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Journal of Medicine and Pharmacy","Tạp chí Y Dược học Cần Thơ",{"EN":487,"VI":488},"\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">04\u002F10\u002F2015 Ministry of Information and Communications allowed Can Tho journal of medicine and pharmacy to operate (102 \u002FGP-BTTTT)\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">07\u002F16\u002F2015 Can Tho journal of medicine and pharmacy is internationally recognized: ISSN 2354-1210\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">In 2016, The journal has been included in the list of medical science journals by The State Council for professorship which is awarded a work score of 0-0.5 points for a published article.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Can Tho Journal of Medicine and Pharmacy welcome original works that haven’t been submitted or published in other medical journals. Posts must contain content related to one of the journal’s categories.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The content published\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The journal is divided into 3 categories:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Scientific research article: are valuable scientific works, which have been researched and accepted.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Overview of medicine, biology and pharmacy: serving the objective of continuing training in the fields of medicine, biology and pharmacy; to systematize classical and modern knowledge.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Update information on new knowledge about medicine, biology, pharmacy in the country and in the world.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Scope\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Publication and introduction of scientific research in the fields:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Medicine (internal medicine, surgery, pediatrics, obstetrics and gynecology, odonto-stomatology, laboratory, oncology, traditional medicine, nursing).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Biology (genetics, biotechnology).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Pharmacology (pharmaceutics, drug quality analysis-control, synthetic pharmaceutical chemistry, biochemistry, pharmacognosy, botany, clinical pharmacy).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- To enhance the quality of undergraduate, postgraduate education, scientifically researching and meet the necessary treatment in hospital.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Introducing the updated domestic and oversea information about science technology to promote scientific research and exchanging technology in local, other universities.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Exchanging pharmaceutical and medical information for social health developing in the Mekong Delta and Vietnam.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The object\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Postgraduate students, student of Can Tho University of Medicine and Pharmacy, scientists from schools, research institutes, hospitals, health centers, pharmaceutical companies of the Mekong Delta; other provinces and regions in Vietnam and other country.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Address\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Headquarters of Can Tho Journal of Medicine and Pharmacy, located Scientific Research and International Cooperation Office: 179 Nguyen Van Cu Street, An Khanh Ward, Ninh Kieu District, Can Tho City, Vietnam.\u003C\u002Fspan>\u003C\u002Fp>","\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Ngày 16\u002F7\u002F2015, Tạp chí Y Dược học Cần Thơ được cấp chỉ số quốc tế: ISSN 2354-1210.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 4\u002F2016, Tạp chí đã được Hội đồng Giáo sư ngành Y đưa vào danh sách các tạp chí khoa học Y học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Năm 2020 Tạp chí Y Dược học Cần Thơ đã được phê duyệt vào danh mục của các Hội đồng Giáo sư ngành Dược học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ ra 12 số\u002Fnăm, 180-200 trang\u002Fsố.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 12\u002F2022 Tạp chí Y Dược học Cần Thơ là thành viên của hệ thống Crossref và từ tháng 01\u002F2023 tạp chí thực hiện bình duyệt online kín 2 chiều nhằm tăng tính minh bạch, tin cậy của các công trình nghiên cứu khoa học và đảm bảo tốt nhất chất lượng khoa học của bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ, mục đích và phạm vi của tạp chí\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ và mục đích hoạt động của tạp chí: xuất bản nhằm mục đích phổ biến kết quả từ các đề tài nghiên cứu khoa học; giao lưu trao đổi khoa học, chia sẻ kinh nghiệm, học tập, đồng thời cập nhật thông tin khoa học mới trong các lĩnh vực y, sinh, dược học trong và ngoài nước.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phạm vi của tạp chí: Tạp chí xuất bản được chia thành 3 chuyên mục: (i) Bài báo nghiên cứu khoa học là kết quả công trình nghiên cứu khoa học có giá trị đã được triển khai nghiên cứu, (ii) Bài tổng quan y, sinh, dược học: phục vụ mục tiêu đào tạo liên tục trong lĩnh vực y, sinh, dược học; nhằm hệ thống hóa những kiến thức kinh điển và hiện đại; (iii) Thông tin cập nhật kiến thức mới về y, sinh, dược học trong nước và trên thế giới.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Chính sách truy cập mở\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ áp dụng chính sách truy cập mở đối với các bài báo đã xuất bản đến với độc giả, nhằm mở rộng cơ hội tiếp cận các kết quả nghiên cứu chất lượng cao và tăng cường trao đổi kiến thức. Tạp chí đăng tải trực tuyến (miễn phí) toàn văn các bài báo được công bố trên website của Tạp chí (https:\u002F\u002Ftapchi.ctump.edu.vn).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đạo đức xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ cam kết tuân thủ đạo đức xuất bản phù hợp với các hướng dẫn và tiêu chuẩn của the Committee on Publication Ethics (COPE), tuân thủ các nguyên tắc của COPE’s Core Practices, Best Practices Guidelines for Journal Editors và Guidelines on Good Publication Practices.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Bản thảo bài báo chỉ được chấp nhận khi được tác giả chịu trách nhiệm chính cam kết các nội dung sau: Các nội dung của bản thảo chưa được đăng tải toàn bộ hoặc một phần ở các tạp chí khác; Tất cả các tác giả đều có đóng góp một cách đáng kể vào quá trình nghiên cứu hoặc chuẩn bị bản thảo và cùng chịu trách nhiệm về các nội dung của bản thảo; Tuân thủ các biện pháp đảm bảo đạo đức nghiên cứu (ví dụ thỏa thuận đồng ý tham gia nghiên cứu).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Cam kết bảo mật\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí cam kết thực hiện và tuân thủ các quy định của luật và các văn bản hướng dẫn liên quan đến bảo mật thông tin cá nhân trên không gian mạng. Các thông tin mà người dùng (tác giả, độc giả, biên tập viên, người phản biện) nhập vào các biểu mẫu trên Hệ thống Quản lý xuất bản trực tuyến của tạp chí chỉ được sử dụng vào các mục đích đã được tuyên bố rõ ràng và sẽ không được cung cấp cho bất kỳ bên thứ ba nào khác, hay dùng vào bất kỳ mục đích nào khác.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phí gửi bài\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng bài: 1.000.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng nhanh: 1.500.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với tác giả là cán bộ viên chức thuộc Trường Đại học Y Dược Cần Thơ thì được hỗ trợ 50% lệ phí gửi đăng bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với sinh viên thực hiện đề tài nghiên cứu khoa học cấp trường được hỗ trợ 100% lệ phí đăng bài ( Tác giả gửi đính kèm “ Quyết định về việc giao tổ chức thực hiện đề tài nghiên cứu khoa học cấp Trường của sinh viên”).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Hình thức nộp lệ phí:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Tiền mặt:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Nộp trực tiếp tại Phòng Tài chính - Kế toán, Trường Đại học Y Dược Cần Thơ, số 179 Nguyễn Văn Cừ, P. An Khánh, Q. Ninh Kiều, thành phố Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Chuyển khoản:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tên Tài khoản: Trường ĐHYD Cần Thơ, Số TK: 0111000115668, tại ngân hàng Vietcombank chi nhánh Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Thời gian: Áp dụng từ ngày 01\u002F02\u002F2023.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">* Phí gửi bài không được hoàn trả khi bài viết bị từ chối hoặc tác giả xin rút bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Quy trình phản biện bài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ thực hiện quy trình phản biện kín hai chiều nghiêm ngặt. Danh tính của những người phản biện không được tiết lộ cho các tác giả và ngược lại. Quy trình thẩm định bài báo đăng gồm các bước sau:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tiếp nhận bản thảo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tác giả liên hệ gửi bản thảo đến Tạp chí qua hệ thống trực tuyến tại website: https:\u002F\u002Ftapchi.ctump.edu.vn. Hướng dẫn về cách đăng ký, gửi bài và chuẩn bị bản thảo được cung cấp trên website của Tạp chí.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sàng lọc sơ bộ\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sau khi Tòa soạn nhận được bài báo của tác giả, Ban Thư ký sẽ tiến hành kiểm tra sơ bộ bài báo (các yêu cầu về nội dung và hình thức). Những bài báo không đúng quy cách hoặc có nội dung không phù hợp hoặc vi phạm bản quyền sẽ bị từ chối (Ban Thư ký thông báo phản hồi đến tác giả trong vòng 1 tuần). Những bài báo đủ điều kiện, được Ban Thư ký tòa soạn chuyển đến Ban Biên tập có cùng chuyên môn với nội dung bài báo để đề xuất người phản biện. Thời gian kể từ khi Ban Biên tập nhận bài báo đến khi đề xuất người phản biện bài báo chậm nhất là 5 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Vòng phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký gửi bài và yêu cầu phản biện đến 02 phản biện độc lập.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Các phản biện gởi nhận xét cho Ban Thư ký. Thời gian từ khi gửi bài cho phản biện đến khi nhận ý kiến của phản biện tối đa là 20 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xử ký kết quả phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Nếu ý kiến đồng ý cho đăng và không cần chỉnh sửa, Ban Thư ký tiếp tục đăng bài theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Nếu ý kiến đồng ý đăng và cần chỉnh sửa, Ban Thư ký sẽ thông tin đến tác giả chỉnh sửa theo yêu cầu của người phản biện. Thời gian chỉnh sửa và gửi lại kéo dài không quá 2 tuần, từ khi tác giả bài báo nhận được thông tin (Quá trình này có thể lặp lại tối đa 2 lần\u002F1 bài báo). Khi có sự thống nhất, đồng ý của người phản biện; bài báo được tiếp tục đăng theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Những bài báo có chất lượng không đạt yêu cầu, cả 2 phản biện không đồng ý cho đăng sẽ bị Tòa soạn từ chối đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký tổng hợp các bản thảo đã được tác giả hoàn thiện sau thẩm định trình Ban Biên tập xem xét, Tổng Biên tập phê duyệt, quyết định bài đăng theo các tiêu chí: sự phù hợp nội dung với tôn chỉ và mục đích, thể loại bài viết (ưu tiên các bài có bài có nghiên cứu chuyên sâu, hàm lượng khoa học cao), đóng góp mới bài báo, bài báo được ưu tiên đăng trong số gần nhất của Tạp chí theo thứ tự: tính thời sự, chất lượng bài báo và thời gian gửi bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Ban Biên tập và Ban Thư ký biên tập bản thảo, chế bản, đọc rà soát lỗi. Thời gian hoàn thành từ 10-15 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Ban Thư ký có trách nhiệm thông báo cho tác giả bài báo (bằng e-mail) về tình hình phê duyệt bài báo, thời gian, số kỳ, tập xuất bản bài báo theo qui định.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">4. Danh sách bài báo theo số Tạp chí được in ấn và phát hành trong năm định kỳ được công bố chính thức trên website: https:\u002F\u002Ftapchi.ctump.edu.vn\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>",{"VOID":490},"wcQ1uqwAAAAJ","2023-05-30T08:17:21.868+00:00",[],[494],{"id":495,"createTime":28,"updateTime":28,"relativeEntities":496,"slug":28,"properties":497,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":507,"parentIds":508,"statistic":28},"6413896b-eca9-442b-a73f-182a58a0ce40",[],{"title":498,"address":501,"country":504,"abbreviation":505},{"EN":499,"VI":500},"Can Tho University of Medicine and Pharmacy","Trường Đại học Y Dược Cần Thơ",{"EN":502,"VI":503},"No 179, Nguyen Van Cu street, An Khanh ward, Ninh Kieu district, Can Tho city, Vietnam","Số 179, đường Nguyễn Văn Cừ, phường An Khánh, quận Ninh Kiều, thành phố Cần Thơ, Việt Nam",{"VOID":15},{"VOID":506},"ctump","http:\u002F\u002Fwww.ctump.edu.vn\u002F",[],[],"https:\u002F\u002Ftapchi.ctump.edu.vn\u002Findex.php\u002Fctump",{"impactFactor":32,"impactFactorByYear":512,"i10Index":32,"i10IndexLast5Year":32,"totalPublication":514,"totalPublicationByYear":515,"totalCitation":520,"totalCitationByYear":521,"totalCitationPerPublication":108,"totalCitationPerPublicationByYear":523,"hindexLast5Year":45,"hindex":45},{"2022":513,"2023":111,"2024":106},0.01,1556,{"2020":47,"2021":516,"2022":517,"2023":518,"2024":519,"2025":122},57,306,801,358,161,{"2021":146,"2022":280,"2023":522},99,{"2021":524,"2022":318,"2023":104},0.23,{"impactFactor":28,"impactFactorByYear":28,"i10Index":123,"i10IndexLast5Year":123,"totalPublication":526,"totalPublicationByYear":527,"totalCitation":526,"totalCitationByYear":528,"totalCitationPerPublication":40,"totalCitationPerPublicationByYear":531,"hindexLast5Year":49,"hindex":49},476,{"0":205,"2019":123,"2021":139,"2022":459,"2023":451,"2024":357,"2025":49,"2026":48},{"2021":42,"2022":123,"2023":161,"2024":529,"2025":360,"2026":530},136,83,{"2021":105,"2022":513,"2023":532,"2024":127,"2025":533,"2026":534},0.62,25.43,13.83,{"id":536,"createTime":537,"updateTime":382,"relativeEntities":538,"slug":539,"properties":540,"entityType":25,"verifyStatus":26,"verifyTime":28,"verifyNote":28,"languages":552,"translateLanguages":28,"viewCount":133,"subjectFields":553,"manageAffiliations":554,"indexDatabases":555,"url":556,"thumbnailPath":557,"statistic":558,"gsStatistic":594,"type":55,"analyzePriority":28},"6984a56a-db70-403b-9cc4-4013e1ceaffa","2023-05-09T06:47:40.346+00:00",[],"T%E1%BA%A1p%20ch%C3%AD%20Nghi%C3%AAn%20c%E1%BB%A9u%20n%C6%B0%E1%BB%9Bc%20ngo%C3%A0i",{"country":541,"issn":542,"title":544,"introduce":547,"gsId":550},{"VOID":15},{"VOID":543},"25252445",{"EN":545,"VI":546},"VNU Journal of Foreign Studies","Tạp chí Nghiên cứu nước ngoài",{"EN":548,"VI":549},"{\"ops\":[{\"insert\":\"\\n\\nThe \\n\"},{\"attributes\":{\"italic\":true},\"insert\":\"VNU Journal of Science\"},{\"insert\":\"\\n was established in 1985 for the publication of national and international research papers in all fields of natural sciences and technology, social sciences and humanities. 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Engl. J. Med., 343, 544, 10.1056\u002FNEJM200008243430804\nCazzola, 1983, Iron loading in congenital dyserythropoietic anaemias and congenital sideroblastic anaemias, Br. J. Haematol., 54, 649, 10.1111\u002Fj.1365-2141.1983.tb02145.x\nPeto, 1983, Iron overload in mild sideroblastic anaemias, Lancet, ii, 375, 10.1016\u002FS0140-6736(83)91498-8\nSimon, 1985, Is the HLA-linked haemochromatosis allele implicated in idiopathic refractory sideroblastic anaemia?, Br. J. Haematol., 60, 75, 10.1111\u002Fj.1365-2141.1985.tb07387.x\nBlacklock, 1981, Serum ferritin in patients with hereditary spherocytosis, Br. J. Haematol., 49, 117, 10.1111\u002Fj.1365-2141.1981.tb07204.x\nEdwards, 1982, Iron overload in hereditary spherocytosis: Association with HLA-linked hemochromatosis, Am. J. Haematol., 13, 101, 10.1002\u002Fajh.2830130202\nFargion, 1986, Association of hereditary spherocytosis and idiopatic hemochromatosis. A synergistic effect in determining iron overload, Am. J. Clin. Pathol., 86, 645, 10.1093\u002Fajcp\u002F86.5.645\nMohler, 1986, Case report: Hemochromatosis heterozygotes may have significant iron overload when they also have hereditary spherocytosis, Am. J. Med. Sci., 292, 320, 10.1097\u002F00000441-198611000-00014\nLin, 1990, Iron overload in untransfused patients with haemoglobin H disease, Acta Haematol., 33, 137\nZanella, 1989, Relative iron deficiency in hereditary spherocytosis, Am. J. Hematol., 31, 81, 10.1002\u002Fajh.2830310202\nBaronciani, 1996, Study of the molecular defects in glucose phosphate isomerase deficient patients affected by chronic hemolytic anemia, Blood, 88, 2306, 10.1182\u002Fblood.V88.6.2306.bloodjournal8862306\nFiorelli, 2000, Chronic non-spherocytic haemolytic disorders associated with glucose-6-phosphate dehydrogenase variants, Balliere's Clin. Haematol., 13, 39, 10.1053\u002Fbeha.1999.0056\nNoel, 1979, Hemochromatose et deficit en pyruvate kinase, Ann. Med. Interne., 130, 679\nSalem, 1980, Iron overload in congenital erythrocyte pyruvate kinase deficiency, Med. J. Aust., i, 531, 10.5694\u002Fj.1326-5377.1980.tb135097.x\nVives-Corrons, 1980, Hereditary erythrocyte pyruvate-kinase (PK) deficiency and chronic hemolytic anemia: Clinical, genetic and molecular studies in six new Spanish patients, Hum. Genet., 53, 401, 10.1007\u002FBF00287063\nLakomek, 1984, Erythrocyte pyruvate kinase deficiency: Characterization of a new variant (PK Aarau), Blut, 48, 123, 10.1007\u002FBF00320334\nRowbotham, 1984, Iron overload associated with congenital pyruvate kinase deficiency and high dose ascorbic acid ingestion, Aust. N. Z. J. Med., 14, 667, 10.1111\u002Fj.1445-5994.1984.tb05022.x\nZanella, 1988, Erythrocyte pyruvate kinase deficiency: 11 new cases, Br. J. Haematol., 69, 399, 10.1111\u002Fj.1365-2141.1988.tb02380.x\nBoivin, 1990, La surcharge en fer dans l'anémie hémolytique congenitale par deficit en pyruvate kinase. Une complication tardive majeure, Presse Med., 19, 1087\nBrabec, 1990, Serum ferritin in patients with various haemolytic disorders, Folia Haematol. Int. Mag. Klin. Morphol. Blutforsch., 117, 219\nDe Braekeleer, 1991, Hemochromatosis and pyruvate-kinase deficiency—Report of a case and review of the literature, Ann. Hematol., 62, 188, 10.1007\u002FBF01703147\nZanella, 1993, Iron status in red cell pyruvate kinase deficiency: Study of Italian cases, Br. J. Haematol., 83, 485, 10.1111\u002Fj.1365-2141.1993.tb04675.x\nEdwards, 1988, Prevalence of hemochromatosis among 11,065 presumably healthy blood donors, N. Engl. J. Med., 318, 1355, 10.1056\u002FNEJM198805263182103\nVelati, 1990, Prevalence of idiopathic haemochromatosis in Italy: Study of 1301 blood donors, Haematologica, 75, 309\nWorwood, 1994, Genetics of haemochromatosis, Balliere's Clin. Haematol., 7, 903, 10.1016\u002FS0950-3536(05)80130-1\nWorwood, 1997, A simple genetic test identifies 90% of UK patients with haemochromatosis, Gut, 41, 841, 10.1136\u002Fgut.41.6.841\nFeder, 1996, A novel MHC class I-like gene is mutated in patients with hereditary hemochromatosis, Nat. Genet., 13, 399, 10.1038\u002Fng0896-399\nFeder, 1997, The hemochromatosis founder mutation in HLA-H disrupts β2-microglobulin interaction and surface expression, J. Biol. Chem., 272, 14025, 10.1074\u002Fjbc.272.22.14025\nFeder, 1998, The hemochromatosis gene product complexes with the transferrin receptor and lowers its affinity for ligand binding, Proc. Natl. Acad. Sci. USA, 95, 1472, 10.1073\u002Fpnas.95.4.1472\nParkkila, 1997, Association of the transferrin receptor in human placenta with HFE, the protein defective in hereditary hemochromatosis, Proc. Natl. Acad. Sci. USA, 94, 13198, 10.1073\u002Fpnas.94.24.13198\nParkkila, 1997, Immunochemistry of HLA-H, the protein defective in patients with hemochromatosis, reveals unique pattern of expression in gastrointestinal tract, Proc. Natl. Acad. Sci. USA, 94, 2534, 10.1073\u002Fpnas.94.6.2534\nSalter-Cid, 1999, Transferrin receptor is negatively modulated by the hemochromatosis protein HFE: Implications for cellular iron homeostasis, Proc. Natl. Acad. Sci. USA, 96, 5434, 10.1073\u002Fpnas.96.10.5434\nBeutler, 1996, Mutation analysis in hereditary hemochromatosis, Blood Cells Mol. Dis., 22, 187, 10.1006\u002Fbcmd.1996.0027\nBorot, 1997, Mutations in the MHC class I-like candidate gene for hemochromatosis in French patients, Immunogenetics, 45, 320, 10.1007\u002Fs002510050211\nCarella, 1997, Mutation analysis of HLA-H gene in Italian hemochromatosis patients, Am. J. Hum. Genet., 60, 828\nPiperno, 1984, Iron overload in hereditary spherocytosis, Haematologica, 69, 90\nBacon, 1999, HFE genotype in patients with hemochromatosis and in patients with liver disease, Ann. Intern. Med., 130, 953, 10.7326\u002F0003-4819-130-12-199906150-00002\nBeutler, 1997, The significance of the 187G (H63D) mutation in hemochromatosis, Am. J. Hum. Genet., 60, 828\nBurt, 1998, The significance of hemochromatosis gene mutations in the general population: Implications for screening, Gut, 43, 830, 10.1136\u002Fgut.43.6.830\nRossi, 2001, Effect of hemochromatosis genotype and lifestyle factors on iron and red cell indices in a community population, Clin. Chem., 47, 202, 10.1093\u002Fclinchem\u002F47.2.202\nDatz, 1998, Heterozygosity for the C282Y mutation in the hemochromatosis gene is associated with increased serum iron, transferrin saturation and hemoglobin in young women: A protective role against iron deficiency, Clin. Chem., 44, 2429, 10.1093\u002Fclinchem\u002F44.12.2429\nRossi, 2000, Compound heterozygous hemochromatosis genotype predicts increased iron and erythrocyte indices in women, Clin. Chem., 46, 162, 10.1093\u002Fclinchem\u002F46.2.162\nZanella, 1997, Molecular characterization of PK-LR gene in pyruvate kinase-deficient Italian patients, Blood, 89, 3847, 10.1182\u002Fblood.V89.10.3847\nZanella, 2000, Red cell pyruvate kinase deficiency: From genetics to clinical manifestations, Balliere's Clin. Haematol., 13, 57, 10.1053\u002Fbeha.1999.0057\nDacie, 1984\nBeutler, 1984\nNathan, 1998\nDeugnier, 1992, Liver pathology in genetic hemochromatosis: A review of 135 homozygous cases and their bioclinical correlations, Gastroenterology, 102, 2050, 10.1016\u002F0016-5085(92)90331-R\nPippard, 1982, Ferrioxamine excretion in iron-loaded man, Blood, 60, 288, 10.1182\u002Fblood.V60.2.288.288\nPootrakul, 1980, Increased transferrin iron saturation in splenectomized thalassaemic patients, Br. J. 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Chem., 269, 10312, 10.1016\u002FS0021-9258(17)34062-0\nMarklund, 1994, The phenotype of a “Cdc2 kinase target site-deficient” mutant of oncoprotein 18 reveals a role of this protein in cell cycle control, J. Biol. Chem., 269, 30626, 10.1016\u002FS0021-9258(18)43860-4\nLuo, 1994, Atweh, GF., Regulation of phosphoprotein p18 in leukemic cells. Cell cycle regulated phosphorylation by p34cdc2 kinase, J. Biol. Chem., 269, 10312, 10.1016\u002FS0021-9258(17)34062-0\nBelmont, 1996, Identification of a protein that interacts with tubulin dimers and increases the catastrophe rate of microtubules, Cell, 84, 623, 10.1016\u002FS0092-8674(00)81037-5\nJiang, 2008, Proteins induced by telomere dysfunction and DNA damage represent biomarkers of human aging and disease, Proc. Natl. Acad. Sci. U. S. A., 105, 11299, 10.1073\u002Fpnas.0801457105\nUchida, 2014, Learning-induced and stathmin-dependent changes in microtubule stability are critical for memory and disrupted in ageing, Nat. Commun., 5, 4389, 10.1038\u002Fncomms5389\nSaetre, 2011, Age-related changes in gene expression are accelerated in Alzheimer’s disease, Synapse, 65, 971, 10.1002\u002Fsyn.20933\nChang, 2001, Oncoprotein 18 levels and phosphorylation mediate megakaryocyte polyploidization in human erythroleukemia cells, Proteomics, 1, 1415, 10.1002\u002F1615-9861(200111)1:11\u003C1415::AID-PROT1415>3.0.CO;2-F\nIancu-Rubin, 2011, Down-regulation of stathmin expression is required for megakaryocyte maturation and platelet production, Blood, 117, 4580, 10.1182\u002Fblood-2010-09-305540\nRabilloud, 1995, Induction of stathmin expression during erythropoietic differentiation, Cell Growth Differ, 6, 1307\nRubin, 2003, Stathmin expression and megakaryocyte differentiation: a potential role in polyploidy, Exp. Hematol., 31, 389, 10.1016\u002FS0301-472X(03)00043-2\nBrattsand, 1993, Quantitative analysis of the expression and regulation of an activation-regulated phosphoprotein (oncoprotein 18) in normal and neoplastic cells, Leukemia, 7, 569\nLiu, 2011, Microtubule assembly affects bone mass by regulating both osteoblast and osteoclast functions: stathmin deficiency produces an osteopenic phenotype in mice, J. Bone Miner. Res., 26, 2052, 10.1002\u002Fjbmr.419\nOzon, 1997, The stathmin family—molecular and biological characterization of novel mammalian proteins expressed in the nervous system, Eur. J. Biochem., 248, 794, 10.1111\u002Fj.1432-1033.1997.t01-2-00794.x\nRowlands, 1995, Stathmin expression is a feature of proliferating cells of most, if not all, cell lineages, Lab. 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Cell Sci., 114, 909, 10.1242\u002Fjcs.114.5.909\nAlli, 2007, Reversal of stathmin-mediated resistance to paclitaxel and vinblastine in human breast carcinoma cells, Mol. Pharmacol., 71, 1233, 10.1124\u002Fmol.106.029702\nMistry, 2006, Therapeutic interactions between stathmin inhibition and chemotherapeutic agents in prostate cancer, Mol. Cancer Ther., 5, 3248, 10.1158\u002F1535-7163.MCT-06-0227\nAlli, 2002, Effect of stathmin on the sensitivity to antimicrotubule drugs in human breast cancer, Cancer Res., 62, 6864\nIancu, 2000, Taxol and anti-stathmin therapy: a synergistic combination that targets the mitotic spindle, Cancer Res., 60, 3537\nMachado-Neto, 2014, Stathmin 1 is involved in the highly proliferative phenotype of high-risk myelodysplastic syndromes and acute leukemia cells, Leuk. 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Cell Biol., 6, 259, 10.1006\u002Fscel.1995.0035\nTaylor, 2001, Endothelial cell protein C receptor plays an important role in protein C activation in vivo, Blood, 97, 1685, 10.1182\u002Fblood.V97.6.1685\nWalker, 1979, The inhibition of blood coagulation by activated Protein C through the selective inactivation of activated Factor V, Biochim. Biophys. Acta, 571, 333, 10.1016\u002F0005-2744(79)90103-7\nFulcher, 1984, Proteolytic inactivation of human factor VIII procoagulant protein by activated human protein C and its analogy with factor V, Blood, 63, 486, 10.1182\u002Fblood.V63.2.486.486\nHanson, 1993, Antithrombotic effects of thrombin-induced activation of endogenous protein C in primates, J. Clin. Invest., 92, 2003, 10.1172\u002FJCI116795\nGriffin, 2002, Activated protein C, Semin. Hematol., 39, 197, 10.1053\u002Fshem.2002.34093\nBernard, 2001, Efficacy and safety of recombinant human activated protein C for severe sepsis, N. Engl. J. 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Biochem.Tokyo, 111, 491, 10.1093\u002Foxfordjournals.jbchem.a123785\nShibata, 2001, Anti-inflammatory, antithrombotic and neuroprotective effects of activated protein C in a murine model of focal ischemic stroke, Circulation, 103, 1799, 10.1161\u002F01.CIR.103.13.1799\nChase, 1967, p-Nitrophenyl-p′-guanidinobenzoate HCl, Biochem. Biophys. Res. Commun., 29, 508, 10.1016\u002F0006-291X(67)90513-X\nGale, 2000, The autolysis loop of activated protein C interacts with factor Va and differentiates between the Arg506 and Arg306 cleavage sites, Blood, 96, 585, 10.1182\u002Fblood.V96.2.585\nTabrizi, 1999, Tissue plasminogen activator (tPA)deficiency exacerbates cerebrovascular fibrin deposition and brain injury in a murine stroke model, Arterioscler. Thromb. Vasc. Biol., 19, 2801, 10.1161\u002F01.ATV.19.11.2801\nJalbert, 1998, Nucleotide structure and characterization of the murine gene encoding anticoagulant protein C, Thromb. Haemost., 79, 310, 10.1055\u002Fs-0037-1615006\nFoster, 1985, The nucleotide sequence of the gene for human protein C, Proc. Natl. Acad. Sci. USA, 82, 4673, 10.1073\u002Fpnas.82.14.4673\nWalker, 1981, Regulation of bovine-activated protein C by protein S, Thromb. Res., 22, 321, 10.1016\u002F0049-3848(81)90125-0\nHolly, 1994, Resistance to inhibition by alpha-1-anti-trypsin and species specificity of a chimeric human\u002Fbovine protein C, Biochemistry, 33, 1876, 10.1021\u002Fbi00173a034\nShen, 1997, Synergistic cofactor function of factor V and protein S to activated protein C in the inactivation of the factor VIIIa-factor IXa complex—species specific interactions of components of the protein C anticoagulant system, Thromb. Haemost., 78, 1030, 10.1055\u002Fs-0038-1657682\nGruber, 1990, Inhibition of thrombus formation by activated recombinant protein C in a primate model of arterial thrombosis, Circulation, 82, 578, 10.1161\u002F01.CIR.82.2.578\nTaylor, 1987, Protein C prevents the coagulopathic and lethal effects of Escherichia coli infusion in the baboon, J. Clin. Invest., 79, 918, 10.1172\u002FJCI112902\nJalbert, 1998, Inactivation of the gene for anticoagulant protein C causes lethal perinatal consumptive coagulopathy in mice, J. Clin. Invest., 102, 1481, 10.1172\u002FJCI3011\nT. Cheng, et al., Activated protein C blocks p53-mediated apoptosis in ischemic human brain endothelium and is neuroprotective, Nat. 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