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Bởi vì cơ chế gây tổn thương do năng lượng tần số vô tuyến là nhiệt, nghiên cứu này được thực hiện nhằm xác định độ dốc nhiệt độ trong mô cơ tim trong quá trình ablation bằng ống thông tần số vô tuyến (RF), và để xác thực một mô hình động lực học đã được xây dựng nhằm mô tả những quan sát này. Các tổn thương được tạo ra bằng cách làm nóng RF trong một mô hình thí nghiệm của thành thất phải (RV) của chó, được cô lập và tưới máu cũng như siêu tưới máu. Công suất RF được điều chỉnh để duy trì nhiệt độ đầu điện cực ở 80°C trong 120 giây cho 153 tổn thương liên tiếp và các độ dốc nhiệt độ theo hướng ra đã được đo lường. Với khoảng cách tăng dần từ điện cực, nhiệt độ của cơ tim giảm theo hình thức hyperbol mà mô hình động lực học đã được xây dựng tiên đoán một cách chính xác (P = 0.0001, r = 0.98). Độ dốc này và kích thước tổn thương kết quả không bị ảnh hưởng bởi tốc độ tưới máu vành. Việc sử dụng giám sát nhiệt độ đầu điện cực như một yếu tố dự đoán kích thước tổn thương đã được thử nghiệm trên 104 tổn thương liên tiếp với nhiệt độ đầu điện cực thay đổi giữa 50 và 85°C. Nhiệt độ đầu điện cực có mối tương quan chặt chẽ với độ sâu của tổn thương (P = 0.0001, r = 0.92) và chiều rộng (P = 0.0001, r = 0.88), và là một yếu tố dự đoán kích thước tổn thương tốt hơn các phép đo về công suất, dòng điện hay năng lượng. Nhiệt độ tại ranh giới giữa mô sống và mô không sống được ước lượng là 47.9°C. Các dữ liệu này chứng minh rằng trong quá trình ablation bằng ống thông tần số vô tuyến, độ dốc nhiệt độ theo hướng ra là một cách tiên đoán rõ ràng và dường như độc lập với tưới máu nội cơ tim nếu nhiệt độ điện cực cố định được duy trì. Việc sử dụng giám sát nhiệt độ đầu điện cực có thể dự đoán chính xác kích thước cuối cùng của tổn thương do tần số vô tuyến gây ra.\u003C\u002Fjats:p>","\u003Cjats:p>Thecharacteristics of radiofrequency catheter ablation induced injury in the heart are not well characterized. Since the mechanism of injury by radiofrequency energy is thermal, this study was performed to determine the temperature gradient in myocardial tissue during radiofrequency (RF) catheter ablation, and to validate a thermodynamic model derived to describe these observations. Lesions were created by RF heating in an experimental model of isolated perfused and superfused canine right ventricular (RV) free wall. RF power output was adjusted to maintain electrode tip temperature at 80°C for 120 seconds in 153 serial lesions and radial temperature gradients were measured. With increasing distance from the electrode, the temperature of the myocardium decreased in a hyperbolic form that was closely predicted by a derived thermodynamic model (P = 0.0001, r = 0.98). This gradient and resultant lesion sizes were unafected by the rate of coronary perfusion. The utility of tip temperature monitoring as a predictor of lesion size was tested in 104 serial lesions with tip temperatures that were varied between 50 and 85°C. The tip temperature correlated closely with lesion depth (P = 0.0001, r = 0.92) and width (P = 0.0001, r = 0.88), and was a better predictor of lesion size than measurements of power, current or energy. The temperature at the margin between viable and nonviable tissue was estimated to be 47.9°C. These data demonstrate that during radiofrequency catheter ablation, the radial temperature gradient is predictably hyperbolic and appears to be independent of intramyocardial perfusion if constant electrode temperature is maintained. The use of tip temperature monitoring can accurately predict the ultimate size of radiofrequency‐induced lesions.\u003C\u002Fjats:p>",{"EN":115,"VI":116},"Tissue Heating During Radiofrequency Catheter Ablation: A Thermodynamic Model and Observations in Isolated Perfused and Superfused Canine Right Ventricular Free Wall","Nhiệt độ mô trong quá trình ablation bằng ống thông tần số vô tuyến: Một mô hình nhiệt động lực học và các quan sát trên thành thất phải của chó cô lập được tưới máu và siêu tưới máu",{"VOID":118},"2472624",{"VOID":120},"10.1111\u002Fj.1540-8159.1989.tb05034.x","PUBLICATION","VERIFIED","2024-10-12T05:19:14.996+00:00","Auto Verify",[126],"EN",[128],"VI","https:\u002F\u002Fonlinelibrary.wiley.com\u002Fdoi\u002F10.1111\u002Fj.1540-8159.1989.tb05034.x",[131,152],{"id":132,"sortIndex":25,"researcher":24,"roles":133,"affiliations":134,"properties":143,"displayName":147,"givenName":24,"familyName":24},"64ef2ef4-4fab-450a-86bd-ff0b170e4694",[],[135],{"id":136,"sortIndex":25,"affiliation":137,"properties":24},"103d7ce6-daa7-4801-ac14-18299f6137c4",{"id":136,"createTime":24,"updateTime":24,"relativeEntities":138,"slug":24,"properties":139,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":142,"statistic":24},[],{"title":140},{"EN":141},"Division of Cardiology, Department of Internal Medicine, and the Division of Nuclear Medicine, Department of Radiology, University of Virginia School of Medicine, Charlottesville, Virginia",[],{"orcid":144,"title":146,"gsAuthor":148,"openalex":150},{"VOID":145},"https:\u002F\u002Forcid.org\u002F0000-0002-5592-8338",{"EN":147},"David E. Haines",{"VOID":149},"[\"pZbyMn8AAAAJ\"]",{"VOID":151},"A5064399230",{"id":153,"sortIndex":154,"researcher":24,"roles":155,"affiliations":156,"properties":163,"displayName":165,"givenName":24,"familyName":24},"a24ab999-2b75-4f1d-b6fe-92d545430c55",1,[],[157],{"id":136,"sortIndex":25,"affiliation":158,"properties":24},{"id":136,"createTime":24,"updateTime":24,"relativeEntities":159,"slug":24,"properties":160,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":162,"statistic":24},[],{"title":161},{"EN":141},[],{"title":164,"openalex":166},{"EN":165},"Denny D. Watson",{"VOID":167},"A5088685513","ARTICLE",{"url":24,"publisher":170,"properties":212},{"id":6,"createTime":7,"updateTime":8,"relativeEntities":171,"slug":10,"properties":172,"entityType":22,"verifyStatus":23,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":25,"subjectFields":177,"manageAffiliations":186,"indexDatabases":197,"url":91,"thumbnailPath":24,"statistic":24,"gsStatistic":24,"type":24,"analyzePriority":24},[],{"country":173,"eissn":174,"issn":175,"title":176},{"VOID":13},{"VOID":15},{"VOID":17},{"EN":19},[178,182],{"id":28,"createTime":24,"updateTime":24,"relativeEntities":179,"label":180,"description":181,"parentId":24,"standard":24,"scholarHubFieldId":24},[],{"EN":31},{},{"id":34,"createTime":24,"updateTime":24,"relativeEntities":183,"label":184,"description":185,"parentId":24,"standard":24,"scholarHubFieldId":24},[],{"EN":37},{},[187,192],{"id":41,"createTime":24,"updateTime":24,"relativeEntities":188,"slug":24,"properties":189,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":191,"statistic":24},[],{"title":190},{"EN":45},[],{"id":48,"createTime":24,"updateTime":24,"relativeEntities":193,"slug":24,"properties":194,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":196,"statistic":24},[],{"title":195},{"EN":52},[],[198,205],{"id":56,"indexDatabase":199,"url":69,"indexYears":24,"academicFieldIds":204,"indexDatabaseRanking":24},{"id":58,"createTime":24,"updateTime":24,"relativeEntities":200,"label":201,"description":202,"key":65,"publicationTags":203,"standard":24},[],{"EN":61,"VI":61},{"EN":63,"VI":64},[67,68],[71,72],{"id":74,"indexDatabase":206,"url":85,"indexYears":86,"academicFieldIds":211,"indexDatabaseRanking":90},{"id":76,"createTime":24,"updateTime":24,"relativeEntities":207,"label":208,"description":209,"key":82,"publicationTags":210,"standard":24},[],{"EN":79,"VI":79},{"EN":79,"VI":81},[84],[88,89],{"issue":213,"pages":215,"volume":217},{"VOID":214},"6",{"VOID":216},"962-976",{"VOID":218},"12",603,{"total":219,"publishYear":221,"statisticByYear":222},1989,{"1989":223,"1990":223,"1991":224,"1992":225,"1993":226,"1994":227,"1995":228,"1996":227,"1997":229,"1998":230,"1999":231,"2000":232,"2001":233,"2002":232,"2003":226,"2004":226,"2005":224,"2006":234,"2007":235,"2008":236,"2009":224,"2010":237,"2011":227,"2012":238,"2013":233,"2014":229,"2015":239,"2016":225,"2017":235,"2018":236,"2019":236,"2020":231,"2021":236,"2022":225,"2023":240,"2024":240,"2025":241,"2026":234},3,9,11,16,23,26,21,32,20,17,22,15,8,13,18,6,30,14,19,"1989-06-01","ERROR_IN_ANALYZE_CITATION",[67,90],[246,249,252,255,258,261,264,267,270,273,276,279,282,285,288,291,294,297,300,303,306,309,312,315,318],{"id":24,"text":247,"url":24,"identifiers":248},"Scheinman MM., 1982, Catheter induced ablation of the atrioventricular junction to control refractory supraventricular arrhythmia, J Am Med Assoc, 248",{},{"id":24,"text":250,"url":24,"identifiers":251},"10.1016\u002FS0735-1097(86)80156-5",{"doi":250},{"id":24,"text":253,"url":24,"identifiers":254},"10.1161\u002F01.CIR.67.3.687",{"doi":253},{"id":24,"text":256,"url":24,"identifiers":257},"10.1016\u002FS0735-1097(84)80394-0",{"doi":256},{"id":24,"text":259,"url":24,"identifiers":260},"10.1016\u002FS0735-1097(85)80179-0",{"doi":259},{"id":24,"text":262,"url":24,"identifiers":263},"10.1161\u002F01.CIR.73.1.10",{"doi":262},{"id":24,"text":265,"url":24,"identifiers":266},"10.1016\u002FS0039-6109(16)40540-2",{"doi":265},{"id":24,"text":268,"url":24,"identifiers":269},"10.3171\u002Fjns.1970.33.4.0415",{"doi":268},{"id":24,"text":271,"url":24,"identifiers":272},"10.1111\u002Fj.1749-6632.1980.tb50749.x",{"doi":271},{"id":24,"text":274,"url":24,"identifiers":275},"10.1016\u002FS0735-1097(87)80388-1",{"doi":274},{"id":24,"text":277,"url":24,"identifiers":278},"Borggrefe M, 1987, Application of transvenous radio‐frequency alternating current ablation in humans, Circulation, 76, IV",{},{"id":24,"text":280,"url":24,"identifiers":281},"Hoyt RH, 1985, Factors influencing trans‐catheter radiofrequency ablation of the myocardium, Circulation, 72, III",{},{"id":24,"text":283,"url":24,"identifiers":284},"10.1115\u002F1.3138197",{"doi":283},{"id":24,"text":286,"url":24,"identifiers":287},"Gottlieb GJ, 1981, Ultrastructural characterization of the border zone surrounding early experimental myocardial infarcts in dogs, Am J Pathol, 103, 292",{},{"id":24,"text":289,"url":24,"identifiers":290},"10.1111\u002Fj.1540-8159.1988.tb06027.x",{"doi":289},{"id":24,"text":292,"url":24,"identifiers":293},"Langberg J, 1987, Radiofrequency catheter ablation in the coronary sinus, J Am Coll Cardiol, 9, 99A",{},{"id":24,"text":295,"url":24,"identifiers":296},"Naccarelli G, 1987, Selective catheter ablation of canine ventricular myocardium with radiofrequency current, J Am Coll Cardiol, 9, 99A",{},{"id":24,"text":298,"url":24,"identifiers":299},"10.1161\u002F01.CIR.78.2.416",{"doi":298},{"id":24,"text":301,"url":24,"identifiers":302},"10.1111\u002Fj.1540-8159.1987.tb06037.x",{"doi":301},{"id":24,"text":304,"url":24,"identifiers":305},"Casman ER, 1984, Theoretical aspects of radiofrequency lesions in the dorsal root entry zone, Neurosurgery, 15, 945",{},{"id":24,"text":307,"url":24,"identifiers":308},"Adams T, 1980, Thermodynamic technique for the quantification of regional blood flow, Am J Physiol, 238, H682",{},{"id":24,"text":310,"url":24,"identifiers":311},"Organ L., 1976, Electrophysiologic principles of radiofrequency lesion making: Int. Symp. Radiofrequency Lesion Making, Appl Neurophysiol, 39, 69",{},{"id":24,"text":313,"url":24,"identifiers":314},"Hoffman E, 1987, Phase displacement between voltage and current during radiofrequency catheter ablation, Circulation, 76, IV",{},{"id":24,"text":316,"url":24,"identifiers":317},"10.1161\u002F01.RES.46.3.387",{"doi":316},{"id":24,"text":319,"url":24,"identifiers":320},"Sears FW, 1955, University physics. Reading, 17",{},false,{"id":323,"createTime":324,"updateTime":324,"relativeEntities":325,"slug":326,"properties":327,"entityType":121,"verifyStatus":122,"verifyTime":324,"verifyNote":124,"languages":340,"translateLanguages":24,"viewCount":25,"primaryUrl":341,"fullTextUrl":24,"authors":342,"publicationType":168,"publisherRelationship":519,"citationCount":569,"citationInfo":570,"publishDate":573,"publishYear":571,"citationAnalyzeStatus":23,"lastCitationAnalyze":24,"indexDatabases":574,"openAccess":24,"references":575,"isForceReanalyzing":321},"375c9c05-379e-4853-a4f9-242fe0cee50f","2025-02-03T20:29:46.191+00:00",[],"Impact-of-Remote-Magnetic-Catheter-Navigation-on-Ablation-Fluoroscopy-and-Procedure-Time",{"openalex":328,"mag":330,"abstract":332,"title":334,"pm":336,"doi":338},{"VOID":329},"W2100656146",{"VOID":331},"2100656146",{"EN":333},"\u003Cjats:p> \u003Cjats:bold>\n            \u003Cjats:italic>Background:\u003C\u002Fjats:italic>\n          \u003C\u002Fjats:bold>\n          \u003Cjats:italic>Remote magnetic catheter navigation (RCN) is gaining acceptance in clinical cardiac electrophysiology, but details regarding how RCN affects procedure execution are not well characterized.\u003C\u002Fjats:italic> \u003C\u002Fjats:p>\u003Cjats:p> \u003Cjats:bold>\n            \u003Cjats:italic>Methods:\u003C\u002Fjats:italic>\n          \u003C\u002Fjats:bold>\n          \u003Cjats:italic>From January 1, 2005, to November 30, 2007, 721 cases were retrospectively analyzed and compared. Of these, 127 used RCN and 594 used manual catheter navigation (MCN). Data including procedure time, fluoroscopy time, ablation catheter, procedural success, and complications were extracted from our procedure database and compared between RCN and MCN.\u003C\u002Fjats:italic> \u003C\u002Fjats:p>\u003Cjats:p> \u003Cjats:bold>\n            \u003Cjats:italic>Results:\u003C\u002Fjats:italic>\n          \u003C\u002Fjats:bold>\n          \u003Cjats:italic>RCN use significantly decreased fluoroscopy time for atrial fibrillation (AF) ablation (−29 minutes, P < 0.001), atrioventricular nodal reentrant tachycardia ablation (−14 minutes, P < 0.001), and atrioventricular reentrant tachycardia ablation (−18 minutes, P = 0.045). While RCN significantly increased mean procedure time for AF (+36 minutes, P = 0.003) and atypical atrial flutter cases (+116 minutes, P = 0.016), RCN AF procedure time diminished with increasing number of cases performed. Two cases of tamponade occurred during AF ablation using MCN (2.2%,\u003C\u002Fjats:italic> 2 \u003Cjats:italic>of 91 cases). No tamponade occurred during all 75 AF ablations with RCN.\u003C\u002Fjats:italic>\u003C\u002Fjats:p>\u003Cjats:p> \u003Cjats:bold>\n            \u003Cjats:italic>Conclusion:\u003C\u002Fjats:italic>\n          \u003C\u002Fjats:bold>\n          \u003Cjats:italic>RCN can reduce fluoroscopy time and may reduce complications during catheter ablation. While it may increase total procedure duration, procedure times decrease with increasing operator experience.\u003C\u002Fjats:italic> \u003C\u002Fjats:p>",{"EN":335},"Impact of Remote Magnetic Catheter Navigation on Ablation Fluoroscopy and Procedure Time",{"VOID":337},"18950296",{"VOID":339},"10.1111\u002Fj.1540-8159.2008.01202.x",[126],"https:\u002F\u002Fonlinelibrary.wiley.com\u002Fdoi\u002F10.1111\u002Fj.1540-8159.2008.01202.x",[343,362,381,397,414,432,450,467,485,502],{"id":344,"sortIndex":25,"researcher":24,"roles":345,"affiliations":346,"properties":355,"displayName":359,"givenName":24,"familyName":24},"02ccfad3-49c8-4f61-8a7c-cc35f827e746",[],[347],{"id":348,"sortIndex":25,"affiliation":349,"properties":24},"fe73fc38-94f5-4182-83d8-db1bf8074cc3",{"id":348,"createTime":24,"updateTime":24,"relativeEntities":350,"slug":24,"properties":351,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":354,"statistic":24},[],{"title":352},{"VI":353},"University of California, San Francisco, California USA",[],{"orcid":356,"title":358,"openalex":360},{"VOID":357},"https:\u002F\u002Forcid.org\u002F0000-0002-5269-2155",{"EN":359},"Albert M. 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The purpose of this study was to compare remote magnetic catheter navigation to manual navigation for the ablation of common supraventricular arrhythmias.\u003C\u002Fjats:italic> \u003C\u002Fjats:p>\u003Cjats:p> \u003Cjats:bold>\n            \u003Cjats:italic>Methods and Results:\u003C\u002Fjats:italic>\n          \u003C\u002Fjats:bold>\n          \u003Cjats:italic>Patients with supraventricular arrhythmias due to atrioventricular (AV) nodal reentry, accessory pathways, or undergoing AV junctional ablation for complete heart block were randomized in a 3:1 ratio between magnetic (Niobe system and Helios II catheter, Stereotaxis, Inc., St. Louis, MO) and manual navigation for radiofrequency ablation at 13 centers. The primary endpoint of the study was total fluoroscopic time. Fifty‐six patients were randomized to magnetic navigation and 15 to manual navigation. AV nodal reentry was the most common arrhythmia in both groups. Total fluoroscopy time was reduced in the magnetic navigation group (median 17.8 minutes, interquartile (IQ) range 9.9,27.8 minutes) compared to manual navigation (27.1, IQ 19.0,48.0, P < 0.05). The acute success rates (91% for magnetic and 87% for manual navigation, P > 0.05) did not differ between groups. The number of lesions delivered was less for magnetic navigation (6, IQ 4,9 vs 10, IQ 7, 26, P < 0.05). Total procedure time (median 151, IQ 111, 221 minutes magnetic and 151, IQ 110, 221 minutes manual) and complication rates (5.4% patients magnetic and 6.7% patients manual) were similar between the groups (both P > 0.05).\u003C\u002Fjats:italic> \u003C\u002Fjats:p>\u003Cjats:p> \u003Cjats:bold>\n            \u003Cjats:italic>Conclusions:\u003C\u002Fjats:italic>\n          \u003C\u002Fjats:bold>\n          \u003Cjats:italic>Remote magnetic catheter navigation reduces fluoroscopic time and radiofrequency lesion deliveries for the ablation of common supraventricular arrhythmias compared to manual catheter navigation.\u003C\u002Fjats:italic> \u003C\u002Fjats:p>",{"EN":616},"Remote Magnetic Versus Manual Catheter Navigation for Ablation of Supraventricular Tachycardias: A Randomized, Multicenter Trial",{"VOID":618},"18811813",{"VOID":620},"10.1111\u002Fj.1540-8159.2008.01183.x",[126],"https:\u002F\u002Fonlinelibrary.wiley.com\u002Fdoi\u002F10.1111\u002Fj.1540-8159.2008.01183.x",[624,641,660,679,694],{"id":625,"sortIndex":25,"researcher":24,"roles":626,"affiliations":627,"properties":636,"displayName":638,"givenName":24,"familyName":24},"b44c3bcf-5af9-48f7-8758-4af4a19c4ee9",[],[628],{"id":629,"sortIndex":25,"affiliation":630,"properties":24},"b362fa1c-2b89-437e-97bc-94c8519da0c5",{"id":629,"createTime":24,"updateTime":24,"relativeEntities":631,"slug":24,"properties":632,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":635,"statistic":24},[],{"title":633},{"VI":634},"Virginia Commonwealth University Medical Center, Richmond, Virginia,",[],{"title":637,"openalex":639},{"EN":638},"Mark A. 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The procedural duration and fluoroscopic exposure to patients and medical staff were recorded and compared among 43 ablation procedures for PAF, 20 for common atrial flutter, and 16 for accessory pathways. Patient radiation exposure was measured by dosimeters placed over the xyphoid, while that of physicians and nurses was measured by dosimeters placed outside and inside the lead apron. The mean fluoroscopy time was\u003C\u002Fjats:italic>\n          \u003Cjats:styled-content>\n              \u003Cjats:italic>57 ± 30\u003C\u002Fjats:italic>\n            \u003C\u002Fjats:styled-content>\n          \u003Cjats:italic>minutes for PAF,\u003C\u002Fjats:italic>\n          \u003Cjats:styled-content>\n              \u003Cjats:italic>20 ± 10\u003C\u002Fjats:italic>\n            \u003C\u002Fjats:styled-content>\n          \u003Cjats:italic>minutes for common flutter, and\u003C\u002Fjats:italic>\n          \u003Cjats:styled-content>\n              \u003Cjats:italic>22 ± 21\u003C\u002Fjats:italic>\n            \u003C\u002Fjats:styled-content>\n          \u003Cjats:italic>minutes for accessory pathway ablation. The patient median radiation exposure was 1110μSv for PAF, compared with 500 μSv for common flutter and 560 μSv for accessory pathway ablation (P < 0.01). The median radiation exposure to physician and nurse inside the lead apron were, respectively, 2 μSv and 3 μSv for PAF, 1 μSv and 2 μSv for common flutter, and <0.5 μSv and 3 μSv for accessory pathway ablations. RF catheter ablation for PAF was associated with prolonged fluoroscopy times and a twofold higher radiation exposure to the patient and physician compared with other ablation procedures. Assuming 300 procedures\u002Fyear, radiation exposure to the medical staff was below the upper recommended annual dose limit. (PACE 2003; 26[Pt. 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There has been much enthusiasm for this technology as a means to reduce radiation exposure to the patient and physician alike, and potentially decrease risks associated with catheter manipulation by less experienced operators. However, there are limited data regarding the acute, intermediate, and long‐term results and procedural characteristics from ablation procedures utilizing this system. We present a review of the outcomes and procedural data available to date.\u003C\u002Fjats:italic>\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:italic>(PACE 2012; 35:1021–1034)\u003C\u002Fjats:italic>\u003C\u002Fjats:p>",{"EN":1075},"Catheter Ablation Utilizing Remote Magnetic Navigation: A Review of Applications and Outcomes",{"VOID":1077},"22510160",{"VOID":1079},"10.1111\u002Fj.1540-8159.2012.03382.x","2025-02-03T20:29:36.752+00:00",[126],"https:\u002F\u002Fonlinelibrary.wiley.com\u002Fdoi\u002F10.1111\u002Fj.1540-8159.2012.03382.x",[1084,1103,1120,1137,1154],{"id":1085,"sortIndex":25,"researcher":24,"roles":1086,"affiliations":1087,"properties":1096,"displayName":1100,"givenName":24,"familyName":24},"07efc3da-366f-461f-a916-8c0196819cdc",[],[1088],{"id":1089,"sortIndex":25,"affiliation":1090,"properties":24},"a1241333-08b1-4f1e-bfef-16f06dbbed51",{"id":1089,"createTime":24,"updateTime":24,"relativeEntities":1091,"slug":24,"properties":1092,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":1095,"statistic":24},[],{"title":1093},{"EN":1094},"From the UCLA Cardiac Arrhythmia Center, David Geffen School of Medicine at UCLA, Los Angeles, California",[],{"orcid":1097,"title":1099,"openalex":1101},{"VOID":1098},"https:\u002F\u002Forcid.org\u002F0000-0002-2541-5726",{"EN":1100},"Jason S. 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A, 2010, Atrial fibrillation ablation: A single center comparison between remote magnetic navigation, cryoballoon and conventional manual pulmonary vein isolation, Indian Pacing Electrophysiol J, 10, 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J, 2008, Mapping of atrial tachycardia by remote magnetic navigation in postoperative patients with congenital heart disease, J Cardiovasc Electrophysiol, 21, 751, 10.1111\u002Fj.1540-8167.2009.01697.x",{"doi":1319},"10.1111\u002Fj.1540-8167.2009.01697.x",{"id":24,"text":1321,"url":24,"identifiers":1322},"10.1111\u002Fj.1540-8159.2008.02283.x",{"doi":1321},{"id":24,"text":1324,"url":24,"identifiers":1325},"10.2174\u002F157340309787048059",{"doi":1324},{"id":24,"text":339,"url":24,"identifiers":1327},{"doi":339},{"id":24,"text":1329,"url":24,"identifiers":1330},"10.1111\u002Fj.1540-8167.2010.01806.x",{"doi":1329},{"id":24,"text":1332,"url":24,"identifiers":1333},"10.1111\u002Fj.1540-8167.2010.01777.x",{"doi":1332},{"id":24,"text":1335,"url":24,"identifiers":1336},"10.7326\u002F0003-4819-155-7-201110040-00004",{"doi":1335},{"id":24,"text":1338,"url":24,"identifiers":1339},"10.1016\u002Fj.jacc.2009.04.050",{"doi":1338},{"id":24,"text":1341,"url":24,"identifiers":1342},"10.1093\u002Feuropace\u002Feuq501",{"doi":1341},{"id":1344,"createTime":1345,"updateTime":1346,"relativeEntities":1347,"slug":1348,"properties":1349,"entityType":121,"verifyStatus":122,"verifyTime":1345,"verifyNote":124,"languages":1365,"translateLanguages":1366,"viewCount":25,"primaryUrl":1367,"fullTextUrl":24,"authors":1368,"publicationType":168,"publisherRelationship":1433,"citationCount":1481,"citationInfo":1482,"publishDate":1485,"publishYear":1483,"citationAnalyzeStatus":23,"lastCitationAnalyze":24,"indexDatabases":1486,"openAccess":24,"references":1487,"isForceReanalyzing":321},"bb3bec9f-3bc0-4afc-8d17-4068a151220e","2024-10-12T20:34:16.410+00:00","2025-01-26T21:06:21.395+00:00",[],"Implantable-Defibrillation-Eight-Years-Clinical-Experience",{"mag":1350,"keywords":1352,"openalex":1353,"abstract":1355,"title":1358,"pm":1361,"doi":1363},{"VOID":1351},"1997142948",{"VI":108},{"VOID":1354},"W1997142948",{"VI":1356,"EN":1357},"\u003Cjats:p>Việc cấy ghép máy khử rung tim tự động đầu tiên được thực hiện vào tháng 2 năm 1980. Việc tích hợp khả năng cắt điện tim vào năm 1982 đã dẫn đến máy khử rung tim cấy ghép tự động AICD™. Giữa ngày 1 tháng 4 năm 1982 và 15 tháng 4 năm 1988, 3610 bệnh nhân tại 236 trung tâm ở Hoa Kỳ và 84 trung tâm quốc tế đã nhận được máy phát xung AICD. Đối tượng bệnh nhân gồm có 2904 nam và 683 nữ với tình trạng loạn nhịp tim thất tái phát và\u002Fhoặc rung thất, độ tuổi trung bình là 59 tuổi (từ 9 đến 96 tuổi). Chẩn đoán chính được báo cáo cho nhóm bệnh nhân này là: bệnh động mạch vành (63,5%), bệnh cơ tim không thiếu máu (12,9%), khác (6,4%) và không xác định (17,2%). Phân suất tống máu thất trái được báo cáo trung bình là 32,8%. Thời gian theo dõi trung bình là 12,2 tháng (dao động từ 0 đến 72 tháng). Trong 385 ca tử vong, 94 ca (24%) là đột ngột. Tỷ lệ sống sót tích lũy (±S.E.) từ tử vong tim đột ngột (SCD) lần lượt là 98.0 ± 0.3%, 96.5 ± 0.5%, 95.2 ± 0.7%, 93.7 ± 1.0%, 93.7 ± 1.0% và 89.7 ± 4.0% ở 12, 24, 36, 48, 60 và 72 tháng. Tỷ lệ tử vong phẫu thuật (30 ngày) là 2.5%. Các tác dụng phụ\u002Fbiến chứng được báo cáo tương tự như máy tạo nhịp. Đến nay, 33% bệnh nhân đã nhận được các cú sốc tự phát từ thiết bị. Tỷ lệ sống sót của máy phát xung AICD từ các lỗi điện và cơ học là 92.8 ± 0.5%, 88.4 ± 0.7%, 86.7 ± 0.8% và 86.4 ± 0.9% ở 12, 18, 24 và 30 tháng. Phân tích dữ liệu cho thấy AICD đã có tác động đáng kể đến sự sống sót của bệnh nhân khỏi SCD.\u003C\u002Fjats:p>","\u003Cjats:p>Implantation of the first automatic defibrillator occurred in February 1980. Incorporation of cardioversion capability in 1982 resulted in the AICD™ automatic implantable cardioverter defibrillator. Between April 1, 1982 and April 15, 1988, 3610 patients in 236 U.S. and 84 international centers received AICD pulse generators. Patient population consisted of 2904 males and 683 females with recurrent ventricular tachycardia and\u002For fibrillation, mean age 59 yrs. (range 9–96 yrs.). Primary diagnoses reported for the patient group were: coronary artery disease (63.5%), nonischemic cardiomyopathy (12.9%), other (6.4%) and unspecified (17.2%). Mean reported LV ejection fraction was 32.8%. Follow‐up averaged 12.2 mo. (range 0–72 mo.). Of 385 deaths, 94 (24%) were sudden. Cumulative percentage survival (±S.E.) from sudden cardiac death (SCD) was 98.0 ± 0.3%, 96.5 ± 0.5%, 95.2 ± 0.7%, 93.7 ± 1.0%, 93.7 ± 1.0% and 89.7 ± 4.0% at 12, 24, 36, 48, 60 and 72 months, respectively. Operative mortality (30 days) was 2.5%. Reported side effects\u002Fcomplications were similar to those of pacemakers. To date, 33% of the patients received spontaneous device countershocks. AICD pulse generator survival from electrical and mechanical failures was 92.8 ± 0.5%, 88.4 ± 0.7%, 86.7 ± 0.8% and 86.4 ± 0.9% at 12, 18, 24 and 30 mos. Data analysis demonstrates that the AICD has had a significant impact on patient survival from SCD.\u003C\u002Fjats:p>",{"EN":1359,"VI":1360},"Implantable Defibrillation: Eight Years Clinical Experience","Đặt máy khử rung tim cấy ghép: Kinh nghiệm lâm sàng tám năm",{"VOID":1362},"2463587",{"VOID":1364},"10.1111\u002Fj.1540-8159.1988.tb06349.x",[126],[128],"https:\u002F\u002Fonlinelibrary.wiley.com\u002Fdoi\u002F10.1111\u002Fj.1540-8159.1988.tb06349.x",[1369,1386,1403,1418],{"id":1370,"sortIndex":25,"researcher":24,"roles":1371,"affiliations":1372,"properties":1381,"displayName":1383,"givenName":24,"familyName":24},"e07fee50-1b9a-4e89-b207-0cc8e20ba858",[],[1373],{"id":1374,"sortIndex":25,"affiliation":1375,"properties":24},"99cfe6af-019b-4dcf-a2ec-105739351032",{"id":1374,"createTime":24,"updateTime":24,"relativeEntities":1376,"slug":24,"properties":1377,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":1380,"statistic":24},[],{"title":1378},{"EN":1379},"Department of Scientific Studies, Cardiac Pacemakers, Inc., St. Paul, MN 55112-5798.",[],{"title":1382,"openalex":1384},{"EN":1383},"Andrea Thomas",{"VOID":1385},"A5065509005",{"id":1387,"sortIndex":154,"researcher":24,"roles":1388,"affiliations":1389,"properties":1398,"displayName":1400,"givenName":24,"familyName":24},"dc0cdae7-1ffd-43d2-943e-8eeae166c41c",[],[1390],{"id":1391,"sortIndex":25,"affiliation":1392,"properties":24},"4f1f6293-ec1e-453e-8692-f475b69b0378",{"id":1391,"createTime":24,"updateTime":24,"relativeEntities":1393,"slug":24,"properties":1394,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":1397,"statistic":24},[],{"title":1395},{"EN":1396},"Departments of Scientific Studies, Clinical Programs, Biostatistics and Medical Records, Cardiac Pacemakers, Inc., St. Paul, MN",[],{"title":1399,"openalex":1401},{"EN":1400},"Suzan A. 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\u003Cjats:italic>Các rối loạn dẫn truyền tâm nhĩ thường gặp ở những người cao tuổi và\u002Fhoặc những người có bệnh lý tim cấu trúc, chủ yếu là bệnh van tim hai lá, bệnh cơ tim phì đại và tăng huyết áp. Các bất thường điện sinh lý và điện cơ phát sinh từ tình trạng này có liên quan đến nguy cơ cao hơn của các loạn nhịp tim tâm nhĩ paroxysmal hoặc kéo dài, bao gồm rung tâm nhĩ, flutter điển hình hoặc không điển hình, hoặc các dạng loạn nhịp tâm nhĩ khác. Mối liên hệ như vậy không phải là ngẫu nhiên vì các bất thường trong dẫn truyền nội và giữa tâm nhĩ làm chậm và phá vỡ (phân tán không gian và thời gian) kích hoạt điện, điều này thúc đẩy việc khởi đầu và duy trì các mạch tái nhập. Các can thiệp điều trị phòng ngừa tạo ra các hiệu ứng khác nhau, đôi khi trái ngược như với hiệu ứng proarrhythmic của các loại thuốc chống loạn nhịp tim nhóm I. Tương tự, kích thích tâm nhĩ có thể thúc đẩy proarrhythmias hoặc có hiệu ứng chống loạn nhịp tùy thuộc vào vị trí và kiểu kích thích. Kích thích tâm nhĩ đa khu vực được thiết kế nhằm cải thiện, trong khả năng có thể, kích hoạt bất thường do các rối loạn dẫn truyền nội hoặc giữa tâm nhĩ tự phát hoặc do kích thích tâm nhĩ đơn vị, đây là các tình huống thường gây ra các loạn nhịp khó điều trị. Việc đồng bộ hóa điện tâm nhĩ cũng có thể được sử dụng để sửa chữa các bất thường cơ học như sự thiếu đồng bộ giữa tâm nhĩ và tâm thất trái do các chậm trễ trong dẫn truyền nội tâm nhĩ.\u003C\u002Fjats:italic> \u003C\u002Fjats:p>","\u003Cjats:p> \u003Cjats:italic>Atrial conduction disorders are frequent in elderly subjects and\u002For those with structural heart diseases, mainly mitral valve disease, hyperthrophic cardiomyopathies, and hypertension. The resultant electrophysiological and electromechanical abnormalities are associated with a higher risk of paroxysmal or persistent atrial tachyarrhythmias, either atrial fibrillation, typical or atypical flutter or other forms of atrial tachycardias. Such an association is not fortuitous because intra‐ and interatrial conduction abnormalities delays disrupt (spatial and temporal dispersion) electrical activation, thus promoting the initiation and perpetuation of reentrant circuits. Preventive therapeutic interventions induce variable, sometimes paradoxical effects as with the proarrhythmic effect of class I antiarrhythmic drugs. Similarly, atrial pacing may promote proarrhythmias or an antiarrhythmic effect according to the pacing site(s) and mode. Multisite atrial pacing was conceived to correct, as much as possible, abnormal activation induced by spontaneous intra‐ or interatrial conduction disorders or by single site atrial pacing, which are situations responsible for commonly refractory arrhythmias. Atrial electrical resynchronization can also be used to correct mechanical abnormalities like left heart AV dyssynchrony resulting from intraatrial conduction delays. 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Mã này được viết tắt là NBC (cho “Mã Chung NASPE\u002FBPEC”) và được phát triển để cho phép mở rộng khái niệm mã chung cho các máy tạo nhịp có tốc độ thoát được điều khiển liên tục bằng cách theo dõi một số biến sinh lý, thay vì được xác định bằng các khoảng thời gian thoát cố định đo từ các kích thích hoặc các sự kiện khử cực được phát hiện, và cho các thiết bị chống nhịp tim nhanh bao gồm các thiết bị đồng bộ và máy khử rung tim. Mã NASPE\u002FBPEC bao gồm một chữ \"R\" ở vị trí thứ tư để chỉ ra sự điều chỉnh tỷ lệ (tạo nhịp tỷ lệ thích ứng), và một trong bốn chữ cái ở vị trí thứ năm để chỉ ra sự hiện diện của khả năng tạo nhịp chống nhịp tim nhanh hoặc của các chức năng đồng bộ hoặc khử rung tim.\u003C\u002Fjats:p>","\u003Cjats:p>A new generic pacemaker code, derived from and compatible with the Revised ICHD Code, was proposed jointly by the North American Society of Pacing and Electrophysiology (NASPE) Mode Code Committee and the British Pacing and Electrophysiology Croup (BPEC), and has been adopted by the NASPE Board of Trustees. It is abbreviated as the NBC (for “NASPE\u002FBPEC Generic”) Code, and was developed to permit extension of the generic‐code concept to pacemakers whose escape rate is continuously controlled by monitoring some physiologic variable, rather than determined by fixed escape intervals measured from stimuli or sensed depolarizations, and to antitachyarrhythmia devices including cardioverters and defibrillators. 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Pacemaker Study Group, Inter‐Society Commission for Heart Disease Resources (ICHD), Circulation, 50, A21, 10.1161\u002Fcirc.50.4.page5",{"doi":2423},"10.1161\u002Fcirc.50.4.page5",{"id":24,"text":2425,"url":24,"identifiers":2426},"10.1111\u002Fj.1540-8159.1981.tb03719.x",{"doi":2425},{"id":24,"text":2428,"url":24,"identifiers":2429},"Parsonnet V, 1983, Implantable cardiac pacemakers: Status report and resource guidelines, 1982. Pacemaker Study Group, Inter‐Society Commission for Heart Disease Resources, Circulation, 68, 227A",{},{"id":24,"text":2431,"url":24,"identifiers":2432},"10.1111\u002Fj.1540-8159.1984.tb04924.x",{"doi":2431},{"id":24,"text":2434,"url":24,"identifiers":2435},"Bernstein AD, 1985, Modern Cardiac Pacing, 307",{},{"id":24,"text":2437,"url":24,"identifiers":2438},"10.1111\u002Fj.1540-8159.1981.tb03718.x",{"doi":2437},{"id":24,"text":2440,"url":24,"identifiers":2441},"Bernstein AD.A new generic code for antitachyarrhythmia devices. (In preparation.).",{},{"id":2443,"createTime":2444,"updateTime":2445,"relativeEntities":2446,"slug":2447,"properties":2448,"entityType":121,"verifyStatus":122,"verifyTime":2444,"verifyNote":124,"languages":2464,"translateLanguages":2465,"viewCount":25,"primaryUrl":2466,"fullTextUrl":24,"authors":2467,"publicationType":168,"publisherRelationship":2581,"citationCount":2631,"citationInfo":2632,"publishDate":2635,"publishYear":2633,"citationAnalyzeStatus":23,"lastCitationAnalyze":24,"indexDatabases":2636,"openAccess":24,"references":2637,"isForceReanalyzing":321},"1237081e-d307-4fcb-8ae6-5bff5ad22117","2024-10-10T09:27:05.674+00:00","2025-01-26T21:03:28.889+00:00",[],"Anatomical-Mechanisms-Explaining-Damage-to-Pacemaker-Leads-Defibrillator-Leads-and-Failure-of-Central-Venous-Catheters-Adjacent-to-the-Sternoclavicular-Joint",{"mag":2449,"keywords":2451,"openalex":2452,"abstract":2454,"title":2457,"pm":2460,"doi":2462},{"VOID":2450},"2010896345",{"VI":108},{"VOID":2453},"W2010896345",{"VI":2455,"EN":2456},"\u003Cjats:p>Tài liệu cho thấy khoảng 93% tổng số trường hợp gãy dây pacemaker xảy ra ở đoạn dây bên phía bên ngoài của điểm vào tĩnh mạch, và sự chèn ép costoclavicular được cho là nguyên nhân. Mặc dù các mạch máu có thể bị chèn ép do chuyển động của xương đòn, nghiên cứu của chúng tôi cho thấy tổn thương dây và catheter trong khu vực đó là do sự kẹt mô mềm hơn là do tiếp xúc với xương. Phẫu tích tám xác người với mười dây cho thấy hai dây vào tĩnh mạch đầu, và không được đưa vào nghiên cứu. Trong số bốn dây còn lại, bốn dây đi qua cơ dưới đòn, hai dây qua dây chằng costoclavicular, và hai dây qua cả hai cấu trúc này trước khi vào tĩnh mạch dưới đòn, tĩnh mạch cảnh trong hoặc tĩnh mạch brachiocephalic. Các nghiên cứu giải phẫu chứng minh rằng sự kẹt bởi cơ dưới đòn hoặc dây chằng costoclavicular có thể gây gấp đi gấp lại liên tục của các dây trong quá trình vận động của đai vai. Cineradiology của các bệnh nhân với sự tắc nghẽn catheter phụ thuộc vào vị trí đã xác nhận sự kẹt bởi cơ dưới đòn. Sự kẹt mô mềm tạo ra một tải trọng tĩnh lên các dây và catheter, và sự gấp lại liên tục quanh điểm kẹt có thể là nguyên nhân gây tổn thương trước đây được cho là do sự chèn ép cycle costoclavicular.\u003C\u002Fjats:p>","\u003Cjats:p>The literature suggests that approximately 93% of all pacemaker lead fractures occur in the segment of the lead lateral to the venous entry, and costoclavicular compression has been implicated. While blood vessels can be compressed by movements of the clavicle, our research suggests that lead and catheter damage in that region is caused by soft tissue entrapment rather than bony contact. Dissection of eight cadavers with ten leads revealed that two entered the cephalic vein, and were not included in the study. Of the other eight leads, four passed through the subclavius muscle, two through the costoclavicular ligament, and two through both these structures before entering the subclavian, internal jugular, or brachiocephalic vein. Anatomical studies demonstrated that entrapment by the subclavius muscle or the costoclavicular ligament could cause repeated flexing of leads during movements of the pectoral girdle. Cineradiology of patients with position dependent catheter occlusion confirmed entrapment by the subclavius muscle. Soft tissue entrapment imposes a static load upon leads and catheters, and repeated flexure about the point of entrapment may be responsible for damage previously ottributed to cyclic costoclavicular compression.\u003C\u002Fjats:p>",{"EN":2458,"VI":2459},"Anatomical Mechanisms Explaining Damage to Pacemaker Leads, Defibrillator Leads, and Failure of Central Venous Catheters Adjacent to the Sternoclavicular Joint","Cơ chế giải thích sự tổn thương dây pacemaker, dây sốc điện và sự thất bại của các catheter tĩnh mạch trung ương gần khớp ức-clavicle",{"VOID":2461},"7681196",{"VOID":2463},"10.1111\u002Fj.1540-8159.1993.tb01607.x",[126],[128],"https:\u002F\u002Fonlinelibrary.wiley.com\u002Fdoi\u002F10.1111\u002Fj.1540-8159.1993.tb01607.x",[2468,2485,2502,2519,2534,2549,2564],{"id":2469,"sortIndex":25,"researcher":24,"roles":2470,"affiliations":2471,"properties":2480,"displayName":2482,"givenName":24,"familyName":24},"59c1c3a5-20dd-4435-968c-e1c2a659e6aa",[],[2472],{"id":2473,"sortIndex":25,"affiliation":2474,"properties":24},"2c04a4d4-34c5-4050-b8b4-ab56664a94f8",{"id":2473,"createTime":24,"updateTime":24,"relativeEntities":2475,"slug":24,"properties":2476,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":2479,"statistic":24},[],{"title":2477},{"EN":2478},"Department of Cell Biology and Neuroanatomy University of Minnesota, Minneapolis 55455.",[],{"title":2481,"openalex":2483},{"EN":2482},"Jean E. Magney",{"VOID":2484},"A5035692908",{"id":2486,"sortIndex":154,"researcher":24,"roles":2487,"affiliations":2488,"properties":2497,"displayName":2499,"givenName":24,"familyName":24},"2ff37c8c-f62d-414d-ad50-e5d38d18bd2a",[],[2489],{"id":2490,"sortIndex":25,"affiliation":2491,"properties":24},"a4905782-9168-4b84-896e-fb6cca8cea3a",{"id":2490,"createTime":24,"updateTime":24,"relativeEntities":2492,"slug":24,"properties":2493,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":2496,"statistic":24},[],{"title":2494},{"EN":2495},"#Cardiac Pacemakers, Inc., St, Paul, Minnesota",[],{"title":2498,"openalex":2500},{"EN":2499},"Denis Flynn",{"VOID":2501},"A5036917316",{"id":2503,"sortIndex":383,"researcher":24,"roles":2504,"affiliations":2505,"properties":2514,"displayName":2516,"givenName":24,"familyName":24},"ce3008f8-7e4d-4aca-a7f8-2d7344fb0d9c",[],[2506],{"id":2507,"sortIndex":25,"affiliation":2508,"properties":24},"b1b7a0bb-a375-453e-a017-8ecfb25be524",{"id":2507,"createTime":24,"updateTime":24,"relativeEntities":2509,"slug":24,"properties":2510,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":2513,"statistic":24},[],{"title":2511},{"VI":2512},"University of Minnesota, Minneapolis, Minnesota",[],{"title":2515,"openalex":2517},{"EN":2516},"Jonathan A. 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Issues (Intermedics Inc.)December 1987 pp.1–2.",{},{"id":24,"text":2648,"url":24,"identifiers":2649},"Stokes K, 1987, A possible new complication of subclavian stick: Conductor fracture, (abstract), PACE, 10, 748",{},{"id":24,"text":2651,"url":24,"identifiers":2652},"10.1111\u002Fj.1540-8159.1989.tb05044.x",{"doi":2651},{"id":24,"text":2654,"url":24,"identifiers":2655},"10.1111\u002Fj.1540-8159.1988.tb06276.x",{"doi":2654},{"id":24,"text":2657,"url":24,"identifiers":2658},"Kallinen LM, 1990, Electrical Therapy for Cardiac Arrhythmias, 320",{},{"id":24,"text":2660,"url":24,"identifiers":2661},"10.1111\u002Fj.1540-8159.1984.tb05654.x",{"doi":2660},{"id":24,"text":2663,"url":24,"identifiers":2664},"Sholder J, 1990, Technical memorandum; Clinical and technical considerations of bipolar coaxial pacing leads, 14",{},{"id":24,"text":2666,"url":24,"identifiers":2667},"10.1111\u002Fj.1540-8159.1988.tb05011.x",{"doi":2666},{"id":24,"text":2669,"url":24,"identifiers":2670},"10.1111\u002Fj.1540-8159.1986.tb06600.x",{"doi":2669},{"id":24,"text":2672,"url":24,"identifiers":2673},"10.1097\u002F00000658-195105000-00002",{"doi":2672},{"id":24,"text":2675,"url":24,"identifiers":2676},"10.1016\u002FS0002-9610(88)80172-7",{"doi":2675},{"id":24,"text":2678,"url":24,"identifiers":2679},"10.1097\u002F00000658-197705000-00011",{"doi":2678},{"id":24,"text":2681,"url":24,"identifiers":2682},"10.1177\u002F000331978904000901",{"doi":2681},{"id":24,"text":2684,"url":24,"identifiers":2685},"10.1016\u002F0002-9610(86)90141-8",{"doi":2684},{"id":24,"text":2687,"url":24,"identifiers":2688},"10.1016\u002F0002-9610(84)90340-4",{"doi":2687},{"id":24,"text":2690,"url":24,"identifiers":2691},"Fitzpatrick AP, 1985, Pacemaker revision for ventricular endocardial electrode failure; Incidence and type of failure in a major implanting center, abstact) PACE 1991, 14, 639",{},{"id":24,"text":2693,"url":24,"identifiers":2694},"Borja AR, 1970, A safe way to perform infraclavicular subclavian vein catheterization, Surg Gynecol Obstet, 130, 673",{},{"id":24,"text":2696,"url":24,"identifiers":2697},"Friesen A, 1977, Percutaneous insertion of a permanent transvenous pacemaker electrode through the subclavian vein, Can J Surg, 20, 131",{},{"id":24,"text":2699,"url":24,"identifiers":2700},"Holmes DR, 1990, Electrical Therapy for Cardiac Arrhythmias, 173",{},{"id":24,"text":2702,"url":24,"identifiers":2703},"10.1007\u002FBF02096462",{"doi":2702},{"id":24,"text":2705,"url":24,"identifiers":2706},"Bearn JG., 1967, Direct observations on the function of the capsule of the sternoclavicular joint in clavicular support, J Anat, 101, 159",{},{"id":24,"text":2708,"url":24,"identifiers":2709},"Telford ED, 1948, Pressure at the cervicobrachial junction, J Bone Joint Surg, 30, 249, 10.1302\u002F0301-620X.30B2.249",{"doi":2710},"10.1302\u002F0301-620X.30B2.249",{"id":24,"text":2712,"url":24,"identifiers":2713},"Moseley HF., 1968, The clavicle: Its anatomy and function, Clin Orthop, 58, 17",{},{"id":24,"text":2715,"url":24,"identifiers":2716},"Steindler A., 1970, Lecture XXVI in Kinesioiogy of the Human Body under Normal and Pathological Conditions",{},{"id":24,"text":2718,"url":24,"identifiers":2719},"Inman VT, 1944, Observations on the function of the shoulder joint, J Bone Joint Surg, 1",{},{"id":24,"text":2721,"url":24,"identifiers":2722},"10.1177\u002F0148607185009006754",{"doi":2721},{"id":24,"text":2724,"url":24,"identifiers":2725},"10.2214\u002Fajr.133.3.467",{"doi":2724},{"id":24,"text":2727,"url":24,"identifiers":2728},"10.1001\u002Farchsurg.1965.01320080052011",{"doi":2727},{"id":24,"text":2730,"url":24,"identifiers":2731},"10.1055\u002Fs-2007-1020206",{"doi":2730},{"id":24,"text":2733,"url":24,"identifiers":2734},"Conklin E, 1975, Four hundred consecutive patients with permanent transvenous pacemakers, J Thor Cardiovasc Snrg, 69, 1, 10.1016\u002FS0022-5223(19)41605-X",{"doi":2735},"10.1016\u002FS0022-5223(19)41605-X",{"id":24,"text":2737,"url":24,"identifiers":2738},"Grogler FM, 1975, Complications of permanent transvenons cardiac pacing,, J Thorac Cardiovasc Surg, 69, 895, 10.1016\u002FS0022-5223(19)40417-0",{"doi":2739},"10.1016\u002FS0022-5223(19)40417-0",{"id":24,"text":2741,"url":24,"identifiers":2742},"10.1111\u002Fj.1540-8159.1986.tb05427.x",{"doi":2741},{"id":24,"text":2744,"url":24,"identifiers":2745},"Troup PJ., 1989, Current Problems in Cardiology, 673",{},{"id":24,"text":2747,"url":24,"identifiers":2748},"10.1016\u002F0002-9149(90)90695-W",{"doi":2747},{"id":24,"text":2750,"url":24,"identifiers":2751},"10.1001\u002Farchsurg.1967.01330140158036",{"doi":2750},{"id":24,"text":2753,"url":24,"identifiers":2754},"10.1016\u002FS0140-6736(63)90791-8",{"doi":2753},{"id":24,"text":2756,"url":24,"identifiers":2757},"10.1159\u002F000145132",{"doi":2756},{"id":24,"text":2759,"url":24,"identifiers":2760},"10.1111\u002Fj.1540-8159.1983.tb04390.x",{"doi":2759},{"id":24,"text":2762,"url":24,"identifiers":2763},"10.1001\u002Farchsurg.1971.01350040115023",{"doi":2762},{"id":24,"text":2765,"url":24,"identifiers":2766},"10.1016\u002F0003-4975(90)90042-5",{"doi":2765},{"id":2768,"createTime":2769,"updateTime":2770,"relativeEntities":2771,"slug":2772,"properties":2773,"entityType":121,"verifyStatus":122,"verifyTime":2769,"verifyNote":124,"languages":2788,"translateLanguages":2789,"viewCount":25,"primaryUrl":2790,"fullTextUrl":24,"authors":2791,"publicationType":168,"publisherRelationship":2937,"citationCount":2987,"citationInfo":2988,"publishDate":2992,"publishYear":2989,"citationAnalyzeStatus":23,"lastCitationAnalyze":24,"indexDatabases":2993,"openAccess":24,"references":2994,"isForceReanalyzing":321},"46fcb10d-8c87-42b6-ae97-6596a7f0a0e1","2024-10-17T07:06:41.065+00:00","2025-01-26T21:02:31.296+00:00",[],"P-Wave-Dispersion-on-12-Lead-Electrocardiography-in-Patients-with-Paroxysmal-Atrial-Fibrillation",{"mag":2774,"keywords":2776,"openalex":2777,"abstract":2779,"title":2782,"pm":2785,"doi":2787},{"VOID":2775},"2086724701",{"VI":108},{"VOID":2778},"W2086724701",{"VI":2780,"EN":2781},"\u003Cjats:p>Thời gian dẫn truyền trong nhĩ và giữa hai nhĩ kéo dài và sự lan truyền không đồng nhất của các xung động xoang đã được chỉ ra ở bệnh nhân mắc hội chứng rung nhĩ. Gần đây, phân tán sóng P (PWD), được cho là phản ánh sự dẫn truyền không đồng nhất của nhĩ, đã được đề xuất là hữu ích trong việc dự đoán rung nhĩ kịch phát (PAF). Chúng tôi đã nghiên cứu 90 bệnh nhân liên tiếp (46 nam, 44 nữ; tuổi trung bình 55 ± 13 năm) có tiền sử rung nhĩ kịch phát vô căn và 70 đối tượng khỏe mạnh (42 nam, 28 nữ; tuổi trung bình 53 ± 14 năm). Thời gian của sóng P được tính toán ở tất cả 12 đạo trình của điện tâm đồ bề mặt. Sự khác biệt giữa thời gian sóng P tối đa và tối thiểu được tính toán và sự khác biệt này được định nghĩa là phân tán sóng P (PWD = Pmax ‐ Pmin). Tất cả bệnh nhân và nhóm đối chứng cũng được đánh giá bằng siêu âm tim để đo đường kính nhĩ trái và phân suất tống máu thất trái (LVEF). Không có sự khác biệt về giới tính (P = 0,26), tuổi tác (P = 0,12), LVEF (66 ± 4% so với 67 ± 5%, P = 0,8) và đường kính nhĩ trái (36 ± 4 mm so với 34 ± 6 mm, P = 0,13) giữa bệnh nhân và đối chứng. Thời gian sóng P tối đa được phát hiện cao hơn đáng kể ở bệnh nhân có tiền sử PAF (116 ± 17 ms) so với đối chứng (101 ± 11 ms, P \u003C 0,001). Phân tán sóng P cũng cao hơn một cách đáng kể ở bệnh nhân so với đối chứng (44 ± 15 ms so với 27 ± 10 ms, P \u003C 0,001). Có một sự tương quan yếu giữa tuổi tác và phân tán sóng P (r = 0,27, P \u003C 0,001). Một giá trị P tối đa là 106 ms phân tách bệnh nhân có PAF khỏi các đối tượng đối chứng với độ nhạy 83%, độ đặc hiệu 72% và độ chính xác dự đoán dương tính 79%. Giá trị phân tán sóng P là 36 ms đã phân tách bệnh nhân khỏi các đối tượng đối chứng với độ nhạy 77%, độ đặc hiệu 82% và độ chính xác dự đoán dương tính 85%. Kết luận, thời gian sóng P tối đa và phân tán sóng P được tính toán trên một điện tâm đồ bề mặt tiêu chuẩn là các dấu hiệu điện tâm đồ đơn giản có thể được sử dụng để xác định những bệnh nhân mắc rung nhĩ kịch phát vô căn.\u003C\u002Fjats:p>","\u003Cjats:p>The prolongation of intraatrial and interatrial conduction time and the inhomogeneous propagation of sinus impulses have been shown in patients with atrial fibrillation. Recently P wave dispersion (PWD), which is believed to reflect inhomogeneous atrial conduction, has been proposed as being useful for the prediction of paroxysmal atrial fibrillation (PAF). Ninety consecutive patients (46 men, 44 women; aged 55 ± 13 years) with a history of idiopathic PAF and 70 healthy subjects (42 men, 28 women; mean age, 53 ± 14 years) were studied. The P wave duration was calculated in all 12 leads of the surface ECC. The difference between the maximum and minimum P wave duration was calculated and this difference was defined as P wave dispersion (PWD = Pmax ‐ Pmin). All patients and controls were also evaluated by echocardiography to measure the left atrial diameter and left ventricular ejection fraction (LVEF). There was no difference between patients and controls in gender (P = 0.26), age (P = 0.12), LVEF (66 ± 4% vs 67 ± 5%, P = 0.8) and left atrial diameter (36 ± 4 mm vs 34 ± 6 mm, P = 0.13). P maximum duration was found to be significantly higher in patients with a history of PAF (116 ± 17 ms) than controls (101 ±11 ms. P < 0.001). P wave dispersion was also significantly higher in patients than in controls (44 ± 15 ms vs 27 ± 10 ms, P < 0.001). There was a weak correlation between age and P wave dispersion (r = 0.27, P < 0.001). A P maximum value of 106 ms separated patients with PAF from control subjects with a sensitivity of 83%, a specificity of 72%, and a positive predictive accuracy of 79%. A P wave dispersion value of 36 ms separated patients from control subjects with a sensitivity of 77%, a specificity of 82%, and a positive predictive accuracy of 85%. In conclusion, P maximum duration and P wave dispersion calculated on a standard surface ECG are simple ECG markers that could be used to identify the patients with idiopathic paroxysmal atrial fibrillation.\u003C\u002Fjats:p>",{"EN":2783,"VI":2784},"P Wave Dispersion on 12‐Lead Electrocardiography in Patients with Paroxysmal Atrial Fibrillation","Phân Tán Sóng P trên Điện Tâm Đồ 12 Đầu Dẫn ở Bệnh Nhân Mắc Rung Nhĩ Kịch 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