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Journal of Medicine and Pharmacy","Tạp chí Y Dược học Cần Thơ",{"EN":575,"VI":576},"\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">04\u002F10\u002F2015 Ministry of Information and Communications allowed Can Tho journal of medicine and pharmacy to operate (102 \u002FGP-BTTTT)\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">07\u002F16\u002F2015 Can Tho journal of medicine and pharmacy is internationally recognized: ISSN 2354-1210\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">In 2016, The journal has been included in the list of medical science journals by The State Council for professorship which is awarded a work score of 0-0.5 points for a published article.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Can Tho Journal of Medicine and Pharmacy welcome original works that haven’t been submitted or published in other medical journals. Posts must contain content related to one of the journal’s categories.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The content published\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The journal is divided into 3 categories:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Scientific research article: are valuable scientific works, which have been researched and accepted.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Overview of medicine, biology and pharmacy: serving the objective of continuing training in the fields of medicine, biology and pharmacy; to systematize classical and modern knowledge.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Update information on new knowledge about medicine, biology, pharmacy in the country and in the world.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Scope\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Publication and introduction of scientific research in the fields:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Medicine (internal medicine, surgery, pediatrics, obstetrics and gynecology, odonto-stomatology, laboratory, oncology, traditional medicine, nursing).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Biology (genetics, biotechnology).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Pharmacology (pharmaceutics, drug quality analysis-control, synthetic pharmaceutical chemistry, biochemistry, pharmacognosy, botany, clinical pharmacy).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- To enhance the quality of undergraduate, postgraduate education, scientifically researching and meet the necessary treatment in hospital.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Introducing the updated domestic and oversea information about science technology to promote scientific research and exchanging technology in local, other universities.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Exchanging pharmaceutical and medical information for social health developing in the Mekong Delta and Vietnam.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The object\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Postgraduate students, student of Can Tho University of Medicine and Pharmacy, scientists from schools, research institutes, hospitals, health centers, pharmaceutical companies of the Mekong Delta; other provinces and regions in Vietnam and other country.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Address\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Headquarters of Can Tho Journal of Medicine and Pharmacy, located Scientific Research and International Cooperation Office: 179 Nguyen Van Cu Street, An Khanh Ward, Ninh Kieu District, Can Tho City, Vietnam.\u003C\u002Fspan>\u003C\u002Fp>","\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Ngày 16\u002F7\u002F2015, Tạp chí Y Dược học Cần Thơ được cấp chỉ số quốc tế: ISSN 2354-1210.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 4\u002F2016, Tạp chí đã được Hội đồng Giáo sư ngành Y đưa vào danh sách các tạp chí khoa học Y học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Năm 2020 Tạp chí Y Dược học Cần Thơ đã được phê duyệt vào danh mục của các Hội đồng Giáo sư ngành Dược học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ ra 12 số\u002Fnăm, 180-200 trang\u002Fsố.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 12\u002F2022 Tạp chí Y Dược học Cần Thơ là thành viên của hệ thống Crossref và từ tháng 01\u002F2023 tạp chí thực hiện bình duyệt online kín 2 chiều nhằm tăng tính minh bạch, tin cậy của các công trình nghiên cứu khoa học và đảm bảo tốt nhất chất lượng khoa học của bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ, mục đích và phạm vi của tạp chí\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ và mục đích hoạt động của tạp chí: xuất bản nhằm mục đích phổ biến kết quả từ các đề tài nghiên cứu khoa học; giao lưu trao đổi khoa học, chia sẻ kinh nghiệm, học tập, đồng thời cập nhật thông tin khoa học mới trong các lĩnh vực y, sinh, dược học trong và ngoài nước.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phạm vi của tạp chí: Tạp chí xuất bản được chia thành 3 chuyên mục: (i) Bài báo nghiên cứu khoa học là kết quả công trình nghiên cứu khoa học có giá trị đã được triển khai nghiên cứu, (ii) Bài tổng quan y, sinh, dược học: phục vụ mục tiêu đào tạo liên tục trong lĩnh vực y, sinh, dược học; nhằm hệ thống hóa những kiến thức kinh điển và hiện đại; (iii) Thông tin cập nhật kiến thức mới về y, sinh, dược học trong nước và trên thế giới.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Chính sách truy cập mở\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ áp dụng chính sách truy cập mở đối với các bài báo đã xuất bản đến với độc giả, nhằm mở rộng cơ hội tiếp cận các kết quả nghiên cứu chất lượng cao và tăng cường trao đổi kiến thức. Tạp chí đăng tải trực tuyến (miễn phí) toàn văn các bài báo được công bố trên website của Tạp chí (https:\u002F\u002Ftapchi.ctump.edu.vn).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đạo đức xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ cam kết tuân thủ đạo đức xuất bản phù hợp với các hướng dẫn và tiêu chuẩn của the Committee on Publication Ethics (COPE), tuân thủ các nguyên tắc của COPE’s Core Practices, Best Practices Guidelines for Journal Editors và Guidelines on Good Publication Practices.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Bản thảo bài báo chỉ được chấp nhận khi được tác giả chịu trách nhiệm chính cam kết các nội dung sau: Các nội dung của bản thảo chưa được đăng tải toàn bộ hoặc một phần ở các tạp chí khác; Tất cả các tác giả đều có đóng góp một cách đáng kể vào quá trình nghiên cứu hoặc chuẩn bị bản thảo và cùng chịu trách nhiệm về các nội dung của bản thảo; Tuân thủ các biện pháp đảm bảo đạo đức nghiên cứu (ví dụ thỏa thuận đồng ý tham gia nghiên cứu).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Cam kết bảo mật\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí cam kết thực hiện và tuân thủ các quy định của luật và các văn bản hướng dẫn liên quan đến bảo mật thông tin cá nhân trên không gian mạng. Các thông tin mà người dùng (tác giả, độc giả, biên tập viên, người phản biện) nhập vào các biểu mẫu trên Hệ thống Quản lý xuất bản trực tuyến của tạp chí chỉ được sử dụng vào các mục đích đã được tuyên bố rõ ràng và sẽ không được cung cấp cho bất kỳ bên thứ ba nào khác, hay dùng vào bất kỳ mục đích nào khác.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phí gửi bài\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng bài: 1.000.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng nhanh: 1.500.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với tác giả là cán bộ viên chức thuộc Trường Đại học Y Dược Cần Thơ thì được hỗ trợ 50% lệ phí gửi đăng bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với sinh viên thực hiện đề tài nghiên cứu khoa học cấp trường được hỗ trợ 100% lệ phí đăng bài ( Tác giả gửi đính kèm “ Quyết định về việc giao tổ chức thực hiện đề tài nghiên cứu khoa học cấp Trường của sinh viên”).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Hình thức nộp lệ phí:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Tiền mặt:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Nộp trực tiếp tại Phòng Tài chính - Kế toán, Trường Đại học Y Dược Cần Thơ, số 179 Nguyễn Văn Cừ, P. An Khánh, Q. Ninh Kiều, thành phố Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Chuyển khoản:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tên Tài khoản: Trường ĐHYD Cần Thơ, Số TK: 0111000115668, tại ngân hàng Vietcombank chi nhánh Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Thời gian: Áp dụng từ ngày 01\u002F02\u002F2023.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">* Phí gửi bài không được hoàn trả khi bài viết bị từ chối hoặc tác giả xin rút bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Quy trình phản biện bài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ thực hiện quy trình phản biện kín hai chiều nghiêm ngặt. Danh tính của những người phản biện không được tiết lộ cho các tác giả và ngược lại. Quy trình thẩm định bài báo đăng gồm các bước sau:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tiếp nhận bản thảo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tác giả liên hệ gửi bản thảo đến Tạp chí qua hệ thống trực tuyến tại website: https:\u002F\u002Ftapchi.ctump.edu.vn. Hướng dẫn về cách đăng ký, gửi bài và chuẩn bị bản thảo được cung cấp trên website của Tạp chí.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sàng lọc sơ bộ\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sau khi Tòa soạn nhận được bài báo của tác giả, Ban Thư ký sẽ tiến hành kiểm tra sơ bộ bài báo (các yêu cầu về nội dung và hình thức). Những bài báo không đúng quy cách hoặc có nội dung không phù hợp hoặc vi phạm bản quyền sẽ bị từ chối (Ban Thư ký thông báo phản hồi đến tác giả trong vòng 1 tuần). Những bài báo đủ điều kiện, được Ban Thư ký tòa soạn chuyển đến Ban Biên tập có cùng chuyên môn với nội dung bài báo để đề xuất người phản biện. Thời gian kể từ khi Ban Biên tập nhận bài báo đến khi đề xuất người phản biện bài báo chậm nhất là 5 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Vòng phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký gửi bài và yêu cầu phản biện đến 02 phản biện độc lập.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Các phản biện gởi nhận xét cho Ban Thư ký. Thời gian từ khi gửi bài cho phản biện đến khi nhận ý kiến của phản biện tối đa là 20 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xử ký kết quả phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Nếu ý kiến đồng ý cho đăng và không cần chỉnh sửa, Ban Thư ký tiếp tục đăng bài theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Nếu ý kiến đồng ý đăng và cần chỉnh sửa, Ban Thư ký sẽ thông tin đến tác giả chỉnh sửa theo yêu cầu của người phản biện. Thời gian chỉnh sửa và gửi lại kéo dài không quá 2 tuần, từ khi tác giả bài báo nhận được thông tin (Quá trình này có thể lặp lại tối đa 2 lần\u002F1 bài báo). Khi có sự thống nhất, đồng ý của người phản biện; bài báo được tiếp tục đăng theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Những bài báo có chất lượng không đạt yêu cầu, cả 2 phản biện không đồng ý cho đăng sẽ bị Tòa soạn từ chối đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký tổng hợp các bản thảo đã được tác giả hoàn thiện sau thẩm định trình Ban Biên tập xem xét, Tổng Biên tập phê duyệt, quyết định bài đăng theo các tiêu chí: sự phù hợp nội dung với tôn chỉ và mục đích, thể loại bài viết (ưu tiên các bài có bài có nghiên cứu chuyên sâu, hàm lượng khoa học cao), đóng góp mới bài báo, bài báo được ưu tiên đăng trong số gần nhất của Tạp chí theo thứ tự: tính thời sự, chất lượng bài báo và thời gian gửi bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Ban Biên tập và Ban Thư ký biên tập bản thảo, chế bản, đọc rà soát lỗi. Thời gian hoàn thành từ 10-15 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Ban Thư ký có trách nhiệm thông báo cho tác giả bài báo (bằng e-mail) về tình hình phê duyệt bài báo, thời gian, số kỳ, tập xuất bản bài báo theo qui định.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">4. Danh sách bài báo theo số Tạp chí được in ấn và phát hành trong năm định kỳ được công bố chính thức trên website: https:\u002F\u002Ftapchi.ctump.edu.vn\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>",{"VOID":578},"wcQ1uqwAAAAJ","2023-05-30T08:17:21.868+00:00",[],[582],{"id":583,"createTime":24,"updateTime":24,"relativeEntities":584,"slug":24,"properties":585,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":595,"parentIds":596,"statistic":24},"6413896b-eca9-442b-a73f-182a58a0ce40",[],{"title":586,"address":589,"country":592,"abbreviation":593},{"EN":587,"VI":588},"Can Tho University of Medicine and Pharmacy","Trường Đại học Y Dược Cần Thơ",{"EN":590,"VI":591},"No 179, Nguyen Van Cu street, An Khanh ward, Ninh Kieu district, Can Tho city, Vietnam","Số 179, đường Nguyễn Văn Cừ, phường An Khánh, quận Ninh Kiều, thành phố Cần Thơ, Việt Nam",{"VOID":112},{"VOID":594},"ctump","http:\u002F\u002Fwww.ctump.edu.vn\u002F",[],[],"https:\u002F\u002Ftapchi.ctump.edu.vn\u002Findex.php\u002Fctump",{"impactFactor":25,"impactFactorByYear":600,"i10Index":25,"i10IndexLast5Year":25,"totalPublication":602,"totalPublicationByYear":603,"totalCitation":608,"totalCitationByYear":609,"totalCitationPerPublication":200,"totalCitationPerPublicationByYear":611,"hindexLast5Year":138,"hindex":138},{"2022":601,"2023":203,"2024":198},0.01,1556,{"2020":140,"2021":604,"2022":605,"2023":606,"2024":607,"2025":214},57,306,801,358,161,{"2021":236,"2022":369,"2023":610},99,{"2021":612,"2022":407,"2023":196},0.23,{"impactFactor":24,"impactFactorByYear":24,"i10Index":86,"i10IndexLast5Year":86,"totalPublication":614,"totalPublicationByYear":615,"totalCitation":614,"totalCitationByYear":616,"totalCitationPerPublication":89,"totalCitationPerPublicationByYear":619,"hindexLast5Year":141,"hindex":141},476,{"0":295,"2019":86,"2021":230,"2022":547,"2023":539,"2024":446,"2025":141,"2026":87},{"2021":135,"2022":86,"2023":251,"2024":617,"2025":448,"2026":618},136,83,{"2021":197,"2022":601,"2023":620,"2024":218,"2025":621,"2026":622},0.62,25.43,13.83,{"id":624,"createTime":625,"updateTime":470,"relativeEntities":626,"slug":627,"properties":628,"entityType":22,"verifyStatus":122,"verifyTime":24,"verifyNote":24,"languages":640,"translateLanguages":24,"viewCount":224,"subjectFields":641,"manageAffiliations":642,"indexDatabases":643,"url":644,"thumbnailPath":645,"statistic":646,"gsStatistic":681,"type":147,"analyzePriority":24},"6984a56a-db70-403b-9cc4-4013e1ceaffa","2023-05-09T06:47:40.346+00:00",[],"T%E1%BA%A1p%20ch%C3%AD%20Nghi%C3%AAn%20c%E1%BB%A9u%20n%C6%B0%E1%BB%9Bc%20ngo%C3%A0i",{"country":629,"issn":630,"title":632,"introduce":635,"gsId":638},{"VOID":112},{"VOID":631},"25252445",{"EN":633,"VI":634},"VNU Journal of Foreign Studies","Tạp chí Nghiên cứu nước ngoài",{"EN":636,"VI":637},"{\"ops\":[{\"insert\":\"\\n\\nThe \\n\"},{\"attributes\":{\"italic\":true},\"insert\":\"VNU Journal of Science\"},{\"insert\":\"\\n was established in 1985 for the publication of national and international research papers in all fields of natural sciences and technology, social sciences and humanities. Since then, the journal has grown in quality, size and scope and now comprises a dozen of serials spanning academic research. 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Report of a fatal laboratory infection complicated by thrombophlebitis, 59, 305",{},{"id":24,"text":1907,"url":24,"identifiers":1908},"Smithburn, 1949, Rift Valley fever. Accidental infections among laboratory workers, 62, 213",{},{"id":24,"text":1910,"url":24,"identifiers":1911},"Gear, 1951, Rift Valley fever in South Africa, 25, 620",{},{"id":24,"text":1913,"url":24,"identifiers":1914},"Shimshony, 1983, Rift Valley fever, 27, 347",{},{"id":24,"text":1916,"url":24,"identifiers":1917},"1998",{},{"id":24,"text":1919,"url":24,"identifiers":1920},"2007",{},{"id":24,"text":1922,"url":24,"identifiers":1923},"Swanepoel, 2004, Rift Valley fever, 1037",{},{"id":24,"text":1925,"url":24,"identifiers":1926},"Meegan, 1979, The Rift Valley fever epizootic in Egypt 1977–78. 1. Description of the epizootic and virological studies, 73, 618, 10.1016\u002F0035-9203(79)90004-X",{"doi":1927},"10.1016\u002F0035-9203(79)90004-X",{"id":24,"text":1929,"url":24,"identifiers":1930},"Meegan, 1981, Rift Valley fever in Egypt: an overview of the epizootics in 1977 and 1978, 3, 100",{},{"id":24,"text":1932,"url":24,"identifiers":1933},"Meegan, 1981, Clinical experience with Rift Valley fever in humans during the 1977 Egyptian epizootic, 3, 114",{},{"id":24,"text":1935,"url":24,"identifiers":1936},"Shimshony, 1999, Disease prevention and preparedness in cases of animal health emergencies in the Middle East, 18, 66, 10.20506\u002Frst.18.1.1150",{"doi":1937},"10.20506\u002Frst.18.1.1150",{"id":24,"text":1939,"url":24,"identifiers":1940},"Hoogstraal, 1979, The Rift Valley fever epizootic in Egypt 1977–78. 2. Ecological and entomological studies, 73, 624, 10.1016\u002F0035-9203(79)90005-1",{"doi":1941},"10.1016\u002F0035-9203(79)90005-1",{"id":24,"text":1943,"url":24,"identifiers":1944},"Abd el-Rahim, 1999, An epizootic of Rift Valley fever in Egypt in 1997, 18, 741, 10.20506\u002Frst.18.3.1195",{"doi":1945},"10.20506\u002Frst.18.3.1195",{"id":24,"text":1947,"url":24,"identifiers":1948},"Abu-Elyazeed, 1996, Prevalence of anti-Rift Valley fever IgM antibody in abattoir workers in the Nile delta during the 1993 outbreak in Egypt, 74, 155",{},{"id":24,"text":1950,"url":24,"identifiers":1951},"Arthur, 1993, Recurrence of Rift Valley fever in Egypt, 342, 1149, 10.1016\u002F0140-6736(93)92128-G",{"doi":1952},"10.1016\u002F0140-6736(93)92128-G",{"id":24,"text":1954,"url":24,"identifiers":1955},"2000, Outbreak of Rift Valley fever—Saudi Arabia, August–October, 2000, 49, 905",{},{"id":24,"text":1957,"url":24,"identifiers":1958},"Jup, 2002, The 2000 epidemic of Rift Valley fever in Saudi Arabia: mosquito vector studies, 16, 245, 10.1046\u002Fj.1365-2915.2002.00371.x",{"doi":1959},"10.1046\u002Fj.1365-2915.2002.00371.x",{"id":24,"text":1961,"url":24,"identifiers":1962},"Miller, 2002, Isolation and genetic characterization of Rift Valley fever virus from Aedes vexans arabiensis, Kingdom of Saudi Arabia, 8, 1492, 10.3201\u002Feid0812.020194",{"doi":1963},"10.3201\u002Feid0812.020194",{"id":24,"text":1965,"url":24,"identifiers":1966},"Bird, 2007, Complete genome analysis of 33 ecologically and biologically diverse Rift Valley fever virus strains reveals widespread virus movement and low genetic diversity due to recent common ancestry, 81, 2805, 10.1128\u002FJVI.02095-06",{"doi":1967},"10.1128\u002FJVI.02095-06",{"id":24,"text":1969,"url":24,"identifiers":1970},"Shoemaker, 2002, Genetic analysis of viruses associated with emergence of Rift Valley fever in Saudi Arabia and Yemen, 2000–01, 8, 1415, 10.3201\u002Feid0812.020195",{"doi":1971},"10.3201\u002Feid0812.020195",{"id":24,"text":1973,"url":24,"identifiers":1974},"2007, Rift Valley fever outbreak—Kenya, November 2006–January 2007, 56, 73",{},{"id":24,"text":1976,"url":24,"identifiers":1977},"Bird, 2008, Multiple virus lineages sharing recent common ancestry were associated with a large Rift Valley fever outbreak among livestock in Kenya during 2006–2007, 82, 11152, 10.1128\u002FJVI.01519-08",{"doi":1978},"10.1128\u002FJVI.01519-08",{"id":24,"text":1980,"url":24,"identifiers":1981},"Davies, 1980, Possible vectors of Rift Valley fever in Kenya, 74, 815, 10.1016\u002F0035-9203(80)90213-8",{"doi":1982},"10.1016\u002F0035-9203(80)90213-8",{"id":24,"text":1984,"url":24,"identifiers":1985},"McIntosh, 1981, Epidemiological aspects of Rift Valley fever in South Africa with reference to vectors, 3, 92",{},{"id":24,"text":1987,"url":24,"identifiers":1988},"Meegan, 1980, Experimental transmission and field isolation studies implicating Culex pipiens as a vector of Rift Valley fever virus in Egypt, 29, 1405, 10.4269\u002Fajtmh.1980.29.1405",{"doi":1989},"10.4269\u002Fajtmh.1980.29.1405",{"id":24,"text":1991,"url":24,"identifiers":1992},"Smithburn, 1948, Rift Valley fever. Isolation of the virus from wild mosquitoes, 29, 107",{},{"id":24,"text":1994,"url":24,"identifiers":1995},"Linthicum, 1989, Transstadial and horizontal transmission of Rift Valley fever virus in Hyalomma truncatum, 41, 491, 10.4269\u002Fajtmh.1989.41.491",{"doi":1996},"10.4269\u002Fajtmh.1989.41.491",{"id":24,"text":1998,"url":24,"identifiers":1999},"Hoch, 1985, Mechanical transmission of Rift Valley fever virus by hematophagous diptera, 34, 188, 10.4269\u002Fajtmh.1985.34.188",{"doi":2000},"10.4269\u002Fajtmh.1985.34.188",{"id":24,"text":2002,"url":24,"identifiers":2003},"Hoch, 1984, Replication of Rift Valley fever virus in the sandfly Lutzomyia longipalpis, 33, 295, 10.4269\u002Fajtmh.1984.33.295",{"doi":2004},"10.4269\u002Fajtmh.1984.33.295",{"id":24,"text":2006,"url":24,"identifiers":2007},"Turell, 1987, Transmission studies in mosquitoes (Diptera: Culicidae) with disseminated Rift valley fever virus infections, 24, 11, 10.1093\u002Fjmedent\u002F24.1.11",{"doi":2008},"10.1093\u002Fjmedent\u002F24.1.11",{"id":24,"text":2010,"url":24,"identifiers":2011},"Meegan, 1989, Rift Valley fever, Vol 4, 51",{},{"id":24,"text":2013,"url":24,"identifiers":2014},"Turell, 2008, Vector competence of selected African mosquito (Diptera: Culicidae) species for Rift Valley fever virus, 45, 102, 10.1093\u002Fjmedent\u002F45.1.102",{"doi":2015},"10.1093\u002Fjmedent\u002F45.1.102",{"id":24,"text":2017,"url":24,"identifiers":2018},"Turell, 1988, Vector competence of a Houston, Texas strain of Aedes albopictus for Rift Valley fever virus, 4, 94",{},{"id":24,"text":2020,"url":24,"identifiers":2021},"Linthicum, 1987, Detection of Rift Valley fever viral activity in Kenya by satellite remote sensing imagery, 235, 1656, 10.1126\u002Fscience.3823909",{"doi":2022},"10.1126\u002Fscience.3823909",{"id":24,"text":2024,"url":24,"identifiers":2025},"Linthicum, 1985, Rift Valley fever virus (family Bunyaviridae, genus Phlebovirus). Isolations from Diptera collected during an inter-epizootic period in Kenya, 95, 197, 10.1017\u002FS0022172400062434",{"doi":2026},"10.1017\u002FS0022172400062434",{"id":24,"text":2028,"url":24,"identifiers":2029},"Logan, 1991, Egg hatching of Aedes mosquitoes during successive floodings in a Rift Valley fever endemic area in Kenya, 7, 109",{},{"id":24,"text":2031,"url":24,"identifiers":2032},"Logan, 1991, Mosquito species collected from a marsh in western Kenya during the long rains, 7, 395",{},{"id":24,"text":2034,"url":24,"identifiers":2035},"Pretorius, 1997, Rift Valley fever virus: a seroepidemiologic study of small terrestrial vertebrates in South Africa, 57, 693, 10.4269\u002Fajtmh.1997.57.693",{"doi":2036},"10.4269\u002Fajtmh.1997.57.693",{"id":24,"text":2038,"url":24,"identifiers":2039},"Weinbren, 1957, Rift Valley fever in a wild field rat (Arvicanthis abyssincus): a possible natural host, 31, 427",{},{"id":24,"text":2041,"url":24,"identifiers":2042},"Youssef, 2001, The potential role of Rattus rattus in enzootic cycle of Rift Valley fever in Egypt. 1—detection of RVF antibodies in R. rattus blood samples by both enzyme linked immuno sorbent assay (ELISA) and immuno-diffusion technique (ID), 76, 431",{},{"id":24,"text":2044,"url":24,"identifiers":2045},"Youssef, 2002, The potential role of Rattus rattus in enzootic cycle of Rift Valley fever in Egypt. 2—application of reverse transcriptase polymerase chain reaction (RT-PCR) in blood samples of Rattus rattus, 77, 133",{},{"id":24,"text":2047,"url":24,"identifiers":2048},"Turell, 1984, Increased mosquito feeding on Rift Valley fever virus-infected lambs, 33, 1232, 10.4269\u002Fajtmh.1984.33.1232",{"doi":2049},"10.4269\u002Fajtmh.1984.33.1232",{"id":24,"text":2051,"url":24,"identifiers":2052},"van Velden, 1977, Rift Valley fever affecting humans in South Africa: a clinicopathological study, 51, 867",{},{"id":24,"text":2054,"url":24,"identifiers":2055},"Weiss, 1957, Rift Valley fever. A review, 5, 431",{},{"id":24,"text":2057,"url":24,"identifiers":2058},"Peters, 1981, Pathogenesis of Rift Valley fever, 3, 21",{},{"id":24,"text":2060,"url":24,"identifiers":2061},"Walker, 1970, The clinical aspects of Rift Valley fever virus in household pets. I. Susceptibility of the dog, 121, 9, 10.1093\u002Finfdis\u002F121.1.9",{"doi":2062},"10.1093\u002Finfdis\u002F121.1.9",{"id":24,"text":2064,"url":24,"identifiers":2065},"Walker, 1970, The clinical aspects of Rift Valley fever virus in household pets. II. Susceptibility of the cat, 121, 19, 10.1093\u002Finfdis\u002F121.1.19",{"doi":2066},"10.1093\u002Finfdis\u002F121.1.19",{"id":24,"text":2068,"url":24,"identifiers":2069},"Easterday, 1962, Experimental Rift Valley fever in calves, goats, and pigs, 23, 1224",{},{"id":24,"text":2071,"url":24,"identifiers":2072},"Easterday, 1962, The pathogenesis of Rift Valley fever in lambs, 23, 470",{},{"id":24,"text":2074,"url":24,"identifiers":2075},"Van der Lugt, 1996, Distribution of viral antigen in tissues of new-born lambs infected with Rift Valley fever virus, 63, 341",{},{"id":24,"text":2077,"url":24,"identifiers":2078},"Erasmus, 1981, The symptomatology and pathology of Rift Valley fever in domestic animals, 3, 77",{},{"id":24,"text":2080,"url":24,"identifiers":2081},"Baskerville, 1992, Comparison of the pathogenicity for pregnant sheep of Rift Valley fever virus and a live attenuated vaccine, 52, 307, 10.1016\u002F0034-5288(92)90029-2",{"doi":2082},"10.1016\u002F0034-5288(92)90029-2",{"id":24,"text":2084,"url":24,"identifiers":2085},"Coetzer, 1977, Hydrops amnii in sheep associated with hydranencephaly and arthrogryposis with Wesselsbron disease and Rift Valley fever viruses as aetiological agents, 44, 119",{},{"id":24,"text":2087,"url":24,"identifiers":2088},"Easterday, 1965, Rift Valley fever, 10, 65",{},{"id":24,"text":2090,"url":24,"identifiers":2091},"Nabeth, 2001, Rift valley fever outbreak, Mauritania, 1998: seroepidemiologic, virologic, entomologic, and zoologic investigations, 7, 1052, 10.3201\u002Feid0706.010627",{"doi":2092},"10.3201\u002Feid0706.010627",{"id":24,"text":2094,"url":24,"identifiers":2095},"Coackley, 1967, Experimental infection of cattle with pantropic Rift Valley fever virus, 8, 399, 10.1016\u002FS0034-5288(18)34598-3",{"doi":2096},"10.1016\u002FS0034-5288(18)34598-3",{"id":24,"text":2098,"url":24,"identifiers":2099},"Gerdes, 2004, Rift Valley fever, 23, 613, 10.20506\u002Frst.23.2.1500",{"doi":2100},"10.20506\u002Frst.23.2.1500",{"id":24,"text":2102,"url":24,"identifiers":2103},"Chevalier, 2005, Rift Valley fever in small ruminants, Senegal, 2003, 11, 1693, 10.3201\u002Feid1111.050193",{"doi":2104},"10.3201\u002Feid1111.050193",{"id":24,"text":2106,"url":24,"identifiers":2107},"LeBreton, 2006, Rift Valley fever in goats, Cameroon, 12, 702, 10.3201\u002Feid1204.051428",{"doi":2108},"10.3201\u002Feid1204.051428",{"id":24,"text":2110,"url":24,"identifiers":2111},"Zeller, 1995, Rift Valley fever epizootic in small ruminants in southern Mauritania (October 1993): risk of extensive outbreaks, 75, 135",{},{"id":24,"text":2113,"url":24,"identifiers":2114},"Zeller, 1997, Enzootic activity of Rift Valley fever virus in Senegal, 56, 265, 10.4269\u002Fajtmh.1997.56.265",{"doi":2115},"10.4269\u002Fajtmh.1997.56.265",{"id":24,"text":2117,"url":24,"identifiers":2118},"Ksiazek, 1989, Rift Valley fever among domestic animals in the recent west African outbreak, 140, 67, 10.1016\u002FS0923-2516(89)80086-X",{"doi":2119},"10.1016\u002FS0923-2516(89)80086-X",{"id":24,"text":2121,"url":24,"identifiers":2122},"Scott, 1963, Rift Valley fever in camels, 86, 229, 10.1002\u002Fpath.1700860131",{"doi":2123},"10.1002\u002Fpath.1700860131",{"id":24,"text":2125,"url":24,"identifiers":2126},"Ali, 1978, Epidemiology of RVF in domestic animals in Egypt, 53, 255",{},{"id":24,"text":2128,"url":24,"identifiers":2129},"Imam, 1978, Epidemic of Rift Valley fever (RVF) in Egypt: isolation of RVF virus from animals, 53, 265",{},{"id":24,"text":2131,"url":24,"identifiers":2132},"Meegan, 1979, An epizootic of Rift Valley fever in Egypt in 1977, 105, 124, 10.1136\u002Fvr.105.6.124",{"doi":2133},"10.1136\u002Fvr.105.6.124",{"id":24,"text":2135,"url":24,"identifiers":2136},"Olaleye, 1996, Rift Valley fever in Nigeria infections in domestic animals, 15, 937, 10.20506\u002Frst.15.3.966",{"doi":2137},"10.20506\u002Frst.15.3.966",{"id":24,"text":2139,"url":24,"identifiers":2140},"Yedloutschnig, 1981, The response of ponies to inoculation with Rift Valley fever virus, 3, 68",{},{"id":24,"text":2142,"url":24,"identifiers":2143},"Davies, 1981, Experimental infection of the African buffalo with the virus of Rift Valley fever, 13, 185, 10.1007\u002FBF02237921",{"doi":2144},"10.1007\u002FBF02237921",{"id":24,"text":2146,"url":24,"identifiers":2147},"Nafady, 1985, Rift Valley fever: pathological changes on the suspected buffalo calves and aborted foetuses, 14, 95",{},{"id":24,"text":2149,"url":24,"identifiers":2150},"Evans, 2008, Prevalence of antibodies against Rift Valley fever virus in Kenyan wildlife, 136, 261",{},{"id":24,"text":2152,"url":24,"identifiers":2153},"House, 1996, Serum antibody to Rift Valley fever in African carnivores, 791, 345, 10.1111\u002Fj.1749-6632.1996.tb53541.x",{"doi":2154},"10.1111\u002Fj.1749-6632.1996.tb53541.x",{"id":24,"text":2156,"url":24,"identifiers":2157},"Boiro, 1987, Isolation of Rift Valley fever virus from bats in the Republic of Guinea [in French], 80, 62",{},{"id":24,"text":2159,"url":24,"identifiers":2160},"Oelofsen, 1999, Could bats act as reservoir hosts for Rift Valley fever virus?, 66, 51",{},{"id":24,"text":2162,"url":24,"identifiers":2163},"Peters, 1997, Viral hemorrhagic fever, 779",{},{"id":24,"text":2165,"url":24,"identifiers":2166},"Bird, 2007, Highly sensitive and broadly reactive quantitative reverse transcription-PCR assay for high-throughput detection of Rift Valley fever virus, 45, 3506, 10.1128\u002FJCM.00936-07",{"doi":2167},"10.1128\u002FJCM.00936-07",{"id":24,"text":2169,"url":24,"identifiers":2170},"McIntosh, 1980, Rift Valley fever in humans in South Africa, 58, 803",{},{"id":24,"text":2172,"url":24,"identifiers":2173},"Alrajhi, 2004, Rift Valley fever encephalitis, 10, 554, 10.3201\u002Feid1003.020817",{"doi":2174},"10.3201\u002Feid1003.020817",{"id":24,"text":2176,"url":24,"identifiers":2177},"Al-Hazmi, 2003, Epidemic Rift Valley fever in Saudi Arabia: a clinical study of severe illness in humans, 36, 245, 10.1086\u002F345671",{"doi":2178},"10.1086\u002F345671",{"id":24,"text":2180,"url":24,"identifiers":2181},"Findlay, 1936, Présence d'anticorps contre la fièvre de la Vallée du Rift dans le sang des Africains, 29, 986",{},{"id":24,"text":2183,"url":24,"identifiers":2184},"Paweska, 2007, Validation of an indirect ELISA based on a recombinant nucleocapsid protein of Rift Valley fever virus for the detection of IgG antibody in humans, 146, 119, 10.1016\u002Fj.jviromet.2007.06.006",{"doi":2185},"10.1016\u002Fj.jviromet.2007.06.006",{"id":24,"text":2187,"url":24,"identifiers":2188},"Niklasson, 1983, Detection of Rift Valley fever virus antigen by enzyme-linked immunosorbent assay, 17, 1026, 10.1128\u002Fjcm.17.6.1026-1031.1983",{"doi":2189},"10.1128\u002Fjcm.17.6.1026-1031.1983",{"id":24,"text":2191,"url":24,"identifiers":2192},"Morvan, 1992, Duration of immunoglobulin M antibodies against Rift Valley fever virus in cattle after natural infection, 86, 675, 10.1016\u002F0035-9203(92)90187-H",{"doi":2193},"10.1016\u002F0035-9203(92)90187-H",{"id":24,"text":2195,"url":24,"identifiers":2196},"Mariner, 1995, Antibodies to hemorrhagic fever viruses in domestic livestock in Niger: Rift valley fever and Crimean Congo hemorrhagic fever, 53, 217, 10.4269\u002Fajtmh.1995.53.217",{"doi":2197},"10.4269\u002Fajtmh.1995.53.217",{"id":24,"text":2199,"url":24,"identifiers":2200},"Ibrahim, 1997, Detection of Rift Valley fever virus in mosquitoes by RT-PCR, 11, 49, 10.1006\u002Fmcpr.1996.0075",{"doi":2201},"10.1006\u002Fmcpr.1996.0075",{"id":24,"text":2203,"url":24,"identifiers":2204},"Sall, 2001, Single-tube and nested reverse transcriptase-polymerase chain reaction for detection of Rift Valley fever virus in human and animal sera, 91, 85, 10.1016\u002FS0166-0934(00)00252-4",{"doi":2205},"10.1016\u002FS0166-0934(00)00252-4",{"id":24,"text":2207,"url":24,"identifiers":2208},"Jupp, 2000, Experimental detection of Rift Valley fever virus by reverse transcription-polymerase chain reaction assay in large samples of mosquitoes, 37, 467, 10.1093\u002Fjmedent\u002F37.3.467",{"doi":2209},"10.1093\u002Fjmedent\u002F37.3.467",{"id":24,"text":2211,"url":24,"identifiers":2212},"Vahhabzadeh, 2001, The first multiplex reverse transcriptase polymerase chain reaction (multiplex RT-PCR) for diagnostic of Lassa-, Marburg-, Ebola-, Rift-Valley- and Yellow fever virus in clinical specimen, 29, 53",{},{"id":24,"text":2214,"url":24,"identifiers":2215},"Garcia, 2001, Quantitative real-time PCR detection of Rift Valley fever virus and its application to evaluation of antiviral compounds, 39, 4456, 10.1128\u002FJCM.39.12.4456-4461.2001",{"doi":2216},"10.1128\u002FJCM.39.12.4456-4461.2001",{"id":24,"text":2218,"url":24,"identifiers":2219},"Drosten, 2002, Rapid detection and quantification of RNA of Ebola and Marburg viruses, Lassa virus, Crimean-Congo hemorrhagic fever virus, Rift Valley fever virus, Dengue virus, and Yellow fever virus by real-time reverse transcription-PCR, 40, 2323, 10.1128\u002FJCM.40.7.2323-2330.2002",{"doi":2220},"10.1128\u002FJCM.40.7.2323-2330.2002",{"id":24,"text":2222,"url":24,"identifiers":2223},"Morrill, 1991, Recombinant human interferon-gamma modulates Rift Valley fever virus infection in the rhesus monkey, 11, 297, 10.1089\u002Fjir.1991.11.297",{"doi":2224},"10.1089\u002Fjir.1991.11.297",{"id":24,"text":2226,"url":24,"identifiers":2227},"Anderson, 1987, Pathogenesis of Rift Valley fever virus (RVFV) in inbred rats, 2, 283, 10.1016\u002F0882-4010(87)90126-4",{"doi":2228},"10.1016\u002F0882-4010(87)90126-4",{"id":24,"text":2230,"url":24,"identifiers":2231},"Kende, 1987, Enhanced therapeutic efficacity of Poly (ICLC) and Ribavirin combinations against Rift Valley virus infection in mice, 31, 986, 10.1128\u002FAAC.31.7.986",{"doi":2232},"10.1128\u002FAAC.31.7.986",{"id":24,"text":2234,"url":24,"identifiers":2235},"Peters, 1986, Prophylaxis of Rift Valley fever with antiviral drugs, immune serum, an interferon inducer, and a macrophage activator, 6, 285, 10.1016\u002F0166-3542(86)90024-0",{"doi":2236},"10.1016\u002F0166-3542(86)90024-0",{"id":24,"text":2238,"url":24,"identifiers":2239},"Randall, 1962, The development of a formalin-killed Rift Valley fever virus vaccine for use in man, 89, 660, 10.4049\u002Fjimmunol.89.5.660",{"doi":2240},"10.4049\u002Fjimmunol.89.5.660",{"id":24,"text":2242,"url":24,"identifiers":2243},"Anderson, 1991, Efficacy of a Rift Valley fever virus vaccine against an aerosol infection in rats, 9, 710, 10.1016\u002F0264-410X(91)90285-E",{"doi":2244},"10.1016\u002F0264-410X(91)90285-E",{"id":24,"text":2246,"url":24,"identifiers":2247},"Randall, 1964, Immunization against Rift Valley fever virus. Studies on the immunogenicity of lyophilized formalin-inactivated vaccine, 93, 293, 10.4049\u002Fjimmunol.93.2.293",{"doi":2248},"10.4049\u002Fjimmunol.93.2.293",{"id":24,"text":2250,"url":24,"identifiers":2251},"Harrington, 1980, Evaluation of a formalin-inactivated Rift Valley fever vaccine in sheep, 41, 1559",{},{"id":24,"text":2253,"url":24,"identifiers":2254},"Pittman, 1999, Immunogenicity of an inactivated Rift Valley fever vaccine in humans: a 12-year experience, 18, 181, 10.1016\u002FS0264-410X(99)00218-2",{"doi":2255},"10.1016\u002FS0264-410X(99)00218-2",{"id":24,"text":2257,"url":24,"identifiers":2258},"Smithburn, 1949, Rift Valley fever; the neurotropic adaptation of the virus and the experimental use of this modified virus as a vaccine, 30, 1",{},{"id":24,"text":2260,"url":24,"identifiers":2261},"Barnard, 1979, Rift Valley fever vaccine—antibody and immune response in cattle to a live and an inactivated vaccine, 50, 155",{},{"id":24,"text":2263,"url":24,"identifiers":2264},"Caplen, 1985, Mutagen-directed attenuation of Rift Valley fever virus as a method for vaccine development, 66, 2271, 10.1099\u002F0022-1317-66-10-2271",{"doi":2265},"10.1099\u002F0022-1317-66-10-2271",{"id":24,"text":2267,"url":24,"identifiers":2268},"Rossi, 1988, Characterization of attenuated strains of Rift Valley fever virus, 69, 817, 10.1099\u002F0022-1317-69-4-817",{"doi":2269},"10.1099\u002F0022-1317-69-4-817",{"id":24,"text":2271,"url":24,"identifiers":2272},"Moussa, 1986, Experimental infection and protection of lambs with a minute plaque variant of Rift Valley fever virus, 35, 660, 10.4269\u002Fajtmh.1986.35.660",{"doi":2273},"10.4269\u002Fajtmh.1986.35.660",{"id":24,"text":2275,"url":24,"identifiers":2276},"Muller, 1995, Characterization of clone 13, a naturally attenuated avirulent isolate of Rift Valley fever virus, which is altered in the small segment, 53, 405, 10.4269\u002Fajtmh.1995.53.405",{"doi":2277},"10.4269\u002Fajtmh.1995.53.405",{"id":24,"text":2279,"url":24,"identifiers":2280},"Collett, 1987, Protective subunit immunogens to Rift Valley fever virus from bacteria and recombinant vaccinia virus, 321",{},{"id":24,"text":2282,"url":24,"identifiers":2283},"Wallace, 2006, Protective immune responses induced by different recombinant vaccine regimes to Rift Valley fever, 24, 7181, 10.1016\u002Fj.vaccine.2006.06.041",{"doi":2284},"10.1016\u002Fj.vaccine.2006.06.041",{"id":24,"text":2286,"url":24,"identifiers":2287},"Wallace, 2007, Improved method for the generation and selection of homogeneous lumpy skin disease virus (SA-Neethling) recombinants, 146, 52, 10.1016\u002Fj.jviromet.2007.06.004",{"doi":2288},"10.1016\u002Fj.jviromet.2007.06.004",{"id":24,"text":2290,"url":24,"identifiers":2291},"Spik, 2006, Immunogenicity of combination DNA vaccines for Rift Valley fever virus, tick-borne encephalitis virus, Hantaan virus, and Crimean Congo hemorrhagic fever virus, 24, 4657, 10.1016\u002Fj.vaccine.2005.08.034",{"doi":2292},"10.1016\u002Fj.vaccine.2005.08.034",{"id":24,"text":2294,"url":24,"identifiers":2295},"Bird, 2008, Rift Valley fever virus lacking the NSs and NSm genes is highly attenuated, confers protective immunity from virulent virus challenge and allows for differential identification of infected and vaccinated animals, 82, 2681, 10.1128\u002FJVI.02501-07",{"doi":2296},"10.1128\u002FJVI.02501-07",{"id":24,"text":2298,"url":24,"identifiers":2299},"Barnard, 1977, An inactivated rift valley fever vaccine, 48, 45",{},{"id":24,"text":2301,"url":24,"identifiers":2302},"Eddy, 1981, Rift Valley fever vaccine for humans, 3, 124",{},{"id":24,"text":2304,"url":24,"identifiers":2305},"Botros, 2006, Adverse response of non-indigenous cattle of European breeds to live attenuated Smithburn Rift Valley fever vaccine, 78, 787, 10.1002\u002Fjmv.20624",{"doi":2306},"10.1002\u002Fjmv.20624",{"id":24,"text":2308,"url":24,"identifiers":2309},"Takehara, 1989, Identification of mutations in the M RNA of a candidate vaccine strain of Rift Valley fever virus, 169, 452, 10.1016\u002F0042-6822(89)90171-2",{"doi":2310},"10.1016\u002F0042-6822(89)90171-2",{"id":24,"text":2312,"url":24,"identifiers":2313},"Saluzzo, 1990, Use of reassortant viruses to map attenuating and temperature-sensitive mutations of the Rift Valley fever virus MP-12 vaccine, 8, 369, 10.1016\u002F0264-410X(90)90096-5",{"doi":2314},"10.1016\u002F0264-410X(90)90096-5",{"id":24,"text":2316,"url":24,"identifiers":2317},"Vialat, 1997, Mapping of the mutations present in the genome of the Rift Valley fever virus attenuated MP12 strain and their putative role in attenuation, 52, 43, 10.1016\u002FS0168-1702(97)00097-X",{"doi":2318},"10.1016\u002FS0168-1702(97)00097-X",{"id":24,"text":2320,"url":24,"identifiers":2321},"Hubbard, 1991, Ability of a mutagenized virus variant to protect young lambs from Rift Valley fever, 52, 50",{},{"id":24,"text":2323,"url":24,"identifiers":2324},"Morrill, 1991, Further evaluation of a mutagen-attenuated Rift Valley fever vaccine in sheep, 9, 35, 10.1016\u002F0264-410X(91)90314-V",{"doi":2325},"10.1016\u002F0264-410X(91)90314-V",{"id":24,"text":2327,"url":24,"identifiers":2328},"Morrill, 1997, Safety of a mutagen-attenuated Rift Valley fever virus vaccine in fetal and neonatal bovids, 58, 1110",{},{"id":24,"text":2330,"url":24,"identifiers":2331},"Hunter, 2002, Teratogenicity of a mutagenised Rift Valley fever virus (MVP 12) in sheep, 69, 95",{},{"id":24,"text":2333,"url":24,"identifiers":2334},"Shimshony, 1981, The influence of information flow on the veterinary policy of Rift Valley fever prevention in Israel, 1978–1979, 3, 160",{},{"id":24,"text":2336,"url":24,"identifiers":2337},"Klopfer-Orgad, 1981, Activities of the Kimron Veterinary Institute in the framework of Rift Valley fever prevention in Israel, 3, 172",{},{"id":2339,"createTime":2340,"updateTime":2341,"relativeEntities":2342,"slug":2343,"properties":2344,"entityType":965,"verifyStatus":23,"verifyTime":2361,"verifyNote":1754,"languages":2362,"translateLanguages":2363,"viewCount":25,"primaryUrl":2364,"fullTextUrl":24,"authors":2365,"publicationType":970,"publisherRelationship":2416,"citationCount":2467,"citationInfo":2468,"publishDate":2471,"publishYear":2469,"citationAnalyzeStatus":23,"lastCitationAnalyze":24,"indexDatabases":2472,"openAccess":24,"references":2473,"isForceReanalyzing":1737},"7aa9ace6-67d4-473a-9d90-c6991e2987ab","2024-11-25T07:12:56.087+00:00","2025-01-12T04:32:39.885+00:00",[],"Population-and-survival-characteristics-of-cats-with-hypertrophic-cardiomyopathy-260-cases-1990-1999-",{"mag":2345,"keywords":2347,"openalex":2349,"abstract":2351,"title":2354,"pm":2357,"doi":2359},{"VOID":2346},"2151393028",{"VI":2348},"",{"VOID":2350},"W2151393028",{"VI":2352,"EN":2353},"\u003Cjats:title>Tóm tắt\u003C\u002Fjats:title>\u003Cjats:p>\u003Cjats:bold>Mục tiêu\u003C\u002Fjats:bold>—Để xác định những đặc điểm quần thể hiện tại, các phát hiện lâm sàng và thời gian sống của mèo bị cardiomyopathy phì đại (HCM).\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Thiết kế\u003C\u002Fjats:bold>—Nghiên cứu hồi cứu.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Động vật\u003C\u002Fjats:bold>—260 mèo mắc HCM.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Quy trình\u003C\u002Fjats:bold>—Thông tin được thu thập từ hồ sơ y tế. Các mèo được phân loại vào một trong bốn nhóm lâm sàng (nhóm suy tim sung huyết [CHF], nhóm huyết khối động mạch [ATE], nhóm ngất xỉu hoặc mèo không có triệu chứng lâm sàng [nhóm dưới lâm sàng]) dựa trên các triệu chứng lâm sàng chính tại sự khám ban đầu.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Kết quả\u003C\u002Fjats:bold>—120 mèo được phân loại vào nhóm CHF, 43 vào nhóm ATE, 10 vào nhóm ngất xỉu và 87 vào nhóm dưới lâm sàng. Các sự kiện trước đó có thể đã kích thích CHF bao gồm việc truyền dịch tĩnh mạch, gây mê, phẫu thuật và việc sử dụng corticosteroid gần đây. Thời gian sống trung bình là 709 ngày (khoảng, 2 đến 4.418 ngày) cho các mèo sống > 24 giờ. Các mèo trong nhóm dưới lâm sàng sống lâu nhất (thời gian sống trung bình, 1.129 ngày; khoảng, 2 đến 3.778 ngày), tiếp theo là các mèo trong nhóm ngất xỉu (654 ngày; khoảng, 28 đến 1.505 ngày), các mèo trong nhóm CHF (563 ngày; khoảng, 2 đến 4.418 ngày) và các mèo trong nhóm ATE (184 ngày; khoảng, 2 đến 2.278 ngày). Nguyên nhân tử vong bao gồm ATE (n = 56), CHF (49), tử vong đột ngột (13) và các nguyên nhân không tim mạch (27). Trong các phân tích đơn biến, thời gian sống có mối tương quan tiêu cực với kích thước tâm nhĩ trái, tuổi, sự phì đại thất phải và chọc dò ngực. Các mèo với chuyển động trước tâm thu của van hai lá sống lâu hơn các mèo không có phát hiện siêu âm này. Trong các phân tích đa biến, chỉ có tuổi và kích thước tâm nhĩ trái vẫn là những yếu tố tiên đoán có ý nghĩa về thời gian sống.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Kết luận và Ý nghĩa lâm sàng\u003C\u002Fjats:bold>—Mặc dù thời gian sống tổng thể của các mèo mắc HCM tương tự như các báo cáo trước đây, thời gian sống của các mèo mắc CHF hoặc ATE lại dài hơn so với những gì đã được báo cáo trước đây. (\u003Cjats:italic>J Am Vet Med Assoc\u003C\u002Fjats:italic>2002;220:202–207)\u003C\u002Fjats:p>","\u003Cjats:title>Abstract\u003C\u002Fjats:title>\u003Cjats:p>\u003Cjats:bold>Objective\u003C\u002Fjats:bold>—To determine current population characteristics of, clinical findings in, and survival times for cats with hypertrophic cardiomyopathy (HCM).\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Design\u003C\u002Fjats:bold>—Retrospective study.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Animals\u003C\u002Fjats:bold>—260 cats with HCM.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Procedure\u003C\u002Fjats:bold>—Information was obtained from the medical records. Cats were classified into 1 of 4 clinical groups (congestive heart failure [CHF] group, arterial thromboembolism [ATE] group, syncope group, or cats without clinical signs [subclinical group]) on the basis of the primary clinical signs at the initial examination.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Results\u003C\u002Fjats:bold>—120 cats were classified in the CHF group, 43 in the ATE group, 10 in the syncope group, and 87 in the subclinical group. Antecedent events that may have precipitated CHF included IV fluid administration, anesthesia, surgery, and recent corticosteroid administration. Median survival time was 709 days (range, 2 to 4,418 days) for cats that survived > 24 hours. Cats in the subclinical group lived the longest (median survival time, 1,129 days; range, 2 to 3,778 days), followed by cats in the syncope group (654 days; range, 28 to 1,505 days), cats in the CHF group (563 days; range, 2 to 4,418 days), and cats in the ATE group (184 days; range, 2 to 2,278 days). Causes of death included ATE (n = 56), CHF (49), sudden death (13), and noncardiac causes (27). In univariate analyses, survival time was negatively correlated with left atrial size, age, right ventricular enlargement, and thoracentesis. Cats with systolic anterior motion of the mitral valve lived longer than cats without this echocardiographic finding. In multivariate analyses, only age and left atrial size remained significant predictors of survival time.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Conclusions and Clinical Relevance\u003C\u002Fjats:bold>—Although overall survival time for cats with HCM was similar to earlier reports, survival times for cats with CHF or ATE were longer than previously reported. 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10.1016\u002FS0195-5616(98)50126-X",{"doi":2520},"10.1016\u002FS0195-5616(98)50126-X",{"id":24,"text":2522,"url":24,"identifiers":2523},"Kittleson MD, 1999, Circulation, 99, 3172, 10.1161\u002F01.CIR.99.24.3172",{"doi":2524},"10.1161\u002F01.CIR.99.24.3172",{"id":24,"text":2526,"url":24,"identifiers":2527},"Freeman LM, 1997, Can J Vet Res, 61, 227",{},{"id":24,"text":2529,"url":24,"identifiers":2530},"Tilley LP, 1977, Vet Clin North Am Small Anim Pract, 7, 415",{},{"id":24,"text":2532,"url":24,"identifiers":2533},"Rush JE, 1998, Vet Clin North Am Small Anim Pract, 28, 1459, 10.1016\u002FS0195-5616(98)50132-5",{"doi":2534},"10.1016\u002FS0195-5616(98)50132-5",{"id":24,"text":2536,"url":24,"identifiers":2537},"Plumb DC. Veterinary drug handbook. 3rd ed. Ames, Iowa: Iowa State University Press, 1999;485-487.",{},{"id":24,"text":2539,"url":24,"identifiers":2540},"Van der Linde-Sipman JS, 1992, Vet Q, 14, 91, 10.1080\u002F01652176.1992.9694340",{"doi":2541},"10.1080\u002F01652176.1992.9694340",{"id":2543,"createTime":2544,"updateTime":2545,"relativeEntities":2546,"slug":2547,"properties":2548,"entityType":965,"verifyStatus":23,"verifyTime":2544,"verifyNote":1754,"languages":2564,"translateLanguages":2565,"viewCount":25,"primaryUrl":2566,"fullTextUrl":24,"authors":2567,"publicationType":970,"publisherRelationship":2614,"citationCount":541,"citationInfo":2664,"publishDate":2666,"publishYear":1859,"citationAnalyzeStatus":23,"lastCitationAnalyze":24,"indexDatabases":2667,"openAccess":24,"references":2668,"isForceReanalyzing":1737},"51e770e3-100a-4308-9b04-bd5c9b557156","2024-11-25T07:12:52.601+00:00","2025-01-12T04:33:38.484+00:00",[],"Prevalence-of-cardiomyopathy-in-apparently-healthy-cats",{"mag":2549,"keywords":2551,"openalex":2552,"abstract":2554,"title":2557,"pm":2560,"doi":2562},{"VOID":2550},"2134992907",{"VI":2348},{"VOID":2553},"W2134992907",{"VI":2555,"EN":2556},"\u003Cjats:title> Tóm tắt \u003C\u002Fjats:title>\n\u003Cjats:p>\u003Cjats:bold>Mục tiêu\u003C\u002Fjats:bold>—Xác định tỷ lệ bệnh cơ tim và mối quan hệ giữa bệnh cơ tim và tiếng tim bất thường ở mèo có vẻ khỏe mạnh. \u003C\u002Fjats:p>\n\u003Cjats:p>\u003Cjats:bold>Thiết kế\u003C\u002Fjats:bold>—Nghiên cứu cắt ngang. \u003C\u002Fjats:p>\n\u003Cjats:p>\u003Cjats:bold>Động vật\u003C\u002Fjats:bold>—103 chú mèo nhà, có vẻ khỏe mạnh, thuộc sở hữu tư nhân. \u003C\u002Fjats:p>\n\u003Cjats:p>\u003Cjats:bold>Quy trình\u003C\u002Fjats:bold>—Mèo được kiểm tra thể chất và siêu âm tim bởi 2 nhà nghiên cứu độc lập. Độ dày thành thất trái được xác định thông qua siêu âm 2 chiều trong các mặt phẳng ngắn và dài. Tăng huyết áp thất trái được xác định khi các phép đo vào cuối tâm trương của vách ngăn liên thất hoặc thành sau lớn hơn hoặc bằng 6 mm. Những chú mèo có tăng huyết áp thất trái nhưng không có giãn thất trái được coi là có bệnh cơ tim phì đại (HCM). Các mối liên quan giữa tiếng tim bất thường và các tốc độ siêu âm Doppler chỉ ra tình trạng tắc nghẽn động mạch thoát tĩnh mạch thất động đã được đánh giá. \u003C\u002Fjats:p>\n\u003Cjats:p>\u003Cjats:bold>Kết quả\u003C\u002Fjats:bold>—Tiếng tim bất thường được phát hiện ở 16 (15,5%; khoảng tin cậy 95%, 9,2% đến 24,0%) mèo; trong số đó, 5 mèo có bệnh cơ tim. Bệnh cơ tim cũng được xác định ở 16 (15,5%; khoảng tin cậy 95%, 9,2% đến 24,0%) mèo; 15 có HCM, và 1 có bệnh cơ tim phải do rối loạn nhịp. Trong số những con mèo có HCM, 11 có tăng huyết áp thất trái từng phần, 3 có tăng huyết áp thất trái lan rộng, và 1 có tăng huyết áp thất trái trên mức chấp nhận với chuyển động trước tâm thu rõ ràng của van 2 lá. Độ nhạy và độ đặc hiệu của việc phát hiện tiếng tim bất thường qua thính chẩn trong chẩn đoán bệnh cơ tim lần lượt là 31% và 87%. Bằng chứng siêu âm về sự gia tăng tốc độ tâm thu muộn trong các động mạch thoát tĩnh mạch thất có liên quan đến sự tồn tại của tiếng tim bất thường. \u003C\u002Fjats:p>\n\u003Cjats:p>\u003Cjats:bold>Kết luận và Ý nghĩa lâm sàng\u003C\u002Fjats:bold>—Bệnh cơ tim phổ biến trong những chú mèo khỏe mạnh được đánh giá trong nghiên cứu này. Ở những chú mèo có vẻ khỏe mạnh, việc phát hiện tiếng tim bất thường không phải là một chỉ thị đáng tin cậy cho bệnh cơ tim.\u003C\u002Fjats:p>","\u003Cjats:title>Abstract\u003C\u002Fjats:title>\n\u003Cjats:p>\u003Cjats:bold>Objective\u003C\u002Fjats:bold>—To determine the prevalence of cardiomyopathy and the relationship between cardiomyopathy and heart murmurs in apparently healthy cats.\u003C\u002Fjats:p>\n\u003Cjats:p>\u003Cjats:bold>Design\u003C\u002Fjats:bold>—Cross-sectional study.\u003C\u002Fjats:p>\n\u003Cjats:p>\u003Cjats:bold>Animals\u003C\u002Fjats:bold>—103 privately owned, apparently healthy domestic cats.\u003C\u002Fjats:p>\n\u003Cjats:p>\u003Cjats:bold>Procedures\u003C\u002Fjats:bold>—Cats were physically and echocardiographically examined by 2 investigators independently. Left ventricular wall thickness was determined via 2-dimensional echocardiography in short-axis and long-axis planes. Left ventricular hypertrophy was identified when end-diastolic measurements of the interventricular septum or posterior wall were ≥ 6 mm. Cats with left ventricular hypertrophy but without left ventricular dilatation were considered to have hypertrophic cardiomyopathy (HCM). The associations between heart murmurs and Doppler echocardiographic velocity profiles indicative of dynamic ventricular outflow tract obstruction were evaluated.\u003C\u002Fjats:p>\n\u003Cjats:p>\u003Cjats:bold>Results\u003C\u002Fjats:bold>—Heart murmurs were detected in 16 (15.5%; 95% confidence interval, 9.2% to 24.0%) cats; of these, 5 had cardiomyopathy. Cardiomyopathy was also identified in 16 (15.5%; 95% confidence interval, 9.2% to 24.0%) cats; 15 had HCM, and 1 had arrhythmogenic right ventricular cardiomyopathy. Of the cats with HCM, 11 had segmental left ventricular hypertrophy, 3 had diffuse left ventricular hypertrophy, and 1 had borderline left ventricular hypertrophy with marked systolic anterior motion of the mitral valve. Sensitivity and specificity of auscultatory detection of a heart murmur for diagnosing cardiomyopathy were 31% and 87%, respectively. Echocardiographic evidence of late systolic acceleration within ventricular outflow tracts was associated with the existence of a heart murmur.\u003C\u002Fjats:p>\n\u003Cjats:p>\u003Cjats:bold>Conclusions and Clinical Relevance\u003C\u002Fjats:bold>—Cardiomyopathy was common in the healthy cats evaluated in this study. In apparently healthy cats, detection of a heart murmur is not a reliable indicator of cardiomyopathy.\u003C\u002Fjats:p>",{"EN":2558,"VI":2559},"Prevalence of cardiomyopathy in apparently healthy cats","Tỷ lệ bệnh cơ tim ở mèo có vẻ khỏe mạnh",{"VOID":2561},"19480619",{"VOID":2563},"10.2460\u002Fjavma.234.11.1398",[126],[125],"https:\u002F\u002Favmajournals.avma.org\u002Fview\u002Fjournals\u002Fjavma\u002F234\u002F11\u002Fjavma.234.11.1398.xml",[2568,2585,2596,2605],{"id":2569,"sortIndex":25,"researcher":24,"roles":2570,"affiliations":2571,"properties":2580,"displayName":2582,"givenName":24,"familyName":24},"a0423b1d-a324-4783-bc4c-8afdd4f5289d",[],[2572],{"id":2573,"sortIndex":25,"affiliation":2574,"properties":24},"dcff266b-f7c6-4b9b-bb64-d7d9a38900cd",{"id":2573,"createTime":24,"updateTime":24,"relativeEntities":2575,"slug":24,"properties":2576,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":2579,"statistic":24},[],{"title":2577},{"EN":2578},"Department of Small Animal Clinical Sciences, Virginia-Maryland Regional College of Veterinary Medicine, Virginia Polytechnic Institute, Blacksburg, VA 24061, USA.",[],{"title":2581,"openalex":2583},{"EN":2582},"Christopher F. Paige",{"VOID":2584},"A5041748892",{"id":2586,"sortIndex":89,"researcher":24,"roles":2587,"affiliations":2588,"properties":2589,"displayName":2593,"givenName":24,"familyName":24},"d739c7c1-501c-4776-9605-fe3cd76ee399",[],[],{"orcid":2590,"title":2592,"openalex":2594},{"VOID":2591},"https:\u002F\u002Forcid.org\u002F0000-0001-6981-8968",{"EN":2593},"Jonathan A. Abbott",{"VOID":2595},"A5078590096",{"id":2597,"sortIndex":86,"researcher":24,"roles":2598,"affiliations":2599,"properties":2600,"displayName":2602,"givenName":24,"familyName":24},"256b7d51-3016-492f-8a07-6b77cf109990",[],[],{"title":2601,"openalex":2603},{"EN":2602},"François Elvinger",{"VOID":2604},"A5066851512",{"id":2606,"sortIndex":135,"researcher":24,"roles":2607,"affiliations":2608,"properties":2609,"displayName":2611,"givenName":24,"familyName":24},"d3ba64df-4580-491c-a7ff-335175a10559",[],[],{"title":2610,"openalex":2612},{"EN":2611},"R. Lee Pyle",{"VOID":2613},"A5112028184",{"url":24,"publisher":2615,"properties":2658},{"id":6,"createTime":7,"updateTime":8,"relativeEntities":2616,"slug":10,"properties":2617,"entityType":22,"verifyStatus":23,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":25,"subjectFields":2622,"manageAffiliations":2627,"indexDatabases":2638,"url":83,"thumbnailPath":24,"statistic":2653,"gsStatistic":24,"type":24,"analyzePriority":24},[],{"country":2618,"eissn":2619,"issn":2620,"title":2621},{"VOID":13},{"VOID":15},{"VOID":17},{"EN":19},[2623],{"id":28,"createTime":24,"updateTime":24,"relativeEntities":2624,"label":2625,"description":2626,"parentId":24,"standard":24,"scholarHubFieldId":24},[],{"EN":31},{},[2628,2633],{"id":35,"createTime":24,"updateTime":24,"relativeEntities":2629,"slug":24,"properties":2630,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":2632,"statistic":24},[],{"title":2631},{"EN":39},[],{"id":42,"createTime":24,"updateTime":24,"relativeEntities":2634,"slug":24,"properties":2635,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":2637,"statistic":24},[],{"title":2636},{"EN":46},[],[2639,2646],{"id":50,"indexDatabase":2640,"url":63,"indexYears":24,"academicFieldIds":2645,"indexDatabaseRanking":24},{"id":52,"createTime":24,"updateTime":24,"relativeEntities":2641,"label":2642,"description":2643,"key":59,"publicationTags":2644,"standard":24},[],{"EN":55,"VI":55},{"EN":57,"VI":58},[61,62],[65],{"id":67,"indexDatabase":2647,"url":78,"indexYears":79,"academicFieldIds":2652,"indexDatabaseRanking":82},{"id":69,"createTime":24,"updateTime":24,"relativeEntities":2648,"label":2649,"description":2650,"key":75,"publicationTags":2651,"standard":24},[],{"EN":72,"VI":72},{"EN":72,"VI":74},[77],[81],{"impactFactor":25,"impactFactorByYear":2654,"i10Index":87,"i10IndexLast5Year":25,"totalPublication":87,"totalPublicationByYear":2655,"totalCitation":90,"totalCitationByYear":2656,"totalCitationPerPublication":98,"totalCitationPerPublicationByYear":2657,"hindexLast5Year":87,"hindex":87},{"2013":86,"2014":87},{"2004":89,"2006":89,"2008":89,"2009":89,"2010":89,"2012":89},{"2004":92,"2006":93,"2008":94,"2009":95,"2010":96,"2012":97},{"2004":92,"2006":93,"2008":94,"2009":95,"2010":96,"2012":97},{"issue":2659,"pages":2661,"volume":2663},{"VOID":2660},"11",{"VOID":2662},"1398-1403",{"VOID":1856},{"total":541,"publishYear":1859,"statisticByYear":2665},{"2012":295,"2013":140,"2014":412,"2015":222,"2016":143,"2017":236,"2018":446,"2019":295,"2020":224,"2021":226,"2022":218,"2023":221,"2024":87},"2009-06-01",[61,82],[2669,2673,2676,2679,2683,2686,2690,2693,2697,2700,2704,2707,2711,2715,2718,2721,2724,2727,2731,2735,2738,2742,2746],{"id":24,"text":2670,"url":24,"identifiers":2671},"Ferasin, 2003, Feline idiopathic cardiomyopathy: a retrospective study of 106 cats (1994–2001), 5, 151, 10.1016\u002FS1098-612X(02)00133-X",{"doi":2672},"10.1016\u002FS1098-612X(02)00133-X",{"id":24,"text":2674,"url":24,"identifiers":2675},"Rush, 2002, Population and survival characteristics of cats with hypertrophic cardiomyopathy: 260 cases (1990–1999), 220, 202, 10.2460\u002Fjavma.2002.220.202",{"doi":2360},{"id":24,"text":2677,"url":24,"identifiers":2678},"Atkins, 1992, Risk factors, clinical signs, and survival in cats with a clinical diagnosis of idiopathic hypertrophic cardiomyopathy: 74 cases (1985–1989), 201, 613",{},{"id":24,"text":2680,"url":24,"identifiers":2681},"Côté, 2004, Assessment of the prevalence of heart murmurs in overtly healthy cats, 225, 384, 10.2460\u002Fjavma.2004.225.384",{"doi":2682},"10.2460\u002Fjavma.2004.225.384",{"id":24,"text":2684,"url":24,"identifiers":2685},"Grover, 2005, Cardiac cats, 37",{},{"id":24,"text":2687,"url":24,"identifiers":2688},"Riesen, 2007, Prevalence of heart disease in symptomatic cats: an overview from 1998 to 2005, 149, 65, 10.1024\u002F0036-7281.149.2.65",{"doi":2689},"10.1024\u002F0036-7281.149.2.65",{"id":24,"text":2691,"url":24,"identifiers":2692},"Braunwald, 2005, Physical examination of the heart and circulation, 77",{},{"id":24,"text":2694,"url":24,"identifiers":2695},"Thomas, 1993, Recommendations for standards in transthoracic two-dimensional echocardiography in the dog and cat. Echocardiography Committee of the Specialty of Cardiology, American College of Veterinary Internal Medicine, 7, 247, 10.1111\u002Fj.1939-1676.1993.tb01015.x",{"doi":2696},"10.1111\u002Fj.1939-1676.1993.tb01015.x",{"id":24,"text":2698,"url":24,"identifiers":2699},"Weyman, 1994, 600",{},{"id":24,"text":2701,"url":24,"identifiers":2702},"Abbott, 2006, Two-dimensional echocardiographic assessment of the feline left atrium, 20, 111, 10.1111\u002Fj.1939-1676.2006.tb02830.x",{"doi":2703},"10.1111\u002Fj.1939-1676.2006.tb02830.x",{"id":24,"text":2705,"url":24,"identifiers":2706},"Fox, 1995, Echocardiographic assessment of spontaneously occurring feline hypertrophic cardiomyopathy. An animal model of human disease, 92, 2645, 10.1161\u002F01.CIR.92.9.2645",{"doi":2493},{"id":24,"text":2708,"url":24,"identifiers":2709},"Fox, 2000, Spontaneously occurring arrhythmogenic right ventricular cardiomyopathy in the domestic cat: a new animal model similar to the human disease, 102, 1863, 10.1161\u002F01.CIR.102.15.1863",{"doi":2710},"10.1161\u002F01.CIR.102.15.1863",{"id":24,"text":2712,"url":24,"identifiers":2713},"Rishniw, 2002, Dynamic right ventricular outflow obstruction: a new cause of systolic murmurs in cats, 16, 547, 10.1111\u002Fj.1939-1676.2002.tb02385.x",{"doi":2714},"10.1111\u002Fj.1939-1676.2002.tb02385.x",{"id":24,"text":2716,"url":24,"identifiers":2717},"Grandy, 1992, Evaluation of the Doppler ultrasonic method of measuring systolic arterial blood pressure in cats, 53, 1166",{},{"id":24,"text":2719,"url":24,"identifiers":2720},"Maron, 2002, Hypertrophic cardiomyopathy: a systematic review, 287, 1308",{},{"id":24,"text":2722,"url":24,"identifiers":2723},"Maron, 2003, American College of Cardiology\u002FEuropean Society of Cardiology clinical expert consensus document on hypertrophic cardiomyopathy. A report of the American College of Cardiology Foundation Task Force on Clinical Expert Consensus Documents and the European Society of Cardiology Committee for Practice Guidelines, 42, 1687",{},{"id":24,"text":2725,"url":24,"identifiers":2726},"Kittleson, 1999, Familial hypertrophic cardiomyopathy in maine coon cats: an animal model of human disease, 99, 3172, 10.1161\u002F01.CIR.99.24.3172",{"doi":2524},{"id":24,"text":2728,"url":24,"identifiers":2729},"Meurs, 2005, A cardiac myosin binding protein C mutation in the Maine Coon cat with familial hypertrophic cardiomyopathy, 14, 3587, 10.1093\u002Fhmg\u002Fddi386",{"doi":2730},"10.1093\u002Fhmg\u002Fddi386",{"id":24,"text":2732,"url":24,"identifiers":2733},"Altman, 1994, Diagnostic tests. 1: sensitivity and specificity, 308, 1552, 10.1136\u002Fbmj.308.6943.1552",{"doi":2734},"10.1136\u002Fbmj.308.6943.1552",{"id":24,"text":2736,"url":24,"identifiers":2737},"Hennekens, 1987, 54",{},{"id":24,"text":2739,"url":24,"identifiers":2740},"Deeks, 2004, Diagnostic tests 4: likelihood ratios, 329, 168, 10.1136\u002Fbmj.329.7458.168",{"doi":2741},"10.1136\u002Fbmj.329.7458.168",{"id":24,"text":2743,"url":24,"identifiers":2744},"Zocchetti, 1997, Relationship between prevalence rate ratios and odds ratios in cross-sectional studies, 26, 220, 10.1093\u002Fije\u002F26.1.220",{"doi":2745},"10.1093\u002Fije\u002F26.1.220",{"id":24,"text":2747,"url":24,"identifiers":2748},"Binder, 2006, Echocardiography-guided genetic testing in hypertrophic cardiomyopathy: septal morphological features predict the presence of myofilament mutations, 81, 459, 10.4065\u002F81.4.459",{"doi":2749},"10.4065\u002F81.4.459",{"id":2751,"createTime":2752,"updateTime":2753,"relativeEntities":2754,"slug":2755,"properties":2756,"entityType":965,"verifyStatus":122,"verifyTime":2752,"verifyNote":2772,"languages":2773,"translateLanguages":2774,"viewCount":25,"primaryUrl":2775,"fullTextUrl":24,"authors":2776,"publicationType":970,"publisherRelationship":2823,"citationCount":2874,"citationInfo":2875,"publishDate":2877,"publishYear":2469,"citationAnalyzeStatus":23,"lastCitationAnalyze":24,"indexDatabases":2878,"openAccess":24,"references":2879,"isForceReanalyzing":1737},"9991d563-0839-4313-9746-a312130362bb","2024-12-18T16:43:17.138+00:00","2025-01-12T04:34:36.899+00:00",[],"Clinical-features-and-outcome-in-dogs-and-cats-with-obsessive-compulsive-disorder-126-cases-1989-2000-",{"mag":2757,"keywords":2759,"openalex":2760,"abstract":2762,"title":2765,"pm":2768,"doi":2770},{"VOID":2758},"2107806442",{"VI":2348},{"VOID":2761},"W2107806442",{"VI":2763,"EN":2764},"\u003Cjats:title>Tóm tắt\u003C\u002Fjats:title>\u003Cjats:p>\u003Cjats:bold>Mục tiêu\u003C\u002Fjats:bold>—Xác định các đặc điểm lâm sàng và kết quả ở chó và mèo mắc chứng rối loạn ám ảnh cưỡng chế (OCD).\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Thiết kế\u003C\u002Fjats:bold>—Nghiên cứu hồi cứu.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Động vật\u003C\u002Fjats:bold>—103 con chó và 23 con mèo.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Các thủ tục\u003C\u002Fjats:bold>—Các hồ sơ của bệnh nhân mắc OCD đã được phân tích để đánh giá các đặc điểm lâm sàng, thuốc đã sử dụng, mức độ thay đổi hành vi và kết quả.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Kết quả\u003C\u002Fjats:bold>—Hầu hết chó mắc OCD đều được lấy từ các nhà gây giống. Số lượng chó đực vượt trội rõ ràng so với chó cái (2:1). Ở một mẫu nhỏ, số mèo cái nhiều hơn mèo đực 2:1. Hầu hết chó bị ảnh hưởng sống trong các hộ gia đình có 2 hoặc nhiều người và có chó hoặc mèo khác, và có một số đào tạo chính thức. Sự tuân thủ của khách hàng đối với việc thay đổi hành vi là cao. Sự kết hợp giữa thay đổi hành vi và thuốc đã dẫn đến sự giảm lớn về cường độ và tần suất OCD ở hầu hết động vật. Clomipramine có hiệu quả điều trị ở chó vượt trội hơn so với amitriptyline. Chỉ có 1 con chó và 1 con mèo bị euthanized do OCD trong suốt nghiên cứu.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Kết luận và Ý nghĩa lâm sàng\u003C\u002Fjats:bold>—OCD ở chó không có vẻ liên quan đến việc thiếu đào tạo, thiếu kích thích trong hộ gia đình, hoặc sự giam cầm xã hội. Ở mèo, OCD có thể liên quan đến căng thẳng về môi trường và xã hội. Chứng rối loạn ám ảnh cưỡng chế xuất hiện vào thời điểm trưởng thành xã hội và có thể có các dạng sporadic và di truyền. Với điều trị thích hợp (thay đổi hành vi kiên định và điều trị bằng clomipramine), tần suất và cường độ của các dấu hiệu lâm sàng ở hầu hết chó và mèo có thể giảm xuống trên 50%. Sự thành công dường như phụ thuộc vào sự hiểu biết và tuân thủ của khách hàng cũng như kỳ vọng hợp lý rằng OCD không thể được chữa khỏi, nhưng có thể được kiểm soát tốt.","\u003Cjats:title>Abstract\u003C\u002Fjats:title>\u003Cjats:p>\u003Cjats:bold>Objective\u003C\u002Fjats:bold>—To determine clinical features and outcome in dogs and cats with obsessive-compulsive disorder (OCD).\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Design\u003C\u002Fjats:bold>—Retrospective study.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Animals\u003C\u002Fjats:bold>—103 dogs and 23 cats.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Procedures\u003C\u002Fjats:bold>—Records of patients with OCD were analyzed for clinical features, medication used, extent of behavior modification, and outcome.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Results\u003C\u002Fjats:bold>—Most dogs affected with OCD had been obtained from breeders. Male dogs significantly outnumbered females (2:1). Female cats outnumbered male cats by 2:1 in a small sample. Most affected dogs lived in households with 2 or more humans and other dogs or cats, and had some formal training. Client compliance with behavior modification was high. A combination of behavior modification and medication resulted in a large decrease in intensity and frequency of OCD in most animals. Clomipramine was significantly more efficacious for treatment in dogs than was amitriptyline. Only 1 dog and 1 cat were euthanatized because of OCD during the study.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Conclusions and Clinical Relevance\u003C\u002Fjats:bold>—OCD in dogs does not appear to be associated with lack of training, lack of household stimulation, or social confinement. In cats, OCD may be associated with environmental and social stress. Obsessive-compulsive disorder appears at the time of social maturity and may have sporadic and heritable forms. With appropriate treatment (consistent behavior modification and treatment with clomipramine), frequency and intensity of clinical signs in most dogs and cats may decrease by > 50%. Success appears to depend on client understanding and compliance and the reasonable expectation that OCD cannot be cured, but can be well controlled. (\u003Cjats:italic>J Am Vet Med Assoc\u003C\u002Fjats:italic>2002;221:1445–1452)\u003C\u002Fjats:p>",{"EN":2766,"VI":2767},"Clinical features and outcome in dogs and cats with obsessive-compulsive disorder: 126 cases (1989–2000)","Các đặc điểm lâm sàng và kết quả ở chó và mèo mắc chứng rối loạn ám ảnh cưỡng chế: 126 trường hợp (1989–2000)",{"VOID":2769},"12458615",{"VOID":2771},"10.2460\u002Fjavma.2002.221.1445","Auto Verify",[126],[125],"https:\u002F\u002Favmajournals.avma.org\u002Fview\u002Fjournals\u002Fjavma\u002F221\u002F10\u002Fjavma.2002.221.1445.xml",[2777,2796],{"id":2778,"sortIndex":25,"researcher":24,"roles":2779,"affiliations":2780,"properties":2789,"displayName":2793,"givenName":24,"familyName":24},"0415542e-72b5-4301-b1cd-ff3450977451",[],[2781],{"id":2782,"sortIndex":25,"affiliation":2783,"properties":24},"ed74ddee-a370-45bf-b2bc-d942760e1477",{"id":2782,"createTime":24,"updateTime":24,"relativeEntities":2784,"slug":24,"properties":2785,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":2788,"statistic":24},[],{"title":2786},{"EN":2787},"Center for Neurobiology and Genetics-Psychiatry Department, School of Medicine, University of Pennsylvania, Philadelphia, PA 19104, USA.",[],{"orcid":2790,"title":2792,"openalex":2794},{"VOID":2791},"https:\u002F\u002Forcid.org\u002F0000-0001-9202-8294",{"EN":2793},"Karen L. 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tiêu\u003C\u002Fjats:bold>—So sánh các chỉ số đau sau phẫu thuật và hành vi ở chó có và không có sự bổ sung truyền ketamine liều thấp vào quản lý perioperative thông thường.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Thiết kế\u003C\u002Fjats:bold>—Nghiên cứu lâm sàng ngẫu nhiên, mù, tiến cứu.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Động vật\u003C\u002Fjats:bold>—27 chú chó thực hiện phẫu thuật cắt cụt chi trước.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Thủ thuật\u003C\u002Fjats:bold>—Các chú chó được gây mê bằng glycopyrrolate, morphine, propofol, và isoflurane. Mười ba chú chó được điều trị bằng ketamine IV, cụ thể như sau: 0,5 mg\u002Fkg (0,23 mg\u002Flb) tiêm bolus trước phẫu thuật, 10 µg\u002Fkg\u002Fphút (4,5 µg\u002Flb\u002Fphút) trong suốt quá trình phẫu thuật, và 2 µg\u002Fkg\u002Fphút (0,9 µg\u002Flb\u002Fphút) trong 18 giờ sau phẫu thuật. Mười bốn chú chó nhận cùng một thể tích dung dịch muối sinh lý (0,9% NaCl). Tất cả các chú chó nhận một liệu trình fentanyl (1 đến 5 µg\u002Fkg\u002Fgiờ [0,45 đến 2,27 µg\u002Flb\u002Fgiờ]) trong 18 giờ sau phẫu thuật. Các chú chó được đánh giá về các dấu hiệu đau trước phẫu thuật, vào thời điểm extubation, và 1, 2, 3, 4, 12, và 18 giờ sau extubation. Chủ sở hữu đánh giá sự thèm ăn, hoạt động, và độ đau ở vết thương của chó vào các ngày hậu phẫu 2, 3, và 4.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Kết quả\u003C\u002Fjats:bold>—Những chú chó nhận truyền ketamine có điểm đau thấp hơn đáng kể trong 12 và 18 giờ sau phẫu thuật và hoạt động nhiều hơn đáng kể vào ngày hậu phẫu thứ 3 so với những chú chó nhận dung dịch muối sinh lý.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Kết luận và Ý nghĩa lâm sàng\u003C\u002Fjats:bold>—Kết quả cho thấy việc truyền ketamine liều thấp trong thời gian phẫu thuật có thể tăng cường khả năng giảm đau và sự thoải mái trong giai đoạn hậu phẫu ở chó. (\u003Cjats:italic>J Am Vet Med Assoc\u003C\u002Fjats:italic>2002;221:72–75)\u003C\u002Fjats:p>","\u003Cjats:title>Abstract\u003C\u002Fjats:title>\u003Cjats:p>\u003Cjats:bold>Objective\u003C\u002Fjats:bold>—To compare indicators of postoperative pain and behavior in dogs with and without a lowdose ketamine infusion added to usual perioperative management.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Design\u003C\u002Fjats:bold>—Prospective, randomized, blinded clinical study.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Animals\u003C\u002Fjats:bold>—27 dogs undergoing forelimb amputation.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Procedure\u003C\u002Fjats:bold>—Dogs were anesthetized with glycopyrrolate, morphine, propofol, and isoflurane. Thirteen dogs were treated with ketamine IV, as follows: 0.5 mg\u002Fkg (0.23 mg\u002Flb) as a bolus before surgery, 10 µg\u002Fkg\u002Fmin (4.5 µg\u002Flb\u002Fmin) during surgery, and 2 µg\u002Fkg\u002Fmin (0.9 µg\u002Flb\u002Fmin) for 18 hours after surgery. Fourteen dogs received the same volume of saline (0.9% NaCl) solution. All dogs received an infusion of fentanyl (1 to 5 µg\u002Fkg\u002Fh [0.45 to 2.27 µg\u002Flb\u002Fh]) for the first 18 hours after surgery. Dogs were evaluated for signs of pain before surgery, at the time of extubation, and 1, 2, 3, 4, 12, and 18 hours after extubation. Owners evaluated their dogs' appetite, activity, and wound soreness on postoperative days 2, 3, and 4.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Results\u003C\u002Fjats:bold>—Dogs that received ketamine infusions had significantly lower pain scores 12 and 18 hours after surgery and were significantly more active on postoperative day 3 than dogs that received saline solution infusions.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Conclusions and Clinical Relevance\u003C\u002Fjats:bold>—Results suggest that perioperative administration of low doses of ketamine to dogs may augment analgesia and comfort in the postoperative surgical period. 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10.1093\u002Fjn\u002F121.suppl_11.S99",{"doi":3797},"10.1093\u002Fjn\u002F121.suppl_11.S99",{"id":24,"text":3799,"url":24,"identifiers":3800},"Meyer, 1999, Digestibility and compatibility of mixed diets and faecal consistency in different breeds of dog, 46, 155, 10.1046\u002Fj.1439-0442.1999.00201.x",{"doi":3801},"10.1046\u002Fj.1439-0442.1999.00201.x",{"id":24,"text":3803,"url":24,"identifiers":3804},"Axelsson, 2013, The genomic signature of dog domestication reveals adaptation to a starch-rich diet, 495, 360, 10.1038\u002Fnature11837",{"doi":3805},"10.1038\u002Fnature11837",{"id":24,"text":3807,"url":24,"identifiers":3808},"2012",{},{"id":24,"text":3810,"url":24,"identifiers":3811},"Nestle, 2010",{},{"id":24,"text":2348,"url":24,"identifiers":3813},{},{"id":24,"text":2348,"url":24,"identifiers":3815},{},{"id":24,"text":3817,"url":24,"identifiers":3818},"Steel, 1997, Thiamine deficiency in a cat associated with the preservation of “pet meat” with sulphur dioxide, 75, 719, 10.1111\u002Fj.1751-0813.1997.tb12252.x",{"doi":3591},{"id":24,"text":3820,"url":24,"identifiers":3821},"Boermans, 2007, Mycotoxins and the pet food industry: toxicological evidence and risk assessment, 119, 95, 10.1016\u002Fj.ijfoodmicro.2007.07.063",{"doi":3822},"10.1016\u002Fj.ijfoodmicro.2007.07.063",{"id":24,"text":3824,"url":24,"identifiers":3825},"Morita, 1995, Vitamin-D toxicosis in cats: natural outbreak and experimental study, 57, 831, 10.1292\u002Fjvms.57.831",{"doi":3826},"10.1292\u002Fjvms.57.831",{"id":24,"text":3828,"url":24,"identifiers":3829},"Bernard, 2004",{},{"id":24,"text":3831,"url":24,"identifiers":3832},"Dijcker, 2012, Dietary and animal-related factors associate with the rate of urinary oxalate and calcium excretion in dogs and cats, 171, 46, 10.1136\u002Fvr.100293",{"doi":3833},"10.1136\u002Fvr.100293",{"id":24,"text":3835,"url":24,"identifiers":3836},"Crissey, 1997, Use of a raw meat-based diet or a dry kibble diet for sand cats (Felis margarita), 75, 2154, 10.2527\u002F1997.7582154x",{"doi":3837},"10.2527\u002F1997.7582154x",{"id":24,"text":3839,"url":24,"identifiers":3840},"Vester, 2010, Influence of feeding raw or extruded feline diets on nutrient digestibility and nitrogen metabolism of African wildcats (Felis lybica), 29, 676, 10.1002\u002Fzoo.20305",{"doi":3841},"10.1002\u002Fzoo.20305",{"id":24,"text":3843,"url":24,"identifiers":3844},"Kerr, 2012, Apparent total tract energy and macronutrient digestibility and fecal fermentative end-product concentrations of domestic cats fed extruded, raw beef-based, and cooked beef-based diets, 90, 515, 10.2527\u002Fjas.2010-3266",{"doi":3845},"10.2527\u002Fjas.2010-3266",{"id":24,"text":3847,"url":24,"identifiers":3848},"Hendriks, 1999, Heat processing changes the protein quality of canned cat foods as measured by a rat bioassay, 77, 669, 10.2527\u002F1999.773669x",{"doi":3849},"10.2527\u002F1999.773669x",{"id":24,"text":3851,"url":24,"identifiers":3852},"Williams, 2006, Lysine content in canine 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Cats",{},{"id":24,"text":3873,"url":24,"identifiers":3874},"Larsson, 2006, Meat consumption and risk of colorectal cancer: a meta-analysis of prospective studies, 119, 2657, 10.1002\u002Fijc.22170",{"doi":3875},"10.1002\u002Fijc.22170",{"id":24,"text":3877,"url":24,"identifiers":3878},"Sugimura, 2004, Heterocyclic amines: mutagens\u002Fcarcinogens produced during cooking of meat and fish, 95, 290, 10.1111\u002Fj.1349-7006.2004.tb03205.x",{"doi":3879},"10.1111\u002Fj.1349-7006.2004.tb03205.x",{"id":24,"text":3881,"url":24,"identifiers":3882},"Knize, 2003, Mutagenic activity and heterocyclic amine carcinogens in commercial pet foods, 539, 195, 10.1016\u002FS1383-5718(03)00164-5",{"doi":3883},"10.1016\u002FS1383-5718(03)00164-5",{"id":24,"text":3885,"url":24,"identifiers":3886},"Freeman, 2001, Evaluation of raw food diets (Erratum published in J Am Vet Med Assoc 2001; 218:1716), 218, 705",{},{"id":24,"text":3888,"url":24,"identifiers":3889},"Taylor, 2009, Diffuse osteopenia and myelopathy in a puppy fed a diet composed of an organic premix and raw ground beef, 234, 1041, 10.2460\u002Fjavma.234.8.1041",{"doi":3890},"10.2460\u002Fjavma.234.8.1041",{"id":24,"text":3892,"url":24,"identifiers":3893},"Dillitzer, 2011, Intake of minerals, trace elements and vitamins in bone and raw food rations in adults dogs, 106, S53, 10.1017\u002FS0007114511002765",{"doi":3894},"10.1017\u002FS0007114511002765",{"id":24,"text":3896,"url":24,"identifiers":3897},"Larsen, 2012, Evaluation of recipes for home-prepared diets for dogs and cats with chronic kidney disease, 240, 532, 10.2460\u002Fjavma.240.5.532",{"doi":3898},"10.2460\u002Fjavma.240.5.532",{"id":24,"text":3900,"url":24,"identifiers":3901},"Heinze, 2012, Assessment of commercial diets and recipes for home-prepared diets recommended for dogs with cancer, 241, 1453, 10.2460\u002Fjavma.241.11.1453",{"doi":3902},"10.2460\u002Fjavma.241.11.1453",{"id":24,"text":3904,"url":24,"identifiers":3905},"Stockman, 2013, Evaluation of recipes of home-prepared maintenance diets for dogs, 242, 1500, 10.2460\u002Fjavma.242.11.1500",{"doi":3906},"10.2460\u002Fjavma.242.11.1500",{"id":24,"text":3908,"url":24,"identifiers":3909},"KuKanich, 2011, Update on Salmonella spp contamination of pet food, treats, and nutritional products and safe feeding recommendations, 238, 1430, 10.2460\u002Fjavma.238.11.1430",{"doi":3910},"10.2460\u002Fjavma.238.11.1430",{"id":24,"text":3601,"url":24,"identifiers":3912},{"doi":3603},{"id":24,"text":3509,"url":24,"identifiers":3914},{},{"id":24,"text":3916,"url":24,"identifiers":3917},"Weese, 2005, Bacteriological evaluation of commercial canine and feline raw diets, 46, 513",{},{"id":24,"text":3919,"url":24,"identifiers":3920},"Finley, 2008, The occurrence and antimicrobial susceptibility of Salmonellae isolated from commercially available canine raw food diets in three Canadian cities, 55, 462, 10.1111\u002Fj.1863-2378.2008.01147.x",{"doi":3921},"10.1111\u002Fj.1863-2378.2008.01147.x",{"id":24,"text":3923,"url":24,"identifiers":3924},"Lenz, 2009, Perceptions, practices, and consequences associated with foodborne pathogens and the feeding of raw meat to dogs, 50, 637",{},{"id":24,"text":3926,"url":24,"identifiers":3927},"Selmi, 2011, Contaminated commercial dehydrated food as source of multiple Salmonella serotypes outbreak in a municipal kennel in Tuscany, 47, 183",{},{"id":24,"text":3929,"url":24,"identifiers":3930},"Behravesh, 2010, Human Salmonella infections linked to contaminated dry dog and cat food, 2006–2008, 126, 477, 10.1542\u002Fpeds.2009-3273",{"doi":3931},"10.1542\u002Fpeds.2009-3273",{"id":24,"text":3933,"url":24,"identifiers":3934},"Chengappa, 1993, Prevalence of Salmonella in raw meat used in diets of racing Greyhounds, 5, 372, 10.1177\u002F104063879300500312",{"doi":3935},"10.1177\u002F104063879300500312",{"id":24,"text":3536,"url":24,"identifiers":3937},{"doi":3538},{"id":24,"text":3939,"url":24,"identifiers":3940},"Leonard, 2011, Evaluation of pet-related management factors and the risk of Salmonella spp. carriage in pet dogs from volunteer households in Ontario (2005–2006), 58, 140, 10.1111\u002Fj.1863-2378.2009.01320.x",{"doi":3941},"10.1111\u002Fj.1863-2378.2009.01320.x",{"id":24,"text":3943,"url":24,"identifiers":3944},"Morley, 2006, Evaluation of the association between feeding raw meat and Salmonella enterica infections at a Greyhound breeding facility, 228, 1524, 10.2460\u002Fjavma.228.10.1524",{"doi":3626},{"id":24,"text":3946,"url":24,"identifiers":3947},"Cui, 2005, Prevalence and antimicrobial resistance of Campylobacter spp and Salmonella serovars in organic chicken from Maryland retail stores, 71, 4108, 10.1128\u002FAEM.71.7.4108-4111.2005",{"doi":3948},"10.1128\u002FAEM.71.7.4108-4111.2005",{"id":24,"text":3950,"url":24,"identifiers":3951},"Bohaychuk, 2006, Occurrence of pathogens in raw and ready-to-eat meat and poultry products collected from the retail marketplace in Edmonton, Alberta, Canada, 69, 2176, 10.4315\u002F0362-028X-69.9.2176",{"doi":3952},"10.4315\u002F0362-028X-69.9.2176",{"id":24,"text":3954,"url":24,"identifiers":3955},"M'ikanatha, 2010, Multidrug-resistant Salmonella isolates from retail chicken meat compared with human clinical isolates, 7, 929, 10.1089\u002Ffpd.2009.0499",{"doi":3956},"10.1089\u002Ffpd.2009.0499",{"id":24,"text":3958,"url":24,"identifiers":3959},"Lestari, 2009, Prevalence and antimicrobial resistance of Salmonella serovars in conventional and organic chicken from Louisiana retail stores, 72, 1165, 10.4315\u002F0362-028X-72.6.1165",{"doi":3960},"10.4315\u002F0362-028X-72.6.1165",{"id":24,"text":3962,"url":24,"identifiers":3963},"Cook, 2012, Campylobacter, Salmonella, Listeria monocytogenes, verotoxigenic Escherichia coli, and Escherichia coli prevalence, enumeration, and subtypes on retail chicken breasts with and without skin, 75, 34, 10.4315\u002F0362-028X.JFP-11-206",{"doi":3964},"10.4315\u002F0362-028X.JFP-11-206",{"id":24,"text":3966,"url":24,"identifiers":3967},"Zhao, 2002, Occurrence of Salmonella enterica serotype Typhimurium DT104A in retail ground beef, 65, 403, 10.4315\u002F0362-028X-65.2.403",{"doi":3968},"10.4315\u002F0362-028X-65.2.403",{"id":24,"text":3970,"url":24,"identifiers":3971},"Mollenkopf, 2011, Salmonella enterica and Escherichia coli harboring blaCMY in retail beef and pork products, 8, 333, 10.1089\u002Ffpd.2010.0701",{"doi":3972},"10.1089\u002Ffpd.2010.0701",{"id":24,"text":3974,"url":24,"identifiers":3975},"Smielewska-Los, 2002, The influence of feeding and maintenance system on the occurrence of Toxoplasma gondii infections in dogs, 5, 231",{},{"id":24,"text":3977,"url":24,"identifiers":3978},"Lopes, 2008, Serological survey of Toxoplasma gondii infection in domestic cats from northeastern Portugal, 155, 184, 10.1016\u002Fj.vetpar.2008.05.007",{"doi":3979},"10.1016\u002Fj.vetpar.2008.05.007",{"id":24,"text":3981,"url":24,"identifiers":3982},"Dubey, 2005, Prevalence of viable Toxoplasma gondii in beef, chicken, and pork from retail meat stores in the United States: risk assessment to consumers, 91, 1082, 10.1645\u002FGE-683.1",{"doi":3983},"10.1645\u002FGE-683.1",{"id":24,"text":3985,"url":24,"identifiers":3986},"Antolova, 2009, The first finding of Echinococcus multilocularis in dogs in Slovakia: an emerging risk for spreading of infection, 56, 53, 10.1111\u002Fj.1863-2378.2008.01154.x",{"doi":3987},"10.1111\u002Fj.1863-2378.2008.01154.x",{"id":24,"text":3989,"url":24,"identifiers":3990},"Pao, 2009, Comparison of the microbial quality of ground beef and ground beef patties from Internet and local retail markets, 72, 1722, 10.4315\u002F0362-028X-72.8.1722",{"doi":3991},"10.4315\u002F0362-028X-72.8.1722",{"id":24,"text":3993,"url":24,"identifiers":3994},"Rumbeiha, 2011, A review of class I and class II pet food recalls involving chemical contaminants from 1996 to 2008, 7, 60, 10.1007\u002Fs13181-010-0123-5",{"doi":3995},"10.1007\u002Fs13181-010-0123-5",{"id":24,"text":2348,"url":24,"identifiers":3997},{},{"id":24,"text":3999,"url":24,"identifiers":4000},"LaFlamme, 2008, Pet feeding practices of dog and cat owners in the United States and Australia, 232, 687, 10.2460\u002Fjavma.232.5.687",{"doi":3252},{"id":24,"text":4002,"url":24,"identifiers":4003},"Michel, 2005, Nutritional requirements across the species, 43",{},{"id":24,"text":4005,"url":24,"identifiers":4006},"Dressman, 1986, Comparison of canine and human gastrointestinal physiology, 3, 123, 10.1023\u002FA:1016353705970",{"doi":4007},"10.1023\u002FA:1016353705970",{"id":24,"text":4009,"url":24,"identifiers":4010},"Aymerich, 2008, Decontamination technologies for meat products, 78, 114, 10.1016\u002Fj.meatsci.2007.07.007",{"doi":4011},"10.1016\u002Fj.meatsci.2007.07.007",{"id":24,"text":4013,"url":24,"identifiers":4014},"Baert, 2009, The efficacy of preservation methods to inactivate foodborne viruses, 131, 83, 10.1016\u002Fj.ijfoodmicro.2009.03.007",{"doi":4015},"10.1016\u002Fj.ijfoodmicro.2009.03.007",{"id":24,"text":4017,"url":24,"identifiers":4018},"Vanlint, 2012, Emergence and stability of high-pressure resistance in different food-borne pathogens, 78, 3234, 10.1128\u002FAEM.00030-12",{"doi":4019},"10.1128\u002FAEM.00030-12",{"id":24,"text":4021,"url":24,"identifiers":4022},"Rousseau, 2007, Incidence and characterization of esophagitis following esophageal foreign body removal in dogs: 60 cases (1999–2003), 17, 159, 10.1111\u002Fj.1476-4431.2007.00227.x",{"doi":4023},"10.1111\u002Fj.1476-4431.2007.00227.x",{"id":24,"text":4025,"url":24,"identifiers":4026},"Gianella, 2009, Oesophageal and gastric endoscopic 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298",{},{"id":24,"text":4044,"url":24,"identifiers":4045},"Finley, 2006, Human health implications of Salmonella-contaminated natural pet treats and raw pet food, 42, 686, 10.1086\u002F500211",{"doi":4046},"10.1086\u002F500211",{"id":24,"text":4048,"url":24,"identifiers":4049},"Aspinall, 2002, Prevalence of Toxoplasma gondii in commercial meat products as monitored by polymerase chain reaction—food for thought?, 32, 1193, 10.1016\u002FS0020-7519(02)00070-X",{"doi":4050},"10.1016\u002FS0020-7519(02)00070-X",{"id":24,"text":4052,"url":24,"identifiers":4053},"Dorny, 2009, Emerging food-borne parasites, 163, 196, 10.1016\u002Fj.vetpar.2009.05.026",{"doi":4054},"10.1016\u002Fj.vetpar.2009.05.026",{"id":24,"text":4056,"url":24,"identifiers":4057},"Dubey, 2011, High prevalence and genotypes of Toxoplasma gondii isolated from goats from a retail meat store, destined for human consumption in the United States, 41, 827, 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10.1016\u002Fj.prevetmed.2012.01.003",{"doi":4088},"10.1016\u002Fj.prevetmed.2012.01.003",{"id":24,"text":4090,"url":24,"identifiers":4091},"Babcock, 2008, Legal implications of zoonoses for clinical veterinarians, 233, 1556, 10.2460\u002Fjavma.233.10.1556",{"doi":4092},"10.2460\u002Fjavma.233.10.1556",{"id":24,"text":4094,"url":24,"identifiers":4095},"Weese, 2006, Survival of Salmonella Copenhagen in food bowls following contamination with experimentally inoculated raw meat: effects of time, cleaning, and disinfection, 47, 887",{},{"id":24,"text":2348,"url":24,"identifiers":4097},{},{"id":24,"text":4099,"url":24,"identifiers":4100},"Baldwin, 2010, AAHA nutritional assessment guidelines for dogs and cats, 46, 285, 10.5326\u002F0460285",{"doi":4101},"10.5326\u002F0460285",{"id":24,"text":4103,"url":24,"identifiers":4104},"Freeman, 2011, WSAVA Nutritional Assessment Guidelines, 52, 385, 10.1111\u002Fj.1748-5827.2011.01079.x",{"doi":4105},"10.1111\u002Fj.1748-5827.2011.01079.x",{"id":4107,"createTime":4108,"updateTime":4109,"relativeEntities":4110,"slug":4111,"properties":4112,"entityType":965,"verifyStatus":23,"verifyTime":4108,"verifyNote":1754,"languages":4128,"translateLanguages":4129,"viewCount":25,"primaryUrl":4130,"fullTextUrl":24,"authors":4131,"publicationType":970,"publisherRelationship":4187,"citationCount":4238,"citationInfo":4239,"publishDate":4242,"publishYear":4240,"citationAnalyzeStatus":23,"lastCitationAnalyze":24,"indexDatabases":4243,"openAccess":24,"references":4244,"isForceReanalyzing":1737},"881cb8a6-2e06-4588-8c2c-8473c5356048","2025-01-07T20:20:59.820+00:00","2025-01-12T04:36:32.740+00:00",[],"Breeds-of-dogs-involved-in-fatal-human-attacks-in-the-United-States-between-1979-and-1998",{"mag":4113,"keywords":4115,"openalex":4116,"abstract":4118,"title":4121,"pm":4124,"doi":4126},{"VOID":4114},"2159645787",{"VI":2348},{"VOID":4117},"W2159645787",{"VI":4119,"EN":4120},"\u003Cjats:title>Tóm tắt\u003C\u002Fjats:title>\u003Cjats:p>\u003Cjats:bold>Mục tiêu\u003C\u002Fjats:bold>—Tóm tắt các giống chó liên quan đến các cuộc tấn công gây tử vong cho con người trong 20 năm và đánh giá các vấn đề chính sách.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Động vật\u003C\u002Fjats:bold>—Các chó mà giống được báo cáo đã tham gia vào các cuộc tấn công con người giữa năm 1979 và 1998 dẫn đến tử vong liên quan đến cắn chó của con người (DBRF).\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Thủ tục\u003C\u002Fjats:bold>—Dữ liệu về DBRF của con người đã được xác định trước đó cho giai đoạn từ 1979 đến 1996 được kết hợp với DBRF của con người được xác định mới cho năm 1997 và 1998. DBRF của con người được xác định bằng cách tìm kiếm thông tin từ các tài khoản tin tức và sử dụng ngân hàng dữ liệu đăng ký của Hiệp hội Nhân đạo Hoa Kỳ.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Kết quả\u003C\u002Fjats:bold>—Trong năm 1997 và 1998, ít nhất 27 người đã chết do các cuộc tấn công từ cắn chó (18 trong năm 1997 và 9 trong năm 1998). Ít nhất 25 giống chó đã tham gia vào 238 trường hợp DBRF của con người trong 20 năm qua. Các chó loại pit bull và Rottweiler đã liên quan đến hơn một nửa số trường hợp tử vong này. Trong số 227 báo cáo với dữ liệu liên quan, 55 (24%) trường hợp tử vong của con người liên quan đến chó không bị kiểm soát ở ngoài tài sản của chủ, 133 (58%) liên quan đến chó không bị kiểm soát trên tài sản của chủ, 38 (17%) liên quan đến chó bị giữ lại trên tài sản của chủ, và 1 (< 1%) liên quan đến chó bị giữ lại bên ngoài tài sản của chủ.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Kết luận\u003C\u002Fjats:bold>—Mặc dù các cuộc tấn công gây tử vong cho con người có vẻ là một vấn đề liên quan đến giống chó (các chó loại pit bull và Rottweiler), nhưng các giống khác có thể cắn và gây tử vong ở tỷ lệ cao hơn. Do những khó khăn vốn có trong việc xác định giống chó một cách chắc chắn, việc thực thi các quy định cụ thể về giống chó đặt ra các vấn đề về hiến pháp và thực tiễn. Các cuộc tấn công gây tử vong chỉ đại diện cho một tỷ lệ nhỏ trong tổng số các chấn thương do cắn chó đối với con người và do đó, không nên là yếu tố chính dẫn dắt chính sách công liên quan đến chó nguy hiểm. Nhiều lựa chọn thực tiễn khác cho các quy định cụ thể về giống chó tồn tại và có tiềm năng trong việc ngăn ngừa các vụ cắn chó. (\u003Cjats:italic>J Am Vet Med Assoc\u003C\u002Fjats:italic>2000;217:836–840)\u003C\u002Fjats:p>","\u003Cjats:title>Abstract\u003C\u002Fjats:title>\u003Cjats:p>\u003Cjats:bold>Objective\u003C\u002Fjats:bold>—To summarize breeds of dogs involved in fatal human attacks during a 20-year period and to assess policy implications.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Animals\u003C\u002Fjats:bold>—Dogs for which breed was reported involved in attacks on humans between 1979 and 1998 that resulted in human dog bite-related fatalities (DBRF).\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Procedure\u003C\u002Fjats:bold>—Data for human DBRF identified previously for the period of 1979 through 1996 were combined with human DBRF newly identified for 1997 and 1998. Human DBRF were identified by searching news accounts and by use of The Humane Society of the United States' registry databank.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Results\u003C\u002Fjats:bold>—During 1997 and 1998, at least 27 people died of dog bite attacks (18 in 1997 and 9 in 1998). At least 25 breeds of dogs have been involved in 238 human DBRF during the past 20 years. Pit bull-type dogs and Rottweilers were involved in more than half of these deaths. Of 227 reports with relevant data, 55 (24%) human deaths involved unrestrained dogs off their owners' property, 133 (58%) involved unrestrained dogs on their owners' property, 38 (17%) involved restrained dogs on their owners' property, and 1 (< 1%) involved a restrained dog off its owner's property.\u003C\u002Fjats:p>\u003Cjats:p>\u003Cjats:bold>Conclusions\u003C\u002Fjats:bold>—Although fatal attacks on humans appear to be a breed-specific problem (pit bull-type dogs and Rottweilers), other breeds may bite and cause fatalities at higher rates. Because of difficulties inherent in determining a dog's breed with certainty, enforcement of breed-specific ordinances raises constitutional and practical issues. Fatal attacks represent a small proportion of dog bite injuries to humans and, therefore, should not be the primary factor driving public policy concerning dangerous dogs. Many practical alternatives to breed-specific ordinances exist and hold promise for prevention of dog bites. (\u003Cjats:italic>J Am Vet Med Assoc\u003C\u002Fjats:italic>2000;217:836–840)\u003C\u002Fjats:p>",{"EN":4122,"VI":4123},"Breeds of dogs involved in fatal human attacks in the United States between 1979 and 1998","Các giống chó liên quan đến các cuộc tấn công gây tử vong cho con người ở Hoa Kỳ từ năm 1979 đến 1998",{"VOID":4125},"10997153",{"VOID":4127},"10.2460\u002Fjavma.2000.217.836",[126],[125],"https:\u002F\u002Favmajournals.avma.org\u002Fview\u002Fjournals\u002Fjavma\u002F217\u002F6\u002Fjavma.2000.217.836.xml",[4132,4149,4160,4169,4178],{"id":4133,"sortIndex":25,"researcher":24,"roles":4134,"affiliations":4135,"properties":4144,"displayName":4146,"givenName":24,"familyName":24},"bb64feeb-285d-4da4-83e8-8e6457bc016e",[],[4136],{"id":4137,"sortIndex":25,"affiliation":4138,"properties":24},"3c266ea5-c879-45d4-ba0e-87e4614b9387",{"id":4137,"createTime":24,"updateTime":24,"relativeEntities":4139,"slug":24,"properties":4140,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":4143,"statistic":24},[],{"title":4141},{"EN":4142},"US Department of Health and Human Services, Centers for Disease Control and Prevention, Atlanta, GA 30341, USA.",[],{"title":4145,"openalex":4147},{"EN":4146},"Jeffrey J. Sacks",{"VOID":4148},"A5110304340",{"id":4150,"sortIndex":89,"researcher":24,"roles":4151,"affiliations":4152,"properties":4153,"displayName":4157,"givenName":24,"familyName":24},"58f0252b-1626-4bfa-8e99-85509174291f",[],[],{"orcid":4154,"title":4156,"openalex":4158},{"VOID":4155},"https:\u002F\u002Forcid.org\u002F0009-0002-3589-7383",{"EN":4157},"Leslie Sinclair",{"VOID":4159},"A5013895369",{"id":4161,"sortIndex":86,"researcher":24,"roles":4162,"affiliations":4163,"properties":4164,"displayName":4166,"givenName":24,"familyName":24},"baccbc61-0608-4a11-94cb-592c1865d5b8",[],[],{"title":4165,"openalex":4167},{"EN":4166},"J Gilchrist",{"VOID":4168},"A5111991564",{"id":4170,"sortIndex":135,"researcher":24,"roles":4171,"affiliations":4172,"properties":4173,"displayName":4175,"givenName":24,"familyName":24},"0dc2b289-9af6-416b-8e33-fbef1e755cad",[],[],{"title":4174,"openalex":4176},{"EN":4175},"Gail Golab",{"VOID":4177},"A5078181451",{"id":4179,"sortIndex":138,"researcher":24,"roles":4180,"affiliations":4181,"properties":4182,"displayName":4184,"givenName":24,"familyName":24},"5117b405-1b5e-414a-b131-92df59e73275",[],[],{"title":4183,"openalex":4185},{"EN":4184},"R. Lockwood",{"VOID":4186},"A5111977508",{"url":24,"publisher":4188,"properties":4231},{"id":6,"createTime":7,"updateTime":8,"relativeEntities":4189,"slug":10,"properties":4190,"entityType":22,"verifyStatus":23,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":25,"subjectFields":4195,"manageAffiliations":4200,"indexDatabases":4211,"url":83,"thumbnailPath":24,"statistic":4226,"gsStatistic":24,"type":24,"analyzePriority":24},[],{"country":4191,"eissn":4192,"issn":4193,"title":4194},{"VOID":13},{"VOID":15},{"VOID":17},{"EN":19},[4196],{"id":28,"createTime":24,"updateTime":24,"relativeEntities":4197,"label":4198,"description":4199,"parentId":24,"standard":24,"scholarHubFieldId":24},[],{"EN":31},{},[4201,4206],{"id":35,"createTime":24,"updateTime":24,"relativeEntities":4202,"slug":24,"properties":4203,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":4205,"statistic":24},[],{"title":4204},{"EN":39},[],{"id":42,"createTime":24,"updateTime":24,"relativeEntities":4207,"slug":24,"properties":4208,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":4210,"statistic":24},[],{"title":4209},{"EN":46},[],[4212,4219],{"id":50,"indexDatabase":4213,"url":63,"indexYears":24,"academicFieldIds":4218,"indexDatabaseRanking":24},{"id":52,"createTime":24,"updateTime":24,"relativeEntities":4214,"label":4215,"description":4216,"key":59,"publicationTags":4217,"standard":24},[],{"EN":55,"VI":55},{"EN":57,"VI":58},[61,62],[65],{"id":67,"indexDatabase":4220,"url":78,"indexYears":79,"academicFieldIds":4225,"indexDatabaseRanking":82},{"id":69,"createTime":24,"updateTime":24,"relativeEntities":4221,"label":4222,"description":4223,"key":75,"publicationTags":4224,"standard":24},[],{"EN":72,"VI":72},{"EN":72,"VI":74},[77],[81],{"impactFactor":25,"impactFactorByYear":4227,"i10Index":87,"i10IndexLast5Year":25,"totalPublication":87,"totalPublicationByYear":4228,"totalCitation":90,"totalCitationByYear":4229,"totalCitationPerPublication":98,"totalCitationPerPublicationByYear":4230,"hindexLast5Year":87,"hindex":87},{"2013":86,"2014":87},{"2004":89,"2006":89,"2008":89,"2009":89,"2010":89,"2012":89},{"2004":92,"2006":93,"2008":94,"2009":95,"2010":96,"2012":97},{"2004":92,"2006":93,"2008":94,"2009":95,"2010":96,"2012":97},{"issue":4232,"pages":4234,"volume":4236},{"VOID":4233},"6",{"VOID":4235},"836-840",{"VOID":4237},"217",180,{"total":4238,"publishYear":4240,"statisticByYear":4241},2000,{"2012":446,"2013":235,"2014":140,"2015":141,"2016":141,"2017":217,"2018":217,"2019":138,"2020":235,"2021":139,"2022":87,"2023":138,"2024":139},"2000-09-01",[61,82],[4245,4248,4252,4255,4258,4262,4265,4268,4271,4275,4278,4281,4285,4289,4292,4296,4300,4303,4306,4309],{"id":24,"text":4246,"url":24,"identifiers":4247},"10.1001\u002Fjama.262.11.1489",{"doi":4246},{"id":24,"text":4249,"url":24,"identifiers":4250},"Sacks JJ, 1996, Pediatrics, 97, 891, 10.1542\u002Fpeds.97.6.891",{"doi":4251},"10.1542\u002Fpeds.97.6.891",{"id":24,"text":4253,"url":24,"identifiers":4254},"Centers, 1997, Morbid Mortal Weekly Rep, 46, 463",{},{"id":24,"text":4256,"url":24,"identifiers":4257},"Lockwood R, 1988, University of Dayton Law Rev, 13, 267",{},{"id":24,"text":4259,"url":24,"identifiers":4260},"Lockwood R, 1987, Anthrozoos, 1, 2, 10.2752\u002F089279388787058713",{"doi":4261},"10.2752\u002F089279388787058713",{"id":24,"text":4263,"url":24,"identifiers":4264},"NEXIS-LEXIS [onlinedatabase available at http:\u002F\u002Fwww.lexisnexis.com\u002Flncc]. Dayton, Ohio: Lexis-Nexis Group; 1999.",{},{"id":24,"text":4266,"url":24,"identifiers":4267},"American Kennel Club. Dog registration statistics, Jan 1, 1990-Dec 31, 1998. New York: American Kennel Club.",{},{"id":24,"text":4269,"url":24,"identifiers":4270},"10.1016\u002F0001-4575(92)90022-B",{"doi":4269},{"id":24,"text":4272,"url":24,"identifiers":4273},"Sacks JJ, 1996, Injury Prev, 2, 52, 10.1136\u002Fip.2.1.52",{"doi":4274},"10.1136\u002Fip.2.1.52",{"id":24,"text":4276,"url":24,"identifiers":4277},"10.1001\u002Fjama.279.1.51",{"doi":4276},{"id":24,"text":4279,"url":24,"identifiers":4280},"10.1001\u002Fjama.281.3.232",{"doi":4279},{"id":24,"text":4282,"url":24,"identifiers":4283},"Gershman KA, 1994, Pediatrics, 93, 913, 10.1542\u002Fpeds.93.6.913",{"doi":4284},"10.1542\u002Fpeds.93.6.913",{"id":24,"text":4286,"url":24,"identifiers":4287},"Pinckney LE, 1982, Pediatrics., 69, 193, 10.1542\u002Fpeds.69.2.193",{"doi":4288},"10.1542\u002Fpeds.69.2.193",{"id":24,"text":4290,"url":24,"identifiers":4291},"Pollock S. Banning pit bulls in the District of Columbia. Memorandum to the Washington Humane Society from the Arnold and Porter Law Firm. Washington, DC, 1999.",{},{"id":24,"text":4293,"url":24,"identifiers":4294},"Burt MR., 1997, J Am Vet Med Assoc, 210, 1139, 10.2460\u002Fjavma.1997.210.8.1139",{"doi":4295},"10.2460\u002Fjavma.1997.210.8.1139",{"id":24,"text":4297,"url":24,"identifiers":4298},"Wapner M, 2000, J Am Vet Med Assoc, 216, 1552, 10.2460\u002Fjavma.2000.216.1552",{"doi":4299},"10.2460\u002Fjavma.2000.216.1552",{"id":24,"text":4301,"url":24,"identifiers":4302},"Companion animals section and division of higher education programs. Guidelines for regulating dangerous or vicious dogs. Washington, DC: Humane Society of the United States, 1987.",{},{"id":24,"text":4304,"url":24,"identifiers":4305},"Lockwood R, 1992, The Humane Society News, 37, 20",{},{"id":24,"text":4307,"url":24,"identifiers":4308},"Hart BL, 1985, J Am Vet Med Assoc, 186, 1175",{},{"id":24,"text":4310,"url":24,"identifiers":4311},"Hart BL, 1985, J Am Vet Med Assoc, 186, 1181",{},{"id":4313,"createTime":4314,"updateTime":4314,"relativeEntities":4315,"slug":4316,"properties":4317,"entityType":965,"verifyStatus":122,"verifyTime":4314,"verifyNote":2772,"languages":4330,"translateLanguages":24,"viewCount":25,"primaryUrl":4331,"fullTextUrl":24,"authors":4332,"publicationType":970,"publisherRelationship":4458,"citationCount":917,"citationInfo":4508,"publishDate":4511,"publishYear":4509,"citationAnalyzeStatus":23,"lastCitationAnalyze":24,"indexDatabases":4512,"openAccess":24,"references":4513,"isForceReanalyzing":1737},"49f829de-480b-454e-b189-de348566eca8","2025-02-03T20:34:19.548+00:00",[],"Detection-of-Toxoplasma-gondii-like-oocysts-in-cat-feces-and-estimates-of-the-environmental-oocyst-burden",{"openalex":4318,"mag":4320,"abstract":4322,"title":4324,"pm":4326,"doi":4328},{"VOID":4319},"W2009036956",{"VOID":4321},"2009036956",{"EN":4323},"\u003Cjats:title>Abstract\u003C\u002Fjats:title>\n\u003Cjats:p>\u003Cjats:bold>Objective\u003C\u002Fjats:bold>—To estimate the analytic sensitivity of microscopic detection of \u003Cjats:italic>Toxoplasma gondii\u003C\u002Fjats:italic> oocysts and the environmental loading of \u003Cjats:italic>T gondii\u003C\u002Fjats:italic> oocysts on the basis of prevalence of shedding by owned and unowned cats.\u003C\u002Fjats:p>\n\u003Cjats:p>\u003Cjats:bold>Design\u003C\u002Fjats:bold>—Cross-sectional survey.\u003C\u002Fjats:p>\n\u003Cjats:p>\u003Cjats:bold>Sample Population\u003C\u002Fjats:bold>—326 fecal samples from cats.\u003C\u002Fjats:p>\n\u003Cjats:p>\u003Cjats:bold>Procedures\u003C\u002Fjats:bold>—Fecal samples were collected from cat shelters, veterinary clinics, cat-owning households, and outdoor locations and tested via ZnSO\u003Cjats:sub>4\u003C\u002Fjats:sub> fecal flotation.\u003C\u002Fjats:p>\n\u003Cjats:p>\u003Cjats:bold>Results\u003C\u002Fjats:bold>—Only 3 (0.9%) samples of feces from 326 cats in the Morro Bay area of California contained \u003Cjats:italic>T gondii\u003C\u002Fjats:italic>–like oocysts. On the basis of the estimated tonnage of cat feces deposited outdoors in this area, the annual burden in the environment was estimated to be 94 to 4,671 oocysts\u002Fm\u003Cjats:sup>2\u003C\u002Fjats:sup> (9 to 434 oocysts\u002Fft\u003Cjats:sup>2\u003C\u002Fjats:sup>).\u003C\u002Fjats:p>\n\u003Cjats:p>\u003Cjats:bold>Conclusions and Clinical Relevance\u003C\u002Fjats:bold>—Despite the low prevalence and short duration of \u003Cjats:italic>T gondii\u003C\u002Fjats:italic> oocyst shedding by cats detected in the present and former surveys, the sheer numbers of oocysts shed by cats during initial infection could lead to substantial environmental contamination. 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