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Journal of Medicine and Pharmacy","Tạp chí Y Dược học Cần Thơ",{"EN":487,"VI":488},"\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">04\u002F10\u002F2015 Ministry of Information and Communications allowed Can Tho journal of medicine and pharmacy to operate (102 \u002FGP-BTTTT)\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">07\u002F16\u002F2015 Can Tho journal of medicine and pharmacy is internationally recognized: ISSN 2354-1210\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">In 2016, The journal has been included in the list of medical science journals by The State Council for professorship which is awarded a work score of 0-0.5 points for a published article.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Can Tho Journal of Medicine and Pharmacy welcome original works that haven’t been submitted or published in other medical journals. Posts must contain content related to one of the journal’s categories.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The content published\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The journal is divided into 3 categories:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Scientific research article: are valuable scientific works, which have been researched and accepted.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Overview of medicine, biology and pharmacy: serving the objective of continuing training in the fields of medicine, biology and pharmacy; to systematize classical and modern knowledge.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Update information on new knowledge about medicine, biology, pharmacy in the country and in the world.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Scope\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Publication and introduction of scientific research in the fields:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Medicine (internal medicine, surgery, pediatrics, obstetrics and gynecology, odonto-stomatology, laboratory, oncology, traditional medicine, nursing).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Biology (genetics, biotechnology).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Pharmacology (pharmaceutics, drug quality analysis-control, synthetic pharmaceutical chemistry, biochemistry, pharmacognosy, botany, clinical pharmacy).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- To enhance the quality of undergraduate, postgraduate education, scientifically researching and meet the necessary treatment in hospital.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Introducing the updated domestic and oversea information about science technology to promote scientific research and exchanging technology in local, other universities.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Exchanging pharmaceutical and medical information for social health developing in the Mekong Delta and Vietnam.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The object\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Postgraduate students, student of Can Tho University of Medicine and Pharmacy, scientists from schools, research institutes, hospitals, health centers, pharmaceutical companies of the Mekong Delta; other provinces and regions in Vietnam and other country.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Address\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Headquarters of Can Tho Journal of Medicine and Pharmacy, located Scientific Research and International Cooperation Office: 179 Nguyen Van Cu Street, An Khanh Ward, Ninh Kieu District, Can Tho City, Vietnam.\u003C\u002Fspan>\u003C\u002Fp>","\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Ngày 16\u002F7\u002F2015, Tạp chí Y Dược học Cần Thơ được cấp chỉ số quốc tế: ISSN 2354-1210.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 4\u002F2016, Tạp chí đã được Hội đồng Giáo sư ngành Y đưa vào danh sách các tạp chí khoa học Y học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Năm 2020 Tạp chí Y Dược học Cần Thơ đã được phê duyệt vào danh mục của các Hội đồng Giáo sư ngành Dược học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ ra 12 số\u002Fnăm, 180-200 trang\u002Fsố.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 12\u002F2022 Tạp chí Y Dược học Cần Thơ là thành viên của hệ thống Crossref và từ tháng 01\u002F2023 tạp chí thực hiện bình duyệt online kín 2 chiều nhằm tăng tính minh bạch, tin cậy của các công trình nghiên cứu khoa học và đảm bảo tốt nhất chất lượng khoa học của bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ, mục đích và phạm vi của tạp chí\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ và mục đích hoạt động của tạp chí: xuất bản nhằm mục đích phổ biến kết quả từ các đề tài nghiên cứu khoa học; giao lưu trao đổi khoa học, chia sẻ kinh nghiệm, học tập, đồng thời cập nhật thông tin khoa học mới trong các lĩnh vực y, sinh, dược học trong và ngoài nước.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phạm vi của tạp chí: Tạp chí xuất bản được chia thành 3 chuyên mục: (i) Bài báo nghiên cứu khoa học là kết quả công trình nghiên cứu khoa học có giá trị đã được triển khai nghiên cứu, (ii) Bài tổng quan y, sinh, dược học: phục vụ mục tiêu đào tạo liên tục trong lĩnh vực y, sinh, dược học; nhằm hệ thống hóa những kiến thức kinh điển và hiện đại; (iii) Thông tin cập nhật kiến thức mới về y, sinh, dược học trong nước và trên thế giới.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Chính sách truy cập mở\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ áp dụng chính sách truy cập mở đối với các bài báo đã xuất bản đến với độc giả, nhằm mở rộng cơ hội tiếp cận các kết quả nghiên cứu chất lượng cao và tăng cường trao đổi kiến thức. Tạp chí đăng tải trực tuyến (miễn phí) toàn văn các bài báo được công bố trên website của Tạp chí (https:\u002F\u002Ftapchi.ctump.edu.vn).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đạo đức xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ cam kết tuân thủ đạo đức xuất bản phù hợp với các hướng dẫn và tiêu chuẩn của the Committee on Publication Ethics (COPE), tuân thủ các nguyên tắc của COPE’s Core Practices, Best Practices Guidelines for Journal Editors và Guidelines on Good Publication Practices.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Bản thảo bài báo chỉ được chấp nhận khi được tác giả chịu trách nhiệm chính cam kết các nội dung sau: Các nội dung của bản thảo chưa được đăng tải toàn bộ hoặc một phần ở các tạp chí khác; Tất cả các tác giả đều có đóng góp một cách đáng kể vào quá trình nghiên cứu hoặc chuẩn bị bản thảo và cùng chịu trách nhiệm về các nội dung của bản thảo; Tuân thủ các biện pháp đảm bảo đạo đức nghiên cứu (ví dụ thỏa thuận đồng ý tham gia nghiên cứu).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Cam kết bảo mật\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí cam kết thực hiện và tuân thủ các quy định của luật và các văn bản hướng dẫn liên quan đến bảo mật thông tin cá nhân trên không gian mạng. Các thông tin mà người dùng (tác giả, độc giả, biên tập viên, người phản biện) nhập vào các biểu mẫu trên Hệ thống Quản lý xuất bản trực tuyến của tạp chí chỉ được sử dụng vào các mục đích đã được tuyên bố rõ ràng và sẽ không được cung cấp cho bất kỳ bên thứ ba nào khác, hay dùng vào bất kỳ mục đích nào khác.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phí gửi bài\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng bài: 1.000.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng nhanh: 1.500.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với tác giả là cán bộ viên chức thuộc Trường Đại học Y Dược Cần Thơ thì được hỗ trợ 50% lệ phí gửi đăng bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với sinh viên thực hiện đề tài nghiên cứu khoa học cấp trường được hỗ trợ 100% lệ phí đăng bài ( Tác giả gửi đính kèm “ Quyết định về việc giao tổ chức thực hiện đề tài nghiên cứu khoa học cấp Trường của sinh viên”).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Hình thức nộp lệ phí:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Tiền mặt:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Nộp trực tiếp tại Phòng Tài chính - Kế toán, Trường Đại học Y Dược Cần Thơ, số 179 Nguyễn Văn Cừ, P. An Khánh, Q. Ninh Kiều, thành phố Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Chuyển khoản:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tên Tài khoản: Trường ĐHYD Cần Thơ, Số TK: 0111000115668, tại ngân hàng Vietcombank chi nhánh Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Thời gian: Áp dụng từ ngày 01\u002F02\u002F2023.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">* Phí gửi bài không được hoàn trả khi bài viết bị từ chối hoặc tác giả xin rút bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Quy trình phản biện bài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ thực hiện quy trình phản biện kín hai chiều nghiêm ngặt. Danh tính của những người phản biện không được tiết lộ cho các tác giả và ngược lại. Quy trình thẩm định bài báo đăng gồm các bước sau:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tiếp nhận bản thảo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tác giả liên hệ gửi bản thảo đến Tạp chí qua hệ thống trực tuyến tại website: https:\u002F\u002Ftapchi.ctump.edu.vn. Hướng dẫn về cách đăng ký, gửi bài và chuẩn bị bản thảo được cung cấp trên website của Tạp chí.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sàng lọc sơ bộ\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sau khi Tòa soạn nhận được bài báo của tác giả, Ban Thư ký sẽ tiến hành kiểm tra sơ bộ bài báo (các yêu cầu về nội dung và hình thức). Những bài báo không đúng quy cách hoặc có nội dung không phù hợp hoặc vi phạm bản quyền sẽ bị từ chối (Ban Thư ký thông báo phản hồi đến tác giả trong vòng 1 tuần). Những bài báo đủ điều kiện, được Ban Thư ký tòa soạn chuyển đến Ban Biên tập có cùng chuyên môn với nội dung bài báo để đề xuất người phản biện. Thời gian kể từ khi Ban Biên tập nhận bài báo đến khi đề xuất người phản biện bài báo chậm nhất là 5 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Vòng phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký gửi bài và yêu cầu phản biện đến 02 phản biện độc lập.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Các phản biện gởi nhận xét cho Ban Thư ký. Thời gian từ khi gửi bài cho phản biện đến khi nhận ý kiến của phản biện tối đa là 20 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xử ký kết quả phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Nếu ý kiến đồng ý cho đăng và không cần chỉnh sửa, Ban Thư ký tiếp tục đăng bài theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Nếu ý kiến đồng ý đăng và cần chỉnh sửa, Ban Thư ký sẽ thông tin đến tác giả chỉnh sửa theo yêu cầu của người phản biện. Thời gian chỉnh sửa và gửi lại kéo dài không quá 2 tuần, từ khi tác giả bài báo nhận được thông tin (Quá trình này có thể lặp lại tối đa 2 lần\u002F1 bài báo). Khi có sự thống nhất, đồng ý của người phản biện; bài báo được tiếp tục đăng theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Những bài báo có chất lượng không đạt yêu cầu, cả 2 phản biện không đồng ý cho đăng sẽ bị Tòa soạn từ chối đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký tổng hợp các bản thảo đã được tác giả hoàn thiện sau thẩm định trình Ban Biên tập xem xét, Tổng Biên tập phê duyệt, quyết định bài đăng theo các tiêu chí: sự phù hợp nội dung với tôn chỉ và mục đích, thể loại bài viết (ưu tiên các bài có bài có nghiên cứu chuyên sâu, hàm lượng khoa học cao), đóng góp mới bài báo, bài báo được ưu tiên đăng trong số gần nhất của Tạp chí theo thứ tự: tính thời sự, chất lượng bài báo và thời gian gửi bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Ban Biên tập và Ban Thư ký biên tập bản thảo, chế bản, đọc rà soát lỗi. Thời gian hoàn thành từ 10-15 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Ban Thư ký có trách nhiệm thông báo cho tác giả bài báo (bằng e-mail) về tình hình phê duyệt bài báo, thời gian, số kỳ, tập xuất bản bài báo theo qui định.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">4. Danh sách bài báo theo số Tạp chí được in ấn và phát hành trong năm định kỳ được công bố chính thức trên website: https:\u002F\u002Ftapchi.ctump.edu.vn\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>",{"VOID":490},"wcQ1uqwAAAAJ","2023-05-30T08:17:21.868+00:00",[],[494],{"id":495,"createTime":28,"updateTime":28,"relativeEntities":496,"slug":28,"properties":497,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":507,"parentIds":508,"statistic":28},"6413896b-eca9-442b-a73f-182a58a0ce40",[],{"title":498,"address":501,"country":504,"abbreviation":505},{"EN":499,"VI":500},"Can Tho University of Medicine and Pharmacy","Trường Đại học Y Dược Cần Thơ",{"EN":502,"VI":503},"No 179, Nguyen Van Cu street, An Khanh ward, Ninh Kieu district, Can Tho city, Vietnam","Số 179, đường Nguyễn Văn Cừ, phường An Khánh, quận Ninh Kiều, thành phố Cần Thơ, Việt Nam",{"VOID":15},{"VOID":506},"ctump","http:\u002F\u002Fwww.ctump.edu.vn\u002F",[],[],"https:\u002F\u002Ftapchi.ctump.edu.vn\u002Findex.php\u002Fctump",{"impactFactor":32,"impactFactorByYear":512,"i10Index":32,"i10IndexLast5Year":32,"totalPublication":514,"totalPublicationByYear":515,"totalCitation":520,"totalCitationByYear":521,"totalCitationPerPublication":108,"totalCitationPerPublicationByYear":523,"hindexLast5Year":45,"hindex":45},{"2022":513,"2023":111,"2024":106},0.01,1556,{"2020":47,"2021":516,"2022":517,"2023":518,"2024":519,"2025":122},57,306,801,358,161,{"2021":146,"2022":280,"2023":522},99,{"2021":524,"2022":318,"2023":104},0.23,{"impactFactor":28,"impactFactorByYear":28,"i10Index":123,"i10IndexLast5Year":123,"totalPublication":526,"totalPublicationByYear":527,"totalCitation":526,"totalCitationByYear":528,"totalCitationPerPublication":40,"totalCitationPerPublicationByYear":531,"hindexLast5Year":49,"hindex":49},476,{"0":205,"2019":123,"2021":139,"2022":459,"2023":451,"2024":357,"2025":49,"2026":48},{"2021":42,"2022":123,"2023":161,"2024":529,"2025":360,"2026":530},136,83,{"2021":105,"2022":513,"2023":532,"2024":127,"2025":533,"2026":534},0.62,25.43,13.83,{"id":536,"createTime":537,"updateTime":382,"relativeEntities":538,"slug":539,"properties":540,"entityType":25,"verifyStatus":26,"verifyTime":28,"verifyNote":28,"languages":552,"translateLanguages":28,"viewCount":133,"subjectFields":553,"manageAffiliations":554,"indexDatabases":555,"url":556,"thumbnailPath":557,"statistic":558,"gsStatistic":594,"type":55,"analyzePriority":28},"6984a56a-db70-403b-9cc4-4013e1ceaffa","2023-05-09T06:47:40.346+00:00",[],"T%E1%BA%A1p%20ch%C3%AD%20Nghi%C3%AAn%20c%E1%BB%A9u%20n%C6%B0%E1%BB%9Bc%20ngo%C3%A0i",{"country":541,"issn":542,"title":544,"introduce":547,"gsId":550},{"VOID":15},{"VOID":543},"25252445",{"EN":545,"VI":546},"VNU Journal of Foreign Studies","Tạp chí Nghiên cứu nước ngoài",{"EN":548,"VI":549},"{\"ops\":[{\"insert\":\"\\n\\nThe \\n\"},{\"attributes\":{\"italic\":true},\"insert\":\"VNU Journal of Science\"},{\"insert\":\"\\n was established in 1985 for the publication of national and international research papers in all fields of natural sciences and technology, social sciences and humanities. 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Since 2005, 150 consecutive patients were treated with primary radiotherapy for localized prostate cancer. Intensity modulated radiotherapy (IMRT) using the simultaneous integrated boost (SIB) technique was practiced in all patients and doses of 73.9 Gy (n = 41) and 76.2 Gy (n = 109) were delivered in 32 and 33 fractions, respectively. The pelvic lymph nodes were treated in 41 high-risk patients. Treatment was delivered using cone-beam CT based image-guided radiotherapy (IGRT). Toxicity was assessed prospectively using CTCAE 3.0; biochemical failure was defined according to the Phoenix definition of nadir + 2 ng\u002Fml. Median follow-up of living patients was 50 months. Gastrointestinal (GI) toxicity was mild with > 80 % of the patients free from any GI toxicity during follow-up and no time trend to increased rates or to higher grade of GI toxicity. Two patients suffered from late grade 3 GI toxicity. Acute genitourinary (GU) toxicity grade 1–2 was observed in 85 % of the patients; most patients recovered quickly within 6 weeks after treatment. The rate of GU toxicity grade ≥ 2 was \u003C 10 % at 6–12 month but increased continuously to 22.4 % at 60 months; grade 3 GU toxicity remained below 5 % during follow-up. The 5-year freedom from biochemical failure (FFBF) was 82 % for all patients and 88, 80, and 78 % for low-, intermediate-, and high-risk disease. Favorable FFBF with simultaneously low rates of toxicity was observed after moderately hypofractionated radiotherapy with 2 Gy-equivalent doses ≥ 80 Gy. Conformal IMRT planning and accurate IGRT treatment delivery may have contributed to these results.",{"EN":1005},"Moderately hypofractionated radiotherapy for localized prostate cancer",{"VOID":1007},"Alongi F, Fogliata A, Navarria P et al (2012) Moderate hypofractionation and simultaneous integrated boost with volumetric modulated arc therapy (RapidArc) for prostate cancer. Report of feasibility and acute toxicity. Strahlenther Onkol 188:990–996\nD’Amico AV, Whittington R, Malkowicz SB et al (1998) Biochemical outcome after radical prostatectomy, external beam radiation therapy, or interstitial radiation therapy for clinically localized prostate cancer. Jama 280:969–974\nGuckenberger M, Ok S, Polat B et al (2010) Toxicity after intensity-modulated, image-guided radiotherapy for prostate cancer. Strahlenther Onkol 186:535–543\nShih HA, Harisinghani M, Zietman AL et al (2005) Mapping of nodal disease in locally advanced prostate cancer: rethinking the clinical target volume for pelvic nodal irradiation based on vascular rather than bony anatomy. Int J Radiat Oncol Biol Phys 63:1262–1269\nRoach M 3rd, Marquez C, Yuo HS et al (1994) Predicting the risk of lymph node involvement using the pre-treatment prostate specific antigen and Gleason score in men with clinically localized prostate cancer. Int J Radiat Oncol Biol Phys 28:33–37\nGuckenberger M, Baier K, Richter A et al (2008) Does Intensity Modulated Radiation Therapy (IMRT) prevent additional toxicity of treating the pelvic lymph nodes compared to treatment of the prostate only? Radiat Oncol 3:3\nGuckenberger M, Meyer J, Wilbert J et al (2007) Precision of image-guided radiotherapy (IGRT) in six degrees of freedom and limitations in clinical practice. Strahlenther Onkol 183:307–313\nPolat B, Guenther I, Wilbert J et al (2008) Intra-fractional uncertainties in image-guided intensity-modulated radiotherapy (IMRT) of prostate cancer. Strahlenther Onkol 184:668–673\nBoer HC de, Os MJ van, Jansen PP, Heijmen BJ (2005) Application of the No Action Level (NAL) protocol to correct for prostate motion based on electronic portal imaging of implanted markers. Int J Radiat Oncol Biol Phys 61:969–983\nAl-Mamgani A, Heemsbergen WD, Levendag PC, Lebesque JV (2010) Subgroup analysis of patients with localized prostate cancer treated within the Dutch-randomized dose escalation trial. Radiother Oncol 96:13–18\nDearnaley DP, Sydes MR, Graham JD et al (2007) Escalated-dose versus standard-dose conformal radiotherapy in prostate cancer: first results from the MRC RT01 randomised controlled trial. The lancet oncology 8:475–487\nRoach M 3rd, DeSilvio M, Lawton C et al (2003) Phase III trial comparing whole-pelvic versus prostate-only radiotherapy and neoadjuvant versus adjuvant combined androgen suppression: Radiation Therapy Oncology Group 9413. J Clin Oncol 21:1904–1911\nPommier P, Chabaud S, Lagrange JL et al (2007) Is there a role for pelvic irradiation in localized prostate adenocarcinoma? Preliminary results of GETUG-01. J Clin Oncol 25:5366–5373\nGanswindt U, Schilling D, Muller AC et al (2011) Distribution of prostate sentinel nodes: a SPECT-derived anatomic atlas. Int J Radiat Oncol Biol Phys 79:1364–1372\nMiralbell R, Roberts SA, Zubizarreta E, Hendry JH (2012) Dose-fractionation sensitivity of prostate cancer deduced from radiotherapy outcomes of 5,969 patients in seven international institutional datasets: alpha\u002Fbeta = 1.4 (0.9–2.2) Gy. Int J Radiat Oncol Biol Phys 82:e17–e24\nKupelian PA, Willoughby TR, Reddy CA et al (2007) Hypofractionated intensity-modulated radiotherapy (70 Gy at 2.5 Gy per fraction) for localized prostate cancer: Cleveland Clinic Experience. Int J Radiat Oncol Biol Phys 68:1424–1430\nArcangeli S, Strigari L, Gomellini S et al (2012) Updated results and patterns of failure in a randomized hypofractionation trial for high-risk prostate cancer. Int J Radiat Oncol Biol Phys 84:1172–1178\nThomson D, Merrick S, Swindell R et al (2012) Dose-escalated hypofractionated intensity-modulated radiotherapy in high-risk carcinoma of the prostate: outcome and late toxicity. Prostate Cancer 2012:450246\nZelefsky MJ, Kollmeier M, Cox B et al (2012) Improved clinical outcomes with high-dose image guided radiotherapy compared with non-IGRT for the treatment of clinically localized prostate cancer. Int J Radiat Oncol Biol Phys 84(1):125–129\nBoda-Heggemann J, Lohr F, Wenz F (2011) kV cone-beam CT-based IGRT: a clinical review. Strahlenther Onkol 187:284–291\nAl-Mamgani A, Heemsbergen WD, Peeters ST, Lebesque JV (2009) Role of intensity-modulated radiotherapy in reducing toxicity in dose escalation for localized prostate cancer. Int J Radiat Oncol Biol Phys 73:685–691\nSheets NC, Goldin GH, Meyer AM et al (2012) Intensity-modulated radiation therapy, proton therapy, or conformal radiation therapy and morbidity and disease control in localized prostate cancer. Jama 307:1611–1620\nEngels B, Soete G, Verellen D, Storme G (2009) Conformal arc radiotherapy for prostate cancer: increased biochemical failure in patients with distended rectum on the planning computed tomogram despite image guidance by implanted markers. Int J Radiat Oncol Biol Phys 74:388–391",{"VOID":1009},"10.1007\u002Fs00066-013-0443-x","PUBLICATION","Auto Verify","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs00066-013-0443-x",[1014,1030,1043],{"id":1015,"sortIndex":32,"researcher":28,"roles":1016,"affiliations":1018,"properties":1027,"displayName":1029,"givenName":28,"familyName":28},"8cec490f-5156-49a1-b3b2-1465fbce166b",[1017],"AUTHOR",[1019],{"id":1020,"sortIndex":32,"affiliation":1021,"properties":28},"2b563f0d-e726-4836-85ea-e5ba6f236198",{"id":1020,"createTime":28,"updateTime":28,"relativeEntities":1022,"slug":28,"properties":1023,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1026,"statistic":28},[],{"title":1024},{"VI":1025},"Klinik und Poliklinik für Strahlentherapie, Universitätsklinikum Würzburg, Würzburg, Germany",[],{"title":1028},{"VI":1029},"M. 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The goal of the present study was to evaluate the prognostic value of VEGF single nucleotide polymorphisms (SNPs) and haplotypes for clinical recurrence after definitive radiotherapy for prostate cancer. The association of seven VEGF-A polymorphisms and their haplotypes with clinical recurrence (defined as the occurrence of local recurrence and\u002For distant metastases) in 496 prostate cancer patients treated with definitive radiotherapy were investigated. Genotypes were determined by 5’-nuclease (TaqMan) assays; haplotypes were analyzed using the Haploview program. Within a median follow-up time of 80 months, 44 patients (9 %) developed clinical recurrences. Haplotype analysis showed two separate blocks of high-linkage disequilibrium, formed by five polymorphisms (− 2578C > A, − 2489C > T, − 1498C > T, − 634G > C, − 7C > T) upstream of the coding sequence (CCCCC, ATTGC, CCCGC, ATTGT) and two polymorphisms (936C > T, 1612G > A) downstream of the coding sequence (CA, CG, TG). Carriers of at least 1 copy of the ATTGC haplotype were at higher risk of recurrence (hazard ratio [HR] 3.83; 95 %CI 1.48–9.90, p = 0.006); for carriers of 2 copies, the HR was 4.85 (95 %CI 1.72–13.6; p = 0.003). In multivariate analysis, patients harboring at least one copy of the ATTGC haplotype remained at increased risk of recurrence (HR 3.63, 95 %CI 1.38–9.55, p = 0.009); in patients carrying 2 copies, the HR was 4.72 (95 %CI 1.64–13.6, p = 0.004). Our findings indicate that the VEGF-A ATTGC haplotype may predict clinical recurrence in prostate cancer patients treated with radiotherapy.",{"EN":1121},"Association of genetic variants in VEGF-A with clinical recurrence in prostate cancer patients treated with definitive radiotherapy",{"VOID":1123},"Bayer C, Vaupel P (2012) Acute versus chronic hypoxia in tumors: controversial data concerning time frames and biological consequences. Strahlenther Onkol 188:616–627\nBerns EM, Klijn JG, Look MP et al (2003) Combined vascular endothelial growth factor and TP53 status predicts poor response to tamoxifen therapy in estrogen receptor-positive advanced breast cancer. Clin Cancer Res 9:1253–1258\nBrogan IJ, Khan N, Isaac K et al (1999) Novel polymorphisms in the promoter and 5’ UTR regions of the human vascular endothelial growth factor gene. Hum Immunol 60:1245–1249\nBurstein HJ, Chen YH, Parker LM et al (2008) VEGF as a marker for outcome among advanced breast cancer patients receiving anti-VEGF therapy with bevacizumab and vinorelbine chemotherapy. Clin Cancer Res 14:7871–7877\nDayyani F, Gallick GE, Logothetis CJ et al (2011) Novel therapies for metastatic castrate-resistant prostate cancer. J Natl Cancer Inst 103:1665–1675\nDuque JL, Loughlin KR, Adam RM et al (1999) Plasma levels of vascular endothelial growth factor are increased in patients with metastatic prostate cancer. Urology 54:523–527\nEng L, Azad AK, Habbous S et al (2012) Vascular endothelial growth factor pathway polymorphisms as prognostic and pharmacogenetic factors in cancer: a systematic review and meta-analysis. Clin Cancer Res 18:4526–4537\nErpolat OP, Gocun PU, Akmansu M et al (2013) Hypoxia-related molecules HIF-1α, CA9, and osteopontin: predictors of survival in patients with high-grade glioma. Strahlenther Onkol 189:147–154\nEscudier B, Szczylik C, Porta C et al (2012) Treatment selection in metastatic renal cell carcinoma: expert consensus. Nat Rev Clin Oncol 9:327–337\nFerrara N, Gerber HP, LeCouter J (2003) The biology of VEGF and its receptors. Nat Med 9:669–676\nFontanini G, Boldrini L, Chine S et al (1999) Expression of vascular endothelial growth factor mRNA in non-small-cell lung carcinomas. Br J Cancer 79:363–369\nGarcía-Closas M, Malats N, Real FX et al (2007) Large-scale evaluation of candidate genes identifies associations between VEGF polymorphisms and bladder cancer risk. PLoS Genet 3:e29\nGeorge DJ, Halabi S, Shepard T et al (2001) Prognostic significance of plasma vascular endothelial growth factor levels in patients with hormone-refractory prostate cancer treated on Cancer and Leukemia Group B 9480. Clin Cancer Res 7:1932–1936\nGreen MM, Hiley CT, Shanks JH et al (2007) Expression of vascular endothelial growth factor (VEGF) in locally invasive prostate cancer is prognostic for radiotherapy outcome. Int J Radiat Oncol Biol Phys 67:84–90\nGrothey A, Ellis LM (2008) Targeting angiogenesis driven by vascular endothelial growth factors using antibody-based therapies. Cancer J 14:170–177\nHicklin DJ, Ellis LM (2005) Role of the vascular endothelial growth factor pathway in tumor growth and angiogenesis. J Clin Oncol 23:1011–1027\nJang MJ, Jeon YJ, Kim JW et al (2013) Association of VEGF and KDR single nucleotide polymorphisms with colorectal cancer susceptibility in Koreans. Mol Carcinog 52:E60–69\nKelly WK, Halabi S, Carducci M et al (2012) Randomized, double-blind, placebo controlled phase III trial comparing docetaxel and prednisone with or without bevacizumab in men with metastatic castration-resistant prostate cancer: CALGB 90401. J Clin Oncol 30:1534–1540\nKim DH, Xu W, Kamel-Reid S et al (2010) Clinical relevance of vascular endothelial growth factor (VEGFA) and VEGF receptor (VEGFR2) gene polymorphism on the treatment outcome following imatinib therapy. Ann Oncol 21:1179–1188\nKobayashi T, Liu X, Wen FQ et al (2006) Smad3 mediates TGFbeta 1 induction of VEGF production in lung fibroblasts. Biochem Biophys Res Commun 339:290–295\nKoukourakis MI, Papazoglou D, Giatromanolaki A et al (2004) VEGF gene sequence variation defines VEGF gene expression status and angiogenic activity in non-small cell lung cancer. Lung Cancer 46:293–298\nLangenbacher M, Abdel-Jalil RJ, Voelter W et al (2013) In vitro hypoxic cytotoxicity and hypoxic radiosensitization. Efficacy of the novel 2-nitroimidazole N,N,N-tris[2-(2-nitro-1H-imidazol-1-yl)ethyl]amine. Strahlenther Onkol 189:246–254\nLee JC, Chow NH, Wang ST et al (2000) Prognostic value of vascular endothelial growth factor expression in colorectal cancer patients. Eur J Cancer 36:748–753\nLi A, Gao P, Wang Z et al (2013) Positive association of the vascular endothelial growth factor-A + 405 GG genotype and poor survival in stage I–II gastric cancer in the Northern Chinese population. Mol Biol Rep 40:2741–2748\nMcShane L, Altman DG, Sauerbrei W et al (2005) for the Statistics Subcommittee of the NCI-EORTC Working Group on Cancer diagnostics. REporting recommendations for tumor MARKer prognostic studies (REMARK). Eur J Cancer 41:1690–1696\nMinchenko A, Salceda S, Bauer T et al (1994) Hypoxia regulatory elements of the human vascular endothelial growth factor gene. Cell Mol Biol Res 40:35–39\nOh SY, Kwon HC, Kim SH et al (2013) The relationship of vascular endothelial growth factor gene polymorphisms and clinical outcome in advanced gastric cancer patients treated with FOLFOX: VEGF polymorphism in gastric cancer. BMC Cancer 13:43\nRenner W, Kotschan S, Hoffmann C et al (2000) A common 936 C\u002FT mutation in the gene for vascular endothelial growth factor is associated with vascular endothelial growth factor plasma levels. J Vasc Res 37:443–448\nSchultz A, Lavie L, Hochberg I et al (1999) Interindividual heterogeneity in the hypoxic regulation of VEGF: significance for the development of the coronary artery collateral circulation. Circulation 100:547–552\nSeibold ND, Schild SE, Gebhard MP et al (2013) Prognostic impact of VEGF and FLT-1 receptor expression in patients with locally advanced squamous cell carcinoma of the head and neck. Strahlenther Onkol 189:639–646\nShariat SF, Anwuri VA, Lamb DJ et al (2004) Association of preoperative plasma levels of vascular endothelial growth factor and soluble vascular cell adhesion molecule-1 with lymph node status and biochemical progression after radical prostatectomy. J Clin Oncol 22:1655–1663\nStephens M, Smith NJ, Donnelly P (2001) A new statistical method for haplotype reconstruction from population data. Am J Hum Genet 68:978–989\nStevens A, Soden J, Brenchley PE et al (2003) Haplotype analysis of the polymorphic human vascular endothelial growth factor gene promoter. Cancer Res 63:812–816\nTimke C, Zieher H, Roth A et al (2008) Combination of vascular endothelial growth factor receptor\u002Fplatelet-derived growth factor receptor inhibition markedly improves radiation tumor therapy. Clin Cancer Res 14:2210–2219\nVergis R, Corbishley CM, Norman AR et al (2008) Intrinsic markers of tumour hypoxia and angiogenesis in localised prostate cancer and outcome of radical treatment: a retrospective analysis of two randomised radiotherapy trials and one surgical cohort study. Lancet Oncol 9:342e351\nWatson CJ, Webb NJ, Bottomley MJ et al (2000) Identification of polymorphisms within the vascular endothelial growth factor (VEGF) gene: correlation with variation in VEGF protein production. Cytokine 12:1232–1235\nYamamori M, Sakaeda T, Nakamura T et al (2004) Association of VEGF genotype with mRNA level in colorectal adenocarcinomas. Biochem Biophys Res Commun 325:144–150\nYuan A, Yu CJ, Chen WJ et al (2000) Correlation of total VEGF mRNA and protein expression with histologic type, tumor angiogenesis, patient survival and timing of relapse in non-small-cell lung cancer. Int J Cancer 89:475–483",{"VOID":1125},"10.1007\u002Fs00066-013-0503-2","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs00066-013-0503-2",[1128,1143,1156,1171,1186,1199],{"id":1129,"sortIndex":32,"researcher":28,"roles":1130,"affiliations":1131,"properties":1140,"displayName":1142,"givenName":28,"familyName":28},"67da2d24-9f22-458f-bf9e-847bd6103b5a",[1017],[1132],{"id":1133,"sortIndex":32,"affiliation":1134,"properties":28},"8059d74f-91e6-45e9-b7d4-d3f0b923f731",{"id":1133,"createTime":28,"updateTime":28,"relativeEntities":1135,"slug":28,"properties":1136,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1139,"statistic":28},[],{"title":1137},{"VI":1138},"Department of Therapeutic Radiology and Oncology, Medical University of Graz, Graz, Austria",[],{"title":1141},{"VI":1142},"T. 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Während zu Beginn des letzten Jahrhunderts die Strahlenschutzproblematik vor allem durch die technische Entwicklung im Fach Strahlentherapie bestimmt wurde, hat in den letzten Jahren die Bedeutung des strahlenschutzgerechten Handelns und Wissens der am Behandlungsprozess beteiligten Personen deutlich zugenommen. Eine neue Herausforderung stellt die strahlenschutzgerechte Behandlung des Patienten dar, um das Risikos stochastischer Späteffekte wie radiogener Sekundärtumoren zu reduzieren.",{"EN":1278},"Strahlenschutz in der Radioonkologie",{"VOID":1280},"Beck HR (1988) Röntgen- und Strahlenschutzverordnung – Geschichtliche Entwicklung der Röntgenverordnung. Strahlenschutz Forschung Prax 30:169–179\nBundesanzeiger (2002) Bekanntmachung zur Durchführung der Strahlenschutzverordnung „Strahlenschutz in der Medizin“ – Richtlinie nach der Verordnung über den Schutz vor Schäden durch ionisierende Strahlen (Strahlenschutzverordnung – StrlSchV vom 24. Juni 2002). Bundesministerium der Justiz 54\nRichtlinie 96\u002F29\u002FEURATOM des Rates vom 13. Mai 1996 zur Festlegung der grundlegenden Sicherheitsnormen für den Schutz der Gesundheit der Arbeitskräfte und der Bevölkerung gegen die Gefahren durch ionisierende Strahlungen. Amtsblatt der Europäischen Gemeinschaften Nr. L 159\u002F1, 39. Jahrgang, 29. Juni 1996\nRichtlinie 97\u002F43\u002FEURATOM des Rates vom 30. Juni 1997 über den Gesundheitsschutz von Personen gegen die Gefahren ionisierender Strahlung bei medizinischer Exposition und zur Aufhebung der Richtlinie 84\u002F466\u002FEuratom. Amtsblatt der Europäischen Gemeinschaften Nr. L 180, 40. Jahrgang, 9. Juli 1997\nVerordnung über den Schutz vor Schäden durch ionisierende Strahlen (Strahlenschutzverordnung – StrlSchV) vom 20. Juli 2001 (BGBl. I S. 1714), zuletzt geändert durch die Verordnung zur Änderung strahlenschutzrechtlicher Verordnungen vom 4. Oktober 2011 (BGBl. I S. 2000)\nGEMEINSAMES MINISTERIALBLATT (1979) Reaktorsicherheit, Sicherheit sonstiger kerntechnischer Anlagen, Strahlenschutz. Strahlenschutz für ermächtigte Ärzte. Bundesministerium des Innern 30\nGiesel FO (1900) Über radioaktive Stoffe. Berichte der Deutschen Chemischen Gesellschaft 33:3569–3571\nHolthusen H (1936) Erfahrungen über die Verträglichkeitsgrenze für Röntgenstrahlen und deren Nutzanwendung zur Verhütung von Schäden. Strahlenther 254–269\nHolthusen H, Meyer H, Mollineus W (1959) Ehrenbuch der Röntgenologen und Radiologen aller Nationen. Urban & Schwarzenberg, München. (Blackwell Wissensch., Bln; 3. erw. Aufl. (Mai 1998) ISBN-13: 978-3894121327)\nKärcher KH (1985) Philosophie, Vergangenheit und Gegenwart des Strahlenschutzes in der Strahlentherapie. Georg Thieme, Stuttgart. New York „25 Jahre medizinischer Strahlenschutz – Rückblick und Ausblick zur Entwicklung des Strahlenschutzes“, S 101–106\nKemmer W (1987) Biomedizinische Forschung aus der Sicht des Strahlenschutzes. Georg Thieme, Stuttgart, „19. Jahrestagung der Vereinigten Deutschen Strahlenschutzärzte e. V. vom 2. bis 3. Juni 1987 in Essen“, S 97–102\nLeppin O (1986) Mitteilung. Dtsch Med Wochenschr 22:454\nLorenz W (1951) Strahlentherapie – Systematisches Inhaltsverzeichnis zum 1.–80. Bd. Urban & Schwarzenberg, München\nMaio G (2012) Ärztliche Hilfe als Geschäftsmodell. Dtsch Arztebl 109:692–695\nMakedanz A (1971) Strahlenbelastung und Schutzwirkung einiger Strahlenschutzmittel in der Kontakttherapie. Radiobiol Radiother 12:447\nMödder U, Busch U (1997) Die Augen des Professors – Wilhelm Conrad Röntgen: Eine Kurzbiographie. Publ. des Deutschen Röntgenmuseums, Vergangenheitsverlag, Berlin\nMolineus W (Hrsg) Ehrenbuch der Radiologen aller Nationen, 3. erw. Aufl. Blackwell Wiss, Berlin\nMuth H (1987) Wissenschaftliche Grundlagen des Strahlenschutzes – Stationen und Wendepunkte einer 60-jährigen Entwicklung. Strahlenther Onkol 163:563–572\nN.N (1984) Verordnung über die Gewährleistung von Atomsicherheit und Strahlenschutz. Gesetzbl. I (DDR) 30:341\nN.N (1985) Anordnung über Maßnahmen bei der Therapie mit ionisierender Strahlung. Gesetzbl. I (DDR) 28:322\nRassow J, Scherer E (1970) Strahlenschutz in der Therapie. „Fortschritte im Strahlenschutz – Synopsis über 10 Jahre Forschung“, Georg Thieme, Stuttgart, S 59–88\n(o A) (1992) Richtlinie „Strahlenschutz in der Medizin“, 2. völlig neu bearbeitete und erweiterte Aufl. H. Hoffmann, Berlin\n(o A) (2011) Richtlinie „Strahlenschutz in der Medizin“, Bundesministerium für Umwelt, Naturschutz und Reaktorsicherheit\nSitzlack G (1974) Einführung in den Strahlenschutz. VEB Verlag Volk und Gesundheit Berlin\nSmith IH (1964) Cobalt-60 teletherapy. Hoeber Medical Division Harper & Row, Publishers\nStrahlenschutzkommission (1998) Empfehlung der Strahlenschutzkommission Nachsorge für Patienten nach Strahlenbehandlung\nStrahlenschutzkommission (2010) Empfehlung der Strahlenschutzkommission Strahlenhygienische Anforderungen an IGRT\nStrahlenschutzkommission (2011) Empfehlung der Strahlenschutzkommission Strahlenhygienische Anforderungen an die hochkonformale Strahlentherapie\n(o A) (1973) Strahlenunfall – Tödlicher Filter. Der Spiegel 12\nVerordnung über den Schutz vor der schädigenden Einwirkung ionisierender Strahlung – Strahlenschutzverordnung – vom 26.11.1969 (B GBl. II S. 627)\nVerordnung über den Schutz vor Schäden durch ionisierende Strahlen (Strahlenschutzverordnung – StrlSchV) vom 13. Oktober 1976. BGBI I S. 2905\nVerordnung über den Schutz vor Schäden durch Röntgenstrahlen (Röntgenverordnung – RÖV) vom 1. März 1973. BGB I S. 173\nVogel H (2006) Das Ehrenmal der Radiologie in Hamburg – Ein Beitrag zur Geschichte der Röntgenstrahlen. Forsch Rontgenstr 178:753–756",{"VOID":1282},"10.1007\u002Fs00066-012-0208-y","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs00066-012-0208-y",[1285,1300],{"id":1286,"sortIndex":32,"researcher":28,"roles":1287,"affiliations":1288,"properties":1297,"displayName":1299,"givenName":28,"familyName":28},"0b8a0740-0348-4817-ac15-89066489fb51",[1017],[1289],{"id":1290,"sortIndex":32,"affiliation":1291,"properties":28},"e7222c8e-67aa-46ba-b048-30d45a6ddef1",{"id":1290,"createTime":28,"updateTime":28,"relativeEntities":1292,"slug":28,"properties":1293,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1296,"statistic":28},[],{"title":1294},{"VI":1295},"Dresden, Deutschland",[],{"title":1298},{"VI":1299},"Th. 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Mai 2016)\nRoedel C, Graeven U, Fietkau R et al (2015) Oxaliplatin added to fluorouracilbased preoperative chemoradiotherapy and postoperative chemotherapy of locally advanced rectal cancer (the German CAO\u002FARO\u002FAIO04 study): final results of the multicentre, openlabel, randomised, phase 3 trial. Lancet Oncol 16:979989\nFerreiroNeira I, Torres NE, Liesenfeld LF et al (2016) XPO1 inhibition enhances radiation response in preclinical models of rectal cancer. Clin Cancer Res 22:16631673\nAbdul Razak AR, MauSoerensen M, Gagrail MY et al (2016) First-in-class, first-in-human phase I study of selinexor, a selective inhibtor of nuclear export, in patients with advanced solid tumors. J Clin Oncol JCO653949. http:\u002F\u002Fwww.awmf.org\u002Fuploads\u002Ftx_szleitlinien\u002F021007OLk_S3_KRK_201408_01.pdf\nGao J, Azmi AS, Aboukameel A et al (2014) Nuclear retention of Fbw7 by specific inhibitors of nuclear export leads to Notch1 degradation in pancreatic cancer. Oncotarget 5:34443454\nCapalbo G, Rödel C, Stauber RH et al (2007) The role of survivin for radiation therapy. Strahlenther Onkol 183:593599\nKwon YW, Kim IJ, Wu D et al (2012) Pten regulates AuroraA and cooperates with Fbxw7 in modulating radiationinduced tumor development. Mol Cancer Res 10:834844",{"VOID":1382},"10.1007\u002Fs00066-016-1060-2","http:\u002F\u002Flink.springer.com\u002F10.1007\u002Fs00066-016-1060-2",[1385,1400],{"id":1386,"sortIndex":32,"researcher":28,"roles":1387,"affiliations":1388,"properties":1397,"displayName":1399,"givenName":28,"familyName":28},"27ff5977-6e9e-43eb-bbb6-dd884eb2e299",[1017],[1389],{"id":1390,"sortIndex":32,"affiliation":1391,"properties":28},"1af8bcb6-5754-495a-9087-2e34315d40ca",{"id":1390,"createTime":28,"updateTime":28,"relativeEntities":1392,"slug":28,"properties":1393,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1396,"statistic":28},[],{"title":1394},{"VI":1395},"Institut für Physiologie, Universität zu Lübeck, Lübeck, Deutschland",[],{"title":1398},{"VI":1399},"Moritz von Fallois",{"id":1401,"sortIndex":40,"researcher":28,"roles":1402,"affiliations":1403,"properties":1410,"displayName":1412,"givenName":28,"familyName":28},"0d43ed1c-26e5-4e33-a52d-7943c5e17e1d",[1017],[1404],{"id":1390,"sortIndex":32,"affiliation":1405,"properties":28},{"id":1390,"createTime":28,"updateTime":28,"relativeEntities":1406,"slug":28,"properties":1407,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1409,"statistic":28},[],{"title":1408},{"VI":1395},[],{"title":1411},{"VI":1412},"Reinhard 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assess the late effect of a prostaglandin, given rectally during irradiation, on late rectal toxicity. In the acute treatment setting no significant differences in reducing the incidence of acute proctitis symptoms in patients receiving misoprostol, however, significantly more rectal bleeding had been reported. A total of 100 patients who had undergone radiotherapy for prostate cancer had been entered into this phase III randomized, placebo-controlled, double-blind study with misoprostol or placebo suppositories. The toxicity was evaluated yearly after cessation of irradiation by the RTOG\u002FLENT-SOMA scale. The median follow-up was 50 months. 20 patients suffered from grade 1, four patients from grade 2 as well, and three patients only from grade 2 toxicity. Frequency, bleeding and urgency were the most commonly reported symptoms. In keeping with other studies and clinical experience, the symptoms peaked within the first 2 years with a median for grade 1 of 13 months and for grade 2 of 15 months. The presence of acute toxicity grade 2 showed a correlation with the development of any late toxicity (p = 0.03). Any acute rectal bleeding was significant correlated with any late rectal bleeding (p = 0.017). Misoprostol given as once-daily suppository for prevention of acute radiation-induced proctitis does neither influence the incidence and severity of radiation-induced acute nor late rectal toxicity. Misoprostol has no negative impact on the incidence and severity of late rectal bleeding, in contrast to acute rectal bleeding. The routine clinical use of misoprostol suppositories cannot be recommended.",{"EN":1478},"Effect of a Prostaglandin – Given Rectally for Prevention of Radiation-Induced Acute Proctitis – on Late Rectal Toxicity",{"VOID":1480},"Arcangeli S, Saracino B, Petrongari MG, et al. Analysis of toxicity in patients with high risk prostate cancer treated with intensity-modulated pelvic radiation therapy and simultaneous integrated dose escalation to prostate area. Radiother Oncol 2007;84:148–55.\nBen-Josef E, Han S, Tobi M, et al. A pilot study of topical intrarectal application of amifostine for prevention of late radiation rectal injury. Int J Radiat Oncol Biol Phys 2002;53:1160–4.\nBen-Josef E, Han S, Tobi M, et al. Intrarectal application of amifostine for the prevention of radiation-induced rectal injury. Semin Radiat Oncol 2002;12:Suppl 1:81–5.\nCox JD, Stetz J, Pajak TF. Toxicity criteria of the Radiation Therapy Oncology Group (RTOG) and the European Organization for Research and Treatment of Cancer (EORTC). Int J Radiat Oncol Biol Phys 1995;31:1341–6.\nDelaney JP, Bonsack ME, Felemovicius I. Misoprostol in the intestinal lumen protects against radiation injury of the mucosa of the small bowel. Radiat Res 1994;137:405–9.\nEl-Bassiouni M, Davis JB, El-Attar I, et al. Target motion variability and on-line positioning accuracy during external-beam radiation therapy of prostate cancer with an endorectal balloon device. Strahlenther Onkol 2006;182:531–6.\nElsayed H, Bölling T, Moustakis C, et al. Organ movements and dose exposures in teletherapy of prostate cancer using a rectal balloon. Strahlenther Onkol 2007;183:617–24.\nEmpey LR, Papp JD, Jewell LD, et al. Mucosal protective effects of vitamin E and misoprostol during acute radiation-induced enteritis in rats. Dig Dis Sci 1992;37:205–14.\nGilbertson TJ, Ruwart MJ, Stryd RP, et al. Partial characterization of the gastrointestinal weight changes produced in the female rat by 16,16-dimethylprostaglandin E2. Prostaglandins 1983;26:745–59.\nHanson WR, DeLaurentiis K. Comparison of in vivo murine intestinal radiation protection by E-prostaglandins. Prostaglandins 1987;33:Suppl:93–104.\nHanson WR, Houseman KA, Collins PW. Radiation protection in vivo by prostaglandins and related compounds of the arachidonic acid cascade. Pharmacol Ther 1988;39:347–56.\nHanson WR, Houseman KA, Nelson AK, et al. Radiation protection of the murine intestine by misoprostol, a prostaglandin E1 analogue, given alone or with WR-2721, is stereospecific. Prostaglandins Leukot Essent Fatty Acids 1988;32:101–5.\nHanson WR, Marks JE, Reddy SP, et al. Protection from radiation-induced oral mucositis by a mouth rinse containing the prostaglandin E1 analog, misoprostol: a placebo controlled double blind clinical trial. Adv Exp Med Biol 1997;400B:811–8.\nHanson WR, Thomas C. 16,16-dimethyl prostaglandin E2 increases survival of murine intestinal stem cells when given before photon radiation. Radiat Res 1983;96:393–8.\nHenriksson R, Franzen L, Littbrand B. Effects of sucralfate on acute and late bowel discomfort following radiotherapy of pelvic cancer. J Clin Oncol 1992;10:969–75.\nHille A, Schmidberger H, Hermann RM, et al. A phase III randomized, placebo-controlled, double-blind study of misoprostol rectal suppositories to prevent acute radiation proctitis in patients with prostate cancer. Int J Radiat Oncol Biol Phys 2005;63:1488–93.\nInternational Commission on Radiation Units and Measurements. ICRU report 50: prescribing, recording, and reporting photon beam therapy. Washington: International Commission on Radiation Units and Measurements, 1993.\nKeelan M, Thomson AB, Clandinin MT, et al. Effect of oral enprostil, a synthetic prostaglandin E2, on intestinal brush border membrane lipid composition following abdominal irradiation in the rat. Clin Invest Med 1989;12:350–6.\nKhan AM, Birk JW, Anderson JC, et al. A prospective randomized placebo-controlled double-blinded pilot study of misoprostol rectal suppositories in the prevention of acute and chronic radiation proctitis symptoms in prostate cancer patients. Am J Gastroenterol 2000;95:1961–6.\nKneebone A, Mameghan H, Bolin T, et al. The effect of oral sucralfate on the acute proctitis associated with prostate radiotherapy: a double-blind, randomized trial. Int J Radiat Oncol Biol Phys 2001;51:628–35.\nKoper JC, Stroom JC, van Puten WL, et al. Acute morbidity reduction using 3DCRT for prostate carcinoma: a randomized study. Int J Radiat Oncol Biol Phys 1999;43:727–34.\nKouvaris J, Kouloulias V, Malas E, et al. Amifostine as radioprotective agent for the rectal mucosa during irradiation of pelvic tumors. A phase II randomized study using various toxicity scales and rectosigmoidoscopy. Strahlenther Onkol 2003;179:167–74.\nLENT SOMA scales for all anatomic sites. Int J Radiat Oncol Biol Phys 1995;31:1049–91.\nLin C, Turner S, Mai T, et al. Late rectal and urinary toxicity from conformal, dose-escalated radiation therapy for prostate cancer: a prospective study of 402 patients. Australas Radiol 2007;51:578–83.\nMartenson JA, Bollinger JW, Sloan JA, et al. Sucralfate in the prevention of treatment-induced diarrhea in patients receiving pelvic radiation therapy: A North Central Cancer Treatment Group phase III double-blind placebo-controlled trial. J Clin Oncol 2000;18:1239–45.\nMichalski JM, Purdy JA, Winter K, et al. Preliminary report of toxicity following 3D radiation therapy for prostate cancer on 3DOG\u002FRTOG 9406. Int J Radiat Oncol Biol Phys 2000;46:391–402.\nMontana GS, Anscher MS, Mansbach CM II, et al. Topical application of WR-2721 to prevent radiation-induced proctosigmoiditis. A phase I\u002FII trial. Cancer 1992;69:2826–30.\nO’Brien PC, Franklin CI, Dear KB, et al. A phase III double-blind randomised study of rectal sucralfate suspension in the prevention of acute radiation proctitis. Radiother Oncol 1997;45:117–23.\nPinkawa M, Siluschek J, Gagel B, et al. Postoperative radiotherapy for prostate cancer: evaluation of target motion and treatment techniques (intensity-modulated versus conformal radiotherapy). Strahlenther Onkol 2007;183:23–9.\nRauch K, Weiland H. Therapy of radiogenic colitis with salicylazosulfapyridine (azulfidine) Strahlentherapie 1972;143:660–3.\nReinhart WH, Muller O, Halter F. Influence of long-term 16,16-dimethyl prostaglandin E2 treatment on the rat gastrointestinal mucosa. Gastroenterology 1983;85:1003–10.\nSanguineti G, Cavey ML, Endres EJ, et al. Does treatment of the pelvic nodes with IMRT increase late rectal toxicity over conformal prostate-only radiotherapy to 76 Gy? Strahlenther Onkol 2006;182:543–9.\nSanguineti G, Franzone P, Marcenaro M, et al. Sucralfate versus mesalazine versus hydrocortisone in the prevention of acute radiation proctitis during conformal radiotherapy for prostate carcinoma. A randomized study. Strahlenther Onkol 2003;179:464–70.\nSchultheiss TE, Lee WR, Hunt MA, et al. Late GI and GU complications in the treatment of prostate cancer. Int J Radiat Oncol Biol Phys 1997;37:3–11.\nShrader-Bogen CL, Kjellberg JL, McPherson CP, et al. Quality of life and treatment outcomes: prostate carcinoma patients’ perspectives after prostatectomy or radiation therapy. Cancer 1997;79:1977–86.\nSimone NL, Ménard C, Soule BP, et al. Intrarectal amifostine during external beam radiation therapy for prostate cancer produces significant improvements in quality of life measured by EPIC score. Int J Radiat Oncol Biol Phys 2008;70:90–5.\nSingh AK, Ménard C, Guion P, et al. Intrarectal amifostine suspension may protect against acute proctitis during radiation therapy for prostate cancer: a pilot study. Int J Radiat Oncol Biol Phys 2006;65:1008–13.\nStorey MR, Pollack A, Zagars G, et al. Complications from radiotherapy dose escalation in prostate cancer: preliminary results of a randomized trial. Int J Radiat Oncol Biol Phys 2000;48:635–42.\nSzepesi S, Jakobi V, Vecsei P, et al. Treatment of radiogenic colitis with a rectal foam containing cortisol. Clinical and pharmacologic data. Strahlenther Onkol 1990;166:271–4.\nThomson AB, Keelan M, Clandinin MT, et al. Lack of protective effect of oral enprostil, a synthetic prostaglandin E2, on intestinal transport and morphology following abdominal irradiation in the rat. Can J Physiol Pharmacol 1989;67:1351–6.\nTrotti A, Byhardt R, Stetz J, et al. Common Toxicity Criteria: version 2.0. an improved reference for grading the acute effects of cancer treatment: impact on radiotherapy. Int J Radiat Oncol Biol Phys 2000;47:13–47.\nWeiss E, Hirnle P, Arnold-Bofinger H, et al. Therapeutic outcome and relation of acute and late side effects in the adjuvant radiotherapy of endometrial carcinoma stage I and II. Radiother Oncol 1999;53:37–44.\nWeiss W, Horninger W, Forthuber BC, et al. Single-institution results of primary external-beam radiation for the treatment of T1–T3 prostate cancer. Strahlenther Onkol 2007;183:321–6.\nWertz H, Lohr F, Dobler B, et al. Dosimetric impact of image-guided translational isocenter correction for 3-D conformal radiotherapy of the prostate. Strahlenther Onkol 2007;183:203–10.\nWidmark A, Fransson P, Tavelin B, et al. Self-assessment questionnaire for evaluating urinary and intestinal late side effects after pelvic radiotherapy in patients with prostate cancer compared with an age-matched control population. Cancer 1994;74:2520–32.\nZelefsky MJ, Levin EJ, Hunt M, et al. Incidence of late rectal and urinary toxicities after three-dimensional conformal radiotherapy and intensity-modulated radiotherapy for localized prostate cancer. Int J Radiat Oncol Biol Phys 2008;70:1124–9.",{"VOID":1482},"10.1007\u002Fs00066-009-1978-8","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs00066-009-1978-8",[1485,1500,1513,1528,1541,1554,1569,1584,1597],{"id":1486,"sortIndex":32,"researcher":28,"roles":1487,"affiliations":1488,"properties":1497,"displayName":1499,"givenName":28,"familyName":28},"9a17687c-7c64-4639-8ed2-65d51ca33187",[1017],[1489],{"id":1490,"sortIndex":32,"affiliation":1491,"properties":28},"5c9f902b-3bde-4c12-bfd9-d28320d34e7e",{"id":1490,"createTime":28,"updateTime":28,"relativeEntities":1492,"slug":28,"properties":1493,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1496,"statistic":28},[],{"title":1494},{"VI":1495},"Department of Radiotherapy and Radiooncology, University of Göttingen, Göttingen, Germany",[],{"title":1498},{"VI":1499},"Tereza Kertesz",{"id":1501,"sortIndex":40,"researcher":28,"roles":1502,"affiliations":1503,"properties":1510,"displayName":1512,"givenName":28,"familyName":28},"87b5043f-bed4-43f0-bb27-b5b50df2bc96",[1017],[1504],{"id":1490,"sortIndex":32,"affiliation":1505,"properties":28},{"id":1490,"createTime":28,"updateTime":28,"relativeEntities":1506,"slug":28,"properties":1507,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1509,"statistic":28},[],{"title":1508},{"VI":1495},[],{"title":1511},{"VI":1512},"Markus K. 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Ewing’s sarcoma arising in the head and neck region is an extremely rare malignant neoplasm. We describe the unusual case of a tumor originating in the larynx of a 68-year-old male with hoarseness and occasional aphonia. We report successful treatment with sequential chemo- and radiotherapy. Complete regression and larynx preservation with voice function recovery was achieved. To our knowledge, this is the first report of this type of tumor in the larynx with cartilage invasion that documents the effectiveness of radiotherapy as an alternative to surgical management. At present, after 30 months of follow-up, the patient is free of tumor relapse and in very good condition.",{"EN":1684},"Ewing’s sarcoma of the larynx",{"VOID":1686},"Whaley JT, Indelicato DJ, Morris CG et al (2010) Ewing tumors of the head and neck. Am J Clin Oncol 33(4):321–326\nAllam A, El-Husseiny G, Khafaga Y et al (1999) Ewing’s sarcoma of the head and neck: a retrospective analysis of 24 cases. Sarcoma 3:11–15\nGreve B, Sheikh-Mounessi F, Kemper B et al (2012) Survivin, a target to modulate the radiosensitivity of Ewing’s sarcoma. Strahlenther Onkol 188(11):1038–1047\nOsborn HA, Wehrli B, Franklin JH et al (2011) Ewing’s sarcoma of the masseter muscle. J Laryngol Otol 125:978–981\nAgir H, Brasch HD, Tan ST (2007) Extra-skeletal Ewing’s sarcoma of the submandibular gland. J Plast Reconstr Aesthet Surg 60:1345–1348\nYang YS, Hong KH (2004) Extraskeletal Ewing’s sarcoma of the larynx. J Laryngol Otol 118:62–64\nUshigome S, Machinami R, Sorensen PH (2002) Ewing sarcoma\u002FPrimitive neuroectodermal tumour (PNET). In: Fletcher CDM, Unni KK, Mertens F (eds) World Health Organisation Classification of Tumours. Pathology and genetics of tumours of soft tissue and bone. IARC Press, Lyon, S 298–300\nFletcher CDM (2007) Peripheral neuroectodermal tumors. In: Fletcher CDM (ed) Diagnostic Histopathology of Tumors, 3rd edn. Churchill Livingstone, Philadelphia, S 1750–1752\nCoffin CM, Belchis D (2006) Immunohistology of Pediatric Neoplasms. In: Dabbs D. Diagnostic immunohistochemistry. 2nd edn. Churchill Livingstone, Philadelphia, S 612–614\nZagar TM, Triche TJ, Kinsella T (2008) Extraosseus Ewing’s sarcoma: 25 years later. J Clin Oncol 26:4230–4232\nUllmann C, Beck JD, Holter W et al (2008) Long-term results following multidisciplinary treatment of localized Ewing’s sarcoma in children and adolescents. Strahlenther Onkol 184(3):137–144\nEary J (2009) Sarcomas. In: Wahl RL (ed) Principles and practice of PET and PET\u002FCT. Wolters Kluwer ǀ Lippincott Williams & Wilkins, Philadelphia, S 392–401",{"VOID":1688},"10.1007\u002Fs00066-013-0356-8","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs00066-013-0356-8",[1691,1706,1719,1734,1749],{"id":1692,"sortIndex":32,"researcher":28,"roles":1693,"affiliations":1694,"properties":1703,"displayName":1705,"givenName":28,"familyName":28},"21ed6e34-04a0-49ea-b144-78963aee3b3c",[1017],[1695],{"id":1696,"sortIndex":32,"affiliation":1697,"properties":28},"b74f5851-4ddc-4c18-bee5-936c4208039b",{"id":1696,"createTime":28,"updateTime":28,"relativeEntities":1698,"slug":28,"properties":1699,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1702,"statistic":28},[],{"title":1700},{"VI":1701},"Department of Radiation and Clinical Oncology, Maria Sklodowska-Curie Memorial Cancer Center and Institute of Oncology, Gliwice Branch, Gliwice, Poland",[],{"title":1704},{"VI":1705},"A. 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