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II. Prevalence and risk of diabetic retinopathy when age at diagnosis is less than 30 years, Arch Ophthalmol, 102, 520, 10.1001\u002Farchopht.1984.01040030398010\nRodriguez-Villalobos, 2005, Diabetic retinopathy: twelve-year incidence and progression, Cir Cir, 73, 79\nKlein, 1994, The Wisconsin Epidemiologic Study of diabetic retinopathy. XIV. Ten-year incidence and progression of diabetic retinopathy, Arch Ophthalmol, 112, 1217, 10.1001\u002Farchopht.1994.01090210105023\nGardner, 2002, Diabetic retinopathy: more than meets the eye, Surv Ophthalmol, 47, S253, 10.1016\u002FS0039-6257(02)00387-9\nBhagat, 2009, Diabetic macular edema: pathogenesis and treatment, Surv Ophthalmol, 54, 1, 10.1016\u002Fj.survophthal.2008.10.001\nEhrlich, 2010, Diabetic macular oedema: physical, physiological and molecular factors contribute to this pathological process, Acta Ophthalmol, 88, 279, 10.1111\u002Fj.1755-3768.2008.01501.x\nAiello, 1994, Vascular endothelial growth factor in ocular fluid of patients with diabetic retinopathy and other retinal disorders, N Engl J Med, 331, 1480, 10.1056\u002FNEJM199412013312203\nBresnick, 1983, Diabetic maculopathy. A critical review highlighting diffuse macular edema, Ophthalmology, 90, 1301, 10.1016\u002FS0161-6420(83)34388-8\nOgata, 2001, Pigment epithelium derived factor as a neuroprotective agent against ischemic retinal injury, Curr Eye Res, 22, 245, 10.1076\u002Fceyr.22.4.245.5506\nMori, 2002, Regression of ocular neovascularization in response to increased expression of pigment epithelium-derived factor, Invest Ophthalmol Vis Sci, 43, 2428\nDesmettre, 2003, Transpupillary thermotherapy (TTT) with short duration laser exposures induce heat shock protein (HSP) hyperexpression on choroidoretinal layers, Lasers Surg Med, 33, 102, 10.1002\u002Flsm.10193\nPeyman, 1971, Chorioretinal diffusion of peroxidase before and after photocoagulation, Invest Ophthalmol, 10, 489\nPeyman, 1972, Peroxidase diffusion in the normal and laser-coagulated primate retina, Invest Ophthalmol, 11, 35\nAntonetti, 1999, Molecular mechanisms of vascular permeability in diabetic retinopathy, Semin Ophthalmol, 14, 240, 10.3109\u002F08820539909069543\nWilson, 2003, Argon laser photocoagulation-induced modification of gene expression in the retina, Invest Ophthalmol Vis Sci, 44, 1426, 10.1167\u002Fiovs.02-0622\n1985, Early Treatment Diabetic Retinopathy Study report number 1. Early Treatment Diabetic Retinopathy Study research group, Arch Ophthalmol, 103, 1796, 10.1001\u002Farchopht.1985.01050120030015\nBlankenship, 1991, Evaluation of a single intravitreal injection of dexamethasone phosphate in vitrectomy surgery for diabetic retinopathy complications, Graefes Arch Clin Exp Ophthalmol, 229, 62, 10.1007\u002FBF00172263\nPacella, 2013, Preliminary results of an intravitreal dexamethasone implant (Ozurdex®) in patients with persistent diabetic macular edema, Clin Ophthalmol, 7, 1423, 10.2147\u002FOPTH.S48364\nCiulla, 2014, Treatment of diabetic macular edema with sustained-release glucocorticoids: intravitreal triamcinolone acetonide, dexamethasone implant, and fluocinolone acetonide implant, Expert Opin Pharmacother, 15, 953, 10.1517\u002F14656566.2014.896899\nTasman, 2012, 358\nGoyal, 2011, Meta-analysis and review on the effect of bevacizumab in diabetic macular edema, Graefe's Aarch Clin Exp Ophthalmol, 249, 15, 10.1007\u002Fs00417-010-1452-4\nvan der Reis, 2011, A systematic review of the adverse events of intravitreal anti-vascular endothelial growth factor injections, Retina, 31, 1449, 10.1097\u002FIAE.0b013e3182278ab4\nLee, 2011, Intravitreal bevacizumab alone versus combined with macular photocoagulation in diabetic macular edema, Korean J Ophthalmol, 25, 299, 10.3341\u002Fkjo.2011.25.5.299\nWang, 2011, Intravitreal bevacizumab combined with\u002Fwithout triamcinolone acetonide in single injection for treatment of diabetic macular edema, Chin Med J, 124, 352\nGrisanti, 2007, Bevacizumab: off-label use in ophthalmology, Indian J Ophthalmol, 55, 417, 10.4103\u002F0301-4738.36474\nOzkiris, 2009, Intravitreal bevacizumab (Avastin) for primary treatment of diabetic macular oedema, Eye, 23, 616, 10.1038\u002Feye.2008.40\nArevalo, 2009, Comparison of two doses of primary intravitreal bevacizumab (Avastin) for diffuse diabetic macular edema: results from the Pan-American Collaborative Retina Study Group (PACORES) at 12-month follow-up, Graefe's Arch Clin Exp Ophthalmol, 247, 735, 10.1007\u002Fs00417-008-1034-x\n1991, Early photocoagulation for diabetic retinopathy. ETDRS report number 9. Early Treatment Diabetic Retinopathy Study Research Group, Ophthalmology, 98, 766\nLee, 1991, Modified grid laser photocoagulation for diffuse diabetic macular edema. Long-term visual results, Ophthalmology, 98, 1594, 10.1016\u002FS0161-6420(91)32082-7\nBandello, 2011, Ranibizumab in the treatment of patients with visual impairment due to diabetic macular edema, Clin Ophthalmol, 5, 1303, 10.2147\u002FOPTH.S17423\nChen, 2014, Ranibizumab monotherapy or combined with laser versus laser monotherapy for diabetic macular edema: a meta-analysis of randomized controlled trials, PLOS ONE, 9, e115797, 10.1371\u002Fjournal.pone.0115797\nMartin, 2012, Ranibizumab and bevacizumab for treatment of neovascular age-related macular degeneration: two-year results, Ophthalmology, 119, 1388, 10.1016\u002Fj.ophtha.2012.03.053\nElman, 2012, Intravitreal ranibizumab for diabetic macular edema with prompt versus deferred laser treatment: three-year randomized trial results, Ophthalmology, 119, 2312, 10.1016\u002Fj.ophtha.2012.08.022\nFaghihi, 2008, Intravitreal bevacizumab versus combined bevacizumab-triamcinolone versus macular laser photocoagulation in diabetic macular edema, Eur J Ophthalmol, 18, 941, 10.1177\u002F112067210801800614\nGroup, 2011, Ranibizumab and bevacizumab for neovascular age-related macular degeneration, N Engl J Med, 364, 1897, 10.1056\u002FNEJMoa1102673\nNguyen, 2010, Two-year outcomes of the ranibizumab for edema of the mAcula in diabetes (READ-2) study, Ophthalmology, 117, 2146, 10.1016\u002Fj.ophtha.2010.08.016\nMassin, 2010, Safety and efficacy of ranibizumab in diabetic macular edema (RESOLVE Study): a 12-month, randomized, controlled, double-masked, multicenter phase II study, Diabetes Care, 33, 2399, 10.2337\u002Fdc10-0493\nMitchell, 2011, The RESTORE study: ranibizumab monotherapy or combined with laser versus laser monotherapy for diabetic macular edema, Ophthalmology, 118, 615, 10.1016\u002Fj.ophtha.2011.01.031\nSchmidt-Erfurth, 2014, Three-year outcomes of individualized ranibizumab treatment in patients with diabetic macular edema: the RESTORE extension study, Ophthalmology, 12, 1045, 10.1016\u002Fj.ophtha.2013.11.041\nTreatment for CI-DME in eyes with very good VA study (Protocol V). ClinicalTrials.gov. clinicaltrials.gov\u002Fct2\u002Fshow\u002FNCT01909791. 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Current drug targets 2009, Curr Drug Targets, 10, 1169, 10.2174\u002F138945009789753183\nTruche, 2016, Continuous renal replacement therapy versus intermittent hemodialysis in intensive care patients: impact on mortality and renal recovery, Intensive Care Med, 42, 1408, 10.1007\u002Fs00134-016-4404-6\nZeng, 2014, Incidence, outcomes, and comparisons across definitions of AKI in hospitalized individuals, Clin J Am Soc Nephrol, 9, 12, 10.2215\u002FCJN.02730313\nGammelager, 2013, Five-year risk of end-stage renal disease among intensive care patients surviving dialysis-requiring acute kidney injury: a nationwide cohort study, Crit Care, 17, R145, 10.1186\u002Fcc12824\nCruz, 2009, Clinical review: RIFLE and AKIN – time for reappraisal, Crit Care, 13, 211, 10.1186\u002Fcc7759\nPaul, 2008, Intensity of renal support in critically ill patients with acute kidney injury, N Engl J Med, 359, 7, 10.1056\u002FNEJMoa0802639\n2012, KDIGO clinical practice guideline for acute kidney injury, Kidney Int, 2, 1\nGaudry, 2016, Initiation strategies for renal-replacement therapy in the intensive care unit, N Engl J Med, 375, 122, 10.1056\u002FNEJMoa1603017\nMcMahon, 2013, Biomarkers in nephrology: core curriculum 2013, Am J Kidney Dis, 62, 165, 10.1053\u002Fj.ajkd.2012.12.022\nChionh, 2013, Use of peritoneal dialysis in AKI: a systematic review, Clin J Am Soc Nephrol, 8, 1649, 10.2215\u002FCJN.01540213\nPonce, 2012, High-volume peritoneal dialysis in acute kidney injury: indications and limitations, Clin J Am Soc Nephrol, 7, 887, 10.2215\u002FCJN.11131111\nPassadakis, 2007, Peritoneal dialysis in patients with acute renal failure, Adv Perit Dial, 23\nKoch, 2012, Comparable outcome of acute unplanned peritoneal dialysis and haemodialysis, Nephrol Dial Transpl, 27, 375, 10.1093\u002Fndt\u002Fgfr262\nKramer, 1977, Arteriovenous haemofiltration: a new and simple method for treatment of over-hydrated patients resistant to diuretics, Klin Wochenschr, 55, 1121, 10.1007\u002FBF01477940\nRicci, 2016, Renal replacement therapy, F1000Research, 5, 103, 10.12688\u002Ff1000research.6935.1\nManns, 1998, Continuous renal replacement therapies: an update, Am J Kidney Dis, 32, 185, 10.1053\u002Fajkd.1998.v32.pm9708602\nRonco, 2015, Renal replacement therapy in acute kidney injury: controversy and consensus, Crit Care, 19, 146, 10.1186\u002Fs13054-015-0850-8\nTolwani, 2012, Continuous renal-replacement therapy for acute kidney injury, N Engl J Med, 367, 2505, 10.1056\u002FNEJMct1206045\nFleming, 2011, Renal replacement therapy review, Organogenesis, 7, 2, 10.4161\u002Forg.7.1.13997\nProwle, 2010, Continuous renal replacement therapy: recent advances and future research, Nat Rev Nephrol, 6, 521, 10.1038\u002Fnrneph.2010.100\nJohn, 2010, Dose adequacy and assessment, 53\nNetwork* TVARFT, 2008, Intensity of renal support in critically ill patients with acute kidney injury, N Engl J Med, 359, 7, 10.1056\u002FNEJMoa0802639\nZarbock, 2014, Sepsis-induced acute kidney injury revisited, Curr Opin Crit Care, 20, 588, 10.1097\u002FMCC.0000000000000153\nGomez, 2014, A unified theory of sepsis-induced acute kidney injury: inflammation, microcirculatory dysfunction, bioenergetics, and the tubular cell adaptation to injury, Shock, 41, 3, 10.1097\u002FSHK.0000000000000052\nProwle, 2009, Renal blood flow during acute renal failure in man, Blood Purif, 28, 216, 10.1159\u002F000230813\nMurugan, 2010, Acute kidney injury in non-severe pneumonia is associated with an increased immune response and lower survival, Kidney Int, 77, 527, 10.1038\u002Fki.2009.502\nAbraham, 2007, Mechanisms of sepsis-induced organ dysfunction, Crit Care Med, 35, 2408, 10.1097\u002F01.CCM.0000282072.56245.91\nWaikar, 2009, Creatinine kinetics and the definition of acute kidney injury, J Am Soc Nephrol, 20, 672, 10.1681\u002FASN.2008070669\nWang, 2015, Fluid balance and mortality in critically ill patients with acute kidney injury: a multicenter prospective epidemiological study, Crit Care, 19, 371, 10.1186\u002Fs13054-015-1085-4\nLiu, 2011, Acute kidney injury in patients with acute lung injury: impact of fluid accumulation on classification of acute kidney injury and associated outcomes, Crit Care Med, 39, 2665, 10.1097\u002FCCM.0b013e318228234b\nThongprayoon, 2015, The impact of fluid balance on diagnosis, staging and prediction of mortality in critically ill patients with acute kidney injury, J Nephrol, 29, 1\nBagshaw, 2007, Septic acute kidney injury in critically ill patients: clinical characteristics and outcomes, Clin J Am Soc Nephrol, 2, 431, 10.2215\u002FCJN.03681106\nLim, 2015, Initiating acute dialysis at earlier Acute Kidney Injury Network stage in critically ill patients without traditional indications does not improve outcome: a prospective cohort study, Nephrology, 20, 148, 10.1111\u002Fnep.12364\nWierstra, 2016, The impact of “early” versus “late” initiation of renal replacement therapy in critical care patients with acute kidney injury: a 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