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Reproducibility of M-mode echocardiographic assessment of left ventricular function. Significance of the temporal range of measurements. Eur Heart J 1984;5: 412-8.",{"doi":754},"10.1093\u002Foxfordjournals.eurheartj.a061675",{"id":23,"text":756,"url":23,"identifiers":757},"Roelandt J, Gibson DG. Recommendations for the standardization of measurements for M-mode echocardiograms. Eur HeartJ 1980;1:375-8.",{"doi":758},"10.1093\u002Feurheartj\u002F1.5.375",{"id":23,"text":760,"url":23,"identifiers":761},"Bowman LK, Lee FA, Jaffe CC, Mattera J, Wackers FJT, Zaret BL. Peak filling rate normalized to mitral stroke volume: a new Doppler echocardiographic filling index validated by radionuclide angiographic techniques. J Am Coll Cardiol 1988;12:937-43.",{"doi":762},"10.1016\u002F0735-1097(88)90458-5",{"id":23,"text":764,"url":23,"identifiers":765},"Bland JM, Altman DG. Statistical methods for assessing agreement between two methods of clinical measurement. Lancet 1986;i:307-10.",{"doi":766},"10.1016\u002FS0140-6736(86)90837-8",{"id":23,"text":768,"url":23,"identifiers":769},"Fast J, van Dam I, Heringa A, et al. Limits of reproducibility of mitral pulsed Doppler spectra. Am J Cardiol 1988;61: 891-4.",{"doi":770},"10.1016\u002F0002-9149(88)90365-7",{"id":23,"text":772,"url":23,"identifiers":773},"Spirito P, Maron BJ, Verter I, Merrill JS. Reproducibility of Doppler echocardiographic measurements of left ventricular diastolic function. Eur Heart J 1988;9:879-86.",{"doi":774},"10.1093\u002Foxfordjournals.eurheartj.a062582",{"id":23,"text":776,"url":23,"identifiers":777},"Jennrich R, Sampson P, Frane J. Analysis of variance and covariance including repeated measures. In: Dixon WS, Brown MB, Engelman L, et al. BMDP statistical software. Berkeley, California: University of California Press, 1985: 359-87.",{},{"id":23,"text":779,"url":23,"identifiers":780},"Nishimura RA, Housmans PR, Hatle LK, Tajik AJ. Assessment of diastolic function of the heart: background and current applications of Doppler echocardiography. Part II. Clinical studies. Mayo Clin Proc 1989;64:181-204.",{},{"id":23,"text":782,"url":23,"identifiers":783},"Ferguson JJ, Manning WJ, Come PC. Pulsed Doppler echocardiographic determination of the time course of left ventricular filling: validation with cineangiography. Am Heart J 1989;117:127-32.",{"doi":784},"10.1016\u002F0002-8703(89)90666-2",{"id":23,"text":786,"url":23,"identifiers":787},"Appleton CP, Hatle LK, Popp RL. Relation of transmitral flow velocity patterns to left ventricular diastolic function: new insights from a combined hemodynamic and Doppler echocardiographic study. J Am Coll Cardiol 1988;12: 426-40.",{"doi":788},"10.1016\u002F0735-1097(88)90416-0",{"id":23,"text":790,"url":23,"identifiers":791},"Smith SA, Stoner JE, Russell AE, Sheppard JM, Aylward PE. Transmitral velocities measured by pulsed Doppler in healthy volunteers: effects of acute changes in blood pressure and heart rate. Br Heart J 1989;61:344-7.",{"doi":792},"10.1136\u002Fhrt.61.4.344",{"id":23,"text":794,"url":23,"identifiers":795},"Choong CY, Herrman HC, Weyman AE, Fifer MA. Preload dependence of Doppler-derived indexes of left ventricular diastolic function in humans. J Am Coll Cardiol 1987;10: 800-8.",{"doi":796},"10.1016\u002FS0735-1097(87)80273-5",{"id":23,"text":798,"url":23,"identifiers":799},"Koskinen P, Kupari M, Nieminen MS, et al. Effects of alcohol on systemic and pulmonary hemodynamics in normal humans. Clin Cardiol 1986;9:479-82.",{"doi":800},"10.1002\u002Fclc.4960091002",{"id":23,"text":802,"url":23,"identifiers":803},"Greenberg BH, Shutz R, Grunkemeier GL, Griswold H. Acute effects of alcohol in patients with congestive heart failure. Ann Intern Med 1982;97:171-5.",{"doi":804},"10.7326\u002F0003-4819-97-2-171",{"id":23,"text":806,"url":23,"identifiers":807},"Bhatia SJS, Theard MA, Plappert T, St John Sutton MG. Effects of altered loading conditions on transmitral Doppler blood flow velocity [Abstract]. Circulation 1987;76 (suppl IV):124.",{},{"id":23,"text":809,"url":23,"identifiers":810},"Thomas G, Haider B, Oldewurtel HA, Lyons MM, Yeh CK, Regan TJ. Progression of myocardial abnormalities in experimental alcoholism. Am J Cardiol 1980;46:233-41.",{"doi":811},"10.1016\u002F0002-9149(80)90063-6",{"id":23,"text":813,"url":23,"identifiers":814},"Ahmed SS, Levinson GE, Fiore JJ, Regan TJ. Spectrum of heart muscle abnormalities related to alcoholism. Clin Cardiol 1980;3:335-41.",{"doi":815},"10.1002\u002Fclc.4960030408",{"id":23,"text":817,"url":23,"identifiers":818},"Silberbauer K, Juhasz M, Ohrenberger G, Hess C. Noninvasive assessment of left ventricular diastolic function by pulsed Doppler echocardiography in young alcoholics. Cardiology 1988;75:431-9.",{"doi":819},"10.1159\u002F000174413",{"id":821,"createTime":822,"updateTime":823,"relativeEntities":824,"slug":825,"properties":826,"entityType":232,"verifyStatus":233,"verifyTime":843,"verifyNote":235,"languages":844,"translateLanguages":23,"viewCount":24,"primaryUrl":845,"fullTextUrl":23,"authors":846,"publicationType":303,"publisherRelationship":868,"citationCount":182,"citationInfo":914,"publishDate":917,"publishYear":915,"citationAnalyzeStatus":22,"lastCitationAnalyze":823,"indexDatabases":918,"openAccess":23,"references":919,"isForceReanalyzing":358},"4c2cb86c-81af-4870-8113-0cca202b34f1","2024-08-31T18:01:23.678+00:00","2026-05-22T12:05:22.469+00:00",[],"Arterial-stiffness-association-with-chronic-inflammatory-disorders-in-the-UK-Biobank-study",{"mag":827,"gsPaper":829,"pmc":831,"openalex":833,"abstract":835,"title":837,"pm":839,"doi":841},{"VOID":828},"2781804242",{"VOID":830},"[\"1156882448047351187\"]",{"VOID":832},"6204972",{"VOID":834},"W2781804242",{"EN":836},"\u003Cjats:sec>\u003Cjats:title>Objective\u003C\u002Fjats:title>\u003Cjats:p>The present study tested the hypothesis that arterial stiffness will be elevated across overall and specific inflammatory disorders compared with an inflammation-free comparison group.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Methods\u003C\u002Fjats:title>\u003Cjats:p>Adults (n=171 125) aged 40–70 years from the UK Biobank who were cardiovascular disease (CVD) free and who had their arterial stiffness assessed at the time of study recruitment between 2006 and 2010 were included. The main exposure was represented by a global measure of chronic inflammatory disorders. Two inflammatory biomarker measures (eg, leucocytes count, granulocytes count) were included as markers of inflammation severity. The arterial stiffness index assessed by a non-invasive technique represented the study primary outcome measure.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Results\u003C\u002Fjats:title>\u003Cjats:p>A total of 5976 (3%) participants diagnosed with inflammatory disorders and 165 149 participants without an inflammatory disorder had data on arterial stiffness. Adjusted linear regression analyses revealed a 14% increment in mean arterial stiffness for chronic inflammatory disorders (beta coefficient (β) 1.14, 95% CI 1.05 to 1.24, P=0.002) compared with no chronic inflammatory disorder. Arterial stiffness tended to increase (P value=0.031) with tertiles of leucocytes and granulocytes count. For instance, mean arterial stiffness values increased from 1.11 (95% CI 0.96 to 1.29) in the first tertile to 1.17 (95% CI 1.02 to 1.34) in the second tertile, and 1.21 (95% CI 1.05 to 1.39) in the third tertile of leucocytes count. There was evidence for similar associations with some of the most common individual inflammatory disorders, including psoriasis and rheumatoid arthritis.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>\u003Cjats:sec>\u003Cjats:title>Conclusion\u003C\u002Fjats:title>\u003Cjats:p>Arterial stiffness was associated with multiple chronic inflammatory disorders. An increasing trend in mean arterial stiffness was also documented with increasing tertiles of different inflammatory biomarkers. Future studies are needed to investigate the discriminant value of arterial stiffness to predict major CVD events within various inflammatory disorders.\u003C\u002Fjats:p>\u003C\u002Fjats:sec>",{"EN":838},"Arterial stiffness association with chronic inflammatory disorders in the UK Biobank 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Increased arterial stiffness in systemic lupus erythematosus (SLE) patients at low risk for cardiovascular disease: a cross-sectional controlled study. 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