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Journal of Medicine and Pharmacy","Tạp chí Y Dược học Cần Thơ",{"EN":487,"VI":488},"\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">04\u002F10\u002F2015 Ministry of Information and Communications allowed Can Tho journal of medicine and pharmacy to operate (102 \u002FGP-BTTTT)\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">07\u002F16\u002F2015 Can Tho journal of medicine and pharmacy is internationally recognized: ISSN 2354-1210\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">In 2016, The journal has been included in the list of medical science journals by The State Council for professorship which is awarded a work score of 0-0.5 points for a published article.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Can Tho Journal of Medicine and Pharmacy welcome original works that haven’t been submitted or published in other medical journals. Posts must contain content related to one of the journal’s categories.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The content published\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The journal is divided into 3 categories:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Scientific research article: are valuable scientific works, which have been researched and accepted.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Overview of medicine, biology and pharmacy: serving the objective of continuing training in the fields of medicine, biology and pharmacy; to systematize classical and modern knowledge.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Update information on new knowledge about medicine, biology, pharmacy in the country and in the world.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Scope\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Publication and introduction of scientific research in the fields:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Medicine (internal medicine, surgery, pediatrics, obstetrics and gynecology, odonto-stomatology, laboratory, oncology, traditional medicine, nursing).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Biology (genetics, biotechnology).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Pharmacology (pharmaceutics, drug quality analysis-control, synthetic pharmaceutical chemistry, biochemistry, pharmacognosy, botany, clinical pharmacy).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- To enhance the quality of undergraduate, postgraduate education, scientifically researching and meet the necessary treatment in hospital.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Introducing the updated domestic and oversea information about science technology to promote scientific research and exchanging technology in local, other universities.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Exchanging pharmaceutical and medical information for social health developing in the Mekong Delta and Vietnam.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The object\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Postgraduate students, student of Can Tho University of Medicine and Pharmacy, scientists from schools, research institutes, hospitals, health centers, pharmaceutical companies of the Mekong Delta; other provinces and regions in Vietnam and other country.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Address\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Headquarters of Can Tho Journal of Medicine and Pharmacy, located Scientific Research and International Cooperation Office: 179 Nguyen Van Cu Street, An Khanh Ward, Ninh Kieu District, Can Tho City, Vietnam.\u003C\u002Fspan>\u003C\u002Fp>","\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Ngày 16\u002F7\u002F2015, Tạp chí Y Dược học Cần Thơ được cấp chỉ số quốc tế: ISSN 2354-1210.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 4\u002F2016, Tạp chí đã được Hội đồng Giáo sư ngành Y đưa vào danh sách các tạp chí khoa học Y học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Năm 2020 Tạp chí Y Dược học Cần Thơ đã được phê duyệt vào danh mục của các Hội đồng Giáo sư ngành Dược học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ ra 12 số\u002Fnăm, 180-200 trang\u002Fsố.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 12\u002F2022 Tạp chí Y Dược học Cần Thơ là thành viên của hệ thống Crossref và từ tháng 01\u002F2023 tạp chí thực hiện bình duyệt online kín 2 chiều nhằm tăng tính minh bạch, tin cậy của các công trình nghiên cứu khoa học và đảm bảo tốt nhất chất lượng khoa học của bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ, mục đích và phạm vi của tạp chí\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ và mục đích hoạt động của tạp chí: xuất bản nhằm mục đích phổ biến kết quả từ các đề tài nghiên cứu khoa học; giao lưu trao đổi khoa học, chia sẻ kinh nghiệm, học tập, đồng thời cập nhật thông tin khoa học mới trong các lĩnh vực y, sinh, dược học trong và ngoài nước.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phạm vi của tạp chí: Tạp chí xuất bản được chia thành 3 chuyên mục: (i) Bài báo nghiên cứu khoa học là kết quả công trình nghiên cứu khoa học có giá trị đã được triển khai nghiên cứu, (ii) Bài tổng quan y, sinh, dược học: phục vụ mục tiêu đào tạo liên tục trong lĩnh vực y, sinh, dược học; nhằm hệ thống hóa những kiến thức kinh điển và hiện đại; (iii) Thông tin cập nhật kiến thức mới về y, sinh, dược học trong nước và trên thế giới.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Chính sách truy cập mở\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ áp dụng chính sách truy cập mở đối với các bài báo đã xuất bản đến với độc giả, nhằm mở rộng cơ hội tiếp cận các kết quả nghiên cứu chất lượng cao và tăng cường trao đổi kiến thức. Tạp chí đăng tải trực tuyến (miễn phí) toàn văn các bài báo được công bố trên website của Tạp chí (https:\u002F\u002Ftapchi.ctump.edu.vn).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đạo đức xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ cam kết tuân thủ đạo đức xuất bản phù hợp với các hướng dẫn và tiêu chuẩn của the Committee on Publication Ethics (COPE), tuân thủ các nguyên tắc của COPE’s Core Practices, Best Practices Guidelines for Journal Editors và Guidelines on Good Publication Practices.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Bản thảo bài báo chỉ được chấp nhận khi được tác giả chịu trách nhiệm chính cam kết các nội dung sau: Các nội dung của bản thảo chưa được đăng tải toàn bộ hoặc một phần ở các tạp chí khác; Tất cả các tác giả đều có đóng góp một cách đáng kể vào quá trình nghiên cứu hoặc chuẩn bị bản thảo và cùng chịu trách nhiệm về các nội dung của bản thảo; Tuân thủ các biện pháp đảm bảo đạo đức nghiên cứu (ví dụ thỏa thuận đồng ý tham gia nghiên cứu).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Cam kết bảo mật\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí cam kết thực hiện và tuân thủ các quy định của luật và các văn bản hướng dẫn liên quan đến bảo mật thông tin cá nhân trên không gian mạng. Các thông tin mà người dùng (tác giả, độc giả, biên tập viên, người phản biện) nhập vào các biểu mẫu trên Hệ thống Quản lý xuất bản trực tuyến của tạp chí chỉ được sử dụng vào các mục đích đã được tuyên bố rõ ràng và sẽ không được cung cấp cho bất kỳ bên thứ ba nào khác, hay dùng vào bất kỳ mục đích nào khác.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phí gửi bài\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng bài: 1.000.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng nhanh: 1.500.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với tác giả là cán bộ viên chức thuộc Trường Đại học Y Dược Cần Thơ thì được hỗ trợ 50% lệ phí gửi đăng bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với sinh viên thực hiện đề tài nghiên cứu khoa học cấp trường được hỗ trợ 100% lệ phí đăng bài ( Tác giả gửi đính kèm “ Quyết định về việc giao tổ chức thực hiện đề tài nghiên cứu khoa học cấp Trường của sinh viên”).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Hình thức nộp lệ phí:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Tiền mặt:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Nộp trực tiếp tại Phòng Tài chính - Kế toán, Trường Đại học Y Dược Cần Thơ, số 179 Nguyễn Văn Cừ, P. An Khánh, Q. Ninh Kiều, thành phố Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Chuyển khoản:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tên Tài khoản: Trường ĐHYD Cần Thơ, Số TK: 0111000115668, tại ngân hàng Vietcombank chi nhánh Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Thời gian: Áp dụng từ ngày 01\u002F02\u002F2023.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">* Phí gửi bài không được hoàn trả khi bài viết bị từ chối hoặc tác giả xin rút bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Quy trình phản biện bài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ thực hiện quy trình phản biện kín hai chiều nghiêm ngặt. Danh tính của những người phản biện không được tiết lộ cho các tác giả và ngược lại. Quy trình thẩm định bài báo đăng gồm các bước sau:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tiếp nhận bản thảo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tác giả liên hệ gửi bản thảo đến Tạp chí qua hệ thống trực tuyến tại website: https:\u002F\u002Ftapchi.ctump.edu.vn. Hướng dẫn về cách đăng ký, gửi bài và chuẩn bị bản thảo được cung cấp trên website của Tạp chí.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sàng lọc sơ bộ\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sau khi Tòa soạn nhận được bài báo của tác giả, Ban Thư ký sẽ tiến hành kiểm tra sơ bộ bài báo (các yêu cầu về nội dung và hình thức). Những bài báo không đúng quy cách hoặc có nội dung không phù hợp hoặc vi phạm bản quyền sẽ bị từ chối (Ban Thư ký thông báo phản hồi đến tác giả trong vòng 1 tuần). Những bài báo đủ điều kiện, được Ban Thư ký tòa soạn chuyển đến Ban Biên tập có cùng chuyên môn với nội dung bài báo để đề xuất người phản biện. Thời gian kể từ khi Ban Biên tập nhận bài báo đến khi đề xuất người phản biện bài báo chậm nhất là 5 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Vòng phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký gửi bài và yêu cầu phản biện đến 02 phản biện độc lập.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Các phản biện gởi nhận xét cho Ban Thư ký. Thời gian từ khi gửi bài cho phản biện đến khi nhận ý kiến của phản biện tối đa là 20 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xử ký kết quả phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Nếu ý kiến đồng ý cho đăng và không cần chỉnh sửa, Ban Thư ký tiếp tục đăng bài theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Nếu ý kiến đồng ý đăng và cần chỉnh sửa, Ban Thư ký sẽ thông tin đến tác giả chỉnh sửa theo yêu cầu của người phản biện. Thời gian chỉnh sửa và gửi lại kéo dài không quá 2 tuần, từ khi tác giả bài báo nhận được thông tin (Quá trình này có thể lặp lại tối đa 2 lần\u002F1 bài báo). Khi có sự thống nhất, đồng ý của người phản biện; bài báo được tiếp tục đăng theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Những bài báo có chất lượng không đạt yêu cầu, cả 2 phản biện không đồng ý cho đăng sẽ bị Tòa soạn từ chối đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký tổng hợp các bản thảo đã được tác giả hoàn thiện sau thẩm định trình Ban Biên tập xem xét, Tổng Biên tập phê duyệt, quyết định bài đăng theo các tiêu chí: sự phù hợp nội dung với tôn chỉ và mục đích, thể loại bài viết (ưu tiên các bài có bài có nghiên cứu chuyên sâu, hàm lượng khoa học cao), đóng góp mới bài báo, bài báo được ưu tiên đăng trong số gần nhất của Tạp chí theo thứ tự: tính thời sự, chất lượng bài báo và thời gian gửi bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Ban Biên tập và Ban Thư ký biên tập bản thảo, chế bản, đọc rà soát lỗi. Thời gian hoàn thành từ 10-15 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Ban Thư ký có trách nhiệm thông báo cho tác giả bài báo (bằng e-mail) về tình hình phê duyệt bài báo, thời gian, số kỳ, tập xuất bản bài báo theo qui định.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">4. Danh sách bài báo theo số Tạp chí được in ấn và phát hành trong năm định kỳ được công bố chính thức trên website: https:\u002F\u002Ftapchi.ctump.edu.vn\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>",{"VOID":490},"wcQ1uqwAAAAJ","2023-05-30T08:17:21.868+00:00",[],[494],{"id":495,"createTime":28,"updateTime":28,"relativeEntities":496,"slug":28,"properties":497,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":507,"parentIds":508,"statistic":28},"6413896b-eca9-442b-a73f-182a58a0ce40",[],{"title":498,"address":501,"country":504,"abbreviation":505},{"EN":499,"VI":500},"Can Tho University of Medicine and Pharmacy","Trường Đại học Y Dược Cần Thơ",{"EN":502,"VI":503},"No 179, Nguyen Van Cu street, An Khanh ward, Ninh Kieu district, Can Tho city, Vietnam","Số 179, đường Nguyễn Văn Cừ, phường An Khánh, quận Ninh Kiều, thành phố Cần Thơ, Việt Nam",{"VOID":15},{"VOID":506},"ctump","http:\u002F\u002Fwww.ctump.edu.vn\u002F",[],[],"https:\u002F\u002Ftapchi.ctump.edu.vn\u002Findex.php\u002Fctump",{"impactFactor":32,"impactFactorByYear":512,"i10Index":32,"i10IndexLast5Year":32,"totalPublication":514,"totalPublicationByYear":515,"totalCitation":520,"totalCitationByYear":521,"totalCitationPerPublication":108,"totalCitationPerPublicationByYear":523,"hindexLast5Year":45,"hindex":45},{"2022":513,"2023":111,"2024":106},0.01,1556,{"2020":47,"2021":516,"2022":517,"2023":518,"2024":519,"2025":122},57,306,801,358,161,{"2021":146,"2022":280,"2023":522},99,{"2021":524,"2022":318,"2023":104},0.23,{"impactFactor":28,"impactFactorByYear":28,"i10Index":123,"i10IndexLast5Year":123,"totalPublication":526,"totalPublicationByYear":527,"totalCitation":526,"totalCitationByYear":528,"totalCitationPerPublication":40,"totalCitationPerPublicationByYear":531,"hindexLast5Year":49,"hindex":49},476,{"0":205,"2019":123,"2021":139,"2022":459,"2023":451,"2024":357,"2025":49,"2026":48},{"2021":42,"2022":123,"2023":161,"2024":529,"2025":360,"2026":530},136,83,{"2021":105,"2022":513,"2023":532,"2024":127,"2025":533,"2026":534},0.62,25.43,13.83,{"id":536,"createTime":537,"updateTime":382,"relativeEntities":538,"slug":539,"properties":540,"entityType":25,"verifyStatus":26,"verifyTime":28,"verifyNote":28,"languages":552,"translateLanguages":28,"viewCount":133,"subjectFields":553,"manageAffiliations":554,"indexDatabases":555,"url":556,"thumbnailPath":557,"statistic":558,"gsStatistic":594,"type":55,"analyzePriority":28},"6984a56a-db70-403b-9cc4-4013e1ceaffa","2023-05-09T06:47:40.346+00:00",[],"T%E1%BA%A1p%20ch%C3%AD%20Nghi%C3%AAn%20c%E1%BB%A9u%20n%C6%B0%E1%BB%9Bc%20ngo%C3%A0i",{"country":541,"issn":542,"title":544,"introduce":547,"gsId":550},{"VOID":15},{"VOID":543},"25252445",{"EN":545,"VI":546},"VNU Journal of Foreign Studies","Tạp chí Nghiên cứu nước ngoài",{"EN":548,"VI":549},"{\"ops\":[{\"insert\":\"\\n\\nThe \\n\"},{\"attributes\":{\"italic\":true},\"insert\":\"VNU Journal of Science\"},{\"insert\":\"\\n was established in 1985 for the publication of national and international research papers in all fields of natural sciences and technology, social sciences and humanities. 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Urban and rural populations were considered separately for developing countries.\u003C\u002Fjats:p>\u003Cjats:p>RESULTS—The prevalence of diabetes for all age-groups worldwide was estimated to be 2.8% in 2000 and 4.4% in 2030. The total number of people with diabetes is projected to rise from 171 million in 2000 to 366 million in 2030. The prevalence of diabetes is higher in men than women, but there are more women with diabetes than men. The urban population in developing countries is projected to double between 2000 and 2030. The most important demographic change to diabetes prevalence across the world appears to be the increase in the proportion of people >65 years of age.\u003C\u002Fjats:p>\u003Cjats:p>CONCLUSIONS—These findings indicate that the “diabetes epidemic” will continue even if levels of obesity remain constant. Given the increasing prevalence of obesity, it is likely that these figures provide an underestimate of future diabetes prevalence.\u003C\u002Fjats:p>","\u003Cjats:p>MỤC TIÊU—Mục tiêu của nghiên cứu này là ước lượng tỷ lệ mắc bệnh đái tháo đường và số lượng người ở mọi độ tuổi mắc bệnh đái tháo đường trong các năm 2000 và 2030.\u003C\u002Fjats:p>\u003Cjats:p>THIẾT KẾ NGHIÊN CỨU VÀ PHƯƠNG PHÁP—Dữ liệu về tỷ lệ mắc bệnh đái tháo đường theo độ tuổi và giới tính từ một số ít quốc gia đã được ngoại suy cho tất cả 191 quốc gia thành viên của Tổ chức Y tế Thế giới và áp dụng cho ước lượng dân số của Liên Hợp Quốc trong năm 2000 và 2030. Dân số đô thị và nông thôn ở các quốc gia đang phát triển được xem xét riêng biệt.\u003C\u002Fjats:p>\u003Cjats:p>KẾT QUẢ—Tỷ lệ mắc bệnh đái tháo đường trên toàn cầu đối với tất cả các nhóm tuổi ước tính là 2.8% vào năm 2000 và 4.4% vào năm 2030. Tổng số người mắc bệnh đái tháo đường được dự đoán sẽ tăng từ 171 triệu người vào năm 2000 lên 366 triệu người vào năm 2030. Tỷ lệ mắc bệnh đái tháo đường cao hơn ở nam giới so với nữ giới, nhưng số lượng nữ giới mắc bệnh đái tháo đường nhiều hơn nam giới. Dân số đô thị ở các quốc gia đang phát triển được dự báo sẽ gấp đôi từ năm 2000 đến 2030. Thay đổi nhân khẩu học quan trọng nhất đối với tỷ lệ mắc bệnh đái tháo đường trên toàn thế giới dường như là sự gia tăng tỷ lệ người >65 tuổi.\u003C\u002Fjats:p>\u003Cjats:p>KẾT LUẬN—Những phát hiện này cho thấy rằng “dịch bệnh đái tháo đường” sẽ tiếp tục ngay cả khi mức béo phì giữ nguyên. 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Diabetes Care 16:157–177, 1993",{"doi":1153},"10.2337\u002Fdiacare.16.1.157",{"id":28,"text":1155,"url":28,"identifiers":1156},"King H, Aubert RE, Herman WH: Global burden of diabetes, 1995–2025: prevalence, numerical estimates, and projections. Diabetes Care 21:1414–1431, 1998",{"doi":1157},"10.2337\u002Fdiacare.21.9.1414",{"id":28,"text":1159,"url":28,"identifiers":1160},"Amos AF, McCarty DJ, Zimmet P: The rising global burden of diabetes and its complications: estimates and projections to the year 2010. Diabet Med 14 (Suppl. 5):S1–S85, 1997",{"doi":1161},"10.1002\u002F(SICI)1096-9136(199712)14:5+\u003CS7::AID-DIA522>3.0.CO;2-R",{"id":28,"text":1163,"url":28,"identifiers":1164},"Pan XR, Yang WY, Li GW, Liu J: Prevalence of diabetes and its risk factors in China, 1994: National Diabetes Prevention and Control Cooperative Group. Diabetes Care 20:1664–1669, 1997",{"doi":1165},"10.2337\u002Fdiacare.20.11.1664",{"id":28,"text":1167,"url":28,"identifiers":1168},"Aspray TJ, Mugusi F, Rashid S, Whiting D, Edwards R, Alberti KG, Unwin NC, Essential Non-Communicable Disease Health Intervention Project: Rural and urban differences in diabetes prevalence in Tanzania: the role of obesity, physical inactivity and urban living. Trans R Soc Trop Med Hyg 94:637–644, 2000",{"doi":1169},"10.1016\u002FS0035-9203(00)90216-5",{"id":28,"text":1171,"url":28,"identifiers":1172},"International Diabetes Federation: Diabetes Atlas 2000. Brussels, International Diabetes Federation, 2000",{},{"id":28,"text":1174,"url":28,"identifiers":1175},"Barendregt JJ, Van Oortmarssen GJ, Vos T, Murray CJ: Generic model for the assessment of disease epidemiology: the computational basis of DisMod II. 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Diabetes 39:390–396, 1990",{"doi":1278},"10.2337\u002Fdiabetes.39.3.390",{"id":28,"text":1280,"url":28,"identifiers":1281},"Suvd J, Gerel B, Otgooloi H, Purevsuren D, Zolzaya H, Roglic G, King H: Glucose intolerance and associated factors in Mongolia. Diabet Med 19:502–508, 2002",{"doi":1282},"10.1046\u002Fj.1464-5491.2002.00737.x",{"id":28,"text":1284,"url":28,"identifiers":1285},"Zimmet P, King H, Taylor R, Raper LR, Balkau B, Borger J, Heriot W, Thoma K: The high prevalence of diabetes mellitus, impaired glucose tolerance and diabetic retinopathy in Nauru: the 1982 survey. Diabetes Res 1:13–18, 1984",{},{"id":28,"text":1287,"url":28,"identifiers":1288},"Mooy JM, Grootenhuis PA, de Vries H, Valkenburg HA, Bouter LM, Kostense PJ, Heine RJ: Prevalence and determinants of glucose intolerance in a Dutch Caucasian population: the Hoorn Study. Diabetes Care 18:1270–1273, 1995",{"doi":1289},"10.2337\u002Fdiacare.18.9.1270",{"id":28,"text":1291,"url":28,"identifiers":1292},"Asfour MG, Lambourne A, Soliman A, Al-Behlani S, Al-Asfoor D, Bold A, Mahtab H, King H: High prevalence of diabetes mellitus and impaired glucose tolerance in the Sultanate of Oman: results of the 1991 national survey. Diabet Med 12:1122–1125, 1995",{"doi":1293},"10.1111\u002Fj.1464-5491.1995.tb00431.x",{"id":28,"text":1295,"url":28,"identifiers":1296},"Shera AS, Rafique G, Khawaja IA, Baqai S, King H: Pakistan National Diabetes Survey: prevalence of glucose intolerance and associated factors in Baluchistan province. Diabetes Res Clin Pract Suppl 44:49–58, 1999",{"doi":1297},"10.1016\u002FS0168-8227(99)00017-0",{"id":28,"text":1299,"url":28,"identifiers":1300},"Shera AS, Rafique G, Khwaja IA, Ara J, Baqai S, King H: Pakistan national diabetes survey: prevalence of glucose intolerance and associated factors in Shikarpur, Sindh Province. Diabet Med 12:1116–1121, 1995",{"doi":1301},"10.1111\u002Fj.1464-5491.1995.tb00430.x",{"id":28,"text":1303,"url":28,"identifiers":1304},"Jimenez JT, Palacios M, Canete F, Barriocanal LA, Medina U, Figueredo R, Martinez S, de Melgarejo MV, Weik S, Kiefer R, Alberti KG, Moreno-Azorero R: Prevalence of diabetes mellitus and associated cardiovascular risk factors in an adult urban population in Paraguay. Diabet Med 15:334–338, 1998",{"doi":1305},"10.1002\u002F(SICI)1096-9136(199804)15:4\u003C334::AID-DIA575>3.0.CO;2-Z",{"id":28,"text":1307,"url":28,"identifiers":1308},"Collins VR, Dowse GK, Toelupe PM, Imo TT, Aloaina FL, Spark RA, Zimmet PZ: Increasing prevalence of NIDDM in the Pacific island population of Western Samoa over a 13-year period. Diabetes Care 17:288–296, 1994",{"doi":1309},"10.2337\u002Fdiacare.17.4.288",{"id":28,"text":1311,"url":28,"identifiers":1312},"el Hazmi MA, Warsy AS, al Swailem AR, al Swailem AM, Sulaimani R: Diabetes mellitus as a health problem in Saudi Arabia. East Mediterr Health J 4:58–67, 1998",{"doi":1313},"10.26719\u002F1998.4.1.58",{"id":28,"text":1315,"url":28,"identifiers":1316},"Tan CE, Emmanuel SC, Tan BY, Jacob E: Prevalence of diabetes and ethnic differences in cardiovascular risk factors: the 1992 Singapore National Health Survey. Diabetes Care 22:241–247, 1999",{"doi":1317},"10.2337\u002Fdiacare.22.2.241",{"id":28,"text":1319,"url":28,"identifiers":1320},"Levitt NS, Katzenellenbogen JM, Bradshaw D, Hoffman MN, Bonnici F: The prevalence and identification of risk factors for NIDDM in urban Africans in Cape Town, South Africa. Diabetes Care 16:601–607, 1993",{"doi":1321},"10.2337\u002Fdiacare.16.4.601",{"id":28,"text":1323,"url":28,"identifiers":1324},"Park Y, Lee H, Koh CS, Min H, Yoo K, Kim Y, Shin Y: Prevalence of diabetes and IGT in Yonchon County, South Korea. Diabetes Care 18:545–548, 1995",{"doi":1325},"10.2337\u002Fdiacare.18.4.545",{"id":28,"text":1327,"url":28,"identifiers":1328},"Castell C, Tresserras R, Serra J, Goday A, Lloveras G, Salleras L: Prevalence of diabetes in Catalonia (Spain): an oral glucose tolerance test-based population study. Diabetes Res Clin Pract Suppl 43:33–40, 1999",{"doi":1329},"10.1016\u002FS0168-8227(98)00125-9",{"id":28,"text":1331,"url":28,"identifiers":1332},"Elbagir MN, Eltom MA, Elmahadi EM, Kadam IM, Berne C: A population-based study of the prevalence of diabetes and impaired glucose tolerance in adults in Northern Sudan. Diabetes Care 19:1126–1128, 1996",{"doi":1333},"10.2337\u002Fdiacare.19.10.1126",{"id":28,"text":1335,"url":28,"identifiers":1336},"Beckles GL: Diabetes Mellitus and Hypertension: Prevalence and Mortality Risk in Urban Trinidad: The St. James Cardiovascular Survey 1977–1985. Washington, D.C., Pan American Health Organization, 1998",{},{"id":28,"text":1338,"url":28,"identifiers":1339},"Papoz L, Ben Khalifa F, Eschwege E, Ben Ayed H: Diabetes mellitus in Tunisia: description in urban and rural populations. Int J Epidemiol 17:419–422, 1988",{"doi":1340},"10.1093\u002Fije\u002F17.2.419",{"id":28,"text":1342,"url":28,"identifiers":1343},"Satman I, Yilmaz T, Sengul A, Salman S, Salman F, Uygur S, Bastar I, Tutuncu Y, Sargin M, Dinccag N, Karsidag K, Kalaca S, Ozcan C, King H: Population-based study of diabetes and risk characteristics in Turkey: results of the Turkish Diabetes Epidemiology Study (TURDEP). Diabetes Care 25:1551–1556, 2002",{"doi":1344},"10.2337\u002Fdiacare.25.9.1551",{"id":28,"text":1346,"url":28,"identifiers":1347},"Harris MI, Flegal KM, Cowie CC, Eberhardt MS, Goldstein DE, Little RR, Wiedmeyer HM, Byrd-Holt DD: Prevalence of diabetes, impaired fasting glucose, and impaired glucose tolerance in U.S. adults: the Third National Health and Nutrition Examination Survey, 1988–1994. Diabetes Care 21:518–524, 1998",{"doi":1348},"10.2337\u002Fdiacare.21.4.518",{"id":28,"text":1350,"url":28,"identifiers":1351},"King H, Abdullaev B, Djumaeva S, Nikitin V, Ashworth L, Dobo MG: Glucose intolerance and associated factors in the Fergana Valley, Uzbekistan. Diabet Med 15:1052–1062, 1998",{"doi":1352},"10.1002\u002F(SICI)1096-9136(1998120)15:12\u003C1052::AID-DIA661>3.0.CO;2-A",false,{"id":1355,"createTime":1356,"updateTime":1357,"relativeEntities":1358,"slug":1359,"properties":1360,"entityType":978,"verifyStatus":26,"verifyTime":1356,"verifyNote":979,"languages":1376,"translateLanguages":1377,"viewCount":32,"primaryUrl":1378,"fullTextUrl":28,"authors":1379,"publicationType":1075,"publisherRelationship":1432,"citationCount":1486,"citationInfo":1487,"publishDate":1498,"publishYear":1488,"citationAnalyzeStatus":884,"lastCitationAnalyze":28,"indexDatabases":1499,"openAccess":28,"references":1500,"isForceReanalyzing":1353},"c850e1c3-8a0f-4ff8-9431-a511e3cf8ebc","2024-09-21T08:07:01.083+00:00","2025-02-02T21:09:04.760+00:00",[],"Global-Burden-of-Diabetes-1995-2025-Prevalence-numerical-estimates-and-projections",{"mag":1361,"keywords":1363,"openalex":1365,"abstract":1367,"title":1370,"pm":1373,"doi":1375},{"VOID":1362},"2031484324",{"VI":1364},"",{"VOID":1366},"W2031484324",{"VI":1368,"EN":1369},"\u003Cjats:sec>\n                  \u003Cjats:title>MỤC TIÊU\u003C\u002Fjats:title>\n                  \u003Cjats:p>Để ước tính tỷ lệ mắc bệnh tiểu đường và số người mắc bệnh tiểu đường từ 20 tuổi trở lên ở tất cả các quốc gia trên thế giới trong ba thời điểm, tức là năm 1995, 2000 và 2025, và để tính toán thêm các tham số khác như tỷ lệ giới tính, tỷ lệ thành phố-nông thôn và cấu trúc độ tuổi của dân số mắc bệnh tiểu đường.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>\n               \u003Cjats:sec>\n                  \u003Cjats:title>THIẾT KẾ NGHIÊN CỨU VÀ PHƯƠNG PHÁP\u003C\u002Fjats:title>\n                  \u003Cjats:p>Các ước tính tỷ lệ mắc bệnh tiểu đường theo độ tuổi đã được áp dụng cho các ước tính và dự báo về dân số của Liên Hợp Quốc cho số người trưởng thành từ 20 tuổi trở lên ở tất cả các quốc gia trên thế giới. Đối với các quốc gia đang phát triển, dân số đô thị và nông thôn được xem xét riêng biệt.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>\n               \u003Cjats:sec>\n                  \u003Cjats:title>KẾT QUẢ\u003C\u002Fjats:title>\n                  \u003Cjats:p>Tỷ lệ mắc bệnh tiểu đường ở người lớn trên toàn cầu được ước tính là 4,0% vào năm 1995 và sẽ tăng lên 5,4% vào năm 2025. Tỷ lệ mắc cao hơn ở các quốc gia phát triển so với các quốc gia đang phát triển. Số người trưởng thành mắc bệnh tiểu đường trên thế giới sẽ tăng từ 135 triệu vào năm 1995 lên 300 triệu vào năm 2025. Phần lớn sự gia tăng số liệu này sẽ xảy ra ở các quốc gia đang phát triển. Sẽ có sự gia tăng 42%, từ 51 triệu lên 72 triệu, ở các quốc gia phát triển và sự gia tăng 170%, từ 84 triệu lên 228 triệu, ở các quốc gia đang phát triển. Do đó, đến năm 2025, >75% số người mắc bệnh tiểu đường sẽ cư trú ở các quốc gia đang phát triển, so với 62% vào năm 1995. Các quốc gia có số người mắc bệnh tiểu đường cao nhất là, và sẽ là vào năm 2025, Ấn Độ, Trung Quốc và Hoa Kỳ. Ở các quốc gia đang phát triển, phần lớn số người mắc bệnh tiểu đường nằm trong độ tuổi từ 45–64. Ở các quốc gia phát triển, phần lớn số người mắc bệnh tiểu đường từ 65 tuổi trở lên. Mô hình này sẽ được làm nổi bật hơn vào năm 2025. Có nhiều phụ nữ mắc bệnh tiểu đường hơn nam giới, đặc biệt là ở các quốc gia phát triển. Trong tương lai, bệnh tiểu đường sẽ ngày càng tập trung nhiều hơn ở khu vực đô thị.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>\n               \u003Cjats:sec>\n                  \u003Cjats:title>KẾT LUẬN\u003C\u002Fjats:title>\n                  \u003Cjats:p>Báo cáo này ủng hộ những dự đoán trước đó về tính chất dịch tễ của bệnh tiểu đường trên thế giới trong quý đầu tiên của thế kỷ 21. Nó cũng cung cấp một bức tranh tạm thời về các đặc điểm của dịch bệnh. Giám sát bệnh tiểu đường trên toàn cầu là bước đầu tiên cần thiết hướng tới việc phòng ngừa và kiểm soát bệnh, điều này hiện được công nhận là một ưu tiên cấp bách.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>","\u003Cjats:sec>\n                  \u003Cjats:title>OBJECTIVE\u003C\u002Fjats:title>\n                  \u003Cjats:p>To estimate the prevalence of diabetes and the number of people with diabetes who are ≥20 years of age in all countries of the world for three points in time, i.e., the years 1995, 2000, and 2025, and to calculate additional parameters, such as sex ratio, urban-rural ratio, and the age structure of the diabetic population.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>\n               \u003Cjats:sec>\n                  \u003Cjats:title>RESEARCH DESIGN AND METHODS\u003C\u002Fjats:title>\n                  \u003Cjats:p>Age-specific diabetes prevalence estimates were applied to United Nations population estimates and projections for the number of adults aged ≥20 years in all countries of the world. For developing countries, urban and rural populations were considered separately\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>\n               \u003Cjats:sec>\n                  \u003Cjats:title>RESULTS\u003C\u002Fjats:title>\n                  \u003Cjats:p>Prevalence of diabetes in adults worldwide was estimated to be 4.0% in 1995 and to rise to 5.4% by the year 2025. It is higher in developed than in developing countries. The number of adults with diabetes in the world will rise from 135 million in 1995 to 300 million in the year 2025. The major part of this numerical increase will occur in developing countries. There will be a 42% increase, from 51 to 72 million, in the developed countries and a 170% increase, from 84 to 228 million, in the developing countries. Thus, by the year 2025, >75% of people with diabetes will reside in developing countries, as compared with 62% in 1995. The countries with the largest number of people with diabetes are, and will be in the year 2025, India, China, and the U.S. In developing countries, the majority of people with diabetes are in the age range of 45–64 years. In the developed countries, the majority of people with diabetes are aged ≥65 years. This pattern will be accentuated by the year 2025. There are more women than men with diabetes, especially in developed countries. In the future, diabetes will be increasingly concentrated in urban areas.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>\n               \u003Cjats:sec>\n                  \u003Cjats:title>CONCLUSIONS\u003C\u002Fjats:title>\n                  \u003Cjats:p>This report supports earlier predictions of the epidemic nature of diabetes in the world during the first quarter of the 21st century. It also provides a provisional picture of the characteristics of the epidemic. Worldwide surveillance of diabetes is a necessary first step toward its prevention and control, which is now recognized as an urgent priority.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>",{"EN":1371,"VI":1372},"Global Burden of Diabetes, 1995–2025: Prevalence, numerical estimates, and projections","Gánh nặng toàn cầu của bệnh tiểu đường, 1995–2025: Tỷ lệ mắc, ước tính số lượng và dự báo",{"VOID":1374},"9727886",{"VOID":1157},[31],[30],"https:\u002F\u002Fdiabetesjournals.org\u002Fcare\u002Farticle\u002F21\u002F9\u002F1414\u002F22253\u002FGlobal-Burden-of-Diabetes-1995-2025-Prevalence",[1380,1396,1413],{"id":1381,"sortIndex":32,"researcher":28,"roles":1382,"affiliations":1383,"properties":1392,"displayName":1072,"givenName":28,"familyName":28},"80291d0b-c02e-4cef-833d-26d5394f87ac",[],[1384],{"id":1385,"sortIndex":32,"affiliation":1386,"properties":28},"0a856902-30a2-4948-887e-dbcb5dcbd4e5",{"id":1385,"createTime":28,"updateTime":28,"relativeEntities":1387,"slug":28,"properties":1388,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1391,"statistic":28},[],{"title":1389},{"VI":1390},"Division of Noncommunicable Diseases, World Health Organization, Geneva, Switzerland",[],{"orcid":1393,"title":1394,"openalex":1395},{"VOID":1070},{"EN":1072},{"VOID":1074},{"id":1397,"sortIndex":40,"researcher":28,"roles":1398,"affiliations":1399,"properties":1408,"displayName":1410,"givenName":28,"familyName":28},"4ac9f8cd-b5c1-45fa-8c7d-04452bba8387",[],[1400],{"id":1401,"sortIndex":32,"affiliation":1402,"properties":28},"42251d3a-6f17-4ad6-8bd3-48dbd499719a",{"id":1401,"createTime":28,"updateTime":28,"relativeEntities":1403,"slug":28,"properties":1404,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1407,"statistic":28},[],{"title":1405},{"EN":1406},"Prudential Center for Health Care Research Atlanta, Georgia",[],{"title":1409,"openalex":1411},{"EN":1410},"Ronald E. Aubert",{"VOID":1412},"A5065552858",{"id":1414,"sortIndex":123,"researcher":28,"roles":1415,"affiliations":1416,"properties":1425,"displayName":1429,"givenName":28,"familyName":28},"e7ddac0e-941f-459c-b65a-f09c8403ab9a",[],[1417],{"id":1418,"sortIndex":32,"affiliation":1419,"properties":28},"c6f8bb29-e38c-40ff-89c1-84b0396755b4",{"id":1418,"createTime":28,"updateTime":28,"relativeEntities":1420,"slug":28,"properties":1421,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1424,"statistic":28},[],{"title":1422},{"EN":1423},"Division of Endocrinology and Metabolism, Department of Internal Medicine, University of Michigan Ann Arbor, Michigan",[],{"orcid":1426,"title":1428,"openalex":1430},{"VOID":1427},"https:\u002F\u002Forcid.org\u002F0000-0002-0502-674X",{"EN":1429},"William H. 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cảm insulin, OGTT, kẹp insulin euglycemic, tỷ lệ tiêu thụ glucose, tiểu đường type 2, chỉ số nhạy cảm insulin.",{"VOID":1606},"W2133795831",{"EN":1608,"VI":1609},"\u003Cjats:p>OBJECTIVE: Several methods have been proposed to evaluate insulin sensitivity from the data obtained from the oral glucose tolerance test (OGTT). However, the validity of these indices has not been rigorously evaluated by comparing them with the direct measurement of insulin sensitivity obtained with the euglycemic insulin clamp technique. In this study, we compare various insulin sensitivity indices derived from the OGTT with whole-body insulin sensitivity measured by the euglycemic insulin clamp technique. RESEARCH DESIGN AND METHODS: In this study, 153 subjects (66 men and 87 women, aged 18-71 years, BMI 20-65 kg\u002Fm2) with varying degrees of glucose tolerance (62 subjects with normal glucose tolerance, 31 subjects with impaired glucose tolerance, and 60 subjects with type 2 diabetes) were studied. After a 10-h overnight fast, all subjects underwent, in random order, a 75-g OGTT and a euglycemic insulin clamp, which was performed with the infusion of [3-3H]glucose. The indices of insulin sensitivity derived from OGTT data and the euglycemic insulin clamp were compared by correlation analysis. RESULTS: The mean plasma glucose concentration divided by the mean plasma insulin concentration during the OGTT displayed no correlation with the rate of whole-body glucose disposal during the euglycemic insulin clamp (r = -0.02, NS). From the OGTT, we developed an index of whole-body insulin sensitivity (10,000\u002Fsquare root of [fasting glucose x fasting insulin] x [mean glucose x mean insulin during OGTT]), which is highly correlated (r = 0.73, P < 0.0001) with the rate of whole-body glucose disposal during the euglycemic insulin clamp. CONCLUSIONS: Previous methods used to derive an index of insulin sensitivity from the OGTT have relied on the ratio of plasma glucose to insulin concentration during the OGTT. Our results demonstrate the limitations of such an approach. We have derived a novel estimate of insulin sensitivity that is simple to calculate and provides a reasonable approximation of whole-body insulin sensitivity from the OGTT.\u003C\u002Fjats:p>","\u003Cjats:p>MỤC ĐÍCH: Đã có nhiều phương pháp được đề xuất để đánh giá độ nhạy cảm insulin từ dữ liệu thu được từ thử nghiệm dung nạp glucose đường uống (OGTT). Tuy nhiên, tính hợp lệ của các chỉ số này chưa được đánh giá nghiêm ngặt bằng cách so sánh với đo lường trực tiếp độ nhạy cảm insulin được thu thập bằng kỹ thuật kẹp insulin euglycemic. Trong nghiên cứu này, chúng tôi so sánh các chỉ số nhạy cảm insulin khác nhau thu được từ OGTT với độ nhạy cảm insulin toàn cơ thể được đo bằng kỹ thuật kẹp insulin euglycemic. PHƯƠNG PHÁP NGHIÊN CỨU: Trong nghiên cứu này, 153 đối tượng (66 nam và 87 nữ, trong độ tuổi 18-71 tuổi, BMI từ 20-65 kg\u002Fm2) với các mức độ dung nạp glucose khác nhau (62 đối tượng có dung nạp glucose bình thường, 31 đối tượng bị suy giảm dung nạp glucose và 60 đối tượng mắc tiểu đường type 2) đã được nghiên cứu. Sau khi nhịn ăn suốt 10 giờ qua đêm, tất cả đối tượng được thực hiện, theo thứ tự ngẫu nhiên, một thử nghiệm OGTT 75 g và một kỹ thuật kẹp insulin euglycemic, được thực hiện với truyền dịch [3-3H]glucose. Các chỉ số độ nhạy cảm insulin thu được từ dữ liệu OGTT và kẹp insulin euglycemic được so sánh bằng phân tích tương quan. KẾT QUẢ: Nồng độ glucose huyết tương trung bình chia cho nồng độ insulin huyết tương trung bình trong OGTT không hiển thị tương quan với tỉ lệ tiêu thụ glucose toàn cơ thể trong kẹp insulin euglycemic (r = -0.02, NS). Từ OGTT, chúng tôi đã phát triển một chỉ số nhạy cảm insulin toàn cơ thể (10,000\u002Fcăn thức bậc hai của [glucose khi đói x insulin khi đói] x [glucose trung bình x insulin trung bình trong OGTT]), có tương quan cao (r = 0.73, P \u003C 0.0001) với tỉ lệ tiêu thụ glucose toàn cơ thể trong kẹp insulin euglycemic. KẾT LUẬN: Các phương pháp trước đây đã được sử dụng để tạo ra chỉ số nhạy cảm insulin từ OGTT dựa vào tỷ lệ nồng độ glucose huyết tương so với nồng độ insulin trong OGTT. Kết quả của chúng tôi chỉ ra hạn chế của phương pháp này. Chúng tôi đã phát triển một ước tính mới về độ nhạy cảm insulin, đơn giản để tính toán và cung cấp một phép xấp xỉ hợp lý cho độ nhạy cảm insulin toàn cơ thể từ OGTT.\u003C\u002Fjats:p>",{"EN":1611,"VI":1612},"Insulin sensitivity indices obtained from oral glucose tolerance testing: comparison with the euglycemic insulin clamp.","Các chỉ số nhạy cảm insulin từ thử nghiệm dung nạp glucose đường uống: so sánh với phương pháp kẹp insulin euglycemic.",{"VOID":1614},"10480510",{"VOID":1616},"10.2337\u002Fdiacare.22.9.1462",[31],[30],"https:\u002F\u002Fdiabetesjournals.org\u002Fcare\u002Farticle\u002F22\u002F9\u002F1462\u002F20552\u002FInsulin-sensitivity-indices-obtained-from-oral",[1621,1640],{"id":1622,"sortIndex":32,"researcher":28,"roles":1623,"affiliations":1624,"properties":1633,"displayName":1637,"givenName":28,"familyName":28},"4472b180-b27e-4fc5-a0f1-0d2f18cc1207",[],[1625],{"id":1626,"sortIndex":32,"affiliation":1627,"properties":28},"e75a42f3-397a-4936-b835-4aa772fe49ff",{"id":1626,"createTime":28,"updateTime":28,"relativeEntities":1628,"slug":28,"properties":1629,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1632,"statistic":28},[],{"title":1630},{"EN":1631},"Department of Medicine, University of Texas Health Science Center at San Antonio, 78284-7886, USA.",[],{"orcid":1634,"title":1636,"openalex":1638},{"VOID":1635},"https:\u002F\u002Forcid.org\u002F0000-0001-8543-1616",{"EN":1637},"Masafumi Matsuda",{"VOID":1639},"A5051524466",{"id":1641,"sortIndex":40,"researcher":28,"roles":1642,"affiliations":1643,"properties":1650,"displayName":1654,"givenName":28,"familyName":28},"9db649d0-8f35-4efa-8b68-9028e9eda779",[],[1644],{"id":1626,"sortIndex":32,"affiliation":1645,"properties":28},{"id":1626,"createTime":28,"updateTime":28,"relativeEntities":1646,"slug":28,"properties":1647,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1649,"statistic":28},[],{"title":1648},{"EN":1631},[],{"orcid":1651,"title":1653,"openalex":1655},{"VOID":1652},"https:\u002F\u002Forcid.org\u002F0000-0003-3839-1724",{"EN":1654},"Ralph A. 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Ở những người có bệnh tiểu đường loại 2 (n = 1.697), rối loạn glucose lúc đói (IFG)\u002Frối loạn dung nạp glucose (IGT) (n = 798), hoặc kháng insulin với dung nạp glucose bình thường (NGT) (n = 1.988), hội chứng chuyển hóa được định nghĩa là sự xuất hiện của ít nhất hai trong số các yếu tố nguy cơ sau: béo phì, huyết áp cao, rối loạn lipid máu hoặc vi albumin niệu. Tử vong do tim mạch đã được đánh giá ở 3.606 người với thời gian theo dõi trung vị là 6,9 năm.\u003C\u002Fjats:p>\n\u003Cjats:p>KẾT QUẢ—Ở nữ và nam giới, lần lượt, hội chứng chuyển hóa được nhìn thấy ở 10% và 15% người có NGT, 42% và 64% người có IFG\u002FIGT, và 78% và 84% người có bệnh tiểu đường loại 2. Nguy cơ mắc bệnh tim mạch và đột quỵ tăng gấp ba lần ở những người có hội chứng (P < 0,001). Tử vong do tim mạch tăng rõ rệt ở những người có hội chứng chuyển hóa (12,0 so với 2,2%, P < 0,001). Trong các thành phần riêng lẻ của hội chứng chuyển hóa, vi albumin niệu có nguy cơ tử vong do tim mạch mạnh nhất (RR 2,80; P = 0,002).\u003C\u002Fjats:p>\n\u003Cjats:p>KẾT LUẬN—Định nghĩa của WHO về hội chứng chuyển hóa xác định những người có tình trạng morbibilty và tử vong tim mạch cao và cung cấp một công cụ để so sánh kết quả từ các nghiên cứu khác nhau.\u003C\u002Fjats:p>","\u003Cjats:p>OBJECTIVE—To estimate the prevalence of and the cardiovascular risk associated with the metabolic syndrome using the new definition proposed by the World Health Organization (WHO).\u003C\u002Fjats:p>\n               \u003Cjats:p>RESEARCH DESIGN AND METHODS—A total of 4,483 subjects aged 35–70 years participating in a large family study of type 2 diabetes in Finland and Sweden (the Botnia study) were included in the analysis of cardiovascular risk associated with the metabolic syndrome. In subjects who had type 2 diabetes (n = 1,697), impaired fasting glucose (IFG)\u002Fimpaired glucose tolerance (IGT) (n = 798), or insulin-resistance with normal glucose tolerance (NGT) (n = 1,988), the metabolic syndrome was defined as presence of at least two of the following risk factors: obesity, hypertension, dyslipidemia, or microalbuminuria. Cardiovascular mortality was assessed in 3,606 subjects with a median follow-up of 6.9 years.\u003C\u002Fjats:p>\n               \u003Cjats:p>RESULTS—In women and men, respectively, the metabolic syndrome was seen in 10 and 15% of subjects with NGT, 42 and 64% of those with IFG\u002FIGT, and 78 and 84% of those with type 2 diabetes. The risk for coronary heart disease and stroke was increased threefold in subjects with the syndrome (P < 0.001). Cardiovascular mortality was markedly increased in subjects with the metabolic syndrome (12.0 vs. 2.2%, P < 0.001). Of the individual components of the metabolic syndrome, microalbuminuria conferred the strongest risk of cardiovascular death (RR 2.80; P = 0.002).\u003C\u002Fjats:p>\n               \u003Cjats:p>CONCLUSIONS—The WHO definition of the metabolic syndrome identifies subjects with increased cardiovascular morbidity and mortality and offers a tool for comparison of results from different studies.\u003C\u002Fjats:p>",{"EN":1743,"VI":1744},"Cardiovascular Morbidity and Mortality Associated With the Metabolic Syndrome","Morbidité và Tử Vong Tim Mạch Liên Quan Đến Hội Chứng Chuyển 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Diabetes 37:1595–1607, 1988",{"doi":1970},"10.2337\u002Fdiab.37.12.1595",{"id":28,"text":1972,"url":28,"identifiers":1973},"Kylin E: Studien ueber das Hypertonie-Hyperglykämie-Hyperurikämiesyndrom. Zentralblatt fuer Innere Medizin 44:105–127, 1923",{},{"id":28,"text":1975,"url":28,"identifiers":1976},"Björntorp P: Abdominal obesity and the metabolic syndrome (Review). Ann Med 24:465–468, 1994",{"doi":1977},"10.3109\u002F07853899209166997",{"id":28,"text":1979,"url":28,"identifiers":1980},"Groop L, Ekstrand A, Forsblom C, Widén E, Groop PH, Teppo AM, Eriksson J: Insulin resistance, hypertension and microalbuminuria in patients with type 2 (non-insulin-dependent) diabetes mellitus. Diabetologia 36:642–647, 1993",{"doi":1981},"10.1007\u002FBF00404074",{"id":28,"text":1983,"url":28,"identifiers":1984},"Mykkänen L, Zaccaro DJ, Wagenknecht LE, Robbins DJ, Gabriel M, Haffner SM: Microalbuminuria is associated with insulin resistance in nondiabetic subjects: the Insulin Resistance Atherosclerosis Study. Diabetes 47:793–800, 1998",{"doi":1985},"10.2337\u002Fdiabetes.47.5.793",{"id":28,"text":1987,"url":28,"identifiers":1988},"Yudkin JS: Abnormalities of coagulation and fibrinolysis in insulin resistance. Evidence for a common antecedent? (Review) Diabetes Care 22 (Suppl. 3):C25–C30, 1999",{},{"id":28,"text":1990,"url":28,"identifiers":1991},"Hanefeld M, Leonhardt W: Das Metabolische Syndrom. Dt Gesundh.-Wesen 36:545–551, 1981",{},{"id":28,"text":1993,"url":28,"identifiers":1994},"DeFronzo RA, Ferrannini E: Insulin resistance: a multifaceted syndrome responsible for NIDDM, obesity, hypertension, dyslipidemia, and atherosclerotic cardiovascular disease (Review). 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Arch Intern Med 149:1514–1520, 1989",{"doi":2005},"10.1001\u002Farchinte.149.7.1514",{"id":28,"text":2007,"url":28,"identifiers":2008},"Modan M, Halkin H, Almog S, Lusky A, Eshkol A, Shefi M, Shirit A, Fuchs Z: Hyperinsulinemia: a link between hypertension, obesity and glucose intolerance. J Clin Invest 75:807–817, 1985",{"doi":2009},"10.1172\u002FJCI111776",{"id":28,"text":2011,"url":28,"identifiers":2012},"Haffner S, Valdez R, Hazuda H, Mitchell B, Morales P, Stern M: Prospective analysis of the insulin-resistance syndrome (syndrome X). Diabetes 41:715–722, 1992",{"doi":2013},"10.2337\u002Fdiabetes.41.6.715",{"id":28,"text":2015,"url":28,"identifiers":2016},"Balkau B, Charles MA: Comments on the provisional report from the WHO consultation: European Group for the Study of Insulin Resistance (EGIR). Diabet Med 16:442–443, 1999",{"doi":2017},"10.1046\u002Fj.1464-5491.1999.00059.x",{"id":28,"text":2019,"url":28,"identifiers":2020},"Bonora E, Kiechl S, Willeit J, Oberhollenzer F, Egger G, Targher G, Alberiche M, Bonadonna RC, Muggeo M: Prevalence of insulin resistance in metabolic disorders: the Bruneck Study. Diabetes 47:1643–1649, 1998",{"doi":2021},"10.2337\u002Fdiabetes.47.10.1643",{"id":28,"text":2023,"url":28,"identifiers":2024},"Rantala AO, Kauma H, Lilja M, Savolainen MJ, Reunanen A, Kesäniemi YA: Prevalence of the metabolic syndrome in drug-treated hypertensive patients and control subjects. J Intern Med 245:163–174, 1999",{"doi":2025},"10.1046\u002Fj.1365-2796.1999.00429.x",{"id":28,"text":2027,"url":28,"identifiers":2028},"Alberti KGMM, Zimmet PZ, for the WHO Consultation: Definition, diagnosis and classification of diabetes mellitus and its complications. Part 1: Diagnosis and classification of diabetes mellitus, provisional report of a WHO consultation. Diabet Med 15:539–553, 1998",{"doi":2029},"10.1002\u002F(SICI)1096-9136(199807)15:7\u003C539::AID-DIA668>3.0.CO;2-S",{"id":28,"text":2031,"url":28,"identifiers":2032},"Groop L, Forsblom C, Lehtovirta M, Tuomi T, Karanko S, Nissén M, Ehrnström BO, Forsén B, Isomaa B, Snickars B, Taskinen MR: Metabolic consequences of a family history of NIDDM (the Botnia Study): evidence for sex-specific parental effects. Diabetes 45:1585–1593, 1996",{"doi":2033},"10.2337\u002Fdiabetes.45.11.1585",{"id":28,"text":2035,"url":28,"identifiers":2036},"Tuomi T, Carlsson Å-L, Li H, Cano L, Isomaa B, Miettinen A, Nissén M, Ehrnström B-O, Forsén B, Lahti K, Forsblom C, Saloranta C, Taskinen MR, Groop L: Clinical and genetic characteristics of type 2 diabetes with and without GAD antibodies. Diabetes 48:150–157, 1999",{"doi":2037},"10.2337\u002Fdiabetes.48.1.150",{"id":28,"text":2039,"url":28,"identifiers":2040},"Lehto M, Tuomi T, Mahtani M, Widén E, Forsblom C, Gullström M, Sarelin L, Isomaa B, Kanninen T, Lehtovirta M, Hyrkkö A, Orho M, Kirby A, Brettin T, Thomas J, Duyk G, Lander ES, Taskinen M-R and Groop L: Characterisation of the MODY3 phenotype, early-onset diabetes caused by an insulin secretion defect. J Clin Invest 99:582–591, 1997",{"doi":2041},"10.1172\u002FJCI119199",{"id":28,"text":2043,"url":28,"identifiers":2044},"Rose G, Blackburn H: Cardiovascular Survey Methods . Geneva, World Health Organization, 1968 (monogr. no. 56)",{},{"id":28,"text":2046,"url":28,"identifiers":2047},"Tunstall-Pedoe H, Kuulasmaa K, Amouyel P, Arveiler D, Rajakangas AM, Pajak A: Myocardial infarction and coronary deaths in the World Health Organization MONICA Project: registration procedures, event rates, and case-fatality rates in 38 populations from 21 countries in four continents. Circulation 90:583–612, 1994",{"doi":2048},"10.1161\u002F01.CIR.90.1.583",{"id":28,"text":2050,"url":28,"identifiers":2051},"Haffner SM, Miettinen H, Stern MP: The homeostasis model in the San Antonio Heart Study. Diabetes Care 20:1087–1092, 1997",{"doi":2052},"10.2337\u002Fdiacare.20.7.1087",{"id":28,"text":2054,"url":28,"identifiers":2055},"Friedewald WT, Levy RI, Fredrickson DS: Estimation of the concentration of low density lipoprotein cholesterol in plasma without use of preparative ultracentrifugation. Clin Chem 18:499–502, 1972",{"doi":2056},"10.1093\u002Fclinchem\u002F18.6.499",{"id":28,"text":2058,"url":28,"identifiers":2059},"Vanhala MJ, Kumpusalo EA, Pitkäjärvi TK, Takala JK: Metabolic syndrome in a middle-aged Finnish population. J Cardiovasc Risk 4:291–295, 1997",{"doi":2060},"10.1097\u002F00043798-199708000-00010",{"id":28,"text":2062,"url":28,"identifiers":2063},"Liese AD, Mayer-Davis EJ, Tyroler HA, Davis CE, Keil U, Schmidt MI, Brancati FL, Heiss G: Familial components of the multiple metabolic syndrome: the ARIC Study. Diabetologia 40:963–970, 1997",{"doi":2064},"10.1007\u002Fs001250050775",{"id":28,"text":2066,"url":28,"identifiers":2067},"Lehto S, Rönnemaa T, Pyörälä K, Laakso M: Cardiovascular risk factors clustering with endogenous hyperinsulinaemia predict death from coronary heart disease in patients with type II diabetes. Diabetologia 43:148–155, 2000",{"doi":2068},"10.1007\u002Fs001250050023",{"id":28,"text":2070,"url":28,"identifiers":2071},"Yki-Järvinen H: Glucose toxicity. Endocrinol Rev 13:415–431, 1992",{"doi":2072},"10.1210\u002Fer.13.3.415",{"id":28,"text":2074,"url":28,"identifiers":2075},"Randle PJ, Garland PB, Hales CN, Newsholme EA: The glucose fatty-acid cycle: its role in insulin sensitivity and the metabolic disturbances of diabetes mellitus. Lancet i:785–789, 1963",{"doi":2076},"10.1016\u002FS0140-6736(63)91500-9",{"id":28,"text":2078,"url":28,"identifiers":2079},"Boden G: Role of fatty acids in the pathogenesis of insulin resistance and NIDDM (Review). Diabetes 46:3–10, 1997",{"doi":2080},"10.2337\u002Fdiabetes.46.1.3",{"id":28,"text":2082,"url":28,"identifiers":2083},"Taskinen MR, Lahdenperä S, Syvänne M: New insights into lipid metabolism in non-insulin-dependent diabetes mellitus. Ann Med 28:335–340, 1996",{"doi":2084},"10.3109\u002F07853899608999090",{"id":28,"text":2086,"url":28,"identifiers":2087},"Hodge AM, Dowse GK, Zimmet PZ: Microalbuminuria, cardiovascular risk factors, and insulin resistance in two populations with a high risk of type 2 diabetes mellitus. Diabet Med 13:441–449, 1996",{"doi":2088},"10.1002\u002F(SICI)1096-9136(199605)13:5\u003C441::AID-DIA99>3.0.CO;2-J",{"id":28,"text":2090,"url":28,"identifiers":2091},"Zavaroni I, Bonini L, Gasparini P, Zuccarelli A, Dall’Aglio E, Barilli L, Cioni F, Strata A, Reaven GM: Dissociation between urinary albumin excretion and variables associated with insulin resistance in a healthy population. J Intern Med 240:151–156, 1996",{"doi":2092},"10.1046\u002Fj.1365-2796.1996.23849000.x",{"id":28,"text":2094,"url":28,"identifiers":2095},"Jensen JS, Borch-Johnsen K, Feldt-Rasmussen B, Appleyard M, Jensen G: Urinary albumin excretion and history of myocardial infarction in a cross-sectional study of 2,613 individuals. J Cardiovascular Risk 4:121–125, 1997",{"doi":2096},"10.1177\u002F174182679700400208",{"id":28,"text":2098,"url":28,"identifiers":2099},"Borch-Johnsen K, Feldt-Rasmussen B, Strandgaard S, Schroll M, Jensen JS: Urinary albumin excretion: an independent predictor of ischemic heart disease. Arterioscl Thromb Vasc Biol 19:1992–1997, 1999",{"doi":2100},"10.1161\u002F01.ATV.19.8.1992",{"id":28,"text":2102,"url":28,"identifiers":2103},"Kuusisto J, Mykkänen L, Pyörälä K, Laakso M: Hyperinsulinemic microalbuminuria: a new risk indicator for coronary heart disease. Circulation 91:831–837, 1995",{"doi":2104},"10.1161\u002F01.CIR.91.3.831",{"id":28,"text":2106,"url":28,"identifiers":2107},"Dinneen SF, Gerstein HC: The association of microalbuminuria and mortality in non-insulin-dependent diabetes mellitus: a systematic overview of the literature. Arch Intern Med 157:1413–1418, 1997",{"doi":2108},"10.1001\u002Farchinte.157.13.1413",{"id":28,"text":2110,"url":28,"identifiers":2111},"Jensen JS, Borch-Johnsen K, Jensen G, Feldt-Rasmussen B: Microalbuminuria reflects a generalized transvascular albumin leakiness in clinically healthy subjects. Clin Science 88:629–633, 1995",{"doi":2112},"10.1042\u002Fcs0880629",{"id":2114,"createTime":2115,"updateTime":2116,"relativeEntities":2117,"slug":2118,"properties":2119,"entityType":978,"verifyStatus":26,"verifyTime":2115,"verifyNote":979,"languages":2135,"translateLanguages":2136,"viewCount":32,"primaryUrl":2137,"fullTextUrl":28,"authors":2138,"publicationType":1075,"publisherRelationship":2190,"citationCount":2243,"citationInfo":2244,"publishDate":2257,"publishYear":1132,"citationAnalyzeStatus":884,"lastCitationAnalyze":28,"indexDatabases":2258,"openAccess":28,"references":2259,"isForceReanalyzing":1353},"03ec08f8-8bce-4da2-b2e0-9e9d5e6059ef","2024-10-01T02:16:10.268+00:00","2025-02-02T21:11:00.279+00:00",[],"Use-and-Abuse-of-HOMA-Modeling",{"mag":2120,"keywords":2122,"openalex":2123,"abstract":2125,"title":2128,"pm":2131,"doi":2133},{"VOID":2121},"2124620553",{"VI":1364},{"VOID":2124},"W2124620553",{"VI":2126,"EN":2127},"\u003Cjats:p>Đánh giá mô hình homeostatic (HOMA) là một phương pháp đánh giá chức năng tế bào β và kháng insulin (IR) dựa trên nồng độ glucose và insulin hoặc C-peptide trong trạng thái cơ bản (nhịn ăn). Nó đã được báo cáo trong hơn 500 ấn phẩm, gấp 20 lần so với việc ước lượng chức năng tế bào β.\u003C\u002Fjats:p>\n               \u003Cjats:p>Bài báo này tóm tắt cơ sở sinh lý của HOMA, một mô hình cấu trúc của trạng thái ổn định insulin và glucose, được xây dựng từ các phản ứng liều sinh lý của sự hấp thu glucose và sản xuất insulin. Sự thoát và hấp thu glucose của gan và ngoại vi được mô hình hóa để phụ thuộc vào nồng độ glucose và insulin trong huyết tương. Sự giảm chức năng tế bào β được mô hình hóa bằng cách thay đổi phản ứng của tế bào β đối với nồng độ glucose trong huyết tương. Mô hình HOMA nguyên bản được mô tả vào năm 1985 với một công thức để ước lượng gần đúng. Mô hình máy tính có sẵn nhưng chưa được sử dụng rộng rãi như các công thức gần đúng. HOMA đã được xác thực dựa trên nhiều phương pháp sinh lý khác nhau.\u003C\u002Fjats:p>\n               \u003Cjats:p>Chúng tôi xem xét việc sử dụng và báo cáo HOMA trong tài liệu và cung cấp hướng dẫn về cách sử dụng thích hợp của nó (ví dụ: trong các nghiên cứu đoàn hệ và dịch tễ học) và sử dụng không thích hợp (ví dụ: đo chức năng tế bào β một cách tách biệt). Mô hình HOMA so sánh một cách thuận lợi với các mô hình khác và có ưu điểm là chỉ yêu cầu một mẫu huyết tương duy nhất được phân tích cho insulin và glucose.\u003C\u002Fjats:p>\n               \u003Cjats:p>Tóm lại, mô hình HOMA đã trở thành một công cụ lâm sàng và dịch tễ học được sử dụng rộng rãi và, khi được sử dụng thích hợp, nó có thể mang lại dữ liệu giá trị. Tuy nhiên, giống như tất cả các mô hình khác, dữ liệu đầu vào chính cần phải đáng tin cậy và cần phải diễn giải kỹ lưỡng.\u003C\u002Fjats:p>","\u003Cjats:p>Homeostatic model assessment (HOMA) is a method for assessing β-cell function and insulin resistance (IR) from basal (fasting) glucose and insulin or C-peptide concentrations. It has been reported in >500 publications, 20 times more frequently for the estimation of IR than β-cell function.\u003C\u002Fjats:p>\n               \u003Cjats:p>This article summarizes the physiological basis of HOMA, a structural model of steady-state insulin and glucose domains, constructed from physiological dose responses of glucose uptake and insulin production. Hepatic and peripheral glucose efflux and uptake were modeled to be dependent on plasma glucose and insulin concentrations. Decreases in β-cell function were modeled by changing the β-cell response to plasma glucose concentrations. The original HOMA model was described in 1985 with a formula for approximate estimation. The computer model is available but has not been as widely used as the approximation formulae. HOMA has been validated against a variety of physiological methods.\u003C\u002Fjats:p>\n               \u003Cjats:p>We review the use and reporting of HOMA in the literature and give guidance on its appropriate use (e.g., cohort and epidemiological studies) and inappropriate use (e.g., measuring β-cell function in isolation). The HOMA model compares favorably with other models and has the advantage of requiring only a single plasma sample assayed for insulin and glucose.\u003C\u002Fjats:p>\n               \u003Cjats:p>In conclusion, the HOMA model has become a widely used clinical and epidemiological tool and, when used appropriately, it can yield valuable data. However, as with all models, the primary input data need to be robust, and the data need to be interpreted carefully.\u003C\u002Fjats:p>",{"EN":2129,"VI":2130},"Use and Abuse of HOMA Modeling","Sử Dụng và Lạm Dụng Mô Hình HOMA",{"VOID":2132},"15161807",{"VOID":2134},"10.2337\u002Fdiacare.27.6.1487",[31],[30],"https:\u002F\u002Fdiabetesjournals.org\u002Fcare\u002Farticle\u002F27\u002F6\u002F1487\u002F22836\u002FUse-and-Abuse-of-HOMA-Modeling",[2139,2156,2173],{"id":2140,"sortIndex":32,"researcher":28,"roles":2141,"affiliations":2142,"properties":2151,"displayName":2153,"givenName":28,"familyName":28},"e847c27e-c8cb-4676-a0cc-5f1ce92461d8",[],[2143],{"id":2144,"sortIndex":32,"affiliation":2145,"properties":28},"f65757ce-6eba-4570-99ca-c122a52b632b",{"id":2144,"createTime":28,"updateTime":28,"relativeEntities":2146,"slug":28,"properties":2147,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2150,"statistic":28},[],{"title":2148},{"EN":2149},"From the Oxford Centre for Diabetes, Endocrinology and Metabolism, The Churchill Hospital, Oxford, U.K.",[],{"title":2152,"openalex":2154},{"EN":2153},"T. 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10.2337\u002Fdiacare.24.3.561",{"doi":3326},"10.2337\u002Fdiacare.24.3.561",{"id":28,"text":3328,"url":28,"identifiers":3329},"Marrero, 2013, Twenty-first century behavioral medicine: a context for empowering clinicians and patients with diabetes: a consensus report, Diabetes Care, 36, 463, 10.2337\u002Fdc12-2305",{"doi":3330},"10.2337\u002Fdc12-2305",{"id":28,"text":3332,"url":28,"identifiers":3333},"Norris, 2002, Self-management education for adults with type 2 diabetes: a meta-analysis of the effect on glycemic control, Diabetes Care, 25, 1159, 10.2337\u002Fdiacare.25.7.1159",{"doi":3334},"10.2337\u002Fdiacare.25.7.1159",{"id":28,"text":3336,"url":28,"identifiers":3337},"Martin, 2012, Recommendations for age-appropriate education of children and adolescents with diabetes and their parents in the European Union, Pediatr Diabetes, 13, 20, 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10.2337\u002Fdiacare.24.6.1069",{"doi":3551},"10.2337\u002Fdiacare.24.6.1069",{"id":28,"text":3553,"url":28,"identifiers":3554},"Kovacs Burns, 2013, Diabetes Attitudes, Wishes and Needs second study (DAWN2™): cross-national benchmarking indicators for family members living with people with diabetes, Diabet Med, 30, 778, 10.1111\u002Fdme.12239",{"doi":3555},"10.1111\u002Fdme.12239",{"id":28,"text":3557,"url":28,"identifiers":3558},"Harkness, 2010, Identifying psychosocial interventions that improve both physical and mental health in patients with diabetes: a systematic review and meta-analysis, Diabetes Care, 33, 926, 10.2337\u002Fdc09-1519",{"doi":3559},"10.2337\u002Fdc09-1519",{"id":28,"text":3561,"url":28,"identifiers":3562},"Bot, 2012, Association of coexisting diabetes and depression with mortality after myocardial infarction, Diabetes Care, 35, 503, 10.2337\u002Fdc11-1749",{"doi":3563},"10.2337\u002Fdc11-1749",{"id":28,"text":3565,"url":28,"identifiers":3566},"Scherrer, 2011, 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intensification: patient views compared with current treatment guidelines, Diabetes Educ, 37, 78, 10.1177\u002F0145721710388427",{"doi":4855},"10.1177\u002F0145721710388427",{"id":28,"text":4857,"url":28,"identifiers":4858},"Schillinger, 2003, Closing the loop: physician communication with diabetic patients who have low health literacy, Arch Intern Med, 163, 83, 10.1001\u002Farchinte.163.1.83",{"doi":4859},"10.1001\u002Farchinte.163.1.83",{"id":28,"text":4861,"url":28,"identifiers":4862},"Rosal, 2011, Randomized trial of a literacy-sensitive, culturally tailored diabetes self-management intervention for low-income Latinos: Latinos en Control, Diabetes Care, 34, 838, 10.2337\u002Fdc10-1981",{"doi":4863},"10.2337\u002Fdc10-1981",{"id":28,"text":4865,"url":28,"identifiers":4866},"Osborn, 2011, Health literacy explains racial disparities in diabetes medication adherence, J Health Commun, 16, 268, 10.1080\u002F10810730.2011.604388",{"doi":4867},"10.1080\u002F10810730.2011.604388",{"id":28,"text":4869,"url":28,"identifiers":4870},"Rothman, 2004, The relationship between literacy and glycemic control in a diabetes disease-management program, Diabetes Educ, 30, 263, 10.1177\u002F014572170403000219",{"doi":4871},"10.1177\u002F014572170403000219",{"id":28,"text":4873,"url":28,"identifiers":4874},"O’Connor, 2011, Impact of electronic health record clinical decision support on diabetes care: a randomized trial, Ann Fam Med, 9, 12, 10.1370\u002Fafm.1196",{"doi":4875},"10.1370\u002Fafm.1196",{"id":28,"text":4877,"url":28,"identifiers":4878},"Garg, 2005, Effects of computerized clinical decision support systems on practitioner performance and patient outcomes: a systematic review, JAMA, 293, 1223, 10.1001\u002Fjama.293.10.1223",{"doi":4879},"10.1001\u002Fjama.293.10.1223",{"id":28,"text":4881,"url":28,"identifiers":4882},"Smith, 2008, Chronic care model and shared care in diabetes: randomized trial of an electronic decision support system, Mayo Clin Proc, 83, 747, 10.4065\u002F83.7.747",{"doi":4883},"10.4065\u002F83.7.747",{"id":28,"text":4885,"url":28,"identifiers":4886},"Jaffe, 2013, Improved blood pressure control associated with a large-scale hypertension program, JAMA, 310, 699, 10.1001\u002Fjama.2013.108769",{"doi":4887},"10.1001\u002Fjama.2013.108769",{"id":28,"text":4889,"url":28,"identifiers":4890},"Davidson, 2007, Effect of a nurse-directed diabetes disease management program on urgent care\u002Femergency room visits and hospitalizations in a minority population, Diabetes Care, 30, 224, 10.2337\u002Fdc06-2022",{"doi":4891},"10.2337\u002Fdc06-2022",{"id":28,"text":4893,"url":28,"identifiers":4894},"Stone, 2010, Active care management supported by home telemonitoring in veterans with type 2 diabetes: the DiaTel randomized controlled trial, Diabetes Care, 33, 478, 10.2337\u002Fdc09-1012",{"doi":4895},"10.2337\u002Fdc09-1012",{"id":28,"text":4897,"url":28,"identifiers":4898},"Berikai, 2007, Gain in patients’ knowledge of diabetes management targets is associated with better glycemic control, Diabetes Care, 30, 1587, 10.2337\u002Fdc06-2026",{"doi":4899},"10.2337\u002Fdc06-2026",{"id":28,"text":4901,"url":28,"identifiers":4902},"Funnell, 2007, National Standards for Diabetes Self-Management Education, Diabetes Care, 30, 1630, 10.2337\u002Fdc07-9923",{"doi":4903},"10.2337\u002Fdc07-9923",{"id":28,"text":4905,"url":28,"identifiers":4906},"Klein, 2004, Weight management through lifestyle modification for the prevention and management of type 2 diabetes: rationale and strategies: a statement of the American Diabetes Association, the North American Association for the Study of Obesity, and the American Society for Clinical Nutrition, Diabetes Care, 27, 2067, 10.2337\u002Fdiacare.27.8.2067",{"doi":4907},"10.2337\u002Fdiacare.27.8.2067",{"id":28,"text":4909,"url":28,"identifiers":4910},"Norris, 2004, Efficacy of pharmacotherapy for weight loss in adults with type 2 diabetes mellitus: a meta-analysis, Arch Intern Med, 164, 1395, 10.1001\u002Farchinte.164.13.1395",{"doi":4911},"10.1001\u002Farchinte.164.13.1395",{"id":28,"text":4913,"url":28,"identifiers":4914},"Tricco, 2012, Effectiveness of quality improvement strategies on the management of diabetes: a systematic review and meta-analysis, Lancet, 379, 2252, 10.1016\u002FS0140-6736(12)60480-2",{"doi":4915},"10.1016\u002FS0140-6736(12)60480-2",{"id":28,"text":4917,"url":28,"identifiers":4918},"O’Connor, 2011, Diabetes performance measures: current status and future directions, Diabetes Care, 34, 1651, 10.2337\u002Fdc11-0735",{"doi":4919},"10.2337\u002Fdc11-0735",{"id":28,"text":4921,"url":28,"identifiers":4922},"Peikes, 2009, Effects of care coordination on hospitalization, quality of care, and health care expenditures among Medicare beneficiaries: 15 randomized trials, JAMA, 301, 603, 10.1001\u002Fjama.2009.126",{"doi":4923},"10.1001\u002Fjama.2009.126",{"id":28,"text":4925,"url":28,"identifiers":4926},"Feifer, 2007, Different paths to high-quality care: three archetypes of top-performing practice sites, Ann Fam Med, 5, 233, 10.1370\u002Fafm.697",{"doi":4927},"10.1370\u002Fafm.697",{"id":28,"text":4929,"url":28,"identifiers":4930},"Reed, 2012, Outpatient electronic health records and the clinical care and outcomes of patients with diabetes mellitus, Ann Intern Med, 157, 482, 10.7326\u002F0003-4819-157-7-201210020-00004",{"doi":4931},"10.7326\u002F0003-4819-157-7-201210020-00004",{"id":28,"text":4933,"url":28,"identifiers":4934},"Cebul, 2011, Electronic health records and quality of diabetes care, N Engl J Med, 365, 825, 10.1056\u002FNEJMsa1102519",{"doi":4935},"10.1056\u002FNEJMsa1102519",{"id":28,"text":4937,"url":28,"identifiers":4938},"Battersby, 2010, Twelve 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ACO rules—striking the balance between participation and transformative potential, N Engl J Med, 365, e6, 10.1056\u002FNEJMp1106012",{"doi":4954},"10.1056\u002FNEJMp1106012",{"id":28,"text":4956,"url":28,"identifiers":4957},"Washington, 2011, The Patient-Centered Outcomes Research Institute—promoting better information, decisions, and health, N Engl J Med, 365, e31, 10.1056\u002FNEJMp1109407",{"doi":4958},"10.1056\u002FNEJMp1109407",{"id":4960,"createTime":4961,"updateTime":4962,"relativeEntities":4963,"slug":4964,"properties":4965,"entityType":978,"verifyStatus":26,"verifyTime":4980,"verifyNote":979,"languages":4981,"translateLanguages":4982,"viewCount":32,"primaryUrl":4983,"fullTextUrl":28,"authors":4984,"publicationType":1075,"publisherRelationship":5277,"citationCount":5329,"citationInfo":5330,"publishDate":5335,"publishYear":1581,"citationAnalyzeStatus":884,"lastCitationAnalyze":28,"indexDatabases":5336,"openAccess":28,"references":5337,"isForceReanalyzing":1353},"bccc4fd8-ff60-4cf6-a935-9f4a7d317d1c","2024-09-26T10:43:39.310+00:00","2025-02-02T21:12:02.508+00:00",[],"Effects-of-Diet-and-Exercise-in-Preventing-NIDDM-in-People-With-Impaired-Glucose-Tolerance-The-Da-Qing-IGT-and-Diabetes-Study",{"mag":4966,"keywords":4968,"openalex":4969,"abstract":4971,"title":4974,"pm":4977,"doi":4979},{"VOID":4967},"2146536851",{"VI":1364},{"VOID":4970},"W2146536851",{"VI":4972,"EN":4973},"\u003Cjats:sec>\n                  \u003Cjats:title>MỤC TIÊU\u003C\u002Fjats:title>\n                  \u003Cjats:p>Các cá nhân có rối loạn dung nạp glucose (IGT) có nguy cơ cao phát triển thành NIDDM. Mục đích của nghiên cứu này là xác định liệu các can thiệp về chế độ ăn uống và tập thể dục ở những người mắc IGT có thể làm chậm sự phát triển của NIDDM, tức là giảm tỷ lệ mắc NIDDM, và do đó giảm tỷ lệ mắc các biến chứng tiểu đường nói chung, chẳng hạn như bệnh tim mạch, thận và đáy mắt, cũng như tỷ lệ tử vong gia tăng liên quan đến các biến chứng này hay không.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>\n               \u003Cjats:sec>\n                  \u003Cjats:title>THIẾT KẾ NGHIÊN CỨU VÀ PHƯƠNG PHÁP\u003C\u002Fjats:title>\n                  \u003Cjats:p>Vào năm 1986, 110.660 nam nữ từ 33 phòng khám y tế ở thành phố Đại Khánh, Trung Quốc, đã được sàng lọc rối loạn dung nạp glucose (IGT) và NIDDM. Trong số những cá nhân đó, 577 người được phân loại (theo tiêu chí của Tổ chức Y tế Thế giới) là có IGT. Các đối tượng được phân ngẫu nhiên theo phòng khám vào một thử nghiệm lâm sàng, hoặc vào nhóm đối chứng hoặc vào một trong ba nhóm điều trị tích cực: chỉ chế độ ăn uống, chỉ tập thể dục, hoặc chế độ ăn cộng với tập thể dục. Các cuộc khảo sát đánh giá theo dõi đã được tiến hành trong các khoảng thời gian 2 năm trong suốt 6 năm để xác định các đối tượng nào phát triển thành NIDDM. Phân tích rủi ro tỷ lệ Cox đã được sử dụng để xác định xem tỷ lệ mắc NIDDM có khác nhau tùy theo sự phân công điều trị hay không.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>\n               \u003Cjats:sec>\n                  \u003Cjats:title>KẾT QUẢ\u003C\u002Fjats:title>\n                  \u003Cjats:p>Tỷ lệ tích lũy mắc bệnh tiểu đường sau 6 năm là 67,7% (Khoảng tin cậy 95%, 59,8–75,2) trong nhóm đối chứng so với 43,8% (Khoảng tin cậy 95%, 35,5–52,3) trong nhóm chế độ ăn, 41,1% (Khoảng tin cậy 95%, 33,4–49,4) trong nhóm tập thể dục và 46,0% (Khoảng tin cậy 95%, 37,3–54,7) trong nhóm chế độ ăn cộng với tập thể dục (P \u003C 0,05). Khi phân tích theo phòng khám, mỗi nhóm can thiệp tích cực đều khác biệt đáng kể so với các phòng khám đối chứng (P \u003C 0,05). Sự giảm tương đối trong tỷ lệ phát triển của bệnh tiểu đường ở các nhóm điều trị tích cực là tương tự khi các đối tượng được phân loại theo gầy hoặc thừa cân (BMI \u003C hoặc ≥ 25 kg\u002Fm2). Trong một phân tích rủi ro tỷ lệ điều chỉnh cho sự khác biệt về BMI cơ bản và glucose lúc đói, các can thiệp chế độ ăn, tập thể dục và chế độ ăn cộng với tập thể dục tương ứng liên quan đến việc giảm 31% (P \u003C 0,03), 46% (P \u003C 0,0005), và 42% (P \u003C 0,005) nguy cơ phát triển bệnh tiểu đường.","\u003Cjats:sec>\n                  \u003Cjats:title>OBJECTIVE\u003C\u002Fjats:title>\n                  \u003Cjats:p>Individuals with impaired glucose tolerance (IGT) have a high risk of developing NIDDM. The purpose of this study was to determine whether diet and exercise interventions in those with IGT may delay the development of NIDDM, i.e., reduce the incidence of NIDDM, and thereby reduce the overall incidence of diabetic complications, such as cardiovascular, renal, and retinal disease, and the excess mortality attributable to these complications.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>\n               \u003Cjats:sec>\n                  \u003Cjats:title>RESEARCH DESIGN AND METHODS\u003C\u002Fjats:title>\n                  \u003Cjats:p>In 1986, 110,660 men and women from 33 health care clinics in the city of Da Qing, China, were screened for IGT and NIDDM. Of these individuals, 577 were classified (using World Health Organization criteria) as having IGT. Subjects were randomized by clinic into a clinical trial, either to a control group or to one of three active treatment groups: diet only, exercise only, or diet plus exercise. Follow-up evaluation examinations were conducted at 2-year intervals over a 6-year period to identify subjects who developed NIDDM. Cox's proportional hazard analysis was used to determine if the incidence of NIDDM varied by treatment assignment.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>\n               \u003Cjats:sec>\n                  \u003Cjats:title>RESULTS\u003C\u002Fjats:title>\n                  \u003Cjats:p>The cumulative incidence of diabetes at 6 years was 67.7% (95% CI, 59.8–75.2) in the control group compared with 43.8% (95% CI, 35.5–52.3) in the diet group, 41.1% (95% CI, 33.4–49.4) in the exercise group, and 46.0% (95% CI, 37.3–54.7) in the diet-plus-exercise group (P < 0.05). When analyzed by clinic, each of the active intervention groups differed significantly from the control clinics (P < 0.05). The relative decrease in rate of development of diabetes in the active treatment groups was similar when subjects were stratified as lean or overweight (BMI < or ≥ 25 kg\u002Fm2). In a proportional hazards analysis adjusted for differences in baseline BMI and fasting glucose, the diet, exercise, and diet-plus-exercise interventions were associated with 31% (P < 0.03), 46% (P < 0.0005), and 42% (P < 0.005) reductions in risk of developing diabetes, respectively.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>\n               \u003Cjats:sec>\n                  \u003Cjats:title>CONCLUSIONS\u003C\u002Fjats:title>\n                  \u003Cjats:p>Diet and\u002For exercise interventions led to a significant decrease in the incidence of diabetes over a 6-year period among those with IGT.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>",{"EN":4975,"VI":4976},"Effects of Diet and Exercise in Preventing NIDDM in People With Impaired Glucose Tolerance: The Da Qing IGT and Diabetes Study","Tác động của chế độ ăn uống và tập thể dục trong việc ngăn ngừa NIDDM ở những người mắc rối loạn dung nạp glucose: Nghiên cứu Da Qing về IGT và bệnh tiểu 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Tổng cộng có 347,978 nam giới trong độ tuổi từ 35 đến 57, được sàng lọc tại 20 trung tâm cho thử nghiệm MRFIT. Biện pháp kết quả là tỷ lệ tử vong do CVD.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>\n               \u003Cjats:sec>\n                  \u003Cjats:title>KẾT QUẢ\u003C\u002Fjats:title>\n                  \u003Cjats:p>Trong số 5163 nam giới báo cáo đã sử dụng thuốc điều trị bệnh tiểu đường, đã có 1092 ca tử vong (603 ca tử vong do CVD) xảy ra trong thời gian theo dõi trung bình 12 năm. Trong số 342,815 nam giới không sử dụng thuốc tiểu đường, đã xác định được 20,867 ca tử vong, trong đó có 8965 trường hợp được xác định là tử vong do CVD. Nguy cơ tuyệt đối của tử vong do CVD cao hơn nhiều đối với nam giới tiểu đường so với nam giới không tiểu đường cho mọi nhóm tuổi, nguồn gốc dân tộc, và mức độ nguy cơ – tổng thể cao gấp ba lần, sau khi điều chỉnh theo độ tuổi, chủng tộc, thu nhập, mức cholesterol huyết thanh, huyết áp tâm thu (sBP) và số lượng thuốc lá đã báo cáo mỗi ngày (P \u003C 0.0001). Đối với cả nam giới có và không có bệnh tiểu đường, mức cholesterol huyết thanh, sBP và thói quen hút thuốc lá là các yếu tố tiên đoán đáng kể tỷ lệ tử vong do CVD. Đối với nam giới tiểu đường có giá trị cao hơn cho từng yếu tố nguy cơ và sự kết hợp của chúng, nguy cơ tuyệt đối tử vong do CVD tăng nhanh hơn so với nam giới không tiểu đường, khiến cho nguy cơ thừa tuyệt đối cho nam giới tiểu đường ngày càng lớn hơn so với nam giới không tiểu đường có mức độ yếu tố nguy cơ cao hơn.","\u003Cjats:sec>\n                  \u003Cjats:title>OBJECTIVE\u003C\u002Fjats:title>\n                  \u003Cjats:p>To assess predictors of CVD mortality among men with and without diabetes and to assess the independent effect of diabetes on the risk of CVD death.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>\n               \u003Cjats:sec>\n                  \u003Cjats:title>RESEARCH DESIGN AND METHODS\u003C\u002Fjats:title>\n                  \u003Cjats:p>Participants in this cohort study were screened from 1973 to 1975; vital status has been ascertained over an average of 12 yr of follow-up (range 11–13 yr). Participants were 347,978 men aged 35–57 yr, screened in 20 centers for MRFIT. The outcome measure was CVD mortality.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>\n               \u003Cjats:sec>\n                  \u003Cjats:title>RESULTS\u003C\u002Fjats:title>\n                  \u003Cjats:p>Among 5163 men who reported taking medication for diabetes, 1092 deaths (603 CVD deaths) occurred in an average of 12 yr of follow-up. Among 342,815 men not taking medication for diabetes, 20,867 deaths were identified, 8965 ascribed to CVD. Absolute risk of CVD death was much higher for diabetic than nondiabetic men of every age stratum, ethnic background, and risk factor level—overall three times higher, with adjustment for age, race, income, serum cholesterol level, sBP, and reported number of cigarettes\u002Fday (P < 0.0001). For men both with and without diabetes, serum cholesterol level, sBP, and cigarette smoking were significant predictors of CVD mortality. For diabetic men with higher values for each risk factor and their combinations, absolute risk of CVD death increased more steeply than for nondiabetic men, so that absolute excess risk for diabetic men was progressively greater than for nondiabetic men with higher risk factor levels.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>\n               \u003Cjats:sec>\n                  \u003Cjats:title>CONCLUSIONS\u003C\u002Fjats:title>\n                  \u003Cjats:p>These findings emphasize the importance of rigorous sustained intervention in people with diabetes to control blood pressure, lower serum cholesterol, and abolish cigarette smoking, and the importance of considering nutritional-hygienic approaches on a mass scale to prevent diabetes.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>",{"EN":5355,"VI":5356},"Diabetes, Other Risk Factors, and 12-Yr Cardiovascular Mortality for Men Screened in the Multiple Risk Factor Intervention Trial","Đái tháo đường, Các yếu tố nguy cơ khác và Tỷ lệ tử vong do bệnh tim mạch trong 12 năm ở nam giới được sàng lọc trong Thử nghiệm Can thiệp Các yếu tố Nguy cơ Đa chiều",{"VOID":5358},"8432214",{"VOID":3777},[31],[30],"https:\u002F\u002Fdiabetesjournals.org\u002Fcare\u002Farticle\u002F16\u002F2\u002F434\u002F17434\u002FDiabetes-Other-Risk-Factors-and-12-Yr",[5364,5383,5400,5417],{"id":5365,"sortIndex":32,"researcher":28,"roles":5366,"affiliations":5367,"properties":5376,"displayName":5380,"givenName":28,"familyName":28},"834967df-c32f-40c2-a0f6-e97ba4d099a3",[],[5368],{"id":5369,"sortIndex":32,"affiliation":5370,"properties":28},"d5a2e490-52c7-464c-b2e5-d68862cb83f8",{"id":5369,"createTime":28,"updateTime":28,"relativeEntities":5371,"slug":28,"properties":5372,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":5375,"statistic":28},[],{"title":5373},{"EN":5374},"Department of Preventive Medicine, Northwestern University Medical School Chicago, Illinois Department of Internal Medicine and Metabolic Diseases, Second Faculty of Medicine, University of Naples Naples, Italy Division Of Biostatistics, School of Public Health, University of Minnesota Minneapolis, Minnesota National Heart, Lung, and Blood Institute Bethesda, Maryland",[],{"orcid":5377,"title":5379,"openalex":5381},{"VOID":5378},"https:\u002F\u002Forcid.org\u002F0000-0002-8557-7225",{"EN":5380},"Jeremiah Stamler",{"VOID":5382},"A5075793582",{"id":5384,"sortIndex":40,"researcher":28,"roles":5385,"affiliations":5386,"properties":5393,"displayName":5397,"givenName":28,"familyName":28},"187fd208-8622-4af1-848c-51ff9740394b",[],[5387],{"id":5369,"sortIndex":32,"affiliation":5388,"properties":28},{"id":5369,"createTime":28,"updateTime":28,"relativeEntities":5389,"slug":28,"properties":5390,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":5392,"statistic":28},[],{"title":5391},{"EN":5374},[],{"orcid":5394,"title":5396,"openalex":5398},{"VOID":5395},"https:\u002F\u002Forcid.org\u002F0000-0002-1083-1428",{"EN":5397},"Olga Vaccaro",{"VOID":5399},"A5001056979",{"id":5401,"sortIndex":123,"researcher":28,"roles":5402,"affiliations":5403,"properties":5410,"displayName":5414,"givenName":28,"familyName":28},"51d8fae5-16c1-4363-b876-aa0e34f8671a",[],[5404],{"id":5369,"sortIndex":32,"affiliation":5405,"properties":28},{"id":5369,"createTime":28,"updateTime":28,"relativeEntities":5406,"slug":28,"properties":5407,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":5409,"statistic":28},[],{"title":5408},{"EN":5374},[],{"orcid":5411,"title":5413,"openalex":5415},{"VOID":5412},"https:\u002F\u002Forcid.org\u002F0000-0001-9986-069X",{"EN":5414},"James D. Neaton",{"VOID":5416},"A5001303096",{"id":5418,"sortIndex":42,"researcher":28,"roles":5419,"affiliations":5420,"properties":5427,"displayName":5431,"givenName":28,"familyName":28},"a8cd4efc-19c1-40ca-9ec0-d5d5d011f75e",[],[5421],{"id":5369,"sortIndex":32,"affiliation":5422,"properties":28},{"id":5369,"createTime":28,"updateTime":28,"relativeEntities":5423,"slug":28,"properties":5424,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":5426,"statistic":28},[],{"title":5425},{"EN":5374},[],{"orcid":5428,"title":5430,"openalex":5432},{"VOID":5429},"https:\u002F\u002Forcid.org\u002F0000-0001-5536-0335",{"EN":5431},"Deborah Wentworth",{"VOID":5433},"A5073137557",{"url":28,"publisher":5435,"properties":5481},{"id":868,"createTime":869,"updateTime":870,"relativeEntities":5436,"slug":872,"properties":5437,"entityType":25,"verifyStatus":884,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":32,"subjectFields":5442,"manageAffiliations":5455,"indexDatabases":5466,"url":950,"thumbnailPath":28,"statistic":28,"gsStatistic":28,"type":28,"analyzePriority":28},[],{"country":5438,"eissn":5439,"issn":5440,"title":5441},{"VOID":875},{"VOID":877},{"VOID":879},{"EN":881},[5443,5447,5451],{"id":887,"createTime":28,"updateTime":28,"relativeEntities":5444,"label":5445,"description":5446,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":890},{},{"id":893,"createTime":28,"updateTime":28,"relativeEntities":5448,"label":5449,"description":5450,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":896},{},{"id":899,"createTime":28,"updateTime":28,"relativeEntities":5452,"label":5453,"description":5454,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":902},{},[5456,5461],{"id":906,"createTime":28,"updateTime":28,"relativeEntities":5457,"slug":28,"properties":5458,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":5460,"statistic":28},[],{"title":5459},{"EN":910},[],{"id":913,"createTime":28,"updateTime":28,"relativeEntities":5462,"slug":28,"properties":5463,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":5465,"statistic":28},[],{"title":5464},{"EN":917},[],[5467,5474],{"id":921,"indexDatabase":5468,"url":927,"indexYears":928,"academicFieldIds":5473,"indexDatabaseRanking":933},{"id":775,"createTime":28,"updateTime":28,"relativeEntities":5469,"label":5470,"description":5471,"key":781,"publicationTags":5472,"standard":28},[],{"EN":778,"VI":778},{"EN":778,"VI":780},[783],[930,931,932],{"id":935,"indexDatabase":5475,"url":947,"indexYears":28,"academicFieldIds":5480,"indexDatabaseRanking":28},{"id":937,"createTime":28,"updateTime":28,"relativeEntities":5476,"label":5477,"description":5478,"key":944,"publicationTags":5479,"standard":28},[],{"EN":940,"VI":940},{"EN":942,"VI":943},[946,813],[949],{"issue":5482,"pages":5484,"volume":5486},{"VOID":5483},"2",{"VOID":5485},"434-444",{"VOID":5487},"16",4080,{"total":5488,"publishYear":5490,"statisticByYear":5491},1993,{"2012":1586,"2013":5492,"2014":684,"2015":529,"2016":334,"2017":1496,"2018":1496,"2019":522,"2020":155,"2021":826,"2022":687,"2023":567,"2024":136},155,"1993-02-01",[946,933],[],{"id":5497,"createTime":5498,"updateTime":5499,"relativeEntities":5500,"slug":5501,"properties":5502,"entityType":978,"verifyStatus":26,"verifyTime":5498,"verifyNote":979,"languages":5521,"translateLanguages":5522,"viewCount":32,"primaryUrl":5523,"fullTextUrl":28,"authors":5524,"publicationType":1075,"publisherRelationship":6203,"citationCount":6257,"citationInfo":6258,"publishDate":6269,"publishYear":6259,"citationAnalyzeStatus":884,"lastCitationAnalyze":28,"indexDatabases":6270,"openAccess":28,"references":6271,"isForceReanalyzing":1353},"e1d52ada-278e-40d4-b79a-4fec3c074abb","2024-09-01T10:20:55.233+00:00","2025-02-02T21:14:00.717+00:00",[],"Global-Prevalence-and-Major-Risk-Factors-of-Diabetic-Retinopathy",{"mag":5503,"keywords":5505,"pmc":5507,"openalex":5509,"abstract":5511,"title":5514,"pm":5517,"doi":5519},{"VOID":5504},"2138480916",{"VI":5506},"Bệnh võng mạc đái tháo đường, Yếu tố nguy cơ, Tỷ lệ mắc, Phân tích tổng hợp, Dân số toàn cầu, Thị lực",{"VOID":5508},"3322721",{"VOID":5510},"W2138480916",{"VI":5512,"EN":5513},"\u003Cjats:sec>\n                  \u003Cjats:title>MỤC TIÊU\u003C\u002Fjats:title>\n                  \u003Cjats:p>Để kiểm tra tỷ lệ toàn cầu và các yếu tố nguy cơ chính của bệnh võng mạc đái tháo đường (DR) và bệnh võng mạc đái tháo đường đe dọa thị lực (VTDR) ở những người bị tiểu đường.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>\n               \u003Cjats:sec>\n                  \u003Cjats:title>THIẾT KẾ NGHIÊN CỨU VÀ PHƯƠNG PHÁP\u003C\u002Fjats:title>\n                  \u003Cjats:p>Một phân tích tổng hợp sử dụng dữ liệu tham gia cá nhân từ các nghiên cứu dân số trên toàn thế giới đã được thực hiện. Một đánh giá tài liệu hệ thống đã được tiến hành để xác định tất cả các nghiên cứu dân số trong các quần thể chung hoặc cá nhân bị tiểu đường mà đã xác định được DR từ các hình ảnh võng mạc. Các nghiên cứu cung cấp dữ liệu cho các điểm cuối DR, bao gồm bất kỳ DR nào, DR phát triển, phù hoàng điểm do tiểu đường, và VTDR, cũng như các yếu tố nguy cơ hệ thống chính. Các ước tính tỷ lệ tổng hợp được chuẩn hóa theo độ tuổi trực tiếp theo dân số thế giới năm 2010 trong độ tuổi từ 20–79.","\u003Cjats:sec>\n                  \u003Cjats:title>OBJECTIVE\u003C\u002Fjats:title>\n                  \u003Cjats:p>To examine the global prevalence and major risk factors for diabetic retinopathy (DR) and vision-threatening diabetic retinopathy (VTDR) among people with diabetes.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>\n               \u003Cjats:sec>\n                  \u003Cjats:title>RESEARCH DESIGN AND METHODS\u003C\u002Fjats:title>\n                  \u003Cjats:p>A pooled analysis using individual participant data from population-based studies around the world was performed. A systematic literature review was conducted to identify all population-based studies in general populations or individuals with diabetes who had ascertained DR from retinal photographs. Studies provided data for DR end points, including any DR, proliferative DR, diabetic macular edema, and VTDR, and also major systemic risk factors. Pooled prevalence estimates were directly age-standardized to the 2010 World Diabetes Population aged 20–79 years.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>\n               \u003Cjats:sec>\n                  \u003Cjats:title>RESULTS\u003C\u002Fjats:title>\n                  \u003Cjats:p>A total of 35 studies (1980–2008) provided data from 22,896 individuals with diabetes. The overall prevalence was 34.6% (95% CI 34.5–34.8) for any DR, 6.96% (6.87–7.04) for proliferative DR, 6.81% (6.74–6.89) for diabetic macular edema, and 10.2% (10.1–10.3) for VTDR. All DR prevalence end points increased with diabetes duration, hemoglobin A1c, and blood pressure levels and were higher in people with type 1 compared with type 2 diabetes.\u003C\u002Fjats:p>\n               \u003C\u002Fjats:sec>\n               \u003Cjats:sec>\n                  \u003Cjats:title>CONCLUSIONS\u003C\u002Fjats:title>\n                  \u003Cjats:p>There are approximately 93 million people with DR, 17 million with proliferative DR, 21 million with diabetic macular edema, and 28 million with VTDR worldwide. Longer diabetes duration and poorer glycemic and blood pressure control are strongly associated with DR. These data highlight the substantial worldwide public health burden of DR and the importance of modifiable risk factors in its occurrence. 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