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287},"Objective: Workers in various industries can be exposed to polycyclic aromatic hydrocarbons (PAHs). The relationship between biomarkers of genotoxic risk, PAH compounds in air (ambient monitoring) and PAH metabolites in urine (internal exposure) were studied in 17 workers exposed to PAHs in a fireproof-material producing plant before and 3 months after the PAH profile was altered in the binding pitch. Methods: Two biomarkers of exposure, specific DNA adducts of (±)-r-7,t-8-dihydroxy-t-9,10-oxy-7,8,9,10-tetrahydrobenzo[a]pyrene (anti-BPDE) and non-specific DNA adduct of 8-oxo-7,8-dihydro-2‘-deoxyguanosine (8-oxodGuo) were determined in white blood cells (WBCs). In addition, DNA strand breaks were analysed in lymphocytes by single-cell gel electrophoresis in a genotoxic risk assessment. Sixteen PAH compounds in air were determined by personal air sampling, and hydroxylated metabolites of phenanthrene, pyrene and naphthalene were determined in urine. Results: After substitution of the binding pitch the concentrations of benzo[a]pyrene in air decreased (P\u003C0.01). No changes could be observed for pyrene, while levels of phenanthrene (P=0.0013) and naphthalene (P=0.0346) in air increased. Consequently, median DNA adduct rates of anti-BPDE decreased after alteration of the production material (from 0.9 to \u003C0.5 adducts\u002F108 nucleotides). No changes in the excretion of 1-hydroxypyrene in urine could be determined, whereas increased levels of 1-, 2+9-, 3- and 4-hydroxyphenanthrene (P\u003C0.0001) and 1-naphthol and 2-naphthol (P=0.0072) were found in urine. In addition, a statistically significant increase in DNA strand break frequencies (P\u003C0.01) and elevated 8-oxodGuo adduct levels (P=0.7819, not statistically significant) were found in the WBCs of exposed workers 3 months after the PAH profile in the binding pitch had been altered. Conclusion: The results presented here show that the increased concentration of naphthalene and\u002For phenanthrene in the air at the work place could induce the formation of DNA strand breaks and alkali-labile sites in WBCs of exposed workers.",{"EN":289,"VI":290},"Genotoxic risk assessment in white blood cells of occupationally exposed workers before and after alteration of the polycyclic aromatic hydrocarbon (PAH) profile in the production material: comparison with PAH air and urinary metabolite levels","Đánh giá nguy cơ độc tính gen trong tế bào bạch cầu của công nhân phơi nhiễm nghề nghiệp trước và sau khi thay đổi thành phần hydrocacbon thơm đa vòng (PAH) trong nguyên liệu sản xuất: so sánh với nồng độ PAH trong không khí và chất chuyển hóa trong nước tiểu",{"VOID":292},"Asami S, Hirano T, Yamaguchi R, Tomioka Y, Itoh H, Kasai H (1996) Increase of a type of oxidative DNA damage, 8-hydroxyguanine, and its repair activity in human leukocytes by cigarette smoking. Cancer Res 56:2546–2549\nBagchi D, Bagchi M, Balmoori J, Vuchetich PJ, Stohs SJ (1998) Induction of oxidative stress and DNA damage by chronic administration of naphthalene to rats. Res Commun Mol Pathol Pharmacol 101:249–257\nBagchi M, Balmoori J, Ye X, Bagchi D, Ray SD, Stohs SJ (2001) Protective effect of melatonin on naphthalene-induced oxidative stress and DNA damage in cultured macrophage J774A.1 cells. Mol Cell Biochem 221:49–55\nBinkova B, Lewtas J, Miskova I, Rossner P, Cerna M, Mrackova G, Peterkova K, Mumford J, Meyer S, Sram R (1996) Biomarker studies in Northern Bohemia. Environ Health Perspect 104 [Suppl 3]:591–597\nBoffetta P, Jourenkova N, Gustavsson P (1997) Cancer risk from occupational and environmental exposure to polycyclic aromatic hydrocarbons. Cancer Causes Control 8:444–472\nBoiteux S (1993) Properties and biological functions of the NTH and FPG proteins of Escherichia coli: two DNA glycosylases that repair oxidative damage in DNA. J Photochem Photobiol B 19:87–96\nCadet J, Berger M, Douki T, Ravanat J-L (1997) Oxidative damage to DNA: formation, measurement and biological significance. Rev Physiol Biochem Pharmacol 31:1–87\nCanova S, Degan P, Peters LD, Livingstone DR, Voltan R, Venier P (1998) Tissue dose, DNA adducts, oxidative DNA damage and CYP1A-immunopositive proteins in mussels exposed to waterborne benzo[a]pyrene. Mutat Res 399:17–30\nCarstensen U, Hou SM, Alexandrie AK, Hogstedt B, Tagesson C, Warholm M, Rannug A, Lambert B, Axmon A, Hagmar L (1999) Influence of genetic polymorphisms of biotransformation enzymes on gene mutations, strand breaks of deoxyribonucleic acid, and micronuclei in mononuclear blood cells and urinary 8-hydroxydeoxyguanosine in potroom workers exposed to polyaromatic hydrocarbons. Scand J Work Environ Health 25:351–360\nCavalieri El, Rogan EG (1995) Central role of radical cations in metabolic activation of polycyclic aromatic hydrocarbons. Xenobiotica 25:677–688\nCollins AR, Dusinska M, Gedik CM, Stetina R (1996) Oxidative damage to DNA: do we have a reliable biomarker? Environ Health Perspect 104 [Suppl 3]:465–469\nConney AH (1982) Induction of microsomal enzymes by foreign chemicals and carcinogenesis by polycyclic aromatic hydrocarbons. G H A Clowes Memorial Lecture. Cancer Res 42:4875–4917\nCostantino JP, Redmond CK, Bearden A (1995) Occupationally related cancer risk among coke oven workers: 30 years of follow-up. J Occup Environ Med 37:597–604\nvan Delft JHM, Baan RA, Roza L (1998) Biological effect of markers for exposure to carcinogenic compound and their relevance for risk assessment. Crit Rev Toxicol 28:477–510\nDuthie SJ, Ma A, Ross MA, Collins AR (1996) Antioxidant supplementation decreases oxidative DNA damage in human lymphocytes. Cancer Res 56:1291–1295\nFrenkel K, Donahue JM, Banjeree S (1988) Benzo[a]pyrene-induced oxidative DNA damage: a possible mechanism for promotion by complete carcinogens. In: Cerutti P, Firdovich I, McCord J (eds) Oxy-radicals in molecular biology pathology. UCLA symposia on molecular and cellular biology, vol 82. Liss, New York, pp 509–524\nGedik CM, Wood SG, Collins AR (1998) Measuring oxidative damage to DNA; HPLC and the comet assay compared. Free Radic Res 29:609–615\nGelboin HV (1980) Benzo[a]pyrene metabolism, activation and carcinogenesis: role and regulation of mixed function oxidases and related enzymes. Physiol Rev 60:1007–1166\nGrollman AP, Moriya M (1993) Mutagenesis by 8-oxoguanine: an enemy within. Trends Genet 9:246–249\nHalliwell B, Gutteridge JMC (1999) Free radicals in biology and medicine. Oxford University Press, Oxford\nHeudorf U, Angerer J (2001) Internal exposure to PAHs of children and adults living in homes with parquet flooring containing high levels of PAHs in the parquet glue. Int Arch Occup Environ Health 74:91–101\nInternational Agency for Research on Cancer (1987) Overall evaluations of carcinogenicity. An updating of IARC monographs on the evaluation of carcinogenic risks to humans. IARC Scientific Publications, nos. 1 to 42 [Suppl 7], IARC, Lyon\nKasai H (1997) Analysis of a form of oxidative DNA damage, 8-hydroxy-2′-deoxyguanosine, as a marker of cellular oxidative stress during carcinogenesis. Mutat Res 387:147–163\nLintelmann J, Angerer J (1999) PAH metabolites. In: Angerer J, Schaller K-H (eds) Analyses of hazardous substances in biological materials, vol 6. Wiley-VCH, Weinheim, pp 163–187\nMarczynski B, Rozynek P, Elliehausen HJ, Korn M, Baur X (1997) Detection of 8-hydroxydeoxyguanosine, a marker of oxidative DNA damage, in white blood cells of workers occupationally exposed to styrene. Arch Toxicol 71:496–500\nMarczynski B, Rihs HP, Rossbach B, Holzer J, Angerer J, Scherenberg M, Hoffmann G, Bräuning T, Wilhelm M (2002) Analysis of 8-oxo-7,8-dihydro-2′-deoxyguanosine and DNA strand breaks in white blood cells of occupationally exposed workers: comparison with ambient monitoring of urinary metabolites and enzyme polymorphisms. Carcinogenesis 23:273–281\nNational Toxicology Program (2000) Technical report on the toxicology and carcinogenesis studies of naphthalene (CAS No. 91-20-3) in F344\u002FN rats (inhalation studies). DHHS, PHS, Technical Report Series 500, Rockville\nNIOSH (1994) Polynuclear aromatic hydrocarbons by HPLC. In: Cassinelli ME, O’Conner PE (eds) NIOSH manual of analytical methods (NMAM), 4th edn. National Institute for Occupational Safety and Health, Washington\nOhnishi S, Kawanishi S (2002) Double base lesions of DNA by a metabolite of carcinogenic benzo[α]pyrene. Biochem Biophys Res Commun 290:778–782\nÖstling O, Johanson KJ (1984) Microelectrophoretic study of radiation-induced DNA damages in individual mammalian cells. Biochem Biophys Res Commun 123:291–298\nPavanello S, Gabbani G, Mastrangelo G, Brugnone F, Maccacaro G, Clonfero E (1999) Influence of GSTM1 genotypes and anti–BPDE-DNA adduct levels in mononuclear white blood cells of humans exposed to PAH. Int Arch Occup Environ Health 72:238–246\nPenning TM, Burczynski ME, Hung C-F, McCoull KD, Palackal NT, Tsuruda LS (1999) Dihydrodiol dehydrogenases and polycyclic aromatic hydrocarbon activation: generation of reactive and redox-active oquinones. Chem Res Toxicol 12:1–18\nPflaum M, Will O, Epe B (1997) Determination of steady-state levels of oxidative DNA base modifications in mammalian cells by means of repair endonucleases. Carcinogenesis 18:2225–2231\nPopp W, Vahrenholz C, Schell C, Grimmer G, Dettbarn G, Kraus R, Brauksiepe A, Schmeling B, Gutzeit T, von Bulow J, Norpoth K (1997) DNA single strand breakage, DNA adducts, and sister chromatid exchange in lymphocytes and phenanthrene and pyrene metabolites in urine of coke oven workers. Occup Environ Med 54:176–183\nPouget J-P, Ravanat J-L, Douki T, Richard M-J, Cadet J (1999) Measurement of DNA base damage in cells exposed to low doses of γ-radiation: comparison between the HPLC-EC and comet assays. Int J Radiat Biol 75:51–58\nPouget J-P, Douki T, Richard M-J, Cadet J (2000) DNA damage induced in cells by γ and UVA radiation as measured by HPLC\u002FGC-MS and HPLC-EC and comet assay. Chem Res Toxicol 13:541–549\nPreuss R, Angerer J (2004) Simultaneous determination of 1- and 2-naphthol in human urine using on-line clean-up column-switching liquid chromatography-fluorescence detection. J Chromatogr B 801:307–316\nPreuss R, Angerer J, Drexler H (2003): Naphthalene— an environmental and occupational toxicant. Int Arch Occup Environ Health 76:556–576\nPryor WA (1997) Cigarette smoke radicals and the role of free radicals in chemical carcinogenicity. Environ Health Perspect 105 [Suppl 4]:875–882\nRojas M, Alexandrov K, Cascorbi I, Brockmoller J, Likhachev A, Pozharisski K, Bouvier G, Auburtin G, Mayer L, Kopp-Schneider A, Roots I, Bartsch H (1998) High benzo[a]pyrene diol epoxide DNA adduct levels in lung and blood cells from subjects with combined CYP1A1 MspI\u002FMsp\u002FI-GSTM1*0\u002F*0 genotypes. Pharmacogenetics 8:109–118\nSayer JM, Chadha A, Agarwal SK, Yeh HJC, Yagi H, Jerina DM (1991) Covalent nucleoside adducts of benzo[a]pyrene 7,8-diol 9,10-epoxides: structural reinvestigation and characterization of a novel adenosine adduct on the ribose moiety. J Org Chem 56:20–29\nScherer G, Meger-Kossien I, Angerer J, Knecht U (2001) Cotinine. In: Angerer J, Schaller K-H (eds) Analyses of hazardous substances in biological materials, vol. 7. Wiley-VCH, Weinheim, pp 171–189\nSingh NP, McCoy MT, Tice RR, Schneider EL (1988) A simple technique for quantitation of low levels of DNA damage in individual cells. Exp Cell Res 175:184–191\nvon Sonntag C (1987) The chemical basis of radiation biology. Taylor and Francis, New York, pp 117–166, 221–294\nStohs SJ, Ohia S, Bagchi D (2002) Naphthalene toxicity and antioxidant nutrients. Toxicology 180:97–105\nTaussky HH (1954) A micro-colorimetric determination of creatinine in urine by the Jaffey’s reaction. J Biol Chem 208:853–861\nWattenberg LW (1980) Inhibition of chemical carcinogenesis by antioxidants. In: Slaga TJ (ed) Carcinogenesis: modifiers of chemical carcinogenesis, vol 5. Raven Press, New York, pp 85–98\nYu D, Berlin JA, Penning TM, Field J (2002) Reactive oxygen species generated by PAH oquinones cause change-in-function mutations in p53. Chem Res Toxicol 15:832–842",{"VOID":294},"10.1007\u002Fs00420-004-0567-5","PUBLICATION",[297],"VI","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs00420-004-0567-5",[300,316,331,344,358,373,386,402],{"id":301,"sortIndex":23,"researcher":22,"roles":302,"affiliations":304,"properties":313,"displayName":315,"givenName":22,"familyName":22},"7587d847-2c77-4d88-9a88-92b6df550c96",[303],"AUTHOR",[305],{"id":306,"sortIndex":23,"affiliation":307,"properties":22},"3b40e9c4-e734-46f1-b94d-1ea5a9126d2e",{"id":306,"createTime":22,"updateTime":22,"relativeEntities":308,"slug":22,"properties":309,"entityType":22,"verifyStatus":22,"verifyTime":22,"verifyNote":22,"languages":22,"translateLanguages":22,"viewCount":22,"url":22,"parentIds":312,"statistic":22},[],{"title":310},{"VI":311},"Research Institute of Occupational Medicine, Ruhr University of Bochum, Bochum, Germany",[],{"title":314},{"VI":315},"B. 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exposure limits (OEL) for nitrogen oxides (NO, NO2) and diesel exhaust (EC-DPM) were reassessed by the German authorities in 2016\u002F2017. We performed a clinical cross-sectional study among salt and potash underground workers exposed to these substances at relatively high levels to examine possible indicators of acute effects on workers’ health. We measured post- versus pre-shift differences in cardiovascular, inflammatory, immune, and respiratory effect biomarkers and assessed their associations with personal exposures measured during the same shift. We also compared post- versus pre-shift differences in biomarker levels between exposure groups defined based on work site and job type. None of the above-ground workers exceeded the OEL for NO2 and only 5% exceeded the OEL for EC-DPM exposure. Among underground workers, 33% of miners and 7% underground maintenance workers exceeded the OEL for NO2; the OEL for EC-DPM was exceeded by 56% of miners and 17% of maintenance workers. Some effect biomarkers (thrombocytes, neutrophils, MPO, TNF-α, IgE, FeNO) showed statistically significant differences between pre- versus post-shift measurements; however, there were no consistent associations between pre- and post-shift differences and exposure group or personal exposure measurements during the shift. We did not find evidence of associations between workplace exposure to NO, NO2 or EC-DPM and clinically relevant indicators of acute cardiovascular, inflammatory and immune, or respiratory effects among salt and potash underground workers in Germany.",{"EN":479},"Underground salt and potash workers exposed to nitrogen oxides and diesel exhaust: assessment of specific effect biomarkers",{"VOID":481},"Ausschuss für Gefahrstoffe (BAuA) (2021) Technische Regel für Gefahrstoffe: TRGS 900 Arbeitsplatzgrenzwerte. BArBl, Heft 1\u002F2006, 41–55. Last update 02.07.2021. https:\u002F\u002Fwww.baua.de\u002FDE\u002FAngebote\u002FRechtstexte-und-Technische-Regeln\u002FRegelwerk\u002FTRGS\u002FTRGS-900.html\nBerhane K, Zhang Y, Salam MT, Eckel SP, Linn WS, Rappaport EB, Bastain TM, Lurmann F, Gilliland FD (2014) Longitudinal effects of air pollution on exhaled nitric oxide: the children’s health study. Occup Environ Med 71(7):507–513. https:\u002F\u002Fdoi.org\u002F10.1136\u002Foemed-2013-101874\nBhatt SP, Balte PP, Schwartz JE, Cassano PA, Couper D, Jacobs DRJ, Kalhan R, O’Connor GT, Yende S, Sanders JL, Umans JG, Dransfield MT, Chaves PH, White WB, Oelsner EC (2019) Discriminative accuracy of FEV1:FVC thresholds for COPD-related hospitalization and mortality. J Am Med Assoc 321(24):2438–2447. https:\u002F\u002Fdoi.org\u002F10.1001\u002Fjama.2019.7233\nBudkowska M, Lebiecka A, Marcinowska Z, Woźniak J, Jastrzębska M, Dołęgowska B (2019) The circadian rhythm of selected parameters of the hemostasis system in healthy people. Thromb Res 182:79–88. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.thromres.2019.08.015\nCooper BG, Stocks J, Hal GL, Culver B, Steenbruggen I, Carter KW, Thompson BR, Graham BL, Miller MR, Ruppel G, Henderson J, Fragoso CAV, Stanojevic S (2017) The Global Lung Function Initiative (GLI) Network: bringing the world’s respiratory reference values together. Breathe 13(3):e56–e64. https:\u002F\u002Fdoi.org\u002F10.1183\u002F20734735.012717\nDahmann D, Monz C, Sönksen H (2007) Exposure assessment in German potash mining. Int Arch Occup Environ Health 81(1):95–107. https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs00420-007-0194-z\nDawson AA, Ogston D (1969) Exercise-induced thrombocytosis. Acta Haematol 42(4):241–246. https:\u002F\u002Fdoi.org\u002F10.1159\u002F000208784\nDweik RA, Boggs PB, Erzurum SC, Irvin CG, Leigh MW, Lundberg JO, Olin A-C, Plummer AL, Taylor DR (2011) An official ATS clinical practice guideline: interpretation of exhaled nitric oxide levels (FeNO) for clinical applications. Am J Respir Crit Care Med 184(5):602–615. https:\u002F\u002Fdoi.org\u002F10.1164\u002Frccm.9120-11ST\nEpstein TG, Kesavalu B, Bernstein CK, Ryan PH, Bernstein JA, Zimmermann N, Lummus Z, Villareal MS, Smith AM, Lenz PH, Bernstein DI (2013) Chronic traffic pollution exposure is associated with eosinophilic, but not neutrophilic inflammation in older adult asthmatics. J Asthma 50(9):983–989. https:\u002F\u002Fdoi.org\u002F10.3109\u002F02770903.2013.832293\nHennig F, Fuks K, Moebus S, Weinmayr G, Memmesheimer M, Jakobs H, Bröcker-Preuss M, Führer-Sakel D, Möhlenkamp S, Erbel R, Jöckel KH, Hoffmann B (2014) Association between source-specific particulate matter air pollution and hs-CRP: local traffic and industrial emissions. Environ Health Perspect 122(7):703–710. https:\u002F\u002Fdoi.org\u002F10.1289\u002Fehp.1307081\nKübler G, Triebel R, Knappe M (2016) Neue Anforderungen aus der Grenzwertdiskussion zu Stickoxide NOx. Kali & Steinsalz 2:7–17\nLotz G, Kersten N (2012) Dosis-wirkungsbeziehungen von exposition und lungenfunktion bei kalibergarbeitern – vergleichende auswertung einer längsschnittstudie im linearen und gemischten modell. Zentralblatt Für Arbeitsmedizin, Arbeitsschutz Und Ergonomie 62(6):304–318\nLotz G, Plitzko S, Gierke E, Tittelbach U, Kersten N, Schneider WD (2008) Dose-response relationships between occupational exposure to potash, diesel exhaust and nitrogen oxides and lung function: Cross—sectional and longitudinal study in two salt mines. Int Arch Occup Environ Health 81(8):1003–1019. https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs00420-007-0294-9\nLotz A, Kendzia B, Gawrych K, Lehnert M, Brüning T, Pesch B (2013) Statistical methods for the analysis of left-censored variables. GMS Med Inform Biom Epidemiol 9(2):1–7\nLotz G, Backé E, Gierke E, Kersten N, Säverin R, Schneider WD, Thürmer H, Tittelbach U (1998) Studie zur Wirkung von Salzstäuben auf das Atmungssystem. In: Schriftenreihe der Bundesanstalt für Arbeitsschutz und Arbeitsmedizin: Forschungsbericht, Fb 791, 1st edn., Wirtschaftsverlag NW Verlag für neue Wissenschaft GmbH, pp 1–112\nLotz G (2006) Längsschnittstudie über Dosis-Wirkungs-Beziehungen bei Belastungen durch Salzstaub, Dieselmotoremissionen und Stickoxide in 2 Kalibergwerken. research project F5130, BAUA, Germany\nLoxham M, Nieuwenhuijsen MJ (2019) Health effects of particulate matter air pollution in underground railway systems — a critical review of the evidence. Part Fibre Toxicol 16(1):12. https:\u002F\u002Fdoi.org\u002F10.1186\u002Fs12989-019-0296-2\nPoursafa P, Kelishadi R (2010) Air pollution, platelet activation and atherosclerosis. Inflamm Allergy 9(5):387–392. https:\u002F\u002Fdoi.org\u002F10.2174\u002F187152810793937982\nQuanjer PH, Stanojevic S, Cole TJ, Baur X, Hall GL, Culver BH, Enright PL, Hankinson JL, Ip MSM, Zheng J, Stocks J (2012) Multi-ethnic reference values for spirometry for the 3–95-yr age range: the global lung function 2012 equations. Eur Respir J 40(6):1324–1343. https:\u002F\u002Fdoi.org\u002F10.1183\u002F09031936.00080312\nRichter DC, Joubert JR, Nell H, Schuurmans MM, Irusen EM (2008) Diagnostic value of post-bronchodilator pulmonary function testing to distinguish between stable, moderate to severe COPD and asthma. Int J Chronic Obstruct Pulm Dis 3(4):693–699. https:\u002F\u002Fdoi.org\u002F10.2147\u002Fcopd.s948\nRiedl MA, Díaz-Sánchez D, Linn WS, Gong H, Clark KW, Effros RM, Miller JW, Cocker DR, Berhane KT (2012) Allergic inflammation in the human lower respiratory tract affected by exposure to diesel exhaust. Research Report 165. Health Effects Institute, Boston\nRückerl R, Hampel R, Breitner S, Cyrys J, Kraus U, Carter J, Dailey L, Devlin RB, Diaz-Sanchez D, Koenig W, Phipps R, Silbajoris R, Soentgen J, Soukup J, Peters A, Schneider A (2014) Associations between ambient air pollution and blood markers of inflammation and coagulation\u002Ffibrinolysis in susceptible populations. Environ Int 70:32–49. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.envint.2014.05.013\nSchulz H, Karrasch S, Bölke G, Cyrys J, Hornberg C, Pickford R, Schneider A, Witt C, Hoffmann B (2018) Atmen: Luftschadstoffe und Gesundheit. Gesellschaft für Pneumologie und Beatmungsmedizin e.V., Berlin. ISBN: 978-3-9817734-4-6\nScientific Committee on Occupational Exposure Limits, (European Commission) (2014a) Recommendation from the Scientific Committee on Occupational Exposure Limits for nitrogen dioxide. SCOEL\u002FSUM\u002F53, 1–17. https:\u002F\u002Fec.europa.eu\u002Fsocial\u002FBlobServlet?docId=12431&langId=en. Accessed 5 Jan 2015\nScientific Committee on Occupational Exposure Limits, (European Commission) (2014b) Recommendation from the Scientific Committee on Occupational Exposure Limits for nitrogen monoxide. SCOEL\u002FSUM\u002F89, 1–14. https:\u002F\u002Fec.europa.eu\u002Fsocial\u002FBlobServlet?docId=3803&langId=en. Accessed 5 Jan 2015\nSproston NR, Ashworth JJ (2018) Role of C-reactive protein at sites of inflammation and infection. Front Immunol 9:754. https:\u002F\u002Fdoi.org\u002F10.3389\u002Ffimmu.2018.00754\nSteenhof M, Janssen NAH, Strak M, Hoek G, Gosens I, Mudway IS, Kelly FJ, Harrison RM, Pieters RHH, Cassee FR, Brunekreef B (2014) Air pollution exposure affects circulating white blood cell counts in healthy subjects: the role of particle composition, oxidative potential and gaseous pollutants — the RAPTES project. 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In dem von uns zusammengestellten Material ist die Tuberkulose bei Quarzstaubexponierten aus dem Gießereibetrieb deutlich häufiger als bei Personen an nichtexponierten Arbeitsplätzen. 2. Die Altersverteilung der Tuberkulosen ist bei Exponierten und Nichtexponierten verschieden: die Tuberkulose nach dem 45. Altersjahr ist bei Exponierten deutlich häufiger als bei Nichtexponierten, im Sinne der „Zusatztuberkulose“ von Redeker. 3. Im Hinblick auf Schwere und Verlaufsform der Tuberkulose ergeben unsere Untersuchungen keinen wesentlichen Unterschied zwischen Exponierten und Nichtexponierten. 4. Ungefähr die Hälfte unserer Tuberkulosefälle bei Quarzexponierten tritt nach einer Expositionsdauer von weniger als 10 Jahren auf. Die durchschnittliche Quarzexposition unserer Tuberkulosefälle beträgt 14,8 Jahre. 5. Unsere Befunde unterstützen die Annahme, daß bereits geringfügige Quarzreaktionen der Lunge ohne manifeste Silikoseentwicklung die Disposition zu Tuberkulose steigern.",{"EN":1453},"Studien zur Beziehung zwischen Gießerei-Silikose und Tuberkulose",{"VOID":1455},"[\"5775488374986511489\"]",{"VOID":1457},"Bertschi, E., u. E. Stiefel: Silikose in einer Großgießerei. Schweiz. med. Wschr. 80, 1163–1166 (1950); 85, 1114–1117 (1955).\nDi Biasi, W.: Bericht über die Silikose-Arbeitstagung. Bochum 1951.\nHögger, E.: Silikosegefährdete Berufsgruppen. In Handbuch für innere Medizin, Bd. IV, S. 849–869. Berlin: Springer 1956.\nLanza, A. J., and R. J. Vane: The prevalence of silicosis in the general population and its effects upon the incidence of tuberculosis. Amer. Rev. Tuberc. 29, 8–16 (1934).\nRedeker, P., u. O. Walter: Entstehung und Entwicklung der Lungenschwindsucht der Erwachsenen. Leipzig: Curt Kabitsch 1929.\nReichmann, V.: Kurzer Überblick über den Stand der Silikoseforschung nebst einem Beitrag über die röntgenologischen und klinischen Beziehungen der Silikose zur Tuberkulose. Beitr. Silikose-Forsch. H. 1 (1949).\nReinl, W., W. Greinert u. W. Rascher: Die Silikosegefahr in den Gießereien. „Gießerei“ H. 13. 347–348 (1955).\nRüttner, J. R.: Die Silikoanthrakose der Gießer. Vjschr. naturf. Ges. Zürich 95, Beih. 2\u002F3, 73–98 (1950).\nFoundry worker's pneumoconiosis in Switzerland (Anthracosilicosis). A. M. A. Arch. Indust. Hyg. 9, 297–305 (1954).\nSchmid, H. J.: Die Silikose im Schaffhauser Industriegebiet. Mitt. naturforsch. Ges. Schaffhausen 18 (1943).\nDie Klinik der Silikose. In Handbuch der inneren Medizin, Bd. IV\u002F3, Berlin-Göttingen-Heidelberg: Springer 1956.\nTeleky, L.: Sterblichkeit und Krankheitsstatistik der Schleifer Solingens, Cronebergs und Remscheids. Arbeit u. Gesundheit H. 9, 31–43 (1928).\nUehlinger, E.: Pathologische Anatomie und experimentelle Pathologie der Stauberkrankungen. In Handbuch der inneren Medizin, Bd. IV, S. 739–750. Berlin-Göttingen-Heidelberg: Springer 1956.\nUehlinger, F.: Untersuchungen über die Silikose in einem Großbetrieb der Metallindustrie. Diss. Zürich 1949.\nWorth, G., u. E. Schiller: Pneumokoniosen. 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The geometric mean concentration of urinary 1-OHP among nearby group (0.048 ± 1.878 μmol\u002Fmol creatinine, GM ± GSD) was approximately 1.3 times higher than that among remote group (0.036 ± 2.425 μmol\u002Fmol creatinine, GM ± GSD), and using multiple regression techniques, the difference was significant (P \u003C 0.0001) after adjusting for confounding variables. When different periods before the sampling of urine were examined, PM10 averages over 2 days, 3 days, and 1 week prior to urine sampling showed significant associations with urinary 1-OHP levels. 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Br J Ind Med 49(11):761–776",{"doi":1145},{"id":1141,"text":1733,"url":1143,"identifiers":1734},"Buckley TJ, Lioy PJ (1992) An examination of the time course from human dietary exposure to polycyclic aromatic hydrocarbons to urinary elimination of 1-hydroxypyrene. Br J Ind Med 92:113–124",{"doi":1145},{"id":1141,"text":1736,"url":1143,"identifiers":1737},"Castaño-Vinyals G, D’Errico A, Malats N et al (2004) Biomarkers of exposure to polycyclic aromatic hydrocarbons from environmental air pollution. Occup Environ Med 61:1–9",{"doi":1145},{"id":22,"text":1739,"url":22,"identifiers":1740},"Chung JC, Callahan PJ, Lyu CW et al (1999) Polycyclic aromatic hydrocarbon exposures of children in low-income families. J Expos Anal Environ Epidemiol 1:193–225",{},{"id":1141,"text":1742,"url":1143,"identifiers":1743},"Dusseldorp A, Kruize H, Brunekreef B et al (1995) Associations of PM10 and airborne iron with respiratory health of adults living near a steel factory. Am J Respir Crit Care Med 152:1932–1939",{"doi":1145},{"id":22,"text":1745,"url":22,"identifiers":1746},"EPA (1995) Preliminary study of the iron and steel category: effluent limitations guidelines and standards",{},{"id":1748,"text":1749,"url":1750,"identifiers":1751},"f04a8ba3-bf56-48e8-b41e-803c9cb7c551","Fiala Z, Vyskocil A, Krajak V et al (2001) Environmental exposure of small children to polycyclic aromatic hydrocarbons. Int Arch Occup Environ Health 74:411–420","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs004200100239",{"doi":1752},"10.1007\u002Fs004200100239",{"id":1141,"text":1754,"url":1143,"identifiers":1755},"Gündel J, Mannschreck C, Büttner K et al (1996) Urinary levels of 1-hydroxypyrene, 1-, 2-, 3- and 4-hydroxyphenanthrene in females living in an industrial area of Germany. 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Toxicol Lett 72(1–3):205–211",{"doi":1145},{"id":1784,"text":1785,"url":1786,"identifiers":1787},"69ef1a78-e170-4ed3-9e31-b6cc5b768975","Jongeneelen FJ (1997) Methods for routine biological monitoring of carcinogenic PAH-mixtures. Sci Total Environ 199:141–149","https:\u002F\u002Fwww.sciencedirect.com\u002Fscience\u002Farticle\u002Fpii\u002FS0048969797000648",{"doi":1788},"10.1016\u002Fs0048-9697(97)00064-8",{"id":1141,"text":1790,"url":1143,"identifiers":1791},"Jongeneelen FJ, Bos RP, Anzion RB et al (1986) Biological monitoring of polycyclic aromatic hydrocarbons Metabolites in urine. Scand J Work Environ Health 12(2):137–143",{"doi":1145},{"id":1793,"text":1794,"url":1795,"identifiers":1796},"17d1286a-d0f3-4bdb-b8cd-435317435a74","Jongeneelen FJ, Anzion RBM, Henderson PTh (1987) Determination of hydroxylated metabolites of polycyclic aromatic hydrocarbons in urine. J Chromatogr 413:227–232","https:\u002F\u002Fwww.sciencedirect.com\u002Fscience\u002Farticle\u002Fpii\u002F037843478780230X",{"doi":1797},"10.1016\u002F0378-4347(87)80230-x",{"id":1141,"text":1799,"url":1143,"identifiers":1800},"Jongeneelen FJ, van Leeuwen FE, Oosterink S et al (1990) Ambient and biological monitoring of coke oven workers: determinants of the internal dose of polycyclic aromatic hydrocarbons. Br J Ind Med 47(7):454–461",{"doi":1145},{"id":1141,"text":1802,"url":1143,"identifiers":1803},"Kang D, Rothman N, Poirier MC (1995) Interindividual differences in the concentration of 1-hydroxypyrene-glucuronide in urine and polycyclic aromatic hydrocarbon-DNA adducts in peripheral white blood cells after charbroiled beef consumption. Carcinogenesis 16:1079–1085",{"doi":1145},{"id":1141,"text":1805,"url":1143,"identifiers":1806},"Kang JW, Cho SH, Kim H et al (2002) Correlation of urinary 1-hydroxypyrene and 2-naphthol with total suspended particulates in ambient air in municipal middle-school students in Korea. Arch Environ Health 57(4):377–382",{"doi":1145},{"id":22,"text":1808,"url":1809,"identifiers":1810},"Kang JW, Lee CH, Rho SI et al (2004) Exposure assessment of polycyclic aromatic hydrocarbons via food by PAH biomarkers. The annual report of KFDA 8:2603–2604 http:\u002F\u002Fwww.kfda.go.kr","http:\u002F\u002Fwww.kfda.go.kr",{},{"id":22,"text":1812,"url":22,"identifiers":1813},"Kim H, Lim HS, Kang JW et al (1999) Effect of occupation, life style and genetic polymorphism of CYP1A1, GSTM1, and GSTT1 on urinary 1-hydroxypyrene and 2-naphthol concentration. Korean J Occup Environ Med 11(4):546–556",{},{"id":1141,"text":1815,"url":1143,"identifiers":1816},"Liberti L, Notarnicola M, Primerano R et al (2006) Air pollution from a large steel factory: polycyclic aromatic hydrocarbon emissions from coke-oven batteries. J Air Waste Manag Assoc 56(3):255–260",{"doi":1145},{"id":22,"text":1818,"url":22,"identifiers":1819},"Lloyd JW (1971) Long term mortality study of steel workers: V. respiratory cancer in coke plant workers. J Occup Med 13(2):53–68",{},{"id":22,"text":1821,"url":22,"identifiers":1822},"Marcias ES, Husar RB (1976) A review of particulate atmospheric mass measurement via the beta attenuation technique. In: Liu BYH (ed) Fine particles. Academic, New York",{},{"id":22,"text":1824,"url":1825,"identifiers":1826},"Ministry of Construction and transportation, Republic of Korea (2004) Statistical yearbook of traffic volume 2004. Ministry of Construction and Transportation, Republic of Korea http:\u002F\u002Fwww.moct.go.kr","http:\u002F\u002Fwww.moct.go.kr",{},{"id":1141,"text":1828,"url":1143,"identifiers":1829},"Mucha AP, Hryhorczuk D, Serdyuk A et al (2006) Urinary 1-hydroxypyrene as a biomarker of PAH exposure in 3-year-old Ukrainian Children. Environ Health Perspect 114:603–609",{"doi":1145},{"id":22,"text":1831,"url":22,"identifiers":1832},"Nelson D, Scheff PA, Keil C (1991) Characterization of volatile organic compounds contained in coke plant emissions. In: The 84th annual meeting & exhibition of air & waste management association, vol 6, Vancouver, pp 79–91",{},{"id":1141,"text":1834,"url":1143,"identifiers":1835},"Pless-Mulloli T, Phillimore P, Moffatt S et al (1998) Lung cancer, proximity to industry, and poverty in northeast England. Environ Health Perspect 106(4):189–196",{"doi":1145},{"id":1141,"text":1837,"url":1143,"identifiers":1838},"Pope CA III (1989) Respiratory disease associated with community air pollution and a steel mill, Utah Valley. Am J Public Health 79(5):623–628",{"doi":1145},{"id":1141,"text":1840,"url":1143,"identifiers":1841},"Rodriguez V, Tardon A, Kogevinas M et al (2000) Lung cancer risk in iron and steel foundry workers: a nested case control study in Asturias, Spain. Am J Ind Med 38(6):644–650",{"doi":1145},{"id":1843,"text":1844,"url":1845,"identifiers":1846},"2adc1caf-8137-48d4-85b7-e024e91aae48","Strickland P, Kang D (1999) Urinary 1-hydroxypyrene and other PAH metabolites as biomarkers of exposure to environmental PAH in air particulate matter. Toxicol Lett 5(108):191–199","https:\u002F\u002Fwww.sciencedirect.com\u002Fscience\u002Farticle\u002Fpii\u002FS0378427499000892",{"doi":1847},"10.1016\u002Fs0378-4274(99)00089-2",{"id":1141,"text":1849,"url":1143,"identifiers":1850},"Strickland P, Kang D, Sithisarankul P (1996) Polycyclic aromatic hydrocarbon metabolites in urine as biomarkers of exposure and effect. Environ Health Perspect 104(5):927–932",{"doi":1145},{"id":1141,"text":1852,"url":1143,"identifiers":1853},"Strunk P, Ortlepp K, Heinz H et al (2002) Ambient and biological monitoring of coke plant workers—determination of exposure to polycyclic aromatic hydrocarbons. Int Arch Occup Environ Health 75(5):354–358",{"doi":1145},{"id":1141,"text":1855,"url":1143,"identifiers":1856},"Viau C, Vyskocil A, Martel L (1995) Background urinary 1-hydroxypyreme levels in non-occupationally exposed individuals in the Province of Quebec, Canada, and comparison with its excretion in workers exposed to PAH mixtures. Sci Total Environ 164:191–194",{"doi":1145},{"id":22,"text":1858,"url":22,"identifiers":1859},"de Vos RH, van Dokkum W, Schouten A et al (1990) Polycyclic aromatic hydrocarbons in Dutch total diet samples (1984–1986). Food Chem Toxicol 28(4):263–268",{},{"id":1861,"text":1862,"url":1863,"identifiers":1864},"1cb09ecf-8bb7-4fb7-8abd-9cb6fe2756cf","Vyskocil A, Fiala Z, Chénier V et al (2000) Assessment of multipathway exposure of small children to PAH. Environ Toxicol Pharmacol 8:111–118","https:\u002F\u002Fwww.sciencedirect.com\u002Fscience\u002Farticle\u002Fpii\u002FS1382668900000326",{"doi":1865},"10.1016\u002Fs1382-6689(00)00032-6",{"id":1141,"text":1867,"url":1143,"identifiers":1868},"van Wijnen JH, Slob R, Jongmans-Liedekerken G et al (1996) Exposure to polycyclic aromatic hydrocarbons among Dutch children. Environ Health Perspect 104(5):530–534",{"doi":1145},{"id":1141,"text":1870,"url":1143,"identifiers":1871},"Xu Z, Brown LM, Pan GW et al (1996) Cancer risks among iron and steel workers in Anshan, China, Part II: case-control studies of lung and stomach cancer. Am J Ind Med 30(1):7–15",{"doi":1145},{"id":1141,"text":1873,"url":1143,"identifiers":1874},"Yang M, Kim S, Lee E et al (2003) Sources of polycyclic aromatic hydrocarbon exposure in non-occupationally exposed Koreans. Environ Mol Mutagen 42:250–257",{"doi":1145},{"id":1876,"createTime":1877,"updateTime":1878,"relativeEntities":1879,"slug":1880,"properties":1881,"entityType":295,"verifyStatus":484,"verifyTime":1892,"verifyNote":486,"languages":22,"translateLanguages":22,"viewCount":23,"primaryUrl":1893,"fullTextUrl":22,"authors":1894,"publicationType":416,"publisherRelationship":1975,"citationCount":2023,"citationInfo":2024,"publishDate":2027,"publishYear":2025,"citationAnalyzeStatus":1136,"lastCitationAnalyze":1878,"indexDatabases":2028,"openAccess":22,"references":22,"isForceReanalyzing":468},"e75462d6-c87c-4681-8158-2c6eb74a91a1","2024-02-18T14:31:12.707+00:00","2026-07-26T01:32:36.309+00:00",[],"Circulatory-disturbances-of-the-foot-in-vibration-syndrome",{"abstract":1882,"title":1884,"gsPaper":1886,"references":1888,"doi":1890},{"EN":1883},"Circulatory disturbances of the foot in patients with vibration syndrome were studied by measuring the skin temperature of both index fingers and great toes through a 3-min immersion of the right foot in cold water at 10°C. Subjects included 11 patients with vibration-induced white finger (VWF) [VWF(+) group], 12 patients without VWF [VWF(−) group], and 20 healthy referents not exposed to vibration. Patients were all male chain saw operators who had scarcely been exposed to vibration of the foot. The prevalence of coldness felt in the upper and lower extremities was > 90% in the VWF(+) group, about 60% in the VWF(−) group, and \u003C 10% in the referents. The extent of the coldness was greatest in the VWF (+) group. The skin temperature of both fingers and toes was lowest in the VWF(+) group, somewhat higher in the VWF(−) group, and highest in the referents both before and after immersion. These findings indicate that patients with vibration syndrome, especially those with VWF, have circulatory disturbances in the foot as well as in the hand. The disturbances in the foot may be related to long-term repeated vasoconstriction in the foot induced by hand-arm vibration through the sympathetic nervous system.",{"EN":1885},"Circulatory disturbances of the foot in vibration syndrome",{"VOID":1887},"[\"5926978963395545233\"]",{"VOID":1889},"Asaba G, Kimura A, Tsubahara A, Sakai H, Shinohara Y (1981) Effect of hot-spring baths on peripheral circulatory disorders in vibration disease (in Japanese). JJA Phys M Baln Clim 45:3–4\nBini G, Hagbarth K-E, Hynninen P, Wallin BG (1980) Regional similarities and differences in thermoregulatory vaso- and sudomotor tone. J Physiol 306:553–565\nBurton AC (1939) The range and variability of the blood flow in the human fingers and the vasomotor regulation of body temperature. Am J Physiol 127:437–453\nEkenvall L, Lindblad LE, Bevegård S, Etzell B-M (1987) High vascular tone but no obliterative lesions in vibration white fingers. Am J Ind Med 12:47–54\nFärkkilä M, Pyykkö I (1979) Blood flow in the contralateral hand during vibration and hand grip contraction of lumberjacks. Scand J Work Environ Health 5:368–374\nFutatsuka M, Ueda T (1982) Quantitative observations on thermal and metabolic responses to cold of workers using vibrating tools. Int Arch Occup Environ Health 49:213–221\nFutatsuka M, Pyykkö I, Färkkilä M, Korhonen O, Starck J (1983) Blood pressure, flow and peripheral resistance of digital arteries in vibration syndrome. Br J Ind Med 40:434–441\nGomibuchi R, Ohi T (1962) So-called “white waxy finger” (in Japanese). JJTOM 15:177–182\nHashiguchi T, Sakakibara H, Furuta M, Yamada S, Horio K, Toibana N (1988) Raynaud's phenomenon in the lower extremities induced by vibration exposure, report of three cases (in Japanese, with English abstract). JJTOM 36:651–657\nHashiguchi T, Sakakibara H, Yamada S (1990) Changes of skin blood flow in the finger and dorsum of the foot during chain saw operation. In: Okada A, Taylor W, Dupuis H (eds) Hand-arm vibration. Kyoei Press, Kanazawa, Japan, pp 133–135\nHedlund U (1989) Raynaud's phenomenon of fingers and toes of miners exposed to local and whole-body vibration and cold. Int Arch Occup Environ Health 61:457–461\nIki M, Kurumatani N, Hirata K, Moriyama T, Satoh M, Arai T (1986) Association between vibration-induced white finger and hearing loss in forestry workers. Scand J Work Environ Health 12:365–370\nKondo T, Sakakibara H, Miyao M, Akamatsu Y, Yamada S, Nakagawa T, Koike Y (1987) Effect of exposure to hand-transmitted vibration on digital skin temperature change. Ind Health 25:41–53\nNakamoto M (1990) Responses of sympathetic nerve system to cold exposure in vibration syndrome subjects and age-matched healthy controls. Int Arch Occup Environ Health 62:177–181\nNasu Y (1977) Changes of the skin temperature caused by local vibratory stimulation in normals and patients with vibration syndrome. Yonago Acta Med 21:83–99\nOkada A (1990) Pathogenic mechanism of vibration-induced white finger (VWF) — recent findings and speculations. In: Okada A, Taylor W, Dupuis H (eds) Hand-arm vibration. Kyoei Press, Kanazawa, Japan, pp 1–8\nOlsen N, Nielsen SL, Voss P (1982) Cold response of digital arteries in chain saw operators. Br J Ind Med 39:82–88\nOlsen N, Fjeldborg P, Brøchner-Mortensen J (1985) Sympathetic and local vasoconstrictor response to cold in vibration induced white finger. Br J Ind Med 42:272–275\nPyykkö I, Hyvärinen J (1973) The physiological basis of the traumatic vasospastic disease: a sympathetic vasoconstrictor reflex triggered by high frequency vibration. Work Environ Health 10:36–47\nPyykkö I, Starck J, Färkkilä M, Hoikkala M, Korhonen O, Nurminen M (1981) Hand-arm vibration in the aetiology of hearing loss in lumberjacks. Br J Ind Med 38:281–289\nSakakibara H, Akamatsu Y, Miyao M, Kondo T, Furuta M, Yamada S, Harada N, Miyake S, Hosokawa M (1988) Correlation between vibration-induced white finger and symptoms of upper and lower extremities. Int Arch Occup Environ Health 60:285–289\nSakakibara H, Iwase S, Mano T, Watanabe T, Kobayashi F, Furuta M, Kondo T, Miyao M, Yamada S (1990) Skin sympathetic activity in the tibial nerve triggered by vibration applied to the hand. Int Arch Occup Environ Health 62:455–458\nSuzuki Y, Nishiyama K, Nagayasu M, Nishihara S (1966) A case of the occupational Raynaud's phenomenon observed on the foot of a vibrating tool operator (in Japanese, with English abstract). Shikoku Acta Med 22:70–72\nToibana N, Ishikawa N (1990) The patients with Raynaud's phenomenon in fingers and toes caused by vibration. In: Okada A, Taylor W, Dupuis H (eds) Hand-arm vibration. Kyoei Press, Kanazawa, Japan, pp 245–248\nYamada S, Suzuki H, Maeda K, Takeuchi Y (1974) Skin temperature of the finger and value of nail press test under various environmental conditions in persons affected by vibration hazards (in Japanese, with English abstract). J Sci Labor 50:439–450",{"VOID":1891},"10.1007\u002FBF00379079","2024-06-24T23:00:22.402+00:00","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002FBF00379079",[1895,1910,1923,1936,1949,1962],{"id":1896,"sortIndex":23,"researcher":22,"roles":1897,"affiliations":1898,"properties":1907,"displayName":1909,"givenName":22,"familyName":22},"71bb3658-93ed-4739-8914-db10ab089c68",[303],[1899],{"id":1900,"sortIndex":23,"affiliation":1901,"properties":22},"cea2136b-e5af-408c-9c51-5964e646532f",{"id":1900,"createTime":22,"updateTime":22,"relativeEntities":1902,"slug":22,"properties":1903,"entityType":22,"verifyStatus":22,"verifyTime":22,"verifyNote":22,"languages":22,"translateLanguages":22,"viewCount":22,"url":22,"parentIds":1906,"statistic":22},[],{"title":1904},{"VI":1905},"Department of Public Health, Nagoya University School of Medicine, Showa-ku, Nagoya, Japan",[],{"title":1908},{"VI":1909},"Hisataka Sakakibara",{"id":1911,"sortIndex":157,"researcher":22,"roles":1912,"affiliations":1913,"properties":1920,"displayName":1922,"givenName":22,"familyName":22},"de624861-7d1e-47d4-8b8c-c418dc6e4ed6",[303],[1914],{"id":1900,"sortIndex":23,"affiliation":1915,"properties":22},{"id":1900,"createTime":22,"updateTime":22,"relativeEntities":1916,"slug":22,"properties":1917,"entityType":22,"verifyStatus":22,"verifyTime":22,"verifyNote":22,"languages":22,"translateLanguages":22,"viewCount":22,"url":22,"parentIds":1919,"statistic":22},[],{"title":1918},{"VI":1905},[],{"title":1921},{"VI":1922},"Toshinori Hashiguchi",{"id":1924,"sortIndex":158,"researcher":22,"roles":1925,"affiliations":1926,"properties":1933,"displayName":1935,"givenName":22,"familyName":22},"ed5c65bc-7f6f-4f3b-9152-d80970ddc102",[303],[1927],{"id":1900,"sortIndex":23,"affiliation":1928,"properties":22},{"id":1900,"createTime":22,"updateTime":22,"relativeEntities":1929,"slug":22,"properties":1930,"entityType":22,"verifyStatus":22,"verifyTime":22,"verifyNote":22,"languages":22,"translateLanguages":22,"viewCount":22,"url":22,"parentIds":1932,"statistic":22},[],{"title":1931},{"VI":1905},[],{"title":1934},{"VI":1935},"Masashi 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