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Intensive care as a positive place to work: workforce wellbeing best practice framework London: Intensive Care Society; 2021. https:\u002F\u002Fwww.cc3n.org.uk\u002Fuploads\u002F9\u002F8\u002F4\u002F2\u002F98425184\u002Fworkforce_wellbeing_best_practice_framework_-_fina.pdf.\nPritchard A. 2022\u002F23 priorities and operational planning guidance London: NHS England and NHS Improvement; 2021. https:\u002F\u002Fwww.england.nhs.uk\u002Fwp-content\u002Fuploads\u002F2021\u002F12\u002FB1160-2022-23-priorities-and-operational-planning-guidance.pdf?utm_campaign=2060619_Political%20Update%2007%2F01%2F2022&utm_medium=dotmailer&utm_source=emailmarketing&dm_i=4D4N,185ZF,1181T7,5N4EQ,1.\nWilliams MT. Microaggressions: clarification, evidence, and impact. Perspect Psychol Sci. 2020;15(1):3–26.\nBaum F, Freeman T, Musolino C, Abramovitz M, De Ceukelaire W, Flavel J, et al. Explaining covid-19 performance: What factors might predict national responses? BMJ. 2021;372: n91.",{"EN":841},"Resilience is ubiquitous in everyday speech, academic literature and governmental policies. Yet it seems to have taken a narrow scope in healthcare, confined to individual and psychological resilience. This short essay aims to broaden the understanding of resilience to organisational levels and calls intensivists to take active roles in fostering resilience for their staff. The article explores firstly the background and etymology of resilience. It then challenges current approaches and briefly signposts some current work in the area. Some examples of structural factors which build individual resilience are listed, followed by a call for intensivists to take active roles to build future resilience. The need for interdisciplinary, cross-sectoral and multi-level approaches is vital to build future healthcare resilience, and we intensivists must continue to be advocates for systemic change.",{"EN":843},"Resilience is a dirty word: misunderstood, and how we can truly build it",{"VOID":845},"10.1186\u002Fs13054-022-04040-x","https:\u002F\u002Fccforum.biomedcentral.com\u002Farticles\u002F10.1186\u002Fs13054-022-04040-x",[848],{"id":849,"sortIndex":21,"researcher":20,"roles":850,"affiliations":851,"properties":870},"bc56f090-78d7-46c8-9ad7-0441ee8f9572",[218],[852,862],{"id":853,"sortIndex":250,"affiliation":854,"properties":861},"b5bf4dcf-d247-4c59-bdb8-1c5f19cb3ed8",{"id":855,"createTime":856,"updateTime":856,"relativeEntities":857,"slug":20,"properties":858,"entityType":40,"verifyStatus":19,"verifyTime":20,"verifyNote":20,"syncStatus":19,"languages":20,"translateLanguages":20,"viewCount":21},"df7ac40b-4c9a-4d26-b349-4b518b50e023","2024-01-27T23:59:14.264+00:00",[],{"title":859},{"VI":860},"National Institute for Health and care Research (NIHR) Academic Clinical Fellow, Humanitarian and Conflict Response Institute, University of Manchester, Manchester, UK",{},{"id":20,"sortIndex":21,"affiliation":863,"properties":20},{"id":864,"createTime":865,"updateTime":865,"relativeEntities":866,"slug":20,"properties":867,"entityType":40,"verifyStatus":19,"verifyTime":20,"verifyNote":20,"syncStatus":19,"languages":20,"translateLanguages":20,"viewCount":21},"670b5ae4-9955-438a-b5df-35e3902848cb","2024-01-27T23:59:14.259+00:00",[],{"title":868},{"VI":869},"ST6 Anaesthesia and Intensive Care Medicine, Health Education Northwest, Manchester, UK",{"title":871},{"VI":872},"Mark Z. 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Implications of gastrointestinal manifestations of COVID-19. Lancet Gastroenterol Hepatol 2020 Published Online May 12, 2020 \nhttps:\u002F\u002Fdoi.org\u002F10.1016\u002FS2468-1253(20)30132-1\n\n.",{"doi":997},"10.1016\u002FS2468-1253(20)30132-1",{"id":20,"text":999,"url":20,"identifiers":1000},"Henry BM, de Oliveira MHS, Benoit J, Lippi G. Gastrointestinal symptoms associated with severity of coronavirus disease 2019 (COVID-19): a pooled analysis. Intern Emerg Med. 2020. \nhttps:\u002F\u002Fdoi.org\u002F10.1007\u002Fs11739-020-02329-9\n\n.",{"doi":1001},"10.1007\u002Fs11739-020-02329-9",{"id":20,"text":1003,"url":20,"identifiers":1004},"Shi X, Gong E, Gao D, Zhang B, Zheng J, Gao Z, et al. Severe acute respiratory syndrome associated coronavirus is detected in intestinal tissues of fatal cases. Am J Gastroenterol. 2005;100(1):169–76.",{"doi":1005},"10.1111\u002Fj.1572-0241.2005.40377.x",{"id":20,"text":1007,"url":20,"identifiers":1008},"Zhou J, Li C, Zhao G, Chu H, Wang D, Yan HH, et al. Human intestinal tract serves as an alternative infection route for Middle East respiratory syndrome coronavirus. Sci Adv. 2017;3(11):eaao4966. \nhttps:\u002F\u002Fdoi.org\u002F10.1126\u002Fsciadv.aao4966\n\n.",{"doi":1009},"10.1126\u002Fsciadv.aao4966",{"id":20,"text":1011,"url":20,"identifiers":1012},"Darnell MER, Subbarao K, Feinstone SM, Taylor DR. Inactivation of the coronavirus that induces severe acute respiratory syndrome, SARS-Co-1. J Virol Methods. 2004;121(1):85–91.",{"doi":1013},"10.1016\u002Fj.jviromet.2004.06.006",{"id":20,"text":1015,"url":20,"identifiers":1016},"Freedberg DE, Lebwohl B, Abrams JA. The impact of proton pump inhibitors on the human gastrointestinal microbiome. Clin Lab Med. 2014;34(4):771–85. \nhttps:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.cll.2014.08.008\n\n.",{"doi":1017},"10.1016\u002Fj.cll.2014.08.008",{"id":1019,"createTime":1020,"updateTime":1021,"relativeEntities":1022,"slug":1023,"properties":1024,"entityType":210,"verifyStatus":211,"verifyTime":1021,"verifyNote":212,"syncStatus":19,"languages":20,"translateLanguages":20,"viewCount":21,"primaryUrl":1033,"fullTextUrl":20,"authors":1034,"publicationType":261,"publisherRelationship":1050,"citationCount":20,"citationInfo":20,"publishDate":1083,"publishYear":1084,"citationAnalyzeStatus":19,"lastCitationAnalyze":20,"indexDatabases":20,"openAccess":20,"references":20,"isForceReanalyzing":298},"473b0b29-69f0-4998-9c1e-802ca8f7b154","2023-12-17T22:24:28.351+00:00","2025-01-15T23:59:05.037+00:00",[],"Antithrombin-in-sepsis-revisited",{"references":1025,"abstract":1027,"title":1029,"doi":1031},{"VOID":1026},"Kountchev J, Bijuklic K, Bellmann R, Wiedermann CJ, Joannidis M: Reduction of D-dimer levels after therapeutic administration of antithrombin in acquired antithrombin deficiency of severe sepsis. Crit Care 2005, 9: R596-R600. 10.1186\u002Fcc3808\nEsmon CT: Role of coagulation inhibitors in inflammation. Thromb Haemost 2001, 86: 51-56.\nLevi M, de Jonge E, van der Poll T: Rationale for restoration of physiological anticoagulant pathways in patients with sepsis and disseminated intravascular coagulation. Crit Care Med 2001,29(Suppl 7):S90-94. 10.1097\u002F00003246-200107001-00028\nMesters RM, Mannucci PM, Coppola R, Keller T, Ostermann H, Kienast J: Factor VIIa and antithrombin III activity during severe sepsis and septic shock in neutropenic patients. Blood 1996, 88: 881-886.\nFourrier F, Chopin C, Goudemand J, Hendrycx S, Caron C, Rime A, Narey P, Lestavel P: Septic shock, multiple organ failure, and disseminated intravascular coagulation. Compared patterns of antithrombin III, protein C, and protein S deficiencies [see comments]. Chest 1992, 101: 816-823.\nMinnema MC, Chang AC, Jansen PM, Lubbers YT, Pratt BM, Whittaker BG, Taylor FB, Hack CE, Friedman B: Recombinant human antithrombin III improves survival and attenuates inflammatory responses in baboons lethally challenged with Escherichia coli. Blood 2000, 95: 1117-1123.\nLevi M, van der Poll T, Buller HR: Bidirectional relation between inflammation and coagulation. Circulation 109: 2698-2704. 10.1161\u002F01.CIR.0000131660.51520.9A\nBernard GR, Vincent JL, Laterre PF, LaRosa SP, Dhainaut JF, Lopez-Rodriguez A, Steingrub JS, Garber GE, Helterbrand JD, Ely EW, Fisher CJ Jr, Recombinant human protein C Worldwide Evaluation in Severe Sepsis (PROWESS) study group: Efficacy and safety of recombinant human activated protein C for severe sepsis. N Engl J Med 2001, 344: 699-709. 10.1056\u002FNEJM200103083441001\nLevi M, ten Cate H, van der Poll T: Disseminated intravascular coagulation: State of the art. Thromb Haemost 1999, 82: 695-705.\nWarren BL, Eid A, Singer P, Pillay SS, Carl P, Novak I, Chalupa P, Penzes I, Kubler A, Knaul S, et al.: Caring for the critically ill patient. High-dose antithrombin III in severe sepsis: a randomized controlled trial. J Am Med Assoc 2001, 286: 1869-1878. 10.1001\u002Fjama.286.15.1869\nBlauhut B, Kramar H, Vinazzer H, Bergmann H: Substitution of antithrombin III in shock and DIC: a randomized study. Thromb Res 1985, 39: 81-89. 10.1016\u002F0049-3848(85)90123-9\nBernard GR, Ely EW, Wright TJ, Stasek JE, Russell JA, Norris PE, Yau SB, Helterbrand JD: Safety and dose relationship of recombinant human activated protein C for coagulopathy in severe sepsis. Crit Care Med 2001, 29: 2051-2059. 10.1097\u002F00003246-200111000-00003\nTaylor FBJ, Toh CH, Hoots WK, Wada H, Levi M: Towards definition, clinical and laboratory criteria, and a scoring system for disseminated intravascular coagulation. Thromb Haemost 2001, 86: 1327-1330.\nDhainaut JF, Yan SB, Joyce DE, Petilla V, Busson BR, Brauelt JT, Saudin D, Levi M: Treatment effects of drotrecogin alfa (activated) in patients with severe sepsis with or without overt disseminated intravascular coagulation. J Thromb Haemost 2004, 2: 1924-1933. 10.1111\u002Fj.1538-7836.2004.00955.x",{"EN":1028},"As the pivotal phase III randomized controlled clinical trial on antithrombin concentrate in patients with severe sepsis did not show a beneficial effect of antithrombin treatment on 28-day mortality, the interest in the potential use of this treatment modality in sepsis has diminished. 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