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Journal of Medicine and Pharmacy","Tạp chí Y Dược học Cần Thơ",{"EN":487,"VI":488},"\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">04\u002F10\u002F2015 Ministry of Information and Communications allowed Can Tho journal of medicine and pharmacy to operate (102 \u002FGP-BTTTT)\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">07\u002F16\u002F2015 Can Tho journal of medicine and pharmacy is internationally recognized: ISSN 2354-1210\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">In 2016, The journal has been included in the list of medical science journals by The State Council for professorship which is awarded a work score of 0-0.5 points for a published article.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Can Tho Journal of Medicine and Pharmacy welcome original works that haven’t been submitted or published in other medical journals. Posts must contain content related to one of the journal’s categories.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The content published\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The journal is divided into 3 categories:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Scientific research article: are valuable scientific works, which have been researched and accepted.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Overview of medicine, biology and pharmacy: serving the objective of continuing training in the fields of medicine, biology and pharmacy; to systematize classical and modern knowledge.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Update information on new knowledge about medicine, biology, pharmacy in the country and in the world.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Scope\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Publication and introduction of scientific research in the fields:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Medicine (internal medicine, surgery, pediatrics, obstetrics and gynecology, odonto-stomatology, laboratory, oncology, traditional medicine, nursing).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Biology (genetics, biotechnology).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Pharmacology (pharmaceutics, drug quality analysis-control, synthetic pharmaceutical chemistry, biochemistry, pharmacognosy, botany, clinical pharmacy).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- To enhance the quality of undergraduate, postgraduate education, scientifically researching and meet the necessary treatment in hospital.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Introducing the updated domestic and oversea information about science technology to promote scientific research and exchanging technology in local, other universities.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Exchanging pharmaceutical and medical information for social health developing in the Mekong Delta and Vietnam.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The object\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Postgraduate students, student of Can Tho University of Medicine and Pharmacy, scientists from schools, research institutes, hospitals, health centers, pharmaceutical companies of the Mekong Delta; other provinces and regions in Vietnam and other country.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Address\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Headquarters of Can Tho Journal of Medicine and Pharmacy, located Scientific Research and International Cooperation Office: 179 Nguyen Van Cu Street, An Khanh Ward, Ninh Kieu District, Can Tho City, Vietnam.\u003C\u002Fspan>\u003C\u002Fp>","\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Ngày 16\u002F7\u002F2015, Tạp chí Y Dược học Cần Thơ được cấp chỉ số quốc tế: ISSN 2354-1210.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 4\u002F2016, Tạp chí đã được Hội đồng Giáo sư ngành Y đưa vào danh sách các tạp chí khoa học Y học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Năm 2020 Tạp chí Y Dược học Cần Thơ đã được phê duyệt vào danh mục của các Hội đồng Giáo sư ngành Dược học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ ra 12 số\u002Fnăm, 180-200 trang\u002Fsố.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 12\u002F2022 Tạp chí Y Dược học Cần Thơ là thành viên của hệ thống Crossref và từ tháng 01\u002F2023 tạp chí thực hiện bình duyệt online kín 2 chiều nhằm tăng tính minh bạch, tin cậy của các công trình nghiên cứu khoa học và đảm bảo tốt nhất chất lượng khoa học của bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ, mục đích và phạm vi của tạp chí\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ và mục đích hoạt động của tạp chí: xuất bản nhằm mục đích phổ biến kết quả từ các đề tài nghiên cứu khoa học; giao lưu trao đổi khoa học, chia sẻ kinh nghiệm, học tập, đồng thời cập nhật thông tin khoa học mới trong các lĩnh vực y, sinh, dược học trong và ngoài nước.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phạm vi của tạp chí: Tạp chí xuất bản được chia thành 3 chuyên mục: (i) Bài báo nghiên cứu khoa học là kết quả công trình nghiên cứu khoa học có giá trị đã được triển khai nghiên cứu, (ii) Bài tổng quan y, sinh, dược học: phục vụ mục tiêu đào tạo liên tục trong lĩnh vực y, sinh, dược học; nhằm hệ thống hóa những kiến thức kinh điển và hiện đại; (iii) Thông tin cập nhật kiến thức mới về y, sinh, dược học trong nước và trên thế giới.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Chính sách truy cập mở\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ áp dụng chính sách truy cập mở đối với các bài báo đã xuất bản đến với độc giả, nhằm mở rộng cơ hội tiếp cận các kết quả nghiên cứu chất lượng cao và tăng cường trao đổi kiến thức. Tạp chí đăng tải trực tuyến (miễn phí) toàn văn các bài báo được công bố trên website của Tạp chí (https:\u002F\u002Ftapchi.ctump.edu.vn).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đạo đức xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ cam kết tuân thủ đạo đức xuất bản phù hợp với các hướng dẫn và tiêu chuẩn của the Committee on Publication Ethics (COPE), tuân thủ các nguyên tắc của COPE’s Core Practices, Best Practices Guidelines for Journal Editors và Guidelines on Good Publication Practices.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Bản thảo bài báo chỉ được chấp nhận khi được tác giả chịu trách nhiệm chính cam kết các nội dung sau: Các nội dung của bản thảo chưa được đăng tải toàn bộ hoặc một phần ở các tạp chí khác; Tất cả các tác giả đều có đóng góp một cách đáng kể vào quá trình nghiên cứu hoặc chuẩn bị bản thảo và cùng chịu trách nhiệm về các nội dung của bản thảo; Tuân thủ các biện pháp đảm bảo đạo đức nghiên cứu (ví dụ thỏa thuận đồng ý tham gia nghiên cứu).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Cam kết bảo mật\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí cam kết thực hiện và tuân thủ các quy định của luật và các văn bản hướng dẫn liên quan đến bảo mật thông tin cá nhân trên không gian mạng. Các thông tin mà người dùng (tác giả, độc giả, biên tập viên, người phản biện) nhập vào các biểu mẫu trên Hệ thống Quản lý xuất bản trực tuyến của tạp chí chỉ được sử dụng vào các mục đích đã được tuyên bố rõ ràng và sẽ không được cung cấp cho bất kỳ bên thứ ba nào khác, hay dùng vào bất kỳ mục đích nào khác.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phí gửi bài\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng bài: 1.000.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng nhanh: 1.500.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với tác giả là cán bộ viên chức thuộc Trường Đại học Y Dược Cần Thơ thì được hỗ trợ 50% lệ phí gửi đăng bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với sinh viên thực hiện đề tài nghiên cứu khoa học cấp trường được hỗ trợ 100% lệ phí đăng bài ( Tác giả gửi đính kèm “ Quyết định về việc giao tổ chức thực hiện đề tài nghiên cứu khoa học cấp Trường của sinh viên”).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Hình thức nộp lệ phí:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Tiền mặt:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Nộp trực tiếp tại Phòng Tài chính - Kế toán, Trường Đại học Y Dược Cần Thơ, số 179 Nguyễn Văn Cừ, P. An Khánh, Q. Ninh Kiều, thành phố Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Chuyển khoản:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tên Tài khoản: Trường ĐHYD Cần Thơ, Số TK: 0111000115668, tại ngân hàng Vietcombank chi nhánh Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Thời gian: Áp dụng từ ngày 01\u002F02\u002F2023.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">* Phí gửi bài không được hoàn trả khi bài viết bị từ chối hoặc tác giả xin rút bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Quy trình phản biện bài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ thực hiện quy trình phản biện kín hai chiều nghiêm ngặt. Danh tính của những người phản biện không được tiết lộ cho các tác giả và ngược lại. Quy trình thẩm định bài báo đăng gồm các bước sau:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tiếp nhận bản thảo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tác giả liên hệ gửi bản thảo đến Tạp chí qua hệ thống trực tuyến tại website: https:\u002F\u002Ftapchi.ctump.edu.vn. Hướng dẫn về cách đăng ký, gửi bài và chuẩn bị bản thảo được cung cấp trên website của Tạp chí.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sàng lọc sơ bộ\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sau khi Tòa soạn nhận được bài báo của tác giả, Ban Thư ký sẽ tiến hành kiểm tra sơ bộ bài báo (các yêu cầu về nội dung và hình thức). Những bài báo không đúng quy cách hoặc có nội dung không phù hợp hoặc vi phạm bản quyền sẽ bị từ chối (Ban Thư ký thông báo phản hồi đến tác giả trong vòng 1 tuần). Những bài báo đủ điều kiện, được Ban Thư ký tòa soạn chuyển đến Ban Biên tập có cùng chuyên môn với nội dung bài báo để đề xuất người phản biện. Thời gian kể từ khi Ban Biên tập nhận bài báo đến khi đề xuất người phản biện bài báo chậm nhất là 5 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Vòng phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký gửi bài và yêu cầu phản biện đến 02 phản biện độc lập.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Các phản biện gởi nhận xét cho Ban Thư ký. Thời gian từ khi gửi bài cho phản biện đến khi nhận ý kiến của phản biện tối đa là 20 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xử ký kết quả phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Nếu ý kiến đồng ý cho đăng và không cần chỉnh sửa, Ban Thư ký tiếp tục đăng bài theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Nếu ý kiến đồng ý đăng và cần chỉnh sửa, Ban Thư ký sẽ thông tin đến tác giả chỉnh sửa theo yêu cầu của người phản biện. Thời gian chỉnh sửa và gửi lại kéo dài không quá 2 tuần, từ khi tác giả bài báo nhận được thông tin (Quá trình này có thể lặp lại tối đa 2 lần\u002F1 bài báo). Khi có sự thống nhất, đồng ý của người phản biện; bài báo được tiếp tục đăng theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Những bài báo có chất lượng không đạt yêu cầu, cả 2 phản biện không đồng ý cho đăng sẽ bị Tòa soạn từ chối đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký tổng hợp các bản thảo đã được tác giả hoàn thiện sau thẩm định trình Ban Biên tập xem xét, Tổng Biên tập phê duyệt, quyết định bài đăng theo các tiêu chí: sự phù hợp nội dung với tôn chỉ và mục đích, thể loại bài viết (ưu tiên các bài có bài có nghiên cứu chuyên sâu, hàm lượng khoa học cao), đóng góp mới bài báo, bài báo được ưu tiên đăng trong số gần nhất của Tạp chí theo thứ tự: tính thời sự, chất lượng bài báo và thời gian gửi bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Ban Biên tập và Ban Thư ký biên tập bản thảo, chế bản, đọc rà soát lỗi. Thời gian hoàn thành từ 10-15 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Ban Thư ký có trách nhiệm thông báo cho tác giả bài báo (bằng e-mail) về tình hình phê duyệt bài báo, thời gian, số kỳ, tập xuất bản bài báo theo qui định.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">4. Danh sách bài báo theo số Tạp chí được in ấn và phát hành trong năm định kỳ được công bố chính thức trên website: https:\u002F\u002Ftapchi.ctump.edu.vn\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>",{"VOID":490},"wcQ1uqwAAAAJ","2023-05-30T08:17:21.868+00:00",[],[494],{"id":495,"createTime":28,"updateTime":28,"relativeEntities":496,"slug":28,"properties":497,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":507,"parentIds":508,"statistic":28},"6413896b-eca9-442b-a73f-182a58a0ce40",[],{"title":498,"address":501,"country":504,"abbreviation":505},{"EN":499,"VI":500},"Can Tho University of Medicine and Pharmacy","Trường Đại học Y Dược Cần Thơ",{"EN":502,"VI":503},"No 179, Nguyen Van Cu street, An Khanh ward, Ninh Kieu district, Can Tho city, Vietnam","Số 179, đường Nguyễn Văn Cừ, phường An Khánh, quận Ninh Kiều, thành phố Cần Thơ, Việt Nam",{"VOID":15},{"VOID":506},"ctump","http:\u002F\u002Fwww.ctump.edu.vn\u002F",[],[],"https:\u002F\u002Ftapchi.ctump.edu.vn\u002Findex.php\u002Fctump",{"impactFactor":32,"impactFactorByYear":512,"i10Index":32,"i10IndexLast5Year":32,"totalPublication":514,"totalPublicationByYear":515,"totalCitation":520,"totalCitationByYear":521,"totalCitationPerPublication":108,"totalCitationPerPublicationByYear":523,"hindexLast5Year":45,"hindex":45},{"2022":513,"2023":111,"2024":106},0.01,1556,{"2020":47,"2021":516,"2022":517,"2023":518,"2024":519,"2025":122},57,306,801,358,161,{"2021":146,"2022":280,"2023":522},99,{"2021":524,"2022":318,"2023":104},0.23,{"impactFactor":28,"impactFactorByYear":28,"i10Index":123,"i10IndexLast5Year":123,"totalPublication":526,"totalPublicationByYear":527,"totalCitation":526,"totalCitationByYear":528,"totalCitationPerPublication":40,"totalCitationPerPublicationByYear":531,"hindexLast5Year":49,"hindex":49},476,{"0":205,"2019":123,"2021":139,"2022":459,"2023":451,"2024":357,"2025":49,"2026":48},{"2021":42,"2022":123,"2023":161,"2024":529,"2025":360,"2026":530},136,83,{"2021":105,"2022":513,"2023":532,"2024":127,"2025":533,"2026":534},0.62,25.43,13.83,{"id":536,"createTime":537,"updateTime":382,"relativeEntities":538,"slug":539,"properties":540,"entityType":25,"verifyStatus":26,"verifyTime":28,"verifyNote":28,"languages":552,"translateLanguages":28,"viewCount":133,"subjectFields":553,"manageAffiliations":554,"indexDatabases":555,"url":556,"thumbnailPath":557,"statistic":558,"gsStatistic":594,"type":55,"analyzePriority":28},"6984a56a-db70-403b-9cc4-4013e1ceaffa","2023-05-09T06:47:40.346+00:00",[],"T%E1%BA%A1p%20ch%C3%AD%20Nghi%C3%AAn%20c%E1%BB%A9u%20n%C6%B0%E1%BB%9Bc%20ngo%C3%A0i",{"country":541,"issn":542,"title":544,"introduce":547,"gsId":550},{"VOID":15},{"VOID":543},"25252445",{"EN":545,"VI":546},"VNU Journal of Foreign Studies","Tạp chí Nghiên cứu nước ngoài",{"EN":548,"VI":549},"{\"ops\":[{\"insert\":\"\\n\\nThe \\n\"},{\"attributes\":{\"italic\":true},\"insert\":\"VNU Journal of Science\"},{\"insert\":\"\\n was established in 1985 for the publication of national and international research papers in all fields of natural sciences and technology, social sciences and humanities. 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Flow to the major masses of brown adipose tissue (BAT) increased in WA rats from a mean of 0.81 ml\u002Fmin (0.92% of CO) at rest to 13.5 ml\u002Fmin (11.4% of CO) during calorigenesis; it increased in CA rats from 2.3 ml\u002Fmin (2.6% of CO) to 57.2 ml\u002Fmin (33.5% of CO). Flow to skeletal muscle increased in WA rats from 12.0 ml\u002Fmin at rest to 15.1 ml\u002Fmin during calorigenesis; it increased in CA rats from 9.9 ml\u002Fmin to 14.5 ml\u002Fmin. Flow to heart and to muscles involved in respiratory movements was two to five times greater during calorigenesis. Flow to most other tissues and organs increased or decreased by less than 40%.Arteriovenous differences in blood oxygen [Formula: see text] across interscapular BAT (IBAT) during rest and during calorigenesis together with measurements of blood flow established that IBAT alone accounted for 14% of the extra O\u003Cjats:sub>2\u003C\u002Fjats:sub> used by CA rats during NA-induced calorigenesis. If during calorigenesis other masses of BAT have an [Formula: see text] as great as that for IBAT, the major masses of BAT together would account for 60% of the calorigenic response of the CA rat. In contrast, even if the skeletal muscle of the CA rat used all the O\u003Cjats:sub>2\u003C\u002Fjats:sub> in the blood flowing through it during calorigenesis, it could not have been responsible for more than 12% of the calorigenic response.The rat, long considered to exemplify major participation of skeletal muscle in nonshivering thermogenesis (NST), now becomes just one of a growing list of species for which there is explicit or circumstantial evidence that NST occurs principally in BAT. It thus becomes reasonable to propose as a general principle that BAT is the primary anatomical site of the NST that is characteristic of many small mammals: CA adults, newborns, and hibernators alike. \u003C\u002Fjats:p>","\u003Cjats:p> Lưu lượng tim (CO) và phân phối tỷ lệ (FD) của các vi cầu nhựa được gán nhãn γ (15 ± 5 μm) tiêm vào tâm thất trái được sử dụng để tính lưu lượng máu đến các cơ quan và mô của chuột trắng được an thần với barbital, được thích nghi với nhiệt độ ấm (WA) hoặc lạnh (CA) khi nghỉ ngơi và sau đó trong phản ứng sinh nhiệt tối đa của chúng với noradrenaline (NA) được truyền vào. Lưu lượng đến các khối mô mỡ nâu chính (BAT) đã tăng ở chuột WA từ trung bình 0.81 ml\u002Fphút (0.92% của CO) khi nghỉ ngơi lên 13.5 ml\u002Fphút (11.4% của CO) trong quá trình sinh nhiệt; nó đã tăng ở chuột CA từ 2.3 ml\u002Fphút (2.6% của CO) lên 57.2 ml\u002Fphút (33.5% của CO). Lưu lượng đến cơ bắp đã tăng ở chuột WA từ 12.0 ml\u002Fphút khi nghỉ ngơi lên 15.1 ml\u002Fphút trong quá trình sinh nhiệt; nó đã tăng ở chuột CA từ 9.9 ml\u002Fphút lên 14.5 ml\u002Fphút. Lưu lượng đến tim và các cơ tham gia vào chuyển động hô hấp cao gấp hai đến năm lần trong quá trình sinh nhiệt. Lưu lượng đến phần lớn các mô và cơ quan khác tăng hoặc giảm dưới 40%. Sự chênh lệch tĩnh mạch động mạch về oxy trong máu [Công thức: xem văn bản] qua mô mỡ nâu giữa bả vai (IBAT) trong quá trình nghỉ ngơi và trong quá trình sinh nhiệt cùng với các phép đo lưu lượng máu cho thấy rằng IBAT chiếm 14% O\u003Cjats:sub>2\u003C\u002Fjats:sub> dư thừa mà chuột CA sử dụng trong quá trình sinh nhiệt do NA gây ra. Nếu trong quá trình sinh nhiệt các khối mô mỡ nâu khác có [Công thức: xem văn bản] cũng lớn như IBAT, các khối mô mỡ nâu chính cùng lại sẽ chiếm 60% phản ứng sinh nhiệt của chuột CA. Ngược lại, ngay cả khi cơ bắp của chuột CA sử dụng tất cả O\u003Cjats:sub>2\u003C\u002Fjats:sub> trong máu chảy qua nó trong quá trình sinh nhiệt, nó không thể chịu trách nhiệm cho hơn 12% phản ứng sinh nhiệt. Chuột, từ lâu được coi là ví dụ điển hình cho sự tham gia chính của cơ bắp trong nhiệt sinh học không run (NST), giờ đây trở thành một trong số ngày càng nhiều loài cho thấy bằng chứng rõ ràng hoặc gián tiếp rằng NST chủ yếu xảy ra ở BAT. Do đó, điều này là hợp lý khi đề xuất như một nguyên tắc chung rằng BAT là vị trí giải phẫu chính của NST mà nhiều loài động vật có vú nhỏ như: Cá thể CA, trẻ sơ sinh và động vật ngủ đông đều có đặc điểm.",{"EN":980,"VI":981},"Nonshivering thermogenesis in the rat. II. Measurements of blood flow with microspheres point to brown adipose tissue as the dominant site of the calorigenesis induced by noradrenaline","Nhiệt sinh học không run ở chuột. II. Các phép đo lưu lượng máu bằng vi cầu chỉ ra mô mỡ nâu là vị trí chiếm ưu thế của sự sinh nhiệt do noradrenaline gây ra",{"VOID":983},"638848",{"VOID":985},"10.1139\u002Fy78-015","PUBLICATION","Author affiliation is blank",[31],[30],"http:\u002F\u002Fwww.nrcresearchpress.com\u002Fdoi\u002F10.1139\u002Fy78-015",[992,1001],{"id":993,"sortIndex":32,"researcher":28,"roles":994,"affiliations":995,"properties":996,"displayName":998,"givenName":28,"familyName":28},"fbee3f29-8d72-403a-9ec9-5fab88617f43",[],[],{"title":997,"openalex":999},{"EN":998},"David O. Foster",{"VOID":1000},"A5052118573",{"id":1002,"sortIndex":40,"researcher":28,"roles":1003,"affiliations":1004,"properties":1005,"displayName":1007,"givenName":28,"familyName":28},"c04e789e-c57f-4709-a6fb-798f97d8f0d3",[],[],{"title":1006,"openalex":1008},{"EN":1007},"M. Lorraine Frydman",{"VOID":1009},"A5044714389","ARTICLE",{"url":28,"publisher":1012,"properties":1062},{"id":868,"createTime":869,"updateTime":870,"relativeEntities":1013,"slug":872,"properties":1014,"entityType":25,"verifyStatus":884,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":32,"subjectFields":1019,"manageAffiliations":1036,"indexDatabases":1047,"url":958,"thumbnailPath":28,"statistic":28,"gsStatistic":28,"type":28,"analyzePriority":28},[],{"country":1015,"eissn":1016,"issn":1017,"title":1018},{"VOID":875},{"VOID":877},{"VOID":879},{"EN":881},[1020,1024,1028,1032],{"id":887,"createTime":28,"updateTime":28,"relativeEntities":1021,"label":1022,"description":1023,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":890},{},{"id":893,"createTime":28,"updateTime":28,"relativeEntities":1025,"label":1026,"description":1027,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":896},{},{"id":899,"createTime":28,"updateTime":28,"relativeEntities":1029,"label":1030,"description":1031,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":902},{},{"id":905,"createTime":28,"updateTime":28,"relativeEntities":1033,"label":1034,"description":1035,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":908},{},[1037,1042],{"id":912,"createTime":28,"updateTime":28,"relativeEntities":1038,"slug":28,"properties":1039,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1041,"statistic":28},[],{"title":1040},{"EN":916},[],{"id":919,"createTime":28,"updateTime":28,"relativeEntities":1043,"slug":28,"properties":1044,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1046,"statistic":28},[],{"title":1045},{"EN":923},[],[1048,1055],{"id":927,"indexDatabase":1049,"url":939,"indexYears":28,"academicFieldIds":1054,"indexDatabaseRanking":28},{"id":929,"createTime":28,"updateTime":28,"relativeEntities":1050,"label":1051,"description":1052,"key":936,"publicationTags":1053,"standard":28},[],{"EN":932,"VI":932},{"EN":934,"VI":935},[938,813],[941,942],{"id":944,"indexDatabase":1056,"url":950,"indexYears":951,"academicFieldIds":1061,"indexDatabaseRanking":957},{"id":775,"createTime":28,"updateTime":28,"relativeEntities":1057,"label":1058,"description":1059,"key":781,"publicationTags":1060,"standard":28},[],{"EN":778,"VI":778},{"EN":778,"VI":780},[783],[953,954,955,956],{"issue":1063,"pages":1065,"volume":1067},{"VOID":1064},"1",{"VOID":1066},"110-122",{"VOID":1068},"56",653,{"total":1069,"publishYear":1071,"statisticByYear":1072},1978,{"2012":145,"2013":51,"2014":46,"2015":146,"2016":49,"2017":145,"2018":205,"2019":46,"2020":45,"2021":42,"2022":45,"2023":46},"1978-02-01",[938,957],[],false,{"id":1078,"createTime":1079,"updateTime":1080,"relativeEntities":1081,"slug":1082,"properties":1083,"entityType":986,"verifyStatus":884,"verifyTime":1079,"verifyNote":987,"languages":1100,"translateLanguages":1101,"viewCount":32,"primaryUrl":1102,"fullTextUrl":28,"authors":1103,"publicationType":1010,"publisherRelationship":1157,"citationCount":1215,"citationInfo":1216,"publishDate":1219,"publishYear":1217,"citationAnalyzeStatus":884,"lastCitationAnalyze":28,"indexDatabases":1220,"openAccess":28,"references":1221,"isForceReanalyzing":1076},"ea880f00-9e35-4671-8c2a-49ee5255de05","2024-10-06T15:13:03.221+00:00","2025-01-05T05:03:46.163+00:00",[],"Role-of-free-radicals-in-catecholamine-induced-cardiomyopathy",{"mag":1084,"keywords":1086,"openalex":1088,"abstract":1090,"title":1093,"pm":1096,"doi":1098},{"VOID":1085},"2098503682",{"VI":1087},"chất chống oxy hóa, vitamin E, kẽm, cơ tim, isoproterenol, gốc tự do, bệnh cơ tim, peroxidation lipid",{"VOID":1089},"W2098503682",{"VI":1091,"EN":1092},"\u003Cjats:p> Nghiên cứu đã xem xét tác động của một chất chống oxy hóa, vitamin E, và một tác nhân ổn định màng, kẽm, lên các thay đổi do isoproterenol gây ra trong cơ tim chuột. Việc điều trị bằng isoproterenol (80 mg\u002Fkg, được tiêm trong 2 ngày với hai liều bằng nhau) đã gây ra rối loạn nhịp tim và tỷ lệ tử vong lên tới 25% trong 24 giờ đầu sau liều tiêm cuối cùng. Các thay đổi vi cấu trúc trong lớp dưới nội tâm mạc và ở các vùng tiêu điểm của lớp ngoài tâm mạc bao gồm sự sưng lên của ty thể, mất myofibril, hoại tử tế bào, xơ hóa, và sự xâm nhập của các bạch cầu đa nhân. Cả mức creatine phosphate và adenosine triphosphate đều giảm đáng kể trong tim của các động vật được điều trị bằng isoproterenol. Việc điều trị trước bằng vitamin E (10 mg∙kg\u003Cjats:sup>−1\u003C\u002Fjats:sup>∙ngày\u003Cjats:sup>−1\u003C\u002Fjats:sup> trong 2 tuần) hoặc kẽm (10 mg\u002Fkg ZnSO\u003Cjats:sub>4\u003C\u002Fjats:sub>, hai lần mỗi ngày trong 7 ngày) đã ngăn chặn những tác động có hại của isoproterenol. Các động vật được duy trì trên chế độ ăn thiếu vitamin E trong 8 tuần được phát hiện nhạy cảm hơn với các thay đổi do isoproterenol gây ra và sự nhạy cảm này đã được đảo ngược sau 2 tuần cho ăn chế độ ăn bình thường kết hợp với điều trị vitamin E. Dựa trên dữ liệu thu được trong nghiên cứu này, tác giả đề xuất rằng các thay đổi do catecholamine có thể liên quan đến các gốc tự do, điều này thông qua việc thúc đẩy quá trình peroxidation lipid có thể làm tăng tính thấm của màng và dẫn đến sự phát triển của bệnh cơ tim. \u003C\u002Fjats:p>","\u003Cjats:p> Effects of an antioxidant, vitamin E, and a membrane stabilizing agent, zinc, were examined on the isoproterenol-induced changes in the rat myocardium. Isoproterenol treatment (80 mg\u002Fkg given over 2 days in two equal doses) caused arrhythmias and 25% mortality within 24 h of the last injection. The ultrastructural changes in the subendocardium and in focal areas of the subepicardium included swelling of mitochondria, loss of myofibrils, cell necrosis, fibrosis, and infiltration of the affected areas by polymorphonucleocytes. Both creatine phosphate and adenosine triphosphate levels were markedly decreased in hearts from isoproterenol-treated animals. Pretreatment of the animals with vitamin E (10 mg∙kg\u003Cjats:sup>−1\u003C\u002Fjats:sup>∙day\u003Cjats:sup>−1\u003C\u002Fjats:sup> for 2 weeks) or zinc (10 mg\u002Fkg ZnSO\u003Cjats:sub>4\u003C\u002Fjats:sub>, twice a day for 7 days) prevented these deleterious effects of isoproterenol. Animals maintained on vitamin E deficient diet for 8 weeks were found to be more sensitive to isoproterenol-induced changes and this increased sensitivity was reversed by a 2-week feeding of the animals on the normal diet coupled with vitamin E treatment. Based on the data obtained in this study it is proposed that catecholamine-induced changes may involve free radicals, which by promoting lipid peroxidation may increase membrane permeability and lead to the development of cardiomyopathy. \u003C\u002Fjats:p>",{"EN":1094,"VI":1095},"Role of free radicals in catecholamine-induced cardiomyopathy","Vai trò của các gốc tự do trong bệnh cơ tim do catecholamine",{"VOID":1097},"7151008",{"VOID":1099},"10.1139\u002Fy82-207",[31],[30],"http:\u002F\u002Fwww.nrcresearchpress.com\u002Fdoi\u002F10.1139\u002Fy82-207",[1104,1115,1126,1137,1146],{"id":1105,"sortIndex":32,"researcher":28,"roles":1106,"affiliations":1107,"properties":1108,"displayName":1112,"givenName":28,"familyName":28},"e15a4863-e701-4cd4-a28c-d057a66b8008",[],[],{"orcid":1109,"title":1111,"openalex":1113},{"VOID":1110},"https:\u002F\u002Forcid.org\u002F0000-0001-8818-9239",{"EN":1112},"Pawan K. Singal",{"VOID":1114},"A5048278410",{"id":1116,"sortIndex":40,"researcher":28,"roles":1117,"affiliations":1118,"properties":1119,"displayName":1123,"givenName":28,"familyName":28},"97f3e27e-ab47-42ea-ace3-529bb8281cc3",[],[],{"orcid":1120,"title":1122,"openalex":1124},{"VOID":1121},"https:\u002F\u002Forcid.org\u002F0000-0002-8302-6796",{"EN":1123},"Navin K. Kapur",{"VOID":1125},"A5000653485",{"id":1127,"sortIndex":123,"researcher":28,"roles":1128,"affiliations":1129,"properties":1130,"displayName":1134,"givenName":28,"familyName":28},"9bab34fa-b221-40f1-b84e-e879c744755c",[],[],{"orcid":1131,"title":1133,"openalex":1135},{"VOID":1132},"https:\u002F\u002Forcid.org\u002F0009-0005-1185-9712",{"EN":1134},"K. S. Dhillon",{"VOID":1136},"A5086364454",{"id":1138,"sortIndex":42,"researcher":28,"roles":1139,"affiliations":1140,"properties":1141,"displayName":1143,"givenName":28,"familyName":28},"fa8e73cd-f505-45cc-8d09-a385129abaa4",[],[],{"title":1142,"openalex":1144},{"EN":1143},"Robert E. Beamish",{"VOID":1145},"A5066389450",{"id":1147,"sortIndex":45,"researcher":28,"roles":1148,"affiliations":1149,"properties":1150,"displayName":1154,"givenName":28,"familyName":28},"ceae4533-48dc-4a4f-9e1a-17579079965a",[],[],{"orcid":1151,"title":1153,"openalex":1155},{"VOID":1152},"https:\u002F\u002Forcid.org\u002F0000-0002-4894-4727",{"EN":1154},"Naranjan S. Dhalla",{"VOID":1156},"A5068857226",{"url":28,"publisher":1158,"properties":1208},{"id":868,"createTime":869,"updateTime":870,"relativeEntities":1159,"slug":872,"properties":1160,"entityType":25,"verifyStatus":884,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":32,"subjectFields":1165,"manageAffiliations":1182,"indexDatabases":1193,"url":958,"thumbnailPath":28,"statistic":28,"gsStatistic":28,"type":28,"analyzePriority":28},[],{"country":1161,"eissn":1162,"issn":1163,"title":1164},{"VOID":875},{"VOID":877},{"VOID":879},{"EN":881},[1166,1170,1174,1178],{"id":887,"createTime":28,"updateTime":28,"relativeEntities":1167,"label":1168,"description":1169,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":890},{},{"id":893,"createTime":28,"updateTime":28,"relativeEntities":1171,"label":1172,"description":1173,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":896},{},{"id":899,"createTime":28,"updateTime":28,"relativeEntities":1175,"label":1176,"description":1177,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":902},{},{"id":905,"createTime":28,"updateTime":28,"relativeEntities":1179,"label":1180,"description":1181,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":908},{},[1183,1188],{"id":912,"createTime":28,"updateTime":28,"relativeEntities":1184,"slug":28,"properties":1185,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1187,"statistic":28},[],{"title":1186},{"EN":916},[],{"id":919,"createTime":28,"updateTime":28,"relativeEntities":1189,"slug":28,"properties":1190,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1192,"statistic":28},[],{"title":1191},{"EN":923},[],[1194,1201],{"id":927,"indexDatabase":1195,"url":939,"indexYears":28,"academicFieldIds":1200,"indexDatabaseRanking":28},{"id":929,"createTime":28,"updateTime":28,"relativeEntities":1196,"label":1197,"description":1198,"key":936,"publicationTags":1199,"standard":28},[],{"EN":932,"VI":932},{"EN":934,"VI":935},[938,813],[941,942],{"id":944,"indexDatabase":1202,"url":950,"indexYears":951,"academicFieldIds":1207,"indexDatabaseRanking":957},{"id":775,"createTime":28,"updateTime":28,"relativeEntities":1203,"label":1204,"description":1205,"key":781,"publicationTags":1206,"standard":28},[],{"EN":778,"VI":778},{"EN":778,"VI":780},[783],[953,954,955,956],{"issue":1209,"pages":1211,"volume":1213},{"VOID":1210},"11",{"VOID":1212},"1390-1397",{"VOID":1214},"60",458,{"total":1215,"publishYear":1217,"statisticByYear":1218},1982,{"2012":122,"2013":357,"2014":146,"2015":205,"2016":127,"2017":205,"2018":357,"2019":42,"2020":49,"2021":46,"2022":46,"2023":46,"2024":40},"1982-11-01",[938,957],[],{"id":1223,"createTime":1224,"updateTime":1225,"relativeEntities":1226,"slug":1227,"properties":1228,"entityType":986,"verifyStatus":884,"verifyTime":1224,"verifyNote":987,"languages":1244,"translateLanguages":1245,"viewCount":32,"primaryUrl":1246,"fullTextUrl":28,"authors":1247,"publicationType":1010,"publisherRelationship":1275,"citationCount":1332,"citationInfo":1333,"publishDate":1336,"publishYear":1334,"citationAnalyzeStatus":884,"lastCitationAnalyze":28,"indexDatabases":1337,"openAccess":28,"references":1338,"isForceReanalyzing":1076},"6ae0bd97-0039-4d1e-b814-51d0a37737ac","2024-10-07T19:05:59.474+00:00","2025-01-05T05:04:40.884+00:00",[],"Inhibition-of-the-immune-response-by-rapamycin-a-new-antifungal-antibiotic",{"mag":1229,"keywords":1231,"openalex":1232,"abstract":1234,"title":1237,"pm":1240,"doi":1242},{"VOID":1230},"1976726113",{"VI":973},{"VOID":1233},"W1976726113",{"VI":1235,"EN":1236},"\u003Cjats:p> Rapamycin, một loại kháng sinh chống nấm mới, được phát hiện là có khả năng ức chế phản ứng miễn dịch ở chuột đồng. Nó hoàn toàn ngăn chặn sự phát triển của hai bệnh lý miễn dịch thí nghiệm (viêm não tủy dị ứng thí nghiệm (EAE) và viêm khớp bổ trợ (AA)) và sự hình thành kháng thể thể dịch (giống IgE). Nó mạnh bằng khoảng một nửa so với cyclophosphamide trong việc ức chế EAE. Trong trường hợp AA và sự hình thành kháng thể, rapamycin và cyclophosphamide có độ mạnh tương đương, trong khi methotrexate lại mạnh hơn. Hoạt tính ức chế miễn dịch của rapamycin có vẻ liên quan đến việc ức chế hệ bạch huyết. \u003C\u002Fjats:p>","\u003Cjats:p> Rapamycin, a new antifungal antibiotic, was found to inhibit the immune response in rats. It totally prevented the development of two experimental immunopathies (experimental allergic encephalomyelitis (EAE) and adjuvant arthritis (AA)) and the formation of humoral (IgE-like) antibody. It was about half as potent as cyclophosphamide in inhibiting EAE. In AA and on antibody formation, rapamycin and cyclophosphamide were about equipotent, whereas methotrexate was more potent. The immunosuppressant activity of rapamycin appears to be related to inhibition of the lymphatic system. \u003C\u002Fjats:p>",{"EN":1238,"VI":1239},"Inhibition of the immune response by rapamycin, a new antifungal antibiotic","Sự ức chế phản ứng miễn dịch bởi rapamycin, một loại kháng sinh chống nấm mới",{"VOID":1241},"843990",{"VOID":1243},"10.1139\u002Fy77-007",[31],[30],"http:\u002F\u002Fwww.nrcresearchpress.com\u002Fdoi\u002F10.1139\u002Fy77-007",[1248,1257,1266],{"id":1249,"sortIndex":32,"researcher":28,"roles":1250,"affiliations":1251,"properties":1252,"displayName":1254,"givenName":28,"familyName":28},"af74fcac-520c-4608-9f36-40613d94efa1",[],[],{"title":1253,"openalex":1255},{"EN":1254},"R. R. Martel",{"VOID":1256},"A5103584079",{"id":1258,"sortIndex":40,"researcher":28,"roles":1259,"affiliations":1260,"properties":1261,"displayName":1263,"givenName":28,"familyName":28},"672065d1-1c09-4c72-85cf-be07f3115d8f",[],[],{"title":1262,"openalex":1264},{"EN":1263},"J. Klicius",{"VOID":1265},"A5012822333",{"id":1267,"sortIndex":123,"researcher":28,"roles":1268,"affiliations":1269,"properties":1270,"displayName":1272,"givenName":28,"familyName":28},"557bdf6b-b44e-41c6-9b6b-e74375c7b2bf",[],[],{"title":1271,"openalex":1273},{"EN":1272},"S. Galet",{"VOID":1274},"A5041096824",{"url":28,"publisher":1276,"properties":1326},{"id":868,"createTime":869,"updateTime":870,"relativeEntities":1277,"slug":872,"properties":1278,"entityType":25,"verifyStatus":884,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":32,"subjectFields":1283,"manageAffiliations":1300,"indexDatabases":1311,"url":958,"thumbnailPath":28,"statistic":28,"gsStatistic":28,"type":28,"analyzePriority":28},[],{"country":1279,"eissn":1280,"issn":1281,"title":1282},{"VOID":875},{"VOID":877},{"VOID":879},{"EN":881},[1284,1288,1292,1296],{"id":887,"createTime":28,"updateTime":28,"relativeEntities":1285,"label":1286,"description":1287,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":890},{},{"id":893,"createTime":28,"updateTime":28,"relativeEntities":1289,"label":1290,"description":1291,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":896},{},{"id":899,"createTime":28,"updateTime":28,"relativeEntities":1293,"label":1294,"description":1295,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":902},{},{"id":905,"createTime":28,"updateTime":28,"relativeEntities":1297,"label":1298,"description":1299,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":908},{},[1301,1306],{"id":912,"createTime":28,"updateTime":28,"relativeEntities":1302,"slug":28,"properties":1303,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1305,"statistic":28},[],{"title":1304},{"EN":916},[],{"id":919,"createTime":28,"updateTime":28,"relativeEntities":1307,"slug":28,"properties":1308,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1310,"statistic":28},[],{"title":1309},{"EN":923},[],[1312,1319],{"id":927,"indexDatabase":1313,"url":939,"indexYears":28,"academicFieldIds":1318,"indexDatabaseRanking":28},{"id":929,"createTime":28,"updateTime":28,"relativeEntities":1314,"label":1315,"description":1316,"key":936,"publicationTags":1317,"standard":28},[],{"EN":932,"VI":932},{"EN":934,"VI":935},[938,813],[941,942],{"id":944,"indexDatabase":1320,"url":950,"indexYears":951,"academicFieldIds":1325,"indexDatabaseRanking":957},{"id":775,"createTime":28,"updateTime":28,"relativeEntities":1321,"label":1322,"description":1323,"key":781,"publicationTags":1324,"standard":28},[],{"EN":778,"VI":778},{"EN":778,"VI":780},[783],[953,954,955,956],{"issue":1327,"pages":1328,"volume":1330},{"VOID":1064},{"VOID":1329},"48-51",{"VOID":1331},"55",443,{"total":1332,"publishYear":1334,"statisticByYear":1335},1977,{"2012":199,"2013":126,"2014":51,"2015":135,"2016":199,"2017":134,"2018":130,"2019":134,"2020":136,"2021":122,"2022":146,"2023":323,"2024":48},"1977-02-01",[938,957],[],{"id":1340,"createTime":1341,"updateTime":1342,"relativeEntities":1343,"slug":1344,"properties":1345,"entityType":986,"verifyStatus":884,"verifyTime":1341,"verifyNote":987,"languages":1362,"translateLanguages":1363,"viewCount":32,"primaryUrl":1364,"fullTextUrl":28,"authors":1365,"publicationType":1010,"publisherRelationship":1430,"citationCount":1488,"citationInfo":1489,"publishDate":1492,"publishYear":1490,"citationAnalyzeStatus":884,"lastCitationAnalyze":28,"indexDatabases":1493,"openAccess":28,"references":1494,"isForceReanalyzing":1076},"ef7678a9-ceaf-4ab0-8055-77372e026e16","2024-09-04T01:30:44.899+00:00","2025-01-05T05:05:40.242+00:00",[],"The-abdominal-visceral-innervation-and-the-emetic-reflex-pathways-pharmacology-and-plasticity",{"mag":1346,"keywords":1348,"openalex":1350,"abstract":1352,"title":1355,"pm":1358,"doi":1360},{"VOID":1347},"2143952462",{"VI":1349},"nôn mửa, dây thần kinh tạng, dây thần kinh phế vị, chồn ferret, tính linh hoạt, các đối kháng serotonin.",{"VOID":1351},"W2143952462",{"VI":1353,"EN":1354},"\u003Cjats:p> Trong những năm gần đây, vai trò của vùng postrema trong phản xạ nôn đã trở nên nổi bật và sự tham gia của hệ thống thần kinh tạng bụng có xu hướng bị bỏ qua. Bài báo này cố gắng cân bằng lại phản xạ bằng cách xem xét các khía cạnh của tài liệu hiện có và bổ sung điều này bằng các nghiên cứu gốc từ con chồn ferret. Với việc con chồn ferret được sử dụng rộng rãi trong các nghiên cứu về nôn mửa và đặc biệt là trong việc phân tích tác động chống nôn của các đối kháng thụ thể 5-HT\u003Cjats:sub>3\u003C\u002Fjats:sub>, cơ hội được lấy để đánh giá tính phù hợp của loài này cho các nghiên cứu về nôn mửa. Kết luận cho thấy rằng con chồn ferret nhạy cảm với nhiều loại kích thích nôn khác nhau bao gồm các chất kích thích dạ dày, agonist thụ thể opiate và dopamine, nhiều thuốc gây độc tế bào và bức xạ. Đối với một số kích thích, nó nhạy hơn các loài khác, và đối với bức xạ, căn cứ vào ED\u003Cjats:sub>100\u003C\u002Fjats:sub> của nó, dường như nó là loài động vật thí nghiệm nhạy cảm nhất trong số các loài được nghiên cứu. Sử dụng kích thích điện của đầu trung tâm của thân thần kinh phế vị ở bụng trên động vật có ý thức và đã gây mê, các nhánh tủy sống đã cho thấy có khả năng gây ra nôn. Qua các nghiên cứu tổn thương, sự tham gia của thần kinh phế vị trong phản ứng nôn với một số thuốc gây độc tế bào (ví dụ: cisplatin, cyclophosphamide, mustine) và bức xạ đã được chứng minh, mặc dù mức độ tác động biến đổi theo các kích thích khác nhau. Một nỗ lực được thực hiện để hòa hợp những quan sát này với các nghiên cứu trước đó về việc cắt bỏ vùng postrema. Vấn đề trong việc giải thích các tác động của tổn thương thần kinh được thảo luận một cách nghiêm túc trong bối cảnh bằng chứng sơ bộ được trình bày ở đây rằng có thể có một mức độ linh hoạt trong con đường nôn sau những tổn thương như vậy. Phạm vi các tác động chống nôn của các đối kháng thụ thể 5-HT\u003Cjats:sub>3\u003C\u002Fjats:sub> được xem xét và một nỗ lực được thực hiện để xác định vị trí mà những tác nhân này hoạt động. Kết quả được trình bày cho thấy mối liên hệ giữa thần kinh phế vị và sự đối kháng thụ thể 5-HT\u003Cjats:sub>3\u003C\u002Fjats:sub>. Những nghiên cứu này được thảo luận cùng với những nghiên cứu khác và dẫn chúng tôi đến việc đề xuất rằng (trong con chồn ferret) các đối kháng thụ thể 5-HT\u003Cjats:sub>3\u003C\u002Fjats:sub> có tác động chống nôn chính bằng cách tác động lên các đầu tận cùng của nhánh tủy sống trong thành phần dạ dày trên với một vị trí phụ nữa là trong nhân tractus solitarius hoặc trước synap trên các đầu tận cùng tủy sống ở hành não nơi mà các vị trí gắn kết cho ligand của thụ thể 5-HT\u003Cjats:sub>3\u003C\u002Fjats:sub> gần đây đã được chứng minh trong loài này. Từ khóa: nôn mửa, dây thần kinh tạng, dây thần kinh phế vị, chồn ferret, tính linh hoạt, các đối kháng serotonin.","\u003Cjats:p> In recent years the role of the area postrema in the emetic reflex has been predominant and the involvement of the abdominal visceral innervation has tended to be overlooked. This paper attempts to redress the balance reflex by reviewing aspects of the existing literature and complementing this with original studies from the ferret. In view of the widespread use of the ferret in studies of emesis and particularly in the characterization of the antiemetic actions of 5-HT\u003Cjats:sub>3\u003C\u002Fjats:sub> receptor antagonist, the opportunity is taken to assess the suitability of this species for studies of emesis. It is concluded that the ferret is sensitive to a wide range of emetic stimuli including intragastric irritants, opiate and dopamine receptor agonists, many cytotoxic drugs, and radiation. For several stimuli it is more sensitive than other species and for radiation on the basis of its ED\u003Cjats:sub>100\u003C\u002Fjats:sub> it appears to be the most sensitive of the laboratory animals studied. Using electrical stimulation of the central end of the dorsal vagal trunk in the abdomen in conscious and anaesthetized animals, the vagal afferents were shown to be capable of eliciting emesis. Using lesioning studies an involvement of the vagus in the emetic response to a number of cytotoxic drugs (e.g., cisplatinum, cyclophosphamide, mustine) and radiation was demonstrated, although the magnitude of the effect varied with the different stimuli. An attempt is made to reconcile these observations with previous studies of area postrema ablation. The problems of interpreting the effects of nerve lesions are critically discussed in light of preliminary evidence presented here that there may be a degree of plasticity in the emetic pathway following such lesions. The range of antiemetic effects of 5-HT\u003Cjats:sub>3\u003C\u002Fjats:sub> receptor antagonists is reviewed and an attempt is made to identify the site(s) at which these agents act. Results are presented that suggest a link between the vagus and 5-HT\u003Cjats:sub>3\u003C\u002Fjats:sub> receptor antagonism. These studies are discussed together with others and lead us to propose that (in the ferret) 5-HT\u003Cjats:sub>3\u003C\u002Fjats:sub> receptor antagonists have their main antiemetic effect by acting on vagal afferent terminals in the wall of the upper gut with an additional minor site either in the nucleus tractus solitarius or presynaptically on the vagal afferent terminals in the medulla where binding sites for 5-HT\u003Cjats:sub>3\u003C\u002Fjats:sub> receptor ligands have recently been demonstrated in this species.Key words: emesis, visceral nerves, vagus nerve, ferret, plasticity, serotonin antagonists. \u003C\u002Fjats:p>",{"EN":1356,"VI":1357},"The abdominal visceral innervation and the emetic reflex: pathways, pharmacology, and plasticity","Sự thần kinh tạng trong bụng và phản xạ nôn: lộ trình, dược lý và tính linh hoạt",{"VOID":1359},"2178756",{"VOID":1361},"10.1139\u002Fy90-047",[31],[30],"http:\u002F\u002Fwww.nrcresearchpress.com\u002Fdoi\u002F10.1139\u002Fy90-047",[1366,1385,1394,1403,1412,1421],{"id":1367,"sortIndex":32,"researcher":28,"roles":1368,"affiliations":1369,"properties":1378,"displayName":1382,"givenName":28,"familyName":28},"1f7244fc-f8dc-4840-9e8d-b7d34718611a",[],[1370],{"id":1371,"sortIndex":32,"affiliation":1372,"properties":28},"2ed7bcc6-27dd-4960-8ad8-538359ba9e8e",{"id":1371,"createTime":28,"updateTime":28,"relativeEntities":1373,"slug":28,"properties":1374,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1377,"statistic":28},[],{"title":1375},{"VI":1376},"Department of Physiology, St. George's Hospital Medical School, London, UK",[],{"orcid":1379,"title":1381,"openalex":1383},{"VOID":1380},"https:\u002F\u002Forcid.org\u002F0000-0001-7099-6964",{"EN":1382},"Paul Andrews",{"VOID":1384},"A5081947723",{"id":1386,"sortIndex":40,"researcher":28,"roles":1387,"affiliations":1388,"properties":1389,"displayName":1391,"givenName":28,"familyName":28},"c2b17838-88c5-4bdf-bb13-47a9fc50086b",[],[],{"title":1390,"openalex":1392},{"EN":1391},"Claudine Davis",{"VOID":1393},"A5004213295",{"id":1395,"sortIndex":123,"researcher":28,"roles":1396,"affiliations":1397,"properties":1398,"displayName":1400,"givenName":28,"familyName":28},"5931c9b1-056b-4a17-ba5d-5c57e8c62906",[],[],{"title":1399,"openalex":1401},{"EN":1400},"Sharon Bingham",{"VOID":1402},"A5049640545",{"id":1404,"sortIndex":42,"researcher":28,"roles":1405,"affiliations":1406,"properties":1407,"displayName":1409,"givenName":28,"familyName":28},"0ce74083-0d99-4e83-a214-88bf531be206",[],[],{"title":1408,"openalex":1410},{"EN":1409},"H. 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Giai đoạn nước bên trong có thể chứa enzyme hoặc các protein khác, các mảnh tế bào hoặc tế bào nguyên vẹn. Các ví dụ về phương pháp chuẩn bị các vi hạt này đã được mô tả chi tiết, và một số tính chất của chúng được trình bày.\u003C\u002Fjats:p>","\u003Cjats:p> Methods have been developed for depositing thin stable semipermeable polymer membranes around aqueous microdroplets (mean diameter down to 5 μ or less) by either interfacial polymerization or interfacial coacervation. The enclosed aqueous phase may contain enzymes or other proteins, cell fragments, or intact cells. Examples of methods for preparing such microcapsules are given in detail, and some of their properties are described. \u003C\u002Fjats:p>",{"EN":1511,"VI":1512},"SEMIPERMEABLE AQUEOUS MICROCAPSULES: I. PREPARATION AND PROPERTIES","MÀNG VI MÀU ĐỊNH HÌNH BÁO TỐ TÍNH NẰM TRONG: I. CHUẨN BỊ VÀ TÍNH CHẤT",{"VOID":1514},"5938982",{"VOID":1516},"10.1139\u002Fy66-013","2024-07-18T23:09:42.587+00:00",[31],[30],"http:\u002F\u002Fwww.nrcresearchpress.com\u002Fdoi\u002F10.1139\u002Fy66-013",[1522,1531,1540],{"id":1523,"sortIndex":32,"researcher":28,"roles":1524,"affiliations":1525,"properties":1526,"displayName":1528,"givenName":28,"familyName":28},"7fecdfb9-49f2-42dd-ac0d-c39cbc27a478",[],[],{"title":1527,"openalex":1529},{"EN":1528},"T. M. S. Chang",{"VOID":1530},"A5037539620",{"id":1532,"sortIndex":40,"researcher":28,"roles":1533,"affiliations":1534,"properties":1535,"displayName":1537,"givenName":28,"familyName":28},"42b3b972-24ce-4d13-8106-d72b469f425e",[],[],{"title":1536,"openalex":1538},{"EN":1537},"F. C. 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SCMLEICHEK. TJ., Nos., 2, 800",{},{"id":28,"text":1620,"url":28,"identifiers":1621},"L' UCTION, Nos., 634, 634",{},{"id":28,"text":1623,"url":28,"identifiers":1624},"AIG., 1964, Science, 144, 1093, 10.1126\u002Fscience.144.3622.1093",{"doi":1625},"10.1126\u002Fscience.144.3622.1093",{"id":28,"text":1627,"url":28,"identifiers":1628},"I' KEHN, 1957, Acad. Sci., 64, 1009, 10.1111\u002Fj.1749-6632.1957.tb52492.x",{"doi":1629},"10.1111\u002Fj.1749-6632.1957.tb52492.x",{"id":28,"text":1631,"url":28,"identifiers":1632},"KENNEDY., 1960, Ann. Surg., 151, 626, 10.1097\u002F00000658-196005000-00002",{"doi":1633},"10.1097\u002F00000658-196005000-00002",{"id":1635,"createTime":1636,"updateTime":1636,"relativeEntities":1637,"slug":1638,"properties":1639,"entityType":986,"verifyStatus":884,"verifyTime":1636,"verifyNote":987,"languages":1652,"translateLanguages":28,"viewCount":32,"primaryUrl":1653,"fullTextUrl":28,"authors":1654,"publicationType":1010,"publisherRelationship":1664,"citationCount":1722,"citationInfo":1723,"publishDate":1726,"publishYear":1724,"citationAnalyzeStatus":884,"lastCitationAnalyze":28,"indexDatabases":1727,"openAccess":28,"references":1728,"isForceReanalyzing":1076},"d1398562-5dfd-45a7-b21b-7dafef00a99c","2025-01-04T20:40:28.694+00:00",[],"The-coactivation-of-antagonist-muscles",{"openalex":1640,"mag":1642,"abstract":1644,"title":1646,"pm":1648,"doi":1650},{"VOID":1641},"W2068096613",{"VOID":1643},"2068096613",{"EN":1645},"\u003Cjats:p> Since Sherrington's convincing demonstration of the reciprocal innervation of opposing muscles, it has generally been thought that antagonist muscles are inactive during most voluntary movements. However, more recent evidence suggests that excitation of Renshaw cells may facilitate antagonist coactivation whereas excitation of Ia inhibitory neurons can induce reciprocal inhibition. A body of evidence has accumulated to indicate some of the circumstances which particularly favour the co-contraction of antagonist muscles. Isometric prehension, either in the precision grip or the power grip, can be shown to be one of the most important examples of antagonist coactivation. Studies of the discharge of single Purkinje cells of the intermediate cerebellar cortex in awake monkeys during performance of a maintained grip revealed that the majority of these neurons are deactivated during antagonist co-contraction. In contrast, other, unidentified neurons of the cerebellar cortex were as a group activated during grasping. It is suggested that the Purkinje cells act to inhibit antagonist muscles during reciprocal inhibition but are themselves inhibited during antagonist coactivation. These results support a suggestion made by Tilney and Pike in 1925 that the cerebellum plays an important role in switching between the coactivation and reciprocal inhibition of antagonist muscles. \u003C\u002Fjats:p>",{"EN":1647},"The coactivation of antagonist muscles",{"VOID":1649},"7032676",{"VOID":1651},"10.1139\u002Fy81-110",[31],"http:\u002F\u002Fwww.nrcresearchpress.com\u002Fdoi\u002F10.1139\u002Fy81-110",[1655],{"id":1656,"sortIndex":32,"researcher":28,"roles":1657,"affiliations":1658,"properties":1659,"displayName":1661,"givenName":28,"familyName":28},"ea8514f8-75b6-48fd-a77c-a6c0995bf6a8",[],[],{"title":1660,"openalex":1662},{"EN":1661},"Allan M. 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Các isoenzym creatine phosphokinase đóng một vai trò quan trọng trong việc vận chuyển năng lượng nội bào từ ti thể đến sợi cơ và các vị trí sử dụng năng lượng khác. Do sự tồn tại của con đường creatine phosphate để vận chuyển năng lượng, nồng độ creatine phosphate trong tế bào rõ ràng là một yếu tố điều tiết quan trọng cho sự co cơ, ảnh hưởng đến lực co bóp bằng cách xác định tốc độ tái sinh ATP có sẵn cho myosin ATPase, và đồng thời kiểm soát sự gia tăng ion canxi kích hoạt vào myoplasm qua màng tế bào.","\u003Cjats:p> This paper summarizes the data concerning the role of the creatine phosphokinase system in muscle cells with main attention to the cardiac muscle. Creatine phosphokinase isoenzymes play a key role in the intracellular energy transport from mitochondria to myofibrils and other sites of energy utilization. Due to the existence of the creatine phosphate pathway for energy transport, intracellular creatine phosphate concentration is apparently an important regulatory factor for muscle contraction which influences the contractile force by determining the rate of regeneration of ATP directly available for myosin ATPase, and at the same time controls the activator calcium entry into the myoplasm across the surface membrane of the cells. \u003C\u002Fjats:p>",{"EN":1745,"VI":1746},"Role of creatine phosphokinase in cellular function and metabolism","Vai trò của creatine phosphokinase trong chức năng tế bào và chuyển hóa",{"VOID":1748},"361188",{"VOID":1750},"10.1139\u002Fy78-113",[31],[30],"http:\u002F\u002Fwww.nrcresearchpress.com\u002Fdoi\u002F10.1139\u002Fy78-113",[1755,1764,1773,1782],{"id":1756,"sortIndex":32,"researcher":28,"roles":1757,"affiliations":1758,"properties":1759,"displayName":1761,"givenName":28,"familyName":28},"6aff781e-8add-4847-a24a-3c0dcc72126c",[],[],{"title":1760,"openalex":1762},{"EN":1761},"V A Saks",{"VOID":1763},"A5085909148",{"id":1765,"sortIndex":40,"researcher":28,"roles":1766,"affiliations":1767,"properties":1768,"displayName":1770,"givenName":28,"familyName":28},"20da86ae-0f1e-4704-8ee9-664b4c7add8b",[],[],{"title":1769,"openalex":1771},{"EN":1770},"Leonid V. 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Câu chuyện về endothelin là độc nhất, vì các chuỗi gene của receptor endothelin và các đối kháng receptor đầu tiên đã có sẵn chỉ trong 4 năm sau khi xác định được chuỗi peptide. Nghiên cứu lâm sàng đầu tiên trên bệnh nhân suy tim sung huyết được công bố chỉ 3 năm sau đó. Tuy nhiên, bất chấp bằng chứng thuyết phục từ các nghiên cứu thực nghiệm về vai trò bệnh sinh của endothelin trong sự phát triển, chức năng tế bào và bệnh tật, nhiều nghiên cứu lâm sàng ban đầu về đối kháng endothelin lại có kết quả tiêu cực. Trong nhiều nghiên cứu này, thiết kế nghiên cứu hoặc việc chọn lựa bệnh nhân không đủ. Ngày nay, đối với các bệnh như tăng huyết áp động mạch phổi, điều trị đối kháng endothelin đã trở thành hiện thực trong y học lâm sàng, và các nghiên cứu lâm sàng đang diễn ra đang đánh giá các chỉ định bổ sung, chẳng hạn như bệnh thận và ung thư. Hai mươi năm sau khi phát hiện ra endothelin, các chất ức chế của nó cuối cùng đã có mặt tại lĩnh vực lâm sàng và đang cung cấp cho chúng ta những lựa chọn mới để điều trị bệnh và kéo dài tuổi thọ cho bệnh nhân. Các chỉ định trong tương lai có thể bao gồm tăng huyết áp động mạch kháng trị, bệnh thận có protein niệu, ung thư, và các bệnh về mô liên kết.\u003C\u002Fjats:p>","\u003Cjats:p>Since its identification as an endothelial cell-derived vasoconstrictor peptide in 1988, endothelin-1, the predominant member of the endothelin peptide family, has received considerable interest in basic medical science and in clinical medicine, which is reflected by more than 20 000 scientific publications on endothelin research in the past 20 years. The story of endothelin is unique as the gene sequences of endothelin receptors and the first receptor antagonists became available within only 4 years of the identification of the peptide sequence.The first clinical study in patients with congestive heart failure was published only 3 years thereafter. Yet, despite convincing experimental evidence of a pathogenetic role for endothelin in development, cell function, and disease, many initial clinical studies on endothelin antagonism were negative. In many of these studies, study designs or patient selection were inadequate. Today, for diseases such as pulmonary hypertension, endothelin antagonist treatment has become reality in clinical medicine, and ongoing clinical studies are evaluating additional indications, such as renal disease and cancer. Twenty years after the discovery of endothelin, its inhibitors have finally arrived in the clinical arena and are now providing us with new options to treat disease and prolong the lives of patients. Possible future indications include resistant arterial hypertension, proteinuric renal disease, cancer, and connective tissue diseases.\u003C\u002Fjats:p>",{"EN":1869,"VI":1870},"Endothelin: 20 years from discovery to therapyThis article is one of a selection of papers published in the special issue (part 2 of 2) on Forefronts in Endothelin.","Endothelin: 20 năm từ phát hiện đến liệu pháp",{"VOID":1872},"18758495",{"VOID":1874},"10.1139\u002Fy08-059","Auto 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Japan",[],{"id":1893,"sortIndex":40,"affiliation":1894,"properties":28},"f090a442-f9a5-4cf8-9f3a-292ab7fbfa35",{"id":1893,"createTime":28,"updateTime":28,"relativeEntities":1895,"slug":28,"properties":1896,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1899,"statistic":28},[],{"title":1897},{"VI":1898},"Howard Hughes Medical Institute, Department of Molecular Genetics, University of Texas Southwestern Medical Center, Dallas, Texas, USA",[],{"id":1901,"sortIndex":123,"affiliation":1902,"properties":28},"7ba7cf57-d7a1-4fc0-b931-64cf649696c8",{"id":1901,"createTime":28,"updateTime":28,"relativeEntities":1903,"slug":28,"properties":1904,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1907,"statistic":28},[],{"title":1905},{"EN":1906},"Klinik und Poliklinik für Innere Medizin, Departement für Innere 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Physicians, 111, 417, 10.1111\u002Fpaa.1999.111.5.417",{"doi":2409},"10.1111\u002Fpaa.1999.111.5.417",{"id":28,"text":2411,"url":28,"identifiers":2412},"Schiffrin E.L., 1999, Hypertension, 34, 876, 10.1161\u002F01.HYP.34.4.876",{"doi":2413},"10.1161\u002F01.HYP.34.4.876",{"id":28,"text":2415,"url":28,"identifiers":2416},"10.1016\u002FS0895-7061(01)02074-X",{"doi":2415},{"id":28,"text":2418,"url":28,"identifiers":2419},"10.1016\u002FS0895-7061(02)03006-6",{"doi":2418},{"id":28,"text":2421,"url":28,"identifiers":2422},"10.1016\u002Fj.vph.2005.03.004",{"doi":2421},{"id":28,"text":2424,"url":28,"identifiers":2425},"10.1161\u002FHYPERTENSIONAHA.107.108126",{"doi":2424},{"id":28,"text":2427,"url":28,"identifiers":2428},"10.1161\u002FCIRCULATIONAHA.107.733212",{"doi":2427},{"id":28,"text":2430,"url":28,"identifiers":2431},"10.1002\u002Fart.22634",{"doi":2430},{"id":28,"text":2433,"url":28,"identifiers":2434},"10.1097\u002F00063198-200309000-00015",{"doi":2433},{"id":28,"text":2436,"url":28,"identifiers":2437},"Shennib H., 1998, Am. 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Biol. Med. (Maywood), 231, 857",{},{"id":28,"text":2474,"url":28,"identifiers":2475},"10.1097\u002F00041552-200303000-00009",{"doi":2474},{"id":28,"text":2477,"url":28,"identifiers":2478},"Wesson D.E., 2006, Kidney Int., 70, 2066, 10.1038\u002Fsj.ki.5001905",{"doi":2479},"10.1038\u002Fsj.ki.5001905",{"id":28,"text":2481,"url":28,"identifiers":2482},"10.1074\u002Fjbc.274.12.8103",{"doi":2481},{"id":28,"text":2484,"url":28,"identifiers":2485},"10.1002\u002Fart.1780340220",{"doi":2484},{"id":28,"text":2487,"url":28,"identifiers":2488},"10.1016\u002F0165-6147(89)90011-4",{"doi":2487},{"id":28,"text":2490,"url":28,"identifiers":2491},"10.1038\u002F332411a0",{"doi":2490},{"id":28,"text":2493,"url":28,"identifiers":2494},"10.1172\u002FJCI7447",{"doi":2493},{"id":28,"text":2496,"url":28,"identifiers":2497},"Yokokawa K., 1991, Ann. Intern. Med., 114, 213, 10.7326\u002F0003-4819-114-3-213",{"doi":2498},"10.7326\u002F0003-4819-114-3-213",{"id":28,"text":2500,"url":28,"identifiers":2501},"Yoshio T., 1995, Ann. Rheum. Dis., 54, 361, 10.1136\u002Fard.54.5.361",{"doi":2502},"10.1136\u002Fard.54.5.361",{"id":28,"text":2504,"url":28,"identifiers":2505},"10.1016\u002Fj.diabres.2007.12.024",{"doi":2504},{"id":2507,"createTime":2508,"updateTime":2509,"relativeEntities":2510,"slug":2511,"properties":2512,"entityType":986,"verifyStatus":884,"verifyTime":2508,"verifyNote":987,"languages":2528,"translateLanguages":2529,"viewCount":32,"primaryUrl":2530,"fullTextUrl":28,"authors":2531,"publicationType":1010,"publisherRelationship":2563,"citationCount":2620,"citationInfo":2621,"publishDate":2624,"publishYear":2622,"citationAnalyzeStatus":884,"lastCitationAnalyze":28,"indexDatabases":2625,"openAccess":28,"references":2626,"isForceReanalyzing":1076},"47fcfcb9-243b-469e-8ada-1f97061aa55c","2024-09-24T09:31:46.296+00:00","2025-01-05T05:09:24.902+00:00",[],"THE-TAIL-OF-THE-RAT-IN-TEMPERATURE-REGULATION-AND-ACCLIMATIZATION",{"mag":2513,"keywords":2515,"openalex":2516,"abstract":2518,"title":2521,"pm":2524,"doi":2526},{"VOID":2514},"2088917071",{"VI":973},{"VOID":2517},"W2088917071",{"VI":2519,"EN":2520},"\u003Cjats:p> Vai trò của đuôi chuột Wistar trắng trong việc điều hòa nhiệt độ đã được nghiên cứu và một chỉ số mới về sự thích nghi đã được phát hiện. Lưu lượng máu tại đuôi được đo bằng phương pháp plethysmography tắc tĩnh mạch ở các nhiệt độ môi trường từ 17 đến 33 °C. Có một hiện tượng giãn mạch đột ngột xảy ra giữa 27 và 30° với lưu lượng tăng từ dưới 5 ml lên khoảng 40 ml\u002F100 ml mô mỗi phút. Đo lường sự mất nhiệt bằng một nhiệt kế gradient trên đuôi cho thấy một giãn mạch phản xạ tương tự ở nhiệt độ giãn mạch quan trọng (T\u003Cjats:sub>CVD\u003C\u002Fjats:sub>). Sau khi giãn mạch, đuôi có thể mất tới 20% tổng sản lượng nhiệt của chuột. Nhiệt độ da của đuôi được sử dụng như một chỉ số giãn mạch để xác định xem nhiệt độ quan trọng có dịch chuyển với sự thích nghi tại 11 °C, 20 °C và 30 °C hay không. Có một sự giảm T\u003Cjats:sub>CVD\u003C\u002Fjats:sub> khoảng 6° sau khi thích nghi với lạnh (T\u003Cjats:sub>CVD\u003C\u002Fjats:sub> = 20 °C cho 11 °C, 26 °C cho 20 °C). Sau khi thích nghi với 30 °C, không có hiện tượng giãn mạch nào được phát hiện ở nhiệt độ lên tới 33 °C. Sự mất nhiệt tối đa của đuôi được tăng cường đáng kể sau khi thích nghi với lạnh. Cơ chế của sự dịch chuyển này có thể là một sự thay đổi trong độ nhạy của các thụ thể nhiệt trên đuôi, do sự tăng cường vách mạch (tăng độ dẫn nhiệt) của các mô địa phương.","\u003Cjats:p> The role of the tail of the Wistar white rat in its temperature regulation was studied, and a new index of acclimatization was found. Blood flow in the tail was measured by venous-occlusion plethysmography at environmental temperatures from 17 to 33 °C. There is an abrupt vasodilation between 27 and 30° with flow rising from less than 5 ml to about 40 ml\u002F100 ml tissue per minute. Measurement of heat loss by a gradient calorimeter on the tail showed a similar reflex vasodilation at a critical vasodilation temperature (T\u003Cjats:sub>CVD\u003C\u002Fjats:sub>). After vasodilation the tail can lose up to 20% of the total heat production of the rat. The skin temperature of the tail was used as an index of vasodilation to determine whether the critical temperature shifted with acclimatization to 11 °C, 20 °C, and 30 °C. There is a decrease in T\u003Cjats:sub>CVD\u003C\u002Fjats:sub> of about 6° after acclimatization to cold (T\u003Cjats:sub>CVD\u003C\u002Fjats:sub> = 20 °C for 11 °C, 26 °C for 20 °C). After acclimatization to 30 °C, no vasodilation was found at temperatures up to 33 °C. The maximum heat loss of the tail is greatly increased after cold acclimatization. The mechanism of the shift is probably a change in sensitivity of thermal receptors on the tail, due to an increased vascularity (increased thermal conductivity) of the local tissues. \u003C\u002Fjats:p>",{"EN":2522,"VI":2523},"THE TAIL OF THE RAT, IN TEMPERATURE REGULATION AND ACCLIMATIZATION","ĐUÔI CỦA RẮN TRONG VIỆC ĐIỀU HÒA NHIỆT ĐỘ VÀ THÍCH NGHI NHIỆT",{"VOID":2525},"14329334",{"VOID":2527},"10.1139\u002Fy65-025",[31],[30],"http:\u002F\u002Fwww.nrcresearchpress.com\u002Fdoi\u002F10.1139\u002Fy65-025",[2532,2541,2552],{"id":2533,"sortIndex":32,"researcher":28,"roles":2534,"affiliations":2535,"properties":2536,"displayName":2538,"givenName":28,"familyName":28},"841bb2d6-b0b2-4c3a-b6f0-ba59010c6c4c",[],[],{"title":2537,"openalex":2539},{"EN":2538},"Richard P. Rand",{"VOID":2540},"A5050355967",{"id":2542,"sortIndex":40,"researcher":28,"roles":2543,"affiliations":2544,"properties":2545,"displayName":2549,"givenName":28,"familyName":28},"b95f91ef-b7bd-494b-98ff-95dded05fc0e",[],[],{"orcid":2546,"title":2548,"openalex":2550},{"VOID":2547},"https:\u002F\u002Forcid.org\u002F0009-0001-4925-6096",{"EN":2549},"Alan C. Burton",{"VOID":2551},"A5018141960",{"id":2553,"sortIndex":123,"researcher":28,"roles":2554,"affiliations":2555,"properties":2556,"displayName":2560,"givenName":28,"familyName":28},"7286331c-e03c-4a8d-8a37-22c0b14f95c3",[],[],{"orcid":2557,"title":2559,"openalex":2561},{"VOID":2558},"https:\u002F\u002Forcid.org\u002F0000-0001-9897-4337",{"EN":2560},"Ing Ts",{"VOID":2562},"A5011170391",{"url":28,"publisher":2564,"properties":2614},{"id":868,"createTime":869,"updateTime":870,"relativeEntities":2565,"slug":872,"properties":2566,"entityType":25,"verifyStatus":884,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":32,"subjectFields":2571,"manageAffiliations":2588,"indexDatabases":2599,"url":958,"thumbnailPath":28,"statistic":28,"gsStatistic":28,"type":28,"analyzePriority":28},[],{"country":2567,"eissn":2568,"issn":2569,"title":2570},{"VOID":875},{"VOID":877},{"VOID":879},{"EN":881},[2572,2576,2580,2584],{"id":887,"createTime":28,"updateTime":28,"relativeEntities":2573,"label":2574,"description":2575,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":890},{},{"id":893,"createTime":28,"updateTime":28,"relativeEntities":2577,"label":2578,"description":2579,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":896},{},{"id":899,"createTime":28,"updateTime":28,"relativeEntities":2581,"label":2582,"description":2583,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":902},{},{"id":905,"createTime":28,"updateTime":28,"relativeEntities":2585,"label":2586,"description":2587,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":908},{},[2589,2594],{"id":912,"createTime":28,"updateTime":28,"relativeEntities":2590,"slug":28,"properties":2591,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2593,"statistic":28},[],{"title":2592},{"EN":916},[],{"id":919,"createTime":28,"updateTime":28,"relativeEntities":2595,"slug":28,"properties":2596,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2598,"statistic":28},[],{"title":2597},{"EN":923},[],[2600,2607],{"id":927,"indexDatabase":2601,"url":939,"indexYears":28,"academicFieldIds":2606,"indexDatabaseRanking":28},{"id":929,"createTime":28,"updateTime":28,"relativeEntities":2602,"label":2603,"description":2604,"key":936,"publicationTags":2605,"standard":28},[],{"EN":932,"VI":932},{"EN":934,"VI":935},[938,813],[941,942],{"id":944,"indexDatabase":2608,"url":950,"indexYears":951,"academicFieldIds":2613,"indexDatabaseRanking":957},{"id":775,"createTime":28,"updateTime":28,"relativeEntities":2609,"label":2610,"description":2611,"key":781,"publicationTags":2612,"standard":28},[],{"EN":778,"VI":778},{"EN":778,"VI":780},[783],[953,954,955,956],{"issue":2615,"pages":2616,"volume":2618},{"VOID":1483},{"VOID":2617},"257-267",{"VOID":2619},"43",234,{"total":2620,"publishYear":2622,"statisticByYear":2623},1965,{"2012":45,"2013":40,"2014":42,"2015":123,"2016":123,"2017":42,"2018":45,"2019":40,"2020":45,"2021":42,"2022":42,"2023":45,"2024":123},"1965-03-01",[938,957],[2627,2630,2634,2637],{"id":28,"text":2628,"url":28,"identifiers":2629},"1958, J. Exptl. Biol., 35, 893",{},{"id":28,"text":2631,"url":28,"identifiers":2632},"I' J. I I N G, 1953, J. Physiol. (London), 122, 570, 10.1113\u002Fjphysiol.1953.sp005021",{"doi":2633},"10.1113\u002Fjphysiol.1953.sp005021",{"id":28,"text":2635,"url":28,"identifiers":2636},"Can I., 1959, Hiochcni. Physioi., 37, 1247",{},{"id":28,"text":2638,"url":28,"identifiers":2639},"Iiosssra, 1959, J. Biochem. PhysioI., 37, 671",{},{"id":2641,"createTime":2642,"updateTime":2643,"relativeEntities":2644,"slug":2645,"properties":2646,"entityType":986,"verifyStatus":26,"verifyTime":2642,"verifyNote":1875,"languages":2663,"translateLanguages":2664,"viewCount":32,"primaryUrl":2665,"fullTextUrl":28,"authors":2666,"publicationType":1010,"publisherRelationship":2805,"citationCount":2862,"citationInfo":2863,"publishDate":2865,"publishYear":1983,"citationAnalyzeStatus":884,"lastCitationAnalyze":28,"indexDatabases":2866,"openAccess":28,"references":2867,"isForceReanalyzing":1076},"e9a735e6-bc19-4b39-9aed-4bebba0d073d","2024-10-08T16:10:34.844+00:00","2025-01-05T05:10:22.391+00:00",[],"Altered-expression-of-epithelial-junctional-proteins-in-atopic-asthma-possible-role-in-inflammation",{"mag":2647,"keywords":2649,"openalex":2651,"abstract":2653,"title":2656,"pm":2659,"doi":2661},{"VOID":2648},"2158340316",{"VI":2650},"bệnh hen phế quản dị ứng, tế bào biểu mô, protein liên kết, viêm nhiễm, bạch cầu ái toan",{"VOID":2652},"W2158340316",{"VI":2654,"EN":2655},"\u003Cjats:p> Các tế bào biểu mô tạo thành một hàng rào chặt chẽ chống lại các kích thích từ môi trường thông qua các mối nối chặt (TJs) và các mối nối dính (AJs). Các khiếm khuyết trong các protein TJ và AJ có thể gây ra những thay đổi về hình thái và tính toàn vẹn của biểu mô và có khả năng dẫn đến việc di chuyển nhanh hơn của các tế bào viêm qua biểu mô. Tính dễ tổn thương của biểu mô phế quản đã được ghi nhận ở các bệnh nhân hen phế quản, nhưng còn rất ít thông tin về sự biểu hiện của các protein TJ và AJ trong bệnh hen. Chúng tôi đã nghiên cứu sự biểu hiện của protein zonula occludens-1 (ZO-1) và các protein AJ như E-cadherin, α-catenin, và β-catenin trong các mẫu sinh thiết phế quản từ những đối tượng không dị ứng, không hen (khỏe mạnh) (n = 14), và những đối tượng hen phế quản dị ứng ổn định (n = 22) trong các điều kiện cơ bản. Việc nhuộm miễn dịch cho các protein này được semi-định lượng cho các thành phần tế bào riêng biệt. E-cadherin, α-catenin và β-catenin có mặt ở màng tế bào và ít hơn trong bào tương. Chỉ có β-catenin được tìm thấy trong nhân tế bào nhất quán với chức năng tiềm năng của nó như một yếu tố phiên mã. ZO-1 có mặt ở màng apicolateral của các tế bào bề mặt. Biểu hiện của α-catenin thấp hơn đáng kể ở những đối tượng mắc hen so với những người không mắc và có mối tương quan nghịch với số lượng bạch cầu ái toan trong biểu mô. Biểu hiện của ZO-1 và E-cadherin thấp hơn đáng kể ở những bệnh nhân hen so với những bệnh nhân không hen. Biểu hiện của β-catenin không khác biệt. Kết quả của chúng tôi gợi ý rằng sự biểu hiện thấp hơn của α-catenin, E-cadherin và (hoặc) ZO-1 trong các bệnh nhân hen phế quản dị ứng góp phần vào một hàng rào biểu mô đường thở bị khuyết tật và một lưu lượng bạch cầu ái toan cao hơn trong biểu mô. \u003C\u002Fjats:p>","\u003Cjats:p> Epithelial cells form a tight barrier against environmental stimuli via tight junctions (TJs) and adherence junctions (AJs). Defects in TJ and AJ proteins may cause changes in epithelial morphology and integrity and potentially lead to faster trafficking of inflammatory cells through the epithelium. Bronchial epithelial fragility has been reported in asthmatic patients, but little is known about the expression of TJ and AJ proteins in asthma. We studied epithelial expression of zonula occludens-1 (ZO-1) and AJ proteins E-cadherin, α-catenin, and β-catenin in bronchial biopsies from nonatopic nonasthmatic (healthy) subjects (n = 14), and stable atopic asthmatic subjects (n = 22) at baseline conditions. Immunostaining for these proteins was semi-quantified for separate cellular compartments. E-cadherin, α-catenin and β-catenin were present in the cellular membrane and less in the cytoplasm. Only β-catenin was present in the nucleus in agreement with its potential function as transcription factor. ZO-1 was present in the apicolateral membrane of superficial cells. α-Catenin expression was significantly lower in subjects with asthma than without and correlated inversely with numbers of eosinophils within the epithelium. ZO-1 and E-cadherin expression were significantly lower in asthmatic than in nonasthmatic subjects. Expression of β-catenin was not different. 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