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Nutr Metab Cardiovasc Dis 18:39–45\nKlein R, Klein BE, Moss SE, Davis MD, DeMets DL (1984) The Wisconsin epidemiologic study of diabetic retinopathy. IV. Diabetic macular edema. Ophthalmology 91:1464–1474\nBallard DJ, Humphrey LL, Melton LJ 3rd, Frohnert PP, Chu PC, O’Fallon WM, Palumbo PJ (1988) Epidemiology of persistent proteinuria in type II diabetes mellitus. Population-based study in Rochester, Minnesota. Diabetes 37:405–412\nHuxley R, Barzi F, Woodward M (2006) Excess risk of fatal coronary heart disease associated with diabetes in men and women: meta-analysis of 37 prospective cohort studies. BMJ 332:73–78\nLee WL, Cheung AM, Cape D, Zinman B (2000) Impact of diabetes on coronary artery disease in women and men: a meta-analysis of prospective studies. Diabetes Care 23:962–968\nNorhammar A, Stenestrand U, Lindbäck J et al (2008) Women younger than 65 years with diabetes mellitus are a high-risk group after myocardial infarction: a report from the Swedish Register of Information and Knowledge about Swedish Heart Intensive Care Admission (RIKS-HIA). Heart 94:1565–1570\nAvogaro A, Giorda C, Maggini M, Mannucci E, Raschetti R, Lombardo F, Spila-Alegiani S, Turco S, Velussi M, Ferrannini E (2007) Incidence of coronary heart disease in type 2 diabetic men and women: impact of microvascular complications, treatment, and geographic location. Diabetes Care 30:1241–1247\nGiorda CB, Avogaro A, Maggini M, Lombardo F, Mannucci E, Turco S, Alegiani SS, Raschetti R, Velussi M, Ferrannini E (2007) Incidence and risk factors for stroke in type2 diabetic patients: the DAI study. Stroke 38:1154–1160\nKrämer HU, Müller H, Stegmaier C, Rothenbacher D, Raum E, Brenner H (2012) Type 2 diabetes mellitus and gender-specific risk for colorectal neoplasia. Eur J Epidemiol. doi:10.1007\u002Fs10654-012-9686-6\nMerlotti D, Gennari L, Dotta F, Lauro D, Nuti R (2010) Mechanisms of impaired bone strength in type 1 and 2 diabetes. Nutr Metab Cardiovasc Dis 20:683–690\nYamaguchi T, Sugimoto T (2011) Bone metabolism and fracture risk in type 2 diabetes mellitus. Endocr J 58:613–624\nMonami M, Cresci B, Colombini A, Pala L, Balzi D, Gori F, Chiasserini V, Marchionni N, Rotella CM, Mannucci E (2008) Bone fractures and hypoglycemic treatment in type 2 diabetic patients: a case-control study. Diabetes Care 31:199–203\nMezuk B, Eaton WW, Albrecht S, Golden SH (2008) Depression and type 2 diabetes over the lifespan: a meta-analysis. Diabetes Care 31:2383–2390\nMannucci E, Rotella F, Ricca V, Moretti S, Placidi GF, Rotella CM (2005) Eating disorders in patients with type 1 diabetes: a meta-analysis. J Endocrinol Invest 28:417–419\nBenvenuti S, Cellai I, Luciani P, Deledda C, Baglioni S, Giuliani C, Saccardi R, Mazzanti B, Dal Pozzo R, Mannucci E, Peri A, Serio M (2007) Rosiglitazone stimulates adipogenesis and decreases osteoblastogenesis in human mesenchymal stem cells. J Endocrinol Invest 30:RC26–RC30\nKahn SE, Haffner SM, Heise MA, ADOPT Study Group et al (2006) Glycemic durability of rosiglitazone, metformin, or glyburide monotherapy. N Engl J Med 355:2427–2443\nMonami M, Dicembrini I, Antenore A, Mannucci E (2011) Dipeptidyl Peptidase 4 inhibitors and bone fractures. A meta-analysis of randomized clinical trials. Diabetes Care 34:2474–2476\nGough S, Belda-Iniesta C, Poole C, Weber M, Russell-Jones D, Falck-Hansen B, Mannucci E, Tuomilehto J (2011) Insulin therapy in diabetes and cancer risk: current understanding and implications for future study. Adv Ther 28(Suppl 5):1–18\nLewis JD, Ferrara A, Peng T, Hedderson M, Bilker WB, Quesenberry CP Jr, Vaughn DJ, Nessel L, Selby J, Strom BL (2011) Risk of bladder cancer among diabetic patients treated with pioglitazone: interim report of a longitudinal cohort study. Diabetes Care 34:916–922\nGuppy A, Jamal-Hanjani M, Pickering L (2011) Anticancer effects of metformin and its potential use as a therapeutic agent for breast cancer. Future Oncol 7:727–736\nBecker DM, Segal J, Vaidya D et al (2006) Sex differences in platelet reactivity and response to low-dose aspirin therapy. JAMA 295:1420–1427\nShen H, Herzog W, Drolet M et al (2009) Aspirin resistance in healthy drug-naive men versus women (from the Heredity and Phenotype Intervention Heart Study). Am J Cardiol 104(4):606–612 (Epub 2009 Jun 21)\nGum PA, Kottke-Marchant K, Poggio ED et al (2001) Profile and prevalence of aspirin resistance in patients with cardiovascular disease. Am J Cardiol 88:230–235\nLee PY, Chen WH, Ng W et al (2005) Low-dose aspirin increases aspirin resistance in patients with coronary artery disease. Am J Med 118:723–727\nBerger JS, Roncaglioni MC, Avanzini F et al (2006) Aspirin for the primary prevention of cardiovascular events in women and men: a sex-specific meta-analysis of randomized controlled trials. JAMA 295:306–313 (Erratum in: JAMA. 2006;295:2002)\nButalia S, Leung AA, Ghali W et al (2011) Aspirin effect on the incidence of major adverse cardiovascular events in patients with diabetes mellitus: a systematic review and meta-analysis. Cardiovasc Diabetol 10:25\nRidker PM, Cook NR, Lee IM et al (2005) A randomized trial of low-dose aspirin in the primary prevention of cardiovascular disease in women. N Engl J Med 352:1293–1304\nBerger JS, Bhatt DL, Cannon CP et al (2009) The relative efficacy and safety of clopidogrel in women and men a sex-specific collaborative meta-analysis. J Am Coll Cardiol 54:1935–1945\nWannamethee SG, Papacosta O, Lawlor DA et al (2012) Do women exhibit greater differences in established and novel risk factors between diabetes and non-diabetes than men? The British Regional Heart Study and British Women’s Heart Health Study. Diabetologia 55:80–87\nMehta SS, Silver RJ, Aaronson A et al (2006) Comparison of aspirin resistance in type 1 versus type 2 diabetes mellitus. Am J Cardiol 97:567–570\nDe Berardis G, Sacco M, Strippoli GFM et al (2009) Aspirin for primary prevention of cardiovascular events in people with diabetes: meta-analysis of randomised controlled trials. BMJ 339:b4531\nOng G, Davis TM, Davis WA et al (2010) Aspirin is associated with reduced cardiovascular and all-cause mortality in type 2 diabetes in a primary prevention setting. The Fremantle Diabetes Study. Diabetes Care 33:317–321\nAmerican Diabetes Association, American Heart Association, American College of Cardiology Foundation, Pignone M, Alberts MJ, Colwell JA et al (2010). Aspirin for primary prevention of cardiovascular events in people with diabetes. J Am Coll Cardiol 55:2878–2886\nETDRS Investigators (1992) Aspirin effects on mortality and morbidity in patients with diabetes mellitus. Early Treatment Diabetic Retinopathy Study report 14. JAMA 268:1292–1300",{"EN":116},"The prevalence and incidence of diabetes is similar in the two sexes but the long-term impact of diabetes on vascular and non-vascular complications is more gender specific. Men, in comparison with women, seem to be at higher risk for micro-vascular complications, such as severe retinopathy and nephropathy. On the other hand, the impact of diabetes on the risk of major cardiovascular events is different in women in comparison with men. Both type 1 and type 2 diabetes are associated with a significant increase in the incidence of bone fractures. Although this phenomenon is present in both sexes, its impact is greater in women, due to the higher baseline incidence of fractures. Diabetes negatively affects mood, leading to an increased risk of depressive disorders, due to the burden and side effects of therapy, together with the fear of complications. This phenomenon can be more evident in women, who are at greater risk of depressive disorders. Non-pharmacological treatments (i.e. diet and exercise), which are the backbone of therapy for type 2 diabetes, do not differ across genders. On the other hand, some drugs could have diverse profiles of action in women and in men. In relation to diabetes, the sex-related difference in platelet activity and platelet inhibitory response to anti-aggregating therapy, reported in the general population, was observed also in diabetic women.",{"EN":118},"Diabetes and sex: from pathophysiology to personalized medicine",{"VOID":120},"10.1007\u002Fs11739-012-0804-y","PUBLICATION","VERIFIED","Auto 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vein thrombosis (DVT) in critically ill patients still represents a clinical challenge. The aim of the study was to investigate whether a systematic ultrasound (US) screening might improve the management of the antithrombotic therapy in intensive care unit (ICU). In this non-randomized diagnostic clinical trial, 100 patients consecutively admitted to ICU of the University Hospital of Perugia were allocated either in the screening group or in the control group. Subjects in the screening group underwent US examination of lower limbs 48 h after admission, and again after 5 days. Subjects in the control group underwent US examination according to the standard of care (SOC) of the enrolling institution. Retrospectively registered at ClinicalTrials.gov (NCT05019092) on 24.08.2021. Lower limb DVT was significantly more frequent in the screening group (\u003Cjats:italic>p\u003C\u002Fjats:italic> < 0.001), as well as the subsequent extension of a pre-existing DVT (\u003Cjats:italic>p\u003C\u002Fjats:italic> = 0.027). In the control group, DVT of large veins was more frequent (\u003Cjats:italic>p\u003C\u002Fjats:italic> = 0.038). Major bleedings were reported in 5 patients, 4 in the non-screening group and in 1 in the screening group. Patients in the screening group started the antithrombotic treatment later (\u003Cjats:italic>p\u003C\u002Fjats:italic> = 0.038), although the frequency, dose and duration of the treatment were not different between the two groups. The duration of stay in ICU was longer in the screening group (\u003Cjats:italic>p\u003C\u002Fjats:italic> = 0.007). Active screening for DVT is associated with an increased diagnosis of DVT. The screening could be associated with a reduced incidence of proximal DVT and a reduction in the bleeding risk.\u003C\u002Fjats:p>",{"EN":248},"Ultrasound screening for asymptomatic deep vein thrombosis in critically ill patients: a pilot 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Ann Intern Med 156:785–795\nHarrell FE Jr, Lee KL, Mark DB (1996) Multivariable prognostic models: issues in developing models, evaluating assumptions and adequacy, and measuring and reducing errors. Stat Med 15:361–387\nMalek MH, Berger DE, Coburn JW (2007) On the inappropriateness of stepwise regression analysis for model building and testing. Eur J Appl Physiol 101:263–264 (author reply 265–266)\nAhluwalia SC, Gross CP, Chaudhry SI, Leo-Summers L, Van Ness PH, Fried TR (2011) Change in comorbidity prevalence with advancing age among persons with heart failure. J Gen Intern Med 26:1145–1151\nMartin-Sanchez FJ, Marino-Genicio R, Rodriguez-Adrada E, Jacob J, Herrero P, Miro O et al (2013) Management of acute heart failure in spanish emergency departments based on age. Rev Esp Cardiol (Engl Ed) 66:715–720\nLee DS, Ezekowitz JA (2014) Risk stratification in acute heart failure. 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Eur Heart J 28:980–988\nButler J, Subacius H, Vaduganathan M, Fonarow GC, Ambrosy AP, Konstam MA et al (2013) Relationship between clinical trial site enrollment with participant characteristics, protocol completion, and outcomes: insights from the EVEREST (efficacy of vasopressin antagonism in heart failure: outcome study with tolvaptan) trial. J Am Coll Cardiol 61:571–579\nHsich EM, Pina IL (2009) Heart failure in women: a need for prospective data. J Am Coll Cardiol 54:491–498\nGalvao M, Kalman J, DeMarco T, Fonarow GC, Galvin C, Ghali JK et al (2006) Gender differences in in-hospital management and outcomes in patients with decompensated heart failure: analysis from the Acute Decompensated Heart Failure National Registry (ADHERE). J Card Fail 12:100–107\nNieminen MS, Brutsaert D, Dickstein K, Drexler H, Follath F, Harjola VP et al (2006) EuroHeart Failure Survey II (EHFS II): a survey on hospitalized acute heart failure patients: description of population. Eur Heart J 27:2725–2736\nSarma S, Mentz RJ, Kwasny MJ, Fought AJ, Huffman M, Subacius H et al (2013) Association between diabetes mellitus and post-discharge outcomes in patients hospitalized with heart failure: findings from the EVEREST trial. Eur J Heart Fail 15:194–202\nJencks SF, Williams MV, Coleman EA (2009) Rehospitalizations among patients in the Medicare fee-for-service program. N Engl J Med 360:1418–1428\nRoss JS, Mulvey GK, Stauffer B, Patlolla V, Bernheim SM, Keenan PS et al (2008) Statistical models and patient predictors of readmission for heart failure: a systematic review. Arch Intern Med 168:1371–1386\nVashi AA, Fox JP, Carr BG, D’Onofrio G, Pines JM, Ross JS et al (2013) Use of hospital-based acute care among patients recently discharged from the hospital. JAMA 309:364–371\nDi Tano G, De Maria R, Gonzini L, Aspromonte N, Di Lenarda A, Feola M et al (2015) The 30-day metric in acute heart failure revisited: data from IN-HF Outcome, an Italian nationwide cardiology registry. Eur J Heart Fail. doi:10.1002\u002Fejhf.290\nTeerlink JR, Metra M, Felker GM, Ponikowski P, Voors AA, Weatherley BD et al (2009) Relaxin for the treatment of patients with acute heart failure (Pre-RELAX-AHF): a multicentre, randomised, placebo-controlled, parallel-group, dose-finding phase IIb study. Lancet 373:1429–1439\nFonarow GC, Peacock WF, Horwich TB, Phillips CO, Givertz MM, Lopatin M et al (2008) Usefulness of B-type natriuretic peptide and cardiac troponin levels to predict in-hospital mortality from ADHERE. Am J Cardiol 101:231–237",{"EN":610},"Acute heart failure (AHF) is a major public health issue due to high incidence and poor prognosis. Only a few studies are available on the long-term prognosis and on outcome predictors in the unselected population attending the emergency department (ED) for AHF. We carried out a 1-year follow-up analysis of 1234 consecutive patients from selected Italian EDs from January 2011 to June 2012 for an episode of AHF. Their prognosis and outcome-associated factors were tested by Cox proportional hazard model. Patients’ mean age was 84, with 66.0 % over 80 years and 56.2 % females. Comorbidities were present in over 50 % of cases, principally a history of acute coronary syndrome, chronic obstructive pulmonary disease, diabetes, chronic kidney disease, valvular heart disease. Death occurred within 6 h in 24 cases (1.9 %). At 30-day follow-up, death was registered in 123 cases (10.0 %): 110 cases (89.4 %) died of cardiovascular events and 13 (10.6 %) of non-cardiovascular causes (cancer, gastrointestinal hemorrhages, sepsis, trauma). At 1-year follow-up, all-cause death was recorded in 50.1 % (over 3 out of 4 cases for cardiovascular origin). Six variables (older age, diabetes, systolic arterial pressure \u003C110 mm\u002FHg, high NT pro-BNP, high troponin levels and impaired cognitive status) were selected as outcome predictors, but with limited discriminant capacity (AUC = 0.649; SE 0.015). Recurrence of AHF was registered in 31.0 %. The study identifies a cluster of variables associated with 1-year mortality in AHF, but their predictive capacity is low. 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J Hepatol 63:743–752\nHelmy A, Al Kahtani K, Al Fadda M (2008) Updates in the pathogenesis, diagnosis and management of ectopic varices. Hepatol Int 2:322–334\nCheng LF, Wang ZQ, Li CZ, Lin WU, Yeo AE, Jin BO (2010) Low incidence of complications from endoscopic gastric variceal obturation with butyl cyanoacrylate. Clin Gastroenterol Hepatol 8:760–766\nLo GH, Lai KH, Cheng JS, Chen MH, Chiang HT (2001) A prospective, randomized trial of butyl cyanoacrylate injection versus band ligation in the management of bleeding varices. Hepatology 33:1060–1064\nRomero-Castro R, Ellrichmann M, Ortiz-Moyano C et al (2013) EUS-guided coil versus cyanoacrylate therapy for the treatment of gastric varices: a multicentre study (with videos). Gastrointest Endosc 78:711–721",{"EN":786},"Cerebral infarction following cyanoacrylate endoscopic therapy of duodenal varices in a patient with a patent foramen ovale",{"VOID":788},"10.1007\u002Fs11739-018-1892-0","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs11739-018-1892-0",[791,818,840,879,898],{"id":792,"sortIndex":185,"researcher":22,"roles":793,"affiliations":794,"properties":815},"31cd5564-a65d-437d-9cd6-982bf552559d",[129],[795,805],{"id":22,"sortIndex":23,"affiliation":796,"properties":22},{"id":797,"createTime":798,"updateTime":799,"relativeEntities":800,"slug":801,"properties":802,"entityType":51,"verifyStatus":21,"verifyTime":22,"verifyNote":22,"syncStatus":21,"languages":22,"translateLanguages":22,"viewCount":23},"1732f9b7-1e32-4902-9485-001ce0904059","2024-01-09T21:21:37.704+00:00","2024-10-13T14:20:53.281+00:00",[],"Universit%C3%A9-Paris-Descartes-Sorbonne-Paris-Cit%C3%A9-Paris-France",{"title":803},{"VI":804},"Université Paris Descartes, 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TA, Metra M, Adamo M, Gardner RS, Baumbach A, Böhm M, Burri H, Butler J, Čelutkienė J, Chioncel O, Cleland JGF, Coats AJS, Crespo-Leiro MG, Farmakis D, Gilard M, Heymans S, Hoes AW, Jaarsma T, Jankowska EA, Lainscak M, Lam CSP, Lyon AR, McMurray JJV, Mebazaa A, Mindham R, Muneretto C, Francesco Piepoli M, Price S, Rosano GMC, Ruschitzka F, Kathrine Skibelund A; ESC Scientific Document Group (2021) ESC Guidelines for the diagnosis and treatment of acute and chronic heart failure. Eur Heart J 2021(42):3599–3726\nOlson TP, Beck KC, Johnson JB, Johnson BD (2006) Competition for intrathoracic space reduces lung capacity in patients with chronic heart failure: a radiographic study. Chest 130:164–171\nVerissimo P, Timenetsky KT, Casalaspo TJA, Gonçalves LHR, Yang ASY, Eid RC (2015) High prevalence of respiratory muscle weakness in hospitalized acute heart failure elderly patients. PLoS ONE 10(2):e0118218\nLena A, Anker MS, Springer J (2020) Muscle wasting and sarcopenia in heart failure—the current state of science. Int J Mol Sci 21:6549\nMiyagi M, Kinugasa Y, Sota T, Yamada K, Ishisugi T, Hirai M, Yanagihara K, Haruki N, Matsubara K, Kato M, Yamamoto K (2018) Diaphragm muscle dysfunction in patients with heart failure. J Card Fail 24:209–216\nKinugasa Y, Miyagi M, Sota T, Yamada K, Ishisugi T, Hirai M, Yanagihara K, Haruki N, Matsubara K, Kato M, Yamamoto K (2018) Dynapenia and diaphragm muscle dysfunction in patients with heart failure. Eur J Prev Cardiol 25:1785–1786\nSpiesshoefer J, Henke C, Kabitz HJ, Bengel P, Schütt K, Nofer JR, Spieker M, Orwat S, Diller GP, Strecker JK, Giannoni A, Dreher M, Randerath WJ, Boentert M, Tuleta I (2021) Heart failure results in inspiratory muscle dysfunction irrespective of left ventricular ejection fraction. Respiration 100:96–108\nScarlata S, Mancini D, Laudisio A, Raffaele AI (2019) Reproducibility of diaphragmatic thickness measured by M-mode ultrasonography in healthy volunteers. Respir Physiol Neurobiol 260:58–62\nScarlata S, Mancini D, Laudisio A, Benigni A, Antonelli Incalzi R (2018) Reproducibility and clinical correlates of supine diaphragmatic motion measured by M-mode ultrasonography in healthy volunteers. Respiration 96:259–266\nLang RM, Badano LP, Mor-Avi V, Afilalo J, Armstrong A, Ernande L et al (2015) Recommendations for cardiac chamber quantification by echocardiography in adults: an update from the American society of echocardiography and the European association of cardiovascular imaging. J Am Soc Echocardiogr 28:1–39\nVolpicelli G, Elbarbary M, Blaivas M, Lichtenstein DA, Mathis G, Kirkpatrick AW, International Liaison Committee on Lung Ultrasound (ILC-LUS) for the International Consensus Conference on Lung Ultrasound (ICC-LUS) (2012) International evidence-based recommendations for point-of-care lung ultrasound. Intensive Care Med 38:577–591\nKelley RC, Ferreira LF (2017) Diaphragm abnormalities in heart failure and aging: mechanisms and integration of cardiovascular and respiratory pathophysiology. Heart Fail Rev 22:191–207\nArutyunov AG, Ilyina KV, Arutyunov GP, Kolesnikova EA, Pchelin VV, Kulagina NP, Tokmin DS, Tulyakova EV (2019) Morphofunctional features of the diaphragm in patients with chronic heart failure. Kardiologiia 59:12–21\nMangner N, Garbade J, Heyne E, van den Berg M, Winzer EB, Hommel J, Sandri M, Jozwiak-Nozdrzykowska J, Meyer AL, Lehmann S, Schmitz C, Malfatti E, Schwarzer M, Ottenheijm CAC, Bowen TS, Linke A, Adams V (2021) Molecular mechanisms of diaphragm myopathy in humans with severe heart failure. Circ Res 128(6):706–719\nMantilla CB, Sieck GC (2003) Plasticity in respiratory motor control. Invited review: mechanisms underlying motor unit plasticity in the respiratory system. J Appl Physiol 94:1230–1241\nManders E, Rain S, Bogaard HJ, Handoko ML, Stienen GJ, Vonk-Noordegraaf A, Ottenheijm CA, de Man FS (2015) The striated muscles in pulmonary arterial hypertension: adaptations beyond the right ventricle. Eur Respir J 46:832–842\nManders E, Bonta PI, Kloek JJ, Symersky P, Bogaard HJ, Hooijman PE, Jasper JR, Malik FI, Stienen GJ, Vonk-Noordegraaf A, de Man FS, Ottenheijm CA (2016) Reduced force of diaphragm muscle fibers in patients with chronic thromboembolic pulmonary hypertension. Am J Physiol Lung Cell Mol Physiol 311(1):L20–L28\nJorbenadze A, Goldberg LR, Shaburishvili T, Zuber M, Mirro M, Fudim M (2022) Synchronized diaphragmatic stimulation for heart failure with a reduced left ventricular ejection fraction using the VisONE system: a first-in-patient study with extended population. Struct Heart 6(6):100103\nZuber M, Roos M, Kobza R, Toggweiler S, Meier R, Erne P (2010) Detection and hemodynamic significance of cardiac pacemaker-induced phrenic nerve stimulation. Congest Heart Fail 16:147–152\nFudim M, Abraham WT, von Bardeleben RS et al (2021) Device therapy in chronic heart failure: JACC state-of-the-art review. J Am Coll Cardiol 78(9):931–956\nGargani L, Girerd N, Platz E, Pellicori P, Stankovic I, Palazzuoli A, Pivetta E, Miglioranza MH, Soliman-Aboumarie H, Agricola E, Volpicelli G, Price S, Donal E, Cosyns B, Neskovic AN (2023) Lung ultrasound in acute and chronic heart failure. A clinical consensus statement of the european association of cardiovascular imaging (EACVI). Eur Heart J Cardiovasc Imaging 24:169",{"EN":957},"Heart failure patients often experience respiratory symptoms due to diaphragmatic involvement, but the diaphragmatic motion in heart failure remains understudied. This research aimed to investigate the correlation between ultrasonographically assessed diaphragmatic motion and thickness with cardiac performance indexes in an emergency setting. Seventy-two acutely decompensated heart failure patients and 100 non-heart failure individuals were enrolled. Diaphragmatic motion and thickness were assessed via ultrasound. Cardiac and respiratory parameters were recorded, and regression analysis was performed. Heart failure patients exhibited reduced diaphragmatic motion at total lung capacity compared to controls, and an inverse association was found between motion and heart failure severity (NYHA stage). Diaphragmatic thickness was also higher in heart failure patients at tidal volume and total lung capacity. Notably, diaphragmatic motion inversely correlated with systolic pulmonary artery pressure. The study highlights diaphragmatic dysfunction in acutely decompensated heart failure, with reduced motion and increased thickness. These changes were associated with cardio-respiratory parameters, specifically systolic pulmonary artery pressure. Monitoring diaphragmatic motion via ultrasound may aid in evaluating heart failure severity and prognosis in emergency settings. Additionally, interventions targeting diaphragmatic function could improve heart failure management. Further research is warranted to enhance heart failure management and patient outcomes.",{"EN":959},"Diaphragmatic ultrasound evaluation in acute heart failure: clinical and functional associations",{"VOID":961},"10.1007\u002Fs11739-024-03531-9","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs11739-024-03531-9",[964,989,1001,1016,1035,1050,1062,1074,1086,1098,1110],{"id":965,"sortIndex":463,"researcher":22,"roles":966,"affiliations":967,"properties":986},"bb3c8f9a-9844-4449-af17-50218572d11f",[129],[968,976],{"id":22,"sortIndex":23,"affiliation":969,"properties":22},{"id":970,"createTime":971,"updateTime":971,"relativeEntities":972,"slug":22,"properties":973,"entityType":51,"verifyStatus":21,"verifyTime":22,"verifyNote":22,"syncStatus":21,"languages":22,"translateLanguages":22,"viewCount":23},"ae59ac23-11a9-435a-af63-a53adfe3b61d","2024-02-18T23:03:32.638+00:00",[],{"title":974},{"VI":975},"Unit of Internal Medicine, Fondazione Policlinico Universitario Campus 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G, Verso M (2006) Therapy insight: venous catheter-related thrombosis in cancer patients. Nat Clin Pract Oncol 3(4):214–222\nAgnelli G, Verso M (2005) Thrombosis and cancer: clinical relevance of a dangerous liaison. Haematologica 90(2):154–156\nVerso M, Agnelli G (2003) Venous thromboembolism associated with long-term use of central venous catheters in cancer patients. J Clin Oncol 21:3665–3675\nBern HM, Lokich JJ, Wallach SR et al (1990) Very low dose of warfarin can prevent thrombosis in central venous catheters: a randomized, prospective trial. Ann Inter Med 112:423–428\nMonreal M, Alastrue A, Rull M et al (1996) Upper extremity deep venous thrombosis in cancer patients with venous access devices. Prophylaxis with a low molecular weight heparin (Fragmin). Thromb Haemost 75:251–253\nKarthaus M, Kretzschmar A, Kröning H et al (2006) A Dalteparin for prevention of catheter-related complications in cancer patients with central venous catheters: final results of a double-blind, placebo-controlled phase III trial. Ann Oncol 17(2):289–296\nCouban S, Goodyear M, Burnell M et al (2005) Randomized placebo-controlled study of low-dose warfarin for the prevention of central venous catheter-associated thrombosis in patients with cancer. J Clin Oncol 23(18):4063–4069\nGeerts WH, Pineo GF, Heit JA et al (2004) Prevention of venous thromboembolism. Chest 126:338S–400S\nYoung AM, Begum G, Billingham LJ (2005) WARP-a multicentre, prospective, randomized controlled trial of thrombosis prophylaxis with warfarin in cancer patients with central venous catheters (CVCs). Ann ASCO Meet 2005; abstr no. 8004\nVerso M, Agnelli G, Bertoglio S et al (2005) Enoxaparin for the prevention of venous thromboembolism associated with central vein catheters: a double-blind, placebo-controlled, randomized study in cancer patients. J Clin Oncol 23(18):4057–4062\nCadman A, Lawrance JAL, Fitzsimmons L, Spencer-Shaw A, Swindell R (2004) To clot or not to clot? That is the question in central venous catheters? Clin Rad 59:349–355\nTesselaar ME, Ouwerkek J, Nooy MA, Rosendaal FR, Osanto S (2004) Risk factors for catheter-related thrombosis in cancer patients. Eur J Cancer 40:2253–2259\nEastridge BJ, Lefor AT (1995) Complication of indwelling venous access devices in cancer patients. J Clin Onc 13(1):233–238\nTimsit JF, Farkas JC, Boyer JM et al (1998) Central vein catheter-related thrombosis in intensive care patients. Incidence, risk factors and relationship with catheter-related sepsis. Chest 114:207–213\nCraft PS, May J, Dorigo A, Hoy C, Plant A (1996) Hickman catheters: left sided insertion, male gender and obesity are associated with an increased risk of complications. Aust NZ J Med 26:33–39\nUnal AE, Bayar S, Arat M, Ihhan O (2003) Malpositioning of Hickman catheters, left versus right sided attempts. Transfus Apheresis Sci 28:9–12\nPetersenJ, Delaney JH, Brakstad MT, Rowbotham RK, Bagley CM (1999) Silicone venous access devices positioned with their tips high in the superior vena cava are more likely to malfunction. Am J Surg 178(1):38–41\nMcGee WT, Ackerman BL, Rouben LR, Prasad VM, Bandi V, Mallory DL (1993) Accurate placement of central venous catheters: a prospective, randomized, multicenter trial. Crit Care Med 21(8):1118–1123\nPuel V, Caudry M, Metayer P et al (1993) Superior vena cava thrombosis related to catheter malposition in cancer chemotherapy given through implanted ports. Cancer 72:2248–2252\nAgnelli G, Verso M (2006) Is antithrombotic prophylaxis required in cancer patients with central venous catheters? No. J Thromb Haemost 4(1):14–15\nLuciani A, Clement O, Halimi P et al (2001) Catheter-related upper extremity deep venous thrombosis in cancer patients: a prospective study based on doppler US. Radiology 220(3):655–660\nKearns PJ, Coleman S, Wehner JH (1996) Complications of long-term catheters: randomized trial of central vs. peripheral tip location. J PEN 20(1):20–24\nGould JR, Carloss HW, Skinner WL (1993) Groshong catheter-associated subclavian venous thrombosis. Am J Med 95(4):419–423\nDe Cicco M, Matovic M, Balestrieri L et al (1997) Central venous thrombosis: an early and frequent complication in cancer patients bearing long term silastic catheter. A prospective study. Thromb Res 86(2):101–113\nBlom JW, Doggen CJM, Osanto S, Rosendaal FR (2005) Malignancies, prothrombotic mutations and the risk of venous thrombosis. JAMA 293:715–722\nRandolph AG, Cook DJ, Gonzales CA, Andrew M (1998) Benefit of heparin in central venous and pulmonary artery catheters: a meta-analysis of randomized controlled trials. Chest 113(1):165–171\nStephens LC, Haire WD, Tarantolo S et al (1997) Normal saline versus heparin flush for maintaining central venous catheter patency during apheresis collection of peripheral blood stem cell. Transfus Sci 18(2):187–193",{"EN":1170},"Deep vein thrombosis of upper limb is a common complication of CVC in patients with cancer. In these patients the risk factors for CVC-related thrombosis are not completely defined. The purpose of this study was to identify the risk factors for CVC-related thrombosis in patients included in a randomized, double-blind, placebo-controlled study aimed at assessing the efficacy and safety of enoxaparin for the prophylaxis of CVC-related thrombosis. CVC-related thrombosis was screened by mandatory venography after 6 weeks of study treatment. A number of patient baseline characteristics were assessed as potential risk factors for CVC-related deep vein thrombosis. Crude associations between risk factors and clinical outcomes were assessed by χ2 test or Fisher’s exact test. Multiple logistic regression analysis was used to identify independent risk factors. A CVC-related thrombosis was found in 50 out of 310 patients (16.1%). At multiple logistic regression analysis, CVC tip misplaced in the upper half of superior vena cava (OR 4.05, 95%CI 1.64–10.02), left-sided CVC insertion (OR 2.29, 95%CI 1.01–5.51) and chest radiotherapy (OR 7.01, 95%CI 1.42–34.66) were independent risk factors for thrombosis. In addition to these risk factors, the presence of distant metastases (OR 9.36, 95%CI 1.53–57.05) increased the risk of thrombosis in patients who received placebo. An inadequate position of the CVC tip, left-sided CVC insertion and chest radiotherapy are independent risk factors for CVC-related thrombosis in cancer patients. Patients with distant metastases have an increased risk for thrombosis in absence of antithrombotic prophylaxis.",{"EN":1172},"Risk factors for upper limb deep vein thrombosis associated with the use of central vein catheter in cancer 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Lancet Oncol. 10(10):943–949\nLee AY, Levine MN, Baker RI, Bowden C, Kakkar AK, Prins M et al (2003) Low-molecular-weight heparin versus a coumarin for the prevention of recurrent venous thromboembolism in patients with cancer. N Engl J Med 349(2):146–153\nLee AY, Kamphuisen PW, Meyer G, Bauersachs R, Janas MS, Jarner MF et al (2015) Tinzaparin vs warfarin for treatment of acute venous thromboembolism in patients with active cancer: a randomized clinical trial. JAMA 314(7):677–686\nHull RD, Pineo GF, Brant RF, Mah AF, Burke N, Dear R et al (2006) Long-term low-molecular-weight heparin versus usual care in proximal-vein thrombosis patients with cancer. Am J Med 119(12):1062–1072\nMeyer G, Marjanovic Z, Valcke J, Lorcerie B, Gruel Y, Solal-Celigny P et al (2002) Comparison of low-molecular-weight heparin and warfarin for the secondary prevention of venous thromboembolism in patients with cancer: a randomized controlled study. Arch Intern Med 162(15):1729–1735\nZwicker JI, Karp RL, Carrier M (2016) A meta-analysis of intracranial hemorrhage in patients with brain tumors receiving therapeutic anticoagulation. J Thromb Haemost 14(9):1736–1740\nEdwin NC, Khoury MN, Sohal D, McCrae KR, Ahluwalia MS, Khorana AA (2016) Recurrent venous thromboembolism in glioblastoma. Thromb Res 137:184–188\nAkl EA, Kahale L, Neumann I, Barba M, Sperati F, Terrenato I et al (2014) Anticoagulation for the initial treatment of venous thromboembolism in patients with cancer. Cochrane Database Syst Rev 6:CD006649\nAlvarado G, Noor R, Bassett R, Papadopoulos NE, Kim KB, Hwu WJ et al (2012) Risk of intracranial hemorrhage with anticoagulation therapy in melanoma patients with brain metastases. Melanoma Res 22(4):310–315\nKahn SR, Lim W, Dunn AS, Cushman M, Dentali F, Akl EA et al (2012) Prevention of VTE in nonsurgical patients: antithrombotic Therapy and Prevention of Thrombosis, 9th ed: American College of Chest Physicians Evidence-Based Clinical Practice Guidelines. Chest 141(2 Suppl):e195S–e226S\nKearon C, Akl EA, Ornelas J, Blaivas A, Jimenez D, Bounameaux H et al (2016) Antithrombotic therapy for VTE disease: CHEST Guideline and Expert Panel Report. Chest 149(2):315–352\nKhorana AA, Carrier M, Garcia DA, Lee AY (2016) Guidance for the prevention and treatment of cancer-associated venous thromboembolism. J Thromb Thrombolysis 41(1):81–91\nFarge D, Debourdeau P, Beckers M, Baglin C, Bauersachs RM, Brenner B et al (2013) International clinical practice guidelines for the treatment and prophylaxis of venous thromboembolism in patients with cancer. J Thromb Haemost 11(1):56–70\nLyman GH, Bohlke K, Khorana AA, Kuderer NM, Lee AY, Arcelus JI et al (2015) Venous thromboembolism prophylaxis and treatment in patients with cancer: american society of clinical oncology clinical practice guideline update 2014. J Clin Oncol 33(6):654–656\nLyman GH, Khorana AA, Kuderer NM, Lee AY, Arcelus JI, Balaban EP et al (2013) Venous thromboembolism prophylaxis and treatment in patients with cancer: American Society of Clinical Oncology clinical practice guideline update. J Clin Oncol 31(17):2189–2204",{"EN":1505},"The decision concerning the introduction of primary and secondary prophylaxis of venous thromboembolism (VTE) in patients with solid brain neoplasms and brain metastases is often challenging due to the concomitant increased risk of intracranial hemorrhage and to limited evidence from available literature. A standardized questionnaire composed of nine multiple-choice questions regarding primary VTE prevention in non-surgical patients during high-risk conditions and VTE secondary prevention in patients with a solid brain neoplasm or cerebral metastases was sent via electronic mail to all the members (n = 2420) of the Italian Federation of the Internal Medicine Hospital Executives’ Associations (FADOI) in June 2015. Three hundred and fifty two physicians (14.5%) returned it (participants' median age 51 years; females 46.9%). The majority of respondents prescribe primary thromboprophylaxis (usually with heparin) in non-surgical patients with solid brain neoplasms and brain metastases in concomitance with high-risk conditions. Full-dose anticoagulation with either low-molecular-weight heparin or fondaparinux is the preferred option for acute VTE (69.6%), while a reduced dose is chosen by 21.0% of physicians. The presence of a highly vascular brain neoplasm histotype mandates the prescription of a reduced-dose antithrombotic regimen in a minority of respondents. Vena cava filter placement is an option for the treatment of acute VTE in more than 6% of respondents. Anticoagulants are often prescribed for both VTE primary prevention and treatment. 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