:592–600\nParker BA, Tschakovsky ME, Augeri AL, Polk DM, Thompson PD, Kiernan FJ (2011) Heterogenous vasodilator pathways underlie flow-mediated dilation in men and women. Am J Physiol Heart Circ Physiol 301:H1118–H1126\nPyke KE, Tschakovsky ME (2005) The relationship between shear stress and flow-mediated dilatation: implications for the assessment of endothelial function. J Physiol 568:357–369\nPyke KE, Tschakovsky ME (2007) Peak vs. total reactive hyperemia: which determines the magnitude of flow-mediated dilation? J Appl Physiol 102:1510–1519\nPyke KE, Hartnett JA, Tschakovsky ME (2008a) Are the dynamic response characteristics of brachial artery flow-mediated dilation sensitive to the magnitude of increase in shear stimulus? J Appl Physiol 105:282–292\nPyke KE, Poitras V, Tschakovsky ME (2008b) Brachial artery flow mediated dilation during handgrip exercise: evidence for endothelial transduction of the mean shear stimulus. Am J Physiol Heart Circ Physiol 294:H2669–H2679\nPyke K, Green DJ, Weisbrod C, Best M, Dembo L, O’Driscoll G, Tschakovsky M (2010) Nitric oxide is not obligatory for radial artery flow-mediated dilation following release of 5 or 10 min distal occlusion. Am J Physiol Heart Circ Physiol 298:H119–H126\nSegal SS, Jacobs TL (2001) Role for endothelial cell conduction in ascending vasodilatation and exercise hyperaemia in hamster skeletal muscle. J Physiol 536:937–946\nShamim-Uzzaman QA, Pfenninger D, Kehrer C, Chakrabarti A, Kacirotti N, Rubenfire M, Brook R, Rajagopalan S (2002) Altered cutaneous microvascular responses to reactive hyperaemia in coronary artery disease: a comparative study with conduit vessel responses. Clin Sci (Lond) 103:267–273\nThijssen DH, Black MA, Pyke KE, Padilla J, Atkinson G, Harris RA, Parker B, Widlansky ME, Tschakovsky ME, Green DJ (2011a) Assessment of flow-mediated dilation in humans: a methodological and physiological guideline. Am J Physiol Heart Circ Physiol 300:H2–H12\nThijssen DH, Tinken TM, Hopkins N, Dawson EA, Cable NT, Green DJ (2011b) The impact of exercise training on the diameter dilator response to forearm ischaemia in healthy men. Acta Physiol (Oxf) 201:427–434\nTinken TM, Thijssen DH, Hopkins N, Dawson EA, Cable NT, Green DJ (2010) Shear stress mediates endothelial adaptations to exercise training in humans. Hypertension 55:312–318\nVictor RG, Seals DR, Mark AL (1987) Differential control of heart rate and sympathetic nerve activity during dynamic exercise. Insight from intraneural recordings in humans. J Clin Invest 79:508–516\nWendelhag I, Fagerberg B, Wikstrand J (1999) Adding ischaemic hand exercise during occlusion of the brachial artery increases the flow-mediated vasodilation in ultrasound studies of endothelial function. Clin Physiol 19:279–283\nWray DW, Uberoi A, Lawrenson L, Richardson RS (2006) Evidence of preserved endothelial function and vascular plasticity with age. Am J Physiol Heart Circ Physiol 290:H1271–H1277\nWray DW, Witman MA, Ives SJ, McDaniel J, Fjeldstad AS, Trinity JD, Conklin JD, Supiano MA, Richardson RS (2011) Progressive handgrip exercise: evidence of nitric oxide-dependent vasodilation and blood flow regulation in humans. Am J Physiol Heart Circ Physiol 300:H1101–H1107\nWray DW, Witman MA, Ives SJ, McDaniel J, Trinity JD, Conklin JD, Supiano MA, Richardson RS (2013) Does brachial artery flow-mediated vasodilation provide a bioassay for NO? 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activities such as running have been shown to be osteogenic. However, investigations have also shown that running may lead to site-specific deficiencies in bone mineral density (BMD) as well as overall low BMD. The purpose of this investigation was to evaluate and compare the BMD of female and male collegiate cross-country runners with non-running controls. In addition, energy availability and disordered eating attitudes and behaviors were assessed. BMD of 60 collegiate cross-country runners and 47 BMI and age-matched non-running controls were measured via DXA scans. Participants completed a Block 2014 Food Frequency Questionnaire and Eating Disorder Examination Questionnaire. Controlling for fat-free mass (FFM), male runners showed greater BMD at the femoral neck (0.934 ± 0.029 vs. 0.866 ± 0.028 g cm2, p \u003C 0.05), total hip (1.119 ± 0.023 vs. 1.038 ± 0.021 g cm2, p \u003C 0.05), and whole body (1.119 ± 0.023 vs. 1.038 ± 0.021 g cm2, p \u003C 0.05) than male controls. The female runners had greater whole-body BMD than female controls (1.143 ± 0.018 vs. 1.087 ± 0.022 g cm2, p \u003C 0.05). Runners scored significantly higher than controls in dietary restraint (1.134 ± 1.24 vs. 0.451 ± 0.75, p \u003C 0.05), male runners were significantly higher than male controls in eating concern (1.344 ± 1.08 vs. 0.113 ± 0.27, p \u003C 0.05) and female runners were significantly higher than male runners in shape concern (1.056 ± 1.27 vs. 0.242 ± 0.31, p \u003C 0.05). Forty-two percent of the male runners and 29% of female runners had an energy availability of less than 30 kcals kg−1FFM. It appears that distance running has beneficial effects on whole-body BMD and site-specific areas. Further research is warranted to further clarify the health effects of eating behaviors and EA of distance runners.",{"EN":2170},"Bone mineral density, energy availability, and dietary restraint in collegiate cross-country runners and non-running controls",{"VOID":2172},"Ainsworth BE, Haskell WL, Herrmann SD, Meckes N, Bassett DR Jr, Tudor-Locke C, Leon AS (2011) 2011 compendium of physical activities: a second update of codes and MET values. Med Sci Sports Exerc 43(8):1575–1581. https:\u002F\u002Fdoi.org\u002F10.1249\u002FMSS.0b013e31821ece12\nAnderson DA, Lundgren JD, Shapiro JR, Paulosky CA (2004) Assessment of eating disorders: review and recommendations for clinical use. Behav Modif 28(6):763–782. https:\u002F\u002Fdoi.org\u002F10.1177\u002F0145445503259851\nBarrack MT, Rauh MJ, Barkai HS, Nichols JF (2008) Dietary restraint and low bone mass in female adolescent endurance runners. Am J Clin Nutr 87(1):36–43\nBarrack MT, Gibbs JC, De Souza MJ, Williams NI, Nichols JF, Rauh MJ, Nattiv A (2014) Higher incidence of bone stress injuries with increasing female athlete triad-related risk factors: a prospective multisite study of exercising girls and women. Am J Sports Med 42(4):949–958. https:\u002F\u002Fdoi.org\u002F10.1177\u002F0363546513520295\nBarrack MT, Fredericson M, Tenforde AS, Nattiv A (2017) Evidence of a cumulative effect for risk factors predicting low bone mass among male adolescent athletes. Br J Sports Med 51(3):200–205. https:\u002F\u002Fdoi.org\u002F10.1136\u002Fbjsports-2016-096698\nBaxter-Jones AD, Mirwald RL, McKay HA, Bailey DA (2003) A longitudinal analysis of sex differences in bone mineral accrual in healthy 8–19-year-old boys and girls. Ann Hum Biol 30(2):160–175\nBaxter-Jones AD, Faulkner RA, Forwood MR, Mirwald RL, Bailey DA (2011) Bone mineral accrual from 8 to 30 years of age: an estimation of peak bone mass. J Bone Miner Res 26(8):1729–1739. https:\u002F\u002Fdoi.org\u002F10.1002\u002Fjbmr.412\nBlack AE (2000) Critical evaluation of energy intake using the Goldberg cut-off for energy intake: basal metabolic rate. A practical guide to its calculation, use and limitations. Int J Obes Relat Metab Disord 24(9):1119–1130\nBlock G, Woods M, Potosky A, Clifford C (1990) Validation of a self-administered diet history questionnaire using multiple diet records. J Clin Epidemiol 43(12):1327–1335\nBlock G, Thompson FE, Hartman AM, Larkin FA, Guire KE (1992) Comparison of two dietary questionnaires validated against multiple dietary records collected during a 1-year period. J Am Diet Assoc 92(6):686–693\nBuckinx F, Landi F, Cesari M et al (2018) Pitfalls in the measurement of muscle mass: a need for a reference standard. J Cachexia Sarcopenia Muscle 9(2):269–278\nBurke LM, Close GL, Lundy B, Mooses M, Morton JP, Tenforde AS (2018a) Relative energy deficiency in sport in male athletes: a commentary on its presentation among selected groups of male athletes. Int J Sport Nutr Exerc Metab 28(4):364–374. https:\u002F\u002Fdoi.org\u002F10.1123\u002Fijsnem.2018-0182\nBurke LM, Lundy B, Fahrenholtz IL, Melin AK (2018b) Pitfalls of conducting and interpreting estimates of energy availability in free-living athletes. Int J Sport Nutr Exerc Metab 28(4):350–363. https:\u002F\u002Fdoi.org\u002F10.1123\u002Fijsnem.2018-0142\nCunningham JJ (1991) Body composition as a determinant of energy expenditure: a synthetic review and a proposed general prediction equation. Am J Clin Nutr 54(6):963–969. https:\u002F\u002Fdoi.org\u002F10.1093\u002Fajcn\u002F54.6.963\nDarcy AM, Hardy KK, Lock J, Hill KB, Peebles R (2013) The Eating Disorder Examination Questionnaire (EDE-Q) among university men and women at different levels of athleticism. Eat Behav 14(3):378–381. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.eatbeh.2013.04.002\nFairburn CG, Cooper Z, O’Connor M (2008) Eating Disorder Examination. In: Fairburn CG (ed) Cognitive behavior therapy and eating disorders, 16.0th edn. Guilford Press, New York\nFredericson M, Chew K, Ngo J, Cleek T, Kiratli J, Cobb K (2007) Regional bone mineral density in male athletes: a comparison of soccer players, runners and controls. Br J Sports Med 41(10):664–668. https:\u002F\u002Fdoi.org\u002F10.1136\u002Fbjsm.2006.030783 (discussion 668)\nGarber CE, Blissmer B, Deschenes MR, Franklin BA, Lamonte MJ, Lee IM, American College of Sports M (2011) American College of Sports Medicine position stand. Quantity and quality of exercise for developing and maintaining cardiorespiratory, musculoskeletal, and neuromotor fitness in apparently healthy adults: guidance for prescribing exercise. Med Sci Sports Exerc 43(7):1334–1359. https:\u002F\u002Fdoi.org\u002F10.1249\u002Fmss.0b013e318213fefb\nGuebels CP, Kam LC, Maddalozzo GF, Manore MM (2014) Active women before\u002Fafter an intervention designed to restore menstrual function: resting metabolic rate and comparison of four methods to quantify energy expenditure and energy availability. Int J Sport Nutr Exerc Metab 24(1):37–46. https:\u002F\u002Fdoi.org\u002F10.1123\u002Fijsnem.2012-0165\nGunter KB, Almstedt HC, Janz KF (2012) Physical activity in childhood may be the key to optimizing lifespan skeletal health. Exerc Sport Sci Rev 40(1):13–21. https:\u002F\u002Fdoi.org\u002F10.1097\u002FJES.0b013e318236e5ee\nHartman AM, Block G, Chan W, Williams J, McAdams M, Banks WL Jr, Robbins A (1996) Reproducibility of a self-administered diet history questionnaire administered three times over three different seasons. Nutr Cancer 25(3):305–315\nHeikura IA, Uusitalo ALT, Stellingwerff T, Bergland D, Mero AA, Burke LM (2018) Low energy availability is difficult to assess but outcomes have large impact on bone injury rates in elite distance athletes. Int J Sport Nutr Exerc Metab 28(4):403–411. https:\u002F\u002Fdoi.org\u002F10.1123\u002Fijsnem.2017-0313\nHind K, Truscott JG, Evans JA (2006) Low lumbar spine bone mineral density in both male and female endurance runners. Bone 39(4):880–885. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.bone.2006.03.012\nHind K, Gannon L, Whatley E, Cooke C, Truscott J (2012) Bone cross-sectional geometry in male runners, gymnasts, swimmers and non-athletic controls: a hip-structural analysis study. Eur J Appl Physiol 112(2):535–541. https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs00421-011-2008-y\nIhle R, Loucks AB (2004) Dose-response relationships between energy availability and bone turnover in young exercising women. J Bone Miner Res 19(8):1231–1240. https:\u002F\u002Fdoi.org\u002F10.1359\u002FJBMR.040410\nKemmler W, Engelke K, Baumann H, Beeskow C, von Stengel S, Weineck J, Kalender WA (2006) Bone status in elite male runners. Eur J Appl Physiol 96(1):78–85. https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs00421-005-0060-1\nKoehler K, Achtzehn S, Braun H, Mester J, Schaenzer W (2013) Comparison of self-reported energy availability and metabolic hormones to assess adequacy of dietary energy intake in young elite athletes. Appl Physiol Nutr Metab 38(7):725–733. https:\u002F\u002Fdoi.org\u002F10.1139\u002Fapnm-2012-0373\nKohl HW, Blair SN, Paffenbarger RS Jr, Macera CA, Kronenfeld JJ (1988) A mail survey of physical activity habits as related to measured physical fitness. Am J Epidemiol 127(6):1228–1239\nKohrt WM, Bloomfield SA, Little KD, Nelson ME, Yingling VR (2004) American College of Sports Medicine Position Stand: physical activity and bone health. Med Sci Sports Exerc 36(11):1985–1996\nLaBrie JW, Boyle S, Earle A, Almstedt HC (2018) Heavy episodic drinking is associated with poorer bone health in adolescent and young adult women. J Stud Alcohol Drugs 79(3):391–398\nLoucks AB (2004) Energy balance and body composition in sports and exercise. J Sports Sci 22(1):1–14. https:\u002F\u002Fdoi.org\u002F10.1080\u002F0264041031000140518\nLoucks AB, Kiens B, Wright HH (2011) Energy availability in athletes. J Sports Sci 29(Suppl 1):S7–15. https:\u002F\u002Fdoi.org\u002F10.1080\u002F02640414.2011.588958\nLuce KH, Engler PA, Crowther JH (2007) Eating disorders and alcohol use: group differences in consumption rates and drinking motives. Eat Behav 8(2):177–184. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.eatbeh.2006.04.003\nMelin A, Tornberg AB, Skouby S, Moller SS, Sundgot-Borgen J, Faber J, Sjodin A (2015) Energy availability and the female athlete triad in elite endurance athletes. Scand J Med Sci Sports 25(5):610–622. https:\u002F\u002Fdoi.org\u002F10.1111\u002Fsms.12261\nMountjoy M, Sundgot-Borgen J, Burke L, Carter S, Constantini N, Lebrun C, Ljungqvist A (2014) The IOC consensus statement: beyond the Female Athlete Triad-Relative Energy Deficiency in Sport (RED-S). Br J Sports Med 48(7):491–497. https:\u002F\u002Fdoi.org\u002F10.1136\u002Fbjsports-2014-093502\nMountjoy M, Sundgot-Borgen J, Burke L, Ackerman KE, Blauwet C, Constantini N, Budgett R (2018) International Olympic Committee (IOC) Consensus Statement on Relative Energy Deficiency in Sport (RED-S): 2018 update. Int J Sport Nutr Exerc Metab 28(4):316–331. https:\u002F\u002Fdoi.org\u002F10.1123\u002Fijsnem.2018-0136\nNattiv A, Loucks AB, Manore MM, Sanborn CF, Sundgot-Borgen J, Warren MP (2007) American College of Sports Medicine position stand. The female athlete triad. Med Sci Sports Exerc 39(10):1867–1882. https:\u002F\u002Fdoi.org\u002F10.1249\u002Fmss.0b013e318149f11100005768-200710000-00026\nPapageorgiou M, Elliott-Sale KJ, Parsons A, Tang JCY, Greeves JP, Fraser WD, Sale C (2017) Effects of reduced energy availability on bone metabolism in women and men. Bone 105:191–199. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.bone.2017.08.019\nPereira MA, FitzerGerald SJ, Gregg EW, Joswiak ML, Ryan WJ, Suminski RR, Zmuda JM (1997) A collection of Physical Activity Questionnaires for health-related research. Med Sci Sports Exerc 29(6 Suppl):S1–205\nQuick VM, Byrd-Bredbenner C (2013) Eating Disorders Examination Questionnaire (EDE-Q): norms for US college students. Eat Weight Disord 18(1):29–35. https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs40519-013-0015-1\nSmock AJ, Hughes JM, Popp KL, Wetzsteon RJ, Stovitz SD, Kaufman BC, Petit MA (2009) Bone volumetric density, geometry, and strength in female and male collegiate runners. Med Sci Sports Exerc 41(11):2026–2032. https:\u002F\u002Fdoi.org\u002F10.1249\u002FMSS.0b013e3181a7a5a2\nStewart AD, Hannan J (2000) Total and regional bone density in male runners, cyclists, and controls. Med Sci Sports Exerc 32(8):1373–1377\nTam N, Santos-Concejero J, Tucker R, Lamberts RP, Micklesfield LK (2018) Bone health in elite Kenyan runners. J Sports Sci 36(4):456–461. https:\u002F\u002Fdoi.org\u002F10.1080\u002F02640414.2017.1313998\nTenforde AS, Fredericson M (2011) Influence of sports participation on bone health in the young athlete: a review of the literature. PMR 3(9):861–867. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.pmrj.2011.05.019\nTenforde AS, Fredericson M, Sayres LC, Cutti P, Sainani KL (2015) Identifying sex-specific risk factors for low bone mineral density in adolescent runners. Am J Sports Med 43(6):1494–1504. https:\u002F\u002Fdoi.org\u002F10.1177\u002F0363546515572142\nTenforde AS, Kraus E, Fredericson M (2016) Bone stress injuries in runners. Phys Med Rehabil Clin N Am 27(1):139–149. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.pmr.2015.08.008\nTenforde AS, Carlson JL, Chang A, Sainani KL, Shultz R, Kim JH, Fredericson M (2017) Association of the female athlete triad risk assessment stratification to the development of bone stress injuries in collegiate athletes. Am J Sports Med 45(2):302–310. https:\u002F\u002Fdoi.org\u002F10.1177\u002F0363546516676262\nTenforde AS, Parziale AL, Popp KL, Ackerman KE (2018) Low bone mineral density in male athletes is associated with bone stress injuries at anatomic sites with greater trabecular composition. Am J Sports Med 46(1):30–36. https:\u002F\u002Fdoi.org\u002F10.1177\u002F0363546517730584\nViner RT, Harris M, Berning JR, Meyer NL (2015) Energy availability and dietary patterns of adult male and female competitive cyclists with lower than expected bone mineral density. Int J Sport Nutr Exerc Metab 25(6):594–602. https:\u002F\u002Fdoi.org\u002F10.1123\u002Fijsnem.2015-0073\nZanker CL, Swaine IL (2000) Responses of bone turnover markers to repeated endurance running in humans under conditions of energy balance or energy restriction. Eur J Appl Physiol 83(4–5):434–440. https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs004210000293",{"VOID":2174},"10.1007\u002Fs00421-019-04164-z","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs00421-019-04164-z",[2177,2192,2205,2220],{"id":2178,"sortIndex":32,"researcher":28,"roles":2179,"affiliations":2180,"properties":2189,"displayName":2191,"givenName":28,"familyName":28},"1428f65f-daee-468e-80a1-2a388dde6300",[1111],[2181],{"id":2182,"sortIndex":32,"affiliation":2183,"properties":28},"0ff5147e-6e00-465d-87c1-7a95a1a9a481",{"id":2182,"createTime":28,"updateTime":28,"relativeEntities":2184,"slug":28,"properties":2185,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2188,"statistic":28},[],{"title":2186},{"VI":2187},"Department of Health and Human Sciences, Loyola Marymount University, Los Angeles, USA",[],{"title":2190},{"VI":2191},"William P. 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ramp exercise the deoxy[Hb + Mb] pattern follows a sigmoid model [f(x) = f\n                        0 + A\u002F(1 + exp−(−c\n                         + dx))], indicating a non-linear muscle blood flow \n                  \n                    \n                  \n                  $$ (\\dot{Q}{\\text{m}})\u002F{\\text{oxygen}}\\;{\\text{uptake}}\\;(\\dot{V}{\\text{O}}_{{2{\\text{m}}}} ) $$\n                -relationship. We hypothesised that in trained cyclists the sigmoid would display a rightward shift, due to an increased oxidative capacity and\u002For higher percentage of slow-twitch fibres. A total of 10 cyclists and 11 physically active students (PA students) performed a relative ramp exercise (±12 min) and a ramp25-exercise (25 W min−1). Deoxy[Hb + Mb] was measured at the M. Vastus Lateralis by NIRS, normalized to the total amplitude of the response and expressed as a function of absolute and relative (%peakP) work rate. The work rate corresponding to c\u002Fd (i.e.50% of the amplitude of the deoxy[Hb + Mb] response) was the only parameter of the sigmoid that differed significantly between cyclists (57.9 ± 4.4% and 60.1 ± 4.1%peakP in the relative and ramp25, respectively) and PAstudents (49.6 ± 4.2% and 48.2 ± 5.1%peakP, respectively), indicating a rightward shift of the sigmoid in the cyclists. These results suggest a change in the time course of C(a–v)O2 as a function of aerobic fitness status.",{"EN":2308},"Pattern of deoxy[Hb + Mb] during ramp cycle exercise: influence of aerobic fitness status",{"VOID":2310},"Armstrong RB, Laughlin MH (1984) Exercise blood flow pattern within and among rat muscles after training. Am J Physiol 246:H59–H68\nBauer TA, Reusch JE, Levi M, Regensteiner JG (2007) Skeletal muscle deoxygenation after the onset of moderate exercise suggests slowed microvascular blood flow kinetics in type 2 diabetes. Diabetes Care 30:2880–2885. doi:10.2337\u002Fdc07-0843\nBeaver WL, Wasserman K, Whipp BJ (1986) A new method for detecting anaerobic threshold by gas exchange. J Appl Physiol 60:2020–2027\nBehnke BJ, McDonough P, Padilla DJ, Musch TI, Poole DC (2003) Oxygen exchange profile in rat muscle of contrasting fibre types. J Physiol 549:597–605. doi:10.1113\u002Fjphysiol.2002.035915\nBelardinelli R, Georgiou D, Barstow TJ (1995) Near-infrared spectroscopy and changes in skeletal muscle oxygenation during incremental exercise in chronic heart failure: a comparison with healthy subjects. G Ital Cardiol 25:715–724\nBoone J, Koppo K, Bouckaert J (2008) The \\( \\dot{V}{\\text{O}_{2}} \\) response to submaximal ramp cycle exercise: influence of ramp slope and training status. Respir Physiol Neurobiol 161:291–297. doi:10.1016\u002Fj.resp.2008.03.008\nCostill DL, Fink WJ, Pollock ML (1976) Muscle fibre composition and enzyme activities of elite distance runners. Med Sci Sports Exerc 8:96–100\nDelorey DS, Kowalchuk JM, Paterson DH (2003) Relationship between pulmonary O2 uptake kinetics and muscle deoxygenation during moderate-intensity exercise. J Appl Physiol 95:113–120\nDelp MD, Armstrong RB (1988) Blood flow in normal and denervated muscle during exercise in conscious rats. Am J Physiol 255:H1509–H1515\nEdgerton VR, Smith JL, Simpson DR (1975) Muscle fibre type populations of humans leg muscles. Histochem J 7:259–266. doi:10.1007\u002FBF01003594\nElder GCB (1977) The heterogeneity of fibre type populations in human muscle. Med Sci Sports Exerc 9:64–65. doi:10.1249\u002F00005768-197721000-00090\nFaulkner JA, Heigenhauser GJ, Schork MA (1977) The cardiac output-oxygen uptake relationship of men during graded bicycle ergometry. Med Sci Sports Exerc 9:148–154\nFerreira LF, Townsend DK, Lutjemeier BJ, Barstow TJ (2005) Muscle capillary blood flow kinetics estimated from pulmonary O2 uptake and near-infrared spectroscopy. J Appl Physiol 98:1820–1828. doi:10.1152\u002Fjapplphysiol.00907.2004\nFerreira LF, McDonough P, Behnke BJ, Musch TI, Poole DC (2006) Blood flow and O2 extraction as a function of O2 uptake in muscle composed of different fibre types. Respir Physiol Neurobiol 153:237–249. doi:10.1016\u002Fj.resp.2005.11.004\nFerreira LF, Koga S, Barstow TJ (2007) Dynamics of noninvasively estimated microvascular O2 extraction during ramp exercise. J Appl Physiol 103:1999–2004. doi:10.1152\u002Fjapplphysiol.01414.2006\nFolkow B, Halicka HD (1968) A comparison between ‘red’ and ‘white’muscle with respect to blood supply, capillary surface area and oxygen uptake during rest and movement. Microvasc Res 1:1–14. doi:10.1016\u002F0026-2862(68)90002-2\nGollnick PD, Saltin B (1982) Significance of skeletal muscle oxidative enzyme enhancement with endurance training. Clin Physiol 2:1–12. doi:10.1111\u002Fj.1475-097X.1982.tb00001.x\nGrassi B, Pogliaghi S, Rampichini S, Quaresima V, Ferrari M, Marconi C, Ceretelli P (2003) Muscle oxygenation and pulmonary gas exchange kinetics during cycle exercise on-transitions in humans. J Appl Physiol 95:149–158\nHarper AJ, Ferreira LF, Lutjemeier BJ, Townsend DK, Barstow TJ (2006) Human femoral artery and estimated muscle capillary blood flow kinetics following the onset of exercise. Exp Physiol 91:661–671. doi:10.1113\u002Fexpphysiol.2005.032904\nJohnson MA, Polgar J, Weightman D, Appleton D (1973) Data on the distribution of human fibre types in 36 human muscles. An autopsy study. J Neurol Sci 18:111–129. doi:10.1016\u002F0022-510X(73)90023-3\nKautz SA, Neptune RR (2002) Biomechanical determinants of pedalling energetics: internal and external work are not independent. Exerc Sport Sci Rev 30:159–165. doi:10.1097\u002F00003677-200210000-00004\nKoga S, Poole DC, Ferreira LF, Whipp BJ, Kondo N, Saitoh T, Ohmae E, Barstow TJ (2008) Spatial heterogeneity of quadriceps muscle deoxygenation kinetics during cycle exercise. J Appl Physiol 103:2049–2056. doi:10.1152\u002Fjapplphysiol.00627.2007\nKime R, Im J, Moser D, Lin Y, Nioka S, Katsumura T, Chance B (2005) Reduced heterogeneity of muscle deoxygenation during heavy bicycle exercise. Med Sci Sports Exerc 37:412–417. doi:10.1249\u002F01.MSS.0000155401.81284.76\nLaughlin MH, Armstrong RB (1982) Muscular blood flow distribution pattern as a function of running speed in rats. Am J Physiol 243:H296–H306\nLaughlin MH, Armstrong RB (1987) Adrenoreceptor effects on rat muscle blood flow during treadmill exercise. J Appl Physiol 62:1465–1472\nLexell J, Henriksson-Larsen K, Sjöstrom M (1983) Distribution of different fibre types in human skeletal muscle. 2. A study of cross-sections of whole m. vastus lateralis. Acta Physiol Scand 117:115–122\nLi YSW, Yuen CWM, Yueng KW, Sin KM (1999) Prediction of the best-fit regression model to correlate instrumental colour measurement and visual assessment. Colour Technol 115:22–31. doi:10.1111\u002Fj.1478-4408.1999.tb00346.x\nLutjemeier BJ, Miura A, Scheuermann BW, Koga S, Townsend DK, Barstow TJ (2005) Muscle contraction-blood flow interactions during upright knee extension exercise in humans. J Appl Physiol 98:1575–1583. doi:10.1152\u002Fjapplphysiol.00219.2004\nMcDonough P, Behnke BJ, Padilla DJ, Musch TI, Poole DC (2005) Control of microvascular oxygen pressures in rat muscles comprised of different fibre types. J Physiol 563:903–913. doi:10.1113\u002Fjphysiol.2004.079533\nMcGuire DK, Levine BD, Williamson JW, Snell PG, Blomqvist CG, Saltin B, Mitchell JH (2001) A 30-year follow-up of the Dallas bedrest and training study. II. Effect of age on cardiovascular adaptation to exercise training. Circulation 104:1358–1366. doi:10.1161\u002Fhc3701.096099\nMizuno M, Tokizawa K, Iwakawa T, Muaoka I (2004) Inflection points of cardiovascular responses and oxygenation are correlated in the distal but not the proximal portions of muscle during incremental exercise. J Appl Physiol 97:867–873. doi:10.1152\u002Fjapplphysiol.00213.2004\nPoole DC, Sexton WL, Behnke BJ, Ferguson CS, Hageman KS, Musch TI (2000) Respiratory muscle blood flows during physiological and chemical hyperpnea in the rat. J Appl Physiol 88:186–194\nProctor DN, Miller JD, Dietz NM, Minson CT, Joyner MJ (2001) Reduced submaximal leg blood flow after high-intensity aerobic training. J Appl Physiol 91:2619–2627\nRichardson RS, Poole DC, Knight DR, Kurdak SS, Hogan MC, Grassi B, Johnson EC, Kendrick KF, Erickson BK, Wagner PD (1993) High muscle blood flow in man: is maximal O2 extraction compromised? J Appl Physiol 75:1911–1916\nSaltin B, Blomqvist G, Mitchell JH, Johnson RL Jr, Wildenthal K, Chapman CB (1968) Response to exercise after bed rest and after training. Circulation 38:VII1–VII78\nSheriff DD, Hakeman AL (2001) Role of speed vs grade in relation to muscle pump function at locomotion onset. J Appl Physiol 91:269–276\nSwanson GD, Hughson RL (1988) On the modelling and interpretation of oxygen uptake kinetics from ramp work rate tests. J Appl Physiol 65:2453–2458\nTrappe S, Harber M, Creer A, Gallagher P, Slivka D, Minchev K, Whitsett D (2006) Single muscle fibre adaptations with marathon training. J Appl Physiol 101:721–727. doi:10.1152\u002Fjapplphysiol.01595.2005\nTschakovsky ME, Hughson RL (2003) Rapid blunting of sympathetic vasoconstriction in the human forearm at the onset of exercise. J Appl Physiol 94:1785–1792\nVan Beekvelt MC, Borghuis MS, Van Engelen BG, Wevers RA, Colier WN (2001) Adipose tissue thickness affects in vivo quantitative near-infrared spectroscopy in human skeletal muscle. Clin Sci 101:21–28. doi:10.1042\u002FCS20000247\nWarner HR, Cox A (1962) A mathematical model of heart rate control by sympathetic and vagus efferent information. J Appl Physiol 17:349–355\nWoodman CR, Schrage WG, Rush JW, Ray CA, Price EM, Hasser EM, Laughlin MH (2001) Hindlimb unweighting decreases endothelium-dependent dilation and eNOS expression in soleus not gastrocnemius. J Appl Physiol 91:1091–1098\nWunsch SA, Muller-Delp J, Delp MD (2000) Time course of vasodilatory responses in skeletal muscle arterioles: role in hyperemia at onset of exercise. 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