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Journal of Medicine and Pharmacy","Tạp chí Y Dược học Cần Thơ",{"EN":487,"VI":488},"\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">04\u002F10\u002F2015 Ministry of Information and Communications allowed Can Tho journal of medicine and pharmacy to operate (102 \u002FGP-BTTTT)\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">07\u002F16\u002F2015 Can Tho journal of medicine and pharmacy is internationally recognized: ISSN 2354-1210\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">In 2016, The journal has been included in the list of medical science journals by The State Council for professorship which is awarded a work score of 0-0.5 points for a published article.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Can Tho Journal of Medicine and Pharmacy welcome original works that haven’t been submitted or published in other medical journals. Posts must contain content related to one of the journal’s categories.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The content published\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The journal is divided into 3 categories:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Scientific research article: are valuable scientific works, which have been researched and accepted.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Overview of medicine, biology and pharmacy: serving the objective of continuing training in the fields of medicine, biology and pharmacy; to systematize classical and modern knowledge.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Update information on new knowledge about medicine, biology, pharmacy in the country and in the world.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Scope\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Publication and introduction of scientific research in the fields:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Medicine (internal medicine, surgery, pediatrics, obstetrics and gynecology, odonto-stomatology, laboratory, oncology, traditional medicine, nursing).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Biology (genetics, biotechnology).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Pharmacology (pharmaceutics, drug quality analysis-control, synthetic pharmaceutical chemistry, biochemistry, pharmacognosy, botany, clinical pharmacy).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- To enhance the quality of undergraduate, postgraduate education, scientifically researching and meet the necessary treatment in hospital.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Introducing the updated domestic and oversea information about science technology to promote scientific research and exchanging technology in local, other universities.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Exchanging pharmaceutical and medical information for social health developing in the Mekong Delta and Vietnam.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The object\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Postgraduate students, student of Can Tho University of Medicine and Pharmacy, scientists from schools, research institutes, hospitals, health centers, pharmaceutical companies of the Mekong Delta; other provinces and regions in Vietnam and other country.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Address\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Headquarters of Can Tho Journal of Medicine and Pharmacy, located Scientific Research and International Cooperation Office: 179 Nguyen Van Cu Street, An Khanh Ward, Ninh Kieu District, Can Tho City, Vietnam.\u003C\u002Fspan>\u003C\u002Fp>","\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Ngày 16\u002F7\u002F2015, Tạp chí Y Dược học Cần Thơ được cấp chỉ số quốc tế: ISSN 2354-1210.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 4\u002F2016, Tạp chí đã được Hội đồng Giáo sư ngành Y đưa vào danh sách các tạp chí khoa học Y học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Năm 2020 Tạp chí Y Dược học Cần Thơ đã được phê duyệt vào danh mục của các Hội đồng Giáo sư ngành Dược học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ ra 12 số\u002Fnăm, 180-200 trang\u002Fsố.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 12\u002F2022 Tạp chí Y Dược học Cần Thơ là thành viên của hệ thống Crossref và từ tháng 01\u002F2023 tạp chí thực hiện bình duyệt online kín 2 chiều nhằm tăng tính minh bạch, tin cậy của các công trình nghiên cứu khoa học và đảm bảo tốt nhất chất lượng khoa học của bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ, mục đích và phạm vi của tạp chí\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ và mục đích hoạt động của tạp chí: xuất bản nhằm mục đích phổ biến kết quả từ các đề tài nghiên cứu khoa học; giao lưu trao đổi khoa học, chia sẻ kinh nghiệm, học tập, đồng thời cập nhật thông tin khoa học mới trong các lĩnh vực y, sinh, dược học trong và ngoài nước.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phạm vi của tạp chí: Tạp chí xuất bản được chia thành 3 chuyên mục: (i) Bài báo nghiên cứu khoa học là kết quả công trình nghiên cứu khoa học có giá trị đã được triển khai nghiên cứu, (ii) Bài tổng quan y, sinh, dược học: phục vụ mục tiêu đào tạo liên tục trong lĩnh vực y, sinh, dược học; nhằm hệ thống hóa những kiến thức kinh điển và hiện đại; (iii) Thông tin cập nhật kiến thức mới về y, sinh, dược học trong nước và trên thế giới.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Chính sách truy cập mở\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ áp dụng chính sách truy cập mở đối với các bài báo đã xuất bản đến với độc giả, nhằm mở rộng cơ hội tiếp cận các kết quả nghiên cứu chất lượng cao và tăng cường trao đổi kiến thức. Tạp chí đăng tải trực tuyến (miễn phí) toàn văn các bài báo được công bố trên website của Tạp chí (https:\u002F\u002Ftapchi.ctump.edu.vn).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đạo đức xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ cam kết tuân thủ đạo đức xuất bản phù hợp với các hướng dẫn và tiêu chuẩn của the Committee on Publication Ethics (COPE), tuân thủ các nguyên tắc của COPE’s Core Practices, Best Practices Guidelines for Journal Editors và Guidelines on Good Publication Practices.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Bản thảo bài báo chỉ được chấp nhận khi được tác giả chịu trách nhiệm chính cam kết các nội dung sau: Các nội dung của bản thảo chưa được đăng tải toàn bộ hoặc một phần ở các tạp chí khác; Tất cả các tác giả đều có đóng góp một cách đáng kể vào quá trình nghiên cứu hoặc chuẩn bị bản thảo và cùng chịu trách nhiệm về các nội dung của bản thảo; Tuân thủ các biện pháp đảm bảo đạo đức nghiên cứu (ví dụ thỏa thuận đồng ý tham gia nghiên cứu).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Cam kết bảo mật\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí cam kết thực hiện và tuân thủ các quy định của luật và các văn bản hướng dẫn liên quan đến bảo mật thông tin cá nhân trên không gian mạng. Các thông tin mà người dùng (tác giả, độc giả, biên tập viên, người phản biện) nhập vào các biểu mẫu trên Hệ thống Quản lý xuất bản trực tuyến của tạp chí chỉ được sử dụng vào các mục đích đã được tuyên bố rõ ràng và sẽ không được cung cấp cho bất kỳ bên thứ ba nào khác, hay dùng vào bất kỳ mục đích nào khác.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phí gửi bài\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng bài: 1.000.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng nhanh: 1.500.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với tác giả là cán bộ viên chức thuộc Trường Đại học Y Dược Cần Thơ thì được hỗ trợ 50% lệ phí gửi đăng bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với sinh viên thực hiện đề tài nghiên cứu khoa học cấp trường được hỗ trợ 100% lệ phí đăng bài ( Tác giả gửi đính kèm “ Quyết định về việc giao tổ chức thực hiện đề tài nghiên cứu khoa học cấp Trường của sinh viên”).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Hình thức nộp lệ phí:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Tiền mặt:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Nộp trực tiếp tại Phòng Tài chính - Kế toán, Trường Đại học Y Dược Cần Thơ, số 179 Nguyễn Văn Cừ, P. An Khánh, Q. Ninh Kiều, thành phố Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Chuyển khoản:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tên Tài khoản: Trường ĐHYD Cần Thơ, Số TK: 0111000115668, tại ngân hàng Vietcombank chi nhánh Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Thời gian: Áp dụng từ ngày 01\u002F02\u002F2023.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">* Phí gửi bài không được hoàn trả khi bài viết bị từ chối hoặc tác giả xin rút bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Quy trình phản biện bài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ thực hiện quy trình phản biện kín hai chiều nghiêm ngặt. Danh tính của những người phản biện không được tiết lộ cho các tác giả và ngược lại. Quy trình thẩm định bài báo đăng gồm các bước sau:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tiếp nhận bản thảo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tác giả liên hệ gửi bản thảo đến Tạp chí qua hệ thống trực tuyến tại website: https:\u002F\u002Ftapchi.ctump.edu.vn. Hướng dẫn về cách đăng ký, gửi bài và chuẩn bị bản thảo được cung cấp trên website của Tạp chí.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sàng lọc sơ bộ\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sau khi Tòa soạn nhận được bài báo của tác giả, Ban Thư ký sẽ tiến hành kiểm tra sơ bộ bài báo (các yêu cầu về nội dung và hình thức). Những bài báo không đúng quy cách hoặc có nội dung không phù hợp hoặc vi phạm bản quyền sẽ bị từ chối (Ban Thư ký thông báo phản hồi đến tác giả trong vòng 1 tuần). Những bài báo đủ điều kiện, được Ban Thư ký tòa soạn chuyển đến Ban Biên tập có cùng chuyên môn với nội dung bài báo để đề xuất người phản biện. Thời gian kể từ khi Ban Biên tập nhận bài báo đến khi đề xuất người phản biện bài báo chậm nhất là 5 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Vòng phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký gửi bài và yêu cầu phản biện đến 02 phản biện độc lập.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Các phản biện gởi nhận xét cho Ban Thư ký. Thời gian từ khi gửi bài cho phản biện đến khi nhận ý kiến của phản biện tối đa là 20 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xử ký kết quả phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Nếu ý kiến đồng ý cho đăng và không cần chỉnh sửa, Ban Thư ký tiếp tục đăng bài theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Nếu ý kiến đồng ý đăng và cần chỉnh sửa, Ban Thư ký sẽ thông tin đến tác giả chỉnh sửa theo yêu cầu của người phản biện. Thời gian chỉnh sửa và gửi lại kéo dài không quá 2 tuần, từ khi tác giả bài báo nhận được thông tin (Quá trình này có thể lặp lại tối đa 2 lần\u002F1 bài báo). Khi có sự thống nhất, đồng ý của người phản biện; bài báo được tiếp tục đăng theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Những bài báo có chất lượng không đạt yêu cầu, cả 2 phản biện không đồng ý cho đăng sẽ bị Tòa soạn từ chối đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký tổng hợp các bản thảo đã được tác giả hoàn thiện sau thẩm định trình Ban Biên tập xem xét, Tổng Biên tập phê duyệt, quyết định bài đăng theo các tiêu chí: sự phù hợp nội dung với tôn chỉ và mục đích, thể loại bài viết (ưu tiên các bài có bài có nghiên cứu chuyên sâu, hàm lượng khoa học cao), đóng góp mới bài báo, bài báo được ưu tiên đăng trong số gần nhất của Tạp chí theo thứ tự: tính thời sự, chất lượng bài báo và thời gian gửi bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Ban Biên tập và Ban Thư ký biên tập bản thảo, chế bản, đọc rà soát lỗi. Thời gian hoàn thành từ 10-15 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Ban Thư ký có trách nhiệm thông báo cho tác giả bài báo (bằng e-mail) về tình hình phê duyệt bài báo, thời gian, số kỳ, tập xuất bản bài báo theo qui định.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">4. Danh sách bài báo theo số Tạp chí được in ấn và phát hành trong năm định kỳ được công bố chính thức trên website: https:\u002F\u002Ftapchi.ctump.edu.vn\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>",{"VOID":490},"wcQ1uqwAAAAJ","2023-05-30T08:17:21.868+00:00",[],[494],{"id":495,"createTime":28,"updateTime":28,"relativeEntities":496,"slug":28,"properties":497,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":507,"parentIds":508,"statistic":28},"6413896b-eca9-442b-a73f-182a58a0ce40",[],{"title":498,"address":501,"country":504,"abbreviation":505},{"EN":499,"VI":500},"Can Tho University of Medicine and Pharmacy","Trường Đại học Y Dược Cần Thơ",{"EN":502,"VI":503},"No 179, Nguyen Van Cu street, An Khanh ward, Ninh Kieu district, Can Tho city, Vietnam","Số 179, đường Nguyễn Văn Cừ, phường An Khánh, quận Ninh Kiều, thành phố Cần Thơ, Việt Nam",{"VOID":15},{"VOID":506},"ctump","http:\u002F\u002Fwww.ctump.edu.vn\u002F",[],[],"https:\u002F\u002Ftapchi.ctump.edu.vn\u002Findex.php\u002Fctump",{"impactFactor":32,"impactFactorByYear":512,"i10Index":32,"i10IndexLast5Year":32,"totalPublication":514,"totalPublicationByYear":515,"totalCitation":520,"totalCitationByYear":521,"totalCitationPerPublication":108,"totalCitationPerPublicationByYear":523,"hindexLast5Year":45,"hindex":45},{"2022":513,"2023":111,"2024":106},0.01,1556,{"2020":47,"2021":516,"2022":517,"2023":518,"2024":519,"2025":122},57,306,801,358,161,{"2021":146,"2022":280,"2023":522},99,{"2021":524,"2022":318,"2023":104},0.23,{"impactFactor":28,"impactFactorByYear":28,"i10Index":123,"i10IndexLast5Year":123,"totalPublication":526,"totalPublicationByYear":527,"totalCitation":526,"totalCitationByYear":528,"totalCitationPerPublication":40,"totalCitationPerPublicationByYear":531,"hindexLast5Year":49,"hindex":49},476,{"0":205,"2019":123,"2021":139,"2022":459,"2023":451,"2024":357,"2025":49,"2026":48},{"2021":42,"2022":123,"2023":161,"2024":529,"2025":360,"2026":530},136,83,{"2021":105,"2022":513,"2023":532,"2024":127,"2025":533,"2026":534},0.62,25.43,13.83,{"id":536,"createTime":537,"updateTime":382,"relativeEntities":538,"slug":539,"properties":540,"entityType":25,"verifyStatus":26,"verifyTime":28,"verifyNote":28,"languages":552,"translateLanguages":28,"viewCount":133,"subjectFields":553,"manageAffiliations":554,"indexDatabases":555,"url":556,"thumbnailPath":557,"statistic":558,"gsStatistic":594,"type":55,"analyzePriority":28},"6984a56a-db70-403b-9cc4-4013e1ceaffa","2023-05-09T06:47:40.346+00:00",[],"T%E1%BA%A1p%20ch%C3%AD%20Nghi%C3%AAn%20c%E1%BB%A9u%20n%C6%B0%E1%BB%9Bc%20ngo%C3%A0i",{"country":541,"issn":542,"title":544,"introduce":547,"gsId":550},{"VOID":15},{"VOID":543},"25252445",{"EN":545,"VI":546},"VNU Journal of Foreign Studies","Tạp chí Nghiên cứu nước ngoài",{"EN":548,"VI":549},"{\"ops\":[{\"insert\":\"\\n\\nThe \\n\"},{\"attributes\":{\"italic\":true},\"insert\":\"VNU Journal of Science\"},{\"insert\":\"\\n was established in 1985 for the publication of national and international research papers in all fields of natural sciences and technology, social sciences and humanities. 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our previous publication we have received critical comments to our conclusions as well as new data that are strengthening our findings. With the new data, 11 suicide attempts among patients on paroxetine against 1 among patients on placebo, we found with a Bayesian technique that the posterior probability that medication with paroxetine is associated with an increased intensity per year of a suicide attempt is from 0.98 to 0.99, depending on the prior. We found that the comment to our article by GSK representatives contained errors, misunderstanding and unwillingness to accept Bayesian principles in the analysis of clinical trials. We were in our previous publication, with preliminary data and a Bayesian approach, able to raise a concern that suicide attempts might be connected with the use of paroxetine. This suspicion has now been confirmed.",{"EN":1002},"Even more suicide attempts in clinical trials with paroxetine randomised against placebo",{"VOID":1004},"Aursnes I, Tvete IF, Gaasemyr J, Natvig B: Suicide attempts in clinical trials with paroxetine randomised against placebo. BMC Medicine. 2005, 3: 14-10.1186\u002F1741-7015-3-14. doi:10.1186\u002F1741-7015-3-14\nHawkes N: Top selling drug linked to increased suicide risk. [http:\u002F\u002Fwww.timesonline.co.uk\u002Farticle\u002F0,,2-1741916,00.html]\nBoseley S: Drug 'can trigger suicide in adults.'. [http:\u002F\u002Fwww.guardian.co.uk\u002Fuk_news\u002Fstory\u002F0,3604,1553676,00.html]\nModell J: Paroxetine association with suicide attempts overstated. [http:\u002F\u002Fwww.biomedcentral.com\u002F1741-7015\u002F3\u002F14\u002Fcomments]\nBriefing Document. 2006 April 5th., [http:\u002F\u002Fwww.gsk.com\u002Fmedia\u002Fparoxetine\u002Fbriefing_doc.pdf]\nGuidance for the use of Bayesian statistics in medical device clinical trials – Draft guidance for industry and FDA staff. [http:\u002F\u002Fwww.fda.gov\u002Fcdrh\u002Fosb\u002Fguidance\u002F1601.html]\nAursnes I, Tvete IF, Gaasemyr J, Natvig B: Clinical efficacies of antihypertensive drugs. Scand Cardiovasc J. 2003, 37: 72-79. 10.1080\u002F14017430310001186.\nThe pre-publication history for this paper can be accessed here:http:\u002F\u002Fwww.biomedcentral.com\u002F1471-244X\u002F6\u002F55\u002Fprepub",{"VOID":1006},"10.1186\u002F1471-244X-6-55","PUBLICATION","Auto Verify","https:\u002F\u002Fbmcpsychiatry.biomedcentral.com\u002Farticles\u002F10.1186\u002F1471-244X-6-55",[1011,1027,1042,1055],{"id":1012,"sortIndex":32,"researcher":28,"roles":1013,"affiliations":1015,"properties":1024,"displayName":1026,"givenName":28,"familyName":28},"b0dbf4fd-7ad1-44dd-8b8f-e0487ec69a8b",[1014],"AUTHOR",[1016],{"id":1017,"sortIndex":32,"affiliation":1018,"properties":28},"944f031e-af5a-4d1f-b173-c204d02b80c0",{"id":1017,"createTime":28,"updateTime":28,"relativeEntities":1019,"slug":28,"properties":1020,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1023,"statistic":28},[],{"title":1021},{"VI":1022},"Department of Pharmacotherapeutics, University of Oslo, Oslo, Norway",[],{"title":1025},{"VI":1026},"Ivar Aursnes",{"id":1028,"sortIndex":40,"researcher":28,"roles":1029,"affiliations":1030,"properties":1039,"displayName":1041,"givenName":28,"familyName":28},"24f7c88e-0828-4aee-89ae-d51061a1e913",[1014],[1031],{"id":1032,"sortIndex":32,"affiliation":1033,"properties":28},"b14f335d-8d3b-4b4e-a7ee-6f77bd9560dc",{"id":1032,"createTime":28,"updateTime":28,"relativeEntities":1034,"slug":28,"properties":1035,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1038,"statistic":28},[],{"title":1036},{"VI":1037},"Department of Mathematics, University of Oslo, Oslo, Norway",[],{"title":1040},{"VI":1041},"Ingunn Fride Tvete",{"id":1043,"sortIndex":123,"researcher":28,"roles":1044,"affiliations":1045,"properties":1052,"displayName":1054,"givenName":28,"familyName":28},"da7884c3-f5ff-4e05-849e-f6c9754090d7",[1014],[1046],{"id":1032,"sortIndex":32,"affiliation":1047,"properties":28},{"id":1032,"createTime":28,"updateTime":28,"relativeEntities":1048,"slug":28,"properties":1049,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1051,"statistic":28},[],{"title":1050},{"VI":1037},[],{"title":1053},{"VI":1054},"Jorund Gaasemyr",{"id":1056,"sortIndex":42,"researcher":28,"roles":1057,"affiliations":1058,"properties":1065,"displayName":1067,"givenName":28,"familyName":28},"ae7bb274-0165-4b53-b358-4fac3db6493e",[1014],[1059],{"id":1032,"sortIndex":32,"affiliation":1060,"properties":28},{"id":1032,"createTime":28,"updateTime":28,"relativeEntities":1061,"slug":28,"properties":1062,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1064,"statistic":28},[],{"title":1063},{"VI":1037},[],{"title":1066},{"VI":1067},"Bent 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is a relatively common disease worldwide with a point prevalence of around 5\u002F1000 in the population. The aim of this present work was to assess the demographic, clinical, familial, and environmental factors associated with schizophrenia in Mali. This was a prospective descriptive study on a series of 164 patients aged at least 12 years who came for a follow-up consultation at the psychiatry department of the University Hospital Center (CHU) Point G in Mali between February 2019 and January 2020 for schizophrenia spectrum disorder as defined by DSM-5 diagnostic criteria. Our results revealed that the male sex was predominant (80.5%). The 25–34 age group was more represented with 44.5%. The place of birth for the majority of our patients was the urban area (52.4%), which also represented the place of the first year of life for the majority of our patients (56.1%). We noted that the unemployed and single people accounted for 56.1 and 61% respectively. More than half of our patients 58.5% reported having reached secondary school level. With the exception of education level, there was a statistically significant difference in the distribution of demographic parameters. Familial schizophrenia cases accounted for 51.7% versus 49.3% for non-familial cases. The different clinical forms were represented by the paranoid form, followed by the undifferentiated form, and the hebephrenic form with respectively 34, 28 and 17.1%. We noted that almost half (48.8%) of patients were born during the cold season. Cannabis use history was not observed in 68.7% of the patients. The proportions of patients with an out-of-school father or an out-of-school mother were 51.2 and 64.2%, respectively. The onset of schizophrenia in the Malian population has been associated with socio-demographic, clinical, genetic and environmental characteristics.",{"EN":1130},"Descriptive study of cases of schizophrenia in the Malian population",{"VOID":1132},"Messias EL, Chen C-Y, Eaton WW. Epidemiology of Schizophrenia: Review of Findings and Myths. Psychiatric Clin North Am 2007;30(3):323–38. DOI: https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.psc.2007.04.007 .\nOrganization WH. The ICD-10 classification of mental and behavioural disorders: clinical descriptions and diagnostic guidelines. World Health Organization; 1992. Available free at https:\u002F\u002Fapps.who.int\u002Firis\u002Fhandle\u002F10665\u002F37958.\nAngermeyer MC, Carta MG, Ghachem R, Matschinger H, Millier A, Refai T, et al. Cultural Variations in Public Beliefs about Mental Disorders: A Comparison between Tunisia and Germany. Clin Pract Epidemiol Ment Health. 2020;16(Suppl-1):70–81.DOI: https:\u002F\u002Fdoi.org\u002F10.2174\u002F1745017902016010070 .\nMednick S, Huttunen MO, Machón RA. Prenatal influenza infections and adult schizophrenia. Schizophr Bull. 1994;20(2):263–7. https:\u002F\u002Fdoi.org\u002F10.1093\u002Fschbul\u002F20.2.263.\nJablensky A. The diagnostic concept of schizophrenia: its history, evolution, and future prospects. Dialogues Clin Neurosci. 2010;12(3):271–87.DOI: https:\u002F\u002Fdoi.org\u002F10.31887\u002FDCNS.2010.12.3\u002Fajablensky .\nSchizophrenia (EU-GEI) EN of NN studying G-EI in. Identifying gene-environment interactions in schizophrenia: contemporary challenges for integrated, large-scale investigations. Schizophr Bull 2014;40(4):729–736.DOI: https:\u002F\u002Fdoi.org\u002F10.1093\u002Fschbul\u002Fsbu069 .\nOuango J-G, Karfo K, Kere M, Ouedraogo M, Kabore G, Ouedraogo A. Concept traditionnel de la folie et difficultés thérapeutiques psychiatriques chez les Moosé du Kadiogo. Santé Mentale au Québec. 1998;23(2):197–211 https:\u002F\u002Fdoi.org\u002F10.7202\u002F032459ar.\nYao YP, Yeo-Tenena YJ-M, Assi-Sedji C, Tetchi EO, Ngongi KPP, Delafosse RCJ. Itinéraires thérapeutiques des schizophrènes à Abidjan. L’information Psychiatrique. 2009;85(5):461–9 https:\u002F\u002Fdoi.org\u002F10.3917\u002Finpsy.8505.0461.\nBudziszewska MD, Babiuch-Hall M, Wielebska K. Love and romantic relationships in the voices of patients who experience psychosis: an interpretive phenomenological analysis. Front Psychol. 2020;11:570928. https:\u002F\u002Fdoi.org\u002F10.3389\u002Ffpsyg.2020.570928.\nVan der Merwe NJ, Karayiorgou M, Ehlers R, Roos JL. Family history identifies sporadic schizoaffective disorder as a subtype for genetic studies. S Afr J Psychiatry. 2020;26:1393. https:\u002F\u002Fdoi.org\u002F10.4102\u002Fsajpsychiatry.v26i0.1393.\nEsan O, Esan A. Sexual dysfunction among patients with schizophrenia in Southwest Nigeria. J Sex Marital Ther. 2018;44(7):657–66. https:\u002F\u002Fdoi.org\u002F10.1080\u002F0092623X.2018.1447055.\nAribi L, Baati I, Amami O. [Schizophrenia and education: about 32 Tunisian students suffering from schizophrenia]. Tunis Med. sept 2014;92(8–9):536–41.PMID: 25815538 .\nMcClain L, Mansour H, Ibrahim I, Klei L, Fathi W, Wood J, et al. Age dependent association of inbreeding with risk for schizophrenia in Egypt. Schizophr Res févr. 2020;216:450–9. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.schres.2019.10.039.\nBen Thabet J, Charfi N, Dalhoum W, Yaich S, Maâlej Bouali M, Omri S, et al. Factors affecting the Management of Somatic Comorbidity in Tunisian patients with schizophrenia. Psychiatr Q mars. 2019;90(1):29–40. https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs11126-018-9605-4.\nBrown AS. Prenatal infection as a risk factor for schizophrenia. Schizophr Bull. 2006;32(2):200–2. https:\u002F\u002Fdoi.org\u002F10.1093\u002Fschbul\u002Fsbj052.\nMoreno JL, Kurita M, Holloway T, López J, Cadagan R, Martínez-Sobrido L, et al. Maternal influenza viral infection causes schizophrenia-like alterations of 5-HT2A and mGlu2 receptors in the adult offspring. J Neurosci. 2011;31(5):1863–72. https:\u002F\u002Fdoi.org\u002F10.1523\u002FJNEUROSCI.4230-10.2011.\nBrown AS, Patterson PH. Maternal infection and schizophrenia: implications for prevention. Schizophr Bull. 2011;37(2):284–90. https:\u002F\u002Fdoi.org\u002F10.1523\u002FJNEUROSCI.4230-10.2011.\nVan Os J, Rutten BP, Poulton R. Gene-environment interactions in schizophrenia: review of epidemiological findings and future directions. Schizophr Bull. 2008;34(6):1066–82. https:\u002F\u002Fdoi.org\u002F10.1093\u002Fschbul\u002Fsbn117.\nAleman A, Kahn RS, Selten J-P. Sex differences in the risk of schizophrenia: evidence from meta-analysis. Arch Gen Psychiatry. 2003;60(6):565–71. https:\u002F\u002Fdoi.org\u002F10.1001\u002Farchpsyc.60.6.565.\nVilain J, Galliot A-M, Durand-Roger J, Leboyer M, Llorca P-M, Schürhoff F, et al. Les facteurs de risque environnementaux de la schizophrénie. L’Encéphale. 2013;39(1):19–28. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.encep.2011.12.007.\nAbel KM, Drake R, Goldstein JM. Sex differences in schizophrenia. Int Review Psychiatry. 2010;22(5):417–28. https:\u002F\u002Fdoi.org\u002F10.3109\u002F09540261.2010.515205.\nMarkham JA. Sex steroids and schizophrenia. Reviews Endocrine Metabolic Disorders. 2012;13(3):187–207. https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs11154-011-9184-2.\nCoulibaly SP, Maïga FI, Landouré G, Dolo H, Mounkor PP, Dara AE, et al. Facteurs associés aux troubles mentaux dans le service de psychiatrie du Centre Hospitalier Universitaire du Point G au Mali. Mali Médical. 2021;36(1):52-57. Available free at https:\u002F\u002Fwww.malimedical.org\u002F2021\u002F04\u002F19\u002Fannee-2021-tome-xxxvi-n1\u002F.\nAlbakaye M, Maiga Y, Kanikomo D, Djibo D. La dimension socioculturelle de l’épilepsie au Mali. African & Middle East Epilepsy Journal. 2012;1(1). Available free at https:\u002F\u002Frevues.imist.ma\u002Findex.php\u002FAMEEJ\u002Farticle\u002Fdownload\u002F3572\u002F2598 .\nMounkoro PP, Coulibaly S, Kamate Z, Coulibaly SP, Traore J, Traore K, et al. Les Troubles Psychiatriques chez les Émigrés au Service de Psychiatrie du CHU Point G (Bamako). Health Sci. Dis.2019;20(6): 55-60. Available free at www.hsd-fmsb.org.\nWeiser M, Werbeloff N, Dohrenwend BP, Levav I, Yoffe R, Davidson M. Do psychiatric registries include all persons with schizophrenia in the general population? A population-based longitudinal study. Schizophr Res. 2012;135(1–3):187–91. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.schres.2011.12.023.\nHoungbé JE, Gansou MG, Agongbonou R, Amonles Y, Tognide CM, Houinato D, et al. Prévalence de la schizophrénie au sud du Bénin. Rev Epidemiol Sante Publique. 2014;62:S226 https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.respe.2014.06.176.\nKebede D, Alem A, Shibre T, Negash A, Fekadu A, Fekadu D, et al. Onset and clinical course of schizophrenia in Butajira-Ethiopia--a community-based study. Soc Psychiatry Psychiatr Epidemiol nov. 2003;38(11):625–31. https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs00127-003-0678-4.\nMarwaha S, Johnson S. Schizophrenia and employment. Soc Psychiatry Psychiatr Epidemiol. 2004;39(5):337–49. https:\u002F\u002Fdoi.org\u002F10.1007\u002Fs00127-004-0762-4.\nLuo Y, Pang L, Zhao Y, Guo C, Zhang L, Zheng X. Gender difference in the association between education and schizophrenia in Chinese adults. BMC Psychiatry. 2020;20(1):296.DOI: https:\u002F\u002Fdoi.org\u002F10.1186\u002Fs12888-020-02700-2 .\nKelly BD, O’Callaghan E, Waddington JL, Feeney L, Browne S, Scully PJ, et al. Schizophrenia and the city: a review of literature and prospective study of psychosis and urbanicity in Ireland. Schizophr Res. 2010;116(1):75–89. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.schres.2009.10.015.\nLundberg P, Cantor-Graae E, Rukundo G, Ashaba S, Ostergren P-O. Urbanicity of place of birth and symptoms of psychosis, depression and anxiety in Uganda. Br J Psychiatry. 2009;195(2):156–62. https:\u002F\u002Fdoi.org\u002F10.1192\u002Fbjp.bp.108.051953.\nPedersen CB, Mortensen PB. Evidence of a dose-response relationship between urbanicity during upbringing and schizophrenia risk. Arch Gen Psychiatry. 2001;58(11):1039–46. https:\u002F\u002Fdoi.org\u002F10.1001\u002Farchpsyc.58.11.1039.\nVassos E, Pedersen CB, Murray RM, Collier DA, Lewis CM. Meta-analysis of the association of urbanicity with schizophrenia. Schizophr Bull. 2012;38(6):1118–23. https:\u002F\u002Fdoi.org\u002F10.1093\u002Fschbul\u002Fsbs096.\nChou I-J, Kuo C-F, Huang Y-S, Grainge MJ, Valdes AM, See L-C, et al. Familial Aggregation and Heritability of Schizophrenia and Co-aggregation of Psychiatric Illnesses in Affected Families. Schizophr Bull. 2017;43(5):1070–8.DOI: https:\u002F\u002Fdoi.org\u002F10.1093\u002Fschbul\u002Fsbw159 .\nLiang S, Deng W, Wang Q, Ma X, Li M, Brown MRG, et al. Performance of Verbal Fluency as an Endophenotype in Patients with Familial versus Sporadic Schizophrenia and Their Parents. 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Arch Gen Psychiatry. 1967;17(4):435–40. https:\u002F\u002Fdoi.org\u002F10.1001\u002Farchpsyc.1967.01730280051005.\nHare EH, Price JS. Mental disorder and season of birth: comparison of psychoses with neurosis. Br J Psychiatry. 1969;115(522):533–40. https:\u002F\u002Fdoi.org\u002F10.1192\u002Fbjp.115.522.533.\nBener A, Dafeeah EE, Samson N. Does consanguinity increase the risk of schizophrenia? Study based on primary health care centre visits. Ment Health Fam Med. 2012;9(4):241–8.\nAhmed AH. Consanguinity and schizophrenia in Sudan. Br J Psychiatry. 1979;134(6):635–6. https:\u002F\u002Fdoi.org\u002F10.1192\u002Fbjp.134.6.635.\nMansour H, Fathi W, Klei L, Wood J, Chowdari K, Watson A, et al. Consanguinity and increased risk for schizophrenia in Egypt. Schizophr Res. 2010;120(1–3):108–12. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.schres.2010.03.026.\nMortensen PB, Pedersen CB, Westergaard T, Wohlfahrt J, Ewald H, Mors O, et al. 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Data were used from subjects with current MDD (n = 655), GAD (n = 107) and comorbid MDD\u002FGAD (n = 266) diagnosis from the Netherlands Study of Depression and Anxiety (NESDA). The Composite Interview Diagnostic Instrument was used to diagnose MDD and GAD. Cognitive profiles were measured using the Leiden Index of Depression Sensitivity, the Anxiety Sensitivity Index, and the Penn State Worry Questionnaire. Results showed that differences in cognitive profiles between single MDD and single GAD subjects were present: scores on hopelessness\u002Fsuicidality and rumination were significantly higher in MDD than GAD, whereas anxiety sensitivity for physical concerns and pathological worry were higher in GAD than MDD. The cognitive profile of comorbid MDD\u002FGAD showed more extreme depression cognitions compared to single disorders, and a similar anxiety profile compared to single GAD subjects. Despite the commonalities in cognitive profiles in MDD and GAD, there are differences suggesting that MDD and GAD have disorder-specific cognitive profiles. 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Behav Res Ther. 2008, 46: 1151-1159. 10.1016\u002Fj.brat.2008.05.009.",{"id":28,"text":1709,"url":28,"identifiers":28},"Norton PJ, Sexton KA, Walker JR, Norton GR: Hierarchical model of vulnerabilities for anxiety: replication and extension with a clinical sample. Cogn Behav Ther. 2005, 34: 50-63. 10.1080\u002F16506070410005401.",{"id":28,"text":1711,"url":28,"identifiers":28},"Norton PJ, Mehta PD: Hierarchical model of vulnerabilities for emotional disorders. Cogn Behav Ther. 2007, 36: 240-254. 10.1080\u002F16506070701628065.",{"id":28,"text":1713,"url":28,"identifiers":28},"Sexton KA, Norton PJ, Walker JR, Norton GR: Hierarchical model of generalized and specific vulnerabilities in anxiety. Cogn Behav Ther. 2003, 32: 82-94. 10.1080\u002F16506070302321.",{"id":28,"text":1715,"url":28,"identifiers":28},"Beck R, Perkins TS, Holder R, Robbins M, Gray M, Allison SH: The cognitive and emotional phenomenology of depression and anxiety: are worry and hopelessness the cognitive correlates of NA and PA?. Cogn Ther Res. 2001, 25: 829-838. 10.1023\u002FA:1012983726272.",{"id":28,"text":1717,"url":28,"identifiers":28},"Beck R, Benedict B, Winkler A: Depression and anxiety: integrating the tripartite and cognitive content-specificity assessment models. Psychopathol Behav Assess. 2003, 25: 251-256. 10.1023\u002FA:1025899029040.",{"id":28,"text":1719,"url":28,"identifiers":28},"Jolly JB, Dyck MJ, Kramer TA, Wherry JN: Integration of positive and negative affectivity and cognitive content-specificity: improved discrimination of anxious and depressive symptoms. 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J Res Pers. 2005, 39: 46-66. 10.1016\u002Fj.jrp.2004.09.006.",{"id":28,"text":1813,"url":28,"identifiers":28},"American Psychiatric Association: Diagnostic and Statistical Manual of Mental Disorders. 1987, Washington, DC: American Psychiatric Association, 3",{"id":28,"text":1815,"url":28,"identifiers":28},"The pre-publication history for this paper can be accessed here:http:\u002F\u002Fwww.biomedcentral.com\u002F1471-244X\u002F14\u002F96\u002Fprepub",{"id":1817,"createTime":1818,"updateTime":1819,"relativeEntities":1820,"slug":1821,"properties":1822,"entityType":1007,"verifyStatus":26,"verifyTime":1831,"verifyNote":1008,"languages":28,"translateLanguages":28,"viewCount":32,"primaryUrl":1832,"fullTextUrl":28,"authors":1833,"publicationType":1068,"publisherRelationship":2026,"citationCount":28,"citationInfo":28,"publishDate":2072,"publishYear":2073,"citationAnalyzeStatus":878,"lastCitationAnalyze":28,"indexDatabases":2074,"openAccess":28,"references":28,"isForceReanalyzing":1119},"0050c7ef-f568-4aed-9f3f-ecc3951c7938","2024-01-19T05:32:20.224+00:00","2025-01-21T02:47:27.543+00:00",[],"Bifrontal-electroconvulsive-therapy-leads-to-improvement-of-cerebral-glucose-hypometabolism-in-frontotemporal-dementia-with-comorbid-psychotic-depression-a-case-report",{"abstract":1823,"title":1825,"references":1827,"doi":1829},{"EN":1824},"Differentiating depression and dementia in elderly patients represents a major clinical challenge for psychiatrists. Pharmacological and non-pharmacological treatment options for both conditions are often used cautiously due to fear of adverse effects. If a clinically indicated therapy is not initiated due to fear of adverse effects, the quality of life of affected patients may significantly be reduced. Here, we describe the case of a 65-year-old woman who presented to the department of psychiatry of a university hospital with depressed mood, pronounced anxiety, and nihilistic thoughts. While several pharmacological treatments remained without clinical response, further behavioral observation in conjunction with 18F-fluoro‐2‐deoxy‐D‐glucose positron emission tomography\u002Fcomputed tomography (18F-FDG PET\u002FCT) revealed the diagnosis of frontotemporal dementia (FTD). To counter the pharmacological treatment resistance of psychotic depression, we decided to perform electroconvulsive therapy (ECT). Remarkably, ten sessions of ECT yielded an almost complete remission of depressive symptoms. In addition, the patient’s delusional ideas disappeared. A follow-up 18F-FDG PET\u002FCT after the ECT series still showed a frontally and parieto-temporally accentuated hypometabolism, albeit with a clear regression compared to the previous image. The follow-up 18F-FDG PET\u002FCT thus corroborated the diagnosis of FTD, while on the other hand it demonstrated the success of ECT. In this case, ECT was a beneficial treatment option for depressive symptoms in FTD. Also, 18F-FDG PET\u002FCT should be discussed as a valuable tool in differentiating depression and dementia and as an indicator of treatment response.",{"EN":1826},"Bifrontal electroconvulsive therapy leads to improvement of cerebral glucose hypometabolism in frontotemporal dementia with comorbid psychotic depression – a case report",{"VOID":1828},"Bang J, Spina S, Miller BL. Frontotemporal dementia. Lancet. 2015;386(10004):1672–82.\nYounes K, Miller BL. 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Usefulness of EEG techniques in distinguishing frontotemporal dementia from Alzheimer’s disease and other dementias. Dis Markers 2018, 2018.\nDucharme S, Dols A, Laforce R, Devenney E, Kumfor F, Van Den Stock J, Dallaire-Théroux C, Seelaar H, Gossink F, Vijverberg E. Recommendations to distinguish behavioural variant frontotemporal dementia from psychiatric disorders. Brain. 2020;143(6):1632–50.\nRascovsky K, Hodges JR, Knopman D, Mendez MF, Kramer JH, Neuhaus J, Van Swieten JC, Seelaar H, Dopper EG, Onyike CU. Sensitivity of revised diagnostic criteria for the behavioural variant of frontotemporal dementia. Brain. 2011;134(9):2456–77.\nRivero-Santana A, Ferreira D, Perestelo-Perez L, Westman E, Wahlund L, Sarria A, Serrano-Aguilar P. Cerebrospinal fluid biomarkers for the differential diagnosis between Alzheimer’s disease and frontotemporal lobar degeneration: systematic review, HSROC analysis, and confounding factors. 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The Montreal Cognitive Assessment, MoCA: a brief screening tool for mild cognitive impairment. J Am Geriatr Soc. 2005;53(4):695–9.\nSotaniemi M, Pulliainen V, Hokkanen L, Pirttilä T, Hallikainen I, Soininen H, Hänninen T. CERAD-neuropsychological battery in screening mild Alzheimer’s disease. Acta Neurol Scand. 2012;125(1):16–23.\nDubois B, Slachevsky A, Litvan I, Pillon B. The FAB: a Frontal Assessment Battery at bedside. Neurology. 2000;55(11):1621–6.\nWang YP, Gorenstein C. Psychometric properties of the Beck Depression Inventory-II: a comprehensive review. Braz J Psychiatry. 2013;35(4):416–31.\nSchroeder K, Andresen B, Naber D, Huber CG. Diagnostic validity of ICD-10 personality dimensions: a multitrait-multimethod analysis of two self-report questionnaires and a structured interview. Psychopathology. 2010;43(2):110–20.\nKobayashi T, Inoue K, Shioda K, Kato S. 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Efficacy and cognitive side effects of electroconvulsive therapy (ECT) in depressed elderly inpatients with coexisting mild cognitive impairment or dementia. J Clin Psychiatry. 2011;72(1):91–7.\nDunne RA, McLoughlin DM. Systematic review and meta-analysis of bifrontal electroconvulsive therapy versus bilateral and unilateral electroconvulsive therapy in depression. World J Biol Psychiatry. 2012;13(4):248–58.\nTakamiya A, Liang KC, Nishikata S, Tarumi R, Sawada K, Kurokawa S, Hirano J, Yamagata B, Mimura M, Kishimoto T. Predicting Individual Remission After Electroconvulsive Therapy Based on Structural Magnetic Resonance Imaging: A Machine Learning Approach. J ECT. 2020;36(3):205–10.\nMo Y, Wei Q, Bai T, Zhang T, Lv H, Zhang L, Ji G, Yu F, Tian Y, Wang K. Bifrontal electroconvulsive therapy changed regional homogeneity and functional connectivity of left angular gyrus in major depressive disorder. Psychiatry Res. 2020;294:113461.\nCano M, Cardoner N, Urretavizcaya M, Martínez-Zalacaín I, Goldberg X, Via E, Contreras-Rodríguez O, Camprodon J, de Arriba-Arnau A, Hernández-Ribas R. Modulation of limbic and prefrontal connectivity by electroconvulsive therapy in treatment-resistant depression: a preliminary study. Brain Stimul. 2016;9(1):65–71.\nSchmidt EZ, Reininghaus B, Enzinger C, Ebner C, Hofmann P, Kapfhammer HP. Changes in brain metabolism after ECT-positron emission tomography in the assessment of changes in glucose metabolism subsequent to electroconvulsive therapy–lessons, limitations and future applications. J Affect Disord. 2008;106(1–2):203–8.\nReininghaus EZ, Reininghaus B, Ille R, Fitz W, Lassnig RM, Ebner C, Annamaria P, Hofmann P, Kapfhammer HP, Reingard A, Fazekas F, Ropele S, Enzinger C. Clinical effects of electroconvulsive therapy in severe depression and concomitant changes in cerebral glucose metabolism–an exploratory study. J Affect Disord. 2013;146(2):290–4.\nLajoie C, Levasseur MA, Paquet N. Complete normalization of severe brain 18F-FDG hypometabolism following electroconvulsive therapy in a major depressive episode. Clin Nucl Med. 2013;38(9):735–6.\nHassamal S, Jolles P, Pandurangi A. Reversal of cerebral glucose hypometabolism on positron emission tomography with electroconvulsive therapy in an elderly patient with a psychotic episode. Psychogeriatrics. 2016;16(6):376–81.\nWilliams D, Campbell K. Electroconvulsive Therapy for the Treatment of the Behavioural and Psychological Symptoms of Dementia: A Review of Clinical Effectiveness and Guidelines 2019, Ottawa (ON):Canadian Agency for Drugs and Technologies in Health.",{"VOID":1830},"10.1186\u002Fs12888-023-04759-z","2025-01-21T02:47:27.542+00:00","https:\u002F\u002Fbmcpsychiatry.biomedcentral.com\u002Farticles\u002F10.1186\u002Fs12888-023-04759-z",[1834,1849,1864,1877,1890,1905,1920,1933,1948,1961,1974,1987,2000,2013],{"id":1835,"sortIndex":32,"researcher":28,"roles":1836,"affiliations":1837,"properties":1846,"displayName":1848,"givenName":28,"familyName":28},"f359dfe8-2a3a-4b33-ad97-8fb956bd3184",[1014],[1838],{"id":1839,"sortIndex":32,"affiliation":1840,"properties":28},"9a22eece-2105-4ab4-96fc-3445ee805705",{"id":1839,"createTime":28,"updateTime":28,"relativeEntities":1841,"slug":28,"properties":1842,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1845,"statistic":28},[],{"title":1843},{"VI":1844},"Department of Psychiatry, Social Psychiatry and Psychotherapy, Hannover Medical School, Hannover, Germany",[],{"title":1847},{"VI":1848},"Sebastian 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purpose of this study is to investigate the possible link between dietary theobromine intake and symptoms of depression. These results are based on the responses of 3637 people who took part in the National Health and Nutrition Examination Survey in 2017–2018. Participants' daily theobromine intake was determined using a 24-h food questionnaire from the 2017–2018 cycle. Presence of depression was defined as a score of 5 or above on the Patient Health Questionnaire. Association between theobromine intake and depression was examined using a multivariate logistic regression adjusting for several relevant sociodemographic, lifestyle and health-related factors. A total of 6903 participants were included in the study. The results of multivariate logistic regression showed a correlation between depressive symptoms and theobromine intake (OR:1.17, 95%CI:1.02–1.34). Our cross-sectional population based study suggests that increased theobromine intake is associated with increased risk for depression. Nevertheless, more investigations are needed to confirm our findings.",{"EN":2085},"Association between dietary theobromine with depression: a population-based study",{"VOID":2087},"Tchekalarova J, Ivanova N, Nenchovska Z, Tzoneva R, Stoyanova T, Uzunova V, et al. Evaluation of neurobiological and antioxidant effects of novel melatonin analogs in mice. Saudi Pharm J. 2020;28:1566–79. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.jsps.2020.10.004.\nWen Y, Liu C, Liao J, Yin Y, Wu D. Incidence and risk factors of depressive symptoms in 4 years of follow-up among mid-aged and elderly community-dwelling Chinese adults: findings from the China health and retirement longitudinal study. BMJ Open. 2019;9:e29529. https:\u002F\u002Fdoi.org\u002F10.1136\u002Fbmjopen-2019-029529.\nAkil H, Gordon J, Hen R, Javitch J, Mayberg H, McEwen B, et al. Treatment resistant depression: a multi-scale, systems biology approach. Neurosci Biobehav Rev. 2018;84:272–88. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.neubiorev.2017.08.019.\nBurcu M, Zito JM, Safer DJ, Magder LS, dosReis S, Shaya FT, et al. Association of antidepressant medications with incident type 2 diabetes among medicaid-insured youths. JAMA Pediatr. 2017;171:1200–7. https:\u002F\u002Fdoi.org\u002F10.1001\u002Fjamapediatrics.2017.2896.\nJackson SE, Smith L, Firth J, Grabovac I, Soysal P, Koyanagi A, et al. Is there a relationship between chocolate consumption and symptoms of depression? A cross-sectional survey of 13,626 US adults. Depress Anxiety. 2019;36:987–95. https:\u002F\u002Fdoi.org\u002F10.1002\u002Fda.22950.\nPérez-Ara MÁ, Gili M, Visser M, Penninx BWJH, Brouwer IA, Watkins E, et al. Associations of non-alcoholic beverages with major depressive disorder history and depressive symptoms clusters in a sample of overweight adults. Nutrients. 2020;12:3202. https:\u002F\u002Fdoi.org\u002F10.3390\u002Fnu12103202.\nMolendijk M, Molero P, Ortuño Sánchez-Pedreño F, Van der Does W, Angel Martínez-González M. Diet quality and depression risk: A systematic review and dose-response meta-analysis of prospective studies. J Affect Disord. 2018;226:346–54. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.jad.2017.09.022.\nGao L, Ge W, Peng C, Guo J, Chen N, He L. Association between dietary theobromine and cognitive function in a representative American population: a cross-sectional study. J Prev Alzheimer’s Dis. 2022;9:449–57. https:\u002F\u002Fdoi.org\u002F10.14283\u002Fjpad.2022.39.\nCurti SA, Taylor EN, Su D, Spankovich C. Prevalence of and characteristics associated with self-reported good hearing in a population with elevated audiometric thresholds. JAMA Otolaryngology Head Neck Surg. 2019;145:626–33. https:\u002F\u002Fdoi.org\u002F10.1001\u002Fjamaoto.2019.1020.\nWang L, Li X, Wang Z, Bancks MP, Carnethon MR, Greenland P, et al. Trends in prevalence of diabetes and control of risk factors in diabetes among US adults, 1999–2018. JAMA. 2021;326:1–13. https:\u002F\u002Fdoi.org\u002F10.1001\u002Fjama.2021.9883.\nLi Z, Wang W, Xin X, Song X, Zhang D. Association of total zinc, iron, copper and selenium intakes with depression in the US adults. J Affect Disord. 2018;228:68–74. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.jad.2017.12.004.\nBailey RL, Fulgoni VLR, Keast DR, Dwyer JT. Dietary supplement use is associated with higher intakes of minerals from food sources. Am J Clin Nutr. 2011;94:1376–81. https:\u002F\u002Fdoi.org\u002F10.3945\u002Fajcn.111.020289.\nKim K, Melough MM, Vance TM, Noh H, Koo SI, Chun OK. Dietary cadmium intake and sources in the US. Nutrients. 2018;11:2. https:\u002F\u002Fdoi.org\u002F10.3390\u002Fnu11010002.\nIob E, Frank P, Steptoe A, Fancourt D. Levels of severity of depressive symptoms among at-risk groups in the UK during the COVID-19 pandemic. JAMA Netw Open. 2020;3:e2026064. https:\u002F\u002Fdoi.org\u002F10.1001\u002Fjamanetworkopen.2020.26064.\nFlint KM, Fairclough DL, Spertus JA, Bekelman DB. Does heart failure-specific health status identify patients with bothersome symptoms, depression, anxiety, and\u002For poorer spiritual well-being? European Heart Journal. Qual Care Clin Outcomes. 2019;5:233–41. https:\u002F\u002Fdoi.org\u002F10.1093\u002Fehjqcco\u002Fqcy061.\nKim K, Song Y, Oh TJ, Choi SH, Jang HC. Association between iron intake and diabetic peripheral neuropathy in type 2 diabetes: significance of iron intake and the ratio between iron intake and polyunsaturated fatty acids intake. Nutrients. 2020;12:3365. https:\u002F\u002Fdoi.org\u002F10.3390\u002Fnu12113365.\nRattan P, Penrice DD, Ahn JC, Ferrer A, Patnaik M, Shah VH, et al. Inverse association of telomere length with liver disease and mortality in the US population. Hepatol Commun. 2022;6:399–410. https:\u002F\u002Fdoi.org\u002F10.1002\u002Fhep4.1803.\nJenkins GP, Evenson KR, Herring AH, Hales D, Stevens J. Cardiometabolic correlates of physical activity and sedentary patterns in U.S. youth. Med Sci Sports Exerc. 2017;49:1826–33. https:\u002F\u002Fdoi.org\u002F10.1249\u002FMSS.0000000000001310.\nLinkens AM, Eussen SJ, Houben AJ, Kroon AA, Schram MT, Reesink KD, et al. Habitual intake of dietary advanced glycation end products is not associated with arterial stiffness of the aorta and carotid artery in adults: the Maastricht study. J Nutr. 2021;151:1886–93. https:\u002F\u002Fdoi.org\u002F10.1093\u002Fjn\u002Fnxab097.\nKing DE, Xiang J, Pilkerton CS. Multimorbidity trends in United States adults, 1988–2014. J Am Board Fam Med. 2018;31:503–13. https:\u002F\u002Fdoi.org\u002F10.3122\u002Fjabfm.2018.04.180008.\nSmith HC, Saxena S, Petersen I. Maternal postnatal depression and completion of infant immunizations: a UK cohort study of 196,329 mother-infant Pairs, 2006–2015. J Clin Psychiatry. 2022;83:20m13575. https:\u002F\u002Fdoi.org\u002F10.4088\u002FJCP.20m13575.\nYoneda M, Sugimoto N, Katakura M, Matsuzaki K, Tanigami H, Yachie A, et al. Theobromine up-regulates cerebral brain-derived neurotrophic factor and facilitates motor learning in mice. J Nutr Biochem. 2017;39:110–6. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.jnutbio.2016.10.002.\nMendiola-Precoma J, Padilla K, Rodríguez-Cruz A, Berumen LC, Miledi R, García-Alcocer G. Theobromine-induced changes in A1 purinergic receptor gene expression and distribution in a rat brain alzheimer’s disease model. J Alzheimer’s Dis. 2017;55:1273–83.\nMartínez-Pinilla E, Oñatibia-Astibia A, Franco R. The relevance of theobromine for the beneficial effects of cocoa consumption. Front Pharmacol. 2015;6:30. https:\u002F\u002Fdoi.org\u002F10.3389\u002Ffphar.2015.00030.\nMitchell ES, Slettenaar M, vd Meer N, Transler C, Jans L, Quadt F, et al. Differential contributions of theobromine and caffeine on mood, psychomotor performance and blood pressure. Physiol Behav. 2011;104:816–22. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.physbeh.2011.07.027.\nIranpour S, Sabour S. Inverse association between caffeine intake and depressive symptoms in US adults: data from National Health and Nutrition Examination Survey (NHANES) 2005–2006. Psychiatry Res. 2019;271:732–9. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.psychres.2018.11.004.\nSmith AP. Caffeine, cognitive failures and health in a non-working community sample. Hum Psychopharmacol. 2009;24:29–34. https:\u002F\u002Fdoi.org\u002F10.1002\u002Fhup.991.\nMumford GK, Benowitz NL, Evans SM, Kaminski BJ, Preston KL, Sannerud CA, et al. Absorption rate of methylxanthines following capsules, cola and chocolate. Eur J Clin Pharmacol. 1996;51:319–25.\nDørheim SK, Bjorvatn B, Eberhard-Gran M. Insomnia and depressive symptoms in late pregnancy: a population-based study. Behav Sleep Med. 2012;10:152–66. https:\u002F\u002Fdoi.org\u002F10.1080\u002F15402002.2012.660588.\nTsujiguchi H, Thi Thu Nguyen T, Goto D, Miyagi S, Kambayashi Y, Hara A, et al. Relationship between the Intake of n-3 polyunsaturated fatty acids and depressive symptoms in elderly japanese people: differences according to sex and weight status. Nutrients. 2019;11:775. https:\u002F\u002Fdoi.org\u002F10.3390\u002Fnu11040775.",{"VOID":2089},"10.1186\u002Fs12888-022-04415-y","https:\u002F\u002Fbmcpsychiatry.biomedcentral.com\u002Farticles\u002F10.1186\u002Fs12888-022-04415-y",[2092,2116,2131,2146],{"id":2093,"sortIndex":32,"researcher":28,"roles":2094,"affiliations":2095,"properties":2113,"displayName":2115,"givenName":28,"familyName":28},"c4276709-8399-40f4-839c-462e831dd6f2",[1014],[2096,2104],{"id":2097,"sortIndex":32,"affiliation":2098,"properties":28},"103924ba-7c88-40f6-8c5d-58fe82b85f3c",{"id":2097,"createTime":28,"updateTime":28,"relativeEntities":2099,"slug":28,"properties":2100,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2103,"statistic":28},[],{"title":2101},{"VI":2102},"Department of Interventional Therapy, Multidisciplinary Team of Vascular Anomalies, Shanghai Ninth People’s hospital, Shanghai Jiao Tong University, Shanghai, People’s Republic of China",[],{"id":2105,"sortIndex":40,"affiliation":2106,"properties":2112},"754f4b41-cc17-487a-96b8-b5f02f10b136",{"id":2105,"createTime":28,"updateTime":28,"relativeEntities":2107,"slug":28,"properties":2108,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2111,"statistic":28},[],{"title":2109},{"EN":2110},"Department of Neurosurgery, Shanghai Ninth People’s Hospital, Shanghai Jiao Tong University, Shanghai, People’s Republic of China",[],{},{"title":2114},{"VI":2115},"Xin-yu 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are part of women with low socioeconomic status and most of them are migrant from rural to central part of Ethiopia, less educated, either with poor, separated, single or divorced family and\u002For dead parents. Housemaid may experience problems like depression and anxiety more than other groups of women. Therefore, the aim of this study was to assess the magnitude and determinants of those problems among housemaids. To assess the magnitude and determinants of depression & anxiety among housemaids in Addis Ababa; Ethiopia; 2018. Community based cross-sectional study was conducted from May 1 to August 30, 2018 among housemaids working in Addis Ababa, Ethiopia. Multistage sampling technique was applied with a total of 826 samples. Quantitative data was employed by using structured questionnaires. The collected data was coded, entered in to Epi-Info version 7 and analyzed by using SPSS version 20. Descriptive, analytical statistical procedures; bivariate and multivariate binary logistic regressions with odds ratios and 95% confidence interval was employed. The statistical significance was declared at p value \u003C 0.05. This study enrolled 862 participants with response rate 99.5%, 99.5% refers to the number of people who actually completed the interview. The result showed prevalence of depression and anxiety among housemaids 27.5% and 32.3% respectively. Among all participants 44.6% (95% CI= 41.0 – 47.9) have mild, 18.5% (95% CI= 15.7 – 21.2) have moderate and 6.1% (95% CI= 4.5 – 7.8) have severe form of comorbid anxiety with depression. Depression (44.9%) and anxiety (41.9%) found more prevalent among the age group 16 to 20. In this study history of parental divorce, participant’s divorce, physical violence and sexual violence are positively associated. Other factors; being less educated and living with relatives; were associated negatively. The prevalence of Depression and Anxiety is found high among housemaid; its prevalence is more among age group 16 to 20 than other age groups. Violence, participant’s divorce, history of parental divorce and contraceptive use has positively associated with depression and anxiety.",{"EN":2219},"Depression, anxiety and associated factors among housemaids working in Addis Ababa Ethiopia",{"VOID":2221},"Allen M, Raghallaigh MN. Women’s Experiences of Gender Based Violence in Tigray, Ethiopia; 2012.\nBaxter A, Scott K, Vos T, Whiteford H. Global prevalence of anxiety disorders: a systematic review and meta-regression. Psychol Med. 2013;43(5):897–910.\nParker G, Fletcher K, Paterson A, Anderson J, Hong M. Gender differences in depression severity and symptoms across depressive sub-types. J Affect Disord. 2014;167:351–7.\nWeissman MM, Klerman GL. Sex differences and the epidemiology of depression. Arch Gen Psychiatry. 1977;34(1):98–111.\nFaravelli C, Scarpato MA, Castellini G, Sauro CL. Gender differences in depression and anxiety: the role of age. Psychiatry Res. 2013;210(3):1301–3.\nKessler RC. Epidemiology of women and depression. J Affect Disord. 2003;74(1):5–13.\nZahid MA, Fido A, Alowaish R, Mohsen MAE-M, Razik MA. Psychiatric morbidity among housemaids in Kuwait III: Vulnerability factors. Int J Soc Psychiatry. 2003;49(2):87–96.\nEl-Hilu SM, Mousa R, Abdulmalek H, Kamel N, Zohdi M, Maher A, Al-Aamriti M. Psychiatric morbidity among foreign housemaids in Kuwait. Int J Soc Psychiatry. 1990;36(4):291–9.\nZahid M, Fido A, Razik M, Mohsen M, El-Sayed A. Psychiatric morbidity among housemaids in Kuwait. Med Princ Pract. 2004;13(5):249–54.\nSales EC, Santana VS. Depressive and anxiety symptoms among housemaids. Am J Ind Med. 2003;44(6):685–91.\nRehn E, Johnson Sirleaf E. Women, War and Peace, Progress of the World's Women. New York: UNIFEM; 2002.\nHsieh Y-CJ, Apostolopoulos Y, Sönmez S. The world at work: hotel cleaners. Occup Environ Med. 2013;70(5):360–4.\nAllen M, Raghallaigh MN. Domestic violence in a developing context: The perspectives of women in northern Ethiopia. Affilia. 2013;28(3):256–72.\nKumar A. Domestic violence in India: Causes, consequences and remedies. Youth ki awaaz. 2010. https:\u002F\u002Fwww.youthkiawaaz.com\u002F2010\u002F02\u002Fdomestic-violence-in-india-causes-consequences-and-remedies-2\u002F.\nLöwe B, Wahl I, Rose M, Spitzer C, Glaesmer H, Wingenfeld K, Schneider A, Brähler E. A 4-item measure of depression and anxiety: validation and standardization of the Patient Health Questionnaire-4 (PHQ-4) in the general population. J Affect Disord. 2010;122(1):86–95.\nEllsberg M, Jansen H, Heise L, Watts CH, Garcia-Moreno C. WHO Multi-country Study on Women's Health and Domestic Violence against Women Study Team. Intimate partner violence and women's physical and mental health in the WHO multi-country study on women's health and domestic violence: an observational study. Lancet. 2008;371(9619):1165–72.\nAudet CM, Wainberg ML, Oquendo MA, Yu Q, Peratikos MB, Duarte CS, Martinho S, Green AF, González-Calvo L, Moon TD. Depression among female heads-of-household in rural Mozambique: A cross-sectional population-based survey. J Affect Disord. 2018;227:48–55.\nDeyessa N, Berhane Y, Alem A, Hogberg U, Kullgren G. Depression among women in rural Ethiopia as related to socioeconomic factors: a community-based study on women in reproductive age groups. Scand J Public Health. 2008;36(6):589–97.\nFenta H, Hyman I, Noh S. Determinants of depression among Ethiopian immigrants and refugees in Toronto. J Nerv Ment Dis. 2004;192(5):363–72.\nKendler KS, Neale MC, Kessler RC, Heath AC, Eaves LJ. Childhood parental loss and adult psychopathology in women: a twin study perspective. Arch Gen Psychiatry. 1992;49(2):109–16.\nEllsberg M, Caldera T, Herrera A, Winkvist A, Kullgren G. Domestic violence and emotional distress among Nicaraguan women: Results from a population-based study. Am Psychol. 1999;54(1):30.\nFinne LB, Christensen JO, Knardahl S. Psychological and social work factors as predictors of mental distress: a prospective study. PLoS One. 2014;9(7):e102514.",{"VOID":2223},"10.1186\u002Fs12888-020-02638-5","https:\u002F\u002Fbmcpsychiatry.biomedcentral.com\u002Farticles\u002F10.1186\u002Fs12888-020-02638-5",[2226,2241,2254,2267,2280],{"id":2227,"sortIndex":32,"researcher":28,"roles":2228,"affiliations":2229,"properties":2238,"displayName":2240,"givenName":28,"familyName":28},"daae9382-6a46-435e-8771-7e8783872459",[1014],[2230],{"id":2231,"sortIndex":32,"affiliation":2232,"properties":28},"5be6995e-1b0c-4fdb-92be-d8b78b5096da",{"id":2231,"createTime":28,"updateTime":28,"relativeEntities":2233,"slug":28,"properties":2234,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2237,"statistic":28},[],{"title":2235},{"VI":2236},"Addis Ababa, Ethiopia",[],{"title":2239},{"VI":2240},"Alem K. Ejigu",{"id":2242,"sortIndex":40,"researcher":28,"roles":2243,"affiliations":2244,"properties":2251,"displayName":2253,"givenName":28,"familyName":28},"abad657a-ef2a-475a-a78f-f769ad1d13d3",[1014],[2245],{"id":2231,"sortIndex":32,"affiliation":2246,"properties":28},{"id":2231,"createTime":28,"updateTime":28,"relativeEntities":2247,"slug":28,"properties":2248,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2250,"statistic":28},[],{"title":2249},{"VI":2236},[],{"title":2252},{"VI":2253},"Zahra R. 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Jilcha",{"id":2281,"sortIndex":45,"researcher":28,"roles":2282,"affiliations":2283,"properties":2290,"displayName":2292,"givenName":28,"familyName":28},"966f4528-5d94-4afa-985c-628006bf5f1c",[1014],[2284],{"id":2231,"sortIndex":32,"affiliation":2285,"properties":28},{"id":2231,"createTime":28,"updateTime":28,"relativeEntities":2286,"slug":28,"properties":2287,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2289,"statistic":28},[],{"title":2288},{"VI":2236},[],{"title":2291},{"VI":2292},"Yodit H. Bezabih",{"url":2224,"publisher":2294,"properties":2335},{"id":868,"createTime":869,"updateTime":870,"relativeEntities":2295,"slug":872,"properties":2296,"entityType":25,"verifyStatus":878,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":32,"subjectFields":2299,"manageAffiliations":2304,"indexDatabases":2315,"url":930,"thumbnailPath":28,"statistic":2330,"gsStatistic":28,"type":55,"analyzePriority":28},[],{"issn":2297,"title":2298},{"VOID":875},{"EN":877},[2300],{"id":881,"createTime":28,"updateTime":28,"relativeEntities":2301,"label":2302,"description":2303,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":884},{},[2305,2310],{"id":888,"createTime":28,"updateTime":28,"relativeEntities":2306,"slug":28,"properties":2307,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2309,"statistic":28},[],{"title":2308},{"EN":892},[],{"id":895,"createTime":28,"updateTime":28,"relativeEntities":2311,"slug":28,"properties":2312,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2314,"statistic":28},[],{"title":2313},{"EN":899},[],[2316,2323],{"id":903,"indexDatabase":2317,"url":909,"indexYears":910,"academicFieldIds":2322,"indexDatabaseRanking":913},{"id":803,"createTime":28,"updateTime":28,"relativeEntities":2318,"label":2319,"description":2320,"key":809,"publicationTags":2321,"standard":28},[],{"EN":806,"VI":806},{"EN":806,"VI":808},[811],[912],{"id":915,"indexDatabase":2324,"url":927,"indexYears":28,"academicFieldIds":2329,"indexDatabaseRanking":28},{"id":917,"createTime":28,"updateTime":28,"relativeEntities":2325,"label":2326,"description":2327,"key":924,"publicationTags":2328,"standard":28},[],{"EN":920,"VI":920},{"EN":922,"VI":923},[926,796],[929],{"impactFactor":32,"impactFactorByYear":2331,"i10Index":934,"i10IndexLast5Year":211,"totalPublication":935,"totalPublicationByYear":2332,"totalCitation":948,"totalCitationByYear":2333,"totalCitationPerPublication":966,"totalCitationPerPublicationByYear":2334,"hindexLast5Year":829,"hindex":829},{"2012":348,"2013":347,"2014":693,"2015":286,"2016":368,"2017":368,"2018":120,"2019":230,"2020":178,"2021":933,"2022":821,"2023":340},{"2001":49,"2002":357,"2003":323,"2004":147,"2005":352,"2006":202,"2007":937,"2008":332,"2009":280,"2010":428,"2011":938,"2012":939,"2013":429,"2014":573,"2015":940,"2016":941,"2017":942,"2018":943,"2019":459,"2020":944,"2021":945,"2022":946,"2023":947,"2024":529},{"2001":601,"2002":129,"2003":360,"2004":950,"2005":938,"2006":941,"2007":951,"2008":952,"2009":953,"2010":572,"2011":954,"2012":955,"2013":956,"2014":957,"2015":958,"2016":959,"2017":960,"2018":961,"2019":962,"2020":963,"2021":964,"2022":965},{"2001":968,"2002":234,"2003":969,"2004":970,"2005":971,"2006":972,"2007":973,"2008":974,"2009":975,"2010":976,"2011":977,"2012":978,"2013":979,"2014":980,"2015":981,"2016":982,"2017":983,"2018":984,"2019":985,"2020":986,"2021":987,"2022":338},{"pages":2336,"volume":2338},{"VOID":2337},"1-11",{"VOID":2339},"20","2020-05-13",2020,[913,926],{"id":2344,"createTime":2345,"updateTime":2346,"relativeEntities":2347,"slug":2348,"properties":2349,"entityType":1007,"verifyStatus":26,"verifyTime":2346,"verifyNote":1008,"languages":28,"translateLanguages":28,"viewCount":32,"primaryUrl":2358,"fullTextUrl":28,"authors":2359,"publicationType":1068,"publisherRelationship":2441,"citationCount":28,"citationInfo":28,"publishDate":2486,"publishYear":2207,"citationAnalyzeStatus":878,"lastCitationAnalyze":28,"indexDatabases":2487,"openAccess":28,"references":28,"isForceReanalyzing":1119},"0087cdc8-c0e3-4d95-9798-6e10ed40a3ff","2024-01-30T05:45:30.901+00:00","2025-01-08T12:41:30.593+00:00",[],"Long-term-outcomes-at-24-and-36-month-follow-up-in-the-intervention-arm-of-the-randomized-controlled-trial-of-Prompt-Mental-Health-Care",{"abstract":2350,"title":2352,"references":2354,"doi":2356},{"EN":2351},"Whether long-term symptom improvement is maintained after treatment in services such as the Norwegian Prompt Mental Health Care (PMHC) and the English Improving Access to Psychological Therapies is not yet known. In this prospective study, we investigate whether improvements observed at 6-month follow-up are maintained at 24- and 36-month follow-up among clients who received PMHC. Data from the treatment arm of the randomized controlled trial of PMHC were used (n = 459). The main outcomes were (reliable) recovery rate and symptoms of depression (PHQ-9) and anxiety (GAD-7). Primary outcome data at 24- and 36-months follow-up were available for 47% and 39% of participants, respectively. Secondary outcomes were work participation, functional status, health-related quality of life, and positive mental well-being. Sensitivity analyses with regard to missing data assumptions were conducted for the primary continuous outcomes. Improvements were maintained at 24- and 36-month follow-up for symptoms of depression and anxiety, (reliable) recovery rate, and health-related quality of life. Small linear improvements since 6-month follow-up were observed for work participation, functional status, and positive mental well-being. Sensitivity analyses did not substantially alter the findings for symptoms of depression and anxiety mentioned above. Our findings support the long-term effectiveness of PMHC, but results should be interpreted with caution due to lacking follow-up data at 24- and 36-month in the control group, and substantial attrition.",{"EN":2353},"Long-term outcomes at 24- and 36-month follow-up in the intervention arm of the randomized controlled trial of Prompt Mental Health Care",{"VOID":2355},"Cuijpers P, Berking M, Andersson G, Quigley L, Kleiboer A, Dobson KS. A meta-analysis of cognitive-behavioural therapy for adult depression, alone and in comparison with other treatments. Can J Psychiatry Revue Canadienne de Psychiatrie. 2013;58(7):376–85.\nHofmann SG, Smits JA. Cognitive-behavioral therapy for adult anxiety disorders: a meta-analysis of randomized placebo-controlled trials. J Clin Psychiatry. 2008;69(4):621–32.\nHofmann SG, Asnaani A, Vonk IJ, Sawyer AT, Fang A. The efficacy of cognitive behavioral therapy: A review of meta-analyses. Cognit Ther Res. 2012;36(5):427–40.\nOtte C. Cognitive behavioral therapy in anxiety disorders: current state of the evidence. Dialogues Clin Neurosci. 2011;13(4):413–21.\nZhang A, Franklin C, Jing S, Bornheimer LA, Hai AH, Himle JA, et al. The effectiveness of four empirically supported psychotherapies for primary care depression and anxiety: A systematic review and meta-analysis. J Affect Disord. 2018;245:1168.\nTwomey C, O’Reilly G, Byrne M. Effectiveness of cognitive behavioural therapy for anxiety and depression in primary care: a meta-analysis. Fam Pract. 2014;32(1):3–15.\nCraske MG, Stein MB, Eley TC, Milad MR, Holmes A, Rapee RM, et al. Anxiety disorders. Nat Rev Dis Primers. 2017;3(1):17024.\nHaby MM, Donnelly M, Corry J, Vos T. Cognitive behavioural therapy for depression, panic disorder and generalized anxiety disorder: a meta-regression of factors that may predict outcome. Aust N Z J Psychiatry. 2006;40(1):9–19.\nClark A, Goodman G, Petitti C. The Persistent Case of Major Depression: An Argument for long-term treatment. J Contemp Psychother. 2017;48:1–8.\nVittengl JR, Clark LA, Dunn TW, Jarrett RB. Reducing relapse and recurrence in unipolar depression: a comparative meta-analysis of cognitive-behavioral therapy’s effects. J Consult Clin Psychol. 2007;75(3):475–88.\nHollon SD, Thase ME, Markowitz JC. Treatment and prevention of depression. Psychol Sci Public Interest. 2002;3(2):39–77.\nHardeveld F, Spijker J, De Graaf R, Nolen W, Beekman A. Prevalence and predictors of recurrence of major depressive disorder in the adult population. Acta Psychiatr Scand. 2010;122(3):184–91.\nYiend J, Paykel E, Merritt R, Lester K, Doll H, Burns T. Long term outcome of primary care depression. J Affect Disord. 2009;118(1–3):79–86.\nBurcusa SL, Iacono WG. Risk for recurrence in depression. Clin Psychol Rev. 2007;27(8):959–85.\nVittengl JR, Clark LA, Dunn TW, Jarrett RB. Reducing relapse and recurrence in unipolar depression: a comparative meta-analysis of cognitive-behavioral therapy’s effects. J Consult Clin Psychol. 2007;75(3):475.\nBruce SE, Yonkers KA, Otto MW, Eisen JL, Weisberg RB, Pagano M, et al. Influence of psychiatric comorbidity on recovery and recurrence in generalized anxiety disorder, social phobia, and panic disorder: a 12-year prospective study. Am J Psychiatry. 2005;162(6):1179–87.\nNorwegian Directorate of Health. Rask Psykisk Helsehjelp – 12 pilotkommuner. Veiledende materiell. [Prompt Mental Health Care - 12 pilot municipalities. Instructional material]. Oslo: Norwegian Directorate of Health; 2013.\nSmith ORF, Alves DE, Knapstad M. Rask psykisk helsehjelp: Evaluering av de første 12 pilotene i Norge [Promt Mental Health Care: Evaluation of the first 12 pilot sites in Norway]. Oslo: Norwegian Institute of Public Health; 2016.\nClark DM, Layard R, Smithies R, Richards DA, Suckling R, Wright B. Improving access to psychological therapy: Initial evaluation of two UK demonstration sites. Behav Res Ther. 2009;47(11):910–20.\nClark DM. Implementing NICE guidelines for the psychological treatment of depression and anxiety disorders: the IAPT experience. Int Rev Psychiatry. 2011;23(4):318–27.\nNational Competence Center For Mental Health Work. Rask Psykisk Helsehjelp [Prompt Mental Health Care] Trondheim, Norway: National Competence Center For Mental Health Work; 2021 [Available from: https:\u002F\u002Fnapha.no\u002Fcontent\u002F13931\u002Frask-psykisk-helsehjelp].\nLervik LV, Knapstad M, Smith ORF. Process evaluation of prompt mental health care (PMHC): the Norwegian version of Improving access to psychological therapies. BMC Health Serv Res. 2020;20:1–17.\nWakefield S, Kellett S, Simmonds-Buckley M, Stockton D, Bradbury A, Delgadillo J. Improving Access to psychological therapies (IAPT) in the United Kingdom: A systematic review and meta-analysis of 10-years of practice-based evidence. Br J Clin Psychol. 2021;60(1):1–37.\nCommunity and Mental health team HaSCIC. Psychological therapies: Annual report on the use of IAPT services England, 2019–2020,. National Health Service, England; 2020.\nAli S, Rhodes L, Moreea O, McMillan D, Gilbody S, Leach C, et al. How durable is the effect of low intensity CBT for depression and anxiety? Remission and relapse in a longitudinal cohort study. Behav Res Ther. 2017;94:1–8.\nClark DM. Realizing the mass public benefit of evidence-based psychological therapies: The IAPT program. Annu Rev Clin Psychol. 2018;14:159–83.\nKnapstad M, Nordgreen T, Smith ORF. Prompt mental health care, the Norwegian version of IAPT: clinical outcomes and predictors of change in a multicenter cohort study. BMC Psychiatry. 2018;18(1):260.\nSmith ORF, Knapstad M, Alves DE, Aaro LE. Initial results of prompt mental health care, the Norwegian version of improving access to psychological therapies. Psychother Psychosom. 2017;86(6):382–4.\nSaether SMM, Knapstad M, Grey N, Smith ORF. Twelve months post-treatment results from the Norwegian version of improving access to psychological therapies. Front Psychol. 2019;10:2303.\nKnapstad M, Lervik LV, Sæther SMM, Aarø LE, Smith ORF. Effectiveness of prompt mental health care, the Norwegian version of improving access to psychological therapies: a randomized controlled trial. Psychother Psychosom. 2020;89(2):90–105.\nSæther SMM, Knapstad M, Grey N, Rognerud MA, Smith OR. Long-term outcomes of prompt mental health care: A randomized controlled trial. Behav Res Ther. 2020;135:103758.\nKnapstad M, Lervik LV, Saether SMM, Aaro LE, Smith ORF. Effectiveness of prompt mental health care, the Norwegian version of improving access to psychological therapies: A randomized controlled trial. Psychother Psychosom. 2020;89(2):90–105.\nSaether SMM, Knapstad M, Grey N, Rognerud MA, Smith ORF. Long-term outcomes of prompt mental health care: A randomized controlled trial. Behav Res Ther. 2020;135:103758.\nKroenke K, Spitzer R, Williams J. The PHQ-9: validity of a brief depression severity measure. [Research Support Non-US Gov’t. 2001;16:606.\nKroenke K, Spitzer RL, Williams JB, Löwe B. The patient health questionnaire somatic, anxiety, and depressive symptom scales: a systematic review. Gen Hosp Psychiatry. 2010;32(4):345–59.\nSpitzer RL, Kroenke K, Williams JB, Löwe B. A brief measure for assessing generalized anxiety disorder: the GAD-7. Arch Intern Med. 2006;166(10):1092–7.\nKroenke K, Spitzer RL, Williams JB, Monahan PO, Löwe B. Anxiety disorders in primary care: prevalence, impairment, comorbidity, and detection. Ann Intern Med. 2007;146(5):317–25.\nMundt JC, Marks IM, Shear MK, Greist JM. The work and social adjustment scale: a simple measure of impairment in functioning. Br J Psychiatry. 2002;180(5):461–4.\nZahra D, Qureshi A, Henley W, Taylor R, Quinn C, Pooler J, et al. The work and social adjustment scale: reliability, sensitivity and value. Int J Psychiatry Clin Pract. 2014;18(2):131–8.\nRabin R, Charro Fd. EQ-SD: a measure of health status from the EuroQol group. Ann Med. 2001;33(5):337–43.\nEuroQol. EQ-5D-5L | Self-complete version on paper [Available from: https:\u002F\u002Feuroqol.org\u002Feq-5d-instruments\u002Feq-5d-5l-available-modes-of-administration\u002Fself-complete-on-paper\u002F].\nDevlin NJ, Shah KK, Feng Y, Mulhern B, van Hout B. Valuing health-related quality of life: An EQ-5D-5L value set for England. Health Econ. 2018;27(1):7–22.\nHansen TM, Helland Y, Augestad LA, Rand K, Stavem K, Garratt A. Elicitation of Norwegian EQ-5D-5L values for hypothetical and experience-based health states based on the EuroQol Valuation Technology (EQ-VT) protocol. Bmj Open. 2020;10:6.\nSobocki P, Ekman M, Ågren H, Krakau I, Runeson B, Mårtensson B, et al. Health-related quality of life measured with EQ-5D in patients treated for depression in primary care. Value Health. 2007;10(2):153–60.\nTennant R, Hiller L, Fishwick R, Platt S, Joseph S, Weich S, et al. The Warwick-Edinburgh mental well-being scale (WEMWBS): development and UK validation. Health Qual Life Outcomes. 2007;5(1):1–13.\nStewart-Brown SL, Platt S, Tennant A, Maheswaran H, Parkinson J, Weich S, et al. The Warwick-Edinburgh mental well-being scale (WEMWBS): a valid and reliable tool for measuring mental well-being in diverse populations and projects. J Epidemiol Community Health. 2011;65(Suppl 2):A38–9.\nBartram DJ, Sinclair JM, Baldwin DS. Further validation of the Warwick-Edinburgh mental well-being scale (WEMWBS) in the UK veterinary profession: Rasch analysis. Qual Life Res. 2013;22(2):379–91.\nSmith OR, Alves DE, Knapstad M, Haug E, Aarø LE. Measuring mental well-being in Norway: validation of the Warwick-Edinburgh mental well-being scale (WEMWBS). BMC Psychiatry. 2017;17(1):1–9.\nHu LT, Bentler PM. Cutoff criteria for fit indexes in covariance structure analysis: Conventional criteria versus new alternatives. Struct Equation Model Multidisciplinary J. 1999;6(1):1–55.\nEnders CK. Applied missing data analysis. New York, NY, US: Guilford Press; 2010.\nvon Brachel R, Hirschfeld G, Berner A, Willutzki U, Teismann T, Cwik J, et al. Long-term effectiveness of cognitive behavioral therapy in routine outpatient care: A 5-to 20-year follow-up study. Psychother Psychosom. 2019;88(4):225–35.\nChambless DL, Ollendick TH. Empirically supported psychological, interventions: Controversies and evidence. Annu Rev Psychol. 2001;52:685–716.\nvan Dis EAM, van Veen SC, Hagenaars MA, Batelaan NM, Bockting CLH, van den Heuvel RM, et al. Long-term outcomes of cognitive behavioral therapy for anxiety-related disorders A systematic review and meta-analysis. JAMA Psychiat. 2020;77(3):265–73.\nCuijpers P, Hollon SD, van Straten A, Bockting C, Berking M, Andersson G. Does cognitive behaviour therapy have an enduring effect that is superior to keeping patients on continuation pharmacotherapy? A meta-analysis. Bmj Open. 2013;3(4):e002542.\nHans E, Hiller W. A meta-analysis of nonrandomized effectiveness studies on outpatient cognitive behavioral therapy for adult anxiety disorders. Clin Psychol Rev. 2013;33(8):954–64.\nHans E, Hiller W. Effectiveness of and dropout from outpatient cognitive behavioral therapy for adult unipolar depression: A meta-analysis of nonrandomized effectiveness studies. J Consult Clin Psychol. 2013;81(1):75–88.\nMcKnight PE, Kashdan TB. The importance of functional impairment to mental health outcomes: A case for reassessing our goals in depression treatment research. Clin Psychol Rev. 2009;29(3):243–59.\nKennedy N, Foy K, Sherazi R, McDonough M, McKeon P. Long-term social functioning after depression treated by psychiatrists: a review. Bipolar Disord. 2007;9(1–2):25–37.",{"VOID":2357},"10.1186\u002Fs12888-022-04227-0","https:\u002F\u002Fbmcpsychiatry.biomedcentral.com\u002Farticles\u002F10.1186\u002Fs12888-022-04227-0",[2360,2393,2406,2428],{"id":2361,"sortIndex":32,"researcher":28,"roles":2362,"affiliations":2363,"properties":2390,"displayName":2392,"givenName":28,"familyName":28},"f4362c0d-fd55-48b3-8de4-33e775b009aa",[1014],[2364,2372,2381],{"id":2365,"sortIndex":32,"affiliation":2366,"properties":28},"fed5e0cd-dac3-474d-8c86-5a705df0adb0",{"id":2365,"createTime":28,"updateTime":28,"relativeEntities":2367,"slug":28,"properties":2368,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2371,"statistic":28},[],{"title":2369},{"VI":2370},"Division of Mental and Physical Health, Department of Health Promotion, Norwegian Institute of Public Health, Bergen, Norway",[],{"id":2373,"sortIndex":40,"affiliation":2374,"properties":2380},"c00419ff-2e66-45fb-8c9e-1eaf8cd9aa11",{"id":2373,"createTime":28,"updateTime":28,"relativeEntities":2375,"slug":28,"properties":2376,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2379,"statistic":28},[],{"title":2377},{"VI":2378},"Centre for Evaluation of Public Health Measure, Norwegian Institute of Public Health, Bergen, Norway",[],{},{"id":2382,"sortIndex":123,"affiliation":2383,"properties":2389},"4188e633-37bd-484f-997b-bedadbc7b565",{"id":2382,"createTime":28,"updateTime":28,"relativeEntities":2384,"slug":28,"properties":2385,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2388,"statistic":28},[],{"title":2386},{"VI":2387},"Department of Teacher Education, NLA University College, Bergen, Norway",[],{},{"title":2391},{"VI":2392},"Otto R. F. Smith",{"id":2394,"sortIndex":40,"researcher":28,"roles":2395,"affiliations":2396,"properties":2403,"displayName":2405,"givenName":28,"familyName":28},"3146cc4d-0d4c-4e4c-accc-217b5040a419",[1014],[2397],{"id":2365,"sortIndex":32,"affiliation":2398,"properties":28},{"id":2365,"createTime":28,"updateTime":28,"relativeEntities":2399,"slug":28,"properties":2400,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2402,"statistic":28},[],{"title":2401},{"VI":2370},[],{"title":2404},{"VI":2405},"Solbjørg M. M. Sæther",{"id":2407,"sortIndex":123,"researcher":28,"roles":2408,"affiliations":2409,"properties":2425,"displayName":2427,"givenName":28,"familyName":28},"b1313bdc-b3fc-4f37-b15c-542fd0406e64",[1014],[2410,2416],{"id":2382,"sortIndex":32,"affiliation":2411,"properties":28},{"id":2382,"createTime":28,"updateTime":28,"relativeEntities":2412,"slug":28,"properties":2413,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2415,"statistic":28},[],{"title":2414},{"VI":2387},[],{"id":2417,"sortIndex":40,"affiliation":2418,"properties":2424},"6dc999cc-25ab-4548-9555-14ab9b6c66a4",{"id":2417,"createTime":28,"updateTime":28,"relativeEntities":2419,"slug":28,"properties":2420,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2423,"statistic":28},[],{"title":2421},{"VI":2422},"Department of Health Promotion and Development, University of Bergen, Bergen, Norway",[],{},{"title":2426},{"VI":2427},"Ellen Haug",{"id":2429,"sortIndex":42,"researcher":28,"roles":2430,"affiliations":2431,"properties":2438,"displayName":2440,"givenName":28,"familyName":28},"e8e9e83f-cfc5-48cf-ac29-4b3d2af557be",[1014],[2432],{"id":2365,"sortIndex":32,"affiliation":2433,"properties":28},{"id":2365,"createTime":28,"updateTime":28,"relativeEntities":2434,"slug":28,"properties":2435,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2437,"statistic":28},[],{"title":2436},{"VI":2370},[],{"title":2439},{"VI":2440},"Marit 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study investigated the impact of psychiatric disorders on Quality of Life (QOL) cross-sectionally and longitudinally in a group of Norwegian tourists severely exposed to the 2004 tsunami. Sixty-two adult Norwegian tsunami survivors were interviewed face to face 2 years post-tsunami (T1) and 58 were interviewed again by telephone 6 years post-tsunami (T2). The majority (81 %) reported direct exposure to the waves, and 14 participants (23 %) lost a close family member in the tsunami. Psychiatric morbidity was measured by structured clinical interviews and QOL was assessed with WHO’s Quality of Life-Bref scale. Multiple linear regression analyses were performed to assess the independent effects of psychiatric disorders on QOL 2 and 6 years after the tsunami. Psychiatric disorders, especially depression, but also PTSD and other anxiety disorders, were associated with reduced QOL. Psychiatric disorders were more strongly related to QOL at 6 years after the tsunami than at 2 years. Psychiatric disorders, and especially depression, is related to reduced QOL in a disaster exposed population. Post-disaster psychiatric disorders, such as PTSD and especially depression, should be addressed properly in the aftermath of disasters.",{"EN":2498},"The relationship between psychiatric morbidity and quality of life: interview study of Norwegian tsunami survivors 2 and 6 years post-disaster",{"VOID":2500},"Sirgy MJ, Michalos AC, Ferriss AL, Easterlin RA, Pavot W, Patrick D. The quality of life (QOL) research movement: past, present and future. Soc Indic Res. 2006;76:343–466.\nWHO QoL Group. The development of the World Health Organization Quality of Life Assessment Instrument (the WHOQoL). In: Orley J, Kuyken W, editors. Quality of Life Assessment: International Perspectives. Heidleberg: Springer Verlag; 1994. p. 44.\nCenters for Disease Control and Prevention, Atlanta, USA. Health related quality of life - Key findings. [www.cdc.gov\u002Fchronicdisease\u002Fabout\u002Fprograms.htm]. Accessed 27 Apr 2016.\nZhang X, Norris SL, Chowdhury FM, Gregg EW, Zhang P. 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J Trauma Stress. 2002;15:313–19.\nHjemdal OK. “Follow-up of disaster victims. The role of general practitioners”, (In Norwegian, English summary). Resource document. Oslo: Norwegian Centre for Violence and Traumatic Stress Studies; 2007. Retrieved from [https:\u002F\u002Fwww.nkvts.no\u002Fcontent\u002Fuploads\u002F2015\u002F08\u002Ftsunamien_fastlegerapport2.pdf], Accessed 27 May 2016.\nMichel PO, Rosendal S, Weisaeth L, Heir T. Use of and satisfaction with support received among survivors from three Scandinavian countries after the 2004 Southeast Asian tsunami. 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disorder is one of the most disabling of all psychiatric illnesses. Despite available pharmacological and psychotherapeutic treatments about 10% of patients remain severely affected and are considered treatment-refractory. For some of these patients deep brain stimulation offers an appropriate treatment method. The scope of this article is to review the published data and to compare different target structures and their effectiveness. PubMed search, last update June 2013, was conducted using the terms “deep brain stimulation” and “obsessive compulsive disorder”. In total 25 studies were found that reported five deep brain stimulation target structures to treat obsessive-compulsive disorder: the anterior limb of the internal capsule (five studies including 14 patients), nucleus accumbens (eight studies including 37 patients), ventral capsule\u002Fventral striatum (four studies including 29 patients), subthalamic nucleus (five studies including 23 patients) and inferior thalamic peduncle (two studies including 6 patients). Despite the anatomical diversity, deep brain stimulation treatment results in similar response rates for the first four target structures. Inferior thalamic peduncle deep brain stimulation results in higher response rates but these results have to be interpreted with caution due to a very small number of cases. Procedure and device related adverse events are relatively low, as well as stimulation or therapy related side effects. Most stimulation related side effects are transient and decline after stimulation parameters have been changed. Deep brain stimulation in treatment-refractory obsessive-compulsive disorder seems to be a relatively safe and promising treatment option. However, based on these studies no superior target structure could be identified. More research is needed to better understand mechanisms of action and response predictors that may help to develop a more personalized approach for these severely affected obsessive compulsive patients.",{"EN":2655},"Deep brain stimulation for treatment-refractory obsessive compulsive disorder: a systematic review",{"VOID":2657},"Murray CJ, Lopez AD: Evidence-based health policy–lessons from the Global Burden of Disease Study. Science. 1996, 274 (5288): 740-743. 10.1126\u002Fscience.274.5288.740.\nRobins LN, Helzer JE, Weissman MM, Orvaschel H, Gruenberg E, Burke JD, Regier DA: Lifetime prevalence of specific psychiatric disorders in three sites. Arch Gen Psychiatry. 1984, 41 (10): 949-958. 10.1001\u002Farchpsyc.1984.01790210031005.\nMcIntyre CC, Hahn PJ: Network perspectives on the mechanisms of deep brain stimulation. 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CNS Spectr. 2005, 10 (12): 966-979. 983\nDenys D, Mantione M, Figee M, van den Munckhof P, Koerselman F, Westenberg H, Bosch A, Schuurman R: Deep brain stimulation of the nucleus accumbens for treatment-refractory obsessive-compulsive disorder. Arch Gen Psychiatry. 2010, 67 (10): 1061-1068. 10.1001\u002Farchgenpsychiatry.2010.122.\nNuttin B, Cosyns P, Demeulemeester H, Gybels J, Meyerson B: Electrical stimulation in anterior limbs of internal capsules in patients with obsessive-compulsive disorder. Lancet. 1999, 354 (9189): 1526-10.1016\u002FS0140-6736(99)02376-4.\nKuhn J, Lenartz D, Mai JK, Huff W, Lee SH, Koulousakis A, Klosterkoetter J, Sturm V: Deep brain stimulation of the nucleus accumbens and the internal capsule in therapeutically refractory Tourette-syndrome. J Neurol. 2007, 254 (7): 963-965. 10.1007\u002Fs00415-006-0404-8.\nVandewalle V, van der Linden C, Groenewegen HJ, Caemaert J: Stereotactic treatment of Gilles de la Tourette syndrome by high frequency stimulation of thalamus. Lancet. 1999, 353 (9154): 724-10.1016\u002FS0140-6736(98)05964-9.\nKuhn J, Lenartz D, Huff W, Lee S, Koulousakis A, Klosterkoetter J, Sturm V: Remission of alcohol dependency following deep brain stimulation of the nucleus accumbens: valuable therapeutic implications?. J Neurol Neurosurg Psychiatry. 2007, 78 (10): 1152-1153. 10.1136\u002Fjnnp.2006.113092.\nMuller UJ, Voges J, Steiner J, Galazky I, Heinze HJ, Moller M, Pisapia J, Halpern C, Caplan A, Bogerts B, Kuhn J: Deep brain stimulation of the nucleus accumbens for the treatment of addiction. Ann N Y Acad Sci. 2013, 1282: 119-128. 10.1111\u002Fj.1749-6632.2012.06834.x.\nMayberg HS, Lozano AM, Voon V, McNeely HE, Seminowicz D, Hamani C, Schwalb JM, Kennedy SH: Deep brain stimulation for treatment-resistant depression. Neuron. 2005, 45 (5): 651-660. 10.1016\u002Fj.neuron.2005.02.014.\nFigee M, Luigjes J, Smolders R, Valencia-Alfonso CE, van Wingen G, de Kwaasteniet B, Mantione M, Ooms P, de Koning P, Vulink N, Levar N, Droge L, van den Munckhof P, Schuurman PR, Nederveen A, van den Brink W, Mazaheri A, Vink M, Denys D: Deep brain stimulation restores frontostriatal network activity in obsessive-compulsive disorder. Nat Neurosci. 2013, 16 (4): 386-387. 10.1038\u002Fnn.3344.\nLuigjes J, de Kwaasteniet BP, de Koning PP, Oudijn MS, van den Munckhof P, Schuurman PR, Denys D: Surgery for psychiatric disorders. World Neurosurg. 2013, 80 (3–4): S31 e17-28-\nMoher D, Liberati A, Tetzlaff J, Altman DG: Preferred reporting items for systematic reviews and meta-analyses: the PRISMA statement. PLoS Med. 2009, 6 (7): e1000097-10.1371\u002Fjournal.pmed.1000097.\nMindus P, Rasmussen SA, Lindquist C: Neurosurgical treatment for refractory obsessive-compulsive disorder: implications for understanding frontal lobe function. J Neuropsychiatry Clin Neurosci. 1994, 6 (4): 467-477.\nNuttin B, Gabriels LA, Cosyns PR, Meyerson BA, Andreewitch S, Sunaert SG, Maes AF, Dupont PJ, Gybels JM, Gielen F, Demeulemeester HG: Long-term electrical capsular stimulation in patients with obsessive-compulsive disorder. Neurosurgery. 2003, 52 (6): 1263-1272. 10.1227\u002F01.NEU.0000064565.49299.9A. discussion 1272–1264\nGabriels L, Cosyns P, Nuttin B, Demeulemeester H, Gybels J: Deep brain stimulation for treatment-refractory obsessive-compulsive disorder: psychopathological and neuropsychological outcome in three cases. Acta Psychiatr Scand. 2003, 107 (4): 275-282. 10.1034\u002Fj.1600-0447.2003.00066.x.\nAnderson D, Ahmed A: Treatment of patients with intractable obsessive-compulsive disorder with anterior capsular stimulation. Case report J Neurosurg. 2003, 98 (5): 1104-1108. 10.3171\u002Fjns.2003.98.5.1104.\nAbelson JL, Curtis GC, Sagher O, Albucher RC, Harrigan M, Taylor SF, Martis B, Giordani B: Deep brain stimulation for refractory obsessive-compulsive disorder. Biol Psychiat. 2005, 57 (5): 510-516. 10.1016\u002Fj.biopsych.2004.11.042.\nServello D, Sassi M, Bastianello S, Poloni GU, Mancini F, Pacchetti C: Electrode displacement after intracerebral hematoma as a complication of a deep brain stimulation procedure. Neuropsychiatr Dis Treat. 2009, 5: 183-187.\nOkun MS, Mann G, Foote KD, Shapira NA, Bowers D, Springer U, Knight W, Martin P, Goodman WK: Deep brain stimulation in the internal capsule and nucleus accumbens region: responses observed during active and sham programming. J Neurol Neurosurg Psychiatry. 2007, 78 (3): 310-314. 10.1136\u002Fjnnp.2006.095315.\nMogenson GJ, Yang CR: The contribution of basal forebrain to limbic-motor integration and the mediation of motivation to action. Adv Exp Med Biol. 1991, 295: 267-290. 10.1007\u002F978-1-4757-0145-6_14.\nSturm V, Lenartz D, Koulousakis A, Treuer H, Herholz K, Klein JC, Klosterkotter J: The nucleus accumbens: a target for deep brain stimulation in obsessive-compulsive- and anxiety-disorders. J Chem Neuroanat. 2003, 26 (4): 293-299. 10.1016\u002Fj.jchemneu.2003.09.003.\nHuff W, Lenartz D, Schormann M, Lee SH, Kuhn J, Koulousakis A, Mai J, Daumann J, Maarouf M, Klosterkotter J, Sturm V: Unilateral deep brain stimulation of the nucleus accumbens in patients with treatment-resistant obsessive-compulsive disorder: Outcomes after one year. Clin Neurol Neurosurg. 2010, 112 (2): 137-143. 10.1016\u002Fj.clineuro.2009.11.006.\nAouizerate B, Cuny E, Martin-Guehl C, Guehl D, Amieva H, Benazzouz A, Fabrigoule C, Allard M, Rougier A, Bioulac B, Tignol J, Burbaud P: Deep brain stimulation of the ventral caudate nucleus in the treatment of obsessive-compulsive disorder and major depression. Case report J Neurosurg. 2004, 101 (4): 682-686. 10.3171\u002Fjns.2004.101.4.0682.\nPlewnia C, Schober F, Rilk A, Buchkremer G, Reimold M, Wachter T, Breit S, Weiss D, Kruger R, Freudenstein D: Sustained improvement of obsessive-compulsive disorder by deep brain stimulation in a woman with residual schizophrenia. Int J Neuropsychopharmacol. 2008, 11 (8): 1181-1183. 10.1017\u002FS1461145708009188.\nShimizu E: [Definition of treatment-refractory obsessive-compulsive disorder based on cognitive behavioral therapy]. Seishin Shinkeigaku Zasshi. 2013, 115 (9): 975-980.\nFranzini A, Messina G, Gambini O, Muffatti R, Scarone S, Cordella R, Broggi G: Deep-brain stimulation of the nucleus accumbens in obsessive compulsive disorder: clinical, surgical and electrophysiological considerations in two consecutive patients. Neurol Sci. 2010, 31 (3): 353-359. 10.1007\u002Fs10072-009-0214-8.\nRoh D, Chang WS, Chang JW, Kim CH: Long-term follow-up of deep brain stimulation for refractory obsessive-compulsive disorder. Psychiatry Res. 2012, 200 (2–3): 1067-1070. 10.1016\u002Fj.psychres.2012.06.018.\nBarcia JA, Reyes L, Arza R, Saceda J, Avecillas J, Yanez R, Garcia-Albea J, Ortiz T, Lopez-Ibor MI, Lopez-Ibor JJ: Deep Brain Stimulation for Obsessive-Compulsive Disorder: Is the Side Relevant?. Stereotact Funct Neurosurg. 2013, 92 (1): 31-36. 10.1159\u002F000353187.\nCorveleyn P, Nuttin B, Gabriels L: Deep brain stimulation in a patient with ocd and the intensive pre- and post-operative psychiatric\u002Fpsychotherapeutic follow-up. A case study. Tijdschr Psychiatr. 2013, 55 (3): 203-208.\nGreenberg BD, Malone DA, Friehs GM, Rezai AR, Kubu CS, Malloy PF, Salloway SP, Okun MS, Goodman WK, Rasmussen SA: Three-year outcomes in deep brain stimulation for highly resistant obsessive-compulsive disorder. Neuropsychopharmacology. 2006, 31 (11): 2384-2393. 10.1038\u002Fsj.npp.1301165.\nGreenberg BD, Gabriels LA, Malone DA, Rezai AR, Friehs GM, Okun MS, Shapira NA, Foote KD, Cosyns PR, Kubu CS, Malloy PF, Salloway SP, Giftakis JE, Rise MT, Machado AG, Baker KB, Stypulkowski PH, Goodman WK, Rasmussen SA, Nuttin BJ: Deep brain stimulation of the ventral internal capsule\u002Fventral striatum for obsessive-compulsive disorder: worldwide experience. Mol Psychiatry. 2010, 15 (1): 64-79. 10.1038\u002Fmp.2008.55.\nGoodman WK, Foote KD, Greenberg BD, Ricciuti N, Bauer R, Ward H, Shapira NA, Wu SS, Hill CL, Rasmussen SA, Okun MS: Deep brain stimulation for intractable obsessive compulsive disorder: pilot study using a blinded, staggered-onset design. Biol Psychiat. 2010, 67 (6): 535-542. 10.1016\u002Fj.biopsych.2009.11.028.\nTsai HC, Chang CH, Pan JI, Hsieh HJ, Tsai ST, Hung HY, Chen SY: Pilot study of deep brain stimulation in refractory obsessive-compulsive disorder ethnic Chinese patients. Psychiatry Clin Neurosci. 2012, 66 (4): 303-312. 10.1111\u002Fj.1440-1819.2012.02352.x.\nMallet L, Mesnage V, Houeto JL, Pelissolo A, Yelnik J, Behar C, Gargiulo M, Welter ML, Bonnet AM, Pillon B, Cornu P, Dormont D, Pidoux B, Allilaire JF, Agid Y: Compulsions, Parkinson’s disease, and stimulation. Lancet. 2002, 360 (9342): 1302-1304. 10.1016\u002FS0140-6736(02)11339-0.\nFontaine D, Mattei V, Borg M, von Langsdorff D, Magnie MN, Chanalet S, Robert P, Paquis P: Effect of subthalamic nucleus stimulation on obsessive-compulsive disorder in a patient with Parkinson disease. Case report J Neurosurg. 2004, 100 (6): 1084-1086. 10.3171\u002Fjns.2004.100.6.1084.\nMallet L, Polosan M, Jaafari N, Baup N, Welter ML, Fontaine D, du Montcel ST, Yelnik J, Chereau I, Arbus C, Raoul S, Aouizerate B, Damier P, Chabardès S, Czernecki V, Ardouin C, Krebs MO, Bardinet E, Chaynes P, Burbaud P, Cornu P, Derost P, Bougerol T, Bataille B, Mattei V, Dormont D, Devaux B, Vérin M, Houeto JL, Pollak P, et al: Subthalamic nucleus stimulation in severe obsessive-compulsive disorder. N Engl J Med. 2008, 359 (20): 2121-2134. 10.1056\u002FNEJMoa0708514.\nChabardes S, Polosan M, Krack P, Bastin J, Krainik A, David O, Bougerol T, Benabid AL: Deep brain stimulation for obsessive-compulsive disorder: subthalamic nucleus target. World Neurosurg. 2013, 80 (3–4): S31 e31-38-\nJimenez-Ponce F, Velasco-Campos F, Castro-Farfan G, Nicolini H, Velasco AL, Salin-Pascual R, Trejo D, Criales JL: Preliminary study in patients with obsessive-compulsive disorder treated with electrical stimulation in the inferior thalamic peduncle. Neurosurgery. 2009, 65 (6 Suppl): 203-209. discussion 209\nJimenez F, Nicolini H, Lozano AM, Piedimonte F, Salin R, Velasco F: Electrical stimulation of the inferior thalamic peduncle in the treatment of major depression and obsessive compulsive disorders. World Neurosurg. 2013, 80 (3–4): S30 e17-25-\nDell'Osso B, Porta M, Servello D, Altamura AC: Deep Brain Stimulation device removal after scar picking behaviors in a patient with treatment-resistant obsessive compulsive disorder. Brain Stimul. 2013, 6 (1): 96-98. 10.1016\u002Fj.brs.2012.01.007.\nVora AK, Ward H, Foote KD, Goodman WK, Okun MS: Rebound symptoms following battery depletion in the NIH OCD DBS cohort: clinical and reimbursement issues. Brain Stimul. 2012, 5 (4): 599-604. 10.1016\u002Fj.brs.2011.10.004.\nWitt K, Kuhn J, Timmermann L, Zurowski M, Woopen C: Deep Brain Stimulation and the Search for Identity. Neuroethics. 2013, 6: 499-511. 10.1007\u002Fs12152-011-9100-1.\nGarnaat SL, Greenberg BD, Sibrava NJ, Goodman WK, Mancebo MC, Eisen JL, Rasmussen SA: Who Qualifies for Deep Brain Stimulation for OCD? Data From a Naturalistic Clinical Sample. J Neuropsychiatry Clin Neurosci. 2014, 26 (1): 81-86.\nRiva-Posse P, Choi KS, Holtzheimer PE, McIntyre CC, Gross RE, Chaturvedi A, Crowell AL, Garlow SJ, Rajendra JK, Mayberg HS: Defining Critical White Matter Pathways Mediating Successful Subcallosal Cingulate Deep Brain Stimulation for Treatment-Resistant Depression.Biol Psychiat 2014, Advanced online publication. Retrieved from doi:10.1016\u002Fj.biopsych.2014.03.029.,",{"VOID":2659},"10.1186\u002Fs12888-014-0214-y","https:\u002F\u002Fbmcpsychiatry.biomedcentral.com\u002Farticles\u002F10.1186\u002Fs12888-014-0214-y",[2662,2677,2690,2705,2727,2742,2757,2770],{"id":2663,"sortIndex":32,"researcher":28,"roles":2664,"affiliations":2665,"properties":2674,"displayName":2676,"givenName":28,"familyName":28},"06e8b7f0-734b-466c-a6b4-760e3a12dc13",[1014],[2666],{"id":2667,"sortIndex":32,"affiliation":2668,"properties":28},"86a665f1-bad9-41db-a342-ebf4a6e42089",{"id":2667,"createTime":28,"updateTime":28,"relativeEntities":2669,"slug":28,"properties":2670,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2673,"statistic":28},[],{"title":2671},{"VI":2672},"Department of Psychiatry and Psychotherapy, University of Cologne, Cologne, Germany",[],{"title":2675},{"VI":2676},"Sina Kohl",{"id":2678,"sortIndex":40,"researcher":28,"roles":2679,"affiliations":2680,"properties":2687,"displayName":2689,"givenName":28,"familyName":28},"cdc3b4fd-25f8-416d-91eb-104585e09f0e",[1014],[2681],{"id":2667,"sortIndex":32,"affiliation":2682,"properties":28},{"id":2667,"createTime":28,"updateTime":28,"relativeEntities":2683,"slug":28,"properties":2684,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2686,"statistic":28},[],{"title":2685},{"VI":2672},[],{"title":2688},{"VI":2689},"Deva M Schönherr",{"id":2691,"sortIndex":123,"researcher":28,"roles":2692,"affiliations":2693,"properties":2702,"displayName":2704,"givenName":28,"familyName":28},"3a3ef4d3-88a5-460e-aa72-78376c749284",[1014],[2694],{"id":2695,"sortIndex":32,"affiliation":2696,"properties":28},"8d7013eb-8746-481b-b510-47705f682def",{"id":2695,"createTime":28,"updateTime":28,"relativeEntities":2697,"slug":28,"properties":2698,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2701,"statistic":28},[],{"title":2699},{"VI":2700},"Department of Psychiatry, Academic Medical Center, Amsterdam, The Netherlands",[],{"title":2703},{"VI":2704},"Judy Luigjes",{"id":2706,"sortIndex":42,"researcher":28,"roles":2707,"affiliations":2708,"properties":2724,"displayName":2726,"givenName":28,"familyName":28},"6cc6cd62-0130-4fc4-9360-b8a9c702ebad",[1014],[2709,2715],{"id":2695,"sortIndex":32,"affiliation":2710,"properties":28},{"id":2695,"createTime":28,"updateTime":28,"relativeEntities":2711,"slug":28,"properties":2712,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2714,"statistic":28},[],{"title":2713},{"VI":2700},[],{"id":2716,"sortIndex":40,"affiliation":2717,"properties":2723},"35810d39-4029-4972-8ec6-e6f4d712fcf6",{"id":2716,"createTime":28,"updateTime":28,"relativeEntities":2718,"slug":28,"properties":2719,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2722,"statistic":28},[],{"title":2720},{"VI":2721},"The Netherlands Institute for Neuroscience, an institute of the Royal Netherlands Academy of Arts and Sciences, Amsterdam, The Netherlands",[],{},{"title":2725},{"VI":2726},"Damiaan Denys",{"id":2728,"sortIndex":45,"researcher":28,"roles":2729,"affiliations":2730,"properties":2739,"displayName":2741,"givenName":28,"familyName":28},"f73d5961-9879-44c5-b98d-5e3733dd1547",[1014],[2731],{"id":2732,"sortIndex":32,"affiliation":2733,"properties":28},"66c82494-82ab-4abd-82db-a90c6ba4d42f",{"id":2732,"createTime":28,"updateTime":28,"relativeEntities":2734,"slug":28,"properties":2735,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2738,"statistic":28},[],{"title":2736},{"VI":2737},"Department of Psychiatry and Psychotherapy, University of Magdeburg, Magdeburg, Germany",[],{"title":2740},{"VI":2741},"Ulf J Mueller",{"id":2743,"sortIndex":46,"researcher":28,"roles":2744,"affiliations":2745,"properties":2754,"displayName":2756,"givenName":28,"familyName":28},"d3c2aef8-5445-413f-8d7f-efd085fda093",[1014],[2746],{"id":2747,"sortIndex":32,"affiliation":2748,"properties":28},"5ca5ce6d-d542-43ab-b0b7-947bf3e39e95",{"id":2747,"createTime":28,"updateTime":28,"relativeEntities":2749,"slug":28,"properties":2750,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2753,"statistic":28},[],{"title":2751},{"VI":2752},"Department of Stereotactic and Functional Neurosurgery, University of Cologne, Cologne, Germany",[],{"title":2755},{"VI":2756},"Doris Lenartz",{"id":2758,"sortIndex":48,"researcher":28,"roles":2759,"affiliations":2760,"properties":2767,"displayName":2769,"givenName":28,"familyName":28},"6c0f2785-10f7-47c9-9908-cf3dd9187f2b",[1014],[2761],{"id":2747,"sortIndex":32,"affiliation":2762,"properties":28},{"id":2747,"createTime":28,"updateTime":28,"relativeEntities":2763,"slug":28,"properties":2764,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2766,"statistic":28},[],{"title":2765},{"VI":2752},[],{"title":2768},{"VI":2769},"Veerle Visser-Vandewalle",{"id":2771,"sortIndex":49,"researcher":28,"roles":2772,"affiliations":2773,"properties":2780,"displayName":2782,"givenName":28,"familyName":28},"b0d16166-954f-4ccb-948b-7b0378431d7a",[1014],[2774],{"id":2667,"sortIndex":32,"affiliation":2775,"properties":28},{"id":2667,"createTime":28,"updateTime":28,"relativeEntities":2776,"slug":28,"properties":2777,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2779,"statistic":28},[],{"title":2778},{"VI":2672},[],{"title":2781},{"VI":2782},"Jens 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plays a role in the regulation of emotional states like depression. The aim of this study was to investigate the plasma nesfatin-1levels in Chinese patients with depression and healthy subjects, and to determine the possible association between the plasma nesfatin-1 level and the severity of depression. A total of 103 depressive patients and 32 healthy subjects were assessed. According to HAMD-17scores, 51, 18, and 34 patients were enrolled in the mild depression, moderate depression, and severe depression groups, respectively. Plasma nesfatin-1 levels were determined by the ELISA method. Differences between groups were compared and associations between plasma nesfatin-1 and other variables were analyzed. The plasma nesfatin-1 was significantly positively correlated with HAMD-17 score (r = 0.651). Compared with healthy controls (8.11 ± 3.31 ng\u002FmL), the plasma nesfatin-1 level significantly increased in patients with mild depression (11.17 ± 3.58 ng\u002FmL), with moderate depression (16.33 ± 8.78 ng\u002FmL), and with severe depression (27.65 ± 8.26 ng\u002FmL) respectively. Plasma nesfatin-1 level (Odds ratio [OR] = 1.269) was an independent indicator for severe depression by multivariate logistic regression analysis. The plasma nesfatin-1 level is positively correlated with the severity of depression. Plasma nesfatin-1 level may be a potential indicator for depression severity.",{"EN":2839},"Plasma nesfatin-1 level is associated with severity of depression in Chinese depressive patients",{"VOID":2841},"Mogi T, Toda H, Yoshino A. Clinical characteristics of patients with diagnostic uncertainty of major depressive disorder. Asian J Psychiatr. 2017;30:159–62.\nSetoyama D, Kato TA, Hashimoto R, Kunugi H, Hattori K, Hayakawa K, et al. Plasma metabolites predict severity of depression and suicidal ideation in psychiatric patients-a multicenter pilot analysis. PLoS One. 2016;11:e0165267.\nHuang TL, Lin CC. Advances in biomarkers of major depressive disorder. Adv Clin Chem. 2015;68:177–204.\nCenik B, Cenik C, Snyder MP, Brown ES. Plasma sterols and depressive symptom severity in a population-based cohort. PLoS One. 2017;12:e0184382.\nLamers F, Milaneschi Y, de Jonge P, Giltay EJ, Penninx BWJH. Metabolic and inflammatory markers: associations with individual depressive symptoms. Psychol Med. 2017; doi: https:\u002F\u002Fdoi.org\u002F10.1017\u002FS0033291717002483.\nAydin S. Role of NUCB2\u002Fnesfatin-1 as a possible biomarker. Curr Pharm Des. 2013;19:6986–92.\nÇelik F, Belviranli M, Okudan N. Circulating levels of leptin, nesfatin-1 and kisspeptin in postmenopausal obese women. Arch Physiol Biochem. 2016;122:195–9.\nEmmerzaal TL, Kozicz T. Nesfatin-1; implication in stress and stress-associated anxiety and depression. Curr Pharm Des. 2013;19:6941–8.\nXu YY, Ge JF, Qin G, Peng YN, Zhang CF, Liu XR, et al. Acute, but not chronic, stress increased the plasma concentration and hypothalamic mRNA expression of NUCB2\u002Fnesfatin-1 in rats. Neuropeptides. 2015;54:47–53.\nKönczöl K, Bodnár I, Zelena D, Pintér O, Papp RS, Palkovits M, et al. Nesfatin-1\u002FNUCB2 may participate in the activation of the hypothalamic-pituitary-adrenal axis in rats. Neurochem Int. 2010;57:189–97.\nGe JF, Xu YY, Qin G, Peng YN, Zhang CF, Liu XR, et al. Depression-like behavior induced by Nesfatin-1 in rats: involvement of increased immune activation and imbalance of synaptic vesicle proteins. Front Neurosci. 2015;9:429.\nGeng LY, Qian FY, Qian JF, Zhang ZJ. The combination of plasma glutamate and physical impairment after acute stroke as a potential indicator for the early-onset post-stroke depression. J Psychosom Res. 2017;96:35–41.\nLiu Z, Zhu Z, Zhao J, Ren W, Cai Y, Wang Q, et al. 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