[{"data":1,"prerenderedAt":-1},["ShallowReactive",2],{"_public_publication_all{\"sortAscending\":false,\"sortField\":\"updateTime\",\"page\":0,\"size\":10,\"facet\":true,\"searchKey\":\"\"}":3},{"meta":4,"data":6},{"total":5},"15168086",[7,386,478,693,941,1021,1132,1255,1369,1530],{"id":8,"createTime":9,"updateTime":10,"relativeEntities":11,"slug":12,"properties":13,"entityType":26,"verifyStatus":27,"verifyTime":28,"verifyNote":29,"syncStatus":30,"languages":31,"translateLanguages":32,"viewCount":34,"primaryUrl":35,"fullTextUrl":31,"authors":36,"publicationType":173,"publisherRelationship":174,"citationCount":31,"citationInfo":31,"publishDate":383,"publishYear":384,"citationAnalyzeStatus":30,"lastCitationAnalyze":31,"indexDatabases":31,"openAccess":31,"references":31,"isForceReanalyzing":385},"7bade3ef-2886-41ff-92d6-ff5f8ae6619d","2024-02-08T07:50:18.340+00:00","2025-01-07T23:59:59.902+00:00",[],"Targeting-ataxia-telangiectasia-mutated-and-Rad3-related-kinase-ATR-in-PTEN-deficient-breast-cancers-for-personalized-therapy",{"references":14,"abstract":16,"title":19,"doi":22,"keywords":24},{"VOID":15},"Abdel-Fatah TM et al (2015) Untangling the ATR-CHEK1 network for prognostication, prediction and therapeutic target validation in breast cancer. Mol Oncol 9:569–585. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.molonc.2014.10.013\nAbdel-Fatah TM et al (2014) DNA polymerase beta deficiency is linked to aggressive breast cancer: a comprehensive analysis of gene copy number, mRNA and protein expression in multiple cohorts. Mol Oncol 8:520–532. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.molonc.2014.01.001\nAlbarakati N et al (2015) Targeting BRCA1-BER deficient breast cancer by ATM or DNA-PKcs blockade either alone or in combination with cisplatin for personalized therapy. Mol Oncol 9:204–217. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.molonc.2014.08.001\nArora A et al (2015) Transcriptomic and protein expression analysis reveals clinicopathological significance of bloom syndrome helicase (BLM) in breast cancer. Mol Cancer Ther 14:1057–1065. https:\u002F\u002Fdoi.org\u002F10.1158\u002F1535-7163.MCT-14-0939\nBassi C et al (2013) Nuclear PTEN controls DNA repair and sensitivity to genotoxic stress. Science 341:395–399. https:\u002F\u002Fdoi.org\u002F10.1126\u002Fscience.1236188\nBose S et al (2002) Reduced expression of PTEN correlates with breast cancer progression. Hum Pathol 33:405–409\nChalhoub N, Baker SJ (2009) PTEN and the PI3-kinase pathway in cancer. Annu Rev Pathol 4:127–150. https:\u002F\u002Fdoi.org\u002F10.1146\u002Fannurev.pathol.4.110807.092311\nDepowski PL et al (2001) Loss of expression of the PTEN gene protein product is associated with poor outcome in breast cancer. Mod Pathol 14:672–676. https:\u002F\u002Fdoi.org\u002F10.1038\u002Fmodpathol.3880371\nFokas E et al (2014) Targeting ATR in DNA damage response and cancer therapeutics. Cancer Treat Rev 40:109–117. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.ctrv.2013.03.002\nKarnitz LM, Zou L (2015) Molecular Pathways: targeting ATR in Cancer Therapy. Clin Cancer Res 21:4780–4785. https:\u002F\u002Fdoi.org\u002F10.1158\u002F1078-0432.CCR-15-0479\nKwok M et al (2016) ATR inhibition induces synthetic lethality and overcomes chemoresistance in TP53- or ATM-defective chronic lymphocytic leukemia cells. Blood 127:582–595. https:\u002F\u002Fdoi.org\u002F10.1182\u002Fblood-2015-05-644872\nMcCabe N et al (2015) Mechanistic rationale to target PTEN-deficient tumor cells with inhibitors of the DNA damage response kinase ATM. Cancer Res 75:2159–2165. https:\u002F\u002Fdoi.org\u002F10.1158\u002F0008-5472.CAN-14-3502\nMcShane LM et al (2005) Reporting recommendations for tumor marker prognostic studies (REMARK). J Natl Cancer Inst 97:1180–1184. https:\u002F\u002Fdoi.org\u002F10.1093\u002Fjnci\u002Fdji237\nMing M, He YY (2012) PTEN in DNA damage repair. Cancer Lett 319:125–129. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.canlet.2012.01.003\nMukherjee A, Karmakar P (2013) Attenuation of PTEN perturbs genomic stability via activation of Akt and down-regulation of Rad51 in human embryonic kidney cells. Mol Carcinog 52:611–618. https:\u002F\u002Fdoi.org\u002F10.1002\u002Fmc.21903\nPerren A et al (1999) Immunohistochemical evidence of loss of PTEN expression in primary ductal adenocarcinomas of the breast. Am J Pathol 155:1253–1260. https:\u002F\u002Fdoi.org\u002F10.1016\u002FS0002-9440(10)65227-3\nPhuah SY et al (2012) Triple-negative breast cancer and PTEN (phosphatase and tensin homologue) loss are predictors of BRCA1 germline mutations in women with early-onset and familial breast cancer, but not in women with isolated late-onset breast cancer. Breast Cancer Res 14:R142. https:\u002F\u002Fdoi.org\u002F10.1186\u002Fbcr3347\nPuc J et al (2005) Lack of PTEN sequesters CHK1 and initiates genetic instability. Cancer Cell 7:193–204. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.ccr.2005.01.009\nSaldivar JC et al (2017) The essential kinase ATR: ensuring faithful duplication of a challenging genome. Nat Rev Mol Cell Biol 18:622–636. https:\u002F\u002Fdoi.org\u002F10.1038\u002Fnrm.2017.67\nSmits VA et al (2010) Mechanisms of ATR-mediated checkpoint signalling. Front Biosci 15:840–853\nSultana R et al (2013) Targeting XRCC1 deficiency in breast cancer for personalized therapy. Cancer Res 73:1621–1634. https:\u002F\u002Fdoi.org\u002F10.1158\u002F0008-5472.CAN-12-2929\nWorby CA, Dixon JE (2014) Pten. Annu Rev Biochem 83:641–669. https:\u002F\u002Fdoi.org\u002F10.1146\u002Fannurev-biochem-082411-113907\nZhao Q et al (2017) Inhibition of Rad51 sensitizes breast cancer cells with wild-type PTEN to olaparib. Biomed Pharmacother 94:165–168. https:\u002F\u002Fdoi.org\u002F10.1016\u002Fj.biopha.2017.07.090",{"VI":17,"EN":18},"Phosphatase và tenin homolog (PTEN), một chất điều hòa tiêu cực của tín hiệu PI3K, có vai trò trong việc sửa chữa DNA. ATR là một cảm biến chính của tổn thương DNA và căng thẳng sao chép. Chúng tôi đã đánh giá liệu tín hiệu ATR có ý nghĩa lâm sàng và có thể được nhắm mục tiêu bởi tính độc hại tổng hợp trong ung thư vú ba âm tính (TNBC) thiếu PTEN hay không. Mức độ protein của PTEN, ATR và pCHK1Ser345 đã được đánh giá trong 1650 mẫu ung thư vú của người. Việc chặn ATR bằng VE-821 được nghiên cứu trên các dòng tế bào TNBC có PTEN (MDA-MB-231) và thiếu PTEN (BT-549, MDA-MB-468). Các nghiên cứu chức năng bao gồm phân tích biểu hiện sửa chữa DNA, xét nghiệm sự phát triển tế bào MTS, FACS (tiến trình chu kỳ tế bào và sự tích tụ γH2AX) và phân tích flow cytometry FITC-annexin V. PTEN hạt nhân thấp liên quan đến mức độ cao hơn, đa hình thái, phân hóa kém, chỉ số phân bào cao hơn, kích thước khối u lớn hơn, âm tính với ER và tỷ lệ sống ngắn hơn (p \u003C 0.05). Trong các khối u có PTEN hạt nhân thấp, mức ATR cao và\u002Fhoặc mức pCHK1ser345 cao cũng liên quan đến mức độ cao hơn, kích thước khối u lớn hơn và tỷ lệ sống kém (tất cả p \u003C 0.05). VE-821 có độc tính chọn lọc trên các tế bào TNBC thiếu PTEN và dẫn đến sự tích tụ các đứt gãy DNA mạch đôi, dừng chu kỳ tế bào và tăng apoptosis. Tín hiệu ATR ảnh hưởng tiêu cực đến sự sống sót trong các ung thư vú thiếu PTEN. Sự ức chế ATR gây ra cái chết tổng hợp trong các tế bào TNBC thiếu PTEN.","Phosphate and tensin homolog (PTEN), a negative regulator of PI3K signaling, is involved in DNA repair. ATR is a key sensor of DNA damage and replication stress. We evaluated whether ATR signaling has clinical significance and could be targeted by synthetic lethality in PTEN-deficient triple-negative breast cancer (TNBC). PTEN, ATR and pCHK1Ser345 protein level was evaluated in 1650 human breast cancers. ATR blockade by VE-821 was investigated in PTEN-proficient- (MDA-MB-231) and PTEN-deficient (BT-549, MDA-MB-468) TNBC cell lines. Functional studies included DNA repair expression profiling, MTS cell-proliferation assay, FACS (cell cycle progression & γH2AX accumulation) and FITC-annexin V flow cytometry analysis. Low nuclear PTEN was associated with higher grade, pleomorphism, de-differentiation, higher mitotic index, larger tumour size, ER negativity, and shorter survival (p values \u003C 0.05). In tumours with low nuclear PTEN, high ATR and\u002For high pCHK1ser345 level was also linked to higher grade, larger tumour size and poor survival (all p values \u003C 0.05). VE-821 was selectively toxic in PTEN-deficient TNBC cells and resulted in accumulation of double-strand DNA breaks, cell cycle arrest, and increased apoptosis. ATR signalling adversely impact survival in PTEN-deficient breast cancers. ATR inhibition is synthetically lethal in PTEN-deficient TNBC cells.",{"VI":20,"EN":21},"Nhắm mục tiêu vào kinase liên quan đến ataxia telangiectasia-mutated và Rad3 (ATR) trong ung thư vú thiếu PTEN để điều trị cá nhân hóa","Targeting ataxia telangiectasia-mutated- and Rad3-related kinase (ATR) in PTEN-deficient breast cancers for personalized therapy",{"VOID":23},"10.1007\u002Fs10549-018-4683-4",{"VI":25},"PTEN, ATR, ung thư vú ba âm tính, độc hại tổng hợp, sửa chữa DNA","PUBLICATION","VERIFIED","2024-12-31T10:04:06.722+00:00","Auto Verify","PENDING",null,[33],"VI",0,"https:\u002F\u002Flink.springer.com\u002Farticle\u002F10.1007\u002Fs10549-018-4683-4",[37,57,84,97,110,122,135,147,160],{"id":38,"sortIndex":39,"researcher":31,"roles":40,"affiliations":42,"properties":54},"fd283aac-3dca-47a2-8766-7d56fce97754",7,[41],"AUTHOR",[43],{"id":31,"sortIndex":34,"affiliation":44,"properties":31},{"id":45,"createTime":46,"updateTime":47,"relativeEntities":48,"slug":49,"properties":50,"entityType":53,"verifyStatus":30,"verifyTime":31,"verifyNote":31,"syncStatus":30,"languages":31,"translateLanguages":31,"viewCount":34},"037d3ce9-7774-463b-bbe7-f0efa714f97f","2024-02-08T07:50:19.133+00:00","2025-06-11T16:49:33.407+00:00",[],"Department-of-Pathology-Division-of-Cancer-and-Stem-Cells-School-of-Medicine-University-of-Nottingham-Nottingham-University-Hospitals-Nottingham-UK",{"title":51},{"VI":52},"Department of Pathology, Division of Cancer and Stem Cells, School of Medicine, University of Nottingham, Nottingham University Hospitals, Nottingham, UK","AFFILIATION",{"title":55},{"VI":56},"Emad A. Rakha",{"id":58,"sortIndex":59,"researcher":31,"roles":60,"affiliations":61,"properties":81},"a9c00bb7-ffc9-4ce9-8093-707b0b292b6c",8,[41],[62,73],{"id":63,"sortIndex":64,"affiliation":65,"properties":72},"46dd2b7b-0d68-409b-9bf1-1da690522364",1,{"id":66,"createTime":67,"updateTime":67,"relativeEntities":68,"slug":31,"properties":69,"entityType":53,"verifyStatus":30,"verifyTime":31,"verifyNote":31,"syncStatus":30,"languages":31,"translateLanguages":31,"viewCount":34},"11c51a23-53ef-4a78-902b-1113e4d48efd","2024-01-11T08:57:20.972+00:00",[],{"title":70},{"VI":71},"Department of Oncology, Nottingham University Hospitals, Nottingham, UK",{},{"id":31,"sortIndex":34,"affiliation":74,"properties":31},{"id":75,"createTime":76,"updateTime":76,"relativeEntities":77,"slug":31,"properties":78,"entityType":53,"verifyStatus":30,"verifyTime":31,"verifyNote":31,"syncStatus":30,"languages":31,"translateLanguages":31,"viewCount":34},"2d7395cc-189c-44a8-8666-e94cb8ee6b1c","2024-02-08T07:50:19.239+00:00",[],{"title":79},{"VI":80},"Translational Oncology, Division of Cancer and Stem Cells, School of Medicine, University of Nottingham, Nottingham University Hospitals, Nottingham, UK",{"title":82},{"VI":83},"Srinivasan Madhusudan",{"id":85,"sortIndex":86,"researcher":31,"roles":87,"affiliations":88,"properties":94},"f9b2f369-d635-4ed3-9d73-fe3135444b53",6,[41],[89],{"id":31,"sortIndex":34,"affiliation":90,"properties":31},{"id":45,"createTime":46,"updateTime":47,"relativeEntities":91,"slug":49,"properties":92,"entityType":53,"verifyStatus":30,"verifyTime":31,"verifyNote":31,"syncStatus":30,"languages":31,"translateLanguages":31,"viewCount":34},[],{"title":93},{"VI":52},{"title":95},{"VI":96},"Ian O. 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Phys., 22, 235, 10.1080\u002F00107518108231524\nWebster, 1983, Laser Focus, 19, 41\nTravis, 1984, Progr. Anal. Atom. Spectry, 7, 199\nGreen, 1967, Appl. Phys. Letters, 29, 727, 10.1063\u002F1.88918\nTravis, 1982, Anal. Chem., 54, 1006A, 10.1021\u002Fac00246a001\nSchenck, 1978, J. Chem. Phys., 69, 5147, 10.1063\u002F1.436461\nCarlson, 1985, Chem. Phys. Letters, 113, 515, 10.1016\u002F0009-2614(85)85023-5\nWalkup, 1983, Phys. Rev. Letters, 50, 1846, 10.1103\u002FPhysRevLett.50.1846\nKlein, 1983, Chem. Phys. Letters, 100, 475, 10.1016\u002F0009-2614(83)87411-9\nMiyazaki, 1983, Phys. Rev. Letters, 50, 1046, 10.1103\u002FPhysRevLett.50.1046\nSuzuki, 1981, Opt. Commun., 38, 364, 10.1016\u002F0030-4018(81)90067-5\nDelsart, 1981, J. Phys., B14, 3355\nLabastie, 1982, J. Phys., B15, 2595\nSuzuki, 1982, J. Mol. Spectry, 93, 423, 10.1016\u002F0022-2852(82)90178-3\nWebster, 1983, J. Chem. Phys., 78, 646, 10.1063\u002F1.444805\nWebster, 1983, J. Chem. Phys., 78, 2121, 10.1063\u002F1.445074\nVasudev, 1982, J. Chem. Phys., 76, 5267, 10.1063\u002F1.442922\nHannaford, 1981, J. Phys., B14, L661\nHannaford, 1982, Phys. Rev. Letters, 48, 1326, 10.1103\u002FPhysRevLett.48.1326\nHannaford, 1982, Opt. Commun., 41, 427, 10.1016\u002F0030-4018(82)90170-5\nJulien, 1982, J. Phys., B15, 2881\nPalmer, 1980, An atlas of uranium emission intensities in a hollow cathode discharge, LAUR-8251-MS, 10.2172\u002F5095995\nDovichi, 1982, Appl. Opt., 21, 1468, 10.1364\u002FAO.21.001468\nLawler, 1979, Phys. Rev. Letters, 42, 1046, 10.1103\u002FPhysRevLett.42.1046\nLyons, 1981, Opt. Commun., 38, 35, 10.1016\u002F0030-4018(81)90302-3\nPfaff, 1984, Mol. Phys., 52, 541, 10.1080\u002F00268978400101391\nLawler, 1980, Phys. Rev., A22, 1025, 10.1103\u002FPhysRevA.22.1025\nDoughty, 1983, Phys. Rev., A28, 773, 10.1103\u002FPhysRevA.28.773\nDreze, 1982, J. Opt. Soc. Am., 72, 912, 10.1364\u002FJOSA.72.000912\nArimondo, 1984, Opt. Letters, 9, 530, 10.1364\u002FOL.9.000530\nDowney, 1984, Intern. J. Mass Spectrom. Ion. Phys., 61, 337, 10.1016\u002F0168-1176(84)87105-0\nKrupenie, 1972, J. Phys. Chem. Ref. Data, 1, 423, 10.1063\u002F1.3253101\nAlbritton, 1977, J. Mol. Spectry, 67, 157, 10.1016\u002F0022-2852(77)90037-6\nCarrington, 1978, Mol. Phys., 35, 1523, 10.1080\u002F00268977800101141\nBjerre, 1985, Phys. Rev., A31, 167, 10.1103\u002FPhysRevA.31.167\nWest, 1975, J. Chem. Phys., 63, 1237, 10.1063\u002F1.431413\nBen-Amar, 1984, Appl. Opt., 24, 4529, 10.1364\u002FAO.23.004529\nShuker, 1983, J. Appl. Phys., 54, 5685, 10.1063\u002F1.331786\nLindinger, 1975, J. Chem. Phys., 62, 4101, 10.1063\u002F1.430286\nHeicklen, 1976",{"EN":395},"Optogalvanic study of A Ne\u002FO2 glow discharge",{"VOID":397},"10.1016\u002F0009-2614(85)87067-6","https:\u002F\u002Fwww.sciencedirect.com\u002Fscience\u002Farticle\u002Fpii\u002F0009261485870676",[400,415],{"id":401,"sortIndex":64,"researcher":31,"roles":402,"affiliations":403,"properties":412},"deba7137-61d0-4109-81f3-58352eb61e19",[41],[404],{"id":31,"sortIndex":34,"affiliation":405,"properties":31},{"id":406,"createTime":407,"updateTime":407,"relativeEntities":408,"slug":31,"properties":409,"entityType":53,"verifyStatus":30,"verifyTime":31,"verifyNote":31,"syncStatus":30,"languages":31,"translateLanguages":31,"viewCount":34},"1a645d4c-de9c-4e15-8b7b-79e77d91e602","2023-12-01T23:59:59.504+00:00",[],{"title":410},{"VI":411},"Chemistry Division, MS G738, Los Alamos National Laboratory, Los Alamos, NM 87545, USA",{"title":413},{"VI":414},"G.L. 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Nature, 2015, 528: 392–395\nMcHugh KJ, Nguyen TD, Linehan AR, Yang D, Behrens AM, Rose S, Tochka ZL, Tzeng SY, Norman JJ, Anselmo AC, Xu X, Tomasic S, Taylor MA, Lu J, Guarecuco R, Langer R, Jaklenec A. Science, 2017, 357: 1138–1142\nZhang YS, Khademhosseini A. Science, 2017, 356: eaaf3627\nYadavali S, Jeong HH, Lee D, Issadore D. Nat Commun, 2018, 9: 1222\nZhang J, Wei H, Tan J, Qiao W, Guan Y, Zhang J. Sci China Chem, 2018, 61: 328–335\nLei Y, Hamada Y, Li J, Cong L, Wang N, Li Y, Zheng W, Jiang X. J Control Release, 2016, 232: 131–142\nLai WF, Shum HC. Nanoscale, 2016, 8: 517–528\nWu S, Li J, Liang H, Wang L, Chen X, Jin G, Xu X, Yang HH. Sci China Chem, 2017, 60: 628–634\nLee TY, Ku M, Kim B, Lee S, Yang J, Kim SH. Small, 2017, 13: 1700646\nMin NG, Ku M, Yang J, Kim SH. Chem Mater, 2016, 28: 1430–1438\nLawrence MJ, Rees GD. Adv Drug Deliv Rev, 2012, 64: 175–193\nAllen TM, Cullis PR. Adv Drug Deliv Rev, 2013, 65: 36–48\nDash TK, Konkimalla VB. J Control Release, 2012, 158: 15–33\nTiwari G, Tiwari R, Sriwastawa B, Bhati L, Pandey S, Pandey P, Bannerjee SK. Int J Pharm Investig, 2012, 2: 2\nYu Y, Fu F, Shang L, Cheng Y, Gu Z, Zhao Y. Adv Mater, 2017, 29: 1605765\nSong Y, Chan YK, Ma Q, Liu Z, Shum HC. ACS Appl Mater Interfaces, 2015, 7: 13925–13933\nShum HC, Zhao Y, Kim SH, Weitz DA. Angew Chem, 2011, 123: 1686–1689\nTang MYH, Shum HC. Lab Chip, 2016, 16: 4359–4365\nSim JY, Lee GH, Kim SH. Small, 2015, 11: 4938–4945\nKim SH, Weitz DA. Angew Chem, 2011, 123: 8890–8893\nShang L, Cheng Y, Zhao Y. Chem Rev, 2017, 117: 7964–8040\nDing W, Li Y, Xia H, Wang D, Tao X. ACS Nano, 2014, 8: 11206–11213\nMao Z, Xu H, Wang D. Adv Funct Mater, 2010, 20: 1053–1074\nZhang YS, Arneri A, Bersini S, Shin SR, Zhu K, Goli-Malekabadi Z, Aleman J, Colosi C, Busignani F, Dell’Erba V, Bishop C, Shupe T, Demarchi D, Moretti M, Rasponi M, Dokmeci MR, Atala A, Khademhosseini A. Biomaterials, 2016, 110: 45–59\nZhang YS, Khademhosseini A. Nanomedicine, 2015, 10: 685–688\nWang J, Zou M, Sun L, Cheng Y, Shang L, Fu F, Zhao Y. Sci China Mater, 2017, 60: 857–865\nZhao Y, Cheng Y, Shang L, Wang J, Xie Z, Gu Z. Small, 2015, 11: 151–174\nZhang L, Feng Q, Wang J, Sun J, Shi X, Jiang X. Angew Chem Int Ed, 2015, 54: 3952–3956\nFeng Q, Liu J, Li X, Chen Q, Sun J, Shi X, Ding B, Yu H, Li Y, Jiang X. Small, 2017, 13: 1603109\nWang J, Sun L, Zou M, Gao W, Liu C, Shang L, Gu Z, Zhao Y. Sci Adv, 2017, 3: e1700004\nKeidel R, Ghavami A, Lugo DM, Lotze G, Virtanen O, Beumers P, Pedersen JS, Bardow A, Winkler RG, Richtering W. Sci Adv, 2018, 4: eaao7086\nKim B, Soo Lee H, Kim J, Kim SH. Chem Commun, 2013, 49: 1865–1867\nGholampour N, Chaemchuen S, Hu ZY, Mousavi B, Van Tendeloo G, Verpoort F. Chem Eng J, 2017, 322: 702–709\nLeong TSH, Martin GJO, Ashokkumar M. Ultrasons Sonochem, 2017, 35: 605–614\nGalvão KCS, Vicente AA, Sobral PJA. Food Bioprocess Technol, 2018, 11: 355–367\nDowding PJ, Atkin R, Vincent B, Bouillot P. Langmuir, 2005, 21: 5278–5284\nHong Y, Gao C, Shi Y, Shen J. Polym Adv Technol, 2005, 16: 622–627\nWang J, Chen W, Sun J, Liu C, Yin Q, Zhang L, Xianyu Y, Shi X, Hu G, Jiang X. Lab Chip, 2014, 14: 1673–1677\nYu Y, Shang L, Gao W, Zhao Z, Wang H, Zhao Y. Angew Chem Int Ed, 2017, 56: 12127–12131\nShang L, Fu F, Cheng Y, Wang H, Liu Y, Zhao Y, Gu Z. J Am Chem Soc, 2015, 137: 15533–15539\nLiu Y, Huang Q, Wang J, Fu F, Ren J, Zhao Y. Sci Bull, 2017, 62: 1283–1290\nWang R, Zhou L, Wang W, Li X, Zhang F. Nat Commun, 2017, 8: 14702\nLi Y, Yan D, Fu F, Liu Y, Zhang B, Wang J, Shang L, Gu Z, Zhao Y. Sci China Mater, 2017, 60: 543–553\nDeng W, Li J, Yao P, He F, Huang C. Macromol Biosci, 2010, 10: 1224–1234\nFeng Q, Sun J, Jiang X. Nanoscale, 2016, 8: 12430–12443\nChen H, Ma Y, Wang X, Wu X, Zha Z. RSC Adv, 2017, 7: 248–255\nHuang C, Yang G, Ha Q, Meng J, Wang S. Adv Mater, 2015, 27: 310–313",{"EN":486},"Microcarriers have attracted increasing interests in drug delivery. In order to develop this technique, it is prone to focus on the generation of functional particles through using simple approaches and novel but accessible materials. Here, inspired by the formation mechanism of tofu that through the mixing of soymilk and brine for cross-linking soybean proteins, we present novel soybean protein microcarriers by using microfluidic generation approach for drug delivery. Since the soybean protein droplets are generated by microfluidic emulsification method, the tofu microparticles present highly monodisperse and homogeneous morphologies. Because of the excellent biocompatibility of the soybean protein and the interconnected porous structures throughout the whole microparticles after freeze-drying, various kinds of drugs and active molecules could be absorbed and loaded in the microcarriers, which makes them versatile for drug delivery. It can be anticipated that the microfluidic-generated tofu microcarriers will have great potential in the biomedical field.",{"EN":488},"Tofu-inspired microcarriers from droplet microfluidics for drug 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China",{"title":508},{"VI":509},"Yuxiao Liu",{"id":511,"sortIndex":149,"researcher":31,"roles":512,"affiliations":513,"properties":519},"876fe8a0-be4d-43bb-902e-99155bd57e8b",[41],[514],{"id":31,"sortIndex":34,"affiliation":515,"properties":31},{"id":499,"createTime":500,"updateTime":501,"relativeEntities":516,"slug":503,"properties":517,"entityType":53,"verifyStatus":30,"verifyTime":31,"verifyNote":31,"syncStatus":30,"languages":31,"translateLanguages":31,"viewCount":34},[],{"title":518},{"VI":506},{"title":520},{"VI":521},"Yuanjin 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