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There is growing evidence suggesting an increased risk of obstetrical complications in women with adenomyosis. An electronic search was conducted among the databases of PubMed, Google Scholar, and Cochrane review from January 2016 to March 2021. Nine observational studies fulfilled the inclusion criteria. Limitations of eligible studies include the presence of a majority of pregnancies achieved by ART, despite ART pregnancies being reported to be associated per se with a higher risk of adverse obstetric outcome; lack of information regarding the presence or absence of endometriosis; nonhomogeneous diagnostic criteria for adenomyosis; lack of the description of the severity of adenomyosis; and different timing of the diagnosis either before or during pregnancy. All studies reported that adenomyosis is associated with preterm delivery. Moreover, available data suggest that adenomyosis is associated with lower pregnancy rates after assisted reproductive techniques (ART) and higher rates of small-for-gestational-age (SGA)\u002Flow–birth weight (LBW) infants, cesarean delivery, and hypertensive disorder of pregnancy (HDP). Further studies on naturally conceived pregnancies, accurately designed in order to consider potential confounding factors, are needed in order to estimate the actual role of adenomyosis on fertility and obstetric and neonatal outcomes. Until more accurate data are available, it seems wise to foresee, when managing pregnant women with adenomyosis, the possible need for additional obstetric and intra-partum care.",{"EN":139},"Adenomyosis and Obstetrical Outcome: a Narrative Mini-Review of the Latest Evidence",{"VOID":141},"10.1007\u002Fs13669-021-00316-1","PUBLICATION","VERIFIED","Auto 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Italy",{"id":163,"sortIndex":92,"affiliation":164,"properties":171},"69c40179-12ec-48fb-b008-b87a751ce70b",{"id":165,"createTime":166,"updateTime":166,"relativeEntities":167,"slug":18,"properties":168,"entityType":38,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19},"2660ee93-1be7-4eb0-8429-448fddc9380f","2023-12-12T09:29:52.351+00:00",[],{"title":169},{"VI":170},"Department of Clinical Sciences and Community Health, Università degli Studi, Milan, Italy",{},{"title":173},{"VI":174},"Vercellini, 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JH, Buchanan EM, Hillson C. Diagnosis and management of ectopic pregnancy. Am Fam Physician. 2014;90:34–40 www.aafp.org\u002F.\nCreanga AA, Shapiro-Mendoza CK, Bish CL, Zane S, Berg CJ, Callaghan WM, et al. Obstet Gynecol. 2011;117:837–43.\nGhaneie A, Grajo JR, Derr C, Kumm TR. Unusual Ectopic Pregnancies,Sonografic Findings And Implications For Management. J Ultrasound Med. 2015;34:951–62 This review provides support for the rare types of ectopic pregnancy ultrasound.\nTenore JL. Ectopic pregnancy. Am Fam Physician. 2000;61:1080–8.\nParker VL, Srinivas M. Non-tubal ectopic pregnancy. Arch Gynecol Obstet. 2016;294:19–27 This review provides support for the clinical management of the rare types of ectopic pregnancies.\nAlkatout I, Honemeyer U, Strauss A, Tinelli A, Malvasi A, Jonat W, et al. Clinical diagnosis and treatment of ectopic pregnancy. Obstet Gynecol Surv. 2013;68:571–81.\nPak JO, Durfee JK, Pedro L, Osborne A, Larkins-Pettigrew M. Retroperitoneal ectopic pregnancy. Obstet Gynecol. 2018;132:1491–3 This case report provides information about the rare retroperitoneal ectopic pregnancy.\nVagg D, Arsala L, Kathurusinghe S, Ang WC. Intramural ectopic pregnancy following myomectomy. J Investig Med High Impact Case Rep. 2018;6:1–4 This case report provides information about the rare intramural ectopic pregnancy.\nMohamed Siraj SM, Min Chern BS. Laparoscopic management of bladder ectopic pregnancy. Gynecol Minim Invasive Ther. 2016;5:124–6.\nSeow KM, Huang LW, Lin YH, Lin MY, Tsai YL, Hwang JL. Cesarean scar pregnancy :issues in management. Ultrasound Obstet Gynecol. 2004;23:247–53.\nAsh A, Smith A, Maxwell D. Caesarean scar pregnancy. Br J Obstet Gynecol. 2007;114:253–63.\nShen L, Fu J, Huang W, Zhu H, Wang Q, Yang S, Wu T. Interventions for non-tubal ectopic pregnancy. Cochrane Database Syst Rev. 2014;7:1–18.\nBenson CB, Doubilet PM. Strategies for conservative treatment of cervical ectopic pregnancy. Ultrasound Obstet Gynecol. 1996;8:371–2.\nCepni I, Ocal P, Erkan S, Erzik B. Conservative treatment of cervical ectopic pregnancy with transvaginal ultrasound guided aspiration and single dose methotrexate. Fertil Steril. 2002;81:1130–2.\nSpitzer D, Steiner H, Graf A, Zajc M, Staudach A. Conservative treatment of cervical pregnancy by curettage and local prostaglandin injection. Hum Reprod. 1997;12:860–6.\nMashiach S, Admon D, Oelsner G, Paz B, Achiron R, Zalel Y. Cervical Shirodkar cerclage may be the treatment modality of choice for cervical pregnancy. Hum Reprod. 2002;17:493–6.\nTrambert JJ, Einstein MH, Banks E, Frost A, Goldberg GL. Uterine artery embolization in the management of vaginal bleeding from cervical pregnancy: a case series. J Reprod Med. 2005;50:844–50.\nChetty M, Elson J. Treating non-tubal ectopic pregnancy. Best Pract Res Clin Obstet Gyneacol. 2009;23:529–38 This review gives information about the clinical management of the non-tubal ectopic preganancies.\nHonemeyer U, Plavsic SK, Kurjak A. Interstisyel ectopic pregnancy: the essential role of ultrasound diagnosis. Donald School J Ultrasound Obstet Gynecol. 2010;4:321–5.\nRaziel A, Schachter M, Mordechai E, Friedler S, Panski M, Ron-EL R. Ovarian pregnancy :a 12- year experience of 19 cases in one instution. Eur J Obstet Gynecol Reprod Biol. 2004;114:92–6.\nDmowski WP, Rana N, Ding J, Wu WT. Retroperitoneal subpancreatic ectopic pregnancy following in vitro fertilization in a patient with previous bilateral salpingectomy: how did it get there? J Assist Reprod Genet. 2002;19:90–3.\nAhmed DM, Fetene AA, Asres EM, Mekonnen AW, Mamuye HG. Cervical stump pregnancy 6 years after subtotal hysterectomy: a case report. J Med Case Rep. 2019;13:135.\nAgarwal N, Odejinmi F. Early abdominal ectopic pregnancy: challenges, update and review of current management. Obstet Gynecol. 2014;16:193–8.\nAyinde OA, Aimakhu CO, Adeyanju OA, Omigbodun AO. Abdominal pregnancy at the university college hospital, Ibadan: a ten-year review. Afr J Reprod Health. 2005;9:123–7.",{"EN":266},"The purpose of this review is to inform readers of recent discussions and findings regarding the underlying aetiology and clinical management of rare ectopic pregnancies. Risk factors, early diagnosis, treatment and results of rare ectopic pregnancies are discussed. New surgical\u002Fmedical modalities and methods that facilitate early diagnosis are shared. With the widespread use of ultrasonography and hcg assays, the diagnosis of ectopic pregnancy is increasing. At the same time, the increase in risk factors (IVF, previous surgeries, pelvic infections) also increases the rare diagnosis of ectopic pregnancy. Accordingly, the probability of detecting rare types increases. There is no clear consensus for the treatment of rare types, but early diagnosis is important to prevent maternal morbidity and mortality, and allows fertility to be preserved in cases in the reproductive age. In conclusion, when ectopic pregnancy is suspected, as well as diagnosis of ectopic pregnancy, non-tubal and other rare types should be considered when no tubal ectopic is detected. Past medical and surgical treatments of the patient should be thoroughly questioned to determine risk factors.",{"EN":268},"Rare Types of Ectopic Pregnancies",{"VOID":270},"10.1007\u002Fs13669-020-00303-y","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs13669-020-00303-y",[273],{"id":274,"sortIndex":19,"researcher":18,"roles":275,"affiliations":276,"properties":285},"da7a9e41-4e9b-4118-b1b6-ff48e33e8083",[152],[277],{"id":18,"sortIndex":19,"affiliation":278,"properties":18},{"id":279,"createTime":280,"updateTime":280,"relativeEntities":281,"slug":18,"properties":282,"entityType":38,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19},"c464d196-eced-436e-b52b-063db6bfbbcb","2024-01-17T03:07:45.099+00:00",[],{"title":283},{"VI":284},"Etlik Zübeyde Hanım Kadın Hastalıkları Eğitim ve Araştırma Hastanesi, Ankara, Turkey",{"title":286},{"VI":287},"Dilek Yüksel",{"url":271,"publisher":289,"properties":315},{"id":6,"createTime":7,"updateTime":8,"relativeEntities":290,"slug":10,"properties":291,"entityType":16,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19,"subjectFields":294,"manageAffiliations":295,"indexDatabases":296,"url":77,"thumbnailPath":18,"statistic":310,"gsStatistic":18,"type":124,"analyzePriority":18},[],{"issn":292,"title":293},{"VOID":13},{"EN":15},[],[],[297,304],{"id":42,"indexDatabase":298,"url":57,"indexYears":18,"academicFieldIds":303,"indexDatabaseRanking":18},{"id":44,"createTime":45,"updateTime":46,"relativeEntities":299,"label":300,"description":301,"key":53,"publicationTags":302,"standard":18},[],{"EN":49,"VI":49},{"VI":51,"EN":52},[55,56],[59],{"id":61,"indexDatabase":305,"url":74,"indexYears":75,"academicFieldIds":18,"indexDatabaseRanking":76},{"id":63,"createTime":64,"updateTime":65,"relativeEntities":306,"label":307,"description":308,"key":71,"publicationTags":309,"standard":18},[],{"EN":68,"VI":68},{"EN":68,"VI":70},[73],{"impactFactor":19,"impactFactorByYear":311,"i10Index":89,"i10IndexLast5Year":19,"totalPublication":90,"totalPublicationByYear":312,"totalCitation":104,"totalCitationByYear":313,"totalCitationPerPublication":113,"totalCitationPerPublicationByYear":314,"hindexLast5Year":103,"hindex":103},{"2014":80,"2015":81,"2016":82,"2017":83,"2018":84,"2019":85,"2020":86,"2021":87,"2022":88,"2023":81},{"2011":92,"2012":93,"2013":94,"2014":95,"2015":96,"2016":97,"2017":96,"2018":98,"2019":99,"2020":99,"2021":100,"2022":101,"2023":102,"2024":103},{"2012":106,"2013":107,"2014":92,"2015":108,"2016":109,"2017":110,"2018":111,"2019":106,"2020":98,"2022":112},{"2012":115,"2013":116,"2014":82,"2015":117,"2016":118,"2017":119,"2018":120,"2019":121,"2020":122,"2022":123},{"volume":316,"pages":318},{"VOID":317},"10",{"VOID":319},"1-6","2021-01-14",2021,{"id":323,"createTime":324,"updateTime":325,"relativeEntities":326,"slug":327,"properties":328,"entityType":142,"verifyStatus":143,"verifyTime":337,"verifyNote":144,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19,"primaryUrl":338,"fullTextUrl":18,"authors":339,"publicationType":224,"publisherRelationship":421,"citationCount":18,"citationInfo":18,"publishDate":453,"publishYear":454,"citationAnalyzeStatus":17,"lastCitationAnalyze":18,"indexDatabases":18,"openAccess":18,"references":18,"isForceReanalyzing":255},"002a0ca2-ae1c-4a6d-85d9-1c045579d885","2024-02-10T16:21:10.844+00:00","2024-12-09T23:18:34.732+00:00",[],"Progesterone-Resistance-Aromatase-and-Inflammation-The-Important-Relationships-Between-Hormones-and-Inflammation",{"references":329,"abstract":331,"title":333,"doi":335},{"VOID":330},"•• Vercellini P, Crosignani P, Somigliana E, et al. ‘Waiting for Godot’: a commonsense approach to the medical treatment of endometriosis. Hum Reprod. 2011;26:3–13. This is an up-to-date critical overview on the current treatments for endometriosis.\nKnapp VJ. How old is endometriosis? Late 17th- and 18th-century European descriptions of the disease. Fertil Steril. 1999;72:10–4.\nKennedy S, Bergqvist A, Chapron C, et al. ESHRE guideline for the diagnosis and treatment of endometriosis. Hum Reprod. 2005;20:2698–704.\nGuo SW. Recurrence of endometriosis and its control. Hum Reprod Update. 2009;15:441–61.\nCrosignani P, Olive D, Bergqvist A, Luciano A. Advances in the management of endometriosis: an update for clinicians. Hum Reprod Update. 2006;12:179–89.\nBarrier BF. Immunology of endometriosis. Clin Obstet Gynecol. 2010;53:397–402.\nBacci M, Capobianco A, Monno A, et al. Macrophages are alternatively activated in patients with endometriosis and required for growth and vascularization of lesions in a mouse model of disease. Am J Pathol. 2009;175:547–56.\n• Capobianco A, Monno A, Cottone L, et al. Proangiogenic Tie(2) macrophages infiltrate human and murine endometriotic lesions and dictate their growth in a mouse model of the disease. Am J Pathol. 2011;179:2651–9. This article is a characterization of macrophages infiltrating endometriotic lesions in terms of angiogenic potential.\nNothnick WB. Treating endometriosis as an autoimmune disease. Fertil Steril. 2001;76:223–31.\nSinaii N, Cleary SD, Ballweg ML, et al. High rates of autoimmune and endocrine disorders, fibromyalgia, chronic fatigue syndrome and atopic diseases among women with endometriosis: a survey analysis. Hum Reprod. 2002;17:2715–24.\nMatarese G, De Placido G, Nikas Y, Alviggi C. Pathogenesis of endometriosis: natural immunity dysfunction or autoimmune disease? Trends Mol Med. 2003;9:223–8.\nHever A, Roth RB, Hevezi P, et al. Human endometriosis is associated with plasma cells and overexpression of B lymphocyte stimulator. Proc Natl Acad Sci U S A. 2007;104:12451–6.\nFlores I, Rivera E, Mousses S, et al. Identification of molecular markers for endometriosis in blood lymphocytes by using deoxyribonucleic acid microarrays. Fertil Steril. 2006;85:1676–83.\nSomigliana E, Vigano P, Parazzini F, et al. Association between endometriosis and cancer: a comprehensive review and a critical analysis of clinical and epidemiological evidence. Gynecol Oncol. 2006;101:331–41.\nVigano P, Somigliana E, Parazzini F, Vercellini P. Bias versus causality: interpreting recent evidence of association between endometriosis and ovarian cancer. Fertil Steril. 2007;88:588–93.\nGentilini D, Perino A, Vigano P, et al. Gene expression profiling of peripheral blood mononuclear cells in endometriosis identifies genes altered in non-gynaecologic chronic inflammatory diseases. Hum Reprod. 2011;26:3109–17.\nKoczan D, Guthke R, Thiesen HJ, et al. Gene expression profiling of peripheral blood mononuclear leukocytes from psoriasis patients identifies new immune regulatory molecules. Eur J Dermatol. 2005;15:251–7.\nLuk T, Malam Z, Marshall JC. Pre-B cell colony-enhancing factor (PBEF)\u002Fvisfatin: a novel mediator of innate immunity. J Leukoc Biol. 2008;83:804–16.\nVoisine P, Ruel M, Khan TA, et al. Differences in gene expression profiles of diabetic and nondiabetic patients undergoing cardiopulmonary bypass and cardioplegic arrest. Circulation. 2004;110:II280–286.\nMote PA, Bartow S, Tran N, Clarke CL. Loss of co-ordinate expression of progesterone receptors A and B is an early event in breast carcinogenesis. Breast Cancer Res Treat. 2002;72:163–72.\nIto K, Utsunomiya H, Yaegashi N, Sasano H. Biological roles of estrogen and progesterone in human endometrial carcinoma–new developments in potential endocrine therapy for endometrial cancer. Endocr J. 2007;54:667–79.\nDe Vivo I, Huggins GS, Hankinson SE, et al. A functional polymorphism in the promoter of the progesterone receptor gene associated with endometrial cancer risk. Proc Natl Acad Sci U S A. 2002;99:12263–8.\nJacobsen BM, Richer JK, Sartorius CA, Horwitz KB. Expression profiling of human breast cancers and gene regulation by progesterone receptors. J Mammary Gland Biol Neoplasia. 2003;8:257–68.\nAttia GR, Zeitoun K, Edwards D, et al. Progesterone receptor isoform A but not B is expressed in endometriosis. J Clin Endocrinol Metab. 2000;85:2897–902.\nIgarashi TM, Bruner-Tran KL, Yeaman GR, et al. Reduced expression of progesterone receptor-B in the endometrium of women with endometriosis and in cocultures of endometrial cells exposed to 2,3,7,8-tetrachlorodibenzo-p-dioxin. Fertil Steril. 2005;84:67–74.\nWu Y, Strawn E, Basir Z, et al. Promoter hypermethylation of progesterone receptor isoform B (PR-B) in endometriosis. Epigenetics. 2006;1:106–11.\nBukulmez O, Hardy DB, Carr BR, et al. Inflammatory status influences aromatase and steroid receptor expression in endometriosis. Endocrinology. 2008;149:1190–204.\nBurney RO, Talbi S, Hamilton AE, et al. Gene expression analysis of endometrium reveals progesterone resistance and candidate susceptibility genes in women with endometriosis. Endocrinology. 2007;148:3814–26.\nGentilini D, Vigano P, Vignali M, et al. Endometrial stromal progesterone receptor-A\u002Fprogesterone receptor-B ratio: no difference between women with and without endometriosis. Fertil Steril. 2010;94:1538–40.\nGentilini D, Vigano P, Somigliana E, et al. Endometrial stromal cells from women with endometriosis reveal peculiar migratory behavior in response to ovarian steroids. Fertil Steril. 2010;93:706–15.\nBackdahl L, Bushell A, Beck S. Inflammatory signalling as mediator of epigenetic modulation in tissue-specific chronic inflammation. Int J Biochem Cell Biol. 2009;41:176–84.\nBromer JG, Zhou Y, Taylor MB, et al. Bisphenol-A exposure in utero leads to epigenetic alterations in the developmental programming of uterine estrogen response. FASEB J. 2010;24:2273–80.\nHeindel JJ, McAllister KA, Worth Jr L, Tyson FL. Environmental epigenomics, imprinting and disease susceptibility. Epigenetics. 2006;1:1–6.\nBruner-Tran KL, Osteen KG. Developmental exposure to TCDD reduces fertility and negatively affects pregnancy outcomes across multiple generations. Reprod Toxicol. 2011;31:344–50.\nBruner-Tran KL, Ding T, Osteen KG. Dioxin and endometrial progesterone resistance. Semin Reprod Med. 2010;28:59–68.\nNayyar T, Bruner-Tran KL, Piestrzeniewicz-Ulanska D, Osteen KG. Developmental exposure of mice to TCDD elicits a similar uterine phenotype in adult animals as observed in women with endometriosis. Reprod Toxicol. 2007;23:326–36.\nStilley JA, Woods-Marshall R, Sutovsky M, et al. Reduced fecundity in female rats with surgically induced endometriosis and in their daughters: a potential role for tissue inhibitors of metalloproteinase 1. Biol Reprod. 2009;80:649–56.\nSignorile PG, Spugnini EP, Mita L, et al. Pre-natal exposure of mice to bisphenol A elicits an endometriosis-like phenotype in female offspring. Gen Comp Endocrinol. 2010;168:318–25.\nSeli E, Berkkanoglu M, Arici A. Pathogenesis of endometriosis. Obstet Gynecol Clin North Am. 2003;30:41–61.\nColette S, Lousse JC, Defrere S, et al. Absence of aromatase protein and mRNA expression in endometriosis. Hum Reprod. 2009;24:2133–41.\nColette S, Donnez J. Endometriosis. N Engl J Med. 2009;360:1911–2. author reply 1912.\nAttar E, Bulun SE. Aromatase inhibitors: the next generation of therapeutics for endometriosis? Fertil Steril. 2006;85:1307–18.\nBulun SE. Endometriosis. N Engl J Med. 2009;360:268–79.\nBulun SE ZK, Takayama K, Noble L, Michael D, Simpson E, Johns A, Putman M, Sasano H, Carr B. Aromatase expression in endometriosis: biology and clinical perspectives. Understanding and managing endometriosis: advances in research and practice. Quebec City: Parthenon Publishing; 1998.\nBulun SE, Zeitoun KM, Takayama K, Sasano H. Molecular basis for treating endometriosis with aromatase inhibitors. Hum Reprod Update. 2000;6:413–8.\nNawathe A, Patwardhan S, Yates D, et al. Systematic review of the effects of aromatase inhibitors on pain associated with endometriosis. BJOG. 2008;115:818–22.\nFerrero S, Venturini PL, Ragni N, et al. Pharmacological treatment of endometriosis: experience with aromatase inhibitors. Drugs. 2009;69:943–52.\nFang Z, Yang S, Gurates B, et al. Genetic or enzymatic disruption of aromatase inhibits the growth of ectopic uterine tissue. J Clin Endocrinol Metab. 2002;87:3460–6.\nNoble LS, Takayama K, Zeitoun KM, et al. Prostaglandin E2 stimulates aromatase expression in endometriosis-derived stromal cells. J Clin Endocrinol Metab. 1997;82:600–6.\nVigano P, Mangioni S, Odorizzi MP, et al. Use of estrogen antagonists and aromatase inhibitors in endometriosis. Curr Opin Investig Drugs. 2003;4:1209–12.\nCasper RF. Letrozole versus clomiphene citrate: which is better for ovulation induction? Fertil Steril. 2009;92:858–9.\nSoysal S, Soysal ME, Ozer S, et al. The effects of post-surgical administration of goserelin plus anastrozole compared to goserelin alone in patients with severe endometriosis: a prospective randomized trial. Hum Reprod. 2004;19:160–7.\nAilawadi RK, Jobanputra S, Kataria M, et al. Treatment of endometriosis and chronic pelvic pain with letrozole and norethindrone acetate: a pilot study. Fertil Steril. 2004;81:290–6.\nAmsterdam LL, Gentry W, Jobanputra S, et al. Anastrazole and oral contraceptives: a novel treatment for endometriosis. Fertil Steril. 2005;84:300–4.\nRemorgida V, Abbamonte HL, Ragni N, et al. Letrozole and norethisterone acetate in rectovaginal endometriosis. Fertil Steril. 2007;88:724–6.\nFerrero S, Camerini G, Seracchioli R, et al. Letrozole combined with norethisterone acetate compared with norethisterone acetate alone in the treatment of pain symptoms caused by endometriosis. Hum Reprod. 2009;24:3033–41.\nAmerican College of Obstetricians and Gynecologists. ACOG Committee Opinion. Aromatase inhibitors in gynecologic practice. Obstet Gynecol. 2008;112:405–7.\nNear AM, Wu AH, Templeman C, et al. Progesterone receptor gene polymorphisms and risk of endometriosis: results from an international collaborative effort. Fertil Steril. 2011;95:40–5.\nAghajanova L, Velarde MC, Giudice LC. The progesterone receptor coactivator Hic-5 is involved in the pathophysiology of endometriosis. Endocrinology. 2009;150:3863–70.\nKao LC, Germeyer A, Tulac S, et al. Expression profiling of endometrium from women with endometriosis reveals candidate genes for disease-based implantation failure and infertility. Endocrinology. 2003;144:2870–81.\nBulun SE, Cheng YH, Yin P, et al. progesterone resistance in endometriosis: link to failure to metabolize estradiol. Mol Cell Endocrinol. 2006;248:94–103.\nAghajanova L, Tatsumi K, Horcajadas JA, et al. Unique transcriptome, pathways and networks in the human endometrial fibroblast response to progesterone in endometriosis. Biol Reprod. 2011;84:801–15.\nBruner-Tran KL, Zhang Z, Eisenberg E, et al. Down-regulation of endometrial matrix metalloproteinase-3 and −7 expression in vitro and therapeutic regression of experimental endometriosis in vivo by a novel nonsteroidal progesterone receptor agonist, tanaproget. J Clin Endocrinol Metab. 2006;91:1554–60.\nNayyar T, Bruner-Tran KL, Piestrzeniewicz-Ulanska D, et al. Developmental exposure of mice to TCDD elicits a similar uterine phenotype in adult animals as observed in women with endometriosis. Reprod Toxicol. 2007;23:326–36.\nVercellini P, Fedele L, Pietropaolo G, et al. Progestogens for endometriosis: forward to the past. Hum Reprod Update. 2003;9:387–96.",{"EN":332},"Endometriosis is a complex gynecologic disorder in which alterations of specific biological processes involving both the endocrine and the immune systems have been identified. Recently, a reduced endometrial responsiveness to progesterone has been suggested as a crucial element in the pathophysiology of the disease but it is unclear whether this reduced sensitivity to the steroid is involved in the disease induction or is simply a consequence of the disease condition. Indeed, endometriosis is associated with a local and systemic inflammatory process that is strongly inter-related to the steroid activity, and both the systems mutually interact in a complex loop that strongly influences the disease development and maintenance. The link between prostaglandin E2 (PGE2), aromatase activity, and local estrogen synthesis represents a paradigm of this loop. A better understanding of the pathophysiology of this interaction should enable researchers to develop targeted therapeutics that may limit this self-supporting pathologic loop.",{"EN":334},"Progesterone Resistance, Aromatase, and Inflammation: The Important Relationships Between Hormones and Inflammation",{"VOID":336},"10.1007\u002Fs13669-012-0013-8","2024-12-09T23:18:34.731+00:00","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs13669-012-0013-8",[340,355,370,382,397,409],{"id":341,"sortIndex":19,"researcher":18,"roles":342,"affiliations":343,"properties":352},"2086d819-1df8-47bb-ac8a-2bb11cd00057",[152],[344],{"id":18,"sortIndex":19,"affiliation":345,"properties":18},{"id":346,"createTime":347,"updateTime":347,"relativeEntities":348,"slug":18,"properties":349,"entityType":38,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19},"4ded3707-4031-42c3-ae70-db2b49543304","2024-02-05T23:35:01.356+00:00",[],{"title":350},{"VI":351},"Obstetrics and Gynecology Unit, San Raffaele Scientific Institute, Milano, Italy",{"title":353},{"VI":354},"Paola Vigano",{"id":356,"sortIndex":89,"researcher":18,"roles":357,"affiliations":358,"properties":367},"dfedba00-f896-45ca-b798-48f222ac1a86",[152],[359],{"id":18,"sortIndex":19,"affiliation":360,"properties":18},{"id":361,"createTime":362,"updateTime":362,"relativeEntities":363,"slug":18,"properties":364,"entityType":38,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19},"39d092a7-1e55-4b4c-b998-b66b1b4c7c73","2024-02-10T16:21:10.928+00:00",[],{"title":365},{"VI":366},"Istituto Ostetrico e Ginecologico “L. Mangiagalli,” University of Milano, Milano, Italy",{"title":368},{"VI":369},"Paolo Vercellini",{"id":371,"sortIndex":150,"researcher":18,"roles":372,"affiliations":373,"properties":379},"b585399e-f155-4b55-821c-b49cd56afa65",[152],[374],{"id":18,"sortIndex":19,"affiliation":375,"properties":18},{"id":346,"createTime":347,"updateTime":347,"relativeEntities":376,"slug":18,"properties":377,"entityType":38,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19},[],{"title":378},{"VI":351},{"title":380},{"VI":381},"Massimo Candiani",{"id":383,"sortIndex":110,"researcher":18,"roles":384,"affiliations":385,"properties":394},"86c9e64e-5674-4c78-9c65-3394f44c0837",[152],[386],{"id":18,"sortIndex":19,"affiliation":387,"properties":18},{"id":388,"createTime":389,"updateTime":389,"relativeEntities":390,"slug":18,"properties":391,"entityType":38,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19},"c86e96f1-a6e9-48e0-8d14-45eece8efea8","2023-12-18T01:47:29.575+00:00",[],{"title":392},{"VI":393},"Department of Obstetrics, Gynecology and Neonatology, Fondazione Cà Granda, Ospedale Maggiore Policlinico, Milano, Italy",{"title":395},{"VI":396},"Edgardo Somigliana",{"id":398,"sortIndex":189,"researcher":18,"roles":399,"affiliations":400,"properties":406},"fd93dff1-8340-41d9-bc78-5c9d0ba059eb",[152],[401],{"id":18,"sortIndex":19,"affiliation":402,"properties":18},{"id":346,"createTime":347,"updateTime":347,"relativeEntities":403,"slug":18,"properties":404,"entityType":38,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19},[],{"title":405},{"VI":351},{"title":407},{"VI":408},"Luca Pagliardini",{"id":410,"sortIndex":92,"researcher":18,"roles":411,"affiliations":412,"properties":418},"d8095d47-23be-4d26-bbd2-cf993858e12e",[152],[413],{"id":18,"sortIndex":19,"affiliation":414,"properties":18},{"id":346,"createTime":347,"updateTime":347,"relativeEntities":415,"slug":18,"properties":416,"entityType":38,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19},[],{"title":417},{"VI":351},{"title":419},{"VI":420},"Elisa Rabellotti",{"url":338,"publisher":422,"properties":448},{"id":6,"createTime":7,"updateTime":8,"relativeEntities":423,"slug":10,"properties":424,"entityType":16,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19,"subjectFields":427,"manageAffiliations":428,"indexDatabases":429,"url":77,"thumbnailPath":18,"statistic":443,"gsStatistic":18,"type":124,"analyzePriority":18},[],{"issn":425,"title":426},{"VOID":13},{"EN":15},[],[],[430,437],{"id":42,"indexDatabase":431,"url":57,"indexYears":18,"academicFieldIds":436,"indexDatabaseRanking":18},{"id":44,"createTime":45,"updateTime":46,"relativeEntities":432,"label":433,"description":434,"key":53,"publicationTags":435,"standard":18},[],{"EN":49,"VI":49},{"VI":51,"EN":52},[55,56],[59],{"id":61,"indexDatabase":438,"url":74,"indexYears":75,"academicFieldIds":18,"indexDatabaseRanking":76},{"id":63,"createTime":64,"updateTime":65,"relativeEntities":439,"label":440,"description":441,"key":71,"publicationTags":442,"standard":18},[],{"EN":68,"VI":68},{"EN":68,"VI":70},[73],{"impactFactor":19,"impactFactorByYear":444,"i10Index":89,"i10IndexLast5Year":19,"totalPublication":90,"totalPublicationByYear":445,"totalCitation":104,"totalCitationByYear":446,"totalCitationPerPublication":113,"totalCitationPerPublicationByYear":447,"hindexLast5Year":103,"hindex":103},{"2014":80,"2015":81,"2016":82,"2017":83,"2018":84,"2019":85,"2020":86,"2021":87,"2022":88,"2023":81},{"2011":92,"2012":93,"2013":94,"2014":95,"2015":96,"2016":97,"2017":96,"2018":98,"2019":99,"2020":99,"2021":100,"2022":101,"2023":102,"2024":103},{"2012":106,"2013":107,"2014":92,"2015":108,"2016":109,"2017":110,"2018":111,"2019":106,"2020":98,"2022":112},{"2012":115,"2013":116,"2014":82,"2015":117,"2016":118,"2017":119,"2018":120,"2019":121,"2020":122,"2022":123},{"volume":449,"pages":451},{"VOID":450},"1",{"VOID":452},"146-152","2012-04-27",2012,{"id":456,"createTime":457,"updateTime":458,"relativeEntities":459,"slug":460,"properties":461,"entityType":142,"verifyStatus":143,"verifyTime":458,"verifyNote":144,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":92,"primaryUrl":470,"fullTextUrl":18,"authors":471,"publicationType":224,"publisherRelationship":514,"citationCount":18,"citationInfo":18,"publishDate":546,"publishYear":547,"citationAnalyzeStatus":17,"lastCitationAnalyze":18,"indexDatabases":18,"openAccess":18,"references":18,"isForceReanalyzing":255},"f5710460-69b4-44d9-902c-f7db19b57b0a","2024-01-28T15:45:04.215+00:00","2024-12-26T23:14:46.936+00:00",[],"Global-Abortion-Policies-Database-a-Descriptive-Analysis-of-Financial-Coverage-for-Abortion-Care",{"references":462,"abstract":464,"title":466,"doi":468},{"VOID":463},"UN General Assembly. Political declaration of the high-level meeting on universal health coverage, 10 October 2019, A\u002FRES\u002F74\u002F2. https:\u002F\u002Fundocsorg\u002Fen\u002FA\u002FRES\u002F74\u002F2. Accessed 27 March 2020.\n• The World Health Report. Research for universal health coverage. Geneva: World Health Organization; 2013. The report focuses on the importance of research in advancing progress towards universal health coverage.\nUN General Assembly. Entry into force of the constitution of the World Health Organization, 17 November 1947, A\u002FRES\u002F131. https:\u002F\u002Fwwwrefworldorg\u002Fdocid\u002F3b00f09554html Accessed 27 Feb 2020.\nThirteenth general programme of work 2019−2023. Geneva: World Health Organization; 2019.\n•• The World Health Report. Health systems financing: the path to universal coverage. Geneva: World Health Organization; 2010. This report identifies what countries can do to modify their financing systems and move towards universal health coverage.\nMathauer I, Mathivet B, Kutzin J. Free health care policies: opportunities and risks for moving towards UHC. Geneva: World Health Organization; 2017.\nIlboudo P, Greco G, Sundby J, Torsvik G. Costs and consequences of abortions to women and their households: a cross-sectional study in Ouagadougou, Burkina Faso. Health Policy Plan. 2015;30(4):500–7. https:\u002F\u002Fdoi.org\u002F10.1093\u002Fheapol\u002Fczu025.\nSundaram A, Vlassoff M, Mugisha F, Bankole A, Singh S, Amanya L, et al. Documenting the individual- and household-level cost of unsafe abortion in Uganda. IPSRH. 2013;39(4):174–84.\nDuggal R. The political economy of abortion in India: cost and expenditure patterns. Reprod Health Matters. 2004;12(24 Suppl):130–7.\nBoonstra H. Abortion in the lives of women struggling financially: why insurance coverage matters. Guttmacher Policy Review: 2016; 19. https:\u002F\u002Fwww.guttmacher.org\u002Fgpr\u002F2016\u002F07\u002Fabortion-lives-women-struggling-financially-why-insurance-coverage-matters. Accessed 27 March 2020.\nPrioritizing essential packages of health services in six countries in Sub-Saharan Africa. 2019. https:\u002F\u002Fwww.who.int\u002Fpmnch\u002Fmedia\u002Fnews\u002F2019\u002FFull-report.pdf. Accessed 27 March 2020.\nHERA Right to Health & Development. Universal health coverage for sexual and reproductive health in the Asia-Pacific region. Belgium. 2018. https:\u002F\u002Fasiapacific.unfpa.org\u002Fsites\u002Fdefault\u002Ffiles\u002Fpub-pdf\u002FUNFPA_SRH_hera_171023_web_optimized.pdf. Accessed 27 March 2020.\n••• Safe abortion: technical and policy guidance for health systems, second edition. Geneva: World Health Organization; 2012. This guideline provides evidence-based guidance on clinical care, as well as information on how to establish and strengthen services, and outlines a human-rights-based approach to laws and policies on safe, comprehensive abortion care.\nJohnson BR, Lavelanet AF, Schlitt S. Global abortion policies database: a new approach to strengthening knowledge on laws, policies, and human rights standards. BMC Int Health Hum Rights. 2018;18:35. https:\u002F\u002Fdoi.org\u002F10.1186\u002Fs12914-018-0174-2.\nLavelanet AF, Schlitt S, Johnson BR, Ganatra B. Global abortion policies database: a descriptive analysis of the legal categories of lawful abortion. BMC Int Health Hum Rights. 2018;18:35. https:\u002F\u002Fdoi.org\u002F10.1186\u002Fs12914-018-0183-1.\nLavelanet AF, Johnson BR, Ganatra B. Global abortion policies database: a descriptive analysis of the regulatory and policy environment related to abortion. Best Pract Res Clin Obstet Gynaecol. 2019;62:25–35. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.bpobgyn.2019.06.002.\nWorld Health Organization. Universal health coverage – key facts. 2019. https:\u002F\u002Fwww.who.int\u002Fen\u002Fnews-room\u002Ffact-sheets\u002Fdetail\u002Funiversal-health-coverage-(uhc). Accessed 27 March 2020.\nGanatra B, Gerdts C, Rossier C, Johnson BR Jr, Tunçalp Ö, Assifi A, et al. Global, regional, and subregional classification of abortions by safety, 2010-14: estimates from a Bayesian hierarchical model. Lancet. 2017;390(10110):2372–81. https:\u002F\u002Fdoi.org\u002F10.1016\u002FS0140-6736(17)31794-4.\nSay L, Chou D, Gemmill AT, OMoller AB, et al. Global causes of maternal death: a WHO systematic analysis. Lancet Glob Health. 2014;2:e323–33. https:\u002F\u002Fdoi.org\u002F10.1016\u002FS2214-109X(14)70227-X.\nGrossman D, Grindlay K, Burns B. Public funding for abortion where broadly legal. Contraception. 2016;94(5):453–60.\nErdman JN, Cook RJ. Decriminalization of abortion – a human rights imperative. Best Pract Res Clin Obstet Gynaecol. 2020;62:11–24. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.bpobgyn.2019.05.004.\nUN Committee for the Elimination of all forms of Discrimination against Women, concluding observations for Slovakia, CEDAW\u002FC\u002FSVK\u002FCO\u002F5–6 (25 November 2015), para 30 and 31.\nUN Committee on the Rights of the Child, concluding observations for Slovakia, CRC\u002FC\u002FSVK\u002FCO\u002F3–5 (20 July 2016), para 39 and 40.\nUN Human Rights Committee. Concluding Observations for Ghana, CCPR\u002FC\u002FGHA\u002FCO\u002F1 (9 August 2016), para 23 and 24.\nUN Committee for the Elimination of all forms of Discrimination against Women, concluding observations for Serbia, CEDAW\u002FC\u002FSRB\u002FCO\u002F2–3 (30 July 2013), para 32 and 33.\nUN Human Rights Committee. Concluding observations for St Vincent and the Grenadines, CCPR\u002FC\u002FVCT\u002FCO\u002F2\u002FAdd.1 (9 May 2019), para 20 and 21.\nHealth worker roles in providing safe abortion care and post-abortion contraception. Geneva: World Health Organization; 2015.",{"EN":465},"The purpose of this review is to report on data available in the Global Abortion Policies Database (GAPD) as of February 2020. The GAPD is a repository of publicly available sources related to abortion for all countries. Research indicates that where individuals must pay for abortion procedures, costs can often be unaffordable and catastrophic for households. Faced with financial barriers, individuals may be deterred from seeking healthcare or may resort to clandestine practices. There are limited countries with publicly available sources related to financial protection for abortion services. Even where legal access may be available on paper, several countries restrict actual access by limiting financial protection to specific types of individuals seeking abortion or certain legal categories of abortion, thus raising questions of equity and possible human rights violations. As countries advance towards universal health coverage (UHC), there is an urgent need to expand the GAPD to include additional data, building the evidence base on national health systems financing, and to consider the implications of financial protection with respect to access to abortion services.",{"EN":467},"Global Abortion Policies Database: a Descriptive Analysis of Financial Coverage for Abortion Care",{"VOID":469},"10.1007\u002Fs13669-020-00294-w","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs13669-020-00294-w",[472,487,502],{"id":473,"sortIndex":189,"researcher":18,"roles":474,"affiliations":475,"properties":484},"9c3b486e-2692-4a58-8968-48f20a91f0a3",[152],[476],{"id":18,"sortIndex":19,"affiliation":477,"properties":18},{"id":478,"createTime":479,"updateTime":479,"relativeEntities":480,"slug":18,"properties":481,"entityType":38,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19},"989d6bd8-44e8-4716-8255-0d67343d58d6","2024-01-28T15:45:04.249+00:00",[],{"title":482},{"VI":483},"Department of Sexual and Reproductive Health and Research and UNDP-UNFPA-UNICEF-WHO-World Bank Special Programme of Research, Development and Research Training in Human Reproduction (HRP), World Health Organization, Geneva 27, Switzerland",{"title":485},{"VI":486},"Veloshnee Govender",{"id":488,"sortIndex":92,"researcher":18,"roles":489,"affiliations":490,"properties":499},"5acf0c68-fea6-41fb-bbf0-f850649e22d2",[152],[491],{"id":18,"sortIndex":19,"affiliation":492,"properties":18},{"id":493,"createTime":494,"updateTime":494,"relativeEntities":495,"slug":18,"properties":496,"entityType":38,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19},"6ae1d3a0-9ead-4ec3-b94e-387aa5a2ec51","2024-01-28T15:45:04.236+00:00",[],{"title":497},{"VI":498},"UNDP-UNFPA-UNICEF-WHO-World Bank Special Programme of Research, Development and Research Training in Human Reproduction (HRP) (Consultant), World Health Organization, Geneva 27, Switzerland",{"title":500},{"VI":501},"Esther Major",{"id":503,"sortIndex":19,"researcher":18,"roles":504,"affiliations":505,"properties":511},"054ccf13-01e3-4151-8daf-f1b643ec5403",[152],[506],{"id":18,"sortIndex":19,"affiliation":507,"properties":18},{"id":478,"createTime":479,"updateTime":479,"relativeEntities":508,"slug":18,"properties":509,"entityType":38,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19},[],{"title":510},{"VI":483},{"title":512},{"VI":513},"Antonella F. 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three-fold purpose of this review is to summarize recent evidence supporting the use of a multidisciplinary team (MDT) approach in patients with placenta accreta spectrum (PAS), to describe essential members and processes of the PAS MDT, and to highlight necessary resources for both planned and unexpected cases of PAS. Maternal outcomes in women with PAS are improved when an antenatal diagnosis occurs and referral is made to centers with MDT specialization and experience in treating PAS. New guidelines unequivocally recommend referral to higher-volume, experienced PAS centers of excellence. The diagnosis and treatment of patients with PAS are complex, resource-intensive, and pose high risk of maternal morbidity and mortality. Recent evidence suggests that stable, experienced MDTs focused on continuous systems improvement at high-volume tertiary care centers are superior to standard obstetric management for patients with PAS. Referral to a center of excellence is indicated when PAS is suspected.",{"EN":664},"Multidisciplinary Teams in the Management of Placenta Accreta Spectrum Disorders",{"VOID":666},"10.1007\u002Fs13669-019-00264-x",[668],"EN","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs13669-019-00264-x",[671,688],{"id":672,"sortIndex":19,"researcher":18,"roles":673,"affiliations":674,"properties":683},"304007ac-4105-4c1c-aaee-d45768feb718",[],[675],{"id":18,"sortIndex":19,"affiliation":676,"properties":18},{"id":677,"createTime":678,"updateTime":678,"relativeEntities":679,"slug":18,"properties":680,"entityType":38,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19},"3c7a7557-e3ef-4446-8f63-09b8586a4a6f","2023-12-12T13:02:08.957+00:00",[],{"title":681},{"VI":682},"University of Utah Health, Salt Lake City, USA",{"title":684,"email":686},{"EN":685},"Brett D. 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Regionalization of care for obstetric hemorrhage and its effect on maternal mortality. Obstet Gynecol. 2010;115:1194–200 In a large nationally-representative sample, the authors convincingly demonstrate a difference in maternal mortality between high- and low-volume hospitals for women undergoing peripartum hysterectomy, 35% of whom had a diagnosis of placenta accreta spectrum.",{"id":18,"text":733,"url":18,"identifiers":18},"• Eller AG, Bennett MA, Sharshiner M, Masheter C, Soisson AP, Dodson M, et al. Maternal morbidity in cases of placenta accreta managed by a multidisciplinary care team compared with standard obstetric care. Obstet Gynecol. 2011;117:331–7 This state-wide evaluation of placenta accreta management was the first and still largest study to show reduced maternal morbidity for women with PAS receiving care within multidisciplinary care setting.",{"id":18,"text":735,"url":18,"identifiers":18},"Shamshirsaz AA, Fox KA, Salmanian B, Diaz-Arrastia CR, Lee W, Baker BW, et al. Maternal morbidity in patients with morbidly adherent placenta treated with and without a standardized multidisciplinary approach. Am J Obstet Gynecol. 2015;212:218.e1–9.",{"id":18,"text":737,"url":18,"identifiers":18},"Shamshirsaz AA, Fox KA, Erfani H, Clark SL, Salmanian B, Baker BW, et al. Multidisciplinary team learning in the management of the morbidly adherent placenta: outcome improvements over time. Am J Obstet Gynecol. 2017;216:612.e1–5.",{"id":18,"text":739,"url":18,"identifiers":18},"Placenta accreta spectrum. Obstetric care consensus no. 7. American College of Obstetricians and Gynecologists. Obstet Gynecol. 2018;132:e259–75.",{"id":18,"text":741,"url":18,"identifiers":18},"Levels of maternal care. Obstetric care consensus no. 2. American College of Obstetricians and Gynecologists. Obstet Gynecol. 2015;125:502–15.",{"id":18,"text":743,"url":18,"identifiers":18},"Jauniaux ERM, Alfirevic Z, Bhide AG, Belfort MA, Burton GJ, Collins SL, Dornan S, Jurkovic D, Kayem G, Kingdom J, Silver R, Sentilhes L on behalf of the Royal College of Obstetricians and Gynaecologists. Placenta Praevia and Placenta Accreta: Diagnosis and Management. Green-top Guideline No. 27a. BJOG 2018",{"id":18,"text":745,"url":18,"identifiers":18},"Wright JD, Silver RM, Bonanno C, Gaddipati S, Lu YS, Simpson LL, et al. Practice patterns and knowledge of obstetricians and gynecologists regarding placenta accreta. J Matern Fetal Neonatal Med. 2013;26:1602–9.",{"id":18,"text":747,"url":18,"identifiers":18},"Fitzpatrick KE, Sellers S, Spark P, Kurinczuk JJ, Brocklehurst P, Knight M. The management and outcomes of placenta accreta, increta, and percreta in the UK: a population-based descriptive study. BJOG. 2014;121:62–71.",{"id":18,"text":749,"url":18,"identifiers":18},"Bailit JL, Grobman WA, Rice MM, Reddy UM, Wapner RJ, Varner MW, et al. Morbidly adherent placenta treatments and outcomes. Obstet Gynecol. 2015;125:683–9.",{"id":18,"text":751,"url":18,"identifiers":18},"Eller AG, Porter TF, Soisson P, Silver RM. Optimal management strategies for placenta accreta. BJOG. 2009;116:648–54.",{"id":18,"text":753,"url":18,"identifiers":18},"Warshak CR, Ramos GA, Eskander R, Benirschke K, Saenz CC, Kelly TF, et al. Effect of predelivery diagnosis in 99 consecutive cases of placenta accreta. Obstet Gynecol. 2010;115:65–9.",{"id":18,"text":755,"url":18,"identifiers":18},"•• Silver, RM FKA, Barton JR, Abuhamad AZ, Simhan H, Huls CK, et al. Center of excellence for placenta accreta. Am J Obstet Gynecol. 2015;212:561–8 This position statement from a group of experts at experienced accreta referral centers first defined the concept and characteristics of a multidisciplinary accreta team.",{"id":18,"text":757,"url":18,"identifiers":18},"Skupski DW, Brady D, Lowenwirt IP, Sample J, Lin SN, Lohana R, et al. Improvement in outcomes of major obstetric hemorrhage through systematic change. Obstet Gynecol. 2017;130(4):770–7.",{"id":18,"text":759,"url":18,"identifiers":18},"Fransen AF, van de Ven J, Schuit E, van Tetering A, Mol BW, Oei SG. Simulation-based team training for multi-professional obstetric care teams to improve patient outcome: a multicentre, cluster randomised controlled trial. BJOG. 2017;124(4):641–50.",{"id":761,"createTime":762,"updateTime":763,"relativeEntities":764,"slug":765,"properties":766,"entityType":142,"verifyStatus":143,"verifyTime":775,"verifyNote":144,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19,"primaryUrl":776,"fullTextUrl":18,"authors":777,"publicationType":224,"publisherRelationship":825,"citationCount":18,"citationInfo":18,"publishDate":857,"publishYear":858,"citationAnalyzeStatus":17,"lastCitationAnalyze":18,"indexDatabases":18,"openAccess":18,"references":18,"isForceReanalyzing":255},"4f91dcfb-1d40-42a8-9c2b-57875a7974c0","2023-12-29T14:22:37.618+00:00","2025-01-15T22:33:54.604+00:00",[],"Point-of-Care-Thromboelastometry-in-the-Management-of-Acute-Obstetric-Hemorrhage",{"references":767,"abstract":769,"title":771,"doi":773},{"VOID":768},"Say L, Chou D, Gemmill A, Tunçalp Ö, Moller AB, Daniels J, et al. Global causes of maternal death: a WHO systematic analysis. Lancet Global Health. 2014;2(6):e323–33.\nCristina Rossi A, Mullin P. The etiology of maternal mortality in developed countries: a systematic review of literature. Arch Gynecol Obstet. 2012;285(6):1499–503.\nCallaghan WM, MacKay AP, Berg CJ. Identification of severe maternal morbidity during delivery hospitalizations, United States, 1991–2003. Am J Obstet Gynecol. 2008;199(2):133–e1.\nLiu S, Joseph KS, Bartholomew S, Fahey J, Lee L, Allen AC, et al. Temporal trends and regional variations in severe maternal morbidity in Canada, 2003 to 2007. J Obstet Gynaecol Can. 2010;32(9):847–55.\nCallaghan WM, Kuklina EV, Berg CJ. Trends in postpartum hemorrhage: United States, 1994–2006. Am J Obstet Gynecol. 2010;202(4):353–e1.\nKnight M, Callaghan WM, Berg C, Alexander S, Bouvier-Colle MH, Ford JB, et al. Trends in postpartum hemorrhage in high resource countries: a review and recommendations from the International Postpartum Hemorrhage Collaborative Group. BMC Pregnancy Childbirth. 2009;9(1):55.\nFuchs KM, Miller RS, Berkowitz RL. Optimizing outcomes through protocols, multidisciplinary drills, and simulation. Semin Perinatol. 2009;33:104–8.\nBrace V, Kernagham D, Penney G. Learning from adverse clinical outcomes: major obstetric haemorrhage in Scotland, 2003–05. BJOG. 2007;114:1388–96.\nJoint Commission. Sentinel event alert, issue 44: preventing maternal death. (January 26, 2010).\nAmerican College of Obstetricians and Gynecologists. ACOG Practice Bulletin No. 76. Postpartum hemorrhage. Obstet Gynecol. 2006;108(4):1039–47.\nCMQCC. Obstetric hemorrhage toolkit: obstetric hemorrhage care guidelines and compendium of best practices reviewed by CDPH-MCAH: 11\u002F24\u002F09. http:\u002F\u002Fwww.cdph.ca.gov\u002Fprograms\u002Fmcah\u002FDocuments\u002FMCAH-OBHemorrhageToolkit.pdf\nRoyal College of Obstetrician and Gynaecologists. Postpartum hemorrhage: prevention and management. 2011.\nHolcomb JB, Jenkins D, Rhee P, Johannigman J, Mahoney P, Mehta S, et al. Damage control resuscitation: directly addressing the early coagulopathy of trauma. J Trauma. 2007;62:307–10.\nBorgman MA, Spinella PC, Perkins JG, Grathwohl KW, Repine T, Beekley AC, et al. The ratio of blood products transfused affects mortality in patients receiving massive transfusions at a combat support hospital. J Trauma. 2007;63(4):805–13.\nHolcomb JB, Wade CE, Michalek JE, Chisholm GB, Zarzabal LA, Schreiber MA, et al. Increased plasma and platelet to red blood cell ratios improves outcome in 466 massively transfused civilian trauma patients. Ann Surg. 2008;248(3):447–58.\nHolcomb JB, Fox EE, Wade CE, PROMMTT Study Group. The Prospective Observational Multicenter Major Trauma Transfusion (PROMMTT) study. J Trauma Acute Care Surg. 2013;75:S1–2.\nDzik WH, Blajchman MA, Fergusson D, Hameed M, Henry B, Kirkpatrick AW, et al. Clinical review: Canadian National Advisory Committee on Blood and Blood Products—massive transfusion consensus conference 2011: report of the panel. Crit Care. 2011;15(6):242.\nMcColl MD, Ramsay JE, Tait RC, Walker ID, McCall F, Conkie JA, et al. Risk factors for pregnancy associated venous thromboembolism. Thromb Haemost. 1997;78(4):1183–8.\nYounis JS, Shukha M, Ben-Ami M, Izhaki I, Blumenfeld Z, Brenner B, et al. Evaluation of protein C global assay during normal pregnancy and after assisted reproduction. Obstet Gynecol. 2010;115:969–74.\nHolmes VA, Wallace JM. Haemostasis in normal pregnancy: a balancing act? Biochem Soc Trans. 2005;33(2):428–32.\nEpiney M, Boehlen F, Boulvain M, Reber G, Antonelli E, Morales M, et al. D‐dimer levels during delivery and the postpartum. J Thromb Haemost. 2005;3(2):268–71.\nHartert H. Blutgerinnungsstudien mit der Thrombelastographie, einem neuen Untersuchungsvefahren. Klin Wochenschr. 1948;26:577–83.\nWeber CF, Görlinger K, Meininger D, Herrmann E, Bingold T, Moritz A, et al. Point-of-care testing: a prospective, randomized clinical trial of efficacy in coagulopathic cardiac surgery patients. Anesthesiology. 2012;117(3):531–47.\nSaner FH, Gieseler RK, Akız H, Canbay A, Görlinger K. Delicate balance of bleeding and thrombosis in end-stage liver disease and liver transplantation. Digestion. 2013;88(3):135–44.\nGoerlinger K, Dirkmann D, Müller-Beissenhirtz H, Paul A, Hartmann M, Saner F. Thromboelastometry-based perioperative coagulation management in visceral surgery and liver transplantation: experience of 10 years and 1105 liver transplants. Liver Transpl. 2010;16(1):S86.\nHuissoud C, Carrabin N, Benchaib M, Fontaine O, Levrat A, Massignon D, et al. Coagulation assessment by rotation thrombelastometry in normal pregnancy. Thromb Haemost. 2009;101(4):755–61.\nde Lange NM, van Rheenen-Flach LE, Lancé MD, Mooyman L, Woiski M, van Pampus EC, et al. Peri-partum reference ranges for ROTEM® thromboelastometry. Br J Anaesth. 2014;112(5):852–9. One of the first articles, which examined ROTEM® thromboelastometry in the peri-partum period. Reference values for ROTEM ® parameters were reported for the 1st time.\nCharbit B, Mandelbrot L, Samain E, Baron G, Haddaoui B, Keita H, et al. The decrease of fibrinogen is an early predictor of the severity of postpartum hemorrhage. J Thromb Haemost. 2007;5(2):266–73.\nHuissoud C, Carrabin N, Audibert F, Levrat A, Massignon D, Berland M, et al. Bedside assessment of fibrinogen level in postpartum haemorrhage by thrombelastometry. BJOG. 2009;116(8):1097–102.\nMallaiah S, Barclay P, Harrod I, Chevannes C, Bhalla A. Introduction of an algorithm for ROTEM-guided fibrinogen concentrate administration in major obstetric haemorrhage. Anaesthesia. 2015;70(2):166–75. This is the first paper, which introduced an algorithm for ROTEM-guided fibrinogen concentrate administration in major obstetric hemorrhage. The study proved that fibrinogen concentrate allowed timely correction of coagulation deficits associated with major obstetric hemorrhage, which, in turn, reduced the requirement for blood component therapy and decreased the risks of recounted complications.\nAbstract. 110th Annual Scientific Meeting of the American Society of Anesthesiologists",{"EN":770},"Massive obstetric hemorrhage is the leading cause of maternal morbidity and direct obstetric death in developed countries. It is also the most frequent reason for maternal ICU admission. Mismanagement of maternal hemorrhage is one of the potentially preventable causes of maternal mortality in the USA. Thromboelastometry (TEM)—previously named rotational thromboelastography (ROTEG) or rotational thromboelastometry (ROTEM)—is an established viscoelastic method for hemostasis testing in whole blood. It investigates the interaction of coagulation factors, their inhibitors, anticoagulant drugs, and blood cells (especially platelets) during clotting and subsequent fibrinolysis. The rotational conditions mimic the sluggish flow of blood in veins. Introduction into clinical practice of point-of-care TEM provides prompt bedside analysis of hemostasis. Utilization of point-of-care TEM allows for the transition from one-size-fits-all fixed-ratio resuscitation to a personalized goal-directed transfusion strategy. Such an individualization of treatment for hemorrhage-related coagulopathy will almost certainly lead to improvements in clinical outcome and a reduced cost of care for patients with massive obstetric hemorrhage.",{"EN":772},"Point-of-Care Thromboelastometry in the Management of Acute Obstetric Hemorrhage",{"VOID":774},"10.1007\u002Fs13669-016-0172-0","2025-01-15T22:33:54.603+00:00","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs13669-016-0172-0",[778,793,810],{"id":779,"sortIndex":189,"researcher":18,"roles":780,"affiliations":781,"properties":790},"74621e75-1b10-47c4-a81b-923da33fc74e",[152],[782],{"id":18,"sortIndex":19,"affiliation":783,"properties":18},{"id":784,"createTime":785,"updateTime":785,"relativeEntities":786,"slug":18,"properties":787,"entityType":38,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19},"9622fb81-a1b1-4411-9943-bd8863203768","2023-12-29T14:22:37.664+00:00",[],{"title":788},{"VI":789},"Clinical Research, Center for Pain Medicine, Western Reserve Hospital, Anesthesiology, Heritage College of Osteopathic Medicine, Ohio University, Cuyahoga Falls, USA",{"title":791},{"VI":792},"Dmitri Souzdalnitski",{"id":794,"sortIndex":92,"researcher":18,"roles":795,"affiliations":796,"properties":807},"3cee8f28-4b4c-425e-8216-2e158e61fed0",[152],[797],{"id":18,"sortIndex":19,"affiliation":798,"properties":18},{"id":799,"createTime":800,"updateTime":801,"relativeEntities":802,"slug":803,"properties":804,"entityType":38,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19},"01251522-69ee-4a30-86c2-a951f4823748","2024-02-08T05:22:00.364+00:00","2025-06-11T14:36:58.288+00:00",[],"Department-of-Anesthesiology-Yale-School-of-Medicine-New-Haven-USA",{"title":805},{"VI":806},"Department of Anesthesiology, Yale School of Medicine, New Haven, USA",{"title":808},{"VI":809},"Zachary Walton",{"id":811,"sortIndex":19,"researcher":18,"roles":812,"affiliations":813,"properties":822},"196a8cd8-8fdd-4f84-a494-102fcf21d079",[152],[814],{"id":18,"sortIndex":19,"affiliation":815,"properties":18},{"id":816,"createTime":817,"updateTime":817,"relativeEntities":818,"slug":18,"properties":819,"entityType":38,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19},"3eb4e696-9b78-48ad-af0f-6e63b9cb3e24","2023-12-29T14:22:37.638+00:00",[],{"title":820},{"VI":821},"Anesthesia Associates of Willimantic, P.C., Willimantic, USA",{"title":823},{"VI":824},"Denis Snegovskikh",{"url":776,"publisher":826,"properties":852},{"id":6,"createTime":7,"updateTime":8,"relativeEntities":827,"slug":10,"properties":828,"entityType":16,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19,"subjectFields":831,"manageAffiliations":832,"indexDatabases":833,"url":77,"thumbnailPath":18,"statistic":847,"gsStatistic":18,"type":124,"analyzePriority":18},[],{"issn":829,"title":830},{"VOID":13},{"EN":15},[],[],[834,841],{"id":42,"indexDatabase":835,"url":57,"indexYears":18,"academicFieldIds":840,"indexDatabaseRanking":18},{"id":44,"createTime":45,"updateTime":46,"relativeEntities":836,"label":837,"description":838,"key":53,"publicationTags":839,"standard":18},[],{"EN":49,"VI":49},{"VI":51,"EN":52},[55,56],[59],{"id":61,"indexDatabase":842,"url":74,"indexYears":75,"academicFieldIds":18,"indexDatabaseRanking":76},{"id":63,"createTime":64,"updateTime":65,"relativeEntities":843,"label":844,"description":845,"key":71,"publicationTags":846,"standard":18},[],{"EN":68,"VI":68},{"EN":68,"VI":70},[73],{"impactFactor":19,"impactFactorByYear":848,"i10Index":89,"i10IndexLast5Year":19,"totalPublication":90,"totalPublicationByYear":849,"totalCitation":104,"totalCitationByYear":850,"totalCitationPerPublication":113,"totalCitationPerPublicationByYear":851,"hindexLast5Year":103,"hindex":103},{"2014":80,"2015":81,"2016":82,"2017":83,"2018":84,"2019":85,"2020":86,"2021":87,"2022":88,"2023":81},{"2011":92,"2012":93,"2013":94,"2014":95,"2015":96,"2016":97,"2017":96,"2018":98,"2019":99,"2020":99,"2021":100,"2022":101,"2023":102,"2024":103},{"2012":106,"2013":107,"2014":92,"2015":108,"2016":109,"2017":110,"2018":111,"2019":106,"2020":98,"2022":112},{"2012":115,"2013":116,"2014":82,"2015":117,"2016":118,"2017":119,"2018":120,"2019":121,"2020":122,"2022":123},{"volume":853,"pages":855},{"VOID":854},"5",{"VOID":856},"244-249","2016-07-05",2016,{"id":860,"createTime":861,"updateTime":862,"relativeEntities":863,"slug":864,"properties":865,"entityType":142,"verifyStatus":143,"verifyTime":862,"verifyNote":144,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19,"primaryUrl":874,"fullTextUrl":18,"authors":875,"publicationType":224,"publisherRelationship":903,"citationCount":18,"citationInfo":18,"publishDate":935,"publishYear":936,"citationAnalyzeStatus":17,"lastCitationAnalyze":18,"indexDatabases":18,"openAccess":18,"references":18,"isForceReanalyzing":255},"9a890b64-24c1-4625-9d46-3a1ebe410e30","2024-02-12T09:48:04.630+00:00","2025-01-17T22:23:48.730+00:00",[],"Urodynamic-Testing-Choosing-Between-Simple-Versus-Complex",{"references":866,"abstract":868,"title":870,"doi":872},{"VOID":867},"Wu JM, Vaughan CP, Goode PS, Redden DT, Burgio KL, Richter HE, et al. Prevalence and trends of symptomatic pelvic floor disorders in U.S. women. Obstet Gynecol. 2014;123(1):141–8. https:\u002F\u002Fdoi.org\u002F10.1097\u002FAOG.0000000000000057.\nWu JM, Hundley AF, Fulton RG, Myers ER. Forecasting the prevalence of pelvic floor disorders in U.S. women: 2010 to 2050. Obstet Gynecol. 2009;114(6):1278–83. https:\u002F\u002Fdoi.org\u002F10.1097\u002FAOG.0b013e3181c2ce96.\n•• Rosier P, Schaefer W, Lose G, Goldman HB, Guralnick M, Eustice S, et al. International continence society good urodynamic practices and terms 2016: urodynamics, uroflowmetry, cystometry, and pressure-flow study. Neurourol Urodyn. 2017;36(5):1243–60. https:\u002F\u002Fdoi.org\u002F10.1002\u002Fnau.23124. The evidence-based International Continence Society standardization and terminology guidelines in order to improve urodynamic testing and reporting for patient care and scientific purposes\nYeung JY, Eschenbacher MA, Pauls RN. Pain and embarrassment associated with urodynamic testing in women. Int Urogynecol J. 2014;25(5):645–50. https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs00192-013-2261-1.\nYiou R, Audureau E, Loche CM, Dussaud M, Lingombet O, Binhas M. Comprehensive evaluation of embarrassment and pain associated with invasive urodynamics. Neurourol Urodyn. 2015;34(2):156–60. https:\u002F\u002Fdoi.org\u002F10.1002\u002Fnau.22521.\nNager CW, Brubaker L, Litman HJ, Zyczynski HM, Varner RE, Amundsen C, et al. A randomized trial of urodynamic testing before stress-incontinence surgery. N Engl J Med. 2012;366(21):1987–97. https:\u002F\u002Fdoi.org\u002F10.1056\u002FNEJMoa1113595.\nvan Leijsen SA, Kluivers KB, Mol BW, Hout J, Milani AL, Roovers JP, et al. Value of urodynamics before stress urinary incontinence surgery: a randomized controlled trial. Obstet Gynecol. 2013;121(5):999–1008. https:\u002F\u002Fdoi.org\u002F10.1097\u002FAOG.0b013e31828c68e3.\nWeidner AC, Myers ER, Visco AG, Cundiff GW, Bump RC. Which women with stress incontinence require urodynamic evaluation? Am J Obstet Gynecol. 2001;184(2):20–7. https:\u002F\u002Fdoi.org\u002F10.1067\u002Fmob.2001.108171.\nClement KD, Lapitan MC, Omar MI, Glazener CM. Urodynamic studies for management of urinary incontinence in children and adults. Cochrane Database Syst Rev. 2013;10:CD003195. https:\u002F\u002Fdoi.org\u002F10.1002\u002F14651858.CD003195.pub3.\nSuskind AM, Cox L, Clemens JQ, Oldendorf A, Stoffel JT, Malaeb B, et al. The value of urodynamics in an academic specialty referral practice. Urology. 2017;105:48–53. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.urology.2017.02.049.\nVerghese TS, Middleton LJ, Daniels JP, Deeks JJ, Latthe PM. The impact of urodynamics on treatment and outcomes in women with an overactive bladder: a longitudinal prospective follow-up study. Int Urogynecol J. 2017; https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs00192-017-3414-4.\nGormley EA, Lightner DJ, Burgio KL, Chai TC, Clemens JQ, Culkin DJ, et al. Diagnosis and treatment of overactive bladder (non-neurogenic) in adults: AUA\u002FSUFU guideline. J Urol. 2012;188(6 Suppl):2455–63. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.juro.2012.09.079.\nCollins CW, Winters JC, American Urological A, Society of Urodynamics Female Pelvic M, Urogenital R. AUA\u002FSUFU adult urodynamics guideline: a clinical review. Urol Clin North Am. 2014;41(3):353–362, vii. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.ucl.2014.04.011.\nHanno PM, Burks DA, Clemens JQ, Dmochowski RR, Erickson D, Fitzgerald MP, et al. AUA guideline for the diagnosis and treatment of interstitial cystitis\u002Fbladder pain syndrome. J Urol. 2011;185(6):2162–70. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.juro.2011.03.064.\nRichter HE, Albo ME, Zyczynski HM, Kenton K, Norton PA, Sirls LT, et al. Retropubic versus transobturator midurethral slings for stress incontinence. N Engl J Med. 2010;362(22):2066–76. https:\u002F\u002Fdoi.org\u002F10.1056\u002FNEJMoa0912658.\nNager CW, Sirls L, Litman HJ, Richter H, Nygaard I, Chai T, et al. Baseline urodynamic predictors of treatment failure 1 year after mid urethral sling surgery. J Urol. 2011;186(2):597–603. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.juro.2011.03.105.\nZyczynski HM, Albo ME, Goldman HB, Wai CY, Sirls LT, Brubaker L, et al. Change in overactive bladder symptoms after surgery for stress urinary incontinence in women. Obstet Gynecol. 2015;126(2):423–30. https:\u002F\u002Fdoi.org\u002F10.1097\u002FAOG.0000000000000929.\n• Jelovsek JE, Hill AJ, Chagin KM, Kattan MW, Barber MD. Predicting risk of urinary incontinence and adverse events after midurethral sling surgery in women. Obstet Gynecol. 2016;127(2):330–40. https:\u002F\u002Fdoi.org\u002F10.1097\u002FAOG.0000000000001269. This is a secondary analysis of four randomized controlled trials of mid-urethral slings investigating possible predictors of developing stress and urgency urinary incontinence and adverse events 12 months after sling surgery\nBrubaker L, Cundiff GW, Fine P, Nygaard I, Richter HE, Visco AG, et al. Abdominal sacrocolpopexy with Burch colposuspension to reduce urinary stress incontinence. N Engl J Med. 2006;354(15):1557–66. https:\u002F\u002Fdoi.org\u002F10.1056\u002FNEJMoa054208.\nWei JT, Nygaard I, Richter HE, Nager CW, Barber MD, Kenton K, et al. A midurethral sling to reduce incontinence after vaginal prolapse repair. N Engl J Med. 2012;366(25):2358–67. https:\u002F\u002Fdoi.org\u002F10.1056\u002FNEJMoa1111967.\nVisco AG, Brubaker L, Nygaard I, Richter HE, Cundiff G, Fine P, et al. The role of preoperative urodynamic testing in stress-continent women undergoing sacrocolpopexy: the Colpopexy and Urinary Reduction Efforts (CARE) randomized surgical trial. Int Urogynecol J Pelvic Floor Dysfunct. 2008;19(5):607–14. https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs00192-007-0498-2.\nNager CW, Albo ME, Fitzgerald MP, McDermott S, Wruck L, Kraus S, et al. Reference urodynamic values for stress incontinent women. Neurourol Urodyn. 2007;26(3):333–40. https:\u002F\u002Fdoi.org\u002F10.1002\u002Fnau.20348.\nBo K, Frawley HC, Haylen BT, Abramov Y, Almeida FG, Berghmans B, et al. An International Urogynecological Association (IUGA)\u002FInternational Continence Society (ICS) joint report on the terminology for the conservative and nonpharmacological management of female pelvic floor dysfunction. Int Urogynecol J. 2017;28(2):191–213. https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs00192-016-3123-4.\nWolf JS Jr, Bennett CJ, Dmochowski RR, Hollenbeck BK, Pearle MS, Schaeffer AJ, et al. Best practice policy statement on urologic surgery antimicrobial prophylaxis. J Urol. 2008;179(4):1379–90. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.juro.2008.01.068.\n•• Norton PA, Nager CW, Brubaker L, Lemack GE, Sirls LT, Holley R, et al. The cost of preoperative urodynamics: a secondary analysis of the ValUE trial. Neurourol Urodyn. 2016;35(1):81–4. https:\u002F\u002Fdoi.org\u002F10.1002\u002Fnau.22684. This is a secondary analysis from the Value of Urodynamic Evaluation (ValUE) trial, a multicenter non-inferiority randomized trial to determine whether a basic office evaluation (OE) is non-inferior in terms of SUI surgery outcomes to office evaluation with addition of urodynamic studies (UDS). This analysis calculates the cost incurred when UDS was performed as a supplement to a basic office evaluation and extrapolates the potential savings of not doing UDS in this patient population on a national basis",{"EN":869},"The purpose of the review is to explore the utility of urodynamic testing and guide readers on how to choose the best between simple and complex urodynamic testing. The literature discussed includes clinical scenarios in which urodynamic testing is usually not necessary, when a simple urodynamic assessment provides sufficient clinical information and when complex testing is particularly helpful in clinical decision-making. The evaluation of a straightforward incontinent woman usually does not require urodynamic testing when planning initial behavioral or pharmacologic therapy. The assessment of stress urinary incontinence and prolapse without incontinence can often be accomplished with simple urodynamics. Refractory or complicated urinary incontinence often necessitates complex urodynamics.",{"EN":871},"Urodynamic Testing: Choosing Between Simple Versus Complex",{"VOID":873},"10.1007\u002Fs13669-017-0229-8","https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs13669-017-0229-8",[876,891],{"id":877,"sortIndex":92,"researcher":18,"roles":878,"affiliations":879,"properties":888},"e52c8784-effd-4df6-904f-de65b04bb44d",[152],[880],{"id":18,"sortIndex":19,"affiliation":881,"properties":18},{"id":882,"createTime":883,"updateTime":883,"relativeEntities":884,"slug":18,"properties":885,"entityType":38,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19},"883f0902-660c-4cd5-8a5e-caf84668f5b3","2023-12-19T12:48:28.691+00:00",[],{"title":886},{"VI":887},"Department of Obstetrics and Gynecology, Medical University of South Carolina, Charleston, USA",{"title":889},{"VI":890},"Steven E. 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JM, Jacobs MV, Manos MM, et al. 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Arch Pediatr Adolesc Med. 2006;160:151–6.",{"doi":1038},"10.1001\u002Farchpedi.160.2.151",{"id":18,"text":1040,"url":18,"identifiers":1041},"Plummer M, Peto J, Franceschi S. Time since first sexual intercourse and the risk of cervical cancer. Int J Canc. 2012;130(11):2638–44.",{"doi":1042},"10.1002\u002Fijc.26250",{"id":18,"text":1044,"url":18,"identifiers":1045},"Discacciati MG, Gimenes F, Pennacchi PC, Faiao-Flores F, Zeferino LC, Derchain SM, et al. MMP-9\u002FRECK imbalance: a mechanism asosociated with high-grade cervical lesions and genital infection by human papillomavirus and Chlamydia trachomatis. Cancer Epidemiol Biomarkers Prev. 2015;24:1539–47.",{"doi":1046},"10.1158\u002F1055-9965.EPI-15-0420",{"id":18,"text":1048,"url":18,"identifiers":1049},"Lehtinen M, Ault KA, Lyytikainen E, Dillner J, Garland SM, Ferris DG, et al. Chlamydia trachomatis infection and risk of cervical intraepithelial neoplasia. Sex Transm Infect. 2011;87:372–6.",{"doi":1050},"10.1136\u002Fsti.2010.044354",{"id":18,"text":1052,"url":18,"identifiers":1053},"WHO Global health sector strategy on sexually transmitted infections 2016–2021. World Health Organization Department of Reproductive Health and Research. 2016.",{},{"id":18,"text":1055,"url":18,"identifiers":1056},"Nonato DR, Alves RR, Ribeiro AA, Saddi VA, Segati KD, Almeida KP, et al. Prevalence and factors associated with coinfection of human papillomavirus and Chlamydia trachomatis in adolescents and young women. Am J Obstetr Gynecol. 2016;215(6):753–9.",{"doi":1057},"10.1016\u002Fj.ajog.2016.07.003",{"id":18,"text":1059,"url":18,"identifiers":1060},"Martel C, Plummer V, Vignat J, Franceschi S. Worldwide burden of cancer attributable to HPV by site, country and HPV type. Int J Cancer. 2017;141(4):664–70.",{"doi":1061},"10.1002\u002Fijc.30716",{"id":18,"text":1063,"url":18,"identifiers":1064},"Guan P, Howell-Jones P, Li N, Bruni L, DeSanjose S, Franceschi S, et al. Human papillomavirus types in 115,789 HPV-positive women: a meta-analysis from cervical infection to cancer. Int J Cancer. 2012;131(10):2349–59.",{"doi":1065},"10.1002\u002Fijc.27485",{"id":18,"text":1067,"url":18,"identifiers":1068},"Zur HH. Papillomaviruses in the causation of human cancers-a brief historical account. Virology. 2009;384:260–5.",{"doi":1069},"10.1016\u002Fj.virol.2008.11.046",{"id":18,"text":1071,"url":18,"identifiers":1072},"Comar M, Monasta L, Seraceni S, Colli C, Luska V, Morassut S, et al. Chlamydia trachomatis and HPV coinfections in HIV negative men from a multi-ethnic area of Northern Italy at high prevalence of cervical malignancies. J Med Virol. 2017;89(9):1654–61.",{"doi":1073},"10.1002\u002Fjmv.24811",{"id":18,"text":1075,"url":18,"identifiers":1076},"Paavonen J. Chlamydia trachomatis infections of the female genital tract: state of the art. Ann Med. 2012;44:18–28.",{"doi":1077},"10.3109\u002F07853890.2010.546365",{"id":18,"text":1079,"url":18,"identifiers":1080},"Zhu H, Shen Z, Luo H, Zhang W, Zhu X. Chlamydia trachomatis infection-associated risk of cervical cancer. Medicine. 2016;95(13):3077.",{"doi":1081},"10.1097\u002FMD.0000000000003077",{"id":18,"text":1083,"url":18,"identifiers":1084},"Jensen KE, Thomsen LT, Schmiedel S, Frederiksen K, Norrild B, van den Brule A, et al. Chlamydia trachomatis and risk of cervical intraepithelial neoplasia grade 3 or worse in women with persistent human papillomavirus infection: a cohort study. Sex Transm Infect. 2014;90:550–5.",{"doi":1085},"10.1136\u002Fsextrans-2013-051431",{"id":18,"text":1087,"url":18,"identifiers":1088},"Anttila T, Saikku P, Koskela P, Bloigu A, Dillner J, Ikaheimo I, et al. Serotypes of Chlamydia trachomatis and risk for development of cervical squamous cell carcinoma. JAMA. 2001;285:47–51.",{"doi":1089},"10.1001\u002Fjama.285.1.47",{"id":18,"text":1091,"url":18,"identifiers":1092},"Tungsrithong N, Kasinpila C, Maneenin C, Namujju PB, Lehtinen M, Anttila A, et al. Lack of significant effects of Chlamydia trachomatis infection on cervical cancer risk in a nested case-control study in North-East Thailand. Asian Pac J Cancer Prev. 2014;15:1497–500.",{"doi":1093},"10.7314\u002FAPJCP.2014.15.3.1497",{"id":18,"text":1095,"url":18,"identifiers":1096},"•• Karim S, Souho T, Benlemlih M, Bennani B. Cervical cancer induction enhancement potential of Chlamydia trachomatis: a systematic review. Curr Microbiol. 2018;75:1667–74. In this recent review and meta analysis, it was found that Chlamydia trachomatis could be an independent predictor for cervical cancer.",{"doi":1097},"10.1007\u002Fs00284-018-1439-7",{"id":18,"text":1099,"url":18,"identifiers":1100},"• Yang X, Siddique A, Khan AA, Wang Q, Malik A, Jan AT, et al. Chlamydia trachomatis infection: their potential implication in the etiology of cervical cancer. J Canc. 2021;12(16):4891–900. In this study, women with Chlamydia trachomatis infections were reported to have an incresed risk for cervical cancer development.",{"doi":1101},"10.7150\u002Fjca.58582",{"id":18,"text":1103,"url":18,"identifiers":1104},"Castle PE, Escoffery C, Schachter J, Rattray C, Schiffman M, Moncada J, et al. Chlamydia trachomatis, herpes simplex virus 2, and human T-cell lymphotrophic virus type 1 are not associated with grade of cervical neoplasia in Jamaican colposcopy patients. Sex Transm Dis. 2003;30:575–80.",{"doi":1105},"10.1097\u002F00007435-200307000-00009",{"id":18,"text":1107,"url":18,"identifiers":1108},"Magalhaes PAF, Miranda CAN, Lima EG, Moizeis RNC, de Lima DBS, Cobucci RNO, et al. Genital tract infection with Chlamydia trachomatis in women attended at a cervical cancer screening program in Northeastern from Brasil. Arch Gynecol Obstet. 2015;291:1095–102.",{"doi":1109},"10.1007\u002Fs00404-014-3514-z",{"id":18,"text":1111,"url":18,"identifiers":1112},"Castellsague X, Pawlita M, Roura E, Margall N, Waterboer T, Bosch FX, et al. Prospective seroepidemiologic study on the role of human papillomavirus and other infections in cervical carcinogenesis: evidence from the EPIC cohort. Int J Cancer. 2014;135:440–52.",{"doi":1113},"10.1002\u002Fijc.28665",{"id":18,"text":1115,"url":18,"identifiers":1116},"Valadan M, Yarandi F, Eftekhar Z, Danvish S, Fathollahi MS, Mirsalehian A. Chlamydia trachomatis and cervical intraepithelial neoplasia in married women in a Middle Eastern community. East Mediterr Health J. 2010;16:304–7.",{"doi":1117},"10.26719\u002F2010.16.3.304",{"id":18,"text":1119,"url":18,"identifiers":1120},"Koskela P, Anttila T, Bjorge T, Brunsvig A, Dillner J, Hakama M, et al. Chlamydia trachomatis infection as a risk factor for invasive cervical cancer. Int J Cancer. 2000;85:35–9.",{"doi":1121},"10.1002\u002F(SICI)1097-0215(20000101)85:1\u003C35::AID-IJC6>3.0.CO;2-A",{"id":18,"text":1123,"url":18,"identifiers":1124},"Smith JS, Bosetti C, Munoz N, Herrero R, Bosch FX, Eluf-Neto C, et al. Chlamydia trachomatis and invasive cervical cancer: a pooled analysis of the IARC multicentric case-control study. Int J Cancer. 2004;111:431–9.",{"doi":1125},"10.1002\u002Fijc.20257",{"id":18,"text":1127,"url":18,"identifiers":1128},"Bhatla N, Puri K, Joseph E, Kriplani A, Iyer VK, Sreenivas V. Association of Chlamydia trachomatis infection with human papillomavirus (HPV) & cervical intraepithelial neoplasia- a pilot study. Indian J Med Res. 2013;137:533–9.",{},{"id":18,"text":1130,"url":18,"identifiers":1131},"Chumduri C, Gurumurthy RK, Zadora PK, Mi Y, Meyer TF. Chlamydia infection promotes host DNA damage and proliferation but impairs the DNA damage response. Cell Host Microbe. 2013;13:746–58.",{"doi":1132},"10.1016\u002Fj.chom.2013.05.010",{"id":18,"text":1134,"url":18,"identifiers":1135},"Kun D, Xiang-lin C, Ming Z, Qi L. Chlamydia inhibits host cell apoptosis by inducing Bag-1 via the MAPK\u002FERK survival pathway. Apoptosis. 2013;18:1083–92.",{"doi":1136},"10.1007\u002Fs10495-013-0865-z",{"id":18,"text":1138,"url":18,"identifiers":1139},"Silva J, Cerqueira F, Medeiros R. Chlamydia trachomatis infection: implications for HPV status and cervical cancer. Arch Gynecol Obstet. 2014;289:715–23.",{"doi":1140},"10.1007\u002Fs00404-013-3122-3",{"id":18,"text":1142,"url":18,"identifiers":1143},"Abreu A, Malaguti N, Souza RP, Uchimura NS, Ferreira EC, Pereira MW, et al. Association of human papillomavirus, Neisseria gonorrhoeae and Chlamydia trachomatis coinfections on the risk of high-grade squamous intraepithelial cervical lesion. Am J Cancer Res. 2016;6(6):1371–83.",{},{"id":18,"text":1145,"url":18,"identifiers":1146},"Escarcega-Tame MA, Lopez-Hurtado M, Escobedo-Guerra MR, Reyes-Maldonado E, Castro-Escarpulli G, Guerra-Infante F. Co-infection between genotypes of the human papillomavirus and Chlamydia trachomatis in Mexican women. Int J STD AIDS. 2020;31(13):1255–62.",{"doi":1147},"10.1177\u002F0956462420947587",{"id":18,"text":1149,"url":18,"identifiers":1150},"Ji Y, Ma XX, Li Z, Peppelenbosch MP, Ma Z, Pan Q. The burden of human papillomavirus and Chlamydia trachomatis coinfection in women: a large cohort study in Inner Mongolia. China J Infect Dis. 2019;219(2):206–14.",{"doi":1151},"10.1093\u002Finfdis\u002Fjiy497",{"id":18,"text":1153,"url":18,"identifiers":1154},"Khan AA, Abuderman A, Ashraf MT, Khan Z. Protein-protein interactions of HPV-Chlamydia trachomatis-human and their potential in cervical cancer. Future Microbiol. 2020;15:509–20.",{"doi":1155},"10.2217\u002Ffmb-2019-0242",{"id":18,"text":1157,"url":18,"identifiers":1158},"Madaan N, Pandhi D, Sharma V, Bhattacharya SN, Guleria K, Mishra K, et al. Association of abnormal cervical cytology with coinfection of human papillomavirus and Chlamydia trachomatis. Indian J Sex Transm Dis AIDS. 2019;40(1):57–63.",{"doi":1159},"10.4103\u002Fijstd.IJSTD_9_19",{"id":18,"text":1161,"url":18,"identifiers":1162},"Mancini F, Vescio F, Mochi S, Accardi L, Bonito P, Ciervo A. HPV and Chlamydia trachomatis coinfection in women with Pap smear abnomality: baseline data of the HPV Pathogen ISS study. Infez Med. 2018;26(2):139–44.",{},{"id":18,"text":1164,"url":18,"identifiers":1165},"• Sangpichai S, Patarapadungkit N, Pientong C, Ekalaksananan T, Chaiwiriyakul S, Thongbor R, et al. Chlamydia tarchomatis infection in high-risk human papillomavirus based on cervical cytology specimen. Asian Pac J Cancer Prev. 2019;20(12):3843–7. This study demonstrated no association between human papillomavirus and Chlamydia trachomatis coinfections with cervical carcinogenesis and abnormal cervical cytology results.",{"doi":1166},"10.31557\u002FAPJCP.2019.20.12.3843",{"id":1168,"createTime":1169,"updateTime":1170,"relativeEntities":1171,"slug":1172,"properties":1173,"entityType":142,"verifyStatus":17,"verifyTime":1170,"verifyNote":1183,"syncStatus":17,"languages":1184,"translateLanguages":18,"viewCount":19,"primaryUrl":1185,"fullTextUrl":18,"authors":1186,"publicationType":224,"publisherRelationship":1216,"citationCount":19,"citationInfo":1243,"publishDate":18,"publishYear":18,"citationAnalyzeStatus":17,"lastCitationAnalyze":18,"indexDatabases":18,"openAccess":18,"references":1245,"isForceReanalyzing":255},"c61eb863-e603-4785-a043-3c7f2a682cf1","2024-04-11T13:36:13.888+00:00","2025-01-22T22:09:07.105+00:00",[],"Surgical-Treatment-of-Adenomyosis",{"keywords":1174,"openalex":1175,"abstract":1177,"title":1179,"doi":1181},{},{"VOID":1176},"W4393090636",{"EN":1178},"\u003Cjats:title>Abstract\u003C\u002Fjats:title>\u003Cjats:sec>\n              \u003Cjats:title>Purpose of Review\u003C\u002Fjats:title>\n              \u003Cjats:p>Adenomyosis is defined as the presence of endometrial tissue within the myometrium. The aim of the review is to describe contemporary surgical approaches for adenomyosis.\u003C\u002Fjats:p>\n            \u003C\u002Fjats:sec>\u003Cjats:sec>\n              \u003Cjats:title>Recent Findings\u003C\u002Fjats:title>\n              \u003Cjats:p>Hysterectomy has been standard practice for the treatment of adenomyosis for many years. However, uterine-sparing interventions have emerged recently allowing patients to retain or even enhance their fertility. If there is no wish for further fertility and no desire for uterine preservation, hysterectomy with bilateral salpingectomy is the gold standard treatment for symptomatic adenomyosis. Otherwise, the objectives of surgery are (a) to remove most (ideally the whole) of the adenomyotic tissue, (b) to preserve the integrity of the endometrial cavity, (c) to reconstruct the uterus, and (d) to preserve the functionality of the ovaries and the tubes. The following surgical methods have been proposed for uterus-sparing treatment of adenomyosis: classical excision of adenomyotic tissue after a single incision of the uterus, wedge resection, double- or triple-flap method, transverse H incision, and the PUSH technique. Post-operative clinical outcomes are in favor of fertility-sparing surgery of adenomyosis. The reduction of dysmenorrhea after uterus-sparing surgery for adenomyosis ranges from 54.6 to 84.6%. The reduction of menorrhagia ranges from 50.0 to 73.7%. The total delivery rate in patients who have undergone any uterus-sparing surgery for adenomyosis is 46.9%.\u003C\u002Fjats:p>\n            \u003C\u002Fjats:sec>\u003Cjats:sec>\n              \u003Cjats:title>Summary\u003C\u002Fjats:title>\n              \u003Cjats:p>In conclusion, hysterectomy has traditionally been the primary treatment for adenomyosis in women. However, contemporary medicine offers several excisional and non-excisional techniques for patients who wish to preserve their fertility.\u003C\u002Fjats:p>\n            \u003C\u002Fjats:sec>",{"EN":1180},"Surgical Treatment of Adenomyosis",{"VOID":1182},"10.1007\u002Fs13669-024-00383-0","Author affiliation is blank",[668],"https:\u002F\u002Flink.springer.com\u002F10.1007\u002Fs13669-024-00383-0",[1187,1196,1205],{"id":1188,"sortIndex":19,"researcher":18,"roles":1189,"affiliations":1190,"properties":1191},"e13fd7d7-e63e-4638-b267-91058e3ac3b1",[],[],{"openalex":1192,"title":1194},{"VOID":1193},"A5094223268",{"EN":1195},"Stavros Bischiniotis",{"id":1197,"sortIndex":189,"researcher":18,"roles":1198,"affiliations":1199,"properties":1200},"d5ee5f52-4bcf-4f41-ac7f-06b8f69510ec",[],[],{"openalex":1201,"title":1203},{"VOID":1202},"A5051998581",{"EN":1204},"Grigoris Grimbizis",{"id":1206,"sortIndex":92,"researcher":18,"roles":1207,"affiliations":1208,"properties":1209},"24f2f5c7-f394-4303-9b80-0112466eb9af",[],[],{"openalex":1210,"orcid":1212,"title":1214},{"VOID":1211},"A5090195995",{"VOID":1213},"https:\u002F\u002Forcid.org\u002F0000-0003-3454-8528",{"EN":1215},"Themistoklis Mikos",{"url":18,"publisher":1217,"properties":18},{"id":6,"createTime":7,"updateTime":8,"relativeEntities":1218,"slug":10,"properties":1219,"entityType":16,"verifyStatus":17,"verifyTime":18,"verifyNote":18,"syncStatus":17,"languages":18,"translateLanguages":18,"viewCount":19,"subjectFields":1222,"manageAffiliations":1223,"indexDatabases":1224,"url":77,"thumbnailPath":18,"statistic":1238,"gsStatistic":18,"type":124,"analyzePriority":18},[],{"issn":1220,"title":1221},{"VOID":13},{"EN":15},[],[],[1225,1232],{"id":42,"indexDatabase":1226,"url":57,"indexYears":18,"academicFieldIds":1231,"indexDatabaseRanking":18},{"id":44,"createTime":45,"updateTime":46,"relativeEntities":1227,"label":1228,"description":1229,"key":53,"publicationTags":1230,"standard":18},[],{"EN":49,"VI":49},{"VI":51,"EN":52},[55,56],[59],{"id":61,"indexDatabase":1233,"url":74,"indexYears":75,"academicFieldIds":18,"indexDatabaseRanking":76},{"id":63,"createTime":64,"updateTime":65,"relativeEntities":1234,"label":1235,"description":1236,"key":71,"publicationTags":1237,"standard":18},[],{"EN":68,"VI":68},{"EN":68,"VI":70},[73],{"impactFactor":19,"impactFactorByYear":1239,"i10Index":89,"i10IndexLast5Year":19,"totalPublication":90,"totalPublicationByYear":1240,"totalCitation":104,"totalCitationByYear":1241,"totalCitationPerPublication":113,"totalCitationPerPublicationByYear":1242,"hindexLast5Year":103,"hindex":103},{"2014":80,"2015":81,"2016":82,"2017":83,"2018":84,"2019":85,"2020":86,"2021":87,"2022":88,"2023":81},{"2011":92,"2012":93,"2013":94,"2014":95,"2015":96,"2016":97,"2017":96,"2018":98,"2019":99,"2020":99,"2021":100,"2022":101,"2023":102,"2024":103},{"2012":106,"2013":107,"2014":92,"2015":108,"2016":109,"2017":110,"2018":111,"2019":106,"2020":98,"2022":112},{"2012":115,"2013":116,"2014":82,"2015":117,"2016":118,"2017":119,"2018":120,"2019":121,"2020":122,"2022":123},{"total":19,"publishYear":18,"statisticByYear":1244},{},[1246,1250,1253,1257,1261,1265,1269,1273,1277,1281,1284,1288,1292,1296,1299,1303,1307,1311,1315,1319,1323,1327,1331,1335],{"id":18,"text":1247,"url":18,"identifiers":1248},"Gaba ND, Polite FG, Keller JM, Young AE. 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