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It is often suggested that sociocultural processes are the main cause of such widespread dissatisfaction. Here, we consider how perceptual effects may influence ideas of body normality and body ideals.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Method:\u003C\u002Fjats:title>\u003Cjats:p>Women who varied on a measure of body dissatisfaction rated a range of bodies for how normal and ideal they looked. They were exposed to either thin or fat bodies, and then they rerated the bodies.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Results:\u003C\u002Fjats:title>\u003Cjats:p>Women's perceptions of body normality and ideal were easily malleable by exposure. In addition, greater body dissatisfaction and internalization of the thin Western ideal were related to (i) a smaller most normal and ideal body, (ii) a greater discrepancy between the most normal and most ideal‐rated body, and (iii) a reduced effect of exposure to fat bodies.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Discussion:\u003C\u002Fjats:title>\u003Cjats:p>Reduced updating of perceptions of body normality and body ideals in response to experience may be one mechanism that maintains body dissatisfaction. © 2008 by Wiley Periodicals, Inc. 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Dordrecht: Kluwer Academic.",{},{"id":24,"text":829,"url":24,"identifiers":830},"Neale, (199, Behavior Genetics, 27, 113, 10.1023\u002FA:1025681223921",{"doi":831},"10.1023\u002FA:1025681223921",{"id":24,"text":833,"url":24,"identifiers":834},"Neumark-Sztainer, (199, Archives of Pediatrics and Adolescent Medicine, 149, 1330, 10.1001\u002Farchpedi.1995.02170250036005",{"doi":835},"10.1001\u002Farchpedi.1995.02170250036005",{"id":24,"text":837,"url":24,"identifiers":838},"Pietiläinen, (199, International Journal of Obesity, 23, 107, 10.1038\u002Fsj.ijo.0800767",{"doi":839},"10.1038\u002Fsj.ijo.0800767",{"id":24,"text":841,"url":24,"identifiers":842},"Pingitore, (199, Obesity Research, 5, 402, 10.1002\u002Fj.1550-8528.1997.tb00662.x",{"doi":843},"10.1002\u002Fj.1550-8528.1997.tb00662.x",{"id":24,"text":845,"url":24,"identifiers":846},"Rierdan, (199, Adolescence, 32, 615",{},{"id":24,"text":848,"url":24,"identifiers":849},"Rissanen, (198, International Journal of Obesity, 12, 391",{},{"id":24,"text":851,"url":24,"identifiers":852},"Rose, (199, Journal of Studies on Alcohol, Suppl, 13, 63",{},{"id":24,"text":854,"url":24,"identifiers":855},"Rutherford, (199, Psychological Medicine, 23, 425, 10.1017\u002FS003329170002852X",{"doi":856},"10.1017\u002FS003329170002852X",{"id":24,"text":858,"url":24,"identifiers":859},"Sands, (199, International Journal of Eating Disorders, 21, 159, 10.1002\u002F(SICI)1098-108X(199703)21:2\u003C159::AID-EAT6>3.0.CO;2-K",{"doi":860},"10.1002\u002F(SICI)1098-108X(199703)21:2\u003C159::AID-EAT6>3.0.CO;2-K",{"id":24,"text":862,"url":24,"identifiers":863},"Sarna, (198, Human Heredity, 30, 71, 10.1159\u002F000153105",{"doi":864},"10.1159\u002F000153105",{"id":24,"text":866,"url":24,"identifiers":867},"Sarna, (197, Human Heredity, 28, 241, 10.1159\u002F000152964",{"doi":868},"10.1159\u002F000152964",{"id":24,"text":870,"url":24,"identifiers":871},"Schur, (200, International Journal of Eating Disorders, 27, 74, 10.1002\u002F(SICI)1098-108X(200001)27:1\u003C74::AID-EAT8>3.0.CO;2-K",{"doi":872},"10.1002\u002F(SICI)1098-108X(200001)27:1\u003C74::AID-EAT8>3.0.CO;2-K",{"id":24,"text":874,"url":24,"identifiers":875},"Silventoinen, (200, Twin Research, 6, 399, 10.1375\u002F136905203770326402",{"doi":876},"10.1375\u002F136905203770326402",{"id":24,"text":878,"url":24,"identifiers":879},"Slof, (200, Obesity Research, 11, 971, 10.1038\u002Foby.2003.134",{"doi":880},"10.1038\u002Foby.2003.134",{"id":24,"text":882,"url":24,"identifiers":883},"Statistics Finland website: Finland in figures: Education. (2001). Last updated: January 9, 2003. http:\u002F\u002Fwww.stat.fi\u002Ftk\u002Ftp\u002Ftasku\u002Ftaskue_koulutus.html (accessed October 24, 2003).",{},{"id":24,"text":885,"url":24,"identifiers":886},"Stice, (200, Developmental Psychology, 37, 597, 10.1037\u002F0012-1649.37.5.597",{"doi":887},"10.1037\u002F0012-1649.37.5.597",{"id":24,"text":889,"url":24,"identifiers":890},"Stice, (200, Developmental Psychology, 38, 669, 10.1037\u002F0012-1649.38.5.669",{"doi":891},"10.1037\u002F0012-1649.38.5.669",{"id":24,"text":893,"url":24,"identifiers":894},"Taylor, (199, Behavior Research and Therapy, 30, 53, 10.1016\u002F0005-7967(92)90096-Y",{"doi":895},"10.1016\u002F0005-7967(92)90096-Y",{"id":24,"text":897,"url":24,"identifiers":898},"Wade, (200, Psychological Medicine, 33, 1395, 10.1017\u002FS0033291703008572",{"doi":899},"10.1017\u002FS0033291703008572",{"id":24,"text":901,"url":24,"identifiers":902},"Wade, (199, Psychological Medicine, 28, 761, 10.1017\u002FS0033291798006989",{"doi":903},"10.1017\u002FS0033291798006989",{"id":24,"text":905,"url":24,"identifiers":906},"Wiseman, (199, International Journal of Eating Disorders, 24, 429, 10.1002\u002F(SICI)1098-108X(199812)24:4\u003C429::AID-EAT10>3.0.CO;2-D",{"doi":907},"10.1002\u002F(SICI)1098-108X(199812)24:4\u003C429::AID-EAT10>3.0.CO;2-D",{"id":909,"createTime":910,"updateTime":911,"relativeEntities":912,"slug":913,"properties":914,"entityType":185,"verifyStatus":186,"verifyTime":910,"verifyNote":188,"languages":933,"translateLanguages":934,"viewCount":25,"primaryUrl":936,"fullTextUrl":24,"authors":937,"publicationType":288,"publisherRelationship":1023,"citationCount":1081,"citationInfo":1082,"publishDate":1085,"publishYear":1083,"citationAnalyzeStatus":23,"lastCitationAnalyze":24,"indexDatabases":1086,"openAccess":24,"references":1087,"isForceReanalyzing":495},"f8a54eea-b920-4a3a-94d5-ec3d738b0da0","2025-01-04T16:03:17.652+00:00","2025-02-20T07:48:46.225+00:00",[],"Dietary-energy-density-and-diet-variety-as-risk-factors-for-relapse-in-anorexia-nervosa-A-replication",{"mag":915,"keywords":917,"pmc":919,"openalex":921,"abstract":923,"title":926,"pm":929,"doi":931},{"VOID":916},"1995556269",{"VI":918},"",{"VOID":920},"4469286",{"VOID":922},"W1995556269",{"EN":924,"VI":925},"\u003Cjats:title>Abstract\u003C\u002Fjats:title>\u003Cjats:sec>\u003Cjats:title>Objective:\u003C\u002Fjats:title>\u003Cjats:p>To replicate our previous findings of an association between energy density and diet variety in recently weight‐restored patients with anorexia nervosa (AN) and clinical outcome in the year following treatment.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Method:\u003C\u002Fjats:title>\u003Cjats:p>Nineteen hospitalized, weight‐restored women with AN completed a food record, from which a diet energy density score (DEDS) and a diet variety score (DVS) were calculated. After hospital discharge, patients were contacted regularly; at the end of one year, clinical outcome was determined using modified Morgan‐Russell criteria. As in our previous study, outcome was dichotomized into “full, good, or fair” and “poor” groups.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Results:\u003C\u002Fjats:title>\u003Cjats:p>Data from 16 subjects were available. The DEDS was significantly lower (\u003Cjats:italic>p\u003C\u002Fjats:italic> < .05) in the poor outcome group (0.7 ± 1) compared with the “full, good, or fair” outcome group (0.9 ± 1). Although the DVS was also lower in the poor outcome group (13.9 ± 2) compared with the “full, good or fair” outcome group (15.7 ± 1.8), this difference was not statistically significant.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Discussion:\u003C\u002Fjats:title>\u003Cjats:p>In recently weight‐restored patients with AN, a lower DEDS, but not DVS, is associated with poor clinical outcome after inpatient treatment. This finding may be important in the assessment of risk for relapse in patients with AN. © 2011 Wiley Periodicals, Inc. (Int J Eat Disord 2012; 45:79–84)\u003C\u002Fjats:p>\u003C\u002Fjats:sec>","\u003Cjats:title>Tóm tắt\u003C\u002Fjats:title>\u003Cjats:sec>\u003Cjats:title>Mục tiêu:\u003C\u002Fjats:title>\u003Cjats:p>Mục tiêu của nghiên cứu này là tái hiện những phát hiện trước đây về mối liên hệ giữa mật độ năng lượng và sự đa dạng trong chế độ ăn ở những bệnh nhân chán ăn tâm thần (AN) đã phục hồi trọng lượng gần đây và kết quả lâm sàng trong năm tiếp theo sau điều trị.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Phương pháp:\u003C\u002Fjats:title>\u003Cjats:p>Mười chín phụ nữ được nhập viện và phục hồi trọng lượng đã hoàn thành hồ sơ thực phẩm, từ đó có thể tính toán điểm mật độ năng lượng trong chế độ ăn (DEDS) và điểm đa dạng trong chế độ ăn (DVS). Sau khi xuất viện, các bệnh nhân được liên lạc thường xuyên; vào cuối một năm, kết quả lâm sàng được xác định bằng tiêu chí Morgan-Russell được điều chỉnh. Giống như trong nghiên cứu trước đó của chúng tôi, kết quả được chia thành hai nhóm \"đầy đủ, tốt hoặc khá\" và \"kém\".\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Kết quả:\u003C\u002Fjats:title>\u003Cjats:p>Dữ liệu từ 16 đối tượng đã có sẵn. DEDS có ý nghĩa thống kê thấp hơn (\u003Cjats:italic>p\u003C\u002Fjats:italic> < .05) ở nhóm có kết quả kém (0.7 ± 1) so với nhóm có kết quả \"đầy đủ, tốt hoặc khá\" (0.9 ± 1). Mặc dù DVS cũng thấp hơn ở nhóm có kết quả kém (13.9 ± 2) so với nhóm có kết quả \"đầy đủ, tốt hoặc khá\" (15.7 ± 1.8), sự khác biệt này không mang tính chất ý nghĩa thống kê.",{"EN":927,"VI":928},"Dietary energy density and diet variety as risk factors for relapse in anorexia nervosa: A replication","Mật độ năng lượng và sự đa dạng trong chế độ ăn như yếu tố nguy cơ tái phát ở bệnh nhân chán ăn tâm thần: Một nghiên cứu lặp lại",{"VOID":930},"21448937",{"VOID":932},"10.1002\u002Feat.20922",[190],[935],"VI","https:\u002F\u002Fonlinelibrary.wiley.com\u002Fdoi\u002F10.1002\u002Feat.20922",[938,955,972,989,1006],{"id":939,"sortIndex":25,"researcher":24,"roles":940,"affiliations":941,"properties":950,"displayName":952,"givenName":24,"familyName":24},"fa894471-1723-49c6-9e83-8bb0cbb31969",[],[942],{"id":943,"sortIndex":25,"affiliation":944,"properties":24},"f0d4bb2b-615a-4637-a012-146a0f295979",{"id":943,"createTime":24,"updateTime":24,"relativeEntities":945,"slug":24,"properties":946,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":949,"statistic":24},[],{"title":947},{"EN":948},"Eating Disorders Research Unit, Department of Psychiatry, College of Physicians and Surgeons of Columbia University, New York State Psychiatric Institute, New York, New York",[],{"title":951,"openalex":953},{"EN":952},"Janet Schebendach",{"VOID":954},"A5109323930",{"id":956,"sortIndex":117,"researcher":24,"roles":957,"affiliations":958,"properties":965,"displayName":969,"givenName":24,"familyName":24},"7967cdfe-a018-40f9-ae7a-1bcb398df4db",[],[959],{"id":943,"sortIndex":25,"affiliation":960,"properties":24},{"id":943,"createTime":24,"updateTime":24,"relativeEntities":961,"slug":24,"properties":962,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":964,"statistic":24},[],{"title":963},{"EN":948},[],{"orcid":966,"title":968,"openalex":970},{"VOID":967},"https:\u002F\u002Forcid.org\u002F0000-0002-9424-1133",{"EN":969},"Laurel Mayer",{"VOID":971},"A5062221893",{"id":973,"sortIndex":118,"researcher":24,"roles":974,"affiliations":975,"properties":982,"displayName":986,"givenName":24,"familyName":24},"fa767386-d0d6-4deb-bad5-39e205281bfc",[],[976],{"id":943,"sortIndex":25,"affiliation":977,"properties":24},{"id":943,"createTime":24,"updateTime":24,"relativeEntities":978,"slug":24,"properties":979,"entityType":24,"verifyStatus":24,"verifyTime":24,"verifyNote":24,"languages":24,"translateLanguages":24,"viewCount":24,"url":24,"parentIds":981,"statistic":24},[],{"title":980},{"EN":948},[],{"orcid":983,"title":985,"openalex":987},{"VOID":984},"https:\u002F\u002Forcid.org\u002F0000-0003-2006-424X",{"EN":986},"Michael J. 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eating (PE) is common across the lifespan and related to psychosocial impairment and limited dietary variety. However, research about PE in non‐Western countries is limited. Because eating behaviors may differ by culture, operationalizing PE in non‐Western countries (e.g., China) is needed. The present study aimed to replicate two previous studies identifying PE profiles in Western countries by using latent profile analysis (LPA) to classify and characterize adults reporting picky eating in a Chinese sample.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Method\u003C\u002Fjats:title>\u003Cjats:p>A sample of 1,068 Chinese young adults completed a battery of questionnaires including the adult eating behavior questionnaire (AEBQ). LPA was utilized to identify eating profiles. The three‐step approach was used to examine predictors of latent memberships and profile differences on various self‐reported measures.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Results\u003C\u002Fjats:title>\u003Cjats:p>The best fit was a four‐profile solution, with two picky eating profiles emerging: \u003Cjats:italic>picky eating\u003C\u002Fjats:italic> and \u003Cjats:italic>severe picky eating\u003C\u002Fjats:italic>. Compared to those in the other two profiles, participants in the \u003Cjats:italic>picky eating\u003C\u002Fjats:italic> profile (19.4%) and \u003Cjats:italic>severe picky eating\u003C\u002Fjats:italic> profile (3.3%) had significantly higher scores on self‐reported eating disorder symptoms and psychological distress, and lower scores on self‐reported food‐related life satisfaction. Relative to the \u003Cjats:italic>picky eating\u003C\u002Fjats:italic> profile, participants in the \u003Cjats:italic>severe picky eating\u003C\u002Fjats:italic> profile reported significantly greater self‐reported eating disorder symptoms, psychological distress, and food‐related dissatisfaction.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Discussion\u003C\u002Fjats:title>\u003Cjats:p>Characterizing PE profiles is an important step toward understanding eating behaviors among Chinese young adults. 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Yet, systematic comparisons of all major EDs on emotional eating patterns are lacking. Furthermore, emotional eating correlates with body mass index (BMI), which also differs between EDs and thus confounds this comparison.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Method\u003C\u002Fjats:title>\u003Cjats:p>Interview‐diagnosed female ED patients (\u003Cjats:italic>n\u003C\u002Fjats:italic> = 204) with restrictive (AN‐R) or binge‐purge anorexia nervosa (AN‐BP), bulimia nervosa (BN), or binge‐eating disorder (BED) completed a questionnaire assessing “negative emotional eating” (sadness, anger, anxiety) and “happiness eating.” ED groups were compared to BMI‐matched healthy controls (HCs; \u003Cjats:italic>n\u003C\u002Fjats:italic> = 172 ranging from underweight to obesity) to exclude BMI as a confound.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Results\u003C\u002Fjats:title>\u003Cjats:p>Within HCs, higher BMI was associated with higher negative emotional eating and lower happiness eating. AN‐R reported the lowest degree of negative emotional eating relative to other EDs and BMI‐matched HCs, and the highest degree of happiness eating relative to other EDs. The BN and BED groups showed higher negative emotional eating compared to BMI‐matched HCs. Patients with AN‐BP occupied an intermediate position between AN‐R and BN\u002FBED and reported less happiness eating compared to BMI‐matched HCs.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Discussion\u003C\u002Fjats:title>\u003Cjats:p>Negative emotional and happiness eating patterns differ across EDs. BMI‐independent emotional eating patterns distinguish ED subgroups and might be related to the occurrence of binge eating versus restriction. Hence, different types of emotional eating can represent fruitful targets for tailored psychotherapeutic interventions. 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Dữ liệu được phân tích theo hai cách. Trong phân tích đầu tiên, tính toàn vẹn nhân tố của bảng hỏi và tính đồng nhất nội bộ của các thang đo con không được tìm thấy là rất thỏa mãn. Dữ liệu cho thấy sự thiếu biến thiên ấn tượng. Các tác giả gợi ý rằng trong một quần thể không có triệu chứng, sự biến thiên của dữ liệu EDI bị ức chế do việc, phù hợp với sách hướng dẫn, các mục EDI 6 điểm ban đầu đã được chuyển đổi thành các mục 4 điểm. Trong phân tích thứ hai, việc chuyển đổi mục này đã bị loại bỏ. Các đặc tính tâm lý đo lường của tất cả các thang đo con EDI trong nghiên cứu hiện tại đã được cải thiện. Các hàm ý cho việc sử dụng EDI chưa chuyển đổi như một công cụ sàng lọc đã được thảo luận.\u003C\u002Fjats:p>","\u003Cjats:title>Abstract\u003C\u002Fjats:title>\u003Cjats:p>In the present study, the validity of the Eating Disorder Inventory (EDI) in a Dutch nonclinical female school population was established. The data were analyzed in two ways. 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Các rối loạn Axis I và Axis II đã được đánh giá thông qua phỏng vấn có cấu trúc; phỏng vấn theo dõi được thực hiện sau mỗi 6-12 tháng để thu thập dữ liệu hàng tuần về triệu chứng rối loạn ăn uống.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Kết quả\u003C\u002Fjats:title>\u003Cjats:p>Phụ nữ mắc ANR và ANBP không có sự khác biệt về tiền sử lạm dụng chất, chứng hay trộm cắp, suy nghĩ tự sát hoặc chẩn đoán rối loạn nhân cách biên giới khi nhập viện, cũng như về tỷ lệ phục hồi, tái phát hoặc tỷ lệ tử vong. Sau 8 năm theo dõi, 62% phụ nữ mắc ANR đã chuyển sang ANBP và chỉ 12% phụ nữ mắc AN chưa bao giờ báo cáo hành vi cuồng ăn\u002Fthải thường xuyên.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Kết luận\u003C\u002Fjats:title>\u003Cjats:p>Các phát hiện về tính bốc đồng, quá trình và kết quả không hỗ trợ cho hệ thống phân loại kiểu loại hiện tại. Tỷ lệ chuyển đổi cao từ ANR sang ANBP trong mẫu nghiên cứu của chúng tôi cho thấy ANR đại diện cho một giai đoạn trong quá trình phát triển của AN thay vì một kiểu loại riêng biệt. © 2002 bởi Wiley Periodicals, Inc. Int J Eat Disord 31: 191–201, 2002; DOI. 10.1002\u002Feat.10016\u003C\u002Fjats:p>\u003C\u002Fjats:sec>","\u003Cjats:title>Abstract\u003C\u002Fjats:title>\u003Cjats:sec>\u003Cjats:title>Objective\u003C\u002Fjats:title>\u003Cjats:p>To compare patients with restricting anorexia nervosa (ANR) and binge\u002Fpurge anorexia nervosa (ANBP) on measures of impulsivity, course, and outcome.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Methods\u003C\u002Fjats:title>\u003Cjats:p>One hundred thirty‐six treatment‐seeking women with AN followed prospectively for 8–12 years were reclassified at intake as 51 ANRs and 85 ANBPs according to the DSM‐IV subtyping classification. Lifetime Axis I and Axis II disorders were assessed using structured interviews; follow‐up interviews were conducted at 6–12‐month intervals to collect weekly data on eating disorder symptomatology.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Results\u003C\u002Fjats:title>\u003Cjats:p>Women with ANR and ANBP did not differ on history of substance abuse, kleptomania, suicidality, or borderline personality diagnosis at intake, or on rates of recovery, relapse, or mortality. By 8 years of follow‐up, 62% of women with ANR crossed over to ANBP prospectively and only 12% of women with AN never reported regular binge\u002Fpurge behaviors.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Conclusions\u003C\u002Fjats:title>\u003Cjats:p>The findings on impulsivity, course, and outcome do not support the current subtyping system. The high crossover rate in our sample from ANR to ANBP suggests that ANR represents a phase in the course of AN rather than a distinct subtype. © 2002 by Wiley Periodicals, Inc. 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Ngoài ra, các nam đồng tính báo cáo sự không thoải mái lớn hơn với xu hướng tình dục của họ. Sau khi kiểm soát sự khác biệt về trầm cảm, tự tin và cảm giác thoải mái với xu hướng tình dục, xu hướng tình dục vẫn tiếp tục giải thích được sự biến thiên có ý nghĩa trong điểm số BULIT‐R, EAT‐26 và BSQ.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Thảo luận\u003C\u002Fjats:title>\u003Cjats:p>Các nghiên cứu trong tương lai có thể được lợi từ việc khám phá các khía cạnh của đồng tính luyến ái có thể cụ thể góp phần vào nguy cơ rối loạn ăn uống ở nam giới. © 2002 bởi Wiley Periodicals, Inc. Int J Eat Disord 31: 300–306, 2002; DOI 10.1002\u002Feat.10036\u003C\u002Fjats:p>\u003C\u002Fjats:sec>","\u003Cjats:title>Abstract\u003C\u002Fjats:title>\u003Cjats:sec>\u003Cjats:title>Objective\u003C\u002Fjats:title>\u003Cjats:p>The current study examined whether homosexuality is a specific risk factor for disordered eating in men.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Method\u003C\u002Fjats:title>\u003Cjats:p>Men (64 heterosexual and 58 homosexual) completed the Beck Depression Inventory (BDI), the Rosenberg Self‐Esteem Scale (RSE), the Masculinity and Femininity scales of the Bem Sex‐Role Inventory (BSRI), the Bulimia Test‐Revised (BULIT‐R), the Eating Attitudes Test (EAT‐26), and the Body Shape Questionnaire (BSQ).\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Results\u003C\u002Fjats:title>\u003Cjats:p>Homosexual men had more pathological scores on the BDI, RSE, BULIT‐R, EAT‐26, and BSQ. Additionally, homosexual men reported greater discomfort with sexual orientation. After controlling for differences in depression, self‐esteem, and comfort with sexual orientation, sexual orientation continued to account for significant variance in BULIT‐R, EAT‐26, and BSQ scores.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Discussion\u003C\u002Fjats:title>\u003Cjats:p>Future research may benefit from exploring aspects of homosexuality that may contribute specifically to risk for disordered eating in men. © 2002 by Wiley Periodicals, Inc. 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tắt\u003C\u002Fjats:title>\u003Cjats:sec>\u003Cjats:title>Mục tiêu\u003C\u002Fjats:title>\u003Cjats:p>Dữ liệu hạn chế cho thấy việc ăn uống rối loạn có thể làm trẻ em dễ bị tăng cân quá mức. Chúng tôi đã nghiên cứu mối quan hệ giữa phản ứng ban đầu với Cuộc kiểm tra rối loạn ăn uống điều chỉnh cho trẻ em (ChEDE) và sự thay đổi chỉ số khối cơ thể (BMI (kg\u002Fm\u003Cjats:sup>2\u003C\u002Fjats:sup>)) ở trẻ em có nguy cơ cao mắc béo phì ở người lớn.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Phương pháp\u003C\u002Fjats:title>\u003Cjats:p>Các trẻ em (từ 6–12 tuổi) đã được kiểm tra ChEDE để đánh giá tình trạng mất kiểm soát khi ăn (LOC), sự kiềm chế chế độ ăn uống, và lo ngại về ăn uống, hình dạng và trọng lượng. Chiều cao và cân nặng được đo ở thời điểm ban đầu và hàng năm.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Kết quả\u003C\u002Fjats:title>\u003Cjats:p>Giữa tháng 7 năm 1999 và tháng 8 năm 2007, 772 lần đo đã được thực hiện trên 143 trẻ em trong thời gian 4.5 ± 1.9 năm. Mất kiểm soát khi ăn (LOC) dự đoán tỷ lệ tăng trưởng BMI cao hơn theo thời gian (\u003Cjats:italic>p\u003C\u002Fjats:italic> = .02). So với những trẻ em không bị LOC, những trẻ báo cáo LOC đã tăng thêm trung bình 2.4 kg trọng lượng mỗi năm.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Kết luận\u003C\u002Fjats:title>\u003Cjats:p>Mất kiểm soát khi ăn (LOC) là yếu tố dự đoán nổi bật về việc tăng cân trong giai đoạn giữa tuổi thơ. Các can thiệp giảm thiểu tình trạng LOC khi ăn nên được đánh giá về khả năng ngăn ngừa tăng cân quá mức ở trẻ em. © 2008 bởi Wiley Periodicals, Inc. Int J Eat Disord 2009\u003C\u002Fjats:p>\u003C\u002Fjats:sec>","\u003Cjats:title>Abstract\u003C\u002Fjats:title>\u003Cjats:sec>\u003Cjats:title>Objective\u003C\u002Fjats:title>\u003Cjats:p>Limited data suggest that disordered‐eating may predispose children to excessive weight gain. We investigated the relationship between baseline responses to the Eating Disorder Examination adapted for Children (ChEDE) and change in BMI (kg\u002Fm\u003Cjats:sup>2\u003C\u002Fjats:sup>) in children at high risk for adult obesity.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Method\u003C\u002Fjats:title>\u003Cjats:p>Children (6–12 years) were administered the ChEDE to assess loss of control (LOC) eating, dietary restraint, and eating, shape, and weight concern. Height and weight were measured at baseline and annually.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Results\u003C\u002Fjats:title>\u003Cjats:p>Between July, 1999, and August, 2007, 772 measurements were obtained from 143 children over 4.5 ± 1.9 years. LOC eating predicted an increased rate of BMI growth over time (\u003Cjats:italic>p\u003C\u002Fjats:italic> = .02). 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tắt\u003C\u002Fjats:title>\u003Cjats:sec>\u003Cjats:title>Mục tiêu\u003C\u002Fjats:title>\u003Cjats:p>Chúng tôi tiến hành một bài tổng quan tài liệu quan trọng về các nghiên cứu đánh giá tình trạng đồng bệnh của rối loạn lo âu (RLA) ở những đối tượng mắc rối loạn ăn uống (RLA) (chán ăn tâm thần và chứng ăn uống không kiểm soát). Trong phần đầu, chúng tôi thảo luận về các vấn đề phương pháp liên quan đến các nghiên cứu đồng bệnh giữa RLA và RLA. Trong phần thứ hai, với việc xem xét các vấn đề phương pháp đã nêu, chúng tôi tóm tắt các phát hiện của những nghiên cứu này.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Phương pháp\u003C\u002Fjats:title>\u003Cjats:p>Chúng tôi đã thực hiện tìm kiếm thủ công và tìm kiếm máy tính (Medline) cho tất cả các nghiên cứu đã công bố về tình trạng đồng bệnh giữa RLA và RLA, giới hạn tìm kiếm của chúng tôi từ năm 1985–2001 để có các tiêu chí chẩn đoán đủ đồng nhất cho cả hai loại rối loạn.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Kết quả\u003C\u002Fjats:title>\u003Cjats:p>Quá ít nghiên cứu bao gồm các nhóm đối chứng và rất ít nghiên cứu so sánh các tiểu nhóm chẩn đoán của những đối tượng RLA, với những kết quả hiếm hoi hoặc mâu thuẫn.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Thảo luận\u003C\u002Fjats:title>\u003Cjats:p>Chúng tôi thảo luận về các kết quả với sự xem xét các vấn đề phương pháp quan sát. Chúng tôi đưa ra hướng dẫn để xem xét các kết quả của các nghiên cứu đã công bố và lên kế hoạch cho các nghiên cứu trong tương lai. © 2002 bởi Wiley Periodicals, Inc. Int J Eat Disord 32: 253–270, 2002.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>","\u003Cjats:title>Abstract\u003C\u002Fjats:title>\u003Cjats:sec>\u003Cjats:title>Objective\u003C\u002Fjats:title>\u003Cjats:p>We conducted a critical literature review on studies assessing the prevalence of anxiety disorders (AD) in subjects with eating disorders (ED) (anorexia nervosa and bulimia nervosa). In the first part, we discuss methodological issues relevant to comorbidity studies between ED and AD. In the second part, taking into account these methodological considerations raised, we summarize the findings of these studies.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Method\u003C\u002Fjats:title>\u003Cjats:p>We performed a manual and computerized search (Medline) for all published studies on comorbidity between ED and AD, limiting our search from 1985–2001 to get sufficiently homogeneous diagnostic criteria for both categories of disorders.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Results\u003C\u002Fjats:title>\u003Cjats:p>Too few studies include control groups and few studies have compared diagnostic subgroups of ED subjects, with scarce or conflicting results.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Discussion\u003C\u002Fjats:title>\u003Cjats:p>We discuss the results taking into account the methodological problems observed. 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