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Journal of Medicine and Pharmacy","Tạp chí Y Dược học Cần Thơ",{"EN":487,"VI":488},"\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">04\u002F10\u002F2015 Ministry of Information and Communications allowed Can Tho journal of medicine and pharmacy to operate (102 \u002FGP-BTTTT)\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">07\u002F16\u002F2015 Can Tho journal of medicine and pharmacy is internationally recognized: ISSN 2354-1210\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">In 2016, The journal has been included in the list of medical science journals by The State Council for professorship which is awarded a work score of 0-0.5 points for a published article.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Can Tho Journal of Medicine and Pharmacy welcome original works that haven’t been submitted or published in other medical journals. Posts must contain content related to one of the journal’s categories.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The content published\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The journal is divided into 3 categories:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Scientific research article: are valuable scientific works, which have been researched and accepted.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Overview of medicine, biology and pharmacy: serving the objective of continuing training in the fields of medicine, biology and pharmacy; to systematize classical and modern knowledge.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Update information on new knowledge about medicine, biology, pharmacy in the country and in the world.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Scope\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Publication and introduction of scientific research in the fields:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Medicine (internal medicine, surgery, pediatrics, obstetrics and gynecology, odonto-stomatology, laboratory, oncology, traditional medicine, nursing).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Biology (genetics, biotechnology).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Pharmacology (pharmaceutics, drug quality analysis-control, synthetic pharmaceutical chemistry, biochemistry, pharmacognosy, botany, clinical pharmacy).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- To enhance the quality of undergraduate, postgraduate education, scientifically researching and meet the necessary treatment in hospital.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Introducing the updated domestic and oversea information about science technology to promote scientific research and exchanging technology in local, other universities.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Exchanging pharmaceutical and medical information for social health developing in the Mekong Delta and Vietnam.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The object\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Postgraduate students, student of Can Tho University of Medicine and Pharmacy, scientists from schools, research institutes, hospitals, health centers, pharmaceutical companies of the Mekong Delta; other provinces and regions in Vietnam and other country.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Address\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Headquarters of Can Tho Journal of Medicine and Pharmacy, located Scientific Research and International Cooperation Office: 179 Nguyen Van Cu Street, An Khanh Ward, Ninh Kieu District, Can Tho City, Vietnam.\u003C\u002Fspan>\u003C\u002Fp>","\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Ngày 16\u002F7\u002F2015, Tạp chí Y Dược học Cần Thơ được cấp chỉ số quốc tế: ISSN 2354-1210.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 4\u002F2016, Tạp chí đã được Hội đồng Giáo sư ngành Y đưa vào danh sách các tạp chí khoa học Y học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Năm 2020 Tạp chí Y Dược học Cần Thơ đã được phê duyệt vào danh mục của các Hội đồng Giáo sư ngành Dược học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ ra 12 số\u002Fnăm, 180-200 trang\u002Fsố.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 12\u002F2022 Tạp chí Y Dược học Cần Thơ là thành viên của hệ thống Crossref và từ tháng 01\u002F2023 tạp chí thực hiện bình duyệt online kín 2 chiều nhằm tăng tính minh bạch, tin cậy của các công trình nghiên cứu khoa học và đảm bảo tốt nhất chất lượng khoa học của bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ, mục đích và phạm vi của tạp chí\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ và mục đích hoạt động của tạp chí: xuất bản nhằm mục đích phổ biến kết quả từ các đề tài nghiên cứu khoa học; giao lưu trao đổi khoa học, chia sẻ kinh nghiệm, học tập, đồng thời cập nhật thông tin khoa học mới trong các lĩnh vực y, sinh, dược học trong và ngoài nước.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phạm vi của tạp chí: Tạp chí xuất bản được chia thành 3 chuyên mục: (i) Bài báo nghiên cứu khoa học là kết quả công trình nghiên cứu khoa học có giá trị đã được triển khai nghiên cứu, (ii) Bài tổng quan y, sinh, dược học: phục vụ mục tiêu đào tạo liên tục trong lĩnh vực y, sinh, dược học; nhằm hệ thống hóa những kiến thức kinh điển và hiện đại; (iii) Thông tin cập nhật kiến thức mới về y, sinh, dược học trong nước và trên thế giới.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Chính sách truy cập mở\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ áp dụng chính sách truy cập mở đối với các bài báo đã xuất bản đến với độc giả, nhằm mở rộng cơ hội tiếp cận các kết quả nghiên cứu chất lượng cao và tăng cường trao đổi kiến thức. Tạp chí đăng tải trực tuyến (miễn phí) toàn văn các bài báo được công bố trên website của Tạp chí (https:\u002F\u002Ftapchi.ctump.edu.vn).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đạo đức xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ cam kết tuân thủ đạo đức xuất bản phù hợp với các hướng dẫn và tiêu chuẩn của the Committee on Publication Ethics (COPE), tuân thủ các nguyên tắc của COPE’s Core Practices, Best Practices Guidelines for Journal Editors và Guidelines on Good Publication Practices.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Bản thảo bài báo chỉ được chấp nhận khi được tác giả chịu trách nhiệm chính cam kết các nội dung sau: Các nội dung của bản thảo chưa được đăng tải toàn bộ hoặc một phần ở các tạp chí khác; Tất cả các tác giả đều có đóng góp một cách đáng kể vào quá trình nghiên cứu hoặc chuẩn bị bản thảo và cùng chịu trách nhiệm về các nội dung của bản thảo; Tuân thủ các biện pháp đảm bảo đạo đức nghiên cứu (ví dụ thỏa thuận đồng ý tham gia nghiên cứu).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Cam kết bảo mật\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí cam kết thực hiện và tuân thủ các quy định của luật và các văn bản hướng dẫn liên quan đến bảo mật thông tin cá nhân trên không gian mạng. Các thông tin mà người dùng (tác giả, độc giả, biên tập viên, người phản biện) nhập vào các biểu mẫu trên Hệ thống Quản lý xuất bản trực tuyến của tạp chí chỉ được sử dụng vào các mục đích đã được tuyên bố rõ ràng và sẽ không được cung cấp cho bất kỳ bên thứ ba nào khác, hay dùng vào bất kỳ mục đích nào khác.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phí gửi bài\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng bài: 1.000.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng nhanh: 1.500.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với tác giả là cán bộ viên chức thuộc Trường Đại học Y Dược Cần Thơ thì được hỗ trợ 50% lệ phí gửi đăng bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với sinh viên thực hiện đề tài nghiên cứu khoa học cấp trường được hỗ trợ 100% lệ phí đăng bài ( Tác giả gửi đính kèm “ Quyết định về việc giao tổ chức thực hiện đề tài nghiên cứu khoa học cấp Trường của sinh viên”).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Hình thức nộp lệ phí:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Tiền mặt:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Nộp trực tiếp tại Phòng Tài chính - Kế toán, Trường Đại học Y Dược Cần Thơ, số 179 Nguyễn Văn Cừ, P. An Khánh, Q. Ninh Kiều, thành phố Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Chuyển khoản:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tên Tài khoản: Trường ĐHYD Cần Thơ, Số TK: 0111000115668, tại ngân hàng Vietcombank chi nhánh Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Thời gian: Áp dụng từ ngày 01\u002F02\u002F2023.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">* Phí gửi bài không được hoàn trả khi bài viết bị từ chối hoặc tác giả xin rút bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Quy trình phản biện bài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ thực hiện quy trình phản biện kín hai chiều nghiêm ngặt. Danh tính của những người phản biện không được tiết lộ cho các tác giả và ngược lại. Quy trình thẩm định bài báo đăng gồm các bước sau:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tiếp nhận bản thảo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tác giả liên hệ gửi bản thảo đến Tạp chí qua hệ thống trực tuyến tại website: https:\u002F\u002Ftapchi.ctump.edu.vn. Hướng dẫn về cách đăng ký, gửi bài và chuẩn bị bản thảo được cung cấp trên website của Tạp chí.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sàng lọc sơ bộ\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sau khi Tòa soạn nhận được bài báo của tác giả, Ban Thư ký sẽ tiến hành kiểm tra sơ bộ bài báo (các yêu cầu về nội dung và hình thức). Những bài báo không đúng quy cách hoặc có nội dung không phù hợp hoặc vi phạm bản quyền sẽ bị từ chối (Ban Thư ký thông báo phản hồi đến tác giả trong vòng 1 tuần). Những bài báo đủ điều kiện, được Ban Thư ký tòa soạn chuyển đến Ban Biên tập có cùng chuyên môn với nội dung bài báo để đề xuất người phản biện. Thời gian kể từ khi Ban Biên tập nhận bài báo đến khi đề xuất người phản biện bài báo chậm nhất là 5 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Vòng phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký gửi bài và yêu cầu phản biện đến 02 phản biện độc lập.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Các phản biện gởi nhận xét cho Ban Thư ký. Thời gian từ khi gửi bài cho phản biện đến khi nhận ý kiến của phản biện tối đa là 20 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xử ký kết quả phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Nếu ý kiến đồng ý cho đăng và không cần chỉnh sửa, Ban Thư ký tiếp tục đăng bài theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Nếu ý kiến đồng ý đăng và cần chỉnh sửa, Ban Thư ký sẽ thông tin đến tác giả chỉnh sửa theo yêu cầu của người phản biện. Thời gian chỉnh sửa và gửi lại kéo dài không quá 2 tuần, từ khi tác giả bài báo nhận được thông tin (Quá trình này có thể lặp lại tối đa 2 lần\u002F1 bài báo). Khi có sự thống nhất, đồng ý của người phản biện; bài báo được tiếp tục đăng theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Những bài báo có chất lượng không đạt yêu cầu, cả 2 phản biện không đồng ý cho đăng sẽ bị Tòa soạn từ chối đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký tổng hợp các bản thảo đã được tác giả hoàn thiện sau thẩm định trình Ban Biên tập xem xét, Tổng Biên tập phê duyệt, quyết định bài đăng theo các tiêu chí: sự phù hợp nội dung với tôn chỉ và mục đích, thể loại bài viết (ưu tiên các bài có bài có nghiên cứu chuyên sâu, hàm lượng khoa học cao), đóng góp mới bài báo, bài báo được ưu tiên đăng trong số gần nhất của Tạp chí theo thứ tự: tính thời sự, chất lượng bài báo và thời gian gửi bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Ban Biên tập và Ban Thư ký biên tập bản thảo, chế bản, đọc rà soát lỗi. Thời gian hoàn thành từ 10-15 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Ban Thư ký có trách nhiệm thông báo cho tác giả bài báo (bằng e-mail) về tình hình phê duyệt bài báo, thời gian, số kỳ, tập xuất bản bài báo theo qui định.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">4. Danh sách bài báo theo số Tạp chí được in ấn và phát hành trong năm định kỳ được công bố chính thức trên website: https:\u002F\u002Ftapchi.ctump.edu.vn\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>",{"VOID":490},"wcQ1uqwAAAAJ","2023-05-30T08:17:21.868+00:00",[],[494],{"id":495,"createTime":28,"updateTime":28,"relativeEntities":496,"slug":28,"properties":497,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":507,"parentIds":508,"statistic":28},"6413896b-eca9-442b-a73f-182a58a0ce40",[],{"title":498,"address":501,"country":504,"abbreviation":505},{"EN":499,"VI":500},"Can Tho University of Medicine and Pharmacy","Trường Đại học Y Dược Cần Thơ",{"EN":502,"VI":503},"No 179, Nguyen Van Cu street, An Khanh ward, Ninh Kieu district, Can Tho city, Vietnam","Số 179, đường Nguyễn Văn Cừ, phường An Khánh, quận Ninh Kiều, thành phố Cần Thơ, Việt Nam",{"VOID":15},{"VOID":506},"ctump","http:\u002F\u002Fwww.ctump.edu.vn\u002F",[],[],"https:\u002F\u002Ftapchi.ctump.edu.vn\u002Findex.php\u002Fctump",{"impactFactor":32,"impactFactorByYear":512,"i10Index":32,"i10IndexLast5Year":32,"totalPublication":514,"totalPublicationByYear":515,"totalCitation":520,"totalCitationByYear":521,"totalCitationPerPublication":108,"totalCitationPerPublicationByYear":523,"hindexLast5Year":45,"hindex":45},{"2022":513,"2023":111,"2024":106},0.01,1556,{"2020":47,"2021":516,"2022":517,"2023":518,"2024":519,"2025":122},57,306,801,358,161,{"2021":146,"2022":280,"2023":522},99,{"2021":524,"2022":318,"2023":104},0.23,{"impactFactor":28,"impactFactorByYear":28,"i10Index":123,"i10IndexLast5Year":123,"totalPublication":526,"totalPublicationByYear":527,"totalCitation":526,"totalCitationByYear":528,"totalCitationPerPublication":40,"totalCitationPerPublicationByYear":531,"hindexLast5Year":49,"hindex":49},476,{"0":205,"2019":123,"2021":139,"2022":459,"2023":451,"2024":357,"2025":49,"2026":48},{"2021":42,"2022":123,"2023":161,"2024":529,"2025":360,"2026":530},136,83,{"2021":105,"2022":513,"2023":532,"2024":127,"2025":533,"2026":534},0.62,25.43,13.83,{"id":536,"createTime":537,"updateTime":382,"relativeEntities":538,"slug":539,"properties":540,"entityType":25,"verifyStatus":26,"verifyTime":28,"verifyNote":28,"languages":552,"translateLanguages":28,"viewCount":133,"subjectFields":553,"manageAffiliations":554,"indexDatabases":555,"url":556,"thumbnailPath":557,"statistic":558,"gsStatistic":594,"type":55,"analyzePriority":28},"6984a56a-db70-403b-9cc4-4013e1ceaffa","2023-05-09T06:47:40.346+00:00",[],"T%E1%BA%A1p%20ch%C3%AD%20Nghi%C3%AAn%20c%E1%BB%A9u%20n%C6%B0%E1%BB%9Bc%20ngo%C3%A0i",{"country":541,"issn":542,"title":544,"introduce":547,"gsId":550},{"VOID":15},{"VOID":543},"25252445",{"EN":545,"VI":546},"VNU Journal of Foreign Studies","Tạp chí Nghiên cứu nước ngoài",{"EN":548,"VI":549},"{\"ops\":[{\"insert\":\"\\n\\nThe \\n\"},{\"attributes\":{\"italic\":true},\"insert\":\"VNU Journal of Science\"},{\"insert\":\"\\n was established in 1985 for the publication of national and international research papers in all fields of natural sciences and technology, social sciences and humanities. 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The Long-Term Intervention with Pravastatin in Ischaemic Disease (LIPID) Study Group, N Engl J Med, 339, 1349, 10.1056\u002FNEJM199811053391902\nSacks, 1996, The effect of pravastatin on coronary events after myocardial infarction in patients with average cholesterol levels. Cholesterol and Recurrent Events Trial investigators, N Engl J Med, 335, 1001, 10.1056\u002FNEJM199610033351401\nLevine, 1995, Cholesterol reduction in cardiovascular disease: clinical benefits and possible mechanisms, N Engl J Med, 332, 512, 10.1056\u002FNEJM199502233320807\nSmith, 2006, AHA\u002FACC guidelines for secondary prevention for patients with coronary and other atherosclerotic vascular disease: 2006 update: endorsed by the National Heart, Lung, and Blood Institute, Circulation, 113, 2363, 10.1161\u002FCIRCULATIONAHA.106.174516\nHunt, 2005, Circulation, 112, e154, 10.1161\u002FCIRCULATIONAHA.105.167586\nSwedberg, 2005, Guidelines for the diagnosis and treatment of chronic heart failure: executive summary (update 2005): The Task Force for the Diagnosis and Treatment of Chronic Heart Failure of the European Society of Cardiology, Eur Heart J, 26, 1115, 10.1093\u002Feurheartj\u002Fehi204\nHorwich, 2002, Low serum total cholesterol is associated with marked increase in mortality in advanced heart failure, J Card Fail, 8, 216, 10.1054\u002Fjcaf.2002.0804216\nRauchhaus, 2003, The relationship between cholesterol and survival in patients with chronic heart failure, J Am Coll Cardiol, 42, 1933, 10.1016\u002Fj.jacc.2003.07.016\nAfsarmanesh, 2006, Total cholesterol levels and mortality risk in nonischemic systolic heart failure, Am Heart J, 152, 1077, 10.1016\u002Fj.ahj.2006.06.015\nHorwich, 2004, Statin therapy is associated with improved survival in ischemic and non-ischemic heart failure, J Am Coll Cardiol, 43, 642, 10.1016\u002Fj.jacc.2003.07.049\nMozaffarian, 2004, Statin therapy is associated with lower mortality among patients with severe heart failure, Am J Cardiol, 93, 1124, 10.1016\u002Fj.amjcard.2004.01.039\nSola, 2005, Statin therapy is associated with improved cardiovascular outcomes and levels of inflammatory markers in patients with heart failure, J Card Fail, 11, 607, 10.1016\u002Fj.cardfail.2005.05.011\nAnker, 2006, Statin use and survival in patients with chronic heart failure: results from two observational studies with 5200 patients, Int J Cardiol, 112, 234, 10.1016\u002Fj.ijcard.2006.03.057\nKrum, 2007, Statins and symptomatic chronic systolic heart failure: a post-hoc analysis of 5010 patients enrolled in Val-HeFT, Int J Cardiol, 119, 48, 10.1016\u002Fj.ijcard.2006.07.106\nOwan, 2005, Epidemiology of diastolic heart failure, Prog Cardiovasc Dis, 47, 320, 10.1016\u002Fj.pcad.2005.02.010\nBhatia, 2006, Outcome of heart failure with preserved ejection fraction in a population-based study, N Engl J Med, 355, 260, 10.1056\u002FNEJMoa051530\nOwan, 2006, Trends in prevalence and outcome of heart failure with preserved ejection fraction, N Engl J Med, 355, 251, 10.1056\u002FNEJMoa052256\nBursi, 2006, Systolic and diastolic heart failure in the community, JAMA, 296, 2209, 10.1001\u002Fjama.296.18.2209\nFukuta, 2005, Statin therapy may be associated with lower mortality in patients with diastolic heart failure: a preliminary report, Circulation, 112, 357, 10.1161\u002FCIRCULATIONAHA.104.519876\nGrundy, 2004, Implications of recent clinical trials for the National Cholesterol Education Program Adult Treatment Panel III guidelines, Circulation, 110, 227, 10.1161\u002F01.CIR.0000133317.49796.0E\nRoik, 2006, Impact of statin therapy on survival in patients with diastolic heart failure, Eur Heart J, 27, 23\nFolkeringa, 2006, Statins associated with reduced mortality in patients admitted for congestive heart failure, J Card Fail, 12, 134, 10.1016\u002Fj.cardfail.2005.10.014\nGo, 2006, Statin therapy and risks for death and hospitalization in chronic heart failure, JAMA, 296, 2105, 10.1001\u002Fjama.296.17.2105\nRay, 2005, Statin use and survival outcomes in elderly patients with heart failure, Arch Intern Med, 165, 62, 10.1001\u002Farchinte.165.1.62\nFoody, 2006, Statins and mortality among elderly patients hospitalized with heart failure, Circulation, 113, 1086, 10.1161\u002FCIRCULATIONAHA.105.591446\nSenni, 1998, Congestive heart failure in the community: a study of all incident cases in Olmsted County, Minnesota, in 1991, Circulation, 98, 2282, 10.1161\u002F01.CIR.98.21.2282\nVasan, 1999, Congestive heart failure in subjects with normal versus reduced left ventricular ejection fraction: prevalence and mortality in a population-based cohort, J Am Coll Cardiol, 33, 1948, 10.1016\u002FS0735-1097(99)00118-7\nAhmed, 2007, Hospitalizations due to unstable angina pectoris in diastolic and systolic heart failure, Am J Cardiol, 99, 460, 10.1016\u002Fj.amjcard.2006.08.056\nCollins, 2003, MRC\u002FBHF Heart Protection Study of cholesterol-lowering with simvastatin in 5963 people with diabetes: a randomised placebo-controlled trial, Lancet, 361, 2005, 10.1016\u002FS0140-6736(03)13636-7\nTonelli, 2003, Pravastatin for secondary prevention of cardiovascular events in persons with mild chronic renal insufficiency, Ann Intern Med, 138, 98, 10.7326\u002F0003-4819-138-2-200301210-00010\nNishikawa, 2004, Statins induce the regression of left ventricular mass in patients with angina, Circ J, 68, 121, 10.1253\u002Fcircj.68.121\nPatel, 2001, Simvastatin induces regression of cardiac hypertrophy and fibrosis and improves cardiac function in a transgenic rabbit model of human hypertrophic cardiomyopathy, Circulation, 104, 317, 10.1161\u002Fhc2801.094031\nIndolfi, 2002, Hydroxymethylglutaryl coenzyme A reductase inhibitor simvastatin prevents cardiac hypertrophy induced by pressure overload and inhibits p21ras activation, Circulation, 106, 2118, 10.1161\u002F01.CIR.0000034047.70205.97\nZile, 2002, New concepts in diastolic dysfunction and diastolic heart failure: Part II: causal mechanisms and treatment, Circulation, 105, 1503, 10.1161\u002Fhc1202.105290\nAurigemma, 2004, Clinical practice: diastolic heart failure, N Engl J Med, 351, 1097, 10.1056\u002FNEJMcp022709\nRedfield, 2004, Understanding diastolic heart failure, N Engl J Med, 350, 1930, 10.1056\u002FNEJMp048064\nBorghi, 2002, Interactions between hypercholesterolemia and hypertension: implications for therapy, Curr Opin Nephrol Hypertens, 11, 489, 10.1097\u002F00041552-200209000-00003\nGlorioso, 1999, Effect of the HMG-CoA reductase inhibitors on blood pressure in patients with essential hypertension and primary hypercholesterolemia, Hypertension, 34, 1281, 10.1161\u002F01.HYP.34.6.1281\nFerrier, 2002, Intensive cholesterol reduction lowers blood pressure and large artery stiffness in isolated systolic hypertension, J Am Coll Cardiol, 39, 1020, 10.1016\u002FS0735-1097(02)01717-5\nSmilde, 2000, The effect of cholesterol lowering on carotid and femoral artery wall stiffness and thickness in patients with familial hypercholesterolaemia, Eur J Clin Invest, 30, 473, 10.1046\u002Fj.1365-2362.2000.00654.x\nDavignon, 2004, Beneficial cardiovascular pleiotropic effects of statins, Circulation, 109, III39, 10.1161\u002F01.CIR.0000131517.20177.5a\nWilkinson, 2002, Nitric oxide regulates local arterial distensibility in vivo, Circulation, 105, 213, 10.1161\u002Fhc0202.101970\nLandmesser, 2005, Simvastatin versus ezetimibe: pleiotropic and lipid-lowering effects on endothelial function in humans, Circulation, 111, 2356, 10.1161\u002F01.CIR.0000164260.82417.3F\nFarrar, 1980, Reduction in pulse wave velocity and improvement of aortic distensibility accompanying regression of atherosclerosis in the rhesus monkey, Circ Res, 47, 425, 10.1161\u002F01.RES.47.3.425\nCorti, 2002, Lipid lowering by simvastatin induces regression of human atherosclerotic lesions: two years' follow-up by high-resolution noninvasive magnetic resonance imaging, Circulation, 106, 2884, 10.1161\u002F01.CIR.0000041255.88750.F0\nO'Rourke, 2001, Diastolic heart failure, diastolic left ventricular dysfunction and exercise intolerance, J Am Coll Cardiol, 38, 803, 10.1016\u002FS0735-1097(01)01452-8\nKass, 2004, What mechanisms underlie diastolic dysfunction in heart failure?, Circ Res, 94, 1533, 10.1161\u002F01.RES.0000129254.25507.d6\nBauersachs, 2001, Improvement of left ventricular remodeling and function by hydroxymethylglutaryl coenzyme a reductase inhibition with cerivastatin in rats with heart failure after myocardial infarction, Circulation, 104, 982, 10.1161\u002Fhc3401.095946\nHayashidani, 2002, Fluvastatin, a 3-hydroxy-3-methylglutaryl coenzyme a reductase inhibitor, attenuates left ventricular remodeling and failure after experimental myocardial infarction, Circulation, 105, 868, 10.1161\u002Fhc0702.104164\nZaca, 2007, Chronic monotherapy with rosuvastatin prevents progressive left ventricular dysfunction and remodeling in dogs with heart failure, J Am Coll Cardiol, 50, 551, 10.1016\u002Fj.jacc.2007.04.050\nSaka, 2006, Pitavastatin improves cardiac function and survival in association with suppression of the myocardial endothelin system in a rat model of hypertensive heart failure, J Cardiovasc Pharmacol, 47, 770, 10.1097\u002F01.fjc.0000211791.22411.0d\nIchihara, 2006, Pravastatin increases survival and suppresses an increase in myocardial matrix metalloproteinase activity in a rat model of heart failure, Cardiovasc Res, 69, 726, 10.1016\u002Fj.cardiores.2005.08.001\nYoung-Xu, 2003, Usefulness of statin drugs in protecting against atrial fibrillation in patients with coronary artery disease, Am J Cardiol, 92, 1379, 10.1016\u002Fj.amjcard.2003.08.040\nSiu, 2003, Prevention of atrial fibrillation recurrence by statin therapy in patients with lone atrial fibrillation after successful cardioversion, Am J Cardiol, 92, 1343, 10.1016\u002Fj.amjcard.2003.08.023\nPatti, 2006, Randomized trial of atorvastatin for reduction of postoperative atrial fibrillation in patients undergoing cardiac surgery: results of the ARMYDA-3 (Atorvastatin for Reduction of MYocardial Dysrhythmia After Cardiac Surgery) study, Circulation, 114, 1455, 10.1161\u002FCIRCULATIONAHA.106.621763\nHumphries, 2007, Statin use and recurrence of atrial fibrillation after successful cardioversion, Am Heart J, 154, 908, 10.1016\u002Fj.ahj.2007.07.015\nMitchell, 2003, Are lipid-lowering drugs also antiarrhythmic drugs? An analysis of the Antiarrhythmics Versus Implantable Defibrillators (AVID) trial, J Am Coll Cardiol, 42, 81, 10.1016\u002FS0735-1097(03)00498-4\nVyas, 2006, Reduction in ventricular tachyarrhythmias with statins in the Multicenter Automatic Defibrillator Implantation Trial (MADIT)-II, J Am Coll Cardiol, 47, 769, 10.1016\u002Fj.jacc.2005.09.053\nGoldberger, 2006, Effects of statin therapy on arrhythmic events and survival in patients with nonischemic dilated cardiomyopathy, J Am Coll Cardiol, 48, 1228, 10.1016\u002Fj.jacc.2006.05.053\nDickinson, 2007, Statin use was associated with reduced mortality in both ischemic and nonischemic cardiomyopathy and in patients with implantable defibrillators: mortality data and mechanistic insights from the Sudden Cardiac Death in Heart Failure Trial (SCD-HeFT), Am Heart J, 153, 573, 10.1016\u002Fj.ahj.2007.02.002\nDeswal, 2001, Cytokines and cytokine receptors in advanced heart failure: an analysis of the cytokine database from the Vesnarinone trial (VEST), Circulation, 103, 2055, 10.1161\u002F01.CIR.103.16.2055\nRauchhaus, 2000, Plasma cytokine parameters and mortality in patients with chronic heart failure, Circulation, 102, 3060, 10.1161\u002F01.CIR.102.25.3060\nGivertz, 1998, New targets for heart-failure therapy: endothelin, inflammatory cytokines, and oxidative stress, Lancet, 352, SI34, 10.1016\u002FS0140-6736(98)90017-4\nOlsson, 2006, Atrial fibrillation and risk of clinical events in chronic heart failure with and without left ventricular systolic dysfunction: results from the Candesartan in Heart failure-Assessment of Reduction in Mortality and Morbidity (CHARM) program, J Am Coll Cardiol, 47, 1997, 10.1016\u002Fj.jacc.2006.01.060\nSolomon, 2004, Effect of candesartan on cause-specific mortality in heart failure patients: the Candesartan in Heart failure Assessment of Reduction in Mortality and Morbidity (CHARM) program, Circulation, 110, 2180, 10.1161\u002F01.CIR.0000144474.65922.AA\nSolomon, 2005, Influence of ejection fraction on cardiovascular outcomes in a broad spectrum of heart failure patients, Circulation, 112, 3738, 10.1161\u002FCIRCULATIONAHA.105.561423\nRauchhaus, 2000, The endotoxin-lipoprotein hypothesis, Lancet, 356, 930, 10.1016\u002FS0140-6736(00)02690-8\nZile, 2005, Treating diastolic heart failure with statins: phat chance for pleiotropic benefits, Circulation, 112, 300, 10.1161\u002FCIRCULATIONAHA.105.551887\nKjekshus, 2005, A statin in the treatment of heart failure? Controlled Rosuvastatin Multinational Study in Heart Failure (CORONA): study design and baseline characteristics, Eur J Heart Fail, 7, 1059, 10.1016\u002Fj.ejheart.2005.09.005\nTavazzi, 2004, Rationale and design of the GISSI heart failure trial: a large trial to assess the effects of n-3 polyunsaturated fatty acids and rosuvastatin in symptomatic congestive heart failure, Eur J Heart Fail, 6, 635, 10.1016\u002Fj.ejheart.2004.03.001\nKjekshus, 2007, Rosuvastatin in older patients with systolic heart failure, N Engl J Med, 357, 2248, 10.1056\u002FNEJMoa0706201\nMasoudi, 2007, Statins for ischemic systolic heart failure, N Engl J Med, 357, 2301, 10.1056\u002FNEJMe0707221\nSenni, 2001, Heart failure with preserved systolic function. 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Association with cell-mediated but not humoral alloimmunity to donor-specific vascular endothelium, Circulation, 92, 205, 10.1161\u002F01.CIR.92.2.205\nCostanzo-Nordin, 1992, Cardiac allograft vasculopathy: relationship with acute cellular rejection and histocompatibility, J Heart Lung Transplant, 11, S90\nYamani, 2004, Does acute cellular rejection correlate with cardiac allograft vasculopathy?, J Heart Lung Transplant, 23, 272, 10.1016\u002FS1053-2498(03)00189-X\nMichaels, 2003, Humoral rejection in cardiac transplantation: risk factors, hemodynamic consequences and relationship to transplant coronary artery disease, J Heart Lung Transplant, 22, 58, 10.1016\u002FS1053-2498(02)00472-2\nMcGiffin, 1995, Cardiac transplant coronary artery disease. 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Cardiac Transplant Research Database, J Heart Lung Transplant, 17, 744\nKapadia, 2001, Impact of lipid abnormalities in development and progression of transplant coronary disease: a serial intravascular ultrasound study, J Am Coll Cardiol, 38, 206, 10.1016\u002FS0735-1097(01)01337-7\nCaforio, 2004, Immune and nonimmune predictors of cardiac allograft vasculopathy onset and severity: multivariate risk factor analysis and role of immunosuppression, Am J Transplant, 4, 962, 10.1111\u002Fj.1600-6143.2004.00434.x\nSharples, 1991, Risk factor analysis for the major hazards following heart transplantation—rejection, infection, and coronary occlusive disease, Transplantation, 52, 244, 10.1097\u002F00007890-199108000-00012\nMehra, 1994, Influence of donor and recipient gender on cardiac allograft vasculopathy. An intravascular ultrasound study, Circulation, 90, II78\nErinc, 2004, The influence of donor gender on allograft vasculopathy: evidence from intravascular ultrasound, Transplant Proc, 36, 3129, 10.1016\u002Fj.transproceed.2004.10.072\nGao, 1987, Clinical and laboratory correlates of accelerated coronary artery disease in the cardiac transplant patient, Circulation, 76, V56\nEscobar, 1994, Cardiac allograft vasculopathy assessed by intravascular ultrasonography and nonimmunologic risk factors, Am J Cardiol, 74, 1042, 10.1016\u002F0002-9149(94)90856-7\nPark, 1996, Lipid disorder and transplant coronary artery disease in long-term survivors of heart transplantation, J Heart Lung Transplant, 15, 572\nPethig, 2000, Systemic inflammatory response in cardiac allograft vasculopathy: high-sensitive C-reactive protein is associated with progressive luminal obstruction, Circulation, 102, III233\nHognestad, 2003, Plasma C-reactive protein as a marker of cardiac allograft vasculopathy in heart transplant recipients, J Am Coll Cardiol, 42, 477, 10.1016\u002FS0735-1097(03)00645-4\nEverett, 1992, Prolonged cytomegalovirus infection with viremia is associated with development of cardiac allograft vasculopathy, J Heart Lung Transplant, 11, S133\nPetrakopoulou, 2004, Cytomegalovirus infection in heart transplant recipients is associated with impaired endothelial function, Circulation, 110, II207\nValantine, 2004, The role of viruses in cardiac allograft vasculopathy, Am J Transplant, 4, 169, 10.1046\u002Fj.1600-6143.2003.00316.x\nHaji, 2006, Donor or recipient hepatitis B seropositivity is associated with allograft vasculopathy, J Heart Lung Transplant, 25, 294, 10.1016\u002Fj.healun.2005.10.007\nHaji, 2004, Donor hepatitis-C seropositivity is an independent risk factor for the development of accelerated coronary vasculopathy and predicts outcome after cardiac transplantation, J Heart Lung Transplant, 23, 277, 10.1016\u002FS1053-2498(03)00148-7\nStampfer, 1992, A prospective study of plasma homocyst(e)ine and risk of myocardial infarction in US physicians, JAMA, 268, 877, 10.1001\u002Fjama.268.7.877\nKutschka, 2004, Increased plasma homocysteine concentrations accelerate cardiac allograft vasculopathy, J Heart Lung Transplant, 23, 1260, 10.1016\u002Fj.healun.2003.09.004\nMiner, 2001, Hyperhomocysteinemia and transplant coronary artery disease in cardiac transplant recipients, Clin Transplant, 15, 258, 10.1034\u002Fj.1399-0012.2001.150407.x\nPotena, 2005, Homocysteine-lowering therapy and early progression of transplant vasculopathy: a prospective, randomized, IVUS-based study, Am J Transplant, 5, 2258, 10.1111\u002Fj.1600-6143.2005.01015.x\nBonaa, 2006, Homocysteine lowering and cardiovascular events after acute myocardial infarction, N Engl J Med, 354, 1578, 10.1056\u002FNEJMoa055227\nYamani, 2003, Donor spontaneous intracerebral hemorrhage is associated with systemic activation of matrix metalloproteinase-2 and matrix metalloproteinase-9 and subsequent development of coronary vasculopathy in the heart transplant recipient, Circulation, 108, 1724, 10.1161\u002F01.CIR.0000087604.27270.5B\nYamani, 2004, Systemic up-regulation of angiotensin II type 1 receptor in cardiac donors with spontaneous intracerebral hemorrhage, Am J Transplant, 4, 1097, 10.1111\u002Fj.1600-6143.2004.00463.x\nYamani, 2005, Systemic activation of integrin alphaVbeta3 in donors with spontaneous intracerebral hemorrhage is associated with subsequent development of vasculopathy in the heart transplant recipient, J Heart Lung Transplant, 24, 1014, 10.1016\u002Fj.healun.2004.06.003\nRadovancevic, 1990, Risk factors for development of accelerated coronary artery disease in cardiac transplant recipients, Eur J Cardiothorac Surg, 4, 309, 10.1016\u002F1010-7940(90)90207-G\nHoang, 1998, Diabetes and dyslipidemia. A new model for transplant coronary artery disease, Circulation, 97, 2160, 10.1161\u002F01.CIR.97.21.2160\nUretsky, 1987, Development of coronary artery disease in cardiac transplant patients receiving immunosuppressive therapy with cyclosporine and prednisone, Circulation, 76, 827, 10.1161\u002F01.CIR.76.4.827\nLim, 1997, Role of compensatory enlargement and shrinkage in transplant coronary artery disease. Serial intravascular ultrasound study, Circulation, 95, 855, 10.1161\u002F01.CIR.95.4.855\nRamasubbu, 2003, Repeated intravascular ultrasound imaging in cardiac transplant recipients does not accelerate transplant coronary artery disease, J Am Coll Cardiol, 41, 1739, 10.1016\u002FS0735-1097(03)00339-5\nTuzcu, 1995, Occult and frequent transmission of atherosclerotic coronary disease with cardiac transplantation. Insights from intravascular ultrasound, Circulation, 91, 1706, 10.1161\u002F01.CIR.91.6.1706\nKapadia, 1998, Development of transplantation vasculopathy and progression of donor-transmitted atherosclerosis: comparison by serial intravascular ultrasound imaging, Circulation, 98, 2672, 10.1161\u002F01.CIR.98.24.2672\nLi, 2006, Influence of pre-existing donor atherosclerosis on the development of cardiac allograft vasculopathy and outcomes in heart transplant recipients, J Am Coll Cardiol, 47, 2470, 10.1016\u002Fj.jacc.2006.01.072\nTuzcu, 2005, Intravascular ultrasound evidence of angiographically silent progression in coronary atherosclerosis predicts long-term morbidity and mortality after cardiac transplantation, J Am Coll Cardiol, 45, 1538, 10.1016\u002Fj.jacc.2004.12.076\nMehra, 1995, The prognostic significance of intimal proliferation in cardiac allograft vasculopathy: a paradigm shift, J Heart Lung Transplant, 14, S207\nKobashigawa, 1995, Effect of pravastatin on outcomes after cardiac transplantation, N Engl J Med, 333, 621, 10.1056\u002FNEJM199509073331003\nFang, 2002, Effect of vitamins C and E on progression of transplant-associated arteriosclerosis: a randomised trial, Lancet, 359, 1108, 10.1016\u002FS0140-6736(02)08154-0\nEisen, 2003, Everolimus for the prevention of allograft rejection and vasculopathy in cardiac-transplant recipients, N Engl J Med, 349, 847, 10.1056\u002FNEJMoa022171\nSpes, 1999, Diagnostic and prognostic value of serial dobutamine stress echocardiography for noninvasive assessment of cardiac allograft vasculopathy: a comparison with coronary angiography and intravascular ultrasound, Circulation, 100, 509, 10.1161\u002F01.CIR.100.5.509\nTona, 2006, Coronary flow velocity pattern and coronary flow reserve by contrast-enhanced transthoracic echocardiography predict long-term outcome in heart transplantation, Circulation, 114, I49\nYoung, 1999, Allograft vasculopathy: diagnosing the nemesis of heart transplantation, Circulation, 100, 458, 10.1161\u002F01.CIR.100.5.458\nMehra, 2002, Comparative beneficial effects of simvastatin and pravastatin on cardiac allograft rejection and survival, J Am Coll Cardiol, 40, 1609, 10.1016\u002FS0735-1097(02)02340-9\nGrigioni, 2006, Long-term safety and effectiveness of statins for heart transplant recipients in routine clinical practice, Transplant Proc, 38, 1507, 10.1016\u002Fj.transproceed.2006.02.071\nKobashigawa, 2005, Ten-year follow-up of a randomized trial of pravastatin in heart transplant patients, J Heart Lung Transplant, 24, 1736, 10.1016\u002Fj.healun.2005.02.009\nSchroeder, 1993, A preliminary study of diltiazem in the prevention of coronary artery disease in heart-transplant recipients, N Engl J Med, 328, 164, 10.1056\u002FNEJM199301213280303\nMehra, 1995, An intravascular ultrasound study of the influence of angiotensin-converting enzyme inhibitors and calcium entry blockers on the development of cardiac allograft vasculopathy, Am J Cardiol, 75, 853, 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Roentgenol, 186, 627, 10.2214\u002FAJR.04.0746\nWeinsaft, 2008, Detection of left ventricular thrombus by delayed-enhancement cardiovascular magnetic resonance prevalence and markers in patients with systolic dysfunction, J Am Coll Cardiol, 52, 148, 10.1016\u002Fj.jacc.2008.03.041\nSechtem, 1986, MRI of the normal pericardium. Part I, AJR Am J Roentgenol, 147, 239, 10.2214\u002Fajr.147.2.239\nSechtem, 1986, MRI of the abnormal pericardium. Part. II, AJR Am J Roentgenol, 147, 245, 10.2214\u002Fajr.147.2.245\nTeraoka, 2005, Delayed contrast enhancement in a patient with perimyocarditis on contrast-enhanced cardiac MRI: case report, Int J Cardiovasc Imaging, 21, 325, 10.1007\u002Fs10554-004-4016-z\nRienmuller, 2004, CT and MR imaging of pericardial disease, Radiol Clin North Am, 42, 587, 10.1016\u002Fj.rcl.2004.03.003\nBreen, 2001, Imaging of the pericardium, J Thorac Imaging, 16, 47, 10.1097\u002F00005382-200101000-00007\nWhite, 1995, MR evaluation of the pericardium, Top Magn Reson Imaging, 7, 258, 10.1097\u002F00002142-199500740-00006\nKastler, 1990, Spin echo MRI in the evaluation of pericardial disease, Comput Med Imaging Graph, 14, 241, 10.1016\u002F0895-6111(90)90004-U\nWang, 2003, CT and MR imaging of pericardial disease, Radiographics, 23, S167, 10.1148\u002Frg.23si035504\nGlockner, 2003, Imaging of pericardial disease, Magn Reson Imaging Clin N Am, 11, 149, 10.1016\u002FS1064-9689(02)00025-9\nFrank, 1999, Magnetic resonance imaging evaluation of myocardial and pericardial disease, J Magn Reson Imaging, 10, 617, 10.1002\u002F(SICI)1522-2586(199911)10:5\u003C617::AID-JMRI5>3.0.CO;2-Z\nRefsum, 1981, Ventricular diastolic pressure-volume relations and the pericardium: effects of changes in blood volume and pericardial effusion in dogs, Circulation, 64, 997, 10.1161\u002F01.CIR.64.5.997\nFreeman, 1984, Pericardial adaptations during chronic cardiac dilation in dogs, Circ Res, 54, 294, 10.1161\u002F01.RES.54.3.294\nFreeman, 1986, Determinants of intrapericardial pressure in dogs, J Appl Phys, 60, 758\nLittle, 2006, Pericardial disease, Circulation, 113, 1622, 10.1161\u002FCIRCULATIONAHA.105.561514\nTsang, 1999, Diagnosis and management of cardiac tamponade in the era of echocardiography, Clin Cardiol, 22, 446, 10.1002\u002Fclc.4960220703\nTsang, 1999, Clinical and echocardiographic characteristics of significant pericardial effusions following cardiothoracic surgery and outcomes of echo-guided pericardiocentesis for management: Mayo Clinic experience, 1979–1998, Chest, 116, 322, 10.1378\u002Fchest.116.2.322\nSingh, 1984, Right ventricular and right atrial collapse in patients with cardiac tamponade: a combined echocardiographic and hemodynamic study, Circulation, 70, 966, 10.1161\u002F01.CIR.70.6.966\nAppleton, 1988, Cardiac tamponade and pericardial effusion: respiratory variation in transvalvular flow velocities studied by Doppler echocardiography, J Am Coll Cardiol, 11, 1020, 10.1016\u002FS0735-1097(98)90060-2\nFrancone, 2005, Real-time cine MRI of ventricular septal motion: a novel approach to assess ventricular coupling, J Magn Reson Imaging, 21, 305, 10.1002\u002Fjmri.20259\nMyers, 1999, Constrictive pericarditis: clinical and pathophysiologic characteristics, Am Heart J, 138, 219, 10.1016\u002FS0002-8703(99)70105-5\nLing, 1999, Constrictive pericarditis in the modern era: evolving clinical spectrum and impact on outcome after pericardiectomy, Circulation, 100, 1380, 10.1161\u002F01.CIR.100.13.1380\nLing, 2000, Calcific constrictive pericarditis: is it still with us?, Ann Intern Med, 132, 444, 10.7326\u002F0003-4819-132-6-200003210-00004\nNishimura, 2001, Constrictive pericarditis in the modern era: a diagnostic dilemma, Heart, 86, 619, 10.1136\u002Fheart.86.6.619\nNishimura, 1985, Constrictive pericarditis: assessment of current diagnostic procedures, Mayo Clin Proc, 60, 397, 10.1016\u002FS0025-6196(12)60850-7\nOh, 2001, Surgical pathology of the parietal pericardium: a study of 344 cases (1993–1999), Cardiovasc Pathol, 10, 157, 10.1016\u002FS1054-8807(01)00076-X\nTalreja, 2003, Constrictive pericarditis in 26 patients with histologically normal pericardial thickness, Circulation, 108, 1852, 10.1161\u002F01.CIR.0000087606.18453.FD\nMertens, 1993, The differentiation between restrictive cardiomyopathy and constrictive pericarditis: the impact of the imaging techniques, Echocardiography, 10, 497, 10.1111\u002Fj.1540-8175.1993.tb00064.x\nMasui, 1992, Constrictive pericarditis and restrictive cardiomyopathy: evaluation with MR imaging, Radiology, 182, 369, 10.1148\u002Fradiology.182.2.1732952\nSengupta, 2004, Accuracy and pitfalls of early diastolic motion of the mitral annulus for diagnosing constrictive pericarditis by tissue Doppler imaging, Am J Cardiol, 93, 886, 10.1016\u002Fj.amjcard.2003.12.029\nGiorgi, 2003, Clinically suspected constrictive pericarditis: MR imaging assessment of ventricular septal motion and configuration in patients and healthy subjects, Radiology, 228, 417, 10.1148\u002Fradiol.2282020345\nHancock, 2004, A clearer view of effusive-constrictive pericarditis, N Engl J Med, 350, 435, 10.1056\u002FNEJMp038199\nHancock, 1971, Subacute effusive-constrictive pericarditis, Circulation, 43, 183, 10.1161\u002F01.CIR.43.2.183\nYamano, 2004, Magnetic resonance imaging differentiated partial from complete absence of the left pericardium in a case of leftward displacement of the heart, Circ J, 68, 385, 10.1253\u002Fcircj.68.385\nFaridah, 2002, Congenital absence of pericardium revisited, Int J Cardiovasc Imaging, 18, 67, 10.1023\u002FA:1014350814067\nOyama, 2004, Computed tomography and magnetic resonance imaging of the pericardium: anatomy and pathology, Magn Reson Med Sci, 3, 145, 10.2463\u002Fmrms.3.145\nVander Salm, 2000, Unusual primary tumors of the heart, Semin Thorac Cardiovasc Surg, 12, 89, 10.1053\u002Fct.2000.5080\nGilkeson, 2003, MR evaluation of cardiac and pericardial malignancy, Magn Reson Imaging Clin N Am, 11, 173, 10.1016\u002FS1064-9689(02)00047-8\nLink, 1995, MR evaluation of cardiac\u002Fjuxtacardiac masses, Top Magn Reson Imaging, 7, 232, 10.1097\u002F00002142-199500740-00004\nMeier, 1994, MRI of right atrial pseudomass: is it really a diagnostic problem?, J Comput Assist Tomogr, 18, 398, 10.1097\u002F00004728-199405000-00010\nKingdon, 2001, Atrial thrombus and central venous dialysis catheters, Am J Kidney Dis, 38, 631, 10.1053\u002Fajkd.2001.26898\nMollet, 2002, Visualization of ventricular thrombi with contrast-enhanced magnetic resonance imaging in patients with ischemic heart disease, Circulation, 106, 2873, 10.1161\u002F01.CIR.0000044389.51236.91\nSrichai, 2006, Clinical, imaging, and pathological characteristics of left ventricular thrombus: a comparison of contrast-enhanced magnetic resonance imaging, transthoracic echocardiography, and transesophageal echocardiography with surgical or pathological validation, Am Heart J, 152, 75, 10.1016\u002Fj.ahj.2005.08.021\nBarkhausen, 2002, Detection and characterization of intracardiac thrombi on MR imaging, AJR Am J Roentgenol, 179, 1539, 10.2214\u002Fajr.179.6.1791539\nOhyama, 2003, Comparison of magnetic resonance imaging and transesophageal echocardiography in detection of thrombus in the left atrial appendage, Stroke, 34, 2436, 10.1161\u002F01.STR.0000090350.73614.0F\nMohrs, 2006, Thrombus detection in the left atrial appendage using contrast-enhanced MRI: a pilot study, AJR Am J Roentgenol, 186, 198, 10.2214\u002FAJR.04.1504\nGrizzard, 2007, Magnetic resonance imaging of pericardial disease and cardiac masses, Cardiol Clin, 25, 111, 10.1016\u002Fj.ccl.2007.02.004",{"VOID":1171},"10.1016\u002Fj.hfc.2009.02.006","https:\u002F\u002Fwww.sciencedirect.com\u002Fscience\u002Farticle\u002Fpii\u002FS155171360900021X",[1174],{"id":1175,"sortIndex":32,"researcher":28,"roles":1176,"affiliations":1177,"properties":1186},"195bf380-cac0-4b1d-b526-8b61d59d200a",[973],[1178],{"id":1179,"sortIndex":32,"affiliation":1180,"properties":28},"e0119f81-1bd1-40ac-88eb-7b579231350f",{"id":1179,"createTime":28,"updateTime":28,"relativeEntities":1181,"slug":28,"properties":1182,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1185,"statistic":28},[],{"title":1183},{"VI":1184},"Virginia Commonwealth University Medical Center Richmond, VA, USA",[],{"title":1187},{"VI":1188},"John D. 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The role of catheter size and duration of support in a multivariate analysis of risk, Eur Heart J, 19, 458, 10.1053\u002Feuhj.1997.0802\nMeisel, 2004, Utilization of low-profile intra-aortic balloon catheters inserted by the sheathless technique in acute cardiac patients: clinical efficacy with a very low complication rate, Clin Cardiol, 27, 600, 10.1002\u002Fclc.4960271104\nArceo, 2003, In-hospital complications of percutaneous intraaortic balloon counterpulsation, Angiology, 54, 577, 10.1177\u002F000331970305400507\nShin, 2000, Acute ischemic hepatic failure resulting from intraaortic balloon pump malposition, Eur J Cardiothorac Surg, 17, 492, 10.1016\u002FS1010-7940(00)00366-3\nOsentowski, 2007, Evaluating the efficacy of intra-aortic balloon pump timing using the auto-timing mode of operation with the datascope CS100, J Extra Corpor Technol, 39, 87\nSanta-Cruz, 2006, Aortic counterpulsation: a review of the hemodynamic effects and indications for use, Catheter Cardiovasc Interv, 67, 68, 10.1002\u002Fccd.20552\nAnderson, 1997, Use of intraaortic balloon counterpulsation in patients presenting with cardiogenic shock: observations from the Gusto-I study. global utilization of streptokinase and TPA for occluded coronary arteries, J Am Coll Cardiol, 30, 708, 10.1016\u002FS0735-1097(97)00227-1\nHochman, 2000, Cardiogenic shock complicating acute myocardial infarction–etiologies, management and outcome: a report from the shock trial registry. Should we emergently revascularize occluded coronaries for cardiogenic shock?, J Am Coll Cardiol, 36, 1063, 10.1016\u002FS0735-1097(00)00879-2\nSanborn, 2000, J Am Coll Cardiol, 36, 1123, 10.1016\u002FS0735-1097(00)00875-5\nBarron, 2001, The use of intra-aortic balloon counterpulsation in patients with cardiogenic shock complicating acute myocardial infarction: data from the National Registry of Myocardial Infarction 2, Am Heart J, 141, 933, 10.1067\u002Fmhj.2001.115295\nSun, 2003, Indications for long-term assist device placement as bridge to transplantation, Cardiol Clin, 21, 51, 10.1016\u002FS0733-8651(03)00004-3\nSun, 2007, Devices for cardiac support and replacement, 587\nRose, 2001, Long-term mechanical left ventricular assistance for end-stage heart failure, N Engl J Med, 345, 1435, 10.1056\u002FNEJMoa012175\nDembitsky, 2004, Left ventricular assist device performance with long-term circulatory support: lessons from the rematch trial, Ann Thorac Surg, 78, 2123, 10.1016\u002Fj.athoracsur.2004.02.030\nRogers, 2007, Chronic mechanical circulatory support for inotrope-dependent heart failure patients who are not transplant candidates: results of the intrepid trial, J Am Coll Cardiol, 50, 741, 10.1016\u002Fj.jacc.2007.03.063\nMadigan, 1999, Surgical management of the patient with an implanted cardiac device: implications of electromagnetic interference, Ann Surg, 230, 639, 10.1097\u002F00000658-199911000-00005\n2004\nPasque, 2002, Adverse events in the use of HeartMate vented electric and Novacor left ventricular assist devices: comparing apples and oranges, J Thorac Cardiovasc Surg, 124, 1063, 10.1067\u002Fmtc.2002.123520\nLazar, 2004, Neurological events during long-term mechanical circulatory support for heart failure: the randomized evaluation of mechanical assistance for the treatment of congestive heart failure (rematch) experience, Circulation, 109, 2423, 10.1161\u002F01.CIR.0000129414.95137.CD\nLong, 2005, Long-term destination therapy with the HeartMate XVE left ventricular assist device: improved outcomes since the rematch study, Congest Heart Fail, 11, 133, 10.1111\u002Fj.1527-5299.2005.04540.x\nMcKellar, 1999, Treatment of infected left ventricular assist device using antibiotic-impregnated beads, Ann Thorac Surg, 67, 554, 10.1016\u002FS0003-4975(98)01243-0\nSajjadian, 2006, Omental transposition flap for salvage of ventricular assist devices, Plast Reconstr Surg, 118, 919, 10.1097\u002F01.prs.0000232419.74219.15\nOchiai, 2002, Predictors of severe right ventricular failure after implantable left ventricular assist device insertion: analysis of 245 patients, Circulation, 106, I198\nDang, 2006, Right heart failure after left ventricular assist device implantation in patients with chronic congestive heart failure, J Heart Lung Transplant, 25, 1, 10.1016\u002Fj.healun.2005.07.008\nPagani, 2006, Improved mechanical reliability of the HeartMate XVE left ventricular assist system, Ann Thorac Surg, 82, 1413, 10.1016\u002Fj.athoracsur.2006.04.057\nMyers, 2007, Management of multiple left ventricular assist device failures in a patient, J Heart Lung Transplant, 26, 98, 10.1016\u002Fj.healun.2006.10.009\nHorton, 2004, Left ventricular assist device malfunction: a systematic approach to diagnosis, J Am Coll Cardiol, 43, 1574, 10.1016\u002Fj.jacc.2003.11.055",{"VOID":1257},"10.1016\u002Fj.hfc.2008.08.002","https:\u002F\u002Fwww.sciencedirect.com\u002Fscience\u002Farticle\u002Fpii\u002FS1551713608001256",[1260],{"id":1261,"sortIndex":32,"researcher":28,"roles":1262,"affiliations":1263,"properties":1275},"945e6b8b-205a-4314-a6ea-a25a06b7a295",[973],[1264],{"id":1265,"sortIndex":32,"affiliation":1266,"properties":1272},"49ef2667-0fc5-423e-903e-5050f8f3ed28",{"id":1265,"createTime":28,"updateTime":28,"relativeEntities":1267,"slug":28,"properties":1268,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1271,"statistic":28},[],{"title":1269},{"EN":1270},"The Ohio State University, Columbus, OH, USA.",[],{"title":1273},{"VI":1274},"The Ohio State University, Columbus, OH, USA",{"title":1276},{"VI":1277},"Brian C. 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Analysis of the organ procurement and transplantation network\u002FU.S. United Network of Organ Sharing data, 1990 to 2005, J Am Coll Cardiol, 50, 1282, 10.1016\u002Fj.jacc.2007.04.099\nFonarow, 1994, Persistently high left ventricular filling pressures predict mortality despite angiotensin converting enzyme inhibition in advanced heart failure, Circulation, 90, I-488\nAbraham, 1998, Systemic hemodynamic, neurohormonal, and renal effects of a steady-state infusion of human brain natriuretic peptide in patients with hemodynamically decompensated heart failure, J Card Fail, 4, 37, 10.1016\u002FS1071-9164(98)90506-1\nLe Jemtel, 2006, Hemodynamic goals are outdated, Circulation, 113, 1027\nBourge, 2008, Randomized controlled trial of an implantable continuous hemodynamic monitor in patients with advanced heart failure. The COMPASS-HF study, J Am Coll Cardiol, 51, 1073, 10.1016\u002Fj.jacc.2007.10.061\nMagalski, 2008, Does lowering filling pressures by using continuously measured intracardiac pressures result in reduced risk of heart failure event?, J Am Coll Cardiol, 51, A80\nLeier, 2007, The physical examination in heart failure—part I, CHF, 13, 41\nDrazner, 2001, Prognostic importance of elevated jugular venous pressure and a third heart sound in patients with heart failure, N Engl J Med, 345, 574, 10.1056\u002FNEJMoa010641\nDrazner, 1999, Relationship between right and left sided filling pressures in 1000 patients with advanced heart failure, J Heart Lung Transplant, 18, 1126, 10.1016\u002FS1053-2498(99)00070-4\nLucus, 2000, Freedom from congestion predicts good survival despite previous class IV symptoms of heart failure, Am Heart J, 140, 840, 10.1067\u002Fmhj.2000.110933\nDaniels, 2007, Natriuretic peptides, J Am Coll Cardiol, 50, 2357, 10.1016\u002Fj.jacc.2007.09.021\nDoust, 2005, How well does B-type natriuretic peptide predict death and cardiac events in patients with heart failure: systematic review, BMJ, 330, 625, 10.1136\u002Fbmj.330.7492.625\nLogeart, 2004, Predischarge B-type natriuretic peptide assay for identifying patients at high risk for readmission after decompensated heart failure, J Am Coll Cardiol, 43, 635, 10.1016\u002Fj.jacc.2003.09.044\nJourdain, 2007, Plasma brain natriuretic peptide-guided therapy to improve outcome in heart failure. The STARS-BNP multicenter study, J AM Coll Cardiol, 49, 1733, 10.1016\u002Fj.jacc.2006.10.081\nValle, 2008, B-type natriuretic peptide–guided treatment for predicting outcome in patients hospitalized in sub-intensive care unit with acute heart failure, J Card Fail, 14, 219, 10.1016\u002Fj.cardfail.2007.10.009\nKirkpatrick, 2007, Echocardiography in heart failure, J Am Coll Cardiol, 50, 381, 10.1016\u002Fj.jacc.2007.03.048\nRohde, 2007, A hemodynamically oriented echocardiography-based strategy in the treatment of congestive heart failure, J Card Fail, 13, 618, 10.1016\u002Fj.cardfail.2007.05.003\nRistow, 2007, Elevated pulmonary artery pressure by Doppler echocardiography predicts hospitalization for heart failure and mortality in ambulatory stable coronary artery disease. 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Essential role for p2t(ac) and alpha(2a) receptors, J Biol Chem, 274, 29108, 10.1074\u002Fjbc.274.41.29108\nFurci, 1991, Heterogeneicity of prostaglandin H2\u002FThromboxane A2 receptors:distinct subtypes mediate vascular smooth muscle contraction and platelet aggregation, J Pharmacol Exp Ther, 258, 74\nWeksler, 1983, Differential inhibition by aspirin of vascular and prostaglandin synthesis in atherosclerotic patients, N Engl J Med, 308, 800, 10.1056\u002FNEJM198304073081402\nWhelton, 1999, Nephrotoxicity of nonsteroidal anti-inflammatory drugs: physiologic foundations and clinical implications, Am J Med, 106, 13S, 10.1016\u002FS0002-9343(99)00113-8\nGainer, 1998, Effect of bradykinin-receptor blockade on the response to angiotensin converting enzyme inhibitor in normotensive and hypertensive subjects, N Engl J Med, 339, 1285, 10.1056\u002FNEJM199810293391804\nAhmed, 2002, Interaction between aspirin and angiotensin–converting enzyme inhibitors: should they be used together in older adults with heart failure?, J Am Geriatr Soc, 50, 1293, 10.1046\u002Fj.1532-5415.2002.50320.x\nTeerlink, 1999, The interaction of ACEI and ASA in heart failure: torn between two lovers, Am Heart J, 138, 193, 10.1016\u002FS0002-8703(99)70099-2\nCleland, 1995, Is aspirin safe for patients with heart failure?, Br Heart J, 74, 215, 10.1136\u002Fhrt.74.3.215\nFrances, 2000, Are we inhibited? Renal insufficiency should not preclude the use of ACEI for patients with myocardial infarction and depressed left ventricular function, Arch Intern Med, 160, 2645, 10.1001\u002Farchinte.160.17.2645\nLather, 2000, Long term ACEI therapy in patients with heart failure or left ventricular dysfunction: a systematic overview of data from individual patients. ACEI Myocardial infarction collaborative group, Lancet, 355, 1575, 10.1016\u002FS0140-6736(00)02212-1\nEras, 2001, NSAIDS and the kidney revisited: are selective cyclooxygenase-2 inhibitors safe?, Am J Med Sci, 321, 181, 10.1097\u002F00000441-200103000-00005\nPerazella, 2000, Drug-induced hyperkalemia. Old culprits and new offenders, Am J Med, 109, 307, 10.1016\u002FS0002-9343(00)00496-4\nKimberly, 1978, Reduction of renal function by newer non steroidal anti-inflammatory drugs, Am J Med, 64, 804, 10.1016\u002F0002-9343(78)90520-X\nTan, 1979, Indomethacin induced prostaglandin inhibition wih hyperkalemia, Ann Intern Med, 90, 783, 10.7326\u002F0003-4819-90-5-783\nKurata, 1999, Syncope caused by Nonsteroidal anti-inflammatory drugs and angiotensin-converting enzyme inhibitors, Jpn Circ J, 63, 1002, 10.1253\u002Fjcj.63.1002\nJolobe, 1999, Evaluation of renal function in elderly patients on ACE inhibitors, Postgrad Med J, 75, 275, 10.1136\u002Fpgmj.75.883.275\nCampbell, 2004, Acute congestive heart failure induced by rofecoxib, J Am Board Fam Pract, 17, 131, 10.3122\u002Fjabfm.17.2.131\nFeenstra, 2002, Association of nonsteroidal anti-inflammatory drugs with first occurrence of heart failure and with relapsing heart failure, Arch Intern Med, 162, 265, 10.1001\u002Farchinte.162.3.265\nHeerdink, 1998, NSAIDS associated with increased risk of CHF in elderly patients taking diuretics, Arch Intern Med, 158, 1108, 10.1001\u002Farchinte.158.10.1108\nChertow, 2005, Acute kidney injury, mortality, length of stay, and costs in hospitalized patients, J Am Soc Nephrol, 16, 3365, 10.1681\u002FASN.2004090740\nKhand, 2000, Is the prognosis of heart failure improving?, J Am Coll Cardiol, 36, 2284, 10.1016\u002FS0735-1097(00)00995-5\nKonstan, 2000, Progress in heart failure management? 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Evidence for increasing population prevalence, Arch Intern Med, 150, 769, 10.1001\u002Farchinte.1990.00390160043010\nMcMurray, 2000, Epidemiology, aetiology, and prognosis of heart failure, Heart, 83, 596, 10.1136\u002Fheart.83.5.596\nCurtis, 2008, Incidence and prevalence of heart failure in the elderly, 1994–2008, Arch Intern Med, 168, 418, 10.1001\u002Farchinternmed.2007.80\nInstitute of Medicine, 2001\nMasoudi, 2004, National patterns of use and effectiveness of angiotensin-converting enzyme inhibitors in older patients with heart failure and left ventricular systolic dysfunction, Circulation, 110, 724, 10.1161\u002F01.CIR.0000138934.28340.ED\nPeterson, 2006, Association between hospital process performance and outcomes among patients with acute coronary syndromes, JAMA, 295, 1912, 10.1001\u002Fjama.295.16.1912\nFonarow, 2007, Association between performance measures and clinical outcomes for patients hospitalized with heart failure, JAMA, 297, 61, 10.1001\u002Fjama.297.1.61\nJha, 2005, Care in U.S. hospitals—the Hospital Quality Alliance program, N Engl J Med, 353, 265, 10.1056\u002FNEJMsa051249\nWilliams, 2005, Quality of care in U.S. hospitals as reflected by standardized measures, 2002–2004, N Engl J Med, 353, 255, 10.1056\u002FNEJMsa043778\nFonarow, 2004, Organized Program to Initiate Lifesaving Treatment in Hospitalized Patients with Heart Failure (OPTIMIZE-HF): rationale and design, Am Heart J, 148, 43, 10.1016\u002Fj.ahj.2004.03.004\nFonarow, 2005, Adherence to heart failure quality-of-care indicators in US hospitals: analysis of the ADHERE Registry, Arch Intern Med, 165, 1469, 10.1001\u002Farchinte.165.13.1469\nFonarow, 2007, Improving the use of evidence-based heart failure therapies in the outpatient setting: the IMPROVE HF performance improvement registry, Am Heart J, 154, 12, 10.1016\u002Fj.ahj.2007.03.030\nLaBresh, 2004, Get with the guidelines for cardiovascular secondary prevention: pilot results, Arch Intern Med, 164, 203, 10.1001\u002Farchinte.164.2.203\nCenters for Medicare and Medicaid. Available at: www.cms.hhs.gov\u002Fquality\u002Fhospital Accessed February 1, 2008.\nFonarow, 2007, Influence of a performance-improvement initiative on quality of care for patients hospitalized with heart failure: results of the Organized Program to Initiate Lifesaving Treatment in Hospitalized Patients With Heart Failure (OPTIMIZE-HF), Arch Intern Med, 167, 1493, 10.1001\u002Farchinte.167.14.1493\nBonow, 2005, Circulation, 112, 1853, 10.1161\u002FCIRCULATIONAHA.105.170072\nKrumholz, 2000, Evaluating quality of care for patients with heart failure, Circulation, 101, E122, 10.1161\u002F01.CIR.101.12.e122\nHernandez, 2007, Sex and racial differences in the use of implantable cardioverter-defibrillators among patients hospitalized with heart failure, JAMA, 298, 1525, 10.1001\u002Fjama.298.13.1525\nBhatt, 2006, International prevalence, recognition, and treatment of cardiovascular risk factors in outpatients with atherothrombosis, JAMA, 295, 180, 10.1001\u002Fjama.295.2.180\nKo, 2004, Lipid-lowering therapy with statins in high-risk elderly patients: the treatment–risk paradox, JAMA, 291, 1864, 10.1001\u002Fjama.291.15.1864\nAnker, 2006, Statin use and survival in patients with chronic heart failure—results from two observational studies with 5200 patients, Int J Cardiol, 112, 234, 10.1016\u002Fj.ijcard.2006.03.057\nDomanski, 2007, Effect of statin therapy on survival in patients with nonischemic dilated cardiomyopathy (from the Beta-blocker Evaluation of Survival Trial [BEST]), Am J Cardiol, 99, 1448, 10.1016\u002Fj.amjcard.2006.12.080\nKrum, 2007, Impact of statin therapy on clinical outcomes in chronic heart failure patients according to beta-blocker use: results of CIBIS II, Cardiology, 108, 28, 10.1159\u002F000095629\nFoody, 2006, Statins and mortality among elderly patients hospitalized with heart failure, Circulation, 113, 1086, 10.1161\u002FCIRCULATIONAHA.105.591446\nGo, 2006, Statin therapy and risks for death and hospitalization in chronic heart failure, JAMA, 296, 2105, 10.1001\u002Fjama.296.17.2105\nCabana, 1999, Why don't physicians follow clinical practice guidelines? A framework for improvement, JAMA, 282, 1458, 10.1001\u002Fjama.282.15.1458\nCarruthers, 1999, Assimilating new therapeutic interventions into clinical practice: How does hypertension compare with other therapeutic areas?, Am Heart J, 138, 256, 10.1016\u002FS0002-8703(99)70319-4\nEisenberg, 1985, Physician utilization. The state of research about physicians' practice patterns, Med Care, 23, 461, 10.1097\u002F00005650-198505000-00010\nLomas, 1989, Do practice guidelines guide practice? The effect of a consensus statement on the practice of physicians, N Engl J Med, 321, 1306, 10.1056\u002FNEJM198911093211906\nBero, 1998, Closing the gap between research and practice: an overview of systematic reviews of interventions to promote the implementation of research findings. The Cochrane Effective Practice and Organization of Care Review Group, BMJ, 317, 465, 10.1136\u002Fbmj.317.7156.465\nGreco, 1993, Changing physicians' practices, N Engl J Med, 329, 1271, 10.1056\u002FNEJM199310213291714\nBoekeloo, 1990, Strategies for increasing house staff management of cholesterol with inpatients, Am J Prev Med, 6, 51\nWinkens, 1995, Randomised controlled trial of routine individual feedback to improve rationality and reduce numbers of test requests, Lancet, 345, 498, 10.1016\u002FS0140-6736(95)90588-X\nOxman, 1995, No magic bullets: a systematic review of 102 trials of interventions to improve professional practice, CMAJ, 153, 1423\nPatel, 2004, State-mandated continuing medical education and the use of proven therapies in patients with an acute myocardial infarction, J Am Coll Cardiol, 44, 192, 10.1016\u002Fj.jacc.2004.03.070\nDavis, 1995, Changing physician performance. A systematic review of the effect of continuing medical education strategies, JAMA, 274, 700, 10.1001\u002Fjama.1995.03530090032018\nSoumerai, 1998, Effect of local medical opinion leaders on quality of care for acute myocardial infarction: a randomized controlled trial, JAMA, 279, 1358, 10.1001\u002Fjama.279.17.1358\nLaPointe, 2006, Multifaceted intervention to promote beta-blocker use in heart failure, Am Heart J, 151, 992, 10.1016\u002Fj.ahj.2005.06.038\nAnsari, 2003, Improving guideline adherence: a randomized trial evaluating strategies to increase beta-blocker use in heart failure, Circulation, 107, 2799, 10.1161\u002F01.CIR.0000070952.08969.5B\nFonarow, 2001, Improved treatment of coronary heart disease by implementation of a Cardiac Hospitalization Atherosclerosis Management Program (CHAMP), Am J Cardiol, 87, 819, 10.1016\u002FS0002-9149(00)01519-8\nButler, 2006, Improved compliance with quality measures at hospital discharge with a computerized physician order entry system, Am Heart J, 151, 643, 10.1016\u002Fj.ahj.2005.05.007\nJohnston, 1994, Effects of computer-based clinical decision support systems on clinician performance and patient outcome. 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Five-year survival following a first admission for heart failure, Eur J Heart Fail, 22, 315, 10.1016\u002FS1388-9842(00)00141-0\nRemme, 2001, Guidelines for the diagnosis and treatment of chronic heart failure, Eur Heart J, 22, 1527, 10.1053\u002Feuhj.2001.2783\nLloyd-Jones, 2004, Lifetime risk for development of atrial fibrillation: the Framingham Heart Study, Circulation, 110, 1042, 10.1161\u002F01.CIR.0000140263.20897.42\nRosamond, 2008, American Heart Association Statistics Committee and Stroke Statistics Subcommittee. Heart disease and stroke statistics—2008 update: a report from the American Heart Association Statistics Committee and Stroke Statistics Subcommittee, Circulation, 117, e25, 10.1161\u002FCIRCULATIONAHA.107.187998\nAnker, 2004, Inflammatory mediators in chronic heart failure: an overview, Heart, 90, 464, 10.1136\u002Fhrt.2002.007005\nvon Haehling, 2002, Cachexia: a therapeutic approach beyond cytokine antagonism, Int J Cardiol, 85, 173, 10.1016\u002FS0167-5273(02)00245-0\nDoehner, 2005, Impaired insulin sensitivity as an independent risk factor for mortality in patients with stable chronic heart failure, J Am Coll Cardiol, 46, 1019, 10.1016\u002Fj.jacc.2005.02.093\nSharma, 2004, Haemoglobin predicts survival in patients with chronic heart failure: a substudy of the ELITE II trial, Eur Heart J, 25, 1021, 10.1016\u002Fj.ehj.2004.04.023\nKomajda, 2006, The impact of new onset anaemia on morbidity and mortality in chronic heart failure: results from COMET, Eur Heart J, 27, 1440, 10.1093\u002Feurheartj\u002Fehl012\nWitte, 2005, The effect of micronutrient supplementation on quality-of-life and left ventricular function in elderly patients with chronic heart failure, Eur Heart J, 26, 2238, 10.1093\u002Feurheartj\u002Fehi442\nJankowska, 2006, Autonomic imbalance and immune activation in chronic heart failure: pathophysiological links, Cardiovasc Res, 70, 434, 10.1016\u002Fj.cardiores.2006.01.013\nKrack, 2005, The importance of the gastrointestinal system in the pathogenesis of heart failure, Eur Heart J, 26, 2368, 10.1093\u002Feurheartj\u002Fehi389\nConraads, 2004, Selective intestinal decontamination in advanced chronic heart failure: a pilot trial, Eur J Heart Fail, 6, 483, 10.1016\u002Fj.ejheart.2003.12.004\nSandek, 2007, Altered intestinal function in patients with chronic heart failure, J Am Coll Cardiol, 50, 1561, 10.1016\u002Fj.jacc.2007.07.016\nAnker, 2002, The syndrome of cardiac cachexia, Int J Cardiol, 85, 51, 10.1016\u002FS0167-5273(02)00233-4\nvon Haehling, 2007, Nutrition, metabolism, and the complex pathophysiology of cachexia in chronic heart failure, Cardiovasc Res, 73, 298, 10.1016\u002Fj.cardiores.2006.08.018\nAnker, 2002, How to RECOVER from RENAISSANCE? The significance of the results of RECOVER, RENAISSANCE, RENEWAL and ATTACH, Int J Cardiol, 86, 123, 10.1016\u002FS0167-5273(02)00470-9\nvon Haehling, 2004, Tumour necrosis factor-alpha and the failing heart: pathophysiology and therapeutic implications, Basic Res Cardiol, 99, 18, 10.1007\u002Fs00395-003-0433-8\nvon Haehling, 2005, Future prospects of anticytokine therapy in chronic heart failure, Expert Opin Investig Drugs, 14, 163, 10.1517\u002F13543784.14.2.163\nLevine, 1990, Elevated circulating levels of tumor necrosis factor in severe chronic heart failure, N Engl J Med, 323, 236, 10.1056\u002FNEJM199007263230405\nMatsumori, 1994, Increased circulating cytokines in patients with myocarditis and cardiomyopathy, Br Heart J, 72, 561, 10.1136\u002Fhrt.72.6.561\nTsutamoto, 1998, Interleukin-6 spillover in the peripheral circulation increases with the severity of heart failure, and the high plasma level of interleukin-6 is an important prognostic predictor in patients with congestive heart failure, J Am Coll Cardiol, 31, 391, 10.1016\u002FS0735-1097(97)00494-4\nGenth-Zotz, 2002, Pathophysiologic quantities of endotoxin-induced tumor necrosis factor-alpha release in whole blood from patients with chronic heart failure, Am J Cardiol, 90, 1226, 10.1016\u002FS0002-9149(02)02839-4\nTriantafilou, 2002, Lipopolysaccharide recognition: CD14, TLRs and the LPS-activation cluster, Trends Immunol, 23, 301, 10.1016\u002FS1471-4906(02)02233-0\nFöldes, 2006, Toll-like receptor modulation in cardiovascular disease: a target for intervention?, Expert Opin Investig Drugs, 15, 857, 10.1517\u002F13543784.15.8.857\nda Silva Correia, 2001, Lipopolysaccharide is in close proximity to each of the proteins in its membrane receptor complex: transfer from CD14 to TLR4 and MD-2, J Biol Chem, 276, 21129, 10.1074\u002Fjbc.M009164200\nShimazu, 1999, MD-2, a molecule that confers lipopolysaccharide responsiveness on Toll-like receptor 4, J Exp Med, 189, 1777, 10.1084\u002Fjem.189.11.1777\nTriantafilou, 2001, A CD14-independent LPS receptor cluster, Nat Immunol, 2, 338, 10.1038\u002F86342\nTriantafilou, 2004, Heat-shock protein 70 and heat-shock protein 90 associate with Toll-like receptor 4 in response to bacterial lipopolysaccharide, Biochem Soc Trans, 32, 636, 10.1042\u002FBST0320636\nGenth-Zotz, 2004, Heat shock protein 70 in patients with chronic heart failure: relation to disease severity and survival, Int J Cardiol, 96, 397, 10.1016\u002Fj.ijcard.2003.08.008\nNiebauer, 1999, Endotoxin and immune activation in chronic heart failure: a prospective cohort study, Lancet, 353, 1838, 10.1016\u002FS0140-6736(98)09286-1\nAxtelle, 2001, IC14, a CD14 specific monoclonal antibody, is a potential treatment for patients with severe sepsis, J Endotoxin Res, 7, 310, 10.1179\u002F096805101101532783\nVerbon, 2001, IC14, an anti-CD14 antibody, inhibits endotoxin-mediated symptoms and inflammatory responses in humans, J Immunol, 166, 3599, 10.4049\u002Fjimmunol.166.5.3599\nManocha, 2002, Novel therapies for sepsis: antiendotoxin therapies, Expert Opin Investig Drugs, 11, 1795, 10.1517\u002F13543784.11.12.1795\nRauchhaus, 2003, The relationship between cholesterol and survival in patients with chronic heart failure, J Am Coll Cardiol, 42, 1933, 10.1016\u002Fj.jacc.2003.07.016\nHorwich, 2002, Low serum total cholesterol is associated with marked increase in mortality in advanced heart failure, J Card Fail, 8, 216, 10.1054\u002Fjcaf.2002.0804216\nHorwich, 2007, Reverse epidemiology beyond dialysis patients: chronic heart failure, geriatrics, rheumatoid arthritis, COPD, and AIDS, Semin Dial, 20, 549, 10.1111\u002Fj.1525-139X.2007.00346.x\nSharma, 2005, Whole blood endotoxin responsiveness in patients with chronic heart failure: the importance of serum lipoproteins, Eur J Heart Fail, 7, 479, 10.1016\u002Fj.ejheart.2004.09.013\nGordon, 1996, Low lipid concentrations in critical illness: implications for preventing and treating endotoxemia, Crit Care Med, 24, 584, 10.1097\u002F00003246-199604000-00006\nMurch, 2007, Lipoproteins in inflammation and sepsis. I. 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Prevention of coronary heart disease with pravastatin in men with hypercholesterolemia, N Engl J Med, 333, 1301, 10.1056\u002FNEJM199511163332001\n1998, The Long-Term Intervention with Pravastatin in Ischaemic Disease (LIPID) Study Group. Prevention of cardiovascular events and death with pravastatin in patients with coronary heart disease and a broad range of initial cholesterol levels, N Engl J Med, 339, 1349, 10.1056\u002FNEJM199811053391902\nSchwartz, 2001, Effects of atorvastatin on early recurrent ischemic events in acute coronary syndromes: the MIRACL study. A randomised controlled trial, JAMA, 285, 1711, 10.1001\u002Fjama.285.13.1711\nVaughan, 2000, The evolving role of statins in the management of atherosclerosis, J Am Coll Cardiol, 35, 1, 10.1016\u002FS0735-1097(99)00525-2\nvon Haehling, 2003, Statins and the role of nitric oxide in chronic heart failure, Heart Fail Rev, 8, 99, 10.1023\u002FA:1022103222857\nvon Haehling, 2005, Statins for heart failure: at the crossroads between cholesterol reduction and pleiotropism?, Heart, 91, 1, 10.1136\u002Fhrt.2004.042515\nWalter, 2004, Effects of statins on endothelium and endothelial progenitor cell recruitment, Semin Vasc Med, 4, 385, 10.1055\u002Fs-2004-869595\nAznar, 2001, Rho signals to cell growth and apoptosis, Cancer Lett, 165, 1, 10.1016\u002FS0304-3835(01)00412-8\nAnker, 2006, Statin use and survival in patients with chronic heart failure: results from two observational studies with 5200 patients, Int J Cardiol, 112, 234, 10.1016\u002Fj.ijcard.2006.03.057\nHorwich, 2004, Statin therapy is associated with improved survival in ischemic and non-ischemic heart failure, J Am Coll Cardiol, 43, 642, 10.1016\u002Fj.jacc.2003.07.049\nNode, 2003, Short-term statin therapy improves cardiac function and symptoms in patients with idiopathic dilated cardiomyopathy, Circulation, 108, 839, 10.1161\u002F01.CIR.0000084539.58092.DE\nSola, 2006, Atorvastatin improves left ventricular systolic function and serum markers of inflammation in nonischemic heart failure, J Am Coll Cardiol, 47, 332, 10.1016\u002Fj.jacc.2005.06.088\nYamada, 2007, Long-term effect of atorvastatin on neurohumoral activation and cardiac function in patients with chronic heart failure: a prospective randomized controlled study, Am Heart J, 153, 1055, 10.1016\u002Fj.ahj.2007.03.027\nBleske, 2006, Neutral effect on markers of heart failure, inflammation, endothelial activation and function, and vagal tone after high-dose HMG-CoA reductase inhibition in non-diabetic patients with non-ischemic cardiomyopathy and average 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decreased rate of severe sepsis, Circulation, 110, 880, 10.1161\u002F01.CIR.0000138932.17956.F1\nMajumdar, 2006, Statins and outcomes in patients admitted to hospital and community acquired pneumonia: population based prospective cohort study, BMJ, 333, 999, 10.1136\u002Fbmj.38992.565972.7C\nMartin, 2007, Effectiveness of statins in reducing the rate of severe sepsis: a retrospective evaluation, Pharmacotherapy, 27, 20, 10.1592\u002Fphco.27.1.20\nvan de Garde, 2006, Statin treatment and reduced risk of pneumonia in patients with diabetes, Thorax, 61, 957, 10.1136\u002Fthx.2006.062885\nMortensen, 2005, The effect of prior statin use on 30-day mortality for patients hospitalized with community-acquired pneumonia, Respir Res, 25, 82, 10.1186\u002F1465-9921-6-82\nGupta, 2007, Statin use and hospitalization for sepsis in patients with chronic kidney disease, JAMA, 297, 1455, 10.1001\u002Fjama.297.13.1455\nThomsen, 2006, Statin use and mortality within 180 days after bacteremia: a population-based cohort study, Crit Care Med, 34, 1080, 10.1097\u002F01.CCM.0000207345.92928.E4\nMonneret, 2006, Persisting low monocyte human leukocyte antigen-DR expression predicts mortality in septic shock, Intensive Care Med, 32, 1175, 10.1007\u002Fs00134-006-0204-8\nReith, 2005, Regulation of MHC class II gene expression by the class II transactivator, Nat Rev Immunol, 5, 793, 10.1038\u002Fnri1708\nSchefold, 2007, A novel selective extracorporeal intervention in sepsis: immunoadsorption of endotoxin, interleukin 6, and complement-activating product 5a, Shock, 28, 418, 10.1097\u002Fshk.0b013e31804f5921\nNierhaus, 2003, Reversal of immunoparalysis by recombinant human granulocyte-macrophage colony-stimulating factor in patients with severe sepsis, Intensive Care Med, 4, 646, 10.1007\u002Fs00134-003-1666-6\n1999, Gruppo Italiano per lo Studio della Sopravvivenza nell'Infarto Miocardico. 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peptide in isolated atrial and ventricular human myocardium: influence of angiotensin II and diastolic fiber length, Circulation, 102, 3074, 10.1161\u002F01.CIR.102.25.3074\nBibbins-Domingo, 2003, B-type natriuretic peptide and ischemia in patients with stable coronary disease: data from the Heart and Soul study, Circulation, 108, 2987, 10.1161\u002F01.CIR.0000103681.04726.9C\nRiddervold, 1991, Rate-induced increase in plasma atrial natriuretic factor can occur independently of changes in atrial wall stretch, Am J Physiol, 260, H1953\nNishimori, 1997, Dexamethasone-induced up-regulation of adrenomedullin and atrial natriuretic peptide genes in cultured rat ventricular myocytes, J Mol Cell Cardiol, 29, 2125, 10.1006\u002Fjmcc.1997.0460\nTsutamoto, 2007, Direct comparison of transcardiac difference between brain natriuretic peptide (BNP) and N-terminal pro-BNP in patients with chronic heart failure, Eur J Heart Fail, 9, 667, 10.1016\u002Fj.ejheart.2007.01.003\nMcCullough, 2003, B-type natriuretic peptide and renal disease, Heart Fail Rev, 8, 355, 10.1023\u002FA:1026195332025\nForfia, 2005, Relationship between B-type natriuretic peptides and pulmonary capillary wedge pressure in the intensive care unit, J Am Coll Cardiol, 45, 1667, 10.1016\u002Fj.jacc.2005.01.046\nHubert, 1983, Obesity as an independent risk factor for cardiovascular disease: a 26-year follow-up of participants in the Framingham Heart Study, Circulation, 67, 968, 10.1161\u002F01.CIR.67.5.968\nTaylor, 2006, B-type natriuretic peptide and N-terminal pro B-type natriuretic peptide are depressed in obesity despite higher left ventricular end diastolic pressures, Am Heart J, 152, 1071, 10.1016\u002Fj.ahj.2006.07.010\nWang, 2004, Impact of obesity on plasma natriuretic peptide levels, Circulation, 109, 594, 10.1161\u002F01.CIR.0000112582.16683.EA\nKrauser, 2005, Effect of body mass index on natriuretic peptide levels in patients with acute congestive heart failure: a ProBNP Investigation of dyspnea in the 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832, 10.1093\u002Feurheartj\u002Fehi772\nIwanaga, 2006, B-type natriuretic peptide strongly reflects diastolic wall stress in patients with chronic heart failure: comparison between systolic and diastolic heart failure, J Am Coll Cardiol, 47, 742, 10.1016\u002Fj.jacc.2005.11.030\nMaeda, 1998, Plasma brain natriuretic peptide as a biochemical marker of high left ventricular end-diastolic pressure in patients with symptomatic left ventricular dysfunction, Am Heart J, 135, 825, 10.1016\u002FS0002-8703(98)70041-9\nMair, 2007, B-type natriuretic peptide (BNP) is more sensitive to rapid hemodynamic changes in acute heart failure than N-terminal proBNP, Clin Chim Acta, 379, 163, 10.1016\u002Fj.cca.2006.12.018\nKazanegra, 2001, A rapid test for B-type natriuretic peptide correlates with falling wedge pressures in patients treated for decompensated heart failure: a pilot study, J Card Fail, 7, 21, 10.1054\u002Fjcaf.2001.23355\nTroughton, 2004, Plasma B-type natriuretic peptide levels in systolic heart failure: importance of left ventricular diastolic function and right ventricular systolic function, J Am Coll Cardiol, 43, 416, 10.1016\u002Fj.jacc.2003.08.046\nParsonage, 2005, Value of B-type natriuretic peptide for identifying significantly elevated pulmonary artery wedge pressure in patients treated for established chronic heart failure secondary to ischemic or idiopathic dilated cardiomyopathy, Am J Cardiol, 95, 883, 10.1016\u002Fj.amjcard.2004.12.021\nKuster, 2002, B-type natriuretic peptide for diagnosis and treatment of congestive heart failure, Swiss Med Wkly, 132, 623\nO'Neill, 2005, B-type natriuretic peptide levels are not a surrogate marker for invasive hemodynamics during management of patients with severe heart failure, Am Heart J, 149, 363, 10.1016\u002Fj.ahj.2004.05.051\nTschope, 2005, The role of NT-proBNP in the diagnostics of isolated diastolic dysfunction: correlation with echocardiographic and invasive measurements, Eur Heart J, 26, 2277, 10.1093\u002Feurheartj\u002Fehi406\nVanderheyden, 2004, Wall stress modulates brain natriuretic peptide production in pressure overload cardiomyopathy, J Am Coll Cardiol, 44, 2349, 10.1016\u002Fj.jacc.2004.09.038\nMottram, 2003, Usefulness of B-type natriuretic peptide in hypertensive patients with exertional dyspnea and normal left ventricular ejection fraction and correlation with new echocardiographic indexes of systolic and diastolic function, Am J Cardiol, 92, 1434, 10.1016\u002Fj.amjcard.2003.08.053\nMottram, 2004, Response of B-type natriuretic peptide to exercise in hypertensive patients with suspected diastolic heart failure: correlation with cardiac function, hemodynamics, and workload, Am Heart J, 148, 365, 10.1016\u002Fj.ahj.2004.02.012\nFukuta, 2007, Elevated left ventricular filling pressure after maximal exercise predicts increased plasma B-type natriuretic peptide levels in patients with impaired relaxation pattern of diastolic filling, J Am Soc Echocardiogr, 20, 832, 10.1016\u002Fj.echo.2007.01.004\nJoung, 2005, Can pro-brain natriuretic peptide be used as a noninvasive predictor of elevated left ventricular diastolic pressures in patients with normal systolic function?, Am Heart J, 150, 1213, 10.1016\u002Fj.ahj.2005.01.014\nAbhayaratna, 2006, Population-based detection of systolic and diastolic dysfunction with amino-terminal pro-B-type natriuretic peptide, Am Heart J, 152, 941, 10.1016\u002Fj.ahj.2006.05.007\nDahlstrom, 2004, Can natriuretic peptides be used for the diagnosis of diastolic heart failure?, Eur J Heart Fail, 6, 281, 10.1016\u002Fj.ejheart.2004.01.005\nRedfield, 2004, Plasma brain natriuretic peptide to detect preclinical ventricular systolic or diastolic dysfunction: a community-based study, Circulation, 109, 3176, 10.1161\u002F01.CIR.0000130845.38133.8F\nHaug, 1993, Plasma brain natriuretic peptide and atrial natriuretic peptide concentrations correlate with left ventricular end-diastolic pressure, Clin Cardiol, 16, 553, 10.1002\u002Fclc.4960160708\nOmland, 1996, Plasma cardiac natriuretic peptide determination as a screening test for the detection of patients with mild left ventricular impairment, Heart, 76, 232, 10.1136\u002Fhrt.76.3.232\nPfister, 2004, Use of NT-proBNP in routine testing and comparison to BNP, Eur J Heart Fail, 6, 289, 10.1016\u002Fj.ejheart.2003.12.012\nMarz, 2007, N-terminal pro-B-type natriuretic peptide predicts total and cardiovascular mortality in individuals with or without stable coronary artery disease: the Ludwigshafen Risk and Cardiovascular Health Study, Clin Chem, 53, 1075, 10.1373\u002Fclinchem.2006.075929\nDokainish, 2004, Optimal noninvasive assessment of left ventricular filling pressures: a comparison of tissue Doppler echocardiography and B-type natriuretic peptide in patients with pulmonary artery catheters, Circulation, 109, 2432, 10.1161\u002F01.CIR.0000127882.58426.7A\nTung, 2004, Utility of B-type natriuretic peptide for the evaluation of intensive care unit shock, Crit Care Med, 32, 1643, 10.1097\u002F01.CCM.0000133694.28370.7F\nShah, 2005, Impact of the pulmonary artery catheter in critically Ill patients: meta-analysis of randomized clinical trials, JAMA, 294, 1664, 10.1001\u002Fjama.294.13.1664\nKnebel, 2005, NT-ProBNP in acute heart failure: correlation with invasively measured hemodynamic parameters during recompensation, J Card Fail, 11, S38, 10.1016\u002Fj.cardfail.2005.04.012\nGegenhuber, 2004, Time course of B-type natriuretic peptide (BNP) and N-terminal proBNP changes in patients with decompensated heart failure, Clin Chem, 50, 454, 10.1373\u002Fclinchem.2003.028712\nTroughton, 2006, BNP for clinical monitoring of heart failure, Heart Fail Clin, 2, 333, 10.1016\u002Fj.hfc.2006.08.006\nO'Hanlon, 2007, The biologic variability of B-type natriuretic peptide and N-terminal pro-B-type natriuretic peptide in stable heart failure patients, J Card Fail, 13, 50, 10.1016\u002Fj.cardfail.2006.09.003\nHare, 2008, Performance of conventional echocardiographic parameters and myocardial measurements in the sequential evaluation of left ventricular ejection function, Am J Cardiol, 10.1016\u002Fj.amjcard.2007.10.037\nPetrie, 2004, Poor concordance of commonly used echocardiographic measures of left ventricular diastolic function in patients with suspected heart failure but preserved systolic function: is there a reliable echocardiographic measure of diastolic dysfunction?, Heart, 90, 511, 10.1136\u002Fhrt.2003.011403\nBurgess, 2006, Diastolic stress echocardiography: hemodynamic validation and clinical significance of estimation of ventricular filling pressure with exercise, J Am Coll Cardiol, 47, 1891, 10.1016\u002Fj.jacc.2006.02.042\nWijbenga, 1999, Cardiac peptides differ in their response to exercise. 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