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Journal of Medicine and Pharmacy","Tạp chí Y Dược học Cần Thơ",{"EN":487,"VI":488},"\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">04\u002F10\u002F2015 Ministry of Information and Communications allowed Can Tho journal of medicine and pharmacy to operate (102 \u002FGP-BTTTT)\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">07\u002F16\u002F2015 Can Tho journal of medicine and pharmacy is internationally recognized: ISSN 2354-1210\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">In 2016, The journal has been included in the list of medical science journals by The State Council for professorship which is awarded a work score of 0-0.5 points for a published article.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Can Tho Journal of Medicine and Pharmacy welcome original works that haven’t been submitted or published in other medical journals. Posts must contain content related to one of the journal’s categories.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The content published\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The journal is divided into 3 categories:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Scientific research article: are valuable scientific works, which have been researched and accepted.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Overview of medicine, biology and pharmacy: serving the objective of continuing training in the fields of medicine, biology and pharmacy; to systematize classical and modern knowledge.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Update information on new knowledge about medicine, biology, pharmacy in the country and in the world.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Scope\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Publication and introduction of scientific research in the fields:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Medicine (internal medicine, surgery, pediatrics, obstetrics and gynecology, odonto-stomatology, laboratory, oncology, traditional medicine, nursing).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Biology (genetics, biotechnology).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Pharmacology (pharmaceutics, drug quality analysis-control, synthetic pharmaceutical chemistry, biochemistry, pharmacognosy, botany, clinical pharmacy).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- To enhance the quality of undergraduate, postgraduate education, scientifically researching and meet the necessary treatment in hospital.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Introducing the updated domestic and oversea information about science technology to promote scientific research and exchanging technology in local, other universities.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Exchanging pharmaceutical and medical information for social health developing in the Mekong Delta and Vietnam.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The object\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Postgraduate students, student of Can Tho University of Medicine and Pharmacy, scientists from schools, research institutes, hospitals, health centers, pharmaceutical companies of the Mekong Delta; other provinces and regions in Vietnam and other country.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Address\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Headquarters of Can Tho Journal of Medicine and Pharmacy, located Scientific Research and International Cooperation Office: 179 Nguyen Van Cu Street, An Khanh Ward, Ninh Kieu District, Can Tho City, Vietnam.\u003C\u002Fspan>\u003C\u002Fp>","\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Ngày 16\u002F7\u002F2015, Tạp chí Y Dược học Cần Thơ được cấp chỉ số quốc tế: ISSN 2354-1210.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 4\u002F2016, Tạp chí đã được Hội đồng Giáo sư ngành Y đưa vào danh sách các tạp chí khoa học Y học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Năm 2020 Tạp chí Y Dược học Cần Thơ đã được phê duyệt vào danh mục của các Hội đồng Giáo sư ngành Dược học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ ra 12 số\u002Fnăm, 180-200 trang\u002Fsố.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 12\u002F2022 Tạp chí Y Dược học Cần Thơ là thành viên của hệ thống Crossref và từ tháng 01\u002F2023 tạp chí thực hiện bình duyệt online kín 2 chiều nhằm tăng tính minh bạch, tin cậy của các công trình nghiên cứu khoa học và đảm bảo tốt nhất chất lượng khoa học của bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ, mục đích và phạm vi của tạp chí\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ và mục đích hoạt động của tạp chí: xuất bản nhằm mục đích phổ biến kết quả từ các đề tài nghiên cứu khoa học; giao lưu trao đổi khoa học, chia sẻ kinh nghiệm, học tập, đồng thời cập nhật thông tin khoa học mới trong các lĩnh vực y, sinh, dược học trong và ngoài nước.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phạm vi của tạp chí: Tạp chí xuất bản được chia thành 3 chuyên mục: (i) Bài báo nghiên cứu khoa học là kết quả công trình nghiên cứu khoa học có giá trị đã được triển khai nghiên cứu, (ii) Bài tổng quan y, sinh, dược học: phục vụ mục tiêu đào tạo liên tục trong lĩnh vực y, sinh, dược học; nhằm hệ thống hóa những kiến thức kinh điển và hiện đại; (iii) Thông tin cập nhật kiến thức mới về y, sinh, dược học trong nước và trên thế giới.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Chính sách truy cập mở\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ áp dụng chính sách truy cập mở đối với các bài báo đã xuất bản đến với độc giả, nhằm mở rộng cơ hội tiếp cận các kết quả nghiên cứu chất lượng cao và tăng cường trao đổi kiến thức. Tạp chí đăng tải trực tuyến (miễn phí) toàn văn các bài báo được công bố trên website của Tạp chí (https:\u002F\u002Ftapchi.ctump.edu.vn).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đạo đức xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ cam kết tuân thủ đạo đức xuất bản phù hợp với các hướng dẫn và tiêu chuẩn của the Committee on Publication Ethics (COPE), tuân thủ các nguyên tắc của COPE’s Core Practices, Best Practices Guidelines for Journal Editors và Guidelines on Good Publication Practices.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Bản thảo bài báo chỉ được chấp nhận khi được tác giả chịu trách nhiệm chính cam kết các nội dung sau: Các nội dung của bản thảo chưa được đăng tải toàn bộ hoặc một phần ở các tạp chí khác; Tất cả các tác giả đều có đóng góp một cách đáng kể vào quá trình nghiên cứu hoặc chuẩn bị bản thảo và cùng chịu trách nhiệm về các nội dung của bản thảo; Tuân thủ các biện pháp đảm bảo đạo đức nghiên cứu (ví dụ thỏa thuận đồng ý tham gia nghiên cứu).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Cam kết bảo mật\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí cam kết thực hiện và tuân thủ các quy định của luật và các văn bản hướng dẫn liên quan đến bảo mật thông tin cá nhân trên không gian mạng. Các thông tin mà người dùng (tác giả, độc giả, biên tập viên, người phản biện) nhập vào các biểu mẫu trên Hệ thống Quản lý xuất bản trực tuyến của tạp chí chỉ được sử dụng vào các mục đích đã được tuyên bố rõ ràng và sẽ không được cung cấp cho bất kỳ bên thứ ba nào khác, hay dùng vào bất kỳ mục đích nào khác.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phí gửi bài\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng bài: 1.000.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng nhanh: 1.500.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với tác giả là cán bộ viên chức thuộc Trường Đại học Y Dược Cần Thơ thì được hỗ trợ 50% lệ phí gửi đăng bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với sinh viên thực hiện đề tài nghiên cứu khoa học cấp trường được hỗ trợ 100% lệ phí đăng bài ( Tác giả gửi đính kèm “ Quyết định về việc giao tổ chức thực hiện đề tài nghiên cứu khoa học cấp Trường của sinh viên”).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Hình thức nộp lệ phí:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Tiền mặt:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Nộp trực tiếp tại Phòng Tài chính - Kế toán, Trường Đại học Y Dược Cần Thơ, số 179 Nguyễn Văn Cừ, P. An Khánh, Q. Ninh Kiều, thành phố Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Chuyển khoản:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tên Tài khoản: Trường ĐHYD Cần Thơ, Số TK: 0111000115668, tại ngân hàng Vietcombank chi nhánh Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Thời gian: Áp dụng từ ngày 01\u002F02\u002F2023.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">* Phí gửi bài không được hoàn trả khi bài viết bị từ chối hoặc tác giả xin rút bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Quy trình phản biện bài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ thực hiện quy trình phản biện kín hai chiều nghiêm ngặt. Danh tính của những người phản biện không được tiết lộ cho các tác giả và ngược lại. Quy trình thẩm định bài báo đăng gồm các bước sau:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tiếp nhận bản thảo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tác giả liên hệ gửi bản thảo đến Tạp chí qua hệ thống trực tuyến tại website: https:\u002F\u002Ftapchi.ctump.edu.vn. Hướng dẫn về cách đăng ký, gửi bài và chuẩn bị bản thảo được cung cấp trên website của Tạp chí.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sàng lọc sơ bộ\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sau khi Tòa soạn nhận được bài báo của tác giả, Ban Thư ký sẽ tiến hành kiểm tra sơ bộ bài báo (các yêu cầu về nội dung và hình thức). Những bài báo không đúng quy cách hoặc có nội dung không phù hợp hoặc vi phạm bản quyền sẽ bị từ chối (Ban Thư ký thông báo phản hồi đến tác giả trong vòng 1 tuần). Những bài báo đủ điều kiện, được Ban Thư ký tòa soạn chuyển đến Ban Biên tập có cùng chuyên môn với nội dung bài báo để đề xuất người phản biện. Thời gian kể từ khi Ban Biên tập nhận bài báo đến khi đề xuất người phản biện bài báo chậm nhất là 5 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Vòng phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký gửi bài và yêu cầu phản biện đến 02 phản biện độc lập.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Các phản biện gởi nhận xét cho Ban Thư ký. Thời gian từ khi gửi bài cho phản biện đến khi nhận ý kiến của phản biện tối đa là 20 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xử ký kết quả phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Nếu ý kiến đồng ý cho đăng và không cần chỉnh sửa, Ban Thư ký tiếp tục đăng bài theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Nếu ý kiến đồng ý đăng và cần chỉnh sửa, Ban Thư ký sẽ thông tin đến tác giả chỉnh sửa theo yêu cầu của người phản biện. Thời gian chỉnh sửa và gửi lại kéo dài không quá 2 tuần, từ khi tác giả bài báo nhận được thông tin (Quá trình này có thể lặp lại tối đa 2 lần\u002F1 bài báo). Khi có sự thống nhất, đồng ý của người phản biện; bài báo được tiếp tục đăng theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Những bài báo có chất lượng không đạt yêu cầu, cả 2 phản biện không đồng ý cho đăng sẽ bị Tòa soạn từ chối đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký tổng hợp các bản thảo đã được tác giả hoàn thiện sau thẩm định trình Ban Biên tập xem xét, Tổng Biên tập phê duyệt, quyết định bài đăng theo các tiêu chí: sự phù hợp nội dung với tôn chỉ và mục đích, thể loại bài viết (ưu tiên các bài có bài có nghiên cứu chuyên sâu, hàm lượng khoa học cao), đóng góp mới bài báo, bài báo được ưu tiên đăng trong số gần nhất của Tạp chí theo thứ tự: tính thời sự, chất lượng bài báo và thời gian gửi bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Ban Biên tập và Ban Thư ký biên tập bản thảo, chế bản, đọc rà soát lỗi. Thời gian hoàn thành từ 10-15 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Ban Thư ký có trách nhiệm thông báo cho tác giả bài báo (bằng e-mail) về tình hình phê duyệt bài báo, thời gian, số kỳ, tập xuất bản bài báo theo qui định.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">4. Danh sách bài báo theo số Tạp chí được in ấn và phát hành trong năm định kỳ được công bố chính thức trên website: https:\u002F\u002Ftapchi.ctump.edu.vn\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>",{"VOID":490},"wcQ1uqwAAAAJ","2023-05-30T08:17:21.868+00:00",[],[494],{"id":495,"createTime":28,"updateTime":28,"relativeEntities":496,"slug":28,"properties":497,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":507,"parentIds":508,"statistic":28},"6413896b-eca9-442b-a73f-182a58a0ce40",[],{"title":498,"address":501,"country":504,"abbreviation":505},{"EN":499,"VI":500},"Can Tho University of Medicine and Pharmacy","Trường Đại học Y Dược Cần Thơ",{"EN":502,"VI":503},"No 179, Nguyen Van Cu street, An Khanh ward, Ninh Kieu district, Can Tho city, Vietnam","Số 179, đường Nguyễn Văn Cừ, phường An Khánh, quận Ninh Kiều, thành phố Cần Thơ, Việt Nam",{"VOID":15},{"VOID":506},"ctump","http:\u002F\u002Fwww.ctump.edu.vn\u002F",[],[],"https:\u002F\u002Ftapchi.ctump.edu.vn\u002Findex.php\u002Fctump",{"impactFactor":32,"impactFactorByYear":512,"i10Index":32,"i10IndexLast5Year":32,"totalPublication":514,"totalPublicationByYear":515,"totalCitation":520,"totalCitationByYear":521,"totalCitationPerPublication":108,"totalCitationPerPublicationByYear":523,"hindexLast5Year":45,"hindex":45},{"2022":513,"2023":111,"2024":106},0.01,1556,{"2020":47,"2021":516,"2022":517,"2023":518,"2024":519,"2025":122},57,306,801,358,161,{"2021":146,"2022":280,"2023":522},99,{"2021":524,"2022":318,"2023":104},0.23,{"impactFactor":28,"impactFactorByYear":28,"i10Index":123,"i10IndexLast5Year":123,"totalPublication":526,"totalPublicationByYear":527,"totalCitation":526,"totalCitationByYear":528,"totalCitationPerPublication":40,"totalCitationPerPublicationByYear":531,"hindexLast5Year":49,"hindex":49},476,{"0":205,"2019":123,"2021":139,"2022":459,"2023":451,"2024":357,"2025":49,"2026":48},{"2021":42,"2022":123,"2023":161,"2024":529,"2025":360,"2026":530},136,83,{"2021":105,"2022":513,"2023":532,"2024":127,"2025":533,"2026":534},0.62,25.43,13.83,{"id":536,"createTime":537,"updateTime":382,"relativeEntities":538,"slug":539,"properties":540,"entityType":25,"verifyStatus":26,"verifyTime":28,"verifyNote":28,"languages":552,"translateLanguages":28,"viewCount":133,"subjectFields":553,"manageAffiliations":554,"indexDatabases":555,"url":556,"thumbnailPath":557,"statistic":558,"gsStatistic":594,"type":55,"analyzePriority":28},"6984a56a-db70-403b-9cc4-4013e1ceaffa","2023-05-09T06:47:40.346+00:00",[],"T%E1%BA%A1p%20ch%C3%AD%20Nghi%C3%AAn%20c%E1%BB%A9u%20n%C6%B0%E1%BB%9Bc%20ngo%C3%A0i",{"country":541,"issn":542,"title":544,"introduce":547,"gsId":550},{"VOID":15},{"VOID":543},"25252445",{"EN":545,"VI":546},"VNU Journal of Foreign Studies","Tạp chí Nghiên cứu nước ngoài",{"EN":548,"VI":549},"{\"ops\":[{\"insert\":\"\\n\\nThe \\n\"},{\"attributes\":{\"italic\":true},\"insert\":\"VNU Journal of Science\"},{\"insert\":\"\\n was established in 1985 for the publication of national and international research papers in all fields of natural sciences and technology, social sciences and humanities. 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đặc điểm của tổn thương do ablation bằng ống thông tần số vô tuyến gây ra trong tim vẫn chưa được xác định rõ ràng. Bởi vì cơ chế gây tổn thương do năng lượng tần số vô tuyến là nhiệt, nghiên cứu này được thực hiện nhằm xác định độ dốc nhiệt độ trong mô cơ tim trong quá trình ablation bằng ống thông tần số vô tuyến (RF), và để xác thực một mô hình động lực học đã được xây dựng nhằm mô tả những quan sát này. Các tổn thương được tạo ra bằng cách làm nóng RF trong một mô hình thí nghiệm của thành thất phải (RV) của chó, được cô lập và tưới máu cũng như siêu tưới máu. Công suất RF được điều chỉnh để duy trì nhiệt độ đầu điện cực ở 80°C trong 120 giây cho 153 tổn thương liên tiếp và các độ dốc nhiệt độ theo hướng ra đã được đo lường. Với khoảng cách tăng dần từ điện cực, nhiệt độ của cơ tim giảm theo hình thức hyperbol mà mô hình động lực học đã được xây dựng tiên đoán một cách chính xác (P = 0.0001, r = 0.98). Độ dốc này và kích thước tổn thương kết quả không bị ảnh hưởng bởi tốc độ tưới máu vành. Việc sử dụng giám sát nhiệt độ đầu điện cực như một yếu tố dự đoán kích thước tổn thương đã được thử nghiệm trên 104 tổn thương liên tiếp với nhiệt độ đầu điện cực thay đổi giữa 50 và 85°C. Nhiệt độ đầu điện cực có mối tương quan chặt chẽ với độ sâu của tổn thương (P = 0.0001, r = 0.92) và chiều rộng (P = 0.0001, r = 0.88), và là một yếu tố dự đoán kích thước tổn thương tốt hơn các phép đo về công suất, dòng điện hay năng lượng. Nhiệt độ tại ranh giới giữa mô sống và mô không sống được ước lượng là 47.9°C. Các dữ liệu này chứng minh rằng trong quá trình ablation bằng ống thông tần số vô tuyến, độ dốc nhiệt độ theo hướng ra là một cách tiên đoán rõ ràng và dường như độc lập với tưới máu nội cơ tim nếu nhiệt độ điện cực cố định được duy trì. Việc sử dụng giám sát nhiệt độ đầu điện cực có thể dự đoán chính xác kích thước cuối cùng của tổn thương do tần số vô tuyến gây ra.\u003C\u002Fjats:p>","\u003Cjats:p>Thecharacteristics of radiofrequency catheter ablation induced injury in the heart are not well characterized. Since the mechanism of injury by radiofrequency energy is thermal, this study was performed to determine the temperature gradient in myocardial tissue during radiofrequency (RF) catheter ablation, and to validate a thermodynamic model derived to describe these observations. Lesions were created by RF heating in an experimental model of isolated perfused and superfused canine right ventricular (RV) free wall. RF power output was adjusted to maintain electrode tip temperature at 80°C for 120 seconds in 153 serial lesions and radial temperature gradients were measured. With increasing distance from the electrode, the temperature of the myocardium decreased in a hyperbolic form that was closely predicted by a derived thermodynamic model (P = 0.0001, r = 0.98). This gradient and resultant lesion sizes were unafected by the rate of coronary perfusion. The utility of tip temperature monitoring as a predictor of lesion size was tested in 104 serial lesions with tip temperatures that were varied between 50 and 85°C. The tip temperature correlated closely with lesion depth (P = 0.0001, r = 0.92) and width (P = 0.0001, r = 0.88), and was a better predictor of lesion size than measurements of power, current or energy. The temperature at the margin between viable and nonviable tissue was estimated to be 47.9°C. These data demonstrate that during radiofrequency catheter ablation, the radial temperature gradient is predictably hyperbolic and appears to be independent of intramyocardial perfusion if constant electrode temperature is maintained. The use of tip temperature monitoring can accurately predict the ultimate size of radiofrequency‐induced lesions.\u003C\u002Fjats:p>",{"EN":1211,"VI":1212},"Tissue Heating During Radiofrequency Catheter Ablation: A Thermodynamic Model and Observations in Isolated Perfused and Superfused Canine Right Ventricular Free Wall","Nhiệt độ mô trong quá trình ablation bằng ống thông tần số vô tuyến: Một mô hình nhiệt động lực học và các quan sát trên thành thất phải của chó cô lập được tưới máu và siêu tưới máu",{"VOID":1214},"2472624",{"VOID":1216},"10.1111\u002Fj.1540-8159.1989.tb05034.x","2024-10-12T05:19:14.996+00:00",[31],[30],"https:\u002F\u002Fonlinelibrary.wiley.com\u002Fdoi\u002F10.1111\u002Fj.1540-8159.1989.tb05034.x",[1222,1243],{"id":1223,"sortIndex":32,"researcher":28,"roles":1224,"affiliations":1225,"properties":1234,"displayName":1238,"givenName":28,"familyName":28},"64ef2ef4-4fab-450a-86bd-ff0b170e4694",[],[1226],{"id":1227,"sortIndex":32,"affiliation":1228,"properties":28},"103d7ce6-daa7-4801-ac14-18299f6137c4",{"id":1227,"createTime":28,"updateTime":28,"relativeEntities":1229,"slug":28,"properties":1230,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1233,"statistic":28},[],{"title":1231},{"EN":1232},"Division of Cardiology, Department of Internal Medicine, and the Division of Nuclear Medicine, Department of Radiology, University of Virginia School of Medicine, Charlottesville, Virginia",[],{"orcid":1235,"title":1237,"gsAuthor":1239,"openalex":1241},{"VOID":1236},"https:\u002F\u002Forcid.org\u002F0000-0002-5592-8338",{"EN":1238},"David E. Haines",{"VOID":1240},"[\"pZbyMn8AAAAJ\"]",{"VOID":1242},"A5064399230",{"id":1244,"sortIndex":40,"researcher":28,"roles":1245,"affiliations":1246,"properties":1253,"displayName":1255,"givenName":28,"familyName":28},"a24ab999-2b75-4f1d-b6fe-92d545430c55",[],[1247],{"id":1227,"sortIndex":32,"affiliation":1248,"properties":28},{"id":1227,"createTime":28,"updateTime":28,"relativeEntities":1249,"slug":28,"properties":1250,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1252,"statistic":28},[],{"title":1251},{"EN":1232},[],{"title":1254,"openalex":1256},{"EN":1255},"Denny D. 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MM., 1982, Catheter induced ablation of the atrioventricular junction to control refractory supraventricular arrhythmia, J Am Med Assoc, 248",{},{"id":28,"text":1320,"url":28,"identifiers":1321},"10.1016\u002FS0735-1097(86)80156-5",{"doi":1320},{"id":28,"text":1323,"url":28,"identifiers":1324},"10.1161\u002F01.CIR.67.3.687",{"doi":1323},{"id":28,"text":1326,"url":28,"identifiers":1327},"10.1016\u002FS0735-1097(84)80394-0",{"doi":1326},{"id":28,"text":1329,"url":28,"identifiers":1330},"10.1016\u002FS0735-1097(85)80179-0",{"doi":1329},{"id":28,"text":1332,"url":28,"identifiers":1333},"10.1161\u002F01.CIR.73.1.10",{"doi":1332},{"id":28,"text":1335,"url":28,"identifiers":1336},"10.1016\u002FS0039-6109(16)40540-2",{"doi":1335},{"id":28,"text":1338,"url":28,"identifiers":1339},"10.3171\u002Fjns.1970.33.4.0415",{"doi":1338},{"id":28,"text":1341,"url":28,"identifiers":1342},"10.1111\u002Fj.1749-6632.1980.tb50749.x",{"doi":1341},{"id":28,"text":1344,"url":28,"identifiers":1345},"10.1016\u002FS0735-1097(87)80388-1",{"doi":1344},{"id":28,"text":1347,"url":28,"identifiers":1348},"Borggrefe M, 1987, Application of transvenous radio‐frequency alternating current ablation in humans, Circulation, 76, IV",{},{"id":28,"text":1350,"url":28,"identifiers":1351},"Hoyt RH, 1985, Factors influencing trans‐catheter radiofrequency ablation of the myocardium, Circulation, 72, III",{},{"id":28,"text":1353,"url":28,"identifiers":1354},"10.1115\u002F1.3138197",{"doi":1353},{"id":28,"text":1356,"url":28,"identifiers":1357},"Gottlieb GJ, 1981, Ultrastructural characterization of the border zone surrounding early experimental myocardial infarcts in dogs, Am J Pathol, 103, 292",{},{"id":28,"text":1359,"url":28,"identifiers":1360},"10.1111\u002Fj.1540-8159.1988.tb06027.x",{"doi":1359},{"id":28,"text":1362,"url":28,"identifiers":1363},"Langberg J, 1987, Radiofrequency catheter ablation in the coronary sinus, J Am Coll Cardiol, 9, 99A",{},{"id":28,"text":1365,"url":28,"identifiers":1366},"Naccarelli G, 1987, Selective catheter ablation of canine ventricular myocardium with radiofrequency current, J Am Coll Cardiol, 9, 99A",{},{"id":28,"text":1368,"url":28,"identifiers":1369},"10.1161\u002F01.CIR.78.2.416",{"doi":1368},{"id":28,"text":1371,"url":28,"identifiers":1372},"10.1111\u002Fj.1540-8159.1987.tb06037.x",{"doi":1371},{"id":28,"text":1374,"url":28,"identifiers":1375},"Casman ER, 1984, Theoretical aspects of radiofrequency lesions in the dorsal root entry zone, Neurosurgery, 15, 945",{},{"id":28,"text":1377,"url":28,"identifiers":1378},"Adams T, 1980, Thermodynamic technique for the quantification of regional blood flow, Am J Physiol, 238, H682",{},{"id":28,"text":1380,"url":28,"identifiers":1381},"Organ L., 1976, Electrophysiologic principles of radiofrequency lesion making: Int. Symp. Radiofrequency Lesion Making, Appl Neurophysiol, 39, 69",{},{"id":28,"text":1383,"url":28,"identifiers":1384},"Hoffman E, 1987, Phase displacement between voltage and current during radiofrequency catheter ablation, Circulation, 76, IV",{},{"id":28,"text":1386,"url":28,"identifiers":1387},"10.1161\u002F01.RES.46.3.387",{"doi":1386},{"id":28,"text":1389,"url":28,"identifiers":1390},"Sears FW, 1955, University physics. Reading, 17",{},{"id":1392,"createTime":1393,"updateTime":1394,"relativeEntities":1395,"slug":1396,"properties":1397,"entityType":965,"verifyStatus":26,"verifyTime":1393,"verifyNote":966,"languages":1414,"translateLanguages":1415,"viewCount":32,"primaryUrl":1416,"fullTextUrl":28,"authors":1417,"publicationType":989,"publisherRelationship":1499,"citationCount":459,"citationInfo":1549,"publishDate":1552,"publishYear":1550,"citationAnalyzeStatus":884,"lastCitationAnalyze":28,"indexDatabases":1553,"openAccess":28,"references":1554,"isForceReanalyzing":1191},"f04a9edd-e19b-4635-81f4-4be894ba8b08","2024-09-17T11:05:10.785+00:00","2025-01-26T21:00:36.129+00:00",[],"Treatment-of-Thalamic-Pain-by-Chronic-Motor-Cortex-Stimulation",{"mag":1398,"keywords":1400,"openalex":1402,"abstract":1404,"title":1407,"pm":1410,"doi":1412},{"VOID":1399},"2089718440",{"VI":1401},"hội chứng đau thalamus, kích thích vỏ não vận động, điều trị đau, nơron thalamus, cơn đau mạn tính",{"VOID":1403},"W2089718440",{"VI":1405,"EN":1406},"\u003Cjats:p>Tất cả các hình thức điều trị, bao gồm cả kích thích mãn tính của nhân chuyển tiếp thalamus, chỉ có thể cung cấp kiểm soát cơn đau thỏa đáng trong khoảng 20%-30% trường hợp hội chứng đau thalamus. Để phát triển phương pháp điều trị hiệu quả hơn cho hội chứng đau thalamus, chúng tôi đã điều tra ảnh hưởng của việc kích thích các vùng não khác nhau đến hoạt động bùng phát quá mức của các nơron thalamus được ghi nhận ở mèo sau khi tước bỏ sự cảm giác của đường spinothalamic. Sự ức chế hoàn toàn, kéo dài của hoạt động bùng phát quá mức được tạo ra bởi việc kích thích vỏ não vận động. Dựa trên phát hiện thực nghiệm này, chúng tôi đã điều trị bảy trường hợp hội chứng đau thalamus bằng cách kích thích mạn tính vỏ não vận động sử dụng điện cực đệm ngoài màng cứng. Kiểm soát cơn đau xuất sắc hoặc tốt đã được đạt được trong tất cả các trường hợp mà không có bất kỳ biến chứng hay tác dụng phụ nào. Trong quá trình kích thích, sự gia tăng lưu lượng máu khu vực của vỏ não và thalamus, sự gia tăng nhiệt độ rõ rệt của các vùng da đau, và sự cải thiện cử động của các chi đau đã được quan sát. Những kết quả này gợi ý rằng hội chứng đau thalamus có thể được điều trị hiệu quả nhất bằng cách kích thích vỏ não vận động mạn tính.\u003C\u002Fjats:p>","\u003Cjats:p>All forms of therapy, including chronic stimulation of the thalamic relay nucleus, can provide satisfactory pain control in only 20%‐30% of cases of thalamic pain syndrome. In order to develop a more effective treatment for fhalamic pain syndrome, we investigated the effects of stimulation of various brain regions on the burst hyperactivity of thalamic neurons recorded in cats after deafferentiation of the spinothalamic pathway. Complete, long‐ term inhibition of the burst hyperactivity was induced by stimulation of the motor cortex, Based on this experimental finding, we treated seven cases of thalamic pain syndrome by chronic motor cortex stimulation employing epidural plate electrodes. Excellent or good pain control was obtained in all cases without any complications or side effects. During the stimulation, an increase in regional blood flow of the cerebral cortex and thalamus, a marked rise in temperature of the painful skin regions, and improved movements of the painful limbs were observed. These results suggest that thalamic pain syndrome can be most effectively treated by chronic motor cortex stimulation.\u003C\u002Fjats:p>",{"EN":1408,"VI":1409},"Treatment of Thalamic Pain by Chronic Motor Cortex Stimulation","Điều trị Đau Thalamus bằng Kích thích Vỏ Não Vận Động Mạn Tính",{"VOID":1411},"1705329",{"VOID":1413},"10.1111\u002Fj.1540-8159.1991.tb04058.x",[31],[30],"https:\u002F\u002Fonlinelibrary.wiley.com\u002Fdoi\u002F10.1111\u002Fj.1540-8159.1991.tb04058.x",[1418,1437,1454,1469,1484],{"id":1419,"sortIndex":32,"researcher":28,"roles":1420,"affiliations":1421,"properties":1430,"displayName":1434,"givenName":28,"familyName":28},"ebe01cfd-de1b-450f-9fc4-0aab48558e0a",[],[1422],{"id":1423,"sortIndex":32,"affiliation":1424,"properties":28},"6157dd9d-4661-4243-8013-59f4a160cde7",{"id":1423,"createTime":28,"updateTime":28,"relativeEntities":1425,"slug":28,"properties":1426,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1429,"statistic":28},[],{"title":1427},{"VI":1428},"Department of Neurological Surgery, Nihon University School of Medicine, Tokyo, Japan",[],{"orcid":1431,"title":1433,"openalex":1435},{"VOID":1432},"https:\u002F\u002Forcid.org\u002F0000-0002-5682-4215",{"EN":1434},"Takashi Takata",{"VOID":1436},"A5034515522",{"id":1438,"sortIndex":40,"researcher":28,"roles":1439,"affiliations":1440,"properties":1449,"displayName":1451,"givenName":28,"familyName":28},"5532b6fb-fb11-46be-8dd1-7c0953703f63",[],[1441],{"id":1442,"sortIndex":32,"affiliation":1443,"properties":28},"ca6647b8-8f5d-414f-8839-82053a54d44e",{"id":1442,"createTime":28,"updateTime":28,"relativeEntities":1444,"slug":28,"properties":1445,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1448,"statistic":28},[],{"title":1446},{"EN":1447},"Department of Neuorological Surgery, Nihon University School of Medicine, Tokyo, Japan",[],{"title":1450,"openalex":1452},{"EN":1451},"Yoichi 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tuổi, và đôi khi ở trẻ em và trẻ sơ sinh. Những năm gần đây đã chứng kiến sự gia tăng đáng kể các bài báo liên quan đến các khía cạnh lâm sàng và cơ bản của bệnh lý này. Đặc trưng bởi sự nâng cao đoạn ST kiểu cove ở các đầu điện cực trước ngực bên phải trên điện tâm đồ (ECG), hội chứng Brugada có nền tảng di truyền mà cho đến nay chỉ liên quan đến các đột biến trong gen SCN5A, gen mã hóa cho chuỗi α của kênh natri. ECG của hội chứng Brugada thường bị ẩn giấu, nhưng có thể bị lộ diện hoặc điều chỉnh bởi một số loại thuốc và tình trạng sinh lý bệnh bao gồm các chất chặn kênh natri, tình trạng sốt, các tác nhân kích thích thần kinh phế vị, thuốc chống trầm cảm ba vòng, cũng như ngộ độc cocaine và propranolol. Tuổi trung bình tại thời điểm chẩn đoán ban đầu hoặc đột tử là 40 ± 22, với bệnh nhân trẻ nhất được chẩn đoán ở độ tuổi 2 ngày và bệnh nhân lớn tuổi nhất ở độ tuổi 84. Bài tổng quan này cung cấp cái nhìn tổng quan về các khía cạnh lâm sàng, di truyền, phân tử và tế bào của hội chứng Brugada, kết hợp với kết quả từ hai hội nghị đồng thuận gần đây. Các tranh cãi liên quan đến phân tầng nguy cơ và các chiến lược dược lý mới được đề xuất cũng được thảo luận.\u003C\u002Fjats:italic> \u003C\u002Fjats:p>","\u003Cjats:p> \u003Cjats:italic>First introduced as a new clinical entity in 1992, the Brugada syndrome is associated with a relatively high risk of sudden death in young adults, and occasionally in children and infants. Recent years have witnessed a striking proliferation of papers dealing with the clinical and basic aspects of the disease. Characterized by a coved‐type ST‐segment elevation in the right precordial leads of the electrocardiogram (ECG), the Brugada syndrome has a genetic basis that thus far has been linked only to mutations in SCN5A, the gene that encodes the α‐subunit of the sodium channel. The Brugada ECG is often concealed, but can be unmasked or modulated by a number of drugs and pathophysiological states including sodium channel blockers, a febrile state, vagotonic agents, tricyclic antidepressants, as well as cocaine and propranolol intoxication. Average age at the time of initial diagnosis or sudden death is 40 ± 22, with the youngest patient diagnosed at 2 days of age and the oldest at 84 years. This review provides an overview of the clinical, genetic, molecular, and cellular aspects of the Brugada syndrome, incorporating the results of two recent consensus conferences. Controversies with regard to risk stratification and newly proposed pharmacologic strategies are discussed.\u003C\u002Fjats:italic> \u003C\u002Fjats:p>",{"EN":1577,"VI":1578},"Brugada Syndrome","Hội chứng 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A, 1993, Recurrent ventricular fibrillation, right bundle‐branch block and persistent ST segment elevation in V1‐V3: A new arrhythmia syndrome? 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Gần đây, phân tán sóng P (PWD), được cho là phản ánh sự dẫn truyền không đồng nhất của nhĩ, đã được đề xuất là hữu ích trong việc dự đoán rung nhĩ kịch phát (PAF). Chúng tôi đã nghiên cứu 90 bệnh nhân liên tiếp (46 nam, 44 nữ; tuổi trung bình 55 ± 13 năm) có tiền sử rung nhĩ kịch phát vô căn và 70 đối tượng khỏe mạnh (42 nam, 28 nữ; tuổi trung bình 53 ± 14 năm). Thời gian của sóng P được tính toán ở tất cả 12 đạo trình của điện tâm đồ bề mặt. Sự khác biệt giữa thời gian sóng P tối đa và tối thiểu được tính toán và sự khác biệt này được định nghĩa là phân tán sóng P (PWD = Pmax ‐ Pmin). Tất cả bệnh nhân và nhóm đối chứng cũng được đánh giá bằng siêu âm tim để đo đường kính nhĩ trái và phân suất tống máu thất trái (LVEF). Không có sự khác biệt về giới tính (P = 0,26), tuổi tác (P = 0,12), LVEF (66 ± 4% so với 67 ± 5%, P = 0,8) và đường kính nhĩ trái (36 ± 4 mm so với 34 ± 6 mm, P = 0,13) giữa bệnh nhân và đối chứng. Thời gian sóng P tối đa được phát hiện cao hơn đáng kể ở bệnh nhân có tiền sử PAF (116 ± 17 ms) so với đối chứng (101 ± 11 ms, P \u003C 0,001). Phân tán sóng P cũng cao hơn một cách đáng kể ở bệnh nhân so với đối chứng (44 ± 15 ms so với 27 ± 10 ms, P \u003C 0,001). Có một sự tương quan yếu giữa tuổi tác và phân tán sóng P (r = 0,27, P \u003C 0,001). Một giá trị P tối đa là 106 ms phân tách bệnh nhân có PAF khỏi các đối tượng đối chứng với độ nhạy 83%, độ đặc hiệu 72% và độ chính xác dự đoán dương tính 79%. Giá trị phân tán sóng P là 36 ms đã phân tách bệnh nhân khỏi các đối tượng đối chứng với độ nhạy 77%, độ đặc hiệu 82% và độ chính xác dự đoán dương tính 85%. Kết luận, thời gian sóng P tối đa và phân tán sóng P được tính toán trên một điện tâm đồ bề mặt tiêu chuẩn là các dấu hiệu điện tâm đồ đơn giản có thể được sử dụng để xác định những bệnh nhân mắc rung nhĩ kịch phát vô căn.\u003C\u002Fjats:p>","\u003Cjats:p>The prolongation of intraatrial and interatrial conduction time and the inhomogeneous propagation of sinus impulses have been shown in patients with atrial fibrillation. Recently P wave dispersion (PWD), which is believed to reflect inhomogeneous atrial conduction, has been proposed as being useful for the prediction of paroxysmal atrial fibrillation (PAF). Ninety consecutive patients (46 men, 44 women; aged 55 ± 13 years) with a history of idiopathic PAF and 70 healthy subjects (42 men, 28 women; mean age, 53 ± 14 years) were studied. The P wave duration was calculated in all 12 leads of the surface ECC. The difference between the maximum and minimum P wave duration was calculated and this difference was defined as P wave dispersion (PWD = Pmax ‐ Pmin). All patients and controls were also evaluated by echocardiography to measure the left atrial diameter and left ventricular ejection fraction (LVEF). There was no difference between patients and controls in gender (P = 0.26), age (P = 0.12), LVEF (66 ± 4% vs 67 ± 5%, P = 0.8) and left atrial diameter (36 ± 4 mm vs 34 ± 6 mm, P = 0.13). P maximum duration was found to be significantly higher in patients with a history of PAF (116 ± 17 ms) than controls (101 ±11 ms. P &lt; 0.001). P wave dispersion was also significantly higher in patients than in controls (44 ± 15 ms vs 27 ± 10 ms, P &lt; 0.001). There was a weak correlation between age and P wave dispersion (r = 0.27, P &lt; 0.001). A P maximum value of 106 ms separated patients with PAF from control subjects with a sensitivity of 83%, a specificity of 72%, and a positive predictive accuracy of 79%. A P wave dispersion value of 36 ms separated patients from control subjects with a sensitivity of 77%, a specificity of 82%, and a positive predictive accuracy of 85%. 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Mặc dù các mạch máu có thể bị chèn ép do chuyển động của xương đòn, nghiên cứu của chúng tôi cho thấy tổn thương dây và catheter trong khu vực đó là do sự kẹt mô mềm hơn là do tiếp xúc với xương. Phẫu tích tám xác người với mười dây cho thấy hai dây vào tĩnh mạch đầu, và không được đưa vào nghiên cứu. Trong số bốn dây còn lại, bốn dây đi qua cơ dưới đòn, hai dây qua dây chằng costoclavicular, và hai dây qua cả hai cấu trúc này trước khi vào tĩnh mạch dưới đòn, tĩnh mạch cảnh trong hoặc tĩnh mạch brachiocephalic. Các nghiên cứu giải phẫu chứng minh rằng sự kẹt bởi cơ dưới đòn hoặc dây chằng costoclavicular có thể gây gấp đi gấp lại liên tục của các dây trong quá trình vận động của đai vai. Cineradiology của các bệnh nhân với sự tắc nghẽn catheter phụ thuộc vào vị trí đã xác nhận sự kẹt bởi cơ dưới đòn. Sự kẹt mô mềm tạo ra một tải trọng tĩnh lên các dây và catheter, và sự gấp lại liên tục quanh điểm kẹt có thể là nguyên nhân gây tổn thương trước đây được cho là do sự chèn ép cycle costoclavicular.\u003C\u002Fjats:p>","\u003Cjats:p>The literature suggests that approximately 93% of all pacemaker lead fractures occur in the segment of the lead lateral to the venous entry, and costoclavicular compression has been implicated. While blood vessels can be compressed by movements of the clavicle, our research suggests that lead and catheter damage in that region is caused by soft tissue entrapment rather than bony contact. Dissection of eight cadavers with ten leads revealed that two entered the cephalic vein, and were not included in the study. Of the other eight leads, four passed through the subclavius muscle, two through the costoclavicular ligament, and two through both these structures before entering the subclavian, internal jugular, or brachiocephalic vein. Anatomical studies demonstrated that entrapment by the subclavius muscle or the costoclavicular ligament could cause repeated flexing of leads during movements of the pectoral girdle. Cineradiology of patients with position dependent catheter occlusion confirmed entrapment by the subclavius muscle. 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Mã này được viết tắt là NBC (cho “Mã Chung NASPE\u002FBPEC”) và được phát triển để cho phép mở rộng khái niệm mã chung cho các máy tạo nhịp có tốc độ thoát được điều khiển liên tục bằng cách theo dõi một số biến sinh lý, thay vì được xác định bằng các khoảng thời gian thoát cố định đo từ các kích thích hoặc các sự kiện khử cực được phát hiện, và cho các thiết bị chống nhịp tim nhanh bao gồm các thiết bị đồng bộ và máy khử rung tim. Mã NASPE\u002FBPEC bao gồm một chữ \"R\" ở vị trí thứ tư để chỉ ra sự điều chỉnh tỷ lệ (tạo nhịp tỷ lệ thích ứng), và một trong bốn chữ cái ở vị trí thứ năm để chỉ ra sự hiện diện của khả năng tạo nhịp chống nhịp tim nhanh hoặc của các chức năng đồng bộ hoặc khử rung tim.\u003C\u002Fjats:p>","\u003Cjats:p>A new generic pacemaker code, derived from and compatible with the Revised ICHD Code, was proposed jointly by the North American Society of Pacing and Electrophysiology (NASPE) Mode Code Committee and the British Pacing and Electrophysiology Croup (BPEC), and has been adopted by the NASPE Board of Trustees. 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Các bất thường điện sinh lý và điện cơ phát sinh từ tình trạng này có liên quan đến nguy cơ cao hơn của các loạn nhịp tim tâm nhĩ paroxysmal hoặc kéo dài, bao gồm rung tâm nhĩ, flutter điển hình hoặc không điển hình, hoặc các dạng loạn nhịp tâm nhĩ khác. Mối liên hệ như vậy không phải là ngẫu nhiên vì các bất thường trong dẫn truyền nội và giữa tâm nhĩ làm chậm và phá vỡ (phân tán không gian và thời gian) kích hoạt điện, điều này thúc đẩy việc khởi đầu và duy trì các mạch tái nhập. Các can thiệp điều trị phòng ngừa tạo ra các hiệu ứng khác nhau, đôi khi trái ngược như với hiệu ứng proarrhythmic của các loại thuốc chống loạn nhịp tim nhóm I. Tương tự, kích thích tâm nhĩ có thể thúc đẩy proarrhythmias hoặc có hiệu ứng chống loạn nhịp tùy thuộc vào vị trí và kiểu kích thích. Kích thích tâm nhĩ đa khu vực được thiết kế nhằm cải thiện, trong khả năng có thể, kích hoạt bất thường do các rối loạn dẫn truyền nội hoặc giữa tâm nhĩ tự phát hoặc do kích thích tâm nhĩ đơn vị, đây là các tình huống thường gây ra các loạn nhịp khó điều trị. Việc đồng bộ hóa điện tâm nhĩ cũng có thể được sử dụng để sửa chữa các bất thường cơ học như sự thiếu đồng bộ giữa tâm nhĩ và tâm thất trái do các chậm trễ trong dẫn truyền nội tâm nhĩ.\u003C\u002Fjats:italic> \u003C\u002Fjats:p>","\u003Cjats:p> \u003Cjats:italic>Atrial conduction disorders are frequent in elderly subjects and\u002For those with structural heart diseases, mainly mitral valve disease, hyperthrophic cardiomyopathies, and hypertension. The resultant electrophysiological and electromechanical abnormalities are associated with a higher risk of paroxysmal or persistent atrial tachyarrhythmias, either atrial fibrillation, typical or atypical flutter or other forms of atrial tachycardias. Such an association is not fortuitous because intra‐ and interatrial conduction abnormalities delays disrupt (spatial and temporal dispersion) electrical activation, thus promoting the initiation and perpetuation of reentrant circuits. Preventive therapeutic interventions induce variable, sometimes paradoxical effects as with the proarrhythmic effect of class I antiarrhythmic drugs. Similarly, atrial pacing may promote proarrhythmias or an antiarrhythmic effect according to the pacing site(s) and mode. Multisite atrial pacing was conceived to correct, as much as possible, abnormal activation induced by spontaneous intra‐ or interatrial conduction disorders or by single site atrial pacing, which are situations responsible for commonly refractory arrhythmias. Atrial electrical resynchronization can also be used to correct mechanical abnormalities like left heart AV dyssynchrony resulting from intraatrial conduction delays. 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RD, 1987, Amiodarone in the management of atrial fibrillation, Arch Intern Med, 147, 1401, 10.1001\u002Farchinte.1987.00370080037009",{"doi":3963},"10.1001\u002Farchinte.1987.00370080037009",{"id":28,"text":3965,"url":28,"identifiers":3966},"10.1111\u002Fj.0954-6820.1980.tb09668.x",{"doi":3965},{"id":28,"text":3968,"url":28,"identifiers":3969},"Swartz JF, 1994, A catheter‐based curative approach to atrial fibrillation in humans, Circulation, 90, I",{},{"id":28,"text":3971,"url":28,"identifiers":3972},"10.1111\u002Fj.1540-8167.1995.tb00758.x",{"doi":3971},{"id":28,"text":3974,"url":28,"identifiers":3975},"10.1111\u002Fj.1540-8167.1995.tb00759.x",{"doi":3974},{"id":28,"text":3977,"url":28,"identifiers":3978},"Coumel P., 1992, Atrial Fibrillation: Mechanisms and Management, 109",{},{"id":3980,"createTime":3981,"updateTime":3982,"relativeEntities":3983,"slug":3984,"properties":3985,"entityType":965,"verifyStatus":26,"verifyTime":3981,"verifyNote":966,"languages":4001,"translateLanguages":4002,"viewCount":32,"primaryUrl":4003,"fullTextUrl":28,"authors":4004,"publicationType":989,"publisherRelationship":4069,"citationCount":613,"citationInfo":4118,"publishDate":4121,"publishYear":4119,"citationAnalyzeStatus":884,"lastCitationAnalyze":28,"indexDatabases":4122,"openAccess":28,"references":4123,"isForceReanalyzing":1191},"bb3bec9f-3bc0-4afc-8d17-4068a151220e","2024-10-12T20:34:16.410+00:00","2025-01-26T21:06:21.395+00:00",[],"Implantable-Defibrillation-Eight-Years-Clinical-Experience",{"mag":3986,"keywords":3988,"openalex":3989,"abstract":3991,"title":3994,"pm":3997,"doi":3999},{"VOID":3987},"1997142948",{"VI":1204},{"VOID":3990},"W1997142948",{"VI":3992,"EN":3993},"\u003Cjats:p>Việc cấy ghép máy khử rung tim tự động đầu tiên được thực hiện vào tháng 2 năm 1980. Việc tích hợp khả năng cắt điện tim vào năm 1982 đã dẫn đến máy khử rung tim cấy ghép tự động AICD™. Giữa ngày 1 tháng 4 năm 1982 và 15 tháng 4 năm 1988, 3610 bệnh nhân tại 236 trung tâm ở Hoa Kỳ và 84 trung tâm quốc tế đã nhận được máy phát xung AICD. Đối tượng bệnh nhân gồm có 2904 nam và 683 nữ với tình trạng loạn nhịp tim thất tái phát và\u002Fhoặc rung thất, độ tuổi trung bình là 59 tuổi (từ 9 đến 96 tuổi). Chẩn đoán chính được báo cáo cho nhóm bệnh nhân này là: bệnh động mạch vành (63,5%), bệnh cơ tim không thiếu máu (12,9%), khác (6,4%) và không xác định (17,2%). Phân suất tống máu thất trái được báo cáo trung bình là 32,8%. Thời gian theo dõi trung bình là 12,2 tháng (dao động từ 0 đến 72 tháng). Trong 385 ca tử vong, 94 ca (24%) là đột ngột. Tỷ lệ sống sót tích lũy (±S.E.) từ tử vong tim đột ngột (SCD) lần lượt là 98.0 ± 0.3%, 96.5 ± 0.5%, 95.2 ± 0.7%, 93.7 ± 1.0%, 93.7 ± 1.0% và 89.7 ± 4.0% ở 12, 24, 36, 48, 60 và 72 tháng. Tỷ lệ tử vong phẫu thuật (30 ngày) là 2.5%. Các tác dụng phụ\u002Fbiến chứng được báo cáo tương tự như máy tạo nhịp. Đến nay, 33% bệnh nhân đã nhận được các cú sốc tự phát từ thiết bị. Tỷ lệ sống sót của máy phát xung AICD từ các lỗi điện và cơ học là 92.8 ± 0.5%, 88.4 ± 0.7%, 86.7 ± 0.8% và 86.4 ± 0.9% ở 12, 18, 24 và 30 tháng. Phân tích dữ liệu cho thấy AICD đã có tác động đáng kể đến sự sống sót của bệnh nhân khỏi SCD.\u003C\u002Fjats:p>","\u003Cjats:p>Implantation of the first automatic defibrillator occurred in February 1980. Incorporation of cardioversion capability in 1982 resulted in the AICD™ automatic implantable cardioverter defibrillator. Between April 1, 1982 and April 15, 1988, 3610 patients in 236 U.S. and 84 international centers received AICD pulse generators. Patient population consisted of 2904 males and 683 females with recurrent ventricular tachycardia and\u002For fibrillation, mean age 59 yrs. (range 9–96 yrs.). Primary diagnoses reported for the patient group were: coronary artery disease (63.5%), nonischemic cardiomyopathy (12.9%), other (6.4%) and unspecified (17.2%). Mean reported LV ejection fraction was 32.8%. Follow‐up averaged 12.2 mo. (range 0–72 mo.). Of 385 deaths, 94 (24%) were sudden. Cumulative percentage survival (±S.E.) from sudden cardiac death (SCD) was 98.0 ± 0.3%, 96.5 ± 0.5%, 95.2 ± 0.7%, 93.7 ± 1.0%, 93.7 ± 1.0% and 89.7 ± 4.0% at 12, 24, 36, 48, 60 and 72 months, respectively. Operative mortality (30 days) was 2.5%. Reported side effects\u002Fcomplications were similar to those of pacemakers. To date, 33% of the patients received spontaneous device countershocks. AICD pulse generator survival from electrical and mechanical failures was 92.8 ± 0.5%, 88.4 ± 0.7%, 86.7 ± 0.8% and 86.4 ± 0.9% at 12, 18, 24 and 30 mos. Data analysis demonstrates that the AICD has had a significant impact on patient survival from SCD.\u003C\u002Fjats:p>",{"EN":3995,"VI":3996},"Implantable Defibrillation: Eight Years Clinical Experience","Đặt máy khử rung tim cấy ghép: Kinh nghiệm lâm sàng tám năm",{"VOID":3998},"2463587",{"VOID":4000},"10.1111\u002Fj.1540-8159.1988.tb06349.x",[31],[30],"https:\u002F\u002Fonlinelibrary.wiley.com\u002Fdoi\u002F10.1111\u002Fj.1540-8159.1988.tb06349.x",[4005,4022,4039,4054],{"id":4006,"sortIndex":32,"researcher":28,"roles":4007,"affiliations":4008,"properties":4017,"displayName":4019,"givenName":28,"familyName":28},"e07fee50-1b9a-4e89-b207-0cc8e20ba858",[],[4009],{"id":4010,"sortIndex":32,"affiliation":4011,"properties":28},"99cfe6af-019b-4dcf-a2ec-105739351032",{"id":4010,"createTime":28,"updateTime":28,"relativeEntities":4012,"slug":28,"properties":4013,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":4016,"statistic":28},[],{"title":4014},{"EN":4015},"Department of Scientific Studies, Cardiac Pacemakers, Inc., St. Paul, MN 55112-5798.",[],{"title":4018,"openalex":4020},{"EN":4019},"Andrea Thomas",{"VOID":4021},"A5065509005",{"id":4023,"sortIndex":40,"researcher":28,"roles":4024,"affiliations":4025,"properties":4034,"displayName":4036,"givenName":28,"familyName":28},"dc0cdae7-1ffd-43d2-943e-8eeae166c41c",[],[4026],{"id":4027,"sortIndex":32,"affiliation":4028,"properties":28},"4f1f6293-ec1e-453e-8692-f475b69b0378",{"id":4027,"createTime":28,"updateTime":28,"relativeEntities":4029,"slug":28,"properties":4030,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":4033,"statistic":28},[],{"title":4031},{"EN":4032},"Departments of Scientific Studies, Clinical Programs, Biostatistics and Medical Records, Cardiac Pacemakers, Inc., St. Paul, MN",[],{"title":4035,"openalex":4037},{"EN":4036},"Suzan A. 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