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Journal of Medicine and Pharmacy","Tạp chí Y Dược học Cần Thơ",{"EN":487,"VI":488},"\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">04\u002F10\u002F2015 Ministry of Information and Communications allowed Can Tho journal of medicine and pharmacy to operate (102 \u002FGP-BTTTT)\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">07\u002F16\u002F2015 Can Tho journal of medicine and pharmacy is internationally recognized: ISSN 2354-1210\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">In 2016, The journal has been included in the list of medical science journals by The State Council for professorship which is awarded a work score of 0-0.5 points for a published article.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Can Tho Journal of Medicine and Pharmacy welcome original works that haven’t been submitted or published in other medical journals. Posts must contain content related to one of the journal’s categories.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The content published\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The journal is divided into 3 categories:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Scientific research article: are valuable scientific works, which have been researched and accepted.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Overview of medicine, biology and pharmacy: serving the objective of continuing training in the fields of medicine, biology and pharmacy; to systematize classical and modern knowledge.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Update information on new knowledge about medicine, biology, pharmacy in the country and in the world.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Scope\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Publication and introduction of scientific research in the fields:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Medicine (internal medicine, surgery, pediatrics, obstetrics and gynecology, odonto-stomatology, laboratory, oncology, traditional medicine, nursing).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Biology (genetics, biotechnology).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Pharmacology (pharmaceutics, drug quality analysis-control, synthetic pharmaceutical chemistry, biochemistry, pharmacognosy, botany, clinical pharmacy).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- To enhance the quality of undergraduate, postgraduate education, scientifically researching and meet the necessary treatment in hospital.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Introducing the updated domestic and oversea information about science technology to promote scientific research and exchanging technology in local, other universities.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Exchanging pharmaceutical and medical information for social health developing in the Mekong Delta and Vietnam.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The object\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Postgraduate students, student of Can Tho University of Medicine and Pharmacy, scientists from schools, research institutes, hospitals, health centers, pharmaceutical companies of the Mekong Delta; other provinces and regions in Vietnam and other country.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Address\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Headquarters of Can Tho Journal of Medicine and Pharmacy, located Scientific Research and International Cooperation Office: 179 Nguyen Van Cu Street, An Khanh Ward, Ninh Kieu District, Can Tho City, Vietnam.\u003C\u002Fspan>\u003C\u002Fp>","\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Ngày 16\u002F7\u002F2015, Tạp chí Y Dược học Cần Thơ được cấp chỉ số quốc tế: ISSN 2354-1210.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 4\u002F2016, Tạp chí đã được Hội đồng Giáo sư ngành Y đưa vào danh sách các tạp chí khoa học Y học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Năm 2020 Tạp chí Y Dược học Cần Thơ đã được phê duyệt vào danh mục của các Hội đồng Giáo sư ngành Dược học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ ra 12 số\u002Fnăm, 180-200 trang\u002Fsố.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 12\u002F2022 Tạp chí Y Dược học Cần Thơ là thành viên của hệ thống Crossref và từ tháng 01\u002F2023 tạp chí thực hiện bình duyệt online kín 2 chiều nhằm tăng tính minh bạch, tin cậy của các công trình nghiên cứu khoa học và đảm bảo tốt nhất chất lượng khoa học của bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ, mục đích và phạm vi của tạp chí\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ và mục đích hoạt động của tạp chí: xuất bản nhằm mục đích phổ biến kết quả từ các đề tài nghiên cứu khoa học; giao lưu trao đổi khoa học, chia sẻ kinh nghiệm, học tập, đồng thời cập nhật thông tin khoa học mới trong các lĩnh vực y, sinh, dược học trong và ngoài nước.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phạm vi của tạp chí: Tạp chí xuất bản được chia thành 3 chuyên mục: (i) Bài báo nghiên cứu khoa học là kết quả công trình nghiên cứu khoa học có giá trị đã được triển khai nghiên cứu, (ii) Bài tổng quan y, sinh, dược học: phục vụ mục tiêu đào tạo liên tục trong lĩnh vực y, sinh, dược học; nhằm hệ thống hóa những kiến thức kinh điển và hiện đại; (iii) Thông tin cập nhật kiến thức mới về y, sinh, dược học trong nước và trên thế giới.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Chính sách truy cập mở\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ áp dụng chính sách truy cập mở đối với các bài báo đã xuất bản đến với độc giả, nhằm mở rộng cơ hội tiếp cận các kết quả nghiên cứu chất lượng cao và tăng cường trao đổi kiến thức. Tạp chí đăng tải trực tuyến (miễn phí) toàn văn các bài báo được công bố trên website của Tạp chí (https:\u002F\u002Ftapchi.ctump.edu.vn).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đạo đức xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ cam kết tuân thủ đạo đức xuất bản phù hợp với các hướng dẫn và tiêu chuẩn của the Committee on Publication Ethics (COPE), tuân thủ các nguyên tắc của COPE’s Core Practices, Best Practices Guidelines for Journal Editors và Guidelines on Good Publication Practices.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Bản thảo bài báo chỉ được chấp nhận khi được tác giả chịu trách nhiệm chính cam kết các nội dung sau: Các nội dung của bản thảo chưa được đăng tải toàn bộ hoặc một phần ở các tạp chí khác; Tất cả các tác giả đều có đóng góp một cách đáng kể vào quá trình nghiên cứu hoặc chuẩn bị bản thảo và cùng chịu trách nhiệm về các nội dung của bản thảo; Tuân thủ các biện pháp đảm bảo đạo đức nghiên cứu (ví dụ thỏa thuận đồng ý tham gia nghiên cứu).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Cam kết bảo mật\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí cam kết thực hiện và tuân thủ các quy định của luật và các văn bản hướng dẫn liên quan đến bảo mật thông tin cá nhân trên không gian mạng. Các thông tin mà người dùng (tác giả, độc giả, biên tập viên, người phản biện) nhập vào các biểu mẫu trên Hệ thống Quản lý xuất bản trực tuyến của tạp chí chỉ được sử dụng vào các mục đích đã được tuyên bố rõ ràng và sẽ không được cung cấp cho bất kỳ bên thứ ba nào khác, hay dùng vào bất kỳ mục đích nào khác.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phí gửi bài\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng bài: 1.000.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng nhanh: 1.500.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với tác giả là cán bộ viên chức thuộc Trường Đại học Y Dược Cần Thơ thì được hỗ trợ 50% lệ phí gửi đăng bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với sinh viên thực hiện đề tài nghiên cứu khoa học cấp trường được hỗ trợ 100% lệ phí đăng bài ( Tác giả gửi đính kèm “ Quyết định về việc giao tổ chức thực hiện đề tài nghiên cứu khoa học cấp Trường của sinh viên”).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Hình thức nộp lệ phí:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Tiền mặt:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Nộp trực tiếp tại Phòng Tài chính - Kế toán, Trường Đại học Y Dược Cần Thơ, số 179 Nguyễn Văn Cừ, P. An Khánh, Q. Ninh Kiều, thành phố Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Chuyển khoản:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tên Tài khoản: Trường ĐHYD Cần Thơ, Số TK: 0111000115668, tại ngân hàng Vietcombank chi nhánh Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Thời gian: Áp dụng từ ngày 01\u002F02\u002F2023.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">* Phí gửi bài không được hoàn trả khi bài viết bị từ chối hoặc tác giả xin rút bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Quy trình phản biện bài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ thực hiện quy trình phản biện kín hai chiều nghiêm ngặt. Danh tính của những người phản biện không được tiết lộ cho các tác giả và ngược lại. Quy trình thẩm định bài báo đăng gồm các bước sau:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tiếp nhận bản thảo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tác giả liên hệ gửi bản thảo đến Tạp chí qua hệ thống trực tuyến tại website: https:\u002F\u002Ftapchi.ctump.edu.vn. Hướng dẫn về cách đăng ký, gửi bài và chuẩn bị bản thảo được cung cấp trên website của Tạp chí.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sàng lọc sơ bộ\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sau khi Tòa soạn nhận được bài báo của tác giả, Ban Thư ký sẽ tiến hành kiểm tra sơ bộ bài báo (các yêu cầu về nội dung và hình thức). Những bài báo không đúng quy cách hoặc có nội dung không phù hợp hoặc vi phạm bản quyền sẽ bị từ chối (Ban Thư ký thông báo phản hồi đến tác giả trong vòng 1 tuần). Những bài báo đủ điều kiện, được Ban Thư ký tòa soạn chuyển đến Ban Biên tập có cùng chuyên môn với nội dung bài báo để đề xuất người phản biện. Thời gian kể từ khi Ban Biên tập nhận bài báo đến khi đề xuất người phản biện bài báo chậm nhất là 5 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Vòng phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký gửi bài và yêu cầu phản biện đến 02 phản biện độc lập.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Các phản biện gởi nhận xét cho Ban Thư ký. Thời gian từ khi gửi bài cho phản biện đến khi nhận ý kiến của phản biện tối đa là 20 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xử ký kết quả phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Nếu ý kiến đồng ý cho đăng và không cần chỉnh sửa, Ban Thư ký tiếp tục đăng bài theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Nếu ý kiến đồng ý đăng và cần chỉnh sửa, Ban Thư ký sẽ thông tin đến tác giả chỉnh sửa theo yêu cầu của người phản biện. Thời gian chỉnh sửa và gửi lại kéo dài không quá 2 tuần, từ khi tác giả bài báo nhận được thông tin (Quá trình này có thể lặp lại tối đa 2 lần\u002F1 bài báo). Khi có sự thống nhất, đồng ý của người phản biện; bài báo được tiếp tục đăng theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Những bài báo có chất lượng không đạt yêu cầu, cả 2 phản biện không đồng ý cho đăng sẽ bị Tòa soạn từ chối đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký tổng hợp các bản thảo đã được tác giả hoàn thiện sau thẩm định trình Ban Biên tập xem xét, Tổng Biên tập phê duyệt, quyết định bài đăng theo các tiêu chí: sự phù hợp nội dung với tôn chỉ và mục đích, thể loại bài viết (ưu tiên các bài có bài có nghiên cứu chuyên sâu, hàm lượng khoa học cao), đóng góp mới bài báo, bài báo được ưu tiên đăng trong số gần nhất của Tạp chí theo thứ tự: tính thời sự, chất lượng bài báo và thời gian gửi bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Ban Biên tập và Ban Thư ký biên tập bản thảo, chế bản, đọc rà soát lỗi. Thời gian hoàn thành từ 10-15 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Ban Thư ký có trách nhiệm thông báo cho tác giả bài báo (bằng e-mail) về tình hình phê duyệt bài báo, thời gian, số kỳ, tập xuất bản bài báo theo qui định.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">4. Danh sách bài báo theo số Tạp chí được in ấn và phát hành trong năm định kỳ được công bố chính thức trên website: https:\u002F\u002Ftapchi.ctump.edu.vn\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>",{"VOID":490},"wcQ1uqwAAAAJ","2023-05-30T08:17:21.868+00:00",[],[494],{"id":495,"createTime":28,"updateTime":28,"relativeEntities":496,"slug":28,"properties":497,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":507,"parentIds":508,"statistic":28},"6413896b-eca9-442b-a73f-182a58a0ce40",[],{"title":498,"address":501,"country":504,"abbreviation":505},{"EN":499,"VI":500},"Can Tho University of Medicine and Pharmacy","Trường Đại học Y Dược Cần Thơ",{"EN":502,"VI":503},"No 179, Nguyen Van Cu street, An Khanh ward, Ninh Kieu district, Can Tho city, Vietnam","Số 179, đường Nguyễn Văn Cừ, phường An Khánh, quận Ninh Kiều, thành phố Cần Thơ, Việt Nam",{"VOID":15},{"VOID":506},"ctump","http:\u002F\u002Fwww.ctump.edu.vn\u002F",[],[],"https:\u002F\u002Ftapchi.ctump.edu.vn\u002Findex.php\u002Fctump",{"impactFactor":32,"impactFactorByYear":512,"i10Index":32,"i10IndexLast5Year":32,"totalPublication":514,"totalPublicationByYear":515,"totalCitation":520,"totalCitationByYear":521,"totalCitationPerPublication":108,"totalCitationPerPublicationByYear":523,"hindexLast5Year":45,"hindex":45},{"2022":513,"2023":111,"2024":106},0.01,1556,{"2020":47,"2021":516,"2022":517,"2023":518,"2024":519,"2025":122},57,306,801,358,161,{"2021":146,"2022":280,"2023":522},99,{"2021":524,"2022":318,"2023":104},0.23,{"impactFactor":28,"impactFactorByYear":28,"i10Index":123,"i10IndexLast5Year":123,"totalPublication":526,"totalPublicationByYear":527,"totalCitation":526,"totalCitationByYear":528,"totalCitationPerPublication":40,"totalCitationPerPublicationByYear":531,"hindexLast5Year":49,"hindex":49},476,{"0":205,"2019":123,"2021":139,"2022":459,"2023":451,"2024":357,"2025":49,"2026":48},{"2021":42,"2022":123,"2023":161,"2024":529,"2025":360,"2026":530},136,83,{"2021":105,"2022":513,"2023":532,"2024":127,"2025":533,"2026":534},0.62,25.43,13.83,{"id":536,"createTime":537,"updateTime":382,"relativeEntities":538,"slug":539,"properties":540,"entityType":25,"verifyStatus":26,"verifyTime":28,"verifyNote":28,"languages":552,"translateLanguages":28,"viewCount":133,"subjectFields":553,"manageAffiliations":554,"indexDatabases":555,"url":556,"thumbnailPath":557,"statistic":558,"gsStatistic":594,"type":55,"analyzePriority":28},"6984a56a-db70-403b-9cc4-4013e1ceaffa","2023-05-09T06:47:40.346+00:00",[],"T%E1%BA%A1p%20ch%C3%AD%20Nghi%C3%AAn%20c%E1%BB%A9u%20n%C6%B0%E1%BB%9Bc%20ngo%C3%A0i",{"country":541,"issn":542,"title":544,"introduce":547,"gsId":550},{"VOID":15},{"VOID":543},"25252445",{"EN":545,"VI":546},"VNU Journal of Foreign Studies","Tạp chí Nghiên cứu nước ngoài",{"EN":548,"VI":549},"{\"ops\":[{\"insert\":\"\\n\\nThe \\n\"},{\"attributes\":{\"italic\":true},\"insert\":\"VNU Journal of Science\"},{\"insert\":\"\\n was established in 1985 for the publication of national and international research papers in all fields of natural sciences and technology, social sciences and humanities. 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Rare, resident cells of bone marrow and periosteum, referred to as mesenchymal stem cells, have been shown to differentiate into a number of mesenchymal phenotypes including bone, cartilage, and adipocytes. Rat bone marrow‐derived mesenchymal stem cells were exposed to 5‐azacytidine beginning 24 h after seeding twice‐passaged cells into culture dishes. After an exposure of 24 h, long, multinucleated myotubes were observed in some of the dishes 7–11 days later. Cells containing Sudan black‐positive droplets in their cytoplasm were also observed. Thus, culture‐propagated rat bone marrow mesenchymal stem cells appear to have the capacity to be induced to differentiate in vitro into myogenic and adipocytic phenotypes, although nonmesenchymal cells (rat brain fibroblasts) cannot be so induced. 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Sau 24 giờ tiếp xúc, các ống cơ dài, đa nhân đã được quan sát thấy trong một số đĩa 7–11 ngày sau đó. Các tế bào chứa các giọt dương tính với Sudan black trong bào tương cũng đã được quan sát. Do đó, các tế bào gốc trung mô đã nuôi cấy từ tủy xương chuột dường như có khả năng được kích thích để phân hóa in vitro thành các kiểu hình cơ và mỡ, mặc dù các tế bào không phải trung mô (tế bào sợi não chuột) thì không thể được kích thích như vậy. 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ceramic vehicles for rat marrow‐derived osteogenic cell delivery: effects of pretreatment with fibronectin or laminin, J Oral Implant, 19, 106",{},{"id":28,"text":1194,"url":28,"identifiers":1195},"Dennis JE, 1991, Osteogenesis in marrow‐derived mesenchymal stem cell porous ceramic composites transplanted subcutaneously: effect of fibronectin and laminin on cell retention and rate of osteogenic expression, Cell Transplant, 1, 2332",{},{"id":28,"text":1197,"url":28,"identifiers":1198},"10.1002\u002Ftera.1420240212",{"doi":1197},{"id":28,"text":1200,"url":28,"identifiers":1201},"10.1097\u002F00007890-196803000-00009",{"doi":1200},{"id":28,"text":1203,"url":28,"identifiers":1204},"Goshima J, 1991, The osteogenic potential of culture‐expanded rat marrow mesenchymal cells assayed in vivo in calcium phosphate ceramic blocks, Clin Orthop Rel Res, 262, 298, 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cells derived from the periosteum, Clin Orthop Rel Res, 259, 223, 10.1097\u002F00003086-199010000-00032",{"doi":1233},"10.1097\u002F00003086-199010000-00032",{"id":28,"text":1235,"url":28,"identifiers":1236},"10.1016\u002F0014-4827(91)90401-F",{"doi":1235},{"id":28,"text":1238,"url":28,"identifiers":1239},"10.1002\u002Fjor.1100090402",{"doi":1238},{"id":28,"text":1241,"url":28,"identifiers":1242},"Nakahara H, 1992, Culture‐expanded periosteal‐derived cells exhibit osteochondrogenic potential in porous calcium phosphate ceramics in vivo, Clin Orthop Rel Res, 276, 291, 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factors limiting force production and exercise endurance time have been briefly described, together with some of the changes occurring at various sites within the muscle and central nervous system. Evidence is presented that, in fatigue of sustained maximal voluntary contractions (MVC) executed by well‐motivated subjects, the reduction in force generating capacity need not be due to a decline in central nervous system (CNS) motor drive or to failing neuromuscular transmission, but can be attributed solely to contractile failure of the muscles involved. However, despite this conclusion, both the integrated electromyogram (EMG) and the mean firing rate of individual motor units do decline progressively during sustained MVC. This, however, does not necessarily result in loss of force since the parallel slowing of muscle contractile speed reduces tetanic fusion frequency. It is suggested that the range of motoneuron firing rates elicited by voluntary effort is regulated and limited for each muscle to the minimum required for maximum force generation, thus preventing neuromuscular transmission failure and optimizing motor control. Such a CNS regulating mechanism would probably require some reflex feedback from the muscle.\u003C\u002Fjats:p>","\u003Cjats:title>Tóm tắt\u003C\u002Fjats:title>\u003Cjats:p>Những yếu tố hạn chế khả năng sản xuất lực và thời gian chịu đựng khi tập thể dục đã được mô tả một cách ngắn gọn, cùng với một số thay đổi xảy ra ở các vị trí khác nhau trong cơ và hệ thần kinh trung ương. Bằng chứng được đưa ra cho thấy, trong tình trạng mỏi khi thực hiện các cơn co bóp tối đa tự nguyện kéo dài (MVC) bởi những cá nhân có động lực cao, sự giảm khả năng tạo ra lực không nhất thiết phải do suy giảm trong động lực thần kinh trung ương (CNS) hoặc do truyền dẫn thần kinh cơ không hiệu quả, mà có thể được quy cho sự thất bại trong co bóp của các cơ tham gia. Tuy nhiên, mặc dù có kết luận này, cả điện cơ tích hợp (EMG) và tần suất phát xung trung bình của các đơn vị vận động cá nhân đều giảm dần trong suốt quá trình thực hiện MVC kéo dài. Tuy nhiên, điều này không nhất thiết dẫn đến mất lực do sự giảm tốc độ co bóp của cơ song song làm giảm tần số hợp nhất co cơ. Có thể đề xuất rằng phạm vi tần suất phát xung của các nơ-ron vận động được kích thích bởi nỗ lực tự nguyện được điều chỉnh và giới hạn cho mỗi cơ đến mức tối thiểu cần thiết cho việc sản xuất lực tối đa, từ đó ngăn chặn sự thất bại trong truyền dẫn thần kinh cơ và tối ưu hóa kiểm soát vận động. Cơ chế điều tiết này của hệ thần kinh trung ương có thể sẽ cần một số phản hồi phản xạ từ cơ.",{"EN":1286,"VI":1287},"Changes in muscle contractile properties and neural control during human muscular fatigue","Những thay đổi trong các đặc tính co bóp của cơ và kiểm soát thần kinh trong quá trình mỏi cơ ở người",{"VOID":1289},"6100456",{"VOID":1291},"10.1002\u002Fmus.880070902",[31],[30],"https:\u002F\u002Fonlinelibrary.wiley.com\u002Fdoi\u002F10.1002\u002Fmus.880070902",[1296,1313],{"id":1297,"sortIndex":32,"researcher":28,"roles":1298,"affiliations":1299,"properties":1308,"displayName":1310,"givenName":28,"familyName":28},"4e1033c4-259d-44f6-9645-e4eafef04c42",[],[1300],{"id":1301,"sortIndex":32,"affiliation":1302,"properties":28},"91f96087-10a8-458c-b8e9-7a32401cf82a",{"id":1301,"createTime":28,"updateTime":28,"relativeEntities":1303,"slug":28,"properties":1304,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1307,"statistic":28},[],{"title":1305},{"EN":1306},"John B. 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AY, 1981, Extent of motor unit activation during effort, J Appl Physiol, 50, 1131, 10.1152\u002Fjappl.1981.51.5.1131",{"doi":1409},"10.1152\u002Fjappl.1981.51.5.1131",{"id":28,"text":1411,"url":28,"identifiers":1412},"Bellemare F, 1984, Localization of human diaphragm fatigue, Fed Proc Abst, 43, 530",{},{"id":28,"text":1414,"url":28,"identifiers":1415},"10.1152\u002Fjappl.1982.53.5.1190",{"doi":1414},{"id":28,"text":1417,"url":28,"identifiers":1418},"10.1152\u002Fjn.1983.50.6.1380",{"doi":1417},{"id":28,"text":1420,"url":28,"identifiers":1421},"10.1113\u002Fjphysiol.1954.sp005161",{"doi":1420},{"id":28,"text":1423,"url":28,"identifiers":1424},"10.1249\u002F00003677-198101000-00002",{"doi":1423},{"id":28,"text":1426,"url":28,"identifiers":1427},"Bigland‐Ritchie B, 1983, Central and peripheral fatigue in intermittent submaximal contractions, Neurosci Abst, 9, 631",{},{"id":28,"text":1429,"url":28,"identifiers":1430},"10.1152\u002Fjn.1983.50.1.313",{"doi":1429},{"id":28,"text":1429,"url":28,"identifiers":1432},{"doi":1429},{"id":28,"text":1434,"url":28,"identifiers":1435},"Bigland‐Ritchie B, 1978, Central and peripheral fatigue in sustained maximum voluntary contractions of human quadriceps muscle, Clin Sci Mol Med, 54, 609",{},{"id":28,"text":1437,"url":28,"identifiers":1438},"10.1016\u002F0014-4886(79)90280-2",{"doi":1437},{"id":28,"text":1440,"url":28,"identifiers":1441},"10.1113\u002Fjphysiol.1982.sp014340",{"doi":1440},{"id":28,"text":1443,"url":28,"identifiers":1444},"10.1002\u002F9780470715420",{"doi":1443},{"id":28,"text":1446,"url":28,"identifiers":1447},"Clamann HP, 1979, Relation between force and fatigability of red and pale skeletal muscles in man, Am J Phys Med, 58, 70",{},{"id":28,"text":1449,"url":28,"identifiers":1450},"10.1113\u002Fjphysiol.1977.sp012072",{"doi":1449},{"id":28,"text":1452,"url":28,"identifiers":1453},"Henneman E, 1980, Medical Physiology, 674",{},{"id":28,"text":1455,"url":28,"identifiers":1456},"10.1113\u002Fjphysiol.1963.sp007094",{"doi":1455},{"id":28,"text":1458,"url":28,"identifiers":1459},"Ikai M, 1967, Muskelkraft und Muskulare Ermdung bei wilkurlieher Anspannung und elekrischer Reizung des Muskels, Sportarz Sportmedizin, 5, 197",{},{"id":28,"text":1461,"url":28,"identifiers":1462},"Krnjevic K, 1958, Failure of neuromuscular propagation in rats, J Physiol (Lond), 140, 440, 10.1113\u002Fjphysiol.1958.sp005944",{"doi":1463},"10.1113\u002Fjphysiol.1958.sp005944",{"id":28,"text":1465,"url":28,"identifiers":1466},"10.1016\u002F0006-8993(81)90266-3",{"doi":1465},{"id":28,"text":1468,"url":28,"identifiers":1469},"10.1113\u002Fjphysiol.1954.sp005070",{"doi":1468},{"id":28,"text":1471,"url":28,"identifiers":1472},"10.1002\u002F9780470715420.ch8",{"doi":1471},{"id":28,"text":1474,"url":28,"identifiers":1475},"10.1113\u002Fjphysiol.1975.sp010904",{"doi":1474},{"id":28,"text":1477,"url":28,"identifiers":1478},"10.1113\u002Fjphysiol.1973.sp010192",{"doi":1477},{"id":28,"text":1480,"url":28,"identifiers":1481},"10.1111\u002Fj.1748-1716.1955.tb01255.x",{"doi":1480},{"id":28,"text":1483,"url":28,"identifiers":1484},"Rohmert W, 1960, Ermittung von Erholung‐spausen fur statische Arbeit des Menschen, Int Angew Physiol, 18, 123",{},{"id":28,"text":1486,"url":28,"identifiers":1487},"10.1113\u002Fjphysiol.1972.sp009691",{"doi":1486},{"id":28,"text":1489,"url":28,"identifiers":1490},"10.1002\u002F9780470715420.ch7",{"doi":1489},{"id":28,"text":1492,"url":28,"identifiers":1493},"Woods JJ, 1983, Linear and non‐linear surface EMG\u002Fforce relationships in human muscles: an anatomical\u002Ffunctional argument for the existence of both, Am J Phys Med, 62, 287",{},{"id":1495,"createTime":1496,"updateTime":1497,"relativeEntities":1498,"slug":1499,"properties":1500,"entityType":1029,"verifyStatus":26,"verifyTime":1496,"verifyNote":1030,"languages":1516,"translateLanguages":1517,"viewCount":32,"primaryUrl":1518,"fullTextUrl":28,"authors":1519,"publicationType":1092,"publisherRelationship":1600,"citationCount":1663,"citationInfo":1664,"publishDate":1667,"publishYear":1665,"citationAnalyzeStatus":884,"lastCitationAnalyze":28,"indexDatabases":1668,"openAccess":28,"references":1669,"isForceReanalyzing":1268},"56b86756-5498-40e3-b453-74457d34c650","2024-10-01T06:40:50.426+00:00","2025-02-12T16:55:43.912+00:00",[],"Measurement-of-muscle-contraction-with-ultrasound-imaging",{"mag":1501,"keywords":1503,"openalex":1504,"abstract":1506,"title":1509,"pm":1512,"doi":1514},{"VOID":1502},"2008276678",{"VI":1279},{"VOID":1505},"W2008276678",{"EN":1507,"VI":1508},"\u003Cjats:title>Abstract\u003C\u002Fjats:title>\u003Cjats:p>To investigate the ability of ultrasonography to estimate muscle activity, we measured architectural parameters (pennation angles, fascicle lengths, and muscle thickness) of several human muscles (tibialis anterior, biceps brachii, brachialis, transversus abdominis, obliquus internus abdominis, and obliquus externus abdominis) during isometric contractions of from 0 to 100% maximal voluntary contraction (MVC). Concurrently, electromyographic (EMG) activity was measured with surface (tibialis anterior only) or fine‐wire electrodes. Most architectural parameters changed markedly with contractions up to 30% MVC but changed little at higher levels of contraction. Thus, ultrasound imaging can be used to detect low levels of muscle activity but cannot discriminate between moderate and strong contractions. Ultrasound measures could reliably detect changes in EMG of as little as 4% MVC (biceps muscle thickness), 5% MVC (brachialis muscle thickness), or 9% MVC (tibialis anterior pennation angle). They were generally less sensitive to changes in abdominal muscle activity, but it was possible to reliably detect contractions of 12% MVC in transversus abdominis (muscle length) and 22% MVC in obliquus internus (muscle thickness). Obliquus externus abdominis thickness did not change consistently with muscle contraction, so ultrasound measures of thickness cannot be used to detect activity of this muscle. Ultrasound imaging can thus provide a noninvasive method of detecting isometric muscle contractions of certain individual muscles. Muscle Nerve 27: 682–692, 2003\u003C\u002Fjats:p>","\u003Cjats:title>Tóm tắt\u003C\u002Fjats:title>\u003Cjats:p>Để khảo sát khả năng của siêu âm trong việc ước lượng hoạt động cơ, chúng tôi đã đo các tham số kiến trúc (góc pennation, độ dài sợi cơ, và độ dày cơ) của một số nhóm cơ ở người (cơ chày trước, cơ nhị đầu cánh tay, cơ cánh tay, cơ ngang bụng, cơ chéo trong bụng, và cơ chéo ngoài bụng) trong các tình huống co cơ isometric từ 0 đến 100% mức co cơ tự nguyện tối đa (MVC). Đồng thời, hoạt động điện cơ (EMG) được đo bằng điện cực bề mặt (chỉ với cơ chày trước) hoặc điện cực dây nhỏ. Hầu hết các tham số kiến trúc thay đổi rõ rệt với các mức co cơ lên tới 30% MVC nhưng thay đổi rất ít ở các mức co cơ cao hơn. Vì vậy, hình ảnh siêu âm có thể được sử dụng để phát hiện mức độ hoạt động cơ thấp nhưng không thể phân biệt giữa các mức co cơ vừa và mạnh. Các phép đo siêu âm có thể đáng tin cậy phát hiện những thay đổi trong EMG ở mức thấp nhất là 4% MVC (độ dày cơ nhị đầu), 5% MVC (độ dày cơ cánh tay), hoặc 9% MVC (góc pennation của cơ chày trước). Chúng thường kém nhạy hơn với những thay đổi trong hoạt động của cơ bụng, nhưng có thể đáng tin cậy phát hiện sự co cơ ở mức 12% MVC trong cơ ngang bụng (chiều dài cơ) và 22% MVC trong cơ chéo trong (độ dày cơ). Độ dày của cơ chéo ngoài bụng không thay đổi nhất quán với sự co cơ, do đó phép đo siêu âm về độ dày không thể được sử dụng để phát hiện hoạt động của cơ này. Vì vậy, hình ảnh siêu âm có thể cung cấp một phương pháp không xâm lấn để phát hiện các cơn co cơ isometric của một số cơ riêng lẻ.",{"EN":1510,"VI":1511},"Measurement of muscle contraction with ultrasound imaging","Đo lường sự co cơ bằng hình ảnh siêu âm",{"VOID":1513},"12766979",{"VOID":1515},"10.1002\u002Fmus.10375",[31],[30],"https:\u002F\u002Fonlinelibrary.wiley.com\u002Fdoi\u002F10.1002\u002Fmus.10375",[1520,1547,1566,1583],{"id":1521,"sortIndex":32,"researcher":28,"roles":1522,"affiliations":1523,"properties":1540,"displayName":1544,"givenName":28,"familyName":28},"b61d2467-13c9-466c-8a2f-690cdf5e0d16",[],[1524,1532],{"id":1525,"sortIndex":32,"affiliation":1526,"properties":28},"4b47a993-becf-4dd3-8a12-b54f05e98efc",{"id":1525,"createTime":28,"updateTime":28,"relativeEntities":1527,"slug":28,"properties":1528,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1531,"statistic":28},[],{"title":1529},{"EN":1530},"Department of Physiotherapy, University of Queensland, Brisbane, Queensland 4072, Australia",[],{"id":1533,"sortIndex":40,"affiliation":1534,"properties":28},"f175c089-b597-45a5-b9f9-9bded1dfd28c",{"id":1533,"createTime":28,"updateTime":28,"relativeEntities":1535,"slug":28,"properties":1536,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1539,"statistic":28},[],{"title":1537},{"EN":1538},"Prince of Wales Medical Research Institute and the University of New South Wales, Sydney, New South Wales 2031, Australia",[],{"orcid":1541,"title":1543,"openalex":1545},{"VOID":1542},"https:\u002F\u002Forcid.org\u002F0000-0002-1206-9107",{"EN":1544},"Paul W. 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359",{},{"id":28,"text":1750,"url":28,"identifiers":1751},"10.1016\u002F0021-9290(94)90020-5",{"doi":1750},{"id":1753,"createTime":1754,"updateTime":1755,"relativeEntities":1756,"slug":1757,"properties":1758,"entityType":1029,"verifyStatus":884,"verifyTime":1754,"verifyNote":1775,"languages":1776,"translateLanguages":1777,"viewCount":32,"primaryUrl":1778,"fullTextUrl":28,"authors":1779,"publicationType":1092,"publisherRelationship":1841,"citationCount":1904,"citationInfo":1905,"publishDate":1908,"publishYear":1906,"citationAnalyzeStatus":884,"lastCitationAnalyze":28,"indexDatabases":1909,"openAccess":28,"references":1910,"isForceReanalyzing":1268},"fa4abe45-2c9e-4231-a8ce-b5049692590a","2024-09-25T08:07:14.597+00:00","2025-02-12T16:56:47.829+00:00",[],"Entries-in-the-Leiden-Duchenne-muscular-dystrophy-mutation-database-An-overview-of-mutation-types-and-paradoxical-cases-that-confirm-the-reading-frame-rule",{"mag":1759,"keywords":1761,"openalex":1763,"abstract":1765,"title":1768,"pm":1771,"doi":1773},{"VOID":1760},"2137703332",{"VI":1762},"Duchenne muscular dystrophy; Becker muscular dystrophy; DMD gene; dystrophin; reading-frame rule; genotype–phenotype correlations; mutation variability",{"VOID":1764},"W2137703332",{"EN":1766,"VI":1767},"\u003Cjats:title>Abstract\u003C\u002Fjats:title>\u003Cjats:p>The severe Duchenne and milder Becker muscular dystrophy are both caused by mutations in the \u003Cjats:italic>DMD\u003C\u002Fjats:italic> gene. This gene codes for dystrophin, a protein important for maintaining the stability of muscle‐fiber membranes. In 1988, Monaco and colleagues postulated an explanation for the phenotypic difference between Duchenne and Becker patients in the reading‐frame rule: In Duchenne patients, mutations induce a shift in the reading frame leading to prematurely truncated, dysfunctional dystrophins. In Becker patients, in‐frame mutations allow the synthesis of internally deleted, but largely functional dystrophins. Currently, over 4700 mutations have been reported in the Leiden DMD mutation database, of which 91% are in agreement with this rule. In this study we provide an update of the mutational variability in the \u003Cjats:italic>DMD\u003C\u002Fjats:italic> gene, particularly focusing on genotype–phenotype correlations and mutations that appear to be exceptions to the reading‐frame rule. Muscle Nerve, 2006\u003C\u002Fjats:p>","\u003Cjats:title>Tóm tắt\u003C\u002Fjats:title>\u003Cjats:p>Bệnh cơ nhỡ Duchenne nặng và bệnh cơ nhỡ Becker nhẹ đều do đột biến trong gen \u003Cjats:italic>DMD\u003C\u002Fjats:italic> gây ra. Gen này mã hóa cho dystrophin, một protein quan trọng trong việc duy trì độ ổn định của màng sợi cơ. Vào năm 1988, Monaco và các đồng nghiệp đã đưa ra giả thuyết giải thích sự khác biệt kiểu hình giữa bệnh nhân Duchenne và bệnh nhân Becker dựa trên quy tắc khung đọc: Ở bệnh nhân Duchenne, các đột biến gây ra sự dịch chuyển trong khung đọc dẫn đến sản xuất dystrophin bị rút ngắn và không chức năng. Ở bệnh nhân Becker, các đột biến đúng khung cho phép tổng hợp dystrophin bị thiếu một phần bên trong, nhưng vẫn chủ yếu có chức năng. Hiện tại, đã có hơn 4700 đột biến được báo cáo trong cơ sở dữ liệu đột biến DMD Leiden, trong đó 91% phù hợp với quy tắc này. Trong nghiên cứu này, chúng tôi cung cấp bản cập nhật về sự biến đổi đột biến trong gen \u003Cjats:italic>DMD\u003C\u002Fjats:italic>, đặc biệt tập trung vào các mối tương quan kiểu gen – kiểu hình và các đột biến dường như là ngoại lệ cho quy tắc khung đọc. Muscle Nerve, 2006\u003C\u002Fjats:p>",{"EN":1769,"VI":1770},"Entries in the Leiden Duchenne muscular dystrophy mutation database: An overview of mutation types and paradoxical cases that confirm the reading‐frame rule","Các mục trong cơ sở dữ liệu đột biến bệnh cơ nhỡ Duchenne Leiden: Tổng quan về các loại đột biến và các trường hợp nghịch lý xác nhận quy tắc khung đọc",{"VOID":1772},"16770791",{"VOID":1774},"10.1002\u002Fmus.20586","Author affiliation is blank",[31],[30],"https:\u002F\u002Fonlinelibrary.wiley.com\u002Fdoi\u002F10.1002\u002Fmus.20586",[1780,1799,1808,1819,1830],{"id":1781,"sortIndex":32,"researcher":28,"roles":1782,"affiliations":1783,"properties":1792,"displayName":1796,"givenName":28,"familyName":28},"2f973413-b9ab-4e9b-84fc-85649d9e1a56",[],[1784],{"id":1785,"sortIndex":32,"affiliation":1786,"properties":28},"a5d17340-3ad5-405b-a393-d1bd8198bcad",{"id":1785,"createTime":28,"updateTime":28,"relativeEntities":1787,"slug":28,"properties":1788,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1791,"statistic":28},[],{"title":1789},{"EN":1790},"Leiden University Medical Center, Department of Human Genetics, P.O. 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This unsatisfactory state may relate to evaluation of different patient groups, different minimal criteria for the diagnosis of neuropathy, and different degrees of surveillance. To made matters worse, patients with polyneuropathy tend to be equated ignoring differences in severity. To remedy this situation, four recommendations are made: (1) population‐based patients should be studied, (2) nerve conduction should be used to set minimal criteria for neuropathy because the test is objective, sensitive, and repeatable, (3) validated tests of symptoms and deficits should also be used because clinical manifestations of neuropathy cannot be accurately inferred from electrophysiologic measurements, and (4) approaches to staging severity of neuropathy should be developed and used in expressing abnormality. To this end minimal criteria for the diagnosis of diabetic polyneuropathy have been proposed, and validated tests to assess neuropathic symptoms and sensory deficits have been developed. In this report we also propose a staging approach utilizing nerve conduction and neurologic history and examination and validated tests of neuropathic symptoms and deficits.\u003C\u002Fjats:p>","\u003Cjats:title>Tóm tắt\u003C\u002Fjats:title>\u003Cjats:p> Tỷ lệ hiện mắc bệnh đa dây thần kinh tiểu đường được báo cáo dao động từ 5 đến 80%. Tình trạng không thỏa đáng này có thể liên quan đến việc đánh giá các nhóm bệnh nhân khác nhau, các tiêu chí tối thiểu khác nhau để chẩn đoán bệnh thần kinh, và các mức độ giám sát khác nhau. Để làm tình hình tồi tệ hơn, bệnh nhân mắc đa dây thần kinh thường bị coi là tương tự nhau mà không xem xét sự khác biệt về mức độ nặng. Để khắc phục tình hình này, bốn khuyến nghị được đưa ra: (1) nên nghiên cứu các bệnh nhân trên cơ sở dân số, (2) sử dụng đo dẫn truyền dây thần kinh để xác định tiêu chí tối thiểu cho bệnh thần kinh vì bài test này là khách quan, nhạy và có thể lặp lại, (3) cũng nên sử dụng các bài kiểm tra đã được xác nhận về triệu chứng và khiếm khuyết vì các biểu hiện lâm sàng của bệnh thần kinh không thể suy luận chính xác từ các đo lường điện sinh lý, và (4) cần phát triển và sử dụng các phương pháp phân độ mức độ nặng của bệnh thần kinh trong việc biểu thị sự bất thường. Để đạt được điều này, các tiêu chí tối thiểu cho chẩn đoán bệnh đa dây thần kinh tiểu đường đã được đề xuất và các bài kiểm tra đã được xác nhận để đánh giá triệu chứng thần kinh và khiếm khuyết cảm giác đã được phát triển. Trong báo cáo này, chúng tôi cũng đề xuất một phương pháp phân độ sử dụng đo dẫn truyền dây thần kinh và tiền sử cũng như khám lâm sàng thần kinh và các bài kiểm tra đã được xác nhận về triệu chứng và khiếm khuyết thần kinh.\u003C\u002Fjats:p>",{"EN":2111,"VI":2112},"Detection, characterization, and staging of polyneuropathy: Assessed in diabetics","Phát hiện, đặc trưng và phân độ của bệnh đa dây thần kinh: Đánh giá trên bệnh nhân tiểu đường",{"VOID":2114},"3277049",{"VOID":2116},"10.1002\u002Fmus.880110106",[31],[30],"https:\u002F\u002Fonlinelibrary.wiley.com\u002Fdoi\u002F10.1002\u002Fmus.880110106",[2121],{"id":2122,"sortIndex":32,"researcher":28,"roles":2123,"affiliations":2124,"properties":2133,"displayName":2137,"givenName":28,"familyName":28},"e64916d4-7b02-4f3e-a38b-ace7486fc725",[],[2125],{"id":2126,"sortIndex":32,"affiliation":2127,"properties":28},"2e739d90-1b01-4c49-a1a2-1b94fceb6f62",{"id":2126,"createTime":28,"updateTime":28,"relativeEntities":2128,"slug":28,"properties":2129,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2132,"statistic":28},[],{"title":2130},{"EN":2131},"Peripheral Nerve Center, Mayo Clinic & Foundation, Rochester, MN 55905",[],{"orcid":2134,"title":2136,"openalex":2138},{"VOID":2135},"https:\u002F\u002Forcid.org\u002F0000-0003-0212-8254",{"EN":2137},"P. 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Dyck",{"VOID":2139},"A5005132437",{"url":28,"publisher":2141,"properties":2196},{"id":868,"createTime":869,"updateTime":870,"relativeEntities":2142,"slug":872,"properties":2143,"entityType":25,"verifyStatus":884,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":32,"subjectFields":2148,"manageAffiliations":2165,"indexDatabases":2176,"url":958,"thumbnailPath":28,"statistic":2191,"gsStatistic":28,"type":28,"analyzePriority":28},[],{"country":2144,"eissn":2145,"issn":2146,"title":2147},{"VOID":875},{"VOID":877},{"VOID":879},{"EN":881},[2149,2153,2157,2161],{"id":887,"createTime":28,"updateTime":28,"relativeEntities":2150,"label":2151,"description":2152,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":890},{},{"id":893,"createTime":28,"updateTime":28,"relativeEntities":2154,"label":2155,"description":2156,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":896},{},{"id":899,"createTime":28,"updateTime":28,"relativeEntities":2158,"label":2159,"description":2160,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":902},{},{"id":905,"createTime":28,"updateTime":28,"relativeEntities":2162,"label":2163,"description":2164,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":908},{},[2166,2171],{"id":912,"createTime":28,"updateTime":28,"relativeEntities":2167,"slug":28,"properties":2168,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2170,"statistic":28},[],{"title":2169},{"EN":916},[],{"id":919,"createTime":28,"updateTime":28,"relativeEntities":2172,"slug":28,"properties":2173,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2175,"statistic":28},[],{"title":2174},{"EN":923},[],[2177,2184],{"id":927,"indexDatabase":2178,"url":933,"indexYears":934,"academicFieldIds":2183,"indexDatabaseRanking":940},{"id":775,"createTime":28,"updateTime":28,"relativeEntities":2179,"label":2180,"description":2181,"key":781,"publicationTags":2182,"standard":28},[],{"EN":778,"VI":778},{"EN":778,"VI":780},[783],[936,937,938,939],{"id":942,"indexDatabase":2185,"url":954,"indexYears":28,"academicFieldIds":2190,"indexDatabaseRanking":28},{"id":944,"createTime":28,"updateTime":28,"relativeEntities":2186,"label":2187,"description":2188,"key":951,"publicationTags":2189,"standard":28},[],{"EN":947,"VI":947},{"EN":949,"VI":950},[953,813],[956,957],{"impactFactor":32,"impactFactorByYear":2192,"i10Index":611,"i10IndexLast5Year":45,"totalPublication":333,"totalPublicationByYear":2193,"totalCitation":969,"totalCitationByYear":2194,"totalCitationPerPublication":983,"totalCitationPerPublicationByYear":2195,"hindexLast5Year":196,"hindex":196},{"2012":235,"2013":961,"2014":962,"2015":48,"2016":963,"2017":579,"2018":964,"2019":965,"2020":348,"2021":966,"2022":967,"2023":123},{"1983":123,"1984":40,"1985":40,"1988":123,"1989":123,"1991":40,"1993":45,"1994":123,"1995":42,"2001":45,"2002":123,"2003":46,"2004":42,"2005":48,"2006":40,"2007":42,"2008":46,"2009":123,"2010":48,"2011":357,"2012":46,"2013":42,"2014":46,"2015":45,"2016":123,"2017":42,"2018":123,"2019":123,"2020":40,"2021":40},{"1983":684,"1984":333,"1985":971,"1988":972,"1989":973,"1991":156,"1993":974,"1994":282,"1995":570,"2001":975,"2002":976,"2003":977,"2004":978,"2005":979,"2006":573,"2007":137,"2008":980,"2009":352,"2010":157,"2011":605,"2012":981,"2013":215,"2014":454,"2015":200,"2016":982,"2017":353,"2018":46,"2019":196,"2020":332,"2021":126},{"1983":985,"1984":333,"1985":971,"1988":617,"1989":986,"1991":156,"1993":982,"1994":987,"1995":157,"2001":988,"2002":989,"2003":829,"2004":990,"2005":991,"2006":573,"2007":992,"2008":993,"2009":994,"2010":995,"2011":996,"2012":997,"2013":160,"2014":998,"2015":999,"2016":1000,"2017":1001,"2018":189,"2019":135,"2020":332,"2021":126},{"issue":2197,"pages":2199,"volume":2201},{"VOID":2198},"1",{"VOID":2200},"21-32",{"VOID":2202},"11",515,{"total":2203,"publishYear":2205,"statisticByYear":2206},1988,{"2012":51,"2013":140,"2014":130,"2015":130,"2016":205,"2017":205,"2018":47,"2019":146,"2020":127,"2021":130,"2022":205,"2023":145,"2024":46},"1988-01-01",[953,940],[2210,2213,2216,2219,2222,2225,2228,2231,2234,2237,2240,2243,2246,2249,2252,2255,2258,2261,2264,2267,2270,2273,2276,2279,2282,2285,2288,2291,2294,2297,2300,2303,2307,2310,2313,2316,2319,2322],{"id":28,"text":2211,"url":28,"identifiers":2212},"Bird TD, 1982, Linkage of Charcot‐Marie‐Tooth neuropathy to the Duffy locus on chromosome 1, Am J. Hum Genet, 34, 388",{},{"id":28,"text":2214,"url":28,"identifiers":2215},"10.1111\u002Fj.1748-1716.1954.tb01167.x",{"doi":2214},{"id":28,"text":2217,"url":28,"identifiers":2218},"Culp WJ, 1982, Abnormal Nerves and Muscles as Impulse Generators, 750",{},{"id":28,"text":2220,"url":28,"identifiers":2221},"Daube JR, 1987, Diabetic Neuropathy",{},{"id":28,"text":2223,"url":28,"identifiers":2224},"10.1136\u002Fjnnp.12.4.259",{"doi":2223},{"id":28,"text":2226,"url":28,"identifiers":2227},"10.1212\u002FWNL.11.10.876",{"doi":2226},{"id":28,"text":2229,"url":28,"identifiers":2230},"Dyck PJ, 1975, Peripheral Neuropathy, 296",{},{"id":28,"text":2232,"url":28,"identifiers":2233},"Dyck PJ, 1982, Cecil Textbook of Medicine, 2153",{},{"id":28,"text":2235,"url":28,"identifiers":2236},"Dyck PJ, 1984, Peripheral Neuropathy, 1557",{},{"id":28,"text":2238,"url":28,"identifiers":2239},"10.2337\u002Fdiacare.10.4.432",{"doi":2238},{"id":28,"text":2241,"url":28,"identifiers":2242},"10.1056\u002FNEJM198602203140801",{"doi":2241},{"id":28,"text":2244,"url":28,"identifiers":2245},"10.1093\u002Fbrain\u002F108.4.861",{"doi":2244},{"id":28,"text":2247,"url":28,"identifiers":2248},"Dyck PJ, 1984, Peripheral Neuropathy, 760",{},{"id":28,"text":2250,"url":28,"identifiers":2251},"Dyck PJ, Diabetic Neuropathy",{},{"id":28,"text":2253,"url":28,"identifiers":2254},"DyckPJ KarnesJ O'BrienPC SwansonCJ: Neuropathy symptom profile in health motor neuron disease diabetic neuropathy and amyloidosis.Neurology (NY) in press.",{},{"id":28,"text":2256,"url":28,"identifiers":2257},"Dyck PJ, 1984, Peripheral Neuropathy, 1103",{},{"id":28,"text":2259,"url":28,"identifiers":2260},"Dyck PJ, 1982, Clinical Neurology, 1",{},{"id":28,"text":2262,"url":28,"identifiers":2263},"Dyck PJ, 1983, Linkage evidence for genetic heterogeneity among kinships with hereditary motor and sensory neuropathy type I, Mayo Clin Proc, 58, 430",{},{"id":28,"text":2265,"url":28,"identifiers":2266},"10.1002\u002Fana.410080608",{"doi":2265},{"id":28,"text":2268,"url":28,"identifiers":2269},"10.1002\u002Fana.410040605",{"doi":2268},{"id":28,"text":2271,"url":28,"identifiers":2272},"Gilliatt RW, 1965, Biochemical Aspects of Neurological Disorders, 117",{},{"id":28,"text":2274,"url":28,"identifiers":2275},"10.1093\u002Fbrain\u002F87.2.215",{"doi":2274},{"id":28,"text":2277,"url":28,"identifiers":2278},"10.1001\u002Farchneurpsyc.1948.02310040011002",{"doi":2277},{"id":28,"text":2280,"url":28,"identifiers":2281},"10.1016\u002FS0140-6736(78)92644-2",{"doi":2280},{"id":28,"text":2283,"url":28,"identifiers":2284},"Kimura J, 1984, Peripheral Neuropathy, 919",{},{"id":28,"text":2286,"url":28,"identifiers":2287},"Lambert EH, 1956, Clinical Examination in Neurology, 287",{},{"id":28,"text":2289,"url":28,"identifiers":2290},"10.1136\u002Fjnnp.33.4.442",{"doi":2289},{"id":28,"text":2292,"url":28,"identifiers":2293},"10.1001\u002Farchneur.1962.04210040117015",{"doi":2292},{"id":28,"text":2295,"url":28,"identifiers":2296},"Low PA, Diabetic Neuropathy",{},{"id":28,"text":2298,"url":28,"identifiers":2299},"10.1016\u002F0304-3959(75)90044-5",{"doi":2298},{"id":28,"text":2301,"url":28,"identifiers":2302},"10.1097\u002F00132985-196104000-00001",{"doi":2301},{"id":28,"text":2304,"url":28,"identifiers":2305},"Palumbo PJ, 1976, Diabetes mellitus: incidence, prevalence, survivorship, and causes of death in Rochester, Minnesota 1945–1970, Diabetes, 25, 566, 10.2337\u002Fdiab.25.7.566",{"doi":2306},"10.2337\u002Fdiab.25.7.566",{"id":28,"text":2308,"url":28,"identifiers":2309},"10.2337\u002Fdiab.14.1.1",{"doi":2308},{"id":28,"text":2311,"url":28,"identifiers":2312},"Rasminsky M, 1984, Peripheral Neuropathy, 911",{},{"id":28,"text":2314,"url":28,"identifiers":2315},"10.2337\u002Fdiab.10.1.46",{"doi":2314},{"id":28,"text":2317,"url":28,"identifiers":2318},"Spencer PS, 1980, Experimental and Clinical Neurotoxicology, 929",{},{"id":28,"text":2320,"url":28,"identifiers":2321},"Thomas PK, 1984, Peripheral Neuropathy, 1773",{},{"id":28,"text":2323,"url":28,"identifiers":2324},"Windebank AJ, 1987, Diabetic Neuropathy",{},{"id":2326,"createTime":2327,"updateTime":2328,"relativeEntities":2329,"slug":2330,"properties":2331,"entityType":1029,"verifyStatus":26,"verifyTime":2346,"verifyNote":1030,"languages":2347,"translateLanguages":2348,"viewCount":32,"primaryUrl":2349,"fullTextUrl":28,"authors":2350,"publicationType":1092,"publisherRelationship":2378,"citationCount":855,"citationInfo":2440,"publishDate":2443,"publishYear":2441,"citationAnalyzeStatus":884,"lastCitationAnalyze":28,"indexDatabases":2444,"openAccess":28,"references":2445,"isForceReanalyzing":1268},"e5dacd9d-cb2b-4c5c-a65e-48fcdb1bec9d","2024-08-30T13:24:51.469+00:00","2025-02-12T17:00:18.228+00:00",[],"The-dystrophin-glycoprotein-complex-cellular-signaling-and-the-regulation-of-cell-survival-in-the-muscular-dystrophies",{"mag":2332,"keywords":2334,"openalex":2335,"abstract":2337,"title":2340,"pm":2343,"doi":2345},{"VOID":2333},"1968210959",{"VI":1279},{"VOID":2336},"W1968210959",{"EN":2338,"VI":2339},"\u003Cjats:title>Abstract\u003C\u002Fjats:title>\u003Cjats:p>Mutations of different components of the dystrophin–glycoprotein complex (DGC) cause muscular dystrophies that vary in terms of severity, age of onset, and selective involvement of muscle groups. Although the primary pathogenetic processes in the muscular dystrophies have clearly been identified as apoptotic and necrotic muscle cell death, the pathogenetic mechanisms that lead to cell death remain to be determined. Studies of components of the DGC in muscle and in nonmuscle tissues have revealed that the DGC is undoubtedly a multifunctional complex and a highly dynamic structure, in contrast to the unidimensional concept of the DGC as a mechanical component in the cell. Analysis of the DGC reveals compelling analogies to two other membrane‐associated protein complexes, namely integrins and caveolins. Each of these complexes mediates signal transduction cascades in the cell, and disruption of each complex causes muscular dystrophies. The signal transduction cascades associated with the DGC, like those associated with integrins and caveolins, play important roles in cell survival signaling, cellular defense mechanisms, and regulation of the balance between cell survival and cell death. This review focuses on the functional components of the DGC, highlighting the evidence of their participation in cellular signaling processes important for cell survival. Elucidating the link between these functional components and the pathogenetic processes leading to cell death is the foremost challenge to understanding the mechanisms of disease expression in the muscular dystrophies due to defects in the DGC. © 2001 John Wiley & Sons, Inc. Muscle Nerve 24: 1575–1594, 2001\u003C\u002Fjats:p>","\u003Cjats:title>Tóm tắt\u003C\u002Fjats:title>\u003Cjats:p>Các đột biến của các thành phần khác nhau trong phức hợp dystrophin–glycoprotein (DGC) gây ra bệnh loạn dưỡng cơ với mức độ nghiêm trọng, tuổi khởi phát và sự tham gia chọn lọc của các nhóm cơ khác nhau. Mặc dù các quá trình bệnh sinh chính trong bệnh loạn dưỡng cơ đã được xác định rõ ràng là sự chết tế bào cơ do apoptotic và hoại tử, nhưng các cơ chế bệnh sinh dẫn đến sự chết tế bào vẫn chưa được xác định. Các nghiên cứu về các thành phần của DGC trong cơ và trong các mô không phải cơ đã chỉ ra rằng DGC chắc chắn là một phức hợp đa chức năng và là một cấu trúc rất linh hoạt, trái ngược với khái niệm một chiều về DGC như một thành phần cơ học trong tế bào. Phân tích DGC tiết lộ những tương đồng đáng kể với hai phức hợp protein liên kết màng khác, đó là integrin và caveolin. Mỗi phức hợp này trung gian các thác tín hiệu truyền thông trong tế bào, và sự gián đoạn của mỗi phức hợp gây ra bệnh loạn dưỡng cơ. Các thác tín hiệu truyền thông liên quan đến DGC, giống như những thác tín hiệu liên quan đến integrin và caveolin, đóng vai trò quan trọng trong tín hiệu sống sót của tế bào, các cơ chế phòng vệ tế bào và điều tiết sự cân bằng giữa sống sót và chết tế bào. Bài tổng quan này tập trung vào các thành phần chức năng của DGC, nhấn mạnh bằng chứng về sự tham gia của chúng trong các quá trình tín hiệu tế bào quan trọng cho sự sống sót của tế bào. Làm rõ liên kết giữa các thành phần chức năng này và các quá trình bệnh sinh dẫn đến sự chết tế bào là thử thách lớn nhất trong việc hiểu các cơ chế biểu hiện bệnh trong bệnh loạn dưỡng cơ do khiếm khuyết trong DGC.",{"EN":2341,"VI":2342},"The dystrophin–glycoprotein complex, cellular signaling, and the regulation of cell survival in the muscular dystrophies","Phức hợp dystrophin–glycoprotein, tín hiệu tế bào và sự điều tiết sống sót của tế bào trong các biến chứng cơ bắp",{"VOID":2344},"11745966",{"VOID":2068},"2024-08-30T13:24:51.468+00:00",[31],[30],"https:\u002F\u002Fonlinelibrary.wiley.com\u002Fdoi\u002F10.1002\u002Fmus.1192",[2351],{"id":2352,"sortIndex":32,"researcher":28,"roles":2353,"affiliations":2354,"properties":2371,"displayName":2375,"givenName":28,"familyName":28},"e634cf75-4738-4ec1-97c2-4df75289ca35",[],[2355,2363],{"id":2356,"sortIndex":32,"affiliation":2357,"properties":28},"f38cd688-4390-4572-9ef3-856047686517",{"id":2356,"createTime":28,"updateTime":28,"relativeEntities":2358,"slug":28,"properties":2359,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2362,"statistic":28},[],{"title":2360},{"EN":2361},"Department of Neurology and Neurological Sciences, Stanford University Medical Center, Room A-343, Stanford, California 94305-5235, USA",[],{"id":2364,"sortIndex":40,"affiliation":2365,"properties":28},"f55aeb4d-a661-46e1-9453-7dcff26d7ed7",{"id":2364,"createTime":28,"updateTime":28,"relativeEntities":2366,"slug":28,"properties":2367,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2370,"statistic":28},[],{"title":2368},{"VI":2369},"Geriatric Research, Education and Clinical Center, VA Palo Alto Health Care System, Palo Alto, California, USA",[],{"orcid":2372,"title":2374,"openalex":2376},{"VOID":2373},"https:\u002F\u002Forcid.org\u002F0000-0001-5843-8564",{"EN":2375},"Thomas A. 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CS, 1995, Phosphorylation of the carboxyl terminal region of dystrophin by mitogen‐activated protein (MAP) kinase, Mol Cell Biochem, 152, 63, 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JG, 1995, Apoptosis precedes necrosis of dystrophin‐deficient muscle, J Cell Sci, 108, 2197, 10.1242\u002Fjcs.108.6.2197",{"doi":3036},"10.1242\u002Fjcs.108.6.2197",{"id":28,"text":3038,"url":28,"identifiers":3039},"10.1038\u002F360591a0",{"doi":3038},{"id":28,"text":3041,"url":28,"identifiers":3042},"10.1073\u002Fpnas.91.18.8307",{"doi":3041},{"id":28,"text":3044,"url":28,"identifiers":3045},"10.1006\u002Fmgme.1998.2763",{"doi":3044},{"id":28,"text":3047,"url":28,"identifiers":3048},"Tome FMS, 1998, Neuromuscular disorders: clinical and molecular genetics, 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JT, 1993, Embryonic mesodermal defects in α5 integrin‐deficient mice, Development, 119, 1093, 10.1242\u002Fdev.119.4.1093",{"doi":3103},"10.1242\u002Fdev.119.4.1093",{"id":28,"text":3105,"url":28,"identifiers":3106},"10.1093\u002Fhmg\u002F9.7.1033",{"doi":3105},{"id":28,"text":3108,"url":28,"identifiers":3109},"10.1006\u002Fbbrc.1996.1123",{"doi":3108},{"id":28,"text":3111,"url":28,"identifiers":3112},"10.1074\u002Fjbc.273.3.1583",{"doi":3111},{"id":28,"text":3114,"url":28,"identifiers":3115},"10.1016\u002FS0387-7604(12)80123-9",{"doi":3114},{"id":28,"text":3117,"url":28,"identifiers":3118},"10.1002\u002Fmus.1033",{"doi":3117},{"id":28,"text":3120,"url":28,"identifiers":3121},"10.1096\u002Ffasebj.13.9001.s91",{"doi":3120},{"id":3123,"createTime":3124,"updateTime":3125,"relativeEntities":3126,"slug":3127,"properties":3128,"entityType":1029,"verifyStatus":26,"verifyTime":3124,"verifyNote":1030,"languages":3144,"translateLanguages":3145,"viewCount":32,"primaryUrl":3146,"fullTextUrl":28,"authors":3147,"publicationType":1092,"publisherRelationship":3167,"citationCount":3229,"citationInfo":3230,"publishDate":3233,"publishYear":3231,"citationAnalyzeStatus":884,"lastCitationAnalyze":28,"indexDatabases":3234,"openAccess":28,"references":3235,"isForceReanalyzing":1268},"ae50b84d-6c1e-43e0-a384-7fb0435b6575","2024-08-31T15:56:02.665+00:00","2025-02-12T17:01:15.437+00:00",[],"Small-fiber-neuropathy",{"mag":3129,"keywords":3131,"openalex":3132,"abstract":3134,"title":3137,"pm":3140,"doi":3142},{"VOID":3130},"2003360699",{"VI":1279},{"VOID":3133},"W2003360699",{"EN":3135,"VI":3136},"\u003Cjats:title>Abstract\u003C\u002Fjats:title>\u003Cjats:p>Small‐fiber neuropathy is a common disorder. It is often “idiopathic” and typically presents with painful feet in patients over the age of 60. Autoimmune mechanisms are often suspected, but rarely identified. Known causes of small‐fiber neuropathy include diabetes mellitus, amyloidosis, toxins, and inherited sensory and autonomic neuropathies. Occasionally, small‐fiber neuropathy is diffuse or multifocal. Depending on the type of small‐fiber neuropathy, autonomic dysfunction can be significant or subclinical. Diagnosis is made on the basis of the clinical features, normal nerve conduction studies, and abnormal specialized tests of small‐fiber function. These specialized studies include assessment of epidermal nerve fiber density as well as sudomotor, quantitative sensory, and cardiovagal testing. The sensitivities of these tests range from 59–88%. Each has certain advantages and disadvantages, and the tests may be complementary. Unless an underlying disease is identified, treatment is usually directed toward alleviation of neuropathic pain. © 2002 Wiley Periodicals, Inc. Muscle Nerve 26: 173–188, 2002\u003C\u002Fjats:p>","\u003Cjats:title>Tóm tắt\u003C\u002Fjats:title>\u003Cjats:p>Bệnh thần kinh sợi nhỏ là một rối loạn phổ biến. Nó thường được coi là \"thiếu thông tin\" và thường xuất hiện với các triệu chứng đau chân ở những bệnh nhân trên 60 tuổi. Các cơ chế tự miễn dịch thường bị nghi ngờ nhưng hiếm khi được xác định. Các nguyên nhân đã biết của bệnh thần kinh sợi nhỏ bao gồm tiểu đường, bệnh amyloidosis, độc tố và các bệnh lý thần kinh cảm giác và tự chủ di truyền. Đôi khi, bệnh thần kinh sợi nhỏ có thể lan rộng hoặc đa ổ. Tùy thuộc vào loại bệnh thần kinh sợi nhỏ, rối loạn tự chủ có thể đáng kể hoặc không triệu chứng. Chẩn đoán được thực hiện dựa trên các đặc điểm lâm sàng, kết quả nghiên cứu dẫn truyền thần kinh bình thường và các xét nghiệm chuyên biệt bất thường về chức năng của sợi nhỏ. Các nghiên cứu chuyên biệt này bao gồm đánh giá mật độ sợi thần kinh biểu bì cũng như xét nghiệm sudomotor, cảm giác định lượng và cardiovagal. Độ nhạy của các xét nghiệm này dao động từ 59–88%. Mỗi xét nghiệm có những ưu nhược điểm nhất định, và các xét nghiệm có thể bổ sung cho nhau. Trừ khi có bệnh lý tiềm ẩn được xác định, điều trị thường hướng đến giảm đau thần kinh.",{"EN":3138,"VI":3139},"Small‐fiber neuropathy","Bệnh thần kinh sợi nhỏ",{"VOID":3141},"12210380",{"VOID":3143},"10.1002\u002Fmus.10181",[31],[30],"https:\u002F\u002Fonlinelibrary.wiley.com\u002Fdoi\u002F10.1002\u002Fmus.10181",[3148],{"id":3149,"sortIndex":32,"researcher":28,"roles":3150,"affiliations":3151,"properties":3160,"displayName":3164,"givenName":28,"familyName":28},"3fad1032-ab24-4497-a6a3-50866cda9d7a",[],[3152],{"id":3153,"sortIndex":32,"affiliation":3154,"properties":28},"a5488fc2-9344-421a-b302-efc07e52674e",{"id":3153,"createTime":28,"updateTime":28,"relativeEntities":3155,"slug":28,"properties":3156,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":3159,"statistic":28},[],{"title":3157},{"EN":3158},"Department of Neurology, University of Pittsburgh School of Medicine, UPMC Presbyterian, 200 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reviewed 180 electroneuromyographic (EMG) studies from patients with acute inflammatory demyelinating polyradiculoneuropathy. EMG criteria suggestive of demyelination were met during the first 5 weeks in 87% of patients; an additional 10% had indeterminate electrodiagnostic evaluations, and 3% demonstrated axonal degeneration only. Motor nerve conduction abnormalities initially predominated, with the nadir of abnormality occurring at week 3. Sensory nerve conduction abnormalities peaked during week 4 and were atypical for polyneuropathy, with 52% of patients having normal sural but abnormal median sensory studies, perhaps reflecting distal nerve involvement. Delayed sensory abnormalities may reflect, in part, secondary involvement related to increased intraneural edema accentuated by compression at sites of anatomic vulnerability. Fibrillation potentials and increased polyphasia appeared between weeks 2 and 5 in proximal and distal muscles simultaneously, which is consistent with either random axonal degeneration at any point along the axon or distal involvement. Resolution of conduction abnormalities began between weeks 6 and 10, with increased mean motor‐evoked amplitude best reflecting functional clinical recovery.\u003C\u002Fjats:p>","\u003Cjats:title>Tóm tắt\u003C\u002Fjats:title>\u003Cjats:p>Chúng tôi đã xem xét 180 nghiên cứu điện thần kinh cơ học (EMG) từ các bệnh nhân bị hội chứng đa rễ thần kinh viêm cấp tính do mất myelin. Các tiêu chí EMG có tính chất gợi ý về sự mất myelin đã được thoả mãn trong 5 tuần đầu ở 87% bệnh nhân; thêm 10% có đánh giá điện chẩn đoán không xác định, và 3% chỉ thể hiện hiện tượng thoái hoá sợi thần kinh. Các bất thường dẫn truyền thần kinh vận động xuất hiện chiếm ưu thế ban đầu, với điểm thấp nhất của sự bất thường xảy ra vào tuần thứ 3. Các bất thường dẫn truyền thần kinh cảm giác đạt đỉnh vào tuần thứ 4 và không điển hình cho bệnh đa dây thần kinh, với 52% bệnh nhân có kết quả khám dọc thần kinh chầy bình thường nhưng nghiên cứu thần kinh cảm giác trung bình bất thường, có thể phản ánh sự tổn thương thần kinh xa. Các bất thường cảm giác chậm có thể phản ánh, một phần, sự tham gia thứ phát liên quan đến việc tăng phù nề trong thần kinh được làm nổi bật bởi sự chèn ép tại các vị trí có thể tổn thương giải phẫu. Các điện thế co giật và sự gia tăng đa hình xuất hiện giữa tuần thứ 2 và 5 ở cả cơ gần và xa cùng một lúc, điều này phù hợp với sự thoái hóa sợi thần kinh ngẫu nhiên ở bất kỳ điểm nào along sợi thần kinh hoặc sự tham gia ở xa. Việc phục hồi các bất thường dẫn truyền bắt đầu từ giữa tuần thứ 6 đến thứ 10, với biên độ vận động kích thích trung bình gia tăng phản ánh tốt nhất sự phục hồi lâm sàng về chức năng.\u003C\u002Fjats:p>",{"EN":3571,"VI":3572},"Sequential electrodiagnostic abnormalities in acute inflammatory demyelinating polyradiculoneuropathy","Các bất thường điện chẩn đoán theo chuỗi trong bệnh đa rễ thần kinh viêm cấp tính do mất myelin",{"VOID":3574},"16758578",{"VOID":3576},"10.1002\u002Fmus.880080609",[31],[30],"https:\u002F\u002Fonlinelibrary.wiley.com\u002Fdoi\u002F10.1002\u002Fmus.880080609",[3581,3598,3607],{"id":3582,"sortIndex":32,"researcher":28,"roles":3583,"affiliations":3584,"properties":3593,"displayName":3595,"givenName":28,"familyName":28},"d5a8c0b2-d599-4949-b7e3-3367e8262bbc",[],[3585],{"id":3586,"sortIndex":32,"affiliation":3587,"properties":28},"c7a2c8e3-02f9-43db-96bc-f3965d72faa0",{"id":3586,"createTime":28,"updateTime":28,"relativeEntities":3588,"slug":28,"properties":3589,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":3592,"statistic":28},[],{"title":3590},{"EN":3591},"Neuromuscular Section, Department of Neurology, University of Michigan Medical Center, Ann Arbor, MI 48109-0010, USA.",[],{"title":3594,"openalex":3596},{"EN":3595},"James W. Albers",{"VOID":3597},"A5003974462",{"id":3599,"sortIndex":40,"researcher":28,"roles":3600,"affiliations":3601,"properties":3602,"displayName":3604,"givenName":28,"familyName":28},"4530f84c-8566-4c5b-903c-b3fa281c0d06",[],[],{"title":3603,"openalex":3605},{"EN":3604},"Peter D. Donofrio",{"VOID":3606},"A5031844525",{"id":3608,"sortIndex":123,"researcher":28,"roles":3609,"affiliations":3610,"properties":3611,"displayName":3613,"givenName":28,"familyName":28},"f5f86a1e-eb49-4fa3-b885-bcd648a98c19",[],[],{"title":3612,"openalex":3614},{"EN":3613},"T K McGonagle",{"VOID":3615},"A5002345294",{"url":28,"publisher":3617,"properties":3672},{"id":868,"createTime":869,"updateTime":870,"relativeEntities":3618,"slug":872,"properties":3619,"entityType":25,"verifyStatus":884,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":32,"subjectFields":3624,"manageAffiliations":3641,"indexDatabases":3652,"url":958,"thumbnailPath":28,"statistic":3667,"gsStatistic":28,"type":28,"analyzePriority":28},[],{"country":3620,"eissn":3621,"issn":3622,"title":3623},{"VOID":875},{"VOID":877},{"VOID":879},{"EN":881},[3625,3629,3633,3637],{"id":887,"createTime":28,"updateTime":28,"relativeEntities":3626,"label":3627,"description":3628,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":890},{},{"id":893,"createTime":28,"updateTime":28,"relativeEntities":3630,"label":3631,"description":3632,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":896},{},{"id":899,"createTime":28,"updateTime":28,"relativeEntities":3634,"label":3635,"description":3636,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":902},{},{"id":905,"createTime":28,"updateTime":28,"relativeEntities":3638,"label":3639,"description":3640,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":908},{},[3642,3647],{"id":912,"createTime":28,"updateTime":28,"relativeEntities":3643,"slug":28,"properties":3644,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":3646,"statistic":28},[],{"title":3645},{"EN":916},[],{"id":919,"createTime":28,"updateTime":28,"relativeEntities":3648,"slug":28,"properties":3649,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":3651,"statistic":28},[],{"title":3650},{"EN":923},[],[3653,3660],{"id":927,"indexDatabase":3654,"url":933,"indexYears":934,"academicFieldIds":3659,"indexDatabaseRanking":940},{"id":775,"createTime":28,"updateTime":28,"relativeEntities":3655,"label":3656,"description":3657,"key":781,"publicationTags":3658,"standard":28},[],{"EN":778,"VI":778},{"EN":778,"VI":780},[783],[936,937,938,939],{"id":942,"indexDatabase":3661,"url":954,"indexYears":28,"academicFieldIds":3666,"indexDatabaseRanking":28},{"id":944,"createTime":28,"updateTime":28,"relativeEntities":3662,"label":3663,"description":3664,"key":951,"publicationTags":3665,"standard":28},[],{"EN":947,"VI":947},{"EN":949,"VI":950},[953,813],[956,957],{"impactFactor":32,"impactFactorByYear":3668,"i10Index":611,"i10IndexLast5Year":45,"totalPublication":333,"totalPublicationByYear":3669,"totalCitation":969,"totalCitationByYear":3670,"totalCitationPerPublication":983,"totalCitationPerPublicationByYear":3671,"hindexLast5Year":196,"hindex":196},{"2012":235,"2013":961,"2014":962,"2015":48,"2016":963,"2017":579,"2018":964,"2019":965,"2020":348,"2021":966,"2022":967,"2023":123},{"1983":123,"1984":40,"1985":40,"1988":123,"1989":123,"1991":40,"1993":45,"1994":123,"1995":42,"2001":45,"2002":123,"2003":46,"2004":42,"2005":48,"2006":40,"2007":42,"2008":46,"2009":123,"2010":48,"2011":357,"2012":46,"2013":42,"2014":46,"2015":45,"2016":123,"2017":42,"2018":123,"2019":123,"2020":40,"2021":40},{"1983":684,"1984":333,"1985":971,"1988":972,"1989":973,"1991":156,"1993":974,"1994":282,"1995":570,"2001":975,"2002":976,"2003":977,"2004":978,"2005":979,"2006":573,"2007":137,"2008":980,"2009":352,"2010":157,"2011":605,"2012":981,"2013":215,"2014":454,"2015":200,"2016":982,"2017":353,"2018":46,"2019":196,"2020":332,"2021":126},{"1983":985,"1984":333,"1985":971,"1988":617,"1989":986,"1991":156,"1993":982,"1994":987,"1995":157,"2001":988,"2002":989,"2003":829,"2004":990,"2005":991,"2006":573,"2007":992,"2008":993,"2009":994,"2010":995,"2011":996,"2012":997,"2013":160,"2014":998,"2015":999,"2016":1000,"2017":1001,"2018":189,"2019":135,"2020":332,"2021":126},{"issue":3673,"pages":3674,"volume":3676},{"VOID":1658},{"VOID":3675},"528-539",{"VOID":3677},"8",{"total":971,"publishYear":3679,"statisticByYear":3680},1985,{"2012":146,"2013":127,"2014":145,"2015":205,"2016":49,"2017":357,"2018":47,"2019":145,"2020":42,"2021":357,"2022":126,"2023":48,"2024":357},"1985-07-01",[953,940],[3684,3687,3690,3693,3696,3699,3702,3705,3708,3711,3714,3717,3720,3723,3726,3729,3732,3735,3738,3741,3744,3747,3750,3753,3756,3759,3762,3765,3768,3771,3774],{"id":28,"text":3685,"url":28,"identifiers":3686},"10.1002\u002Fmus.880040606",{"doi":3685},{"id":28,"text":3688,"url":28,"identifiers":3689},"10.1002\u002Fmus.880010405",{"doi":3688},{"id":28,"text":3691,"url":28,"identifiers":3692},"10.1111\u002Fj.1600-0404.1982.tb06851.x",{"doi":3691},{"id":28,"text":3694,"url":28,"identifiers":3695},"Arnason BGW, 1975, Peripheral Neuropathy, 1110",{},{"id":28,"text":3697,"url":28,"identifiers":3698},"10.1002\u002Fana.410090703",{"doi":3697},{"id":28,"text":3700,"url":28,"identifiers":3701},"Asbury AK, 1978, Criteria for diagnosis of Guillain‐Barre syndrome, Ann Neurol, 3, 565",{},{"id":28,"text":3703,"url":28,"identifiers":3704},"Ballantyne JP, 1982, A quantitative assessment of reinnervation in the polyneuropathies, Muscle Nerve, 5, S127",{},{"id":28,"text":3706,"url":28,"identifiers":3707},"10.1093\u002Fbrain\u002F107.1.219",{"doi":3706},{"id":28,"text":3709,"url":28,"identifiers":3710},"BrownWFFeasbyTE: Sensory evoked potentials in Guillain‐Barre polyneuropathy.J Neurol Neurosurg Psychiatry in press.",{},{"id":28,"text":3712,"url":28,"identifiers":3713},"10.1212\u002FWNL.29.5.662",{"doi":3712},{"id":28,"text":3715,"url":28,"identifiers":3716},"10.1001\u002Farchneur.1974.00490360014004",{"doi":3715},{"id":28,"text":3718,"url":28,"identifiers":3719},"10.1002\u002Fmus.880050206",{"doi":3718},{"id":28,"text":3721,"url":28,"identifiers":3722},"Gilliatt R, 1982, Electrophysiology of peripheral neuropathies—an overview, Muscle Nerve, 5, S108",{},{"id":28,"text":3724,"url":28,"identifiers":3725},"Kennedy RH, 1978, Guillain‐Barre syndrome. A 42‐year epidemiologic and clinical study, Mayo Clin Proc, 53, 93",{},{"id":28,"text":3727,"url":28,"identifiers":3728},"10.1002\u002Fmus.880060706",{"doi":3727},{"id":28,"text":3730,"url":28,"identifiers":3731},"Lambert EH, 1963, Nerve conduction in the Guillain‐Barre syndrome, Bull Am Assoc Electromyogr Electrodiagn, 10, 13",{},{"id":28,"text":3733,"url":28,"identifiers":3734},"Lewis RA, 1980, Electrodiagnostic distinctions between chronic acquired and familial demyelinating neuropathies, Neurology, 30, 371",{},{"id":28,"text":3736,"url":28,"identifiers":3737},"10.1136\u002Fjnnp.40.2.156",{"doi":3736},{"id":28,"text":3739,"url":28,"identifiers":3740},"McDonald WI, 1980, The Physiology of Peripheral Nerve Disease, 265",{},{"id":28,"text":3742,"url":28,"identifiers":3743},"10.1002\u002Fana.410090705",{"doi":3742},{"id":28,"text":3745,"url":28,"identifiers":3746},"10.1136\u002Fjnnp.34.5.607",{"doi":3745},{"id":28,"text":3748,"url":28,"identifiers":3749},"Murray NMF, 1980, The sural sensory action potential in Guillain‐Barre syndrome, Muscle Nerve, 3, 444",{},{"id":28,"text":3751,"url":28,"identifiers":3752},"10.1002\u002Fana.410090713",{"doi":3751},{"id":28,"text":3754,"url":28,"identifiers":3755},"10.1212\u002FWNL.18.12.1143",{"doi":3754},{"id":28,"text":3757,"url":28,"identifiers":3758},"Prineas JW, 1967, The Landry‐Guillain‐Barre syndrome: a survey and a clinical report of 127 cases, Acta Neurol Scand, 43, 427",{},{"id":28,"text":3760,"url":28,"identifiers":3761},"Raman PT, 1962, Prognostic significance of electrodiagnostic studies in the Guillain‐Barre syndrome, J Neurol Neurosurg Psychiatry, 25, 321",{},{"id":28,"text":3763,"url":28,"identifiers":3764},"Roberge FA, 1978, Abnormal Neuronal Discharges, 389",{},{"id":28,"text":3766,"url":28,"identifiers":3767},"10.1016\u002F0022-510X(78)90234-4",{"doi":3766},{"id":28,"text":3769,"url":28,"identifiers":3770},"10.1002\u002Fana.410090706",{"doi":3769},{"id":28,"text":3772,"url":28,"identifiers":3773},"10.1136\u002Fjnnp.46.2.168",{"doi":3772},{"id":28,"text":3775,"url":28,"identifiers":3776},"10.1212\u002FWNL.7.2.124",{"doi":3775},{"id":3778,"createTime":3779,"updateTime":3780,"relativeEntities":3781,"slug":3782,"properties":3783,"entityType":1029,"verifyStatus":26,"verifyTime":3779,"verifyNote":1030,"languages":3799,"translateLanguages":3800,"viewCount":32,"primaryUrl":3801,"fullTextUrl":28,"authors":3802,"publicationType":1092,"publisherRelationship":3845,"citationCount":3908,"citationInfo":3909,"publishDate":3912,"publishYear":3910,"citationAnalyzeStatus":884,"lastCitationAnalyze":28,"indexDatabases":3913,"openAccess":28,"references":3914,"isForceReanalyzing":1268},"d6530fce-96eb-45f1-ad58-7d18dfceda75","2024-09-11T10:50:39.717+00:00","2025-02-12T17:03:12.358+00:00",[],"The-estimated-numbers-and-relative-sizes-of-thenar-motor-units-as-selected-by-multiple-point-stimulation-in-young-and-older-adults",{"mag":3784,"keywords":3786,"openalex":3787,"abstract":3789,"title":3792,"pm":3795,"doi":3797},{"VOID":3785},"2129239771",{"VI":1279},{"VOID":3788},"W2129239771",{"EN":3790,"VI":3791},"\u003Cjats:title>Abstract\u003C\u002Fjats:title>\u003Cjats:p>Multiple point stimulation (MPS) is described as a method of estimating the numbers of motor units in the median innervated thenar muscles of young and older control subjects. Stimulation at multiple sites along the course of the median nerve was employed to collect a sample of the lowest threshold, all‐or‐nothing surface‐recorded motor unit action potentials (S‐MUAPs). The average, negative peak area, and peak‐to‐peak amplitude of the sample of S‐MUAPs was determined and divided into the corresponding value for the maximal compound muscle action potential to derive the motor unit estimate (MUE). In 37 trials from 17 younger subjects (20—40 years), the mean MUE was 288 ± 95 SD based on negative peak area and, in 33 trials from 20 older subjects, mean values were 139 ± 68. In 23 young and older subjects, MPS was performed on at least two occasions and the MUEs were found to be highly correlated (\u003Cjats:italic>r\u003C\u002Fjats:italic> = 0.88). © 1993 John Wiley & Sons, Inc.\u003C\u002Fjats:p>","\u003Cjats:title>Tóm tắt\u003C\u002Fjats:title>\u003Cjats:p>Kích thích nhiều điểm (MPS) được mô tả như một phương pháp ước lượng số lượng các đơn vị vận động trong các cơ nắm tay được chi phối bởi dây thần kinh giữa ở cả người trẻ và người lớn tuổi. Kích thích tại nhiều điểm dọc theo đường đi của dây thần kinh giữa đã được sử dụng để thu thập mẫu các điện thế hành động của đơn vị vận động được ghi nhận trên bề mặt với ngưỡng kích thích thấp nhất (S‐MUAPs) theo nguyên tắc tất cả hoặc không có. Diện tích đỉnh âm trung bình, diện tích đỉnh và biên độ đỉnh-đỉnh của mẫu S‐MUAPs đã được xác định và chia cho giá trị tương ứng của điện thế hành động cơ hợp nhất tối đa để tính toán ước tính đơn vị vận động (MUE). Trong 37 thử nghiệm từ 17 đối tượng trẻ (20—40 tuổi), giá trị trung bình MUE là 288 ± 95 SD dựa trên diện tích đỉnh âm và, trong 33 thử nghiệm từ 20 đối tượng lớn tuổi, giá trị trung bình là 139 ± 68. Trong 23 đối tượng trẻ và lớn tuổi, MPS đã được thực hiện ít nhất hai lần và các MUE được tìm thấy có sự tương quan cao (\u003Cjats:italic>r\u003C\u002Fjats:italic> = 0.88). © 1993 John Wiley & Sons, Inc.\u003C\u002Fjats:p>",{"EN":3793,"VI":3794},"The estimated numbers and relative sizes of thenar motor units as selected by multiple point stimulation in young and older adults","Số lượng và kích thước tương đối của các đơn vị vận động của cơ nắm tay được ước lượng bằng phương pháp kích thích nhiều điểm ở người trẻ và người lớn tuổi",{"VOID":3796},"8455648",{"VOID":3798},"10.1002\u002Fmus.880160404",[31],[30],"https:\u002F\u002Fonlinelibrary.wiley.com\u002Fdoi\u002F10.1002\u002Fmus.880160404",[3803,3820],{"id":3804,"sortIndex":32,"researcher":28,"roles":3805,"affiliations":3806,"properties":3815,"displayName":3817,"givenName":28,"familyName":28},"336d4e90-0c41-47ab-9c9d-407634fc68cc",[],[3807],{"id":3808,"sortIndex":32,"affiliation":3809,"properties":28},"f8fa623d-fc96-432e-a8f3-41e759049885",{"id":3808,"createTime":28,"updateTime":28,"relativeEntities":3810,"slug":28,"properties":3811,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":3814,"statistic":28},[],{"title":3812},{"EN":3813},"Faculty of Kinesiology, University Hospital, London, Ontario, Canada.",[],{"title":3816,"openalex":3818},{"EN":3817},"Timothy J. 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Activity",{},{"id":28,"text":3943,"url":28,"identifiers":3944},"10.1159\u002F000140989",{"doi":3943},{"id":28,"text":3946,"url":28,"identifiers":3947},"10.1002\u002Fmus.880141114",{"doi":3946},{"id":28,"text":3949,"url":28,"identifiers":3950},"Henneman E, 1981, Handbook of Physiology, 423",{},{"id":28,"text":2277,"url":28,"identifiers":3952},{"doi":2277},{"id":28,"text":3954,"url":28,"identifiers":3955},"10.1136\u002Fjnnp.39.10.973",{"doi":3954},{"id":28,"text":3957,"url":28,"identifiers":3958},"Komori T, 1991, Characteristics of single ‘F’ motor units at different stimulus intensities, Muscle Nerve, 14, 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The purpose of this study was to develop an evidence‐based guideline for the use of neuromuscular ultrasound in the diagnosis of carpal tunnel syndrome (CTS). Methods: Two questions were asked: (1) What is the accuracy of median nerve cross‐sectional area enlargement as measured with ultrasound for the diagnosis of CTS? (2) What added value, if any, does neuromuscular ultrasound provide over electrodiagnostic studies alone for the diagnosis of CTS? A systematic review was performed, and studies were classified according to American Academy of Neurology criteria for rating articles of diagnostic accuracy (question 1) and for screening articles (question 2). Results: Neuromuscular ultrasound measurement of median nerve cross‐sectional area at the wrist is accurate and may be offered as a diagnostic test for CTS (Level A). Neuromuscular ultrasound probably adds value to electrodiagnostic studies when diagnosing CTS and should be considered in screening for structural abnormalities at the wrist in those with CTS (Level B). Muscle Nerve 46: 287–293, 2012\u003C\u002Fjats:p>","\u003Cjats:title>Tóm tắt\u003C\u002Fjats:title>\u003Cjats:p>Giới thiệu: Mục tiêu của nghiên cứu này là phát triển một hướng dẫn dựa trên bằng chứng cho việc sử dụng siêu âm thần kinh cơ trong chẩn đoán hội chứng ống cổ tay (CTS). Phương pháp: Hai câu hỏi được đặt ra: (1) Độ chính xác của việc đo diện tích mặt cắt ngang của dây thần kinh giữa bằng siêu âm trong chẩn đoán CTS là bao nhiêu? (2) Siêu âm thần kinh cơ có giá trị bổ sung gì, nếu có, so với các nghiên cứu điện chẩn đoán đơn thuần trong chẩn đoán CTS? Một đánh giá hệ thống đã được thực hiện và các nghiên cứu được phân loại theo tiêu chí của Học viện Thần kinh Hoa Kỳ để đánh giá độ chính xác chẩn đoán (câu hỏi 1) và các bài báo sàng lọc (câu hỏi 2). Kết quả: Đo lường diện tích mặt cắt ngang của dây thần kinh giữa tại cổ tay bằng siêu âm là chính xác và có thể được đề xuất như một bài kiểm tra chẩn đoán cho CTS (Cấp A). Siêu âm thần kinh cơ có khả năng bổ sung giá trị cho các nghiên cứu điện chẩn đoán khi chẩn đoán CTS và nên được xem xét trong việc sàng lọc các bất thường cấu trúc tại cổ tay ở những người mắc CTS (Cấp B). Muscle Nerve 46: 287–293, 2012\u003C\u002Fjats:p>",{"EN":4011,"VI":4012},"Evidence‐based guideline: Neuromuscular ultrasound for the diagnosis of carpal tunnel syndrome","Hướng dẫn dựa trên bằng chứng: Siêu âm thần kinh cơ cho chẩn đoán hội chứng ống cổ tay",{"VOID":4014},"22806381",{"VOID":4016},"10.1002\u002Fmus.23389",[31],[30],"https:\u002F\u002Fonlinelibrary.wiley.com\u002Fdoi\u002F10.1002\u002Fmus.23389",[4021,4040,4057,4074,4093,4120,4139,4158,4175],{"id":4022,"sortIndex":32,"researcher":28,"roles":4023,"affiliations":4024,"properties":4033,"displayName":4037,"givenName":28,"familyName":28},"a7b19caa-d950-4147-a5b9-99017fda7c85",[],[4025],{"id":4026,"sortIndex":32,"affiliation":4027,"properties":28},"41b65e05-c27c-478e-8807-d281f7f429e5",{"id":4026,"createTime":28,"updateTime":28,"relativeEntities":4028,"slug":28,"properties":4029,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":4032,"statistic":28},[],{"title":4030},{"VI":4031},"Wake Forest University",[],{"orcid":4034,"title":4036,"openalex":4038},{"VOID":4035},"https:\u002F\u002Forcid.org\u002F0000-0001-8898-1162",{"EN":4037},"Catherine French",{"VOID":4039},"A5074019565",{"id":4041,"sortIndex":40,"researcher":28,"roles":4042,"affiliations":4043,"properties":4050,"displayName":4054,"givenName":28,"familyName":28},"013748c9-16d4-4d7f-b824-bd6105785f6b",[],[4044],{"id":4026,"sortIndex":32,"affiliation":4045,"properties":28},{"id":4026,"createTime":28,"updateTime":28,"relativeEntities":4046,"slug":28,"properties":4047,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":4049,"statistic":28},[],{"title":4048},{"VI":4031},[],{"orcid":4051,"title":4053,"openalex":4055},{"VOID":4052},"https:\u002F\u002Forcid.org\u002F0000-0003-2026-6511",{"EN":4054},"Michael S. 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