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Journal of Medicine and Pharmacy","Tạp chí Y Dược học Cần Thơ",{"EN":487,"VI":488},"\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">04\u002F10\u002F2015 Ministry of Information and Communications allowed Can Tho journal of medicine and pharmacy to operate (102 \u002FGP-BTTTT)\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">07\u002F16\u002F2015 Can Tho journal of medicine and pharmacy is internationally recognized: ISSN 2354-1210\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">In 2016, The journal has been included in the list of medical science journals by The State Council for professorship which is awarded a work score of 0-0.5 points for a published article.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Can Tho Journal of Medicine and Pharmacy welcome original works that haven’t been submitted or published in other medical journals. Posts must contain content related to one of the journal’s categories.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The content published\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The journal is divided into 3 categories:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Scientific research article: are valuable scientific works, which have been researched and accepted.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Overview of medicine, biology and pharmacy: serving the objective of continuing training in the fields of medicine, biology and pharmacy; to systematize classical and modern knowledge.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Update information on new knowledge about medicine, biology, pharmacy in the country and in the world.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Scope\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Publication and introduction of scientific research in the fields:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Medicine (internal medicine, surgery, pediatrics, obstetrics and gynecology, odonto-stomatology, laboratory, oncology, traditional medicine, nursing).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Biology (genetics, biotechnology).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Pharmacology (pharmaceutics, drug quality analysis-control, synthetic pharmaceutical chemistry, biochemistry, pharmacognosy, botany, clinical pharmacy).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- To enhance the quality of undergraduate, postgraduate education, scientifically researching and meet the necessary treatment in hospital.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Introducing the updated domestic and oversea information about science technology to promote scientific research and exchanging technology in local, other universities.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Exchanging pharmaceutical and medical information for social health developing in the Mekong Delta and Vietnam.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The object\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Postgraduate students, student of Can Tho University of Medicine and Pharmacy, scientists from schools, research institutes, hospitals, health centers, pharmaceutical companies of the Mekong Delta; other provinces and regions in Vietnam and other country.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Address\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Headquarters of Can Tho Journal of Medicine and Pharmacy, located Scientific Research and International Cooperation Office: 179 Nguyen Van Cu Street, An Khanh Ward, Ninh Kieu District, Can Tho City, Vietnam.\u003C\u002Fspan>\u003C\u002Fp>","\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Ngày 16\u002F7\u002F2015, Tạp chí Y Dược học Cần Thơ được cấp chỉ số quốc tế: ISSN 2354-1210.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 4\u002F2016, Tạp chí đã được Hội đồng Giáo sư ngành Y đưa vào danh sách các tạp chí khoa học Y học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Năm 2020 Tạp chí Y Dược học Cần Thơ đã được phê duyệt vào danh mục của các Hội đồng Giáo sư ngành Dược học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ ra 12 số\u002Fnăm, 180-200 trang\u002Fsố.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 12\u002F2022 Tạp chí Y Dược học Cần Thơ là thành viên của hệ thống Crossref và từ tháng 01\u002F2023 tạp chí thực hiện bình duyệt online kín 2 chiều nhằm tăng tính minh bạch, tin cậy của các công trình nghiên cứu khoa học và đảm bảo tốt nhất chất lượng khoa học của bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ, mục đích và phạm vi của tạp chí\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ và mục đích hoạt động của tạp chí: xuất bản nhằm mục đích phổ biến kết quả từ các đề tài nghiên cứu khoa học; giao lưu trao đổi khoa học, chia sẻ kinh nghiệm, học tập, đồng thời cập nhật thông tin khoa học mới trong các lĩnh vực y, sinh, dược học trong và ngoài nước.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phạm vi của tạp chí: Tạp chí xuất bản được chia thành 3 chuyên mục: (i) Bài báo nghiên cứu khoa học là kết quả công trình nghiên cứu khoa học có giá trị đã được triển khai nghiên cứu, (ii) Bài tổng quan y, sinh, dược học: phục vụ mục tiêu đào tạo liên tục trong lĩnh vực y, sinh, dược học; nhằm hệ thống hóa những kiến thức kinh điển và hiện đại; (iii) Thông tin cập nhật kiến thức mới về y, sinh, dược học trong nước và trên thế giới.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Chính sách truy cập mở\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ áp dụng chính sách truy cập mở đối với các bài báo đã xuất bản đến với độc giả, nhằm mở rộng cơ hội tiếp cận các kết quả nghiên cứu chất lượng cao và tăng cường trao đổi kiến thức. Tạp chí đăng tải trực tuyến (miễn phí) toàn văn các bài báo được công bố trên website của Tạp chí (https:\u002F\u002Ftapchi.ctump.edu.vn).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đạo đức xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ cam kết tuân thủ đạo đức xuất bản phù hợp với các hướng dẫn và tiêu chuẩn của the Committee on Publication Ethics (COPE), tuân thủ các nguyên tắc của COPE’s Core Practices, Best Practices Guidelines for Journal Editors và Guidelines on Good Publication Practices.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Bản thảo bài báo chỉ được chấp nhận khi được tác giả chịu trách nhiệm chính cam kết các nội dung sau: Các nội dung của bản thảo chưa được đăng tải toàn bộ hoặc một phần ở các tạp chí khác; Tất cả các tác giả đều có đóng góp một cách đáng kể vào quá trình nghiên cứu hoặc chuẩn bị bản thảo và cùng chịu trách nhiệm về các nội dung của bản thảo; Tuân thủ các biện pháp đảm bảo đạo đức nghiên cứu (ví dụ thỏa thuận đồng ý tham gia nghiên cứu).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Cam kết bảo mật\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí cam kết thực hiện và tuân thủ các quy định của luật và các văn bản hướng dẫn liên quan đến bảo mật thông tin cá nhân trên không gian mạng. Các thông tin mà người dùng (tác giả, độc giả, biên tập viên, người phản biện) nhập vào các biểu mẫu trên Hệ thống Quản lý xuất bản trực tuyến của tạp chí chỉ được sử dụng vào các mục đích đã được tuyên bố rõ ràng và sẽ không được cung cấp cho bất kỳ bên thứ ba nào khác, hay dùng vào bất kỳ mục đích nào khác.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phí gửi bài\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng bài: 1.000.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng nhanh: 1.500.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với tác giả là cán bộ viên chức thuộc Trường Đại học Y Dược Cần Thơ thì được hỗ trợ 50% lệ phí gửi đăng bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với sinh viên thực hiện đề tài nghiên cứu khoa học cấp trường được hỗ trợ 100% lệ phí đăng bài ( Tác giả gửi đính kèm “ Quyết định về việc giao tổ chức thực hiện đề tài nghiên cứu khoa học cấp Trường của sinh viên”).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Hình thức nộp lệ phí:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Tiền mặt:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Nộp trực tiếp tại Phòng Tài chính - Kế toán, Trường Đại học Y Dược Cần Thơ, số 179 Nguyễn Văn Cừ, P. An Khánh, Q. Ninh Kiều, thành phố Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Chuyển khoản:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tên Tài khoản: Trường ĐHYD Cần Thơ, Số TK: 0111000115668, tại ngân hàng Vietcombank chi nhánh Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Thời gian: Áp dụng từ ngày 01\u002F02\u002F2023.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">* Phí gửi bài không được hoàn trả khi bài viết bị từ chối hoặc tác giả xin rút bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Quy trình phản biện bài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ thực hiện quy trình phản biện kín hai chiều nghiêm ngặt. Danh tính của những người phản biện không được tiết lộ cho các tác giả và ngược lại. Quy trình thẩm định bài báo đăng gồm các bước sau:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tiếp nhận bản thảo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tác giả liên hệ gửi bản thảo đến Tạp chí qua hệ thống trực tuyến tại website: https:\u002F\u002Ftapchi.ctump.edu.vn. Hướng dẫn về cách đăng ký, gửi bài và chuẩn bị bản thảo được cung cấp trên website của Tạp chí.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sàng lọc sơ bộ\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sau khi Tòa soạn nhận được bài báo của tác giả, Ban Thư ký sẽ tiến hành kiểm tra sơ bộ bài báo (các yêu cầu về nội dung và hình thức). Những bài báo không đúng quy cách hoặc có nội dung không phù hợp hoặc vi phạm bản quyền sẽ bị từ chối (Ban Thư ký thông báo phản hồi đến tác giả trong vòng 1 tuần). Những bài báo đủ điều kiện, được Ban Thư ký tòa soạn chuyển đến Ban Biên tập có cùng chuyên môn với nội dung bài báo để đề xuất người phản biện. Thời gian kể từ khi Ban Biên tập nhận bài báo đến khi đề xuất người phản biện bài báo chậm nhất là 5 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Vòng phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký gửi bài và yêu cầu phản biện đến 02 phản biện độc lập.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Các phản biện gởi nhận xét cho Ban Thư ký. Thời gian từ khi gửi bài cho phản biện đến khi nhận ý kiến của phản biện tối đa là 20 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xử ký kết quả phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Nếu ý kiến đồng ý cho đăng và không cần chỉnh sửa, Ban Thư ký tiếp tục đăng bài theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Nếu ý kiến đồng ý đăng và cần chỉnh sửa, Ban Thư ký sẽ thông tin đến tác giả chỉnh sửa theo yêu cầu của người phản biện. Thời gian chỉnh sửa và gửi lại kéo dài không quá 2 tuần, từ khi tác giả bài báo nhận được thông tin (Quá trình này có thể lặp lại tối đa 2 lần\u002F1 bài báo). Khi có sự thống nhất, đồng ý của người phản biện; bài báo được tiếp tục đăng theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Những bài báo có chất lượng không đạt yêu cầu, cả 2 phản biện không đồng ý cho đăng sẽ bị Tòa soạn từ chối đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký tổng hợp các bản thảo đã được tác giả hoàn thiện sau thẩm định trình Ban Biên tập xem xét, Tổng Biên tập phê duyệt, quyết định bài đăng theo các tiêu chí: sự phù hợp nội dung với tôn chỉ và mục đích, thể loại bài viết (ưu tiên các bài có bài có nghiên cứu chuyên sâu, hàm lượng khoa học cao), đóng góp mới bài báo, bài báo được ưu tiên đăng trong số gần nhất của Tạp chí theo thứ tự: tính thời sự, chất lượng bài báo và thời gian gửi bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Ban Biên tập và Ban Thư ký biên tập bản thảo, chế bản, đọc rà soát lỗi. Thời gian hoàn thành từ 10-15 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Ban Thư ký có trách nhiệm thông báo cho tác giả bài báo (bằng e-mail) về tình hình phê duyệt bài báo, thời gian, số kỳ, tập xuất bản bài báo theo qui định.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">4. Danh sách bài báo theo số Tạp chí được in ấn và phát hành trong năm định kỳ được công bố chính thức trên website: https:\u002F\u002Ftapchi.ctump.edu.vn\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>",{"VOID":490},"wcQ1uqwAAAAJ","2023-05-30T08:17:21.868+00:00",[],[494],{"id":495,"createTime":28,"updateTime":28,"relativeEntities":496,"slug":28,"properties":497,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":507,"parentIds":508,"statistic":28},"6413896b-eca9-442b-a73f-182a58a0ce40",[],{"title":498,"address":501,"country":504,"abbreviation":505},{"EN":499,"VI":500},"Can Tho University of Medicine and Pharmacy","Trường Đại học Y Dược Cần Thơ",{"EN":502,"VI":503},"No 179, Nguyen Van Cu street, An Khanh ward, Ninh Kieu district, Can Tho city, Vietnam","Số 179, đường Nguyễn Văn Cừ, phường An Khánh, quận Ninh Kiều, thành phố Cần Thơ, Việt Nam",{"VOID":15},{"VOID":506},"ctump","http:\u002F\u002Fwww.ctump.edu.vn\u002F",[],[],"https:\u002F\u002Ftapchi.ctump.edu.vn\u002Findex.php\u002Fctump",{"impactFactor":32,"impactFactorByYear":512,"i10Index":32,"i10IndexLast5Year":32,"totalPublication":514,"totalPublicationByYear":515,"totalCitation":520,"totalCitationByYear":521,"totalCitationPerPublication":108,"totalCitationPerPublicationByYear":523,"hindexLast5Year":45,"hindex":45},{"2022":513,"2023":111,"2024":106},0.01,1556,{"2020":47,"2021":516,"2022":517,"2023":518,"2024":519,"2025":122},57,306,801,358,161,{"2021":146,"2022":280,"2023":522},99,{"2021":524,"2022":318,"2023":104},0.23,{"impactFactor":28,"impactFactorByYear":28,"i10Index":123,"i10IndexLast5Year":123,"totalPublication":526,"totalPublicationByYear":527,"totalCitation":526,"totalCitationByYear":528,"totalCitationPerPublication":40,"totalCitationPerPublicationByYear":531,"hindexLast5Year":49,"hindex":49},476,{"0":205,"2019":123,"2021":139,"2022":459,"2023":451,"2024":357,"2025":49,"2026":48},{"2021":42,"2022":123,"2023":161,"2024":529,"2025":360,"2026":530},136,83,{"2021":105,"2022":513,"2023":532,"2024":127,"2025":533,"2026":534},0.62,25.43,13.83,{"id":536,"createTime":537,"updateTime":382,"relativeEntities":538,"slug":539,"properties":540,"entityType":25,"verifyStatus":26,"verifyTime":28,"verifyNote":28,"languages":552,"translateLanguages":28,"viewCount":133,"subjectFields":553,"manageAffiliations":554,"indexDatabases":555,"url":556,"thumbnailPath":557,"statistic":558,"gsStatistic":594,"type":55,"analyzePriority":28},"6984a56a-db70-403b-9cc4-4013e1ceaffa","2023-05-09T06:47:40.346+00:00",[],"T%E1%BA%A1p%20ch%C3%AD%20Nghi%C3%AAn%20c%E1%BB%A9u%20n%C6%B0%E1%BB%9Bc%20ngo%C3%A0i",{"country":541,"issn":542,"title":544,"introduce":547,"gsId":550},{"VOID":15},{"VOID":543},"25252445",{"EN":545,"VI":546},"VNU Journal of Foreign Studies","Tạp chí Nghiên cứu nước ngoài",{"EN":548,"VI":549},"{\"ops\":[{\"insert\":\"\\n\\nThe \\n\"},{\"attributes\":{\"italic\":true},\"insert\":\"VNU Journal of Science\"},{\"insert\":\"\\n was established in 1985 for the publication of national and international research papers in all fields of natural sciences and technology, social sciences and humanities. 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Hyg., 68, 23, 10.4269\u002Fajtmh.2003.68.23\nAhlbom, 1994, Neonatal exposure to a type-I pyrethroid (bioallethrin) induces dose-response changes in brain muscarinic receptors and behaviour in neonatal and adult mice, Brain Res., 645, 318, 10.1016\u002F0006-8993(94)91666-7\nAziz, 2001, Neurodevelopmental consequences of gestational exposure (GD14-GD20) to low dose deltamethrin in rats, Neurosci. Lett., 300, 161, 10.1016\u002FS0304-3940(01)01543-9\nImamura, 2002, Neonatal exposure of newborn mice to pyrethroid (permethrin) represses activity-dependent c-fos mRNA expression in cerebellum, Arch. Toxicol., 76, 392, 10.1007\u002Fs00204-002-0358-2\nJin, 2014, Exposure to bifenthrin causes immunotoxicity and oxidative stress in male mice, Environ. Toxicol., 29, 991, 10.1002\u002Ftox.21829\nMoniz, 1990, Effects of exposure to a pyrethroid insecticide during lactation on the behavior of infant and adult rats, Braz. J. Med. Biol. Res., 23, 45\nPrater, 2002, Single-dose topical exposure to the pyrethroid insecticide, permethrin in C57BL\u002F6N mice: effects on thymus and spleen, Food. Chem. Toxicol., 40, 1863, 10.1016\u002FS0278-6915(02)00163-1\nShafer, 2008, Complete inhibition of spontaneous activity in neuronal networks in vitro by deltamethrin and permethrin, Neurotoxicology, 29, 203, 10.1016\u002Fj.neuro.2008.01.002\nKojima, 2004, Screening for estrogen and androgen receptor activities in 200 pesticides by in vitro reporter gene assays using Chinese hamster ovary cells, Environ. Health Perspect., 112, 524, 10.1289\u002Fehp.6649\nSun, 2014, Pyrethroid and their metabolite, 3-phenoxybenzoic acid showed similar (anti)estrogenic activity in human and rat estrogen receptor alpha-mediated reporter gene assays, Environ. Toxicol. Pharmacol., 37, 371, 10.1016\u002Fj.etap.2013.11.031\nZhang, 2007, Permethrin may disrupt testosterone biosynthesis via mitochondrial membrane damage of Leydig cells in adult male mouse, Endocrinology, 148, 3941, 10.1210\u002Fen.2006-1497\nManikkam, 2012, Pesticide and insect repellent mixture (permethrin and DEET) induces epigenetic transgenerational inheritance of disease and sperm epimutations, Reprod. Toxicol., 34, 708, 10.1016\u002Fj.reprotox.2012.08.010\nVincenzo, 2000\nFAO, 1987, Pesticide residues in food – 1987, part II, toxicology\nBrachet, 1940, La detection histochimique des acides pentose nucleiques, Compt. Rend. Soc. Biol., 133, 88\nCooper, 2009, Cell, 136, 777, 10.1016\u002Fj.cell.2009.02.011\nMateus, 2008, Cell proliferation and apoptosis in keratocystic odontogenic tumors, Med. Oral Patol. Oral. Cir. Bucal, 13, 697\nRussell, 1990\nKim, 2005, Potential estrogenic and antiandrogenic effects of permethrin in rats, J. Reprod. Dev., 51, 201, 10.1262\u002Fjrd.16060\nSharpe, 2001, Hormones and testis development and the possible adverse effects of environmental chemicals, Toxicol. Lett., 120, 221, 10.1016\u002FS0378-4274(01)00298-3\nAtanassova, 1999, Permanent effects of neonatal estrogen exposure in rats on reproductive hormone levels, Sertoli cell number, and the efficiency of spermatogenesis in adulthood, Endocrinology, 140, 5364, 10.1210\u002Fendo.140.11.7108\nAtanassova, 2005, Evidence that androgens and oestrogens as well as follicle-stimulating hormone, can alter Sertoli cell number in the neonatal rat, J. Endocrinol., 184, 107, 10.1677\u002Fjoe.1.05884\nSharpe, 1998, Abnormalities in functional development of the Sertoli cells in rats treated neonatally with diethylstilbestrol: a possible role for estrogens in Sertoli cell development, Biol. Reprod., 59, 1084, 10.1095\u002Fbiolreprod59.5.1084\nImanishi, 2013, Prenatal exposure to permethrin influences vascular development of fetal brain and adult behavior in mice offspring, Environ. Toxicol., 28, 617, 10.1002\u002Ftox.20758\nNasuti, 2007, Dopaminergic system modulation, behavioral changes, and oxidative stress after neonatal administration of pyrethroids, Toxicology, 229, 194, 10.1016\u002Fj.tox.2006.10.015\nShin, 2007, Polyamines in spermatocytes and residual bodies of rat testis, Histochem. Cell. 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inhibitors, J Exp Zool, 216, 445, 10.1002\u002Fjez.1402160313\nKupfer, 1982, Estrogenic actions of chlorinated hydrocarbons, 121\nKupfer, 1983, Estrogenic action of DDT analogs, Am J Ind Med, 4, 163, 10.1002\u002Fajim.4700040115\nBulger, 1985, Estrogenic activity of pesticides and other xenobiotics on the uterus and male reproductive tract, 1\nBulger, 1978, Studies on the in vivo and in vitro estrogenic activities of methoxychlor and its metabolites, Biochem Pharmacol, 27, 2417, 10.1016\u002F0006-2952(78)90354-4\nEroschenko, 1990, Morphologic and biochemical alterations in reproductive tracts of neonatal female mice treated with the pesticide methoxychlor, Biol Reprod, 42, 575, 10.1095\u002Fbiolreprod42.3.573\nHarris, 1974, Effect of several dietary levels of technical methoxychlor on reproduction in rats, J Agr Food Chem, 22, 114, 10.1021\u002Fjf60196a018\nGray, 1989, Antifertility effect of methoxychlor in female rats: Dose- and time-dependent blockade of pregnancy, Toxicol Appl Pharmacol, 97, 454, 10.1016\u002F0041-008X(89)90250-0\nMulder, 1979, Ketamine and xylazine anesthesia in the mouse, UM\u002FSAL, 74, 569\nHeinz, 1987, The influence of chlordecone and oestrogen on the secretion of proteinaceous molecules of the mouse uterus, Environ Pollut, 46, 297, 10.1016\u002F0269-7491(87)90174-6\nSAS Institute Inc., 1988, SAS\u002FSTAT User's Guide, 549\nMartin, 1973, Hypertrophy and hyperplasia in the mouse uterus after oestrogen treatment: an autoradiographic study, J Endocrinol, 56, 133, 10.1677\u002Fjoe.0.0560133\nFinn, 1981, Hormonal control of cell death in the luminal epithelium of the mouse uterus, J Endocrinol, 91, 335, 10.1677\u002Fjoe.0.0910335\nBranham, 1985, The postnatal ontogeny of rat uterine glands and age-related effects of 17β-estradiol, Endocrinology, 117, 2229, 10.1210\u002Fendo-117-5-2229\nMartin, 1984, On the source of uterine “luminal fluid” proteins in the mouse, J Reprod Fertil, 71, 73, 10.1530\u002Fjrf.0.0710073\nStancel, 1980, The 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Neuropsychopharmacol. Biol. Psychiatry, 52, 45, 10.1016\u002Fj.pnpbp.2013.09.014\nMetz, 2015, Marijuana use in pregnancy and lactation: a review of the evidence, Am. J. Obstet. Gynecol., 213, 761, 10.1016\u002Fj.ajog.2015.05.025\nSchonhofen, 2015, Cannabidiol exposure during neuronal differentiation sensitizes cells against redox-active neurotoxins, Mol. Neurobiol., 52, 26, 10.1007\u002Fs12035-014-8843-1\nGunn, 2015, The effects of prenatal cannabis exposure on fetal development and pregnancy outcomes: a protocol, BMJ Open, 5, 10.1136\u002Fbmjopen-2014-007227\nHiguera-Matas, 2015, Long-term consequences of perinatal and adolescent cannabinoid exposure on neural and psychological processes, Neurosci. Biobehav. Rev., 55, 119, 10.1016\u002Fj.neubiorev.2015.04.020\nBara, 2018, Sex-dependent effects of in utero cannabinoid exposure on cortical function, Elife, 7, 10.7554\u002FeLife.36234\nFriedrich, 2016, The grass isn’t always greener: the effects of cannabis on embryological development, BMC Pharmacol. Toxicol., 17, 45, 10.1186\u002Fs40360-016-0085-6\nU.S. Environmental Protection Agency (US EPA), 2014, 1\nAkhtar, 2006, Transplacental disposition and teratogenic effects of chlorpyrifos in rats, J. Toxicol. Sci., 31, 521, 10.2131\u002Fjts.31.521\nRaber, 2015, Understanding dabs: contamination concerns of cannabis concentrates and cannabinoid transfer during the act of dabbing, J. Toxicol. Sci., 40, 797, 10.2131\u002Fjts.40.797\nLankenau, 2017, Marijuana practices and patterns of use among young adult medical marijuana patients and non-patient marijuana users, Drug Alcohol Depend., 170, 181, 10.1016\u002Fj.drugalcdep.2016.10.025\nShiplo, 2016, Medical cannabis use in Canada: vapourization and modes of delivery, Harm Reduct. J., 13, 30, 10.1186\u002Fs12954-016-0119-9\nSexton, 2016, A cross-sectional survey of medical cannabis users: patterns of use and perceived efficacy, Cannabis Cannabinoid Res., 1, 131, 10.1089\u002Fcan.2016.0007\nCalifornia Bureau of Marijuana Control, 2017, 1\nSullivan, 2013, Determination of pesticide residues in Cannabis smoke, J. 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ovarian dysfunction, Endocrine Journal, 51, 165, 10.1507\u002Fendocrj.51.165\nSuzuki, 2002, Developmental effects of perinatal exposure to bisphenol-A and diethylstilbestrol on reproductive organs in female mice, Reproductive Toxicology, 16, 107, 10.1016\u002FS0890-6238(02)00005-9\nXu, 2002, Bisphenol A induces apoptosis and G2-to-M arrest of ovarian granulosa cells, Biochemical and Biophysical Research Communications, 292, 456, 10.1006\u002Fbbrc.2002.6644\nPeretz, 2011, Bisphenol A impairs follicle growth, inhibits steroidogenesis, and downregulates rate-limiting enzymes in the estradiol biosynthesis pathway, Toxicological Sciences, 119, 209, 10.1093\u002Ftoxsci\u002Fkfq319\nPeretz, 2012, Bisphenol A inhibits follicle growth and induces atresia in cultured mouse antral follicles independently of the genomic estrogenic pathway, Biology of Reproduction, 87, 63, 10.1095\u002Fbiolreprod.112.101899\nLenie, 2008, Continuous exposure to bisphenol A during in vitro follicular development induces meiotic abnormalities, Mutation Research, 651, 71, 10.1016\u002Fj.mrgentox.2007.10.017\nVandenberg, 2009, Bisphenol-A and the great divide: a review of controversies in the field of endocrine disruption, Endocrine Reviews, 30, 75, 10.1210\u002Fer.2008-0021\nSengupta, 2013, Molecular mechanism of action of bisphenol and bisphenol A mediated by oestrogen receptor alpha in growth and apoptosis of breast cancer cells, British Journal of Pharmacology, 169, 167, 10.1111\u002Fbph.12122\nHugo, 2008, Bisphenol A at environmentally relevant doses inhibits adiponectin release from human adipose tissue explants and adipocytes, Environmental Health Perspectives, 116, 1642, 10.1289\u002Fehp.11537\nVinas, 2013, Bisphenol S disrupts estradiol-induced nongenomic signaling in a rat pituitary cell line: effects on cell functions, Environmental Health Perspectives, 121, 352, 10.1289\u002Fehp.1205826\nTeng, 2013, Bisphenol A affects androgen receptor function via multiple mechanisms, Chemico-Biological Interactions, 203, 556, 10.1016\u002Fj.cbi.2013.03.013\nMoriyama, 2002, Thyroid hormone action is disrupted by bisphenol A as an antagonist, Journal of Clinical Endocrinology and Metabolism, 87, 5185, 10.1210\u002Fjc.2002-020209\nRubin, 2011, Bisphenol A: an endocrine disruptor with widespread exposure and multiple effects, Journal of Steroid Biochemistry and Molecular Biology, 127, 27, 10.1016\u002Fj.jsbmb.2011.05.002\nPoland, 1982, 2,3,7,8-Tetrachlorodibenzo-p-dioxin and related halogenated aromatic hydrocarbons: examination of the mechanism of toxicity, Annual Review of Pharmacology and Toxicology, 22, 517, 10.1146\u002Fannurev.pa.22.040182.002505\nPuga, 2009, The aryl hydrocarbon receptor cross-talks with multiple signal transduction pathways, Biochemical Pharmacology, 77, 713, 10.1016\u002Fj.bcp.2008.08.031\nAbel, 2010, An introduction to the molecular basics of aryl hydrocarbon receptor biology, Biological Chemistry, 391, 1235, 10.1515\u002Fbc.2010.128\nPollenz, 2002, The mechanism of AH receptor protein down-regulation (degradation) and its impact on AH receptor-mediated gene regulation, Chemico-Biological Interactions, 141, 41, 10.1016\u002FS0009-2797(02)00065-0\nHernandez-Ochoa, 2009, The role of the aryl hydrocarbon receptor in the female reproductive system, Biochemical Pharmacology, 77, 547, 10.1016\u002Fj.bcp.2008.09.037\nBussmann, 2006, An aryl hydrocarbon receptor agonist amplifies the mitogenic actions of estradiol in granulosa cells: evidence of involvement of the cognate receptors, Biology of Reproduction, 74, 417, 10.1095\u002Fbiolreprod.105.043901\nSafe, 2003, Inhibitory aryl hydrocarbon receptor-estrogen receptor alpha cross-talk and mechanisms of action, Chemical Research in Toxicology, 16, 807, 10.1021\u002Ftx034036r\nNishizawa, 2005, Effects of in utero exposure to bisphenol A on mRNA expression of arylhydrocarbon and retinoid receptors in murine embryos, Journal of Reproduction and Development, 51, 315, 10.1262\u002Fjrd.16008\nBonefeld-Jorgensen, 2007, Endocrine-disrupting potential of bisphenol A, bisphenol A dimethacrylate, 4-n-nonylphenol, and 4-n-octylphenol in vitro: new data and a brief review, Environmental Health Perspectives, 115, 69, 10.1289\u002Fehp.9368\nSwedenborg, 2010, AhR and ARNT modulate ER signaling, Toxicology, 268, 132, 10.1016\u002Fj.tox.2009.09.007\nKarman, 2012, 2,3,7,8-Tetrachlorodibenzo-p-dioxin activates the aryl hydrocarbon receptor and alters sex steroid hormone secretion without affecting growth of mouse antral follicles in vitro, Toxicology and Applied Pharmacology, 261, 88, 10.1016\u002Fj.taap.2012.03.015\nFlint, 2012, Bisphenol A exposure, effects, and policy: a wildlife perspective, Journal of Environment Management, 104, 19, 10.1016\u002Fj.jenvman.2012.03.021\nVandenberg, 2012, Urinary, circulating, and tissue biomonitoring studies indicate widespread exposure to bisphenol A, Cien Saude Colet, 17, 407, 10.1590\u002FS1413-81232012000200015\nSchmidt, 1996, Characterization of a murine Ahr null allele: involvement of the Ah receptor in hepatic growth and development, Proceedings of the National Academy of Sciences of the United States of America, 93, 6731, 10.1073\u002Fpnas.93.13.6731\nBenedict, 2000, Physiological role of the aryl hydrocarbon receptor in mouse ovary development, Toxicological Sciences, 56, 382, 10.1093\u002Ftoxsci\u002F56.2.382\nHernandez-Ochoa, 2010, The ability of the aryl hydrocarbon receptor to regulate ovarian follicle growth and estradiol biosynthesis in mice depends on stage of sexual maturity, Biology of Reproduction, 83, 698, 10.1095\u002Fbiolreprod.110.087015\nMiller, 2005, Methoxychlor directly affects ovarian antral follicle growth and atresia through Bcl-2- and Bax-mediated pathways, Toxicological Sciences, 88, 213, 10.1093\u002Ftoxsci\u002Fkfi276\nAbbott, 1999, RT-PCR quantification of AHR, ARNT, GR, and CYP1A1 mRNA in craniofacial tissues of embryonic mice exposed to 2,3,7,8-tetrachlorodibenzo-p-dioxin and hydrocortisone, Toxicological Sciences, 47, 76, 10.1093\u002Ftoxsci\u002F47.1.76\nCraig, 2013, Di-n-butyl phthalate disrupts the expression of genes involved in cell cycle and apoptotic pathways in mouse ovarian antral follicles, Biology of Reproduction, 88, 23, 10.1095\u002Fbiolreprod.112.105122\nPfaffl, 2001, A new mathematical model for relative quantification in real-time RT-PCR, Nucleic Acids Research, 29, e45, 10.1093\u002Fnar\u002F29.9.e45\nWozniak, 2005, Xenoestrogens at picomolar to nanomolar concentrations trigger membrane estrogen receptor-alpha-mediated Ca2+ fluxes and prolactin release in GH3\u002FB6 pituitary tumor cells, Environmental Health Perspectives, 113, 431, 10.1289\u002Fehp.7505\nQuirk, 2004, Ovarian follicular growth and atresia: the relationship between cell proliferation and survival, Journal of Animal Science, 82, E40, 10.2527\u002F2004.8213_supplE40x\nMatsuda, 2012, Follicular growth and atresia in mammalian ovaries: regulation by survival and death of granulosa cells, Journal of Reproduction and Development, 58, 44, 10.1262\u002Fjrd.2011-012\nHsu, 1996, Targeted overexpression of Bcl-2 in ovaries of transgenic mice leads to decreased follicle apoptosis, enhanced folliculogenesis, and increased germ cell tumorigenesis, Endocrinology, 137, 4837, 10.1210\u002Fendo.137.11.8895354\nRiou, 1999, Susceptibility of differentiated thyrocytes in primary culture to undergo apoptosis after exposure to hydrogen peroxide: relation with the level of expression of apoptosis regulatory proteins, Bcl-2 and Bax, Endocrinology, 140, 1990, 10.1210\u002Fendo.140.5.6725\nNishizawa, 2005, Effects of exposure in utero to bisphenol a on the expression of aryl hydrocarbon receptor, related factors, and xenobiotic metabolizing enzymes in murine embryos, Journal of Reproduction and Development, 51, 593, 10.1262\u002Fjrd.17026\nKruger, 2008, Plastic components affect the activation of the aryl hydrocarbon and the androgen receptor, Toxicology, 246, 112, 10.1016\u002Fj.tox.2007.12.028\nImanishi, 2003, Effects of oral exposure of bisphenol A on mRNA expression of nuclear receptors in murine placentae assessed by DNA microarray, Journal of Reproduction and Development, 49, 329, 10.1262\u002Fjrd.49.329\nBasavarajappa, 2012, Methoxychlor inhibits growth and induces atresia through the aryl hydrocarbon receptor pathway in mouse ovarian antral follicles, Reproductive Toxicology, 34, 16, 10.1016\u002Fj.reprotox.2012.03.007\nCooper, 2008, Maternal tobacco use and its preimplantation effects on fertility: more reasons to stop smoking, Seminars in Reproductive Medicine, 26, 204, 10.1055\u002Fs-2008-1042959\nShiromizu, 1985, Murine oocyte destruction following intraovarian treatment with 3-methylcholanthrene or 7,12-dimethylbenz(a)anthracene: protection by alpha-naphthoflavone, Teratogenesis, Carcinogenesis, and Mutagenesis, 5, 463, 10.1002\u002Ftcm.1770050609\nShiromizu, 1984, The effect of intraovarian injection of benzo(a)pyrene on primordial oocyte number and ovarian aryl hydrocarbon [benzo(a)pyrene] hydroxylase activity, Toxicology and Applied Pharmacology, 76, 18, 10.1016\u002F0041-008X(84)90025-5\nHolcomb, 1994, Inhibition of 7,12-dimethylbenzanthracene-induced rat mammary tumor growth by 2,3,7,8-tetrachlorodibenzo-p-dioxin, Cancer Letters, 82, 43, 10.1016\u002F0304-3835(94)90144-9\nMlynarcikova, 2005, Alterations in steroid hormone production by porcine ovarian granulosa cells caused by bisphenol A and bisphenol A dimethacrylate, Molecular and Cellular Endocrinology, 244, 57, 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Guidance for industry-labelling guidance for cisapride tablets. OGD-L-4. Internet, 1997.\nFunabashi, 1993, Reproductive and developmental toxicity studies of mosapride citrate. 2. Teratogenicity study in rats, Yakuri to Chiryo, 21, 3423\nFunabashi, 1993, Reproductive and developmental toxicity studies of mosapride citrate. 4. Teratogenicity study in rabbits, Yakuri to Chiryo, 21, 3469\nADRAC on cisapride, acyclovir, SSRIs. Scripta 1996;2158:27.\nWarning on cisapride interactions. Scripta 1996;2123:21.\nBran, 1995, Long QT syndrome during high-dose cisapride, Arch. Internal Med., 155, 765, 10.1001\u002Farchinte.155.7.765\nLewin, 1996, Cisapride-induced long QT interval [see comments], J. Pediatr., 128, 279, 10.1016\u002FS0022-3476(96)70409-2\nWysowski, 1996, Cisapride and fatal arrhythmia [letter], N. Engl. J. Med., 335, 290, 10.1056\u002FNEJM199607253350416\nCarlsson, 1997, Electrophysiological characterisation of the prokinetic agents cisapride and mosapride in vivo and in vitro: implications for proarrhythmic potential?, J. Pharmacol. Exp. Ther., 282, 220\nMacNeil, 1997, The side effect profile of class III antiarrhythmic drugs: focus on d,l-sotalol, Am. J. Cardiol., 80, 90G, 10.1016\u002FS0002-9149(97)00718-2\nDavies, 1996, Developmental changes in ionic channel activity in the embryonic murine heart, Circul. Res., 78, 15, 10.1161\u002F01.RES.78.1.15\nWang, 1996, Developmental changes in the delayed rectifier K+ channels in mouse heart, Circul. Res., 79, 79, 10.1161\u002F01.RES.79.1.79\nSpence, 1994, Effects of the class III antiarrhythmic dofetilide (UK-68,798) on the heart rate of midgestation rat embryos, in vitro, Teratology, 49, 282, 10.1002\u002Ftera.1420490408\nAzarbayjani, 2001, Phenytoin-induced cleft palate: evidence for embryonic cardiac bradyarrhythmia due to inhibition of delayed rectifier K+ channels resulting in hypoxia-reoxygenation damage, Teratology, 63, 152, 10.1002\u002Ftera.1026\nWebster, 1996, Teratogenic potential of almokalant, dofetilide, and d-sotalol: drugs with potassium channel blocking activity, Teratology, 53, 168, 10.1002\u002F(SICI)1096-9926(199603)53:3\u003C168::AID-TERA4>3.0.CO;2-0\nBan, 1994, Embryotoxic effects of L-691,121, a class III antiarrhythmic agent, in rats, Arch. Toxicol., 69, 65, 10.1007\u002Fs002040050139\nAbrahamsson, 1994, Induction of rhythm abnormalities in the fetal rat heart. 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In: Kavlock RJ, Daston GP, editors. Drug toxicity in embryonic development II; advances in understanding mechanisms of birth defects: mechanistic understanding of human developmental toxicants. Berlin, Heidelberg: Springer, 1997. p. 161–90.\nDanielsson, 1997, Initiation of phenytoin teratogenesis: pharmacologically induced embryonic bradycardia and arrhythmia resulting in hypoxia and possible free radical damage at reoxygenation, Teratology, 56, 271, 10.1002\u002F(SICI)1096-9926(199710)56:4\u003C271::AID-TERA6>3.0.CO;2-1\nStein, 1990, Effects of calcium channel blockers on the development of early rat post-implantation embryos in culture, Arch. Toxicol., 64, 623, 10.1007\u002FBF01974690\nNew, 1978, Whole-embryo culture and the study of mammalian embryos during organogenesis, Biol. Rev. Camb. Philos. Soc., 53, 81, 10.1111\u002Fj.1469-185X.1978.tb00993.x\nMcCallum, 1991, Cisapride: a new class of prokinetic agent. The ACG Committee on FDA-related matters. American College of Gastroenterology, Am. J. Gastroenterol., 86, 135\nEMEA. Points to consider: the assessment of the potential for QT interval prolongation by non-cardivascular medicinal products (EMEACPMP\u002F986\u002F96). Internet, 1997.\nManson JM, Kang YJ. Test methods for assessing female reproductive and developmental toxicology. In: Hayes AW, editor. Principles and methods of toxicology. New York: Raven Press, 1994. p. 989–1037.\nWellfelt, 1999, Teratogenicity of the class III antiarrhythmic drug almokalant. Role of hypoxia and reactive oxygen species, Reprod. Toxicol., 13, 93, 10.1016\u002FS0890-6238(98)00066-5\nSköld AC, Wellfelt K, Danielsson BR, Stage-specific skeletal and visceral defects of the IKr-blocker almokalant: further evidence for teratogenicity via a hypoxia-related mechanism. Teratology 2001;64:292–300.\nCarlsson, 1993, Proarrhythmic effects of the class III agent almokalant: importance of infusion rate, QT dispersion and early afterdepolarisations [see comments], Cardiovasc. Res., 27, 2186, 10.1093\u002Fcvr\u002F27.12.2186\nKowey, 1997, Pharmacologic and pharmacokinetic profile of class III antiarrhythmic drugs, Am. J. Cardiol., 80, 16G, 10.1016\u002FS0002-9149(97)00710-8\nNobile, 1997, Inhibition of delayed rectifier K+ channels by phenytoin in rat neuroblastoma cells, Br. J. Pharmacol., 120, 647, 10.1038\u002Fsj.bjp.0700969\nFinnell, 1991, Parental epilepsy, anticonvulsant drugs, and reproductive outcome: epidemiologic and experimental findings spanning three decades. 1: Animal studies, Reprod. Toxicol., 5, 281, 10.1016\u002F0890-6238(91)90090-3\nDanielson, 1992, Histopathological and hemodynamic studies supporting hypoxia and vascular disruption as explanation to phenytoin teratogenicity, Teratology, 46, 485, 10.1002\u002Ftera.1420460513\nDanielsson, 1995, Phenytoin causes phalangeal hypoplasia in the rabbit fetus at clinically relevant free plasma concentrations, Teratology, 52, 252, 10.1002\u002Ftera.1420520503\nDanielsson, 2000, Pharmacokinetic data support pharmacologically induced embryonic dysrhythmia as explanation to fetal hydantoin syndrome in rats, Toxicol. Appl. Pharmacol., 163, 164, 10.1006\u002Ftaap.1999.8858\nViskin, 1999, Long QT syndromes and torsade de pointes, Lancet, 354, 1625, 10.1016\u002FS0140-6736(99)02107-8\nBailey, 1997, Cisapride use during human pregnancy: a prospective, controlled multicentre study, Digest. Dis. Sci., 42, 1848, 10.1023\u002FA:1018898707449\nDansky, 1991, Parental epilepsy anticonvulsant drugs, and reproductive outcome: epidemiologic and experimental findings spanning three decades. 2: Human studies, Reprod. Toxicol., 5, 301, 10.1016\u002F0890-6238(91)90091-S\nBabin, 1985, Possible embryofetopathy caused by beta-blocker (sotalol) taken throughout the pregnancy, Pediatrie, 40, 129",{"VOID":1953},"10.1016\u002Fs0890-6238(02)00042-4","https:\u002F\u002Fwww.sciencedirect.com\u002Fscience\u002Farticle\u002Fpii\u002FS0890623802000424",[1956,1980,1993,2006],{"id":1957,"sortIndex":32,"researcher":28,"roles":1958,"affiliations":1959,"properties":1977,"displayName":1979,"givenName":28,"familyName":28},"edd7fee8-74c1-4b29-af37-14a2becdb22a",[955],[1960,1968],{"id":1961,"sortIndex":32,"affiliation":1962,"properties":28},"46fa6a5c-993f-4b9d-ba4c-8de8c6d1912f",{"id":1961,"createTime":28,"updateTime":28,"relativeEntities":1963,"slug":28,"properties":1964,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1967,"statistic":28},[],{"title":1965},{"VI":1966},"Division of Toxicology, Department of Pharmaceutical 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N. Y. Acad. Sci., 1076, 691, 10.1196\u002Fannals.1371.053\nAbadin, 2007\nEFSA, 2013, Scientific opinion on the safety and efficacy of iodine compounds (E2) as feed additives for all species: calcium iodate anhydrous and potassium iodide, based on a dossier submitted by HELM AG1, EFSA J., 11, 3101, 10.2903\u002Fj.efsa.2013.3101\nEFSA, 2011, Scientific Opinion on Safety and efficacy of Sel-Plex® (organic form of selenium produced by Saccharomyces cerevisiae CNCM I-3060) for all species, EFSA J., 9, 2110, 10.2903\u002Fj.efsa.2011.2110\nEuropean Council, 2005\nEuropean Commission, 2011\nEMA, 2005\nEuropean Parliament and Council, 2006\nDownes, 2012, Juvenile toxicity: are we asking the right questions?, Toxicol. Pathol., 40, 830, 10.1177\u002F0192623312439124\nEuropean Parliament and Council, 2006\nEuropean Parliament and Council, 2012\nCoughtrie, 1988, The inadequacy of perinatal glucuronidation: immunoblot analysis of the developmental expression of individual UDP-glucuronosyltransferase isoenzymes in rat and human liver microsomes, Mol. Pharmacol., 34, 729\nMaranghi, 2012, Targeted toxicological testing to investigate the role of endocrine disrupters in puberty disorders, Reprod. Toxicol., 33, 290, 10.1016\u002Fj.reprotox.2012.01.009\nCarr, 2014, Low level chlorpyrifos exposure increases anandamide accumulation in juvenile rat brain in the absence of brain cholinesterase inhibition, Neurotoxicology, 43, 82, 10.1016\u002Fj.neuro.2013.12.009\nCarr, 2013, Induction of endocannabinoid levels in juvenile rat brain following developmental chlorpyrifos exposure, Toxicol. 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Rev. Toxicol., 30, 197, 10.1080\u002F10408440091159194\nShin, 2015, High doses of caffeine reduce in vivo osteogenic activity in prepubertal rats, J. Anat., 227, 10, 10.1111\u002Fjoa.12332\nTollefsen, 2014, Applying adverse outcome pathways (AOPs) to support integrated approaches to testing and assessment (IATA), Regul. Toxicol. Pharm., 70, 629, 10.1016\u002Fj.yrtph.2014.09.009\nEFSA PPR Panel (EFSA Panel on Plant Protection Products and their Residues), 2017, Scientific Opinion on the investigation into experimental toxicological prope rties of plant protection products having a potential link to Parkinson’s disease and childhood leukaemia, EFSA J., 15, 4691\nDe Schaepdrijver, 2008, Real life juvenile toxicity case studies: the good, the bad and the ugly, Reprod. Toxicol., 26, 54, 10.1016\u002Fj.reprotox.2008.04.002\nRemick, 2015, Juvenile toxicology: relevance and challenges for toxicologists and pathologist, Toxicol. Pathol., 43, 1166, 10.1177\u002F0192623315595883\nVo, 2010, Potential estrogenic effect (s) of parabens at the prepubertal stage of a postnatal female rat model, Reprod. Toxicol., 29, 306, 10.1016\u002Fj.reprotox.2010.01.013\nCalamandrei, 2006, Efficient testing strategies for evaluation of xenobiotics with neuroendocrine activity, Reprod. Toxicol., 22, 164, 10.1016\u002Fj.reprotox.2006.04.003\nVeldhuis, 2006, Somatotropic and gonadotropic axes linkages in infancy, childhood, and the puberty-adult transition, Endocr. 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