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Journal of Medicine and Pharmacy","Tạp chí Y Dược học Cần Thơ",{"EN":487,"VI":488},"\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">04\u002F10\u002F2015 Ministry of Information and Communications allowed Can Tho journal of medicine and pharmacy to operate (102 \u002FGP-BTTTT)\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">07\u002F16\u002F2015 Can Tho journal of medicine and pharmacy is internationally recognized: ISSN 2354-1210\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">In 2016, The journal has been included in the list of medical science journals by The State Council for professorship which is awarded a work score of 0-0.5 points for a published article.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Can Tho Journal of Medicine and Pharmacy welcome original works that haven’t been submitted or published in other medical journals. Posts must contain content related to one of the journal’s categories.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The content published\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The journal is divided into 3 categories:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Scientific research article: are valuable scientific works, which have been researched and accepted.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Overview of medicine, biology and pharmacy: serving the objective of continuing training in the fields of medicine, biology and pharmacy; to systematize classical and modern knowledge.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Update information on new knowledge about medicine, biology, pharmacy in the country and in the world.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Scope\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Publication and introduction of scientific research in the fields:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Medicine (internal medicine, surgery, pediatrics, obstetrics and gynecology, odonto-stomatology, laboratory, oncology, traditional medicine, nursing).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Biology (genetics, biotechnology).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">+ Pharmacology (pharmaceutics, drug quality analysis-control, synthetic pharmaceutical chemistry, biochemistry, pharmacognosy, botany, clinical pharmacy).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- To enhance the quality of undergraduate, postgraduate education, scientifically researching and meet the necessary treatment in hospital.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Introducing the updated domestic and oversea information about science technology to promote scientific research and exchanging technology in local, other universities.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">- Exchanging pharmaceutical and medical information for social health developing in the Mekong Delta and Vietnam.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">The object\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Postgraduate students, student of Can Tho University of Medicine and Pharmacy, scientists from schools, research institutes, hospitals, health centers, pharmaceutical companies of the Mekong Delta; other provinces and regions in Vietnam and other country.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Address\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Headquarters of Can Tho Journal of Medicine and Pharmacy, located Scientific Research and International Cooperation Office: 179 Nguyen Van Cu Street, An Khanh Ward, Ninh Kieu District, Can Tho City, Vietnam.\u003C\u002Fspan>\u003C\u002Fp>","\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Ngày 16\u002F7\u002F2015, Tạp chí Y Dược học Cần Thơ được cấp chỉ số quốc tế: ISSN 2354-1210.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 4\u002F2016, Tạp chí đã được Hội đồng Giáo sư ngành Y đưa vào danh sách các tạp chí khoa học Y học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Năm 2020 Tạp chí Y Dược học Cần Thơ đã được phê duyệt vào danh mục của các Hội đồng Giáo sư ngành Dược học được tính điểm công trình 0-0,5 điểm cho một bài báo đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ ra 12 số\u002Fnăm, 180-200 trang\u002Fsố.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Từ tháng 12\u002F2022 Tạp chí Y Dược học Cần Thơ là thành viên của hệ thống Crossref và từ tháng 01\u002F2023 tạp chí thực hiện bình duyệt online kín 2 chiều nhằm tăng tính minh bạch, tin cậy của các công trình nghiên cứu khoa học và đảm bảo tốt nhất chất lượng khoa học của bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ, mục đích và phạm vi của tạp chí\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tôn chỉ và mục đích hoạt động của tạp chí: xuất bản nhằm mục đích phổ biến kết quả từ các đề tài nghiên cứu khoa học; giao lưu trao đổi khoa học, chia sẻ kinh nghiệm, học tập, đồng thời cập nhật thông tin khoa học mới trong các lĩnh vực y, sinh, dược học trong và ngoài nước.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phạm vi của tạp chí: Tạp chí xuất bản được chia thành 3 chuyên mục: (i) Bài báo nghiên cứu khoa học là kết quả công trình nghiên cứu khoa học có giá trị đã được triển khai nghiên cứu, (ii) Bài tổng quan y, sinh, dược học: phục vụ mục tiêu đào tạo liên tục trong lĩnh vực y, sinh, dược học; nhằm hệ thống hóa những kiến thức kinh điển và hiện đại; (iii) Thông tin cập nhật kiến thức mới về y, sinh, dược học trong nước và trên thế giới.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Chính sách truy cập mở\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ áp dụng chính sách truy cập mở đối với các bài báo đã xuất bản đến với độc giả, nhằm mở rộng cơ hội tiếp cận các kết quả nghiên cứu chất lượng cao và tăng cường trao đổi kiến thức. Tạp chí đăng tải trực tuyến (miễn phí) toàn văn các bài báo được công bố trên website của Tạp chí (https:\u002F\u002Ftapchi.ctump.edu.vn).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đạo đức xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ cam kết tuân thủ đạo đức xuất bản phù hợp với các hướng dẫn và tiêu chuẩn của the Committee on Publication Ethics (COPE), tuân thủ các nguyên tắc của COPE’s Core Practices, Best Practices Guidelines for Journal Editors và Guidelines on Good Publication Practices.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Bản thảo bài báo chỉ được chấp nhận khi được tác giả chịu trách nhiệm chính cam kết các nội dung sau: Các nội dung của bản thảo chưa được đăng tải toàn bộ hoặc một phần ở các tạp chí khác; Tất cả các tác giả đều có đóng góp một cách đáng kể vào quá trình nghiên cứu hoặc chuẩn bị bản thảo và cùng chịu trách nhiệm về các nội dung của bản thảo; Tuân thủ các biện pháp đảm bảo đạo đức nghiên cứu (ví dụ thỏa thuận đồng ý tham gia nghiên cứu).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Cam kết bảo mật\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí cam kết thực hiện và tuân thủ các quy định của luật và các văn bản hướng dẫn liên quan đến bảo mật thông tin cá nhân trên không gian mạng. Các thông tin mà người dùng (tác giả, độc giả, biên tập viên, người phản biện) nhập vào các biểu mẫu trên Hệ thống Quản lý xuất bản trực tuyến của tạp chí chỉ được sử dụng vào các mục đích đã được tuyên bố rõ ràng và sẽ không được cung cấp cho bất kỳ bên thứ ba nào khác, hay dùng vào bất kỳ mục đích nào khác.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Phí gửi bài\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng bài: 1.000.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Lệ phí gửi đăng nhanh: 1.500.000đ\u002Fbài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với tác giả là cán bộ viên chức thuộc Trường Đại học Y Dược Cần Thơ thì được hỗ trợ 50% lệ phí gửi đăng bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Đối với sinh viên thực hiện đề tài nghiên cứu khoa học cấp trường được hỗ trợ 100% lệ phí đăng bài ( Tác giả gửi đính kèm “ Quyết định về việc giao tổ chức thực hiện đề tài nghiên cứu khoa học cấp Trường của sinh viên”).\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Hình thức nộp lệ phí:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Tiền mặt:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Nộp trực tiếp tại Phòng Tài chính - Kế toán, Trường Đại học Y Dược Cần Thơ, số 179 Nguyễn Văn Cừ, P. An Khánh, Q. Ninh Kiều, thành phố Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Chuyển khoản:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tên Tài khoản: Trường ĐHYD Cần Thơ, Số TK: 0111000115668, tại ngân hàng Vietcombank chi nhánh Cần Thơ.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Thời gian: Áp dụng từ ngày 01\u002F02\u002F2023.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">* Phí gửi bài không được hoàn trả khi bài viết bị từ chối hoặc tác giả xin rút bài viết.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Quy trình phản biện bài báo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tạp chí Y Dược học Cần Thơ thực hiện quy trình phản biện kín hai chiều nghiêm ngặt. Danh tính của những người phản biện không được tiết lộ cho các tác giả và ngược lại. Quy trình thẩm định bài báo đăng gồm các bước sau:\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tiếp nhận bản thảo\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Tác giả liên hệ gửi bản thảo đến Tạp chí qua hệ thống trực tuyến tại website: https:\u002F\u002Ftapchi.ctump.edu.vn. Hướng dẫn về cách đăng ký, gửi bài và chuẩn bị bản thảo được cung cấp trên website của Tạp chí.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sàng lọc sơ bộ\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Sau khi Tòa soạn nhận được bài báo của tác giả, Ban Thư ký sẽ tiến hành kiểm tra sơ bộ bài báo (các yêu cầu về nội dung và hình thức). Những bài báo không đúng quy cách hoặc có nội dung không phù hợp hoặc vi phạm bản quyền sẽ bị từ chối (Ban Thư ký thông báo phản hồi đến tác giả trong vòng 1 tuần). Những bài báo đủ điều kiện, được Ban Thư ký tòa soạn chuyển đến Ban Biên tập có cùng chuyên môn với nội dung bài báo để đề xuất người phản biện. Thời gian kể từ khi Ban Biên tập nhận bài báo đến khi đề xuất người phản biện bài báo chậm nhất là 5 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Vòng phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký gửi bài và yêu cầu phản biện đến 02 phản biện độc lập.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Các phản biện gởi nhận xét cho Ban Thư ký. Thời gian từ khi gửi bài cho phản biện đến khi nhận ý kiến của phản biện tối đa là 20 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xử ký kết quả phản biện\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Nếu ý kiến đồng ý cho đăng và không cần chỉnh sửa, Ban Thư ký tiếp tục đăng bài theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Nếu ý kiến đồng ý đăng và cần chỉnh sửa, Ban Thư ký sẽ thông tin đến tác giả chỉnh sửa theo yêu cầu của người phản biện. Thời gian chỉnh sửa và gửi lại kéo dài không quá 2 tuần, từ khi tác giả bài báo nhận được thông tin (Quá trình này có thể lặp lại tối đa 2 lần\u002F1 bài báo). Khi có sự thống nhất, đồng ý của người phản biện; bài báo được tiếp tục đăng theo qui trình.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Những bài báo có chất lượng không đạt yêu cầu, cả 2 phản biện không đồng ý cho đăng sẽ bị Tòa soạn từ chối đăng.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">Xuất bản\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">1. Ban Thư ký tổng hợp các bản thảo đã được tác giả hoàn thiện sau thẩm định trình Ban Biên tập xem xét, Tổng Biên tập phê duyệt, quyết định bài đăng theo các tiêu chí: sự phù hợp nội dung với tôn chỉ và mục đích, thể loại bài viết (ưu tiên các bài có bài có nghiên cứu chuyên sâu, hàm lượng khoa học cao), đóng góp mới bài báo, bài báo được ưu tiên đăng trong số gần nhất của Tạp chí theo thứ tự: tính thời sự, chất lượng bài báo và thời gian gửi bài.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">2. Ban Biên tập và Ban Thư ký biên tập bản thảo, chế bản, đọc rà soát lỗi. Thời gian hoàn thành từ 10-15 ngày.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">3. Ban Thư ký có trách nhiệm thông báo cho tác giả bài báo (bằng e-mail) về tình hình phê duyệt bài báo, thời gian, số kỳ, tập xuất bản bài báo theo qui định.\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>\u003Cp>\u003Cspan style=\"color: rgb(0, 0, 0);\">4. Danh sách bài báo theo số Tạp chí được in ấn và phát hành trong năm định kỳ được công bố chính thức trên website: https:\u002F\u002Ftapchi.ctump.edu.vn\u003C\u002Fspan>\u003C\u002Fp>\u003Cp>\u003Cbr>\u003C\u002Fp>",{"VOID":490},"wcQ1uqwAAAAJ","2023-05-30T08:17:21.868+00:00",[],[494],{"id":495,"createTime":28,"updateTime":28,"relativeEntities":496,"slug":28,"properties":497,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":507,"parentIds":508,"statistic":28},"6413896b-eca9-442b-a73f-182a58a0ce40",[],{"title":498,"address":501,"country":504,"abbreviation":505},{"EN":499,"VI":500},"Can Tho University of Medicine and Pharmacy","Trường Đại học Y Dược Cần Thơ",{"EN":502,"VI":503},"No 179, Nguyen Van Cu street, An Khanh ward, Ninh Kieu district, Can Tho city, Vietnam","Số 179, đường Nguyễn Văn Cừ, phường An Khánh, quận Ninh Kiều, thành phố Cần Thơ, Việt Nam",{"VOID":15},{"VOID":506},"ctump","http:\u002F\u002Fwww.ctump.edu.vn\u002F",[],[],"https:\u002F\u002Ftapchi.ctump.edu.vn\u002Findex.php\u002Fctump",{"impactFactor":32,"impactFactorByYear":512,"i10Index":32,"i10IndexLast5Year":32,"totalPublication":514,"totalPublicationByYear":515,"totalCitation":520,"totalCitationByYear":521,"totalCitationPerPublication":108,"totalCitationPerPublicationByYear":523,"hindexLast5Year":45,"hindex":45},{"2022":513,"2023":111,"2024":106},0.01,1556,{"2020":47,"2021":516,"2022":517,"2023":518,"2024":519,"2025":122},57,306,801,358,161,{"2021":146,"2022":280,"2023":522},99,{"2021":524,"2022":318,"2023":104},0.23,{"impactFactor":28,"impactFactorByYear":28,"i10Index":123,"i10IndexLast5Year":123,"totalPublication":526,"totalPublicationByYear":527,"totalCitation":526,"totalCitationByYear":528,"totalCitationPerPublication":40,"totalCitationPerPublicationByYear":531,"hindexLast5Year":49,"hindex":49},476,{"0":205,"2019":123,"2021":139,"2022":459,"2023":451,"2024":357,"2025":49,"2026":48},{"2021":42,"2022":123,"2023":161,"2024":529,"2025":360,"2026":530},136,83,{"2021":105,"2022":513,"2023":532,"2024":127,"2025":533,"2026":534},0.62,25.43,13.83,{"id":536,"createTime":537,"updateTime":382,"relativeEntities":538,"slug":539,"properties":540,"entityType":25,"verifyStatus":26,"verifyTime":28,"verifyNote":28,"languages":552,"translateLanguages":28,"viewCount":133,"subjectFields":553,"manageAffiliations":554,"indexDatabases":555,"url":556,"thumbnailPath":557,"statistic":558,"gsStatistic":594,"type":55,"analyzePriority":28},"6984a56a-db70-403b-9cc4-4013e1ceaffa","2023-05-09T06:47:40.346+00:00",[],"T%E1%BA%A1p%20ch%C3%AD%20Nghi%C3%AAn%20c%E1%BB%A9u%20n%C6%B0%E1%BB%9Bc%20ngo%C3%A0i",{"country":541,"issn":542,"title":544,"introduce":547,"gsId":550},{"VOID":15},{"VOID":543},"25252445",{"EN":545,"VI":546},"VNU Journal of Foreign Studies","Tạp chí Nghiên cứu nước ngoài",{"EN":548,"VI":549},"{\"ops\":[{\"insert\":\"\\n\\nThe \\n\"},{\"attributes\":{\"italic\":true},\"insert\":\"VNU Journal of Science\"},{\"insert\":\"\\n was established in 1985 for the publication of national and international research papers in all fields of natural sciences and technology, social sciences and humanities. 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▪ Abstract  Flavonoids comprise the most common group of plant polyphenols and provide much of the flavor and color to fruits and vegetables. More than 5000 different flavonoids have been described. The six major subclasses of flavonoids include the flavones (e.g., apigenin, luteolin), flavonols (e.g., quercetin, myricetin), flavanones (e.g., naringenin, hesperidin), catechins or flavanols (e.g., epicatechin, gallocatechin), anthocyanidins (e.g., cyanidin, pelargonidin), and isoflavones (e.g., genistein, daidzein). Most of the flavonoids present in plants are attached to sugars (glycosides), although occasionally they are found as aglycones. Interest in the possible health benefits of flavonoids has increased owing to their potent antioxidant and free-radical scavenging activities observed in vitro. There is growing evidence from human feeding studies that the absorption and bioavailability of specific flavonoids is much higher than originally believed. However, epidemiologic studies exploring the role of flavonoids in human health have been inconclusive. Some studies support a protective effect of flavonoid consumption in cardiovascular disease and cancer, other studies demonstrate no effect, and a few studies suggest potential harm. Because there are many biological activities attributed to the flavonoids, some of which could be beneficial or detrimental depending on specific circumstances, further studies in both the laboratory and with populations are warranted. \u003C\u002Fjats:p>",{"EN":961},"D\u003Cscp>IETARY\u003C\u002Fscp> F\u003Cscp>LAVONOIDS\u003C\u002Fscp>: Bioavailability, Metabolic Effects, and Safety",{"VOID":963},"12055336",{"VOID":965},"10.1146\u002Fannurev.nutr.22.111401.144957","PUBLICATION","Auto Verify",[31],"https:\u002F\u002Fwww.annualreviews.org\u002Fdoi\u002F10.1146\u002Fannurev.nutr.22.111401.144957",[971,1004],{"id":972,"sortIndex":32,"researcher":28,"roles":973,"affiliations":974,"properties":999,"displayName":1001,"givenName":28,"familyName":28},"b2628bee-4faf-4df3-8ead-439d8522758f",[],[975,983,991],{"id":976,"sortIndex":32,"affiliation":977,"properties":28},"e2dec727-1bfa-4aad-99f0-b3a00a286cd5",{"id":976,"createTime":28,"updateTime":28,"relativeEntities":978,"slug":28,"properties":979,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":982,"statistic":28},[],{"title":980},{"VI":981},"Department of Pediatrics, University of Minnesota Medical School, Minneapolis, Minnesota 55455, USA",[],{"id":984,"sortIndex":40,"affiliation":985,"properties":28},"79c25dcc-9386-4553-a511-441e4f281e1a",{"id":984,"createTime":28,"updateTime":28,"relativeEntities":986,"slug":28,"properties":987,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":990,"statistic":28},[],{"title":988},{"EN":989},"Department of Pediatrics, University of Minnesota Cancer Center, Minneapolis, Minnesota 55455;",[],{"id":992,"sortIndex":123,"affiliation":993,"properties":28},"470ec327-469d-4320-a02a-dcbfec0d8d55",{"id":992,"createTime":28,"updateTime":28,"relativeEntities":994,"slug":28,"properties":995,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":998,"statistic":28},[],{"title":996},{"EN":997},"Division of Epidemiology, University of Minnesota School of Public Health, Minneapolis, Minnesota 55455;",[],{"title":1000,"openalex":1002},{"EN":1001},"Julie A. 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Analysis of flavonoids in fruits, vegetables and beverages by HPCL-UV and LC-MS and estimation of the total daily flavonoid intake in Denmark. InPolyphenols in Food, ed. R Amado, H Andersson, S Bardocz, F Serra, pp. 39–40. Luxembourg: Off. Official Publications Eur. Commun.",{},{"id":28,"text":1230,"url":28,"identifiers":1231},"10.1016\u002FS0891-5849(98)00190-7",{"doi":1230},{"id":28,"text":1233,"url":28,"identifiers":1234},"Makela S, Poutanen M, Kostian ML, Lehtimaki N, Salo L, et al. 1995. Inhibition of 17B-hydroxysteroid oxidoreductase by flavonoids in breast and prostate cancer cells. 3rd Int. Conf. Phytoestrogens. Dec. 3–6, Little Rock, AK",{},{"id":28,"text":1236,"url":28,"identifiers":1237},"Mao TK, 1999, Int. J. 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Invest., 46, 159",{},{"id":28,"text":1287,"url":28,"identifiers":1288},"Wenzel U, 2000, Cancer Res., 60, 3823",{},{"id":28,"text":1290,"url":28,"identifiers":1291},"10.1093\u002Fjn\u002F125.9.2307",{"doi":1290},{"id":28,"text":1293,"url":28,"identifiers":1294},"10.1093\u002Fjn\u002F124.6.825",{"doi":1293},{"id":28,"text":1296,"url":28,"identifiers":1297},"10.1093\u002Foxfordjournals.aje.a009738",{"doi":1296},false,{"id":1300,"createTime":1301,"updateTime":1301,"relativeEntities":1302,"slug":1303,"properties":1304,"entityType":966,"verifyStatus":26,"verifyTime":1301,"verifyNote":967,"languages":1317,"translateLanguages":28,"viewCount":32,"primaryUrl":1318,"fullTextUrl":28,"authors":1319,"publicationType":1033,"publisherRelationship":1337,"citationCount":1386,"citationInfo":1387,"publishDate":1391,"publishYear":1388,"citationAnalyzeStatus":884,"lastCitationAnalyze":28,"indexDatabases":1392,"openAccess":28,"references":1393,"isForceReanalyzing":1298},"3d2e008c-8c3e-4ccc-8667-25f12fc37c8b","2024-10-08T15:22:25.190+00:00",[],"Fuel-Metabolism-in-Starvation",{"openalex":1305,"mag":1307,"abstract":1309,"title":1311,"pm":1313,"doi":1315},{"VOID":1306},"W2168854431",{"VOID":1308},"2168854431",{"EN":1310},"\u003Cjats:p>▪ Abstract  This article, which is partly biographical and partly scientific, summarizes a life in academic medicine. It relates my progress from benchside to bedside and then to academic and research administration, and concludes with the teaching of human biology to college undergraduates. My experience as an intern (anno 1953) treating a youngster in diabetic ketoacidosis underscored our ignorance of the controls in human fuel metabolism. Circulating free fatty acids were then unknown, insulin could not be measured in biologic fluids, and β-hydroxybutyric acid, which was difficult to measure, was considered by many a metabolic poison. The central role of insulin and the metabolism of free fatty acids, glycerol, glucose, lactate, and pyruvate, combined with indirect calorimetry, needed characterization in a near-steady state, namely prolonged starvation. This is the main topic of this chapter. Due to its use by brain, D-β-hydroxybutyric acid not only has permitted man to survive prolonged starvation, but also may have therapeutic potential owing to its greater efficiency in providing cellular energy in ischemic states such as stroke, myocardial insufficiency, neonatal stress, genetic mitochondrial problems, and physical fatigue.\u003C\u002Fjats:p>",{"EN":1312},"Fuel Metabolism in Starvation",{"VOID":1314},"16848698",{"VOID":1316},"10.1146\u002Fannurev.nutr.26.061505.111258",[31],"https:\u002F\u002Fwww.annualreviews.org\u002Fdoi\u002F10.1146\u002Fannurev.nutr.26.061505.111258",[1320],{"id":1321,"sortIndex":32,"researcher":28,"roles":1322,"affiliations":1323,"properties":1332,"displayName":1334,"givenName":28,"familyName":28},"c66bdddf-41cb-4250-a0da-4d8ff6d55b53",[],[1324],{"id":1325,"sortIndex":32,"affiliation":1326,"properties":28},"7f48b94a-895e-43c2-b40f-b7f9e57cf5ab",{"id":1325,"createTime":28,"updateTime":28,"relativeEntities":1327,"slug":28,"properties":1328,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1331,"statistic":28},[],{"title":1329},{"VI":1330},"Department of Medicine, Harvard Medical School, Boston, Massachusetts 02115.",[],{"title":1333,"openalex":1335},{"EN":1334},"George F. Cahill",{"VOID":1336},"A5017780499",{"url":28,"publisher":1338,"properties":1380},{"id":868,"createTime":869,"updateTime":870,"relativeEntities":1339,"slug":872,"properties":1340,"entityType":25,"verifyStatus":884,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":32,"subjectFields":1345,"manageAffiliations":1354,"indexDatabases":1365,"url":943,"thumbnailPath":28,"statistic":28,"gsStatistic":28,"type":28,"analyzePriority":28},[],{"country":1341,"eissn":1342,"issn":1343,"title":1344},{"VOID":875},{"VOID":877},{"VOID":879},{"EN":881},[1346,1350],{"id":887,"createTime":28,"updateTime":28,"relativeEntities":1347,"label":1348,"description":1349,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":890},{},{"id":893,"createTime":28,"updateTime":28,"relativeEntities":1351,"label":1352,"description":1353,"parentId":28,"standard":28,"scholarHubFieldId":28},[],{"EN":896},{},[1355,1360],{"id":900,"createTime":28,"updateTime":28,"relativeEntities":1356,"slug":28,"properties":1357,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1359,"statistic":28},[],{"title":1358},{"EN":904},[],{"id":907,"createTime":28,"updateTime":28,"relativeEntities":1361,"slug":28,"properties":1362,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":1364,"statistic":28},[],{"title":1363},{"EN":911},[],[1366,1373],{"id":915,"indexDatabase":1367,"url":921,"indexYears":922,"academicFieldIds":1372,"indexDatabaseRanking":926},{"id":786,"createTime":28,"updateTime":28,"relativeEntities":1368,"label":1369,"description":1370,"key":792,"publicationTags":1371,"standard":28},[],{"EN":789,"VI":789},{"EN":789,"VI":791},[794],[924,925],{"id":928,"indexDatabase":1374,"url":940,"indexYears":28,"academicFieldIds":1379,"indexDatabaseRanking":28},{"id":930,"createTime":28,"updateTime":28,"relativeEntities":1375,"label":1376,"description":1377,"key":937,"publicationTags":1378,"standard":28},[],{"EN":933,"VI":933},{"EN":935,"VI":936},[939,813],[942],{"issue":1381,"pages":1382,"volume":1384},{"VOID":1079},{"VOID":1383},"1-22",{"VOID":1385},"26",1114,{"total":1386,"publishYear":1388,"statisticByYear":1389},2006,{"2012":434,"2013":201,"2014":200,"2015":50,"2016":156,"2017":325,"2018":560,"2019":209,"2020":1089,"2021":210,"2022":1390,"2023":212,"2024":332},111,"2006-08-01",[939,926],[1394,1397,1400,1404,1408,1411,1414,1417,1420,1423,1426,1429,1432,1435,1438,1441,1444,1447,1450,1454,1457,1460,1463,1466,1469,1472,1475,1478,1481,1484,1487,1490,1494,1497,1500,1504,1507,1510,1513,1516,1519,1522,1525,1528,1531,1534,1537,1540,1543,1546,1549,1552,1555,1558,1561,1564,1568,1571,1574,1577,1580,1583,1586,1589,1592,1595,1598,1601,1604,1607,1610,1613,1616,1619,1622,1625,1628,1631,1634,1637,1640,1643],{"id":28,"text":1395,"url":28,"identifiers":1396},"10.1089\u002F152091501750220073",{"doi":1395},{"id":28,"text":1398,"url":28,"identifiers":1399},"10.1093\u002Fajcn\u002F28.5.507",{"doi":1398},{"id":28,"text":1401,"url":28,"identifiers":1402},"Ashmore J, 1957, J. Biol. Chem., 224, 225, 10.1016\u002FS0021-9258(18)65024-0",{"doi":1403},"10.1016\u002FS0021-9258(18)65024-0",{"id":28,"text":1405,"url":28,"identifiers":1406},"Benedict FG. 1915. A study of prolonged fasting.Carnegie Inst. Wash.Publ. 203",{"doi":1407},"10.5962\u002Ft.174877",{"id":28,"text":1409,"url":28,"identifiers":1410},"10.1172\u002FJCI112299",{"doi":1409},{"id":28,"text":1412,"url":28,"identifiers":1413},"10.1056\u002FNEJM197003052821026",{"doi":1412},{"id":28,"text":1415,"url":28,"identifiers":1416},"Cahill GF, 2005, Pharos, 68, 8",{},{"id":28,"text":1418,"url":28,"identifiers":1419},"Cahill GF, 1972, Handbook of Physiology, Section 7: Endocrinology, 563",{},{"id":28,"text":1421,"url":28,"identifiers":1422},"10.1152\u002Fajplegacy.1958.192.3.491",{"doi":1421},{"id":28,"text":1424,"url":28,"identifiers":1425},"10.1016\u002F0002-9343(59)90316-X",{"doi":1424},{"id":28,"text":1427,"url":28,"identifiers":1428},"10.1172\u002FJCI105481",{"doi":1427},{"id":28,"text":1430,"url":28,"identifiers":1431},"Cahill GF, 2003, Trans. Am. Clin. Climatol. 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Physiol., 243, E251",{},{"id":28,"text":1482,"url":28,"identifiers":1483},"10.1002\u002Fbmb.2005.49403304243",{"doi":1482},{"id":28,"text":1485,"url":28,"identifiers":1486},"10.1016\u002F0026-0495(82)90024-5",{"doi":1485},{"id":28,"text":1488,"url":28,"identifiers":1489},"10.1210\u002Fjcem-38-4-521",{"doi":1488},{"id":28,"text":1491,"url":28,"identifiers":1492},"Heinbecker P, 1928, J. Biol. Chem., 80, 461, 10.1016\u002FS0021-9258(18)83867-4",{"doi":1493},"10.1016\u002FS0021-9258(18)83867-4",{"id":28,"text":1495,"url":28,"identifiers":1496},"10.1152\u002Fajplegacy.1969.217.3.784",{"doi":1495},{"id":28,"text":1498,"url":28,"identifiers":1499},"10.1172\u002FJCI105610",{"doi":1498},{"id":28,"text":1501,"url":28,"identifiers":1502},"Kashiwaya Y, 1994, J. Biol. Chem., 269, 25502, 10.1016\u002FS0021-9258(18)47278-X",{"doi":1503},"10.1016\u002FS0021-9258(18)47278-X",{"id":28,"text":1505,"url":28,"identifiers":1506},"10.1073\u002Fpnas.97.10.5440",{"doi":1505},{"id":28,"text":1508,"url":28,"identifiers":1509},"10.1161\u002F01.STR.11.5.506",{"doi":1508},{"id":28,"text":1511,"url":28,"identifiers":1512},"10.1128\u002Fjb.177.3.491-496.1995",{"doi":1511},{"id":28,"text":1514,"url":28,"identifiers":1515},"10.1146\u002Fannurev.nutr.20.1.339",{"doi":1514},{"id":28,"text":1517,"url":28,"identifiers":1518},"10.1002\u002F(SICI)1520-7560(199911\u002F12)15:6\u003C412::AID-DMRR72>3.0.CO;2-8",{"doi":1517},{"id":28,"text":1520,"url":28,"identifiers":1521},"Lardy HA, 1945, Arch. Biochem., 6, 53",{},{"id":28,"text":1523,"url":28,"identifiers":1524},"10.3181\u002F00379727-102-25306",{"doi":1523},{"id":28,"text":1526,"url":28,"identifiers":1527},"10.1136\u002Fbmj.317.7171.1481",{"doi":1526},{"id":28,"text":1529,"url":28,"identifiers":1530},"10.1172\u002FJCI106552",{"doi":1529},{"id":28,"text":1532,"url":28,"identifiers":1533},"McKinlay WL. 1976.The Last Voyage of the Karluk. A Survivor's Memoir of Arctic Disaster. New York: Martin's Press. 168pp.",{},{"id":28,"text":1535,"url":28,"identifiers":1536},"10.1093\u002Fajcn\u002F54.1.81",{"doi":1535},{"id":28,"text":1538,"url":28,"identifiers":1539},"10.1016\u002FS1474-4422(02)00043-1",{"doi":1538},{"id":28,"text":1541,"url":28,"identifiers":1542},"10.1002\u002Fbmb.2005.49403304246",{"doi":1541},{"id":28,"text":1544,"url":28,"identifiers":1545},"10.1172\u002FJCI106016",{"doi":1544},{"id":28,"text":1547,"url":28,"identifiers":1548},"10.1172\u002FJCI105650",{"doi":1547},{"id":28,"text":1550,"url":28,"identifiers":1551},"10.1093\u002Fajcn\u002F68.1.12",{"doi":1550},{"id":28,"text":1553,"url":28,"identifiers":1554},"10.1172\u002FJCI106940",{"doi":1553},{"id":28,"text":1556,"url":28,"identifiers":1557},"Pozefsky T, 1968, Assoc. Am. Physicians, 81, 258",{},{"id":28,"text":1559,"url":28,"identifiers":1560},"10.1042\u002Fbst0260125",{"doi":1559},{"id":28,"text":1562,"url":28,"identifiers":1563},"10.1172\u002FJCI107584",{"doi":1562},{"id":28,"text":1565,"url":28,"identifiers":1566},"Renold AE, Cahill GF Jr, eds. 1965.Handbook of Physiology, Section 5: Adipose Tissue.Washington, DC: Am. Physiol. Soc. 824pp.",{"doi":1567},"10.1097\u002F00000441-196611000-00016",{"id":28,"text":1569,"url":28,"identifiers":1570},"10.1007\u002F978-3-642-58444-2_8",{"doi":1569},{"id":28,"text":1572,"url":28,"identifiers":1573},"10.1007\u002Fs00592-003-0037-7",{"doi":1572},{"id":28,"text":1575,"url":28,"identifiers":1576},"10.1152\u002Fphysrev.1980.60.1.143",{"doi":1575},{"id":28,"text":1578,"url":28,"identifiers":1579},"10.1002\u002Fajpa.1330350605",{"doi":1578},{"id":28,"text":1581,"url":28,"identifiers":1582},"10.1177\u002F08830738050200010501",{"doi":1581},{"id":28,"text":1584,"url":28,"identifiers":1585},"Ruderman NB, 1976, Gluconeogenesis: Its Regulation in Mammalian Species, 515",{},{"id":28,"text":1587,"url":28,"identifiers":1588},"10.1042\u002Fbj1380001",{"doi":1587},{"id":28,"text":1590,"url":28,"identifiers":1591},"10.1172\u002FJCI107410",{"doi":1590},{"id":28,"text":1593,"url":28,"identifiers":1594},"10.1172\u002FJCI107016",{"doi":1593},{"id":28,"text":1596,"url":28,"identifiers":1597},"10.1096\u002Ffasebj.9.8.7768357",{"doi":1596},{"id":28,"text":1599,"url":28,"identifiers":1600},"10.1016\u002FS0022-3476(81)80583-5",{"doi":1599},{"id":28,"text":1602,"url":28,"identifiers":1603},"Schneider ED, Sagan D. 2005.Into the Cool: Energy Flow, Thermodynamics and Life.Chicago: Univ. Chicago Press. 362pp.",{},{"id":28,"text":1605,"url":28,"identifiers":1606},"10.2337\u002Fdiab.14.12.771",{"doi":1605},{"id":28,"text":1608,"url":28,"identifiers":1609},"Stadtlander Lu JJ, Morgan-Boyd RL, Nelson RA, Peper RL. 2000. Serum chemistries in black bears in active and denning sites.FASEB J.14(4), Abstract 451.5",{},{"id":28,"text":1611,"url":28,"identifiers":1612},"10.1126\u002Fscience.1070534",{"doi":1611},{"id":28,"text":1614,"url":28,"identifiers":1615},"Suzuki M, 2001, J. Pharmacol., 87, 143",{},{"id":28,"text":1617,"url":28,"identifiers":1618},"10.1074\u002Fjbc.C500213200",{"doi":1617},{"id":28,"text":1620,"url":28,"identifiers":1621},"Tilney NL. 2003.Transplant, from Myth to Reality.New York: Donnelley. 320pp.",{},{"id":28,"text":1623,"url":28,"identifiers":1624},"10.1016\u002FS0140-6736(03)13105-4",{"doi":1623},{"id":28,"text":1626,"url":28,"identifiers":1627},"10.1212\u002F01.WNL.0000152046.11390.45",{"doi":1626},{"id":28,"text":1629,"url":28,"identifiers":1630},"10.1301\u002Fnr.2003.oct.327-341",{"doi":1629},{"id":28,"text":1632,"url":28,"identifiers":1633},"10.1242\u002Fjeb.01064",{"doi":1632},{"id":28,"text":1635,"url":28,"identifiers":1636},"10.1016\u002Fj.plefa.2003.09.007",{"doi":1635},{"id":28,"text":1638,"url":28,"identifiers":1639},"10.1080\u002F152165401753311780",{"doi":1638},{"id":28,"text":1641,"url":28,"identifiers":1642},"10.1042\u002Fcs0860677",{"doi":1641},{"id":28,"text":1644,"url":28,"identifiers":1645},"10.1042\u002Fbj0820090",{"doi":1644},{"id":1647,"createTime":1648,"updateTime":1649,"relativeEntities":1650,"slug":1651,"properties":1652,"entityType":966,"verifyStatus":26,"verifyTime":1667,"verifyNote":967,"languages":1668,"translateLanguages":1669,"viewCount":32,"primaryUrl":1670,"fullTextUrl":28,"authors":1671,"publicationType":1033,"publisherRelationship":1691,"citationCount":1740,"citationInfo":1741,"publishDate":1744,"publishYear":1742,"citationAnalyzeStatus":884,"lastCitationAnalyze":28,"indexDatabases":1745,"openAccess":28,"references":1746,"isForceReanalyzing":1298},"c5d6fc58-3fb6-4a6d-bda1-3fd7a1d899d4","2024-09-12T14:44:04.984+00:00","2024-12-29T10:37:30.171+00:00",[],"HOMOCYSTEINE-METABOLISM",{"openalex":1653,"abstract":1655,"title":1658,"keywords":1661,"pm":1663,"doi":1665},{"VOID":1654},"W4212986840",{"EN":1656,"VI":1657},"\u003Cjats:p> ▪ Abstract  Homocysteine is a sulfur amino acid whose metabolism stands at the intersection of two pathways: remethylation to methionine, which requires folate and vitamin B\u003Cjats:sub>12\u003C\u002Fjats:sub> (or betaine in an alternative reaction); and transsulfuration to cystathionine, which requires pyridoxal-5′-phosphate. The two pathways are coordinated by S-adenosylmethionine, which acts as an allosteric inhibitor of the methylenetetrahydrofolate reductase reaction and as an activator of cystathionine β-synthase. Hyperhomocysteinemia, a condition that recent epidemiological studies have shown to be associated with increased risk of vascular disease, arises from disrupted homocysteine metabolism. Severe hyperhomocysteinemia is due to rare genetic defects resulting in deficiencies in cystathionine beta synthase, methylenetetrahydrofolate reductase, or in enzymes involved in methyl-B\u003Cjats:sub>12\u003C\u002Fjats:sub> synthesis and homocysteine methylation. Mild hyperhomocysteinemia seen in fasting conditions is due to mild impairment in the methylation pathway (i.e. folate or B\u003Cjats:sub>12\u003C\u002Fjats:sub> deficiencies or methylenetetrahydrofolate reductase thermolability). Post–methionine-load hyperhomocysteinemia may be due to heterozygous cystathionine β-synthase defect or B\u003Cjats:sub>6\u003C\u002Fjats:sub> deficiency. Early studies with nonphysiological high homocysteine levels showed a variety of deleterious effects on endothelial or smooth muscle cells in culture. More recent studies with human beings and animals with mild hyperhomocysteinemia provided encouraging results in the attempt to understand the mechanism that underlies this relationship between mild elevations of plasma homocysteine and vascular disease. The studies with animal models indicated the possibility that the effect of elevated homocysteine is multifactorial, affecting both the vascular wall structure and the blood coagulation system. \u003C\u002Fjats:p>","\u003Cjats:p> ▪ Tóm tắt Homocysteine là một axit amin lưu huỳnh, có chuyển hóa nằm ở giao điểm của hai con đường: tái methyl hóa thành methionine, cần folate và vitamin B\u003Cjats:sub>12\u003C\u002Fjats:sub> (hoặc betaine trong phản ứng thay thế); và transsulfuration thành cystathionine, cần pyridoxal-5′-phosphate. Hai con đường này được điều hòa bởi S-adenosylmethionine, hoạt động như một chất ức chế allosteric của phản ứng methylenetetrahydrofolate reductase và như một hoạt hóa viên của cystathionine β-synthase. Tình trạng tăng homocysteine trong máu, mà các nghiên cứu dịch tễ học gần đây đã chứng minh có liên quan đến nguy cơ cao hơn về bệnh mạch máu, phát sinh từ sự rối loạn chuyển hóa homocysteine. Tăng homocysteine nghiêm trọng là do các khiếm khuyết di truyền hiếm gặp dẫn đến thiếu hụt cystathionine beta synthase, methylenetetrahydrofolate reductase, hoặc các enzyme liên quan đến tổng hợp methyl-B\u003Cjats:sub>12\u003C\u002Fjats:sub> và methyl hóa homocysteine. Tăng homocysteine nhẹ trong các điều kiện nhịn ăn là do sự suy giảm nhẹ trong con đường methyl hóa (có nghĩa là thiếu hụt folate hoặc B\u003Cjats:sub>12\u003C\u002Fjats:sub> hoặc độ bền nhiệt của methylenetetrahydrofolate reductase). Tăng homocysteine sau khi nạp methionine có thể do khiếm khuyết heterozygous cystathionine β-synthase hoặc thiếu hụt B\u003Cjats:sub>6\u003C\u002Fjats:sub>. Các nghiên cứu sớm với mức homocysteine cao phi sinh lý đã cho thấy nhiều tác động có hại đối với tế bào nội mạc hoặc tế bào cơ trơn trong nuôi cấy. Các nghiên cứu gần đây với con người và động vật có tăng homocysteine nhẹ đã cung cấp kết quả khích lệ trong nỗ lực hiểu cơ chế nằm sau mối quan hệ giữa sự gia tăng nhẹ nồng độ homocysteine huyết tương và bệnh mạch máu. Những nghiên cứu với các mô hình động vật chỉ ra khả năng tác động của homocysteine tăng có tính đa yếu tố, ảnh hưởng đến cả cấu trúc thành mạch và hệ thống đông máu.",{"EN":1659,"VI":1660},"HOMOCYSTEINE METABOLISM","CHUYỂN HÓA HOMOCYSTEINE",{"VI":1662},"tăng homocysteine, chuyển hóa homocysteine, bệnh mạch máu, cystathionine, 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Although such environmental factors appear unrelated, they generally influence consumption volume by inhibiting consumption monitoring and by suggesting alternative consumption norms. For researchers, this review suggests that redirecting the focus of investigations to the psychological mechanisms behind consumption will raise the profile and impact of research. For health professionals, this review underscores how small structural changes in personal environments can reduce the unknowing overconsumption of food.\u003C\u002Fjats:p>",{"EN":2213},"ENVIRONMENTAL FACTORS THAT INCREASE THE FOOD INTAKE AND CONSUMPTION VOLUME OF UNKNOWING 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In press",{"doi":2556},"10.1002\u002Feat.20195",{"id":28,"text":2558,"url":28,"identifiers":2559},"10.1146\u002Fannurev.psych.53.100901.135103",{"doi":2558},{"id":28,"text":2404,"url":28,"identifiers":2561},{},{"id":28,"text":2563,"url":28,"identifiers":2564},"10.1037\u002F0022-3514.50.6.1253",{"doi":2563},{"id":28,"text":2566,"url":28,"identifiers":2567},"10.1111\u002Fj.1467-6494.1979.tb00617.x",{"doi":2566},{"id":28,"text":2569,"url":28,"identifiers":2570},"10.1016\u002FS0899-9007(02)00784-0",{"doi":2569},{"id":28,"text":2572,"url":28,"identifiers":2573},"10.2307\u002F3152079",{"doi":2572},{"id":28,"text":2575,"url":28,"identifiers":2576},"10.1111\u002Fj.1471-6712.1996.tb00304.x",{"doi":2575},{"id":28,"text":2578,"url":28,"identifiers":2579},"10.3758\u002FBF03329832",{"doi":2578},{"id":28,"text":2581,"url":28,"identifiers":2582},"Rodin J. 1974. Effects of distraction on the performance of obese and normal subjects. 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Effects of manipulating salience of food upon consumption by obese and normal eaters. See Ref.115a, pp.43–51",{},{"id":28,"text":2623,"url":28,"identifiers":2624},"Roth DA, 2000, Dissert. Abstract Int., 61, 590 (Abstr.)",{},{"id":28,"text":2626,"url":28,"identifiers":2627},"10.3758\u002FBF03202667",{"doi":2626},{"id":28,"text":2629,"url":28,"identifiers":2630},"10.1111\u002F1467-9280.00073",{"doi":2629},{"id":28,"text":2632,"url":28,"identifiers":2633},"10.1111\u002F1467-9280.02452",{"doi":2632},{"id":28,"text":2635,"url":28,"identifiers":2636},"10.1016\u002F0950-3293(93)90309-T",{"doi":2635},{"id":28,"text":2638,"url":28,"identifiers":2639},"Schachter S, 1971, Emotion, Obesity, and Crime",{},{"id":28,"text":2641,"url":28,"identifiers":2642},"10.1037\u002Fh0030817",{"doi":2641},{"id":28,"text":2644,"url":28,"identifiers":2645},"Schachter S, Friedman LN. 1974. The effects of work and cue prominence on eating behavior. 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Bad popcorn in big buckets: portion size can influence intake as much as taste. In press",{"doi":2724},"10.1016\u002FS1499-4046(06)60278-9",{"id":28,"text":2726,"url":28,"identifiers":2727},"10.1038\u002Fsj.ijo.0802291",{"doi":2726},{"id":28,"text":2729,"url":28,"identifiers":2730},"Wansink B, Painter JE, Van Ittersum, K. 2004. Bowl-size, spoon-size, and consumption intake at the ice cream social. In press",{},{"id":28,"text":2732,"url":28,"identifiers":2733},"Wansink B, Painter JE, Lee Y-K. 2004. Proximity's influence on estimated and actual candy consumption. In press",{},{"id":28,"text":2735,"url":28,"identifiers":2736},"Wansink B, Painter JE, North J. 2004. The bottomless bowl: visual cues of portion size influence intake, consumption norms, estimation, and satiation. In press",{"doi":2737},"10.1038\u002Foby.2005.12",{"id":28,"text":2739,"url":28,"identifiers":2740},"10.1016\u002FS0950-3293(00)00031-8",{"doi":2739},{"id":28,"text":2742,"url":28,"identifiers":2743},"10.1086\u002F378621",{"doi":2742},{"id":28,"text":2745,"url":28,"identifiers":2746},"Wansink B, Van Ittersum K. 2004. Illusive consumption behavior and the DelBoeuf illusion: Are the eyes really bigger than the stomach?In press",{},{"id":28,"text":2748,"url":28,"identifiers":2749},"10.1016\u002FS0195-6663(84)80035-5",{"doi":2748},{"id":28,"text":2751,"url":28,"identifiers":2752},"10.1017\u002FS002966519900107X",{"doi":2751},{"id":28,"text":2754,"url":28,"identifiers":2755},"10.1038\u002Foby.2001.130",{"doi":2754},{"id":28,"text":2757,"url":28,"identifiers":2758},"10.1056\u002FNEJM200003233421206",{"doi":2757},{"id":28,"text":2760,"url":28,"identifiers":2761},"10.2105\u002FAJPH.92.2.246",{"doi":2760},{"id":2763,"createTime":2764,"updateTime":2765,"relativeEntities":2766,"slug":2767,"properties":2768,"entityType":966,"verifyStatus":26,"verifyTime":2785,"verifyNote":967,"languages":2786,"translateLanguages":2787,"viewCount":32,"primaryUrl":2788,"fullTextUrl":28,"authors":2789,"publicationType":1033,"publisherRelationship":2828,"citationCount":2877,"citationInfo":2878,"publishDate":2881,"publishYear":2879,"citationAnalyzeStatus":884,"lastCitationAnalyze":28,"indexDatabases":2882,"openAccess":28,"references":2883,"isForceReanalyzing":1298},"eca2bc81-d40b-4f28-885e-c0d78bcad2ab","2024-12-01T10:17:07.909+00:00","2025-01-09T20:33:43.057+00:00",[],"P-scp-EROXISOMAL-scp-%CE%B2-O-scp-XIDATION-AND-scp-P-scp-EROXISOME-scp-P-scp-ROLIFERATOR-scp-A-scp-CTIVATED-scp-R-scp-ECEPTOR-scp-%CE%B1-An-Adaptive-Metabolic-System",{"mag":2769,"keywords":2771,"openalex":2773,"abstract":2775,"title":2778,"pm":2781,"doi":2783},{"VOID":2770},"2104492370",{"VI":2772},"",{"VOID":2774},"W2104492370",{"VI":2776,"EN":2777},"\u003Cjats:p>▪ Tóm tắt  Oxi hóa β xảy ra trong cả ty thể và peroxisome. Ty thể xúc tác quá trình oxi hóa β của phần lớn axit béo chuỗi ngắn, chuỗi vừa và chuỗi dài được hấp thụ từ chế độ ăn uống, và con đường này cấu thành quá trình chính mà qua đó axit béo được oxi hóa để tạo ra năng lượng. Peroxisome tham gia vào quá trình rút ngắn chuỗi oxi hóa β của các axit béo chuỗi dài và chuỗi rất dài như acyl-coenzyme (CoA), diacyl-CoAs, este CoA của eicosanoid, axit béo có nhánh methyl 2, và este CoA của các chất trung gian acid mật di- và trihydroxycoprostanoic, đồng thời trong quá trình này, chúng tạo ra H\u003Cjats:sub>2\u003C\u002Fjats:sub>O\u003Cjats:sub>2\u003C\u002Fjats:sub>. Axit béo chuỗi dài và chuỗi rất dài (VLCFAs) cũng được chuyển hóa bởi hệ thống oxi hóa ω-CYP4A cytochrome P450 thành các axit dicarboxylic, mà sẽ là substrat cho quá trình oxi hóa β của peroxisome. Hệ thống oxi hóa β của peroxisome bao gồm (a) một con đường truyền thống có khả năng xúc tác acyl-CoAs chuỗi thẳng thông qua fatty acyl-CoA oxidase, protein L-bifunctional, và thiolase, và (b) một con đường không thể kích hoạt thứ hai xúc tác quá trình oxi hóa các axit béo acyl-CoA nhánh methyl 2 thông qua branched-chain acyl-CoA oxidase (pristanoyl-CoA oxidase\u002Ftrihydroxycoprostanoyl-CoA oxidase), protein D-bifunctional, và protein vận chuyển sterol (SCP)x. Các gen mã hóa con đường oxi hóa β truyền thống trong gan được điều chỉnh chuyển hóa bởi thụ thể kích hoạt bởi peroxisome proliferator α (PPARα). Bằng chứng từ chuột thiếu PPARα, fatty acyl-CoA oxidase peroxisome và một số enzym khác trong hai con đường oxi hóa β peroxisome chỉ ra tầm quan trọng thiết yếu của PPARα và fatty acyl-CoA oxidase peroxisome truyền thống trong chuyển hóa năng lượng, và trong sự phát triển của gan nhiễm mỡ, viêm gan nhiễm mỡ và ung thư gan.\u003C\u002Fjats:p>","\u003Cjats:p>▪ Abstract  β-Oxidation occurs in both mitochondria and peroxisomes. Mitochondria catalyze the β-oxidation of the bulk of short-, medium-, and long-chain fatty acids derived from diet, and this pathway constitutes the major process by which fatty acids are oxidized to generate energy. Peroxisomes are involved in the β-oxidation chain shortening of long-chain and very-long-chain fatty acyl-coenzyme (CoAs), long-chain dicarboxylyl-CoAs, the CoA esters of eicosanoids, 2-methyl-branched fatty acyl-CoAs, and the CoA esters of the bile acid intermediates di- and trihydroxycoprostanoic acids, and in the process they generate H\u003Cjats:sub>2\u003C\u002Fjats:sub>O\u003Cjats:sub>2\u003C\u002Fjats:sub>. Long-chain and very-long-chain fatty acids (VLCFAs) are also metabolized by the cytochrome P450 CYP4A ω-oxidation system to dicarboxylic acids that serve as substrates for peroxisomal β-oxidation. The peroxisomal β-oxidation system consists of (a) a classical peroxisome proliferator–inducible pathway capable of catalyzing straight-chain acyl-CoAs by fatty acyl-CoA oxidase, L-bifunctional protein, and thiolase, and (b) a second noninducible pathway catalyzing the oxidation of 2-methyl-branched fatty acyl-CoAs by branched-chain acyl-CoA oxidase (pristanoyl-CoA oxidase\u002Ftrihydroxycoprostanoyl-CoA oxidase), D-bifunctional protein, and sterol carrier protein (SCP)x. The genes encoding the classical β-oxidation pathway in liver are transcriptionally regulated by peroxisome proliferator–activated receptor α (PPARα). Evidence derived from mice deficient in PPARα, peroxisomal fatty acyl-CoA oxidase, and some of the other enzymes of the two peroxisomal β-oxidation pathways points to the critical importance of PPARα and of the classical peroxisomal fatty acyl-CoA oxidase in energy metabolism, and in the development of hepatic steatosis, steatohepatitis, and liver cancer.\u003C\u002Fjats:p>",{"EN":2779,"VI":2780},"P\u003Cscp>EROXISOMAL\u003C\u002Fscp>β-O\u003Cscp>XIDATION AND\u003C\u002Fscp>P\u003Cscp>EROXISOME\u003C\u002Fscp>P\u003Cscp>ROLIFERATOR\u003C\u002Fscp>–A\u003Cscp>CTIVATED\u003C\u002Fscp>R\u003Cscp>ECEPTOR\u003C\u002Fscp>α: An Adaptive Metabolic System","Oxi hóa β-Peroxisome và Thụ thể được kích hoạt bởi Peroxisome Proliferator α: Một Hệ thống Thích ứng Chuyển hóa",{"VOID":2782},"11375435",{"VOID":2784},"10.1146\u002Fannurev.nutr.21.1.193","2024-12-01T10:17:07.903+00:00",[31],[30],"https:\u002F\u002Fwww.annualreviews.org\u002Fdoi\u002F10.1146\u002Fannurev.nutr.21.1.193",[2790,2809],{"id":2791,"sortIndex":32,"researcher":28,"roles":2792,"affiliations":2793,"properties":2802,"displayName":2806,"givenName":28,"familyName":28},"6a6b7357-be46-49de-a428-1211c164db51",[],[2794],{"id":2795,"sortIndex":32,"affiliation":2796,"properties":28},"a14a1363-3f25-473e-a16a-f28e1d65dade",{"id":2795,"createTime":28,"updateTime":28,"relativeEntities":2797,"slug":28,"properties":2798,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":2801,"statistic":28},[],{"title":2799},{"EN":2800},"Department of Pathology, Northwestern University Medical School, Chicago, Illinois 60611, USA. jkreddy@northwestern.edu",[],{"orcid":2803,"title":2805,"openalex":2807},{"VOID":2804},"https:\u002F\u002Forcid.org\u002F0000-0002-6452-4933",{"EN":2806},"Janardan K. 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Protein này là một thành viên của họ oxydase đa đồng, một nhóm protein được bảo toàn trong quá trình tiến hóa, sử dụng đồng để kết hợp quá trình oxi hóa cơ chất với việc khử oxy thành nước bằng bốn electron. Mặc dù cần có đồng để ceruloplasmin hoạt động, nhưng protein này không đóng vai trò thiết yếu trong việc vận chuyển hoặc chuyển hóa kim loại này. Aceruloplasminemia là một bệnh thoái hóa thần kinh do sự mất chức năng di truyền của gen ceruloplasmin. Đặc điểm của rối loạn này đã chỉ ra vai trò sinh lý quan trọng của ceruloplasmin trong việc xác định tốc độ thoát sắt từ các tế bào có kho dự trữ sắt có thể mobilizable và đã cung cấp những hiểu biết mới về chuyển hóa và dinh dưỡng sắt ở người.","\u003Cjats:p> ▪ Abstract  Ceruloplasmin is a serum ferroxidase that contains greater than 95% of the copper found in plasma. This protein is a member of the multicopper oxidase family, an evolutionarily conserved group of proteins that utilize copper to couple substrate oxidation with the four-electron reduction of oxygen to water. Despite the need for copper in ceruloplasmin function, this protein plays no essential role in the transport or metabolism of this metal. Aceruloplasminemia is a neurodegenerative disease resulting from inherited loss-of-function mutations in the ceruloplasmin gene. Characterization of this disorder revealed a critical physiological role for ceruloplasmin in determining the rate of iron efflux from cells with mobilizable iron stores and has provided new insights into human iron metabolism and nutrition. \u003C\u002Fjats:p>",{"EN":3848,"VI":3849},"C\u003Cscp>ERULOPLASMIN\u003C\u002Fscp> M\u003Cscp>ETABOLISM AND\u003C\u002Fscp> F\u003Cscp>UNCTION\u003C\u002Fscp>","Chuyển hóa và Chức năng của Ceruloplasmin",{"VOID":3851},"12055353",{"VOID":3853},"10.1146\u002Fannurev.nutr.22.012502.114457","2024-12-11T10:09:33.653+00:00",[31],[30],"https:\u002F\u002Fwww.annualreviews.org\u002Fdoi\u002F10.1146\u002Fannurev.nutr.22.012502.114457",[3859,3876],{"id":3860,"sortIndex":32,"researcher":28,"roles":3861,"affiliations":3862,"properties":3871,"displayName":3873,"givenName":28,"familyName":28},"09f412bf-9306-4232-af0e-048b799be8ab",[],[3863],{"id":3864,"sortIndex":32,"affiliation":3865,"properties":28},"ee0f54df-79a2-45b4-b6a7-cd602715069e",{"id":3864,"createTime":28,"updateTime":28,"relativeEntities":3866,"slug":28,"properties":3867,"entityType":28,"verifyStatus":28,"verifyTime":28,"verifyNote":28,"languages":28,"translateLanguages":28,"viewCount":28,"url":28,"parentIds":3870,"statistic":28},[],{"title":3868},{"EN":3869},"Edward Mallinckrodt Department of Pediatrics, Washington University School of Medicine, St. Louis, Missouri 63110, USA.",[],{"title":3872,"openalex":3874},{"EN":3873},"Nathan E. 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Nó bao gồm một polypeptide chuỗi đơn với hai thùy hình cầu và tương đối kháng lại sự phân hủy bởi protease. Các cDNA hoàn chỉnh cho lactoferrin từ sữa người, bạch cầu trung tính và sữa bò đã được báo cáo, và các protein tái tổ hợp đã được sản xuất. Nhờ vào các đặc tính liên kết với sắt, lactoferrin được đề xuất là có vai trò trong việc hấp thu sắt qua niêm mạc ruột và hoạt động như một tác nhân kìm khuẩn bằng cách giữ lại sắt từ các vi khuẩn cần sắt. Sự hiện diện của nó trong bạch cầu trung tính và việc giải phóng trong quá trình viêm cho thấy rằng lactoferrin cũng liên quan đến việc tiêu diệt thực bào và phản ứng miễn dịch. Thêm vào đó, lactoferrin có thể hoạt động theo những cách không liên quan đến việc liên kết với sắt, ví dụ như một yếu tố tăng trưởng và một tác nhân diệt khuẩn. Bài đánh giá này cố gắng đánh giá các chức năng được đề xuất này và ý nghĩa sinh học của chúng một cách chi tiết hơn. \u003C\u002Fjats:p>","\u003Cjats:p> Lactoferrin is an 80-kDa, iron-binding glycoprotein present in milk and, to a lesser extent, in exocrine fluids such as bile and tears. It consists of a single-chain polypeptide with two gobular lobes and is relatively resistant to proteolysis. The complete cDNAs for lactoferrin from human milk, neutrophils, and bovine milk have been reported, and recombinant proteins have been produced. Owing to its iron-binding properties, lactoferrin has been proposed to play a role in iron uptake by the intestinal mucosa and to act as a bacteriostatic agent by withholding iron from iron-requiring bacteria. Its presence in neutrophils and its release during inflammation suggest that lactoferrin is also involved in phagocytic killing and immune responses. Additionally, lactoferrin may function in ways not related to iron-binding, e.g. as a growth factor and as a bactericidal agent. 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